wanyu/IteraTeR_full_sent huggingface dataset:
Original:  However, due to increasing amounts of heterogeneous real-world network data with nodes and edges of different types, we extend three recent state-of-the-art homogeneous NA methods, WAVE, MAGNA++, and SANA, to allow for heterogeneous NA for the first time.
Revised:  However, due to increasing amounts of heterogeneous real-world network data with nodes or edges of different types, we extend three recent state-of-the-art homogeneous NA methods, WAVE, MAGNA++, and SANA, to allow for heterogeneous NA for the first time.
Edit intent: fluency
Original:  We introduce several algorithmic novelties to achieve this . Namely, these existing NA methods compute homogeneous graphlet-based node similarities and then find high-scoring alignments with respect to these similarities, while simultaneously maximizing the number of conserved edges.
Revised:  We introduce several algorithmic novelties . Namely, these existing NA methods compute homogeneous graphlet-based node similarities and then find high-scoring alignments with respect to these similarities, while simultaneously maximizing the number of conserved edges.
Edit intent: coherence
Original:  We introduce several algorithmic novelties to achieve this . Namely, these existing NA methods compute homogeneous graphlet-based node similarities and then find high-scoring alignments with respect to these similarities, while simultaneously maximizing the number of conserved edges.
Revised:  We introduce several algorithmic novelties to achieve this . Namely, these existing methods compute homogeneous graphlet-based node similarities and then find high-scoring alignments with respect to these similarities, while simultaneously maximizing the number of conserved edges.
Edit intent: clarity
Original:  We introduce several algorithmic novelties to achieve this . Namely, these existing NA methods compute homogeneous graphlet-based node similarities and then find high-scoring alignments with respect to these similarities, while simultaneously maximizing the number of conserved edges.
Revised:  We introduce several algorithmic novelties to achieve this . Namely, these existing NA methods compute homogeneous graphlet-based node similarities and then find high-scoring alignments with respect to these similarities, while simultaneously maximizing the amount of conserved edges.
Edit intent: clarity
Original:  In evaluations on synthetic networks and real-world biological network data , our proposed heterogeneous NA methods lead to higher-quality alignments and better robustness to noise in the data than their homogeneous counterparts  .
Revised:  In evaluations on synthetic and real-world biological network data , our proposed heterogeneous NA methods lead to higher-quality alignments and better robustness to noise in the data than their homogeneous counterparts .
Edit intent: fluency
Original:  In evaluations on synthetic networks and real-world biological network data , our proposed heterogeneous NA methods lead to higher-quality alignments and better robustness to noise in the data than their homogeneous counterparts  .
Revised:  In evaluations on synthetic networks and real-world biological networks , our proposed heterogeneous NA methods lead to higher-quality alignments and better robustness to noise in the data than their homogeneous counterparts .
Edit intent: fluency
Original:  In evaluations on synthetic networks and real-world biological network data , our proposed heterogeneous NA methods lead to higher-quality alignments and better robustness to noise in the data than their homogeneous counterparts  .
Revised:  In evaluations on synthetic networks and real-world biological network data , our proposed heterogeneous NA methods lead to higher-quality alignments and better robustness to noise in the data than their homogeneous counterparts . The software and data from this work is available upon request .
Edit intent: meaning-changed
Original:  Agents face quadratic costs-of-carry on their positions and as a consequence, their marginal valuation of the asset decreases when the magnitude of their position increases , as it would be the case for risk-averse agents.
Revised:  Risk-neutral agents face quadratic costs-of-carry on their positions and as a consequence, their marginal valuation of the asset decreases when the magnitude of their position increases , as it would be the case for risk-averse agents.
Edit intent: meaning-changed
Original:  Agents face quadratic costs-of-carry on their positions and as a consequence, their marginal valuation of the asset decreases when the magnitude of their position increases , as it would be the case for risk-averse agents.
Revised:  Agents face quadratic costs-of-carry on positions and thus their marginal valuations decrease with the size of their position increases , as it would be the case for risk-averse agents.
Edit intent: clarity
Original:  Agents face quadratic costs-of-carry on their positions and as a consequence, their marginal valuation of the asset decreases when the magnitude of their position increases , as it would be the case for risk-averse agents.
Revised:  Agents face quadratic costs-of-carry on their positions and as a consequence, their marginal valuation of the asset decreases when the magnitude of their position , as it would be the case for risk-averse agents.
Edit intent: clarity
Original:  In the equilibrium models of investors with heterogeneous beliefs that followed the original work by Harrison and Kreps , investors are risk-neutral, short-selling is prohibited and agents face a constant marginal cost of carrying positions.
Revised:  In the equilibrium models of heterogeneous beliefs that followed the original work by Harrison and Kreps , investors are risk-neutral, short-selling is prohibited and agents face a constant marginal cost of carrying positions.
Edit intent: clarity
Original:  In the equilibrium models of investors with heterogeneous beliefs that followed the original work by Harrison and Kreps , investors are risk-neutral, short-selling is prohibited and agents face a constant marginal cost of carrying positions.
Revised:  In the equilibrium models of investors with heterogeneous beliefs that followed Harrison-Kreps , investors are risk-neutral, short-selling is prohibited and agents face a constant marginal cost of carrying positions.
Edit intent: clarity
Original:  In the equilibrium models of investors with heterogeneous beliefs that followed the original work by Harrison and Kreps , investors are risk-neutral, short-selling is prohibited and agents face a constant marginal cost of carrying positions.
Revised:  In the equilibrium models of investors with heterogeneous beliefs that followed the original work by Harrison and Kreps , investors are risk-neutral, short-selling is prohibited and agents face constant marginal costs of carrying positions.
Edit intent: fluency
Original:  The resulting resale option guarantees that the equilibrium price exceeds the price of the asset in a static buy-and-hold model where speculation is ruled out . Our model features three main novelties. First, increasing marginal costs entail that the price depends on the exogenous supply.
Revised:  The resulting resale option guarantees that the equilibrium price exceeds the price of the asset in a static buy-and-hold model where speculation is ruled out ; the difference is identified as a bubble. In our model increasing marginal costs entail that the price depends on the exogenous supply.
Edit intent: meaning-changed
Original:  Third, we introduce the possibility of short-selling ; then the resale option for agents with short positions partly compensates the resale option for long agents. We characterize the unique equilibrium of our model through a Hamilton-Jacobi-Bellman equation of a novel form and use it to derive several comparative statics results .
Revised:  Third, we introduce the possibility of short-selling . A Hamilton-Jacobi-Bellman equation of a novel form and use it to derive several comparative statics results .
Edit intent: coherence
Original:  We characterize the unique equilibrium of our model through a Hamilton-Jacobi-Bellman equation of a novel form and use it to derive several comparative statics results .
Revised:  We characterize the unique equilibrium of our model through a Hamilton-Jacobi-Bellman equation of a novel form quantifies precisely the influence of the costs-of-carry on the price. An unexpected decrease in shorting costs may lead to the collapse of a bubble; this links the financial innovations that facilitated shorting of MBSs to the subsequent collapse of prices .
Edit intent: meaning-changed
Original: We investigate the dynamical effects of non-Gaussian asymmetric stable L\'evy fluctuations on the evolution of the transcription factor activator in a genetic regulation system . The noisy fluctuations arise from the synthesis reaction rate .
Revised: We investigate the dynamical effects of non-Gaussian asymmetric stable L\'evy fluctuations on the evolution of the transcription factor activator in a genetic regulation system , when the synthesis reaction rate .
Edit intent: coherence
Original:  The noisy fluctuations arise from the synthesis reaction rate . We compute two deterministic quantities, the mean first exit time (MFET) and the first escape probability (FEP), in order to examine the likelihood for transcriptions: The mean time scale for the system exits the low concentration state (the longer the exit time, the less likely for transcription) and the switch probability from low concentration states to high concentration states (corresponding to likelihood for transcription) .
Revised:  The noisy fluctuations arise from the synthesis reaction rate is under additive and multiplicative asymmetric stable L\'evy fluctuations .
Edit intent: meaning-changed
Original:  in the probability distributions of noise, we find that the fluctuations in the synthesis reaction rate lead to peculiar transitions to high concentrations and thus to possible transcriptions, such as realizing higher likelihood of transcription for larger positive skewness (i.e., asymmetry)
Revised:  in the probability distributions of noise, we find that via examining the mean first exit time (MFET) and the first escape probability (FEP), the asymmetric fluctuations, interacting with nonlinearity in the system, lead to peculiar transitions to high concentrations and thus to possible transcriptions, such as realizing higher likelihood of transcription for larger positive skewness (i.e., asymmetry)
Edit intent: meaning-changed
Original:  in the probability distributions of noise, we find that the fluctuations in the synthesis reaction rate lead to peculiar transitions to high concentrations and thus to possible transcriptions, such as realizing higher likelihood of transcription for larger positive skewness (i.e., asymmetry)
Revised:  in the probability distributions of noise, we find that the fluctuations in the synthesis reaction rate lead to peculiar likelihood for transcription. This includes, in the additive noise case, realizing higher likelihood of transcription for larger positive skewness (i.e., asymmetry)
Edit intent: meaning-changed
Original:  index \beta, causing a bifurcation for the likelihood of transcription at the critical non-Gaussianity index value \alpha=1 (i.e., beyond which the likelihood for transcription suddenly increases ), and achieving a turning point at the threshold value \beta \approx 0.55 (i.e., beyond which the likelihood for transcription  reversed for \alpha values).
Revised:  index \beta, causing a stochastic bifurcation at the non-Gaussianity index value \alpha=1 (i.e., beyond which the likelihood for transcription suddenly increases ), and achieving a turning point at the threshold value \beta \approx 0.55 (i.e., beyond which the likelihood for transcription reversed for \alpha values).
Edit intent: clarity
Original:  index \beta, causing a bifurcation for the likelihood of transcription at the critical non-Gaussianity index value \alpha=1 (i.e., beyond which the likelihood for transcription suddenly increases ), and achieving a turning point at the threshold value \beta \approx 0.55 (i.e., beyond which the likelihood for transcription  reversed for \alpha values).
Revised:  index \beta, causing a bifurcation for the likelihood of transcription at the critical non-Gaussianity index value \alpha=1 (i.e., it is a separating point or line for the likelihood for transcription suddenly increases ), and achieving a turning point at the threshold value \beta \approx 0.55 (i.e., beyond which the likelihood for transcription reversed for \alpha values).
Edit intent: meaning-changed
Original:  index \beta, causing a bifurcation for the likelihood of transcription at the critical non-Gaussianity index value \alpha=1 (i.e., beyond which the likelihood for transcription suddenly increases ), and achieving a turning point at the threshold value \beta \approx 0.55 (i.e., beyond which the likelihood for transcription  reversed for \alpha values).
Revised:  index \beta, causing a bifurcation for the likelihood of transcription at the critical non-Gaussianity index value \alpha=1 (i.e., beyond which the likelihood for transcription ), and achieving a turning point at the threshold value \beta \approx 0.55 (i.e., beyond which the likelihood for transcription reversed for \alpha values).
Edit intent: clarity
Original:  index \beta, causing a bifurcation for the likelihood of transcription at the critical non-Gaussianity index value \alpha=1 (i.e., beyond which the likelihood for transcription suddenly increases ), and achieving a turning point at the threshold value \beta \approx 0.55 (i.e., beyond which the likelihood for transcription  reversed for \alpha values).
Revised:  index \beta, causing a bifurcation for the likelihood of transcription at the critical non-Gaussianity index value \alpha=1 (i.e., beyond which the likelihood for transcription suddenly increases ), and achieving a turning point at the threshold value \beta \approx -0.5 (i.e., beyond which the likelihood for transcription reversed for \alpha values).
Edit intent: meaning-changed
Original:  index \beta, causing a bifurcation for the likelihood of transcription at the critical non-Gaussianity index value \alpha=1 (i.e., beyond which the likelihood for transcription suddenly increases ), and achieving a turning point at the threshold value \beta \approx 0.55 (i.e., beyond which the likelihood for transcription  reversed for \alpha values).
Revised:  index \beta, causing a bifurcation for the likelihood of transcription at the critical non-Gaussianity index value \alpha=1 (i.e., beyond which the likelihood for transcription suddenly increases ), and achieving a turning point at the threshold value \beta \approx 0.55 (i.e., beyond which the likelihood for transcription suddenly reversed for \alpha values).
Edit intent: fluency
Original:  We conduct a series of numerical experiments about `regulating' the likelihood of gene transcription by tuning asymmetric stable L\'evy noise indexes.
Revised:  While in the multiplicative noise case, non-Gaussianity index value \alpha=1 is a separating point or line for both the mean first exit time (MFET) and the first escape probability (FEP). We also investigate the noise enhanced stability phenomenon. Additionally, we are able to specify the regions in the whole parameter space for the asymmetric noise, in which we attain desired likelihood for transcription. We have conducted a series of numerical experiments about `regulating' the likelihood of gene transcription by tuning asymmetric stable L\'evy noise indexes.
Edit intent: meaning-changed
Original:  We have characterized yeast telomere chromatin structure, phenotypic heritability, and telosome segregation in both wild-type [MRE11] and a specific class of mre11 mutants.
Revised:  In this study, we characterize yeast telomere chromatin structure, phenotypic heritability, and telosome segregation in both wild-type [MRE11] and a specific class of mre11 mutants.
Edit intent: clarity
Original:  We have characterized yeast telomere chromatin structure, phenotypic heritability, and telosome segregation in both wild-type [MRE11] and a specific class of mre11 mutants.
Revised:  We have characterized yeast telomere chromatin structure, phenotypic heritability, and chromatin segregation in both wild-type [MRE11] and a specific class of mre11 mutants.
Edit intent: clarity
Original:  We have characterized yeast telomere chromatin structure, phenotypic heritability, and telosome segregation in both wild-type [MRE11] and a specific class of mre11 mutants. Wild-type strains confer a telomere size of 300 bp of G+T simple sequence DNA . This DNA  is embedded in a telosome: a micrococcal nuclease-resistant non-nucleosomal particle.
Revised:  We have characterized yeast telomere chromatin structure, phenotypic heritability, and telosome segregation in both wild-type [MRE11] and A470 motif alleles. MRE11 strains confer a telomere size of 300 bp of G+T simple sequence DNA . This DNA is embedded in a telosome: a micrococcal nuclease-resistant non-nucleosomal particle.
Edit intent: meaning-changed
Original:  Wild-type strains confer a telomere size of 300 bp of G+T simple sequence DNA . This DNA  is embedded in a telosome: a micrococcal nuclease-resistant non-nucleosomal particle.
Revised:  Wild-type strains confer a telomere size of 300 base pairs of G+T simple sequence DNA . This DNA is embedded in a telosome: a micrococcal nuclease-resistant non-nucleosomal particle.
Edit intent: clarity
Original:  Wild-type strains confer a telomere size of 300 bp of G+T simple sequence DNA . This DNA  is embedded in a telosome: a micrococcal nuclease-resistant non-nucleosomal particle.
Revised:  Wild-type strains confer a telomere size of 300 bp of G+T irregular simple sequence repeats . This DNA is embedded in a telosome: a micrococcal nuclease-resistant non-nucleosomal particle.
Edit intent: clarity
Original:  Wild-type strains confer a telomere size of 300 bp of G+T simple sequence DNA . This DNA  is embedded in a telosome: a micrococcal nuclease-resistant non-nucleosomal particle.
Revised:  Wild-type strains confer a telomere size of 300 bp of G+T simple sequence DNA . This DNA and a portion of subtelomeric DNA is embedded in a telosome: a micrococcal nuclease-resistant non-nucleosomal particle.
Edit intent: meaning-changed
Original:  Wild-type strains confer a telomere size of 300 bp of G+T simple sequence DNA . This DNA  is embedded in a telosome: a micrococcal nuclease-resistant non-nucleosomal particle.
Revised:  Wild-type strains confer a telomere size of 300 bp of G+T simple sequence DNA . This DNA is embedded in a telosome: an MNase-resistant non-nucleosomal particle.
Edit intent: clarity
Original:  The mre11A470Tmutation confers shorter 150 bp telomeric embedded within a telosome and is more resistant to micrococcal nuclease digestion than wild-type cells Interestingly, real-time qPCR suggests a lower or more transient binding of the mutant relative to the wild-type .
Revised:  Chromatin immunoprecipitation shows a three to four-fold lower occupancy of Mre11A470T proteins than wild-type cells Interestingly, real-time qPCR suggests a lower or more transient binding of the mutant relative to the wild-type .
Edit intent: meaning-changed
Original:  The mre11A470Tmutation confers shorter 150 bp telomeric embedded within a telosome and is more resistant to micrococcal nuclease digestion than wild-type cells Interestingly, real-time qPCR suggests a lower or more transient binding of the mutant relative to the wild-type .
Revised:  The mre11A470Tmutation confers shorter 150 bp telomeric embedded within a telosome and is more resistant to micrococcal nuclease digestion than wild-type proteins in telosomes. Telosomes containing the Mre11A470T protein confer a greater resistance to MNase digestion than wild-type .
Edit intent: clarity
Original:  The mre11A470Tmutation confers shorter 150 bp telomeric embedded within a telosome and is more resistant to micrococcal nuclease digestion than wild-type cells Interestingly, real-time qPCR suggests a lower or more transient binding of the mutant relative to the wild-type . Yet the introduction of wild-type Mre11 does not change the telomere size of the mutant cells and maintains the telosome structure of mutant cells.
Revised:  The mre11A470Tmutation confers shorter 150 bp telomeric embedded within a telosome and is more resistant to micrococcal nuclease digestion than wild-type cells Interestingly, real-time qPCR suggests a lower or more transient binding of the mutant relative to the wild-type telosomes. The integration of a wild-type Mre11 does not change the telomere size of the mutant cells and maintains the telosome structure of mutant cells.
Edit intent: coherence
Original:  Yet the introduction of wild-type Mre11 does not change the telomere size of the mutant cells and maintains the telosome structure of mutant cells. Similarly, the introduction of mre11A470T gene into a wild type genomic locus leads to a diffuse/wild-type chromatin phenotype. Our data show that telosomes predominantly restrict both MRE11  and  mre11A470T telosomes exclusively to sister chromatids, suggesting conservative segregation.
Revised:  Yet the introduction of wild-type MRE11 and mre11A470T telosomes exclusively to sister chromatids, suggesting conservative segregation.
Edit intent: coherence
Original:  Our data show that telosomes predominantly restrict both MRE11  and  mre11A470T telosomes exclusively to sister chromatids, suggesting conservative segregation.
Revised:  Our data show that telosomes predominantly restrict both MRE11 allele into an ectopic locus in the genome of a mre11A470T mutant and mre11A470T telosomes exclusively to sister chromatids, suggesting conservative segregation.
Edit intent: meaning-changed
Original:  Our data show that telosomes predominantly restrict both MRE11  and  mre11A470T telosomes exclusively to sister chromatids, suggesting conservative segregation. To show this, we used ectopic integration to characterize cells that carry either the MRE11 or mre11A470T alleles at the genomic g%DIFDELCMD < ] %%% site, and a second opposing mre11A470T or MRE11 allele, respectively, at an ectopic site.
Revised:  Our data show that telosomes predominantly restrict both MRE11 and mre11A470T g%DIFDELCMD < ] %%% site, and a second opposing mre11A470T or MRE11 allele, respectively, at an ectopic site.
Edit intent: clarity
Original:  To show this, we used ectopic integration to characterize cells that carry either the MRE11 or mre11A470T alleles at the genomic g%DIFDELCMD < ] %%% site, and a second opposing mre11A470T or MRE11 allele, respectively, at an ectopic site.
Revised:  To show this, we used ectopic integration to characterize cells that carry either the MRE11 or mre11A470T alleles at the genomic %DIFDELCMD < ] %%% site, and a second opposing mre11A470T or MRE11 allele, respectively, at an ectopic site.
Edit intent: fluency
Original:  To show this, we used ectopic integration to characterize cells that carry either the MRE11 or mre11A470T alleles at the genomic g%DIFDELCMD < ] %%% site, and a second opposing mre11A470T or MRE11 allele, respectively, at an ectopic site. We find that segregation of MRE11 or mre11A470T telosomes is dependent primarily on its initial genotype, but not on any subsequent genotypic change supporting the conservative segregation of a replicating chromatid
Revised:  To show this, we used ectopic integration to characterize cells that carry either the MRE11 or mre11A470T alleles at the genomic g%DIFDELCMD < ] %%% allele at an ectopic site in a wild-type strain lead to unexpectedly differing results. In each case, the replicated sister chromatids inherit telosomes containing only the protein encoded by the genomic mre11 locus, even in the presence of protein encoded by the opposing ectopic allele. We hypothesize that the telosome segregates by a conservative mechanism. These data support a mechanism for the linkage between sister chromatid replication and maintenance of either identical mutant or identical wild-type telosomes after replication of sister chromatids. These data suggest the presence of an active mechanism for chromatin segregation in yeast.
Edit intent: meaning-changed