Talk:Hypokalemia

message: According to NCBI http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2769974/  — Preceding unsigned comment added by Hippypink (talk • contribs) 16:36, 4 September 2013 (UTC)
thread_title:  Can be Caused by Hypoglycemia 

message: As I understand hypokalemia, and its causes, I believe it can also be genetic.
:I'm glad you understand hypokalemia! Honestly, there are a few hereditary conditions in which there is low potassium (e.g. Bartter syndrome, periodic paralysis), but the vast majority is not genetic. JFW | T@lk 06:50, 1 August 2005 (UTC)
replies: text: Decreased extracellular potassium levels cause a greater difference between intracellular and extracellular potassium levels, thus creating hyperpolarization. Still potassium is indeed needed for repolarization. But even with low potassium levels, the cell is able to pump potassium into the cell (using NaKATPase). The repolarization will take longer though. The first place where this will be problematic is in the heart with hypokalemia leading to arrythmia's. A total lack of potassium would lead to death very quickly. --WS 11:15, 15 December 2005 (UTC)
thread_title:  Hypokalemia 

message: Noticed that User:Danieliralevy removed the Muscular Dystrophy template I placed on the page yesterday.  I have no qualms with him doing so but am hoping to resolve the reasoning why others have sated on the main Muscular Dystrophy page that Hypokalemia is classified as muscular dystrophy under "Less Common Myophathies", "Periodic Paralysis".  It may well be that the muscular dystrophy page is wrong and would like to correct that if so.  Any thoughts/ideas/sources would be appreciated.  Theflyer 04:33, 8 March 2007 (UTC)
replies: text: Muscular dystrophy is an inherited disorder that causes progressive muscle weakness (myopathy) and loss of muscle mass (atrophy) due to defects in genes required for normal functioning of muscle.  Severe hypokalemia (and hyperkalemia) can cause muscle weakness, but the symptoms only occur until the electrolyte disorder is corrected.  A seperate set of hereditary disorders, hypokalemic & hyperkalemic periodic paralysis, are hereditary (genetic).  These disorders are paroxysmal (come on as acute attacks) and are associated with defective ion channels that can either make the muscle more sensitive to the abnormal potassium concentration, and/or can cause shifts of potassium in or out of the cells which causes the electrolyte disorder.  The periodic paralyses are nueromuscular disorders, but certainly not muscular dystrophies.  I believe that putting that long block of muscle disorders on a hyperkalemia or hypokalemia page is not at all appropriate.  However, I can certainly add a few lines to the causes and pathophysiology sections.Dan Levy 15:19, 8 March 2007 (UTC)
replies: text: Dan, Thank you for the response.  Please feel free to add information as you feel appropriate to the Hypokalemia page about its relationship to MD.  I do not have a problem with the Hypokalemia's editors deciding against using the Muscular Dystrophy template.  More importantly, it appears we may need to fix something in Wikipedia because the Muscular Dystrophy article does link Hypokalemic and Hyperkalemic, which you claim are different diseases from hypokalemia and hyperkalemia.  If this is true, then we definitely need to break those redirects and make separate articles for the hereditary versions.  I'm also engaging over on the Talk:Muscular dystrophy page to see if I can get better sourcing/information on why Hypokalemic and Hyperkalemia are "Generally classified as Muscular Dystrophy".  Theflyer 14:15, 10 March 2007 (UTC)
thread_title: Relationship of Hypokalemia and Muscular Dystrophy

message: Removed explicit mention of Cohn's syndrome as that's covered under adrenal tumors a few words later.  Some cases of Cushing's syndrome (mostly ectopic ACTH-producing tumors) are thought to overwhelm the 11-β hydroxylase enzyme of the renal tubules.  This gives an effect like glycyrrhizin and can cause hypokalemia. Dan Levy 19:08, 9 March 2007 (UTC)
thread_title:  Cushing's 

message: Does potassium deficiency cause hair loss.  I am going through chemo which shouldn't be causing hair loss, but am losing some, not all.  Thought maybe due to my postassium deficiency?
:No relationship between the two.  I'll elaborate more on the TALK page for 72.47.38.162 Dan Levy 21:17, 15 June 2007 (UTC)
thread_title:  Hair Loss 

message: I've heard of tetany as a symptom of hypocalcaemia before , but not hypokalaemia; is there a source for this?  —Preceding unsigned comment added by Heyeyya (talk • contribs) 14:22, 24 October 2010 (UTC)
replies: text: Agreed.  I see tetany in a textbook, but it doesn't seem to be reported in the primary literature. D.I.L. (talk) 13:50, 25 October 2010 (UTC)
thread_title:  Tetany? 

message: I'm not sure I get the physiological mechanism behind K+ excretion in the kidney in alkalosis following excessive vomiting.
The article states that "an acute rise of plasma HCO3- concentration (caused by vomiting, for example) will exceed the capacity of the renal proximal tubule to reabsorb this anion, and potassium will be excreted as an obligate cation partner to the bicarbonate."
Am I right in thinking the following:
The rise in serum bicarbonate results from Henderson-Hasselbalch style buffering of the lost H+ from the vomit via carbonic acid?
I gather that bicarb reabsorption occurs after dissociation by carbonic anhydrase to CO2 and OH-, which, combined with H+, cross into the luminal cell, where they reform HCO3-. With an excess of serum and thus filtrate bicarb, there is an insufficient amount of H+ to permit transport of HCO3- back into the cell. I presume this is what is meant by exceeding the capacity to reabsorb this anion.
I'm not quite sure where potassium fits in, however. I had read that there is an H+/K+ antiporter in alpha intercalated cells of the cortical collecting tubule. Would minimal H+ as possible inactivate the antiporter and prevent reabsorption of potassium from the urine?  
I'm not quite sure what is meant by K+ being the obligate cation. What exactly does this mean, and how is this borne out physiologically? Are there more channels involved than what I've mentioned?
You'll excuse me if I've missed the point, I'm just trying to get my head round this one as a student studying some of the unpleasant consequences that can occur in bulimia.
Ultimatebeej (talk) 23:14, 10 February 2011 (UTC)
thread_title:  Hypokalaemia in hyperemetic alkalosis 

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thread_title:  External links modified 

message: Currently the article claims the following
: The hypokalemia is thought to be from the combination of the diuretic effect of caffeine and copious fluid intake, although it may also be related to diarrhea caused by heavy fructose ingestion. A physiological response to hypercapnia, blood potassium (as well as calcium) helps offset acidosis, which is consistent with chronic, extreme consumption of carbonated beverages.
AFAIK, not much of any CO2 present in carbonated drinks makes it into the circulatory system - so I find this highly dubious. Maybe someone more knowledgeable can shed some light here? Tony Mach (talk) 11:28, 3 November 2016 (UTC)
thread_title:  Carbonated beverages and acidosis 

message: *An in depth description of an ECG is not needed in a caption "The 12 leads are displayed in 4 columns with 3 leads per column as well as a Lead 2 rhythm strip across the entire length at the bottom. "
*Diagnosis is based on a blood potassium not "Blood chemistries"
*Our goal is not to make the first sentence of an article as difficult to parse as possible. This does not need to go in the first sentence we are not a dictionary. "(derived from hypo- under; kalium - potassium, and emia - condition of the blood),"
*"The speed at which potassium should be replaced" was perfect fine. No need to change it to "rapidity"
*Not sure why this was removed "Hyperkalemia refers to a high level of potassium in the blood serum."
Doc James (talk · contribs · email) 08:25, 15 March 2019 (UTC)
thread_title: Text

message: — Assignment last updated by Jessicacariello (talk) 14:56, 14 February 2023 (UTC)
thread_title: Wiki Education assignment: STS 1010

message: (sorry for my poor English!) Reading
https://en.wikipedia.org/wiki/Hypokalemia#Urinary_loss I cannot find the possibility (maybe) of a low intake in sodium causing the body to expel / reject potassium in urine, thus creating a (secondary) hypokalemia in an attempt to keep an acceptable balance between sodium and potassium. Do I misunderstand something? Or can a low intake in sodium create an hypokalemia? I cannot find references about that, but would not it be logical? Or am I missing something? Thanks to you experts for clarifying this question! Regards! 130.239.235.218 (talk) 14:16, 2 September 2023 (UTC)
thread_title:  Sodium and hypokalemia