com a presenca do H. pylori causando infeccao gastrica.E, os pacientes dispepticos que afirmam possuirem halitose na sua maioria tambem sao portadores desta bacteria no estomago. Espera-se que pesquisas futuras esclarecam o verdadeiro mecanismo de participacao de H. pylori na contribuicao do mau halito. ABSTRACT The aim of this work is to promote a research among patients who have chronic dyspeptic complains with a high indication for digestive endoscopic investigation, its findings, Helicobacter pylori research in gastric mucosa and its possible correlation with the increase on volatile sulphur compounds in the oral cavity. Dyspepsia is defined as abdominal or retrosternal pain or discomfort, pyrosis, nausea, vomit and any other symptom related to the proximal digestive tube. It is classified as organic when it is secondary to anatomic lesions (gastro-duodenum lesions, cancer, esophagitis, biliopancreatic diseases) and non-ulcerous when the symptoms were neither related to organic disease nor to physical exercises. The understanding of upper gastrointestinal tract diseases was revolutionized since the isolation of the bacterium H. pylori on its tissues. Its relationship with the ethiopathogeny of the ulcerous peptic disease, gastric cancer, and gastric lymphoma is a real fact. The observation of gastroenterologic investigators about the improvement of halitosis complaints in patients, who had a clearing of H. pylori in the gastric mucosa, after its treatment, suggests the hypothesis that this bacterium would be involved in the ethiopathogeny of the formation of Volatile Sulphur Compounds and other gases compound bad breath. When taking measurements of Volatile Sulphur Compounds in these patients and comparing the findings of H. pylori in the gastric mucosa, we can observe an increase of gases that are part of bad odor in patients who are positive for this infection. Key-words: Dyspepsia. Helicobacter pylori. Volatile Sulphur Compounds. Halitosis REFERENCIAS BIBLIOGRAFICASAvcu N, Avcu F, Beyan C, Ural AU, Kaptan K, Ozyurt M, Nevruz O, Yalcin A, Turkey A. The relationship between gastric-oral helicobacter pylori and oral hygiene in patients with vitamin B12 - deficiency anemia. Oral surg oral med oral pathol oral radiol endod 2001; 92:166-9.Bernander S, Dalen J, Gastrin B, Hedenborg L, Lamke LO, Ohrn R. Absence of helicobacter pylori in dental plaques in helicobacter pylori positive dyspeptic patients. Eur. J. Clin. Microbiol. Infect. Dis. 1993; 282.Bussac G. Helicobacter pylori and the oral environment. Pract periodontics aesthet dent 1999; 11(8): 918-22.Cave David R. Transmission and epidemiology of helicobacter pylori. Am J Med 1996; 100:12S-18S.Deltenre M, Kasten E. How come Ive got it? (A review of helicobacter pylori transmission). Eur J Gastroent Hepatol 2000; 12(.)479-82.Eapen T, Chuancang J, David SC, Chuanfu L, Donald AF. The role of the oral cavity in helicobacter pylori infection. The American Journal of Gastroenterology 1997; 92(12): 2148.Hoshi K, YamanoY, Mitsunaga A, Shimizu S, Kagawa J, Ogiuch H. Gastrointestinal diseases and halitosis: association of gastric helicobacter pylori infection. Internation Dental journal 2002; 52 (Suppl 3): 207-11.Ierardi E, Amoruso A, La Notte T, Francavilla R, Castellaneta S, Marazza E, Monno AR, Francavilla A. Halitosis and helicobacter pylor: a possible relationship. Digestive diseases and sciences. Dec 1998; 43 (12): 2733-37.Kilmartin CM. Dental implications of helicobacter pylori. J can dent