wanyu/IteraTeR_v2 huggingface dataset:
Original: Waman Bapuji Metre ( 14 February 1906 – 21 November 1970), known as Dada (meaning Elder brother in Marathi language), was an Indian petroleum geologist.
Revised: Waman Bapuji Metre ( 14 February 1906 – 21 November 1970), known as Dada (meaning Elder brother in Marathi 
Edit intent: coherence
Original:  For his " significant contribution to the growth of the oil industry in the country " , he was awarded India's third-highest civilian award, the Padma Bhushan , in 1968  
Revised:  For his " significant contribution to the growth of the oil industry in the country ' , he was awarded India's third-highest civilian award, the Padma Bhushan , in 1968 
Edit intent: fluency
Original:  For his " significant contribution to the growth of the oil industry in the country " , he was awarded India's third-highest civilian award, the Padma Bhushan , in 1968  
Revised:  For his " significant contribution to the growth of the oil industry in the country " , he was awarded India's third-highest civilian award, the Padma Bhushan in 1968 
Edit intent: fluency
Original:  For his " significant contribution to the growth of the oil industry in the country " , he was awarded India's third-highest civilian award, the Padma Bhushan , in 1968  
Revised:  For his " significant contribution to the growth of the oil industry in the country " , he was awarded India's third-highest civilian award, the Padma Bhushan , in 1968 Early lif
Edit intent: fluency
Original: Some examples Although cancer syndromes exhibit an increased risk of cancer, the risk varies.
Revised: Examples Although cancer syndromes exhibit an increased risk of cancer, the risk varies.
Edit intent: clarity
Original:  Genetic Screening Genetic testing can be used to identify mutated genes or chromosomes that are passed through generations.
Revised:  Genetic screening Genetic testing can be used to identify mutated genes or chromosomes that are passed through generations.
Edit intent: fluency
Original:  Werner's Syndrome has a prevalence of 1 in 200,000 live births in the U.S., but it affects individuals in Japan in 1 in 20,000-40,000 cases . 303x303px|This is a quick overview of how CRISPR Cas9 works in gene editing . 1 in 40 Ashkenazi Jews have a BRCA mutation, this is a huge contrast from the general population in the United States where 1 in 400 people are affected.
Revised:  Werner's Syndrome has a prevalence of 1 in 200,000 live births in the U.S., but it affects individuals in Japan in 1 in 20,000-40,000 cases . 1 in 40 Ashkenazi Jews have a BRCA mutation, this is a huge contrast from the general population in the United States where 1 in 400 people are affected.
Edit intent: coherence
Original:  Given the prevalence of breast and colon cancer, the most widely recognized syndromes include hereditary breast-ovarian cancer syndrome (HBOC) and hereditary non-polyposis colon cancer ( HNPCC, Lynch syndrome).
Revised:  Given the prevalence of breast and colon cancer, the most widely recognized syndromes include hereditary breast-ovarian cancer syndrome and hereditary non-polyposis colon cancer ( HNPCC, Lynch syndrome).
Edit intent: clarity
Original:  Given the prevalence of breast and colon cancer, the most widely recognized syndromes include hereditary breast-ovarian cancer syndrome (HBOC) and hereditary non-polyposis colon cancer ( HNPCC, Lynch syndrome).
Revised:  Given the prevalence of breast and colon cancer, the most widely recognized syndromes include hereditary breast-ovarian cancer syndrome (HBOC) and hereditary non-polyposis colon cancer ( Lynch syndrome).
Edit intent: clarity
Original:  Fanconi anemia Fanconi anemia (FA) is a disorder with a wide clinical spectrum, including: early onset and increased risk of cancer;
Revised:  Fanconi anemia Fanconi anemia is a disorder with a wide clinical spectrum, including: early onset and increased risk of cancer;
Edit intent: clarity
Original:  Hereditary breast and ovarian cancer Hereditary breast-ovarian cancer syndrome (HBOC) is an autosomal dominant genetic disorder caused by genetic mutations of the BRCA1 and BRCA2 genes.
Revised:  Hereditary breast and ovarian cancer Hereditary breast-ovarian cancer syndrome is an autosomal dominant genetic disorder caused by genetic mutations of the BRCA1 and BRCA2 genes.
Edit intent: clarity
Original:  Hereditary non-polyposis colon cancer Hereditary non-polyposis colon cancer (HNPCC) , also known as Lynch syndrome, is an autosomal dominant cancer syndrome that increases the risk of colorectal cancer.
Revised:  Hereditary non-polyposis colon cancer Hereditary non-polyposis colon cancer , also known as Lynch syndrome, is an autosomal dominant cancer syndrome that increases the risk of colorectal cancer.
Edit intent: clarity
Original:  HNPCC is also associated with an early onset of colorectal cancer.
Revised:  Hereditary non-polyposis colon cancer is also associated with an early onset of colorectal cancer.
Edit intent: clarity
Original:  This example pedigree chart shows how inheritance of x-linked Fanconi anemia might occur through several generations.
Revised:  This example pedigree chart shows how inheritance of X-linked Fanconi anemia might occur through several generations.
Edit intent: fluency
Original:  Hereditary paraganglioma-pheochromocytoma syndrome Most cases of familial paraganglioma are caused by mutations in the succinate dehydrogenase ( SDH;
Revised:  Hereditary paraganglioma-pheochromocytoma syndrome Most cases of familial paraganglioma are caused by mutations in the succinate dehydrogenase ( 
Edit intent: coherence
Original:  Nevoid basal cell carcinoma syndrome (NBCCS) , also known as Gorlin syndrome, is an autosomal dominant cancer syndrome in which the risk of basal cell carcinoma is very high.
Revised:  Nevoid basal cell carcinoma syndrome , also known as Gorlin syndrome, is an autosomal dominant cancer syndrome in which the risk of basal cell carcinoma is very high.
Edit intent: clarity
Original:  Von Hippel–Lindau disease Von Hippel–Lindau (VHL) disease is a rare, autosomal dominant genetic condition that predisposes individuals to benign and malignant tumors.
Revised:  Von Hippel–Lindau disease Von Hippel–Lindau disease is a rare, autosomal dominant genetic condition that predisposes individuals to benign and malignant tumors.
Edit intent: clarity
Original:  The most common tumors in VHL are central nervous system and retinal hemangioblastomas, clear cell renal carcinomas, pheochromocytomas, pancreatic neuroendocrine tumours, pancreatic cysts, endolymphatic sac tumors and epididymal papillary cystadenomas.
Revised:  The most common tumors in Von Hippel–Lindau disease are central nervous system and retinal hemangioblastomas, clear cell renal carcinomas, pheochromocytomas, pancreatic neuroendocrine tumours, pancreatic cysts, endolymphatic sac tumors and epididymal papillary cystadenomas.
Edit intent: clarity
Original:  VHL results from a mutation in the von Hippel–Lindau tumor suppressor gene on chromosome 3p25.3. Xeroderma pigmentosum (XP) is an autosomal recessive disorder characterized by sensitivity to ultra-violet (UV) light, massively increased risk of sunburn and increased risk of skin cancers.
Revised:  Von Hippel–Lindau disease results from a mutation in the von Hippel–Lindau tumor suppressor gene on chromosome 3p25.3. Xeroderma pigmentosum (XP) is an autosomal recessive disorder characterized by sensitivity to ultra-violet (UV) light, massively increased risk of sunburn and increased risk of skin cancers.
Edit intent: clarity
Original:  VHL results from a mutation in the von Hippel–Lindau tumor suppressor gene on chromosome 3p25.3. Xeroderma pigmentosum (XP) is an autosomal recessive disorder characterized by sensitivity to ultra-violet (UV) light, massively increased risk of sunburn and increased risk of skin cancers.
Revised:  VHL results from a mutation in the von Hippel–Lindau tumor suppressor gene on chromosome 3p25.3. Xeroderma pigmentosum is an autosomal recessive disorder characterized by sensitivity to ultra-violet (UV) light, massively increased risk of sunburn and increased risk of skin cancers.
Edit intent: clarity
Original:  Primary care physicians can identify people who are at risk of heridatary cancer syndrome.
Revised:  Primary care physicians can identify people who are at risk of a hereditary cancer syndrome.
Edit intent: fluency
Original:  Scientific understanding of cancer susceptibility syndromes is actively expanding: additional syndromes are being found, the underlying biology is becoming clearer, and commercialization of diagnostic genetics methodology is improving clinical access . Given the prevalence of breast and colon cancer, the most widely recognized syndromes include hereditary breast-ovarian cancer syndrome and hereditary non-polyposis colon cancer (Lynch syndrome).
Revised:  Scientific understanding of cancer susceptibility syndromes is actively expanding: additional syndromes are being found, the underlying biology is becoming clearer, and genetic testing is improving detection, treatment, and prevention of cancer syndromes . Given the prevalence of breast and colon cancer, the most widely recognized syndromes include hereditary breast-ovarian cancer syndrome and hereditary non-polyposis colon cancer (Lynch syndrome).
Edit intent: clarity
Original:  Police: Anthony Kirkland suspect in several murders - FOX19.com-Cincinnati News, Weather Sports He was paroled in URL
Revised:  Sports He was paroled in URL
Edit intent: coherence
Original:  Police: Anthony Kirkland suspect in several murders - FOX19.com-Cincinnati News, Weather Sports He was paroled in URL
Revised:  Police: Anthony Kirkland suspect in several murders - FOX19.com-Cincinnati News, Weather He was paroled in URL
Edit intent: fluency
Original:  768 p. . For example, Fraser et al. generated one of the first estimates of  for pandemic H1N1 influenza in 2009 by using a coalescent-based analysis of 11 hemagglutinin sequences in combination with prior data about the infectious period for influenza.
Revised:  768 p. . For example, one of the first estimates of for pandemic H1N1 influenza in 2009 by using a coalescent-based analysis of 11 hemagglutinin sequences in combination with prior data about the infectious period for influenza.
Edit intent: clarity
Original:  768 p. . For example, Fraser et al. generated one of the first estimates of  for pandemic H1N1 influenza in 2009 by using a coalescent-based analysis of 11 hemagglutinin sequences in combination with prior data about the infectious period for influenza.
Revised:  768 p. . For example, Fraser et al. generated one of the first estimates of was for pandemic H1N1 influenza in 2009 by using a coalescent-based analysis of 11 hemagglutinin sequences in combination with prior data about the infectious period for influenza.
Edit intent: fluency
Original:  Volz et al. proposed that the general formula for the rate of coalescence will be: .
Revised:  The general formula for the rate of coalescence will be: .
Edit intent: clarity
Original:  Volz et al. proposed that the general formula for the rate of coalescence will be: .
Revised:  Volz et al. proposed that the general formula for the rate of coalescence is: .
Edit intent: clarity
Original:  This rate, derived for the SIR model at equilibrium, is equivalent to the rate of coalescence given by the more general formula provided by Volz et al . Rates of coalescence can similarly be derived for epidemiological models with superspreaders or other transmission heterogeneities, for models with individuals who are exposed but not yet infectious, and for models with variable infectious periods, among others.
Revised:  This rate, derived for the SIR model at equilibrium, is equivalent to the rate of coalescence given by the more general formula . Rates of coalescence can similarly be derived for epidemiological models with superspreaders or other transmission heterogeneities, for models with individuals who are exposed but not yet infectious, and for models with variable infectious periods, among others.
Edit intent: clarity
Original:  However, recent work by Rambaut et al. and Russell et al. has shown that temperate epidemics usually emerge from a global reservoir rather than emerging from within the previous season's genetic diversity.
Revised:  However, temperate epidemics usually emerge from a global reservoir rather than emerging from within the previous season's genetic diversity.
Edit intent: clarity
Original:  This work, and more recent workby Bedford et al. and Bahl et al. , has suggested that the global persistence of the influenza population is driven by viruses being passed from epidemic to epidemic, with no individual region in the world showing continual persistence.
Revised:  This and subsequent work , has suggested that the global persistence of the influenza population is driven by viruses being passed from epidemic to epidemic, with no individual region in the world showing continual persistence.
Edit intent: clarity
Original:  However, through joint epidemiological and evolutionary simulations, Bedford et al. show that their estimates of migration rates  appear robust to a large degree of undersampling or oversampling of a particular region.
Revised:  However, estimates of migration rates appear robust to a large degree of undersampling or oversampling of a particular region.
Edit intent: coherence
Original:  Figure 2. Idealized caricatures of virus phylogenies that show the effects of population subdivision, distinguishing between a structured host population (A) and an unstructured host population (B).
Revised:  Idealized caricatures of virus phylogenies that show the effects of population subdivision, distinguishing between a structured host population (A) and an unstructured host population (B).
Edit intent: clarity
Original:  Figure 3. Idealized caricatures of virus phylogenies that show the effects of immune escape where selection results in an unbalanced tree (A) and neutral dynamics results in a balance tree (B).
Revised:  Idealized caricatures of virus phylogenies that show the effects of immune escape where selection results in an unbalanced tree (A) and neutral dynamics results in a balance tree (B).
Edit intent: clarity
Original:  Figure 4. A gene genealogy illustrating internode intervals.
Revised:  A gene genealogy illustrating internode intervals.
Edit intent: clarity
Original:  250px| Figure 5. Phylogenetic tree of the HA1 region of the HA gene of influenza A (H3N2) from viruses sampled between 1968 and 2002. + Table 1. Estimated annual growth rates of for early HIV sub-epidemics.
Revised:  250px| Phylogenetic tree of the HA1 region of the HA gene of influenza A (H3N2) from viruses sampled between 1968 and 2002. + Table 1. Estimated annual growth rates of for early HIV sub-epidemics.
Edit intent: clarity
Original:  250px| Figure 5. Phylogenetic tree of the HA1 region of the HA gene of influenza A (H3N2) from viruses sampled between 1968 and 2002. + Table 1. Estimated annual growth rates of for early HIV sub-epidemics.
Revised:  250px| Figure 5. Phylogenetic tree of the HA1 region of the HA gene of influenza A (H3N2) from viruses sampled between 1968 and 2002. + Estimated annual growth rates of for early HIV sub-epidemics.
Edit intent: clarity
Original: Sources of phylodynamic variation Idealized caricatures of virus phylogenies that distinguish between exponential growth (A) and constant size (B) of the virus population.
Revised: Sources of phylodynamic variation Idealized caricatures of virus phylogenies that distinguish between virus population with (A) and constant size (B) of the virus population.
Edit intent: clarity
Original: Sources of phylodynamic variation Idealized caricatures of virus phylogenies that distinguish between exponential growth (A) and constant size (B) of the virus population.
Revised: Sources of phylodynamic variation Idealized caricatures of virus phylogenies that distinguish between exponential growth (A) exponential growth (B) of the virus population.
Edit intent: clarity
Original: Sources of phylodynamic variation Idealized caricatures of virus phylogenies that distinguish between exponential growth (A) and constant size (B) of the virus population.
Revised: Sources of phylodynamic variation Idealized caricatures of virus phylogenies that distinguish between exponential growth (A) and constant size (B) or constant size.
Edit intent: clarity
Original:  The relative lengths of internal versus external branches will be affected by changes in viral population size over time (see figure 1) Rapid expansion of a virus in a population will be reflected by a "star-like" tree, in which external branches are long relative to internal branches.
Revised:  The relative lengths of internal versus external branches will be affected by changes in viral population size over time Rapid expansion of a virus in a population will be reflected by a "star-like" tree, in which external branches are long relative to internal branches.
Edit intent: coherence
Original:  The phylogeny of HIV provides a good example of a star-like tree, as the prevalence of HIV infection rose rapidly throughout the 1980s ( caricatured by figure 1A ).
Revised:  The phylogeny of HIV provides a good example of a star-like tree, as the prevalence of HIV infection rose rapidly throughout the 1980s ( exponential growth ).
Edit intent: clarity
Original:  The phylogeny of hepatitis B virus (caricatured by figure 1B) instead reflects a viral population that has remained roughly constant in size . Similarly, trees reconstructed from viral sequences isolated from chronically infected individuals can be used to gauge changes in viral population sizes within a host.
Revised:  The phylogeny of hepatitis B virus instead reflects a viral population that has remained roughly constant in size . Similarly, trees reconstructed from viral sequences isolated from chronically infected individuals can be used to gauge changes in viral population sizes within a host.
Edit intent: clarity
Original:  The phylogeny of hepatitis B virus (caricatured by figure 1B) instead reflects a viral population that has remained roughly constant in size . Similarly, trees reconstructed from viral sequences isolated from chronically infected individuals can be used to gauge changes in viral population sizes within a host.
Revised:  The phylogeny of hepatitis B virus (caricatured by figure 1B) instead reflects a viral population that has remained roughly consistent (constant size) . Similarly, trees reconstructed from viral sequences isolated from chronically infected individuals can be used to gauge changes in viral population sizes within a host.
Edit intent: clarity
Original:  The clustering of taxa on a viral phylogeny will be affected by host population structure (see figure 2) Viruses within similar hosts, such as hosts that reside in the same geographic region, are expected to be more closely related genetically if transmission occurs more commonly between them.
Revised:  The clustering of taxa on a viral phylogeny will be affected by host population structure Viruses within similar hosts, such as hosts that reside in the same geographic region, are expected to be more closely related genetically if transmission occurs more commonly between them.
Edit intent: coherence
Original:  The phylogenies of measles and rabies virus (caricatured by figure 2A) illustrate viruses with strong spatial structure .
Revised:  The phylogenies of measles and rabies virus illustrate viruses with strong spatial structure .
Edit intent: clarity
Original:  The phylogenies of measles and rabies virus (caricatured by figure 2A) illustrate viruses with strong spatial structure .
Revised:  The phylogenies of measles and rabies virus (caricatured by figure 2A) illustrate viruses with spatially structured host population .
Edit intent: clarity
Original:  Tree balance will be affected by selection, most notably immune escape (see figure 3) The effect of directional selection on the shape of a viral phylogeny is exemplified by contrasting the trees of influenza virus and HIV's surface proteins.
Revised:  Tree balance will be affected by selection, most notably immune escape The effect of directional selection on the shape of a viral phylogeny is exemplified by contrasting the trees of influenza virus and HIV's surface proteins.
Edit intent: coherence
Original:  An example of this is the phylogeny of the HIV envelope protein inferred from sequences isolated from different individuals in a population ( caricatured by figure 3B ). phylogenies of the HIVf envelope protein from chronically infected hosts resemble influenza's ladder-like tree (caricatured by figure 3A) .
Revised:  An example of this is the phylogeny of the HIV envelope protein inferred from sequences isolated from different individuals in a population ( caricatured by figure 3B ). phylogenies of the HIVf envelope protein from chronically infected hosts resemble influenza's ladder-like tree .
Edit intent: coherence
Original:  For instance, although ladder-like trees such as the one shown in figure 3A could reflect the presence of directional selection, ladder-like trees could also reflect sequential genetic bottlenecks that might occur with rapid spatial spread, as in the case of rabies virus.
Revised:  For instance, although ladder-like trees could reflect the presence of directional selection, ladder-like trees could also reflect sequential genetic bottlenecks that might occur with rapid spatial spread, as in the case of rabies virus.
Edit intent: clarity
Original:  This time interval is labeled , and at its end there are extant lineages remaining (see figure 4) .
Revised:  This time interval is labeled , and at its end there are extant lineages remaining .
Edit intent: coherence
Original:  A basic question is whether geographic character labels are more clustered on a phylogeny than expected under a simple nonstructured model (see figure 3) .
Revised:  A basic question is whether geographic character labels are more clustered on a phylogeny than expected under a simple nonstructured model .
Edit intent: coherence
Original:  Of central interest to the study of viral phylodynamics is the distinctive phylogenetic tree of epidemic influenza A/H3N2, which shows a single predominant trunk lineage that persists through time and side branches that persist for only 1–5 years before going extinct (see figure 5) . Phylodynamic techniques have provided insight into the relative selective effects of mutations to different sites and different genes across the influenza virus genome.
Revised:  Of central interest to the study of viral phylodynamics is the distinctive phylogenetic tree of epidemic influenza A/H3N2, which shows a single predominant trunk lineage that persists through time and side branches that persist for only 1–5 years before going extinct . Phylodynamic techniques have provided insight into the relative selective effects of mutations to different sites and different genes across the influenza virus genome.
Edit intent: coherence
Original:  Phylogenetic analysis of H3N2 influenza has shown that putative epitope sites of the HA protein evolve approximately 3.5 times faster on the trunk of the phylogeny than on side branches (see figure 5) .
Revised:  Phylogenetic analysis of H3N2 influenza has shown that putative epitope sites of the HA protein evolve approximately 3.5 times faster on the trunk of the phylogeny than on side branches .
Edit intent: coherence
Original:  Qualitatively similar to the phylogeny of influenza A/H3N2’s hemagglutinin protein (see figure 5) , influenza A/H1N1 exhibits a ladder-like phylogeny with relatively low genetic diversity at any point in time and rapid lineage turnover.
Revised:  Qualitatively similar to the phylogeny of influenza A/H3N2’s hemagglutinin protein , influenza A/H1N1 exhibits a ladder-like phylogeny with relatively low genetic diversity at any point in time and rapid lineage turnover.
Edit intent: coherence
Original:  The rapid early growth of HIV-1 in Central Africa is reflected in the star-like phylogenies of the virus (caricatured in figure 2) , with most coalescent events occurring in the distant past.
Revised:  The rapid early growth of HIV-1 in Central Africa is reflected in the star-like phylogenies of the virus , with most coalescent events occurring in the distant past.
Edit intent: clarity