wanyu/IteraTeR_v2 huggingface dataset:
Original:  Dezember 2011. 2007 drehte Anne Cuneo ein filmisches Porträt des „ Pioniers in Sachen Kunstausstellungen “ Weblinks Category:2005 deaths Category:1913 births Category:Swiss people Category:People from Züric
Revised:  Dezember 2011. 2007 drehte Anne Cuneo ein filmisches Porträt des „ Pioniers in Sachen Kunstausstellungen " Weblinks Category:2005 deaths Category:1913 births Category:Swiss people Category:People from Züric
Edit intent: fluency
Original:  Dezember 2011. 2007 drehte Anne Cuneo ein filmisches Porträt des „ Pioniers in Sachen Kunstausstellungen “ Weblinks Category:2005 deaths Category:1913 births Category:Swiss people Category:People from Züric
Revised:  Dezember 2011. 2007 drehte Anne Cuneo ein filmisches Porträt des „ Pioniers in Sachen Kunstausstellungen “ Weblinks Category:2005 deaths Category:1913 births Category:Swiss people Category:People from Züri
Edit intent: fluency
Original:  Clinical diagnosis can be made based on a combination of medical history, physical examination, and computer tomography.
Revised:  Clinical diagnosis can be made based on a combination of medical history, physical examination, and computed tomography.
Edit intent: fluency
Original:  Age-related bony degenerations permit air to traverse freely into the periorbital soft tissues, which imply potential risks for periorbital emphysema.
Edit intent: coherence
Original:  Determining the degree of virilization or masculinisation: In females, the Prader staging system should be used if the karyotyping  In males, the external masculinization score should be used.
Revised:  Determining the degree of virilization or masculinisation: In females, the Prader staging system should be used if the karyotyping . In males, the external masculinization score should be used.
Edit intent: fluency
Original:  Tests are conducted within 36 hours of birth to days 15-90 days post birth which is a time frame in which can be amplified to 6 months of age.
Revised:  Tests are conducted within 36 hours after birth and/or 15-90 days post birth which is a time frame in which can be amplified to 6 months of age.
Edit intent: clarity
Original:  Tests are conducted within 36 hours of birth to days 15-90 days post birth which is a time frame in which can be amplified to 6 months of age.
Revised:  Tests are conducted within 36 hours of birth to days 15-90 days post birth . The testing time frame is amplified to 6 months of age.
Edit intent: clarity
Original:  Basal cortisol levels and adrenocorticotropic hormone (ACTH) is essential in diagnosing panhypopituitarism and enzymatic disorders affecting the adrenal steroidogenesis.
Revised:  Basal cortisol levels and adrenocorticotropic hormone (ACTH) is essential in diagnosing panhypopituitarism and enzymatic disorders affecting adrenal steroidogenesis.
Edit intent: fluency
Original:  Abnormalities that require immediate medical evaluation occur in approximately 1 in 1000 to 4500 live births.
Edit intent: coherence
Original:  Afterwards, if the family or individual wishes,  can partake in different management and treatment options for their conditions (e.g. hormone therapy).
Revised:  Afterwards, if the family or individual wishes, they can partake in different management and treatment options for their conditions (e.g. hormone therapy).
Edit intent: clarity
Original:  Overview Testosterone.
Revised:  Overview Chemical Structure of Testosterone
Edit intent: clarity
Original:  In the normal prenatal stages of fetal development, the fetus is exposed to testosterone - albeit more in male fetus than female . Upon the presence of 5α-reductase enzyme, testosterone is converted to dihydrotestosterone (i.e. DHT).
Revised:  In the normal prenatal stages of foetal development, the fetus is exposed to testosterone - albeit more in male fetus than female . Upon the presence of 5α-reductase enzyme, testosterone is converted to dihydrotestosterone (i.e. DHT).
Edit intent: fluency
Original:  In the normal prenatal stages of fetal development, the fetus is exposed to testosterone - albeit more in male fetus than female . Upon the presence of 5α-reductase enzyme, testosterone is converted to dihydrotestosterone (i.e. DHT).
Revised:  In the normal prenatal stages of fetal development, the fetus is exposed to testosterone - albeit more in male fetuses than female ones . Upon the presence of 5α-reductase enzyme, testosterone is converted to dihydrotestosterone (i.e. DHT).
Edit intent: clarity
Original:  Males with Klinefelter syndrome usually have a karyotype of 47,XXY as a result of having 2 or more X chromosomes.
Revised:  Males with Klinefelter syndrome usually have a karyotype of 47,XXY as a result of having two or more X chromosomes.
Edit intent: fluency
Original:  The incidence for 47,XXY is 1 in 500 males, but more severe and rarer cases of Klinefelter syndrome are when 3 or more X chromosomes are present.
Revised:  The incidence for 47,XXY is 1 in 500 males, but more severe and rarer cases of Klinefelter syndrome are when three or more X chromosomes are present.
Edit intent: fluency
Original:  The incidence is 1 in 2500 live-born females, while most patients do not survive for more than 1 year after birth.
Revised:  The incidence is 1 in 2500 live-born females, while most patients do not survive for more than one year after birth.
Edit intent: fluency
Original:  Abnormal genital development Genital abnormality can occur in the penis, scrotum or testes in males ;
Revised:  Abnormal genital development Genital abnormality can occur in the penis, scrotum or testes in males ,
Edit intent: fluency
Original:  Others These other anomalies are not classified in the above categories, which includes : Severe early-onset intrauterine growth restriction, isolated hypospadias, congenital hypogonadotropic, hypogonadism and cryptorchidism in males ;
Revised:  Others These other anomalies are not classified in the above categories, which includes Severe early-onset intrauterine growth restriction, isolated hypospadias, congenital hypogonadotropic, hypogonadism and cryptorchidism in males ;
Edit intent: fluency
Original:  Others These other anomalies are not classified in the above categories, which includes : Severe early-onset intrauterine growth restriction, isolated hypospadias, congenital hypogonadotropic, hypogonadism and cryptorchidism in males ;
Revised:  Others These other anomalies are not classified in the above categories, which includes : severe early-onset intrauterine growth restriction, isolated hypospadias, congenital hypogonadotropic, hypogonadism and cryptorchidism in males ;
Edit intent: fluency
Original:  Others These other anomalies are not classified in the above categories, which includes : Severe early-onset intrauterine growth restriction, isolated hypospadias, congenital hypogonadotropic, hypogonadism and cryptorchidism in males ;
Revised:  Others These other anomalies are not classified in the above categories, which includes : Severe early-onset intrauterine growth restriction, isolated hypospadias, congenital hypogonadotropic, hypogonadism and cryptorchidism in males ,
Edit intent: fluency
Original:  Malformation syndromes, Müllerian agenesis/hypoplasia, Uterine anomalies, vaginal atresia and labial adhesions in females.
Revised:  Malformation syndromes, Müllerian agenesis/hypoplasia, uterine anomalies, vaginal atresia and labial adhesions in females.
Edit intent: fluency
Original:  These genetic abnormalities stem from the prenatal stage of the individual as a result of their genetics but can also have secondary maternal factors resulting from hormones such as endocrine-disrupting chemicals (EDCs) or environmental factors.
Revised:  These genetic abnormalities stem from the prenatal stage of the individual as a result of their genetics . However, they can also have secondary maternal factors resulting from hormones such as endocrine-disrupting chemicals (EDCs) or environmental factors.
Edit intent: coherence
Original:  These genetic abnormalities stem from the prenatal stage of the individual as a result of their genetics but can also have secondary maternal factors resulting from hormones such as endocrine-disrupting chemicals (EDCs) or environmental factors.
Revised:  These genetic abnormalities stem from the prenatal stage of the individual as a result of their genetics but can also have secondary maternal factors as a result of endocrine disruptors such as hormones or other environmental factors.
Edit intent: clarity
Original:  Sexual differentiation occurs  during the prenatal development of a fetus.
Revised:  Sexual differentiation occurs through various processes during the prenatal development of a fetus.
Edit intent: clarity
Original:  236x236px|Human karyotype (male) .
Revised:  236x236px|Human karyotype (male) 
Edit intent: fluency
Original:  Peripheral blood is collected for karyotyping, and it helps classify the patient in one of the 3 main categories of DSD: Chromosomal variation, gonadal development disorders and abnormal genital development.
Revised:  Peripheral blood is collected for karyotyping, and it helps classify the patient in one of the three main categories of DSD: Chromosomal variation, gonadal development disorders and abnormal genital development.
Edit intent: fluency
Original:  Peripheral blood is collected for karyotyping, and it helps classify the patient in one of the 3 main categories of DSD: Chromosomal variation, gonadal development disorders and abnormal genital development.
Revised:  Peripheral blood is collected for karyotyping, and it helps classify the patient in one of the 3 main categories of DSD: chromosomal variation, gonadal development disorders and abnormal genital development.
Edit intent: fluency
Original:  FISH analysis can be supplemented through the use of sex chromosome specific probes.
Edit intent: coherence
Original:  17-Hydroxyprogesterone - Screens for congenital adrenal hyperplasia (CAH), commonly found in patients with 46, XX DSD.
Revised:  17-Hydroxyprogesterone can be used to screen for congenital adrenal hyperplasia (CAH), commonly found in patients with 46, XX DSD.
Edit intent: coherence
Original:  Tests are conducted within 36 hours after birth and/or 15-90 days post birth .
Revised:  Tests are conducted within 36 hours of birth to days 15-90 days post birth .
Edit intent: clarity
Original:  Basal cortisol levels and adrenocorticotropic hormone (ACTH) is essential in diagnosing panhypopituitarism and enzymatic disorders affecting  adrenal steroidogenesis.
Revised:  Basal cortisol levels and adrenocorticotropic hormone (ACTH) is essential in diagnosing panhypopituitarism and enzymatic disorders affecting the adrenal steroidogenesis.
Edit intent: fluency
Original:  Anti Mullerian Hormone - Used for evaluating the function of Sertoli cells.
Revised:  The anti-Müllerian hormone is used for evaluating the function of Sertoli cells.
Edit intent: clarity
Original:   See Also Referenc
Revised:  People who live with conditions regarding sexual abnormalities may encounter various mental and physical health problems. This may include traumatic experience with their own bodies, dissatisfaction with body image, low-self esteem, anxiety, depression, bipolar disorders, eating disorders, personality disorders, schizophrenia disorders, trauma and stress-related disorders, etc. See Also Referenc
Edit intent: coherence
Original:   See Also Referenc
Revised:  See Also Reference
Edit intent: fluency
Original:  Individuals with congenital (inborn) discrepancy between sex chromosome, gonadal, and the appearance of their external genitalia are categorised as having a disorder of sexual development (DSD).
Revised:  Individuals with congenital (inborn) discrepancy between sex chromosome, gonadal, and their internal and external genitalia are categorised as having a disorder of sexual development (DSD).
Edit intent: clarity
Original:  Individuals with congenital (inborn) discrepancy between sex chromosome, gonadal, and the appearance of their external genitalia are categorised as having a disorder of sexual development (DSD).
Revised:  Individuals with congenital (inborn) discrepancy between sex chromosome, gonadal, and the appearance of their external genitalia are categorised as individuals with a disorder of sexual development (DSD).
Edit intent: fluency
Original:  Individuals with congenital (inborn) discrepancy between sex chromosome, gonadal, and the appearance of their external genitalia are categorised as having a disorder of sexual development (DSD).
Revised:  Individuals with congenital (inborn) discrepancy between sex chromosome, gonadal, and the appearance of their external genitalia are categorised as having a disorder of sex development (DSD).
Edit intent: fluency
Original:  The reason for this is because psychosexual development is influenced by numerous factors that include, but  not limited to  gender differences in brain structure, genes associated with sexual development, prenatal androgen exposure, interactions with family, and cultural and societal factors.
Revised:  The reason for this is because psychosexual development is influenced by numerous factors that include, but are not limited to gender differences in brain structure, genes associated with sexual development, prenatal androgen exposure, interactions with family, and cultural and societal factors.
Edit intent: fluency
Original:  The reason for this is because psychosexual development is influenced by numerous factors that include, but  not limited to  gender differences in brain structure, genes associated with sexual development, prenatal androgen exposure, interactions with family, and cultural and societal factors.
Revised:  The reason for this is because psychosexual development is influenced by numerous factors that include, but not limited to , gender differences in brain structure, genes associated with sexual development, prenatal androgen exposure, interactions with family, and cultural and societal factors.
Edit intent: fluency
Original:   A team of experts, or patient support groups, are usually recommended for cases related to sexual anomalies.
Revised:  Because of the complex and multifaceted factors involved, communication and psychosexual support are all important. A team of experts, or patient support groups, are usually recommended for cases related to sexual anomalies.
Edit intent: coherence
Original:  This team of experts are usually derived from a variety of disciplines like paediatricians , neonatologists, paediatric urologists, paediatric general surgeons, endocrinologists, geneticists, radiologists, psychologists and social workers.
Revised:  This team of experts are usually derived from a variety of disciplines including pediatricians , neonatologists, paediatric urologists, paediatric general surgeons, endocrinologists, geneticists, radiologists, psychologists and social workers.
Edit intent: clarity
Original:  This team of experts are usually derived from a variety of disciplines like paediatricians , neonatologists, paediatric urologists, paediatric general surgeons, endocrinologists, geneticists, radiologists, psychologists and social workers.
Revised:  This team of experts are usually derived from a variety of disciplines like paediatricians , neonatologists, pediatric urologists, pediatric general surgeons, endocrinologists, geneticists, radiologists, psychologists and social workers.
Edit intent: fluency
Original:   Overview Chemical Structure of Testosterone In the normal prenatal stages of foetal development, the fetus is exposed to testosterone - albeit more in male fetuses than female ones.
Revised:  Overview Chemical structure of testosterone In the normal prenatal stages of foetal development, the fetus is exposed to testosterone - albeit more in male fetuses than female ones.
Edit intent: coherence
Original:   Overview Chemical Structure of Testosterone In the normal prenatal stages of foetal development, the fetus is exposed to testosterone - albeit more in male fetuses than female ones.
Revised:  Overview Chemical Structure of Testosterone In the normal prenatal stages of fetal development, the fetus is exposed to testosterone - albeit more in male fetuses than female ones.
Edit intent: coherence
Original:  Upon the presence of  5α-reductase enzyme, testosterone is converted to dihydrotestosterone (i.e. DHT).
Revised:  Upon the presence of the 5α-reductase enzyme, testosterone is converted to dihydrotestosterone (i.e. DHT).
Edit intent: fluency
Original:  If DHT is present,  male external genitalia will develop.
Revised:  If DHT is present, the male external genitalia will develop.
Edit intent: clarity
Original:  The genital  tubercle forms the penis , urethral folds form the urethra and genital swelling forms the scrotum .
Revised:  Development of male external genitalia: tubercle forms the penis , urethral folds form the urethra and genital swelling forms the scrotum .
Edit intent: clarity
Original:  The genital  tubercle forms the penis , urethral folds form the urethra and genital swelling forms the scrotum .
Revised:  The genital tubercle forms the penis Urethral folds forms the urethra Genital swelling forms the scrotum .
Edit intent: coherence
Original:  The genital  tubercle forms the penis , urethral folds form the urethra and genital swelling forms the scrotum .
Revised:  The genital tubercle forms the penis , urethral folds form the urethra and genital swelling forms the scrotum 
Edit intent: fluency
Original:  On the other hand, if DHT is not present and maternal placenta estrogen is  , then the development of female external genitalia occurs.
Revised:  On the other hand, if maternal placenta estrogen is , then the development of female external genitalia occurs.
Edit intent: coherence
Original:  The genital   tubercle forms the clitoris , the urethral folds form the labia minora and the genital swelling forms the labia majora .
Revised:  tubercle forms the clitoris , the urethral folds form the labia minora and the genital swelling forms the labia majora .
Edit intent: coherence
Original:  The genital   tubercle forms the clitoris , the urethral folds form the labia minora and the genital swelling forms the labia majora .
Revised:  The genital tubercle forms the clitoris Urethral folds forms the labia minora and the genital swelling forms the labia majora .
Edit intent: fluency
Original:  The genital   tubercle forms the clitoris , the urethral folds form the labia minora and the genital swelling forms the labia majora .
Revised:  The genital tubercle forms the clitoris , the urethral folds form the labia minora and the genital swelling forms the labia majora 
Edit intent: fluency
Original:  Chemical Structure of Dihydrotestosterone Males with Klinefelter syndrome usually have a karyotype of 47,XXY as a result of having two or more X chromosomes.
Revised:  Males with Klinefelter syndrome usually have a karyotype of 47,XXY as a result of having two or more X chromosomes.
Edit intent: coherence
Original:  The incidence for 47,XXY is 1 in 500 males, but more severe and rarer cases of Klinefelter syndrome are when three or more X chromosomes are present .
Revised:  The incidence for 47,XXY is 1 in 500 males, but severe and rare cases of Klinefelter syndrome are when three or more X chromosomes are present .
Edit intent: clarity
Original:  The incidence for 47,XXY is 1 in 500 males, but more severe and rarer cases of Klinefelter syndrome are when three or more X chromosomes are present .
Revised:  The incidence for 47,XXY is 1 in 500 males, but more severe and rarer cases of Klinefelter syndrome presents as three or more X chromosomes are present .
Edit intent: clarity
Original:  The incidence for 47,XXY is 1 in 500 males, but more severe and rarer cases of Klinefelter syndrome are when three or more X chromosomes are present .
Revised:  The incidence for 47,XXY is 1 in 500 males, but more severe and rarer cases of Klinefelter syndrome are when three or more X chromosomes .
Edit intent: clarity
Original:  Turner syndrome is classified as aneuploidy or structural rearrangement of  X chromosome.
Revised:  Turner syndrome is classified as aneuploidy or structural rearrangement of the X chromosome.
Edit intent: fluency
Original:  However, unsolved diagnosis and malignancy still cause difficulties in sex determination of these patients.
Revised:  However, unsolved diagnosis and malignancy still represent difficulties in the sex determination of these patients.
Edit intent: clarity
Original:  Abnormal genital development Genital abnormality can occur in the penis, scrotum or testes in males , vagina and labia in females.
Revised:  Abnormal genital development Genital abnormality can occur in the penis, scrotum or testes in males ; and vagina and labia in females.
Edit intent: coherence
Original:  Sometimes, ambiguous genitalia could occur, of which the external genitalia do not appear to be male or femaleclearly .
Revised:  Sometimes, ambiguous genitalia could occur, where the clear distinction of external genitalia is absent in both male and female .
Edit intent: clarity
Original:  Hence, examination , usually at birth, is done and the sex of the patient will be determined through imaging and blood tests.
Revised:  Hence, examination (typically at birth) is carried out where the sex of the patient will be determined through imaging and blood tests.
Edit intent: clarity
Original:  Abnormal genital development includes disorders of foetal origin, disorders in androgen synthesis or action, disorders in anti-Müllerian hormone synthesis or action.
Revised:  Abnormal genital development includes disorders of fetal origin, disorders in androgen synthesis or action, disorders in anti-Müllerian hormone synthesis or action.
Edit intent: fluency
Original:  Others These other anomaliesare not classified in the above categories, which includes severe early-onset intrauterine growth restriction, isolated hypospadias, congenital hypogonadotropic  , hypogonadism and cryptorchidism in males, Malformation syndromes, Müllerian agenesis/hypoplasia, uterine anomalies, vaginal atresia and labial adhesions in females . Causes Sexual anomalies are often caused by genetic reasons.
Revised:  Others In addition to the aforementioned sexual anomalies, there are other unclassified sexual anomalies. In males, this includes severe early-onset intrauterine growth restriction, isolated hypospadias, congenital hypogonadotropic , hypogonadism and cryptorchidism in males, Malformation syndromes, Müllerian agenesis/hypoplasia, uterine anomalies, vaginal atresia and labial adhesions in females . Causes Sexual anomalies are often caused by genetic reasons.
Edit intent: clarity
Original:  Others These other anomaliesare not classified in the above categories, which includes severe early-onset intrauterine growth restriction, isolated hypospadias, congenital hypogonadotropic  , hypogonadism and cryptorchidism in males, Malformation syndromes, Müllerian agenesis/hypoplasia, uterine anomalies, vaginal atresia and labial adhesions in females . Causes Sexual anomalies are often caused by genetic reasons.
Revised:  Others These other anomaliesare not classified in the above categories, which includes severe early-onset intrauterine growth restriction, isolated hypospadias, congenital hypogonadotropic hypogonadism , hypogonadism and cryptorchidism in males, Malformation syndromes, Müllerian agenesis/hypoplasia, uterine anomalies, vaginal atresia and labial adhesions in females . Causes Sexual anomalies are often caused by genetic reasons.
Edit intent: clarity