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Posterior leukoencephalopathy following cisplatin, bleomycin and vinblastine therapy for germ cell tumor of the ovary. | 1Related
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Posterior leukoencephalopathy following cisplatin, bleomycin and vinblastine therapy for germ cell tumor of the ovary. | 1Related
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Posterior leukoencephalopathy following cisplatin, bleomycin and vinblastine therapy for germ cell tumor of the ovary. | 1Related
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After 5 days of treatment with IL-2, the patient developed a hemorrhagic lesion that progressed to toxic epidermal necrolysis, as well as grade 4 pancytopenia. | 1Related
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After 5 days of treatment with IL-2, the patient developed a hemorrhagic lesion that progressed to toxic epidermal necrolysis, as well as grade 4 pancytopenia. | 1Related
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After 5 days of treatment with IL-2, the patient developed a hemorrhagic lesion that progressed to toxic epidermal necrolysis, as well as grade 4 pancytopenia. | 1Related
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OBJECTIVE: To report a case of cutaneous and hematologic toxicity in a patient treated with IL-2. | 1Related
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Subcutaneous IL-2 is safe and well tolerated, with a mortality rate <3%. | 1Related
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Toxic epidermal necrolysis associated with interleukin-2. | 1Related
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A case of anaphylactoid reaction due solely to the use of Gelofusine in a patient with non-haemorrhagic hypovolaemia is presented, with a discussion on the management and the use of allergy identification jewellery. | 1Related
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Gelofusine allergy--the need for identification jewellery. | 1Related
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The authors describe valproate-induced hyperammonemia and mental status changes in an 88-year-old man, the first known reported case in an elderly patient. | 1Related
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The authors describe valproate-induced hyperammonemia and mental status changes in an 88-year-old man, the first known reported case in an elderly patient. | 1Related
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Valproate-induced hyperammonemia as a cause of altered mental status. | 1Related
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Valproate-induced hyperammonemia as a cause of altered mental status. | 1Related
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Leiomyosarcoma in urinary bladder after cyclophosphamide therapy for retinoblastoma and review of bladder sarcomas. | 1Related
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The epivodes of NMS occured under treatment with clozapine, risperidone, and amisulpride. | 1Related
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The epivodes of NMS occured under treatment with clozapine, risperidone, and amisulpride. | 1Related
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The epivodes of NMS occured under treatment with clozapine, risperidone, and amisulpride. | 1Related
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A 73-year-old woman presented with fever and cough 2 weeks after completing the third cycle of fludarabine for chronic lymphocytic leukemia (CLL). | 1Related
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A 73-year-old woman presented with fever and cough 2 weeks after completing the third cycle of fludarabine for chronic lymphocytic leukemia (CLL). | 1Related
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Fludarabine induced lung toxicity must be considered in all patients who develop unexplained lung disease while receiving fludarabine. | 1Related
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Fludarabine induced lung toxicity must be considered in all patients who develop unexplained lung disease while receiving fludarabine. | 1Related
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Fludarabine induced lung toxicity must be considered in all patients who develop unexplained lung disease while receiving fludarabine. | 1Related
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Multiple pulmonary nodules: an unusual presentation of fludarabine pulmonary toxicity: case report and review of literature. | 1Related
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Multiple pulmonary nodules: an unusual presentation of fludarabine pulmonary toxicity: case report and review of literature. | 1Related
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This case extends the spectrum of fludarabine pulmonary toxicity to include pulmonary nodules. | 1Related
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This case extends the spectrum of fludarabine pulmonary toxicity to include pulmonary nodules. | 1Related
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To our knowledge, four cases of interstitial pneumonitis associated with fludarabine have been reported in medical literature. | 1Related
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Epoprostenol may be associated rarely with severe erythroderma. | 1Related
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Severe erythroderma as a complication of continuous epoprostenol therapy. | 1Related
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We report a case of a patient with pulmonary hypertension and undifferentiated connective tissue disease who, after 2 months of treatment with epoprostenol, presented with rapidly progressive erythema, scaling, nausea and vomiting, and fever. | 1Related
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We report a case of a patient with pulmonary hypertension and undifferentiated connective tissue disease who, after 2 months of treatment with epoprostenol, presented with rapidly progressive erythema, scaling, nausea and vomiting, and fever. | 1Related
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We report a case of a patient with pulmonary hypertension and undifferentiated connective tissue disease who, after 2 months of treatment with epoprostenol, presented with rapidly progressive erythema, scaling, nausea and vomiting, and fever. | 1Related
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We report a case of a patient with pulmonary hypertension and undifferentiated connective tissue disease who, after 2 months of treatment with epoprostenol, presented with rapidly progressive erythema, scaling, nausea and vomiting, and fever. | 1Related
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We report a case of a patient with pulmonary hypertension and undifferentiated connective tissue disease who, after 2 months of treatment with epoprostenol, presented with rapidly progressive erythema, scaling, nausea and vomiting, and fever. | 1Related
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A 50-year-old diabetic and hypertensive male patient is reported who had ticlopidine-induced marrow aplasia partially responsive to colony-stimulating factors and corticosteroids, but experienced complete recovery with cyclosporine. | 1Related
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There is no consensus on the treatment of ticlopidine-induced marrow aplasia. | 1Related
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Ticlopidine-induced marrow aplasia treated with cyclosporine. | 1Related
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A 53-year-old man developed lower leg edema 4 weeks after rosiglitazone was increased from 4 mg once/day to 4 mg twice/day. | 1Related
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A 75-year-old man developed bilateral lower leg edema 6 months after switching from troglitazone to pioglitazone. | 1Related
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A 77-year-old man developed ankle, hand, and facial swelling 2 weeks after starting rosiglitazone. | 1Related
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Two cases of polymorphic ventricular tachycardia induced by the administration of verapamil against paroxysmal supraventricular tachycardia. | 1Related
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Verapamil is widely used for the termination of paroxysmal supraventricular tachycardia (PSVT) with little proarrhythmic effect. | 1Related
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We describe two cases of PSVT that changed to non-sustained polymorphic ventricular tachycardia after administration of verapamil. | 1Related
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Epsilon-aminocaproic acid and renal complications: case report and review of the literature. | 1Related
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Neonatal mydriasis: intravenous lidocaine adverse reaction. | 1Related
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We present a neonate with a seizure disorder who acutely developed pupillary mydriasis secondary to lidocaine overdose. | 1Related
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We suggest adding this side effect to the list of untoward effects of lidocaine and to the differential diagnosis of fixed dilated pupils in neonates treated with lidocaine. | 1Related
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Myelodysplasia terminating in acute myeloid leukemia in a hairy cell leukemia patient treated with 2-deoxycoformycin. | 1Related
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Myelodysplasia terminating in acute myeloid leukemia in a hairy cell leukemia patient treated with 2-deoxycoformycin. | 1Related
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To the best of our knowledge only two previous cases of AML have been linked to treatment of HCL with purine analogs, both with 2-chlorodeoxyadenosine. | 1Related
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CONCLUSIONS: Peripheral administration of low-dose vasopressin for septic shock should be discouraged because of the risk of ischemic skin complications. | 1Related
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OBJECTIVE: To describe a case of severe skin necrosis resulting from peripheral intravenous administration of low-dose vasopressin in a patient with catecholamine-resistant septic shock. | 1Related
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Skin necrosis after extravasation of low-dose vasopressin administered for septic shock. | 1Related
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Mitomycin-C induced hemolytic uremic syndrome: a case report. | 1Related
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Mitomycin-C is used widely in the treatment of malignancies and is associated with serious dose related adverse effects including the occurrence of hemolytic uremic syndrome. | 1Related
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Although vasculitis has been reported in the course of hairy cell leukaemia, it has only rarely been reported as the consequence of cladribine treatment. | 1Related
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Systemic vasculitis complicating hairy cell leukaemia treatment with cladribine. | 1Related
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We describe a 73-year-old woman who developed serious systemic vasculitis with associated thrombocytopenia in the course of treatment with cladribine. | 1Related
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We describe a 73-year-old woman who developed serious systemic vasculitis with associated thrombocytopenia in the course of treatment with cladribine. | 1Related
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Drug-induced mammary hyperplasias have been reported as rare complications of D-penicillamine and Neothetazone. | 1Related
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Drug-induced mammary hyperplasias have been reported as rare complications of D-penicillamine and Neothetazone. | 1Related
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Gigantomastia induced by bucillamine. | 1Related
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Retrospectively, bucillamine was believed to be the cause of the giant hypertrophy because of its structural similarity to D-penicillamine, which was the subject of an abundance of reports of mammary hyperplasia. | 1Related
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Retrospectively, bucillamine was believed to be the cause of the giant hypertrophy because of its structural similarity to D-penicillamine, which was the subject of an abundance of reports of mammary hyperplasia. | 1Related
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The authors report the first case of bucillamine-induced giant mammary hyperplasia. | 1Related
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The authors describe three families in whom the occurrence of FVS in all the siblings strongly suggests hereditary susceptibility to valproic acid-induced adverse outcome. | 1Related
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The fetal valproate syndrome (FVS) is characterized by distinctive facial appearance, major and minor malformations, and developmental delay. | 1Related
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The fetal valproate syndrome (FVS) is characterized by distinctive facial appearance, major and minor malformations, and developmental delay. | 1Related
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The fetal valproate syndrome (FVS) is characterized by distinctive facial appearance, major and minor malformations, and developmental delay. | 1Related
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The fetal valproate syndrome (FVS) is characterized by distinctive facial appearance, major and minor malformations, and developmental delay. | 1Related
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The fetal valproate syndrome (FVS) is characterized by distinctive facial appearance, major and minor malformations, and developmental delay. | 1Related
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Valproate embryopathy in three sets of siblings: further proof of hereditary susceptibility. | 1Related
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Generalised pustular psoriasis induced by cyclosporin a withdrawal responding to the tumour necrosis factor alpha inhibitor etanercept. | 1Related
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We report a 50-year-old male patient with a 15-year history of psoriasis including mutilating psoriatic arthritis, in whom the withdrawal of cyclosporin A induced a generalised pustular exacerbation and a aggravation of the joint condition. | 1Related
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We report a 50-year-old male patient with a 15-year history of psoriasis including mutilating psoriatic arthritis, in whom the withdrawal of cyclosporin A induced a generalised pustular exacerbation and a aggravation of the joint condition. | 1Related
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The first case concerns a 70-year-old man who developed severe aplastic anemia 7 weeks after treatment with 500 mg of ticlopidine daily. | 1Related
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The second was an 82-year-old man receiving ticlopidine for 2 years when, during a febrile episode, he was found neutropenic with marrow aplasia. | 1Related
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The second was an 82-year-old man receiving ticlopidine for 2 years when, during a febrile episode, he was found neutropenic with marrow aplasia. | 1Related
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Ticlopidine-induced aplastic anemia (TIAA) is considered very uncommon. | 1Related
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Ticlopidine-induced aplastic anemia (TIAA) is considered very uncommon. | 1Related
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Ticlopidine-induced aplastic anemia: two new case reports, review, and meta-analysis of 55 additional cases. | 1Related
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When the data of the 57 patients are evaluated, a reversible direct cytotoxic effect of ticlopidine on the pluripotent/bipotent hematopoietic progenitor stem cell is proposed. | 1Related
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Fixed drug eruption to rofecoxib. | 1Related
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Rofecoxib, used for dysmenorrhea, caused a herpetiform fixed drug eruption predominantly involving the lips with classic clinical and histological findings in a red-brown lesion on the dorsal hand. | 1Related
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Rofecoxib, used for dysmenorrhea, caused a herpetiform fixed drug eruption predominantly involving the lips with classic clinical and histological findings in a red-brown lesion on the dorsal hand. | 1Related
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A patient who had been treated with large doses of thyroid hormone for several years developed features of secondary hypothyroidism after thyroid hormone withdrawal. | 1Related
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BACKGROUND: Gemcitabine has mild renal toxicity, but cases of gemcitabine-associated hemolytic-uremic syndrome (HUS) have been reported. | 1Related
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BACKGROUND: Gemcitabine has mild renal toxicity, but cases of gemcitabine-associated hemolytic-uremic syndrome (HUS) have been reported. | 1Related
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BACKGROUND: Gemcitabine has mild renal toxicity, but cases of gemcitabine-associated hemolytic-uremic syndrome (HUS) have been reported. | 1Related
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BACKGROUND: Gemcitabine has mild renal toxicity, but cases of gemcitabine-associated hemolytic-uremic syndrome (HUS) have been reported. | 1Related
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BACKGROUND: Gemcitabine has mild renal toxicity, but cases of gemcitabine-associated hemolytic-uremic syndrome (HUS) have been reported. | 1Related
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CONCLUSION: There are only a few confirmed cases of gemcitabine-associated HUS despite the widespread use of the drug. | 1Related
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Gemcitabine-associated hemolytic-uremic syndrome. | 1Related
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Mean time between initiation of gemcitabine therapy and onset of HUS was 7.4 +/- 3.5 months, or 21.9 +/- 10.9 doses of gemcitabine. | 1Related
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Mean time between initiation of gemcitabine therapy and onset of HUS was 7.4 +/- 3.5 months, or 21.9 +/- 10.9 doses of gemcitabine. | 1Related
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METHODS: A case is presented of a 45-year-old woman on prolonged gemcitabine treatment for ovarian cancer who developed HUS and recovered after drug discontinuation. | 1Related
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RESULTS: Including our own patient, a total of 26 cases of gemcitabine-associated HUS were identified. | 1Related
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An amiodarone optic neuropathy has been described. | 1Related
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