Court Opinion

ID: 9562132
Source: CourtListenerOpinion
Date Created: 2023-08-21 18:22:12.44668+00
Date Added: 2024-06-11T09:17:13.255380
License: Public Domain

Rosellini, J.
(dissenting) — In my opinion, this case should never have been submitted to the jury, since there was neither medical nor non-medical evidence on which the jury could reasonably base a finding that the plaintiff’s disability was caused by the negligence of either of the defendants. However, it did go to the jury and the jury returned the only reasonable verdict supported by the evidence — a verdict in favor of both defendants. In order to set that verdict aside and order a new trial, the majority has found it necessary to introduce more than one “innovation” in the law, and that without any discussion of the reasons for the existing rules of law or what it considers the justification for changing them, other than the broad assumption that medical testimony is hard to get.
It is not only the law but the evidence in this case which is being subjected to considerable revision in order to reach the desired result. In discussing the testimony of Dr. Mal-den, the plaintiff’s neurologist and expert witness, the majority opinion effectively disposes of the doctor’s testimony that an abnormal reaction to the proper drug, properly administered, was a possible cause of the plaintiff’s condition. It concedes that the doctor did testify that such a reaction was a possible cause, but it fails to observe that his testimony was uncontradicted and unimpeached, and that the plaintiff acknowledges that hyperergy remains “suspect” as a possible cause. Instead, the majority resorts to a report by two Philadelphia doctors on a study made in the University of Pennsylvania Hospital of some 10,000 patients who received spinal anesthesia and did not develop cauda equina syndrome. From the fact that they found no instance of the syndrome in their study, these doctors volunteered the opinion that such syndromes must be due to negligence (if indeed they ever occur!).
Giving this hearsay opinion the status of evidence in the *492case, even though it was not introduced therein, the majority says the jury was entitled to disregard Dr. Malden’s testimony concerning the possibility of hyperergic reaction and treat the other possibilities as the only possible causes of the plaintiff’s condition. Thus by this bootstrap technique does the majority render the doctrine of res ipsa loquitur applicable.
I say that not only were the opinions of Doctors Vandam and Dripps not introduced at the trial and not before the jury for its consideration, but their opinions do not represent any prevailing medical view insofar as I have been able to ascertain. Since the majority can resort to independent medical research to fortify its view that the judgment should be reversed, I believe that I can legitimately resort to the products of such research to fortify my position that the verdict of the jury and the judgment entered thereon should be sustained.
My research, as well as the evidence in this case, shows that the term “cauda equina syndrome” describes a group of symptoms or effects which are of the type suffered by the plaintiff. It is ridiculous to say that they do not exist. My research also shows that there is no agreement among doctors as to the cause, although there seems to be a general agreement that the cause is unknown. See my footnote 3, infra.
But before approaching that aspect of the matter, I would like to review the testimony of Dr. Malden in a little more detail.
The plaintiff’s attorney posed to Dr. Malden a hypothetical question which embodied the plaintiff’s theory of what her evidence had demonstrated at that point in the proceedings. The doctor was asked to assume that the facts contained in the hypothetical were true and to state whether he had an opinion as to the cause of the plaintiff’s condition. He replied that he had. When asked to give that opinion, he answered:
My opinion is that it must have been the effect of . . . the substance in the ampules on the spinal cord *493and on the nerve roots which has resulted in findings in Mrs. Douglas’ case.
In amplifying this, the doctor said:
In describing, in thinking about a patient which I faced, and in trying to decide what was the cause of her condition, I cannot look into a crystal ball. I must follow a pattern of medical thinking, which is the accepted medical procedure. In doing this I must logically follow a number of steps, and in following these steps logically, as a physician and in the course of my proper medical practice, I can arrive at a conclusion, and it is because of following these correct logical steps that I, on the basis of the hypothetical question and in relation to it, would say that medically I have to consider that this was a substance in the ampule, whether — and there is a number of further possibilities what could be — well, the substance in the ampule could have been an entirely foreign substance. The other correct medical thinking would be that the substance which was in the ampule was the correct substance, but the quantity was wrong.
. . . Then I would have to medically think that the substance which was in the ampule was the correct substance, but was contaminated. There is one other possibility. That is, that the substance which was in the ampule was the correct substance in correct quantity, but it is still this substance that in this patient has caused the particular result.
On cross-examination, he stated rather succinctly that there were in his mind three possibilities: one, that a contaminant somehow got into the ampule or into the syringe through which it was administered; two, that the substance in the ampule was not procaine hydrochloride at all but some foreign substance; and three, that the plaintiff simply had an unusual reaction to the drug.
The majority seems to feel that the doctor did not accord much dignity to this latter possibility. However, far from discounting this latter possibility, Dr. Malden took some pains to explain the difference between idiosyncratic reaction or allergic reaction, which he had indeed eliminated as a possible cause, and abnormal reaction. He said:
*494An idiosyncratic reaction is an entirely different reaction, not only in quantity, but in quality. If I give an aspirin to somebody, I expect them — let’s take a patient who has a temperature. I give them aspirin to lower the temperature. If I give an aspirin to a normal patient, I expect the temperature to come down for four hours, and again, go up again. Now, let’s take another patient. I give him a tablet of aspirin, and this patient drops dead. That is an idiosyncratic reaction. It did not affect his temperature at all, but he just dropped dead, or he had a bronchial spasm. Now, take the same aspirin; I give a normal dose to the patient, and the patient’s temperature, instead of dropping down five degrees and five-tenths of a degree and staying down for four hours, drops down precipitously and stays down for three days. That is an abnormal reaction to a normal dose; but the actual quality of the response is normal. The temperature dropped and stayed down. It dropped too much, it stayed down too long. This is called hyperergy.
Dr. Malden agreed that such a possibility was consistent with the facts, inasmuch as the plaintiff did respond to the anesthetic in the expected manner — that is, she was anesthetized while the operation was performed. Also, consistent with this theory of a possible cause is the fact that the plaintiff’s symptoms are anesthetic in nature. It is as though the anesthetic never wore off completely.
At this point I would like to inject an observation of my own concerning the possible causes. It seems to me that Dr. Malden’s second possibility — that the substance in the ampule was not procaine hydrochloride at all — should reasonably be discounted, inasmuch as the desired and expected results of anesthesia were obtained. It would be strange indeed if some foreign substance, say epsom salts or sugar (the substance was in the form of crystals), should anesthetize the patient and produce the very beneficial relaxation of the abdominal muscles for which procaine hydrochloride and its kindred anesthetic drugs are noted. I am inclined to believe that, had Dr. Malden given this possibility (the possibility of a foreign substance rather than an anesthetic in the ampule) the same critical consideration which he apparently gave to the possibility that the *495needle was injected in the wrong place, he would have concluded that it was a highly unlikely one. And, as a matter of fact, the trial court concluded and so instructed the jury that there was no competent evidence that the drug in the ampule was not pure procaine hydrochloride; and no error has been assigned to the giving of that instruction.
If that possibility is ehminated, it leaves only two possibilities: (1) that a contaminant somehow got into the ampule or into the syringe, or (2) that the plaintiff suffered an abnormal reaction. (Dr. Malden did not think an excessive amount of anesthetic was given and there was no evidence that the amount given was excessive.) As I read the majority opinion, it has now come to the conclusion that the lay juror is capable of diagnosing the cause of a condition which develops postoperatively and that a lay judge also may engage in the diagnostic procedure. I do not know whether, under the new approach, one is disqualified proportionately to the amount of medical knowledge which he is able to obtain; but proceeding on the assumption that a little medical knowledge does not automically disqualify the diagnostician, I have taken the liberty of conducting a less-than-exhaustive but nevertheless enlightening research on the subject of neurological disorders following spinal anesthesia. It was my aim not only to ascertain how universal was the opinion of Doctors Vandam and Dripps, but also to satisfy myself that the plaintiff in this case was the victim of the well-known and much-publicized “conspiracy of silence” on the part of the medical profession, and that this was the reason she was unable to prove that her condition was caused by negligence on the part of her doctor and/or the manufacturer of the drug. (Possible negligence on the part of the hospital must be discounted, inasmuch as the hospital, which supposedly sterilized the ampules and the instruments, is not a defendant!)
I will refer further to the fruits of this research later in this dissent; here I simply wish to state that I found that medical knowledge is not passed about from doctor to doctor by word of mouth (in order to conceal it from the *496layman) but is written down and published copiously.2 The medical profession has not ignored the cauda equina syndrome, nor has it ignored the fact that other neurological conditions sometimes manifest themselves after the administering of anesthetics. On the contrary, extensive research has been done and experiments have been performed, and much has been written. Still, there is no agreement as to the probable causes of the syndrome, and it is true that, within the medical profession, the exact cause is not known.3 Possible causes have been suggested and some have been eliminated. One of the possibilities suggested is contaminants in the ampule or in the equipment with which the anesthetic is administered. However, doubt has been expressed that such a small quantity of a contaminant could produce such widespread damage.4
Since Dr. Malden stated on cross-examination that, in his opinion, the manner in which Dr. Bussabarger performed the surgery and administered the spinal was within the *497standard of care in the area, it would appear that he did not believe that the plaintiff’s condition was the result of negligence on Dr. Bussabarger’s part.
In light of these considerations, if I were called upon to guess whether Dr. Malden, in his own mind, had eliminated the possibility of an abnormal reaction as the cause of the plaintiff’s condition, I would guess that he had not done so, but rather that he favored it as the most probable explanation. However, Dr. Malden was not asked to state his preference among the possibilities, although he said that he had one.
I have discussed this question of Dr. Malden’s testimony first and at some length because it is crucial to the disposition which the majority makes of this case. If the possibility of an abnormal reaction is not eliminated, how can it conceivably be said that this is a proper case for the application of res ipsa loquitur? That latin expression, of course, simply means “[T]he thing speaks for itself.” If the “thing” says that it could have been caused by negligence or it could have happened without negligence, it certainly does not speak “negligence.”
I will not further extend this already lengthy dissent by discussing the relative merits of those recent cases which have held that a jury instruction on the “doctrine” of res ipsa loquitur was proper and Ball v. Mudge, 64 Wn.2d 247, 391 P.2d 201 (1964), holding that a jury instruction is not necessary. I will assume the latter case has been overruled, albeit sub silentio.
Nevertheless, I believe that the instruction was properly refused in this case for a number of reasons. In order to make these clear, it is necessary to state the conditions which must be shown to exist before the doctrine comes into play. First, the accident or occurrence must be of a kind which does not happen ordinarily in the absence of someone’s negligence. Second, the agency or instrumentality producing the injury must be within the exclusive control of the defendant. Third, the plaintiff must not have contributed to the injury. Horner v. Northern Pac. Beneficial Ass’n Hosps., Inc., 62 Wn.2d 351, 382 P.2d 518 (1963).
*498As this court said in that case, negligence can be inferred (1) when the act causing the injury is so palpably negligent that it may be inferred as a matter of law, i.e., leaving foreign objects, sponges, scissors, etc., in the body, or amputation of a wrong member; (2) when the general experience and observation of mankind teaches that the result would not be expected without negligence; and (3) when proof by experts in an esoteric field creates an inference that negligence caused the injuries.
As I have previously pointed out, the first condition can be found to be present in this case only if the testimony of Dr. Malden is mutilated. He said the cause is not known and, in the present state of medical knowledge, cannot be known. The medical publications which I have examined bear him out in this.5
The record is devoid of evidence that any negligence on the part of either the doctor or the defendant drug company caused the plaintiff’s condition. Much less is there evidence of a “palpably negligent” act causing the injury. Likewise, it can hardly be said that the common experience and observation of mankind teaches that the result would not be expected without negligence. If such is the case, the “common experience of mankind” teaches more than years of experimentation and research have been able to teach the medical profession.
Finally, the medical evidence does not raise an inference of negligence if Dr. Malden’s testimony is allowed to stand as it was given, uncontradicted and unimpeached. Only by introducing medical opinions from outside the record, which in themselves are untrustworthy because they are based on a comparatively small sample and are illogical in their assumptions, and which are not shared by other equally competent doctors, can such an inference be raised. To use such a device to supply missing evidence constitutes, in my opinion, a shocking denial of due process.
*499I agree with the majority that there was no evidence of negligence on the part of the defendant drug company. There was also, of course, no evidence of negligence on the part of the defendant Bussabarger. But the testimony on which they rely to establish the applicability of res ipsa loquitur, that of Dr. Malden, did not assign any greater degree of probability to one possible cause than to another. As far as Dr. Malden’s testimony reveals, the injury could as well have been caused by a foreign substance in the ampule as by an overdose or improper injection by the defendant doctor, and might equally as well have been caused by hyperergy. I therefore do not believe the evidence of probable causation by the defendant Bussabarger is sufficient to render the doctrine applicable.
But assuming that, as the majority seems to reason, hy-perergy can be eliminated on the ground that such a cause would deprive the plaintiff of a right of recovery against someone, but that negligence of the defendant drug company is not eliminated by the fact that there was no evidence to support it, I do not think the case cited by the majority supports its holding. It says that even though there is specific circumstantial evidence of negligence on the part of one defendant, recovery may be had against another under the doctrine of res ipsa loquitur.
Of course, in this case, there was no specific evidence of negligence of the drug company. The circumstantial evidence was the same as the circumstantial evidence against the doctor (and against hyperergy) — that is, the happening of the injury. If that evidence points to the negligence of one, it cannot at the same time speak of the negligence of the other.6 The case cited by the majority, McCarty v. *500Hosang, 154 F. Supp. 852 (W.D. Mo. 1957), involved a three-car collision. The car in front of the plaintiff’s car dropped a piece of luggage, causing the plaintiff to have to stop suddenly. His car was rammed from the rear by a third car. He invoked specific negligence of the driver of that automobile. The court held that this did not preclude him from invoking res ipsa loquitur against the driver of the car in front of him. In that case, there were two concurring independent causes of the accident. The plaintiff was not relying on the same circumstance to prove the negligence of each, as is being done in this case. Consequently that case is not in point and does not support the majority position.
Even the plaintiff acknowledges that the case of Seneris v. Haas, 45 Cal. 2d 811, 291 P.2d 915, 53 A.L.R.2d 124 (1955), on which she relies as an analogous case (and indeed it is on the facts, and supports the view of the majority in this case, although they do not see fit to cite it), goes to lengths which this court has not indicated in the past that it would go in allowing the jury to infer negligence of multiple defendants. Anticipating this court might perceive the il-logic of that decision, the plaintiff makes an argument in her brief that her evidence tended to eliminate detergents, contaminants, and foreign materials (except alcohol) as causes of her condition, as well as bacteria, preexisting disease, fracture, and idiosyncrasy; that the trial court concluded as a matter of law that the anesthetic was “pure, undiluted, uncontaminated procaine hydrochloride”; and that therefore the jury could find the cause of the plaintiff’s condition was negligence of the doctor, supposedly either in administering the injection in the wrong place or in administering too much anesthetic.
In other words, the instruction which the plaintiff contends should have been given would have advised the jury *501that it could infer negligence of the doctor only if it found that the plaintiff’s condition was not caused by negligence of the drug company or by some factor for which the doctor would not be responsible. And even in this contention, the plaintiff has not suggested that she presented evidence eliminating hyperergy as a causative factor. In developing her argument that the doctrine of res ipsa loquitur is applicable, she simply suggests that the jury could reject that possibility. The majority has gone further and eliminated this possibility itself, relieving the jury of the burden.
There is, of course, no suggestion that the plaintiff’s negligence caused her condition. This freedom from contributory fault is the only condition of res ipsa loquitur which is present here, but it is evidently this one fact which is directing the outcome of the case.7 That is to say, sympathy for the plaintiff has caused the court to abandon the rules of law which are calculated to produce a result that accords with accepted concepts of justice. Ordinarily, we hold to the theory that, because one person has been injured, another should not have to pay for that loss unless he is responsible. And, ordinarily, we say the burden is on the plaintiff to prove that it was the defendant’s act which produced the harm. The rationale of res ipsa loquitur is that the defendant is in a better position to explain how the accident happened and should be required to do so because the probabilities are that his negligence did in fact cause the injury. It is not the mere fact that he is in a better position to get at the evidence. That may be true in many cases in which it is not contended that the doctrine of res ipsa has any application.
It has been suggested that, in medical malpractice cases, the plaintiff is not at such a great disadvantage as he once was, since he has at his disposal discovery procedures *502which will enable him to ascertain the pertinent facts.8 In any event, the mere fact that the defendant has greater access to the facts9 does not in itself raise an implication of negligence. It does not do so logically, any more than it does legally, in my view. This case illustrates that point. The plaintiff developed a syndrome that has been known to occur even though the anesthetic is administered under the most careful procedures; and which, in spite of earnest efforts to ascertain its cause so that it can be avoided if possible and its treatment can be better effected (this is the type of consideration which inspires medical research ordinarily, not the avoidance of lawsuits), continues to mystify those in the best position to understand it.
Not content to cast the burden of proving the cause of the injury on the defendants10 (in spite of the fact that the plaintiff’s own evidence established that this is an *503occurrence which may very well result without negligence), the majority has also decided that the jury should be allowed to speculate on the cause of the injury. Medical science does not know the cause but perhaps the jury can filch it from the non-medical testimony.
The instruction given by the court that malpractice of the doctor must be proven by medical testimony was proper under the rulings of this court. In Teig v. St. John’s Hosp., 63 Wn.2d 369, 387 P.2d 527 (1963) and Richison v. Nunn, 57 Wn.2d 1, 340 P.2d 793 (1959), we said that negligence of a doctor by reason of his departure from the recognized standard of practice must be established by medical testimony, unless his negligence is so apparent that even a layman would have no difficulty in recognizing it. Other cases so holding are Woods v. Pommerening, 44 Wn.2d 867, 271 P.2d 705, 45 A.L.R.2d 1267 (1954); Cochran v. Harrison Memorial Hosp., 42 Wn.2d 264, 254 P.2d 752 (1953); and Fritz v. Horsfall, 24 Wn.2d 14, 163 P.2d 148 (1945). The requirement in cases such as this, that negligence of the physician must be shown by medical testimony, is supported by the overwhelming weight of authority. See annotation, 81 A.L.R.2d 597. Since this was not a case in which the negligence of the defendant was so apparent that a layman would recognize it, the trial court, in my opinion, properly gave the instruction that the negligence of the defendant doctor must be established by medical testimony.
This is obviously so because the ordinary layman does not know and has no way of knowing what acceptable medical procedures are. This is an area for experts and of course they must be medical experts.
The evidence which the majority feels that the jury had a right to consider was all medical evidence. It was all testimony of doctors or evidence furnished by them — the fact that, for a stomach operation, insertion of the needle above the second lumbar vertebra is not considered proper procedure; the fact that, if the needle struck the spinal cord, there would be an immediate reaction from the patient; the fact that the plaintiff’s condition was diagnosed *504as cauda equina syndrome and paraplegia, and the amount of anesthetic injected (testified to by Dr. Bussabarger and shown on his records introduced in evidence). I think the majority is not really concerned that the jury was not permitted to consider non-medical evidence; but obviously the concern of the majority is that the jury may have felt inhibited by the instruction if it was inclined to arrive at its own diagnosis of the cause of the syndrome.
While there is some discussion of standard of care in the majority’s disposition of the question whether a jury verdict must rest on medical testimony in a malpractice case when the negligence, if any, is not of a type which even a layman would recognize, it is apparent that it is primarily the question of causation which it feels should be submitted to the jury, whether or not there has been medical testimony tending to establish the probable cause. There was, of course, in this case, no testimony of probabilities but only testimony as to possibilities. This court has consistently held that, in a case of this kind where the layman is not in a position to diagnose the cause of a given condition, there must be expert testimony to establish the cause; and the testimony must be at least that the act relied upon probably caused the condition, rather than it possibly caused it.
In a recent opinion written by Weaver, J., Glazer v. Adams, 64 Wn.2d 144, 147-48, 391 P.2d 195 (1965), I find this exposition:
In Bland v. King Cy., 55 Wn. (2d) 902, 905, 342 P. (2d) 599 (1959), this court said:
“The test to be applied in considering the sufficiency of the evidence to establish cause of death was announced by this court in Seattle-Tacoma Shipbuilding Co. v. Department of Labor & Industries, 26 Wn. (2d) 233, 241, 173 P. (2d) 786 (1946), as follows:
“ ‘The general rule, from which this court has never deviated, is stated in 135 A. L. R. 517, as follows: ’
“ ‘ “It appears to be well settled that medical testimony as to the possibility of a causal relation between a given accident or injury and the subsequent death or impaired physical or mental condition of the person injured is not sufficient, standing alone, to establish such relation. By *505testimony as to possibility is meant testimony in which the witness asserts that the accident or injury ‘might have,’ ‘may have,’ or ‘could have’ caused, or ‘possibly did’ cause the subsequent physical condition or death or that a given physical condition (or death) ‘might have,’ ‘may have,’ or ‘could have’ resulted or ‘possibly did’ result from a previous accident or injury — testimony, that is, which is confined to words indicating the possibility or chance of the existence of the causal relation in question and does not include words indicating the probability or likelihood of its existence.”
“ ‘In Anton v. Chicago, M. & St. P. R. Co., 92 Wash. 305, 159 Pac. 115, this court expressed its views with respect to such evidence, in the following language:
“ ‘ “Taking the opinion of the witness [a medical man] for the appellant, as quoted above, at its full worth, we think it is no more than a statement of a possibility or possibly a probability, more or less remote, that the tuberculosis is a result of the injury. This is not enough. The law demands that verdicts rest upon testimony and not upon conjecture and speculation. There must be some proofs connecting the consequence with the cause relied upon. The testimony, whether direct or circumstantial, must reasonably exclude every hypothesis other than the one relied on.” ’ ”
In Orcutt v. Spokane Cy., 58 Wn. (2d) 846, 364 P. (2d) 1102 (1961), we pointed out that the
“. . . medical testimony must at least be that the injury ‘probably’ or ‘more likely than not’ caused the subsequent condition, rather than that the accident or injury ‘might have,’ ‘could have,’ or ‘possibly did’ cause the subsequent condition. . . .”
A corollary of this rule is the equally well-established rule (which would ordinarily control the disposition of this case) that a verdict cannot be founded on mere theory or speculation, and if there is nothing more tangible to proceed upon than two or more conjectural theories, under one or more of which the defendant would be liable, and under one or more of which there would be no liability upon him, the jury will not be allowed to resort to speculation to determine the facts. Schmidt v. Pioneer United Dairies, 60 Wn.2d 271, 373 P.2d 764 (1962); Mason v. Turner, 48 Wn.2d *506145, 291 P.2d 1023 (1956); Arnold v. Sanstol, 43 Wn.2d 94, 260 P.2d 327 (1953). There must be evidence which would at least support a finding that there is a greater probability that the injury occurred in such a way as to fasten liability upon the person charged therewith, than there is that it happened in a way for which the person charged would not be liable. Kilmer v. Bean, 48 Wn.2d 848, 296 P.2d 992 (1956); Mason v. Turner, supra.
Those rules will apparently apply henceforth in all cases except medical malpractice cases. In medical malpractice cases, again because of the “conspiracy of silence” I take it, medical testimony of causation is not necessary, and the jury may put together two and two, or three and four, or however many bits of evidence it can add up in a given case, and arrive at the cause of a condition or disease — and this in the face of uncontradicted medical testimony that the cause, in the present state of medical knowledge, cannot be ascertained. In doing so, it may choose among possibilities, under one or more of which the defendant is not responsible for the plaintiff’s injury.
The facts of this case as they are presented in the record demonstrate the absurdity of such a rule. As I have indicated earlier, Dr. Malden, the plaintiff’s neurologist and expert witness, testified that he considered the possibility that the plaintiff’s condition was caused by insertion of the needle at a point between T-12 and L-l in drawing (1), which is borrowed from the case of Seneris v. Haas, 45 Cal. 2d 811, 291 P.2d 915 (1955), (see appendix) rather than between L-3 and L-4, the normal and customary place for the injection where the surgery is to be performed on the abdomen, and that it penetrated the spinal cord. (There would be no harm in injecting between T-12 and L-l if the spinal cord was not struck, but it is not done unless an unusually high level of anesthesia is required because of the danger of injuring the cord and because no useful purpose is served by an injection higher than is needed for the particular surgery.)
He stated, however, that he had definitely ruled out that possibility for a number of reasons. The first was that the *507nerve roots which were involved in the plaintiff’s difficulties (her difficulties were with her lower abdomen and her legs and feet) were located in the third and fourth lumbar vertebrae level. If the needle had been inserted at a higher level, he said, one would expect nerve roots at a higher level to be affected. That is to say, she would have been paralyzed or anesthetized at a higher level of her body. The anesthetic would not be expected to bypass certain nerves on its way down the subarachnoid space and only affect those below L-3 and L-4.
Dr. Malden also found no evidence of needle damage to the spinal cord. (This does not mean necessarily that he found no damage to the cord, but simply he found no evidence of needle trauma.)11
Another reason which Dr. Malden gave for ruling out needle trauma as a probable cause of the plaintiff’s disease was that, when the spinal cord is damaged, there is never any improvement, whereas the plaintiff’s condition had improved somewhat which indicated that only nerve roots were involved. I could not ascertain from the doctor’s testimony on this point whether he meant that there is never improvement where there is puncture damage, or that there is never improvement if there is any kind of damage.
Dr. Malden also stated that the fact that, when the needle was inserted, spinal fluid was withdrawn and was mixed with the procaine crystals to form a solution used for the injection of the anesthetic, indicated very strongly that the needle was injected in the proper place (the sub-arachnoid chamber — see drawing (2) ) and not in the spinal cord. This makes infinite sense to me. It would seem, would it not, that, if the needle were injected in the cord and the attempt was made to draw out some fluid in the customary *508way, what the doctor would get in the syringe would be gray matter which is what the cord is made-up of, rather than spinal fluid which can only be found outside the cord.
Also, Dr. Malden found no external evidence that an injection had been made at a point higher than L-3. He found some marks between L-3 and L-4 which could be attributed to an injection at that place.
In sum, Dr. Malden stated that he considered it “highly unlikely” that the plaintiff’s condition was caused by the injection of the needle in a improper place. Now what evidence is there in opposition to this?
Dr. Durkin, who examined the plaintiff, concluded from her reflexes and symptoms that she had suffered some cord damage as well as damage to the nerve roots. He was not asked by the plaintiff’s attorney to give his opinion of a possible cause of it, and an attempt on the part of defense counsel to elicit such an opinion was thwarted by the objection that it was beyond the scope of the direct examination, an objection which was sustained by the court. I note that the majority does not suggest that the testimony of Dr. Durkin would support an inference that the needle was injected in the wrong place. And indeed such an inference would be contrary to the evidence. Dr. Malden, who ruled out needle trauma as a possible cause of the plaintiff’s condition, stated that there was a possibility of cord injury. The injury, of course, could just as well be caused by the anesthetic or other substance injected, just as the injury to the nerve roots is caused by it. The anesthetic moves about in the subarachnoid space after it is injected and it is not surprising that it comes in contact with the spinal cord.
The majority has set forth the evidence which it believes is adequate to support a finding that the needle was injected between T-12 and L-l. It is as follows:
In the instant case, defendant-Dr. Bussabarger testified that when a patient is undergoing a stomach operation, injection of spinal anesthesia above the second lumbar vertebra is not proper procedure. Plaintiff produced further expert testimony to show that if the needle insertion were too high, particularly if it hit the spinal cord, there *509likely would be a spasm or some immediate reaction from the patient. Plaintiff testified that she felt a sharp pain low in her back on the entry of the spinal needle, and complained of the hurt before losing consciousness. In addition, one of plaintiff’s witnesses, Dr. Durkin, testified that he found some evidence of damage to her spinal cord. . . .
. . . Various hospital records admitted into evidence at trial showed the diagnosis of plaintiff’s post-operative condition to be cord damage, which could well indicate improperly high injection of the spinal anesthetic.
It can certainly be conceded for purposes of this case that injection of the needle above the second lumbar vertebra would involve an unnecessary risk. The question is, was the needle so injected. The majority says that such an injection would cause “some immediate reaction,” and that the plaintiff testified she felt a sharp pain low in her back on the entry of the spinal needle. Now, to say the least, it escapes my perception how this proves anything but that the needle was inserted exactly where Dr. Bussabarger said he inserted it (low on the back between L-3 and L-4, not in the middle of the back between T-12 and L-l). There was certainly no evidence that the patient feels no pain if the needle is inserted in the proper place. The evidence was to the contrary.12 There was also no testimony or other evidence that pain in the low back on insertion of the needle means that the needle has been inserted in the wrong place.
What is there left on which the jury is to base a finding that needle trauma or injection of the anesthetic into the *510spinal cord13 caused the plaintiff’s condition? She was diagnosed at one time as having cauda equina syndrome and paraplegia. The plaintiff’s symptoms are certainly those of the cauda equina syndrome. There is no question about that. It is conceded on all sides. It produces varying degrees of paraplegia (paralysis of trunk and lower extremities). But what does this prove about its cause? Precisely nothing. That is what this lawsuit is about — what caused the plaintiff’s cauda equina syndrome. The record says that no one knows, but most probably it was not negligence in injecting the needle in the wrong place. It may have been contaminants in the syringe or in the anesthetic; and it may well have been simply the anesthetic itself producing, in the plaintiff’s particular body, an effect which is rarely produced by the anesthetic but which is sometimes produced and the mechanism of which is not understood by medical science. It may be the effect of the anesthetic on the blood vessels in the anesthetized area (see note 11, supra).
It may possibly be that the concentration of the drug caused it, even though the concentration used was within what are ordinarily considered safe limits. Here again, the majority would permit the jury to speculate on whether too much anesthetic was given. There is not one scintilla of evidence that too much anesthetic was given. The evidence was that the amount varies according to the weight of the patient, the length of time required by the operation, the degree of muscle relaxation required, and the way the patient’s body responds to the drug.14
*511My research reveals that the total amount of anesthetic given is not considered to be a factor, but the total amount given at any one injection. As common knowledge would affirm, I think, the effects of an anesthetic do wear off. Now the continuous spinal anesthetic, which Dr. Bussabarger chose to use in the plaintiff’s case, is recommended for use with the old and the very young because smaller amounts are given at one time than would be given in a single injection and thus it is easier on the patient.15
So it would appear that Dr. Bussabarger chose a method of anesthesia that is considered advantageous for the most vulnerable patients. He administered, in all, 700 milligrams of procaine over a period of approximately 3 hours and 5 minutes. There was no evidence that this was an excessive amount or that it was administered in an excessively concentrated solution. For aught that appears in the record, if the doctor had used less anesthetic the patient would not have remained anesthetized throughout the operation and then we would have had a different lawsuit before us.
The only evidence, relied upon by the plaintiff to show that the anesthetic was administered in an excessive amount, is a brochure which accompanied the anesthetic and gave a recommended dosage of 150 to 200 milligrams (300 only in rare instances). In answer to an interrogatory of the plaintiff, the defendant drug company said that this recommended dosage was for single injections and not for continuous spinal. This accords with what I have been able to learn from medical treatises on the subject.16 The largest *512single injection given to the plaintiff was the initial injection of 200 milligrams.
The majority does not state what evidence the jury was entitled to believe if it chose to decide that the plaintiff’s condition was attributable to the amount of anesthetic administered to her; and this is understandable inasmuch as the record is devoid of any evidence that would support such a finding.
The plaintiff, in evaluating Dr. Malden’s testimony that hyperergy remains suspect as a cause of her condition, states that he made the false assumption that the anesthetic was within normal limits. She neglects to mention that he made the assumption only because he was asked to do so by the plaintiff’s counsel, who formulated the hypothetical question posed to him. I consider it fair to assume that the facts which the doctor was asked to assume were facts which the plaintiff felt her evidence had established at that point. It is certainly true that no evidence was presented, before or after the hypothetical question was posed, which would support a finding that the amount of anesthetic was outside the normal limits.
Thus, in removing the requirement that a finding of malpractice be based on medical testimony in a case such as this, the majority not only permits the jury to speculate, it invites the jury to do so; it even implies that the jury will be remiss in its duty if it does not do so.
In my opinion, the refusal of the plaintiff’s requested instruction on standard of care was not reversible error. There was no evidence that the standard of care in Raymond was any lower than the standard of care in Tacoma and Olympia, and in fact the testimony of the defendant doctor himself was that the standard would be the same. I fail to see how the jury, if it followed the instruction and listened to the evidence and gave the words used their ordinary meaning (It is the general rule that the jury is presumed to do these things. Strandberg v. Northern Pac. Ry., 59 Wn.2d 259, 367 P.2d 137 (1961); Allen v. B. F. Goodrich Co., 67 Wn.2d 587, 408 P.2d 900 (1965)), could *513have failed to consider the evidence of Tacoma and Olympia practices. But more significant than this is the fact that there was no evidence that the defendant deviated from any standard of care, save in one respect. The only evidence of a practice anywhere which is different from that followed by the defendant was evidence that procaine ampules are autoclaved in Olympia, rather than sterilized in alcohol as they were at that time in the hospital where the operation was performed, and evidence that autoclaving is considered a safer means of sterilization generally. This accords with my reading on the subject, incidentally.17
A contaminant in the drug was listed as a possible cause of the plaintiff’s condition by Dr. Malden. However, again he did not name it as a probable cause, nor did any other witness, nor was there any evidence from which the jury could infer that there was a crack in an ampule used by the doctor (sterilized by the hospital, I would assume, but again for purposes of argument I am conceding his responsibility for the sterilization method). My reading on the subject of contaminants as a possible cause of neurological changes reveals that they may produce such changes; but the changes are likely to take the form of decomposition of the cells or inflammation of the arachnoid membrane rather than atrophy of the cauda equina, which autopsy has shown to be the effect upon the nerves where that syndrome is present.18
In any case, there was no medical testimony that alcohol probably produced the damage, and the majority does not suggest that there was “non-medical” evidence upon which the jury could reasonably base a finding that it was negli*514gence in the sterilizing of the ampules which led to the plaintiff’s injury.
Evidence was not offered that, in Raymond, it is good practice to insert the anesthetic needle between T-12 and L-l when performing abdominal surgery nor to give an excessive amount of anesthetic. The evidence was to the contrary. So, assuming the majority is correct — that the jury was entitled to find that one of these two acts was performed by the defendant doctor and produced the injury — the jury would presumably have found the act to be negligent, even though it followed the instruction given and was denied the benefit of the requested instruction.
In short, I do not see how the giving of the requested instruction and the withholding of the instruction given would have or could have affected the outcome of the case, assuming that the jury based its verdict on the evidence presented to it; and I am required to assume this if the jury system is to be given credence. There was simply no evidence of a standard of care in Raymond different from the standard of care generally in regard to those practices, one of which the majority feels the jury was entitled to find was the cause of the injury.
Of course, if my analysis of the record and my concept of the law is correct — that is, if there was no testimony tending to establish a probable cause of the injury, and if there must be testimony as to probable cause if the subject matter is not one which is readily within the grasp of the lay juror without the aid of experts — then any error in giving instructions on the standard of care was clearly harmless. If the cause of the condition is not established, then the standard of care is entirely immaterial. It does not matter whether the doctor’s procedure was negligent if that negligence did not cause the plaintiff’s injuries. That should be axiomatic. An error which does not materially affect the merits of the controversy is not prejudicial and does not require reversal. Reid Co. v. M-B Contracting Co., 46 Wn.2d 784, 285 P.2d 121 (1955).
The new rule laid down in Pederson v. Dumouchel, 72 Wn.2d 73, 431 P.2d 973 (1967), pertaining to the standard *515of care in medical malpractice cases is a good one, I think, and should be applied in all proper cases. But I do not think it should be treated like a new toy to be brought out and admired and used at every opportunity, regardless of whether the evidence in the case warrants its use or whether its application will affect the outcome.
This court has now singled out a class to be dealt with rather harshly. It is the class of those involved in the healing arts. The court says doctor, beware. It is not your duty simply to do the best you can for the sick and the injured. It is your duty, in the eyes of the courts, to see to it that the patient does not develop adverse side effects of the procedures which you use or react in an unexpected manner to your treatment. Failing in this, you will have to compensate the patient if you cannot explain how you managed to fail in these endeavors while still exercising a very high standard of care.
In the case of Hall v. United States, 136 F. Supp. 187, 199 (1955), the plaintiff wife developed a cauda equina syndrome, following the administration of a spinal anesthetic, just as the plaintiff did in this case. The case was tried to the court without a jury, which found that the condition could have resulted from any one of four causes, one of which was idiosyncrasy or natural sensitivity to the drug. The court felt that this was the most probable cause. In dismissing the action, the judge made a comment which is appropriate here. He said:
Any person of normal sensibilities must be deeply concerned over a tragedy such as that disclosed by this record and must feel the utmost sympathy for Mrs. Hall. Yet, however remarkable the advances made by medical science, there remains much to be learned; and members of the medical profession cannot be held responsible for circumstances beyond their knowledge and ability as human beings to anticipate and prevent. Accordingly, the law wisely requires of them only that they possess and use the reasonable knowledge, ability and skill of their colleagues.
See also Ayers v. Parry, 192 F.2d 181 (1951) and 53 A.L.R.2d 158.
*516I only trust that this court’s new zeal to see that the injured patient is compensated, regardless of whether the defendant was at fault, will not be extended to the field of the law with the result that it will become legal malpractice not to win the client’s case. This case illustrates how difficult it may sometimes be for a lawyer to advise his client about the probable outcome of a case if he relies on precedent and not on intuition. However, I can imagine less harm to humanity in the long run if the lawyer decides not to try the case because of the risk of losing, than if the doctor decides not to perform the operation or not to advise the use of the proper anesthetic because of the fear of a lawsuit which he may well be helpless to defend.19
In my opinion, the court would be doing less of a disservice to the public and the medical profession (not to mention the coherency of the common law) if it simply held that, henceforth, those who administer to the sick will be liable.for conditions of the patient which develop during the operation or treatment or which come to light immediately thereafter, regardless of whether those conditions are attributable to any act of the defendant. But since this would clearly be an act of legislation on the part of the court (it is not simply a “development” (!) in the common law), it would perhaps be better, while we are about it, to extend the liability to those considered most advantageously situated to spread the loss1 — that is, the hospitals, *517the doctors, and the manufacturers. Nurses, being salaried employees, are not in such a good position to pass the loss on to other patients and likely should be spared the bankruptcy which one would probably have to undergo after one massive judgment was taken against her. And while we are unblushingly legislating, it would perhaps be well to require that all these prospective defendants carry insurance and that insurance companies provide the insurance; and perhaps it would also be well to limit the amount of recovery for each type of condition or disability. I say this because, in the field of workmen’s compensation, which is analogous and in which the legislature has seen fit to act (RCW 51), there are numerous controls and restrictions provided in order to render the imposition of liability without fault less onerous to the employer and to prevent inequalities in compensation which would probably be the rule if each injured workman were allowed to go to the jury on the question of damages alone.
However, I do not base my dissent on a concern that doctors will fail to exercise their best judgment in treating their patients because of a fear of lawsuits. For one thing, I do not imagine that the majority of doctors are so easily deterred from doing their duty. But more important, my primary concern is in the area of my own duty. It is the effect of this decision upon the law which troubles me — the emasculation of rules which have been tested by time and experience and found to promote the effecting of justice among litigants and have been properly relied upon by members of the bench and bar. It is true that there are many laymen who laugh at the law as enunciated by judges and regard it as ponderous and impractical. But I had thought that that evaluation of the judicial function was not shared by my fellows on the bench. I had supposed that, on the contrary, they had a high regard for the methods of judicial reasoning which, in addition to imposing a duty to employ logic and the perceptions derived from common experience, require that the evidence not be disregarded and that legal precedents be respected and not be departed from in the absence of compelling reasons to con-*518elude that they are based on false assumptions. I find no such compelling reasons in the case before us. On the contrary, it seems to me that those rules are just as serviceable here as they have been in the past in arriving at a just result.
The formulation of public policy is primarily a function for the legislature. It has at hand the means of investigation to assure itself of the pertinent facts, it is responsive to the will of the people, and it has the means to effectuate that policy when it is formulated. This does not mean that the court should never be influenced by public policy considerations; but when a supposed public policy and the well-founded principles of justice upon which the common law is built are in direct conflict, it seems to me that the change in the law should be made by the legislature.
This court is simply not equipped to impose absolute liability in a form which maintains some semblance of justice for the party who is made the insurer. A glance at the workmen's compensation statutes, which I have referred to, shows how many factors there are to consider if a satisfactory system of “shifting the loss” is to be achieved. The legislature even finds it advisable to set up a board to administer this insurance program and thus spare both the injured workman and the employer unnecessary expenses, delays, inconveniences, and embarrassment of litigation. Certainly, it seems to me if the doctor is to be made the insurer of his patients, he should be provided protection from lawsuits in which his competency and integrity are publicly questioned and he is given the onus of malpractice, even though he has done all that he knew to do and all that medical science, in its present state of development, has told him that he could do.
This court has recently effected a change in the common law which I consider a wise one and one which is within the natural evolution of the common law. It has recognized that a factual assumption upon which an exception to the general rule of negligence was formerly engrafted by the courts is now ill founded. I refer, of course, to the case of *519Pederson v. Dumouchel, 72 Wn.2d 73, 431 P.2d 973 (1967), holding that a plaintiff in a malpractice case will no longer be required to show that the defendant doctor departed from the standard of care in the area in which he practices, but may show that he failed to exercise reasonable care under the circumstances and thus may show what the practice is in the profession generally. This, it seems obvious to me, is a return to a basic principle of common law and is only a departure from a court-made exception to the principle, just as the rule of charitable immunity was such a departure.20
Now that a plaintiff may freely go outside his area to find his experts there should be a relaxing of the fear that he will be met with a “conspiracy of silence,” inasmuch as a “foreign” expert will not need to fear local reprisal. I am of the opinion that this court should at least allow the effects of this new rule to be observed and see if it is not sufficiently efficacious to lighten the plaintiff’s burden before it initiates other “reforms” of much more questionable validity.
Furthermore, I do not think that a plaintiff’s attorney need be unduly handicapped by lack of information in testing the expertise of a medical witness. Medical knowledge has been written down and is accessible to the lawyer. If he confronts the witness with this knowledge, I rather doubt that the doctor would allow his reluctance to incriminate a colleague to overcome his concern for his own reputation as a man competent in his field and so profess ignorance of matters which reasonable competence would require him to know.
As far as the plaintiff’s being a victim of a conspiracy of silence in this case is concerned, I find no evidence of it in the record. On the contrary, her expert witness testified quite openly and freely for her, naming every possibility of negligence which has occurred to any student of this dis*520ease, as far as I have been able to ascertain. I see no reason to suppose that he refused to testify that one possible cause was a more probable cause than another for any reason other than the reason that he could not honestly do so. This is, I am convinced, a condition which sometimes develops after anesthesia (and it follows other forms of anesthesia —not just spinal anesthesia21) and for which medical science has not yet found a satisfactory explanation, but the causes and mechanism of which dedicated medical men are earnestly endeavoring to ascertain. It seems reasonable to conclude, however, at this point, that the anesthetic itself, acting upon the patient’s body in some apparently freakish manner, is the agent which produces the damage, since the nerve roots involved are those normally affected by the anesthetic and the effect upon them is anesthetic in nature.
The plaintiff has made and argued a number of assignments of error which are not mentioned in the opinion of the majority. I assume, therefore, that the majority finds them to be without merit as I do and gives its tacit approval to the repetition of the rulings on a new trial. It might be advisable, however, I should think, to at least give some indication that the court has not overlooked these assignments. This might forestall any chance of their being raised and argued if this case is again appealed by the plaintiff, in which case it is not inconceivable that they might be made the basis for the ordering of another new trial if the court’s determination that the plaintiff shall recover damages has not abated by that time.
I suppose it is anticlimactic to state that I would affirm the judgment entered on the verdict of the jury in this case and save my legal reforms to be effectuated when a more patent miscarriage of justice is presented for our review.
*521Appendix

*522

 See, for example, Indexa Medica which guides the reader to numerous periodicals in many languages.

 A. Faulconer, Jr. (M.D., M.S. in Anesthesiology, Head, Section of Anesthesiology, Mayo Clinic, Professor of Anesthesiology, Mayo Graduate School of Medicine, University of Minnesota, Rochester, Minn.) & T. Keys (A. B., M. A. Librarian, Mayo Clinic, Associate Professor of History of Medicine, Mayo Graduate School of Medicine, University of Minnesota, Rochester, Minn.), II Foundations of Anesthesiology (1965); Schwarz & Bevilacqua, Paraplegia Following Spinal Anesthesia, 10 Archives of Neurology, at 308 (March 1964); Keating and Luke, Anesthetic Accidents (2ded. 1961).
In his book Dr. Keating says that there is no evidence that neurological reactions are due to faulty or careless technique; that, although they are rare, they occur in well-managed teaching hospitals in Great Britain, the United States, and the continent of Europe, and in the hands of competent anesthetists. On the other hand, as stated by the majority, Doctors Vandam and Dripps, who conducted a study of 10,098 spinals given at the University of Pennsylvania (reported in 2 Foundations of Anesthesiology, at 888 (1965)) and found no instance of a neurological disorder attributable to the anesthesia, concluded from their experience that such disorders can be avoided if proper precautions are taken. Others who have written on the subject have opinions which fall between these two extremes. Thus the doctors are not in agreement as to possible causes, much less probable causes.

 N. Greene, Neurological Sequelae of Spinal Anesthesia, Anesthesiology 22:682 (1961).

 See note 3, supra. See, also, E. Hudon, Anesthetic Death, Infection, Thrombosis and Disease, Ins. L. J., No. 430, at 711 (1958).

 It is true that the California Supreme Court has held in Ybarra v. Spangard, 25 Cal. 2d 486, 154 P.2d 687 (1944), 208 P.2d 445 (Cal. Dist. Ct. App. 1949) that every person who might have caused the plaintiff’s injury is vulnerable under the doctrine of res ipsa loquitur and must prove his innocence or pay. That case, however, has been roundly criticized. See, for examples, W. Seavey, Res Ipsa Loquitur: Tabula in Naufragio, 63 Harv. L. Rev. 643 (1950); O. C. Adamson, II, Medical Malpractice: Misuse of Res Ipsa Loquitur, 46 Minn. L. Rev. 1043 (1962); The Application of Res Ipsa Loquitur in Medical Malpractice *500Cases, 60 Nw. U. L. Rev. 852 (1966); W. Prosser, Res Ipsa Loquitur in California, 37 Cal. L. Rev. 183 (1949). Those criticisms apply equally to Seneris v. Haas, 45 Cal. 2d 811, 291 P.2d 915, 53 A.L.R.2d 124 (1955), relied upon by the appellant, which also applied the doctrine in a case like this.

 And yet the makeup of the plaintiff’s body or preexisting physical condition may well be a contributing factor in her disease, according to the record in this case. See also the sources cited in note 3, supra.

 The Application of Res Ipsa Loquitur in Medical Malpractice Cases, note 6, supra.

 But, for example, does the doctor have greater access to knowledge of the plaintiff’s previous physical condition? This may be and often is a factor in these postoperative diseases. A. Marinacci, Neurological Aspects of Complications of Spinal Anesthesia with Medicolegal Implications, Bulletin of the Los Angeles Neurological Society, 25:170 (1960).
Dr. Marinacci says that there is only one way to be certain that a postoperative complication is attributable to the anesthesia, and that is to perform electromyogram tests (not to be confused with myelogram). The plaintiff was not given these tests, as far as the record reveals, but her neurologist satisfied himself that her syndrome was attributable to the anesthesia.
Dr. Marinacci also reports that of 542 patients given electromyo-grams, counterfeit symptom-complexes appeared in a ratio of 100 to 1. I am not suggesting that there is any possibility that this was the case here, but it is of interest in indicating how ill founded a supposition may be that, because a condition followed the administering of anesthesia, it is attributable to the anesthesia — and even (if res ipsa loquitur is to be used so indiscriminately) to negligence of the doctor or someone involved in the administering of the anesthesia. And it also demonstrates rather vividly, it seems to me, that an assumption that the doctor has access to all the facts is not warranted. See, also, note 7, supra.

 Only by proving the actual cause can either definitively prove freedom from negligence, on remand of Ybarra v. Spangard, supra, none of the defendants was able to establish the actual cause of the plaintiff’s injury and all were held liable.

 Cord damage is sometimes found in these cases and is caused by the anesthetic or other substance which is injected, according to Dr. Marinacci. See his article cited in note 9, supra: Drs. Schwarz and Bevilaequa (note 3) say that there is cord damage in most human cases. These doctors seem to feel that the secret of the cause may be found (at some future date) through study of the vascular changes which occur under the effects of the anesthesia. They do not seem much impressed with the idea of puncture wounds as a probable cause.

 There was medical testimony that the needle causes pain on injection and also produces sharp pain if it touches a nerve root. The normal sensations of injection are described in Wasmuth, Anesthesia and the Law, (1961) at 57. The author says:
Inasmuch as many people have an intense fear of spinal puncture, it is not surprising that the number of malpractice cases with this procedure is increasing. A patient does not relish the pain caused by the piercing of the skin of his back, or the snapping sensation as the longitudinal ligament is pierced, or the sudden twinge of pain if the nerve filaments are touched by the spinal needle.

 Keating (see note 3, supra) says that cord puncture and injection of solution probably would produce immediate collapse. “[I]f the patient survives long enough,” evidence of transverse myelitis might be observed.

 The following authority confirms the medical testimony on this:
“Some patients require much more intra-spinal procaine to produce analgesia than others. There is no set dose of ether for a given case, but it is given under control as needed and the dose varies greatly. The same is true in operating under spinal anesthesia. The dose should be given as needed and under control.
“Spinal anesthesia is the choice for so many operative procedures *511and the results are not as satisfactory when it has to be supplemented by other anesthetic agents.” II Foundations of Anesthesiology, at 886, note 3, supra.

 “The [continuous spinal] method is very useful when either the scope or the duration of the operation is uncertain. It enables minimal dosage to be given without fear of inadequate analgesia, and so is desirable in the aged, the very young and the physically handicapped.” J. A. Lee & R. S. Atkinson, A Synopsis of Anesthesia (5th ed. 1964), at 341.

 See, A Synopsis of Anesthesia, note 15, supra; J. Lundy, Clinical Anesthesia (1942).

 Greene, note 4, supra. The danger is that the sterilizing solution may seep through an undetected crack in the ampule and contaminate the anesthetic. But Dr. Greene says that large quantities of alcohol would have to be injected to produce neurological changes in man. On animals experimented with, smaller quantities (but more than the amount that would probably seep through a crack in the ampule) produced such changes.

 Greene, note 4, supra; Marinacci, note 9, supra; Marino & Pyrdek, Spinal Anesthesia and the Law of Professional Liability, 3 Ill. Continuing Legal Education, No. 4 (Oct. 1965) at 79.

 Wasmuth, in Anesthesia & the Law (note 12, supra) speaking of Seneris v. Haas, supra, says, at 56:
“The effect of this decision and others, which practically hold the physician strictly liable for untoward results, upon the practice of anesthesiology in the State of California, has been profound. The use of spinal analgesia in general surgery since the California case has declined progressively, whereas it has increased in other sections of the country.
“It is a sad commentary on the courts’ misunderstanding of scientific facts, when a physician feels that he must deny the patient the advantages of a particular type of anesthesia because of the possibility of his involvement in a malpractice suit.”
See also Marino & Pyrdek, note 18, supra. Dr. Greene (note 4, supra) also expresses regret that the courts have stigmatized spinal anesthesia with the doctrine of res ipsa loquitur.

 See, Friend v. Cove Methodist Church, 65 Wn.2d 174, 396 P.2d 546 (1964), wherein we recognized that the assumptions on which the rule oí charitable immunity were based are now known to be invalid.

 See Greene, note 4, supra.