D2-dopaminergic blockade does not influence post-exercise ketosis in non-athletes. 
Metoclopramide has previously been shown to inhibit the ketosis of starvation in rats and humans.  The effect of D2-dopaminergic blockade on post-exercise ketosis was, therefore, studied in 6 carbohydrate-starved non-athletic persons who had just completed a 9-km walk in mountainous terrain.  There were nine control subjects who went on the walk, but who did not ingest metoclopramide.  Metoclopramide (0.15 mg.kg-1 body mass) caused a highly significant rise in the plasma prolactin concentration, but did not influence blood concentrations of 3-hydroxybutyrate, free fatty acid, glucose, insulin or glucagon.  Unlike ketosis in starvation, therefore, neither prolactin, nor the D2-dopaminergic system play a part in the genesis of post-exercise ketosis.