Patent RU-2307592-C1 (RU)
Inventor: GRECHISHNIKOV VLADIMIR VLADIMI
IPC: A61B10/00
Filed: 2006-01-10
Granted: 2007-10-10
Abstract: FIELD: medicine. 
     SUBSTANCE: method involves carrying out basic diagnostic examination. Focal and local estimation is carried out in marginal, lateral, apical, interradical, furcation periodontium tissue segments, including alveolar process periosteum within examined jaw sector. Pain sensitivity and tooth mobility is estimated under percussion. Inflammatory-destructive injury criteria characteristic of focal forms are defined in spreading from marginal segment towards the apical one. Dental deposit quantity is determined from formula of OHI-S=DI-S+CI-S, where DI-S is the quantity of soft dental deposit, CI is the supra- and subgingival tartar availability. Gingival retraction is estimated along tooth surface from oral, vestibular, approximal surfaces. This parameter is determined on four dental surfaces adjacent with gingiva, within the sector under study. Points are subsequently summed on each tooth part into single value on sector. Pocket depth is estimated in the same way on four dental surfaces, and points are summed on each dental surface within the sector. Discharge from the pocket is estimated. Alveolar partition destruction is determined. Points are subsequently summed within the boundaries of the sector under study. Gingival hemorrhage degree is determined. Papillary marginal alveolar index value is determined in the like way by papilla inflammation, marginal gingiva inflammation and alveolar gum taken into account. Then, periodontium tissue state estimation criteria characteristic of local injury forms are determined. Focus destruction size, destruction focus localization greater than 0.6 cm with periapical, periradical and furcation zone of periodontium are taken into account together with the data on destruction focus localization greater than 0.6 cm including cyste, cystogranulema, diffuse destruction having dark patch in the center and an lightening towards periphery with bone pattern from 0.6 sm and more having periosteum injury being retained; data on destruction development in periradical, interradical, periapical and furcation periodontium segments in a combination with untreated pulpitis, untreated periodontitis which has been filled root channel to apex, the root canal with unfinished filling having 1/3 left to the apex, the root canal with unfinished filling having 2/3 left to Multiroot tooth being the case, the points are summed on each root. Etiologic data are estimated after preliminary electric odontodiagnostic examination as: pulpitic tooth having the coronal part undestroyed or with a seal being available; destruction in lateral, interradical or furcational segment -retrograde pulpitis or filling-toxic one, pulpitic tooth with carious cavity being available, destruction in lateral, interradical or furcational segment, root perforation without tool break in pericervical quarter, perforation without break in the second tooth root quarter beginning from the crown, perforation without break in the third quarter, perforation without break in apical quarter, perforation without break in furcation zone, perforation with the tool break, pin inlay or other alien body brought into periodontium, in pericervical quarter, perforation with break or exit of foreign body into the second root quarter, perforation in the third root quarter with break or exit of foreign body, perforation in apical zone with break or exit of foreign body, perforation with break or exit of foreign body into bifurcation zone. Data on root fracture available in combination with destructive process in periodontium: root fracture in apical one-third, root fracture in middle one-third, root fracture in pericervical one-third, root fracture in dental cervix. Then the received values are summed and the received values are separately compared to the focal and the local destructive process form. Their proportion being greater or equal to 4:1, the process is considered to be focal one with distribution from marginal segment in apical direction. Their proportion being smaller or equal to 1:4, the process develops in local way with propagation into deep periodontium structures or to alveolar process surface. The way allows to estimate with character of defeat, , distribution of inflammatory destructive process in periodontium fabrics, and, accordingly, to pick up a complex of necessary actions for regenerative therapy of periodontium, to predict an outcome of therapeutic influence. 
     EFFECT: high reliability in estimating injury nature, spread and localization of inflammatory processes in periodontium tissue; high accuracy of therapy outcome prognosis. 
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