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Massive CBZ OD may produce a reversible encephalopathy that includes cortical hyperexcitability, a profound burst-suppression EEG pattern, and cranial nerve areflexia. | 1Related
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Massive CBZ OD may produce a reversible encephalopathy that includes cortical hyperexcitability, a profound burst-suppression EEG pattern, and cranial nerve areflexia. | 1Related
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Massive CBZ OD may produce a reversible encephalopathy that includes cortical hyperexcitability, a profound burst-suppression EEG pattern, and cranial nerve areflexia. | 1Related
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Massive CBZ OD may produce a reversible encephalopathy that includes cortical hyperexcitability, a profound burst-suppression EEG pattern, and cranial nerve areflexia. | 1Related
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Massive plasmocytosis due to methimazole-induced bone marrow toxicity. | 1Related
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Massive plasmocytosis due to methimazole-induced bone marrow toxicity. | 1Related
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To our knowledge this is the first report of pancytopenia due to MMI, where the usual hypoplasia found is replaced by massive plasmocytosis. | 1Related
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To our knowledge this is the first report of pancytopenia due to MMI, where the usual hypoplasia found is replaced by massive plasmocytosis. | 1Related
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We report a case of fulminant hepatic failure associated with didanosine and masquerading as a surgical abdomen and compare the clinical, biologic, histologic, and ultrastructural findings with reports described previously. | 1Related
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This report details a case of bilateral avascular necrosis of the femoral heads in a patient receiving 'standard' doses of dexamethasone as part of the antiemetic regimen used in cisplatin-based combination chemotherapy. | 1Related
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Although no coagulation study was done and the Meckel's diverticulum is normally associated with bleeding, the particular intensity of the following hemorrhage may have been favored by metformin. | 1Related
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An obese patient, not diabetic, treated with metformin for some weeks, was referred to us with severe inferior digestive hemorrhage, diagnosed with Meckel's diverticulum. | 1Related
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Digestive hemorrhage caused by a Meckel's diverticulum in a metformin-treated patient: is there any connection? | 1Related
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Ulcer became worse after tobramycin and gentamycin treatment for 2 days. | 1Related
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Ulcer became worse after tobramycin and gentamycin treatment for 2 days. | 1Related
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Possible linkage of amprenavir with intracranial bleeding in an HIV-infected hemophiliac. | 1Related
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The bleeding resolved on discontinuation of APV. | 1Related
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We report the occurrence of spontaneous intracranial bleeding in an human immunodeficiency virus (HIV)-infected adolescent with hemophilia A who was receiving amprenavir (APV). | 1Related
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According to the Naranjo probability scale, the relationship of gemcitabine treatment with cutaneous eruption in our patient is possible. | 1Related
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CONCLUSIONS: We report the first case of gemcitabine-induced LABD. | 1Related
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Linear immunoglobulin A bullous dermatosis induced by gemcitabine. | 1Related
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OBJECTIVE: To report a case of linear immunoglobulin (Ig) A bullous dermatosis (LABD) induced by gemcitabine. | 1Related
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OBJECTIVE: To report a case of linear immunoglobulin (Ig) A bullous dermatosis (LABD) induced by gemcitabine. | 1Related
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Twenty-four hours after the administration of gemcitabine, a symmetric, bullous, herpetiform eruption appeared on his trunk and upper limbs. | 1Related
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Vancomycin is the most frequently implicated drug, but other agents have been reported to cause LABD. | 1Related
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Although taxol has shown significant activity in advanced ovarian cancer, peripheral neuropathy is likely to become the major dose-limiting toxicity. | 1Related
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A rare case of advanced ovarian carcinoma who developed difficulty walking 25 days after treatment with weekly paclitaxel. | 1Related
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We describe a case of advanced ovarian carcinoma who developed difficulty walking because of marked pain in the lower extremities and loss of proprioception 25 days after treatment with weekly taxol (80 mg/m(2)x3). | 1Related
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We describe a case of advanced ovarian carcinoma who developed difficulty walking because of marked pain in the lower extremities and loss of proprioception 25 days after treatment with weekly taxol (80 mg/m(2)x3). | 1Related
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We describe a case of advanced ovarian carcinoma who developed difficulty walking because of marked pain in the lower extremities and loss of proprioception 25 days after treatment with weekly taxol (80 mg/m(2)x3). | 1Related
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Relief by naloxone of morphine-induced spasm of the sphincter of Oddi in a post-cholecystectomy patient. | 1Related
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We present the case of a patient who had undergone cholecystectomy previously, but in whom morphine given in the Emergency Department precipitated pain consistent with biliary colic; the pain resolved promptly after administration of naloxone. | 1Related
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We present the case of a patient who had undergone cholecystectomy previously, but in whom morphine given in the Emergency Department precipitated pain consistent with biliary colic; the pain resolved promptly after administration of naloxone. | 1Related
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Myasthenia gravis during low-dose IFN-alpha therapy for chronic hepatitis C. | 1Related
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A patient with rheumatoid arthritis developed an acute intrahepatic cholestasis after 100 mg of sodium aurothiomalate. | 1Related
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Acute lung injury associated with 5-fluorouracil and oxaliplatinum combined chemotherapy. | 1Related
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Acute lung injury associated with 5-fluorouracil and oxaliplatinum combined chemotherapy. | 1Related
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Diarrhoea, T-CD4+ lymphopenia and bilateral patchy pulmonary infiltrates developed in a male 60 yrs of age, who was treated with oxaliplatinum and 5-fluorouracil for unresectable rectum carcinoma. | 1Related
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Diarrhoea, T-CD4+ lymphopenia and bilateral patchy pulmonary infiltrates developed in a male 60 yrs of age, who was treated with oxaliplatinum and 5-fluorouracil for unresectable rectum carcinoma. | 1Related
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Diarrhoea, T-CD4+ lymphopenia and bilateral patchy pulmonary infiltrates developed in a male 60 yrs of age, who was treated with oxaliplatinum and 5-fluorouracil for unresectable rectum carcinoma. | 1Related
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Diarrhoea, T-CD4+ lymphopenia and bilateral patchy pulmonary infiltrates developed in a male 60 yrs of age, who was treated with oxaliplatinum and 5-fluorouracil for unresectable rectum carcinoma. | 1Related
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Diarrhoea, T-CD4+ lymphopenia and bilateral patchy pulmonary infiltrates developed in a male 60 yrs of age, who was treated with oxaliplatinum and 5-fluorouracil for unresectable rectum carcinoma. | 1Related
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Diarrhoea, T-CD4+ lymphopenia and bilateral patchy pulmonary infiltrates developed in a male 60 yrs of age, who was treated with oxaliplatinum and 5-fluorouracil for unresectable rectum carcinoma. | 1Related
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It is concluded that the aforementioned pathological manifestations were due to chemotherapy and included a pulmonary adverse reaction, a feature never previously associated with oxaliplatinum and 5-fluorouracil regimens. | 1Related
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It is concluded that the aforementioned pathological manifestations were due to chemotherapy and included a pulmonary adverse reaction, a feature never previously associated with oxaliplatinum and 5-fluorouracil regimens. | 1Related
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BACKGROUND: Accutane a teratogenic prescription drug licensed to treat severe, recalcitrant nodular acne. | 1Related
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RESULTS: The estimated number of Accutane prescriptions for reproductive-aged women has more than doubled in the past 10 years; it is the most widely used teratogenic drug in the United States, with approximately 2.5 per 1,000 reproductive-aged women exposed to Accutane in 1999. | 1Related
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A 62-year-old woman treated with pranlukast for 2 months developed interstitial pneumonitis with a high fever. | 1Related
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A 62-year-old woman treated with pranlukast for 2 months developed interstitial pneumonitis with a high fever. | 1Related
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Acute interstitial pneumonia induced by ONO-1078 (pranlukast), a leukotriene receptor antagonist. | 1Related
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Acute interstitial pneumonia induced by ONO-1078 (pranlukast), a leukotriene receptor antagonist. | 1Related
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An asymptomatic HIV-infected woman experienced right-sided renal colicky pain during treatment with indinavir. | 1Related
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Indinavir-associated nephrolithiasis and chronic interstitial nephritis were the only possible causes identified in this patient. | 1Related
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Indinavir-associated nephrolithiasis and chronic interstitial nephritis were the only possible causes identified in this patient. | 1Related
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Papillary necrosis associated with the HIV protease inhibitor indinavir. | 1Related
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Physicians should be aware that indinavir nephrolithiasis may cause papillary necrosis. | 1Related
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Physicians should be aware that indinavir nephrolithiasis may cause papillary necrosis. | 1Related
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The HIV protease inhibitor indinavir may cause nephrolithiasis and interstitial nephritis. | 1Related
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The HIV protease inhibitor indinavir may cause nephrolithiasis and interstitial nephritis. | 1Related
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The renal consequences of indinavir-associated nephrotoxicity are uncertain. | 1Related
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We report a case of papillary necrosis in a patient treated with indinavir. | 1Related
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We describe 2 male patients, a 49-year-old with psoriatic arthritis and impaired renal function and a 43-year-old renal transplant recipient, who both sustained a marked decline in glomerular filtration rate in conjunction with a selective inhibitor of cyclooxygenase-2 (COX-2), rofecoxib. | 1Related
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CONCLUSIONS: LTG overdose may result in a severe but reversible encephalopathy, a previously undescribed phenomenon. | 1Related
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Evaluation revealed elevated serum LTG levels and no other etiology for encephalopathy. | 1Related
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METHODS: A 55-year-old woman became stuporous after overdose with lamotrigine (LTG) and valproic acid (VPA) tablets. | 1Related
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METHODS: A 55-year-old woman became stuporous after overdose with lamotrigine (LTG) and valproic acid (VPA) tablets. | 1Related
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METHODS: A 55-year-old woman became stuporous after overdose with lamotrigine (LTG) and valproic acid (VPA) tablets. | 1Related
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METHODS: A 55-year-old woman became stuporous after overdose with lamotrigine (LTG) and valproic acid (VPA) tablets. | 1Related
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PURPOSE: To report that acute lamotrigine poisoning may result in severe encephalopathy. | 1Related
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Stupor from lamotrigine toxicity. | 1Related
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The cause of death was determined to be acute intoxication by olanzapine, and the manner of death was accidental. | 1Related
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This article describes the case of a 25-year-old man found dead at home who had been prescribed olanzapine for schizophrenia. | 1Related
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Lichenoid drug eruption to salsalate. | 1Related
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This eruption emerged after 1 month of therapy with salsalate, persisted for as long as salsalate was administered, and cleared within 3 weeks of discontinuing the medication. | 1Related
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We describe a patient who experienced a lichenoid eruption after the initiation of salsalate for relief of arthritic pain. | 1Related
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Amphotericin B-induced seizures in a patient with AIDS. | 1Related
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CASE SUMMARY: A 46-year-old African-American man experienced recurrent grand mal seizures during intravenous infusion of amphotericin B, then petit mal seizures as the infusion was stopped and the drug concentrations decreased with time. | 1Related
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CONCLUSIONS: Amphotericin B seems to be the probable cause of the seizures. | 1Related
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Despite administration of phenytoin and lorazepam, the seizures persisted and occurred only during amphotercin B administration. | 1Related
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Didanosine also has a potential for inducing seizures. | 1Related
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OBJECTIVE: To report a case of multiple episodes of seizure activity in an AIDS patent following amphotericin B infusion. | 1Related
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The time course of events suggested that amphotericin B was the cause of the seizures in this AIDS patient. | 1Related
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To date, only three cases of seizures associated with amphotericin B have been reported in the literature, but healthcare providers should be aware of the potential for this rare adverse effect. | 1Related
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Vocal cord paralysis as a consequence of peritonsillar infiltration with bupivacaine. | 1Related
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We present the case of a 5-year-old girl who developed bilateral vocal cord paralysis following preoperative peritonsillar bupivacaine infiltration. | 1Related
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A 53 year old Greenlandic male was admitted twice over a period of 4 years with a new complete right bundle branch block after ingestion of 10 g and 4 g of carbamazepine respectively. | 1Related
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Carbamazepine induced right bundle branch block in a Greenlandic patient. | 1Related
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Cicatricial entropion associated with chronic dipivefrin application. | 1Related
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CONCLUSIONS: Cicatrization in the substantia propria of the conjunctiva by excessive lymphocytic infiltration after topically administered antiglaucoma drugs including dipivefrin is a possible mechanism of action for entropion. | 1Related
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Nine eyes from 6 patients, 74 years to 90 years of age, referred by ophthalmologists for repair of cicatricial entropion after at least 2 years of twice-a-day application of dipivefrin. | 1Related
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PURPOSE: To report patients who presented to the oculoplastics department for repair of cicatrical entropion after topical use of dipivefrin. | 1Related
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A 34-year-old lady developed a constellation of dermatitis, fever, lymphadenopathy and hepatitis, beginning on the 17th day of a course of oral sulphasalazine for sero-negative rheumatoid arthritis. | 1Related
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A 34-year-old lady developed a constellation of dermatitis, fever, lymphadenopathy and hepatitis, beginning on the 17th day of a course of oral sulphasalazine for sero-negative rheumatoid arthritis. | 1Related
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A 34-year-old lady developed a constellation of dermatitis, fever, lymphadenopathy and hepatitis, beginning on the 17th day of a course of oral sulphasalazine for sero-negative rheumatoid arthritis. | 1Related
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A 34-year-old lady developed a constellation of dermatitis, fever, lymphadenopathy and hepatitis, beginning on the 17th day of a course of oral sulphasalazine for sero-negative rheumatoid arthritis. | 1Related
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It is thought that the clinico-pathological features and chronology of this case bore the hallmarks of the so-called "3-week sulphasalazine syndrome", a rare, but often fatal, immunoallergic reaction to sulphasalazine. | 1Related
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It is thought that the clinico-pathological features and chronology of this case bore the hallmarks of the so-called "3-week sulphasalazine syndrome", a rare, but often fatal, immunoallergic reaction to sulphasalazine. | 1Related
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It is thought that the clinico-pathological features and chronology of this case bore the hallmarks of the so-called "3-week sulphasalazine syndrome", a rare, but often fatal, immunoallergic reaction to sulphasalazine. | 1Related
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It is thought that the clinico-pathological features and chronology of this case bore the hallmarks of the so-called "3-week sulphasalazine syndrome", a rare, but often fatal, immunoallergic reaction to sulphasalazine. | 1Related
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It is thought that the clinico-pathological features and chronology of this case bore the hallmarks of the so-called "3-week sulphasalazine syndrome", a rare, but often fatal, immunoallergic reaction to sulphasalazine. | 1Related
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