female transsexuals is similar to that of heterosexual women-control. The female-to-male transsexual had a INAH3 volume and number of neurons within the range of male control, although that treatment with testosterone that person out stopped three years before her death (3) (http://1drv.ms / 1qcY97M). The results and comparisons with the control group indicate that the feminization of the interstitial nucleus of the anterior hypothalamus 3 (INAH3) from transgender woman was not due to estrogen treatment performed and proposes that sexual inversion. INAH3 in transsexual people is at least part, a marker of early sexual differentiation of the brain and that the changes in INAH3 and can belong to a complex network that may structurally and functionally be related to gender identity (3) (http://1drv.ms/1qcY97M ). I know it is complicated for those who are outside the biological sciences, neurological, etc, but take a look at the figures, especially http://1drv.ms/1qcY97M figure 8, which shows that male brains have more cells in the same region (black dots) INAH3 than female brains ... and that transgender men-to-women seem more like brains of women. In the branch of the science of genetics several studies have been conducted to investigate hereditary origins of homosexuality. One of the best known studies in this direction attempts to establish a correlation between male homosexuality with the Xq28 gene. It is effectively a thesis that puts homosexuality not as a choice or lifestyle, but rather as a result of genetic variation. Consider, however, that there are studies that contradict the influence of the Xq28 gene for explaining homosexual. In a new study conducted by KAIST was possible to artificially create homosexual behavior in female rats through genetic manipulation. In this study, some genes related to balance the hormone estrogen have been intentionally suppressed by checking a statistically relevant homosexual preference to a control group, launching new evidence that homosexual behavior in mammals may have a genetic factor. "Epigenetic 'tags' linked to homosexuality in men... Twin study reveals five DNA markers that are associated with sexual orientation.Twin studies and family trees provide strong evidence that sexual orientation is at least partly genetic. When one identical twin is gay, there is about a 20% chance that the other will be as well1. But because this rate is not 100%, it is thought that environmental factors play a role as well. One of the best characterized is the 'older brother effect': the chance of a man being gay increases by 33% for each older brother he has2. The reason is not clear, although one hypothesis holds that the mothers immune system begins to react against male antigens and alter the fetuss development.To search for factors that could mediate a link between environment and genes, geneticist Eric Vilain at the University of California, Los Angeles (UCLA), and his colleagues looked at epigenetic markers chemical changes to DNA that affect how genes are expressed, but not the information they contain. These 'epi-marks' can be inherited, but can also be altered by environmental factors such as smoking, and are not always shared by identical twinsThe researchers collected DNA