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Clinicians have been aware of lithium toxicity for many years and traditionally have administered thiazide diuretics for lithium-induced polyuria and nephrogenic diabetes insipidus. | 1Related
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Clinicians have been aware of lithium toxicity for many years and traditionally have administered thiazide diuretics for lithium-induced polyuria and nephrogenic diabetes insipidus. | 1Related
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Treatment of lithium-induced diabetes insipidus with amiloride. | 1Related
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Systemic capillary leak syndrome after granulocyte colony-stimulating factor (G-CSF). | 1Related
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Systemic capillary leak syndrome after granulocyte colony-stimulating factor (G-CSF). | 1Related
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We present two cases in which both patients suffered with CLS, which we believe was caused following administration of granulocyte colony-stimulating factor, to our knowledge not described in the intensive care patient previously. | 1Related
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FK506, which began to be administered 12 days earlier, rose to a level of 44 ng/mL (normal range, 10-20 ng/mL) 1 day before neurologic abnormalities began. | 1Related
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Rapid identification of speech loss linked to FK506 may be important because reduction or cessation of the drug may be associated with reverse of speech loss. | 1Related
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Tacrolimus (FK506), an immunosuppressant, has been associated with mutism in adults after liver transplant. | 1Related
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Tacrolimus (FK506), an immunosuppressant, has been associated with mutism in adults after liver transplant. | 1Related
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Tacrolimus (FK506)-induced mutism after liver transplant. | 1Related
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Tacrolimus (FK506)-induced mutism after liver transplant. | 1Related
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Does the use of insulin in a patient with liver dysfunction increase water retention in the body, i.e. cause insulin oedema? | 1Related
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Soon after introduction of insulin therapy, she developed severe anasarca, including marked peripheral oedema, ascites and pleural effusion. | 1Related
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Soon after introduction of insulin therapy, she developed severe anasarca, including marked peripheral oedema, ascites and pleural effusion. | 1Related
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Soon after introduction of insulin therapy, she developed severe anasarca, including marked peripheral oedema, ascites and pleural effusion. | 1Related
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Soon after introduction of insulin therapy, she developed severe anasarca, including marked peripheral oedema, ascites and pleural effusion. | 1Related
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The induced hyperglycaemia could not be controlled sufficiently, despite a high dose of insulin (> 110 units/day), suggesting the existence of insulin insensitivity and hyperinsulinaemia. | 1Related
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The induced hyperglycaemia could not be controlled sufficiently, despite a high dose of insulin (> 110 units/day), suggesting the existence of insulin insensitivity and hyperinsulinaemia. | 1Related
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We conjectured that the side effects of insulin, such as anti-natriuresis and increased vascular permeability, might be pronounced in the presence of the hepatic dysfunction that accompanies insulin insensitivity, hyperinsulinaemia and hypoalbuminaemia. | 1Related
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We conjectured that the side effects of insulin, such as anti-natriuresis and increased vascular permeability, might be pronounced in the presence of the hepatic dysfunction that accompanies insulin insensitivity, hyperinsulinaemia and hypoalbuminaemia. | 1Related
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We conjectured that the side effects of insulin, such as anti-natriuresis and increased vascular permeability, might be pronounced in the presence of the hepatic dysfunction that accompanies insulin insensitivity, hyperinsulinaemia and hypoalbuminaemia. | 1Related
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We conjectured that the side effects of insulin, such as anti-natriuresis and increased vascular permeability, might be pronounced in the presence of the hepatic dysfunction that accompanies insulin insensitivity, hyperinsulinaemia and hypoalbuminaemia. | 1Related
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We conjectured that the side effects of insulin, such as anti-natriuresis and increased vascular permeability, might be pronounced in the presence of the hepatic dysfunction that accompanies insulin insensitivity, hyperinsulinaemia and hypoalbuminaemia. | 1Related
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While 40 mg/day of prednisolone improved hepatic dysfunction dramatically, her diabetic milieu deteriorated seriously. | 1Related
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Intrathecal methotrexate-induced acute cerebellar syndrome. | 1Related
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We describe a patient who developed acute cerebellar syndrome after prophylactic intrathecal methotrexate administration and recovered spontaneously. | 1Related
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Angioimmunoblastic lymphadenopathy with dysproteinemia following doxycycline administration. | 1Related
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We report a case of AILD in an 80-year-old male who presented with a generalized pruritic maculopapular eruption and fever following doxycycline administration. | 1Related
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We report a case of AILD in an 80-year-old male who presented with a generalized pruritic maculopapular eruption and fever following doxycycline administration. | 1Related
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We report a case of AILD in an 80-year-old male who presented with a generalized pruritic maculopapular eruption and fever following doxycycline administration. | 1Related
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Anaphylaxis from isoniazid is a possible side effect to this commonly prescribed antibiotic. | 1Related
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Isoniazid-induced anaphylaxis. | 1Related
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A case is reported in which severe hypocalcemia, with a low plasma parathyroid hormone (PTH) concentration, resulted from the therapeutic use of magnesium sulfate for toxemia of pregnancy. | 1Related
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A case is reported in which severe hypocalcemia, with a low plasma parathyroid hormone (PTH) concentration, resulted from the therapeutic use of magnesium sulfate for toxemia of pregnancy. | 1Related
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A case is reported in which severe hypocalcemia, with a low plasma parathyroid hormone (PTH) concentration, resulted from the therapeutic use of magnesium sulfate for toxemia of pregnancy. | 1Related
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Carbamazepine-related hyponatremia following cardiopulmonary bypass. | 1Related
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We discuss the association between carbamazepine and hyponatremia and the causes of hyponatremia after cardiopulmonary bypass. | 1Related
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After 5-ASA was discontinued, the polyneuropathy symptoms recovered gradually. | 1Related
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Sensorimotor polyneuropathy with 5-aminosalicylic acid: a case report. | 1Related
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This clinical course suggests that the sensorimotor polyneuropathy may have been caused by 5-ASA. | 1Related
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When SASP was changed to 5-aminosalicylic acid (5-ASA), his skin eruptions were resolved, however, he developed weakness and atrophy in his right arm as well as progressive worsening of the dysesthesia in his legs and gait disturbance. | 1Related
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When SASP was changed to 5-aminosalicylic acid (5-ASA), his skin eruptions were resolved, however, he developed weakness and atrophy in his right arm as well as progressive worsening of the dysesthesia in his legs and gait disturbance. | 1Related
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When SASP was changed to 5-aminosalicylic acid (5-ASA), his skin eruptions were resolved, however, he developed weakness and atrophy in his right arm as well as progressive worsening of the dysesthesia in his legs and gait disturbance. | 1Related
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When SASP was changed to 5-aminosalicylic acid (5-ASA), his skin eruptions were resolved, however, he developed weakness and atrophy in his right arm as well as progressive worsening of the dysesthesia in his legs and gait disturbance. | 1Related
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When SASP was changed to 5-aminosalicylic acid (5-ASA), his skin eruptions were resolved, however, he developed weakness and atrophy in his right arm as well as progressive worsening of the dysesthesia in his legs and gait disturbance. | 1Related
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When SASP was changed to 5-aminosalicylic acid (5-ASA), his skin eruptions were resolved, however, he developed weakness and atrophy in his right arm as well as progressive worsening of the dysesthesia in his legs and gait disturbance. | 1Related
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When SASP was changed to 5-aminosalicylic acid (5-ASA), his skin eruptions were resolved, however, he developed weakness and atrophy in his right arm as well as progressive worsening of the dysesthesia in his legs and gait disturbance. | 1Related
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When SASP was changed to 5-aminosalicylic acid (5-ASA), his skin eruptions were resolved, however, he developed weakness and atrophy in his right arm as well as progressive worsening of the dysesthesia in his legs and gait disturbance. | 1Related
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Disseminated cellulitic cryptococcosis in the setting of prednisone monotherapy for pemphigus vulgaris. | 1Related
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In both cases, high fever, skin rash, liver dysfunction and atypical lymphocytosis developed 3 weeks after initiating treatment with SASP. | 1Related
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In both cases, high fever, skin rash, liver dysfunction and atypical lymphocytosis developed 3 weeks after initiating treatment with SASP. | 1Related
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In both cases, high fever, skin rash, liver dysfunction and atypical lymphocytosis developed 3 weeks after initiating treatment with SASP. | 1Related
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In both cases, high fever, skin rash, liver dysfunction and atypical lymphocytosis developed 3 weeks after initiating treatment with SASP. | 1Related
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Slow acetylator genotypes as a possible risk factor for infectious mononucleosis-like syndrome induced by salazosulfapyridine. | 1Related
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We report two patients with infectious mononucleosis-like syndrome induced by salazosulfapyridine (SASP). | 1Related
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We report two patients with infectious mononucleosis-like syndrome induced by salazosulfapyridine (SASP). | 1Related
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A 45-year-old woman with steroid-dependent Crohn's colitis, successfully managed with maintenance infliximab infusions and methotrexate, developed a lupus-like syndrome eight months after her initial infusion. | 1Related
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A 45-year-old woman with steroid-dependent Crohn's colitis, successfully managed with maintenance infliximab infusions and methotrexate, developed a lupus-like syndrome eight months after her initial infusion. | 1Related
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A lupus-like syndrome associated with infliximab therapy. | 1Related
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Infliximab therapy may cause a lupus-like syndrome that is reversible upon discontinuing this agent. | 1Related
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We report in detail an unusual adverse reaction to infliximab therapy, a drug-induced lupus-like clinical syndrome. | 1Related
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Chronic myelogenous leukemia (CML), hepatitis C, and interferon alpha (IFNalpha) have all been associated with renal dysfunction. | 1Related
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Chronic myelogenous leukemia (CML), hepatitis C, and interferon alpha (IFNalpha) have all been associated with renal dysfunction. | 1Related
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Despite the underlying hepatitis C, this case represents renal abnormalities consistent with IFNalpha therapy for CML. | 1Related
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Interferon-alpha-induced focal segmental glomerulosclerosis in chronic myelogenous leukemia: a case report and review of the literature. | 1Related
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The renal biopsy showed focal segmental glomerulosclerosis, which has only been previously reported in two cases of CML treated with IFNalpha. | 1Related
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Severe autoimmune hemolytic anemia following rituximab therapy in a patient with a lymphoproliferative disorder. | 1Related
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The pathophysiological mechanisms remain unknown, although the drug could act through massive cytokines liberation after destruction of CD20 positive cells by rituximab. | 1Related
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The pathophysiological mechanisms remain unknown, although the drug could act through massive cytokines liberation after destruction of CD20 positive cells by rituximab. | 1Related
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We report the first case, to our knowledge, of rituximab-related autoimmune hemolytic anemia. | 1Related
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Early-onset acute transverse myelitis following hepatitis B vaccination and respiratory infection: case report. | 1Related
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In this paper, we report a case of 3 years-old boy who developed acute onset tetraparesia following a viral respiratory infecction and hepatitis B vaccination. | 1Related
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Although it would be expected that, like other type IA toxicities, diphenhydramine-induced cardiotoxicity could be responsive to hypertonic sodium bicarbonate, this finding is largely unappreciated. | 1Related
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Diphenhydramine-induced wide complex dysrhythmia responds to treatment with sodium bicarbonate. | 1Related
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However, because diphenhydramine also exhibits type IA sodium channel blockade, cardiac toxicity is also possible. | 1Related
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We describe 3 cases of diphenhydramine-induced cardiac toxicity that were responsive to bicarbonate. | 1Related
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We describe a patient with extranodal non-Hodgkin lymphoma who developed systemic candidiasis after treatment with a cyclophosphamide-based chemotherapy regimen. | 1Related
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Stevens-Johnson syndrome in a boy with nephrotic syndrome during prednisolone therapy. | 1Related
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We report a case of SJS in a 14-year-old male with nephrotic syndrome, who was treated with oral prednisolone for 6 weeks. | 1Related
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A 25-year-old man with a history of mid-borderline (BB) Hansen's disease developing a reversal reaction after starting dapsone and rifampin therapy is presented. | 1Related
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A 25-year-old man with a history of mid-borderline (BB) Hansen's disease developing a reversal reaction after starting dapsone and rifampin therapy is presented. | 1Related
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However, in order to avoid neuropathic side effects, patients under thalidomide therapy should be monitored every 6 months with nerve conduction studies while taking the drug. | 1Related
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After a two-month interruption of interferon administration, natural interferon alpha was given but followed by another episode of the same neurological manifestations. | 1Related
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Case 1, a 62-year-old woman, developed bilateral optic neuritis with decreased sensation of vibration and increased deep tendon reflex in the lower extremities after a seven-month use of recombinant interferon alpha-2a for chronic active hepatitis C. | 1Related
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Case 1, a 62-year-old woman, developed bilateral optic neuritis with decreased sensation of vibration and increased deep tendon reflex in the lower extremities after a seven-month use of recombinant interferon alpha-2a for chronic active hepatitis C. | 1Related
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Case 1, a 62-year-old woman, developed bilateral optic neuritis with decreased sensation of vibration and increased deep tendon reflex in the lower extremities after a seven-month use of recombinant interferon alpha-2a for chronic active hepatitis C. | 1Related
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Case 2, a 29-year-old woman, developed bilateral optic neuritis combined with numbness of the lower extremities as well as bowel and bladder dysfunction after a 22-month use of recombinant interferon alpha-2b for chronic myelogenous leukemia. | 1Related
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Case 2, a 29-year-old woman, developed bilateral optic neuritis combined with numbness of the lower extremities as well as bowel and bladder dysfunction after a 22-month use of recombinant interferon alpha-2b for chronic myelogenous leukemia. | 1Related
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Case 2, a 29-year-old woman, developed bilateral optic neuritis combined with numbness of the lower extremities as well as bowel and bladder dysfunction after a 22-month use of recombinant interferon alpha-2b for chronic myelogenous leukemia. | 1Related
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CONCLUSIONS: Optic neuritis in combination with other neurological signs, simulating multiple sclerosis, should be included in the list of adverse effects of recombinant and natural interferon alpha administration. | 1Related
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CONCLUSIONS: Optic neuritis in combination with other neurological signs, simulating multiple sclerosis, should be included in the list of adverse effects of recombinant and natural interferon alpha administration. | 1Related
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Multiple sclerosis-like disease secondary to alpha interferon. | 1Related
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PURPOSE: To describe bilateral optic neuritis that occurred as an adverse effect of recombinant and natural interferon alpha administration. | 1Related
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Acute abdomen due to endometriosis in a premenopausal woman taking tamoxifen. | 1Related
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Acute abdomen due to endometriosis in a premenopausal woman taking tamoxifen. | 1Related
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We describe a premenopausal woman who, while having tamoxifen due to a diagnosis of in situ ductal carcinoma, developed endometriosis requiring surgery. | 1Related
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A case report of fatal dapsone-induced agranulocytosis in an Indian mid-borderline leprosy patient. | 1Related
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Fatal agranulocytosis in an Indian male receiving 100mg of dapsone daily, hospitalized for mid-borderline leprosy in type I reaction with triple nerve paralysis is reported. | 1Related
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Various case reports concerning dapsone-induced agranulocytosis are reviewed. | 1Related
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