Physical O
mapping O
220 O
kb O
centromeric O
of O
the O
human B
MHC I
and O
DNA O
sequence O
analysis O
of O
the O
43 O
- O
kb O
segment O
including O
the O
RING1 B
, O
HKE6 B
, O
and O
HKE4 B
genes I
. O

The O
synthesis O
of O
endo O
- O
adduct O
[ O
4aS O
, O
5S O
, O
8R O
, O
8aR O
, O
SS O
] O
- O
9d O
resulting O
from O
cycloaddition O
on O
the O
substituted O
C O
( O
2 O
) O
- O
C O
( O
3 O
) O
double O
bond O
was O
achieved O
in O
a O
chemo O
- O
and O
diastereoselective O
way O
from O
quinone O
1d O
in O
the O
presence O
of O
ZnBr O
( O
2 O
) O
. O

DNA O
elements O
recognizing O
NF B
- I
Y I
and O
Sp1 B
regulate O
the O
human O
multidrug I
- I
resistance I
gene I
promoter I
. O

Radioimmunoassay O
of O
plasma O
gonadotropins I
; O
problems O
of O
specificity O

Patients O
randomized O
into O
the O
active O
treatment O
groups O
A30 O
( O
n O
= O
49 O
) O
and O
A60 O
( O
n O
= O
48 O
) O
received O
topical O
treatment O
with O
3 O
. O
0 O
% O
diclofenac O
in O
2 O
. O
5 O
% O
hyaluronan O
gel O
0 O
. O
5 O
g O
twice O
daily O
for O
30 O
or O
60 O
days O
, O
respectively O
. O

As O
tat B
itself O
dramatically O
increases O
HIV O
- O
1 O
gene O
expression O
, O
it O
too O
is O
presumably O
regulated O
in O
the O
latent O
state O
, O
and O
may O
also O
be O
activated O
by O
mitogenic O
stimulation O
. O

To O
clarify O
the O
difference O
, O
both O
the O
Crk B
II I
and O
Crk B
II I
- I
23 I
, O
proteins O
were O
expressed O
in O
E O
. O
coli O
and O
examined O
their O
binding O
capacity O
in O
vitro O
. O

CONCLUSIONS O
: O
Systemic O
factors O
that O
increase O
bone O
mineral O
density O
appear O
to O
be O
involved O
in O
the O
pathogenesis O
of O
ossification O
of O
the O
posterior O
longitudinal O
ligament O
, O
and O
these O
factors O
may O
be O
activated O
after O
middle O
age O
. O

Thus O
, O
in O
V O
. O
furnissii O
, O
the O
E B
. I
coli I
manX I
equivalent I
comprises O
two O
genes O
, O
which O
are O
separated O
in O
the O
genome O
by O
two O
other O
genes O
of O
the O
ptsM B
complex I
. O

Id B
- I
1H I
and O
Id B
- I
2H I
seem O
to O
be O
human B
homologues I
of I
mouse I
Id I
- I
1 I
and O
Id B
- I
2 I
, O
respectively O
, O
and O
have O
potential O
to O
encode O
154 O
and O
135 O
amino O
acid O
proteins O
. O

Characterization O
of O
insertion O
mutations O
in O
the O
Saccharomyces B
cerevisiae I
MSH1 I
and O
MSH2 B
genes I
: O
evidence O
for O
separate O
mitochondrial O
and O
nuclear O
functions O
. O

Transcription O
of O
FN B
promoter I
- O
chloramphenicol B
acetyltransferase I
fusion O
genes O
carrying O
the O
base O
substitution O
in O
one O
or O
more O
of O
these O
G O
- O
rich O
sequences O
both O
in O
vivo O
and O
in O
vitro O
revealed O
that O
the O
base O
substitution O
in O
any O
G O
- O
rich O
sequence O
results O
in O
reduction O
of O
promoter O
activity O
, O
although O
the O
downstream O
GC O
box O
( O
GCd O
) O
plays O
a O
primary O
role O
. O

Morphologically O
, O
corrugation O
of O
the O
internal O
elastic O
lamina O
of O
vessels O
was O
often O
observed O
in O
the O
vehicle O
- O
treated O
group O
, O
but O
was O
not O
prominent O
in O
the O
GLNVA O
- O
treated O
groups O
or O
healthy O
controls O
. O

A O
GT O
- O
rich O
sequence O
binding O
the O
transcription O
factor O
Sp1 I
is O
crucial O
for O
high O
expression O
of O
the O
human B
type I
VII I
collagen I
gene I
( O
COL7A1 B
) O
in O
fibroblasts O
and O
keratinocytes O
. O

Each O
received O
20 O
mL O
of O
one O
of O
three O
test O
solutions O
: O
levobupivacaine O
control O
, O
levobupivacaine O
and O
fentanyl O
2 O
microg O
/ O
mL O
, O
or O
levobupivacaine O
and O
fentanyl O
3 O
microg O
/ O
mL O
. O

The O
XPR2 B
gene I
from O
Yarrowia O
lipolytica O
encodes O
an O
inducible O
alkaline O
extracellular O
protease O
. O

CONCLUSIONS O
: O
This O
study O
shows O
that O
the O
annual O
rate O
of O
de O
novo O
aneurysm O
formation O
is O
relatively O
high O
( O
0 O
. O
89 O
% O
) O
and O
that O
the O
cumulative O
risk O
becomes O
significant O
after O
9 O
years O
. O

It O
was O
found O
that O
Nopp140 B
binds O
primarily O
to O
the O
CK2 B
regulatory I
subunit I
, O
beta O
. O

Juvenile O
idiopathic O
inflammatory O
myopathies O
: O
the O
value O
of O
magnetic O
resonance O
imaging O
in O
the O
detection O
of O
muscle O
involvement O
. O

Previous O
analysis O
of O
this O
motif O
in O
the O
lactose B
permease I
( O
A O
. O

Combined O
chemotherapy O
with O
VM O
26 O
and O
BCNU O
for O
recurrent O
malignant O
gliomas O
after O
operation O
and O
irradiation O
. O

Like O
other O
IAPs B
, O
ch B
- I
IAP1 I
contains O
the O
N B
- I
terminal I
baculovirus I
IAP I
repeats I
and O
C O
- O
terminal O
RING O
finger O
motifs O
. O

Effects O
of O
long O
- O
term O
use O
of O
raloxifene O
, O
a O
selective O
estrogen B
receptor I
modulator O
, O
on O
thyroid O
function O
test O
profiles O
. O

The O
donors O
of O
10 O
patients O
were O
ABO O
- O
incompatible O
, O
and O
for O
five O
pairs O
the O
ABO O
incompatibility O
was O
major O
. O

Alternative O
splicing O
of O
the O
human B
serotonin I
transporter I
gene I
. O

Interestingly O
, O
basal O
MAPK B
, O
but O
not O
Raf B
- I
1 I
, O
activity O
was O
constitutively O
enhanced O
in O
Jak1 B
- O
deficient O
HeLa O
cells O
. O

This O
case O
report O
describes O
the O
treatment O
of O
a O
patient O
with O
periodontosis O
. O

The O
aim O
of O
this O
work O
was O
to O
organize O
chemical O
data O
in O
a O
client O
- O
server O
environment O
using O
Database O
Management O
System O
and O
Web O
fashion O
for O
the O
client O
interface O
. O

Cerumen O
management O
in O
hearing O
conservation O
: O
the O
Dallas O
( O
Texas O
) O
Independent O
School O
District O
program O
. O

Lactate O
, O
pyruvate O
and O
actual O
pH O
values O
in O
the O
venous O
blood O
of O
newborn O
calves O

This O
article O
focuses O
on O
a O
general O
approach O
to O
the O
use O
of O
ARV O
agents O
. O

Platelets O
and O
RBCs O
from O
each O
animal O
were O
labeled O
with O
51Cr O
and O
99mTc O
, O
respectively O
, O
and O
were O
rapidly O
injected O
into O
the O
right O
atrium O
while O
blood O
was O
sampled O
from O
the O
ascending O
aorta O
. O

The O
comparisons O
of O
swimming O
performance O
before O
and O
after O
treatments O
with O
deltamethrin O
have O
shown O
a O
significant O
effect O
on O
locomotory O
ability O
of O
rainbow O
trout O
which O
at O
the O
end O
of O
strong O
exposure O
( O
e O
. O
g O
. O

Protection O
levels O
of O
33 O
. O
3 O
% O
, O
36 O
. O
6 O
% O
and O
37 O
. O
0 O
% O
were O
achieved O
in O
groups O
which O
had O
received O
, O
respectively O
, O
1000 O
20 O
- O
krad O
cercariae O
, O
1000 O
10 O
- O
krad O
cercariae O
, O
2000 O
40 O
- O
krad O
cercariae O
and O
2000 O
60 O
- O
krad O
cercariae O
. O

In O
Types O
I O
and O
II O
, O
capillary O
walls O
became O
thinner O
and O
more O
capillary O
lumens O
were O
visiable O
. O

Thus O
, O
primate O
vs O
. O
rodent O
promoter O
selectivity O
mediated O
by O
the O
TBP B
- O
TAFI B
complex O
is O
likely O
to O
be O
the O
result O
of O
cumulative O
subtle O
differences O
between O
individual O
subunits O
that O
lead O
to O
species O
- O
specific O
properties O
of O
RNA B
polymerase I
I I
transcription O
. O

The O
effects O
of O
pretreatment O
with O
the O
non O
- O
competitive O
NMDA O
antagonist O
( O
+ O
) O
MK O
- O
801 O
on O
the O
behavioral O
alterations O
induced O
by O
repeated O
restraint O
stress O
were O
investigated O
. O

When O
gene O
conversion O
is O
initiated O
by O
a O
double O
- O
strand O
break O
( O
DSB O
) O
, O
any O
nonhomologous O
DNA O
that O
may O
be O
present O
at O
the O
ends O
must O
be O
removed O
before O
new O
DNA O
synthesis O
can O
be O
initiated O
. O

In O
this O
reconstituted O
system O
, O
RNAP B
IIA I
, O
but O
not O
RNAP B
IIB I
, O
can O
transcribe O
from O
the O
DHFR B
promoter I
. O

The O
cDNA O
clones O
encode O
a O
polypeptide O
of O
657 O
amino O
acids O
with O
a O
bHLH B
( O
basic B
- I
helix I
- I
loop I
- I
helix I
) O
domain O
, O
characteristic O
of O
a O
large O
family O
of O
transcription O
factors O
, O
and O
a O
PAS B
( O
Per B
- I
Arnt I
- I
Sim I
) O
domain O
in O
the O
amino O
- O
terminal O
half O
region O
. O

FTF O
( O
Joint O
Council O
of O
Civil O
Servants O
and O
Functionaries O
) O
: O
clarification O
about O
membership O
in O
the O
main O
organizations O

Lumbar O
spine O
bone O
mineral O
density O
( O
BMD O
) O
by O
dual O
- O
energy O
X O
- O
ray O
absorptiometry O
( O
DXA O
) O
increased O
by O
0 O
. O
6 O
% O
, O
3 O
. O
6 O
% O
and O
8 O
. O
1 O
% O
after O
48 O
weeks O
in O
groups O
L O
, O
M O
and O
H O
respectively O
, O
responses O
in O
groups O
M O
and O
H O
being O
significantly O
higher O
than O
in O
L O
( O
p O
< O
0 O
. O
05 O
, O
Mann O
- O
Whitney O
U O
- O
test O
) O
. O

Synergy O
between O
interferon B
- I
gamma I
and O
tumor B
necrosis I
factor I
- I
alpha I
in O
transcriptional O
activation O
is O
mediated O
by O
cooperation O
between O
signal B
transducer I
and I
activator I
of I
transcription I
1 I
and O
nuclear B
factor I
kappaB I
. O

1H O
and O
15N O
magnetic O
resonance O
assignments O
, O
secondary O
structure O
, O
and O
tertiary O
fold O
of O
Escherichia B
coli I
DnaJ I
( I
1 I
- I
78 I
) I
. O

A O
cDNA O
for O
human B
microfibril I
- I
associated I
glycoprotein I
- I
2 I
( O
MAGP B
- I
2 I
) O
was O
used O
to O
screen O
a O
human O
leukocyte O
genomic O
DNA O
library O
in O
EMBL O
- O
3 O
vector O
. O

Furthermore O
, O
ERK B
phosphorylation O
was O
substantially O
prolonged O
in O
LC O
/ O
BRY B
- O
treated O
cells O
compared O
to O
those O
exposed O
to O
BRY B
alone O
, O
and O
pretreatment O
with O
the O
highly O
specific O
MEK B
inhibitors O
, O
PD98059 O
, O
U0126 O
, O
and O
SL327 O
, O
opposed O
ERK B
activation O
while O
protecting O
cells O
from O
LC O
/ O
BRY B
- O
induced O
lethality O
. O

The O
cholesterol B
7alpha I
- I
hydroxylase I
gene I
( O
CYP7A B
) O
is O
transcriptionally O
regulated O
by O
a O
number O
of O
factors O
, O
including O
hormones O
, O
bile O
acids O
, O
and O
diurnal O
rhythm O
. O

CNS O
preventive O
treatment O
without O
cranial O
irradiation O
was O
effective O
in O
all O
the O
groups O
of O
ALL O
patients O
. O

Additionally O
, O
the O
members O
of O
this O
family O
display O
strong O
sequence O
homologies O
between O
their O
larger O
C O
- O
terminal O
effector O
binding O
/ O
oligomerization O
domains O
. O

Aspartame O
is O
a O
synthetic O
sweetener O
commonly O
used O
in O
soft O
drinks O
and O
many O
foods O
. O

The O
rat O
anococcygeus O
muscle O
was O
used O
in O
postsynaptic O
studies O
. O

The O
purpose O
of O
this O
presentation O
is O
to O
review O
the O
current O
state O
of O
knowledge O
regarding O
5 O
, O
8 O
, O
11 O
, O
14 O
- O
eicosatetraynoic O
acid O
( O
ETYA O
, O
Ro O
3 O
- O
1428 O
) O
and O
its O
effects O
on O
lipid O
metabolism O
. O

A O
Meis1 B
cDNA I
clone I
that O
encoded O
a O
novel O
member O
of O
the O
homeobox B
gene I
family I
was O
identified O
. O

The O
S B
. I
typhimurium I
aspartyl I
/ I
asparaginyl I
beta I
- I
hydroxylase I
homologue I
( O
designated O
lpxO B
) O
was O
cloned O
into O
pBluescriptSK O
and O
expressed O
in O
Escherichia O
coli O
K O
- O
12 O
, O
which O
does O
not O
contain O
lpxO B
. O

The O
levels O
of O
soluble O
L B
- I
selectin I
and O
ICAM B
- I
1 I
in O
the O
serum O
were O
determined O
by O
the O
ELISA O
method O
. O

The O
highest O
TTX O
content O
is O
10 O
. O
0 O
microg O
/ O
g O
in O
N O
. O
livescens O
. O

On O
the O
other O
hand O
hypokalemia O
, O
induced O
by O
diuretics O
, O
may O
also O
be O
accompanied O
by O
a O
significant O
depletion O
of O
total O
body O
K O
, O
bringing O
about O
more O
general O
consequences O
. O

Glutathione B
reductase I
activities O
in O
liver O
, O
kidney O
, O
lung O
, O
and O
brain O
were O
not O
affected O
by O
diet O
. O

Here O
, O
we O
describe O
the O
nucleotide O
( O
nt O
) O
sequence O
and O
genomic O
organization O
of O
ipiB B
and O
ipiO B
. O

Vancomycin O
and O
tetracycline O
eliminated O
toxin O
in O
stools O
and O
delayed O
death O
. O

Soluble O
fibrin B
monomer I
complexes I
in O
thromboembolism O

Comparison O
to O
the O
murine O
homologues O
of O
these O
molecules O
reveals O
a O
high O
degree O
of O
conservation O
between O
the O
products O
of O
one O
of O
these O
genes O
, O
Fc B
gamma I
RIIb I
, O
and O
the O
murine B
beta I
gene I
in O
primary O
sequence O
, O
splicing O
pattern O
, O
and O
tissue O
distribution O
. O

Acidosis O
provoked O
by O
30 O
% O
CO2 O
irreversibly O
increased O
the O
BDRT O
, O
however O
the O
TRT O
were O
not O
changed O
. O

Others O
as O
for O
example O
C O
- O
11 O
labeled O
short O
chain O
fatty O
acids O
are O
on O
the O
horizon O
. O

The O
average O
progression O
was O
11 O
. O
6 O
( O
SD O
9 O
. O
0 O
) O
. O

80 O
bp O
) O
surrounding O
the O
site O
of O
transcription O
initiation O
. O

Ultrastructure O
and O
toxin O
formation O
of O
Cor O
. O
diphtheriae O
PW O
- O
8 O
cultured O
in O
the O
presence O
of O
penicillin O

EMT B
was O
localized O
to O
chromosome O
5q31 O
- O
32 O
, O
a O
region O
that O
contains O
the O
genes O
for O
several O
growth O
factors O
and O
receptors O
as O
well O
as O
early O
activation O
genes O
, O
particularly O
those O
involved O
in O
the O
hematopoietic O
system O
. O

Tbx6 B
maps O
to O
chromosome O
7 O
and O
does O
not O
appear O
to O
be O
linked O
to O
any O
known O
mutation O
. O

Kaposi O
' O
s O
eruptive O
disease O
. O

It O
is O
not O
tested O
yet O
, O
whether O
the O
extent O
of O
glaucoma O
damage O
should O
be O
better O
quantified O
using O
reference O
plane O
1 O
or O
2 O
. O

Importin B
beta I
mediates O
nuclear O
translocation O
of O
Smad B
3 I
. O

The O
encoded O
protein O
contains O
an O
amino O
terminal O
PDZ B
domain I
, O
followed O
by O
a O
predicted O
coiled O
- O
coil O
region O
, O
a O
PEST O
domain O
, O
and O
a O
carboxy O
- O
terminal O
SAM O
domain O
. O

Using O
lasers O
, O
PRK O
involves O
reshaping O
the O
cornea O
so O
that O
its O
refractive O
power O
is O
increased O
. O

However O
, O
sphincter O
length O
decreased O
with O
age O
from O
24 O
. O
3 O
to O
14 O
. O
8 O
mm O
. O
, O
maximal O
urethral O
pressure O
from O
88 O
. O
7 O
to O
55 O
cm O
. O
water O
and O
maximal O
urethral O
pressure O
during O
voluntary O
contraction O
from O
221 O
. O
4 O
to O
166 O
. O
3 O
cm O
. O
water O
. O

Several O
studies O
have O
demonstrated O
that O
the O
corticotropin B
- I
releasing I
factor I
test O
( O
CRF B
) O
is O
useful O
for O
the O
aetiological O
diagnosis O
of O
Cushing O
' O
s O
syndrome O
: O
in O
Cushing O
' O
s O
disease O
, O
as O
opposed O
to O
ectopic O
ACTH B
secretion O
syndrome O
, O
the O
hypothalamus O
- O
pituitary O
- O
adrenal O
( O
HPA O
) O
axis O
can O
still O
be O
stimulated O
by O
CRF B
. O

Transcription O
initiation O
from O
this O
region O
was O
also O
detected O
in O
vivo O
, O
when O
the O
cloned O
rRNA B
gene I
cluster O
was O
present O
on O
a O
multi O
- O
copy O
plasmid O
. O

The O
results O
of O
the O
transient O
- O
transfection O
assay O
showed O
that O
the O
Sp1 B
binding I
element I
located O
in O
the O
core O
region O
( O
positions O
- O
- O
64 O
to O
+ O
1 O
) O
of O
the O
alpha2 B
- I
integrin I
promoter I
plays O
an O
essential O
role O
in O
the O
alpha2 B
- I
integrin I
promoter I
activity O
and O
its O
downregulation O
by O
Erb B
- I
B2 I
. O

Principal O
component O
analysis O
using O
psychosocial O
factors O
in O
women O
showed O
two O
psychosocial O
structures O
, O
i O
. O
e O
. O
the O
second O
principal O
( O
high O
SOC O
, O
high O
lifestyle O
, O
and O
low O
stress O
) O
and O
the O
4th O
principal O
components O
( O
high O
supernatural O
HLC O
, O
and O
high O
PHLC O
) O
. O

Expression O
of O
FNCAT B
increased O
on O
serum O
treatment O
indicating O
that O
the O
region O
of O
the O
FN B
gene I
between O
positions O
+ O
69 O
and O
- O
510 O
bp O
mediated O
serum O
responsiveness O
. O

Interaction O
of O
the O
v B
- I
rel I
protein I
with O
an O
NF B
- I
kappa I
B I
DNA I
binding I
site I
. O

This O
increase O
is O
not O
the O
result O
of O
alterations O
in O
the O
deposition O
of O
inhaled O
particles O
of O
capsaicin O
brought O
about O
by O
volume O
restriction O
. O

Providing O
appropriate O
care O
to O
both O
sexes O
. O

To O
test O
this O
hypothesis O
, O
we O
developed O
a O
system O
of O
transient O
transfection O
of O
rat O
adipocytes O
with O
constructs O
containing O
glyceraldehyde B
- I
3 I
- I
phosphate I
dehydrogenase I
( O
GAPDH B
) O
and O
fatty B
acid I
synthetase I
( O
FAS B
) O
promoters O
fused O
to O
gene O
reporter O
CAT B
. O

In O
the O
green O
unicellular O
alga O
Chlamydomonas O
eugametos O
, O
cellular O
division O
is O
readily O
synchronized O
by O
light O
/ O
dark O
cycles O
. O

SHBG B
concentrations O
in O
women O
were O
also O
significantly O
higher O
at O
T O
- O
2 O
and O
T O
- O
3 O
when O
compared O
to O
T O
- O
1 O
values O
. O

Simvastatin O
decreased O
levels O
of O
total O
cholesterol O
by O
20 O
. O
8 O
% O
, O
LDL B
cholesterol I
by O
29 O
. O
7 O
% O
, O
triglycerides O
by O
13 O
. O
6 O
% O
, O
apolipoprotein B
B I
by O
22 O
. O
4 O
% O
, O
alpha O
- O
tocopherol O
by O
16 O
. O
2 O
% O
, O
beta O
- O
carotene O
by O
19 O
. O
5 O
% O
, O
and O
ubiquinol O
- O
10 O
by O
22 O
. O
0 O
% O
( O
P O
< O
. O
001 O
for O
all O
) O
and O
increased O
levels O
of O
HDL B
cholesterol I
by O
7 O
. O
0 O
% O
( O
P O
< O
. O
001 O
) O
and O
serum B
insulin I
by O
13 O

. O
2 O
% O
( O
P O
= O
. O
005 O
) O
. O

The O
more O
severe O
the O
CRF O
, O
the O
more O
likely O
that O
total O
HDL B
and O
HDL2 B
cholesterol I
will O
be O
low O
. O

They O
are O
growth O
- O
inhibited O
by O
TGF B
- I
beta1 I
. O

To O
investigate O
the O
in O
vivo O
role O
of O
this O
chimeric O
bZIP I
protein I
in O
oncogenic O
transformation O
, O
its O
expression O
was O
directed O
to O
the O
lymphoid O
compartments O
of O
transgenic O
mice O
. O

This O
is O
consistent O
with O
the O
need O
for O
a O
nuclear B
retinoid I
receptor I
function O
in O
RA O
- O
induced O
ERK2 B
activation O
. O

In O
the O
virus O
- O
infected O
cells O
, O
translation O
of O
the O
M1 B
protein I
was O
reduced O
to O
10 O
to O
20 O
% O
of O
that O
of O
the O
wild O
- O
type O
virus O
; O
however O
, O
the O
translation O
of O
neither O
the O
nucleoprotein O
nor O
NS1 B
was O
significantly O
interfered O
with O
, O
indicating O
the O
important O
role O
of O
NS1 B
in O
translational O
stimulation O
of O
the O
M1 B
protein I
. O

Some O
cells O
were O
excited O
, O
others O
inhibited O
by O
, O
and O
only O
some O
were O
directionally O
sensitive O
to O
the O
optomotor O
stimulus O
. O

LT B
- I
beta I
transcription O
is O
maximal O
in O
the O
thymic O
medulla O
and O
in O
splenic O
white O
pulp O
. O

A O
novel O
Cdc42Hs B
mutant I
induces O
cellular O
transformation O
. O

Interactions O
were O
also O
detected O
between O
full B
- I
length I
HSF1 I
and O
the O
small O
subunit O
( O
gamma O
) O
of O
TFIIA B
. O

We O
report O
that O
Nim1 B
possesses O
intrinsic O
serine B
- I
kinase I
, O
threonine B
- I
kinase I
and O
tyrosine B
- I
kinase I
activities O
. O

A O
short O
conserved O
motif O
is O
required O
for O
repressor O
domain O
function O
in O
the O
myeloid O
- I
specific I
transcription I
factor I
CCAAT I
/ I
enhancer I
- I
binding I
protein I
epsilon I
. O

Androgen O
ablation O
- O
- O
50 O
years O
later O
. O

In O
conclusion O
, O
ORCA3 B
regulates O
jasmonate O
- O
responsive O
expression O
of O
the O
Str B
gene I
via O
direct O
interaction O
with O
the O
JERE B
. O

Also O
, O
the O
absence O
of O
PdNi B
between O
the O
segments O
reduces O
the O
heat O
production O
of O
the O
seed O
. O

Theoretical O
mechanisms O
of O
hyperuricemia O
. O

ORF3 O
encodes O
a O
putative O
periplasmic B
c I
- I
type I
cytochrome I
with O
a O
molecular O
mass O
of O
94 O
, O
000 O
Da O
and O
contains O
seven O
c O
- O
heme O
- O
binding O
motifs O
but O
shows O
no O
sequence O
homology O
to O
occ B
or O
ORF1 O
. O

Doxazosin O
produced O
increases O
in O
both O
mean O
and O
maximum O
urinary O
flow O
rates O
of O
1 O
. O
01 O
ml O
/ O
s O
and O
3 O
. O
2 O
ml O
/ O
s O
respectively O
, O
compared O
with O
0 O
. O
21 O
ml O
/ O
s O
and O
2 O
. O
2 O
ml O
/ O
s O
on O
placebo O
. O

Hypoxia O
decreased O
the O
rate O
of O
spontaneous O
impulse O
initiation O
in O
SA O
nodal O
fibers O
by O
decreasing O
the O
slope O
of O
diastolic O
depolarization O
. O

BACKGROUND O
- O
- O
Platelet B
activating I
factor I
( O
PAF B
) O
has O
been O
implicated O
in O
the O
pathogenesis O
of O
airway O
hyperresponsiveness O
in O
asthma O
. O

TFEC B
RNA I
is O
found O
in O
many O
tissues O
of O
adult O
rats O
, O
but O
the O
relative O
concentrations O
of O
TFEC B
and O
TFE3 B
RNAs I
vary O
considerably O
in O
these O
different O
tissues O
. O

Our O
results O
show O
that O
unlike O
the O
case O
for O
sos1 B
, O
sos2 B
gene I
function O
is O
dispensable O
for O
normal O
mouse O
development O
, O
growth O
, O
and O
fertility O
. O

Studies O
of O
HS2 B
- I
gamma I
gene I
reporter I
constructs I
carrying O
CACCC O
box O
deletions O
revealed O
that O
the O
CACCC O
box O
sequence O
of O
the O
gamma O
gene O
promoter O
mediates O
the O
activation O
of O
the O
gamma O
gene O
by O
FKLF B
. O

Advisory O
Committee O
report O
recommends O
that O
US O
make O
amends O
for O
human O
radiation O
experiments O
. O

Animals O
were O
divided O
into O
six O
equal O
groups O
as O
follows O
: O
( O
1 O
) O
no O
oral O
drug O
( O
water O
) O
, O
no O
hemodilution O
, O
no O
IV O
drug O
( O
saline O
) O
; O
( O
2 O
) O
oral O
water O
, O
hemodilution O
, O
IV O
saline O
; O
( O
3 O
) O
oral O
water O
, O
no O
hemodilution O
, O
IV O
propranolol O
; O
( O
4 O
) O
oral O
water O
, O
hemodilution O
, O
IV O
propranolol O
; O
( O
5 O
) O
oral O
propranolol O
, O
no O
hemodilution O
, O
IV O
saline O
; O
and O
( O
6 O
) O
oral O
propranolol O
, O
hemodilution O
, O
IV O
saline O
. O

First O
, O
a O
GATA B
- I
1 I
motif I
was O
found O
to O
bind O
nuclear O
factor O
( O
s O
) O
, O
presumably O
the O
GATA B
- I
1 I
factor I
, O
present O
in O
K O
- O
562 O
cell O
extracts O
and O
in O
living O
K O
- O
562 O
cells O
. O

BPN O
can O
be O
experimentally O
produced O
by O
intratracheal O
inoculation O
of O
microorganisms O
in O
high O
concentrations O
and O
ventilator O
- O
associated O
BPN O
by O
ventilating O
baboons O
with O
oleic O
- O
acid O
lung O
injury O
. O

A O
mouth O
asymmetry O
study O
. O

Additionally O
, O
these O
findings O
have O
important O
implications O
for O
the O
understanding O
of O
the O
mechanisms O
of O
HIV O
- O
1 O
latency O
and O
reactivation O
. O

This O
inhibition O
was O
due O
to O
competition O
from O
the O
EMC O
virus O
IRES O
present O
in O
pEP B
- I
2A I
transcripts I
, O
as O
well O
as O
the O
expression O
of O
proteolytically O
active O
2Apro B
. O

Some O
may O
affect O
the O
folding O
pathway O
or O
the O
affinity O
for O
chaperonins B
. O

Diet O
imbalances O
and O
aflatoxicosis O

48 O
. O
35 O
+ O
/ O
- O
5 O
. O
07 O
mg O
/ O
l O
in O
R3 O
; O
p O
< O
0 O
. O
05 O
) O
and O
in O
the O
AUC O
( O
0 O
. O
50 O
h O
) O
( O
1429 O
. O
92 O
+ O
/ O
- O
284 O
. O
23 O
in O
PR O
vs O
. O

The O
effect O
on O
FIV B
LTR I
promoter I
activity O
of O
progressively O
deleting O
these O
nuclear O
factor O
binding O
sites O
was O
examined O
by O
linking O
LTR O
deletion O
mutants O
to O
the O
chloramphenicol B
acetyltransferase I
( O
CAT B
) O
gene O
. O

A O
series O
of O
sequence O
fragments O
were O
placed O
5 O
' O
to O
the O
chloramphenicol B
acetyltransferase I
( O
CAT B
) O
reporter O
gene O
and O
ability O
to O
mediate O
transcription O
of O
CAT B
in O
response O
to O
IFN B
gamma I
or O
LPS O
treatment O
was O
studied O
following O
transient O
transfection O
in O
the O
macrophage O
- O
like O
cell O
line O
RAW O
264 O
. O
7 O
. O

As O
part O
of O
a O
large O
Dutch O
prospective O
multicenter O
study O
( O
Netherlands O
Cooperative O
Study O
on O
the O
Adequacy O
of O
Dialysis O
- O
2 O
) O
, O
we O
consecutively O
included O
all O
new O
patients O
with O
end O
- O
stage O
renal O
disease O
for O
whom O
residual O
renal O
function O
could O
be O
obtained O
0 O
to O
4 O
weeks O
before O
the O
start O
of O
dialysis O
therapy O
. O

Module O
2 O
of O
RifA B
lacks O
a O
beta B
- I
ketoacyl I
: I
acyl I
carrier I
protein I
reductase I
( O
KR B
) O
domain O
and O
the O
one O
in O
module O
3 O
has O
an O
apparently O
inactive O
NADPH O
binding O
motif O
, O
similar O
to O
one O
found O
in O
the O
Er B
PKS I
, O
while O
the O
other O
eight O
KR B
domains O
of O
the O
Rf B
PKS I
should O
be O
functional O
. O

In O
murine O
cells O
, O
FPGS B
expression O
is O
controlled O
by O
two O
promoters O
that O
, O
as O
we O
show O
here O
, O
vary O
substantially O
in O
their O
efficiency O
, O
at O
least O
in O
the O
context O
of O
a O
reporter O
gene O
assay O
. O

Should O
we O
use O
thiazide O
diuretics O
in O
hypertensive O
patients O
with O
non O
- O
insulin B
- O
dependent O
diabetes O
mellitus O
? O

Reduced O
glutathione O
in O
whole O
blood O
decreases O
within O
a O
blood O
Se O
range O
of O
1 O
. O
01 O
to O
2 O
. O
28 O
micrograms O
in O
the O
high O
Se O
area O
. O

Sucrose O
produced O
in O
source O
leaves O
is O
the O
predominant O
carbon O
source O
for O
developing O
sink O
tissues O
in O
most O
higher O
plants O
. O

A O
personal O
view O
of O
future O
trends O
in O
the O
field O
of O
immunoassay O
concludes O
this O
review O
. O

In O
accordance O
with O
Ann O
Arbor O
classification O
, O
we O
observed O
12 O
CS O
IA O
( O
21 O
. O
3 O
% O
) O
, O
2 O
CS O
IB O
( O
3 O
. O
5 O
% O
) O
, O
14 O
CS O
IIA O
( O
25 O
. O
4 O
% O
) O
and O
27 O
CS O
IIB O
( O
49 O
. O
7 O
% O
) O
. O

Numerous O
genes O
of O
this O
family O
have O
been O
identified O
in O
animals O
, O
with O
the O
largest O
number O
found O
in O
vertebrates O
. O

The O
GST B
- O
CBL B
- O
LZIP I
fusion O
protein O
contains O
a O
binding O
site O
for O
the O
SH2 B
domain I
of O
the O
p85 B
subunit I
of O
phosphatidylinositol B
3 I
- I
kinase I
, O
which O
mapped O
to O
Tyr731 O
, O
which O
is O
present O
in O
the O
sequence O
YEAM O
. O

We O
have O
previously O
shown O
that O
UNC B
- I
4 I
expression O
in O
the O
VA O
motor O
neurons O
specifies O
the O
wild O
- O
type O
pattern O
of O
presynaptic O
input O
. O

Significant O
differences O
were O
observed O
in O
the O
composition O
of O
nuclear O
transcription O
factors O
bound O
to O
cis O
- O
elements O
within O
the O
LDH B
/ I
C I
proximal I
promoter I
region I
in O
somatic O
versus O
germ O
cells O
. O

Above O
this O
transition O
temperature O
the O
calcium O
antagonist O
lowers O
fm O
more O
pronouncedly O
than O
below O
. O

( O
1986 O
) O
described O
optic O
nerve O
degenerations O
in O
patients O
with O
Alzheimer O
' O
s O
disease O
and O
Sadun O
published O
a O
dropout O
of O
retinal O
ganglion O
cells O
that O
range O
from O
30 O
% O
to O
60 O
% O
. O

It O
has O
become O
clear O
that O
there O
is O
not O
one O
mediator O
responsible O
for O
ALI O
, O
but O
rather O
a O
complex O
interplay O
exists O
between O
diverse O
proinflammatory O
( O
e O
. O
g O
. O
, O
lipopolysaccharide O
, O
complement O
products O
, O
cytocains O
, O
chemocains O
, O
reactive O
oxygen O
species O
and O
arachidonic O
acid O
products O
) O
and O
anti O
- O
inflammatory O
( O
IL B
- I
10 I
, O
IL B
- I
1 I
- I
RA I
, O
PGI2 O
) O
mediators O
. O

We O
then O
supplemented O
these O
spliceosomes O
with O
purified O
proteins O
or O
yeast O
extract O
fractions O
as O
a O
functional O
assay O
for O
second O
- O
step O
splicing O
factors O
. O

Contrast O
radiography O
appears O
to O
be O
an O
especially O
inaccurate O
method O
to O
document O
PUD O
in O
CF O
because O
the O
duodenal O
mucosa O
typically O
appears O
nodular O
and O
distorted O
with O
poor O
definition O
of O
the O
mucosal O
folds O
. O

We O
now O
show O
that O
the O
FRAP B
Ser2035 I
- I
- I
> I
Ala I
mutant I
displays O
similar O
binding O
affinity O
when O
compared O
with O
the O
wild O
- O
type O
protein O
, O
whereas O
all O
other O
mutations O
at O
this O
site O
, O
including O
mimics O
of O
phosphoserine O
, O
abolish O
binding O
, O
presumably O
due O
to O
either O
unfavorable O
steric O
interactions O
or O
induced O
conformational O
changes O
. O

A O
Brassica O
cDNA O
clone O
encoding O
a O
bifunctional O
hydroxymethylpyrimidine B
kinase I
/ O
thiamin B
- I
phosphate I
pyrophosphorylase I
involved O
in O
thiamin O
biosynthesis O
. O

Titres O
of O
antistreptolysin B
O I
in O
mothers O
of O
children O
affected O
with O
Down O
' O
s O
syndrome O
. O

Differential O
decay O
of O
a O
polycistronic O
Escherichia O
coli O
transcript O
is O
initiated O
by O
RNaseE B
- O
dependent O
endonucleolytic O
processing O
. O

Defects O
in O
RT6 B
expression O
coincide O
with O
increased O
susceptibility O
in O
animal O
models O
for O
insulin B
- O
dependent O
diabetes O
mellitus O
and O
other O
autoimmune O
diseases O
. O

By O
means O
of O
early O
detection O
and O
correction O
of O
the O
possible O
causes O
, O
the O
goal O
of O
increasing O
therapeutic O
efficacy O
can O
be O
achieved O
. O

We O
have O
previously O
described O
the O
partial O
purification O
of O
Ap4A B
hydrolase I
from O
S O
. O
pombe O
[ O
Robinson O
, O
de O
la O
Pena O
and O
Barnes O
( O
1993 O
) O
Biochim O
. O

Effect O
of O
a O
new O
positive O
inotropic O
agent O
( O
OPC O
- O
8212 O
) O
on O
intracellular O
potential O
of O
isolated O
canine O
ventricular O
papillary O
muscle O

The O
unc B
- I
101 I
, O
sli B
- I
1 I
, O
and O
rok B
- I
1 I
genes I
encode O
a O
distinct O
set O
of O
negative O
regulators O
of O
vulval O
differentiation O
. O

Thirty O
- O
seven O
patients O
with O
severe O
temporal O
lobe O
epilepsy O
were O
studied O
interictally O
with O
[ O
18F O
] O
fluorodeoxyglucose O
- O
PET O
in O
each O
of O
three O
conditions O
: O
resting O
, O
during O
emotional O
speech O
, O
and O
while O
performing O
a O
visual O
recognition O
task O
. O

SYC1 B
( O
suppressor B
of I
yeast I
cbf5 I
- I
1 I
) O
was O
identified O
as O
a O
multicopy O
suppressor O
of O
cbf5 B
- I
1 I
and O
subsequently O
was O
found O
to O
be O
identical O
to O
RRN3 B
, O
an O
RNA B
polymerase I
I I
transcription I
factor I
. O

Newly O
hatched O
F1 O
nymphs O
of O
Aiolopus O
thalassinus O
( O
Fabr O
. O
) O
were O
fed O
on O
food O
treated O
with O
various O
concentrations O
of O
HgCl2 O
, O
CdCl2 O
, O
and O
PbCl2 O
until O
the O
end O
of O
adult O
life O
. O

Neuronal O
activity O
of O
58 O
dopaminergic O
( O
DA O
) O
and O
200 O
non O
- O
dopaminergic O
( O
non O
- O
DA O
) O
neurons O
in O
the O
ventral O
tegmental O
area O
( O
VTA O
) O
of O
female O
monkeys O
was O
recorded O
, O
and O
correlation O
to O
bar O
press O
feeding O
, O
sensory O
stimulation O
and O
change O
in O
motivation O
was O
investigated O
. O

Both O
sequence O
analysis O
and O
restriction O
fragment O
length O
polymorphism O
analysis O
revealed O
a O
high O
degree O
of O
sequence O
( O
97 O
% O
homology O
) O
and O
structural O
similarity O
among O
members O
of O
the O
ART B
- I
CH I
family O
, O
indicating O
their O
common O
origin O
and O
recent O
penetration O
into O
chicken O
DNA O
. O

Three O
of O
these O
positions O
are O
novel O
in O
that O
they O
had O
not O
been O
identified O
to O
be O
important O
for O
binding O
PC B
by O
previous O
crystallographic O
analysis O
. O

We O
demonstrate O
inhibition O
of O
2 O
- O
deoxyglucose O
uptake O
in O
isolated O
primary O
rat O
adipocytes O
and O
3T3 O
- O
L1 O
adipocytes O
pretreated O
with O
U73122 O
. O

The O
elevated O
levels O
of O
inositol O
phosphates O
resulting O
from O
N B
( I
epi I
) I
alpha I
qQ209L I
expression O
were O
similar O
to O
those O
obtained O
with O
carbachol O
activation O
of O
the O
M1 B
muscarinic I
acetylcholine I
receptor I
. O

In O
addition O
, O
wild O
- O
type O
and O
mutated O
DNA O
templates O
were O
used O
as O
probes O
in O
DNase B
I I
protection O
experiments O
to O
determine O
sites O
of O
protein O
- O
DNA O
interaction O
. O

We O
tested O
the O
hypothesis O
that O
PEH O
is O
associated O
with O
reductions O
in O
TPR O
and O
sympathetic O
nerve O
activity O
( O
SNA O
) O
. O

Salt O
wars O
. O

RESULTS O
: O
BMK O
- O
1 O
and O
other O
19 O
contents O
significantly O
decreased O
depolarization O
parameters O
Vmax O
, O
APA O
, O
OS O
, O
and O
MDP O
, O
which O
was O
similar O
to O
that O
of O
TTX O
and O
different O
from O
that O
of O
Nim O
and O
BaCl2 O
. O

In O
the O
latter O
, O
the O
cells O
and O
adjacent O
matrix O
had O
several O
characteristics O
of O
fibrocartilage O
, O
including O
chondrocytes O
. O

Mannich O
bases O
of O
vanillin O
oxime O
. O

All O
exon O
/ O
intron O
splice O
junctions O
follow O
the O
GT O
/ O
AG O
rule O
. O

Previous O
work O
has O
shown O
that O
glucose O
repression O
of O
PRB1 B
transcription O
is O
not O
mediated O
by O
HXK2 B
or O
by O
the O
SNF1 B
, O
SNF4 B
, O
and O
SNF6 B
genes I
( O
C O
. O

Comparison O
of O
the O
encoded O
ERCC3Dm B
protein I
with O
the O
homologous O
proteins O
of O
mouse O
and O
man O
shows O
a O
strong O
amino O
acid O
conservation O
( O
71 O
% O
identity O
) O
, O
especially O
in O
the O
postulated O
DNA O
binding O
region O
and O
seven O
' B
helicase I
' I
domains O
. O

The O
pyridostigmine O
inhibited O
AChE B
recovered O
only O
in O
the O
100 O
mumol O
kg O
- O
1 O
kg O
oxime O
groups O
at O
the O
end O
of O
the O
experiment O
. O

In O
the O
case O
of O
a O
moderate O
buccal O
Class O
II O
tendency O
, O
55 O
% O
of O
the O
molars O
were O
in O
a O
Class O
I O
relationship O
when O
evaluated O
from O
the O
lingual O
side O
, O
whereas O
45 O
% O
belonged O
to O
mild O
( O
1st O
degrees O
) O
Class O
II O
with O
molar O
rotations O
present O
in O
81 O
% O
of O
the O
cases O
. O

Mutation O
of O
this O
threonine O
to O
isoleucine O
had O
no O
observable O
effect O
on O
either O
nuclear O
localization O
of O
E1 B
or O
DNA O
replication O
of O
the O
intact O
viral O
genome O
. O

Serum B
prolactin I
and O
oestradiol O
levels O
at O
different O
stages O
of O
puberty O
. O

This O
suggests O
that O
the O
duration O
of O
varicocele O
per O
se O
could O
affect O
DHT O
seminal O
plasma O
levels O
. O

Left O
ventricular O
responses O
to O
dopamine O
in O
dilated O
cardiomyopathy O
as O
assessed O
by O
two O
- O
dimensional O
echocardiography O
and O
compared O
with O
findings O
of O
thallium O
- O
201 O
scintigraphy O
. O

Detailed O
comparison O
of O
the O
urinary O
excretion O
of O
purines O
in O
a O
patient O
with O
the O
Lesch O
- O
Nyhan O
syndrome O
and O
a O
control O
subject O
. O

This O
enzyme O
is O
distinct O
from O
other O
known O
E3s B
, O
including O
E3 B
alpha I
/ O
UBR1 B
, O
E3 B
beta I
, O
and O
E6 B
- I
AP I
. O

This O
evidence O
, O
together O
with O
the O
ability O
of O
a O
carboxyl O
- O
terminal O
coding O
sequence O
starting O
from O
the O
BamHI B
site I
to O
complement O
a O
shy1 B
mutant I
, O
suggests O
that O
the O
Shy1p B
contains O
two O
domains O
that O
can O
be O
separately O
expressed O
to O
form O
a O
functional O
protein O
. O

The O
zinc B
finger I
protein I
A20 I
is O
a O
tumor B
necrosis I
factor I
( O
TNF B
) O
- O
and O
interleukin B
1 I
( O
IL B
- I
1 I
) O
- O
inducible O
protein O
that O
negatively O
regulates O
nuclear B
factor I
- I
kappa I
B I
( O
NF B
- I
kappaB I
) O
- O
dependent O
gene O
expression O
. O

A O
visibly O
distinct O
muscular O
hypertrophy O
( O
mh O
) O
, O
commonly O
known O
as O
double O
muscling O
, O
occurs O
with O
high O
frequency O
in O
the O
Belgian O
Blue O
and O
Piedmontese O
cattle O
breeds O
. O

For O
the O
population O
of O
patients O
with O
melanoma O
levels O
III O
and O
IV O
significant O
differences O
in O
survival O
rate O
were O
associated O
with O
thickness O
( O
less O
than O
2 O
mm O
or O
greater O
than O
or O
equal O
to O
2 O
mm O
; O
p O
less O
than O
0 O
, O
001 O
) O
, O
though O
not O
with O
level O
. O

At O
least O
one O
such O
complex O
is O
eliminated O
by O
preincubating O
the O
nuclear O
extract O
with O
an O
antibody O
with O
broad O
cross O
- O
reactivity O
with O
Ets B
- I
1 I
and O
Ets B
- I
2 I
proteins I
, O
thus O
confirming O
that O
an O
ETS B
transcription I
factor I
( I
s I
) I
recognizes O
the O
- O
12 O
motif O
. O

Mutant B
beta2 I
- I
adrenergic I
receptors I
with O
a O
Tyr O
- O
to O
- O
Phe O
substitution O
at O
Tyr O
- O
350 O
do O
not O
display O
agonist O
- O
induced O
desensitization O
, O
Src B
recruitment O
, O
or O
Src B
activation O
. O

Role O
of O
distinct O
mitogen B
- I
activated I
protein I
kinase I
pathways O
and O
cooperation O
between O
Ets B
- I
2 I
, O
ATF B
- I
2 I
, O
and O
Jun B
family I
members O
in O
human B
urokinase I
- I
type I
plasminogen I
activator I
gene I
induction O
by O
interleukin B
- I
1 I
and O
tetradecanoyl O
phorbol O
acetate O
. O

Immunoblot O
analysis O
of O
purified O
and O
protease O
- O
digested O
intracellular O
mature O
virus O
( O
IMV O
) O
and O
extracellular O
enveloped O
virus O
( O
EEV O
) O
showed O
that O
the O
A36R B
proteins I
were O
present O
on O
the O
surface O
of O
EEV O
with O
type O
II O
membrane O
topology O
, O
but O
were O
absent O
from O
IMV O
. O

Here O
we O
demonstrate O
that O
several O
genes O
associated O
with O
NHEJ O
perform O
essential O
functions O
in O
the O
repair O
of O
endonuclease O
- O
induced O
DSBs O
in O
vivo O
. O

The O
failure O
suggests O
that O
these O
assembly O
competent O
but O
complementation O
- O
negative O
alpha B
mutants I
lack O
an O
as O
yet O
unidentified O
function O
( O
s O
) O
. O

Psychopathological O
, O
vegetative O
, O
and O
vital O
parameters O
were O
assessed O
every O
hour O
. O

RESULTS O
: O
Considering O
an O
inulin O
clearance O
of O
less O
than O
80 O
ml O
/ O
min O
/ O
1 O
. O
73 O
m2 O
, O
the O
cut O
- O
off O
value O
for O
serum O
creatinine O
was O
11 O
. O
5 O
mumol O
/ O
liter O
for O
men O
and O
90 O
mumol O
/ O
liter O
for O
women O
. O

These O
data O
may O
indicate O
redundancy O
between O
members O
of O
the O
NGFI B
- I
B I
/ O
Nurr1 B
/ O
Nor1 B
subfamily O
and O
could O
explain O
why O
no O
phenotypic O
disturbances O
have O
yet O
been O
found O
in O
mice O
in O
which O
the O
NGFI B
- I
B I
gene I
has O
been O
inactivated O
. O

Manual O
sample O
clean O
- O
up O
procedures O
as O
well O
as O
the O
addition O
of O
an O
internal O
standard O
are O
not O
needed O
. O

To O
directly O
address O
this O
issue O
, O
we O
used O
a O
tetracycline O
- O
regulated O
system O
in O
human O
U2OS O
osteosarcoma O
cells O
and O
thus O
found O
that O
BCL6 B
mediates O
growth O
suppression O
associated O
with O
impaired O
S O
phase O
progression O
and O
apoptosis O
. O

TAAG O
sites O
that O
serve O
as O
functional O
late O
promoters O
in O
OpMNPV O
were O
found O
to O
mediate O
transcription O
initiation O
at O
only O
basal O
levels O
in O
the O
context O
of O
the O
AcMNPV O
genome O
, O
suggesting O
that O
late O
promoter O
activation O
may O
be O
virus O
specific O
within O
the O
family O
Baculoviridae O
. O

Stability O
of O
125 O
I O
- O
labeled O
insulin B
used O
in O
radioimmunoassay O
of O
insulin B
. O

In O
absence O
of O
i O
. O
m O
. O
medication O
, O
levels O
over O
1 O
, O
000 O
U O
must O
be O
carefully O
screened O
in O
order O
to O
rule O
out O
SNM O
or O
organic O
pathology O
associated O
. O

Paclitaxel O
caused O
a O
rapid O
and O
transient O
increase O
in O
c B
- I
Jun I
NH2 I
- I
terminal I
kinase I
( O
JNK B
) O
activity O
, O
a O
proposed O
mediator O
of O
stress O
activation O
pathways O
. O

These O
results O
indicate O
that O
H B
- I
2RIIBP I
( I
i I
) I
is O
a O
member O
of O
the O
superfamily O
of O
nuclear B
hormone I
receptors I
and O
( O
ii O
) O
may O
regulate O
not O
only O
MHC B
class I
I I
genes I
but O
also O
genes O
containing O
the O
ERE O
and O
related O
sequences O
. O

Two O
courses O
of O
chemotherapy O
( O
CBCDA O
+ O
VDS O
) O
failed O
to O
gain O
any O
improvement O
, O
and O
the O
pain O
resulting O
from O
recurrent O
bone O
metastases O
was O
managed O
mainly O
by O
the O
administration O
of O
the O
best O
supportive O
care O
. O

We O
show O
here O
that O
Spb1p B
is O
able O
to O
bind O
[ O
( O
3 O
) O
H O
] O
AdoMet O
in O
vitro O
, O
suggesting O
that O
it O
is O
a O
novel O
methylase O
, O
whose O
possible O
substrates O
will O
be O
discussed O
. O

RESULTS O
: O
Lateral O
PF O
OA O
was O
more O
common O
than O
medial O
PF O
OA O
( O
P O
< O
0 O
. O
0001 O
) O
. O

CONCLUSION O
: O
Urbanisation O
is O
associated O
with O
an O
increase O
in O
the O
prevalence O
rates O
of O
some O
risk O
behaviours O
. O

However O
, O
additional O
studies O
on O
cell O
lines O
and O
whole O
animals O
are O
required O
to O
understand O
GnRH B
signaling O
in O
the O
context O
of O
other O
hormones O
during O
the O
reproductive O
cycle O
of O
mouse O
and O
human O
. O

Serum O
angiogenin I
concentrations O
in O
young O
patients O
with O
diabetes O
mellitus O
. O

This O
duplicated O
genomic O
region O
is O
also O
linked O
tightly O
to O
D1Z2 B
, O
a O
genetic O
marker O
containing O
a O
highly O
polymorphic O
VNTR O
( O
variable O
number O
tandem O
repeat O
) O
consisting O
of O
an O
unusual O
40 O
- O
bp O
reiterated O
sequence O
. O

ATF6 B
, O
a O
basic B
- I
leucine I
zipper I
protein I
, O
was O
isolated O
by O
binding O
to O
SRF B
and O
in O
particular O
to O
its O
transcriptional O
activation O
domain O
. O

Cetn1 B
possesses O
the O
sequence O
features O
of O
an O
expressed O
retroposon O
: O
the O
gene O
lacks O
introns O
, O
the O
open O
reading O
frame O
is O
not O
interrupted O
by O
stop O
codons O
, O
and O
the O
coding O
region O
is O
flanked O
by O
a O
pair O
of O
direct O
repeats O
. O

The O
largest O
one O
probably O
corresponds O
to O
the O
precursor O
form O
of O
PS2 B
in O
E O
. O
coli O
. O

The O
Aspergillus B
uvsH I
gene I
encodes O
a O
product O
homologous O
to O
yeast B
RAD18 I
and O
Neurospora B
UVS I
- I
2 I
. O

On O
average O
these O
maximum O
potency O
estimates O
were O
within O
one O
order O
of O
magnitude O
of O
the O
inverse O
maximum O
dose O
tested O
. O

VAV B
and O
SOCS1 B
form O
a O
protein O
complex O
through O
interactions O
between O
the O
VAV B
NH I
( I
2 I
) I
- I
terminal I
regulatory I
region I
and O
the O
SH2 B
domain I
of O
SOCS1 B
in O
a O
phosphotyrosine O
- O
independent O
manner O
. O

The O
originally O
embryonic B
gamma I
- I
globin I
locus I
duplicated O
and O
acquired O
a O
novel O
( O
fetal O
) O
pattern O
of O
expression O
in O
a O
defined O
time O
period O
( O
55 O
- O
40 O
million O
years O
ago O
) O
during O
primate O
phylogeny O
. O

Clostridium O
botulinum O
in O
marine O
sediment O

Northern O
blot O
analysis O
with O
GPR37 B
probes O
revealed O
a O
main O
3 O
. O
8 O
- O
kb O
mRNA O
and O
a O
less O
abundant O
8 O
- O
kb O
mRNA O
, O
both O
expressed O
in O
human O
brain O
tissues O
, O
particularly O
in O
corpus O
callosum O
, O
medulla O
, O
putamen O
, O
and O
caudate O
nucleus O
. O

Our O
studies O
indicate O
no O
single O
trans O
- O
acting O
factor O
is O
absolutely O
essential O
for O
enhancer O
activity O
, O
and O
that O
the O
enhancer O
activity O
of O
MerI B
is O
mediated O
via O
a O
combinatorial O
and O
additive O
mechanism O
. O

In O
human O
WI38 O
cells O
, O
the O
E1B B
19K I
gene I
mutant I
viruses O
had O
a O
substantial O
growth O
advantage O
over O
the O
wild O
- O
type O
virus O
, O
yielding O
500 O
- O
fold O
- O
higher O
titers O
. O

To O
elucidate O
the O
mechanism O
of O
these O
hormonal O
effects O
, O
we O
have O
studied O
the O
regulatory O
regions O
of O
the O
PFK B
- I
2 I
gene I
in O
transfection O
experiments O
. O

Consequently O
, O
during O
the O
evolution O
of O
mammals O
, O
it O
is O
the O
CKbeta4GT B
- I
I I
gene I
lineage O
that O
has O
been O
recruited O
for O
the O
biosynthesis O
of O
lactose O
. O

Furthermore O
, O
disruption O
of O
pcr1 B
reduced O
expression O
of O
fbp1 B
, O
a O
glucose O
- O
repressible O
gene O
negatively O
regulated O
by O
PKA B
. O

In O
solution O
, O
[ O
( O
125 O
) O
I O
] O
- O
labeled O
ankyrin B
was O
found O
by O
ND O
- O
PAGE3 B
to O
enhance O
the O
affinity O
of O
spectrin B
self O
- O
association O
by O
10 O
- O
fold O
. O

Transcription O
analysis O
of O
the O
EcoRI B
D I
region I
of I
the I
baculovirus I
Autographa I
californica I
nuclear I
polyhedrosis I
virus I
identifies O
an O
early O
4 O
- O
kilobase O
RNA O
encoding O
the O
essential O
p143 B
gene I
. O

Anesthesia O
in O
the O
radical O
surgery O
of O
arteriosclerotic O
coronary O
disease O

5 O
Intravenous O
prizidilol O
hydrochloride O
decreases O
resting O
blood O
pressure O
and O
left O
ventricular O
area O
, O
increases O
pulse O
rate O
and O
has O
virtually O
no O
effect O
on O
left O
ventricular O
ejection O
fraction O
. O

In O
this O
study O
, O
the O
subcellular O
location O
, O
domain O
structure O
, O
and O
biochemical O
function O
of O
metaxin B
were O
investigated O
. O

The O
human O
cDNA O
was O
cloned O
and O
sequenced O
; O
it O
was O
shown O
to O
have O
an O
open O
reading O
frame O
encoding O
a O
296 O
- O
amino O
- O
acid O
protein O
in O
which O
could O
be O
identified O
four O
peptides O
previously O
identified O
by O
micro O
- O
sequencing O
purified O
protein O
. O

Our O
aim O
was O
to O
evaluate O
this O
technique O
by O
cross O
- O
calibrating O
the O
DXA O
method O
with O
the O
carcass O
chemical O
analysis O
in O
a O
heterogeneous O
population O
of O
nondiabetic O
Wistar O
and O
diabetic O
GK O
rats O
( O
21 O
animals O
were O
used O
for O
precision O
error O
and O
reproducibility O
determinations O
and O
26 O
were O
used O
for O
accuracy O
studies O
) O
. O

Flux O
of O
the O
paramyxovirus O
hemagglutinin B
- O
neuraminidase I
glycoprotein O
through O
the O
endoplasmic O
reticulum O
activates O
transcription O
of O
the O
GRP78 B
- O
BiP B
gene O
. O

The O
preference O
for O
third O
base O
codon O
in O
Y O
position O
prolines O
is O
U O
for O
the O
alpha B
2 I
( I
VI I
) I
collagen I
as O
it O
is O
for O
the O
human B
fibrillar I
collagen I
genes I
. O

Small O
cell O
tumors O
in O
children O
: O
contribution O
to O
the O
solution O
of O
the O
problem O
of O
differential O
diagnosis O
with O
immunohistochemistry O
and O
electron O
microscopy O

Detonation O
- O
traumatic O
load O
during O
military O
service O

In O
static O
conditions O
, O
body O
sway O
was O
assessed O
using O
a O
conventional O
force O
platform O
with O
eyes O
open O
and O
with O
eyes O
closed O
. O

Soggy B
, O
a O
spermatocyte O
- O
specific O
gene O
, O
lies O
3 O
. O
8 O
kb O
upstream O
of O
and O
antipodal O
to O
TEAD B
- I
2 I
, O
a O
transcription O
factor O
expressed O
at O
the O
beginning O
of O
mouse O
development O
. O

Teratospermia O
is O
therefore O
an O
important O
factor O
in O
evaluating O
fertilization O
capacity O
. O

Results O
of O
the O
study O
suggest O
that O
Chinese O
medicine O
use O
among O
this O
population O
depends O
on O
health O
conditions O
, O
and O
that O
having O
a O
regular O
source O
of O
care O
for O
Chinese O
medicine O
as O
well O
as O
a O
preference O
for O
Chinese O
medicine O
are O
two O
predictors O
for O
its O
use O
. O

The O
greatest O
risk O
of O
preterm O
prelabour O
rupture O
of O
membranes O
( O
PPROM O
) O
is O
preterm O
delivery O
. O

DISCUSSION O
: O
The O
higher O
absolute O
knee O
extension O
strength O
measures O
of O
leg O
and O
the O
similar O
extension O
strength O
of O
the O
trunk O
in O
the O
obese O
sample O
compared O
to O
the O
lean O
might O
be O
explained O
by O
the O
training O
effect O
of O
weight O
bearing O
and O
support O
of O
a O
larger O
body O
mass O
. O

Residues O
of O
Stauffer O
R O
- O
3828 O
and O
its O
oxygen O
analogue O
in O
the O
body O
tissues O
of O
cattle O
fed O
R O
- O
3828 O
in O
the O
diet O
. O

The O
goals O
of O
these O
experiments O
were O
to O
determine O
whether O
lactational O
anestrus O
would O
be O
prolonged O
by O
a O
48 O
- O
h O
fast O
at O
days O
13 O
and O
14 O
postpartum O
( O
pp O
) O
and O
, O
if O
so O
, O
to O
determine O
whether O
this O
effect O
could O
be O
reversed O
by O
treatment O
with O
the O
Ob B
protein I
leptin B
. O

In O
pituitary O
GH3 O
cells O
, O
vitamin O
D O
increases O
the O
levels O
of O
PRL B
transcripts I
and O
stimulates O
the O
PRL B
promoter I
. O

However O
, O
electron O
microscopy O
revealed O
that O
while O
sec4 B
mutant I
cells O
accumulate O
secretory O
vesicles O
, O
ypt31 B
/ I
32 I
mutant I
cells O
accumulate O
aberrant O
Golgi O
structures O
. O

METHODS O
: O
We O
retrospectively O
analyzed O
the O
data O
of O
20 O
patients O
who O
underwent O
both O
gated O
fluorine O
18 O
deoxyglucose O
( O
FDG O
) O
- O
PET O
and O
equilibrium O
radionuclide O
angiography O
( O
ERNA O
) O
. O

However O
, O
it O
is O
now O
suggested O
that O
the O
use O
of O
more O
slowly O
digested O
starchy O
foods O
may O
have O
positive O
health O
benefits O
. O

Northern O
blot O
analysis O
demonstrated O
that O
the O
genes O
are O
transcribed O
separately O
. O

Complete O
activation O
of O
signal B
transducer I
and I
activator I
of I
transcription I
1 I
( O
STAT1 B
) O
requires O
phosphorylation O
at O
both O
Y701 O
and O
a O
conserved O
PMS B
( I
727 I
) I
P I
sequence O
. O

Over O
half O
of O
the O
children O
initially O
unresponsive O
to O
H O
. O
influenzae O
type O
b O
meningitis O
subsequently O
developed O
specific O
antibodies O
. O

It O
is O
important O
to O
understand O
the O
etiology O
of O
HITT O
because O
of O
its O
devastating O
clinical O
consequences O
. O

Five O
sequence O
variants O
were O
identified O
. O

The O
Ras B
GTPase I
- I
activating I
- I
protein I
- I
related I
human I
protein I
IQGAP2 I
harbors O
a O
potential O
actin B
binding I
domain I
and O
interacts O
with O
calmodulin B
and O
Rho B
family I
GTPases I
. O

We O
anticipated O
SNS O
function O
after O
CVA O
to O
be O
asymmetric O
and O
selected O
null O
hypotheses O
of O
bilaterally O
symmetric O
SSR O
latencies O
and O
amplitudes O
irrespective O
of O
side O
of O
stimulation O
and O
/ O
or O
recording O
. O

Partial O
hepatectomy O
in O
metastatic O
Wilms O
' O
tumor O
. O

The O
adoption O
of O
a O
twisted O
structure O
of O
importin B
- I
beta I
is O
essential O
for O
the O
protein O
- O
protein O
interaction O
required O
for O
nuclear O
transport O
. O

The O
presence O
of O
a O
short O
( O
70 O
bp O
) O
intron O
near O
the O
N O
- O
terminus O
of O
the O
atX B
gene I
was O
predicted O
that O
contains O
the O
canonical O
GT O
and O
AG O
dinucleotides O
at O
its O
5 O
' O
- O
and O
3 O
' O
- O
splicing O
junctions O
. O

Moreover O
, O
a O
hybrid O
protein O
composed O
of O
a O
PvALF B
activation I
domain I
and O
the O
DNA O
binding O
and O
dimerization O
domain O
of O
ROM2 B
activated O
gene O
expression O
, O
indicating O
that O
ROM2 B
recognizes O
the O
DLEC2 B
enhancer I
in O
vivo O
; O
consequently O
, O
ROM2 B
functions O
as O
a O
DNA O
binding O
site O
- O
dependent O
repressor O
. O

Tc O
- O
99m O
HMPAO O
brain O
SPECT O
compared O
to O
CT O
and O
EEG O
after O
seizures O
in O
childhood O
. O

We O
found O
that O
the O
central O
nonconserved O
region O
of O
the O
large O
subunit O
is O
not O
essential O
for O
function O
and O
likely O
acts O
as O
a O
spacer O
between O
the O
conserved O
N O
- O
and O
C O
- O
terminal O
regions O
. O

On O
the O
other O
hand O
, O
NE O
transport O
and O
antagonist O
( O
[ O
125I O
] O
RTI O
- O
55 O
) O
binding O
assays O
on O
whole O
LLC O
- O
NET O
cells O
treated O
with O
tunicamycin O
reveal O
a O
pronounced O
reduction O
in O
NE O
transport O
activity O
and O
hNET B
membrane O
density O
paralleled O
by O
an O
inability O
of O
NET B
proteins I
to O
replenish O
the O
higher O
M O
( O
r O
) O
hNET B
pool O
. O

We O
show O
that O
the O
upstream O
region O
of O
the O
beta B
- I
MyHC I
gene I
( O
- O
5518 O
to O
- O
2490 O
relative O
to O
the O
transcriptional O
start O
site O
) O
directed O
high O
levels O
of O
transcriptional O
activity O
only O
when O
stably O
integrated O
, O
but O
not O
when O
expressed O
extrachromosomally O
in O
transient O
assays O
. O

DNase B
I I
protection O
analysis O
as O
well O
as O
oligonucleotide O
competition O
experiments O
indicate O
that O
this O
binding O
is O
sequence O
specific O
. O

Plasmid O
pSP64E6E7 O
which O
contains O
the O
reading O
frames O
of O
both O
E6 B
and O
E7 B
was O
constructed O
in O
order O
to O
study O
the O
expression O
of O
both O
proteins O
in O
a O
coupled O
transcription O
/ O
rabbit O
reticulocyte O
translation O
system O
. O

In O
transient O
transfections O
using O
luciferase B
reporter I
genes I
driven O
by O
1 O
kb O
of O
the O
5 O
' O
flanking O
DNA O
of O
the O
three O
CALM B
genes I
, O
the O
promoter O
activity O
correlated O
with O
the O
endogenous O
CALM I
transcriptional O
activity O
, O
but O
only O
when O
the O
5 O
' O
untranslated O
regions O
were O
included O
in O
the O
constructs O
. O

These O
findings O
suggest O
that O
PGE1 O
is O
effective O
in O
increasing O
PVF O
in O
the O
liver O
transplanted O
condition O
; O
however O
, O
the O
hepatic O
circulatory O
improvement O
attributed O
to O
this O
agent O
would O
be O
limited O
to O
the O
first O
few O
days O
following O
transplantation O
. O

The O
derepressed O
expression O
of O
fixN B
was O
not O
observed O
in O
a O
purH B
mutant I
. O

Nearly O
1 O
. O
5 O
million O
American O
men O
age O
65 O
and O
older O
have O
osteoporosis O
, O
and O
another O
3 O
. O
5 O
million O
are O
at O
risk O
. O

Both O
viruses O
express O
the O
mil B
/ O
raf B
oncogene O
product O
as O
a O
gag B
- I
fusion I
polyprotein I
, O
while O
the O
myc B
oncogene I
of O
MH2 O
is O
expressed O
via O
a O
subgenomic O
mRNA O
. O

CONCLUSION O
: O
DaunoXome O
has O
an O
improved O
pharmacokinetic O
profile O
compared O
with O
free O
daunorubicin O
, O
and O
is O
well O
tolerated O
. O

This O
regimen O
may O
be O
considered O
front O
- O
line O
therapy O
when O
autologous O
stem O
cell O
transplantation O
is O
not O
feasible O
and O
when O
a O
rapid O
response O
is O
particularly O
important O
. O

Recombinant O
protein O
encoded O
by O
ESX B
( O
for O
epithelial O
- O
restricted O
with O
serine O
box O
) O
exhibits O
Ets B
- O
like O
DNA O
binding O
specificity O
in O
electrophoretic O
mobility O
shift O
assays O
and O
, O
in O
transient O
transfection O
assays O
, O
transactivates O
Ets B
- O
responsive I
promoter I
elements O
including O
that O
found O
in O
the O
HER2 B
/ O
neu B
oncogene O
. O

In O
area O
CA1 O
of O
the O
hippocampus O
, O
numbers O
of O
normal O
neurons O
were O
increased O
11 O
- O
to O
14 O
- O
fold O
by O
MK O
- O
801 O
treatment O
( O
p O
< O
0 O
. O
01 O
) O
. O

The O
temporal O
changes O
in O
the O
plasma O
concentration O
of O
immunoreactive B
atrial I
natriuretic I
factor I
( O
iANF B
) O
were O
studied O
in O
six O
conscious O
dogs O
with O
an O
arteriovenous O
( O
AV O
) O
fistula O
, O
a O
model O
of O
chronic O
high O
- O
output O
heart O
failure O
. O

The O
revaluation O
at O
the O
end O
of O
the O
study O
showed O
a O
good O
compliance O
for O
the O
proposed O
diet O
scheme O
by O
children O
, O
but O
a O
poor O
compliance O
by O
their O
families O
. O

The O
delta O
G O
values O
of O
these O
regions O
were O
higher O
, O
i O
. O
e O
. O
potential O
secondary O
structural O
elements O
were O
fewer O
, O
than O
in O
TIR O
of O
genes O
from O
E O
. O
coli O
. O

Two O
closely O
related O
groups O
of O
transcripts O
, O
Sagrp1 B
and O
Sagrp2 B
, O
controlled O
by O
a O
circadian O
rhythm O
have O
been O
isolated O
. O

Of O
921 O
specimens O
, O
95 O
yielded O
non O
- O
albicans O
species O
, O
mainly O
from O
patients O
with O
low O
CD4 B
lymphocyte O
counts O
and O
extensive O
previous O
azole O
exposure O
. O

These O
findings O
suggest O
that O
eIF5 B
- O
eIF2beta B
interaction O
plays O
an O
essential O
role O
in O
eIF5 B
function O
in O
eukaryotic O
cells O
. O

Three O
simple O
methods O
of O
detecting O
malnutrition O
on O
medical O
wards O
. O

Compared O
with O
controls O
, O
patients O
in O
the O
unmedicated O
state O
had O
low O
smooth O
pursuit O
gain O
, O
had O
a O
higher O
rate O
of O
corrective O
catch O
- O
up O
saccades O
, O
and O
tended O
to O
spend O
less O
time O
engaged O
in O
the O
tracking O
task O
. O

Cytokine O
- O
mediated O
I B
kappa I
B I
alpha I
reappearance O
was O
completely O
blocked O
by O
the O
protein O
synthesis O
inhibitor O
cycloheximide O
. O

Development O
of O
P O
carinii O
pneumonia O
was O
associated O
with O
the O
stage O
of O
Kaposi O
' O
s O
sarcoma O
, O
B O
subtype O
disease O
, O
and O
the O
presence O
of O
0 O
. O
20 O
X O
10 O
( O
9 O
) O
/ O
L O
( O
200 O
/ O
mm3 O
) O
or O
fewer O
CD4 B
cells O
at O
study O
entry O
. O

The O
effects O
of O
tilmicosin O
administration O
in O
the O
feed O
at O
400 O
mg O
/ O
kg O
and O
an O
injection O
therapy O
of O
clinically O
diseased O
pigs O
with O
long O
- O
acting O
oxytetracycline O
( O
Terramycine O
LA O
) O
at O
20 O
mg O
/ O
kg O
bodyweight O
were O
compared O
. O

Those O
participating O
in O
this O
investigation O
( O
65 O
centers O
= O
79 O
% O
) O
received O
a O
series O
of O
computer O
disks O
containing O
50 O
99mTc O
- O
DMSA O
studies O
. O

Pathogenesis O
of O
fever O
. O

Two O
full O
- O
length O
clones O
( O
pBCH1 B
and O
pBCH2 B
) O
were O
isolated O
. O

Childbirth O
and O
authoritative O
knowledge O
. O

The O
ends O
of O
the O
barrel O
are O
capped O
by O
short O
helices O
. O

Non O
- O
finger O
- O
coding O
modules O
conserved O
among O
members O
of O
subfamilies O
of O
zinc B
finger I
genes I
have O
been O
described O
in O
the O
murine O
genome O
( O
finger O
- O
associated O
boxes O
, O
or O
FAX O
domain O
) O
and O
the O
human O
genome O
( O
Kruppel B
- I
associated I
boxes I
, O
or O
KRAB B
domain I
) O
. O

This O
combination O
is O
considered O
pathognomonic O
for O
factitious O
hyperinsulinism O
. O

Comparison O
of O
the O
5 O
' O
flanking O
regions O
of O
the O
mouse O
J B
kappa I
elements I
, O
including O
the O
conserved O
putative O
recombination O
target O
sequences O
, O
shows O
no O
obvious O
differences O
consistent O
with O
the O
variation O
in O
recombinational O
efficiency O
, O
so O
we O
conclude O
that O
, O
although O
the O
consensus O
heptamer O
and O
nonamer O
signals O
may O
be O
sufficient O
to O
identify O
a O
recombination O
site O
, O
the O
probability O
that O
that O
site O
will O
be O
used O
depends O
also O
on O
other O
determinants O
. O

The O
melanoma O
growth O
stimulatory I
activity O
/ O
growth I
- I
regulated I
protein I
, O
CXCL1 B
, O
is O
constitutively O
expressed O
at O
high O
levels O
during O
inflammation O
and O
progression O
of O
melanocytes O
into O
malignant O
melanoma O
. O

Genetic O
analysis O
places O
CWH43 B
upstream O
of O
the O
BCK2 B
branch O
of O
the O
PKC1 B
signalling O
pathway O
, O
since O
cwh43 B
mutations O
were O
synthetic O
lethal O
with O
pkc1 B
deletion O
, O
whereas O
the O
cwh43 B
defects O
could O
be O
rescued O
by O
overexpression O
of O
BCK2 B
and O
not O
by O
high O
- O
copy O
- O
number O
expression O
of O
genes O
encoding O
downstream O
proteins O
of O
the O
PKC1 B
pathway O
However O
, O
unlike O
BCK2 B
, O
whose O
disruption O
in O
a O
cln3 B
mutant I
resulted O
in O
growth O
arrest O
in O
G O
( O
1 O
) O
, O
no O
growth O
defect O
was O
observed O
in O
a O
double O
cwh43 B
cln3 B
mutants I
. O

Twelve O
( O
92 O
% O
) O
showed O
cognitive O
dysfunction O
including O
impairments O
in O
memory O
, O
attention O
, O
and O
affective O
disturbances O
( O
anxiety O
, O
depression O
, O
irritability O
, O
and O
poor O
frustration O
tolerance O
) O
. O

To O
clarify O
the O
mechanisms O
that O
regulate O
transcription O
of O
the O
GTP B
cyclohydrolase I
I I
gene I
and O
to O
generate O
multiple O
species O
of O
mRNA O
, O
we O
isolated O
genomic O
DNA O
clones O
for O
the O
human B
and I
mouse I
GTP I
cyclohydrolase I
I I
genes I
. O

Therefore O
, O
a O
structural O
interaction O
between O
PsaL B
and O
PsaI B
may O
stabilize O
the O
association O
of O
PsaL B
with O
the O
photosystem O
I O
core O
. O

Reversal O
of O
' O
refractory O
septic O
shock O
' O
by O
infusion O
of O
amrinone O
and O
angiotensin B
II I
in O
an O
anthracycline O
- O
treated O
patient O
. O

The O
present O
study O
deals O
with O
the O
screening O
of O
11 O
of O
these O
plants O
against O
the O
opportunistic O
pathogen O
fungus O
Candida O
albicans O
. O

Negative O
modulation O
of O
alpha1 B
( I
I I
) I
procollagen I
gene I
expression O
in O
human O
skin O
fibroblasts O
: O
transcriptional O
inhibition O
by O
interferon B
- I
gamma I
. O

On O
the O
basis O
of O
this O
homology O
, O
we O
examined O
the O
potential O
role O
of O
c B
- I
Rel I
in O
controlling O
IL B
- I
2R I
alpha I
transcription O
. O

Expression O
of O
CBP B
/ O
p300 B
potentiated O
HS2 O
- O
mediated O
transactivation O
. O

We O
next O
developed O
a O
rapid O
purification O
method O
for O
bacterial O
recombinant O
MyoD B
- O
bHLH O
domain O
by O
affinity O
chromatography O
using O
a O
calmodulin B
- O
Sepharose O
column O
and O
investigated O
the O
phosphorylation O
of O
that O
peptide O
by O
PKC B
and O
its O
interactions O
with O
calmodulin B
and O
S100a B
. O

According O
to O
the O
penetration O
of O
Ca O
- O
45 O
, O
the O
microleakage O
level O
was O
scored O
for O
each O
section O
. O

HL B
is O
hypothesized O
to O
directly O
couple O
HDL B
lipid O
metabolism O
to O
tissue O
/ O
cellular O
lipid O
metabolism O
. O

Epstein B
- I
Barr I
virus I
nuclear I
antigen I
2 I
exerts O
its O
transactivating O
function O
through O
interaction O
with O
recombination B
signal I
binding I
protein I
RBP B
- I
J I
kappa I
, O
the O
homologue O
of O
Drosophila B
Suppressor I
of I
Hairless I
. O

Maduromycosis O
in O
Italy O

Two O
patient O
groups O
were O
studied O
: O
Type O
I O
- O
- O
with O
a O
new O
ePTFE O
graft O
; O
and O
Type O
II O
- O
- O
with O
thrombectomy O
and O
/ O
or O
revision O
of O
a O
previously O
placed O
ePTFE O
graft O
. O

The O
author O
describes O
the O
technique O
in O
detail O
and O
presents O
the O
findings O
for O
25 O
patients O
( O
10 O
men O
and O
15 O
women O
) O
examined O
for O
suspected O
ileostomy O
dysfunction O
, O
recurrent O
Crohn O
' O
s O
disease O
or O
ileal O
obstruction O
remote O
from O
the O
stoma O
. O

Increased O
susceptibility O
of O
aphasics O
to O
a O
distractor O
task O
in O
the O
recall O
of O
verbal O
commands O
. O

Lasting O
paraplegia O
caused O
by O
loss O
of O
lumbar O
spinal O
cord O
interneurons O
in O
rats O
: O
no O
direct O
correlation O
with O
motor O
neuron O
loss O
. O

The O
existence O
of O
universal O
non O
- O
specific O
mechanism O
of O
action O
of O
external O
factors O
upon O
the O
living O
tissue O
based O
on O
the O
DEL O
- O
reaction O
is O
suggested O
. O

Rv O
was O
7 O
. O
7 O
+ O
/ O
- O
1 O
. O
4 O
mmHg O
. O
ml O
- O
1 O
. O
min O
. O
100 O
g O
- O
1 O
and O
Cv O
was O
0 O
. O
59 O
+ O
/ O
- O
0 O
. O
25 O
ml O
/ O
mmHg O
, O
tau O
v O
calculated O
from O
the O
product O
of O
Rv O
and O
Cv O
was O
4 O
. O
20 O
+ O
/ O
- O
1 O
. O
58 O
s O
and O
from O
the O
ratio O
of O
delta O
V O
to O
delta O
Q O
was O
4 O
. O
95 O
+ O
/ O
- O
1 O
. O
53 O
s O
( O
P O
= O
NS O
) O
at O
a O
mean O
Pel O
of O
17 O
. O
6 O
+ O
/ O
- O
3 O
. O
7 O
mmHg O
. O
delta O
V O
was O
also O
produced O
by O
changing O
Pv O

; O
the O
average O
tau O
v O
( O
1 O
. O
95 O
+ O
/ O
- O
0 O
. O
37 O
s O
) O
, O
was O
shorter O
than O
that O
with O
changes O
in O
flow O
. O

We O
constructed O
a O
stable O
, O
doubly O
transfected O
cell O
line O
( O
TIS O
- O
10 O
) O
carrying O
a O
chromosomally O
integrated O
ptetO7 B
- O
CMV B
- I
L I
reporter O
construct O
and O
expressing O
the O
TetR B
- O
KRAB B
protein O
. O

We O
suggest O
that O
electrical O
stimulation O
of O
the O
NTS O
in O
rats O
undergoing O
such O
surgical O
preparation O
to O
observe O
the O
pressor O
response O
and O
/ O
or O
increase O
in O
pVP O
, O
represents O
a O
rapid O
approach O
for O
screening O
the O
neurosecretory O
function O
of O
the O
central O
neural O
integration O
to O
release O
vasopressin B
. O

To O
study O
adaptive O
processes O
following O
spinal O
cord O
injury O
, O
unstructured O
audiotaped O
interviews O
were O
conducted O
on O
an O
almost O
daily O
basis O
with O
a O
30 O
- O
year O
- O
old O
divorced O
male O
during O
the O
first O
3 O
months O
of O
his O
initial O
comprehensive O
inpatient O
rehabilitation O
. O

Melioidosis O
in O
Sika O
deer O
( O
Cervus O
nippon O
nippon O
) O
. O

Enterobius O
vermicularis O
eggs O
were O
demonstrated O
during O
microscopic O
examination O
of O
a O
smear O
taken O
from O
the O
posterior O
fornix O
of O
the O
vagina O
. O

Together O
with O
previous O
work O
our O
results O
suggest O
that O
C B
/ I
EBP I
may O
be O
a O
general O
cooperation O
partner O
for O
v B
- I
Myb I
in O
myelomonocytic O
cells O
. O

When O
medical O
aid O
was O
sought O
a O
false O
history O
was O
given O
and O
the O
true O
nature O
of O
the O
child O
' O
s O
illness O
, O
heat O
stroke O
, O
was O
not O
determined O
until O
after O
death O
. O

Both O
DMP2 B
and O
DMP3 B
are O
closely O
localized O
on O
mouse O
chromosome O
5q21 O
, O
corresponding O
to O
human O
chromosome O
4q21 O
. O

This O
article O
expands O
the O
analysis O
of O
a O
numeric O
example O
included O
in O
the O
SAS O
GLM O
procedure O
to O
cover O
several O
crucial O
statistical O
aspects O
relevant O
to O
ANCOVA O
, O
and O
to O
highlight O
the O
meaning O
of O
the O
statistical O
results O
obtained O
. O

A O
novel O
alternative O
spliced O
variant O
of O
the O
transcription B
factor I
AP2alpha I
is O
expressed O
in O
the O
murine O
ocular O
lens O
. O

Fig1p B
and O
Fig2p B
are O
likely O
to O
act O
at O
the O
cell O
surface O
as O
Fig1 B
: O
: O
beta B
- I
gal I
and O
Fig2 B
: O
: O
beta B
- I
gal I
fusion I
proteins I
localize O
to O
the O
periphery O
of O
mating O
cells O
. O

Crystals O
of O
the O
triple O
mutant O
A42D O
/ O
D47P O
/ O
A63L O
, O
which O
are O
stable O
for O
days O
in O
its O
oxidized O
form O
, O
were O
grown O
from O
ammonium O
sulfate O
, O
with O
the O
cell O
constants O
a O
= O
b O
= O
34 O
. O
3 O
A O
and O
c O
= O
111 O
. O
8 O
A O
belonging O
to O
space O
group O
P3 O
( O
2 O
) O
21 O
. O

Nevertheless O
, O
PEG O
influenced O
in O
vitro O
drug O
availability O
considerably O
, O
by O
increasing O
both O
drug O
solubility O
and O
dissolution O
rate O
. O

Normal O
replication O
of O
DNA O
A O
still O
carrying O
the O
AC3 B
ORF I
mutation I
was O
found O
in O
extracts O
from O
these O
plants O
. O

Such O
saturation O
often O
occurs O
for O
several O
recording O
sweeps O
after O
large O
amplitude O
signals O
such O
as O
eye O
blinks O
are O
rejected O
. O

Muscle O
and O
liver O
biopsies O
demonstrated O
the O
same O
anomalies O
, O
again O
without O
branching O
enzyme O
deficiency O
in O
the O
liver O
. O

Possibility O
of O
using O
gynecologic O
nurses O
in O
the O
independently O
administered O
health O
centers O

Airborne O
rabies O
encephalitis O
: O
demonstration O
of O
rabies O
virus O
in O
the O
human O
central O
nervous O
system O
. O

T1 O
and O
T2 O
values O
were O
calculated O
from O
guinea O
pig O
brain O
in O
vivo O
at O
0 O
. O
5 O
T O
. O

Autophosphorylation O
on O
the O
major O
phosphorylation O
site O
Y1235 O
upregulates O
the O
kinase O
activity O
of O
the O
receptor O
, O
increasing O
the O
Vmax O
of O
the O
phosphotransfer O
reaction O
. O

Clinical O
development O
of O
interleukin B
- I
10 I
. O

By O
cloning O
genes O
encoding O
benzene O
- O
degradative O
enzymes O
, O
we O
found O
that O
strain O
JS150 O
also O
carries O
genes O
for O
a O
toluene B
/ I
benzene I
- I
2 I
- I
monooxygenase I
. O

Our O
data O
suggest O
that O
association O
of O
TBP B
with O
the O
TATA O
box O
may O
be O
regulated O
, O
directly O
or O
indirectly O
, O
by O
a O
substrate O
of O
Snf1 B
. O

Fusion O
proteins O
were O
isolated O
by O
affinity O
chromatography O
on O
APTG B
- O
Sepharose O
. O

It O
differed O
from O
cued O
recall O
only O
in O
the O
instructions O
, O
which O
directed O
subjects O
away O
from O
the O
memory O
aspects O
of O
the O
test O
and O
asked O
them O
to O
complete O
each O
three O
- O
letter O
cue O
with O
the O
first O
word O
that O
came O
to O
mind O
. O

Real O
- O
time O
two O
- O
dimensional O
echocardiographic O
studies O
of O
the O
mitral O
valve O
in O
short O
- O
axis O
view O
were O
obtained O
from O
10 O
normal O
subjects O
. O

RESULTS O
: O
Sixty O
- O
one O
percent O
of O
the O
patients O
had O
damage O
within O
7 O
years O
of O
onset O
( O
mean O
3 O
. O
8 O
yrs O
) O
. O

It O
was O
previously O
shown O
that O
Vi B
antigen I
expression O
was O
regulated O
by O
a O
system O
similar O
to O
the O
rcs B
regulatory I
system I
involved O
in O
colanic O
acid O
synthesis O
in O
Escherichia O
coli O
. O

The O
map O
is O
based O
on O
the O
CEPH O
reference O
pedigrees O
and O
includes O
over O
4000 O
new O
genotypes O
, O
our O
previously O
reported O
data O
plus O
29 O
allele O
systems O
from O
the O
published O
CEPH O
version O
5 O
database O
, O
and O
was O
constructed O
using O
the O
program O
package O
CRI O
- O
MAP O
. O

To O
identify O
the O
GRK6 B
homologue I
on O
chromsome O
13 O
, O
several O
sets O
of O
closely O
- O
spaced O
primers O
were O
designed O
based O
on O
the O
GRK6 B
cDNA I
sequence I
and O
then O
used O
to O
amplify O
human O
genomic O
DNA O
by O
PCR O
. O

In O
the O
absence O
of O
Mg2 O
+ O
, O
the O
extent O
of O
destabilization O
of O
tRNAPhe B
is O
greater O
but O
appears O
to O
be O
confined O
to O
internal O
regions O
of O
the O
acceptor O
and O
T B
psi I
C I
helices I
, O
as O
evidenced O
by O
the O
selectively O
enhanced O
exchange O
rates O
for O
imino O
protons O
associated O
with O
these O
base O
pairs O
. O

Studies O
in O
26 O
patients O
. O

Serum B
alpha I
- I
amylase I
, O
trypsin B
, O
trypsin B
inhibitor O
, O
lipase B
and O
total O
protease O
activity O
in O
the O
pancreatic O
tissue O
was O
studied O
as O
indicator O
of O
the O
treatment O
efficacy O
with O
5 O
- O
fluorouracil O
electro O
- O
cumulation O
. O

1 O
The O
effects O
of O
high O
doses O
of O
piretanide O
, O
a O
new O
diuretic O
agent O
chemically O
related O
to O
frusemide O
and O
bumetanide O
were O
evaluated O
in O
twelve O
patients O
with O
severe O
chronic O
renal O
insufficiency O
( O
creatinine O
clearance O
below O
25 O
ml O
/ O
min O
) O
. O

Synthesis O
of O
22 O
- O
oxavitamin O
D3 O
analogues O
. O

This O
effect O
is O
independent O
of O
the O
Gal4 B
protein I
, O
as O
it O
operates O
in O
a O
gal4 B
mutant I
background O
as O
well O
. O

Consistent O
with O
the O
large O
pocket O
of O
Rb B
binding O
to O
TAF B
( I
II I
) I
250 I
, O
the O
large O
pocket O
domains O
of O
both O
p107 B
and O
p130 B
are O
able O
to O
bind O
to O
TAF B
( I
II I
) I
250 I
in O
vivo O
. O

50 O
- O
nucleotide O
) O
intron O
to O
AA O
. O

The O
educational O
program O
was O
the O
significant O
factor O
that O
influenced O
the O
change O
of O
concern O
trajectories O
, O
and O
the O
recurrent O
/ O
non O
- O
recurrent O
factor O
influenced O
the O
change O
of O
concern O
only O
in O
pamphlet O
group O
. O

Amplification O
of O
4q21 O
- O
q22 O
and O
the O
MXR B
gene I
in O
independently O
derived O
mitoxantrone O
- O
resistant O
cell O
lines O
. O

Activation O
of O
the O
receptor O
induces O
Ret B
phosphorylation O
that O
leads O
the O
survival O
- O
promoting O
effects O
. O

In O
patients O
in O
which O
these O
criteria O
were O
not O
met O
only O
the O
reproducibility O
of O
the O
VO2 O
obtained O
in O
both O
the O
P O
- O
MET O
and O
the O
C O
- O
MET O
indicated O
that O
the O
maximal O
VO2 O
for O
these O
4 O
patients O
had O
been O
reached O
. O

This O
paper O
describes O
a O
course O
of O
therapy O
with O
an O
agoraphobic O
female O
patient O
. O

A O
previously O
developed O
kinetic O
model O
was O
used O
to O
identify O
major O
chemical O
reaction O
pathways O
involving O
PAH O
in O
the O
afterburner O
. O

Henry O
Quastler O
brought O
experience O
from O
an O
actual O
x O
- O
ray O
treatment O
, O
and O
Dr O
. O

Critical O
review O
of O
tests O
for O
the O
HBs B
antigen I
in O
the O
detection O
of O
infectious O
blood O

A O
direct O
statistically O
reliable O
correction O
of O
decrease O
of O
the O
enzymes O
with O
the O
initial O
state O
of O
the O
patients O
has O
been O
established O
in O
an O
analysis O
of O
25 O
patients O
aged O
from O
18 O
to O
60 O
years O
in O
prosthesis O
of O
+ O
the O
mitral O
valve O
( O
n O
- O
9 O
) O
and O
closed O
mitral O
commissurotomy O
( O
n O
- O
16 O
) O
. O

DSS4 B
- I
1 I
is O
a O
dominant O
suppressor O
of O
sec4 B
- I
8 I
that O
encodes O
a O
nucleotide B
exchange I
protein I
that O
aids O
Sec4p B
function O
. O

These O
findings O
indicate O
that O
KCTG B
can O
contribute O
to O
improved O
monitoring O
in O
high O
- O
risk O
pregnancies O
. O

The O
MI O
site O
is O
predictive O
of O
hemodynamic O
left O
ventricular O
dysfunction O
both O
at O
rest O
and O
during O
exercise O
: O
anterior O
MIs O
are O
more O
impaired O
than O
inferior O
MIs O
. O

The O
vaccine O
was O
highly O
immunogenic O
, O
since O
111 O
of O
113 O
patients O
( O
98 O
% O
) O
produced O
anti B
- I
HBs I
( O
10 O
mIU O
/ O
ml O
or O
more O
) O
. O

The O
interleukin B
- I
2 I
receptor I
alpha I
( I
IL B
- I
2R I
alpha I
) O
chain O
gene O
contains O
a O
sequence O
similar O
to O
the O
immunoglobulin B
( I
Ig I
) I
kappa I
( I
kappa I
) O
enhancer O
NF B
- I
kappa I
B I
binding I
site I
. O

Comparison O
of O
the O
derived O
amino O
acid O
sequence O
with O
that O
of O
the O
HCMV B
UL99 I
gene I
product I
reveals O
34 O
. O
8 O
% O
identity O
in O
an O
overlap O
of O
66 O
amino O
acids O
. O

Although O
mutant B
pex5delta I
cells O
expressing O
a O
yeast B
/ I
tobacco I
Pex5p I
chimaera I
failed O
to O
import O
a O
GFP B
- O
Eci1p B
reporter O
protein O
, O
they O
were O
able O
to O
grow O
on O
oleic O
acid O
. O

Consistent O
with O
this O
observation O
, O
introduction O
of O
Smad B
sites I
into O
a O
TGFbeta B
- O
insensitive O
LEF1 B
/ O
TCF B
target O
gene O
confers O
cooperative O
TGFbeta B
and O
Wnt B
responsiveness O
to O
the O
promoter O
. O

In O
analogy O
to O
human B
CD70 I
, O
mCD70 B
transcript I
levels O
are O
strongly O
but O
transiently O
up O
- O
regulated O
during O
lymphocyte O
activation O
, O
which O
is O
in O
line O
with O
a O
role O
for O
the O
CD27 B
- O
CD70 B
receptor O
pair O
early O
in O
the O
immune O
response O
. O

Serum B
IgM I
levels O
showed O
a O
highly O
significant O
increase O
in O
all O
types O
of O
brain O
tumour O
when O
compared O
to O
controls O
. O

Beyond O
the O
first O
year O
post O
- O
renal O
transplantation O
there O
was O
no O
difference O
in O
C O
( O
IO O
) O
between O
LRD O
and O
CAD O
allografts O
. O

Vk8 B
/ O
Jk2 B
and O
Vk1 B
/ O
Jk5 B
rearrangements O
encoded O
the O
respective O
L B
chain I
V O
- O
regions O
. O

The O
phenomena O
of O
nursing O
are O
deeply O
rooted O
in O
the O
human O
condition O
. O

Clinical O
findings O
plaque O
index O
, O
PI O
; O
gingival O
index O
, O
GI O
; O
bleeding O
on O
probing O
, O
BOP O
; O
pus O
discharge O
, O
pus O
; O
and O
probing O
depth O
, O
PD O
at O
both O
PT O
- O
01 O
and O
control O
sites O
were O
measured O
at O
every O
visit O
for O
4 O
weeks O
. O

One O
of O
these O
elements O
resembles O
the O
binding O
site O
of O
a O
previously O
identified O
cellular O
" O
transcription O
" O
factor O
. O

NER O
was O
also O
measured O
by O
a O
plasmid O
host O
cell O
re O
- O
activation O
assay O
using O
a O
vector O
containing O
a O
luciferase B
reporter I
gene I
. O

Plasma O
Pi O
, O
tibia O
breaking O
strength O
, O
and O
percentage O
of O
tibia O
ash O
were O
increased O
by O
raising O
dietary O
Pav O
in O
the O
presence O
of O
. O
392 O
% O
A1 O
with O
either O
level O
of O
Ca O
. O

A O
total O
of O
114 O
animals O
were O
infected O
by O
E O
. O
coli O
O2 O
: O
K1 O
: O
H4 O
by O
the O
hematogenic O
route O
. O

In O
the O
fungus O
Neurospora O
crassa O
, O
nit B
- I
2 I
, O
the O
major O
nitrogen O
regulatory O
gene O
, O
activates O
the O
expression O
of O
unlinked O
structural O
genes O
that O
specify O
nitrogen O
- O
catabolic O
enzymes O
during O
conditions O
of O
nitrogen O
limitation O
. O

The O
matrix O
of O
the O
CBs O
contains O
the O
diagnostic O
protein O
p80 B
- O
coilin B
, O
which O
is O
colocalized O
with O
the O
U7 B
small I
nuclear I
ribonucleoprotein I
( O
snRNP O
) O
, O
whereas O
the O
attached O
and O
embedded O
B O
- O
snurposomes O
contain O
splicing O
snRNPs O
. O

During O
an O
observation O
period O
of O
12 O
weeks O
after O
halving O
atenolol O
from O
a O
mean O
dose O
of O
82 O
mg O
to O
41 O
mg O
, O
no O
significant O
changes O
in O
systolic O
and O
diastolic O
blood O
pressure O
or O
in O
response O
rate O
( O
defined O
as O
a O
diastolic O
blood O
pressure O
of O
95 O
mm O
Hg O
or O
less O
) O
were O
observed O
. O

Comparative O
volumetric O
analysis O
is O
defined O
as O
the O
difference O
between O
mean O
corpuscular O
erythrocyte O
volume O
in O
peripheral O
blood O
( O
MCVB O
) O
diluted O
in O
urine O
supernatant O
after O
centrifugation O
and O
mean O
corpuscular O
volume O
of O
urinary O
erythrocytes O
( O
MCVU O
) O
. O

PATIENTS O
: O
33 O
HIV O
- O
infected O
, O
zidovudine O
- O
experienced O
patients O
with O
serum O
HIV O
RNA O
levels O
of O
at O
least O
20 O
, O
000 O
copies O
/ O
mL O
and O
CD4 B
counts O
ranging O
from O
50 O
to O
400 O
cells O
/ O
mm3 O
. O

Within O
the O
last O
few O
years O
, O
the O
WWW O
has O
grown O
enormously O
. O

These O
data O
, O
taken O
together O
, O
suggest O
that O
CRF B
produces O
its O
behavioral O
activating O
and O
anxiogenic O
effects O
, O
at O
least O
in O
part O
, O
by O
increasing O
the O
activity O
of O
LC O
noradrenergic O
neurons O
. O

On O
the O
basis O
of O
8 O
cases O
, O
the O
authors O
report O
different O
clinical O
pictures O
all O
caused O
by O
cardiac O
toxicity O
of O
5FU O
. O

The O
sequence O
of O
Vac1p B
contains O
two O
putative O
zinc O
- O
binding O
RING O
motifs O
, O
a O
zinc O
finger O
motif O
, O
and O
a O
coiled O
- O
coil O
motif O
. O

Although O
cardiac O
output O
decreased O
slightly O
with O
PCF O
, O
hemodynamic O
changes O
due O
to O
PCF O
were O
unlikely O
to O
account O
for O
the O
observed O
fall O
in O
PaO2 O
. O

Anti B
- I
Jo I
- I
1 I
antibody I
was O
not O
present O
. O

The O
first O
operon O
, O
orf1 B
- O
tolQRA B
, O
is O
iron O
regulated O
throughout O
growth O
, O
but O
iron O
- O
regulated O
expression O
of O
tolB B
and O
oprL B
fusions I
occurs O
only O
in O
late O
log O
phase O
. O

S O
. O
aureus O
leakage O
into O
the O
totally O
submerged O
test O
specimens O
was O
detected O
in O
1 O
of O
5 O
samples O
incubated O
for O
4 O
weeks O
, O
while O
no O
leakage O
was O
detected O
in O
specimens O
incubated O
for O
3 O
, O
5 O
, O
6 O
, O
7 O
, O
and O
8 O
weeks O
. O

We O
examined O
the O
contribution O
of O
a O
cryptic O
plasmid O
, O
pRmeGR4b O
, O
to O
the O
nodulation O
of O
Medicago O
sativa O
by O
strain O
GR4 O
of O
Rhizobium O
meliloti O
. O

It O
also O
appears O
that O
in O
patients O
with O
laboratory O
diagnosis O
of O
prerenal O
acute O
renal O
failure O
( O
i O
. O
e O
. O
, O
a O
RFT O
less O
than O
1 O
. O
0 O
) O
, O
the O
response O
to O
treatment O
is O
unpredictable O
and O
in O
fact O
may O
have O
a O
worse O
prognosis O
than O
in O
those O
with O
a O
RFI O
greater O
than O
1 O
. O
0 O
( O
5 O
/ O
7 O
deaths O
vs O
10 O
/ O
48 O
deaths O
) O
. O

Paraplegia O
associated O
with O
intraaortic O
balloon O
pump O
counterpulsation O
. O

CTP O
, O
GDP O
, O
GTP O
, O
ITP O
) O
did O
not O
affect O
the O
response O
. O

Using O
green B
fluorescent I
protein I
fusions I
we O
demonstrate O
that O
the O
SYT B
, O
SSX B
and O
the O
SYT B
- O
SSX B
proteins O
are O
nuclear O
proteins O
. O

The O
anaesthetic O
management O
of O
41 O
patients O
who O
underwent O
cardiac O
transplantation O
during O
a O
40 O
month O
period O
at O
Clinica O
Puerta O
de O
Hierro O
is O
reviewed O
. O

Acute O
respiratory O
distress O
syndrome O
. O

These O
data O
suggested O
that O
mutant B
PS1 I
may O
cause O
disease O
as O
a O
result O
of O
reduction O
in O
PS1 B
function O
. O

The O
analysis O
of O
the O
organization O
of O
the O
sequence O
of O
the O
human O
ABP B
/ O
DAO B
gene O
reveals O
that O
the O
2 O
. O
4 O
- O
kilobase O
messenger O
RNA O
is O
transcribed O
from O
two O
close O
origins O
identifying O
the O
proximal O
promoter O
. O

Most O
important O
, O
after O
all O
, O
is O
not O
the O
ionization O
technique O
but O
the O
stage O
with O
a O
47 O
. O
1 O
% O
five O
- O
year O
survival O
rate O
in O
T1N0 O
as O
compared O
to O
T2N0 O
with O
28 O
. O
6 O
% O
and O
T1N1 O
with O
19 O
. O
4 O
% O
. O

To O
investigate O
the O
step O
of O
spliceosome O
assembly O
at O
which O
Snu17p B
acts O
, O
we O
have O
used O
nondenaturing O
gel O
electrophoresis O
. O

We O
also O
show O
that O
an O
internal O
fragment O
of O
U24 B
methylation I
guide I
snoRNA I
, O
encompassing O
the O
upstream O
antisense O
element O
and O
the O
D O
' O
and O
C O
' O
box O
motifs O
, O
can O
support O
the O
site O
- O
specific O
methylation O
of O
rRNA O
. O

Mammalian B
ribonucleotide I
reductase I
shows O
S O
- O
phase O
specific O
expression O
and O
consists O
of O
two O
non O
- O
identical O
subunits O
, O
proteins O
R1 I
( O
large O
subunit O
) O
and O
R2 B
( O
small O
subunit O
) O
. O

Effects O
of O
dexamethasone O
on O
the O
development O
of O
radiation O
nephropathy O
in O
the O
rat O
. O

Recombinant B
MsERK1 I
( O
rMsERK1 B
) O
, O
when O
overexpressed O
in O
Escherichia O
coli O
, O
is O
recognized O
by O
antibodies O
raised O
against O
MAP B
kinases I
from I
rat I
, I
Xenopus I
, I
and I
sea I
star I
and O
by O
anti B
- I
phosphotyrosine I
antibodies I
. O

The O
identification O
of O
SRP54sc B
and O
SRP54sp B
provides O
the O
first O
evidence O
for O
SRP B
related I
proteins I
in O
yeast O
. O

We O
previously O
demonstrated O
that O
AKT2 B
, O
a O
member O
of O
protein B
kinase I
B I
family I
, O
is O
activated O
by O
a O
number O
of O
growth O
factors O
via O
Ras B
and O
PI B
3 I
- I
kinase I
signaling O
pathways O
. O

So O
it O
is O
sometimes O
more O
convenient O
to O
use O
the O
back O
Radon O
transform O
R O
- O
1 O
and O
then O
to O
correct O
the O
result O
, O
taking O
into O
account O
attenuation O
. O

Before O
flecainide O
, O
all O
patients O
had O
easily O
inducible O
VT O
that O
was O
morphologically O
identical O
to O
their O
spontaneously O
occurring O
arrhythmia O
. O

An O
additional O
dose O
of O
20 O
- O
25 O
Gy O
was O
delivered O
to O
the O
site O
of O
original O
involvement O
using O
an O
implant O
when O
feasible O
. O

The O
prominent O
fracture O
of O
women O
older O
than O
75 O
years O
is O
the O
hip O
fracture O
( O
type O
II O
osteoporosis O
) O
. O

The O
expected O
products O
of O
the O
cloned O
bph B
genes I
, O
except O
bphA3 B
, O
were O
observed O
in O
E O
. O
coli O
in O
an O
in O
vitro O
transcription O
- O
translation O
system O
. O

It O
is O
our O
conclusion O
that O
Sonoclot O
coagulation O
analysis O
is O
unlikely O
to O
identify O
patients O
with O
prolonged O
bleeding O
time O
in O
whom O
platelet O
count O
and O
other O
coagulation O
factors O
are O
normal O
. O

RESULTS O
: O
Analysis O
by O
Pearson O
' O
s O
correlation O
showed O
that O
the O
subjects O
who O
had O
higher O
scores O
of O
SSMS O
during O
the O
drum O
rotation O
generated O
the O
following O
: O
a O
) O
a O
higher O
rating O
of O
over O
- O
all O
sickness O
( O
r O
= O
0 O
. O
76 O
) O
; O
b O
) O
a O
higher O
ratio O
of O
spectral O
power O
of O
EGG O
at O
4 O
- O
9 O
cycles O
per O
minute O
( O
cpm O
) O
between O
drum O
rotation O
and O
baseline O
periods O
( O
r O
= O
0 O
. O
63 O
) O
; O
c O
) O
a O
higher O
net O
percent O
increase O
of O
spectral O
power O
in O
the O
EEG O
frequency O
band O
0 O
. O
5 O
- O
4 O
Hz O
between O
drum O
rotation O
and O
baseline O
periods O
on O
C3 O
( O
r O
= O
0 O
. O
29 O
) O

and O
C4 O
( O
r O
= O
0 O
. O
31 O
) O
; O
d O
) O
a O
higher O
ratio O
of O
spectral O
power O
of O
EEG O
frequency O
band O
0 O
. O
5 O
- O
4 O
Hz O
between O
drum O
rotation O
and O
baseline O
periods O
on O
C3 O
( O
r O
= O
0 O
. O
31 O
) O
; O
and O
e O
) O
a O
higher O
level O
of O
net O
increase O
in O
skin O
conductance O
from O
baseline O
to O
drum O
rotation O
( O
r O
= O
0 O
. O
30 O
) O
. O

Salts O
of O
these O
four O
metal O
ions O
may O
be O
added O
to O
the O
growth O
medium O
to O
facilitate O
selective O
isolation O
of O
Acinetobacter O
. O

The O
consensus O
mammalian O
ER O
stress O
response O
element O
( O
ERSE O
) O
conserved O
among O
grp B
promoters I
consists O
of O
a O
tripartite O
structure O
CCAAT O
( O
N9 O
) O
CCACG O
, O
with O
N O
being O
a O
strikingly O
GC O
- O
rich O
region O
of O
9 O
bp O
. O

Acute O
toxicity O
tests O
were O
conducted O
to O
measure O
the O
response O
of O
first O
instar O
Toxorhynchites O
splendens O
to O
commonly O
used O
mosquito O
adulticides O
: O
malathion O
, O
naled O
and O
resmethrin O
. O

A O
positive O
correlation O
was O
also O
established O
between O
the O
level O
of O
FP9C B
binding O
and O
the O
degree O
of O
cell O
differentiation O
in O
vitro O
. O

Effects O
of O
acute O
exposure O
to O
cold O
on O
pulmonary O
arterial O
blood O
pressure O
in O
awake O
rats O
. O

When O
labeled O
by O
the O
described O
method O
, O
99mTc O
on O
DTPA O
coupled O
antibodies O
shows O
instability O
in O
serum O
but O
superior O
stability O
than O
99mTc O
on O
antibodies O
without O
the O
attached O
DTPA O
. O

Unlike O
CSK B
, O
the O
SH3 B
domain I
of O
HYL B
was O
unique O
since O
the O
ALYDY B
motif I
was O
absent O
. O

As O
the O
mechanism O
of O
the O
intractability O
, O
cell O
function O
involved O
in O
the O
defense O
against O
infection O
was O
evaluated O
. O

88 O
, O
3739 O
- O
3743 O
) O
) O
. O

OBJECTIVE O
: O
To O
report O
a O
case O
of O
fulminant O
neuropathy O
with O
severe O
quadriparesis O
associated O
with O
vincristine O
chemotherapy O
. O

The O
median O
survival O
time O
was O
18 O
months O
. O

Hemorrheological O
evaluation O
and O
proposed O
pharmacological O
uses O
. O

Its O
phosphorylation O
strongly O
potentiates O
its O
ability O
to O
activate O
transcription O
of O
the O
c B
- I
fos I
promoter I
through O
a O
ternary O
complex O
assembled O
on O
the O
c B
- I
fos I
serum I
response I
element I
. O

Fus3p B
and O
Kss1p B
together O
increase O
the O
expression O
of O
CLN3 B
and O
PCL2 B
genes I
that O
promote O
budding O
, O
and O
Kss1p B
inhibits O
the O
MAP B
kinase I
cascade O
. O

Consistent O
with O
our O
observation O
that O
AP B
- I
1 I
binding O
does O
not O
contribute O
to O
c B
- I
Jun I
coactivation O
is O
the O
observation O
that O
the O
activation O
of O
PU B
. I
1 I
by O
c B
- I
Jun I
is O
blocked O
by O
overexpression O
of O
c B
- I
Fos I
. O

A O
possible O
correlation O
between O
mandibular O
growth O
retardation O
and O
palatal O
clefting O
is O
discussed O
. O

Time O
dependence O
of O
frequency O
potentiation O
in O
the O
isolated O
guinea O
- O
pig O
' O
s O
atrium O
. O

We O
found O
4 O
highly O
conserved O
regions O
to O
vertebrate B
Hsp90 I
exclusively O
and O
27 O
amino O
acids O
conserved O
among O
but O
differing O
between O
Hsp90 B
alpha I
and O
Hsp90 B
beta I
sequences I
. O

Characterization O
of O
trinucleotide O
- O
and O
tandem O
repeat O
- O
containing O
transcripts O
obtained O
from O
human O
spinal O
cord O
cDNA O
library O
by O
high O
- O
density O
filter O
hybridization O
. O

We O
present O
here O
the O
complete O
primary O
structure O
of O
human B
gp330 I
, O
the O
human O
variant O
of O
the O
principal O
kidney O
autoantigen O
causing O
Heymann O
membranous O
glomerulonephritis O
in O
rats O
. O

The O
platelet B
membrane I
glycoprotein I
IIb I
/ I
IIIa I
receptor I
inhibitor O
abciximab O
is O
used O
for O
the O
treatment O
of O
patients O
undergoing O
high O
- O
risk O
percutaneous O
coronary O
interventions O
and O
is O
used O
in O
approximately O
one O
third O
of O
coronary O
interventions O
in O
the O
United O
States O
and O
a O
growing O
number O
of O
procedures O
in O
Europe O
. O

In O
the O
conscious O
animals O
the O
increase O
reached O
statistical O
significance O
when O
the O
animals O
were O
exposed O
to O
12 O
% O
oxygen O
in O
nitrogen O
, O
which O
produced O
a O
fall O
in O
arterial O
PaO2 O
of O
44 O
. O
7 O
+ O
/ O
- O
5 O
. O
0 O
% O
. O

Prenatal O
diagnosis O
of O
genetic O
diseases O
. O

Human B
epidermal I
growth I
factor I
( O
EGF B
) O
, O
a O
naturally O
occurring O
protein O
, O
has O
been O
implicated O
in O
the O
protection O
of O
gastrointestinal O
mucosal O
integrity O
. O

A O
ready O
- O
made O
prosthesis O
is O
placed O
on O
the O
external O
side O
of O
the O
ribs O
vertically O
bridging O
the O
flailed O
chest O
segment O
, O
and O
fixed O
to O
the O
first O
upper O
and O
first O
lower O
intact O
rib O
as O
well O
as O
to O
the O
mobile O
segments O
of O
the O
affected O
ribs O
. O

DESIGN O
: O
Prospective O
case O
study O
( O
Canadian O
Task O
Force O
classification O
II O
- O
2 O
) O
. O

Site O
- O
directed O
mutagenesis O
of O
the O
T4 B
endonuclease I
V I
gene I
: O
role O
of O
lysine O
- O
130 O
. O

A O
minimal O
pheromone O
induction O
domain O
, O
delineated O
as O
residues O
301 O
to O
335 O
of O
Ste12p B
, O
is O
dependent O
on O
the O
pheromone O
mitogen B
- I
activated I
protein I
( I
MAP I
) I
kinase I
pathway O
for O
induction O
activity O
. O

The O
library O
will O
thus O
be O
useful O
for O
the O
selection O
of O
cosmid O
clones O
which O
carry O
CDC B
genes I
from O
yeast O
by O
complementing O
first O
, O
with O
the O
vectorial O
yeast O
gene I
URA1 I
, O
the O
pyrimidine O
auxotrophy O
of O
most O
cdc B
- I
strains O
and O
then O
, O
with O
the O
respective O
CDC B
wild I
- I
type I
genes I
, O
of O
the O
temperature O
- O
sensitive O
mutant O
alleles O
. O

These O
mutant B
rps7 I
leaders I
were O
ligated O
into O
an O
aadA B
expression I
cassette I
and O
transformed O
into O
the O
chloroplast O
of O
C O
. O
reinhardtii O
and O
into O
E O
. O
coli O
. O

In O
addition O
, O
both O
activated O
Ral B
- I
GDS I
- I
like I
factor I
and O
Raf B
stimulate O
cyclin B
D I
( I
1 I
) I
transcription O
and O
E2F B
activity O
and O
act O
in O
synergy O
with O
PI B
3 I
- I
kinase I
. O

From O
the O
present O
results O
a O
concept O
of O
hormone O
- O
dependent O
AR B
activation O
is O
proposed O
, O
which O
requires O
a O
functional O
, O
direct O
or O
indirect O
intramolecular O
interaction O
between O
the O
TAD O
and O
the O
LBD O
. O

Like O
the O
DMA O
, O
but O
unlike O
all O
other O
mammalian B
class I
II I
A I
genes I
, O
the O
zebrafish O
gene O
codes O
for O
two O
cysteine O
residues O
which O
might O
potentially O
be O
involved O
in O
the O
formation O
of O
a O
disulfide O
bond O
in O
the O
alpha O
1 O
domain O
. O

Down O
- O
regulation O
of O
Id1 B
mRNA I
correlated O
with O
mitogenesis O
and O
occurred O
when O
quiescent O
cells O
were O
treated O
with O
growth O
factors O
that O
activate O
G B
protein I
- I
coupled I
receptors I
and O
receptor B
protein I
tyrosine I
kinases I
but O
not O
with O
a O
non O
- O
mitogenic O
cAMP O
analog O
. O

Secretogranin B
II I
( O
SgII B
) O
is O
a O
secretory O
polypeptide O
stored O
in O
large O
dense O
core O
vesicles O
of O
neuroendocrine O
and O
neuronal O
cells O
. O

CONCLUSIONS O
: O
In O
the O
results O
of O
this O
study O
, O
SDB O
, O
even O
snoring O
, O
was O
independently O
associated O
with O
hypertension O
in O
both O
men O
and O
women O
. O

The O
electron O
spin O
resonance O
study O
also O
demonstrated O
that O
the O
formation O
of O
superoxide O
- O
DMPO O
spin O
adduct O
was O
strongly O
inhibited O
by O
a O
selective O
cyclooxygenase B
- I
2 I
inhibitor O
, O
etodolac O
, O
in O
a O
concentration O
- O
dependent O
manner O
. O

BK O
- O
induced O
translocation O
and O
overexpression O
of O
PKC B
isoforms I
as O
well O
as O
coexpression O
of O
inactive O
or O
constitutively O
active O
mutants O
of O
different O
PKC B
isozymes I
provided O
evidence O
for O
a O
role O
of O
the O
diacylglycerol O
- O
sensitive O
PKCs B
alpha I
and I
epsilon I
in O
BK O
signaling O
toward O
MAPK B
. O

Embryonal O
mortality O

A O
lead O
phthalocyanin O
method O
for O
the O
demonstration O
of O
acid B
hydrolases I
in O
plant O
and O
animal O
tissues O
. O

Here O
we O
describe O
the O
characterization O
of O
cDNAs O
encoding O
two O
unusual O
E2Fs B
, O
E2F B
- I
4 I
and O
E2F B
- I
5 I
, O
each O
identified O
by O
the O
ability O
of O
their O
gene O
product O
to O
interact O
with O
p130 B
in O
a O
yeast O
two O
- O
hybrid O
system O
. O

Interestingly O
, O
unlike O
PAK65 B
, O
HPK1 B
does O
not O
contain O
the O
small O
GTPase I
Rac1 B
/ O
Cdc42 B
- O
binding O
domain O
and O
does O
not O
bind O
to O
either O
Rac1 B
or O
Cdc42 B
, O
suggesting O
that O
HPK1 B
. I
activation O
is O
Rac1 B
/ O
Cdc42 B
- O
independent O
. O

The O
results O
were O
compared O
with O
the O
findings O
in O
pair O
- O
fed O
non O
- O
treated O
animals O
( O
Control O
Group O
) O
. O

Tumor B
necrosis I
factor I
levels O
were O
measured O
by O
enzyme O
- O
linked O
immunoabsorbent O
assay O
in O
plasma O
samples O
obtained O
from O
the O
hyperthermic O
isolated O
limb O
perfusion O
circuit O
and O
systemic O
circulation O
. O

However O
, O
only O
plasma O
fibrinogen B
concentrations O
showed O
statistically O
significant O
positive O
associations O
with O
IMT O
in O
both O
groups O
. O

Without O
a O
team O
, O
there O
is O
little O
hope O
for O
fetal O
survival O
; O
mortality O
will O
be O
80 O
- O
100 O
% O
. O

Analysis O
of O
deletion O
and O
substitution O
mutations O
in O
the O
MER2 B
5 O
' O
exon O
demonstrates O
that O
the O
unusually O
large O
size O
of O
this O
exon O
plays O
an O
important O
role O
in O
splicing O
regulation O
. O

To O
study O
the O
signaling O
pathways O
that O
regulate O
cytoskeletal O
rearrangements O
in O
T O
lymphocytes O
, O
we O
set O
up O
a O
conjugate O
formation O
assay O
using O
Jurkat O
T O
cells O
as O
effectors O
and O
cell O
- O
sized O
latex O
beads O
coated O
with O
various O
antibodies O
as O
artificial O
APCs O
. O

Reexamination O
of O
the O
cdc4 B
- I
1 I
mutant I
revealed O
that O
, O
in O
addition O
to O
being O
defective O
in O
the O
onset O
of O
S O
phase O
, O
it O
is O
also O
defective O
in O
G O
( O
2 O
) O
/ O
M O
transition O
when O
released O
from O
hydroxyurea O
- O
induced O
S O
- O
phase O
arrest O
. O

All O
such O
polypeptides O
containing O
263 O
or O
more O
residues O
derived O
from O
the O
C O
- O
terminus O
of O
P130gag B
- O
fps B
( O
i O
. O
e O
. O
residues O
920 O
- O
1182 O
) O
were O
enzymatically O
active O
as O
tyrosine B
kinases I
. O

A O
structural O
model O
for O
Vpu B
is O
proposed O
in O
which O
the O
membrane O
anchor O
precedes O
a O
region O
comprising O
two O
amphipathic O
alpha O
- O
helices O
of O
opposed O
polarity O
, O
joined O
by O
a O
strongly O
acidic O
turn O
that O
protrudes O
into O
the O
cytoplasm O
and O
contains O
the O
CK B
- I
2 I
phosphorylation I
sites O
. O

The O
recombinant O
containing O
the O
full B
- I
length I
polyhedrin I
leader I
sequence I
gave O
levels O
of O
N B
mRNA I
comparable O
to O
those O
of O
AcNPV B
polyhedrin I
mRNA I
. O

Immune O
and O
clinical O
impact O
of O
Lactobacillus O
acidophilus O
on O
asthma O
. O

Thus O
, O
CARP B
is O
a O
YB B
- I
1 I
associated I
factor I
and O
represents O
the O
first O
identified O
cardiac O
- O
restricted O
downstream O
regulatory O
gene O
in O
the O
homeobox B
gene I
Nkx2 B
- I
5 I
pathway O
and O
may O
serve O
as O
a O
negative O
regulator O
of O
HF B
- I
1 I
- O
dependent O
pathways O
for O
ventricular O
muscle O
gene O
expression O
. O

Each O
receptor O
must O
therefore O
engage O
a O
unique O
subset O
of O
the O
available O
signaling O
elements O
- O
- O
at O
least O
partly O
through O
the O
selection O
of O
proteins O
with O
src B
- I
homology I
2 I
domains I
( O
SH2 B
proteins I
) O
. O

A O
proposed O
method O
. O

A O
model O
for O
the O
SMase B
- O
sphingomyelin I
complex O
structure O
was O
built O
to O
investigate O
how O
the O
SMase B
specifically O
recognizes O
its O
substrate O
. O

Two O
estimates O
of O
the O
fractal O
exponent O
are O
considered O
which O
do O
not O
suffer O
from O
this O
limit O
: O
one O
derived O
from O
the O
Allan O
variance O
, O
which O
was O
developed O
by O
the O
authors O
, O
and O
one O
based O
on O
the O
periodogram O
. O

Births O
, O
marriages O
, O
divorces O
, O
and O
deaths O
for O
July O
1997 O
. O

In O
conclusion O
, O
in O
coronary O
artery O
disease O
patients O
, O
exercise O
- O
redistribution O
201Thallium O
cardiac O
imaging O
with O
reinjection O
at O
rest O
can O
identify O
severely O
ischemic O
but O
still O
viable O
myocardium O
and O
may O
be O
particularly O
useful O
in O
the O
prognosis O
of O
such O
patients O
. O

However O
, O
the O
recovery O
curve O
of O
the O
R2 O
component O
of O
the O
blink O
reflex O
diminished O
in O
patients O
with O
tension O
- O
type O
headache O
compared O
with O
the O
other O
groups O
. O

When O
recombinant B
gE I
produced O
in O
HeLa O
cells O
was O
probed O
with O
the O
same O
antiphosphotyrosine B
antibody I
, O
a O
dimeric O
gE B
form O
at O
130 O
kDa O
was O
detected O
on O
the O
cell O
surface O
. O

Transforming B
growth I
factor I
- I
beta I
( O
TGF B
- I
beta I
) O
inhibits O
cell O
cycle O
progression O
, O
in O
part O
through O
up O
- O
regulation O
of O
gene O
expression O
of O
the O
p21 B
( O
WAF1 B
/ O
Cip1 B
) O
( O
p21 B
) O
cell O
cycle O
inhibitor O
. O

The O
results O
suggest O
that O
both O
superficial O
and O
deep O
cortical O
venous O
drainage O
of O
the O
cat O
kidney O
should O
be O
considered O
when O
measuring O
renal B
renin I
release O
. O

CONCLUSION O
: O
Detection O
of O
recurrent O
head O
and O
neck O
cancer O
is O
feasible O
with O
FDG O
PET O
. O

There O
were O
no O
differences O
in O
the O
incidence O
of O
fractured O
zonas O
. O

DSIP O
was O
found O
to O
significantly O
increase O
slow O
- O
waves O
( O
delta O
sleep O
) O
in O
the O
sleep O
EEG O
. O

Accuracy O
and O
predictive O
value O
of O
ultrasound O
in O
acute O
rejection O
. O

7 O
- O
Nitroindazole O
( O
7NI O
) O
, O
a O
relatively O
selective O
neuronal B
nitric I
oxide I
synthase I
( O
nNOS B
) O
inhibitor O
. O
was O
intraperitoneally O
administered O
to O
the O
guinea O
pigs O
30 O
minutes O
before O
the O
onset O
of O
local O
anoxia O
. O

Mutations O
that O
extend O
the O
specificity O
of O
the O
endonuclease O
activity O
of O
lambda B
terminase I
. O

The O
gene O
set O
in O
this O
mitochondrial O
genome O
is O
a O
subset O
of O
that O
of O
Reclinomonas B
americana O
, O
an O
amoeboid O
protozoan O
. O

An O
evaluation O
of O
a O
positive O
control O
for O
platelet O
neutralization O
procedure O
testing O
with O
seven O
commercial O
activated O
partial O
thromboplastin B
time O
reagents O
. O

The O
prosomal B
RNA I
- I
binding I
protein I
p27K I
is O
a O
member O
of O
the O
alpha B
- I
type I
human I
prosomal I
gene I
family I
. O

A O
single O
clone O
, O
which O
was O
conserved O
and O
had O
near O
- O
perfect O
homology O
to O
eight O
human O
/ O
rodent O
expressed O
sequence O
tags O
, O
was O
used O
as O
template O
for O
5 O
' O
and O
3 O
' O
rapid O
amplification O
of O
cDNA O
ends O
and O
SPICE O
( O
system O
for O
polymerase O
chain O
reaction O
amplification O
of O
cDNA O
ends O
) O
reactions O
to O
obtain O
the O
3 O
. O
6 O
- O
kb O
cDNA O
, O
LGL2 B
( O
Genbank O
, O
AF I
110195 I
) O
encoding O
a O
deduced O
polypeptide O
( O
lgl2 B
) O
of O
963 O
amino O
acids O
. O

These O
tiny O
premature O
infants O
also O
received O
PN O
for O
significantly O
longer O
periods O
of O
time O
, O
and O
the O
longer O
the O
infusions O
were O
administered O
the O
greater O
was O
the O
risk O
of O
cholestasis O
developing O
. O

The O
SM O
- O
specific O
selection O
of O
exon O
2 O
results O
from O
the O
inhibition O
of O
exon O
3 O
. O

It O
is O
always O
present O
and O
has O
a O
discrete O
origin O
and O
insertion O
. O

Medical O
management O
of O
early O
- O
stage O
breast O
cancer O
. O

Five O
girls O
with O
Turner O
' O
s O
syndrome O
aged O
12 O
to O
17 O
years O
showed O
LH B
values O
( O
5 O
. O
0 O
- O
14 O
. O
5 O
IU O
/ O
12 O
h O
) O
at O
the O
upper O
end O
of O
or O
slightly O
above O
the O
normal O
range O
and O
pathologically O
high O
values O
( O
22 O
. O
2 O
- O
43 O
. O
5 O
IU O
/ O
12 O
h O
) O
for O
FSH B
. O

In O
vitro O
death O
assays O
with O
transient O
overexpression O
of O
deletion O
constructs O
of O
both O
isoforms O
using O
beta B
- I
galactosidase I
as O
a O
reporter O
gene O
in O
MCF7 O
cells O
suggest O
the O
following O
: O
1 O
) O
the O
nucleotide O
binding O
domain O
may O
act O
as O
a O
negative O
regulator O
of O
the O
killing O
activity O
of O
DEFCAP B
; O
2 O
) O
the O
LRR B
/ O
CARD B
represents O
a O
putative O
constitutively O
active O
inducer O
of O
apoptosis O
; O
3 O
) O
the O
killing O
activity O
of O
LRR B
/ O
CARD B
is O
inhibitable O
by O
benzyloxycarbonyl O
- O
Val O
- O
Ala O
- O

Asp O
( O
OMe O
) O
- O
fluoromethyl O
ketone O
and O
to O
a O
lesser O
extent O
by O
Asp O
- O
Glu O
- O
Val O
- O
Asp O
( O
OMe O
) O
- O
fluoromethyl O
ketone O
; O
and O
4 O
) O
the O
CARD O
is O
critical O
for O
killing O
activity O
of O
DEFCAP O
. O

In O
6 O
patients O
( O
9 O
kidneys O
) O
irrigation O
with O
Solution O
G O
in O
the O
renal O
pelvis O
was O
performed O
for O
the O
dissolution O
of O
their O
infectious O
stones O
. O

The O
use O
of O
sigma B
54 I
promoters I
, O
known O
to O
require O
cognate O
binding O
proteins O
, O
could O
allow O
the O
fine O
- O
tuning O
that O
provides O
the O
temporal O
ordering O
of O
flagellar B
gene I
transcription O
. O

The O
association O
of O
I B
- I
92 I
with O
p92 B
, O
p84 B
, O
p75 B
, O
p73 B
, O
p69 B
, O
and O
p57 B
was O
completely O
reversible O
after O
treatment O
with O
the O
detergent O
deoxycholate O
( O
DOC O
) O
. O

To O
our O
knowledge O
no O
transcription O
factors O
have O
previously O
been O
identified O
that O
exhibit O
androgen O
- O
dependent O
expression O
in O
the O
epididymis O
. O

Focal O
or O
regional O
8 O
- O
to O
20 O
- O
Hz O
low O
- O
voltage O
epileptiform O
paroxysms O
in O
either O
hippocampus O
( O
10 O
patients O
) O
, O
amygdala O
( O
1 O
patient O
) O
, O
or O
both O
( O
1 O
patient O
) O
preceded O
initial O
motionless O
staring O
. O

The O
lack O
of O
heterozygous O
positions O
essentially O
facilitates O
on O
the O
one O
hand O
the O
data O
analysis O
and O
on O
the O
other O
hand O
the O
detection O
of O
new O
alleles O
. O

Epithelial O
cell O
- O
specific O
activation O
is O
achieved O
by O
the O
cooperative O
interaction O
of O
apparently O
ubiquitous O
transcriptional O
factors O
. O

Human O
parvovirus O
B19 O
gene O
expression O
from O
the O
viral B
p6 I
promoter I
( O
B19p6 B
) O
is O
restricted O
to O
primary O
human O
hematopoietic O
cells O
undergoing O
erythroid O
differentiation O
. O

The O
effectiveness O
and O
pathogenetic O
expediency O
of O
correcting O
disorders O
of O
lipid O
metabolism O
in O
atherosclerosis O
with O
enterosorbents O
is O
substantiated O
. O

To O
prevent O
section O
wrinkles O
usually O
encountered O
with O
the O
use O
of O
coated O
single O
- O
hole O
grids O
, O
a O
simple O
method O
was O
developed O
. O

The O
Cryotherapy O
for O
Retinopathy O
of O
Prematurity O
Study O
has O
shown O
that O
this O
number O
can O
be O
halved O
with O
treatment O
. O

Ultrasound O
- O
guided O
fine O
- O
needle O
aspiration O
biopsy O
( O
US O
- O
FNAB O
) O
revealed O
normal O
- O
looking O
squamous O
cells O
. O

Bleeding O
was O
successfully O
prevented O
in O
28 O
patients O
with O
the O
use O
of O
a O
combined O
treatment O
incorporating O
IV O
desmopressin O
, O
an O
antifibrinolytic O
agent O
( O
tranexamic O
acid O
) O
, O
and O
local O
methods O
( O
surgical O
glue O
and O
compression O
techniques O
) O
. O

Hetero O
- O
oligomerization O
of O
Smad4 B
with O
the O
pathway O
- O
restricted O
SMAD B
proteins I
is O
essential O
for O
Smad4 B
- O
mediated O
transcription O
. O

This O
fragment O
contains O
two O
complete O
endo I
- I
beta I
- I
1 I
, I
4 I
- I
glucanase I
- I
encoding I
genes I
, O
designated O
celCCC B
and O
celCCG B
. O

One O
of O
them O
is O
its O
limited O
sensitivity O
to O
weak O
interactions O
, O
which O
are O
common O
in O
the O
mammalian O
cerebral O
cortex O
. O

Analysis O
of O
the O
5 O
' O
and O
3 O
' O
termini O
of O
the O
transcripts O
produced O
in O
vivo O
from O
the O
puf B
operon I
of O
R B
. I
sphaeroides I
PUF I
delta I
348 I
- I
420 I
( O
three O
transcripts O
; O
0 O
. O
59 O
, O
0 O
. O
64 O
, O
and O
2 O
. O
63 O
kilobases O
) O
lacking O
the O
puf B
- I
intercistronic I
terminator I
structure O
were O
identical O
to O
those O
of O
the O
corresponding O
puf B
transcripts I
derived O
from O
wild O
- O
type O
R O
. I
sphaeroides O
2 I
. I
4 I
. I
1 I
( O
four O
transcripts O
; O
0 O
. O
50 O
, O
0 O
. O
66 O
, O
0 O
. O
71 O
, O
and O
2 O
. O
7 O
kilobases O
) O
showing O

that O
the O
transcripts O
begin O
and O
end O
at O
the O
same O
sites O
. O

The O
optimum O
cooling O
rate O
from O
RT O
to O
5 O
degrees O
C O
resulting O
in O
maximum O
survival O
of O
human O
spermatozoa O
was O
found O
to O
be O
0 O
. O
5 O
to O
1 O
degree O
per O
minute O
when O
cooled O
from O
RT O
to O
5 O
degrees O
C O
and O
subsequently O
frozen O
- O
thawed O
in O
liquid O
nitrogen O
( O
LN2 O
) O
. O

The O
cerebellar O
cortical O
vessels O
: O
they O
are O
compared O
to O
the O
cerebral O
vessels O
. O

Transesophageal O
echocardiography O
risk O
factors O
for O
stroke O
in O
nonvalvular O
atrial O
fibrillation O
. O

22 O
- O
Veneto O
Region O
) O

Preventive O
health O
counseling O
tailored O
to O
the O
needs O
of O
this O
group O
may O
be O
most O
beneficial O
. O

Strains O
lacking O
a O
functional O
RPL16A B
gene I
grow O
as O
rapidly O
as O
wild O
type O
, O
whereas O
those O
containing O
a O
null O
allele O
of O
RPL16B B
grow O
more O
slowly O
than O
wild O
type O
. O

Neonatal O
hyperthyroidism O

After O
Northern O
blot O
hybridization O
, O
two O
Cx31 B
transcripts I
of O
2 O
. O
2 O
and O
1 O
. O
8 O
kb O
were O
detected O
in O
total O
RNA O
of O
the O
human O
keratinocyte O
cell O
line O
HaCaT O
and O
two O
transcripts O
of O
2 O
. O
2 O
and O
1 O
. O
9 O
kb O
in O
total O
RNA O
of O
E6 B
/ O
E7 B
transfected O
human O
keratinocytes O
( O
HEK O
cells O
) O
. O

In O
summary O
, O
FGFR3 B
signaling O
pathway O
utilizes O
two O
GRB2 B
- I
containing I
complexes I
; O
Shc B
. O
GRB2 B
. O
Sos B
and O
80K O
- I
H I
. O
pp66 B
. O
GRB2 B
. O
Sos B
; O
these O
two O
complexes O
may O
alternatively O
link O
FGFG3 B
to O
mitogen B
- I
activated I
protein I
kinase I
. O

In O
an O
effort O
to O
identify O
factors O
involved O
in O
the O
expression O
of O
this O
important O
erythroid O
- O
specific O
regulatory O
protein O
, O
we O
have O
isolated O
the O
mouse B
EKLF I
gene I
and O
systematically O
analyzed O
the O
promoter O
region O
. O

Their O
main O
representatives O
are O
the O
American O
diagnostic O
and O
statistical O
manual O
of O
mental O
disorders O
, O
DSM O
- O
III O
, O
its O
revised O
version O
DSM O
- O
III O
- O
R O
and O
the O
new O
international O
classification O
of O
mental O
disorders O
iCD O
- O
10 O
. O

Any O
opacity O
on O
a O
chest O
radiograph O
has O
a O
wide O
differential O
diagnosis O
. O

At O
the O
same O
acoustic O
power O
, O
the O
rate O
of O
chloride O
formation O
with O
20 O
kHz O
ultrasound O
was O
greater O
when O
a O
probe O
with O
a O
larger O
tip O
area O
was O
used O
, O
but O
significantly O
less O
than O
the O
rate O
with O
900 O
kHz O
. O

In O
contrast O
to O
Torpedo B
, O
where O
only O
a O
single O
transcript O
is O
seen O
, O
the O
mouse O
expresses O
several O
mRNAs O
encoding O
different O
isoforms O
. O

Four O
families O
of O
homing B
endonucleases I
have O
been O
identified O
, O
including O
the O
LAGLIDADG O
, O
the O
His O
- O
Cys O
box O
, O
the O
GIY O
- O
YIG O
and O
the O
H B
- I
N I
- I
H I
endonucleases I
. O

Convergence O
of O
trigeminal O
input O
with O
visceral O
and O
phrenic O
inputs O
on O
primate O
C1 O
- O
C2 O
spinothalamic O
tract O
neurons O
. O

The O
GvpE B
protein I
involved O
in O
the O
regulation O
of O
gas O
vesicles O
synthesis O
in O
halophilic O
archaea O
has O
been O
identified O
as O
the O
transcriptional O
activator O
for O
the O
promoter O
located O
upstream O
of O
the O
gvpA B
gene I
encoding O
the O
major O
gas B
vesicle I
structural I
protein I
GvpA I
. O

Nosographic O
problems O

In O
early O
midgestation O
embryos O
it O
is O
expressed O
in O
telencephalic O
, O
diencephalic O
and O
mesencephalic O
regions O
but O
from O
day O
11 O
. O
75 O
of O
gestation O
its O
expression O
disappears O
from O
dorsal O
telencephalon O
and O
is O
confined O
to O
diencephalic O
and O
mesencephalic O
regions O
. O

The O
training O
programs O
had O
a O
duration O
of O
30 O
days O
, O
5 O
days O
/ O
week O
, O
and O
was O
performed O
on O
a O
motor O
- O
driven O
treadmill O
. O

Phylogenetic O
analysis O
using O
a O
neighbor O
- O
joining O
algorithm O
showed O
closest O
similarity O
of O
the O
horse B
mu I
chain I
- I
encoding I
constant I
region I
gene I
to O
human O
and O
dog O
sequences O
. O

Using O
probes O
from O
these O
regions O
, O
rearrangements O
have O
been O
identified O
in O
each O
of O
nine O
cases O
of O
t O
( O
8 O
; O
21 O
) O
AML O
examined O
. O

Cloning O
and O
nucleotide O
sequence O
analysis O
reveals O
that O
BUR6 B
encodes O
a O
homolog O
of O
DRAP1 B
( O
also O
called O
NC2alpha B
) O
, O
a O
mammalian O
repressor O
of O
basal O
transcription O
. O

BRAP2 B
also O
shares O
significant O
homology O
with O
a O
hypothetical O
protein O
from O
yeast O
Saccharomyces O
cerevisiae O
, O
especially O
in O
the O
zinc O
finger O
region O
. O

Earlier O
published O
data O
, O
indicating O
Sp1 B
binding O
to O
the O
R1 B
alpha I
/ I
beta I
regions O
, O
could O
not O
be O
confirmed O
, O
suggesting O
that O
the O
R1 B
initiator O
element O
may O
function O
independent O
of O
Sp1 B
. O

In O
addition O
, O
reduced O
metabolism O
to O
cotinine O
from O
the O
proliposomes O
and O
MP O
was O
apparently O
responsible O
for O
the O
sustained O
plasma O
nicotine O
levels O
. O

Activation O
of O
HIV O
- O
1 O
requires O
the O
binding O
of O
host O
cell O
transcription O
factors O
to O
cis O
elements O
in O
the O
proviral B
long I
terminal I
repeat I
( O
LTR O
) O
. O

The O
yeast B
mitochondrial I
Hsp70 I
, O
Ssc1p B
, O
functions O
as O
a O
molecular O
chaperone O
with O
its O
partner O
proteins O
, O
Mdj1p B
( O
DnaJ B
homologue I
) O
and O
Yge1p B
( O
GrpE B
homologue I
) O
. O

Increased O
TRE O
DNA O
binding O
failed O
to O
lead O
to O
increased O
transactivation O
correlating O
with O
the O
inability O
of O
SB O
203580 O
to O
increase O
phosphorylation O
of O
these O
AP B
- I
1 I
proteins I
. O

The O
rice O
gene O
consists O
of O
four O
exons O
. O

However O
, O
and O
in O
contrast O
to O
other O
MMP B
genes I
, O
no O
significative O
synergistic O
effect O
on O
CAT B
activity O
between O
the O
AP B
- I
1 I
and O
PEA B
- I
3 I
elements I
found O
in O
the O
collagenase B
- I
3 I
gene I
promoter I
was O
found O
. O

The O
equation O
for O
mean O
trabecular O
number O
passed O
the O
test O
, O
whereas O
the O
validity O
of O
the O
equations O
for O
mean O
trabecular O
separation O
and O
Tb O
. O
Wi O
appeared O
limited O
. O

Methodological O
considerations O
limiting O
causal O
assertions O
permissible O
with O
nonexperimental O
data O
are O
discussed O
. O

In O
contrast O
, O
postmitotic O
neurons O
possessed O
extremely O
low O
levels O
of O
E2F1 B
protein I
as O
assessed O
by O
the O
electrophoretic O
mobility O
shift O
assay O
and O
Western O
blotting O
. O

Recurrence O
of O
the O
hepatic O
cyst O
after O
partial O
excision O
and O
drainage O
was O
complicated O
by O
fistula O
formation O
between O
the O
cyst O
and O
the O
duodenum O
. O

Reversal O
of O
mefloquine O
resistance O
in O
rodent O
Plasmodium O
. O

Possible O
consequences O
of O
such O
binder O
distribution O
through O
powder O
aggregates O
are O
discussed O
. O

The O
human B
parvalbumin I
mRNA I
contains O
the O
putative O
polyadenylation O
signal O
AATAAA O
13 O
nucleotides O
upstream O
from O
the O
polyadenylation O
site O
. O

The O
delta O
Mo O
+ O
SV O
tumor O
DNAs O
from O
B O
- O
lineage O
tumors O
were O
typically O
rearranged O
at O
the O
immunoglobulin B
gene I
loci I
and O
contained O
germ O
line O
configurations O
of O
the O
T B
- I
cell I
receptor I
beta I
gene I
. O

The O
experimental O
results O
suggest O
equilibrium O
complex O
formation O
between O
OTC B
and O
PVP O
, O
it O
being O
mainly O
the O
complex O
which O
is O
i O
. O
v O
. O
injected O
when O
both O
products O
are O
combined O
in O
an O
injection O
formulation O
. O

We O
conclude O
that O
the O
MMF O
lesion O
is O
secondary O
to O
intramuscular O
injection O
of O
aluminium O
hydroxide O
- O
containing O
vaccines O
, O
shows O
both O
long O
- O
term O
persistence O
of O
aluminium O
hydroxide O
and O
an O
ongoing O
local O
immune O
reaction O
, O
and O
is O
detected O
in O
patients O
with O
systemic O
symptoms O
which O
appeared O
subsequently O
to O
vaccination O
. O

To O
examine O
the O
effect O
of O
the O
cAMP O
signal O
transduction O
pathway O
on O
transcription O
of O
the O
gene O
encoding O
the O
catalytic O
subunit O
of O
glucose B
- I
6 I
- I
phosphatase I
( O
G6Pase B
) O
, O
G6Pase B
- O
chloramphenicol B
acetyltransferase I
( O
CAT B
) O
fusion O
genes O
were O
transiently O
transfected O
into O
either O
the O
liver O
- O
derived O
HepG2 O
or O
kidney O
- O
derived O
LLC O
- O
PK O
cell O
line O
. O

Whereas O
the O
sequence O
of O
alpha B
- I
actinin I
( O
Noegel O
, O
A O
. O
, O
W O
. O

By O
contrast O
, O
clonidine O
( O
1 O
micrograms O
/ O
kg O
) O
elicited O
an O
immediate O
and O
prolonged O
fall O
in O
blood O
pressure O
and O
heart O
rate O
when O
given O
into O
the O
vertebral O
artery O
, O
but O
not O
intravenously O
. O

The O
shift O
from O
complex O
I O
to O
complex O
II O
seen O
only O
in O
SCM O
- O
1 O
- O
producer O
T O
cell O
lines O
upon O
activation O
was O
completely O
suppressed O
by O
cyclosporin O
A O
. O

( O
We O
observed O
a O
ferritin B
value O
as O
high O
as O
47 O
mg O
/ O
L O
in O
one O
patient O
. O
) O
Results O
with O
all O
these O
kits O
did O
not O
inter O
- O
compare O
well O
for O
ferritin B
concentrations O
greater O
than O
300 O
micrograms O
/ O
L O
, O
a O
finding O
that O
casts O
further O
doubt O
on O
the O
controversial O
use O
of O
serum O
ferritin B
measurement O
in O
cases O
of O
iron O
overload O
. O

Post O
- O
CPB O
cardiac O
index O
was O
superior O
in O
group O
B O
( O
3 O
. O
9 O
+ O
/ O
- O
0 O
. O
3 O
vs O
. O

ER B
beta I
was O
less O
potent O
than O
ER B
alpha I
in O
activating O
E2 O
- O
stimulated O
ERELuc B
activity O
( O
4 O
- O
vs O
. O

The O
expression O
pattern O
of O
LjEmx B
changed O
dramatically O
during O
embryogenesis O
; O
expression O
was O
seen O
initially O
in O
the O
entire O
neural O
tube O
and O
mesoderm O
, O
which O
were O
secondarily O
downregulated O
, O
and O
secondarily O
in O
cranial O
nerve O
ganglia O
and O
in O
the O
craniofacial O
mesenchyme O
. O

Disruption O
of O
PML B
subnuclear O
domains O
by O
the O
acidic O
IE1 B
protein I
of O
human O
cytomegalovirus O
is O
mediated O
through O
interaction O
with O
PML B
and O
may O
modulate O
a O
RING O
finger O
- O
dependent O
cryptic O
transactivator O
function O
of O
PML B
. O

Furthermore O
, O
the O
GC O
- O
rich O
sequences O
could O
confer O
Sp1 B
- O
dependent O
transactivation O
to O
a O
heterologous O
prolactin B
minimal I
promoter I
. O

Twelve O
patients O
of O
leprosy O
with O
arthritis O
and O
161 O
patients O
without O
arthritis O
were O
studied O
for O
immunological O
parameters O
like O
immunoglobulins B
( O
IgG B
, O
IgM B
, O
IgA B
) O
, O
C B
- I
reactive I
proteins I
and O
rheumatoid B
factor I
. O

Several O
reports O
assert O
that O
prolactin B
affects O
the O
delta O
5 O
and O
delta O
4 O
pathways O
through O
its O
effect O
on O
the O
activity O
of O
3beta B
- I
hydroxysteroid I
dehydrogenase I
( O
3beta B
- I
OHSD I
) O
. O

The O
NrfC B
polypeptide I
, O
M O
( O
r O
) O
24 O
, O
567 O
, O
contains O
16 O
cysteine O
residues O
arranged O
in O
four O
clusters O
typical O
of O
the O
CooF B
super O
- I
family I
of O
non B
- I
haem I
iron I
- I
sulphur I
proteins I
. O

Zonography O
in O
urology O
( O
author O
' O
s O
transl O
) O

( O
i O
) O
p60 B
bound O
fast O
- O
migrating O
, O
underprocessed O
wild B
- I
type I
ICP22 I
and O
ICP22 B
lacking O
the O
carboxyl O
- O
terminal O
24 O
amino O
acids O
but O
not O
ICP22 B
lacking O
the O
carboxyl O
- O
terminal O
40 O
amino O
acids O
, O
whereas O
the O
previously O
identified O
cellular B
protein I
p78 I
( O
R O
. O

Stress O
thallium O
- O
201 O
myocardial O
imaging O
was O
used O
in O
two O
angina O
- O
free O
patients O
with O
severe O
congestive O
heart O
failure O
to O
identify O
clinically O
silent O
areas O
of O
ischemic O
myocardium O
and O
to O
distinguish O
between O
scar O
and O
reversibly O
ischemic O
myocardium O
as O
a O
cause O
for O
akinesia O
of O
left O
ventricular O
wall O
segments O
. O

The O
community O
- O
acquired O
bacteremia O
was O
mainly O
due O
to O
E O
. O
coli O
. O

Y1 B
, O
Y2 B
, O
and O
Y4 B
/ O
PP1 B
. O

Low O
plasma O
glucose O
concentrations O
that O
may O
or O
may O
not O
be O
sufficiently O
low O
to O
result O
in O
symptoms O
can O
be O
observed O
as O
a O
concomitant O
of O
several O
diverse O
diseases O
. O

Developmentally O
- O
regulated O
interaction O
of O
a O
transcription O
factor O
complex O
containing O
CDP B
/ O
cut B
with O
the O
early O
histone B
H3 I
gene I
promoter I
of O
the O
sea O
urchin O
Tetrapygus O
niger O
is O
associated O
with O
changes O
in O
chromatin O
structure O
and O
gene O
expression O
. O

Procedures O
with O
perfected O
method O

After O
a O
shift O
to O
37 O
degrees O
C O
, O
the O
mutant O
Rat7 I
- I
1p I
/ O
Nup159 B
- I
1p I
is O
lost O
from O
the O
nuclear O
rim O
of O
rat7 O
- O
1 O
cells O
and O
NPCs O
, O
which O
are O
clustered O
together O
in O
these O
cells O
grown O
under O
permissive O
conditions O
become O
substantially O
less O
clustered O
. O

The O
mRNA O
for O
this O
protein O
is O
expressed O
in O
the O
T O
- O
ALL O
cell O
line O
Jurkat O
and O
has O
been O
designated O
HUG1 B
, O
for O
HOX11 B
Upstream I
Gene I
. O

The O
STS O
sequence O
WI O
- O
14920 O
is O
in O
fact O
derived O
from O
the O
3 O
' O
- O
untranslated O
region O
of O
the O
human B
PUNC I
gene I
. O

Its O
activity O
was O
twice O
that O
of O
a O
construct O
where O
the O
CAT B
gene I
was O
driven O
by O
the O
H B
- I
2Kb I
5 I
' I
enhancer I
region I
( O
H2TF1 B
/ O
KBF1 I
site O
) O
and O
comparable O
to O
that O
of O
pRSVCAT B
construct I
carrying O
the O
strong O
Rous B
sarcoma I
virus I
LTR I
enhancer I
. O

ORF3 O
appears O
to O
encode O
the O
homologue O
of O
the O
well O
- O
conserved O
proteasomal B
26S I
regulatory I
subunit I
. O

The O
tam B
A I
gene I
of I
Aspergillus I
nidulans I
encodes O
a O
739 O
- O
amino O
acid O
protein O
with O
similarity O
to O
Uga35p B
/ O
Dal81p B
/ O
DurLp B
of O
Saccharomyces O
cerevisiae O
. O

Little O
honoured O
in O
his O
own O
country O
: O
statues O
in O
recognition O
of O
Edward O
Jenner O
MD O
FRS O
. O

A O
pharmacokinetic O
interaction O
between O
LY354740 O
and O
diazepam O
, O
leading O
to O
the O
lowering O
of O
the O
plasma O
level O
of O
free O
diazepam O
, O
was O
also O
demonstrated O
. O

The O
promoter O
of O
this O
methyltransferase O
gene O
lacks O
an O
identifiable O
TATA O
box O
but O
is O
characterized O
by O
a O
CpG O
island O
which O
begins O
approximately O
723 O
nucleotides O
upstream O
of O
the O
major O
transcriptional O
start O
site O
and O
extends O
through O
exon O
1 O
and O
into O
the O
first O
intron O
. O

No O
promoter O
activity O
could O
be O
detected O
with O
various O
mouse O
Fli B
- I
1 I
promoter O
- O
CAT B
constructs O
containing O
600 O
bp O
of O
the O
5 O
' O
flanking O
region O
, O
the O
complete O
exon O
1 O
, O
the O
5 O
' O
end O
of O
intron O
1 O
and O
/ O
or O
retroviral O
LTR I
sequence O
. O

The O
D O
. O
discoideum O
proteins O
were O
entirely O
conserved O
over O
the O
four O
regions O
known O
to O
be O
important O
for O
GTP O
binding O
and O
all O
contained O
the O
C O
- O
terminal O
CAAX O
aa O
motifs O
shared O
by O
other O
Rho B
proteins I
. O

An O
81 O
- O
nt O
tandem O
duplication O
of O
the O
C O
- O
terminal O
coding O
region O
is O
located O
adjacent O
to O
the O
termination O
codon O
of O
the O
CyIIIb B
gene I
, O
a O
potential O
relic O
of O
a O
slipped O
mispairing O
and O
replication O
event O
. O

Only O
53 O
% O
of O
all O
injuries O
and O
12 O
. O
5 O
% O
of O
serious O
injuries O
involved O
children O
, O
and O
in O
contrast O
to O
the O
1950s O
and O
early O
1960s O
, O
young O
adults O
appear O
at O
greatest O
risk O
in O
the O
1980s O
. O

The O
method O
was O
tested O
on O
7 O
rats O
with O
48 O
- O
hr O
- O
old O
myocardial O
infarction O
and O
4 O
sham O
- O
operated O
controls O
. O

Here O
we O
examine O
the O
population O
of O
gammaS B
transcripts I
in O
adult O
human O
lens O
and O
the O
structure O
of O
the O
human B
CRYGS I
genes I
. O

The O
slope O
of O
QD O
versus O
QEMF O
for O
the O
four O
tubes O
was O
near O
unity O
. O

The O
extent O
of O
the O
odontoblast O
process O
in O
human O
dentin O
. O

Mouse B
macrophage I
beta I
subunit I
( O
CD11b B
) O
cDNA O
for O
the O
CR3 B
complement I
receptor I
/ O
Mac B
- I
1 I
antigen I
. O

Enoxacin O
has O
been O
shown O
to O
be O
an O
effective O
well O
tolerated O
and O
convenient O
treatment O
for O
gonorrhoea O
. O

We O
assessed O
cardiovascular O
variables O
and O
blood O
O2 O
contents O
in O
order O
to O
characterize O
O2 O
transport O
in O
ponies O
during O
treadmill O
exercise O
. O

The O
findings O
are O
discussed O
with O
reference O
to O
possible O
mechanisms O
by O
which O
parasite O
development O
might O
be O
controlled O
. O

The O
80 O
kb O
preceding O
the O
expression O
site O
has O
few O
, O
if O
any O
, O
functional O
ORFs O
, O
but O
contains O
50 O
bp O
repeats O
, O
INGI B
retrotransposon I
- I
like I
elements I
, O
and O
novel O
4 O
- O
12 O
kb O
repeats O
found O
near O
other O
telomeres O
. O

The O
predicted O
proteins O
show O
strong O
homology O
to O
an O
ABA O
- O
inducible O
glycine O
- O
rich O
protein O
from O
maize O
embryos O
and O
to O
the O
mammalian O
RNA B
- I
binding I
protein I
A1 I
of O
the O
heterogeneous O
nuclear O
ribonucleoprotein O
complex O
involved O
in O
pre O
- O
mRNA O
splicing O
. O

Increase O
in O
[ O
Ca O
+ O
+ O
] O
counteracted O
the O
effects O
of O
verapamil O
on O
RAD O
. O

We O
compared O
the O
quantities O
collected O
with O
both O
pollen O
traps O
during O
February O
, O
March O
and O
April O
1988 O
and O
1989 O
. O

Cbl B
is O
a O
signaling O
molecule O
with O
multiple O
functional O
domains O
: O
an O
SH2 B
domain I
which O
binds O
phosphotyrosine O
residues O
, O
a O
RING B
finger O
domain O
which O
acts O
as O
a O
ubiquitin B
ligase I
, O
a O
proline O
- O
rich O
region O
which O
serves O
as O
a O
docking O
site O
for O
SH3 B
- I
containing I
proteins I
, O
phosphotyrosine O
residues O
which O
serve O
as O
docking O
sites O
for O
SH2 B
- I
containing I
proteins I
such O
a O
CrkL B
and O
the O
p85 B
subunit I
of O
PI B
3 I
- I
kinase I
, O
and O
a O
nuclear O
localization O
signal O
. O

Surprisingly O
, O
apo B
- I
iso I
- I
1 I
- I
cytochrome I
c I
is O
absent O
in O
cyc3 B
- I
strains O
, O
although O
apo B
- I
iso I
- I
2 I
- I
cytochrome I
c I
is O
present O
at O
approximately O
the O
same O
level O
at O
which O
holo B
- I
iso I
- I
2 I
- I
cytochrome I
c I
is O
found O
in O
CYC3 B
+ I
strains O
. O

Here O
, O
we O
identify O
the O
analytical O
form O
of O
the O
PDF O
of O
one O
such O
measure O
, O
the O
order O
parameter O
in O
the O
low O
temperature O
phase O
of O
the O
2D O
XY O
model O
. O

The O
ventricular O
demand O
pacemaker O
( O
VVI O
) O
was O
implanted O
three O
years O
ago O
and O
because O
of O
further O
impairment O
of O
cardiac O
performance O
an O
av O
- O
sequential O
pacemaker O
( O
DDD O
) O
was O
used O
to O
restore O
atrio O
- O
ventricular O
synchronisation O
. O

It O
was O
predicted O
that O
the O
Stroop O
task O
would O
trigger O
greater O
consumption O
of O
ice O
cream O
than O
a O
fearful O
film O
, O
and O
that O
this O
effect O
would O
be O
more O
pronounced O
for O
binge O
- O
eaters O
than O
non O
- O
binge O
- O
eaters O
. O

Our O
data O
are O
in O
line O
with O
the O
hypothesis O
that O
E2F B
functions O
as O
a O
growth O
- O
and O
cell O
cycle O
regulated O
tethering O
factor O
between O
Sp1 B
and O
the O
basic O
transcription O
machinery O
. O

There O
was O
a O
positive O
correlation O
between O
change O
from O
baseline O
in O
parietal O
lobe O
gray O
- O
matter O
cytosolic O
choline O
, O
expressed O
in O
terms O
of O
choline O
/ O
creatine O
resonance O
ratios O
, O
and O
cognitive O
performance O
as O
measured O
with O
the O
Alzheimer O
' O
s O
Disease O
Assessment O
Scale O
Cognitive O
Subscale O
. O

Colonization O
increased O
with O
level O
of O
care O
: O
from O
9 O
per O
cent O
in O
independent O
residents O
of O
apartments O
to O
60 O
per O
cent O
in O
patients O
on O
an O
acute O
hospital O
ward O
( O
P O
less O
than O
0 O
. O
0001 O
) O
. O

IRA2 B
, O
a O
second O
gene O
of O
Saccharomyces O
cerevisiae O
that O
encodes O
a O
protein O
with O
a O
domain O
homologous O
to O
mammalian B
ras I
GTPase I
- I
activating I
protein I
. O

Exon O
A1a O
encodes O
most O
of O
the O
5 O
' O
- O
untranslated O
region O
. O

Co O
- O
operativity O
of O
functional O
domains O
in O
the O
muscle O
- O
specific O
transcription O
factor I
Myf I
- I
5 I
. O

Creatinine O
and O
creatine B
phosphokinase I
( O
CPK B
) O
. O

In O
our O
initial O
studies O
, O
we O
treated O
MCF O
- O
7 O
cells O
with O
paclitaxel O
, O
which O
results O
in O
the O
arrest O
of O
cells O
in O
G1 O
with O
4n O
DNA O
content O
( O
pseudo O
G1 O
) O
. O

These O
alterations O
occurred O
at O
both O
the O
mRNA O
and O
protein O
levels O
but O
did O
not O
significantly O
affect O
the O
subcellular O
distribution O
of O
any O
of O
the O
four O
isoforms O
. O

The O
glyoxysomal O
and O
plastid I
molecular I
chaperones I
( O
70 B
- I
kDa I
heat I
shock I
protein I
) O
of O
watermelon O
cotyledons O
are O
encoded O
by O
a O
single O
gene O
. O

Suspected O
pelvic O
endometriosis O
was O
prospectively O
evaluated O
in O
31 O
women O
with O
T1 O
- O
and O
T2 O
- O
weighted O
conventional O
spin O
- O
echo O
( O
CSE O
) O
magnetic O
resonance O
imaging O
alone O
and O
in O
combination O
with O
T1 O
- O
weighted O
fat O
- O
suppressed O
( O
T1FS O
) O
and O
gadolinium O
- O
enhanced O
T1FS O
( O
Gd O
- O
T1FS O
) O
spin O
- O
echo O
techniques O
. O

Seroconversion O
after O
hepatitis O
B O
vaccination O
. O

A O
case O
of O
bilateral O
testicular O
germ O
cell O
tumors O
in O
a O
23 O
- O
year O
- O
old O
male O
is O
reported O
. O

Total O
body O
BMD O
( O
r O
= O
0 O
. O
30 O
; O
P O
= O
0 O
. O
02 O
) O
and O
femoral O
neck O
BMD O
( O
r O
= O
0 O
. O
39 O
; O
P O
= O
0 O
. O
002 O
) O
were O
positively O
correlated O
with O
weight O
- O
bearing O
activity O
but O
not O
with O
non O
- O
weight O
- O
bearing O
activity O
. O

The O
effects O
of O
castor O
oil O
, O
alone O
, O
as O
well O
as O
in O
combination O
with O
PGI2 O
and O
indomethacin O
on O
gastrointestinal O
functions O
have O
been O
examined O
in O
rats O
. O

Checking O
for O
patient O
compliance O
, O
samples O
( O
n O
= O
10 O
) O
with O
a O
FK O
I O
concentration O
of O
0 O
ng O
/ O
mL O
were O
re O
- O
analyzed O
. O

K O
. O

The O
hematopoietic O
form O
of O
PTPN6 B
transcript I
is O
initiated O
at O
a O
downstream O
promoter O
separated O
by O
7 O
kb O
from O
the O
first O
. O

The O
residual O
small O
rRNAs O
in O
[ O
poky B
] O
appear O
to O
be O
synthesized O
via O
the O
upstream O
promoter O
( O
s O
) O
, O
but O
are O
missing O
37 O
- O
44 O
nucleotides O
from O
their O
5 O
' O
ends O
, O
indicating O
either O
that O
pre O
- O
rRNAs O
are O
processed O
abnormally O
or O
that O
abnormal O
5 O
' O
RNA O
ends O
are O
unstable O
. O

Extra O
dose O
due O
to O
extravehicular O
activity O
during O
the O
NASA4 O
mission O
measured O
by O
an O
on O
- O
board O
TLD O
system O
. O

Unconventional O
mRNA O
processing O
in O
the O
expression O
of O
two O
calcineurin B
B I
isoforms I
in O
Dictyostelium O
. O

The O
deduced O
amino O
acid O
sequence O
of O
this O
open O
reading O
frame O
is O
significantly O
homologous O
to O
the O
HSV B
1 I
UL49 I
. I
5 I
gene I
product I
, O
and O
as O
with O
UL49 B
. I
5 I
, O
it O
contains O
a O
potential O
signal O
sequence O
and O
transmembrane O
domain O
characteristic O
of O
membrane O
- O
associated O
proteins O
. O

It O
has O
been O
demonstrated O
that O
stimulation O
of O
the O
pontomesencephalic O
parabrachial O
region O
( O
PBR O
) O
by O
microinjection O
of O
cholinergic O
drugs O
or O
electricity O
in O
the O
cat O
produces O
potent O
pain O
suppression O
which O
is O
not O
antagonized O
by O
the O
opiate O
antagonist O
, O
naloxone O
. O

GAGA B
factor I
is O
known O
to O
remodel O
the O
chromatin O
structure O
in O
concert O
with O
nucleosome B
- I
remodeling I
factor I
NURF I
in O
a O
Drosophila O
embryonic O
S150 O
extract O
. O

Also O
, O
like O
TUB B
, O
it O
has O
a O
wider O
pattern O
of O
tissue O
expression O
than O
either O
TULP1 B
or O
TULP2 B
. O

After O
nine O
months O
GFR O
improved O
spontaneously O
to O
32 O
ml O
/ O
min O
/ O
1 O
. O
73 O
m2 O
despite O
no O
improvement O
in O
his O
hypertension O
. O

First O
, O
ste12Delta B
cells O
differ O
from O
cells O
with O
disruptions O
of O
the O
upstream O
signaling O
elements O
( O
e O
. O
g O
. O
, O
ste4Delta B
, O
ste20Delta B
, O
ste5Delta B
, O
ste11Delta B
, O
ste7Delta B
, O
or O
fus3Delta B
kss1Delta B
cells O
) O
in O
that O
they O
clearly O
retain O
some O
capacity O
for O
inducing O
Ste3p B
phosphorylation O
. O

Herein O
, O
we O
report O
that O
CRE O
- O
decoy O
oligonucleotide O
treatment O
results O
in O
an O
increase O
in O
the O
p53 B
protein I
level O
in O
MCF O
- O
7 O
human O
breast O
cancer O
cells O
that O
express O
wild B
- I
type I
p53 I
. O

The O
selective O
5 B
- I
HT2 I
receptor I
blocker O
ketanserin O
was O
found O
to O
reduce O
maximal O
urethral O
pressures O
in O
healthy O
females O
by O
about O
40 O
% O
without O
reducing O
blood O
pressure O
. O

It O
thus O
seems O
likely O
that O
genistein O
affects O
a O
common O
pathway O
downstream O
of O
these O
signals O
. O

Studies O
in O
B O
. O
glabrata O
, O
B O
. O
straminea O
and O
B O
. O
tenagophila O
under O
outdoor O
conditions O
. O

The O
electrocardiogram O
in O
tachycardia O
. O

Identification O
of O
a O
putative O
chromosomal O
replication O
origin O
from O
Helicobacter O
pylori O
and O
its O
interaction O
with O
the O
initiator B
protein I
DnaA I
. O

The O
primary O
structure O
of O
cholesterol B
esterase I
displayed O
no O
significant O
homology O
with O
other O
lipases B
, O
although O
the O
putative O
lipid O
interfacial O
recognition O
site O
of O
G O
- O
X O
- O
S O
- O
X O
- O
G O
is O
present O
in O
the O
cholesterol B
esterase I
sequence O
. O

The O
essential O
questions O
about O
hepatitis O
C O

The O
transcriptional O
initiation O
site O
of O
RAG1 B
was O
localized O
at O
A O
, O
26 O
bp O
upstream O
of O
the O
putative O
translational O
initiation O
codon O
, O
ATG O
, O
by O
the O
primer O
extension O
assay O
. O

Coronary O
arteriographies O
and O
ventriculographies O
corresponding O
to O
274 O
consecutive O
patients O
( O
January O
, O
1975 O
- O
October O
, O
1978 O
) O
with O
significant O
coronary O
lesions O
are O
reviewed O
. O

Visualization O
of O
the O
cells O
by O
phase O
contrast O
microscopy O
indicated O
that O
murine B
PKCepsilon I
expression O
in O
the O
presence O
of O
glycerol O
resulted O
in O
a O
significant O
increase O
in O
the O
number O
of O
yeast O
cells O
exhibiting O
very O
small O
buds O
. O

Sonography O
depicted O
the O
true O
morphology O
of O
these O
cystic O
lesions O
more O
clearly O
than O
CT O
, O
and O
the O
sonographic O
findings O
virtually O
excluded O
uncomplicated O
hepatic O
cyst O
as O
a O
diagnosis O
. O

Protein B
kinase I
C I
transiently O
activated O
heteromeric B
N I
- I
methyl I
- I
D I
- I
aspartate I
receptor I
channels O
independent O
of O
the O
phosphorylatable O
C O
- O
terminal O
splice O
domain O
and O
of O
consensus O
phosphorylation O
sites O
. O

A O
24 O
- O
year O
- O
old O
pregnant O
woman O
started O
to O
have O
hyperemesis O
gravidarum O
6 O
weeks O
before O
admission O
. O

Highly O
significant O
similarities O
were O
detected O
between O
the O
N O
- O
terminal O
region O
of O
P30 B
and O
those O
of O
GENA B
[ O
the O
product O
of O
another O
unidentified O
gene O
( O
geneA B
) O
located O
upstream O
of O
the O
aceEF B
- O
lpd B
operon O
] O
, O
and O
GNTR B
( O
a O
putative O
transcriptional O
repressor O
of O
the O
gluconate B
operon I
of I
Bacillus I
subtilis I
) O
. O

In O
vitro O
DNA O
binding O
assays O
indicate O
that O
the O
elements O
identified O
can O
specifically O
interact O
with O
c B
- I
Ets I
- I
1 I
protein I
. O

They O
are O
found O
to O
have O
several O
advantages O
over O
the O
conventional O
indices O
of O
skewness O
and O
kurtosis O
( O
square O
root O
of O
b1 O
and O
b2 O
) O
and O
no O
serious O
drawbacks O
. O

They O
elicit O
similar O
regional O
cerebral O
blood O
flow O
( O
rCBF O
) O
patterns O
, O
even O
though O
sign O
language O
is O
dependent O
on O
spatial O
information O
. O

Siglec B
- I
9 I
is O
predicted O
to O
contain O
three O
extracellular O
immunoglobulin B
- I
like I
domains I
that O
comprise O
an O
N O
- O
terminal O
V O
- O
set O
domain O
and O
two O
C2 O
- O
set O
domains O
, O
a O
transmembrane O
region O
and O
a O
cytoplasmic O
tail O
containing O
two O
putative O
tyrosine O
- O
based O
signaling O
motifs O
. O

Determination O
of O
the O
LD O
50 O
( O
30 O
) O
during O
infant O
period O
and O
growth O
period O

The O
availability O
of O
a O
commercial O
program O
( O
PCNONLIN O
) O
is O
needed O
to O
carry O
out O
matrix O
handling O
calculations O
. O

The O
introduction O
gives O
also O
the O
history O
of O
Research O
Programme O
MZ O
- O
XVII O
and O
its O
implementation O
in O
several O
regions O
of O
Poland O
. O

The O
fecal O
excretion O
is O
found O
to O
be O
only O
one O
third O
of O
that O
measured O
in O
urine O
. O

The O
COR B
biosynthetic I
gene I
cluster I
in I
P I
. I
syringae I
pv O
. O
glycinea O
PG4180 O
is O
encoded O
by O
a O
32 O
- O
kb O
region O
which O
contains O
both O
the O
structural O
and O
regulatory O
genes O
needed O
for O
COR B
synthesis O
. O

Model O
experiments O
on O
pheasants O
using O
single O
doses O
of O
the O
insecticide O
( O
Lindane O
) O
the O
herbicide O
( O
Terbutryn O
) O
a O
mineral O
fertilizer O
( O
calcium O
ammonium O
nitrate O
) O
and O
the O
fungicide O
( O
HCB O
) O
. O

In O
this O
technique O
, O
the O
posterior O
wall O
of O
the O
neopharynx O
consists O
only O
of O
the O
prevertebral O
tissue O
, O
while O
the O
flap O
forms O
the O
anterior O
and O
lateral O
walls O
. O

The O
most O
common O
hosts O
were O
equines O
( O
31 O
% O
) O
, O
bovines O
( O
25 O
% O
) O
and O
raccoons O
( O
19 O
% O
) O
. O

A O
single O
HD O
session O
using O
cellulose O
triacetate O
or O
polysulfone O
membrane O
significantly O
increased O
water O
content O
both O
at O
forearm O
and O
lower O
leg O
( O
p O
< O
0 O
. O
05 O
) O
. O

Both O
claR B
and O
car B
are O
expressed O
as O
monocistronic O
transcripts O
; O
the O
level O
of O
transcript O
declined O
rapidly O
after O
48h O
in O
complex O
media O
, O
but O
low O
sustained O
levels O
of O
both O
transcripts O
were O
observed O
in O
defined O
GSPG O
medium O
until O
96h O
. O

claR B
and O
car B
were O
not O
significantly O
expressed O
in O
mutants O
disrupted O
in O
the O
ccaR B
gene I
, O
a O
regulatory O
gene O
that O
controls O
positively O
clavulanic O
acid O
and O
cephamycin O
biosynthesis O
. O

Whereas O
mutant O
6C4 O
specified O
a O
wild B
- I
type I
- I
size I
Pol I
protein I
, O
we O
detected O
no O
full B
- I
length I
Pol I
protein I
in O
7E4 O
- O
infected O
cell O
extracts O
. O

The O
hepatic O
isoform O
of O
6 B
- I
phosphofructo I
- I
2 I
- I
kinase I
/ O
fructose B
- I
2 I
, I
6 I
- I
bisphosphatase I
( O
PF2K B
/ O
Fru B
- I
2 I
, I
6 I
- I
BPase I
) O
is O
transcriptionally O
stimulated O
by O
glucocorticoids O
, O
whereas O
insulin B
blocks O
this O
stimulatory O
effect O
. O

Being O
implicated O
in O
insulin B
and O
GK B
gene I
regulations O
as O
a O
common O
transcription O
factor O
, O
IPF1 B
/ O
STF B
- I
1 I
/ O
PDX B
- I
1 I
is O
likely O
to O
play O
an O
essential O
role O
in O
maintaining O
normal O
beta O
- O
cell O
functions O
. O

The O
Caenorhabditis B
elegans I
NK I
- I
2 I
class I
homeoprotein I
CEH I
- I
22 I
is O
involved O
in O
combinatorial O
activation O
of O
gene O
expression O
in O
pharyngeal O
muscle O
. O

Mouse O
embryo O
cells O
( O
C57BL O
/ O
6 O
, O
H O
- O
2b O
) O
transformed O
by O
the O
E1A B
and O
E1B B
genes I
of I
adenovirus I
type I
5 I
( O
Ad5E1 B
MEC I
) O
are O
highly O
immunogenic O
. O

The O
largest O
age O
adjusted O
differences O
between O
men O
with O
low O
and O
normal O
mood O
were O
for O
the O
AH4 O
( O
3 O
points O
, O
t O
= O
5 O
. O
6 O
, O
p O
< O
0 O
. O
0001 O
) O
and O
the O
CAMCOG O
( O
2 O
points O
, O
t O
= O
5 O
. O
8 O
, O
p O
< O
0 O
. O
0001 O
) O
. O

Our O
findings O
suggest O
that O
resting O
Tl O
- O
201 O
scintigraphy O
has O
limited O
value O
in O
the O
detection O
of O
coronary O
artery O
disease O
in O
patients O
with O
Hurler O
syndrome O
. O

We O
demonstrate O
that O
the O
WRM B
- I
1 I
protein I
binds O
to O
LIT B
- I
1 I
in O
vivo O
and O
that O
WRM B
- I
1 I
can O
activate O
the O
LIT B
- I
1 I
protein I
kinase I
when O
coexpressed O
in O
vertebrate O
tissue O
culture O
cells O
. O

RESULTS O
: O
All O
primary O
tumors O
were O
positive O
for O
CAM5 B
. I
2 I
. O

Nuclear O
extracts O
from O
Sertoli O
cells O
were O
found O
to O
cause O
an O
E O
- O
box O
gel O
shift O
when O
the O
cells O
were O
stimulated O
to O
differentiate O
in O
culture O
, O
but O
not O
under O
basal O
conditions O
. O

On O
the O
sixth O
postirradiation O
day O
the O
absorption O
of O
phenobarbitone O
, O
sulphafurazole O
and O
mecamylamine O
had O
returned O
to O
the O
control O
level O
, O
but O
the O
absorption O
of O
quinidine O
and O
isoniazid O
was O
still O
retarded O
. O

To O
determine O
the O
transactivation O
potential O
of O
each O
of O
the O
four O
Ahr B
Sp1 I
sites I
, O
we O
fused O
the O
Ahr B
promoter I
to O
a O
luciferase B
( O
LUC B
) O
reporter O
gene O
and O
transfected O
the O
construct O
into O
the O
Drosophila O
cell O
line O
Schneider O
- O
2 O
, O
which O
contains O
no O
Sp1 B
or O
Sp1 B
- I
like I
factors I
. O

We O
determined O
that O
Hop B
( I
T42 I
) I
contains O
a O
glutamic O
acid O
- O
to O
- O
lysine O
substitution O
at O
amino O
acid O
residue O
695 O
( O
E695K O
) O
. O

The O
organotins O
. O

Results O
of O
these O
studies O
indicate O
that O
binding O
of O
biotin O
to O
the O
protein O
results O
in O
protection O
of O
regions O
of O
the O
central O
domain O
in O
the O
vicinity O
of O
the O
active O
site O
and O
the O
C O
- O
terminal O
domain O
from O
chemical O
cleavage O
. O

The O
psh3 B
( I
+ I
) I
gene I
encodes O
a O
protein O
of O
215 O
amino O
acids O
, O
which O
shares O
a O
high O
degree O
of O
structural O
and O
functional O
similarity O
with O
Shr3p B
. O

The O
impairment O
of O
the O
nocturnal O
secretion O
was O
related O
to O
the O
subjects O
' O
age O
and O
, O
for O
the O
GH B
secretory O
pattern O
only O
, O
also O
to O
the O
MMSE O
score O
. O

Thus O
, O
the O
antibody O
class O
switch O
appears O
to O
be O
directed O
by O
induction O
of O
accessibility O
, O
as O
assayed O
by O
transcription O
of O
germ O
line O
CH B
genes I
. O

This O
growth O
- O
inhibitory O
effect O
was O
suppressed O
by O
the O
mpk1 B
delta I
mutation I
, O
suggesting O
that O
hyperactivation O
of O
the O
Mpk1 B
pathway O
is O
toxic O
to O
cells O
. O

The O
chicken O
genome O
contains O
two O
functional O
nonallelic O
beta1 B
, I
4 I
- I
galactosyltransferase I
genes I
. O

Recombinant B
Human I
Erythropoietin I
and O
Platinum O
- O
Based O
Chemotherapy O
In O
Advanced O
Ovarian O
Cancer O

Homodimers O
of O
RIP60 B
( O
replication B
initiation I
- I
region I
protein I
60 I
kDA I
) O
purified O
from O
nuclear O
extract O
bind O
two O
ATT O
- O
rich O
sites O
in O
oribeta O
and O
foster O
the O
formation O
of O
a O
twisted O
720 O
bp O
DNA O
loop O
in O
vitro O
. O

Endodontic O
treatment O
of O
deciduous O
teeth O
using O
the O
formocresol O
amputation O
method O

Some O
of O
these O
physiological O
responses O
are O
regulated O
via O
activation O
of O
transcription O
factors O
such O
as O
activator B
protein I
1 I
( O
AP B
- I
1 I
) O
. O

To O
test O
the O
role O
of O
myb B
family I
members I
in O
progression O
through O
the O
cell O
cycle O
, O
we O
comicroinjected O
c B
- I
myc I
and O
myb B
expression O
vectors O
into O
serum O
- O
deprived O
quiescent O
SMCs O
. O

Overall O
graft O
and O
patient O
survival O
after O
HAT O
were O
33 O
. O
3 O
% O
and O
75 O
% O
, O
respectively O
. O

LAC9 B
is O
a O
DNA O
- O
binding O
protein O
that O
regulates O
transcription O
of O
the O
lactose B
- I
galactose I
regulon I
in O
Kluyveromyces I
lactis O
. O

We O
determined O
the O
complete O
nucleotide O
sequence O
of O
the O
gypsy B
element I
present O
at O
the O
forked B
locus I
of I
Drosophila I
melanogaster I
in O
the O
f1 B
allele I
. O

The O
same O
high O
degree O
of O
sequence O
homology O
between O
the O
two O
F B
. I
diplosiphon I
PC B
alpha I
and O
PC B
beta I
sequences I
( O
85 O
and O
77 O
% O
, O
respectively O
) O
was O
found O
at O
both O
the O
nucleotide O
and O
amino O
acid O
levels O
, O
and O
similar O
results O
were O
obtained O
for O
interspecies O
comparisons O
. O

A O
wound O
model O
for O
decubitus O
and O
leg O
ulcers O
consisting O
of O
human O
dermal O
fibroblasts O
in O
type B
I I
collagen I
dermal O
" O
equivalent O
" O
matrix O
( O
DEM O
) O
was O
exposed O
in O
vitro O
to O
electric O
fields O
similar O
to O
postulated O
endogenous O
fields O
in O
wounds O
. O

A O
group O
of O
factors O
known O
as O
activating B
transcription I
factors I
( O
ATF B
) O
have O
been O
found O
to O
bind O
to O
the O
latter O
and O
related O
sequences O
found O
upstream O
of O
early O
adenovirus O
promoters O
induced O
by O
E1A B
, O
and O
these O
factors O
are O
highly O
homologous O
to O
the O
CREB B
protein I
. O

In O
addition O
to O
these O
cases O
, O
patients O
w O
with O
high O
Loa O
microfilaremia O
also O
developed O
milder O
neurologic O
manifestations O
causing O
functional O
impairment O
lasting O
for O
at O
least O
one O
week O
after O
treatment O
. O

The O
vertebrate O
transcription O
factors O
TCF I
( O
T B
cell I
factor I
) O
and O
LEF B
( O
lymphocyte B
enhancer I
binding I
factor I
) O
interact O
with O
beta B
- I
catenin I
and O
are O
hypothesized O
to O
mediate O
Wingless B
/ O
Wnt B
signaling O
. O

Some O
of O
these O
DNA O
: O
protein O
complexes O
were O
also O
present O
, O
but O
at O
lower O
levels O
, O
in O
nuclear O
extracts O
from O
untransformed O
rat O
cells O
suggesting O
the O
possible O
involvement O
of O
cellular O
factors O
in O
the O
mechanism O
of O
down O
- O
regulation O
mediated O
by O
Ad12 B
E1A I
. O

Hypocalcemia O
is O
usually O
due O
to O
either O
a O
disturbance O
in O
the O
parathyroid B
hormone I
- O
adenylate B
cyclase I
system O
or O
a O
disturbance O
in O
vitamin O
D O
metabolism O
. O

Activity O
was O
reconstituted O
, O
however O
, O
by O
combining O
fractions O
that O
were O
enriched O
in O
the O
two O
components O
. O

If O
failure O
to O
solve O
invisible O
displacements O
was O
due O
to O
increased O
memory O
requirements O
, O
then O
the O
primates O
should O
perform O
at O
chance O
level O
on O
all O
3 O
problems O
. O

In O
seven O
patients O
( O
7 O
. O
8 O
% O
) O
with O
new O
Q O
- O
waves O
and O
a O
pathologic O
CK B
- O
MB B
profile O
( O
group O
II O
) O
troponin B
T I
reached O
median O
levels O
of O
10 O
. O
47 O
micrograms O
/ O
l O
( O
quartile O
6 O
. O
34 O
- O
12 O
. O
50 O
micrograms O
/ O
l O
) O
( O
P O
< O
0 O
. O
001 O
I O
vs O
II O
) O
. O

Mineralized O
bone O
nodule O
formation O
in O
vitro O
by O
cell O
populations O
from O
young O
adult O
rabbit O
alveolar O
bone O
. O

Mouse B
GRK6 I
- I
C I
displays O
none O
of O
these O
motifs O
. O

Comparisons O
of O
the O
efficacy O
of O
benazepril O
and O
hydrochlorothiazide O
alone O
and O
in O
combination O
have O
shown O
that O
benazepril O
20 O
mg O
once O
daily O
is O
as O
effective O
as O
or O
more O
effective O
in O
lowering O
diastolic O
blood O
pressure O
than O
hydrochlorothiazide O
25 O
mg O
once O
daily O
and O
that O
the O
combination O
of O
benazepril O
20 O
mg O
and O
hydrochlorothiazide O
25 O
mg O
has O
a O
possibly O
synergistic O
effect O
on O
diastolic O
blood O
pressure O
. O

The O
132 B
- I
bp I
PHO8p I
fragment I
, O
connected O
at O
position O
- O
281 O
of O
the O
5 O
' O
upstream O
region O
of O
a O
HIS5 B
' I
- I
' I
lacZ I
fused I
gene I
, O
could O
sense O
Pi B
signals O
in O
vivo O
, O
but O
a O
20 O
- O
bp O
synthetic O
oligonucleotide O
having O
the O
same O
sequence O
from O
- O
544 O
to O
- O
525 O
of O
the O
PHO8p B
DNA I
could O
not O
. O

1988 O
) O
. O

Sequences O
within O
the O
UAS2 O
element O
of O
the O
ENO2 B
gene I
bound O
a O
second O
protein O
which O
corresponded O
to O
the O
ABFI B
( O
autonomously B
replicating I
sequence I
- I
binding I
factor I
) O
protein O
. O

Plasmid O
subclones O
of O
recombinant O
phage O
lambda O
Asm152 O
were O
able O
to O
complement O
both O
Escherichia B
coli I
gltB I
and O
A B
. I
sesbaniae I
Asm I
- I
Vi I
mutants I
; O
NADPH B
- I
glutamate I
synthase I
activity O
was O
detected O
in O
all O
such O
strains O
complemented O
to O
Asm B
+ I
. O

The O
sequence O
of O
LZ321 B
matched O
that O
of O
RREB1 B
, O
a O
transcription O
factor O
that O
bound O
to O
a O
Ras B
responsive I
element I
( O
RRE B
) O
very O
different O
from O
the O
sequence O
with O
which O
we O
isolated O
LZ321 B
. O

Steady O
- O
state O
levels O
of O
murine B
MMR I
mRNA I
were O
measured O
in O
the O
macrophage O
cell O
line O
J774E O
, O
which O
is O
known O
to O
express O
the O
protein O
at O
the O
cell O
surface O
. O

Basal O
level O
expression O
was O
reduced O
to O
20 O
and O
50 O
% O
when O
the O
UGA O
stop O
codon O
was O
replaced O
by O
UAG O
or O
UAA O
, O
respectively O
, O
consistent O
with O
the O
finding O
that O
in O
E O
. O
coli O
translation O
terminates O
more O
efficiently O
at O
UAG O
and O
UAA O
than O
at O
UGA O
. O

Discriminant O
function O
analysis O
suggests O
that O
AUDIT O
scores O
can O
successfully O
identify O
SAT O
- O
positive O
and O
SAT O
- O
negative O
patients O
; O
the O
analysis O
accounted O
for O
42 O
. O
5 O
% O
of O
the O
variance O
and O
correctly O
classified O
84 O
. O
6 O
% O
of O
the O
sample O
. O

Coronatine B
( O
COR B
) O
is O
a O
plasmid O
- O
encoded O
phytotoxin O
synthesized O
by O
several O
pathovars O
of O
phytopathogenic O
Pseudomonas O
syringae O
. O

Both O
the O
5 O
' O
and O
3 O
' O
untranslated O
regions O
also O
show O
significant O
similarity O
to O
the O
murine O
gene O
, O
with O
79 O
and O
70 O
% O
sequence O
identity O
, O
respectively O
. O

One O
patient O
exhibited O
a O
non O
- O
sense O
mutation O
( O
codon O
388 O
) O
, O
which O
changed O
a O
glutamine O
codon O
( O
CAG O
) O
to O
a O
stop O
codon O
( O
TAG O
) O
. O

The O
transcriptional O
activity O
of O
P B
- I
450 I
( I
11 I
beta I
) I
gene I
was O
studied O
with O
an O
in O
vitro O
transcription O
system O
using O
nuclear O
extracts O
prepared O
from O
bovine O
adrenal O
cortex O
. O

Renal O
dysplasia O
with O
multisystem O
malformation O
- O
- O
a O
study O
of O
9 O
cases O
. O

This O
bud O
morphology O
results O
at O
least O
in O
part O
from O
a O
cell O
cycle O
delay O
imposed O
by O
the O
Cdc28p B
- I
inhibitory I
kinase I
Swe1p I
. O

Eleven O
of O
the O
12 O
exons O
have O
complete O
sequence O
homology O
with O
the O
RBM B
- I
1 I
sequence I
. O

Copyright O
1998 O
Academic O
Press O
. O

Laparoscopic O
examination O
1 O
year O
after O
surgery O
revealed O
an O
enlarged O
, O
thin O
- O
walled O
, O
and O
fluid O
- O
filled O
uterine O
segment O
cranial O
to O
the O
midcornus O
occlusion O
sites O
in O
all O
animals O
. O

The O
three O
proteins O
copurified O
through O
several O
biochemical O
fractionation O
steps O
and O
could O
be O
coimmunoprecipitated O
by O
using O
antibodies O
against O
GCD1 B
or O
GCD2 B
. O

Cellular O
fractionation O
and O
Percoll O
gradient O
centrifugation O
combined O
with O
immunoblotting O
show O
that O
p67 B
cofractionates O
with O
nuclei O
and O
is O
enriched O
in O
resistant O
structure O
that O
is O
insoluble O
in O
2 O
M O
NaCl O
, O
25 O
mM O
lithium O
3 O
, O
5 O
' O
- O
diiodosalicylate O
, O
and O
1 O
% O
Triton O
but O
is O
soluble O
in O
8 O
M O
urea O
. O

For O
the O
first O
time O
also O
a O
genomic O
sequence O
for O
a O
red B
algal I
lhc I
gene I
is O
presented O
. O

Elimination O
of O
brush O
border O
as O
well O
as O
of O
glomerular O
marker O
proteins O
was O
significantly O
lower O
after O
intravenous O
injection O
of O
low O
- O
osmolar O
CM O
iopamidol O
370 O
( O
832 O
mOsm O
/ O
kg O
) O
than O
after O
meglumine O
diatrizoate O
76 O
( O
2100 O
mOsm O
/ O
kg O
) O
. O

Raman O
scattering O
from O
VO2 O
single O
crystals O
: O
A O
study O
of O
the O
effects O
of O
surface O
oxidation O
. O

Combination O
chemotherapy O
in O
advanced O
ovarian O
cancer O
. O

Making O
sense O
out O
of O
oxygen O
sensor O
. O

In O
comparison O
to O
a O
silent O
baseline O
, O
CBF O
increases O
were O
observed O
in O
auditory O
cortex O
bilaterally O
and O
in O
the O
right O
superior O
parietal O
, O
right O
dorsolateral O
frontal O
, O
and O
right O
premotor O
regions O
, O
with O
no O
modulation O
as O
a O
function O
of O
attentional O
condition O
. O

In O
vitro O
, O
c B
- I
Src I
phosphorylated O
FAK B
Tyr O
- O
925 O
in O
a O
glutathione B
S I
- I
transferase I
- O
FAK B
C O
- O
terminal O
domain O
fusion O
protein O
, O
whereas O
FAK B
did O
not O
. O

METHODS O
AND O
RESULTS O
: O
The O
effect O
of O
prolonged O
nitrate O
therapy O
between O
2 O
days O
and O
6 O
weeks O
during O
healing O
after O
infarction O
on O
serial O
parameters O
of O
ventricular O
remodeling O
( O
scar O
expansion O
, O
scar O
thinning O
, O
ventricular O
dilation O
, O
and O
hypertrophy O
) O
and O
function O
( O
asynergy O
or O
akinesis O
plus O
dyskinesis O
and O
ejection O
fraction O
) O
by O
serial O
two O
- O
dimensional O
echocardiography O
, O
hemodynamics O
, O
postmortem O
topography O
( O
computerized O
planimetry O
, O
geometric O
maps O
, O
and O
radiographs O
) O
, O
and O
collagen B
content O
( O
hydroxyproline O

) O
was O
studied O
in O
64 O
instrumented O
dogs O
randomized O
2 O
days O
after O
left O
anterior O
descending O
coronary O
artery O
ligation O
to O
various O
nitrate O
regimens O
( O
n O
= O
32 O
) O
over O
the O
first O
2 O
weeks O
( O
subgroup O
1 O
: O
2 O
% O
transdermal O
nitroglycerin O
at O
8 O
AM O
and O
4 O
PM O
, O
n O
= O
6 O
; O
subgroup O
2 O
: O
2 O
% O
transdermal O
nitroglycerin O
plus O
2 O
. O
6 O
mg O
of O
sustained O
- O
release O
oral O
nitroglycerin O
at O
8 O
AM O
, O
3 O
PM O
, O
and O
10 O
PM O
, O
n O
= O
5 O
; O
subgroup O
3 O
: O
oral O
isosorbide O
dinitrate O
, O
30 O
mg O
at O
8 O
AM O
and O
4 O

PM O
, O
n O
= O
11 O
) O
or O
6 O
weeks O
( O
subgroup O
4 O
: O
isosorbide O
dinitrate O
, O
n O
= O
10 O
) O
and O
in O
matching O
controls O
( O
n O
= O
32 O
) O
. O

The O
essential O
oil O
composition O
of O
three O
Zingiberaceae O
widely O
used O
as O
medicinal O
aromatic O
plants O
from O
S O
. O

Gel O
shift O
analysis O
of O
protein O
binding O
from O
nuclear O
extracts O
to O
these O
caveolin B
promoter I
DNA I
sequences I
, O
together O
with O
DNase B
I I
footprinting O
, O
confirmed O
nucleoprotein O
binding O
to O
the O
SRE O
- O
like O
elements O
as O
part O
of O
the O
transcriptional O
response O
to O
LDL B
- I
FC I
. O

A O
new O
model O
for O
objective O
assessment O
of O
cervical O
ripening O
: O
the O
effect O
of O
prostaglandin O
E2 O
and O
prelabor O
contractility O
. O

CONCLUSIONS O
: O
The O
MC O
/ O
UA O
ratio O
improves O
the O
sensitivity O
for O
the O
prediction O
of O
poor O
perinatal O
outcome O
when O
it O
is O
combined O
with O
the O
NST O
. O

Evaluation O
of O
materials O
and O
technics O
in O
vascular O
surgery O
using O
111 O
- O
Indium O

The O
claustrocortical O
connection O
was O
investigated O
in O
13 O
cats O
with O
selective O
injections O
of O
30 O
% O
HRP O
in O
the O
three O
subdivisions O
of O
the O
auditory O
cortex O
. O
( O
ABSTRACT O
TRUNCATED O
AT O
400 O
WORDS O
) O

Applying O
the O
experience O
mentioned O
above O
, O
4 O
- O
META O
was O
used O
to O
bond O
a O
proprietary O
photocuring O
microfilled O
composite O
material O
to O
Class O
V O
cavities O
in O
freshly O
extracted O
human O
teeth O
. O

The O
phP1 B
mutation I
was O
induced O
by O
insertion O
of O
a O
1 O
. O
2 O
- O
kb O
P I
element O
into O
the O
5 O
' O
transcribed O
nontranslated O
region O
of O
the O
proximal O
polyhomeotic O
gene O
. O

METHOD O
: O
Six O
instruments O
were O
reviewed O
: O
the O
Berg O
Balance O
Scale O
( O
Berg O
) O
, O
the O
Clinical O
Test O
of O
Sensory O
Interaction O
and O
Balance O
( O
CTSIB O
) O
, O
the O
Functional O
Reach O
Test O
, O
the O
Tinetti O
Balance O
Test O
of O
the O
Performance O
- O
Oriented O
Assessment O
of O
Mobility O
Problems O
( O
Tinetti O
) O
, O
the O
Timed O
" O
Up O
and O
Go O
" O
Test O
( O
TU O
& O
GT O
) O
, O
and O
the O
Physical O
Performance O
Test O
( O
PPT O
) O
. O

Genome O
timeline O
. O

Initial O
computer O
based O
similarity O
searches O
identified O
human B
retinoblastoma I
binding I
protein I
1 I
( O
RBP B
- I
1 I
) O
, O
Drosophila B
melanogaster I
male I
specific I
lethal I
- I
3 I
( O
Msl B
- I
3 I
) O
, O
S O
. O
pombe O
altered B
polarity I
- I
13 I
( O
Alp13 B
) O
and O
S B
. I
cerevisiae I
Eaf3p I
, O
a O
component O
of O
the O
yeast O
NuA4 I
HAT B
complex I
( O
Galarneau O
et O
al O
. O
, O
2000 O
. O

The O
pressure O
measurements O
under O
steady O
flow O
conditions O
showed O
that O
the O
hemodynamic O
performance O
( O
including O
pressure O
gradient O
and O
effective O
orifice O
area O
) O
of O
SPAB O
is O
superior O
to O
that O
of O
its O
stented O
counterpart O
, O
especially O
in O
the O
smaller O
sizes O
. O

Serum B
prolactin I
rapidly O
decreased O
after O
institution O
of O
treatment O
, O
with O
actual O
normalization O
( O
less O
than O
20 O
ng O
/ O
ml O
) O
by O
the O
3rd O
month O
. O

We O
report O
five O
of O
13 O
evaluable O
patients O
undergoing O
allogeneic O
sibling O
BM O
or O
PBSC O
transplantation O
for O
MM O
between O
1990 O
and O
1997 O
who O
met O
the O
criteria O
for O
adjuvant O
alpha B
- I
IFN I
therapy O
. O

Moist O
healing O
versus O
wet O
- O
to O
- O
dry O
. O

In O
this O
report O
, O
we O
identify O
Tyr319 O
as O
a O
functionally O
important O
phosphorylation O
site O
in O
the O
ZAP B
- I
70 I
interdomain I
B I
region I
. O

These O
findings O
suggest O
that O
low O
- O
power O
laser O
irradiation O
can O
be O
used O
for O
promotion O
of O
vascularization O
and O
take O
of O
tissue O
transplants O
. O

RESEARCH O
DESIGN O
AND O
METHODS O
: O
A O
total O
of O
26 O
IDDM O
patients O
with O
normoalbuminuria O
were O
randomized O
into O
two O
groups O
, O
with O
one O
group O
receiving O
placebo O
( O
n O
= O
13 O
, O
age O
36 O
+ O
/ O
- O
3 O
years O
, O
BMI O
24 O
. O
5 O
+ O
/ O
- O
1 O
. O
1 O
kg O
/ O
m2 O
) O
and O
the O
other O
group O
receiving O
an O
average O
of O
15 O
mg O
lisinopril O
daily O
( O
n O
= O
13 O
, O
age O
34 O
+ O
/ O
- O
2 O
years O
, O
BMI O
24 O
. O
4 O
+ O
/ O
- O
0 O
. O
9 O
kg O
/ O
m2 O
) O
. O

Previously O
, O
a O
cDNA O
( O
GT2 B
) O
encoding O
this O
protein O
was O
isolated O
from O
a O
mouse O
3T3 O
- O
L1 O
adipocyte O
library O
and O
was O
sequenced O
. O

Expression O
of O
human O
complement B
receptor I
type I
2 I
( O
CR2 B
/ O
CD21 B
) O
is O
primarily O
restricted O
to O
mature O
B O
cells O
and O
follicular O
dendritic O
cells O
. O

The O
6 O
. O
5 O
- O
kb O
genomic O
fragment O
contains O
the O
complete O
coding O
region O
of O
MyoD B
, O
distributed O
over O
three O
exons O
, O
plus O
2 O
. O
3 O
kb O
of O
5 O
' O
- O
noncoding O
sequence O
and O
1 O
. O
4 O
kb O
of O
3 O
' O
- O
noncoding O
sequence O
. O

Synthetic O
studies O
on O
furan O
derivatives O
by O
the O
wittig O
reaction O

The O
apparent O
binding O
constant O
of O
6 O
to O
calf O
thymus O
DNA O
is O
1 O
. O
68 O
X O
10 O
( O
5 O
) O
M O
- O
1 O
whereas O
netropsin O
under O
similar O
conditions O
gives O
a O
value O
of O
1 O
. O
85 O
X O
10 O
( O
7 O
) O
M O
- O
1 O
. O

Phenotypic O
screening O
of O
mutations O
in O
Pmr1 B
, O
the O
yeast O
secretory O
pathway I
Ca2 B
+ I
/ I
Mn2 I
+ I
- I
ATPase I
, O
reveals O
residues O
critical O
for O
ion O
selectivity O
and O
transport O
. O

Molecular O
cloning O
of O
cDNAs O
encoding O
alpha O
- O
subunits O
of O
guanine B
nucleotide I
- I
binding I
regulatory I
proteins I
( O
G B
- I
proteins I
) O
has O
revealed O
the O
existence O
of O
nine O
species O
of O
alpha O
- O
subunits O
. O

No O
transactivation O
of O
the O
ovalbumin B
promoter I
( O
pLovTATA B
) O
template O
control O
was O
observed O
. O

Pros O
and O
cons O
of O
selective O
inhibition O
of O
cyclooxygenase B
- I
2 I
versus O
dual O
lipoxygenase B
/ O
cyclooxygenase B
inhibition O
: O
is O
two O
better O
than O
one O
? O

At O
11 O
. O
5 O
years O
of O
follow O
- O
up O
, O
521 O
major O
coronary O
disease O
events O
had O
occurred O
, O
261 O
fatal O
and O
260 O
non O
- O
fatal O
. O

The O
etiology O
was O
established O
in O
73 O
( O
76 O
. O
8 O
% O
) O
out O
of O
95 O
cases O
. O

Morphometry O
of O
the O
intestine O
of O
the O
pig O
. O

Serum O
zinc O
and O
copper O
levels O
and O
urine O
copper O
concentrations O
in O
men O
were O
significantly O
lower O
than O
in O
women O
, O
while O
there O
were O
no O
differences O
in O
serum O
or O
urinary O
zinc O
and O
copper O
levels O
with O
age O
. O

HDE B
was O
found O
to O
be O
exclusively O
targeted O
to O
and O
imported O
into O
peroxisomes O
in O
both O
heterologous O
expression O
systems O
. O

GeneCalling O
analysis O
was O
successful O
in O
detecting O
members O
of O
complex O
metabolic O
pathways O
and O
uncovering O
novel O
genes O
that O
were O
either O
coincidentally O
regulated O
or O
directly O
involved O
in O
such O
pathways O
. O

Transformations O
with O
circular O
plasmids O
yielded O
slowly O
and O
irregularly O
growing O
geneticin B
- O
resistant O
mycelia O
in O
which O
1 O
% O
of O
nuclei O
contained O
plasmid O
sequences O
. O

CONCLUSION O
: O
In O
these O
patients O
control O
of O
ventricular O
response O
rate O
with O
either O
HBA O
+ O
VVIR O
pacemaker O
or O
atrioventricular O
modifying O
drugs O
+ O
VVI O
pacemaker O
will O
lead O
to O
a O
significant O
improvement O
in O
exercise O
duration O
and O
quality O
of O
life O
. O

The O
other O
regions O
include O
potential O
binding O
sites O
for O
transcription B
factors I
ATF I
, O
NF1 B
, O
and O
a O
CCAAT B
- I
binding I
protein I
. O

Both O
purH B
and O
purD B
genes I
constitute O
a O
single O
operon O
and O
are O
coregulated O
in O
expression O
by O
purines O
as O
other O
purine O
genes O
are O
. O

The O
patient O
demographics O
( O
means O
+ O
/ O
- O
standard O
deviations O
) O
were O
as O
follows O
: O
age O
, O
57 O
+ O
/ O
- O
12 O
years O
; O
sex O
, O
nine O
males O
and O
three O
females O
; O
APACHE O
II O
score O
, O
15 O
+ O
/ O
- O
3 O
; O
diagnosis O
, O
9 O
of O
12 O
patients O
with O
pneumonia O
. O

In O
situ O
hybridization O
reveals O
strong O
signals O
for O
Zep B
mRNA I
in O
the O
cerebellum O
and O
olfactory O
bulb O
with O
moderate O
signals O
detected O
in O
the O
hippocampus O
and O
cortex O
. O

TNF B
- I
alpha I
effect O
was O
eliminated O
by O
a O
2 O
- O
bp O
substitution O
mutation O
in O
the O
NF B
- I
kappa I
B1 I
binding I
half I
site I
of O
the O
NF B
- I
kappa I
B I
cis I
element I
. O

UV O
- O
cross O
- O
linking O
studies O
suggested O
that O
these O
two O
complexes O
represent O
one O
and O
two O
molecules O
of O
ADR1 B
bound O
to O
DNA O
. O

As O
already O
available O
for O
the O
other O
known O
mammalian O
members O
of O
this O
enzyme O
family O
, O
we O
here O
define O
structural O
and O
functional O
features O
of O
human O
lymphoma I
proprotein I
convertase I
( O
LPC B
) O
. O

The O
M O
- O
phase O
induction O
activity O
of O
Cdc2 B
- O
Cdc13 B
is O
inhibited O
by O
Wee1 B
tyrosine B
kinase I
, O
which O
phosphorylates O
Cdc2 B
on O
tyrosine O
- O
15 O
. O

Nucleotide O
sequencing O
revealed O
a O
2076 O
- O
base O
pair O
open O
reading O
frame O
encoding O
a O
692 O
- O
amino O
acid O
protein O
. O

But O
it O
is O
possible O
that O
the O
two O
hemispheres O
differ O
in O
the O
manner O
in O
which O
they O
make O
use O
of O
semantic O
structures O
: O
the O
left O
hemisphere O
in O
a O
selective O
manner O
, O
appropriate O
for O
the O
solution O
of O
a O
specific O
task O
, O
and O
the O
right O
hemisphere O
in O
a O
more O
diffuse O
and O
global O
manner O
. O

Supportive O
therapy O
with O
xenogenous O
peptides O
in O
patients O
with O
metastatic O
breast O
cancer O
undergoing O
aggressive O
chemotherapy O
( O
modified O
AC O
- O
protocol O
) O
: O
a O
prospective O
, O
randomized O
double O
- O
blind O
study O

The O
pregnancies O
were O
terminated O
at O
19 O
and O
12 O
weeks O
of O
gestation O
, O
respectively O
. O

Single O
substitutions O
of O
three O
highly O
conserved O
phenylalanine O
residues O
( O
Phe O
- O
15 O
, O
Phe O
- O
17 O
, O
Phe O
- O
27 O
) O
by O
alanine O
and O
substitution O
of O
one O
histidine O
( O
His O
- O
29 O
) O
by O
glutamine O
, O
all O
located O
within O
the O
putative O
RNA O
- O
binding O
sites O
RNP B
- I
1 I
and O
RNP B
- I
2 I
, O
abolished O
the O
nucleic O
acid O
- O
binding O
activity O
of O
CspB B
. O

The O
cases O
with O
terminal O
forces O
of O
left O
ventricular O
activation O
in O
the O
same O
direction O
as O
the O
delta O
wave O
, O
superiorly O
and O
to O
the O
left O
at O
- O
60 O
degrees O
or O
inferiorly O
and O
to O
the O
right O
at O
+ O
120 O
degrees O
, O
forming O
a O
single O
deflection O
of O
over O
0 O
, O
12 O
seconds O
' O
duration O
, O
are O
the O
result O
of O
delayed O
activation O
of O
the O
anterior O
or O
posterior O
fascicle O
of O
the O
left O
bundle O
after O
a O
long O
delay O
. O

The O
genes O
encoding O
these O
carotenoids O
in O
E O
. O
herbicola O
Eho13 O
are O
clustered O
in O
a O
7 O
kb O
DNA O
fragment O
. O

The O
results O
show O
that O
while O
a O
larger O
polypeptide O
substrate O
carrying O
the O
HD1 B
/ I
3C I
site O
was O
processed O
more O
efficiently O
than O
a O
polypeptide O
substrate O
carrying O
the O
POL B
/ I
Zn I
site O
, O
cleavage O
of O
the O
synthetic O
peptide O
substrates O
containing O
these O
two O
cleavage O
sites O
occurred O
at O
similar O
efficiencies O
. O

Using O
transgenic O
lines O
a O
detailed O
analysis O
of O
the O
Hoxa B
- I
7 I
enhancer O
- O
directed O
expression O
during O
embryogenesis O
was O
performed O
. O

lacZ B
expression O
was O
first O
detected O
in O
the O
allantois O
at O
day O
7 O
. O
5 O
p O
. O
c O
. O
and O
in O
mesoderm O
and O
ectoderm O
at O
day O
8 O
. O
5 O
of O
gestation O
. O

An O
ERp60 B
- I
like I
protein I
from O
the O
filarial O
parasite O
Dirofilaria O
immitis O
has O
both O
transglutaminase B
and O
protein B
disulfide I
isomerase I
activity O
. O

At O
promoters O
that O
initiate O
with O
+ O
1 O
GGG O
, O
T7 B
RNAP I
synthesizes O
a O
ladder O
of O
poly O
( O
G O
) O
products O
as O
a O
result O
of O
slippage O
of O
the O
transcript O
on O
the O
three O
C O
residues O
in O
the O
template O
strand O
from O
+ O
1 O
to O
+ O
3 O
. O

Serum O
levels O
of O
angiotensin B
converting I
enzyme I
were O
well O
maintained O
. O

The O
UL3 B
ORF I
of O
204 O
amino O
acids O
shows O
significant O
homology O
to O
UL3 B
( O
nuclear O
phosphoprotein O
) O
of O
HSV O
- O
1 O
( O
62 O
% O
) O
and O
PRV O
( O
53 O
% O
) O
. O

On O
Western O
blots O
, O
the O
same O
antibodies O
recognized O
the O
recombinant O
protein O
migrating O
slightly O
slower O
on O
SDS O
/ O
PAGE O
than O
chicken B
axonin I
- I
1 I
. O

For O
routine O
medical O
applications O
, O
no O
sophisticated O
adjustment O
of O
the O
CHESS O
pulse O
is O
needed O
, O
as O
reported O
in O
previous O
methods O
. O

The O
10 O
eyes O
injected O
with O
human O
RPE O
cells O
showed O
STAGE O
2 O
or O
less O
in O
4 O
weeks O
. O

This O
report O
establishes O
that O
the O
effect O
of O
homozygous O
sickle O
cell O
disease O
on O
growth O
patterns O
in O
childhood O
is O
apparent O
before O
the O
age O
of O
6 O
years O
. O

The O
remaining O
77 O
nucleotides O
at O
the O
3 O
' O
end O
of O
domain O
II O
and O
all O
of O
domains O
III O
( O
655 O
nucleotides O
) O
and O
IV O
( O
770 O
nucleotides O
) O
are O
not O
present O
in O
DIssE O
RNA O
. O

The O
biological O
actions O
of O
VHR B
in O
oocytes O
clearly O
distinguish O
it O
from O
other O
dual O
specificity O
phosphatases O
, O
which O
have O
shown O
inhibitory O
effects O
when O
tested O
in O
oocytes O
. O

The O
presence O
of O
chloroquine O
in O
saliva O
from O
seven O
healthy O
volunteers O
for O
21 O
days O
after O
a O
single O
600 O
mg O
oral O
dose O
of O
the O
drug O
was O
established O
by O
chromatographic O
and O
spectroscopic O
methods O
. O

The O
gp41 B
peptide I
( O
Glu O
- O
Leu O
- O
Asp O
- O
Lys O
- O
Trp O
- O
Ala O
) O
fused O
to O
the O
C O
- O
terminus O
of O
Sj B
GST I
forms O
a O
loop O
stabilized O
by O
symmetry O
- O
related O
GSTs B
. O

Comparison O
of O
bioreactive O
and O
immunoreactive O
gastrin I
. O

Also O
, O
PTx O
had O
no O
effect O
on O
shear O
- O
dependent O
activation O
of O
JNK B
. O

A O
score O
( O
APACHE O
II O
) O
was O
calculated O
to O
assess O
the O
severity O
of O
disease O
. O

We O
tested O
several O
growth O
regulatory O
genes O
that O
are O
repressed O
in O
senescent O
cells O
for O
ability O
to O
restore O
activity O
to O
T B
[ I
K1 I
] I
. O

We O
analyzed O
the O
contribution O
of O
the O
three O
different O
types O
of O
UV O
- O
inducible O
mitogen B
- I
activated I
protein I
( I
MAP I
) I
kinases I
( O
ERK B
, O
JNK B
/ O
SAPK B
, O
and O
p38 B
) O
to O
the O
activation O
of O
the O
murine B
uPA I
promoter I
by O
UV O
. O

The O
high O
degree O
of O
sequence O
conservation O
together O
with O
the O
ability O
to O
direct O
nucleolar O
protein O
transport O
supports O
the O
hypothesis O
that O
MAK16 B
proteins I
play O
a O
key O
role O
in O
the O
biogenesis O
of O
60S B
subunits I
. O

Characterization O
of O
a O
nuclear B
deformed I
epidermal I
autoregulatory I
factor I
- I
1 I
( I
DEAF B
- I
1 I
) I
- O
related I
( O
NUDR B
) O
transcriptional O
regulator O
protein O
. O

Here O
we O
show O
that O
the O
sck1 B
gene I
, O
cloned O
as O
a O
high O
copy O
number O
suppressor O
of O
a O
mutation O
in O
git3 B
, O
is O
able O
to O
suppress O
the O
defects O
conferred O
by O
a O
mutation O
in O
any O
of O
these O
git B
genes I
. O

Hamdija O
Karamehmedovic O
; O
Ibn O
al O
- O
Nefis O
, O
" O
Mudzez O
al O
- O
Kanum O
" O
, O
Republicki O
zavod O
za O
zdravstvenu O
zastitu O
Sarajevo O
, O
1961 O
, O
1 O
- O
219 O
; O
Mr O
ph O
Samuel O
Elazar O
, O
Ajnija O
Omanic O
: O
" O
Bibliografija O
medicinskih O
djela O
u O
SR O
BiH O
do O
1895 O
. O
" O
Medicinska O
knjiga O
Beograd O
- O
Zagreb O
1984 O
; O
Besides O
, O
the O
great O
contribution O
in O
bibliography O
was O
given O
by O
dr O
Lujo O
Taler O
, O
prof O
. O

dr O
. O

The O
longest O
cDNA O
insert O
identified O
was O
2 O
. O
2 O
kb O
and O
encoded O
the O
entire O
462 O
- O
amino O
acid O
open O
reading O
frame O
of O
rat B
CgA I
including O
an O
18 O
- O
amino O
acid O
hydrophobic O
signal O
peptide O
. O

We O
have O
used O
systemic O
application O
of O
the O
ototoxic O
drug O
amikacin O
, O
to O
induce O
total O
cochlear O
haircell O
loss O
in O
the O
chinchilla O
, O
in O
order O
to O
create O
an O
animal O
model O
of O
profound O
deafness O
. O

Cotransfection O
with O
C B
/ I
EBPbeta I
and O
GATA B
- I
1 I
expression O
vectors O
produced O
a O
5 O
- O
fold O
increase O
compared O
with O
cotransfection O
with O
the O
C B
/ I
EBPbeta I
or O
GATA B
- I
1 I
expression O
vectors O
individually O
. O

Among O
the O
different O
epithelial O
cell O
lines O
tested O
, O
only O
RTS3b O
cells O
allowed O
an O
expression O
pattern O
similar O
to O
that O
observed O
in O
naturally O
infected O
benign O
condylomas O
. O

In O
contrast O
to O
the O
p16 B
- O
mediated O
G1 O
block O
, O
G1 O
arrest O
mediated O
by O
the O
cdk B
inhibitors O
p21Cip1 B
or O
p27Kip1 B
cannot O
be O
bypassed O
either O
by O
inactivation O
of O
pRb B
or O
overexpression O
of O
E2F B
family I
members I
. O

In O
addition O
, O
the O
authors O
found O
one O
an3 B
allele I
( O
an3 B
- I
W138A I
) O
in O
which O
a O
dTph1 B
element O
had O
inserted O
30 O
bp O
upstream O
the O
translation O
start O
, O
without O
inactivating O
the O
gene O
. O

Case O
of O
nephrotic O
syndrome O
caused O
by O
gold O
preparations O

To O
investigate O
the O
role O
of O
bHLH B
proteins I
in O
MC3T3 O
- O
E1 O
osteoblasts O
, O
which O
undergo O
a O
developmental O
sequence O
in O
vitro O
, O
we O
analyzed O
the O
transcriptional O
control O
of O
osteocalcin B
gene I
expression O
by O
stable O
transfection O
of O
an O
osteocalcin B
promoter I
- O
luciferase B
chimeric O
gene O
( O
p637OC B
- O
luc B
) O
and O
assessed O
the O
role O
of O
E O
- O
box O
cis O
- O
acting O
elements O
in O
osteocalcin B
promoter I
by O
DNA O
binding O
assays O
. O

In O
summary O
, O
the O
only O
treatment O
- O
related O
effect O
noted O
in O
this O
study O
was O
hydrocarbon O
nephropathy O
in O
male O
rats O
, O
which O
is O
not O
considered O
relevant O
for O
human O
health O
. O

In O
this O
study O
, O
we O
show O
that O
HNF B
- I
4 I
, O
a O
member O
of O
the O
steroid B
hormone I
receptor I
superfamily I
, O
binds O
the O
AF O
- O
1 O
site O
on O
the O
apoB B
promoter I
and O
through O
it O
activates O
transcription O
in O
transient O
transfection O
assays O
in O
both O
liver O
and O
non O
- O
liver O
cell O
lines O
, O
HepG2 O
and O
HeLa O
, O
respectively O
. O

Gastrointestinal O
accumulation O
of O
indium O
- O
111 O
labelled O
granulocytes O
in O
reactive O
arthritis O
. O

Acute O
intoxication O
with O
cypermethrin O
( O
NRDC O
149 O
) O
. O

We O
have O
previously O
shown O
that O
the O
transcriptional O
activator O
CREM B
is O
highly O
expressed O
in O
postmeiotic O
cells O
. O

No O
interaction O
with O
epidural O
anesthesia O
was O
observed O
. O

In O
late O
October O
, O
1974 O
, O
Staphylococcus O
aureus O
postoperative O
wound O
infection O
was O
recorded O
in O
a O
nonhuman O
primate O
( O
Macaca O
mulatta O
) O
which O
had O
recently O
undergone O
surgical O
operation O
. O

Twenty O
- O
six O
calves O
were O
subjected O
to O
a O
technique O
of O
cryoablation O
in O
order O
to O
establish O
an O
animal O
model O
of O
complete O
cardiac O
denervation O
. O

Our O
current O
study O
aims O
at O
clarifying O
the O
role O
of O
myristoylation O
in O
caveolar O
targeting O
using O
well O
- O
characterized O
acylation O
mutants O
of O
two O
model O
proteins O
, O
namely O
Gi1 B
alpha I
and O
c B
- I
Src I
. O

The O
effect O
of O
deferoxamine O
B O
on O
the O
blood O
sugar O
behavior O
in O
intravenous O
glucose O
loading O
in O
healthy O
persons O

We O
infer O
that O
RecA B
- O
mediated O
cleavage O
of O
UmuD B
is O
another O
role O
for O
RecA B
in O
SOS O
mutagenesis O
, O
probably O
activating O
UmuD B
for O
its O
mutagenic O
function O
. O

SETTING O
: O
Participants O
in O
the O
Physicians O
' O
Health O
Study O
, O
a O
randomized O
trial O
of O
aspirin O
and O
beta O
- O
carotene O
among O
U O
. O
S O
. O
male O
physicians O
. O

The O
prevalence O
of O
microalbuminuria O
, O
defined O
as O
an O
UAER O
in O
the O
range O
of O
15 O
- O
150 O
micrograms O
min O
- O
1 O
in O
an O
overnight O
urine O
sample O
, O
was O
3 O
% O
( O
95 O
% O
C O
. O
I O
. O
interval O
: O
1 O
. O
9 O
- O
4 O
. O
0 O
) O
. O

The O
Rep B
proteins I
of O
adeno I
- I
associated I
virus I
type O
2 I
( O
AAV O
) O
are O
known O
to O
bind O
to O
Rep B
recognition I
sequences I
( O
RRSs B
) O
in O
the O
AAV O
inverted O
terminal O
repeats O
( O
ITRs O
) O
, O
the O
AAV B
p5 I
promoter I
, O
and O
the O
preferred O
AAV O
integration O
site O
in O
human O
chromosome O
19 O
, O
called O
AAVS1 B
. O

LIP O
and O
DIP O
are O
less O
common O
IIPs O
, O
both O
characterized O
by O
ground O
- O
glass O
attenuation O
. O

The O
levels O
of O
TPAR1 B
mRNAs I
were O
dramatically O
down O
- O
regulated O
in O
regenerating O
rat O
liver O
when O
compared O
to O
normal O
adult O
liver O
. O

Copyright O
1999 O
Academic O
Press O
. O

The O
authors O
suggest O
that O
alprazolam O
may O
have O
enhanced O
specificity O
for O
a O
subpopulation O
of O
benzodiazepine B
receptors I
. O

Southern O
hybridization O
analysis O
of O
BenR B
and O
BenS B
transformants O
suggested O
that O
plasmid O
integration O
occurred O
most O
frequently O
at O
the O
chromosomal B
bens I
locus I
, O
however O
evidence O
for O
gene O
conversion O
and O
heterologous O
recombination O
was O
also O
observed O
. O

The O
analysis O
of O
the O
effects O
of O
eight O
signaling O
molecules O
in O
the O
TGFbeta B
, O
FGF B
, O
Hh B
, O
Wnt B
, O
and O
EGF B
families I
in O
tooth O
explant O
cultures O
revealed O
that O
the O
expression O
of O
edar B
was O
induced O
by O
activinbetaA B
, O
whereas O
Wnt6 B
induced O
ectodysplasin B
expression O
. O

In O
mouse O
, O
two O
high O
- O
affinity O
binding O
sites O
with O
an O
apparent O
dissociation O
constant O
( O
Kd O
) O
of O
50 O
to O
100 O
nM O
have O
been O
mapped O
in O
the O
5 O
' I
ETS I
upstream O
from O
the O
early O
pre O
- O
rRNA O
processing O
site O
. O

Single O
oblique O
- O
view O
mammography O
for O
periodic O
screening O
for O
breast O
cancer O
in O
women O
. O

Radiative O
corrections O
to O
pi O
l2 O
decays O
. O

This O
deletion O
disrupts O
the O
PU B
. I
1 I
Ets B
domain O
. O

In O
turn O
, O
production O
of O
sufficient O
amounts O
of O
TraR B
apparently O
is O
sensitive O
to O
a O
cellular O
function O
requiring O
RNase B
D I
. O

The O
predicted O
product O
exhibits O
91 O
% O
amino O
acid O
identity O
to O
the O
murine B
voltage I
- I
gated I
potassium I
channel I
protein I
Kv1 I
. I
7 I
( O
Kcna7 B
) O
, O
which O
plays O
an O
important O
role O
in O
the O
repolarization O
of O
cell O
membranes O
. O

Phytohemagglutinin B
( O
PHA B
) O
, O
the O
seed B
lectin I
of I
the I
common I
bean I
, O
accumulates O
in O
protein O
storage O
vacuoles O
of O
storage O
parenchyma O
cells O
in O
cotyledons O
. O

This O
dynamic O
nature O
may O
be O
relevant O
to O
the O
ability O
of O
E47 B
both O
to O
homodimerize O
and O
to O
heterodimerize O
with O
MyoD B
, O
Id B
, O
and O
Tal1 B
. O

Blood O
samples O
( O
30 O
ml O
) O
were O
collected O
via O
an O
indwelling O
arm O
catheter O
at O
rest O
, O
and O
at O
minutes O
13 O
and O
28 O
of O
exercise O
for O
determinations O
of O
plasma O
EPI B
, O
serum O
free O
fatty O
acid O
( O
FFA O
) O
, O
serum O
glycerol O
( O
GLY O
) O
, O
blood O
glucose O
( O
GLU O
) O
, O
and O
blood O
lactate O
( O
LA O
) O
concentrations O
. O

Among O
these O
, O
four O
patients O
had O
the O
AIDS O
syndrome O
, O
while O
42 O
individuals O
were O
HIV O
carriers O
. O

Subjects O
were O
73 O
male O
and O
female O
employees O
of O
the O
Xerox O
Corporation O
joining O
a O
newly O
developed O
health O
fitness O
program O
. O

Overexpression O
of O
p50 B
in O
transient O
cotransfection O
studies O
using O
the O
proximal O
CRP B
promoter I
( O
- O
125 O
/ O
+ O
9 O
) O
linked O
to O
a O
luciferase B
reporter I
caused O
a O
3 O
- O
fold O
increase O
of O
luciferase B
activity O
, O
while O
C B
/ I
EBPbeta I
overexpression O
caused O
an O
18 O
- O
fold O
increase O
; O
simultaneous O
overexpression O
of O
both O
transcription O
factors O
increased O
luciferase B
activity O
approximately O
600 O
- O
fold O
. O

Our O
results O
suggest O
that O
it O
might O
be O
possible O
to O
augment O
IN O
function O
in O
vivo O
through O
a O
heterologous O
domain O
. O

While O
differential O
expression O
of O
the O
two O
transcripts O
was O
not O
found O
, O
the O
promoter O
controlling O
LT1 B
/ O
LT2 B
transcription O
is O
regulated O
in O
a O
cell O
cycle O
- O
dependent O
manner O
. O

Schottky O
- O
barrier O
heights O
of O
Ti O
and O
TiSi2 O
on O
n O
- O
type O
and O
p O
- O
type O
Si O
( O
100 O
) O
. O

A O
survey O
of O
children O
with O
HBsAg B
markers O
related O
to O
their O
parents O
HBV O
markers O
. O

Immunological O
studies O
also O
failed O
to O
demonstrate O
any O
significant O
change O
except O
for O
a O
significant O
increase O
of O
natural O
killer O
( O
NK O
) O
cell O
activity O
after O
IFN B
- I
gamma I
infusion O
. O

Fetal O
PaO2 O
fell O
somewhat O
during O
the O
recovery O
stages O
in O
both O
NIT O
and O
control O
groups O
. O

Genetic O
analyses O
indicate O
that O
most O
of O
the O
dominant O
mutants O
are O
cis O
- O
acting O
and O
that O
the O
recessive O
mutants O
define O
a O
minimum O
of O
three O
complementation O
groups O
, O
indicating O
that O
defects O
in O
several O
different O
genes O
can O
restore O
higher O
levels O
of O
HIS4C B
expression O
. O

Rat B
cholesterol I
side I
- I
chain I
cleavage I
cytochrome I
P I
- I
450 I
( O
P B
- I
450scc I
) O
gene O
. O

Histopathologic O
studies O
displaying O
spongiform O
changes O
in O
the O
gray O
matter O
, O
neuronal O
loss O
, O
and O
atrogliosis O
confirmed O
the O
clinical O
diagnosis O
of O
Creutzfeldt O
- O
Jakob O
disease O
. O

Pig O
uPA B
promoter O
- O
CAT B
constructs O
were O
more O
active O
than O
mouse O
constructs O
in O
this O
assay O
. O

Cloning O
the O
cDNA O
for O
a O
new O
human B
zinc I
finger I
protein I
defines O
a O
group O
of O
closely O
related O
Kruppel B
- I
like I
transcription I
factors I
. O

By O
computed O
homology O
search O
, O
we O
noticed O
significant O
similarities O
between O
US3 B
PK I
and O
p21 B
- I
activated I
kinase I
( O
PAK B
) O
, O
which O
is O
activated O
by O
the O
Cdc42 B
or O
Rac B
. O

The O
mean O
amount O
of O
blood O
loss O
prior O
to O
time O
of O
injection O
was O
4 O
. O
5 O
units O
( O
a O
range O
of O
3 O
to O
10 O
units O
) O
. O

In O
response O
to O
acoustical O
stimulation O
the O
properties O
of O
response O
latency O
, O
discharge O
pattern O
, O
frequency O
tuning O
, O
binaural O
interaction O
, O
and O
habituation O
were O
examined O
to O
allow O
an O
appraisal O
of O
the O
differentiation O
of O
the O
MGB O
by O
electrophysiological O
means O
. O

These O
8 B
- I
oxoguanine I
DNA I
glycosylases I
, O
hOgg1 B
( O
human O
) O
and O
mOgg1 B
( O
murine O
) O
, O
are O
homologous O
to O
each O
other O
and O
to O
yeast B
Ogg1 I
. O

The O
CHED B
protein I
includes O
the O
consensus O
ATP O
binding O
and O
phosphorylation O
domains O
characteristic O
of O
kinases O
, O
displays O
34 O
- O
42 O
% O
identically O
aligned O
amino O
acid O
residues O
with O
other O
cdc2 B
- I
related I
kinases I
, O
and O
is O
considerably O
longer O
at O
its O
amino O
and O
carboxyl O
termini O
. O

CONCLUSIONS O
: O
These O
results O
suggest O
that O
, O
by O
interacting O
with O
cellular O
transcription O
factors O
and O
cofactors O
, O
vIRF B
- I
2 I
may O
modulate O
the O
expression O
of O
the O
early O
inflammatory O
genes O
and O
potentially O
deregulate O
the O
immune O
system O
. O

However O
, O
in O
7 O
patients O
with O
normal O
CT O
results O
the O
latter O
method O
showed O
areas O
of O
cerebral O
activity O
anatomically O
correlated O
with O
neurological O
signs O
or O
vascular O
lesions O
. O

We O
report O
that O
Gcn5 B
, O
a O
histone B
H3 I
acetylase I
, O
plays O
a O
central O
role O
in O
initiation O
of O
meiosis O
via O
effects O
on O
IME2 B
expression O
. O

The O
carboxy O
- O
proximal O
regions O
of O
the O
VP1 B
, O
which O
contain O
very O
low O
amino O
acid O
homology O
, O
displayed O
evidence O
of O
conservation O
in O
structural O
features O
such O
as O
a O
hydrophilic O
, O
highly O
basic O
domain O
. O

Interferon B
- I
alpha I
inducibility O
of O
IFI16 B
may O
be O
regulated O
by O
an O
interferon B
- I
alpha I
/ I
beta I
- I
stimulated I
response I
consensus I
element I
in O
the O
5 O
' O
UT O
exon O
, O
as O
a O
similar O
motif O
is O
conserved O
in O
the O
corresponding O
position O
in O
the O
related O
myeloid B
cell I
nuclear I
differentiation I
antigen I
gene I
. O
( O
ABSTRACT O
TRUNCATED O
AT O
400 O
WORDS O
) O

DESIGN O
- O
- O
A O
randomised O
double O
blind O
placebo O
controlled O
parallel O
arm O
trial O
. O

Genealogical O
analysis O
suggested O
mainly O
a O
mother O
- O
to O
- O
offspring O
transmission O
of O
this O
STLV B
- I
1 I
. O

These O
back O
mutations O
led O
to O
a O
modest O
decrease O
in O
kinase O
activity O
, O
decreased O
tumorigenic O
potential O
in O
chickens O
, O
and O
an O
unexpected O
increase O
in O
transforming O
activity O
in O
rat O
cells O
. O

The O
quantification O
limit O
was O
50 O
ng O
/ O
ml O
for O
each O
of O
PHT O
, O
m O
- O
HPPH O
and O
p O
- O
HPPH O
. O

Individual O
intolerance O
to O
betaxolol O
will O
continue O
to O
occur O
and O
care O
is O
always O
required O
when O
patients O
with O
cardiac O
or O
respiratory O
dysfunction O
are O
exposed O
to O
any O
beta O
- O
antagonist O
. O

Pretoria O
Pasteurisation O
is O
feasible O
and O
reliable O
under O
a O
range O
of O
conditions O
. O

In O
the O
present O
study O
we O
use O
a O
tnaC B
- O
UGA O
- O
' O
lacZ I
construct O
lacking O
the O
tnaC B
- O
tnaA B
spacer O
region O
to O
analyze O
the O
effect O
of O
TnaC B
synthesis O
on O
the O
behavior O
of O
the O
ribosome O
that O
translates O
tnaC B
. O

The O
in O
vitro O
activity O
of O
KP O
- O
103 O
, O
a O
novel O
triazole O
derivative O
, O
against O
pathogenic O
fungi O
that O
cause O
dermatomycoses O
and O
its O
therapeutic O
efficacy O
against O
plantar O
tinea O
pedis O
and O
cutaneous O
candidiasis O
in O
guinea O
pigs O
were O
investigated O
. O

Restriction O
analysis O
of O
the O
isolated O
genomic O
clones O
indicated O
that O
the O
endogenous O
sequences O
abutting O
the O
3 O
' O
ends O
of O
the O
94 O
- O
A O
and O
94 O
- O
K O
transgenes O
are O
separated O
by O
less O
than O
20 O
kb O
, O
providing O
strong O
support O
for O
the O
single O
integration O
model O
. O

Serum O
triglycerides O
also O
showed O
an O
increase O
from O
birth O
to O
6 O
months O
of O
age O
, O
but O
a O
decrease O
from O
6 O
months O
to O
1 O
years O
of O
age O
. O

Female O
Wistar O
rats O
receiving O
alcohol O
( O
5 O
% O
) O
in O
drinking O
water O
during O
lactation O
( O
N O
= O
7 O
) O
were O
compared O
to O
normal O
controls O
fed O
ad O
libitum O
( O
N O
= O
6 O
) O
. O

Antisense O
oligonucleotides O
complementary O
to O
the O
5 O
' O
end O
of O
PKC B
- I
zeta I
mRNA I
sequences I
significantly O
reduced O
the O
collagen B
lattice O
- O
stimulated O
alpha2 B
and O
MMP B
- I
1 I
mRNA I
levels O
. O

Overexpression O
of O
a O
HIF B
- I
1alpha I
construct I
with O
deletions O
of O
the O
basic O
domain O
and O
carboxy O
terminus O
blocked O
reporter O
gene O
activation O
by O
endogenous B
HIF I
- I
1 I
in O
hypoxic O
cells O
. O

We O
hypothesized O
that O
conjugated O
estrogens O
, O
which O
contain O
several O
vasoactive O
estrogenic O
compounds O
, O
may O
favorably O
influence O
the O
vasomotor O
response O
to O
acetylcholine O
in O
men O
. O

In O
Experiment O
2 O
, O
levels O
were O
: O
amprolium O
and O
ethopabate O
, O
. O
02 O
% O
; O
salinomycin O
, O
55 O
mg O
/ O
kg O
; O
monensin O
, O
99 O
mg O
/ O
kg O
; O
and O
lasalocid O
, O
110 O
mg O
/ O
kg O
. O

Residues O
crucial O
for O
Ras B
interaction O
with O
GDP B
- I
GTP I
exchangers I
. O

4 O
. O

In O
serum O
- O
starved O
NIH O
3T3 O
cells O
, O
v B
- I
raf I
increased O
mdr1 B
promoter I
activity O
approximately O
10 O
- O
fold O
compared O
to O
a O
v B
- I
raf I
frame O
- O
shift O
control O
. O

Searching O
the O
human O
DNA O
data O
base O
of O
expressed O
sequence O
tags O
( O
EST O
) O
revealed O
novel O
partial O
sequences O
similar O
to O
, O
but O
distinct O
from O
, O
the O
sequences O
of O
the O
previously O
known O
PEF B
proteins I
. O

Under O
control O
conditions O
and O
in O
the O
presence O
of O
3 O
micromol O
/ O
kg O
MIB O
and O
VER O
the O
maximal O
effect O
of O
noradrenaline O
was O
reached O
at O
0 O
. O
1 O
micromol O
/ O
kg O
whereas O
in O
the O
presence O
of O
10 O
micromol O
/ O
kg O
MIB O
and O
VER O
it O
was O
reached O
at O
a O
dose O
of O
1 O
micromol O
/ O
kg O
. O

It O
is O
also O
affected O
by O
the O
HAP2 B
/ I
3 I
/ I
4 I
transcription I
factor I
complex I
and O
by O
SNF1 B
and O
SSN6 B
. O

The O
effective O
use O
of O
superoxide B
dismutase I
from O
human O
erythrocytes O
in O
the O
late O
stages O
of O
experimental O
influenza O
infection O

Two O
different O
genes O
( O
polA B
and O
polB B
) O
encoding O
family B
B I
DNA I
polymerases I
were O
cloned O
from O
the O
organism O
by O
PCR O
using O
degenerated O
primers O
based O
on O
the O
two O
conserved O
motifs O
( O
motif O
A O
and O
B O
) O
. O

The O
effects O
on O
survival O
of O
adjuvant O
treatments O
, O
including O
pre O
- O
or O
postoperative O
systemic O
or O
postoperative O
intra O
- O
arterial O
chemotherapy O
, O
are O
currently O
under O
evaluation O
. O

Structure O
and O
regulation O
of O
the O
luteinizing B
hormone I
receptor I
gene I
. O

The O
disease O
processes O
that O
affect O
transplant O
patients O
both O
before O
and O
after O
transplantation O
are O
not O
seen O
frequently O
in O
the O
general O
practice O
of O
gastroenterology O
. O

METHODS O
: O
Single O
white O
flash O
ERG O
, O
photopic O
ERG O
, O
scotopic O
ERG O
and O
flicker O
ERG O
were O
recorded O
in O
30 O
cases O
of O
unilateral O
CRVO O
. O

However O
, O
to O
encourage O
good O
contact O
between O
the O
farmers O
and O
the O
inseminating O
personnel O
, O
it O
is O
beneficial O
that O
herdsmen O
are O
present O
when O
cows O
are O
inseminated O
. O

Soft O
independent O
modelling O
of O
class O
analogy O
( O
SIMCA O
) O
is O
applied O
to O
identify O
near O
- O
infrared O
( O
NIR O
) O
spectra O
of O
ten O
excipients O
used O
in O
the O
pharmaceutical O
industry O
. O

Multiple O
organ O
failure O
( O
MOF O
) O
following O
major O
trauma O
occurs O
in O
response O
to O
perfusion O
deficits O
, O
a O
persistent O
inflammatory O
focus O
, O
or O
a O
persistent O
focus O
of O
dead O
and O
/ O
or O
injured O
tissue O
. O

Western O
immunoblot O
analysis O
detected O
p55gag B
and O
its O
cleavage O
products O
p39 B
and O
p27 B
in O
purified O
particles O
derived O
by O
expression O
of O
gag B
and O
gag B
- O
pol B
, O
respectively O
. O

Cefminox O
shows O
greater O
in O
vivo O
activity O
than O
that O
expected O
for O
the O
MICs O
, O
excellent O
efficacy O
and O
safety O
. O

CONCLUSION O
: O
high O
resolution O
MF O
- O
ERG O
seems O
more O
sensitive O
than O
low O
resolution O
MF O
- O
ERG O
. O

3 O
- O
5 O
- O
fold O
increases O
in O
EGF B
receptor I
but O
not O
p185 B
( O
neu B
) O
tyrosine O
phosphorylation O
occur O
following O
Gi B
- I
coupled I
receptor I
stimulation O
. O

Next O
, O
to O
examine O
the O
regulation O
mechanism O
of O
CD44 B
/ O
ERM B
interaction O
in O
vivo O
, O
we O
reexamined O
the O
immunoprecipitated O
CD44 B
/ O
ERM B
complex O
from O
BHK O
cells O
and O
found O
that O
it O
contains O
Rho B
- I
GDP I
dissociation I
inhibitor I
( O
GDI B
) O
, O
a O
regulator O
of O
Rho B
GTPase I
. O

The O
results O
showed O
a O
significant O
group O
effect O
only O
on O
the O
active O
task O
. O

On O
days O
7 O
, O
21 O
, O
and O
35 O
postinoculation O
( O
PI O
) O
, O
all O
birds O
received O
sheep O
erythrocytes O
intravenously O
. O

To O
determine O
cis O
- O
acting O
elements O
controlling O
the O
rat O
B B
- I
50 I
/ O
GAP B
- I
43 I
gene O
expression O
, O
the O
genomic O
DNA O
encoding O
exon O
1 O
and O
the O
5 O
' O
flanking O
sequence O
was O
isolated O
. O

In O
this O
paper O
, O
the O
authors O
used O
measured O
weight O
for O
1 O
, O
259 O
white O
women O
aged O
65 O
- O
74 O
years O
from O
the O
Epidemiologic O
Follow O
- O
up O
Study O
of O
the O
First O
National O
Health O
and O
Nutrition O
Examination O
Survey O
to O
examine O
the O
effect O
of O
overweight O
on O
coronary O
heart O
disease O
incidence O
( O
mean O
length O
of O
follow O
- O
up O
, O
14 O
years O
) O
. O

Through O
genomic O
library O
screening O
and O
PCR O
- O
based O
genomic O
walking O
we O
have O
now O
cloned O
the O
mouse B
E I
- I
Tmod I
gene I
, O
whose O
coding O
region O
spans O
approximately O
60kb O
containing O
nine O
exons O
and O
eight O
introns O
. O

TNF B
- I
alpha I
stimulated O
these O
changes O
in O
part O
by O
increasing O
transcription O
and O
stabilization O
of O
RNA O
for O
amphiregulin B
, O
an O
EGF B
receptor I
ligand I
, O
and O
amphiregulin B
directly O
increased O
HPV O
- O
16 O
E6 B
/ O
E7 B
and O
cyclin B
A I
RNAs I
. O

The O
data O
obtained O
included O
1 O
) O
the O
mean O
frequency O
of O
abnormal O
teased O
myelinated O
fibers O
and O
its O
upper O
limit O
value O
of O
95 O
% O
confidence O
interval O
, O
and O
2 O
) O
the O
mean O
densities O
of O
total O
, O
large O
and O
small O
myelinated O
fibers O
and O
of O
unmyelinated O
fibers O
and O
their O
lower O
limit O
value O
of O
95 O
% O
confidence O
interval O
for O
each O
decade O
. O

Fusions O
between O
the O
GAL B
DNA I
binding I
domain I
and O
full B
- I
length I
mSin3 I
were O
also O
capable O
of O
repression O
. O

None O
of O
the O
19 O
women O
with O
probable O
RA O
and O
100 O
of O
the O
116 O
women O
with O
definite O
RA O
met O
the O
1987 O
criteria O
. O

The O
initial O
oral O
administration O
of O
the O
1 O
and O
2 O
mg O
/ O
kg O
doses O
of O
enoximone O
improved O
central O
hemodynamic O
parameters O
with O
apparent O
preferential O
reduction O
of O
limb O
vascular O
resistance O
and O
augmentation O
of O
blood O
flow O
to O
the O
limb O
region O
( O
peripheral O
musculoskeletal O
system O
) O
. O

Antibodies O
against O
REAP B
- I
1 I
inhibit O
in O
vitro O
RNA O
editing O
reactions O
confirming O
its O
role O
in O
RNA O
editing O
. O

The O
full O
- O
length O
cDNA O
was O
cloned O
and O
sequenced O
, O
and O
the O
inferred O
amino O
acid O
sequence O
was O
found O
to O
encode O
a O
novel O
protein O
, O
which O
we O
named O
cystatin B
M I
, O
with O
40 O
% O
homology O
to O
human B
family I
2 I
cystatins I
and O
similar O
overall O
structure O
. O

42nd O
annual O
meeting O
of O
the O
American O
Academy O
of O
Oral O
Pathology O
. O

BAG B
- I
1 I
also O
prevented O
growth O
arrest O
following O
UV O
- O
irradiation O
- O
induced O
genotoxic O
injury O
without O
interfering O
with O
accumulation O
of O
p53 B
protein I
or O
p21 B
( O
waf B
- I
1 I
) O
expression O
. O

The O
side O
effect O
of O
pulmonary O
fibrosis O
occurs O
in O
20 O
to O
30 O
percent O
of O
patients O
receiving O
this O
drug O
. O

These O
larger O
sizes O
, O
however O
, O
are O
consistent O
with O
predictions O
from O
the O
DNA O
sequence O
of O
the O
pol B
gene I
. O

Primary O
care O
programs O
for O
the O
indigent O
. O

N0 O
disease O
is O
more O
favorable O
than O
N1 O
or O
N2 O
disease O
. O

In O
serial O
recall O
from O
short O
- O
term O
memory O
, O
repeated O
items O
are O
recalled O
well O
when O
close O
together O
( O
repetition O
facilitation O
) O
, O
but O
not O
when O
far O
apart O
( O
repetition O
inhibition O
; O
the O
Ranschburg O
effect O
) O
. O

In O
addition O
, O
two O
cis O
- O
acting O
elements O
direct O
the O
first O
zygotic O
expression O
of O
Kr B
in O
a O
striped O
subpattern O
within O
the O
central O
region O
of O
the O
blastoderm O
embryo O
. O

Two O
hundred O
and O
eighty O
- O
three O
patients O
, O
217 O
females O
( O
median O
age O
24 O
years O
, O
range O
0 O
. O
5 O
- O
73 O
) O
and O
66 O
males O
( O
median O
age O
20 O
years O
, O
range O
0 O
. O
75 O
- O
72 O
) O
, O
were O
examined O
. O

The O
Robert O
Wood O
Johnson O
Foundation O
: O
billion O
dollar O
force O
for O
health O
- O
care O
change O
. O

We O
have O
isolated O
a O
unique O
murine B
FLT3 I
cDNA I
that O
codes O
for O
a O
variant O
isoform O
of O
FLT3 B
, O
devoid O
of O
the O
fifth O
Ig B
- I
like I
domain O
, O
by O
comparison O
with O
the O
prototypic O
form O
. O

P53 B
mutations O
also O
offer O
new O
approaches O
to O
the O
study O
of O
the O
origins O
of O
mutations O
in O
human O
cancer O
. O

( O
1993 O
) O
8 O
, O
94 O
- O
99 O
) O
. O

Desarrollo O
de O
un O
modelo O
matematico O
general O
para O
los O
procesos O
fermentativos O
, O
Cinetica O
de O
la O
degradacion O
anaerobia O
, O
Ph O
. O
D O
. O

Particle O
bombardment O
of O
barley O
aleurone O
with O
a O
B22EL8 B
promoter I
- O
GUS B
( O
beta B
- I
glucuronidase I
) O
construct O
demonstrates O
that O
the O
promoter O
( O
3 O
kb O
) O
is O
active O
in O
developing O
barley O
grains O
. O

Changes O
in O
BHR O
were O
found O
to O
correlate O
significantly O
with O
changes O
in O
the O
levels O
of O
24 O
h O
mean O
SO2 O
, O
NO2 O
and O
smoke O
; O
48 O
h O
mean O
NO2 O
and O
smoke O
; O
24 O
h O
lag O
NO2 O
; O
although O
the O
effect O
was O
only O
small O
, O
accounting O
for O
approximately O
10 O
% O
of O
the O
variability O
in O
within O
- O
subject O
BHR O
between O
visits O
. O

The O
N O
- O
terminal O
sequences O
of O
isoforms O
beta O
to O
epsilon O
contain O
a O
PEST O
region O
which O
could O
induce O
rapid O
intracellular O
degradation O
of O
isoforms O
beta O
and O
delta O
. O

Characterization O
of O
regions O
of O
fibronectin B
besides O
the O
arginine O
- O
glycine O
- O
aspartic O
acid O
sequence O
required O
for O
adhesive O
function O
of O
the O
cell O
- O
binding O
domain O
using O
site O
- O
directed O
mutagenesis O
. O

The O
clinical O
effects O
of O
valproate O
and O
ethosuximide O
can O
be O
related O
to O
this O
differential O
modulation O
of O
thalamocortical O
excitability O
. O

Elements O
regulating O
cell O
- O
and O
stage O
- O
specific O
expression O
of O
the O
C B
. I
elegans I
MyoD I
family I
homolog I
hlh B
- I
1 I
. O

An O
analysis O
of O
the O
c B
- I
kit I
5 I
' I
flanking I
region I
using O
the O
bacterial B
chloramphenicol I
acetyltransferase I
gene I
( O
CAT B
assay O
) O
in O
human O
erythroleukemia O
HEL O
cells O
, O
which O
express O
the O
endogenous O
c B
- I
kit I
mRNA I
at O
high O
levels O
, O
showed O
that O
a O
region O
from O
- O
180 O
to O
- O
22 O
is O
important O
for O
the O
expression O
of O
the O
c B
- I
kit I
gene I
. O

After O
5 O
days O
stimulation O
with O
G B
- I
CSF I
( O
10 O
micrograms O
/ O
kg O
) O
1l O
of O
blood O
was O
drawn O
, O
kept O
unprocessed O
for O
3 O
days O
and O
reinfused O
24 O
h O
after O
completion O
of O
chemotherapy O
. O

OAT O
has O
become O
safer O
in O
recent O
years O
, O
particularly O
if O
monitored O
in O
special O
anticoagulation O
clinics O
. O

However O
, O
the O
relative O
substrate O
specificity O
of O
RSK3 B
differed O
from O
that O
reported O
for O
other O
isoforms O
. O

Our O
data O
indicate O
that O
the O
various O
subunits O
of O
the O
human B
P I
- I
TEFb I
complex I
may O
play O
distinct O
roles O
at O
multiple O
stages O
to O
mediate O
Tat B
activation O
of O
HIV O
- O
1 O
transcription O
elongation O
. O

Corresponding O
oligonucleotides O
were O
used O
to O
screen O
a O
rat O
liver O
cDNA O
library O
constructed O
in O
the O
plasmid O
cloning O
vector O
, O
pcDV O
. O

In O
fifty O
non O
selected O
ductal O
carcinomas O
of O
the O
breast O
we O
found O
that O
a O
marked O
tumoral O
inflammatory O
infiltrate O
( O
P O
less O
than O
0 O
. O
025 O
) O
, O
perinodal O
tumoral O
infiltrate O
( O
P O
less O
than O
0 O
. O
01 O
) O
, O
sinus O
catarrh O
( O
P O
less O
than O
0 O
. O
05 O
) O
, O
follicular O
hyperplasia O
( O
P O
less O
than O
0 O
. O
025 O
) O
, O
mixed O
pattern O
in O
lymph O
nodes O
( O
P O
less O
than O
0 O
. O
01 O
) O
and O
with O
54 O
years O
of O
age O
or O
younger O
( O
P O
less O
than O
0 O
. O
01 O
) O
correlated O
significantly O
with O
lymph O
node O
metastases O
and O
/ O

or O
high O
histologic O
grade O
. O

Capillary O
tortuosity O
was O
a O
function O
of O
sarcomere O
length O
in O
all O
animals O
and O
this O
relationship O
was O
not O
changed O
by O
hypoxia O
. O

Sox8 B
maps O
to O
the O
t B
complex I
on O
mouse O
chromosome O
17 O
and O
to O
human O
chromosome O
16p13 O
. O
3 O
, O
a O
region O
associated O
with O
the O
microphthalmia O
- O
cataract O
syndrome O
CATM O
and O
the O
alpha O
- O
thalassemia O
/ O
mental O
retardation O
syndrome O
ATR O
- O
16 O
. O

Directional O
growth O
of O
a O
smectic O
- O
A O
- O
smectic O
- O
B O
interface O
lying O
along O
a O
forbidden O
orientation O
. O

The O
TCR B
beta I
enhancer I
was O
active O
on O
both O
the O
minimal O
simian B
virus I
40 I
promoter I
and O
a O
TCR B
beta I
variable I
gene I
promoter I
in O
both O
TCR B
alpha I
/ I
beta I
+ I
and O
TCR B
gamma I
/ I
delta I
+ I
T O
cells O
. O

U O
. O

Recently O
, O
we O
demonstrated O
that O
platelet B
- I
derived I
growth I
factor I
( I
PDGF I
) I
- I
beta I
receptor I
stimulation O
, O
but O
not O
various O
other O
growth O
factors O
, O
inhibits O
transcription O
of O
alpha1D B
- I
, O
but O
not O
alpha1A B
- I
or I
alpha1B I
- I
ARs I
, O
resulting O
in O
reduced O
norepinephrine O
- O
mediated O
SMC O
growth O
. O

Southern O
blot O
analysis O
following O
reverse O
transcription O
and O
PCR O
showed O
that O
B2R B
is O
expressed O
in O
most O
mouse O
tissues O
, O
except O
the O
liver O
and O
spleen O
, O
which O
is O
consistent O
with O
the O
wide O
distribution O
of O
B2R B
activity O
as O
deduced O
from O
pharmacological O
studies O
. O

Long O
- O
term O
renal O
function O
in O
on O
- O
heart O
- O
beating O
donor O
kidney O
transplantation O
: O
a O
single O
- O
center O
experience O
. O

The O
study O
of O
the O
intensity O
of O
pain O
evoked O
by O
heat O
is O
relatively O
exhaustive O
: O
the O
influence O
of O
various O
local O
, O
stimulus O
- O
dependent O
or O
general O
factors O
upon O
threshold O
values O
has O
been O
well O
studied O
, O
as O
has O
the O
relation O
between O
pain O
and O
stimulus O
intensities O
. O

The O
vaccine O
was O
at O
least O
80 O
% O
efficacious O
against O
Chlamydia O
and O
Campylobacter O
spp O
and O
appeared O
to O
be O
protective O
. O

The O
centromeric O
dodeca O
- O
satellite O
of O
Drosophila O
forms O
altered O
DNA O
structures O
in O
vitro O
in O
which O
its O
purine O
- O
rich O
strand O
( O
G O
- O
strand O
) O
forms O
stable O
fold O
- O
back O
structures O
, O
while O
the O
complementary O
C O
- O
strand O
remains O
unstructured O
. O

Pneumatic O
dilation O
of O
the O
lower O
esophageal O
sphincter O
was O
accomplished O
by O
endoscopic O
visualization O
and O
positioning O
of O
a O
modified O
polyurethane O
dilator O
( O
90 O
F O
diameter O
) O
without O
fluoroscopy O
in O
17 O
consecutive O
patients O
with O
advanced O
symptomatic O
achalasia O
. O

Mean O
CD4 B
percentages O
were O
lower O
postpartum O
( O
21 O
. O
9 O
+ O
/ O
- O
3 O
. O
4 O
) O
than O
prepartum O
( O
45 O
. O
1 O
+ O
/ O
- O
6 O
. O
7 O
) O
in O
all O
five O
patients O
studied O
, O
although O
proliferative O
responses O
to O
the O
mitogens O
phytohemagglutinin B
and O
pokeweed O
were O
unchanged O
during O
the O
study O
period O
. O

The O
second O
system O
( O
VacFix O
) O
uses O
a O
0 O
. O
15 O
mm O
thick O
plastic O
bag O
loosely O
filled O
with O
1 O
mm O
polysterol O
spheres O
. O

Mean O
24 O
h O
urinary O
excretion O
values O
confirmed O
this O
observation O
. O

However O
, O
the O
safety O
of O
design O
4 O
was O
assured O
as O
long O
as O
all O
patients O
received O
three O
courses O
of O
chemotherapy O
, O
which O
is O
unusual O
in O
phase O
I O
studies O
in O
Japan O
. O

The O
results O
indicate O
that O
the O
RNA O
in O
T7 B
RNA I
polymerase I
is O
not O
free O
of O
steric O
interactions O
in O
the O
ternary O
complex O
and O
not O
available O
for O
structure O
formation O
until O
it O
is O
at O
least O
10 O
bases O
away O
from O
the O
site O
of O
polymerization O
. O

The O
STP1 B
locus I
is O
located O
on O
chromosome O
IV O
close O
to O
at O
least O
two O
other O
genes O
involved O
in O
RNA O
splicing O
: O
PRP3 B
and O
SPP41 B
. O

An O
ICU O
length O
of O
stay O
shorter O
than O
24 O
h O
was O
not O
related O
to O
the O
frequency O
of O
group O
C O
errors O
. O

In O
vitro O
, O
SHIP B
catalyzes O
the O
conversion O
of O
the O
phosphoinositide B
3 I
- I
kinase I
( O
PI3K B
) O
product O
phosphatidylinositol O
3 O
, O
4 O
, O
5 O
- O
trisphosphate O
( O
PIP3 O
) O
into O
phosphatidylinositol O
3 O
, O
4 O
- O
bisphosphate O
. O

Like O
pimethixene O
and O
cyproheptadine O
, O
WA O
335 O
has O
no O
distinct O
antagonistic O
qualities O
against O
bradykinin B
. O

To O
confirm O
the O
data O
obtained O
using O
the O
HPLC O
- O
ESI O
- O
MS O
procedure O
, O
fractions O
of O
the O
glycosides O
from O
four O
berries O
were O
separated O
, O
hydrolyzed O
, O
silylated O
and O
the O
sugars O
were O
analyzed O
using O
gas O
chromatography O
- O
mass O
spectrometry O
. O

This O
clone O
will O
be O
useful O
for O
identifying O
the O
genetic O
determinants O
of O
FIV O
- O
Oma B
' O
s O
biological O
activities O
. O

The O
WEB2 B
gene I
encodes O
a O
1 O
, O
522 O
- O
amino O
- O
acid O
protein O
with O
homology O
to O
nucleic B
acid I
- I
dependent I
ATPases I
. O

Evidence O
that O
the O
AT1A B
receptor I
activates O
transcription O
factor O
- O
Stat91 B
and O
/ O
or O
a O
related O
protein O
. O

There O
was O
91 O
% O
concordance O
between O
ST O
- O
RD O
/ O
RI O
/ O
LRD O
201Tl O
and O
R O
- O
ST O
/ O
N O
+ O
Inf O
99Tcm O
- O
tetrofosmin O
imaging O
regarding O
reversibility O
. O

The O
two O
complexes O
were O
detected O
with O
a O
similar O
activation O
kinetics O
upon O
IL B
- I
4 I
stimulation O
. O

These O
results O
suggest O
that O
dauricine O
and O
verapamil O
have O
a O
salutary O
effect O
in O
extracorporeal O
circulation O
. O

These O
results O
demonstrate O
the O
preferential O
endometrial O
cell O
- O
type O
expression O
of O
BTEB B
and O
suggest O
its O
regulatory O
role O
in O
pregnancy O
- O
associated O
endometrial O
epithelial O
gene O
expression O
. O

Spen B
is O
the O
only O
known O
homeotic B
protein I
with O
RNP O
binding O
motifs O
, O
which O
indicates O
that O
splicing O
, O
transport O
, O
or O
other O
RNA O
regulatory O
steps O
are O
involved O
in O
the O
diversification O
of O
segmental O
morphology O
. O

Long O
- O
term O
follow O
- O
up O
of O
kidney O
donors O
: O
a O
longitudinal O
study O
. O

Recent O
research O
on O
lipolysis O
. O

CONCLUSIONS O
: O
The O
present O
study O
showed O
it O
is O
possible O
to O
use O
a O
modified O
Stroop O
task O
as O
a O
measure O
of O
implicit O
processing O
of O
alcohol O
stimuli O
. O

Six O
patients O
( O
24 O
% O
) O
developed O
sepsis O
and O
only O
one O
survived O
. O

None O
of O
the O
10 O
sequences O
has O
hitherto O
been O
recognized O
as O
part O
of O
the O
p53 B
signaling O
pathway O
. O

The O
Prompt O
and O
Praise O
condition O
was O
superior O
to O
the O
other O
two O
conditions O
in O
encouraging O
participation O
in O
low O
- O
interest O
recreational O
activities O
. O

The O
majority O
of O
the O
human B
Ig I
heavy I
chain I
( I
IgH I
) I
constant I
( I
C I
) I
region I
locus I
has O
been O
cloned O
and O
mapped O
. O

A O
. O

We O
analyzed O
right O
ventricular O
size O
and O
function O
and O
201Tl O
uptake O
to O
determine O
if O
there O
was O
a O
relationship O
between O
201Tl O
uptake O
and O
systolic O
function O
in O
19 O
patients O
with O
pulmonary O
artery O
hypertension O
who O
were O
being O
evaluated O
for O
heart O
- O
lung O
transplantation O
. O

The O
transcriptional O
start O
site O
( O
s O
) O
of O
the O
IA B
- I
2 I
gene I
was O
mapped O
by O
5 O
' O
rapid O
amplification O
of O
cDNA O
ends O
to O
97 O
bp O
upstream O
of O
the O
translational O
start O
site O
. O

The O
RAG B
- I
2 I
gene I
encodes O
a O
component O
of O
the O
V B
( I
D I
) I
J I
recombinase I
which O
is O
essential O
for O
the O
assembly O
of O
antigen O
receptor O
genes O
in O
B O
and O
T O
lymphocytes O
. O

The O
lamina O
densa O
was O
always O
interrupted O
by O
numerous O
small O
gaps O
and O
in O
some O
areas O
the O
basement O
membrane O
could O
not O
be O
identified O
over O
a O
long O
distance O
. O

We O
have O
characterized O
21 O
mutations O
in O
the O
type B
VII I
collagen I
gene I
( O
COL7A1 B
) O
encoding O
the O
anchoring O
fibrils O
, O
18 O
of O
which O
were O
not O
previously O
reported O
, O
in O
patients O
from O
15 O
unrelated O
families O
with O
recessive O
dystrophic O
epidermolysis O
bullosa O
( O
RDEB O
) O
. O

The O
data O
when O
used O
to O
stimulate O
the O
collection O
of O
the O
information O
at O
antenatal O
or O
postnatal O
visits O
, O
nonetheless O
provide O
an O
accurate O
description O
of O
under O
- O
3 O
mortality O
trends O
and O
differences O
for O
the O
two O
periods O
examined O
- O
- O
before O
1984 O
and O
before O
1989 O
. O

Both O
residues O
are O
conserved O
in O
the O
three O
phosphorylated O
paralogs O
but O
are O
absent O
in O
the O
ones O
that O
were O
not O
substrates O
of O
RsbT B
: O
YetI B
and O
YezB B
, O
each O
of O
which O
bears O
only O
one O
of O
the O
conserved O
residues O
; O
and O
YtvA B
, O
which O
lacks O
both O
residues O
and O
instead O
possesses O
an O
N O
- O
terminal O
PAS B
domain O
. O

There O
is O
a O
good O
correlation O
between O
the O
occurrence O
in O
any O
particular O
vascular O
segment O
of O
the O
transient O
contractile O
response O
and O
intrinsic O
tone O
as O
assessed O
by O
relaxation O
to O
papaverine O
( O
10 O
( O
- O
6 O
) O
M O
) O
. O

Both O
atracurium O
1 O
- O
100 O
mumol O
litre O
- O
1 O
and O
laudanosine O
1 O
- O
50 O
mumol O
litre O
- O
1 O
enhanced O
the O
release O
of O
3H O
- O
NA O
evoked O
by O
field O
stimulation O
( O
2 O
Hz O
, O
24 O
stimuli O
) O
, O
but O
did O
not O
affect O
resting O
release O
. O

S16 O
, O
16 O
. O
8 O
% O
, O
P O
= O
0 O
. O
001 O
) O
. O

The O
Zn B
- I
15 I
DNA O
- O
binding O
domain O
comprises O
three O
zinc O
fingers O
separated O
by O
unusually O
long O
linker O
sequences O
that O
would O
be O
expected O
to O
interrupt O
specific O
DNA O
site O
recognition O
. O

Replication O
regions O
from O
plant O
- O
pathogenic O
Pseudomonas O
syringae O
plasmids O
are O
similar O
to O
ColE2 O
- O
related O
replicons O
. O

There O
has O
been O
no O
significant O
toxicity O
associated O
with O
repeated O
5 O
- O
FU O
instillation O
into O
the O
airway O
. O

Twenty O
male O
volunteers O
were O
studied O
during O
almokalant O
infusion O
aiming O
at O
plasma O
concentrations O
( O
Cpl O
) O
of O
20 O
, O
50 O
, O
100 O
, O
and O
150 O
nmol O
/ O
l O
. O

Consistently O
, O
we O
show O
that O
phosphorylation O
by O
protein B
kinase I
A I
inhibits O
Sfl1 B
DNA O
binding O
in O
vitro O
, O
and O
that O
a O
tpk2Delta B
mutation I
increases O
the O
levels O
of O
Sfl1 B
protein I
associated O
with O
specific O
promoter O
elements O
in O
vivo O
. O

The O
response O
sequences O
were O
localized O
between O
- O
67 O
and O
+ O
30 O
in O
the O
simian B
cytomegalovirus I
IE94 I
promoter I
and O
upstream O
of O
position O
+ O
9 O
in O
the O
HCMV B
IE68 I
promoter I
. O

Some O
CHOP B
- O
C B
/ I
EBP I
heterodimers O
apparently O
bind O
to O
alternative O
DNA O
sequence O
and O
thereby O
regulate O
the O
transcription O
of O
other O
genes O
. O

To O
identify O
and O
determine O
the O
function O
of O
Ino2p B
in O
yeast O
cells O
, O
we O
raised O
antibodies O
to O
a O
beta B
- I
galactosidase I
/ O
Ino2 B
fusion O
protein O
. O

Northern O
blot O
analyses O
were O
used O
to O
examine O
the O
regulated O
expression O
of O
the O
cys B
- I
14 I
+ I
gene I
. O

Typicality O
is O
probably O
a O
better O
representation O
of O
Alexander O
, O
Dunbar O
and O
others O
' O
conclusions O
than O
specificity O
, O
which O
was O
always O
too O
absolute O
a O
term O
. O

Additions O
of O
NH4 O
+ O
up O
to O
200 O
micrograms O
1 O
( O
- O
1 O
) O
were O
not O
stimulatory O
, O
whereas O
at O
1 O
. O
0 O
mg O
1 O
( O
- O
1 O
) O
levels O
, O
Ke O
was O
50 O
% O
greater O
than O
for O
NO3 O
- O
enriched O
medium O
. O

Cardiovascular O
drug O
use O
and O
hospitalizations O
attributable O
to O
type O
2 O
diabetes O
. O

The O
arrangement O
of O
amino O
acids O
in O
the O
helix O
1 O
and O
helix O
2 O
regions O
is O
quite O
similar O
to O
those O
of O
Mxi B
and O
Mad B
, O
but O
different O
from O
those O
of O
E2F B
- I
1 I
and O
DP B
- I
1 I
. O

They O
contain O
a O
large O
( O
0 O
. O
9 O
X O
10 O
( O
3 O
) O
to O
1 O
. O
88 O
X O
10 O
( O
3 O
) O
base O
- O
pairs O
) O
intron O
in O
the O
middle O
of O
the O
gene O
and O
are O
further O
interrupted O
close O
to O
the O
5 O
' O
end O
of O
the O
gene O
. O

The O
present O
study O
also O
emphasizes O
the O
need O
of O
establishing O
dose O
- O
response O
curves O
to O
correctly O
assess O
the O
relative O
contribution O
of O
the O
different O
regions O
of O
steroid B
hormone I
receptors I
in O
activation O
of O
transcription O
. O

Routine O
and O
default O
retrieval O
home O
visits O
were O
given O
to O
ensure O
maximal O
drug O
compliance O
. O

D O
. O
, O
and O
Long O
, O
C O
. O

By O
contrast O
with O
ama B
- I
1 I
, O
rpc B
- I
1 I
was O
not O
deleted O
by O
mDf4 B
or O
larger O
deficiencies O
examined O
, O
indicating O
that O
these O
genes O
are O
no O
closer O
than O
150 O
kb O
. O

Accordingly O
, O
constitutive O
downregulation O
of O
expression O
of O
accessory O
molecules O
of O
the O
MHC B
class I
I I
pathway O
can O
reveal O
differences O
between O
H2 B
class I
I I
alleles I
in O
antigen O
presentation O
not O
encountered O
when O
the O
expression O
levels O
are O
augmented O
. O

Anovulatory O
cycles O
with O
an O
apparently O
normal O
level O
of O
blood B
prolactin I
. O

Legionella O
antibodies O
in O
domestic O
animals O

By O
differential O
screening O
of O
a O
Xenopus O
laevis O
egg O
cDNA O
library O
, O
we O
have O
isolated O
a O
2 O
, O
111 O
bp O
cDNA O
which O
corresponds O
to O
a O
maternal O
mRNA O
specifically O
deadenylated O
after O
fertilisation O
. O

Similar O
immunological O
disturbances O
were O
observed O
in O
SCI O
but O
not O
in O
the O
PC2 O
subgroup O
, O
i O
. O
e O
. O
patients O
examined O
later O
than O
6 O
months O
after O
injury O
. O

This O
study O
demonstrates O
that O
Fluosol O
significantly O
attenuates O
neutrophil O
adherence O
, O
cytotoxicity O
, O
and O
enzyme O
release O
in O
an O
in O
vitro O
model O
of O
microvascular O
injury O
. O

Using O
the O
same O
electrical O
analog O
, O
we O
present O
an O
analysis O
that O
allows O
calculation O
of O
these O
parameters O
, O
as O
well O
as O
the O
corner O
frequency O
of O
the O
network O
( O
f1 O
) O
, O
without O
need O
for O
similar O
assumptions O
. O

We O
further O
examined O
the O
interaction O
of O
JAK2 B
with O
the O
GHR B
cytoplasmic I
domain I
by O
two O
lines O
of O
investigation O
. O

These O
p21 B
- O
E2F B
complexes O
, O
while O
present O
in O
young O
G1 O
cells O
at O
very O
low O
levels O
, O
were O
elevated O
in O
senescent O
cells O
. O

There O
is O
little O
similarity O
among O
the O
sequences O
upstream O
from O
the O
CAP O
site O
of O
the O
Spec2 B
genes I
except O
the O
TATA O
consensus O
sequence O
and O
a O
repeating O
trinucleotide O
, O
AAC O
. O

Thus O
, O
d O
- O
delta3 O
- O
carene O
was O
about O
four O
times O
more O
potent O
as O
a O
sensory O
irritant O
than O
I O
- O
beta O
- O
pinene O
, O
whereas O
the O
difference O
with O
I O
- O
alpha O
- O
pinene O
was O
more O
marked O
; O
as O
a O
sensory O
irritant O
, O
I O
- O
alpha O
- O
pinene O
is O
almost O
inactive O
. O

The O
reconstituted O
RNA B
polymerases I
containing O
the O
mutant O
alpha O
subunits O
were O
examined O
for O
their O
response O
to O
transcription O
activation O
by O
cAMP B
- I
CRP I
and O
the O
rrnBP1 B
UP I
element I
. O

The O
study O
drugs O
( O
combination O
vinblastine O
- O
CCNU O
and O
single O
- O
agent O
triazinate O
or O
dactinomycin O
) O
failed O
to O
provide O
any O
meaningful O
antitumor O
activity O
for O
these O
patients O
with O
advanced O
renal O
cell O
cancer O
. O

Our O
results O
indicate O
that O
GCN4 B
contains O
two O
activation O
domains O
of O
similar O
potency O
that O
can O
function O
independently O
to O
promote O
high O
- O
level O
transcription O
of O
the O
target O
genes O
HIS3 B
and O
HIS4 B
. O

Those O
diagnosed O
as O
having O
affective O
disorder O
( O
n O
= O
96 O
) O
, O
according O
to O
DSM O
- O
IIIR O
criteria O
, O
were O
compared O
with O
the O
other O
non O
- O
affective O
suicide O
attempters O
( O
n O
= O
161 O
) O
. O

Tax B
was O
demonstrated O
to O
interact O
directly O
with O
CREB1 B
but O
not O
with O
other O
bZIP B
proteins I
, O
including O
CREB2 B
and O
Jun B
. O

Anxiogenic O
effects O
of O
pentagastrin O
in O
patients O
with O
social O
phobia O
and O
healthy O
controls O
. O

Ischaemia O
and O
reperfusion O
injury O
in O
the O
kidney O
: O
current O
status O
and O
future O
direction O
. O

Non O
- O
dialyzable O
transfer B
factor I

We O
also O
demonstrate O
that O
a O
p110RB1 B
mutant I
, O
which O
is O
refractory O
to O
cell O
cycle O
phosphorylation O
but O
intact O
in O
E1a B
/ O
large B
T I
antigen I
- O
binding O
properties O
, O
represses O
EIIaE B
with O
50 O
- O
to O
80 O
- O
fold O
greater O
efficiency O
than O
wild B
- I
type I
p110RB1 I
. O

Subgroup O
analysis O
failed O
to O
show O
any O
benefit O
for O
etoposide O
in O
patients O
with O
monocytic O
or O
myelomonocytic O
disease O
, O
or O
in O
any O
other O
diagnostic O
subgroup O
. O

The O
IGFBP B
- I
1 I
IRS I
and O
PEPCK B
IRS I
both O
bind O
the O
alpha O
and O
beta O
forms O
of O
hepatic B
nuclear I
factor I
3 I
( O
HNF B
- I
3 I
) O
, O
although O
the O
latter O
does O
so O
with O
a O
sixfold O
- O
lower O
relative O
affinity O
. O

Polyglutamine O
- O
containing O
protein O
fragments O
have O
been O
shown O
to O
accumulate O
as O
aggregates O
in O
the O
nucleus O
and O
in O
the O
cytoplasm O
, O
and O
to O
induce O
cell O
death O
when O
expressed O
in O
cultured O
cells O
, O
leading O
to O
the O
proposal O
that O
polyglutamine O
aggregation O
is O
an O
important O
step O
in O
the O
pathogenesis O
. O

It O
is O
suggested O
that O
on O
presentation O
of O
tail O
tip O
necrosis O
in O
kittens O
a O
diagnosis O
of O
neonatal O
isoerythrolysis O
or O
isoagglutination O
should O
be O
considered O
. O

Microscopically O
, O
cartilage O
showed O
markedly O
stunted O
and O
disorganized O
endochondral O
ossification O
. O

Our O
data O
provide O
the O
first O
evidence O
that O
differential O
splicing O
in O
the O
extracellular O
region O
of O
a O
receptor O
gene O
generates O
receptor O
variants O
with O
different O
ligand O
- O
binding O
specificities O
. O

Sucrose O
- O
specific O
signalling O
represses O
translation O
of O
the O
Arabidopsis B
ATB2 I
bZIP I
transcription I
factor I
gene I
. O

Wolf O
- O
Hirschhorn O
syndrome O
( O
WHS O
) O
is O
a O
malformation O
syndrome O
associated O
with O
a O
hemizygous O
deletion O
of O
the O
distal O
short O
arm O
of O
chromosome O
4 O
( O
4p16 O
. O
3 O
) O
. O

Our O
aim O
was O
to O
check O
whether O
, O
during O
PG O
- O
DS O
administration O
, O
serum B
lipase I
activity O
changes O
simultaneously O
with O
serum B
amylase I
activity O
, O
and O
, O
if O
so O
, O
what O
the O
reason O
is O
for O
the O
detected O
change O
. O

A O
long O
- O
term O
follow O
- O
up O
study O
of O
a O
children O
' O
s O
psychiatric O
day O
treatment O
center O
. O

Sequencing O
and O
restriction O
enzyme O
analysis O
demonstrated O
that O
this O
single O
- O
copy O
gene O
contains O
11 O
exons O
and O
spans O
9596 O
base O
pairs O
. O

In O
phase O
II O
studies O
, O
MDL O
72 O
, O
974A O
is O
proving O
to O
be O
a O
useful O
adjunct O
to O
conventional O
therapy O
. O

However O
, O
no O
consensus O
on O
indications O
exists O
and O
, O
according O
to O
the O
literature O
, O
results O
are O
contradictory O
. O

The O
effect O
of O
the O
NMDA B
- I
receptor I
antagonist O
( O
+ O
/ O
- O
) O
- O
CPP O
on O
the O
conditioned O
- O
reflex O
activation O
of O
an O
operant O
reaction O
in O
the O
brain O
electrical O
self O
- O
stimulation O
test O
in O
rats O

Within O
any O
cell O
line O
, O
several O
Tcfs B
and O
TLEs B
are O
co O
- O
expressed O
. O

Constructs O
containing O
fragments O
of O
the O
upstream O
region O
of O
the O
cytotactin B
gene I
fused O
to O
a O
promoterless O
gene O
for O
chloramphenicol B
acetyltransferase I
were O
transiently O
transfected O
into O
chicken O
embryo O
fibroblasts O
to O
define O
functional O
promoter O
sequences O
. O

We O
investigated O
the O
pattern O
of O
evolution O
of O
bronchial B
albumin I
, O
IgA B
, O
and O
IgG B
levels O
in O
ventilated O
ICU O
patients O
in O
relation O
to O
nosocomial O
pneumonia O
. O

Mortality O
among O
patients O
with O
CAVB O
is O
still O
high O
and O
has O
not O
declined O
within O
the O
last O
decade O
. O

Mutations O
away O
from O
splice O
site O
recognition O
sequences O
might O
cis O
- O
modulate O
alternative O
splicing O
of O
goat B
alpha I
s1 I
- I
casein I
transcripts I
. O

SETTING O
: O
A O
private O
office O
- O
based O
fertility O
program O
. O

Promoter O
, O
spliced O
leader O
, O
and O
coding O
sequence O
for O
BICP4 B
, O
the O
largest O
of O
the O
immediate B
- I
early I
proteins I
of O
bovine O
herpesvirus O
1 O
. O

The O
limb O
defects O
, O
oligodactyly O
and O
syndactyly O
, O
have O
been O
traced O
to O
improper O
differentiation O
of O
the O
apical O
ectodermal O
ridge O
( O
AER O
) O
and O
shortening O
of O
the O
anteroposterior O
limb O
axis O
. O

Our O
data O
suggest O
that O
the O
ZF B
- O
HD B
class O
of O
homeodomain B
proteins I
may O
be O
involved O
in O
the O
establishment O
of O
the O
characteristic O
expression O
pattern O
of O
the O
C4 B
PEPCase I
gene I
. O

There O
was O
no O
correlation O
between O
starting O
platelet B
- I
associated I
IgG I
levels O
or O
changes O
in O
platelet B
- I
associated I
IgG I
levels O
with O
therapy O
and O
the O
increment O
in O
the O
patient O
' O
s O
platelet O
count O
. O

We O
investigated O
whether O
two O
WT1 B
splice I
variants I
lacking O
or O
including O
a O
three O
- O
amino O
- O
acid O
( O
KTS O
) O
insertion O
between O
the O
third O
and O
fourth O
zinc O
finger O
in O
the O
DNA O
- O
binding O
domain O
could O
repress O
the O
IR B
promoter I
in O
vitro O
. O

An O
oligomer O
synthesized O
to O
this O
region O
of O
homology O
produced O
two O
DNA O
- O
protein O
complexes O
with O
oviduct O
nuclear O
proteins O
. O

The O
deduced O
amino O
acid O
sequence O
exhibits O
95 O
. O
3 O
and O
76 O
. O
1 O
% O
identity O
with O
the O
CAD B
sequences I
of I
hamster I
and I
Squalus I
acanthias I
. O

In O
contrast O
translocation O
of O
G1 B
to O
the O
Golgi O
was O
not O
observed O
when O
G1 B
was O
coexpressed O
with O
NSm B
, O
although O
NSm B
itself O
was O
still O
detected O
in O
the O
Golgi O
. O

Since O
simian B
virus I
40 I
( I
SV40 I
) I
large I
T I
antigen I
and O
human B
papillomavirus I
type I
16 I
( I
HPV16 I
) I
E7 I
protein I
also O
bind O
host O
regulatory O
factors O
, O
we O
investigated O
whether O
these O
viral O
proteins O
can O
complement O
E1A B
mutants I
which O
are O
defective O
in O
early B
gene I
activation O
. O

We O
further O
showed O
a O
significant O
repression O
of O
promoter O
activity O
( O
> O
40 O
fold O
) O
by O
E2 B
- I
2 I
( O
lacking O
the O
amino O
acid O
sequence O
RSRS O
) O
when O
the O
E47 B
reporter I
construct I
, O
containing O
a O
hexameric O
E O
- O
box O
site O
, O
was O
used O
. O

Similar O
observations O
were O
obtained O
when O
these O
mutated O
versions O
of O
site O
A O
were O
evaluated O
by O
transient O
cotransfection O
assays O
in O
CV1 O
cells O
. O

The O
shear O
bond O
strength O
recorded O
for O
the O
Novabond O
system O
was O
significantly O
lower O
( O
17 O
MPa O
) O
. O

A O
400 O
- O
bp O
fragment O
( O
bp O
- O
339 O
through O
+ O
71 O
) O
of O
the O
CD22 B
promoter I
region I
was O
subcloned O
into O
a O
pGEM O
- O
chloramphenicol B
acetyltransferase I
vector O
and O
after O
transfection O
into O
B O
and O
T O
cells O
was O
found O
to O
be O
active O
in O
both O
B O
and O
T O
cells O
. O

RESULTS O
: O
There O
were O
no O
significant O
differences O
in O
the O
mean O
serum O
vitamin O
A O
, O
E O
concentrations O
and O
vitamin O
E O
/ O
cholesterol O
ratio O
between O
pregnant O
women O
with O
normal O
hemoglobin B
and O
hemoglobinopathies O
, O
while O
confounding O
variables O
that O
might O
affect O
serum O
vitamin O
levels O
i O
. O
e O
. O
maternal O
age O
, O
gravida O
, O
BMI O
, O
gestational O
age O
, O
hematocrit O
, O
hemoglobin B
, O
mean O
corpuscular B
hemoglobin I
concentration O
and O
blood O
group O
were O
not O
different O
. O

Fourteen O
samples O
, O
seven O
each O
of O
IR64 O
and O
IR66 O
were O
studied O
with O
regard O
to O
moisture O
content O
, O
germination O
test O
, O
abnormal O
seedlings O
, O
speed O
of O
germination O
, O
conductance O
of O
leachates O
, O
total O
dehydrogenase B
activity O
, O
total O
free O
amino O
acids O
, O
total O
soluble O
sugar O
, O
fat O
acidity O
, O
gelatinization O
temperature O
, O
gel O
consistency O
, O
amylose O
content O
, O
translucency O
, O
and O
per O
cent O
whiteness O
. O

Electrophoretic O
mobility O
- O
shift O
assays O
between O
viral O
RNA O
and O
BMV B
CP I
peptides I
with O
either O
proline O
or O
alanine O
substitutions O
revealed O
that O
the O
interaction O
is O
nonspecific O
. O

Thus O
, O
over O
a O
wide O
range O
of O
arterial O
dimensions O
, O
results O
obtained O
with O
caliper O
estimates O
of O
luminal O
area O
and O
diameter O
are O
comparable O
to O
those O
obtained O
with O
videodensitometry O
using O
the O
XR O
- O
70 O
system O
. O

CONCLUSIONS O
: O
Neurodevelopmental O
assessment O
at O
1 O
year O
is O
predictive O
of O
school O
performance O
and O
outcome O
in O
the O
adolescent O
period O
. O

We O
reviewed O
the O
different O
published O
cases O
of O
acute O
high O
dose O
methotrexate O
neurotoxicity O
and O
the O
different O
underlying O
mechanisms O
. O

Drug O
therapy O
of O
AAPMC O
is O
directed O
at O
reducing O
the O
amount O
of O
Cl O
. O
difficile O
in O
the O
colon O
and O
promoting O
normalization O
of O
intestinal O
flora O
. O

The O
yield O
of O
endocervical O
cells O
and O
the O
incidence O
of O
dysplasia O
was O
determined O
for O
both O
the O
prenatal O
and O
the O
postpartum O
Pap O
smears O
performed O
for O
this O
group O
of O
patients O
. O

No O
significant O
difference O
in O
plasma O
ET B
levels O
was O
found O
between O
cardiac O
and O
peripheral O
sampling O
sites O
( O
pulmonary O
artery O
; O
1 O
. O
07 O
+ O
/ O
- O
0 O
. O
28 O
, O
right O
atrium O
; O
1 O
. O
02 O
+ O
/ O
- O
0 O
. O
28 O
, O
peripheral O
artery O
; O
1 O
. O
12 O
+ O
/ O
- O
0 O
. O
23 O
, O
peripheral O
vein O
; O
1 O
. O
14 O
+ O
/ O
- O
0 O
. O
38 O
fmol O
/ O
ml O
: O
N O
. O
S O
. O
) O
, O
or O
among O
patients O
with O
uncomplicated O
MI O
, O
unstable O
angina O
( O
1 O
. O
00 O
+ O
/ O
- O
0 O
. O
32 O
fmol O
/ O
ml O
) O
, O
and O
healthy O
subjects O
( O
1 O
. O
01 O
+ O
/ O
- O
0 O
. O
29 O

fmol O
/ O
ml O
) O
. O

Dose O
- O
dependent O
pharmacokinetics O
: O
emphasis O
on O
phase O
I O
metabolism O
. O

Inhibition O
of O
DNA B
topoisomerase I
II I
alpha I
gene I
expression O
by O
the O
p53 B
tumor I
suppressor I
. O

Activation O
of O
HIV O
gene O
expression O
by O
UV O
must O
therefore O
involve O
additional O
cellular O
processes O
, O
such O
as O
those O
triggered O
by O
DNA O
damage O
, O
for O
generation O
of O
a O
full O
gene O
expression O
response O
. O

4 O
. O

Microsporum O
persicolor O
, O
an O
easily O
misinterpreted O
dermatophyt O

We O
propose O
that O
, O
while O
Y O
. O
lipolytica O
CEN O
DNA O
is O
essential O
for O
plasmid O
maintenance O
, O
this O
function O
can O
be O
supplied O
by O
several O
sub O
- O
fragments O
which O
, O
together O
, O
form O
the O
active O
chromosomal O
centromere O
. O

In O
this O
second O
clinical O
paper O
, O
the O
genetic O
features O
of O
the O
case O
of O
microphthalmia O
in O
sheep O
reported O
by O
Wagenaar O
is O
discussed O
in O
greater O
detail O
. O

Our O
study O
reveals O
a O
consequence O
of O
adhesion O
receptor O
- O
mediated O
signaling O
in O
T O
cells O
, O
which O
is O
potentially O
important O
in O
the O
regulation O
of O
T O
cell O
activation O
, O
including O
production O
of O
cytokines O
and O
expression O
of O
proto O
- O
oncogenes O
, O
many O
of O
which O
are O
controlled O
through O
3 O
' O
AU O
- O
rich O
elements O
. O

The O
gap B
protein I
knirps I
mediates O
both O
quenching O
and O
direct O
repression O
in O
the O
Drosophila O
embryo O
. O

The O
family O
histories O
were O
obtained O
from O
the O
parents O
. O

The O
DW O
directions O
uniformly O
sampled O
the O
diffusion O
ellipsoid O
surface O
. O

Here O
we O
demonstrate O
that O
CD40 B
or O
IgM B
receptor I
stimulation O
of O
primary O
B O
cells O
results O
in O
transactivation O
of O
this O
enhancer O
. O

This O
cell O
line O
expresses O
productively O
rearranged I
immunoglobulin B
genes I
as O
well O
as O
the O
Oct B
- I
2 I
gene I
at O
high O
levels O
which O
are O
comparable O
to O
those O
observed O
in O
activated O
murine O
splenic O
B O
cells O
. O

The O
relationship O
between O
CSF B
somatostatin I
and O
peripheral O
thyroid O
hormones O
was O
assessed O
in O
11 O
affectively O
ill O
patients O
before O
and O
during O
carbamazepine O
treatment O
. O

Both O
RNA14 B
and O
RNA15 B
wild I
- I
type I
genes I
, O
when O
cloned O
on O
a O
multicopy O
plasmid O
, O
are O
able O
to O
suppress O
the O
temperature O
- O
sensitive O
phenotype O
of O
strains O
bearing O
either O
the O
rna14 B
or O
the O
rna15 B
mutation I
, O
suggesting O
that O
the O
encoded O
proteins O
could O
interact O
with O
each O
other O
. O

Treatments O
were O
two O
adrenocorticotrophin B
( O
ACTH B
) O
preparations O
given O
intramuscularly O
at O
2 O
. O
2 O
U O
/ O
kg O
, O
one O
of O
the O
ACTH B
preparations O
given O
intramuscularly O
at O
1 O
U O
/ O
kg O
and O
a O
synthetic O
polypeptide O
with O
ACTH B
- O
like O
activity O
( O
tetracosactrin B
, O
cosyntropin B
) O
given O
intravenously O
at O
5 O
micrograms O
/ O
kg O
. O

It O
is O
very O
probable O
that O
this O
change O
was O
due O
to O
mechanical O
effects O
induced O
by O
injecting O
a O
hypertonic O
solution O
of O
MT O
- O
141 O
at O
a O
rate O
of O
70 O
- O
- O
130 O
ml O
/ O
dog O
. O

It O
is O
not O
well O
known O
that O
giant O
cell O
arteritis O
can O
cause O
fatal O
complications O
due O
to O
rupture O
of O
aortic O
aneurysms O
or O
cerebral O
and O
myocardial O
infarctions O
. O

PRIP B
contains O
two O
LXXLL O
signature O
motifs O
. O

Basal O
urinary O
17 O
- O
hydroxycorticoid O
( O
17 O
- O
OHCS O
) O
values O
were O
elevated O
in O
9 O
/ O
12 O
patients O
and O
low O
dose O
dexamethasone O
suppression O
test O
favoured O
Cushing O
' O
s O
syndrome O
in O
8 O
/ O
9 O
patients O
. O

Like O
the O
human O
gene O
, O
the O
Drosophila O
gene O
encodes O
at O
least O
three O
isoforms O
of O
full B
length I
dystrophin I
- I
like I
proteins I
( O
dmDLP1 B
, O
dmDLP2 B
and O
dmDLP3 B
, O
) O
, O
regulated O
by O
different O
promoters O
located O
at O
the O
5 O
' O
end O
of O
the O
gene O
, O
and O
a O
smaller O
product O
regulated O
by O
an O
internal O
promoter O
( O
dmDp186 B
) O
. O

Accordingly O
, O
infection O
of O
IFN B
- O
pretreated O
HeLa O
S3 O
cells O
with O
an O
E3L B
- O
deficient O
VV O
( O
VVDeltaE3L O
) O
resulted O
in O
increased O
phosphorylation O
levels O
of O
both O
PKR B
and O
eIF2alpha B
. O

The O
goal O
of O
our O
work O
was O
to O
determine O
hearing O
thresholds O
in O
patients O
with O
hearing O
impairment O
due O
to O
hereditary O
motor O
and O
sensory O
neuropathy O
( O
HMSN O
I O
) O
. O

Influence O
of O
dietary O
supplementation O
with O
Streptococcus O
faecium O
M O
- O
74 O
on O
broiler O
body O
weight O
, O
feed O
conversion O
, O
carcass O
characteristics O
, O
and O
intestinal O
microbial O
colonization O
. O

The O
results O
reveal O
preferences O
for O
particular O
initiating O
nucleotides O
( O
ATP O
> O
or O
= O
GTP O
> O
UTP O
> O
> O
CTP O
) O
and O
for O
starting O
positions O
downstream O
of O
the O
Pribnow O
box O
( O
7 O
> O
> O
6 O
and O
8 O
> O
9 O
> O
10 O
) O
. O

Trimethylcolchicinic O
acid O
methyl O
ether O
d O
- O
tartrate O
( O
TMCA O
; O
NSC O
- O
36351 O
) O
was O
administered O
daily O
by O
mouth O
to O
71 O
patients O
with O
malignant O
lymphomas O
. O

Gene O
transcripts O
are O
detected O
on O
tissues O
such O
as O
bovine O
liver O
, O
kidney O
, O
lung O
, O
and O
brain O
. O

Assessment O
of O
ammonia O
and O
urea O
blood O
content O
in O
metabolic O
acid O
- O
base O
disorders O
in O
white O
rats O
. O

The O
immediate O
and O
remote O
results O
of O
surgical O
treatment O
of O
renal O
adenocarcinomas O
. O

Vibrio O
cholerae O
iron O
transport O
: O
haem B
transport I
genes O
are O
linked O
to O
one O
of O
two O
sets O
of O
tonB B
, O
exbB B
, O
exbD B
genes I
. O

The O
predicted O
amino O
- O
acid O
sequence O
includes O
an O
N O
- O
terminal O
signal O
sequence O
of O
27 O
amino O
acids O
, O
a O
27 O
amino O
- O
acid O
pro O
- O
region O
, O
a O
251 O
amino O
- O
acid O
catalytic O
domain O
typical O
of O
a O
serine B
protease I
with O
trypsin B
- I
like I
specificity O
, O
and O
a O
C O
- O
terminal O
hydrophobic O
extension O
which O
is O
predicted O
to O
function O
as O
a O
membrane O
anchor O
. O

To O
examine O
the O
biochemical O
mechanism O
of O
Tax1 B
transactivation O
, O
we O
have O
developed O
an O
in O
vitro O
transactivation O
assay O
in O
which O
wild B
- I
type I
Tax1 I
is O
able O
to O
specifically O
transactivate O
a O
polymerase B
II I
promoter I
through O
upstream O
Tax1 B
- I
responsive I
elements I
. O

Interestingly O
, O
PMA O
- O
, O
PDGF B
- O
, O
and O
serum O
- O
induced O
GHR B
proteolysis O
was O
associated O
with O
substantial O
decreases O
in O
GH B
- O
induced O
activation O
of O
Janus B
kinase I
- I
2 I
, O
which O
were O
also O
prevented O
by O
IC3 O
. O

Thus O
, O
IF O
- O
laser O
therapy O
has O
a O
certain O
antioxidative O
effect O
by O
increasing O
SOD B
activity O
in O
RA O
patients O
' O
blood O
cells O
and O
reducing O
the O
production O
of O
highly O
reactive O
oxygen O
and O
nitrogen O
forms O
. O

Reactive O
mesothelial O
cells O
, O
usually O
seen O
as O
exfoliated O
cells O
, O
were O
consistently O
strongly O
positive O
for O
alpha B
1 I
- I
antichymotrypsin I
and O
more O
variably O
for O
alpha B
1 I
- I
antitrypsin I
and O
lysozyme B
. O

Recombinant O
propeptides O
containing O
mutations O
of O
one O
of O
the O
three O
tryptophan O
residues O
were O
three O
orders O
of O
magnitude O
less O
effective O
as O
inhibitors O
of O
mature B
cathepsin I
S I
than O
the O
wild O
- O
type O
propeptide O
. O

The O
highest O
incidence O
was O
observed O
for O
V O
. O
alginolyticus O
( O
92 O
- O
100 O
% O
) O
, O
followed O
by O
V O
. O
parahaemolyticus O
( O
67 O
- O
92 O
% O
) O
, O
V O
. O
fluvialis O
( O
34 O
- O
67 O
% O
) O
, O
V O
. O
vulnificus O
( O
8 O
- O
17 O
% O
) O
, O
V O
. O
furnissii O
( O
0 O
- O
17 O
% O
) O
, O
V O
. O
mimicus O
( O
0 O
- O
17 O
% O
) O
and O
V O
. O
cholerae O
non O
- O
O1 O
( O
0 O
- O
8 O
% O
) O
. O

Acute O
morphine O
treatment O
did O
not O
increase O
the O
locomotor O
activity O
of O
mice O
withdrawn O
for O
1 O
day O
, O
after O
withdrawal O
for O
3 O
days O
the O
increase O
was O
similar O
to O
that O
in O
controls O
, O
and O
after O
5 O
days O
the O
increase O
was O
clearly O
larger O
than O
in O
controls O
. O

In O
patients O
with O
active O
TB O
, O
cytokines O
that O
were O
elevated O
in O
serum O
were O
IFN B
- I
gamma I
, O
IL B
- I
6 I
and O
IL B
- I
10 I
. O

Isolation O
and O
properties O
of O
Drosophila B
melanogaster I
ferritin I
- O
- O
molecular O
cloning O
of O
a O
cDNA O
that O
encodes O
one O
subunit O
, O
and O
localization O
of O
the O
gene O
on O
the O
third O
chromosome O
. O

We O
show O
that O
Fos B
down O
regulates O
several O
immediate B
- I
early I
genes I
( O
c B
- I
fos I
, O
Egr B
- I
1 I
, O
and O
Egr B
- I
2 I
) O
after O
mitogenic O
stimulation O
. O

This O
study O
demonstrates O
a O
novel O
link O
between O
alterations O
in O
platelet B
- I
derived I
growth I
factor I
( O
PDGF B
) O
regulation O
of O
ornithine B
decarboxylase I
( O
ODC B
) O
expression O
during O
malignant O
conversion O
. O

Circulating O
immune O
complexes O
were O
determined O
by O
the O
method O
of O
phagocytosis O
and O
immunofluorescence O
. O

The O
human B
IFI16 I
gene I
is O
a O
member O
of O
an O
interferon B
- I
inducible I
family O
of O
mouse O
and O
human O
genes O
closely O
linked O
on O
syntenic O
regions O
of O
chromosome O
1 O
. O

In O
agreement O
with O
this O
model O
, O
elimination O
of O
the O
ADAM13 B
cytoplasmic I
domain I
increased O
developmental O
alterations O
attributable O
to O
ADAM13 B
proteolytic O
activity O
. O

Diabetic O
osteopathy O
. O

A O
third O
GEF B
, O
GRP3 B
( O
KIAA0846 B
) O
, O
activated O
both O
Ras B
and O
Rap1 B
and O
shared O
significant O
sequence O
homology O
with O
the O
calcium O
- O
and I
diacylglycerol I
- I
activated I
GEFs I
, O
GRP1 B
and O
GRP2 B
. O

A O
new O
simple O
identification O
of O
rheumatoid B
factor I
on O
nitrocellulose O
was O
developed O
that O
allows O
quantitative O
detection O
. O

Comparisons O
of O
defective O
HTLV O
- O
I O
proviruses O
predict O
the O
mode O
of O
origin O
and O
coding O
potential O
of O
internally O
deleted O
genomes O
. O

Reversal O
of O
biliary O
sphincter O
spasm O
with O
low O
dose O
glucagon B
during O
operative O
cholangiography O
. O

Previous O
studies O
have O
shown O
that O
the O
IME1 B
gene I
is O
required O
for O
sporulation O
and O
the O
expression O
of O
meiosis O
specific O
genes O
in O
Saccharomyces O
cerevisiae O
. O

A O
case O
comparison O
of O
acquired O
immune O
deficiency O
syndrome O
( O
AIDS O
) O
in O
homosexual O
males O
with O
spindle O
- O
endothelial O
cell O
abnormalities O
and O
with O
recrudescent O
melioidosis O
. O

Intrathecal O
injections O
of O
small O
volumes O
of O
the O
alpha B
2 I
- I
adrenoceptor I
agonists O
, O
xylazine O
and O
clonidine O
, O
into O
the O
cervical O
region O
of O
the O
spinal O
cord O
of O
conscious O
unrestrained O
sheep O
produced O
a O
dose O
- O
dependent O
analgesia O
of O
the O
forelimbs O
as O
measured O
using O
a O
mechanical O
pressure O
device O
. O

Second O
, O
CGGA O
- O
containing O
sequences O
placed O
at O
- O
88 O
in O
the O
delta O
MTV O
- O
CAT B
reporter O
plasmid O
conferred O
insulin B
responsiveness O
to O
the O
mammary B
tumor I
virus I
promoter I
. O

In O
the O
absence O
of O
c B
- I
fos I
gene I
expression O
, O
c B
- I
Fos I
protein I
appears O
to O
be O
replaced O
by O
proteins O
of O
Fos B
family I
( O
Fra B
- I
1 I
) O
and O
ATF B
family I
( O
ATF B
- I
2 I
and O
ATFa B
) O
. O

The O
dopamine B
D1 I
dopamine I
receptor I
antagonist O
SCH O
23390 O
dose O
dependently O
( O
7 O
. O
5 O
- O
30 O
micrograms O
/ O
kg O
s O
. O
c O
. O
) O
antagonized O
the O
stimulant O
locomotor O
effect O
on O
both O
drugs O
but O
did O
not O
prevent O
their O
antiimmobility O
effect O
on O
the O
behavioral O
despair O
test O
. O

The O
results O
obtained O
show O
that O
expression O
of O
the O
gene O
is O
low O
at O
23 O
degrees O
C O
and O
is O
induced O
rapidly O
at O
37 O
degrees O
C O
. O

Characterization O
of O
Chlamydomonas O
reinhardtii O
zygote O
- O
specific O
cDNAs O
that O
encode O
novel O
proteins O
containing O
ankyrin B
repeats O
and O
WW B
domains O
. O

Adenomas O
in O
the O
upper O
gastrointestinal O
tract O
were O
found O
in O
all O
of O
the O
patients O
: O
Vater O
' O
s O
papilla O
- O
- O
12 O
, O
duodenum O
- O
- O
nine O
, O
and O
gastric O
antrum O
- O
- O
seven O
. O

Microenvironments O
dominated O
by O
cyanobacteria O
( O
BPC O
) O
had O
a O
higher O
pH O
( O
pH O
7 O
- O
8 O
) O
than O
those O
dominated O
by O
lichen O
( O
LTL O
) O
( O
pH O
4 O
. O
5 O
- O
5 O
. O
5 O
) O
. O

A O
total O
of O
64 O
uncomplicated O
Type O
- O
2 O
diabetic O
individuals O
of O
higher O
middle O
class O
to O
rich O
socio O
- O
economic O
status O
were O
studied O
. O

A O
drop O
of O
methylprednisolone O
acetate O
or O
vehicle O
constituent O
was O
placed O
on O
the O
dissected O
plantar O
nerve O
proximal O
to O
the O
stimulating O
electrodes O
after O
recording O
control O
responses O
( O
A O
- O
fibre O
volley O
in O
the O
sciatic O
nerve O
and O
C O
- O
fibre O
evoked O
reflex O
discharge O
in O
flexor O
motoneurons O
) O
. O

Basic B
helix I
- I
loop I
- I
helix I
proteins I
can O
act O
at O
the O
E O
- O
box O
within O
the O
serum O
response O
element O
of O
the O
c B
- I
fos I
promoter I
to O
influence O
hormone O
- O
induced O
promoter O
activation O
in O
Sertoli O
cells O
. O

Using O
yeast O
two O
- O
hybrid O
screening O
, O
we O
isolated O
histone B
H3 I
as O
an O
interacting O
factor O
of O
CIA B
. O

Concentrations O
of O
Co O
, O
Cu O
, O
Fe O
, O
Hg O
, O
Mn O
, O
Sb O
, O
Se O
and O
Zn O
in O
IAEA O
milk O
( O
dry O
) O
standard O
A O
- O
11 O
were O
re O
- O
evaluated O
with O
the O
help O
of O
instrumental O
and O
radiochemical O
neutron O
activation O
analysis O
( O
NAA O
) O
. O

Gallstone O
- O
formation O
has O
traditionally O
been O
attributed O
to O
supersaturation O
of O
bile O
with O
cholesterol O
. O

Sixteen O
patients O
on O
maintenance O
hemodialysis O
underwent O
dialysis O
with O
either O
cuprophane O
( O
n O
= O
8 O
) O
or O
polymethylmethacrylate O
( O
PMMA O
; O
n O
= O
8 O
) O
membranes O
for O
1 O
week O
and O
then O
switched O
to O
the O
opposite O
membrane O
during O
the O
second O
week O
. O

Gene O
expression O
is O
in O
part O
controlled O
by O
chromatin O
remodeling O
factors O
and O
the O
acetylation O
state O
of O
nucleosomal O
histones I
. O

Each O
patient O
was O
seen O
for O
5 O
to O
13 O
postsurgical O
maintenance O
visits O
. O

Furthermore O
, O
Grap B
is O
associated O
with O
a O
Ras B
guanine I
nucleotide I
exchange I
factor I
mSos1 B
, O
primarily O
through O
its O
N O
- I
terminal O
SH3 B
domain I
. O

Recurrent O
infections O
, O
episodic O
lymphopenia O
and O
impaired O
cellular O
immunity O
. O

After O
3 O
months O
of O
intervention O
, O
sustained O
physical O
training O
was O
associated O
with O
the O
decrease O
of O
FVII B
and O
PAI B
- I
1 I
levels O
. O

Thirty O
- O
three O
percent O
had O
elevated O
titers O
( O
above O
1 O
: O
20 O
dilution O
) O
. O

Xa4 B
is O
a O
dominantly O
inherited O
rice O
gene O
that O
confers O
resistance O
to O
Philippine O
race O
1 O
of O
the O
bacterial O
blight O
pathogen O
Xanthomonas O
oryzae O
pv O
. O
oryzae O
in O
rice O
. O

In O
uraemic O
patients O
, O
similar O
maximum O
serum O
concentrations O
were O
found O
after O
a O
single O
7 O
. O
5 O
mg O
/ O
kg O
iv O
dose O
. O

The O
effect O
of O
buthionine O
sulfoximine O
( O
BSO O
) O
and O
disulfiram O
( O
DSF O
) O
on O
the O
urotoxicity O
induced O
by O
cyclophosphamide O
( O
CPA O
) O
was O
examined O
in O
mice O
. O

All O
the O
previously O
mapped O
structural O
protein O
- O
encoding O
genes O
of O
phage O
LL O
- O
H O
were O
included O
in O
the O
sequence O
. O

When O
a O
longer O
- O
714 O
to O
+ O
78 O
fragment O
of O
the O
PBGD B
promoter I
is O
used O
, O
the O
- O
70 O
GATA B
mutant I
still O
displays O
erythroid O
- O
specific O
activity O
and O
is O
cis O
- O
activated O
by O
the O
5 B
' I
HS I
- I
2 I
enhancer I
, O
while O
the O
- O
100 O
CCACC O
mutant O
is O
completely O
inactive O
in O
the O
absence O
or O
in O
the O
presence O
of O
the O
5 B
' I
HS I
- I
2 I
enhancer I
. O

Surprisingly O
, O
in O
adult O
animals O
, O
the O
suprabasal O
expression O
of O
dnRXR B
alpha I
significantly O
reduced O
the O
ability O
of O
topically O
applied O
tRA O
to O
stimulate O
proliferation O
of O
undifferentiated O
keratinocytes O
in O
the O
basal O
layer O
of O
epidermis O
. O

In O
many O
other O
contractile B
protein I
gene I
families O
, O
genes O
encoding O
cardiac O
isoforms O
are O
expressed O
early O
in O
skeletal O
muscle O
development O
and O
are O
later O
repressed O
. O

1995 O
. O

For O
il8ra B
, O
it O
is O
formed O
from O
two O
exons O
, O
whereas O
for O
il8rb B
, O
seven O
distinct O
neutrophil O
mRNAs O
are O
formed O
by O
alternative O
splicing O
of O
11 O
exons O
. O

Endocarditis O
remains O
a O
life O
- O
threatening O
disease O
with O
substantial O
morbidity O
and O
mortality O
. O

The O
gene O
encoding O
the O
cellulase B
( O
Avicelase I
) O
Cel1 B
from I
Streptomyces I
reticuli I
and O
analysis O
of O
protein O
domains O
. O

From O
this O
comparison O
of O
aa O
sequences O
, O
the O
ATPK7 B
protein I
is O
considered O
to O
be O
a O
member O
of O
a O
novel O
subfamily O
of O
Ser B
/ I
Thr I
PKs I
in O
plants O
. O

Of O
these O
, O
23 O
( O
13 O
. O
6 O
% O
) O
were O
due O
to O
GIB O
( O
Upper O
GIB O
= O
19 O
, O
Lower O
GIB O
= O
4 O
) O
. O

The O
E O
. O
multilocularis O
/ O
Mongolian O
gerbil O
system O
can O
replace O
the O
natural O
canid O
hosts O
as O
a O
new O
way O
to O
obtain O
infective O
eggs O
and O
to O
analyze O
host O
- O
parasite O
interactions O
. O

E1 B
recognition I
sequences I
in O
the O
bovine O
papillomavirus O
type O
1 O
origin O
of O
DNA O
replication O
: O
interaction O
between O
half O
sites O
of O
the O
inverted O
repeats O
. O

We O
found O
that O
secretion O
of O
chitinolytic O
enzymes O
by O
cultured O
L O
. O
major O
parasites O
is O
inhibited O
by O
blood O
or O
hemoglobin B
, O
and O
hence O
these O
enzymes O
are O
apparently O
absent O
from O
the O
blood O
- O
fed O
infected O
flies O
, O
where O
the O
cardiac O
valve O
appears O
undamaged O
. O

Patients O
in O
the O
mucinous O
cyst O
group O
had O
significantly O
lower O
CA B
125 I
cystic O
fluid O
levels O
compared O
with O
women O
with O
endometriomas O
and O
dermoids O
( O
P O
& O
lt O
; O
0 O
. O
05 O
) O
. O

SATB1 B
interacted O
with O
CDP B
through O
its O
DNA O
- O
binding O
domain O
, O
as O
demonstrated O
by O
glutathione B
S I
- I
transferase I
( O
GST B
) O
pull O
- O
down O
assays O
. O

Plasma B
prolactin I
concentrations O
in O
pinealectomized O
ewes O
receiving O
melatonin O
treatment O
and O
in O
pineal O
intact O
ewes O
maintained O
under O
a O
non O
- O
24 O
- O
hour O
photoperiod O
. O

It O
is O
targeted O
to O
peroxisomes O
when O
expressed O
in O
mammalian O
cells O
and O
yeast O
. O

The O
transcriptional O
start O
site O
, O
identified O
by O
primer O
extension O
and O
S1 B
nuclease I
assay O
, O
is O
34 O
nt O
upstream O
of O
the O
translation O
initiation O
codon O
. O

Nonlinearities O
in O
cochlear O
hydrodynamics O
. O

Electronic O
structure O
and O
optical O
properties O
of O
the O
B12O2 O
crystal O
. O

Spongiosa O
regeneration O
in O
the O
millipore O
chamber O

The O
predicted O
5 O
' O
and O
3 O
' O
ends O
of O
the O
transcript O
are O
in O
very O
good O
agreement O
with O
the O
previously O
determined O
size O
of O
the O
LEU3 B
message I
. O

This O
cDNA O
corresponded O
to O
FGF B
- I
binding I
protein I
( O
FGF B
- I
BP I
) O
, O
a O
secreted O
protein O
previously O
shown O
to O
bind O
acidic B
and I
basic I
FGF I
and O
to O
modulate O
their O
activities O
. O

Analysis O
of O
replicative O
intermediates O
shows O
that O
plasmid O
YRp7 O
, O
which O
contains O
the O
chromosomal O
replicator O
ARS1 B
, O
initiates O
bidirectional O
replication O
in O
a O
100 O
bp O
region O
within O
the O
sequence O
required O
for O
autonomous O
replication O
in O
vivo O
. O

Such O
results O
suggest O
that O
there O
are O
no O
differences O
in O
the O
BMR O
of O
age O
- O
and O
weight O
- O
matched O
Asian O
Indian O
males O
, O
other O
tropical O
populations O
and O
Americans O
. O

Rewarding O
properties O
of O
methylphenidate O
: O
sensitization O
by O
prior O
exposure O
to O
the O
drug O
and O
effects O
of O
dopamine B
D1 I
- I
and I
D2 I
- I
receptor I
antagonists O
. O

Similar O
to O
what O
was O
observed O
for O
P O
. O
putida O
, O
a O
psrA B
null I
mutant I
of O
P O
. O
aeruginosa O
also O
showed O
a O
90 O
% O
reduction O
in O
rpoS B
promoter I
activity O
; O
both O
mutants O
could O
be O
complemented O
for O
rpoS B
promoter I
activity O
when O
the O
psrA B
gene I
was O
provided O
in O
trans O
. O

psrA B
mutants I
of O
both O
Pseudomonas O
species O
lost O
the O
ability O
to O
induce O
rpoS B
expression O
at O
stationary O
phase O
, O
but O
they O
retained O
the O
ability O
to O
produce O
quorum O
- O
sensing O
autoinducer O
molecules O
. O

The O
role O
of O
the O
putative O
C2 B
domain O
of O
Nedd4 B
has O
not O
been O
elucidated O
. O

ZF87 B
specifically O
binds O
the O
ME1a1 B
element I
with O
higher O
affinity O
than O
the O
ME1a2 B
element I
. O

We O
found O
a O
significant O
increase O
of O
thromboxane O
as O
evidenced O
by O
the O
major O
urinary O
metabolite O
2 O
, O
3 O
- O
dinorthromboxane O
B2 O
in O
the O
oestrogen O
group O
as O
compared O
to O
the O
orchidectomy O
group O
. O

Multivariable O
logistic O
regression O
showed O
that O
mean O
blood O
glucose O
level O
for O
the O
first O
2 O
days O
( O
p O
= O
0 O
. O
002 O
) O
, O
obesity O
( O
p O
< O
0 O
. O
002 O
) O
, O
and O
use O
of O
the O
internal O
mammary O
artery O
( O
p O
< O
0 O
. O
02 O
) O
were O
all O
independent O
predictors O
of O
deep O
wound O
infection O
. O

However O
the O
precise O
function O
of O
DNA B
- I
PK I
in O
DNA O
double O
- O
strand O
break O
repair O
is O
not O
known O
. O

This O
new O
receptor O
, O
TOR B
( O
thymus B
orphan I
receptor I
) O
, O
is O
most O
closely O
related O
in O
both O
its O
DNA O
- O
binding O
domain O
and O
ligand O
- O
binding O
domain O
, O
90 O
% O
and O
53 O
% O
, O
respectively O
, O
to O
ROR B
alpha I
/ O
RZR B
alpha I
and O
clusters O
with O
these O
two O
receptors O
and O
RZR B
beta I
in O
a O
phylogenetic O
tree O
, O
when O
both O
the O
DNA O
- O
binding O
domain O
and O
the O
ligand O
- O
binding O
domain O
sequences O
of O
nuclear O
receptors O
are O
compared O
. O

In O
this O
report O
, O
we O
have O
examined O
whether O
PTP1B B
effects O
transformation O
induced O
by O
p210 B
bcr B
- O
abl B
. O

S1 B
nuclease I
analysis O
of O
the O
RNA O
from O
a O
number O
of O
QT6 O
- O
LD O
clones O
gave O
similar O
results O
, O
indicating O
that O
the O
LD O
population O
was O
composed O
of O
viruses O
with O
similar O
but O
not O
identical O
deletion O
endpoints O
. O

Association O
of O
Smads B
with O
lymphoid B
enhancer I
binding I
factor I
1 I
/ O
T B
cell I
- I
specific I
factor I
mediates O
cooperative O
signaling O
by O
the O
transforming B
growth I
factor I
- I
beta I
and O
wnt B
pathways O
. O

The O
three O
OAS B
genes I
are O
flanked O
by O
markers O
WI B
- I
10614 I
( O
cen B
) O
and O
D12S2293 B
( O
tel B
) O
and O
are O
contained O
within O
three O
sets O
of O
overlapping O
cosmid O
clones O
. O

22 O
. O
6 O
% O
of O
patients O
were O
found O
to O
be O
positive O
at O
histology O
in O
the O
corpus O
mucosa O
; O
all O
but O
one O
of O
these O
patients O
had O
elevated O
circulating O
IgG B
to O
H O
. O
pylori O
( O
Group O
C O
) O
. O

Rottlerin O
also O
inhibited O
the O
activation O
of O
MAP B
kinase I
kinase I
( O
MEK B
) O
in O
response O
to O
activated O
Raf B
, O
but O
had O
no O
effect O
upon O
c B
- I
Raf I
activity O
or O
ERK B
activation O
by O
activated O
MEK B
. O

Quasidiatomic O
study O
of O
Ly O
- O
alpha O
- O
producing O
H2 O
+ O
- O
Ne O
collisions O
at O
keV O
energies O
. O

The O
NH O
( O
2 O
) O
- O
terminus O
of O
all O
three O
proteins O
contains O
a O
POZ O
domain O
, O
a O
conserved O
120 O
amino O
acid O
motif O
involved O
in O
transcriptional O
repression O
and O
protein O
dimerization O
. O

The O
isolation O
of O
crt5 B
- I
262 I
, O
an O
additional O
cdc B
allele I
of O
POL1 B
/ O
CDC17 B
, O
suggests O
for O
the O
first O
time O
that O
directly O
blocking O
DNA O
replication O
can O
provide O
a O
signal O
to O
induce O
the O
DNA O
damage O
response O
. O

crt2 B
mutants I
show O
a O
defect O
in O
basal O
level O
expression O
of O
RNR1 B
- O
lacZ B
reporter O
constructs O
. O

Greer O
, O
and O
L O
. O
A O
. O

We O
prospectively O
studied O
serum B
prolactin I
( O
PRL B
) O
elevation O
after O
different O
types O
of O
documented O
seizures O
in O
17 O
patients O
. O

However O
, O
both O
peptides O
1 O
- O
49 O
and O
63 O
- O
87 O
bound O
to O
PDE B
alpha I
/ I
beta I
in O
a O
solid O
- O
phase O
binding O
assay O
. O

Values O
of O
K1 O
and O
Vd O
were O
significantly O
increased O
in O
the O
tumour O
tissue O
. O

The O
mutation O
also O
preferentially O
limits O
( O
compared O
with O
total O
protein O
synthesis O
) O
translation O
of O
an O
induced O
gene O
that O
depends O
on O
five O
AGA O
codons O
, O
i O
. O
e O
. O
, O
the O
lambda B
cI I
repressor I
gene I
. O

Five O
RNA O
transcripts O
of O
about O
1 O
. O
2 O
to O
1 O
. O
7 O
kilobases O
were O
mapped O
to O
a O
part O
of O
the O
genome O
of O
insect O
iridescent O
virus O
type O
6 O
( O
Chilo O
iridescent O
virus O
; O
CIV O
) O
between O
genome O
coordinates O
0 O
. O
832 O
and O
0 O
. O
856 O
within O
the O
EcoRI B
DNA I
fragment I
F I
. O

Other O
similar O
patients O
must O
be O
found O
before O
it O
is O
established O
that O
the O
colloid O
cyst O
is O
part O
of O
the O
nevoid O
basal O
cell O
carcinoma O
syndrome O
. O

Portions O
of O
the O
3 O
' O
- O
coding O
and O
3 O
' O
- O
untranslated O
regions O
were O
found O
to O
be O
missing O
from O
the O
7 O
. O
2 O
- O
and O
4 B
. I
8 I
- I
kb I
topo I
II I
alpha I
mRNAs I
by O
Northern O
blot O
analysis O
. O

In O
addition O
, O
a O
double O
mutant O
( O
E482A O
, O
D489A O
) O
which O
removed O
negative O
charges O
along O
one O
side O
of O
the O
helix O
had O
negligible O
effects O
on O
fusion O
activity O
. O

A O
GTP O
/ O
GDP O
exchange O
assay O
was O
used O
to O
assess O
the O
potential O
role O
of O
Ras B
in O
the O
pathway O
leading O
to O
ERK B
phosphorylation O
; O
DADLE O
failed O
to O
stimulate O
GTP O
/ O
GDP O
exchange O
in O
comparison O
to O
PMA O
. O

Phenotypic O
analysis O
demonstrates O
that O
trio B
and O
Abl B
cooperate O
in O
regulating O
axon O
outgrowth O
in O
the O
embryonic O
central O
nervous O
system O
( O
CNS O
) O
. O

Recombinant B
caspases I
are O
typically O
produced O
in O
Escherichia O
coli O
expression O
systems O
with O
the O
attendant O
problems O
of O
solubilization O
, O
re O
- O
folding O
and O
activation O
of O
the O
protease O
. O

The O
gene B
encoding I
rat I
peptidylglycine I
alpha I
- I
amidating I
monooxygenase I
( O
PAM B
) O
contains O
26 O
protein O
- O
coding O
exons O
. O

This O
bound O
[ O
3H O
] O
iloprost B
with O
high O
affinity O
and O
stimulated O
cAMP O
production O
when O
exposed O
to O
agonist O
. O

Structure O
of O
the O
connective O
stroma O
of O
the O
epididymis O
in O
the O
zebu O

Free O
protein B
S I
deficiency O
in O
acute O
ischemic O
stroke O
. O

In O
a O
series O
of O
papers O
, O
the O
staging O
system O
for O
melanoma O
was O
critically O
analyzed O
, O
and O
many O
shortcomings O
were O
identified O
. O

Reproduction O
and O
maternal O
response O
of O
the O
rat O
when O
thiamine O
intake O
is O
limited O
. O

Total O
meso O
- O
pore O
volume O
and O
surface O
area O
ranged O
from O
0 O
. O
004 O
- O
0 O
. O
08 O
cm3 O
g O
( O
- O
1 O
) O
and O
from O
0 O
. O
33 O
- O
6 O
. O
9 O
m2 O
g O
( O
- O
1 O
) O
respectively O
, O
accounting O
for O
up O
33 O
% O
of O
the O
BET O
surface O
area O
. O

The O
NF B
- I
kappa I
B I
precursor I
p100 B
( O
lyt B
- I
10 I
, O
p97 B
, O
p98 B
) O
generates O
after O
proteolytic O
processing O
a O
52 O
kDa O
subunit O
, O
which O
can O
bind O
to O
kappa B
B I
- I
motifs I
. O

CONCLUSION O
: O
The O
biodistribution O
of O
111In O
IgG B
is O
similar O
to O
that O
of O
99mTc O
- O
HMPAO O
- O
labeled O
leukocytes O
. O

Based O
on O
29 O
determinations O
of O
the O
glucose O
from O
sinigrin O
, O
analyzed O
under O
different O
conditions O
, O
accuracy O
of O
the O
total O
glucosinolate O
determination O
was O
94 O
. O
8 O
+ O
/ O
- O
7 O
. O
3 O
% O
. O

Receptor B
- I
type I
serine I
/ I
threonine I
kinases I
( O
RSKs B
) O
have O
been O
organized O
into O
two O
distinct O
classes O
known O
as O
types O
I O
and O
II O
on O
the O
basis O
of O
sequence O
similarity O
. O

In O
a O
previous O
study O
of O
transforming B
growth I
factor I
- I
beta1 I
- O
mediated O
inhibition O
of O
the O
cyclin B
A I
promoter I
, O
we O
mapped O
the O
inhibitory O
effect O
to O
the O
ATF B
site I
; O
in O
the O
present O
study O
of O
IFN B
- I
gamma I
treatment O
, O
functional O
analysis O
by O
transient O
transfection O
showed O
that O
inhibition O
of O
the O
cyclin B
A I
promoter I
persisted O
despite O
mutation O
of O
the O
ATF B
, O
NF B
- I
Y I
, O
or O
CDE O
elements I
. O

The O
RsmA B
- I
mutant I
, O
like O
its O
parent O
, O
produces O
N O
- O
( O
3 O
- O
oxohexanoyl O
) O
- O
L O
- O
homoserine O
lactone O
( O
HSL O
) O
, O
a O
starvation O
/ O
cell O
density O
- O
sensing O
signal O
required O
for O
extracellular O
enzyme O
production O
. O

The O
inactive O
X O
allele O
, O
however O
, O
is O
hypermethylated O
in O
leukocytes O
, O
presumably O
reflecting O
early O
X O
inactivation O
events O
that O
become O
important O
for O
gene O
dosage O
in O
expressing O
lineages O
. O

Intensive O
therapy O
of O
viral O
hepatitis O

Also O
, O
the O
RVW O
hypertrophy O
, O
the O
IVS O
hypertrophy O
, O
and O
the O
RV O
high O
pressure O
load O
to O
the O
LV O
through O
the O
IVS O
may O
be O
related O
to O
the O
small O
LV O
, O
high O
EF O
, O
and O
abnormal O
two O
chamber O
inflow O
in O
the O
PS O
group O
before O
BV O
. O

Analysis O
of O
the O
DNA O
- O
binding O
and O
transcriptional O
activation O
functions O
of O
human B
Fli I
- I
1 I
protein I
. O

This O
pattern O
of O
expression O
and O
differential O
processing O
suggests O
a O
role O
for O
inrpk1 B
in O
some O
aspect O
of O
SD O
photoperiodic O
- O
induced O
flowering O
in O
morning O
glory O
. O

Progressive O
immunodeficiency O
in O
a O
patient O
with O
IgA B
deficiency O

RMR O
was O
positively O
correlated O
with O
TEF O
( O
r O
= O
0 O
. O
613 O
, O
p O
< O
0 O
. O
01 O
) O
and O
net O
TEF O
( O
r O
= O
0 O
. O
648 O
, O
p O
< O
0 O
. O
005 O
) O
in O
the O
luteal O
but O
not O
the O
follicular O
phase O
. O

ESR O
spectra O
of O
Er3 O
+ O
in O
SmB6 O
single O
crystals O
: O
Dynamic O
Jahn O
- O
Teller O
effect O
in O
a O
mixed O
- O
valence O
compound O
. O

The O
N O
- O
terminal O
exon1 O
had O
no O
homology O
at O
the O
amino O
acid O
level O
with O
NGFI B
- I
B I
, O
the O
mammalian O
homologue O
. O

This O
paper O
describes O
a O
longitudinal O
study O
in O
which O
clinical O
parameters O
and O
aspartate B
aminotransferase I
( O
AST B
) O
in O
gingival O
crevicular O
fluid O
( O
GCF O
) O
were O
monitored O
bimonthly O
over O
a O
6 O
- O
12 O
months O
period O
in O
970 O
sites O
from O
7 O
treated O
periodontitis O
patients O
. O

We O
conclude O
from O
these O
observations O
that O
the O
cTnC O
3 O
' O
Ile O
element O
is O
a O
composite O
enhancer O
that O
functions O
through O
the O
combined O
interactions O
of O
at O
least O
five O
regulatory O
elements O
and O
their O
cognate O
binding O
factors O
: O
three O
or O
four O
E O
- O
boxes O
, O
a O
MEF B
- I
2 I
site I
, O
and O
a O
MEF B
- I
3 I
site I
. O

The O
analysis O
consisted O
of O
1 O
) O
fitting O
first O
- O
and O
second O
- O
order O
autoregressive O
models O
( O
AR1 O
and O
AR2 O
) O
and O
2 O
) O
obtaining O
the O
power O
spectra O
of O
the O
data O
by O
fast O
- O
Fourier O
transform O
. O

We O
identified O
Tyr O
( O
112 O
) O
and O
Leu O
( O
175 O
) O
within O
the O
RNA O
- O
binding O
domain O
of O
the O
U1 B
70K I
protein I
to O
be O
cross O
- O
linked O
to O
G O
( O
28 O
) O
and O
U O
( O
30 O
) O
in O
stem O
- O
loop O
I O
, O
respectively O
. O

Kudzu O
extract O
shows O
potential O
for O
moderating O
alcohol O
abuse O
. O

Furthermore O
, O
the O
data O
suggest O
an O
immunological O
non O
- O
responsiveness O
to O
enterotoxin B
A I
in O
a O
considerable O
portion O
of O
the O
patients O
. O

BCV O
mRNAs O
5 O
and O
5 O
- O
1 O
appear O
to O
be O
used O
for O
the O
synthesis O
of O
the O
12 O
. O
7 O
- O
and O
9 O
. O
5 O
- O
kDa O
proteins O
, O
respectively O
, O
which O
demonstrates O
a O
pattern O
of O
expression O
strikingly O
different O
from O
that O
utilized O
by O
MHV O
. O

Disaster O
medicine O

The O
longest O
S1 O
- O
S2 O
interval O
at O
which O
S2 O
failed O
to O
capture O
( O
effective O
refractory O
period O
) O
was O
longer O
by O
5 O
. O
5 O
+ O
/ O
- O
8 O
. O
2 O
msec O
at O
twice O
threshold O
( O
0 O
. O
05 O
less O
than O
p O
less O
than O
0 O
. O
10 O
) O
and O
5 O
. O
5 O
+ O
/ O
- O
3 O
. O
5 O
msec O
at O
5 O
times O
threshold O
stimulation O
( O
p O
less O
than O
0 O
. O
01 O
) O
when O
the O
basic O
drive O
and O
extrastimuli O
were O
delivered O
to O
separate O
sites O
. O

The O
use O
of O
specific O
somatic O
cell O
hybrids O
have O
allowed O
us O
to O
locate O
the O
YAC O
contig O
telomeric O
to O
the O
D3F15S2 B
locus I
, O
in O
a O
region O
which O
is O
frequently O
deleted O
in O
lung O
carcinomas O
. O

Experiments O
were O
performed O
without O
polarization O
or O
with O
cathodic O
and O
anodic O
polarization O
of O
the O
adsorbent O
. O

Chronic O
active O
hepatitis O
and O
cirrhosis O
of O
the O
liver O
. O

This O
regulation O
is O
mediated O
by O
several O
kinases O
that O
phosphorylate O
specific O
residues O
in O
the O
different O
functional O
domains O
of O
the O
p53 B
molecule I
. O

These O
data O
show O
that O
the O
processing O
of O
specific O
target O
transcripts O
, O
such O
as O
the O
P B
- I
element I
mRNA I
, O
is O
regulated O
by O
a O
functional O
PSI B
- O
U1 B
snRNP O
interaction O
in O
Drosophila O
. O

Effect O
of O
dimethyl O
sulfoxide O
on O
cooling O
rates O
of O
unrestrained O
rats O
. O

In O
four O
calves O
given O
Haemonchus O
contortus O
larvae O
, O
the O
serum B
pepsinogen I
concentration O
rose O
quickly O
to O
reach O
a O
mean O
of O
3 O
. O
5 O
iu O
tyrosine O
on O
day O
14 O
after O
infection O
. O

These O
results O
strongly O
suggest O
that O
HSV O
- O
1 O
infection O
inhibits O
host O
cell O
splicing O
through O
the O
action O
of O
ICP27 B
. O

The O
T O
( O
CCO2 O
) O
was O
related O
to O
the O
PCO2 O
by O
a O
Pearson O
product O
coefficient O
of O
0 O
. O
79 O
( O
p O
< O
. O
0005 O
) O
, O
with O
a O
mean O
difference O
of O
1 O
. O
94 O
( O
T O
( O
CCO2 O
) O
> O
P O
( O
CO2 O
) O
and O
95 O
% O
confidence O
interval O
of O
- O
0 O
. O
12 O
to O
4 O
. O
07 O
. O

Transformation O
of O
chicken O
embryo O
fibroblasts O
( O
CEF O
) O
with O
the O
Gag B
- O
Crk B
fusion O
protein O
results O
in O
the O
elevation O
of O
tyrosine O
phosphorylation O
on O
specific O
cellular O
proteins O
with O
molecular O
weights O
of O
130 O
, O
000 O
, O
110 O
, O
000 O
, O
and O
70 O
, O
000 O
( O
p130 B
, O
p110 B
, O
and O
p70 B
, O
respectively O
) O
, O
an O
event O
which O
has O
been O
correlated O
with O
cell O
transformation O
. O

Urease B
activity O
, O
judged O
as O
the O
amount O
of O
ammonia O
production O
from O
urea O
, O
could O
be O
measured O
at O
25 O
ng O
per O
tube O
( O
S O
/ O
N O
= O
1 O
. O
5 O
) O
with O
Jack B
bean I
meal I
urease I
. O

Here O
, O
we O
provide O
direct O
evidence O
that O
the O
meiotic O
defect O
caused O
by O
either O
unregulated O
cAPK B
activity O
or O
unregulated O
ran1 B
+ I
kinase I
activity O
is O
due O
to O
inability O
to O
induce O
transcription O
of O
the O
mei2 B
+ I
gene I
, O
which O
is O
required O
for O
meiotic O
initiation O
. O

Optical O
rotation O
of O
the O
second O
harmonic O
radiation O
from O
retinal O
in O
bacteriorhodopsin B
monomers I
in O
Langmuir O
- O
Blodgett O
film O
: O
evidence O
for O
nonplanar O
retinal O
structure O
. O

Ultrasonography O
is O
a O
diagnostic O
alternative O
which O
may O
replace O
arthrography O
. O

In O
K562 O
cells O
, O
using O
the O
bicistronic O
vector O
, O
selection O
with O
colchicine O
led O
to O
at O
least O
20 O
- O
fold O
higher O
expression O
of O
the O
MDR1 B
gene I
product I
than O
did O
selection O
with O
G418 O
, O
suggesting O
that O
the O
stringent O
MDR1 B
selection O
system O
is O
very O
efficient O
for O
obtaining O
overexpression O
of O
foreign O
genes O
. O

In O
this O
report O
, O
several O
health O
behaviors O
were O
investigated O
in O
relation O
to O
maintaining O
mobility O
during O
4 O
years O
of O
follow O
- O
up O
among O
6 O
, O
981 O
men O
and O
women O
aged O
65 O
years O
and O
older O
with O
intact O
mobility O
at O
baseline O
between O
1981 O
and O
1983 O
who O
lived O
in O
one O
of O
three O
communities O
: O
East O
Boston O
, O
Massachusetts O
; O
Iowa O
and O
Washington O
counties O
, O
Iowa O
; O
and O
New O
Haven O
, O
Connecticut O
. O

Our O
analysis O
indicates O
that O
a O
gene O
correction O
mechanism O
has O
been O
operating O
on O
the O
Hbbs O
chromosome O
to O
keep O
beta B
s I
and O
beta B
t I
evolving O
in O
concert O
, O
whereas O
on O
the O
Hbbd O
chromosome O
, O
beta B
dmin I
has O
diverged O
considerably O
from O
beta B
dmaj I
. O

Structure O
and O
regulation O
of O
KGD2 B
, O
the O
structural O
gene O
for O
yeast B
dihydrolipoyl I
transsuccinylase I
. O

In O
dehydrated O
animals O
at O
Ta O
38 O
degrees O
C O
, O
Tb O
and O
EWL O
were O
both O
significantly O
altered O
( O
P O
less O
than O
0 O
. O
001 O
) O
from O
the O
normally O
hydrated O
state O
and O
were O
measured O
at O
39 O
. O
9 O
+ O
/ O
- O
0 O
. O
2 O
degree O
C O
and O
0 O
. O
84 O
+ O
/ O
- O
0 O
. O
09 O
W O
. O

kg O
- O
1 O
respectively O
. O

We O
identify O
YY1 B
recognition I
sequences I
within O
the O
vitamin O
D O
response O
element O
( O
VDRE O
) O
of O
the O
osteocalcin B
gene I
that O
are O
critical O
for O
YY1 B
- O
dependent O
repression O
of O
vitamin O
D O
- O
enhanced O
promoter O
activity O
. O

Back O
- O
ground O
factors O
which O
affect O
the O
outcome O
of O
steroid O
withdrawal O
therapy O
in O
patients O
with O
chronic O
type O
B O
hepatitis O
- O
- O
statistical O
evaluation O
of O
the O
importance O
of O
the O
mode O
of O
HBV O
transmission O

Point O
mutations O
of O
Stat3 B
within O
the O
interacting O
domains O
blocked O
both O
physical O
interaction O
of O
Stat3 B
with O
c B
- I
Jun I
and O
their O
cooperation O
in O
IL B
- I
6 I
- O
induced O
transcription O
directed O
by O
the O
alpha B
( I
2 I
) I
- I
macroglobulin I
enhancer I
. O

Three O
patients O
with O
Mirizzi O
type O
II O
syndrome O
and O
two O
patients O
with O
Mirizzi O
type O
I O
syndrome O
were O
treated O
laparoscopically O
. O

Synchronous O
multicentric O
giant O
cell O
tumour O
: O
a O
case O
report O
with O
review O
of O
literature O
. O

From O
the O
comparison O
of O
the O
nod B
box I
sequences I
of O
( O
brady O
) O
rhizobia O
with O
a O
more O
divergent O
nod B
box I
from O
Azorhizobium O
caulinodans O
, O
we O
now O
propose O
a O
modular O
build O
- O
up O
of O
the O
nod B
box I
with O
the O
sequence O
A O
- O
T O
- O
C O
- O
N9 O
- O
G O
- O
A O
- O
T O
as O
the O
binding O
target O
of O
the O
NodD B
protein I
( O
the O
NodD B
box I
) O
. O

The O
prgX B
gene I
is O
adjacent O
to O
prgQ B
which O
provides O
the O
promoter O
for O
prgB B
expression O
. O

METHODS O
: O
A O
discriminant O
function O
predicting O
surgery O
outcome O
( O
seizure O
- O
free O
vs O
. O
non O
- O
seizure O
- O
free O
) O
was O
computed O
separately O
for O
samples O
of O
patients O
with O
left O
( O
n O
= O
79 O
) O
and O
right O
( O
n O
= O
62 O
) O
temporal O
lobectomy O
( O
LATL O
, O
RATL O
) O
. O

It O
is O
likely O
that O
these O
recordings O
are O
incomplete O
; O
the O
actual O
number O
of O
activated O
neurons O
is O
estimated O
to O
be O
about O
300 O
in O
the O
acutely O
sensitized O
preparation O
. O

The O
possibility O
to O
use O
this O
method O
for O
common O
diagnostic O
problems O
is O
indicated O
. O

Genetic O
analysis O
of O
the O
role O
of O
herpes B
simplex I
virus I
type I
1 I
glycoprotein I
K I
in O
infectious O
virus O
production O
and O
egress O
. O

A O
conventional O
N O
- O
terminal O
signal O
sequence O
was O
not O
detected O
in O
the O
NodO B
protein I
. O

Dynamic O
expression O
of O
broad B
- I
complex I
isoforms I
mediates O
temporal O
control O
of O
an O
ecdysteroid O
target O
gene O
at O
the O
onset O
of O
Drosophila O
metamorphosis O
. O

The O
study O
was O
carried O
out O
according O
to O
the O
BSI O
( O
1980 O
) O
recommendations O
for O
testing O
restorative O
materials O
in O
vivo O
. O

Cellular O
lysates O
were O
analyzed O
for O
luciferase B
and O
beta B
- I
galactosidase I
activities O
. O

Cytokines O
and O
steroid O
hormones O
use O
different O
sets O
of O
signal O
transduction O
pathways O
, O
which O
seem O
to O
be O
unrelated O
. O

These O
findings O
suggest O
that O
the O
proteolipids O
of O
the O
vacuolar B
H I
+ I
- I
ATPases I
were O
evolved O
in O
parallel O
with O
the O
eubacterial O
proteolipid O
, O
from O
a O
common O
ancestral O
gene O
that O
underwent O
gene O
duplication O
. O

This O
mutant O
also O
functions O
in O
vivo O
as O
a O
trans O
- O
acting O
dominant O
negative O
regulator O
: O
the O
transcriptional O
inducibility O
of O
the O
HIV B
long I
terminal I
repeat I
( O
which O
contains O
two O
potential O
NF B
- I
kappa I
B I
binding I
sites I
) O
by O
phorbol O
ester O
( O
PMA O
) O
is O
inhibited O
when O
it O
is O
co O
- O
transfected O
into O
CD4 B
+ I
T O
cells O
with O
the O
delta B
SP I
mutant I
. O

In O
addition O
, O
measurements O
of O
shielded O
and O
unshielded O
syringes O
containing O
99mTc O
- O
labelled O
radiopharmaceuticals O
were O
carried O
out O
. O

This O
study O
tests O
the O
hypothesis O
that O
O2 O
is O
a O
critical O
component O
in O
myocardial O
protection O
afforded O
by O
BCP O
. O

Biomass O
growth O
monitoring O
using O
pressure O
drop O
in O
a O
cocurrent O
biofilter O

This O
alternative O
splice O
acceptor O
site O
is O
also O
used O
by O
a O
biologically O
active O
provirus O
with O
an O
efficiency O
of O
approximately O
5 O
% O
compared O
with O
the O
upstream O
site O
. O

Homology O
is O
much O
higher O
( O
64 O
% O
) O
between O
the O
28 O
- O
kDa O
protein O
and O
regions O
that O
are O
strongly O
conserved O
among O
the O
members O
of O
the O
serine B
protease I
family I
. O

We O
have O
isolated O
and O
structurally O
characterized O
genomic O
DNA O
and O
cDNA O
sequences O
encoding O
ribulose B
- I
1 I
, I
5 I
- I
bisphosphate I
carboxylase I
/ I
oxygenase I
( I
Rbu I
- I
P2 I
carboxylase I
) I
activase I
from O
barley O
( O
Hordeum O
vulgare O
L O
. O
) O
. O

Expression O
of O
putative O
constitutively O
active O
forms O
of O
DdMEK1 B
in O
a O
ddmek1 B
null O
background O
is O
capable O
, O
at O
least O
partially O
, O
of O
complementing O
the O
small O
aggregate O
size O
defect O
and O
the O
ability O
to O
activate O
guanylyl B
cyclase I
. O

Constant O
slowing O
of O
median O
MNCV O
and O
SNCV O
and O
ulnar O
SNCV O
without O
changes O
in O
morphology O
, O
amplitude O
and O
duration O
of O
MAP O
and O
SAP O
have O
been O
observed O
in O
92 O
patients O
, O
with O
persistent O
abnormalities O
in O
64 O
cases O
after O
six O
months O
. O

Moreover O
, O
such O
mutations O
lead O
to O
a O
dramatic O
transition O
in O
chromatin O
structure O
: O
the O
DNase B
I I
hypersensitive O
region O
is O
replaced O
by O
two O
stable O
, O
sequence O
- O
positioned O
nucleosomes O
. O

During O
chronic O
treatment O
with O
salmon B
calcitonin I
, O
alkaline B
phosphatase I
activity O
and O
urinary O
hydroxyproline O
excretion O
decrease O
on O
an O
average O
of O
50 O
% O
in O
patients O
with O
Paget O
' O
s O
disease O
. O

187 O
- O
200 O
. O

Because O
of O
the O
structural O
similarities O
the O
new O
gene O
will O
be O
termed O
scERV2 B
from O
now O
on O
. O

Experience O
in O
a O
Kenya O
district O
hospital O

The O
2 O
. O
3 O
x O
10 O
( O
3 O
) O
base O
SUF12 B
+ I
transcript I
contains O
an O
open O
reading O
frame O
sufficient O
to O
encode O
a O
88 O
x O
10 O
( O
3 O
) O
Mr O
protein O
. O

Cortical O
somatosensory O
evoked O
potentials O
to O
median O
nerve O
stimulation O
revealed O
significantly O
delayed O
peak O
latencies O
of O
N20 O
, O
P20 O
, O
P25 O
, O
and O
N26 O
, O
although O
N16 O
latency O
was O
normal O
. O

A O
new O
family O
of O
rate O
- O
adaptive O
pacemakers O
accomplishes O
this O
circadian O
rate O
variation O
by O
modeling O
the O
patient O
' O
s O
sleep O
- O
wake O
cycle O
using O
a O
time O
- O
of O
- O
day O
clock O
inside O
the O
device O
. O

Progestins O
: O
present O
and O
future O
. O

Bilirubin O
and O
red O
cell O
metabolism O
in O
relation O
to O
neonatal O
jaundice O
. O

Each O
of O
these O
materials O
were O
placed O
by O
means O
of O
three O
different O
placement O
techniques O
( O
Bulk O
pack O
, O
Horizontal O
layering O
and O
Vertical O
layering O
) O
into O
class O
II O
cavities O
in O
extracted O
human O
molars O
. O

This O
ligand O
- O
independent O
pathway O
can O
function O
through O
another O
androgen O
- O
regulated O
promoter O
as O
shown O
by O
the O
use O
of O
the O
mouse O
mammary O
tumor O
virus O
MMTV O
- O
CAT B
reporter O
. O

Stress O
- O
induced O
suppression O
of O
the O
prolactin B
afternoon O
surge O
in O
ovariectomized O
, O
estrogen O
- O
treated O
rats O
and O
the O
nocturnal O
surge O
in O
pseudopregnant O
rats O
are O
accompanied O
by O
an O
increase O
in O
median O
eminence O
dihydroxyphenylacetic O
acid O
concentrations O
. O

The O
sensitivity O
for O
ergometry O
and O
treadmill O
testing O
was O
75 O
and O
62 O
% O
respectively O
. O

Morphologic O
evidence O
that O
rhe O
renal O
calyx O
and O
pelvis O
control O
ureteric O
activity O
in O
the O
rabbit O
. O

Children O
with O
repeated O
proteinuria O
during O
follow O
- O
up O
tend O
to O
have O
a O
higher O
incidence O
of O
pathologic O
findings O
on O
the O
i O
. O
v O
. O
- O
pyelogram O
. O

Parelaphostrongylus O
odocoilei O
is O
redescribed O
from O
worms O
collected O
from O
the O
type O
host O
( O
Odocoileus O
hemionus O
columbianus O
) O
in O
California O
, O
as O
well O
as O
material O
from O
experimentally O
infected O
mule O
deer O
( O
O O
. O
h O
. O
heminus O
) O
in O
Alberta O
. O

The O
carboxyl O
globular O
domain O
was O
found O
to O
be O
encoded O
by O
six O
exons O
which O
appear O
to O
delineate O
its O
structural O
subdomains O
. O

Expression O
of O
the O
Asp O
but O
not O
the O
Ala O
gB B
mutation O
resulted O
in O
an O
increase O
in O
the O
steady O
- O
state O
expression O
of O
gB B
at O
the O
plasma O
membrane O
( O
PM O
) O
in O
U373 O
cells O
. O

Thus O
integration B
host I
factor I
is O
required O
for O
normal O
type O
1 O
fimbriae O
phase O
variation O
in O
E O
. O
coli O
. O

Insulin B
therapy O
has O
been O
expanded O
by O
development O
of O
human B
insulin I
and O
new O
modes O
of O
injection O
, O
including O
insulin B
pumps O
. O

Because O
levels O
of O
Vo2DIR O
and O
Vo2INDIR O
were O
similar O
in O
both O
groups O
, O
we O
pooled O
data O
from O
septic O
and O
control O
animals O
. O

Further O
interventions O
were O
: O
nephrectomy O
( O
2x O
) O
, O
resection O
and O
ligation O
of O
the O
inferior O
vena O
cava O
( O
1x O
) O
, O
resection O
and O
replacement O
of O
the O
left O
renal O
vein O
( O
1x O
) O
. O

OBJECTIVES O
: O
The O
present O
study O
tested O
the O
hypothesis O
that O
stimulating O
the O
central O
noradrenergic O
system O
using O
the O
new O
noradrenaline O
re O
- O
uptake O
inhibitor O
reboxetine O
would O
result O
in O
a O
dose O
- O
dependent O
enhancement O
of O
memory O
for O
emotional O
material O
in O
man O
. O

We O
examined O
the O
mechanisms O
by O
which O
two O
different O
types O
of O
photonic O
radiation O
, O
short O
wavelength O
UV O
( O
UV O
- O
C O
) O
and O
gamma O
radiation O
, O
activate O
transcription O
factor O
NF I
- I
kappaB I
. O

However O
, O
coligation O
of O
Fc B
gamma I
RIIB1 I
with O
B B
cell I
Ag I
receptors I
( O
BCR B
) O
inhibits O
BCR B
- O
mediated O
signaling O
by O
a O
mechanism O
that O
may O
involve O
recruitment O
of O
phosphatases O
SHP B
- I
1 I
, O
SHP B
- I
2 I
, O
and O
the O
SH2 B
containing I
inositol I
5 I
' I
phosphatase I
( O
SHIP B
) O
to O
the O
phosphorylated O
Fc B
gamma I
RIIB1 I
immunoreceptor I
tyrosine I
- I
based I
inhibitory I
motif I
. O

Using O
the O
standard O
scoring O
, O
children O
with O
full O
scale O
IQ O
< O
or O
= O
84 O
on O
the O
Wechsler O
Preschool O
and O
Primary O
Scale O
of O
Intelligence O
at O
age O
4 O
- O
5 O
years O
were O
poorly O
identified O
( O
sensitivity O
54 O
% O
) O
from O
the O
composite O
S O
- O
B O
IV O
score O
at O
age O
3 O
. O

11 O
: O
5801 O
- O
5812 O
, O
1991 O
) O
present O
evidence O
that O
the O
Vps18 B
/ O
Pep3 B
protein O
colocalizes O
with O
the O
Vps11 B
/ O
Pep5 B
protein O
to O
the O
cytosolic O
face O
of O
the O
vacuolar O
membrane O
. O

This O
study O
includes O
200 O
patients O
treated O
from O
1964 O
to O
1978 O
, O
with O
an O
age O
range O
from O
15 O
to O
102 O
years O
, O
who O
required O
329 O
generators O
. O

We O
conclude O
that O
the O
BR B
- I
C I
early I
gene I
directly O
activates O
late O
gene O
transcription O
by O
interacting O
with O
late O
gene O
cis O
- O
acting O
regulatory O
elements O
and O
that O
this O
interaction O
is O
responsible O
for O
the O
temporal O
linkage O
of O
early O
and O
late O
ecdysone O
- O
induced O
gene O
expression O
. O

The O
specificity O
and O
sensitivity O
of O
this O
method O
( O
limit O
of O
detection O
in O
plasma O
0 O
. O
025 O
micrograms O
/ O
mL O
and O
< O
or O
= O
0 O
. O
0125 O
micrograms O
/ O
mL O
for O
febantel O
and O
its O
metabolites O
, O
respectively O
) O
were O
sufficiently O
high O
to O
enable O
us O
to O
characterize O
the O
time O
course O
of O
the O
drug O
in O
the O
plasma O
after O
oral O
administration O
of O
therapeutic O
doses O
to O
sheep O
. O

We O
have O
identified O
the O
gene O
encoding O
sigma B
E I
using O
a O
genetic O
screen O
designed O
to O
isolate O
trans O
- O
acting O
mutations O
that O
abolish O
expression O
from O
either O
htrA B
or O
rpoHP3 B
, O
two O
promoters O
recognized O
uniquely O
by O
sigma B
E I
- I
containing I
RNA I
polymerase I
. O

Cytokinetics O
of O
the O
Krebs O
2 O
carcinoma O
. O

Through O
effective O
use O
of O
laboratory O
testing O
database O
, O
it O
will O
be O
possible O
to O
shift O
away O
our O
vague O
management O
of O
pre O
- O
analytic O
phase O
of O
quality O
control O
so O
far O
to O
its O
established O
system O
based O
on O
objective O
evaluation O
. O

These O
patients O
developed O
severe O
diabetic O
symptoms O
including O
glucose O
intolerance O
, O
weight O
loss O
, O
impaired O
energy O
utilization O
, O
and O
nerve O
and O
brain O
disorders O
that O
were O
refractory O
to O
insulin B
. O

One O
promoter O
, O
p53P1 B
, O
is O
located O
100 O
- O
250 O
bp O
upstream O
of O
the O
218 O
- O
bp O
noncoding O
first O
exon O
; O
a O
second O
, O
stronger O
promoter O
, O
p53P2 B
, O
maps O
within O
the O
first O
intron O
. O

Both O
PMP20 B
gene I
products I
contain O
the O
carboxyl O
- O
terminal O
sequence O
AKL O
, O
similar O
to O
the O
putative O
SKL B
peroxisomal I
sorting I
sequence I
( O
Gould O
, O
S O
. O

The O
likelihood O
of O
death O
was O
more O
than O
3 O
times O
higher O
among O
patients O
in O
the O
ERA O
- O
II O
group O
( O
mortality O
risk O
ratio O
3 O
. O
82 O
[ O
95 O
% O
CI O
1 O
. O
48 O
% O
to O
9 O
. O
84 O
] O
, O
p O
= O
0 O
. O
006 O
) O
. O

Barium O
mucosal O
coating O
was O
judged O
to O
be O
better O
in O
the O
members O
to O
whom O
magnesium O
sulphate O
was O
administered O
( O
p O
= O
0 O
. O
0007 O
) O
. O

In O
both O
immortalized O
and O
normal O
diploid O
human O
cells O
, O
wt O
AAV O
targeted O
integration O
to O
ch O
- O
19 O
. O

The O
corresponding O
tetrapeptide O
sequences O
SSPD O
and O
SATD O
for O
human O
and O
mouse B
PKC I
- I
epsilon I
, O
respectively O
, O
are O
unusual O
for O
caspase B
- I
3 I
. O

The O
lipopolysaccharide B
- I
binding I
protein I
is O
a O
secretory O
class O
1 O
acute O
- O
phase O
protein O
whose O
gene O
is O
transcriptionally O
activated O
by O
APRF B
/ O
STAT B
/ I
3 I
and O
other O
cytokine O
- O
inducible O
nuclear O
proteins O
. O

Tritium O
concentrations O
in O
environmental O
water O
samples O
were O
found O
to O
be O
determined O
within O
an O
accuracy O
of O
10 O
% O
by O
this O
method O
when O
Vi O
/ O
Vf O
was O
14 O
- O
25 O
. O

This O
region O
does O
not O
contain O
a O
consensus O
estrogen O
response O
element O
, O
and O
ER B
binding O
to O
this O
DNA O
sequence O
was O
not O
observed O
. O

These O
proteins O
, O
called O
variant B
surface I
glycoproteins I
( O
VSGs B
) O
, O
are O
expressed O
from O
a O
specific O
locus O
, O
the O
VSG B
gene I
expression I
site I
( O
ES O
) O
. O

The O
strangulated O
intestinal O
pathologies O
of O
horses O
are O
accompanied O
by O
a O
local O
activation O
of O
the O
neutrophils O
, O
that O
can O
be O
revealed O
by O
measuring O
the O
tissular O
enzymatic O
activity O
of O
the O
granulocytic B
enzyme I
myeloperoxidase I
( O
MPO B
) O
. O

A O
deletion O
analysis O
of O
the O
FAS1 B
promoter I
lacking O
the O
previously O
characterized O
inositol O
/ O
choline O
- O
responsive O
- O
element O
motif O
defined O
a O
region O
( O
nucleotides O
- O
760 O
to O
- O
850 O
) O
responsible O
for O
most O
of O
the O
remaining O
activation O
potency O
. O

Here O
we O
report O
the O
fabrication O
of O
single O
- O
molecule O
transistors O
based O
on O
individual O
C60 O
molecules O
connected O
to O
gold O
electrodes O
. O

The O
homeodomain B
transcription I
factor I
CDP B
/ O
cut B
interacts O
with O
the O
cell O
cycle O
regulatory O
element O
of O
histone B
H4 I
genes I
packaged O
into O
nucleosomes O
. O

They O
encode O
proteins O
whose O
core O
regions O
display O
great O
similarity O
to O
Aspergillus B
HAPB I
, O
HAPC B
and O
HAPE B
and O
to O
known O
HAP B
homologs I
from O
other O
organisms O
. O

Furthermore O
, O
the O
cognate O
binding O
protein O
is O
present O
in O
both O
rat O
and O
human O
( O
HeLa O
) O
cell O
nuclear O
extracts O
. O

Transient O
cotransfection O
analyses O
indicate O
that O
the O
cooperative O
association O
of O
NF B
- I
IL I
- I
6 I
and O
RelA B
with O
the O
IL B
- I
8 I
promoter I
results O
in O
synergistic O
transcriptional O
activation O
. O

Heart O
rate O
during O
the O
behavioral O
avoidance O
test O
was O
shown O
to O
be O
as O
susceptible O
to O
experimental O
demand O
as O
other O
studies O
have O
shown O
approach O
behaviors O
to O
be O
influenced O
. O

Function O
was O
retained O
when O
one O
copy O
of O
the O
sequence O
was O
present O
, O
suggesting O
that O
this O
sequence O
represents O
an O
essential O
element O
. O

Decreased O
plasma O
level O
of O
antithrombin B
III I
was O
assumed O
to O
be O
one O
of O
the O
major O
factors O
underlying O
hypercoagulable O
state O
in O
nephrotic O
syndrome O
. O

NO2 O
pollution O
on O
major O
trunk O
roads O
frequently O
exceeded O
British O
and O
European O
Union O
air O
quality O
standards O
, O
while O
particle O
pollution O
was O
lower O
. O

Cotransfection O
of O
pINV B
- I
2473 O
, O
a O
construct O
containing O
2473 O
base O
pairs O
of O
hINV B
upstream I
sequence I
linked O
to O
luciferase B
, O
with O
POU B
homeodomain I
transcription I
factors I
Oct1 B
, O
Oct2 B
, O
Brn4 B
, O
SCIP B
, O
Skn1a B
or O
Skn1i B
, O
results O
in O
a O
strong O
suppression O
of O
basal O
promoter O
activity O
. O

87 O
, O
7270 O
- O
7274 O
) O
. O

I O
. O

Transcripts O
initiating O
at O
the O
CAGT O
motif O
( O
proximal O
transcripts O
) O
were O
abolished O
by O
deletion O
of O
the O
proximal O
TATA O
box O
located O
at O
- O
29 O
relative O
to O
CAGT O
. O

Genomic O
DNA O
blot O
analysis O
indicates O
that O
the O
trout B
L2 I
locus I
has O
a O
cluster O
- O
like O
organization O
similar O
to O
the O
trout B
L1 I
locus I
and O
the O
IgL B
locus I
of O
several O
teleost O
fish O
. O

Although O
the O
energy O
- O
based O
DFT O
was O
not O
affected O
by O
isoproterenol O
( O
from O
6 O
. O
1 O
+ O
/ O
- O
1 O
. O
5 O
to O
6 O
. O
0 O
+ O
/ O
- O
1 O
. O
7 O
J O
) O
, O
it O
was O
decreased O
to O
3 O
. O
7 O
+ O
/ O
- O
1 O
. O
6 O
J O
in O
the O
third O
stage O
by O
infusion O
of O
E4031 O
and O
isoproterenol O
( O
p O
< O
0 O
. O
01 O
vs O
. O
baseline O
and O
vs O
. O
isoproterenol O
) O
. O

Gore O
& O
Associates O
, O
Inc O
. O
) O
standard O
wall O
graft O
segments O
varying O
in O
length O
from O
4 O
to O
12 O
cm O
. O

The O
dynamics O
of O
N O
- O
interaction O
and O
N O
- O
retention O
during O
3 O
hr O
CPB O
was O
quantified O
with O
autologous O
In O
- O
111 O
labeled O
neutrophils O
( O
INN O
) O
in O
4 O
groups O
of O
20 O
Yorkshire O
pigs O
( O
28 O
- O
35 O
kg O
, O
5 O
sham O
; O
5 O
CPB O
, O
1 O
hr O
; O
5 O
CPB O
, O
3 O
hr O
and O
5 O
CPB O
with O
heparinized O
circuit O
, O
3 O
hr O
) O
; O
anesthetized O
pigs O
were O
injected O
with O
INN O
( O
500 O
- O
650 O
microCi O
) O
, O
30 O
min O
before O
CPB O
and O
heparinized O
, O
and O
underwent O
CPB O
with O
a O
roller O
pump O
, O
a O
hollow O

fiber O
OX O
( O
Bentley O
CM O
50 O
, O
5 O
. O
0 O
m2 O
) O
and O
AF O
( O
Bentley O
AF O
025 O
, O
0 O
. O
25 O
m2 O
) O
at O
2 O
. O
5 O
- O
3 O
. O
6 O
l O
/ O
min O
for O
3 O
hr O
. O

The O
region O
immediately O
following O
the O
promoter O
and O
5 O
' O
to O
ORF1 O
has O
a O
potential O
transcription O
terminator O
sequence O
. O

Studies O
on O
recurrences O
in O
gingival O
hyperplasia O

Preface O
and O
acknowledgements O
. O

We O
conclude O
that O
clonidine O
3 O
micrograms O
/ O
kg O
produces O
sedation O
comparable O
to O
diazepam O
0 O
. O
2 O
mg O
/ O
kg O
and O
also O
attenuates O
the O
intubation O
response O
without O
increasing O
the O
incidence O
of O
complications O
. O

Pancreatic O
expression O
of O
the O
glucagon B
gene I
depends O
on O
multiple O
transcription O
factors O
interacting O
with O
at O
least O
three O
DNA O
control O
elements O
: O
G1 O
, O
the O
upstream O
promoter O
element O
, O
and O
G2 O
and O
G3 O
, O
two O
enhancer O
- O
like O
sequences O
. O

We O
have O
compared O
the O
ability O
of O
GST B
- O
Bem3 B
to O
serve O
as O
a O
GAP O
for O
Cdc42Hs B
relative O
to O
other O
members O
of O
the O
rho B
- O
GAP B
subfamily O
and O
found O
the O
following O
order O
of O
potency O
: O
human B
platelet I
Cdc42Hs I
GAP B
> O
p190 B
> O
Bem3 B
> O
break O
point I
cluster I
region I
protein I
, O
whereas O
p85 B
, O
like O
Bem2 B
, O
shows O
no O
GAP B
activity O
or O
any O
ability O
to O
bind O
to O
the O
GTP O
- O
bound O
form O
of O
Cdc42Hs B
. O

CONCLUSION O
: O
Men O
presenting O
with O
urethritis O
and O
women O
presenting O
with O
PID O
both O
have O
significantly O
greater O
peripheral O
blood O
mononuclear O
cell O
proliferative O
responses O
to O
the O
DK20 O
strain O
of O
C O
trachomatis O
than O
controls O
. O

The O
PDSS O
identified O
17 O
( O
94 O
% O
) O
of O
the O
women O
diagnosed O
with O
major O
postpartum O
depression O
, O
the O
EPDS O
identified O
14 O
of O
these O
women O
( O
78 O
% O
) O
, O
and O
the O
BDI O
- O
II O
identified O
10 O
of O
the O
18 O
women O
( O
56 O
% O
) O
. O

Men O
and O
women O
entering O
psychotherapy O
more O
often O
had O
negative O
Wish O
elements O
than O
others O
. O

Single O
amino O
acid O
substitutions O
were O
shown O
to O
result O
from O
the O
mutations O
pdr1 B
- I
2 I
( O
M308I O
) O
, O
pdr1 B
- I
3 I
( O
F815S O
) O
, O
pdr1 B
- I
6 I
( O
K302Q O
) O
, O
pdr1 B
- I
7 I
( O
P298A O
) O
and O
pdr1 B
- I
8 I
( O
L1036 I
W I
) O
, O
whereas O
the O
intragenic O
suppressor O
mutant O
pdr1 B
- I
100 I
is O
deleted O
for O
the O
two O
amino O
acids O
L537 O
and O
A538 O
. O

Both O
merR B
genes I
consist O
of O
a O
408 O
bp O
ORF O
coding O
for O
135 O
amino O
acids O
. O

Amniotic O
fluid O
ionic O
concentration O
in O
response O
to O
chronic O
fetal B
vasopressin I
infusion O
. O

The O
same O
slice O
was O
imaged O
three O
times O
each O
with O
sequences O
using O
spatial O
presaturation O
or O
not O
. O

Molecular O
cloning O
of O
the O
alpha B
- I
globin I
transcription I
factor I
CP2 I
. O

The O
most O
distal O
active O
site O
in O
TIGA B
is O
created O
by O
excision O
of O
a O
66 O
- O
bp O
intron O
. O

Denaturing O
gradient O
gel O
electrophoresis O
( O
DGGE O
) O
with O
sequence O
analysis O
showed O
that O
PGCL4 B
is O
a O
major O
member O
in O
the O
female O
mammary O
gland O
, O
and O
in O
the O
submaxillary O
and O
lachrymal O
glands O
of O
both O
sexes O
, O
while O
the O
counterpart O
in O
male O
liver O
and O
the O
coagulate O
glands O
was O
found O
to O
be O
PGCL1 B
. O

All O
possessed O
cutaneous O
receptive O
fields O
on O
the O
distal O
segments O
of O
digits O
2 O
, O
3 O
, O
or O
4 O
. O

Sesame O
seed O
should O
also O
be O
considered O
a O
cause O
of O
allergic O
reactions O
to O
drug O
products O
and O
cosmetics O
. O

Ligand O
binding O
of O
multi B
- I
chain I
antigen I
receptors I
and O
hematopoietin B
/ I
cytokine I
receptors I
results O
in O
rapid O
activation O
of O
protein B
tyrosine I
kinase I
( O
PTK B
) O
- O
dependent O
signalling O
molecules O
such O
as O
phosphatidylinositol B
3 I
- I
kinase I
( O
PI B
3 I
- I
kinase I
) O
. O

The O
UASB O
reactors O
treated O
a O
VFA O
mixture O
( O
with O
an O
acetate O
: O
propionate O
: O
butyrate O
ratio O
of O
5 O
: O
3 O
: O
2 O
on O
COD O
basis O
) O
or O
acetate O
as O
the O
sole O
substrate O
at O
different O
COD O
: O
sulfate O
ratios O
. O

The O
authors O
emphasize O
absence O
of O
specific O
signs O
of O
vagal O
involvement O
, O
importance O
for O
diagnosis O
of O
surgical O
extirpation O
, O
and O
mildness O
of O
post O
operative O
course O
. O

Now O
: O
pleasure O
in O
work O

This O
article O
reviews O
current O
concepts O
of O
pathophysiology O
and O
summarises O
clinical O
features O
, O
natural O
history O
and O
available O
treatments O
. O

Previous O
data O
suggested O
a O
subtle O
increase O
in O
serum O
P O
at O
the O
time O
of O
hCG B
injection O
without O
LH B
surge O
reduces O
the O
PR O
of O
women O
having O
oocyte O
retrievals O
for O
IVF O
; O
this O
study O
compared O
PRs O
of O
recipients O
in O
a O
shared O
oocyte O
program O
according O
to O
the O
donors O
' O
pre O
- O
hCG B
P O
level O
. O

Adequacy O
of O
the O
Haldane O
transformation O
in O
the O
computation O
of O
exercise O
V O
O2 O
in O
man O
. O

M O
. O
, O
Adrich O
, O
Z O
. O
, O
Fournet O
, O
B O
. O
, O
Capon O
, O
C O
. O
, O
Bonicel O
, O
J O
. O

Association O
between O
hyperhomocysteinemia O
and O
ischemic O
heart O
disease O
in O
Sri O
Lankans O
. O

Radiological O
imaging O
such O
as O
UGI O
series O
and O
CT O
scan O
was O
useful O
to O
arrive O
at O
an O
accurate O
diagnosis O
. O

The O
architecture O
of O
the O
hRap1 B
Myb B
domain O
is O
very O
close O
to O
that O
of O
each O
of O
the O
Myb B
domains I
from O
TRF1 B
, O
scRap1p B
and O
c B
- I
Myb I
. O

80 O
v B
- I
ets I
- I
encoded I
amino I
- I
acids I
located O
immediately O
after O
the O
v B
- I
myb I
/ O
v B
- I
ets I
junction O
are O
not O
found O
in O
P54 B
/ I
56c I
- I
ets I
, O
the O
translation O
product O
of O
the O
c B
- I
ets I
proto I
- I
oncogene I
, O
nor O
in O
a O
set O
of O
cellular O
proteins O
of O
64 O
, O
62 O
, O
and O
60 O
kDa O
related O
to O
but O
distinct O
from O
P54 B
/ I
56c I
- I
ets I
. O

We O
have O
raised O
antibodies O
against O
a O
peptide O
specific O
to O
the O
predicted O
protein O
product O
of O
this O
second O
mRNA O
. O

Because O
controlled O
- O
release O
niacin O
seems O
to O
be O
more O
potent O
than O
crystalline O
niacin O
, O
product O
substitution O
without O
dose O
adjustment O
should O
be O
avoided O
. O

Interobserver O
agreement O
of O
the O
Nottingham O
histologic O
grading O
scheme O
for O
infiltrating O
duct O
carcinoma O
breast O
. O

This O
dose O
of O
Na3 O
citrate O
produced O
no O
clinical O
symptoms O
suggestive O
of O
hypocalcaemia O
in O
these O
subjects O
, O
even O
though O
the O
use O
of O
acid O
- O
citrate O
- O
dextrose O
, O
NIH O
formula O
A O
( O
ACD O
- O
A O
) O
under O
identical O
conditions O
has O
been O
reported O
to O
reduce O
significantly O
the O
level O
of O
total O
calcium O
in O
serum O
, O
and O
concomitantly O
increase O
the O
number O
or O
reactions O
occurring O
in O
donors O
. O

Latex O
products O
( O
gloves O
, O
balloons O
, O
and O
condoms O
) O
directly O
bound O
IgE B
from O
all O
four O
patients O
. O

The O
phosphoprotein B
pUL69 I
of I
human I
cytomegalovirus I
( I
HCMV I
) I
, O
which O
is O
a O
herpesvirus O
of O
considerable O
medical O
importance O
in O
immunosuppressed O
patients O
and O
newborns O
, O
has O
previously O
been O
identified O
as O
an O
early O
- O
late O
viral O
protein O
that O
can O
stimulate O
several O
viral O
and O
cellular O
promoters O
and O
thus O
exerts O
a O
rather O
broad O
activation O
pattern O
. O

Southern O
blot O
analysis O
performed O
on O
genomic O
DNA O
demonstrated O
altered O
CpG O
methylation O
within O
intron O
1 O
in O
DNA O
from O
all O
BCC O
compared O
to O
normal O
, O
mortal O
human O
mammary O
epithelial O
cells O
( O
HMEC O
) O
. O

Previous O
studies O
have O
shown O
that O
TGFbeta1 B
expression O
is O
upregulated O
in O
mouse O
keratinocytes O
infected O
with O
a O
v O
- O
rasHa O
retrovirus O
, O
although O
the O
functional O
significance O
of O
this O
has O
not O
been O
clear O
. O

An O
increased O
incidence O
of O
ATN O
has O
been O
reported O
since O
the O
introduction O
of O
cyclosporin O
A O
. O

Using O
a O
GTP O
- O
dependent O
, O
brefeldin O
A O
- O
sensitive O
in O
vitro O
AP B
- I
1 I
binding O
assay O
, O
we O
have O
determined O
here O
the O
parameters O
of O
the O
AP B
- I
1 I
binding O
reaction O
. O

The O
overall O
rate O
of O
biohydrogenation O
of O
C18 O
: O
2 O
was O
14 O
. O
3 O
% O
/ O
h O
, O
but O
declined O
1 O
. O
2 O
% O
/ O
h O
for O
each O
percentage O
unit O
increase O
in O
C18 O
: O
2 O
added O
to O
the O
substrate O
. O

Plasma B
enteroglucagon I
was O
measured O
before O
and O
during O
three O
hours O
after O
a O
standard O
meal O
in O
21 O
untreated O
adult O
patients O
with O
suspected O
coeliac O
disease O
who O
all O
had O
villous O
atrophy O
of O
the O
small O
intestinal O
mucosa O
and O
malabsorption O
, O
and O
in O
nine O
control O
subjects O
. O

After O
control O
for O
body O
mass O
index O
, O
trait O
anxiety O
and O
anger O
- O
in O
remained O
independent O
predictors O
of O
diastolic O
blood O
pressure O
among O
the O
women O
. O

Regulation O
of O
the O
human O
stress B
response I
gene I
GADD153 I
expression O
: O
role O
of O
ETS1 B
and O
FLI B
- I
1 I
gene I
products I
. O

The O
PRP4 B
protein I
of I
Saccharomyces I
cerevisiae I
is O
an O
essential O
part O
of O
the O
U4 B
/ O
U6 B
snRNP O
, O
a O
component O
of O
the O
mRNA O
splicing O
apparatus O
. O

132 O
varix O
ligations O
were O
performed O
during O
44 O
separate O
EVL O
sessions O
. O

Here O
RFX B
/ O
X2BP I
/ O
DNA O
complexes O
were O
formed O
on O
all O
class O
II O
isotypes O
regardless O
of O
the O
ability O
of O
the O
X O
box O
region O
to O
bind O
either O
factor O
individually O
. O

The O
psi B
zeta I
gene I
has O
a O
nonsense O
mutation O
in O
exon O
1 O
but O
has O
identical O
promoter O
sequence O
and O
RNA O
processing O
sites O
to O
the O
zeta B
gene I
, O
raising O
the O
possibility O
that O
both O
psi B
zeta I
and O
zeta I
are O
transcriptionally O
active O
. O

Prospects O
of O
chemosterilant O
and O
genetic O
control O
of O
rodents O
. O

Serum B
PRL I
concentrations O
significantly O
increased O
after O
MCP O
administration O
in O
normal O
women O
, O
hyperprolactinemic O
patients O
with O
normal O
sella O
and O
patients O
with O
microadenoma O
, O
but O
not O
in O
macroadenoma O
patients O
with O
and O
without O
suprasellar O
expansion O
( O
SSE O
) O
. O

The O
Id4 B
protein I
contains O
a O
HLH B
domain O
highly O
conserved O
among O
the O
dnHLH B
proteins I
from O
mouse O
and O
drosophila O
. O

The O
coding O
region O
of O
nifA B
was O
determined O
using O
a O
translational O
lacZ B
fusion I
and O
by O
site O
- O
directed O
mutagenesis O
to O
identify O
which O
of O
four O
in O
frame O
AUG O
codons O
was O
used O
. O

Peptide O
competition O
studies O
have O
localized O
a O
cyclin B
A I
interaction I
site I
to O
a O
Lys381Lys382Leu383Met384Phe385 O
sequence O
within O
C O
- O
terminal O
negative O
regulatory O
domain O
of O
human B
p53 I
. O

Heterodimerization O
mutant B
RXR I
failed O
to O
alter O
GFP B
- O
VDR B
and O
nlsGFP B
- O
VDR B
distribution O
or O
activity O
. O

Like O
troponin B
Cs I
and O
calmodulins B
, O
PfCPK B
also O
contains O
four O
EF O
hand O
calcium O
- O
binding O
motifs O
. O

A O
third O
group O
( O
n O
= O
10 O
) O
was O
assigned O
to O
receive O
the O
canalith O
repositioning O
maneuver O
without O
mastoid O
vibration O
. O

Upstream O
of O
the O
afa B
- I
3 I
gene I
cluster I
, O
a O
1 O
. O
2 O
- O
kb O
region O
was O
found O
to O
be O
96 O
% O
identical O
to O
the O
RepFIB B
sequence I
of O
one O
of O
the O
enterotoxigenic O
E O
. O
coli O
plasmids O
( O
P307 O
) O
, O
suggesting O
a O
common O
ancestor O
plasmid O
. O

The O
cats O
with O
stage O
- O
2 O
lymphomas O
that O
were O
FeLV O
- O
test O
negative O
had O
the O
best O
response O
to O
treatment O
. O

We O
hypothesize O
that O
the O
ability O
of O
these O
acidic O
activators O
to O
specifically O
interact O
with O
multiple O
components O
of O
the O
transcription O
initiation O
complex O
likely O
underlies O
the O
dramatic O
functional O
synergy O
exhibited O
by O
this O
class O
of O
activation O
domains O
in O
vivo O
. O

Where O
the O
resonator O
has O
two O
holes O
, O
these O
terms O
should O
be O
somewhat O
modified O
: O
A O
is O
the O
combined O
area O
of O
the O
two O
holes O
, O
L O
is O
16 O
/ O
3pi O
r O
( O
~ O
1 O
. O
7r O
) O
for O
a O
simple O
hole O
in O
a O
thin O
- O
walled O
vessel O
and O
r O
is O
the O
radius O
of O
one O
hole O
( O
Seto O
, O
1971 O
) O
. O

Histological O
and O
immunophenotypic O
studies O
revealed O
12 O
large O
cell O
lymphomas O
( O
11 O
B O
cell O
and O
one O
T O
cell O
) O
, O
two O
small O
noncleaved O
cell O
lymphomas O
( O
B O
- O
cell O
phenotype O
) O
, O
and O
five O
low O
grade O
B O
- O
cell O
lymphomas O
( O
two O
small O
lymphocytic O
and O
three O
follicular O
mixed O
lymphomas O
) O
. O

The O
deviation O
site O
is O
equivalent O
to O
the O
exon O
1 O
/ O
exon O
2 O
splice O
site O
of O
the O
mouse B
C I
- I
subunit I
. O

The O
determination O
of O
diacetyl O
, O
2 O
, O
3 O
- O
pentanedione O
and O
acetoin O
was O
performed O
in O
two O
steps O
. O

There O
also O
may O
be O
osmotic O
challenges O
to O
mucosal O
cell O
function O
as O
evidenced O
by O
the O
different O
reaction O
rates O
with O
hyper O
- O
and O
hypotonic O
saline O
. O

In O
F9 O
, O
which O
is O
a O
prototype O
of O
embryonal O
carcinoma O
cells O
expressing O
hst B
, O
the O
expression O
of O
hst B
gene I
is O
positively O
regulated O
by O
a O
downstream O
octamer O
motif O
that O
functions O
as O
an O
enhancer O
. O

The O
reading O
achievement O
of O
students O
with O
learning O
disabilities O
who O
received O
reading O
instruction O
through O
the O
DISTAR O
program O
was O
compared O
to O
that O
of O
similar O
students O
using O
basal O
reader O
materials O
. O

Two O
putative O
ATP O
- O
binding O
domains O
were O
identified O
, O
one O
in O
the O
amino O
- O
terminal O
half O
of O
the O
argA B
- I
encoded I
protein I
and O
the O
other O
in O
the O
carboxy O
- O
terminal O
half O
. O

Chronic O
granulocytic O
leukemia O
in O
children O
. O

Purified O
phosphorylated O
PhoP B
( O
PhoPP B
) O
had O
a O
half O
- O
life O
of O
approximately O
2 O
. O
5 O
h O
, O
which O
was O
reduced O
to O
about O
15 O
min O
by O
addition O
of O
the O
same O
molar O
amount O
of O
* B
PhoR I
( O
the O
cytoplasmic O
region O
of O
PhoR B
) O
. O

This O
transformation O
suppression O
by O
Rb O
was O
further O
shown O
to O
be O
due O
to O
transcriptional O
repression O
of O
neu B
using O
Rb O
expressing O
effector O
plasmid O
and O
neu B
promoter I
- O
chloramphenicol B
acetyltransferase I
reporter O
gene O
. O

Before O
therapy O
was O
started O
, O
trabecular O
bone O
alterations O
could O
be O
observed O
which O
were O
typical O
for O
Paget O
' O
s O
disease O
of O
bone O
as O
there O
were O
bulky O
trabeculare O
, O
increased O
remodelling O
surfaces O
and O
giant O
osteoclasts O
. O

T O
. O
infestans O
, O
T O
. O
delpontei O
, O
T O
. O
rubrovaria O
, O
T O
. O
sordida O
, O
T O
. O
guasayana O
and O
T O
. O
vitticeps O
from O
infestans O
subgroup O
and O
T O
. O
pallidipennis O
from O
rubrofasciata O
, O
were O
studied O
. O

Overexpression O
of O
N O
- O
terminal O
mutations O
disturbs O
mitosis O
and O
produces O
elongated O
cells O
, O
Using O
a O
PCR O
approach O
, O
we O
isolated O
a O
putative O
homologue O
of O
Prp4 B
from O
human O
and O
mouse O
cells O
. O

1 O
. O

Biochemical O
studies O
and O
studies O
of O
cells O
expressing O
mutant B
IL I
- I
2 I
receptors I
indicate O
that O
IL B
- I
2 I
- O
induced O
tyrosine B
kinase I
activation O
initiates O
a O
complex O
signaling O
cascade O
. O

BACKGROUND O
: O
Peripheral O
kappa B
receptor I
agonists O
may O
provide O
a O
new O
therapeutic O
approach O
for O
the O
treatment O
of O
functional O
dyspepsia O
. O

Fourteen O
eyes O
( O
38 O
. O
9 O
% O
) O
developed O
DLK O
after O
an O
epithelial O
defect O
, O
representing O
an O
odds O
ratio O
of O
13 O
times O
. O

The O
yeast O
genome O
contains O
a O
single O
ORD1 B
gene I
that O
resides O
on O
chromosome O
XI O
. O

The O
system O
consists O
of O
a O
TV O
unit O
compatible O
to O
IBM O
PC O
/ O
AT O
and O
software O
. O

The O
N O
- O
terminal O
sequence O
of O
the O
extracellular B
MEP20 I
, O
TKVAS B
, O
was O
found O
at O
aa O
194 O
- O
198 O
within O
the O
ORF O
. O

Isolates O
of O
B O
. O
burgdorferi O
from O
humans O
, O
rodents O
, O
and O
I O
. O
dammini O
are O
usually O
indistinguishable O
, O
but O
strains O
of O
B O
. O
burgdorferi O
with O
different O
major O
proteins O
have O
been O
identified O
. O

Cloning O
of O
the O
GATA B
- I
binding I
protein I
that O
regulates O
endothelin B
- I
1 I
gene I
expression O
in O
endothelial O
cells O
. O

The O
Synechococcus B
gene I
rps1 I
encoding O
S1 B
is O
located O
1 O
. O
1 O
kb O
downstream O
from O
psbB B
, O
which O
encodes O
the O
photosystem B
II I
P680 I
chlorophyll I
a I
apoprotein I
. O

Two O
- O
dimensional O
echocardiographic O
parasternal O
long O
- O
and O
short O
- O
axis O
views O
were O
obtained O
during O
graded O
bleeding O
by O
rapid O
withdrawal O
of O
blood O
from O
an O
arterial O
cannula O
, O
with O
increments O
of O
5 O
% O
each O
up O
to O
30 O
% O
of O
calculated O
blood O
volume O
. O

A O
comparison O
of O
the O
rate O
of O
recovery O
and O
time O
for O
detection O
of O
mycobacteria O
from O
clinical O
specimens O
of O
the O
newly O
developed O
Mycobacteria O
Growth O
Indicator O
Tube O
( O
MGIT O
) O
, O
the O
biphasic O
Septi O
- O
Chek O
, O
and O
the O
egg O
- O
based O
Ogawa O
medium O
was O
made O
. O

As O
in O
IncP O
alpha O
plasmids O
, O
these O
operons O
are O
transcribed O
from O
a O
bidirectional O
promoter O
region O
consisting O
of O
trfAp B
for O
the O
trfA B
operon I
and O
trbAp B
and O
trbBp B
for O
the O
trb B
operon I
. O

CONCLUSIONS O
: O
These O
principally O
structural O
studies O
support O
the O
hypothesis O
that O
the O
thrombus O
is O
a O
self O
- O
sustaining O
entity O
that O
may O
have O
significance O
in O
the O
pathophysiologic O
mechanism O
of O
abdominal O
aortic O
aneurysms O
. O

The O
BHV B
- I
1 I
UL24 I
, O
UL25 B
, O
UL26 B
and O
UL26 B
. I
5 I
transcripts I
all O
terminated O
at O
a O
common O
3 O
' O
- O
polyadenylation O
site O
and O
varied O
significantly O
in O
their O
relative O
abundance O
. O

Electrophoretic O
mobility O
shift O
analysis O
of O
protein O
- O
DNA O
complexes O
formed O
with O
nuclear O
proteins O
isolated O
from O
I3C O
- O
treated O
and O
- O
untreated O
cells O
, O
in O
combination O
with O
supershift O
assays O
using O
Sp1 B
antibodies I
, O
demonstrated O
that O
the O
Sp1 B
- I
binding I
site I
in O
the O
CDK6 B
promoter I
forms O
a O
specific O
I3C O
- O
responsive O
DNA O
- O
protein O
complex O
that O
contains O
the O
Sp1 B
transcription I
factor I
. O

These O
results O
suggest O
that O
both O
the O
distinct O
DNA O
binding O
properties O
of O
PMLRAR B
homodimers I
and O
the O
sequestration O
of O
RXR B
by O
PMLRARs B
may O
contribute O
to O
the O
molecular O
mechanisms O
which O
underlie O
the O
pathogenesis O
of O
APL O
. O

The O
analysis O
showed O
that O
the O
morning O
active O
( O
MA O
) O
individuals O
rose O
earlier O
and O
went O
to O
bed O
earlier O
than O
the O
evening O
active O
( O
EA O
) O
individuals O
, O
and O
the O
former O
had O
a O
longer O
sleep O
length O
than O
the O
latter O
during O
days O
with O
a O
morning O
shift O
, O
while O
the O
opposite O
was O
true O
for O
afternoon O
and O
night O
shifts O
. O

This O
ERD O
is O
recorded O
over O
the O
contralateral O
central O
region O
. O

Deletion O
mutations O
were O
constructed O
, O
removing O
residues O
2 O
- O
30 O
, O
31 O
- O
60 O
, O
61 O
- O
90 O
, O
and O
49 O
- O
78 O
of O
the O
N O
- O
terminal O
cytoplasmic O
domain O
, O
as O
well O
as O
a O
missense O
mutation O
of O
a O
dileucine O
motif O
. O

Kluyveromyces O
lactis O
, O
a O
budding O
yeast O
related O
to O
Saccharomyces O
cerevisiae O
, O
can O
grow O
on O
a O
wider O
variety O
of O
substrates O
and O
shows O
less O
sensitivity O
to O
glucose O
repression O
than O
does O
Saccharomyces O
cerevisiae O
. O

Low O
- O
affinity O
E2 B
- I
binding I
site I
mediates O
downmodulation O
of O
E2 B
transactivation O
of O
the O
human B
papillomavirus I
type I
8 I
late I
promoter I
. O

Untransformed O
3T3 O
cells O
carry O
abundant O
1 B
. I
9 I
kb I
Hox I
1 I
. I
3 I
RNA I
, O
whereas O
the O
methylcholanthrene O
- O
transformed O
MB66 O
and O
LTK O
- O
cells O
or O
3T3 O
cells O
transformed O
by O
the O
oncogenes O
src B
, O
fos B
or O
SV40 B
T I
antigen I
express O
only O
low O
levels O
. O

Although O
analysis O
of O
the O
similarity O
between O
the O
44 O
- O
kDa O
protein O
and O
the O
E B
. I
coli I
RecA I
protein I
did O
not O
show O
any O
significant O
homology O
between O
them O
, O
it O
revealed O
their O
identity O
by O
five O
amino O
- O
acid O
residues O
involved O
in O
the O
formation O
of O
the O
epitope O
that O
recognized O
the O
paratope O
of O
the O
RecA B
protein I
antibody I
for O
subsequent O
epitope O
- O
paratope O
binding O
of O
these O
proteins O
. O

Ureases B
were O
purified O
from O
the O
recombinant O
cells O
and O
shown O
to O
be O
identical O
to O
control O
enzyme O
when O
analyzed O
by O
gel O
filtration O
chromatography O
and O
sodium O
dodecyl O
sulfate O
- O
polyacrylamide O
gel O
electrophoresis O
; O
however O
, O
in O
every O
case O
the O
activity O
levels O
correlated O
to O
nickel O
contents O
as O
analyzed O
by O
atomic O
absorption O
analysis O
. O

Deoxyuridine O
- O
suppression O
test O
and O
bone O
marrow O
culture O
for O
the O
diagnosis O
of O
macrocytic O
refractory O
anaemias O
. O

Finally O
, O
synthetic O
peptides O
corresponding O
to O
the O
mutant O
proteins O
were O
assessed O
for O
the O
ability O
to O
act O
as O
substrates O
for O
PR B
. O

These O
psoralens O
are O
being O
used O
in O
research O
projects O
sponsored O
by O
the O
National O
Toxicology O
Program O
. O

Root O
surface O
caries O
incidence O
in O
the O
groups O
inoculated O
with O
A O
. O
viscosus O
and O
A O
. O
viscosus O
plus O
S O
. O
sobrinus O
did O
not O
differ O
. O

These O
results O
demonstrate O
that O
the O
entire O
E3L B
gene I
is O
required O
for O
pathogenesis O
in O
the O
mouse O
model O
. O

The O
development O
of O
various O
pathological O
lesions O
in O
thymus O
, O
spleen O
, O
lymph O
nodes O
and O
bone O
- O
marrow O
is O
frequently O
observed O
. O

Recently O
, O
we O
have O
demonstrated O
that O
mouse O
microglial O
cells O
, O
the O
brain O
macrophages O
, O
express O
both O
IL B
- I
15 I
and O
IL B
- I
15 I
/ O
IL B
- I
2 I
receptors I
. O

We O
have O
analyzed O
the O
role O
of O
the O
HU B
protein I
in O
invertasome O
assembly O
when O
the O
enhancer O
is O
located O
at O
variable O
positions O
close O
to O
one O
of O
the O
recombination O
sites O
. O

The O
mode O
of O
action O
is O
often O
suggested O
as O
an O
explanation O
for O
the O
different O
slopes O
of O
concentration O
- O
effect O
curves O
. O

This O
may O
be O
another O
example O
of O
the O
principle O
- O
- O
less O
is O
more O
. O

A O
wide O
conservation O
of O
iscSUA B
genes I
in O
nature O
and O
evidence O
that O
NifU B
and O
NifS B
participate O
in O
the O
mobilization O
of O
iron O
and O
sulfur O
for O
nitrogenase B
- O
specific O
iron O
- O
sulfur O
cluster O
formation O
suggest O
that O
the O
products O
of O
the O
iscSUA B
genes I
could O
play O
a O
general O
role O
in O
the O
formation O
or O
repair O
of O
iron O
- O
sulfur O
clusters O
. O

On O
transfer O
from O
glucose O
, O
succinate O
, O
malate O
, O
or O
glycerol O
medium O
to O
citrate O
medium O
, O
the O
Cit B
+ I
Escherichia O
coli O
strains O
showed O
a O
delay O
of O
36 O
to O
48 O
h O
before O
growth O
. O

These O
results O
suggest O
that O
camptothecin O
resistance O
in O
CEM O
/ O
DOX O
cells O
is O
due O
to O
different O
mechanism O
( O
s O
) O
than O
topoisomerase B
- O
or O
P B
- I
glycoprotein I
- O
associated O
multidrug O
resistance O
. O

Jornvall O
, O
B O
. O

The O
pattern O
of O
expression O
of O
this O
and O
other O
Chi26 B
/ O
Chi33 B
chimeric O
promoters O
suggest O
that O
the O
E O
- O
region O
contains O
cis O
- O
acting O
sequences O
which O
activate O
transcription O
in O
aleurone O
and O
silence O
transcription O
in O
leaves O
. O

Spontaneous O
coronary O
artery O
spasm O
during O
coronary O
angiography O
in O
a O
patient O
with O
exercise O
- O
induced O
ST O
segment O
elevation O
. O

Four O
newborns O
in O
this O
group O
became O
HBsAg B
carriers O
. O

Effectively O
, O
the O
upstream O
, O
housekeeping O
- O
type O
promoter O
responds O
to O
FIXK B
and O
positively O
regulates O
the O
downstream O
, O
sigma B
54 I
- I
type I
promoter I
. O

K O
. O
, O
Weiss O
, O
M O
. O

New O
TB O
respirator O
standards O
mean O
more O
choices O
at O
less O
cost O
. O

Electrophoretic O
mobility O
- O
shift O
assays O
using O
oligonucleotides O
derived O
from O
these O
sites O
demonstrated O
formation O
of O
specific O
DNA O
- O
protein O
complexes O
. O

A O
5789 O
- O
nucleotide O
- O
long O
EcoRI B
fragment I
from O
the O
genome O
of O
Thermotoga O
maritima O
, O
identified O
by O
cross O
- O
hybridization O
to O
L11 B
, O
L1 B
, O
L10 B
, O
and O
L12 B
ribosomal I
protein I
gene I
sequences I
from O
Escherichia O
coli O
, O
was O
cloned O
and O
sequenced O
. O

Background O
reduction O
with O
various O
agents O
had O
a O
prominent O
effect O
on O
DTIn1 O
as O
well O
as O
99mTc O
- O
DTPA O
biodistribution O
. O

These O
data O
suggest O
that O
reduction O
in O
PRA O
may O
have O
contributed O
to O
the O
hemodynamic O
effects O
of O
this O
peak B
III I
PDE I
inhibitor O
. O

The O
following O
parameters O
were O
measured O
: O
onset O
time O
( O
time O
interval O
from O
injection O
to O
maximal O
or O
total O
block O
) O
, O
T125 O
/ O
75 O
( O
time O
for O
T1 O
to O
reach O
25 O
% O
or O
75 O
% O
of O
control O
) O
, O
TOF70 O
( O
time O
for O
TOF O
ratio O
to O
reach O
70 O
% O
of O
control O
) O
, O
heart O
rate O
and O
blood O
pressure O
. O

A O
. O

Prematurely O
inactivating O
p42 B
MAPK I
in O
egg O
extracts O
resulted O
in O
a O
corresponding O
hastening O
of O
the O
first O
mitosis O
. O

The O
use O
of O
ALAD B
activity O
and O
EP O
as O
cumulative O
lead O
exposure O
indicators O
is O
suggested O
. O

Binding O
of O
C B
/ I
EBPs I
to O
all O
three O
spi B
2 I
. I
3 I
3 I
' I
UTR I
repressor I
sites I
, O
although O
rather O
weak O
, O
was O
confirmed O
by O
electrophoretic O
mobility O
shift O
assays O
that O
otherwise O
failed O
to O
reveal O
specific O
interactions O
with O
other O
liver O
nuclear O
proteins O
in O
vitro O
. O

Cell O
cycle O
regulatory O
components O
have O
been O
largely O
conserved O
in O
eukaryotes O
; O
however O
, O
orthologs O
of O
neither O
CAK1 B
nor O
csk1 B
have O
been O
identified O
in O
other O
species O
to O
date O
. O

In O
this O
study O
, O
we O
cloned O
the O
full O
- O
length O
cDNA O
of O
mouse B
PAX4 I
by O
RACE O
( O
rapid O
amplification O
of O
cDNA O
ends O
) O
using O
RNA O
from O
MIN6 O
cells O
, O
a O
mouse O
insulinoma O
cell O
line O
. O

Kohtz O
, O
J O
. O

Hence O
the O
GAP1 B
gene I
encodes O
a O
protein O
with O
characteristics O
typical O
of O
integral O
membrane O
proteins O
translocating O
ligants O
across O
cellular O
membranes O
. O

The O
results O
suggest O
that O
proenzyme O
processing O
is O
not O
essential O
for O
secretion O
of O
PC2 B
, O
but O
peptides O
containing O
mutations O
that O
affect O
the O
ability O
of O
the O
propeptide O
( O
and O
cleavage O
sites O
) O
to O
fold O
within O
the O
catalytic O
pocket O
are O
not O
transferred O
beyond O
the O
early O
stages O
of O
the O
secretory O
pathway O
. O

These O
results O
suggest O
that O
transcription O
influences O
aspects O
of O
gene O
conversion O
after O
initiation O
, O
such O
as O
strand O
invasion O
and O
/ O
or O
mismatch O
repair O
( O
MMR O
) O
. O

Important O
prognostic O
factors O
for O
the O
results O
of O
physiotherapeutic O
exercises O
in O
intermittent O
claudication O

Finally O
, O
in O
samples O
obtained O
from O
two O
patients O
with O
drug O
refractory O
ALL O
, O
BAC O
- O
derived O
probes O
applied O
to O
archived O
marrow O
cells O
demonstrated O
that O
a O
breakpoint O
occurred O
between O
MDR1 B
and O
sequences O
500 O
- O
1000 O
KB O
telomeric O
to O
MDR1 B
, O
consistent O
with O
a O
random O
chromosomal O
rearrangement O
. O

Application O
of O
the O
method O
using O
an O
11 O
- O
year O
microseismicity O
record O
revealed O
systematic O
spatial O
and O
temporal O
changes O
in O
the O
slip O
rate O
that O
were O
synchronous O
with O
earthquake O
activity O
and O
other O
independent O
measures O
of O
fault O
- O
zone O
slip O
. O

PC12 O
cultures O
that O
had O
been O
" O
primed O
" O
in O
this O
way O
showed O
an O
accelerated O
and O
augmented O
differentiation O
response O
to O
nerve B
growth I
factor I
. O

One O
of O
these O
clones O
encodes O
Xenopus B
N I
- I
ras I
. O

The O
Molteno O
implant O
is O
an O
effective O
procedure O
in O
the O
treatment O
of O
complicated O
and O
refractory O
cases O
of O
glaucoma O
in O
high O
- O
risk O
eyes O
. O

The O
median O
levels O
of O
t B
- I
PA I
Ag I
and O
PAI B
in O
plasma O
were O
respectively O
10 O
. O
7 O
ng O
/ O
ml O
( O
interquartile O
range O
8 O
. O
6 O
ng O
/ O
ml O
) O
and O
15 O
. O
7 O
IU O
/ O
ml O
( O
interquartile O
range O
12 O
. O
2 O
IU O
/ O
ml O
) O
. O

In O
the O
remaining O
91 O
patients O
( O
group O
2 O
) O
, O
Pseudomonas O
( O
18 O
) O
, O
coagulase O
- O
negative O
Staphylococci O
( O
15 O
) O
, O
Staphylococcus O
epidermidis O
( O
23 O
) O
, O
Staphylococcus O
aureus O
( O
16 O
) O
, O
Corynebacterium O
species O
( O
12 O
) O
, O
and O
others O
( O
seven O
) O
were O
isolated O
. O

Stamps O
in O
cardiology O
: O
de O
Musset O
sign O
. O

A O
factor O
present O
in O
nuclear O
extracts O
of O
wounded O
potato O
tubers O
was O
found O
to O
bind O
specifically O
to O
nucleotides O
located O
between O
- O
135 O
to O
- O
105 O
, O
suggesting O
that O
this O
region O
contains O
important O
cis O
- O
regulatory O
elements O
. O

The O
leucine O
zipper O
region O
of O
ATF B
proteins I
is O
also O
similar O
to O
that O
of O
the O
AP B
- I
1 I
/ O
c B
- I
jun I
family O
of O
transcription O
factors O
, O
whose O
DNA O
- O
binding O
site O
differs O
from O
the O
ATF B
- I
binding I
site I
at O
a O
single O
position O
. O

The O
R260A O
mutant O
, O
as O
expected O
, O
was O
unable O
to O
support O
mRNA O
synthesis O
in O
vitro O
in O
a O
transcription O
reconstitution O
reaction O
as O
well O
as O
transcription O
in O
vivo O
of O
a O
minigenome O
using O
a O
reverse O
genetic O
approach O
. O

PEAP O
was O
significantly O
higher O
in O
IDC O
patients O
compared O
to O
controls O
( O
48 O
. O
7 O
+ O
/ O
- O
32 O
. O
6 O
vs O
. O

Uterine O
contractions O
began O
with O
a O
mean O
latency O
of O
62 O
min O
in O
the O
PGF2 O
alpha O
infused O
women O
, O
in O
controls O
uterine O
activity O
remained O
unchanged O
. O

Were O
this O
hypothesis O
correct O
, O
the O
requirement O
for O
elF4A B
should O
correlate O
with O
the O
degree O
of O
mRNA O
secondary O
structure O
. O

In O
spite O
of O
the O
presence O
of O
a O
physician O
, O
the O
real O
pathology O
of O
an O
indian O
population O
of O
Colombie O
is O
not O
very O
well O
known O
. O

Taken O
together O
, O
these O
results O
demonstrate O
that O
the O
BSMV B
coat I
protein I
is O
the O
sole O
translation O
product O
of O
the O
genomic O
RNA O
beta O
, O
whereas O
sgRNA B
beta I
1 I
serves O
as O
a O
messenger O
for O
translation O
of O
the O
beta B
b I
protein I
, O
and O
sgRNA B
beta I
2 I
functions O
as O
a O
messenger O
for O
translation O
of O
beta B
c I
and O
beta B
d I
and O
the O
newly O
discovered O
beta B
d I
' I
protein I
. O

A O
retrospective O
study O
of O
clozapine O
and O
electroencephalographic O
abnormalities O
in O
schizophrenic O
patients O
. O

The O
concepts O
were O
developed O
by O
Mines O
and O
Garrey O
during O
the O
next O
10 O
years O
. O

When O
the O
measured O
beta B
- I
galactosidase I
activity O
in O
the O
pWP O
- O
transfected O
cells O
was O
normalized O
to O
the O
pCH110 O
- O
transfected O
cells O
( O
an O
appropriate O
control O
if O
the O
SV40 B
early I
region I
promoter I
functions O
constitutively O
in O
various O
cell O
lines O
) O
, O
the O
results O
suggested O
that O
the O
promoter O
region O
of O
the O
PLP B
gene I
contains O
the O
information O
necessary O
for O
initiation O
of O
transcription O
in O
a O
C6 O
cell O
- O
specific O
manner O
. O

PKK B
exists O
in O
three O
discernible O
forms O
at O
steady O
state O
: O
an O
underphosphorylated O
form O
of O
100 O
kDa O
; O
a O
soluble O
, O
cytosolic O
, O
phosphorylated O
form O
of O
110 O
kDa O
; O
and O
a O
phosphorylated O
, O
detergent O
- O
insoluble O
form O
of O
112 O
kDa O
. O

The O
P B
- I
ITIM I
- O
compelled O
multi O
- O
phosphoprotein O
complex O
binds O
to O
and O
activates O
SHP B
- I
2 I
, O
which O
in O
turn O
dephosphorylates O
SHIP B
and O
Shc B
and O
probably O
other O
substrates O
. O

When O
PCR O
was O
used O
to O
amplify O
agar O
cultures O
which O
do O
not O
express O
the O
fimbriae O
, O
the O
switch O
region O
was O
OFF O
only O
. O

Mechanisms O
of O
action O
of O
local O
anaesthetics O
. O

Student O
health O

We O
now O
report O
that O
both O
HSCR O
mutations O
impair O
the O
fixation O
of O
Shc B
to O
RET B
and O
consequently O
prevent O
its O
phosphorylation O
. O

The O
only O
quantities O
which O
were O
significantly O
different O
between O
appendicitis O
and O
a O
normal O
appendix O
were O
sex O
, O
duration O
of O
symptoms O
, O
anorexia O
and O
vomiting O
. O

We O
propose O
that O
U73 B
is O
involved O
in O
methylation O
of O
the O
G1739 O
residue O
of O
the O
human B
28S I
rRNA I
. O

At O
the O
start O
of O
the O
performance O
ride O
, O
plasma O
glucose O
averaged O
4 O
. O
2 O
+ O
/ O
- O
0 O
. O
2 O
, O
5 O
. O
2 O
+ O
/ O
- O
0 O
. O
1 O
, O
and O
5 O
. O
7 O
+ O
/ O
- O
0 O
. O
2 O
mmol O
. O
l O
- O
1 O
in O
CON O
, O
CHO O
- O
7 O
, O
and O
CHO O
- O
0 O
/ O
21 O
, O
respectively O
( O
all O
different O
, O
P O
< O
0 O
. O
05 O
) O
. O

Therefore O
prostaglandin O
E2 O
is O
characterized O
in O
relation O
to O
prostaglandin O
F2 O
- O
alpha O
not O
only O
by O
a O
stronger O
uterine O
effectiveness O
, O
but O
also O
at O
the O
same O
time O
a O
pronounced O
influence O
on O
the O
cardiovascular O
system O
. O

Copyright O
1999 O
Academic O
Press O
. O

Ganglia O
are O
cysts O
which O
frequently O
occur O
in O
the O
proximity O
of O
joints O
. O

Lysates O
of O
COS O
cells O
transfected O
with O
modified O
hGrzB B
cDNA I
were O
able O
to O
hydrolyze O
tert O
- O
butyloxycarbonyl O
- O
Ala O
- O
Ala O
- O
Asp O
- O
thiobenzyl O
ester O
( O
Boc O
- O
Ala O
- O
Ala O
- O
Asp O
- O
SBzl O
) O
, O
whereas O
lysates O
transfected O
with O
unmodified O
hGrzB B
cDNA I
were O
inactive O
. O

Proteins O
PBP I
- I
1 I
and O
PBP B
- I
2 I
bind O
to O
two O
of O
the O
three O
promoter O
elements O
in O
the O
trypanosomatid O
Leptomonas O
seymouri O
. O

Expression O
of O
medical O
efficiency O
by O
integration O
indicators O
with O
the O
rational O
utilization O
of O
budgeted O
funds O

The O
present O
study O
tries O
to O
associate O
some O
of O
the O
causes O
of O
hot O
ischaemia O
during O
the O
agony O
of O
the O
corpse O
donor O
and O
to O
evaluate O
those O
that O
may O
be O
caused O
by O
sympathetic O
efference O
during O
removal O
. O

The O
enzyme O
encoded O
by O
the O
cloned O
fragment O
is O
equally O
active O
on O
pyruvate O
and O
hydroxypyruvate O
, O
indicating O
that O
the O
enzyme O
has O
both O
D B
- I
lactate I
and I
D I
- I
glycerate I
dehydrogenase I
activities O
. O

Noise O
in O
STM O
due O
to O
atoms O
moving O
in O
the O
tunneling O
space O
. O

The O
human O
sequence O
contains O
the O
element O
TGACTCT O
that O
also O
is O
found O
in O
a O
murine B
ras I
- I
responsive I
enhancer I
. O

There O
is O
an O
overall O
functional O
similarity O
between O
IL B
- I
6 I
and O
c B
- I
fos I
promoters I
, O
since O
transfection O
of O
excess O
amounts O
of O
either O
promoter O
DNA O
into O
intact O
HeLa O
cells O
modulates O
the O
function O
of O
the O
heterologous O
promoter O
construct O
. O

Enhanced O
phospholipid O
hydrolysis O
with O
free O
fatty O
acid O
and O
thromboxane O
accumulation O
, O
increased O
release O
of O
excitatory O
amino O
acids O
, O
and O
decreased O
tissue O
magnesium O
levels O
may O
each O
serve O
to O
worsen O
secondary O
tissue O
damage O
and O
diminish O
neurologic O
recovery O
after O
spinal O
cord O
injury O
associated O
with O
acute O
alcohol O
intoxication O
. O

To O
determine O
whether O
the O
transcript O
encodes O
an O
active O
enzyme O
, O
we O
developed O
a O
novel O
transient O
embryonic O
excision O
assay O
in O
which O
zebrafish O
fertilized O
eggs O
were O
co O
- O
injected O
with O
RNA O
transcribed O
in O
vitro O
using O
the O
Tol2 B
cDNA I
as O
a O
template O
and O
a O
plasmid O
DNA O
harboring O
a O
nonautonomous O
Tol2 B
element I
, O
which O
has O
a O
deletion O
in O
the O
transposase B
coding O
region O
. O

The O
overall O
objective O
response O
rate O
( O
RR O
) O
was O
50 O
% O
[ O
95 O
% O
confidence O
interval O
( O
CI O
) O
= O
29 O
- O
71 O
% O
] O
including O
four O
complete O
responses O
and O
eight O
partial O
responses O
. O

We O
suggest O
that O
this O
relatively O
small O
deletion O
affects O
a O
segment O
containing O
3 B
' I
VH I
genes I
with O
important O
regulatory O
functions O
, O
the O
loss O
of O
which O
leads O
to O
the O
ali O
phenotype O
. O

Two O
SH2 B
domains I
of O
p120 B
Ras B
GTPase I
- I
activating I
protein I
bind O
synergistically O
to O
tyrosine O
phosphorylated O
p190 B
Rho B
GTPase I
- I
activating I
protein I
. O

p120 B
GTPase B
- I
activating I
protein I
( O
GAP B
) O
is O
a O
negative O
regulator O
of O
Ras B
that O
functions O
at O
a O
key O
relay O
point O
in O
signal O
transduction O
pathways O
that O
control O
cell O
proliferation O
. O

A O
homologue O
of O
the O
MAP B
/ O
ERK B
family O
of O
protein O
kinase O
genes O
is O
expressed O
in O
vegetative O
and O
in O
female O
reproductive O
organs O
of O
Petunia O
hybrida O
. O

Its O
prognostic O
impact O
is O
superior O
to O
the O
T1 O
/ O
2 O
one O
( O
RR O
= O
2 O
. O
9 O
; O
p O
= O
0 O
. O
0010 O
) O
. O

It O
encodes O
a O
sequence O
- O
specific O
transcription O
factor O
that O
controls O
, O
in O
concert O
with O
other O
gene O
products O
, O
differentiative O
pathways O
of O
tissues O
in O
which O
Scr B
is O
expressed O
. O

CONCLUSION O
: O
DMIPP O
detects O
regionally O
hypoperfused O
myocardium O
, O
in O
which O
agreement O
between O
MBF O
and O
fatty O
acid O
uptake O
deteriorates O
. O

The O
effect O
of O
guaiphenesin O
on O
absorption O
and O
bioavailability O
of O
paracetamol O
from O
composite O
analgesic O
preparations O
. O

Transcription O
of O
the O
inserted O
gene O
into O
the O
predicted O
subgenomic O
polyadenylated O
mRNA O
was O
demonstrated O
by O
Northern O
( O
RNA O
) O
blot O
hybridization O
analysis O
, O
and O
the O
encoded O
protein O
was O
detected O
by O
enzyme O
assay O
and O
by O
radioimmunoprecipitation O
. O

These O
results O
suggest O
that O
panretinal O
photocoagulation O
offers O
a O
highly O
effective O
means O
of O
dealing O
with O
early O
and O
moderately O
advanced O
cases O
of O
angle O
neovascularization O
. O

NBSII B
is O
essential O
for O
ribosome O
- O
stimulated O
activity O
. O

Sixteen O
patients O
( O
25 O
. O
4 O
% O
) O
encountered O
rejection O
while O
weaning O
at O
median O
period O
of O
9 O
. O
5 O
months O
( O
range O
, O
1 O
- O
63 O
months O
) O
from O
the O
start O
of O
weaning O
. O

ALT B
( O
alanine B
aminotransferase I
) O
and O
Hb B
( O
hemoglobin B
) O
appeared O
to O
be O
predictive O
for O
efficacy O
. O

Theophylline O
QID O
, O
TID O
, O
BID O
and O
now O
QD O
? O

A O
report O
on O
24 O
- O
hour O
dosing O
with O
slow O
- O
release O
theophylline O
formulations O
with O
emphasis O
on O
analyses O
of O
data O
used O
to O
obtain O
Food O
and O
Drug O
Administration O
approval O
for O
Theo O
- O
24 O
. O

Signal O
transduction O
in O
fibroblasts O
stably O
transformed O
by O
[ B
Val12 I
] I
Ras I
- O
- O
the O
activities O
of O
extracellular B
- I
signal I
- I
regulated I
kinase I
and O
Jun B
N I
- I
terminal I
kinase I
are O
only O
moderately O
increased O
, O
and O
the O
activity O
of O
the O
delta O
- O
inhibitor O
of O
c B
- I
Jun I
is O
not O
alleviated O
. O

Time O
for O
action O
on O
hepatitis O
B O
immunisation O
. O

Escherichia O
coli O
cells O
expressing O
a O
mutant B
glyV I
( I
glycine B
tRNA I
) I
gene I
have O
a O
UVM O
- O
constitutive O
phenotype O
: O
implications O
for O
mechanisms O
underlying O
the O
mutA B
or O
mutC B
mutator O
effect O
. O

By O
24 O
h O
, O
there O
was O
no O
significant O
difference O
in O
FFA O
levels O
from O
shams O
. O

This O
result O
suggests O
that O
the O
target O
protein O
specificity O
of O
E2s B
can O
be O
altered O
by O
the O
addition O
of O
appropriate O
C O
- O
terminal O
extensions O
, O
thus O
providing O
a O
way O
to O
modify O
the O
selectivity O
of O
the O
ubiquitin B
system O
. O

Here O
we O
report O
the O
cloning O
of O
the O
hemF B
gene I
, O
encoding O
the O
aerobic B
coproporphyrinogen I
III I
oxidase I
from I
Escherichia I
coli I
, O
by O
functional O
complementation O
of O
a O
Saccharomyces B
cerevisiae I
HEM13 I
mutant I
. O

This O
study O
was O
undertaken O
to O
evaluate O
the O
physical O
and O
biological O
characteristics O
of O
nebulized O
interleukin B
2 I
liposomes O
. O

The O
atopic O
disposition O
, O
indicated O
by O
positive O
skin O
reactions O
and O
IgE B
antibody I
titers O
etc O
. O
, O
and O
the O
bronchial O
reactivity O
to O
inhaled O
acetylcholine O
were O
examined O
on O
the O
following O
three O
groups O
: O
( O
1 O
) O
20 O
young O
adults O
with O
a O
history O
of O
childhood O
asthma O
who O
have O
been O
symptom O
- O
free O
for O
more O
than O
4 O
yr O
; O
( O
2 O
) O
20 O
current O
asthmatics O
, O
and O
( O
3 O
) O
20 O
healthy O
young O
adults O
. O

Previous O
studies O
showed O
that O
a O
palindromic O
sequence O
located O
at O
- O
159 O
base O
pairs O
is O
essential O
for O
induction O
of O
cutinase B
gene I
in O
Fusarium O
solani O
f O
. O

sp O
. O
pisi O
( O
Nectria O
haematococca O
mating O
type O
VI O
) O
by O
the O
hydroxy O
fatty O
acids O
from O
plant O
cutin B
and O
that O
a O
50 O
- O
kDa O
nuclear O
protein O
binds O
to O
a O
promoter O
that O
contains O
this O
element O
. O

This O
preliminary O
study O
reports O
an O
initial O
evaluation O
of O
HMPAO O
- O
SPECT O
imaging O
for O
assessing O
regional O
alterations O
in O
brain O
function O
during O
opiate O
dependence O
and O
withdrawal O
. O

We O
conclude O
that O
these O
cDNAs O
belong O
to O
a O
novel O
GST B
class O
hereby O
designated O
Kappa O
, O
with O
the O
rat B
GST I
subunit I
13 I
gene I
designated O
rGSTK1 B
and O
the O
human O
gene O
being O
called O
hGSTK1 B
. O

Based O
on O
PCR O
strategies O
and O
expression O
studies O
, O
we O
define O
the O
genomic O
organization O
of O
the O
FUT8b B
gene I
. O

The O
present O
study O
demonstrates O
that O
the O
proto B
- I
oncogene I
c I
- I
jun I
represses O
transcription O
of O
the O
human B
CG I
alpha I
and O
CG B
beta I
promoters I
. O

c B
- I
Jun I
repressed O
the O
CG B
alpha I
promoter I
through O
a O
canonical O
cAMP O
response O
element O
( O
CRE O
) O
that O
is O
known O
to O
bind O
c B
- I
Jun I
and O
other O
members O
of O
the O
B B
- I
Zip I
transcription I
factor I
family I
. O

Pulse O
monitors O
in O
outpatient O
dental O
anaesthesia O
. O

Previous O
studies O
using O
partial O
sequences O
have O
suggested O
the O
potential O
of O
26S B
or O
large B
- I
subunit I
( I
LSU I
) O
rDNA I
for O
phylogeny O
retrieval O
at O
taxonomic O
levels O
comparable O
to O
those O
investigated O
with O
18S B
rDNA I
. O

The O
sensitivity O
of O
99mTc O
- O
IgG B
scintigraphy O
ranged O
between O
71 O
% O
and O
100 O
% O
. O

The O
index O
patient O
, O
a O
10 O
- O
year O
- O
old O
boy O
, O
presents O
typical O
symptoms O
of O
OPD O
type O
I O
together O
with O
bowing O
of O
the O
long O
bones O
and O
abnormalities O
of O
the O
thorax O
and O
spinal O
column O
. O

The O
cause O
for O
the O
increase O
in O
plasma O
ANP B
levels O
in O
the O
active O
phase O
remains O
to O
be O
determined O
. O

A O
genomic O
clone O
representing O
the O
5 O
' O
part O
of O
the O
message O
was O
also O
isolated O
. O

Antibiotics O
and O
CDCA O
- O
mediation O
are O
essential O
for O
symptomatic O
therapy O
. O

The O
main O
side O
- O
effects O
were O
myelosuppression O
, O
mucositis O
and O
peripheral O
neuropathy O
, O
which O
were O
all O
common O
and O
often O
severe O
. O

The O
column O
densities O
of O
frozen O
molecular O
hydrogen O
and O
methanol O
are O
inferred O
to O
be O
about O
2 O
. O
5 O
x O
10 O
( O
18 O
) O
and O
3 O
. O
0 O
x O
10 O
( O
19 O
) O
, O
respectively O
. O

Similarly O
, O
an O
increase O
in O
CAT B
expression O
from O
the O
same O
construct O
( O
pBLCAT B
- O
ENDOA B
) O
was O
also O
observed O
in O
7AQS2 O
. O
1 O
cells O
. O

The O
prevalence O
of O
antibodies O
to O
HRES B
- I
1 I
peptides I
pep14 I
- I
24 I
and O
pep117 B
- I
127 I
was O
determined O
in O
65 O
normal O
blood O
donors O
and O
146 O
patients O
with O
immunological O
disorders O
. O

We O
recently O
reported O
the O
cloning O
and O
sequencing O
of O
the O
alpha B
7 I
integrin I
chain I
and O
its O
regulated O
expression O
during O
the O
development O
of O
skeletal O
muscle O
( O
Song O
et O
al O
. O

The O
contractile O
effects O
of O
oxytocin B
, O
prostaglandin O
F2 O
alpha O
and O
their O
combined O
use O
on O
human O
pregnant O
myometrium O
were O
studied O
in O
vitro O
. O

These O
involve O
( O
1 O
) O
subcloning O
a O
promoterless O
sucCD B
fragment I
downstream O
of O
the O
lac B
promoter I
in O
M13mp10 O
, O
and O
( O
2 O
) O
precise O
splicing O
of O
the O
suc B
coding I
regions I
with O
the O
efficient O
atpE B
ribosome I
- I
binding I
site I
and O
expression O
from O
the O
thermoinducible O
lambda O
promoters O
in O
the O
pJLA503 O
vector O
. O

These O
mutations O
prevent O
FKBP12 B
- O
rapamycin O
binding O
to O
TOR2 B
, O
as O
assayed O
with O
the O
two O
- O
hybrid O
system O
. O

Transcription O
of O
eukaryotic O
tRNA O
genes O
is O
dependent O
on O
the O
A O
- O
and O
B O
- O
Box O
internal O
control O
regions O
( O
ICRs O
) O
and O
the O
upstream O
transcription O
modulatory O
region O
. O

Failure O
to O
demonstrate O
a O
major O
anti O
- O
inflammatory O
effect O
with O
alpha O
tocopherol O
supplementation O
( O
400 O
IU O
/ O
day O
) O
in O
normal O
subjects O
. O

Results O
on O
the O
use O
of O
amiodarone O
in O
the O
prevention O
of O
paroxysmal O
AF O
have O
been O
equivocal O
; O
this O
may O
be O
attributed O
to O
differences O
in O
defining O
paroxysmal O
AF O
. O

High O
- O
level O
expression O
of O
sccypB B
as O
well O
as O
of O
sccypA B
cloned O
into O
the O
expression O
vector O
pIJ702 O
did O
not O
produce O
detectable O
changes O
in O
growth O
and O
morphology O
of O
S O
. O
chrysomallus O
and O
S O
. O
lividans O
. O

Fas B
( O
APO B
- I
1 I
/ O
CD95 B
) O
, O
which O
is O
a O
member O
of O
the O
tumor B
necrosis I
factor I
receptor I
superfamily I
, O
is O
a O
cell O
surface O
receptor O
that O
induces O
apoptosis O
. O

In O
the O
future O
, O
transoesophageal O
echocardiography O
may O
be O
used O
to O
measure O
variations O
in O
wall O
thickness O
which O
change O
the O
global O
loading O
conditions O
in O
the O
basal O
midwall O
compartments O
of O
the O
left O
ventricle O
. O

We O
studied O
18 O
dogs O
, O
anaesthetised O
and O
spontaneously O
breathing O
both O
room O
air O
and O
after O
the O
inhalation O
of O
a O
gas O
mixture O
containing O
10 O
% O
CO2 O
, O
20 O
. O
9 O
% O
O2 O
, O
and O
69 O
. O
1 O
% O
N2 O
, O
to O
determine O
the O
role O
of O
histamine O
, O
serotonin O
, O
and O
acidaemia O
in O
pulmonary O
hypertension O
produced O
by O
hypercapnia O
. O

Sequencing O
of O
this O
fragment O
showed O
that O
the O
MRP13 B
coding I
region I
specifies O
a O
324 O
- O
amino O
- O
acid O
basic O
protein O
with O
a O
calculated O
Mr O
of O
37 O
, O
366 O
. O

Viral B
complementary I
15 I
S I
mRNA I
that O
directs O
the O
synthesis O
of O
N B
protein I
and O
hybridizes O
to O
the O
predicted O
N B
gene I
DNA I
has O
been O
identified O
in O
infected O
cell O
extracts O
. O

Electrophoretic O
mobility O
shift O
assays O
and O
DNase B
I I
protection O
analyses O
revealed O
that O
FP B
I I
was O
bound O
by O
the O
transcription O
factor O
PU B
. I
1 I
/ O
Spi B
- I
1 I
. O

Retroviral O
vector O
producer O
cell O
populations O
and O
cell O
clones O
were O
established O
for O
each O
chLTR B
vector O
, O
and O
all O
were O
capable O
of O
yielding O
high O
vector O
titers O
( O
> O
10 O
( O
5 O
) O
G418R O
cfu O
/ O
ml O
on O
NIH O
- O
3T3 O
) O
. O

However O
, O
a O
few O
sites O
in O
the O
genomes O
of O
EC O
cells O
permit O
M O
- O
MuLVneo O
delta B
Enh I
proviral O
expression O
. O

Increasing O
insulin B
sensitivity O
in O
type O
I O
diabetic O
patients O
should O
alert O
clinicians O
to O
the O
possibility O
of O
glucocorticoid O
deficiency O
. O

However O
, O
differential O
utilization O
of O
these O
determinants O
mediate O
RAR B
- O
RXR B
heterodimer O
binding O
to O
DR B
- O
2 O
and O
DR B
- O
5 O
RAREs O
. O

Galactorrhoea O
is O
a O
rare O
presentation O
of O
an O
empty O
sella O
syndrome O
. O

The O
addition O
of O
novel O
techniques O
, O
such O
as O
histopathologic O
ultrastaging O
, O
immunohistochemistry O
staining O
, O
and O
reverse B
transcriptase I
polymerase O
chain O
reaction O
assays O
, O
will O
help O
increase O
the O
accuracy O
and O
rate O
of O
detection O
of O
disease O
. O

Several O
of O
the O
drugs O
used O
in O
the O
management O
of O
MAC O
have O
been O
associated O
with O
significant O
drug O
interactions O
involving O
the O
cytochrome B
P450 I
( O
CYP B
) O
enzyme O
system O
. O

Arterial O
compliance O
measurements O
using O
intraarterial O
pulse O
contour O
analysis O
and O
a O
modified O
Windkessel O
model O
were O
carried O
out O
in O
19 O
patients O
with O
isolated O
systolic O
hypertension O
( O
> O
or O
= O
160 O
/ O
< O
or O
= O
90 O
mm O
Hg O
) O
and O
compared O
to O
measurements O
in O
29 O
patients O
with O
essential O
hypertension O
( O
diastolic O
blood O
pressure O
[ O
BP O
] O
> O
or O
= O
95 O
mm O
Hg O
) O
and O
47 O
normotensive O
control O
subjects O
. O

Rather O
than O
seeking O
a O
blanket O
characterization O
of O
an O
essentially O
heterogeneous O
group O
, O
it O
may O
be O
more O
useful O
to O
consider O
idiom O
comprehension O
a O
secondary O
manifestation O
of O
semantic O
and O
/ O
or O
pragmatic O
difficulties O
. O

Methylprednisolone O
and O
U O
- O
74006F O
were O
equally O
effective O
at O
preventing O
neurological O
dysfunction O
compared O
to O
the O
control O
group O
( O
p O
less O
than O
0 O
. O
01 O
) O
; O
U O
- O
78517F O
was O
slightly O
less O
effective O
than O
U O
- O
74006F O
and O
methylprednisolone O
but O
was O
significantly O
better O
than O
vehicle O
in O
preventing O
neurological O
dysfunction O
. O

The O
women O
in O
the O
intermediate O
calcium O
group O
had O
approximately O
the O
same O
mean O
BMD O
values O
as O
those O
in O
the O
low O
calcium O
group O
. O

International O
efforts O
are O
now O
being O
made O
to O
standardize O
haemolytic O
test O
conditions O
and O
the O
present O
study O
is O
meant O
as O
a O
contribution O
to O
this O
. O

The O
transition O
from O
expression O
of O
early O
meiotic O
genes O
to O
expression O
of O
middle O
sporulation O
- O
specific O
genes O
occurs O
at O
about O
the O
time O
that O
cells O
exit O
from O
pachytene O
and O
form O
the O
meiosis O
I O
spindle O
. O

NF B
- I
kappaB I
DNA I
- I
protein I
binding O
and O
ICAM B
- I
1 I
promoter I
activity O
were O
enhanced O
by O
IL B
- I
1beta I
and O
these O
effects O
were O
inhibited O
by O
tyrphostin O
23 O
, O
but O
not O
by O
PD O
98059 O
or O
SB O
203580 O
. O

Peripheral O
neuropathy O
in O
the O
cherry O
- O
red O
spot O
- O
myoclonus O
syndrome O
( O
sialidosis O
type O
I O
) O
. O

Using O
the O
two O
- O
hybrid O
screening O
method O
, O
we O
have O
identified O
an O
additional O
component O
of O
the O
CCAAT B
- I
binding I
factor I
. O

The O
myb B
proto I
oncogene I
product I
( O
c B
- I
Myb I
) O
is O
a O
transcriptional O
regulator O
and O
its O
expression O
and O
function O
are O
tightly O
linked O
to O
the O
cellular O
entry O
into O
S O
phase O
and O
DNA O
synthesis O
. O

Both O
the O
p38 B
kinase I
inhibitor O
SB205380 O
( O
SB O
) O
and O
cotransfection O
of O
a O
dominant O
- O
negative O
mutant O
of O
p38 B
kinase I
reduced O
IL B
- I
1beta I
stimulation O
of O
the O
hBNP B
promoter I
. O

CLINICAL O
FEATURES O
: O
This O
30 O
- O
yr O
- O
old O
gravida O
V O
para O
0 O
woman O
presented O
to O
the O
anaesthesia O
consultation O
clinic O
at O
37 O
- O
wk O
gestation O
to O
discuss O
pain O
relief O
options O
for O
labour O
and O
delivery O
. O

The O
TaqI B
and O
HaeIII B
RFLPs O
will O
provide O
tools O
for O
the O
genetic O
analysis O
of O
CR2 B
. O

In O
addition O
, O
because O
SB203580 O
had O
no O
effect O
of O
TNF B
- O
or O
ceramide O
- O
induced O
apoptosis O
, O
our O
results O
strongly O
argue O
against O
a O
role O
for O
p38 B
MAPK I
in O
the O
induction O
of O
TNF B
- O
- O
or O
ceramide O
- O
induced O
apoptosis O
. O

Cognitive O
measures O
of O
attention O
included O
the O
Stroop O
and O
Controlled O
Oral O
Word O
Association O
Test O
using O
the O
letters O
" O
C O
, O
" O
" O
F O
, O
" O
and O
" O
L O
" O
( O
COWAT O
, O
CFL O
version O
) O
. O

Ptx1 B
belongs O
to O
an O
expanding O
family O
of O
bicoid B
- I
related I
vertebrate I
homeobox I
genes I
. O

Effects O
of O
dietary O
lipids O
on O
whole O
- O
body O
retention O
and O
organ O
distribution O
of O
organic O
and O
inorganic O
mercury O
in O
mice O
. O

Differential O
transcription O
of O
plastome O
- O
encoded O
genes O
in O
the O
mesophyll O
and O
bundle O
- O
sheath O
chloroplasts O
of O
the O
monocotyledonous O
NADP B
- I
malic I
enzyme I
- O
type O
C4 O
plants O
maize O
and O
Sorghum O
. O

Sequential O
immunoprecipitation O
showed O
all O
detectable O
p53 B
to O
be O
of O
the O
PAb246 B
+ I
class O
in O
each O
LPV O
- O
transformed O
cell O
line O
, O
suggesting O
that O
the O
stable O
p53 B
was O
indeed O
wild O
type O
. O

Tissue O
- O
specific O
gene O
regulation O
of O
eukaryotic O
organisms O
is O
to O
a O
large O
extent O
mediated O
by O
transcription O
factors O
that O
interact O
with O
genomic O
DNA O
sequences O
in O
a O
sequence O
- O
specific O
manner O
. O

Enhanced O
quantum O
correlations O
in O
bound O
higher O
- O
order O
solitons O
Quantum O
effects O
in O
N O
- O
bound O
solitons O
can O
be O
drastically O
enhanced O
compared O
to O
the O
fundamental O
soliton O
. O

The O
data O
suggest O
that O
compromised O
fibrinolytic O
capacity O
may O
be O
a O
contributing O
factor O
in O
the O
development O
of O
thrombosis O
in O
patients O
with O
the O
lupus O
anticoagulant O
. O

The O
MEE O
was O
significantly O
greater O
than O
the O
BEE O
and O
significantly O
less O
than O
the O
calculated O
energy O
expenditure O
. O

These O
results O
suggest O
that O
E6010 O
may O
be O
of O
clinical O
value O
in O
the O
treatment O
of O
coronary O
occlusion O
. O

To O
better O
understand O
the O
relationship O
between O
expression O
of O
an O
oncogene O
and O
genetic O
instability O
, O
we O
have O
studied O
a O
cell O
line O
expressing O
an O
activated O
human B
Ha I
- I
ras I
under O
the O
control O
of O
bacterial B
lactose I
operon I
regulatory I
elements I
for O
changes O
in O
methotrexate O
resistance O
and O
dihydrofolate B
reductase I
( O
dhfr B
) O
gene O
amplification O
following O
mutant B
Ha I
- I
ras I
induction O
. O

On O
a O
variety O
of O
fronts O
, O
chronic O
bacterial O
infection O
is O
being O
found O
to O
be O
significantly O
associated O
with O
the O
development O
of O
atherosclerosis O
and O
the O
clinical O
complications O
of O
unstable O
angina O
, O
myocardial O
infarction O
, O
and O
stroke O
. O

Long O
- O
term O
results O
and O
curative O
mechanisms O
of O
vesicoureteral O
reflux O
by O
endoscopic O
injection O
of O
blood O
. O

Inspection O
of O
X O
. O
tropicalis O
, O
mouse O
and O
human B
U6 I
DNA I
upstream I
sequences I
revealed O
the O
presence O
of O
a O
TATA O
box O
as O
well O
as O
of O
the O
proximal O
and O
enhancer O
( O
octamer O
motif O
) O
elements O
contained O
in O
snRNA O
genes O
transcribed O
by O
RNA B
polymerase I
II I
. O

Characterization O
of O
an O
unusual O
tRNA O
- O
like O
sequence O
found O
inserted O
in O
a O
Neurospora O
retroplasmid O
. O

Repeated O
time O
course O
studies O
in O
the O
same O
patient O
were O
reproducible O
although O
the O
absolute O
concentrations O
showed O
some O
variation O
. O

The O
present O
work O
represents O
an O
attempt O
to O
develop O
a O
method O
for O
measuring O
relative O
blood O
flow O
in O
intestinal O
capillaries O
, O
by O
the O
use O
of O
sodium O
fluorescein O
( O
Na O
- O
F O
) O
as O
an O
indicator O
substance O
. O

The O
human B
PAX8 I
gene I
generates O
at O
least O
five O
different O
alternatively O
spliced O
transcripts O
encoding O
different O
PAX8 B
isoforms I
. O

These O
data O
show O
that O
both O
Sp1 B
and O
Sp3 B
transcription I
factors I
upregulate O
HGF B
promoter I
activity O
by O
binding O
to O
the O
Sp1 B
binding I
site I
at O
position O
- O
318 O
to O
- O
303 O
bp O
in O
the O
HGF B
gene I
promoter I
. O

After O
a O
1 O
- O
h O
rest O
( O
for O
T2 O
return O
to O
baseline O
) O
, O
subjects O
repeated O
the O
5 O
- O
min O
protocol O
, O
followed O
by O
a O
final O
MRI O
/ O
MRS O
. O

The O
cells O
arrest O
in O
the O
G1 O
phase O
of O
the O
cell O
cycle O
and O
grow O
a O
projection O
towards O
one O
another O
forming O
a O
shmoo O
projection O
. O

Mammalian O
homologues O
of O
the O
Polycomb B
- I
group I
gene I
Enhancer I
of I
zeste I
mediate O
gene O
silencing O
in O
Drosophila O
heterochromatin O
and O
at O
S O
. O
cerevisiae O
telomeres O
. O

With O
six O
reports O
in O
the O
literature O
that O
show O
there O
is O
an O
order O
to O
melanoma O
nodal O
metastases O
and O
that O
the O
SLN O
histology O
is O
reflective O
of O
the O
histology O
of O
the O
remainder O
of O
the O
nodal O
basin O
, O
the O
more O
conservative O
SLN O
biopsy O
can O
be O
performed O
to O
adequately O
stage O
nodally O
the O
patient O
with O
melanoma O
. O

The O
results O
obtained O
with O
[ B
F304 I
] I
R2 I
indicate O
that O
structural O
changes O
in O
E B
. I
coli I
R2 I
in O
the O
vicinity O
of O
this O
helix O
distortion O
can O
influence O
the O
catalytic O
activity O
of O
the O
holoenzyme O
. O

Cloning O
and O
characterization O
of O
the O
5 O
' O
flanking O
region O
of O
human B
ATP I
- I
citrate I
lyase I
gene I
. O

Cryoglobulinaemia O
among O
maintenance O
haemodialysis O
patients O
and O
its O
relation O
to O
hepatitis O
C O
infection O
. O

The O
S3 B
protein I
also O
contains O
a O
DNA O
repair O
activity O
, O
efficiently O
processing O
8 O
- O
oxoguanine O
residues O
in O
DNA O
via O
an O
N B
- I
glycosylase I
/ O
apurinic B
- I
apyrimidinic I
( I
AP I
) I
lyase I
activity O
. O

25 O
patients O
received O
20 O
mgs O
of O
hyoscine O
N O
- O
butyl O
bromide O
and O
the O
rest O
5 O
mgs O
propinoxate O
, O
i O
. O
v O
. O
plus O
diazepam O
. O

We O
present O
here O
a O
statistical O
technique O
we O
developed O
to O
identify O
the O
sequence O
elements O
that O
may O
be O
responsible O
for O
this O
cell O
cycle O
regulation O
. O

Computed O
tomographic O
( O
CT O
) O
findings O
of O
pancreatic O
ductal O
adenocarcinoma O
were O
studied O
with O
the O
combined O
method O
of O
early O
enhancement O
CT O
and O
high O
dose O
enhancement O
CT O
in O
72 O
carcinomas O
. O

No O
other O
transcripts O
of O
different O
length O
could O
be O
detected O
after O
prolonged O
exposure O
. O

We O
conclude O
, O
therefore O
, O
that O
in O
the O
absence O
of O
E1A B
, O
E4orf4 B
is O
sufficient O
by O
itself O
to O
trigger O
a O
p53 B
- O
independent O
apoptosis O
pathway O
that O
may O
operate O
independently O
of O
the O
known O
zVAD B
- O
inhibitable O
caspases B
, O
and O
that O
may O
involve O
an O
as O
yet O
uncharacterized O
mechanism O
. O

The O
antiserum O
used O
for O
immunoblotting O
specifically O
supershifts O
uteroglobin B
promoter I
- I
protein I
complexes I
in O
gel O
shift O
experiments O
. O

The O
nucleotide O
sequence O
of O
the O
estrogen B
receptor I
gene I
of I
Oreochromis I
aureus I
( O
OaER B
) O
indicates O
that O
the O
hormone O
- O
binding O
E O
domain O
is O
composed O
of O
4 O
exons O
interspersed O
by O
short O
introns O
of O
only O
0 O
. O
18 O
- O
1 O
. O
3 O
kb O
each O
. O

The O
expression O
of O
Scmh1 B
and O
rae28 B
/ O
mph1 B
is O
well O
correlated O
in O
most O
tissues O
of O
embryos O
. O

The O
nucleoporin B
98 I
gene I
( O
NUP98 B
) O
, O
which O
is O
rearranged O
in O
several O
acute O
myeloid O
leukemia O
translocations O
, O
is O
located O
within O
this O
region O
. O

We O
have O
isolated O
and O
sequenced O
cDNA O
sequences O
corresponding O
to O
two O
novel O
genes O
that O
map O
to O
Xq28 O
. O

A O
new O
direct O
hemagglutination O
test O
( O
HI O
- O
GONAVIS O
) O
for O
urinary B
LH I
was O
compared O
with O
the O
serum O
radioimmuno O
- O
assay O
of O
LH B
. O

Diffusion O
epidemic O
models O
with O
incubation O
and O
crisscross O
dynamics O
. O

CERIP B
interaction O
. O

The O
encoded O
Arabidopsis B
U3 I
snRNAs I
can O
be O
folded O
into O
a O
secondary O
structure O
which O
is O
more O
similar O
to O
that O
of O
U3 B
RNAs I
from O
lower O
eukaryotes O
rather O
than O
from O
metazoa O
. O

Therapy O
and O
prevention O
of O
irreversible O
shock O
with O
pharmacological O
doses O
of O
some O
corticosteroids O

Fentanyl O
citrate O
( O
0 O
. O
1 O
mL O
) O
and O
2H5 O
- O
fentanyl O
( O
internal O
standard O
, O
0 O
. O
05 O
mL O
, O
100 O
mg O
/ O
L O
) O
were O
extracted O
with O
Toxi O
- O
A O
tubes O
( O
Toxi O
- O
Lab O
, O
Irvine O
, O
CA O
) O
and O
analyzed O
by O
gas O
chromatography O
- O
mass O
spectrometry O
. O

Positive O
effects O
of O
radioactive O
therapy O
in O
Guarapari O

The O
LT B
- I
beta I
gene I
is O
expressed O
in O
lymphoid O
cells O
and O
organs O
, O
but O
little O
is O
known O
about O
its O
inducible O
regulation O
. O

Ciprofloxacin O
attained O
a O
peak O
serum O
concentration O
of O
1 O
. O
2 O
mg O
/ O
l O
and O
a O
serum O
half O
- O
life O
of O
34 O
min O
. O

Dmyd B
clone O
encodes O
a O
polypeptide O
of O
332 O
amino O
acids O
with O
82 O
% O
identity O
to O
MyoD B
in O
the O
41 O
amino O
acids O
of O
the O
putative O
helix O
- O
loop O
- O
helix O
region O
and O
100 O
% O
identity O
in O
the O
13 O
amino O
acids O
of O
the O
basic O
domain O
proposed O
to O
contain O
the O
essential O
recognition O
code O
for O
muscle O
- O
specific O
gene O
activation O
. O

Identification O
of O
an O
osteocalcin B
gene I
promoter I
sequence I
that O
binds O
AP1 B
. O

Treatment O
is O
based O
on O
antifungal O
drugs O
, O
most O
frequently O
nystatin O
, O
amphotericin O
B O
and O
ketoconazole O
. O

Mutations O
abolishing O
MalK B
function O
not O
only O
result O
in O
inability O
to O
transport O
maltose O
but O
also O
cause O
constitutive O
expression O
of O
the O
maltose B
regulon I
. O

Drosophila B
melanogaster I
casein I
kinase I
II I
( O
DmCKII B
) O
is O
composed O
of O
catalytic O
( O
alpha O
) O
and O
regulatory O
( O
beta O
) O
subunits O
associated O
as O
an O
alpha2beta2 B
heterotetramer I
. O

The O
aims O
of O
this O
study O
were O
to O
examine O
whether O
the O
EORTC O
( O
European O
Organisation O
for O
Research O
and O
Treatment O
of O
Cancer O
) O
QLQ O
- O
C30 O
core O
questionnaire O
alone O
could O
distinguish O
between O
two O
clinically O
different O
groups O
of O
patients O
and O
to O
design O
a O
module O
, O
which O
included O
relevant O
patient O
- O
defined O
gastric O
cancer O
- O
specific O
variables O
. O

On O
both O
indocyanine O
green O
video O
and O
fluorescein O
angiography O
, O
hypofluorescent O
lesions O
were O
almost O
the O
same O
size O
as O
the O
light O
yellow O
area O
and O
the O
dark O
yellow O
center O
of O
a O
placoid O
lesion O
. O

We O
have O
measured O
percentage O
O3 O
uptake O
in O
30 O
adult O
Sprague O
- O
Dawley O
rats O
exposed O
, O
nose O
only O
, O
for O
1 O
hr O
to O
0 O
. O
3 O
, O
0 O
. O
6 O
, O
or O
1 O
. O
0 O
ppm O
O3 O
. O

The O
signal O
- O
to O
- O
noise O
ratio O
( O
SNR O
) O
was O
calculated O
in O
the O
T2 O
- O
weighted O
sequence O
. O

Cell O
rounding O
was O
abrogated O
by O
expression O
of O
either O
kinase O
- O
dead O
forms O
of O
US3 B
PK I
or O
a O
mutant O
protein O
lacking O
the O
acidic O
cluster O
in O
the O
kinase O
regulatory O
domain O
. O

Three O
distinct O
human B
HDACs I
are O
homologous O
to O
RPD3 B
, O
a O
yeast O
transcriptional O
regulator O
. O

The O
4 O
- O
nt O
sequence O
of O
the O
rotavirus O
3 O
' O
TE O
represents O
by O
far O
the O
shortest O
of O
any O
of O
the O
sequence O
enhancers O
known O
to O
stimulate O
translation O
. O

OBJECTIVE O
: O
The O
audiologic O
presentation O
of O
vestibular O
schwannoma O
( O
VS O
) O
associated O
with O
neurofibromatosis O
type O
2 O
( O
NF2 O
) O
has O
not O
been O
well O
characterized O
. O

Expression O
of O
these O
chemokines O
is O
similar O
in O
that O
both O
require O
the O
NF B
- I
kappa I
B I
element I
and O
additional O
regions O
such O
as O
the O
CAAT B
/ I
enhancer I
binding I
protein I
( O
C B
/ I
EBP I
) O
element O
of O
the O
IL B
- I
8 I
promoter I
. O

The O
meaning O
of O
hope O
in O
health O
and O
illness O
. O

Assessment O
of O
two O
anion O
- O
exchange O
resins O
for O
direct O
use O
in O
the O
screening O
method O
for O
urinary O
porphobilinogen O
. O

Aggravation O
of O
peripheral O
- O
blood O
cytopenia O
during O
the O
first O
weeks O
and O
hypocellularity O
of O
bone O
- O
marrow O
aspirates O
at O
the O
end O
of O
therapy O
suggest O
that O
low O
- O
dose O
Ara O
C O
exerts O
its O
main O
activity O
by O
suppression O
of O
leukaemic O
growth O
, O
rather O
than O
by O
induction O
of O
differentiation O
in O
malignant O
cells O
. O

All O
depressive O
groups O
showed O
improved O
error O
measures O
post O
- O
treatment O
. O

Triaxiality O
in O
the O
proton O
- O
neutron O
interacting O
boson O
model O
: O
Perturbed O
O O
( O
6 O
) O
symmetry O
with O
application O
to O
the O
mass O
A O

The O
best O
sequences O
for O
pelvic O
study O
are O
, O
in O
our O
experience O
, O
IR O
or O
short O
TR O
spin O
- O
echo O
for O
T1 O
- O
weighted O
images O
, O
and O
spin O
- O
echo O
with O
1200 O
TR O
for O
T2 O
images O
. O

Cells O
were O
co O
- O
infected O
simultaneously O
with O
IBV O
, O
the O
recombinant O
FPV O
( O
rFPV O
) O
containing O
the O
D B
- O
RNA I
sequence O
and O
a O
second O
rFPV O
expressing O
T7 B
RNA I
polymerase I
for O
the O
initial O
expression O
of O
the O
D B
- I
RNA I
transcript I
, O
subsequently O
rescued O
by O
helper O
IBV O
. O

Seasonal O
variation O
of O
the O
cadmium O
content O
of O
Murex O
trunculus O
in O
a O
non O
- O
cadmium O
polluted O
environment O
. O

These O
results O
are O
discussed O
in O
relation O
to O
predicted O
isoform B
L I
diversity O
across O
human O
tissues O
and O
cells O
. O

Doctors O
given O
intrathecal O
methotrexate O
had O
headache O
when O
20 O
gauge O
standard O
needle O
was O
used O
. O

The O
AP B
- I
1 I
recruited O
by O
ARF1 B
. I
GTP I
is O
released O
from O
the O
Golgi O
membrane O
by O
treatment O
with O
1 O
M O
Tris O
- O
HCl O
( O
pH O
7 O
) O
or O
upon O
reincubation O
at O
37 O
degreesC O
, O
whereas O
AP B
- I
1 I
recruited O
with O
GTPgammaS B
or O
by O
a O
constitutively O
active O
point O
mutant O
, O
ARF1 B
( I
Q71L I
) I
, O
remains O
membrane O
bound O
after O
either O
treatment O
. O

The O
formation O
of O
tumorlike O
lesions O
in O
the O
cockroach O
leucophaea O
maderae O
after O
anal O
blockage O
. O

Fine O
tangled O
pili O
expressed O
by O
Haemophilus O
ducreyi O
are O
a O
novel O
class O
of O
pili O
. O

We O
found O
remarkable O
differences O
in O
the O
expression O
patterns O
of O
MTB B
- I
Zf I
mRNA I
and O
two O
other O
hybridizable O
mRNAs O
of O
5kb O
and O
8 O
. O
5 O
kb O
when O
human O
brain O
and O
primary O
brain O
tumors O
were O
compared O
. O

The O
two O
redox O
centers O
in O
the O
protein O
, O
FAD O
and O
a O
[ O
4Fe4S O
] O
+ O
2 O
, O
+ O
1 O
cluster O
, O
are O
present O
in O
a O
64 O
- O
kDa O
monomer O
. O

The O
pra2 B
gene I
encodes O
a O
pea B
( I
Pisum I
sativum I
) I
small I
GTPase I
belonging O
to O
the O
YPT B
/ O
rab B
family O
, O
and O
its O
expression O
is O
down O
- O
regulated O
by O
light O
, O
mediated O
by O
phytochrome B
. O

Carotid O
body O
chemoreceptor O
activity O
as O
recorded O
from O
the O
petrosal O
ganglion O
in O
cats O
. O

Human B
and I
murine I
cytotoxic I
T I
lymphocyte I
serine I
proteases I
: O
subsite O
mapping O
with O
peptide O
thioester O
substrates O
and O
inhibition O
of O
enzyme O
activity O
and O
cytolysis O
by O
isocoumarins O
. O

To O
explore O
further O
the O
role O
of O
the O
carboxyl O
- O
terminal O
domain O
in O
determining O
the O
cell O
cycle O
function O
of O
CDC34 B
, O
we O
constructed O
and O
characterized O
genes O
encoding O
chimeric B
E2s I
incorporating O
sequences O
from O
CDC34 B
and O
the O
related O
but O
functionally O
distinct O
E2 B
RAD6 I
( O
UBC2 B
) O
. O

MAPKK B
kinase I
/ O
MEKK B
phosphorylates O
and O
activates O
its O
downstream O
protein O
kinase O
, O
MAPK B
kinase I
/ O
MEK B
, O
which O
in O
turn O
activates O
MAPK B
. O

Kondo O
effect O
in O
Cu O
( O
Fe O
) O
films O
. O

However O
, O
ADAR1 B
and O
the O
related O
deaminase O
ADAR2 B
showed O
significant O
expression O
in O
all O
regions O
of O
the O
brain O
examined O
, O
including O
cortex O
, O
hippocampus O
, O
olfactory O
bulb O
, O
and O
striatum O
, O
where O
the O
5 B
- I
HT2CR I
pre I
- I
mRNA I
was O
extensively O
edited O
. O

To O
investigate O
its O
transcription O
, O
1 O
. O
1 O
kilobases O
of O
the O
5 O
' O
- O
flanking O
sequence O
were O
fused O
to O
a O
luciferase B
reporter I
gene I
. O

Influence O
of O
copper O
addition O
. O

The O
0 O
. O
018 O
- O
inch O
FloWire O
provides O
a O
high O
- O
fidelity O
continuous O
Doppler O
signal O
. O

Monosulfates O
of O
16 O
- O
oxygenated O
ketonic O
C19 O
steroids O
in O
adult O
human O
urine O
. O

Under O
anaerobic O
conditions O
( O
a O
50 O
- O
day O
experiment O
) O
these O
organisms O
degraded O
15 O
- O
18 O
% O
of O
the O
TEM O
, O
20 O
- O
25 O
% O
of O
some O
alkanes O
, O
and O
15 O
- O
18 O
% O
of O
selected O
polycyclic O
aromatic O
hydrocarbons O
. O

In O
addition O
, O
WR O
- O
3689 O
, O
WR O
- O
109342 O
, O
and O
WR O
- O
168643 O
were O
used O
with O
per O
os O
administration O
to O
determine O
hematopoietic O
lethality O
. O

Further O
, O
we O
will O
review O
our O
evaluation O
of O
the O
survival O
rate O
and O
prognostic O
factors O
for O
9 O
, O
262 O
patients O
from O
1981 O
to O
1996 O
. O

MEASURES O
: O
SF O
- O
36 O
questionnaires O
were O
completed O
by O
patients O
at O
both O
initial O
and O
discharge O
examinations O
. O

In O
contrast O
, O
corticotropin B
releasing I
factor I
( O
CRF B
) O
, O
injected O
centrally O
produces O
a O
suppression O
of O
punished O
and O
non O
- O
punished O
responding O
in O
the O
conflict O
test O
consistent O
with O
its O
hypothesized O
role O
in O
mediating O
behavioral O
responses O
to O
stress O
. O

Temporal O
recruitment O
of O
the O
mSin3A B
- O
histone B
deacetylase I
corepressor O
complex O
to O
the O
ETS B
domain I
transcription I
factor I
Elk I
- I
1 I
. O

The O
cluster O
was O
mapped O
to O
nucleotides O
- O
464 O
to O
- O
434 O
( O
relative O
to O
nucleotide O
A O
in O
the O
initiation O
codon O
) O
in O
genomic O
DNA O
. O

In O
addition O
, O
FOP B
- O
FGFR1 B
- O
expressing O
cells O
show O
constitutive O
phosphorylation O
of O
the O
positive O
regulator O
of O
translation O
p70S6 B
kinase I
; O
this O
phosphorylation O
is O
inhibited O
by O
PI3 B
- I
kinase I
and O
mTOR B
( O
mammalian B
target I
of I
rapamycin I
) O
inhibitors O
. O

The O
authors O
report O
two O
cases O
of O
orbital O
cavernous O
hemangioma O
diagnosed O
by O
Tc O
- O
99m O
RBC O
SPECT O
. O

Ciprofloxacin O
levels O
in O
serum O
and O
blister O
fluid O
at O
the O
end O
of O
the O
dosing O
interval O
( O
8 O
h O
) O
were O
superior O
or O
almost O
superior O
to O
MICs O
for O
sensitive O
organisms O
including O
Pseudomonas O
aeruginosa O
. O

Antibiotic O
prophylaxis O
in O
a O
surgical O
setting O

We O
identified O
clones O
of O
Chlamydomonas O
genomic O
DNA O
that O
rescued O
the O
Ca O
( O
2 O
+ O
) O
- O
dependent O
axonemal O
microtubule O
severing O
defect O
of O
fa1 B
mutants I
. O

These O
changes O
persisted O
during O
the O
first O
week O
. O

Hyperlipemia O
has O
been O
recently O
described O
in O
children O
with O
migraine O
, O
being O
suggested O
that O
this O
alteration O
is O
in O
the O
base O
of O
the O
disease O
. O

Plasma B
hemopexin I
homeostasis O
during O
the O
acute O
phase O
response O
. O

The O
newly O
identified O
repressor O
element O
is O
a O
rare O
example O
of O
a O
naturally O
occurring O
perfect O
palindromic O
binding O
motif O
for O
the O
NF B
- I
kappa I
B I
/ O
Rel B
family O
of O
transcription O
factors O
. O

Tryptophan O
induction O
prevents O
Rho B
- O
dependent O
transcription O
termination O
in O
the O
leader O
region O
of O
the O
operon O
. O

There O
remains O
the O
physiological O
problem O
- O
- O
one O
which O
has O
always O
been O
foremost O
in O
Moruzzi O
' O
s O
thinking O
about O
the O
intrinsic O
regulation O
of O
brain O
activity O
- O
- O
of O
how O
the O
separate O
actions O
of O
the O
different O
arousal O
systems O
are O
brought O
together O
into O
a O
functional O
whole O
. O

The O
nramp2 B
gene I
is O
comprised O
of O
17 O
exons O
and O
spans O
more O
than O
36 O
kb O
. O

The O
immune O
response O
seems O
to O
be O
partially O
responsible O
for O
both O
protection O
and O
the O
destructive O
consequences O
of O
chlamydial O
infection O
. O

Results O
: O
hepatic O
hydatidosis O
: O
177 O
patients O
. O

This O
is O
similar O
to O
previous O
reports O
of O
muscle B
creatine I
phosphokinase I
release O
in O
psychiatric O
patients O
. O

The O
majority O
of O
putative O
TSC2 B
mutations I
were O
found O
in O
sporadic O
rather O
than O
TSC2 B
- O
linked O
families O
. O

Murine B
ELAM I
- I
1 I
is O
encoded O
by O
a O
single O
- O
copy O
gene O
, O
spanning O
about O
13 O
kb O
, O
which O
is O
structurally O
organized O
into O
14 O
exons O
and O
13 O
introns O
; O
very O
similar O
to O
that O
of O
its O
human O
counterpart O
. O

A O
biopsy O
specimen O
taken O
from O
the O
frontal O
branch O
of O
the O
right O
superficial O
artery O
revealed O
segmental O
intimal O
thickening O
consistent O
with O
intimal O
fibroplasia O
type O
FMD O
upon O
histological O
examination O
. O

Thus O
in O
this O
study O
we O
used O
in O
vitro O
methods O
to O
directly O
evaluate O
whether O
isolated O
human O
saphenous O
vein O
segments O
respond O
to O
vasoconstrictor O
agents O
at O
arterial O
pressure O
levels O
. O

We O
found O
that O
the O
PRP20 B
gene I
is O
identical O
to O
the O
yeast B
SRM1 I
gene I
( O
Clark O
and O
Sprague O
1989 O
) O
. O

Basal O
JNK B
MAPK I
kinase I
activity O
was O
also O
specifically O
induced O
by O
deltaEGFR B
, O
which O
correlated O
with O
increased O
phosphorylation O
of O
a O
54 B
- I
kDa I
JNK2 I
protein I
observed O
in O
deltaEGFR B
- O
containing O
cells O
. O

Deletion O
analysis O
defines O
distinct O
functional O
domains O
for O
protein O
- O
protein O
and O
nucleic O
acid O
interactions O
in O
the O
ORF1 B
protein I
of O
mouse O
LINE O
- O
1 O
. O

PMA O
produced O
similar O
results O
, O
but O
the O
induction O
of O
the O
WT O
AP1 B
c B
- I
jun I
promoter O
- O
CAT B
plasmid O
was O
smaller O
. O

The O
determination O
of O
antithrombin B
III I
( O
AT B
III I
) O
and O
its O
clinical O
significance O

One O
ORF O
extends O
from O
nucleotides O
415 O
to O
1620 O
, O
encodes O
402 O
amino O
acids O
, O
and O
is O
preceded O
by O
a O
ribosome O
- O
binding O
site O
. O

Glucose O
consumption O
of O
the O
human O
brain O
under O
the O
influence O
of O
intravenous O
infusions O
of O
glucose O
, O
glucagon B
and O
glucose B
- I
insulin I

D O
. O
, O
Hughes O
, O
F O
. O

Deletion O
of O
CDC5 B
was O
lethal O
and O
resulted O
in O
a O
dumbbell O
- O
shaped O
terminal O
morphology O
, O
with O
the O
nuclei O
almost O
divided O
but O
still O
connected O
. O

These O
cDNAs O
were O
found O
to O
be O
derived O
from O
the O
3 O
" O
- O
untranslated O
region O
( O
3 O
" O
- O
UTR O
) O
of O
the O
methyl B
- I
CpG I
- I
binding I
protein I
2 I
gene I
( O
MeCP2 B
) O
. O

A O
) O
During O
O2 O
- O
ischemia O
both O
extracellular O
[ O
K O
+ O
] O
and O
change O
of O
pH O
in O
the O
subepicardium O
are O
significantly O
less O
than O
in O
the O
midmyocardium O
. O

The O
generation O
of O
dorso O
- O
ventral O
polarity O
during O
Drosophila O
embryogenesis O
is O
regulated O
by O
the O
action O
of O
12 O
maternally O
expressed O
gene O
products O
, O
the O
dorsal O
group O
. O

Ultimobranchial O
body O
and O
parathyroid O
glands O
of O
the O
freshwater O
snake O
Natrix O
piscator O
in O
response O
to O
vitamin O
D3 O
administration O
. O

These O
observations O
suggest O
that O
the O
AF O
- O
1 O
region O
of O
PPARalpha B
is O
partially O
silenced O
by O
corepressor O
proteins O
, O
which O
might O
interact O
in O
a O
phosphorylation O
- O
dependent O
manner O
. O

Two O
authors O
, O
two O
journal O
titles O
, O
two O
drug O
names O
, O
and O
two O
topics O
of O
a O
general O
medical O
nature O
were O
retrieved O
under O
identical O
circumstances O
and O
conditions O
on O
the O
hosts O
DATA O
- O
STAR O
, O
DIALOG O
, O
DIMDI O
, O
and O
STN O
. O

However O
, O
in O
a O
variation O
of O
this O
assay O
in O
which O
the O
protease O
is O
omitted O
, O
the O
mutant O
enzymes O
exhibited O
substantial O
levels O
of O
prolyl B
isomerase I
activity O
( O
5 O
- O
20 O
% O
of O
wild O
- O
type O
) O
, O
revealing O
that O
these O
mutations O
confer O
sensitivity O
to O
protease O
digestion O
and O
that O
the O
classic O
in O
vitro O
assay O
for O
prolyl B
isomerase I
activity O
may O
be O
misleading O
. O

As O
a O
test O
of O
this O
hypothesis O
, O
we O
predicted O
that O
mice O
which O
have O
altered O
expression O
of O
class O
I O
gene O
products O
, O
the O
beta2 B
- I
microglobulin I
knockout O
mice O
, O
[ O
beta2m B
( O
- O
/ I
- O
) O
] O
, O
would O
develop O
Fe O
overload O
. O

Nucleotide O
sequence O
of O
the O
gag B
gene I
and O
gag B
- O
pol B
junction O
of O
feline O
leukemia O
virus O
. O

Clinical O
trial O
endpoints O
based O
on O
magnitude O
of O
reduction O
in O
HIV O
- O
1 O
RNA O
levels O
provide O
an O
important O
complement O
to O
endpoints O
based O
on O
percentage O
of O
patients O
achieving O
complete O
virologic O
suppression O
. O

It O
also O
describes O
the O
preliminary O
results O
of O
a O
Phase O
II O
trial O
going O
on O
at O
the O
Central O
Institute O
for O
Tumors O
in O
Zagreb O
, O
using O
a O
combination O
of O
cis O
- O
DDP O
, O
5 O
- O
fluorouracil O
and O
vincristine O
. O

On O
the O
basis O
of O
the O
physical O
interactions O
between O
Sxl B
and O
Snf B
, O
we O
present O
a O
model O
for O
Sxl B
splicing O
regulation O
. O

Though O
it O
has O
been O
established O
that O
skinfold O
anthropometry O
has O
severe O
limitations O
as O
a O
method O
of O
deriving O
total O
body O
fat O
( O
TBF O
) O
, O
the O
possibility O
that O
the O
problem O
might O
be O
related O
more O
to O
the O
assumptions O
implicit O
in O
densitometry O
has O
to O
be O
addressed O
. O

The O
average O
mean O
difference O
compared O
with O
the O
low O
and O
the O
intermediate O
calcium O
group O
was O
11 O
% O
for O
the O
femoral O
neck O
, O
8 O
- O
11 O
% O
for O
the O
lumbar O
spine O
and O
5 O
- O
6 O
% O
for O
total O
body O
BMDs O
. O

We O
cloned O
the O
BamHI B
- I
F I
fragment I
from O
the O
left O
end O
of O
Ad4 O
in O
pUC13 O
- O
1 O
between O
the O
SalI B
and O
BamHI B
sites I
in O
order O
to O
carry O
out O
the O
structural O
analysis O
of O
the O
E1A B
region I
of I
Ad4 I
. O

We O
reviewed O
81 O
patients O
with O
dementia O
and O
autopsy O
findings O
of O
Alzheimer O
' O
s O
disease O
( O
AD O
) O
to O
identify O
patients O
with O
seizures O
or O
myoclonus O
after O
onset O
of O
dementia O
. O

BACKGROUND O
: O
In O
lung O
protective O
strategy O
, O
positive O
end O
- O
expiratory O
pressure O
( O
PEEP O
) O
slightly O
higher O
than O
the O
Pflex O
( O
the O
airway O
pressure O
corresponding O
to O
the O
lower O
inflection O
point O
( O
LIP O
) O
on O
the O
inspiratory O
pressure O
- O
volume O
( O
P O
- O
V O
) O
curve O
measured O
with O
ZEEP O
) O
is O
generally O
recommended O
. O

3 O
. O

In O
both O
RAW O
cells O
and O
the O
microglia O
cell O
line O
EOC20 O
, O
two O
IFN B
- I
gamma I
- I
activated I
transcription I
factors I
, O
STAT B
- I
1alpha I
and O
IRF B
- I
1 I
, O
bind O
the O
GAS B
and O
IRF B
elements I
, O
respectively O
. O

Thermal O
changes O
of O
the O
bovine O
uterus O
following O
administration O
of O
estradiol O
- O
17beta O
. O

The O
starting O
dose O
was O
8 O
mg O
/ O
m2 O
administered O
intravenously O
( O
IV O
) O
as O
a O
short O
infusion O
daily O
for O
5 O
days O
, O
repeated O
every O
3 O
weeks O
with O
dose O
adjustments O
depending O
on O
patient O
tolerance O
. O

In O
agreement O
with O
other O
authors O
, O
we O
recommend O
the O
procedure O
as O
a O
safe O
one O
, O
providing O
satisfactory O
results O
. O

After O
inhibition O
of O
monoamine B
oxidase I
with O
pargyline O
( O
10 O
mg O
/ O
kg O
. O
i O
. O
v O
. O
) O
, O
the O
effects O
of O
tyramine O
( O
1000 O
micrograms O
) O
injected O
into O
the O
hepatic O
artery O
were O
potentiated O
. O

Helenalin B
triggers O
a O
CD95 B
death I
receptor I
- O
independent O
apoptosis O
that O
is O
not O
affected O
by O
overexpression O
of O
Bcl B
- I
x I
( I
L I
) I
or O
Bcl B
- I
2 I
. O

Of O
the O
47 O
women O
with O
AGCUS O
, O
16 O
had O
intraepithelial O
or O
invasive O
neoplasms O
( O
34 O
% O
; O
95 O
% O
confidence O
interval O
, O
21 O
- O
49 O
% O
) O
, O
including O
9 O
low O
or O
high O
grade O
squamous O
intraepithelial O
lesions O
, O
1 O
adenocarcinoma O
in O
situ O
of O
the O
cervix O
, O
3 O
adenocarcinomas O
of O
the O
cervix O
, O
2 O
adenocarcinomas O
of O
the O
endometrium O
and O
1 O
adenoid O
basal O
cell O
carcinoma O
of O
the O
cervix O
. O

The O
DF3 B
/ O
MUC1 B
gene O
encodes O
a O
high B
molecular I
weight I
mucin I
- I
like I
glycoprotein I
which O
is O
overexpressed O
at O
the O
transcriptional O
level O
in O
the O
majority O
of O
human O
breast O
cancers O
. O

The O
organization O
of O
the O
MBP B
- I
A I
gene I
is O
very O
similar O
to O
the O
arrangement O
of O
the O
gene O
encoding O
the O
highly O
homologous O
pulmonary B
surfactant I
apoprotein I
, O
although O
one O
of O
the O
intron O
positions O
is O
shifted O
by O
a O
single O
amino O
acid O
. O

Hip O
fracture O
mortality O
and O
prospective O
payment O
system O
. O

These O
results O
indicate O
that O
patients O
with O
SS O
and O
evidence O
of O
exercise O
- O
induced O
ST O
- O
segment O
depression O
may O
have O
decreased O
myocardial O
oxygen O
supply O
due O
to O
low O
hemoglobin B
levels O
and O
increased O
myocardial O
oxygen O
demand O
( O
elevated O
double O
products O
) O
when O
compared O
to O
subjects O
with O
SS O
who O
do O
not O
have O
exercise O
- O
induced O
ST O
- O
segment O
depression O
. O

Although O
hSSTR5 B
displays O
approximately O
75 O
% O
sequence O
identity O
with O
rat B
SSTR5 I
, O
the O
two O
receptors O
display O
significantly O
different O
pharmacological O
profiles O
, O
especially O
with O
respect O
to O
their O
binding O
affinities O
for O
the O
SST O
analogue O
SMS O
201 O
- O
995 O
. O

A O
significant O
and O
specific O
reduction O
in O
binaural O
peak O
amplitude O
and O
area O
of O
the O
echo O
- O
evoked O
middle O
- O
latency O
component O
Pa O
was O
observed O
. O

EMBO O
( O
Eur O
. O

Using O
this O
oligonucleotide O
as O
a O
probe O
, O
an O
8 B
- I
kilobase I
HindIII I
fragment I
of O
genomic O
DNA O
was O
isolated O
and O
subjected O
to O
Sanger O
dideoxy O
DNA O
sequencing O
. O

Arg O
- O
130 O
, O
Gly O
- O
132 O
and O
Lys O
- O
167 O
are O
conserved O
in O
all O
members O
of O
the O
type B
IB I
topoisomerase I
family I
. O

Four O
cDNAs O
( O
Kox4 B
, O
Kox7 B
, O
Kox12 B
, O
and O
Kox15 B
) O
were O
identified O
that O
match O
one O
or O
more O
genomic O
clones O
; O
these O
matches O
were O
confirmed O
by O
nucleotide O
sequence O
analysis O
. O

2 O
. O

Prevalence O
of O
use O
, O
claims O
volume O
, O
and O
expenditures O
were O
compared O
for O
cisapride O
, O
proton O
pump O
inhibitors O
, O
histamine B
- I
2 I
receptor I
antagonists O
, O
and O
the O
prokinetic O
agent O
metoclopramide O
during O
these O
periods O
. O

Relaxant O
effect O
of O
prostaglandin O
E1 O
( O
PGE1 O
) O
and O
papaverine O
( O
PAP O
) O
were O
measured O
in O
strips O
of O
corpus O
cavernosum O
smooth O
muscle O
taken O
from O
a O
healthy O
control O
group O
of O
men O
( O
A O
; O
n O
= O
5 O
) O
, O
from O
arteriogenically O
impotent O
men O
( O
B O
; O
n O
= O
6 O
) O
and O
from O
additionally O
diabetic O
impotent O
men O
( O
C O
; O
n O
= O
5 O
) O
with O
venous O
leakage O
. O

Primer O
extension O
experiments O
using O
dbcAMP O
- O
differentiated O
U937 O
RNA O
indicated O
a O
single O
transcriptional O
initiation O
site O
. O

In O
contrast O
, O
group O
II O
showed O
a O
significant O
improvement O
in O
peak O
VO2 O
with O
rate O
adaptive O
AV O
delay O
compared O
to O
fixed O
AV O
delay O
programming O
( O
P O
less O
than O
0 O
. O
05 O
) O
. O

Effect O
of O
family O
visits O
on O
the O
blood O
pressure O
and O
heart O
rate O
of O
patients O
in O
the O
coronary O
- O
care O
unit O
. O

Sequence O
analysis O
and O
electrophoretic O
mobility O
shift O
experiments O
suggest O
that O
GnSE B
response I
elements I
interact O
, O
in O
these O
two O
regions O
, O
with O
GATA B
- I
and O
LIM B
- I
related I
factors I
, O
respectively O
. O

The O
transfer O
by O
conjugation O
to O
E O
. O
coli O
K12 O
of O
ampicillin O
and O
carbenicillin O
resistance O
was O
obtained O
with O
14 O
strains O
: O
( O
E O
. O
coli O
: O
9 O
, O
C O
. O
freundii O
: O
1 O
, O
K O
. O
pneumoniae O
: O
1 O
, O
E O
. O
cloacae O
: O
2 O
, O
P O
. O
stuartii O
: O
1 O
) O
. O

Wild B
type I
MEKK1 I
enhances O
promoter O
activity O
and O
the O
activity O
can O
be O
inhibited O
by O
dominant B
negative I
MEKK1 I
, O
MEK1 B
, O
MEK7 B
, O
MEK3 B
, O
p38 B
/ O
RK B
, O
and O
c B
- I
Jun I
. O

250 O
, O
302 O
- O
311 O
] O
. O

The O
studies O
permit O
considering O
the O
detected O
shifts O
in O
the O
immunity O
system O
an O
important O
component O
of O
the O
pathogenesis O
of O
chronic O
nontumorous O
diseases O
of O
the O
parotid O
glands O
. O

Coexistence O
of O
Turner O
' O
s O
syndrome O
and O
schizophrenia O

An O
scrR B
promoter I
fragment I
, O
which O
dose O
not O
contain O
a O
sequence O
resembling O
OB B
, O
was O
not O
shifted O
by O
the O
fusion O
protein O
. O

Using O
the O
conflict O
drinking O
test O
as O
a O
model O
, O
we O
studied O
in O
rats O
the O
effect O
of O
the O
nonselective O
beta B
- I
adrenoceptor I
blockers O
pindolol O
and O
cyanopindolol O
which O
bind O
to O
5 B
- I
HT1A I
and I
5 I
- I
HT1B I
receptors I
, O
and O
of O
the O
selective O
beta B
1 I
- I
and I
beta I
2 I
- I
adrenoceptor I
antagonists O
betaxolol O
and O
ICI O
118 O
, O
551 O
, O
respectively O
, O
which O
have O
a O
negligible O
affinity O
for O
5 B
- I
HT I
receptors I
. O

Although O
several O
other O
signal O
transduction O
molecules O
also O
contain O
tandemly O
occurring O
SH3 B
and O
SH2 B
domains I
, O
the O
function O
of O
these O
closely O
spaced O
domains O
is O
not O
well O
understood O
. O

Glycoprotein O
biosynthesis O
in O
Saccharomyces O
cerevisiae O
: O
ngd29 B
, O
an O
N O
- O
glycosylation O
mutant O
allelic O
to O
och1 B
having O
a O
defect O
in O
the O
initiation O
of O
outer O
chain O
formation O
. O

CONCLUSIONS O
: O
In O
the O
majority O
of O
patients O
with O
severe O
ulcerative O
colitis O
, O
circulating O
concentrations O
of O
NOX O
are O
increased O
at O
presentation O
and O
fall O
promptly O
during O
parenteral O
steroid O
therapy O
, O
irrespective O
of O
clinical O
outcome O
. O

The O
duration O
of O
naloxone O
appears O
to O
be O
dose O
- O
related O
, O
statistically O
significant O
up O
to O
approximately O
1 O
. O
5 O
hr O
. O

But O
if O
one O
equalizes O
the O
upper O
limits O
of O
normal O
for O
both O
markers O
to O
a O
common O
95 O
% O
specificity O
, O
the O
tumour O
- O
indicating O
sensitivity O
of O
CA B
19 I
- I
9 I
clearly O
surpasses O
that O
of O
CA B
50 O
. O

These O
motions O
were O
found O
in O
the O
N O
- O
and O
C O
- O
terminal O
tails O
, O
in O
segment O
33 O
- O
35 O
which O
forms O
the O
turn O
between O
beta O
- O
strands O
S1 O
and O
S2 O
, O
and O
residues O
47 O
- O
52 O
located O
in O
a O
long O
loop O
preceding O
strand O
S3 O
. O

Genomic O
Southern O
analysis O
reveals O
10 O
to O
15 O
hybridising O
fragments O
, O
suggesting O
that O
maize O
contains O
a O
small O
gene O
family O
. O

Custom O
- O
made O
orthoses O
play O
an O
invaluable O
role O
in O
achieving O
a O
wide O
range O
of O
therapeutic O
goals O
: O
improving O
function O
and O
pinch O
, O
stabilizing O
individual O
joints O
, O
preventing O
positional O
contractures O
, O
protecting O
joints O
from O
trauma O
, O
stretching O
the O
intrinsic O
muscles O
, O
correcting O
joint O
- O
and O
soft O
- O
tissue O
contractures O
, O
and O
controlling O
inflammation O
. O

Is O
exposure O
to O
a O
patient O
with O
Crohn O
' O
s O
disease O
an O
environmental O
factor O
for O
developing O
the O
disease O
. O

The O
ZAG1 B
protein I
from O
in O
vitro O
translations O
binds O
to O
a O
consensus O
target O
site O
that O
is O
recognized O
by O
the O
AG B
protein I
. O

The O
uap100 B
cis O
- O
acting O
, O
up O
- O
promoter O
, O
constitutive O
mutation O
is O
a O
duplication O
that O
comprises O
two O
GATA B
sites O
and O
suppresses O
weakly O
the O
AREA B
zinc I
finger I
mutation I
but O
does O
not O
alleviate O
the O
need O
for O
functional O
UAY B
and O
AREA B
proteins I
. O

Pituitary O
thyrotropic O
adenoma O
associated O
with O
congenital O
hypothyroidism O
. O

This O
region O
was O
unfavourable O
, O
due O
to O
malaria O
in O
the O
past O
. O

Indeed O
, O
after O
a O
single O
oral O
dose O
, O
the O
role O
of O
the O
mu B
- I
receptor I
agonist O
component O
of O
the O
antinociceptive O
effect O
of O
tramadol O
appears O
to O
be O
minor O
, O
with O
most O
of O
the O
analgesic O
effect O
being O
attributable O
to O
nonopioid O
properties O
of O
the O
parent O
compound O
. O

CHIEF O
OUTCOME O
MEASURES O
: O
Endoluminal O
release O
of O
prostacyclin O
( O
6 O
- O
Keto O
- O
PGF1 O
alpha O
) O
and O
thromboxane O
B2 O
( O
TxB2 O
) O
, O
patency O
, O
EC O
coverage O
and O
cell O
identity O
. O

Somatic O
and O
visceral O
inputs O
to O
the O
thoracic O
spinal O
cord O
of O
the O
cat O
: O
effects O
of O
noxious O
stimulation O
of O
the O
biliary O
system O
. O

We O
have O
produced O
the O
Ure2 B
protein I
to O
high O
yield O
in O
Escherichia O
coli O
from O
a O
synthetic O
gene O
. O

Transfection O
of O
both O
Calu O
- O
6 O
and O
JEG O
- O
3 O
cells O
with O
a O
PKA B
expression O
vector O
resulted O
in O
a O
10 O
- O
fold O
induction O
of O
human B
renin I
transcriptional O
activity O
in O
constructs O
containing O
the O
native O
or O
consensus O
CRE O
and O
5 O
- O
fold O
activation O
in O
a O
construct O
containing O
a O
nonfunctional O
CRE O
. O

We O
undertook O
this O
study O
to O
examine O
the O
relationship O
between O
systemic O
oxygen O
delivery O
( O
DO2 O
) O
and O
tissue O
oxygen O
tension O
( O
TPO2 O
) O
in O
hypovolemic O
shock O
. O

White O
female O
cases O
were O
more O
likely O
to O
have O
worked O
as O
registered O
nurses O
than O
were O
the O
controls O
( O
OR O
= O
1 O
. O
9 O
, O
95 O
% O
CI O
= O
1 O
. O
0 O
- O
3 O
. O
5 O
) O
. O

METHODS O
: O
Children O
with O
attention O
- O
deficit O
/ O
hyperactivity O
disorder O
( O
ADHD O
; O
n O
= O
282 O
) O
, O
all O
subtypes O
, O
ages O
6 O
to O
12 O
years O
, O
were O
randomized O
to O
placebo O
( O
n O
= O
90 O
) O
, O
immediate O
- O
release O
methylphenidate O
( O
IR O
MPH O
) O
3 O
times O
a O
day O
( O
tid O
; O
dosed O
every O
4 O
hours O
; O
n O
= O
97 O
) O
, O
or O
OROS O
MPH O
once O
a O
day O
( O
qd O
; O
n O
= O
95 O
) O
in O
a O
double O
- O
blind O
, O
28 O
- O
day O
trial O
. O

Immunologic O
tolerance O
in O
rats O
during O
13 O
weeks O
of O
inhalation O
exposure O
to O
trimellitic O
anhydride O
. O

They O
cause O
temporary O
disability O
, O
invalidity O
and O
mortality O
of O
a O
great O
part O
of O
the O
productive O
population O
, O
significantly O
damaging O
the O
economic O
status O
of O
the O
country O
. O

Cloning O
and O
sequence O
analysis O
of O
a O
chymotrypsinlike B
protease I
from I
Treponema I
denticola I
. O

METHODS O
AND O
RESULTS O
: O
The O
QTd O
and O
JTd O
intervals O
in O
39 O
children O
with O
long O
QT O
syndrome O
were O
compared O
to O
those O
of O
50 O
normal O
age O
- O
matched O
children O
. O

V B
- I
ErbB I
- O
mediated O
complex O
formation O
and O
transformation O
have O
been O
shown O
to O
occur O
independently O
of O
Ras B
activation O
. O

Both O
parameters O
were O
determined O
in O
an O
individual O
eye O
by O
applying O
the O
dye O
first O
by O
iontophoresis O
and O
then O
by O
intravitreal O
injection O
, O
which O
allows O
the O
influence O
of O
fluorescein O
glucuronide O
on O
the O
fluorophotometric O
measurements O
to O
be O
excluded O
. O

Organization O
of O
exons O
suggests O
that O
, O
similar O
to O
the O
human B
VEGF I
gene I
, O
alternative O
splicing O
generates O
the O
120 O
- O
, O
164 O
- O
, O
and O
188 O
- O
amino O
acid O
isoforms O
, O
but O
does O
not O
predict O
a O
fourth O
VEGF B
isoform I
corresponding O
to O
human B
VEGF206 I
. O

Electron O
microscopy O
of O
pH O
7 O
. O
5 O
- O
treated O
TMV O
particles O
incubated O
in O
SPN O
- O
treated O
wheatgerm O
extract O
or O
rabbit O
reticulocyte O
lysate O
, O
showed O
that O
approximately O
10 O
% O
of O
virions O
complexed O
with O
one O
ribosome O
and O
approximately O
10 O
% O
with O
two O
bound O
ribosomes O
, O
confirming O
that O
omega O
at O
least O
had O
been O
uncoated O
. O

The O
sequence O
contained O
five O
repeats O
of O
the O
hypothetical O
leucine O
zipper O
motif O
: O
one O
is O
in O
the O
N O
- O
terminal O
globular O
domain O
, O
and O
four O
are O
in O
the O
central O
alpha O
- O
helical O
stalk O
. O

Postheparin B
lipoprotein I
lipase I
, O
measured O
using O
fat O
emulsion O
as O
substrate O
, O
also O
was O
significantly O
greater O
in O
female O
rats O
compared O
with O
males O
. O

The O
authors O
report O
a O
case O
of O
a O
painful O
wrist O
related O
to O
a O
" O
Manieux O
muscle O
" O
of O
the O
dorsal O
aspect O
of O
the O
hand O
. O

Genomic O
organization O
of O
the O
human B
multidrug I
resistance I
( O
MDR1 I
) I
gene I
and O
origin O
of O
P B
- I
glycoproteins I
. O

The O
response O
to O
nitroglycerin O
, O
however O
, O
remained O
intact O
regardless O
of O
initial O
reperfusion O
pressure O
. O

Deletion O
analysis O
revealed O
that O
only O
the O
TM O
domain O
of O
E2 B
was O
required O
for O
Golgi O
targeting O
. O

In O
addition O
, O
putative O
binding O
sites O
for O
SH3 B
and O
SH2 B
domains I
are O
present O
in O
the O
amino O
- O
terminal O
half O
of O
the O
molecule O
. O

Since O
nearly O
all O
transformants O
involving O
homeologous O
DNAs O
carried O
a O
single O
recombinant O
plasmid O
in O
both O
Pms O
+ O
and O
Pms O
- O
strains O
, O
stable O
heteroduplex O
DNA O
appears O
less O
likely O
than O
for O
homologous O
DNAs O
. O

All O
rights O
reserved O
. O

One O
new O
ORF O
, O
YBR0224 B
, O
was O
detected O
, O
coding O
for O
a O
protein O
with O
918 O
amino O
acids O
. O

A O
sequence O
- O
ready O
3 O
- O
Mb O
PAC O
contig O
covering O
16 O
breakpoints O
of O
the O
Wilms B
tumor I
/ I
anirida I
region O
of O
human O
chromosome O
11p13 O
. O

The O
MAP B
kinase I
kinase I
kinase I
MLK2 B
co O
- O
localizes O
with O
activated O
JNK B
along O
microtubules O
and O
associates O
with O
kinesin B
superfamily I
motor O
KIF3 B
. O

Bone O
scintigraphy O
is O
an O
excellent O
screening O
test O
for O
bone O
lesions O
and O
Ga O
- O
67 O
scintigraphy O
is O
a O
useful O
tool O
for O
detecting O
inflammatory O
lesions O
. O

Spontaneous O
fracture O
of O
the O
tarsal O
bone O
in O
2 O
cases O
of O
leprosy O

They O
are O
transduced O
through O
TM B
to O
the O
cytoplasmic O
portion O
( O
cytoloops O
and O
the O
C O
- O
terminal O
tail O
) O
of O
the O
receptor O
and O
then O
, O
transferred O
to O
cytoplasmic O
signaling O
molecules O
, O
such O
as O
G B
protein I
. O

Serum B
alkaline I
phosphatase I
and O
creatine B
kinase I
activities O
were O
significantly O
increased O
in O
the O
A O
praelongus O
- O
fed O
pigs O
and O
significantly O
decreased O
in O
Na2SeO4 O
- O
fed O
pigs O
. O

This O
group O
showed O
the O
presence O
of O
bacteria O
infiltrated O
at O
the O
dentinal O
tubules O
coronally O
and O
into O
the O
root O
canals O
. O

Seven O
Zebu O
cattle O
were O
also O
exposed O
to O
challenge O
at O
the O
same O
time O
. O

However O
, O
the O
quasiparticle O
scattering O
rate O
tau O
( O
- O
1 O
) O
is O
such O
that O
Planck O
' O
s O
over O
2pi O
/ O
tau O
approximately O
6t O
( O
perpendicular O
) O
, O
implying O
that O
kappa O
- O
( O
BEDT O
- O
TTF O
) O
2Cu O
( O
NCS O
) O
( O
2 O
) O
meets O
the O
criterion O
used O
to O
identify O
interlayer O
incoherence O
. O

The O
effect O
on O
kidney O
length O
was O
considerably O
prolonged O
compared O
with O
the O
vascular O
effect O
. O

Although O
polyubiquitin O
chains I
linked O
through O
Lys O
( O
29 O
) O
of O
ubiquitin B
have O
been O
implicated O
in O
the O
targeting O
of O
certain O
substrates O
to O
proteasomes O
, O
the O
signaling O
properties O
of O
these O
chains O
are O
poorly O
understood O
. O

Diabetic O
osteopenia O
, O
a O
known O
chronic O
complication O
of O
diabetes O
, O
has O
been O
demonstrated O
with O
alterations O
in O
the O
calcium O
- O
vitamin O
D O
endocrine O
system O
. O

An O
in O
vitro O
Raf B
- I
1 I
kinase I
assay O
, O
however O
, O
failed O
to O
detect O
LPS O
- O
induced O
Raf B
- I
1 I
kinase I
activity O
in O
RAW O
264 O
. O
7 O
cells O
, O
suggesting O
that O
in O
RAW O
264 O
. O
7 O
cells O
, O
Raf B
- I
1 I
kinase I
is O
not O
an O
activating O
component O
of O
the O
LPS O
signaling O
pathway O
regulating O
MAPK B
activity O
or O
sIL B
- I
1Ra I
promoter I
activity O
. O

Phosphorylation O
of O
the O
ras B
GTPase I
- I
activating I
protein I
( O
GAP B
) O
by O
the O
p93c B
- I
fes I
protein I
- I
tyrosine I
kinase I
in O
vitro O
and O
formation O
of O
GAP B
- O
fes B
complexes O
via O
an O
SH2 B
domain O
- O
dependent O
mechanism O
. O

Gingival O
retraction O
for O
class O
V O
restorations O
. O

Two O
methods O
for O
the O
routine O
determination O
of O
blood O
hemoglobin B
oxygen O
affinity O
are O
described O
. O

Thirty O
minutes O
after O
reperfusion O
, O
lung O
lymph O
flow O
( O
QL O
) O
increased O
from O
4 O
. O
3 O
to O
8 O
. O
3 O
ml O
/ O
30 O
minutes O
( O
p O
less O
than O
0 O
. O
05 O
) O
, O
lymph O
/ O
plasma O
protein O
ratio O
was O
unchanged O
from O
0 O
. O
6 O
, O
and O
the O
lymph O
protein O
clearance O
increased O
from O
2 O
. O
6 O
to O
4 O
. O
6 O
ml O
/ O
30 O
minutes O
( O
p O
less O
than O
0 O
. O
05 O
) O
, O
data O
consistent O
with O
increased O
microvascular O
permeability O
. O

U O
. O
S O
. O
A O
. O

RET B
/ O
PTC B
oncogenes O
, O
generated O
by O
chromosomal O
rearrangements O
in O
papillary O
thyroid O
carcinomas O
, O
are O
constitutively O
activated O
versions O
of O
proto B
- I
RET I
, O
a O
gene O
coding O
for O
a O
receptor B
- I
type I
tyrosine I
kinase I
( O
TK B
) O
whose O
ligand O
is O
still O
unknown O
. O

PCC O
6803 O
mutant O
impaired O
in O
cytochrome B
c I
maturation O
. O

STUDY O
OBJECTIVE O
: O
to O
characterize O
gas O
exchange O
and O
cardiopulmonary O
performance O
during O
maximal O
progressive O
arm O
crank O
exercise O
. O

In O
this O
study O
, O
we O
show O
that O
the O
activating O
effect O
of O
this O
mutation O
is O
not O
the O
result O
of O
creating O
an O
exonic O
splicing O
enhancer O
( O
ESE O
) O
or O
disrupting O
a O
putative O
secondary O
structure O
. O

The O
kinase O
domains O
encoded O
in O
the O
COOH O
- O
terminal O
moiety O
are O
94 O
% O
conserved O
; O
the O
NH2 O
- O
terminal O
moieties O
are O
approximately O
65 O
% O
homologous O
, O
suggesting O
this O
region O
may O
encode O
sequences O
conferring O
differential O
regulation O
of O
the O
two O
kinases O
. O

In O
rats O
exposed O
to O
Cd O
for O
30 O
d O
, O
the O
levels O
of O
urinary O
excretion O
of O
cAMP O
after O
treatment O
with O
parathyroid B
hormone I
( O
PTH B
) O
, O
parathyroidectomy O
( O
PTX O
) O
, O
or O
1 O
alpha O
- O
hydroxycholecalciferol O
( O
1 O
alpha O
- O
OH O
- O
D3 O
) O
showed O
almost O
the O
same O
patterns O
as O
those O
of O
control O
rats O
: O
the O
response O
of O
urinary O
cAMP O
to O
treatment O
with O
PTH B
was O
not O
influenced O
by O
continuous O
oral O
administration O
of O
Cd O
. O

The O
goal O
of O
this O
investigation O
was O
to O
determine O
whether O
BMIPP O
uptake O
can O
be O
used O
to O
differentiate O
viable O
myocardium O
from O
scar O
tissue O
soon O
after O
coronary O
thrombolysis O
for O
acute O
myocardial O
infarction O
. O

There O
are O
three O
types O
of O
problems O
: O
problems O
arising O
before O
the O
drug O
enters O
the O
body O
( O
solubilization O
, O
stabilization O
and O
improved O
acceptability O
) O
; O
problems O
associated O
with O
the O
penetration O
and O
fate O
of O
the O
drug O
in O
the O
body O
( O
absorption O
, O
barrier O
crossing O
, O
duration O
of O
action O
) O
; O
problems O
relating O
to O
the O
therapeutic O
target O
( O
selective O
local O
delivery O
) O
. O

In O
the O
absence O
of O
shock O
, O
sepsis O
, O
or O
other O
identifiable O
causes O
of O
lactic O
acidosis O
, O
the O
severe O
anemia O
( O
hemoglobin B
1 O
. O
2 O
g O
/ O
dl O
) O
appeared O
to O
be O
the O
primary O
etiologic O
factor O
. O

The O
cumulative O
experience O
in O
liver O
transplantation O
since O
the O
introduction O
of O
cyclosporine O
A O
has O
confirmed O
its O
efficacy O
in O
the O
treatment O
of O
diverse O
liver O
diseases O
in O
children O
and O
adults O
. O

Northern O
blot O
analysis O
of O
liver O
, O
muscle O
, O
fat O
, O
and O
pituitary O
RNA O
from O
normal O
( O
DwDw O
) O
chickens O
shows O
a O
major O
transcript O
of O
4 O
. O
3 O
kilobases O
( O
kb O
) O
and O
three O
minor O
transcripts O
( O
0 O
. O
8 O
, O
1 O
. O
7 O
, O
and O
3 O
. O
2 O
kb O
) O
, O
which O
correspond O
to O
the O
cGHR B
. O

Copyright O
1999 O
Academic O
Press O
. O

Despite O
a O
lowering O
of O
her O
plasma O
ACTH B
concentration O
during O
therapy O
with O
valproic O
acid O
, O
the O
patient O
' O
s O
tumour O
showed O
no O
evidence O
of O
regression O
while O
she O
was O
taking O
the O
drug O
. O

The O
Htf9 B
- I
a I
gene I
encodes O
the O
RanBP1 B
protein I
, O
a O
major O
partner O
of O
the O
Ran B
GTPase I
. O

STUDY O
DESIGN O
: O
Retrospective O
review O
. O

Sequence O
divergence O
of O
approximately O
16 O
% O
reduced O
transformation O
frequencies O
by O
at O
least O
10 O
- O
fold O
. O

Our O
studies O
suggest O
that O
the O
helicase B
assembly I
protein I
is O
responsible O
for O
loading O
T4 B
gene I
41 I
helicase I
specifically O
at O
replication O
forks O
, O
and O
that O
its O
binding O
sites O
for O
each O
arm O
must O
hold O
more O
than O
six O
, O
but O
not O
more O
than O
12 O
nucleotides O
. O

During O
the O
1st O
week O
of O
treatment O
all O
subjects O
received O
either O
mianserin O
30 O
mg O
or O
clomipramine O
75 O
mg O
once O
daily O
. O

CBD O
exploration O
was O
avoided O
in O
7 O
patients O
with O
normal O
POC O
. O

The O
human B
SHBG I
proximal I
promoter I
was O
analyzed O
by O
DNase B
I I
footprinting O
, O
and O
the O
functional O
significance O
of O
6 O
footprinted O
regions O
( O
FP1 O
- O
FP6 O
) O
within O
the O
proximal O
promoter O
was O
studied O
in O
human O
HepG2 O
hepatoblastoma O
cells O
. O

We O
exposed O
Dorper O
- O
cross O
ewes O
at O
approximately O
120 O
- O
135 O
days O
of O
gestation O
to O
a O
hot O
( O
40 O
degrees O
C O
, O
60 O
% O
relative O
humidity O
) O
and O
a O
cold O
( O
4 O
degrees O
C O
, O
90 O
% O
relative O
humidity O
) O
environment O
and O
to O
treadmill O
exercise O
( O
2 O
. O
1 O
km O
/ O
h O
, O
5 O
degrees O
gradient O
) O
and O
measured O
fetal O
lamb O
and O
ewe O
body O
temperatures O
using O
previously O
implanted O
abdominal O
radiotelemeters O
. O

Previously O
identified O
mutations O
in O
this O
variant O
have O
been O
within O
or O
close O
to O
the O
coding O
region O
of O
exon O
1 O
of O
the O
HMBS B
gene I
, O
the O
only O
exon O
that O
is O
expressed O
solely O
in O
the O
ubiquitous O
isoform O
. O

As O
an O
approach O
to O
the O
determination O
of O
structure O
- O
function O
relationships O
in O
the O
PRP4 B
protein I
, O
we O
have O
isolated O
more O
than O
fifty O
new O
alleles O
of O
the O
PRP4 B
gene I
through O
random O
and O
site O
- O
directed O
mutagenesis O
, O
and O
have O
analyzed O
the O
phenotypes O
of O
many O
of O
them O
. O

Over O
a O
period O
of O
five O
training O
sessions O
( O
2 O
. O
5 O
days O
) O
, O
the O
animals O
learned O
not O
to O
breathe O
, O
and O
the O
number O
of O
stimuli O
received O
in O
the O
fifth O
session O
was O
significantly O
lower O
than O
in O
the O
first O
session O
. O

The O
5 O
' O
- O
flanking O
region O
of O
the O
gene O
, O
approximately O
1 O
. O
1 O
kbp O
long O
, O
had O
no O
known O
regulatory O
elements O
for O
transcription O
such O
as O
TATA O
, O
GC O
, O
and O
CCAAT O
boxes O
. O

We O
find O
that O
Fus3p B
and O
Kss1p B
both O
control O
G1 O
arrest O
through O
multiple O
functions O
that O
operate O
in O
parallel O
with O
Far1p B
. O

Tag B
expression O
induced O
apoptosis O
in O
mammary O
epithelial O
cells O
during O
late O
pregnancy O
. O

The O
composition O
of O
renal O
stones O
analysed O
by O
infrared O
spectroscopy O
. O

14 O
. O
9 O
x O
109 O
L O
- O
1 O
) O
was O
equal O
for O
both O
groups O
, O
but O
CCI O
values O
were O
significantly O
higher O
for O
the O
bedside O
filtered O
PC O
( O
14 O
. O
1 O
+ O
/ O
- O
9 O
. O
5 O
vs O
. O

In O
this O
study O
, O
the O
case O
of O
a O
patient O
presenting O
a O
second O
hepatocellular O
carcinoma O
13 O
years O
after O
resection O
of O
a O
first O
tumor O
of O
the O
same O
type O
is O
reported O
. O

The O
time O
- O
dependent O
changes O
in O
measured O
facility O
of O
outflow O
or O
" O
washout O
phenomenon O
" O
appeared O
to O
result O
from O
the O
gradual O
dissolution O
of O
the O
hyaluronidase B
- O
sensitive O
component O
of O
the O
barriers O
to O
aqueous O
outflow O
in O
the O
canine O
eye O
. O

The O
factors O
defective O
in O
groups O
A O
( O
CIITA B
) O
, O
C B
( O
RFX5 B
) O
and O
D B
( I
RFXAP I
) O
have O
been O
identified O
. O

It O
differed O
significantly O
by O
country O
( O
from O
2 O
. O
4 O
% O
in O
the O
United O
Kingdom O
to O
24 O
. O
7 O
% O
in O
Portugal O
) O
and O
by O
transmission O
category O
( O
2 O
. O
7 O
% O
among O
homo O
/ O
bisexuals O
, O
5 O
. O
8 O
% O
among O
injecting O
drug O
users O
, O
13 O
. O
6 O
% O
among O
heterosexual O
subgroup O
1 O
) O
. O

On O
a O
national O
level O
, O
undiscounted O
costs O
are O
expected O
to O
increase O
up O
to O
approximately O
DF1 O
42 O
million O
annually O
during O
the O
first O
40 O
years O
after O
introduction O
of O
the O
preventative O
strategy O
. O

By O
deletion O
analysis O
, O
we O
identified O
the O
regions O
of O
c B
- I
Jun I
encoding O
transformation O
and O
transactivation O
functions O
. O

A O
survey O
of O
274 O
late O
detected O
cases O
of O
CDH O
born O
in O
the O
years O
1970 O
- O
- O
74 O
is O
presented O
. O

To O
test O
for O
progesterone B
receptor I
- O
mediated O
activation O
of O
transcription O
in O
yeast O
, O
reporter O
plasmids O
were O
constructed O
to O
transform O
yeast O
cells O
expressing O
PRB B
or O
C1C2 B
receptors I
. O

Fli B
- I
1 I
is O
a O
proto O
- O
oncogene O
which O
is O
rearranged O
in O
tumors O
induced O
by O
three O
different O
retroviruses O
, O
Cas O
- O
Br O
- O
E O
, O
F O
- O
MuLV O
, O
and O
10A1 O
. O

Ionic O
composition O
( O
Na O
+ O
, O
K O
+ O
, O
Ca O
+ O
2 O
, O
and O
Mg2 O
+ O
) O
of O
cerebrospinal O
fluid O
and O
feeding O
behavior O
in O
sheep O
. O

Materials O
are O
presented O
of O
a O
comparative O
study O
of O
morphological O
and O
cultural O
properties O
and O
virulence O
of O
liophylized O
cultures O
of O
Leishmania O
tropica O
major O
after O
their O
rehydration O
or O
after O
a O
long O
passage O
on O
the O
NNN O
medium O
. O

CONCLUSIONS O
: O
These O
findings O
imply O
that O
the O
spinal O
cord O
is O
not O
a O
simple O
command O
- O
carrying O
medium O
from O
the O
brain O
to O
the O
limbs O
, O
and O
implies O
that O
some O
computational O
activities O
take O
place O
at O
the O
spinal O
cord O
level O
. O

The O
101st O
annual O
meeting O
of O
the O
Japanese O
Ophthalmological O
Society O
. O

The O
results O
corroborate O
the O
proposed O
physiological O
function O
of O
CCoAOMT B
in O
elicited O
plant O
cells O
and O
may O
shed O
new O
light O
on O
the O
sequential O
action O
of O
trans O
- O
active O
factors O
in O
the O
regulation O
of O
phenylpropanoid O
genes O
. O

In O
src B
- O
transformed O
cells O
GAP B
forms O
heteromeric O
complexes O
, O
notably O
with O
a O
highly O
tyrosine O
phosphorylated O
62 O
- O
kDa O
protein O
( O
p62 B
) O
. O

A O
study O
of O
characteristics O
in O
adult O
asthmatics O
who O
are O
sensitized O
by O
cats O
and O
dogs O
allergens O

PrRP B
rapidly O
and O
transiently O
activated O
extracellular B
signal I
- I
regulated I
protein I
kinase I
( O
ERK B
) O
in O
both O
types O
of O
cells O
. O

Cell O
- O
free O
extracts O
of O
E O
. O
coli O
( O
fpg B
mutY B
) O
harboring O
pYSB10 B
possess O
an O
enzymatic O
activity O
that O
cleaves O
a O
34 O
- O
mer O
oligonucleotide O
containing O
a O
single O
8 O
- O
oxoG O
opposite O
a O
cytosine O
( O
8 O
- O
OxoG O
/ O
C O
) O
. O

Estrogen O
pretreated O
castrated O
males O
also O
showed O
no O
change O
in O
myocardial O
blood O
flow O
after O
17 O
beta O
- O
estradiol O
, O
but O
coronary O
flow O
velocity O
decreased O
( O
P O
< O
0 O
. O
05 O
) O
compared O
with O
baseline O
1 O
h O
after O
the O
200 O
ng O
/ O
kg O
dose O
( O
from O
1 O
. O
69 O
+ O
/ O
- O
0 O
. O
61 O
to O
1 O
. O
41 O
+ O
/ O
- O
0 O
. O
42 O
kHz O
) O
and O
diastolic O
coronary O
resistance O
increased O
significantly O
( O
P O
< O
0 O
. O
01 O
) O
compared O
with O
control O
at O
this O
time O
( O
51 O
+ O
/ O
- O
15 O
compared O
with O
39 O
+ O
/ O
- O
14 O
mmHg O
/ O
mkHz O
) O
. O

Acute O
renal O
vein O
thrombosis O
in O
renal O
allografts O
: O
detection O
with O
duplex O
Doppler O
US O
. O

Several O
DNA O
clones O
were O
obtained O
by O
this O
procedure O
, O
one O
of O
which O
, O
clone O
18 O
, O
is O
reported O
and O
characterized O
here O
. O

Long O
- O
range O
mapping O
by O
pulse O
- O
field O
gel O
electrophoresis O
indicated O
that O
the O
three O
mdr B
genes I
are O
closely O
linked O
on O
a O
genomic O
DNA O
segment O
of O
approximately O
625 O
kilobases O
. O

These O
sequences O
were O
further O
tested O
for O
the O
presence O
of O
an O
enhancer O
element O
, O
by O
their O
ability O
to O
activate O
a O
fusion O
reporter O
gene O
consisting O
of O
the O
CAT B
gene I
linked O
to O
the O
gamma B
- I
globin I
gene I
promoter I
, O
in O
erythroid O
( O
K562 O
) O
and O
non O
- O
erythroid O
( O
HeLa O
) O
cells O
. O

Two O
sibs O
with O
identical O
features O
of O
short O
- O
limbed O
dwarfism O
, O
a O
normal O
skull O
and O
face O
and O
normal O
intelligence O
are O
described O
. O

This O
article O
describes O
a O
program O
that O
resulted O
in O
a O
75 O
- O
percent O
reduction O
of O
inventory O
over O
a O
five O
- O
year O
interval O
. O

Further O
comparative O
analysis O
identified O
an O
ADNP B
paralog I
( O
33 O
% O
identity O
and O
46 O
% O
similarity O
) O
, O
indicating O
that O
these O
genes O
belong O
to O
a O
novel O
protein O
family O
with O
a O
nine O
- O
zinc O
finger O
motif O
followed O
by O
a O
homeobox O
domain O
. O

All O
patients O
received O
marrow O
from O
HLA O
- O
identical O
sibling O
donors O
, O
underwent O
similar O
myeloablative O
regimens O
, O
and O
had O
similar O
pretreatment O
characteristics O
. O

In O
female O
toads O
, O
Bufo O
bufo O
, O
feeding O
at O
different O
rates O
, O
the O
uncorrected O
value O
of O
n O
was O
0 O
. O
44 O
, O
when O
metabolism O
and O
growth O
were O
expressed O
as O
kJ O
kJ O
- O
1 O
. O

METHODS O
: O
Two O
patients O
developed O
cervical O
myelopathy O
or O
radiculopathy O
after O
spinal O
manipulation O
therapy O
, O
and O
magnetic O
resonance O
imaging O
showed O
herniated O
cervical O
discs O
at O
C4 O
- O
C5 O
and O
C6 O
- O
C7 O
, O
respectively O
. O

Subclones O
stably O
expressing O
alpha1A B
- I
, O
alpha1B B
- I
, I
and I
alpha1D I
- I
ARs I
under O
control O
of O
an O
inducible O
promoter O
, O
or O
at O
high O
and O
low O
receptor O
density O
, O
were O
isolated O
and O
characterized O
. O

Analysis O
of O
WT1 B
target I
gene I
expression O
in O
stably O
transfected O
cell O
lines O
. O

Soluble O
recombinant B
RI I
and O
soluble O
recombinant B
C I
alpha I
can O
associate O
in O
vitro O
and O
be O
activated O
by O
cyclic O
AMP O
. O

IRIS B
/ O
GAS B
DNA O
also O
formed O
a O
number O
of O
specific O
complexes O
with O
constitutively O
expressed O
factors O
, O
none O
of O
which O
were O
affected O
by O
the O
above O
Abs O
. O

Simulation O
results O
based O
on O
the O
presented O
model O
are O
compared O
with O
the O
experimental O
data O
for O
two O
types O
of O
cMUTs O
reported O
in O
the O
recent O
literature O
. O

Tre O
during O
exercise O
at O
35 O
W O
at O
23 O
degrees O
C O
correlated O
r O
= O
- O
70 O
with O
Vo2max O
and O
r O
= O
0 O
. O
80 O
with O
Tre O
during O
exercise O
in O
heat O
. O

Electrophoretic O
mobility O
- O
shift O
assays O
with O
nuclear O
extracts O
from O
COS O
- O
1 O
cells O
transfected O
with O
expression O
vectors O
for O
SMADs B
1 I
- I
5 I
indicate O
that O
SMAD3 B
forms O
a O
complex O
with O
a O
migration O
similar O
to O
that O
of O
the O
endogenous O
TGF B
- I
beta I
- I
specific I
complex I
observed O
in O
fibroblast O
extracts O
. O

The O
newly O
identified O
NF B
kappa I
B I
enhancer I
( O
TGGAAATTCC O
) O
is O
bound O
by O
a O
TNF B
alpha I
- O
induced O
nuclear O
protein O
and O
appears O
to O
be O
the O
key O
element O
in O
rapid O
transcription O
induction O
by O
TNF B
alpha I
( O
and O
TPA O
) O
, O
while O
transactivation O
of O
this O
element O
is O
repressed O
by O
the O
ligand O
- O
bound O
glucocorticoid B
receptor I
. O

Our O
results O
showed O
that O
the O
5 O
- O
hydroxytryptamine O
( O
5 O
- O
HT O
) O
content O
in O
platelets O
was O
: O
( O
1 O
) O
increased O
in O
the O
subgroup O
of O
anti O
- O
social O
alcoholics O
; O
( O
2 O
) O
transiently O
and O
differently O
altered O
in O
alcoholics O
compared O
to O
opiate O
addicts O
; O
and O
( O
3 O
) O
lowered O
in O
drinking O
alcoholics O
and O
normal O
in O
alcoholics O
who O
were O
drinking O
as O
well O
as O
smoking O
( O
that O
may O
occur O
via O
MAO B
- I
B I
inhibition O
by O
smoke O
) O
. O

In O
the O
second O
group O
, O
which O
consisted O
of O
babies O
of O
33 O
to O
36 O
weeks O
of O
gestational O
age O
, O
haptoglobin B
was O
detected O
in O
6 O
infants O
, O
with O
levels O
less O
than O
25 O
mg O
/ O
dl O
in O
3 O
. O

The O
polypeptide O
encoded O
by O
the O
cDNA O
contained O
450 O
amino O
acids O
with O
a O
calculated O
M O
( O
r O
) O
of O
52 O
, O
057 O
, O
and O
showed O
significant O
homology O
with O
human B
and I
mouse I
CAD I
( O
22 O
% O
identity O
) O
. O

Correlation O
of O
alpha O
activity O
between O
the O
frontal O
and O
occipital O
cortex O
. O

In O
situ O
localization O
of O
PCP B
- I
A1 I
transcripts I
revealed O
that O
they O
accumulate O
specifically O
in O
pollen O
at O
the O
late O
binucleate O
/ O
trinucleate O
stage O
of O
development O
rather O
than O
in O
the O
tapetum O
, O
which O
previously O
was O
taken O
to O
be O
the O
principal O
source O
of O
the O
pollen O
coat O
. O

Successful O
resolution O
of O
progressive O
multifocal O
leukoencephalopathy O
after O
combination O
therapy O
with O
cidofovir O
and O
cytosine O
arabinoside O
. O

The O
species O
composition O
and O
abundance O
upon O
treatment O
of O
manure O
with O
polyethylene O
, O
polystyrene O
and O
glass O
particles O
was O
similar O
to O
that O
of O
the O
treatment O
control O
, O
i O
. O
e O
. O
natural O
and O
inert O
sand O
. O

However O
, O
H19 B
and O
IGF2 B
lie O
within O
a O
larger O
imprinted O
domain O
, O
and O
the O
gene O
specificity O
of O
H19 B
epimutation O
has O
been O
a O
persistent O
question O
. O

Here O
we O
report O
the O
cloning O
, O
by O
either O
peptide O
sequencing O
or O
by O
sequence O
similarity O
to O
the O
human O
cDNAs O
, O
of O
the O
S O
. O
cerevisiae O
genes O
RFC1 B
, O
RFC2 B
, O
RFC3 B
, O
RFC4 B
, O
and O
RFC5 B
. O

It O
is O
of O
interest O
that O
, O
aside O
from O
starvation O
, O
the O
nutrition O
catastrophes O
of O
the O
past O
, O
including O
scurvy O
( O
vitamin O
C O
deficiency O
) O
resulting O
from O
lack O
of O
fresh O
vegetables O
and O
fruit O
and O
beriberi O
( O
vitamin O
B1 O
deficiency O
) O
from O
consumption O
of O
polished O
rice O
, O
are O
forgotten O
and O
only O
of O
interest O
as O
history O
. O

This O
sequence O
can O
interact O
with O
at O
least O
two O
proteins O
in O
a O
fibroblast O
nuclear O
extract O
that O
have O
binding O
characteristics O
of O
an O
Ets B
family I
protein I
and O
a O
serum B
response I
factor I
- I
like I
protein I
. O

As O
much O
thrombin B
was O
formed O
during O
cardiopulmonary O
bypass O
( O
measured O
by O
the O
prothrombin B
activation I
fragment I
F1 I
+ I
2 I
and O
thrombin B
- O
antithrombin B
complexes O
) O
as O
in O
normal O
patients O
, O
showing O
that O
factor B
XII I
was O
not O
necessary O
for O
thrombin B
generation O
. O

The O
molecular O
event O
( O
s O
) O
underlying O
the O
functional O
specificities O
of O
these O
receptors O
( O
in O
regulating O
the O
expression O
of O
their O
native O
target O
genes O
) O
is O
a O
very O
important O
issue O
that O
remains O
poorly O
understood O
. O

MUS81 B
encodes O
a O
novel O
helix B
- I
hairpin I
- I
helix I
protein I
involved O
in O
the O
response O
to O
UV O
- O
and O
methylation O
- O
induced O
DNA O
damage O
in O
Saccharomyces O
cerevisiae O
. O

Treatment O
of O
chronic O
hepatitis O
B O
with O
interferon B
: O
experience O
in O
western O
countries O
. O

Several O
positive O
clones O
were O
isolated O
, O
one O
of O
which O
encoded O
the O
C O
- O
terminal O
253 O
amino O
acids O
of O
a O
putative O
RNA B
helicase I
, O
a O
DEAD B
box I
protein I
designated O
DDX3 B
. O

Kerstin O
takes O
the O
abbreviated O
medical O
education O
. O

In O
vitro O
binding O
studies O
previously O
showed O
that O
NF B
- I
kappa I
B I
p50 B
homodimer I
binds O
the O
switch B
nuclear I
B I
- I
site I
protein I
( O
SNIP B
) O
of O
the O
S B
gamma I
3 I
tandem I
repeat I
. O

In O
this O
case O
myogenic O
factors O
are O
not O
initially O
expressed O
and O
these O
migratory O
cells O
are O
characterized O
by O
the O
expression O
of O
the O
paired B
- I
box I
gene I
Pax3 I
. O

It O
is O
concluded O
that O
the O
atherogenic O
process O
includes O
stimulation O
of O
collagen B
and O
elastin B
synthesis O
. O

These O
results O
indicate O
that O
( O
1 O
) O
RXR B
- O
TR B
heterodimers O
play O
a O
role O
in O
basal O
transactivation O
and O
T3 O
suppression O
of O
negatively O
regulated O
genes O
, O
and O
( O
2 O
) O
RXRs B
increase O
the O
dominant O
negative O
effect O
of O
some O
mutant B
TRs I
on O
specific O
negative O
TREs O
. O

Phenotypic O
responses O
of O
additional O
mutants O
, O
including O
exo1 B
, O
srs2 B
, O
rad5 B
, O
and O
rdh54 B
strains O
, O
suggest O
roles O
in O
recombinational O
repair O
, O
but O
not O
in O
NHEJ O
. O

Furthermore O
, O
the O
induction O
of O
cytochrome B
P I
- I
450 I
1A1 I
observed O
in O
the O
cormorant O
indicates O
that O
the O
Ah B
receptor I
- O
mediated O
process O
, O
by O
which O
TCDD O
and O
related O
chemicals O
exert O
many O
of O
their O
toxicities O
, O
has O
been O
activated O
. O

RESULT O
( O
S O
) O
: O
A O
prospective O
study O
was O
conducted O
with O
44 O
cycles O
for O
34 O
couples O
with O
nafarelin O
acetate O
( O
group O
1 O
) O
and O
47 O
cycles O
for O
40 O
couples O
with O
buserelin O
acetate O
( O
group O
2 O
) O
with O
a O
long O
IVF O
protocol O
; O
68 O
cycles O
for O
46 O
couples O
with O
nafarelin O
acetate O
( O
group O
3 O
) O
and O
56 O
cycles O
for O
39 O
couples O
with O
buserelin O
acetate O
( O
group O
4 O
) O
with O
a O
short O
IVF O
protocol O
; O
39 O
cycles O
for O
32 O
couples O
with O
nafarelin O
acetate O
( O
group O
5 O
) O
and O
50 O
cycles O
for O
30 O
couples O
with O
buserelin O
acetate O
( O
group O
6 O
) O
with O
a O
long O

ICSI O
protocol O
; O
and O
87 O
cycles O
for O
60 O
couples O
with O
nafarelin O
acetate O
( O
group O
7 O
) O
and O
81 O
cycles O
for O
61 O
couples O
with O
buserelin O
acetate O
( O
group O
8 O
) O
with O
a O
short O
ICSI O
protocol O
. O

This O
study O
demonstrates O
the O
feasibility O
and O
potential O
usefulness O
of O
syngeneic O
BM O
Txp O
in O
myeloma O
. O

5 O
. O
3 O
+ O
/ O
- O
6 O
. O
6 O
for O
risperidone O
( O
P O
= O
0 O
. O
09 O
) O
. O

In O
this O
work O
, O
we O
determined O
the O
sequences O
of O
eight O
VirF B
- I
binding I
sites I
from O
four O
different O
genes O
, O
by O
DNase B
I I
or O
hydroxyl O
radical O
footprinting O
. O

Although O
they O
are O
similar O
to O
the O
IL9R B
gene I
( O
approximately O
90 O
% O
identity O
) O
, O
none O
of O
these O
copies O
encodes O
a O
functional O
receptor O
: O
none O
of O
them O
contains O
sequences O
homologous O
to O
the O
5 O
' O
flanking O
region O
or O
exon O
1 O
of O
the O
IL9R B
gene I
, O
and O
the O
remaining O
ORFs O
have O
been O
inactivated O
by O
various O
point O
mutations O
and O
deletions O
. O

Type O
II O
neuroleptanalgesia O
in O
odonto O
- O
stomatology O

273 O
, O
27420 O
- O
27429 O
) O
a O
putative O
peroxisome B
proliferator I
- I
activated I
receptor I
response I
element I
( O
PPRE O
) O
is O
present O
from O
- O
458 O
to O
- O
474 O
. O

While O
reviewing O
gastric O
specimens O
from O
215 O
baboons O
, O
we O
found O
diffuse O
giant O
mucosal O
folds O
in O
2 O
specimens O
and O
multiple O
giant O
mucosal O
nodules O
in O
another O
3 O
. O

To O
investigate O
whether O
the O
two O
RPG B
- I
boxes I
mediate O
transcription O
activation O
of O
both O
the O
L46 B
and O
S24 B
gene I
, O
two O
experimental O
strategies O
were O
followed O
: O
cloning O
of O
the O
respective O
genes O
on O
multicopy O
vectors O
and O
construction O
of O
fusion O
genes O
. O

When O
the O
opioid O
antagonist O
was O
administered O
to O
animals O
24 O
hr O
after O
termination O
of O
single O
or O
multiple O
exposures O
to O
restraint O
, O
NALT O
- O
induced O
increases O
in O
basal O
plasma O
LH I
were O
significantly O
attenuated O
in O
the O
chronically O
stressed O
rats O
compared O
to O
animals O
subjected O
to O
stress O
only O
once O
or O
not O
at O
all O
. O
( O
ABSTRACT O
TRUNCATED O
AT O
250 O
WORDS O
) O

The O
psychosocial O
features O
of O
people O
using O
self O
- O
ignition O
as O
a O
method O
of O
suicide O
are O
consistent O
with O
those O
of O
suicide O
in O
general O
. O

This O
was O
further O
suggested O
by O
transactivation O
assays O
in O
which O
mouse O
fibroblasts O
were O
transiently O
transfected O
with O
a O
human B
beta I
- I
globin I
reporter I
gene I
in O
the O
absence O
and O
presence O
of O
an O
LKLF B
cDNA I
construct I
. O

Plasma O
colony B
- I
stimulating I
factor I
analysis O
and O
the O
clinical O
significance O

Thus O
, O
in O
addition O
to O
the O
previously O
characterized O
FAS B
- I
binding I
factor I
1 I
interacting O
with O
the O
inositol O
/ O
choline O
- O
responsive O
- O
element O
motif O
, O
a O
second O
motif O
common O
to O
the O
promoter O
regions O
of O
both O
FAS B
genes I
could O
be O
identified O
. O

Reconstruction O
time O
for O
a O
64 O
x O
64 O
matrix O
is O
approximately O
45 O
s O
/ O
transaxial O
slice O
. O

Sequence O
analysis O
identifies O
a O
ras B
- I
associating I
( O
RA B
) O
- O
like O
domain O
in O
the O
N O
- O
termini O
of O
band B
4 I
. I
1 I
/ O
JEF B
domains O
and O
in O
the O
Grb7 B
/ I
10 I
/ I
14 I
adapter I
family I
. O

Patients O
received O
BPB O
using O
bupivacaine O
2 O
mg O
kg O
- O
1 O
with O
adrenaline O
1 O
in O
200 O
, O
000 O
, O
either O
with O
or O
without O
hyaluronidase B
3000 O
iu O
, O
in O
a O
volume O
of O
0 O
. O
5 O
ml O
per O
2 O
. O
54 O
cm O
of O
the O
patient O
' O
s O
height O
. O

Rhinovirus B
2A I
protease I
and O
foot B
- I
and I
- I
mouth I
- I
disease I
virus I
L I
protease I
were O
used O
to O
analyze O
the O
association O
of O
eIF4G B
with O
eIF4A B
, O
eIF4E B
, O
and O
eIF3 B
. O

One O
hundred O
cDNA O
clones O
were O
sequenced O
and O
8 O
RTKs B
were O
identified O
, O
as O
well O
as O
12 O
non O
- I
RTKs B
and O
2 O
serine B
/ I
threonine I
kinases I
. O

Roller O
pumps O
, O
coronary O
suction O
and O
an O
open O
cardiotomy O
reservoir O
were O
used O
. O

Furthermore O
, O
it O
was O
found O
that O
the O
Shb B
- O
overexpressing O
cells O
extended O
neurites O
in O
response O
to O
epidermal B
growth I
factor I
. O

Levels O
of O
p53 B
were O
substantially O
increased O
by O
E1A B
expression O
during O
adenovirus O
infection O
. O

In O
cases O
of O
intracapsular O
prostatic O
cancer O
the O
level O
of O
prostatic B
acid I
phosphatase I
( O
PAP B
) O
measured O
by O
radioimmunoassay O
was O
1 O
. O
4 O
+ O
/ O
- O
0 O
. O
8 O
micrograms O
/ O
l O
. O

Healthy O
subjects O
received O
the O
following O
regimens O
, O
dosed O
to O
steady O
state O
: O
trovafloxacin O
300 O
mg O
/ O
24 O
h O
; O
ciprofloxacin O
400 O
mg O
/ O
12 O
h O
; O
trovafloxacin O
300 O
mg O
/ O
24 O
h O
plus O
cefepime O
2 O
g O
/ O
12 O
h O
, O
and O
ciprofloxacin O
400 O
mg O
/ O
12 O
h O
plus O
cefepime O
2 O
g O
/ O
12 O
h O
. O

In O
EGF B
receptor I
- O
mediated O
signaling O
, O
the O
protein O
kinase O
PKB B
/ O
Akt B
and O
the O
mitogen B
- I
activated I
protein I
kinase I
c I
- I
Jun I
N I
- I
terminal I
kinase I
, O
but O
not O
extracellular B
signal I
- I
regulated I
kinase I
2 I
, O
function O
downstream O
of O
phosphatidylinositol B
3 I
- I
kinase I
( O
PI B
3 I
- I
kinase I
) O
. O

Immunoprecipitation O
of O
cell O
lysates O
with O
anti B
- I
phosphotyrosine I
and O
immunoblotting O
showed O
phosphorylated O
forms O
of O
the O
mitogenic O
pathway O
proteins O
Shc B
and O
MAPK B
in O
addition O
to O
p185 B
( O
neu B
) O
, O
suggesting O
that O
the O
Ras B
to O
MAPK B
mitogenic O
pathway O
is O
activated O
. O

Acad O
. O

We O
suggest O
that O
this O
effect O
of O
Myc B
is O
mediated O
by O
its O
action O
upstream O
of O
cyclin B
E I
- O
CDK2 B
, O
and O
occurs O
via O
the O
neutralization O
of O
p27 B
( O
Kip1 B
) O
family O
proteins O
, O
rather O
than O
induction O
of O
Cdc25A B
. O

Bile O
reflux O
demonstrated O
by O
99m O
Tc O
disofenin O
. O

These O
data O
suggest O
that O
certain O
of O
the O
nuclear O
protein O
import O
functions O
of O
NTF2 B
and O
Ran B
/ O
TC4 B
are O
closely O
linked O
and O
that O
NTF2 B
may O
serve O
to O
modulate O
a O
transport O
step O
involving O
Ran B
/ O
TC4 B
. O

Almost O
every O
image O
that O
appears O
during O
a O
therapy O
session O
reveals O
a O
symbolic O
nature O
, O
but O
the O
very O
concept O
of O
symbol O
is O
not O
free O
from O
ambiguity O
some O
positions O
originated O
in O
different O
areas O
of O
investigation O
are O
mentioned O
, O
ending O
with O
a O
description O
of O
the O
traits O
that O
, O
according O
to O
the O
writer O
, O
bestow O
a O
symbolic O
character O
to O
images O
. O

The O
severity O
of O
the O
psychomotor O
retardation O
varied O
from O
mild O
to O
severe O
. O

Both O
oligonucleotide O
cross O
- O
competition O
and O
antibody O
supershift O
experiments O
established O
that O
the O
double O
- O
strand O
binding O
protein O
is O
equivalent O
to O
Sp1 B
. O

The O
wet O
and O
dry O
methods O
of O
the O
HA O
synthesis O
are O
considered O
. O

All O
these O
abnormalities O
returned O
to O
normal O
after O
removal O
of O
the O
tumor O
. O

Although O
FKH2 B
is O
redundant O
with O
FKH1 B
in O
controlling O
pseudohyphal O
growth O
, O
the O
two O
genes O
have O
different O
functions O
in O
silencing O
HMRa B
. O

Molecular O
analysis O
indicated O
that O
inactivation O
of O
H B
- I
2L I
expression O
in O
nearly O
every O
null O
clone O
resulted O
from O
an O
apparent O
deletion O
or O
rearrangement O
of O
5 O
' O
- O
flanking O
and O
5 O
' O
- O
coding O
H B
- I
2L I
sequences I
, O
with O
breakpoints O
consistently O
mapping O
to O
within O
a O
550 O
bp O
GC O
- O
rich O
region O
between O
exon O
1 O
and O
the O
middle O
of O
intron O
2 O
. O

However O
, O
C B
/ I
EBP I
beta I
stimulated O
transcription O
primarily O
through O
the O
cAMP O
- O
responsive O
element O
( O
CRE O
) O
, O
which O
maps O
between O
positions O
- O
77 O
to O
- O
94 O
, O
but O
not O
at O
the O
more O
5 O
' O
- O
binding O
sites O
. O

Heliox O
improves O
pulsus O
paradoxus O
and O
peak O
expiratory O
flow O
in O
nonintubated O
patients O
with O
severe O
asthma O
. O

50 O
- O
fold O
increase O
in O
foreign O
protein O
production O
. O

Ion O
- O
Bernstein O
- O
wave O
heating O
and O
improved O
confinement O
in O
the O
Alcator O
C O
tokamak O
. O

However O
, O
the O
molecular O
mechanisms O
by O
which O
specific O
cis O
- O
and O
trans O
- O
acting O
factors O
control O
activity O
of O
the O
prodynorphin B
promoter I
are O
not O
as O
clearly O
defined O
. O

Finally O
, O
antisense O
mediated O
reduction O
of O
Elk B
- I
1 I
in O
GH4 O
cells O
decreased O
insulin B
- O
increased O
prolactin B
gene I
expression O
and O
confirmed O
the O
requirement O
for O
Elk B
- I
1 I
for O
insulin B
- O
increased O
prolactin B
gene I
expression O
. O

Substitutions O
of O
Asp11 B
led O
to O
dominant O
lethality O
. O

Three O
modern O
hematology O
analyzers O
( O
Abbott O
Cell O
- O
Dyn O
3000 O
, O
Coulter O
STKS O
, O
and O
Sysmex O
NE O
- O
8000 O
) O
with O
high O
throughput O
and O
5 O
- O
part O
differential O
capability O
were O
evaluated O
using O
a O
protocol O
designed O
by O
a O
quality O
team O
. O

Each O
subject O
was O
injected O
in O
the O
antecubital O
vein O
with O
7 O
mg O
/ O
kg O
of O
sodium O
fluorescein O
( O
25 O
% O
solution O
) O
and O
measurements O
were O
taken O
1 O
hr O
postinjection O
at O
4 O
. O
5 O
mm O
and O
7 O
. O
5 O
mm O
from O
the O
retina O
. O

The O
cleft O
walls O
are O
lined O
with O
highly O
conserved O
residues O
and O
NADP O
is O
bound O
along O
one O
wall O
. O

cDNA O
cloning O
, O
sequencing O
and O
chromosome O
mapping O
of O
a O
non B
- I
erythroid I
spectrin I
, O
human B
alpha I
- I
fodrin I
. O

The O
aromatic B
hydrocarbon I
receptor I
( O
AHR B
) O
is O
a O
ligand O
- O
activated O
transcription O
factor O
that O
regulates O
the O
expression O
of O
several O
drug O
- O
metabolizing O
enzymes O
and O
has O
been O
implicated O
in O
immunosuppression O
, O
teratogenesis O
, O
cell O
- O
specific O
hyperplasia O
, O
and O
certain O
types O
of O
malignancies O
and O
toxicities O
. O

( O
1996 O
) O
J O
. O

To O
define O
the O
molecular O
mechanism O
regulating O
FGFR B
- I
1 I
gene I
expression O
in O
proliferating O
myoblasts O
and O
post O
- O
mitotic O
muscle O
fibers O
, O
we O
have O
isolated O
and O
partially O
characterized O
the O
avian B
FGFR I
- I
1 I
gene I
promoter I
. O

Moreover O
, O
FACT B
specifically O
interacts O
with O
nucleosomes O
and O
histone B
H2A B
/ O
H2B B
dimers O
, O
indicating O
that O
it O
may O
work O
by O
promoting O
nucleosome O
disassembly O
upon O
transcription O
. O

Measures O
of O
sedation O
, O
BIS O
, O
deltaBIS O
( O
absolute O
change O
of O
BIS O
after O
a O
painful O
stimulus O
) O
, O
memory O
, O
and O
drug O
concentration O
were O
obtained O
at O
each O
target O
drug O
concentration O
. O

ER B
and O
DR O
were O
significantly O
correlated O
with O
MIB O
- O
1 O
LI O
( O
P O
< O
0 O
. O
01 O
and O
P O
< O
0 O
. O
05 O
, O
respectively O
) O
, O
but O
RI O
and O
Td O
/ O
Te O
were O
not O
. O

Positive O
linear O
correlations O
were O
found O
when O
the O
AUC O
( O
0 O
- O
12 O
) O
( O
r O
= O
0 O
. O
68 O
; O
P O
less O
than O
0 O
. O
01 O
) O
, O
the O
maximum O
plasma O
concentration O
( O
r O
= O
0 O
. O
34 O
; O
P O
less O
than O
0 O
. O
01 O
) O
, O
the O
minimum O
plasma O
concentration O
( O
r O
= O
0 O
. O
55 O
; O
P O
less O
than O
0 O
. O
01 O
) O
, O
and O
the O
elimination O
t1 O
/ O
2 O
( O
r O
= O
0 O
. O
46 O
; O
P O
less O
than O
0 O
. O
01 O
) O
were O
regressed O
with O
age O
. O

Psychophysical O
evidence O
is O
given O
for O
the O
existence O
of O
two O
distinct O
systems O
in O
human O
vision O
: O
a O
fast O
, O
sign O
- O
invariant O
system O
concerned O
with O
extracting O
contours O
and O
a O
slower O
, O
sign O
- O
sensitive O
system O
concerned O
with O
assigning O
surface O
color O
. O

Peripheral O
visual O
acuity O
with O
monovision O
and O
other O
contact O
lens O
corrections O
for O
presbyopia O
. O

Fourteen O
Centers O
participated O
in O
this O
trial O
. O

The O
program O
for O
myogenic O
differentiation O
is O
subject O
to O
negative O
control O
by O
several O
peptide O
growth O
factors O
and O
by O
the O
products O
of O
mutationally O
activated O
ras B
oncogenes I
, O
which O
persistently O
activate O
intracellular O
cascades O
normally O
triggered O
by O
specific O
growth O
factors O
. O

Tetrazepam O
: O
a O
benzodiazepine O
which O
dissociates O
sedation O
from O
other O
benzodiazepine O
activities O
. O

The O
difference O
between O
the O
effects O
of O
the O
two O
dose O
levels O
of O
Z O
. O

Mutation O
of O
the O
distal O
E2F B
binding I
site I
in O
the O
cdc25A B
promoter I
abolished O
E2 B
- O
induced O
repression O
, O
whereas O
mutation O
of O
the O
proximal O
E2F B
site I
or O
the O
E2 B
site I
had O
no O
effect O
. O

Plasmid O
pNK21 O
, O
in O
which O
2 O
. O
05 O
- O
kb O
sequence O
covering O
the O
region O
encoding O
the O
nitrilase B
was O
was O
placed O
under O
the O
control O
of O
the O
lac B
promoter I
, O
directed O
overproduction O
of O
enzymatically O
active O
nitrilase B
in O
response O
to O
addition O
of O
isopropyl O
beta O
- O
D O
- O
thiogalactopyranoside O
in O
Escherichia O
coli O
. O

A O
method O
for O
determining O
the O
toxicity O
of O
neutralizers O
for O
antimicrobial O
agents O
to O
A O
. O
castellanii O
was O
also O
evaluated O
. O

Expression O
in O
all O
lines O
was O
decreased O
by O
the O
inclusion O
of O
regions O
further O
upstream O
, O
and O
extinguished O
by O
the O
inclusion O
of O
the O
first O
intron O
. O

Static O
and O
dynamic O
MRI O
of O
the O
normal O
and O
pathological O
female O
pelvic O
floor O

IENF O
density O
at O
the O
calf O
was O
lower O
than O
that O
obtained O
from O
skin O
at O
more O
proximal O
sites O
, O
indicating O
the O
length O
dependency O
of O
small O
- O
fiber O
loss O
in O
these O
neuropathies O
. O

These O
results O
demonstrate O
a O
specific O
association O
of O
SIV O
and O
HIV B
- I
2 I
nef I
, O
but O
not O
HIV B
- I
1 I
nef I
, O
with O
TCRzeta B
. O

Insertion O
of O
alternatively O
spliced O
exon O
W O
into O
CREB B
mRNA I
during O
spermatogenesis O
results O
in O
a O
polycistronic O
RNA O
that O
encodes O
two O
novel O
internally O
translated O
CREB B
repressor I
isoforms I
called O
I B
- I
CREBs I
, O
consisting O
of O
the O
carboxy O
- O
terminal O
DNA O
- O
binding O
domain O
devoid O
of O
the O
transactivation O
domains O
. O

A O
continuous O
- O
flow O
BIPAP O
system O
consists O
of O
a O
high O
- O
flow O
CPAP O
system O
, O
a O
reservoir O
bag O
, O
and O
a O
pneumatically O
controlled O
membrane O
valve O
in O
the O
expiratory O
limb O
. O

Older O
females O
had O
lumbar O
( O
0 O
. O
6 O
+ O
/ O
- O
1 O
. O
3 O
) O
and O
TB O
( O
1 O
. O
1 O
+ O
/ O
- O
1 O
. O
1 O
) O
BMD O
Z O
scores O
greater O
than O
0 O
( O
both O
, O
p O
< O
0 O
. O
001 O
) O
. O

Smooth O
pursuit O
gain O
and O
the O
percentage O
of O
total O
eye O
movement O
due O
to O
various O
saccadic O
subtypes O
were O
computed O
using O
infrared O
oculography O
and O
computerized O
pattern O
recognition O
software O
. O

Upon O
incubation O
of O
the O
most O
highly O
purified O
fractions O
with O
Mn O
- O
ATP O
or O
Mg O
- O
ATP O
, O
p40 B
was O
the O
only O
protein O
phosphorylated O
on O
tyrosine O
. O

Consistent O
with O
this O
hypothesis O
, O
alpha1 B
- I
PDX I
prevents O
cleavage O
of O
BMP B
- I
4 I
in O
an O
oocyte O
translation O
assay O
. O

A O
p53 B
cDNA I
deletion I
mutant I
( O
delta B
pro I
AE I
) O
, O
which O
lacks O
this O
entire O
proline O
- O
rich O
domain O
( O
deleted O
for O
amino O
acids O
62 O
- O
91 O
) O
, O
was O
created O
and O
characterized O
for O
a O
variety O
of O
p53 B
functions O
. O

Multiple O
aortic O
thrombi O
associated O
with O
protein B
C I
and I
S I
deficiency O
. O

Our O
results O
suggest O
that O
Bry1 B
/ O
Skn7 B
can O
influence O
the O
expression O
of O
MCB B
- O
and O
SCB O
- O
driven O
gene O
expression O
in O
budding O
yeast O
, O
perhaps O
including O
genes O
involved O
in O
cell O
wall O
metabolism O
, O
via O
a O
two O
- O
component O
signal O
transduction O
pathway O
which O
activates O
Bry1 B
/ O
Skn7 B
in O
response O
to O
an O
unidentified O
signal O
. O

( O
1992 O
) O
J O
. O

A O
constructed O
phylogenetic O
tree O
suggests O
that O
the O
UbcP1 B
protein I
may O
represent O
a O
member O
of O
a O
distinct O
subfamily O
of O
E2s B
. O

Platelet B
factor I
4 I
levels O
in O
patients O
with O
coronary O
artery O
disease O
. O

We O
functionally O
characterised O
AtXPO1 B
by O
its O
interaction O
with O
NESs B
of O
animal O
and O
plant O
proteins O
, O
which O
is O
inhibited O
by O
the O
cytotoxin O
leptomycin O
B O
( O
LMB O
) O
, O
and O
also O
by O
its O
interaction O
with O
the O
small B
GTPase I
Ran1 I
in O
the O
yeast O
two O
- O
hybrid O
system O
. O

A O
backward O
look O
at O
urinary O
tract O
infections O
. O

The O
expression O
of O
cor14b B
was O
strongly O
impaired O
in O
the O
barley B
albino I
mutant I
an I
, O
suggesting O
the O
involvement O
of O
a O
plastidial O
factor O
in O
the O
control O
of O
gene O
expression O
. O

MRS O
allowed O
the O
differentiation O
of O
the O
following O
metabolites O
in O
5 O
patients O
: O
N O
- O
acetyl O
- O
aspartate O
( O
NAA O
) O
, O
creatine O
and O
phosphocreatine O
, O
phosphorylcholine O
and O
glycerophosphorylcholine O
, O
beta O
- O
and O
gamma O
- O
glutamate O
( O
GLU O
) O
. O

Overall O
, O
free O
- O
chlorine O
treatments O
( O
0 O
. O
3 O
or O
1 O
. O
0 O
mg O
/ O
L O
) O
showed O
significantly O
lower O
heterotrophic O
plate O
numbers O
than O
those O
without O
free O
chlorine O
. O

S1 B
nuclease I
protection O
mapping O
and O
primer O
extension O
analysis O
allowed O
us O
to O
propose O
that O
the O
A O
residue O
located O
19 O
bp O
upstream O
from O
the O
translation O
initiation O
codon O
is O
the O
site O
of O
transcription O
initiation O
. O

Various O
stimuli O
inactivate O
IkappaB B
alpha I
by O
triggering O
phosphorylation O
of O
the O
N O
- O
terminal O
residues O
Ser32 O
and O
Ser36 O
. O

There O
was O
no O
difference O
in O
the O
day O
- O
42 O
cure O
rates O
between O
the O
QC7 O
( O
n O
= O
65 O
) O
and O
A7 O
( O
n O
= O
64 O
) O
regimens O
with O
an O
efficacy O
of O
100 O
% O
in O
both O
, O
confirmed O
by O
parasite O
genotyping O
. O

In O
addition O
, O
the O
results O
suggest O
that O
N O
region O
diversity O
at O
V O
( O
D O
) O
J O
junctions O
within O
rearranged O
immunoglobulin B
and O
T B
cell I
receptor I
gene I
loci I
can O
only O
be O
introduced O
after O
the O
generation O
of O
RAG B
- O
catalyzed O
DNA O
double O
- O
strand O
breaks O
, O
i O
. O
e O
. O
during O
the O
DNA O
end O
joining O
phase O
of O
the O
V O
( O
D O
) O
J O
recombination O
reaction O
. O

We O
conclude O
that O
99mTc O
- O
HL91 O
is O
a O
potent O
marker O
of O
myocardial O
viability O
when O
used O
during O
the O
early O
acute O
phase O
after O
reperfusion O
. O

Three O
patients O
treated O
with O
amphotericin O
B O
, O
single O
course O
as O
well O
as O
multiple O
courses O
, O
and O
other O
antifungal O
agents O
( O
hydroxystilbamidine O
and O
miconazole O
) O
have O
all O
relapsed O
. O

An O
infant O
of O
8 O
months O
with O
congenital O
glaucoma O
and O
hemophilia O
A O
lost O
one O
eye O
due O
to O
haemorrhages O
after O
trabeculotomy O
in O
an O
eye O
hospital O
. O

Anderson O
Hospital O
and O
Tumor O
Institute O
at O
Houston O
during O
a O
30 O
- O
year O
interval O
( O
1944 O
to O
1974 O
) O
, O
with O
a O
minimum O
of O
ten O
years O
of O
follow O
- O
up O
, O
we O
determined O
that O
the O
major O
prognostic O
factor O
of O
survival O
was O
the O
number O
of O
positive O
nodes O
. O

We O
have O
now O
isolated O
cDNA O
for O
an O
invertebrate B
Pax I
- I
6 I
protein I
from I
sea I
urchin I
embryos I
. O

To O
study O
structural O
diversity O
of O
the O
three O
homologous O
loci O
encoding O
a O
KN1 B
- I
like I
homeobox I
protein I
in O
the O
hexaploid O
wheat O
genome O
, O
we O
isolated O
clones O
from O
a O
cDNA O
library O
of O
young O
spikes O
of O
Japanese O
common O
wheat O
cultivar O
' O
Norin O
26 O
' O
. O

Whether O
or O
not O
these O
novel O
repeated O
sequences O
throughout O
the O
SMA O
region O
are O
involved O
in O
the O
disease O
remains O
to O
be O
determined O
. O

Thus O
, O
Shp2 B
regulates O
phosphotyrosine O
- O
signalling O
events O
during O
the O
complex O
ectodermal O
- O
mesenchymal O
interactions O
that O
regulate O
mammalian O
budding O
morphogenesis O
. O

Moyamoya O
is O
an O
intriguing O
and O
controversial O
syndrome O
. O

However O
, O
elimination O
of O
the O
kinase O
activity O
of O
DCAMKL1 B
has O
no O
detectable O
effect O
on O
its O
microtubule O
polymerization O
activity O
. O

We O
have O
thus O
cloned O
two O
yeast O
homologs I
of I
mammalian I
p220 I
. O

The O
glucose O
effect O
on O
the O
pyruvate B
kinase I
gene I
is O
reversibly O
antagonized O
by O
agents O
increasing O
intracellular O
cAMP O
. O

An O
identical O
polypeptide O
was O
detected O
by O
Western O
blot O
analysis O
of O
K1F O
virions O
. O

Patients O
with O
PLM O
show O
excessive O
daytime O
sleepiness O
or O
insomnia O
. O

Coexpression O
of O
CREB B
- I
binding I
protein I
( O
CBP B
) O
/ O
p300 B
with O
TReP B
- I
132 I
has O
an O
additive O
effect O
on O
promoter O
activity O
, O
and O
the O
proteins O
were O
demonstrated O
to O
interact O
physically O
. O

It O
is O
believed O
that O
the O
operation O
is O
effective O
because O
it O
cuts O
the O
spinothalamic O
tract O
( O
STT O
) O
, O
a O
primary O
pathway O
carrying O
nociceptive O
information O
from O
the O
spinal O
cord O
to O
the O
brain O
in O
humans O
. O

Catalase B
plays O
a O
key O
role O
as O
an O
antioxidant O
, O
protecting O
aerobic O
organisms O
from O
the O
toxic O
effects O
of O
hydrogen O
peroxide O
, O
and O
in O
some O
cases O
has O
been O
postulated O
to O
be O
a O
virulence O
factor O
. O

Recovery O
rates O
of O
energy O
level O
and O
intracellular O
pH O
during O
stimulation O
at O
100Hz O
were O
greater O
than O
those O
observed O
during O
stimulation O
at O
30Hz O
. O

Exhaled O
NO O
was O
assessed O
by O
controlled O
- O
flow O
chemoluminescence O
after O
adjusting O
for O
trapped O
air O
and O
after O
generating O
pressure O
in O
the O
oral O
cavity O
that O
was O
sufficient O
to O
close O
the O
soft O
palate O
( O
Eco O
Physics O
CLD O
77 O
AM O
analyzer O
) O
. O

Recent O
developments O
in O
the O
study O
of O
growth O
factors O
: O
GRF B
and O
somatomedins B
. O

New O
concepts O
of O
condyloma O
acuminata O
in O
children O
. O

This O
is O
the O
first O
clinical O
case O
report O
of O
NIHF O
due O
to O
fetal O
Kasabach O
- O
Merritt O
syndrome O
that O
was O
prenatally O
diagnosed O
by O
sonography O
, O
computerized O
tomography O
, O
and O
percutaneous O
umbilical O
blood O
sampling O
. O

Acetoacetate B
decarboxylase I
( O
ADC B
) O
( O
EC4 B
. I
1 I
. I
1 I
. I
4 I
) O
of O
Clostridium O
acetobutylicum O
DSM O
792 O
was O
purified O
to O
homogeneity O
, O
and O
its O
first O
25 O
N O
- O
terminal O
amino O
acids O
were O
determined O
. O

Transfer O
of O
health O
care O
to O
natives O
holds O
much O
promise O
, O
lecturers O
say O
. O

The O
sequence O
of O
the O
127 B
residue I
NrfF I
polypeptide I
, O
M O
( O
r O
) O
14 O
, O
522 O
, O
is O
strikingly O
similar O
to O
the O
CcI2 B
protein I
of O
R O
. O
capsulatus O
, O
especially O
in O
the O
putative O
haem O
- O
binding O
motif O
, O
RCPQCQNQN O
. O
( O
ABSTRACT O
TRUNCATED O
AT O
400 O
WORDS O
) O

CAT B
assays O
demonstrated O
that O
overexpression O
of O
RXRalpha B
conferred O
the O
best O
RA O
response O
, O
consistent O
with O
our O
previous O
observation O
that O
9 O
- O
cis O
- O
RA O
is O
more O
potent O
than O
all O
- O
trans O
- O
RA O
for O
inducing O
the O
expression O
of O
the O
AFP B
gene I
. O

For O
rotating O
stimuli O
presented O
to O
the O
dominant O
eye O
, O
this O
class O
of O
neurons O
responded O
best O
to O
rotation O
of O
the O
visual O
world O
about O
an O
axis O
oriented O
near O
the O
horizontal O
plane O
and O
approximately O
45 O
degrees O
azimuth O
. O

None O
of O
these O
patients O
developed O
clinical O
events O
before O
disappearance O
of O
the O
phospholipid O
- O
dependent O
inhibitors O
of O
coagulation O
. O

In O
spite O
of O
the O
variability O
of O
DD95 O
with O
regard O
to O
body O
weight O
, O
the O
recovery O
of O
neuromuscular O
transmission O
in O
the O
patients O
of O
the O
three O
groups O
is O
comparable O
. O

We O
have O
studied O
some O
of O
the O
parameters O
governing O
the O
expression O
of O
a O
foreign O
promoter O
- O
reporter O
gene O
construct O
incorporated O
into O
herpes O
simplex O
virus O
( O
HSV O
) O
type O
1 O
. O

Relation O
between O
clinical O
and O
roentgenological O
scores O
and O
measures O
of O
lung O
function O
in O
cystic O
fibrosis O
, O
with O
special O
reference O
to O
pulmonary O
Xenon133 O
elimination O
. O

Each O
fusion O
leads O
, O
in O
principle O
, O
to O
the O
same O
effect O
: O
The O
ret B
tyrosine I
kinase I
is O
uncoupled O
from O
its O
stringent O
physiological O
regulation O
by O
replacement O
of O
its O
5 O
' O
end O
and O
is O
aberrantly O
activated O
by O
the O
5 O
' O
parts O
of O
fused O
genes O
in O
thyrocytes O
that O
do O
not O
normally O
express O
ret B
tyrosine I
kinase I
. O

He O
had O
complained O
of O
fever O
and O
right O
hypochondralgia O
2 O
months O
after O
being O
operated O
for O
appendicitis O
. O

In O
the O
predicted O
amino O
acid O
sequence O
of O
NPS1 B
, O
sequences O
homologous O
to O
the O
catalytic O
domain O
of O
protein O
kinases O
were O
found O
. O

Carcinoma O
- O
related O
protein O
production O
may O
have O
played O
a O
role O
in O
the O
development O
of O
the O
observed O
renal O
lesions O
. O

These O
results O
provide O
strong O
evidence O
that O
conserved O
interhelical O
packing O
interactions O
in O
the O
gp41 B
core I
are O
important O
determinants O
of O
HIV O
- O
1 O
entry O
and O
its O
inhibition O
. O

Lag O
phases O
were O
14 O
. O
89 O
hours O
+ O
/ O
- O
0 O
. O
77 O
, O
13 O
. O
33 O
hours O
+ O
/ O
- O
0 O
. O
50 O
, O
20 O
. O
22 O
hours O
+ O
/ O
- O
0 O
. O
76 O
, O
and O
20 O
. O
00 O
hours O
+ O
/ O
- O
0 O
. O
79 O
, O
respectively O
, O
for O
endothelial O
cell O
- O
induced O
LDL B
oxidation O
. O

Forty O
- O
eight O
pregnant O
adult O
and O
122 O
fetal O
guinea O
pigs O
were O
sacrificed O
at O
intervals O
throughout O
gestation O
and O
the O
carcasses O
analyzed O
for O
a O
variety O
of O
growth O
parameters O
. O

Here O
we O
demonstrate O
the O
ability O
of O
oligonucleotides O
containing O
the O
C4T O
sequence O
to O
confer O
heat O
shock O
inducibility O
on O
the O
reporter O
gene O
and O
show O
that O
the O
presence O
of O
two O
such O
elements O
produces O
more O
than O
additive O
effects O
on O
induction O
. O

The O
HPV B
E1 I
and O
E2 B
proteins I
along O
with O
cellular O
factors O
, O
are O
required O
for O
replication O
of O
the O
viral O
genome O
. O

The O
observed O
alterations O
in O
lung O
functions O
in O
these O
subjects O
indicate O
that O
individuals O
performing O
heavy O
continuous O
exercise O
are O
more O
likely O
to O
be O
affected O
by O
lower O
O3 O
levels O
. O

In O
ten O
other O
endotoxin O
- O
albumin B
- O
treated O
pigs O
PGE1 O
infusion O
( O
0 O
. O
25 O
micrograms O
X O
kg O
- O
1 O
X O
min O
- O
1 O
) O
was O
begun O
after O
established O
pulmonary O
and O
cardiovascular O
dysfunction O
, O
for O
closer O
mimicking O
of O
clinical O
use O
. O

The O
main O
new O
features O
of O
this O
version O
are O
that O
end O
- O
tidal O
PO2 O
instead O
of O
inspiratory O
PO2 O
can O
be O
kept O
constant O
, O
and O
that O
the O
correcting O
activity O
of O
both O
controllers O
( O
capnostat O
and O
oxystat O
) O
is O
proportional O
to O
the O
magnitude O
of O
the O
difference O
between O
the O
actual O
and O
the O
adjusted O
end O
- O
tidal O
PCO2 O
or O
PO2 O
. O

Like O
the O
human B
erythrocyte I
P4 I
. I
2 I
, O
mouse B
erythrocyte I
P4 I
. I
2 I
contains O
regions O
strikingly O
homologous O
with O
the O
transglutaminase B
( O
TGase B
) O
proteins O
although O
it O
too O
most O
likely O
lacks O
TGase B
crosslinking O
activity O
. O

It O
spans O
20 O
kilobases O
, O
consists O
of O
7 O
exons O
and O
6 O
introns O
, O
and O
contains O
a O
TATA O
motif O
24 O
nucleotides O
upstream O
of O
the O
transcriptional O
start O
site O
. O

Nine O
months O
after O
the O
end O
of O
the O
vaccination O
anti B
- I
HBsAg I
levels O
had O
dropped O
to O
9 O
+ O
/ O
- O
4 O
IU O
/ O
1 O
( O
M O
+ O
/ O
- O
SE O
) O
, O
with O
a O
geometric O
mean O
of O
5 O
IU O
/ O
1 O
, O
in O
the O
nine O
remaining O
evaluable O
patients O
. O

CONCLUSION O
: O
Ultrasound O
biomicroscopy O
is O
a O
useful O
tool O
for O
the O
evaluation O
of O
APVR O
, O
which O
is O
difficult O
by O
ordinary O
methods O
. O

It O
is O
postulated O
that O
the O
synthesis O
of O
the O
peptidoglycan O
layer O
was O
affected O
by O
the O
antimetabolites O
since O
the O
morphological O
effects O
were O
strikingly O
similar O
to O
those O
caused O
by O
treatment O
of O
E O
. O
cloacae O
with O
disodium O
edetate O
plus O
lysozyme B
. O

DRAP27 B
is O
recognized O
by O
CD9 B
antibodies I
. O

Furthermore O
we O
elucidate O
the O
subtle O
highly O
anisotropic O
interchain O
correlations O
and O
reveal O
the O
detailed O
atomic O
structure O
of O
the O
low O
- O
temperature O
( O
8x2 O
) O
phase O
. O

Control O
oligomers O
of O
scrambled O
sequence O
but O
identical O
base O
composition O
were O
ineffective O
, O
and O
no O
TFO O
- O
induced O
recombination O
was O
seen O
in O
a O
control O
LTK O
( O
- O
) O
cell O
line O
carrying O
an O
otherwise O
identical O
dual O
TK B
gene I
construct I
lacking O
the O
30 O
- O
bp O
polypurine O
target O
site O
. O

To O
quantify O
the O
normal O
coupling O
patterns O
, O
fresh O
cadaveric O
human O
lumbar O
spine O
specimens O
( O
L1 O
- O
S1 O
) O
were O
used O
. O

One O
cytoplasmic O
target O
which O
reflects O
the O
functional O
state O
of O
the O
plastids O
is O
protein B
kinase I
C I
. O

These O
three O
chromosomes O
share O
the O
transferrin B
gene I
( O
TF B
) O
, O
the O
myosin B
light I
polypeptide I
3 I
gene I
( O
MYL3 B
) O
, O
and O
the O
acylpeptide B
hydrolase I
gene I
( O
APEH B
) O
. O

( O
i O
) O
Several O
ribosomal O
proteins O
were O
synthesized O
in O
substantial O
excess O
. O

This O
experience O
would O
recommend O
consideration O
of O
home O
nutritional O
support O
in O
patients O
with O
systemic O
scleroderma O
when O
the O
disease O
is O
relatively O
stable O
and O
no O
major O
organ O
failure O
is O
present O
. O

Based O
on O
our O
sequence O
data O
, O
ORF2 O
from O
most O
isolates O
excluding O
G1 O
encode O
truncated O
49 O
aa O
( O
pORF2a B
) O
because O
of O
an O
in O
- O
frame O
stop O
codon O
, O
although O
ORF2s O
from O
most O
G1 O
isolates O
encode O
202 O
aa O
( O
pORF2ab B
) O
. O

In O
the O
present O
study O
we O
prospectively O
compared O
side O
effects O
occurring O
in O
12 O
patients O
after O
the O
first O
administration O
of O
low O
- O
dose O
OKT3 O
( O
0 O
. O
5 O
mg O
twice O
daily O
) O
induction O
therapy O
with O
those O
in O
10 O
patients O
who O
were O
treated O
with O
a O
conventional O
dose O
of O
OKT3 O
( O
5 O
mg O
daily O
) O
for O
acute O
rejection O
. O

These O
data O
indicate O
that O
SB O
203580 O
sensitive O
p38 B
MAP I
kinases I
are O
not O
involved O
in O
okadaic O
acid O
mediated O
increases O
in O
TRE B
DNA O
binding O
and O
transactivation O
. O

Together O
, O
these O
results O
indicate O
that O
YTS1 B
is O
a O
bifunctional O
protein O
active O
in O
both O
splicing O
and O
protein O
synthesis O
. O

Oscillatory O
flow O
is O
reestablished O
in O
the O
model O
at O
the O
capillary O
- O
cerebrospinal O
fluid O
and O
capillary O
- O
venous O
interfaces O
. O

Sgs B
- I
4 I
is O
one O
of O
the O
eight O
known O
genes O
coding O
for O
larval O
secretion I
proteins I
in O
Drosophila O
melanogaster O
. O

We O
conclude O
that O
although O
the O
G3 O
sequence O
contains O
two O
protein O
- O
binding O
motifs O
, O
the O
organization O
of O
the O
G3 B
enhancer I
- I
like I
element I
is O
not O
bipartite O
. O

Human B
myotonic I
dystrophy I
protein I
kinase I
( O
DMPK B
) O
is O
a O
member O
of O
a O
novel O
class O
of O
multidomain O
protein O
kinases O
that O
regulate O
cell O
size O
and O
shape O
in O
a O
variety O
of O
organisms O
. O

The O
human B
BRCA1 I
promoter I
also O
contains O
a O
conserved O
E2F B
site I
and O
is O
similarly O
regulated O
by O
E2F1 B
and O
Rb B
. O

The O
translated O
sequence O
of O
the O
FLI B
LRR I
associated I
protein I
( O
FLAP B
) O
encodes O
a O
novel O
protein O
not O
represented O
in O
the O
data O
base O
. O

Corticosteroid O
treatment O
before O
, O
during O
, O
or O
after O
fludarabine O
treatment O
in O
patients O
with O
alkylator O
- O
resistant O
, O
low O
- O
grade O
lymphoid O
malignancies O
who O
have O
not O
received O
PCP O
prophylaxis O
is O
associated O
with O
an O
increased O
risk O
of O
opportunistic O
pulmonary O
infections O
. O

In O
this O
respect O
, O
the O
promoter O
structure O
of O
COX B
genes I
resemble O
those O
of O
many O
house O
- O
keeping O
genes O
. O

In O
vitro O
, O
cytokine O
- O
or O
hypoxia O
- O
induced O
up O
- O
regulation O
of O
Fas B
expression O
is O
associated O
with O
RTC B
apoptosis O
. O

The O
site O
of O
acetylation O
of O
Tat B
was O
mapped O
to O
the O
double O
- O
lysine O
motif O
in O
a O
highly O
conserved O
region O
, O
( O
49 O
) O
RKKRRQ O
( O
54 O
) O
, O
of O
the O
basic O
RNA O
- O
binding O
motif O
of O
Tat B
. O

These O
results O
suggest O
that O
the O
PVB O
therapy O
is O
not O
sufficient O
to O
cure O
the O
cases O
with O
choriocarcinoma O
element O
or O
bulky O
metastasis O
. O

The O
KEMAR O
measurements O
show O
that O
the O
two O
models O
give O
an O
improvement O
of O
the O
signal O
- O
to O
- O
noise O
ratio O
of O
approximately O
7 O
. O
5 O
dB O
in O
a O
diffuse O
sound O
field O
. O

Indirect O
determination O
of O
maximal O
O2 O
consumption O
in O
man O
. O

Using O
unidirectional O
PCR O
we O
isolated O
a O
361 O
- O
bp O
5 O
' O
promoter O
region O
and O
delineated O
the O
intronic O
/ O
exonic O
boundaries O
which O
include O
a O
non O
- O
coding O
exon O
1 O
, O
a O
single O
intron O
, O
and O
a O
coding O
exon O
2 O
, O
a O
structure O
that O
is O
typical O
of O
genes O
of O
the O
RNase B
A I
superfamily I
. O

When O
severe O
hypoxia O
was O
acutely O
produced O
by O
ventilation O
with O
low O
- O
oxygen O
mixtures O
in O
experimental O
( O
PaO2 O
, O
23 O
. O
7 O
+ O
/ O
- O
1 O
. O
7 O
torr O
) O
and O
control O
animals O
( O
PaO2 O
, O
26 O
. O
3 O
+ O
/ O
- O
1 O
. O
0 O
torr O
) O
, O
plasma B
insulin I
responses O
were O
markedly O
inhibited O
in O
both O
. O

Uptake O
of O
colicins O
required O
different O
domains O
in O
TonB B
, O
for O
colicin B
B I
and I
M I
around O
residue O
160 O
and O
for O
colicin B
Ia I
, O
a O
domain O
closer O
to O
the O
C O
- O
terminal O
end O
. O

D O
. O

When O
lipid O
X O
was O
combined O
with O
ticarcillin O
, O
survival O
differences O
were O
both O
significant O
and O
prolonged O
. O

Central O
treatment O
unit O
saves O
nurses O
, O
adds O
income O
, O
improves O
care O
. O

Gold O
flaxseed O
( O
whole O
or O
ground O
) O
fed O
at O
levels O
of O
5 O
or O
15 O
% O
were O
compared O
to O
a O
1 O
. O
5 O
% O
menhaden O
oil O
or O
a O
typical O
control O
layer O
ration O
. O

It O
is O
dangerous O
to O
label O
such O
conditions O
as O
' O
inappropriate O
' O
secretion O
of O
ADH B
since O
the O
maintenance O
of O
circulating O
volume O
is O
at O
least O
as O
important O
a O
physiological O
requirement O
as O
the O
defence O
of O
tonicity O
. O

The O
complete O
nucleotide O
sequence O
of O
odontoglossum O
ringspot O
virus O
( O
Cy O
- O
1 O
strain O
) O
genomic O
RNA O
. O

An O
enhancement O
of O
benign O
liver O
tumors O
( O
tumorigenic O
effect O
) O
was O
observed O
only O
in O
CF1 O
male O
mice O
( O
17 O
/ O
56 O
at O
the O
only O
tested O
dose O
: O
400 O
ppm O
vs O
. O

Cells O
were O
cotransfected O
with O
this O
plasmid O
, O
and O
the O
appropriate O
responder O
plasmids O
and O
clonies O
were O
selected O
on O
the O
basis O
of O
their O
resistance O
to O
Geneticin B
( O
via O
the O
neomycin B
aminoglycoside I
phosphotransferase I
gene I
) O
. O

This O
C O
- O
terminal O
domain O
contains O
several O
YxxI O
/ O
L O
motifs O
reminiscent O
of O
LMP2A B
and O
a O
putative O
TRAF B
binding I
site I
as O
in O
LMP1 B
. O

Omitting O
part O
of O
the O
experimental O
NOE O
- O
derived O
distances O
results O
in O
reduced O
restraint O
violations O
and O
lower O
R O
factors O
but O
impairs O
structural O
convergence O
in O
the O
rMD O
refinement O
. O

Cardiac O
lesion O
in O
exotoxic O
shock O

Risk O
factors O
for O
atherosclerosis O
related O
to O
nutrition O
are O
hypercholesterolemia O
, O
hyperglycemia O
- O
diabetes O
, O
and O
for O
hypertension O
, O
obesity O
, O
high O
salt O
intake O
, O
and O
excessive O
use O
of O
alcohol O
. O

This O
mutation O
was O
associated O
with O
reduced O
or O
absent O
expression O
of O
TGF B
- I
beta I
RI I
protein I
and O
p53 B
protein I
in O
tumor O
tissues O
. O

SCAN O
boxes O
are O
found O
in O
eight O
other O
genes O
in O
the O
GenBank O
database O
, O
five O
of O
which O
are O
also O
in O
the O
Kruppel B
family I
of O
zinc B
finger I
proteins I
lacking O
KRAB B
A I
and I
B I
domains I
and O
thereby O
define O
a O
new O
subclass O
of O
zinc B
finger I
proteins I
. O

However O
, O
they O
had O
no O
cross O
- O
resistance O
to O
quinolone O
, O
josamycin O
and O
tylosin O
. O

Seventy O
- O
nine O
percent O
of O
the O
children O
screened O
and O
91 O
. O
0 O
% O
of O
the O
children O
with O
PbB O
at O
least O
10 O
micrograms O
/ O
dL O
were O
Hispanic O
. O

Normal O
dogs O
were O
exposed O
to O
either O
10 O
, O
15 O
, O
or O
30 O
Gy O
of O
X O
rays O
to O
a O
single O
hemisphere O
and O
the O
gross O
and O
histopathologic O
changes O
were O
evaluated O
qualitatively O
. O

YACs O
selected O
from O
our O
contig O
will O
be O
the O
starting O
point O
for O
the O
cloning O
of O
the O
LGMD2B B
gene I
and O
thereby O
establish O
the O
biological O
basis O
for O
this O
form O
of O
muscular O
dystrophy O
and O
its O
relationship O
with O
the O
other O
limb O
- O
girdle O
muscular O
dystrophies O
. O

The O
amount O
of O
ERCC1 B
detectable O
by O
immunoblotting O
is O
reduced O
in O
group O
1 O
, O
group O
4 O
and O
XP B
- I
F I
extracts O
. O

In O
the O
plant O
malate B
synthases I
, O
the O
extension O
is O
probably O
involved O
in O
routing O
to O
the O
microbodies O
, O
since O
it O
contains O
the O
potential O
peroxisomal O
targeting O
signal O
, O
Ser O
- O
Arg O
/ O
Lys O
- O
Leu O
, O
at O
the O
carboxy O
terminus O
. O

Optimized O
USF B
and O
MYC B
DNA I
- I
binding I
sites I
, O
which O
differ O
in O
the O
nucleotides O
bordering O
the O
hexanucleotide O
box O
displace O
the O
E B
- I
C4 I
factor I
in O
competition O
assays O
but O
with O
lesser O
efficiency O
than O
the O
E B
- I
C4 I
site I
itself O
. O

Screening O
of O
human O
cDNA O
libraries O
has O
identified O
two O
different O
5 O
' O
- O
termini O
and O
alternatively O
spliced O
forms O
of O
the O
human B
Fli I
- I
1 I
gene I
( O
Fli B
- I
1b I
) O
, O
suggesting O
the O
possible O
existence O
of O
two O
independent O
promoters O
. O

A O
surge O
in O
the O
rate O
of O
ascorbyl O
radical O
production O
, O
directly O
correlated O
with O
oxyradical O
stress O
and O
an O
abrupt O
fall O
in O
superoxide B
dismutase I
activity O
in O
the O
granuloma O
, O
indicates O
' O
switching O
on O
' O
of O
a O
free O
radical O
- O
dependent O
machinery O
for O
the O
formation O
of O
granuloma O
after O
vasectomy O
. O

After O
5 O
- O
h O
thermal O
induction O
of O
cells O
carrying O
the O
runaway O
recombinant B
pBS1 I
, O
protein B
B2 I
constituted O
40 O
% O
of O
the O
soluble O
protein O
fraction O
of O
the O
cells O
. O

A O
regulatory O
element O
of O
the O
empty B
spiracles I
homeobox I
gene I
is O
composed O
of O
three O
distinct O
conserved O
regions O
that O
bind O
regulatory O
proteins O
. O

Peak O
reactive O
hyperemia O
( O
mL O
. O
min O
- O
1 O
. O
100 O
mL O
- O
1 O
) O
was O
determined O
in O
the O
calf O
and O
forearm O
immediately O
before O
and O
after O
12 O
weeks O
of O
training O
. O

These O
characteristics O
add O
to O
the O
suitability O
of O
NM441 O
as O
an O
effective O
prodrug O
of O
NM394 O
. O

Vorozole O
( O
Rivizor O
) O
is O
a O
potent O
and O
stereospecific O
inhibitor O
of O
aromatase B
having O
shown O
promising O
endocrine O
effects O
in O
phase O
I O
studies O
. O

In O
5 O
cases O
, O
combined O
sensitization O
to O
mercury O
and O
other O
metal O
salts O
, O
particularly O
gold O
sodium O
thiosulfate O
( O
GST O
) O
and O
palladium O
chloride O
( O
PDC O
) O
, O
was O
observed O
. O

Gene O
structure O
and O
precursor O
processing O
. O

The O
content O
of O
monoamine O
metabolites O
, O
5 O
- O
hydroxyindoleacetic O
acid O
, O
5 O
- O
HIAA O
, O
and O
homovanillic O
acid O
( O
HVA O
) O
, O
and O
- O
- O
for O
17 O
patients O
- O
- O
tryptophan O
, O
in O
the O
cerebrospinal O
fluid O
was O
determined O
. O

One O
month O
after O
administration O
of O
131I O
, O
the O
29 O
cats O
evaluated O
were O
clinically O
improved O
, O
and O
24 O
( O
83 O
% O
) O
of O
the O
29 O
cats O
evaluated O
had O
normal O
serum O
T4 O
concentrations O
, O
3 O
cats O
( O
10 O
% O
) O
remained O
hyperthyroxinemic O
, O
and O
2 O
cats O
( O
7 O
% O
) O
were O
hypothyroxinemic O
. O

These O
findings O
suggest O
that O
T3R B
can O
repress O
or O
activate O
transcription O
while O
tethered O
to O
the O
LBD O
of O
GAL4 B
- O
RXR B
and O
that O
heterodimerization O
can O
occur O
in O
vivo O
without O
stabilization O
by O
hormone O
response O
elements O
. O

Most O
interestingly O
, O
although O
the O
effector O
plasmid O
containing O
the O
ICP27 B
gene I
had O
little O
effect O
on O
its O
own O
, O
two O
different O
and O
marked O
effects O
depending O
on O
the O
target O
were O
observed O
when O
ICP27 B
was O
combined O
with O
ICP4 B
or O
ICP0 B
or O
both O
. O

In O
renal O
vein O
thrombosis O
, O
similar O
pattern O
to O
acute O
tubular O
necrosis O
was O
found O
but O
RI O
venography O
was O
helpful O
. O

TOM1 B
encodes O
a O
member O
of O
the O
hect B
- I
domain I
- I
containing I
E3 I
ubiquitin I
- I
protein I
ligase I
family O
that O
is O
required O
for O
growth O
at O
elevated O
temperatures O
. O

Genomic O
clones O
containing O
the O
C B
. I
briggsae I
gene I
are O
able O
to O
completely O
rescue O
the O
unc B
- I
119 I
phenotype O
in O
transgenic O
C O
. O
elegans O
mutants O
. O

The O
rapid O
decrease O
of O
plasma O
potassium O
during O
the O
first O
two O
hours O
of O
standard O
HD O
causes O
a O
membrane O
hyperpolarization O
. O

Region O
- O
specific O
enhancers O
near O
two O
mammalian B
homeo I
box I
genes I
define O
adjacent O
rostrocaudal O
domains O
in O
the O
central O
nervous O
system O
. O

In O
this O
study O
, O
we O
report O
the O
cloning O
and O
sequencing O
of O
several O
overlapping O
cDNAs O
encoding O
approximately O
4 O
. O
1 O
kb O
of O
the O
human B
homologue I
of I
Wnt I
- I
5A I
. O

Subtype O
- O
and O
response O
element O
- O
dependent O
differences O
in O
transactivation O
by O
peroxisome B
proliferator I
- I
activated I
receptors I
alpha I
and I
gamma I
. O

MRI O
visualization O
of O
proteoglycan O
depletion O
in O
articular O
cartilage O
via O
intravenous O
administration O
of O
Gd O
- O
DTPA O
. O

We O
then O
developed O
a O
yeast O
artificial O
chromosome O
( O
YAC O
) O
contig O
for O
this O
region O
. O

We O
suggest O
that O
the O
induction O
of O
IFNA B
promoter I
region I
requires O
cooperation O
between O
alpha B
F1 I
binding I
proteins I
and O
IRF B
- I
1 I
. O

Heterologous O
hybridization O
with O
a O
rps12 B
gene I
specific O
probe O
from O
Euglena O
has O
revealed O
the O
presence O
of O
rps12 B
homologous I
sequences I
within O
the O
inverted O
repeat O
of O
Spirodela O
chloroplast O
DNA O
on O
the O
fragment O
BamHI B
- I
V I
. O

To O
investigate O
the O
effect O
of O
hyperthyroidism O
on O
the O
pattern O
and O
time O
course O
of O
O2 O
uptake O
( O
VO2 O
) O
following O
the O
transition O
from O
rest O
to O
exercise O
, O
six O
patients O
and O
six O
healthy O
subjects O
performed O
cycle O
exercise O
at O
an O
average O
work O
rate O
( O
WR O
) O
of O
18 O
and O
20 O
W O
respectively O
. O

However O
, O
to O
date O
, O
no O
viable O
in O
vitro O
- O
generated O
deletion O
mutant O
of O
PSTVd B
has O
been O
reported O
. O

This O
raises O
the O
possibility O
that O
recombination O
occurred O
between O
corresponding O
LTR O
and O
vif B
loci I
of O
the O
quasi O
- O
species O
present O
in O
the O
isolates O
described O
here O
. O

In O
HMEC O
stably O
transfected O
with O
an O
ER B
mutant I
containing O
a O
deletion O
in O
the O
second O
zinc O
finger O
of O
the O
DNA O
- O
binding O
domain O
, O
E B
and O
HT O
had O
different O
effects O
on O
EBBP B
gene I
expression O
; O
EBBP B
regulation O
by O
E B
was O
dramatically O
reduced O
while O
the O
effects O
of O
HT O
were O
augmented O
. O

The O
gene B
atp6 I
, O
encoding O
subunit O
6 O
of O
the O
mitochondrial B
F0 I
- I
ATPase I
complex I
, O
has O
been O
characterized O
from O
both O
the O
normal O
( O
fertile O
) O
and O
Ogura O
( O
male O
- O
sterile O
) O
radish O
cytoplasms O
in O
order O
to O
determine O
if O
previously O
identified O
atp6 B
transcriptional O
differences O
could O
play O
a O
role O
in O
cytoplasmic O
male O
sterility O
. O

When O
the O
same O
single O
- O
amino O
- O
acid O
mutations O
were O
directly O
introduced O
into O
the O
parental O
PV O
/ O
CBV4 O
- O
2A O
genome O
, O
chimeric O
viruses O
with O
a O
large O
- O
plaque O
phenotype O
and O
a O
wild O
- O
type O
PV O
- O
like O
growth O
pattern O
were O
obtained O
upon O
transfection O
, O
an O
observation O
demonstrating O
that O
these O
point O
mutations O
alone O
had O
a O
drastic O
effect O
on O
the O
growth O
of O
the O
PV O
/ O
CBV4 O
chimeric O
virus O
. O

The O
gag B
- O
myc B
proteins O
encoded O
by O
these O
variants O
efficiently O
localized O
to O
the O
cell O
nucleus O
. O

Hpr1 B
forms O
, O
together O
with O
Tho2 B
, O
Mft1 B
, O
and O
Thp2 B
, O
the O
THO B
complex I
, O
which O
controls O
transcription O
elongation O
and O
genome O
stability O
in O
Saccharomyces O
cerevisiae O
. O

UPR O
elements O
present O
in O
other O
ER B
chaperone I
genes I
, O
such O
as O
yeast B
KAR2 I
( O
BiP B
) O
, O
mammalian B
GRP78 I
( O
BiP B
) O
, O
and O
GRP94 B
, O
function O
in O
an O
analogous O
manner O
to O
that O
in O
FKB2 B
. O

The O
objective O
of O
this O
research O
was O
to O
determine O
if O
ergotamine O
, O
an O
ergopeptine O
alkaloid O
isolated O
from O
Neotyphodium O
- O
infected O
grasses O
and O
associated O
with O
toxicoses O
in O
livestock O
, O
altered O
plasma O
concentrations O
of O
reproductive O
hormones O
in O
follicular O
phase O
heifers O
and O
in O
cows O
given O
a O
progestin O
implant O
. O

Acute O
spontaneous O
subdural O
haematoma O
. O

Strains O
carrying O
a O
snf1 B
mutation I
are O
unable O
to O
grow O
on O
sucrose O
, O
galactose O
, O
maltose O
, O
melibiose O
, O
or O
nonfermentable O
carbon O
sources O
; O
utilization O
of O
these O
carbon O
sources O
is O
regulated O
by O
glucose O
repression O
. O

2 O
) O
Depending O
on O
the O
structure O
, O
certain O
compounds O
are O
sequestered O
in O
the O
cornea O
( O
presumably O
the O
stroma O
) O
and O
form O
a O
release O
system O
into O
the O
anterior O
aqueous O
. O

The O
results O
of O
the O
neural O
network O
were O
compared O
with O
those O
of O
a O
density O
mask O
( O
thresholds O
, O
- O
750 O
/ O
- O
300 O
H O
) O
, O
with O
a O
radiologist O
serving O
as O
the O
gold O
standard O
. O

These O
data O
also O
suggest O
the O
existence O
of O
a O
mechanism O
by O
which O
regulatory O
subunits O
modulate O
the O
PI B
3 I
- I
kinase I
- O
mediated O
signals O
, O
independent O
of O
the O
kinase O
activity O
, O
possibly O
through O
subcellular O
localization O
of O
the O
catalytic O
subunit O
or O
interaction O
with O
additional O
signaling O
molecules O
. O

These O
results O
suggest O
that O
the O
PVB O
therapy O
is O
not O
sufficient O
to O
cure O
the O
cases O
with O
choriocarcinoma O
element O
or O
bulky O
metastasis O
. O

Right O
- O
sided O
hemiplegia O
of O
5 O
years O
duration O
occurred O
in O
the O
remaining O
case O
. O

Plasmid O
subclones O
of O
the O
hr1a B
- O
containing O
AcMNPV O
HindIII B
- I
N I
fragment I
were O
examined O
for O
their O
ability O
to O
replicate O
in O
virus O
- O
infected O
( O
Spodoptera O
frugiperda O
) O
Sf9 O
cells O
, O
and O
to O
stimulate O
transcription O
when O
linked O
in O
cis O
with O
a O
39K O
gene O
promoter O
- O
beta B
- I
glucuronidase I
fusion O
and O
cotransfected O
into O
cells O
along O
with O
a O
plasmid O
( O
ple O
- O
1 O
) O
containing O
the O
gene O
encoding O
the O
trans O
- O
acting O
factor O
IE B
- I
1 I
. O

Transfection O
of O
H B
chain I
loss O
variant O
myeloma O
with O
the O
complete O
12 O
kb O
construct O
, O
termed O
238H B
- I
Cmicro I
, O
resulted O
in O
secretion O
of O
intact O
Ig B
pairing O
238H B
- O
Cmicro B
, O
with O
a O
lambda B
L I
chain I
; O
however O
, O
transfectant O
Ig B
lacked O
autoreactivity O
and O
pathogenicity O
. O

Divergent O
regions O
of O
the O
posterior O
left O
hemisphere O
used O
for O
decoding O
and O
storage O
of O
information O
emerged O
in O
each O
working O
memory O
versus O
control O
task O
comparison O
. O

In O
normal O
B O
cells O
stimulated O
by O
LPS O
or O
IL B
- I
4 I
, O
new O
complexes O
appear O
that O
bind O
to O
C B
/ I
EBP I
and O
NF B
- I
IL I
- I
4 I
elements I
, O
respectively O
, O
located O
within O
the O
- O
125 O
/ O
- O
101 O
region O
. O

Here O
, O
we O
describe O
the O
crystal O
structure O
at O
2 O
. O
5 O
A O
resolution O
of O
a O
fragment O
of O
the O
integrase B
of I
Rous I
sarcoma I
virus I
( O
residues O
49 O
- O
286 O
) O
containing O
both O
the O
conserved O
catalytic O
domain O
and O
a O
modulatory O
DNA O
- O
binding O
domain O
( O
C O
domain O
) O
. O

Vectorcardiography O
in O
coronary O
patients O
with O
the O
electrocardiographic O
RS O
segment O
in O
the O
V1 O
lead O

The O
B O
. O
aphidicola O
argS B
- O
rrn B
DNA O
fragments O
from O
endosymbionts O
from O
seven O
species O
of O
aphids O
had O
promoter O
activities O
in O
E O
. O
coli O
which O
ranged O
from O
6 O
to O
135 O
% O
of O
that O
observed O
with O
a O
comparable O
DNA O
fragment O
of O
E B
. I
coli I
rrnB I
. O

The O
adeno B
- I
associated I
virus I
type I
2 I
( I
AAV I
- I
2 I
) I
Rep78 I
/ O
Rep68 B
regulatory O
proteins O
are O
pleiotropic O
effectors O
of O
viral O
and O
cellular O
DNA O
replication O
, O
of O
cellular O
transformation O
by O
viral O
and O
cellular O
oncogenes O
, O
and O
of O
homologous O
and O
heterologous O
gene O
expression O
. O

Role O
of O
the O
CCAAT B
/ I
enhancer I
binding I
protein I
- I
alpha I
transcription I
factor I
in O
the O
glucocorticoid O
stimulation O
of O
p21waf1 B
/ O
cip1 B
gene O
promoter O
activity O
in O
growth O
- O
arrested O
rat O
hepatoma O
cells O
. O

Long O
- O
term O
ambulatory O
measurement O
of O
transcutaneous O
arterial O
CO2 O
pressure O
( O
PCO2 O
) O
offers O
an O
opportunity O
to O
test O
directly O
the O
co O
- O
occurrence O
of O
panic O
and O
hyperventilation O
under O
natural O
conditions O
. O

The O
highly O
conserved O
region O
of O
U6 B
snRNA I
has O
a O
structural O
similarity O
with O
the O
catalytic O
domain O
of O
the O
negative O
strand O
of O
the O
satellite O
RNA O
of O
tobacco O
ring O
spot O
virus O
[ O
( O
- O
) O
sTRSV O
] O
, O
suggesting O
that O
the O
highly O
conserved O
region O
of O
U6 B
snRNA I
forms O
the O
catalytic O
center O
. O

Here O
we O
show O
that O
one O
nudF B
suppressor O
also O
suppresses O
hs O
- O
mutations O
in O
nudA B
, O
nudC B
, O
and O
nudG B
and O
deletions O
in O
nudA B
and O
nudF B
. O

Treatment O
of O
cells O
for O
10 O
min O
with O
TNF B
- I
alpha I
resulted O
in O
activation O
of O
p44 B
/ I
42 I
MAPK I
, O
p38 B
, O
and O
JNK B
. O

This O
report O
describes O
the O
isolation O
and O
recombinant O
expression O
of O
a O
cDNA O
clone O
encoding O
HER4 B
, O
the O
fourth O
member O
of O
the O
human B
epidermal I
growth I
factor I
receptor I
( O
EGFR B
) O
family O
. O

In O
conclusion O
, O
when O
pulmonary O
abnormalities O
are O
found O
in O
HTLV O
- O
1 O
carriers O
, O
we O
should O
be O
careful O
in O
determining O
whether O
such O
pulmonary O
involvements O
are O
etiologically O
related O
to O
HTLV O
- O
1 O
infection O
. O

A O
clinical O
trial O
of O
a O
new O
mitomycin O
C O
derivative O
, O
KW O
- O
2083 O
( O
7 O
- O
N O
- O
( O
p O
- O
hydroxyphenyl O
) O
- O
mitomycin O
C O
) O

Excess O
recombinant B
PTB I
squelches O
the O
splicing O
switch O
and O
reestablishes O
exon O
skipping O
as O
the O
predominant O
splicing O
pathway O
. O

A O
child O
presumed O
to O
have O
the O
21 B
- I
hydroxylase I
deficiency O
form O
of O
congenital O
adrenal O
hyperplasia O
was O
studied O
extensively O
as O
an O
infant O
. O

Sixtieth O
anniversary O
of O
Angiotensin B
. O

Inability O
of O
niacin O
to O
protect O
from O
in O
vivo O
hyperoxia O
or O
in O
vitro O
microsomal O
lipid O
peroxidation O
. O

MDA O
- O
MB O
- O
468 O
cells O
were O
stably O
transfected O
with O
either O
a O
plasmid O
having O
a O
CMV O
promoter O
- O
driven O
rabbit B
beta I
- I
globin I
gene I
or O
plasmids O
having O
a O
CMV O
promoter O
- O
driven O
chimeric O
gadd45 B
5 I
" I
- I
UTR I
- O
rabbit B
beta I
- I
globin I
gene O
, O
where O
the O
entire O
gadd45 B
5 I
" I
- I
UTR I
( O
from O
+ O
1 O
to O
+ O
298 O
) O
or O
a O
45 O
bp O
subfragment O
of O
the O
gadd45 B
5 I
" I
- I
UTR I
( O
from O
+ O
10 O
to O
+ O
55 O
) O
was O
positioned O
at O
the O
5 O
" O
- O
end O
of O

the O
rabbit B
beta I
- I
globin I
gene I
. O

The O
University O
of O
North O
Carolina O
caries O
risk O
assessment O
was O
conducted O
between O
1986 O
and O
1989 O
with O
5000 O
children O
initially O
in O
grades O
1 O
and O
5 O
from O
low O
fluoride O
sites O
in O
South O
Carolina O
and O
Maine O
. O

Following O
EGF B
or O
NGF B
stimulation O
of O
the O
v B
- I
CrkPC12 I
cells O
, O
the O
v B
- I
Crk I
protein I
itself O
became O
tyrosine O
phosphorylated O
within O
1 O
min O
. O

BCR B
- O
ABL B
expression O
confers O
cross O
- O
resistance O
to O
multiple O
genotoxic O
anticancer O
drugs O
by O
inhibition O
of O
the O
apoptotic O
response O
to O
DNA O
damage O
in O
association O
with O
cell O
cycle O
arrest O
at O
the O
G2 O
- O
M O
restriction O
point O
. O

RESULTS O
: O
After O
lavage O
, O
a O
rapid O
decrease O
in O
arterial O
pH O
and O
PaO2 O
, O
and O
an O
increase O
in O
PaCO2 O
and O
PIP O
were O
observed O
in O
all O
animals O
. O

CONCLUSIONS O
: O
Elevation O
of O
TBARS O
and O
decrease O
of O
GSH O
show O
the O
presence O
of O
oxidative O
stress O
during O
the O
PFD O
treatment O
with O
hemodiafilter O
SG3 O
. O

Ozone O
uptake O
was O
assessed O
in O
awake O
, O
spontaneously O
breathing O
Fischer O
- O
344 O
Sprague O
- O
Dawley O
, O
and O
Long O
- O
Evans O
rats O
and O
Hartley O
guinea O
pigs O
to O
provide O
data O
on O
the O
dosimetry O
of O
O3 O
in O
small O
laboratory O
animals O
. O

E O
. O
R O
. O
C O
. O
P O
. O

is O
an O
important O
advance O
in O
diagnosis O
. O

Deletion O
analysis O
of O
the O
3 O
. O
5kb O
DNA O
fragment O
revealed O
that O
the O
region O
between O
- O
125 O
to O
+ O
1 O
, O
containing O
a O
single O
Sp1 B
binding I
site I
, O
is O
essential O
for O
transcription O
of O
the O
embigin B
gene I
. O

Resistance O
to O
Mup O
was O
classified O
as O
low O
( O
minimal O
inhibitory O
concentration O
[ O
MIC O
] O
> O
or O
= O
8 O
microg O
/ O
mL O
) O
or O
high O
( O
MIC O
> O
or O
= O
512 O
microg O
/ O
mL O
) O
degree O
. O

Extragonadal O
endodermal O
sinus O
tumors O
in O
the O
head O
and O
neck O
are O
very O
rare O
. O

Lesions O
of O
this O
kind O
resemble O
those O
in O
vitamin O
A O
- O
deficient O
chickens O
and O
are O
the O
first O
to O
be O
induced O
by O
excess O
vitamin O
E O
. O

Essential O
fatty O
acid O
deficiency O
in O
childhood O

The O
protein B
kinase I
C I
inhibitor O
GF O
- O
109203X O
abolished O
the O
activation O
by O
phorbol O
ester O
and O
inhibited O
the O
effect O
of O
CCK B
by O
78 O
% O
but O
had O
no O
effect O
on O
EGF B
- O
activated O
MAPK B
activity O
. O

Activated O
FGFR3 B
predominantly O
interacts O
with O
GRB2 B
. O
Sos B
in O
complex O
with O
a O
previously O
identified O
90 O
- O
kDa O
protein O
and O
designated O
protein B
80K I
- I
H I
. O

The O
rate O
of O
collagen B
synthesis O
in O
normal O
scar O
was O
approximately O
constant O
between O
6 O
months O
and O
20 O
years O
after O
the O
initial O
wounding O
, O
but O
in O
both O
hypertrophic O
scar O
and O
keloid O
the O
rate O
was O
initially O
approximately O
twice O
that O
in O
normal O
scar O
, O
and O
2 O
- O
3 O
years O
after O
wounding O
it O
fell O
to O
approximately O
the O
same O
level O
as O
in O
normal O
scar O
. O

Evidence O
for O
a O
role O
of O
Smad6 B
in O
chick O
cardiac O
development O
. O

Excellent O
results O
were O
recorded O
in O
390 O
( O
83 O
. O
69 O
% O
) O
of O
the O
patients O
, O
improvement O
in O
46 O
( O
9 O
. O
87 O
% O
) O
where O
in O
the O
majority O
a O
varicose O
complex O
was O
involved O
. O

Mutational O
analysis O
of O
yeast B
CEG1 I
demonstrated O
that O
four O
of O
the O
five O
conserved O
motifs O
are O
essential O
for O
capping B
enzyme I
function O
in O
vivo O
. O

We O
report O
on O
a O
case O
discovered O
in O
a O
13 O
year O
- O
old O
girl O
presenting O
with O
anaemia O
. O

The O
LIF B
appeared O
to O
be O
released O
by O
the O
patient O
' O
s O
peripheral O
blood O
lymphocytes O
when O
cultured O
with O
optimal O
doses O
of O
propranolol O
. O

STUDY O
SELECTION O
: O
Not O
applicable O
. O

Co O
- O
expression O
of O
PLD1 B
in O
COS O
- O
7 O
cells O
with O
the O
two O
recombinant B
CK2 I
subunits I
, O
alpha O
or O
beta O
, O
suggests O
that O
the O
association O
of O
PLD1 B
with O
the O
kinase O
is O
through O
the O
beta O
subunit O
. O

For O
capsules O
containing O
only O
the O
drug O
, O
the O
value O
of O
T50 O
increased O
as O
the O
particle O
size O
of O
the O
drug O
decreased O
. O

The O
LAB O
in O
the O
cecum O
( O
mean O
9 O
. O
4 O
log O
CFU O
/ O
g O
) O
increased O
slightly O
with O
increasing O
abattoir O
holding O
time O
. O

Our O
objective O
was O
to O
find O
possible O
predictors O
for O
the O
expression O
and O
progression O
of O
LJM O
and O
to O
evaluate O
the O
relationship O
between O
LJM O
and O
other O
long O
- O
term O
complications O
of O
insulin B
- O
dependent O
diabetes O
mellitus O
. O

Human B
recombinant I
erythropoietin I
( O
r B
- I
HuEPO I
, O
EprexR O
) O
was O
administered O
to O
8 O
children O
with O
chronic O
renal O
failure O
and O
high O
transfusion O
requirement O
. O

CONCLUSIONS O
: O
LHB O
is O
simple O
, O
easy O
and O
safe O
to O
implement O
, O
and O
is O
the O
only O
technique O
capable O
of O
maintaining O
independent O
upper O
and O
lower O
body O
perfusion O
pressure O
. O

Northern O
analyses O
further O
confirm O
that O
the O
expression O
of O
endogenous B
alpha I
- I
ENaC I
gene I
in O
salivary O
Pa O
- O
4 O
cells O
is O
suppressed O
by O
an O
ectopic O
HMGI B
- I
C I
overexpression O
. O

Removal O
of O
cyclin B
- I
dependant I
kinases I
( O
cdks B
) O
or O
cdk2 B
from O
these O
extracts O
using O
affinity O
matrices O
severely O
inhibits O
initiation O
of O
S O
phase O
. O

Gel O
mobility O
shift O
analyses O
reveal O
that O
FRTL O
- O
5 O
thyroid O
cell O
nuclear O
extracts O
form O
a O
specific O
protein O
/ O
DNA O
complex O
with O
this O
region O
, O
which O
is O
prevented O
by O
the O
TTF B
- I
1 I
binding I
element I
from O
the O
TG B
promoter I
; O
FRT O
and O
BRL O
cell O
nuclear O
extracts O
do O
not O
have O
TTF B
- I
1 I
and O
do O
not O
form O
this O
complex O
. O

The O
Sty1 B
kinase I
is O
stimulated O
by O
a O
variety O
of O
different O
stress O
conditions O
including O
osmotic O
and O
oxidative O
stress O
and O
heat O
shock O
. O

No O
viral O
RNA O
replication O
could O
be O
detected O
in O
cells O
transfected O
with O
mutant O
RNAs O
. O

Acad O
. O

Comparison O
of O
the O
genes O
coding O
for O
mouse O
and O
human B
p36 I
( O
annexin B
II I
) O
and O
mouse O
, O
rat O
and O
human B
p35 I
( O
annexin B
I I
) O
and O
pigeon B
cp35 I
( O
an O
annexin B
I I
- I
related I
protein I
) O
shows O
strong O
genomic O
structural O
conservation O
supporting O
the O
hypothesis O
that O
these O
genes O
had O
a O
common O
ancestor O
. O

When O
incubated O
with O
infective O
larvae O
which O
had O
penetrated O
mouse O
skin O
, O
both O
normal O
and O
immune O
cells O
attached O
to O
larvae O
in O
the O
absence O
of O
serum O
. O

A O
31 O
- O
year O
- O
old O
man O
with O
primary O
myelofibrosis O
initially O
received O
low O
dose O
Ara O
C O
. O

Collectively O
, O
these O
studies O
suggest O
that O
the O
major O
EGF B
- O
stimulated O
mitotic O
growth O
pathways O
may O
not O
be O
absolutely O
linked O
to O
the O
stat91 B
signaling O
pathways O
and O
that O
such O
transcription O
complexes O
are O
more O
complex O
than O
previously O
reported O
. O

FEurea O
was O
also O
measured O
during O
stable O
graft O
function O
, O
7 O
- O
14 O
days O
prior O
to O
allograft O
dysfunction O
. O

Nonsuicidal O
mortality O
in O
late O
- O
life O
depression O
. O

Few O
triers O
or O
users O
had O
received O
SLT O
counseling O
from O
their O
dentist O
despite O
high O
dental O
utilization O
rates O
. O

However O
, O
UV O
resistance O
is O
only O
minimally O
restored O
, O
and O
mutant O
cells O
remain O
sensitive O
to O
gamma O
radiation O
. O

The O
oxalate B
oxidase I
method O
results O
in O
a O
mean O
and O
reference O
interval O
for O
oxalate O
excretion O
that O
are O
comparable O
with O
those O
by O
isotope O
dilution O
, O
gas O
- O
chromatographic O
, O
colorimetric O
, O
and O
other O
enzymic O
procedures O
. O

In O
addition O
, O
we O
show O
that O
expression O
of O
antisense O
mRNA O
for O
p130CAS B
resulted O
in O
reversion O
of O
the O
transformed O
phenotype O
of O
all O
these O
cell O
lines O
. O

Starches O
had O
no O
effect O
. O

Maximal O
Expiratory O
Flow O
Rates O
such O
as O
Peak O
Expiratory O
Flow O
Rate O
( O
PEFR O
) O
, O
rates O
at O
25 O
% O
, O
50 O
% O
and O
75 O
% O
of O
forced O
vital O
capacity O
( O
V O
max O
25 O
% O
, O
V O
max O
50 O
% O
and O
V O
max O
75 O
% O
) O
and O
forced O
expiratory O
flow O
during O
the O
middle O
half O
of O
forced O
vital O
capacity O
( O
FEF O
25 O
- O
75 O
% O
) O
were O
measured O
in O
273 O
healthy O
non O
- O
smoking O
adults O
( O
144 O
males O
, O
129 O
females O
) O
aged O
15 O
- O
63 O
years O
living O
in O
Madras O
. O

The O
amino O
acid O
sequence O
of O
the O
protein O
encoded O
by O
MSK1 B
is O
homologous O
to O
yeast B
cytoplasmic I
lysyl I
- I
tRNA I
synthetase I
and O
to O
the O
product O
of O
the O
herC B
gene I
, O
which O
has O
recently O
been O
suggested O
to O
code O
for O
the O
Escherichia O
coli O
enzyme O
. O

Similar O
to O
the O
D B
, I
B I
/ I
B I
' I
and I
E I
proteins I
, O
the O
F B
and I
G I
proteins I
do O
not O
possess O
any O
of O
the O
known O
RNA O
binding O
motifs O
, O
suggesting O
that O
other O
types O
of O
RNA O
- O
protein O
interactions O
occur O
in O
the O
snRNP O
core O
. O

These O
observations O
suggest O
that O
clr4 B
+ I
and O
rik1 B
+ I
must O
play O
a O
role O
in O
the O
assembly O
of O
Swi6p B
into O
a O
transcriptionally O
silent O
, O
inaccessible O
chromatin O
structure O
at O
fission O
yeast O
centromeres O
which O
is O
required O
to O
facilitate O
interactions O
with O
spindle O
microtubules O
and O
to O
ensure O
normal O
chromosome O
segregation O
. O

Characterization O
of O
a O
DEAD B
box I
ATPase I
/ O
RNA B
helicase I
protein O
of O
Arabidopsis O
thaliana O
. O

Amplitude O
of O
late O
surface O
( O
P2 O
and O
N2 O
) O
and O
depth O
( O
B O
and O
C O
) O
components O
significantly O
decreased O
when O
patients O
shifted O
from O
SWS O
IV O
to O
PS O
and O
increased O
from O
PS O
to O
W2 O
. O

Oligo O
- O
[ O
alpha O
] O
- O
deoxynucleotides O
can O
be O
derived O
by O
stabilizing O
( O
intercalating O
) O
agents O
or O
reactive O
groups O
( O
cleaving O
reagents O
, O
cross O
- O
linkers O
. O
. O
. O
) O
. O

This O
vector O
should O
be O
applicable O
for O
high O
- O
throughput O
characterization O
of O
new O
open O
reading O
frames O
found O
in O
genome O
sequencing O
. O

The O
subjects O
were O
17 O
patients O
with O
android O
obesity O
( O
four O
men O
and O
13 O
women O
) O
aged O
21 O
to O
58 O
years O
with O
a O
body O
mass O
index O
( O
BMI O
) O
ranging O
from O
32 O
. O
0 O
to O
52 O
. O
2 O
kg O
/ O
m2 O
and O
an O
abdominal O
- O
gluteal O
ratio O
greater O
than O
1 O
. O
0 O
. O

No O
universally O
accepted O
standardized O
classification O
system O
for O
acne O
vulgaris O
exists O
, O
although O
there O
is O
a O
strong O
need O
for O
it O
. O

Blood O
samples O
for O
determination O
of O
fibrinolytic O
activity O
and O
factor B
VIII I
in O
plasma O
were O
obtained O
before O
and O
immediately O
after O
the O
end O
of O
compression O
and O
application O
of O
a O
stocking O
, O
respectively O
. O

Neither O
symptoms O
, O
virions O
, O
nor O
viral O
RNA O
was O
detectable O
in O
plants O
inoculated O
with O
this O
mutant O
or O
a O
mutant O
with O
a O
frameshift O
mutation O
in O
the O
coat B
gene I
. O

The O
cumulative O
length O
of O
the O
RGSV O
genome O
, O
including O
RNAs O
5 O
and O
6 O
, O
was O
25142 O
nt O
. O

Risk O
ratios O
decreased O
with O
increasing O
age O
and O
increased O
with O
number O
of O
breast O
biopsies O
, O
family O
history O
of O
breast O
cancer O
, O
estrogen O
use O
, O
time O
between O
screenings O
, O
no O
comparison O
with O
previous O
mammograms O
, O
and O
the O
radiologist O
' O
s O
tendency O
to O
call O
mammograms O
abnormal O
. O

The O
AtNMT1 B
expression O
profile O
indicated O
ubiquity O
in O
roots O
, O
stem O
, O
leaves O
, O
flowers O
, O
and O
siliques O
( O
approximately O
1 O
. O
7 O
kb O
transcript O
and O
approximately O
50 O
kDa O
immunoreactive O
polypeptide O
) O
but O
a O
greater O
level O
in O
the O
younger O
tissue O
, O
which O
are O
developmentally O
very O
active O
. O

The O
ossification O
was O
located O
on O
the O
left O
side O
of O
C3 O
- O
4 O
. O

At O
a O
cutoff O
point O
for O
KISA O
% O
of O
100 O
, O
280 O
of O
281 O
participants O
( O
99 O
. O
6 O
% O
) O
were O
correctly O
classified O
. O

Following O
replicate O
control O
measurements O
, O
test O
ramps O
were O
repeated O
in O
the O
presence O
of O
sodium O
nitroprusside O
( O
1 O
microM O
) O
and O
phentolamine O
( O
1 O
microM O
) O
to O
eliminate O
potential O
smooth O
muscle O
and O
alpha B
1 I
- I
adrenoceptor I
effects O
, O
respectively O
. O

Behavioral O
effects O
of O
benzodiazepine O
ligands O
in O
non O
- O
dependent O
, O
diazepam O
- O
dependent O
and O
diazepam O
- O
withdrawn O
baboons O
. O

This O
paper O
reports O
estimates O
of O
the O
prevalence O
of O
mental O
retardation O
and O
associated O
factors O
based O
on O
a O
population O
survey O
of O
2 O
- O
to O
9 O
- O
year O
- O
old O
children O
in O
Greater O
Karachi O
, O
Pakistan O
. O

Thus O
, O
an O
increase O
in O
lactate O
concentration O
of O
the O
magnitude O
observed O
during O
alkali O
therapy O
need O
not O
indicate O
a O
worsening O
of O
the O
metabolic O
picture O
in O
lactic O
acidosis O
. O

Adjusting O
a O
steady O
- O
state O
warfarin O
dose O
depends O
on O
the O
measured O
INR O
values O
and O
clinical O
factors O
: O
the O
dose O
does O
not O
need O
to O
be O
adjusted O
for O
a O
single O
INR O
that O
is O
slightly O
out O
of O
range O
, O
and O
most O
changes O
should O
alter O
the O
total O
weekly O
dose O
by O
5 O
% O
to O
20 O
% O
. O

All O
of O
this O
region O
is O
part O
of O
a O
prominent O
CpG O
island O
that O
may O
be O
acting O
as O
an O
extended O
, O
enhancer O
- O
independent O
promoter O
. O

Comparison O
of O
Healon O
and O
Amvisc O
. O

Large O
volumes O
of O
bone O
marrow O
may O
be O
required O
for O
certain O
types O
of O
autologous O
bone O
marrow O
transplants O
. O

Further O
analysis O
of O
the O
CAP59 B
locus I
of O
Cryptococcus O
neoformans O
: O
structure O
defined O
by O
forced O
expression O
and O
description O
of O
a O
new O
ribosomal B
protein I
- I
encoding I
gene I
. O

We O
speculate O
that O
the O
enhancing O
effect O
of O
glucose O
and O
galactose O
on O
fructose O
absorption O
may O
be O
due O
to O
activation O
of O
the O
fructose B
carrier I
. O

Virol O
. O

The O
DFGF B
- I
R I
protein I
may O
thus O
participate O
in O
receiving O
spatial O
cues O
that O
guide O
tracheal O
cell O
outgrowth O
. O

The O
data O
from O
animal O
experiments O
indicate O
that O
the O
incidence O
of O
solid O
tumors O
in O
marrow O
transplant O
patients O
may O
still O
rise O
in O
the O
coming O
decades O
. O

Patients O
adopt O
new O
sets O
based O
partly O
on O
the O
original O
topic O
and O
partly O
on O
their O
own O
personal O
idiosyncratic O
concerns O
. O

The O
NS B
- I
2 I
isoforms I
were O
resolved O
at O
a O
pI O
value O
close O
to O
5 O
. O
5 O
as O
three O
groups O
of O
unevenly O
phosphorylated O
polypeptides O
, O
each O
composed O
of O
at O
least O
two O
protein O
species O
. O

Prevalence O
of O
rheumatic O
diseases O
in O
a O
rural O
population O
in O
western O
India O
: O
a O
WHO O
- O
ILAR O
COPCORD O
Study O
. O

A O
. O
nidulans O
transformants O
secreted O
high O
amounts O
of O
PGI B
and O
PGII B
in O
comparison O
to O
the O
previously O
characterized O
A O
. O
niger O
transformants O
and O
a O
novel O
polygalacturonase B
( O
PGC B
) O
was O
produced O
at O
high O
levels O
by O
A O
. O
nidulans O
transformed O
with O
the O
subcloned O
pgaC B
gene I
. O

This O
interaction O
is O
functional O
as O
it O
leads O
to O
retargeting O
of O
NF B
- I
kappa I
B I
p50 B
from O
the O
nucleus O
to O
the O
cytoplasm O
. O

The O
orf2 B
gene I
product I
is O
a O
protein O
of O
79 O
amino O
acids O
with O
characteristics O
similar O
to O
those O
of O
the O
Tat B
( I
transactivator I
) I
proteins I
of O
the O
ungulate O
lentiviruses O
. O

The O
use O
of O
medication O
is O
of O
minor O
importance O
in O
the O
treatment O
of O
flight O
phobia O
. O

Elevated O
total O
serum O
calcium O
to O
a O
level O
of O
11 O
. O
9 O
+ O
/ O
- O
0 O
. O
2 O
mg O
/ O
dl O
( O
p O
less O
than O
0 O
. O
001 O
compared O
to O
control O
group O
) O
developed O
in O
the O
gentamicin O
/ O
1 O
, O
25 O
( O
OH O
) O
2 O
vitamin O
D3 O
group O
on O
day O
4 O
, O
2 O
days O
prior O
to O
pronounced O
structural O
damage O
, O
and O
continued O
to O
be O
elevated O
through O
day O
7 O
. O

Rac2 B
, O
a O
member O
of O
the O
Rho B
family I
of O
GTPases B
, O
is O
highly O
expressed O
in O
myeloid O
cells O
and O
is O
a O
regulator O
of O
the O
NADPH B
- I
oxidase I
complex I
. O

In O
vitro O
kinase O
assays O
on O
isolated O
microvilli O
and O
microvillar O
fractions O
enriched O
in O
the O
putative O
signal O
transduction O
particle O
showed O
a O
high O
specific O
activity O
of O
tyrosine B
kinase I
activity O
compared O
to O
that O
of O
membranes O
from O
EGF B
receptor I
- O
overexpressing O
A431 O
cells O
maximally O
activated O
by O
EGF B
. O

Effect O
of O
infantile O
undernutrition O
on O
adult O
sucrose O
solution O
consumption O
in O
the O
rat O
. O

In O
the O
studies O
described O
in O
this O
report O
, O
we O
have O
identified O
two O
potential O
Ets B
binding I
sites I
, O
EBS1 B
and O
EBS2 B
, O
which O
are O
conserved O
in O
both O
the O
human B
and I
murine I
interleukin I
- I
2 I
enhancers I
. O

KatA B
, O
AhpCF B
, O
heme B
biosynthesis O
enzymes O
, O
and O
MrgA B
are O
also O
induced O
upon O
entry O
into O
stationary O
phase O
under O
conditions O
of O
iron O
and O
manganese O
limitation O
. O

Although O
the O
Gly252Arg O
substitution O
observed O
in O
UM O
: O
JG5 O
is O
non O
- O
conservative O
, O
it O
was O
not O
possible O
to O
distinguish O
whether O
it O
is O
a O
mutation O
or O
a O
polymorphism O
. O

This O
region O
shows O
46 O
% O
identity O
with O
the O
calmodulin B
- I
binding I
region I
of O
rat B
brain I
Ca2 I
+ I
/ I
calmodulin I
- I
dependent I
protein I
kinase I
II I
and O
32 O
% O
identity O
with O
the O
equivalent O
region O
of O
chicken B
smooth I
muscle I
myosin I
light I
chain I
kinase I
. O

The O
perforated O
splint O
gives O
superior O
results O
by O
virtue O
of O
the O
fact O
that O
it O
does O
not O
require O
to O
be O
removed O
for O
purposes O
of O
hygiene O
and O
can O
thus O
produce O
a O
satisfactory O
result O
even O
in O
those O
patients O
who O
fail O
to O
grasp O
the O
technique O
of O
removal O
and O
replacement O
of O
the O
splint O
. O

It O
contains O
31 O
exons O
and O
a O
high O
proportion O
of O
class O
0 O
introns O
, O
alternative O
splicing O
of O
which O
results O
in O
significant O
levels O
of O
variant O
transcripts O
that O
maintain O
the O
original O
open O
reading O
frame O
of O
MRP B
mRNA I
. O

Issues O
discussed O
include O
the O
validity O
of O
the O
MMPI O
Mf O
scale O
, O
questions O
of O
sample O
representativeness O
, O
whether O
" O
straights O
" O
can O
do O
adequate O
research O
on O
homosexuality O
, O
differences O
in O
heterosexual O
and O
homosexual O
sexuality O
, O
the O
role O
of O
subculture O
acculturation O
in O
homosexual O
psychological O
adjustment O
, O
and O
the O
use O
of O
cluster O
- O
analysis O
to O
generate O
typologies O
of O
homosexualities O
. O

A O
brief O
discussion O
for O
the O
relationship O
among O
these O
formulas O
is O
given O
. O

Lamivudine O
therapy O
for O
acute O
hepatitis O
B O
infection O
following O
peripheral O
blood O
stem O
cell O
transplantation O
. O

These O
findings O
suggest O
that O
NMDA B
receptor I
blockade O
may O
ameliorate O
the O
dyskinetic O
complications O
of O
long O
- O
term O
levodopa O
therapy O
, O
without O
diminishing O
the O
beneficial O
effects O
on O
parkinsonian O
signs O
. O

Furthermore O
, O
the O
secondary O
stress O
responses O
were O
more O
pronounced O
and O
the O
ability O
to O
recover O
from O
them O
seemed O
to O
be O
impaired O
in O
exposed O
fish O
as O
compared O
to O
unexposed O
fish O
. O

The O
distinct O
spatial O
pattern O
of O
expression O
, O
and O
unusual O
amino O
acid O
sequence O
in O
its O
DNA O
binding O
domain O
, O
may O
indicate O
a O
particular O
role O
for O
DTEF B
- I
1 I
in O
cell O
- O
specific O
gene O
regulation O
. O

As O
usual O
tests O
, O
such O
as O
" O
time O
" O
of O
cephalin O
are O
slightly O
sensible O
to O
LMW O
- O
Hep O
, O
only O
anti B
- I
Xa I
activity O
can O
be O
used O
. O

Usher O
syndrome O
1C O
( O
USH1C O
) O
is O
a O
congenital O
condition O
manifesting O
profound O
hearing O
loss O
, O
the O
absence O
of O
vestibular O
function O
, O
and O
eventual O
retinal O
degeneration O
. O

Implantation O
is O
contraindicated O
for O
severe O
dysplasia O
of O
the O
cochlea O
and O
for O
the O
recently O
described O
variety O
of O
x O
- O
linked O
deafness O
with O
deficient O
bone O
at O
the O
fundus O
of O
the O
internal O
auditory O
meatus O
. O

Over O
9 O
kb O
of O
DNA O
sequence O
was O
obtained O
for O
one O
clone O
( O
A1 O
) O
with O
a O
total O
IGS B
length O
of O
approximately O
12 O
. O
4 O
kb O
. O

Scleraxis B
overexpression O
enhanced O
expression O
of O
the O
aggrecan B
gene I
, O
which O
is O
not O
normally O
expressed O
at O
high O
levels O
in O
these O
osteoblastic O
cells O
. O

This O
study O
demonstrates O
that O
the O
exposure O
to O
sulphur O
mustard O
results O
in O
very O
low O
androgen O
levels O
and O
hypo O
- O
responsiveness O
to O
GnRH B
in O
the O
first O
five O
weeks O
and O
normalization O
by O
the O
twelfth O
week O
after O
injury O
. O

New O
results O
of O
importance O
for O
optimization O
of O
the O
postoperative O
course O
] O
Complications O
after O
major O
surgery O
may O
be O
related O
to O
factors O
in O
the O
surgical O
stress O
response O
with O
endocrine O
- O
metabolic O
and O
inflammatory O
changes O
. O

Four O
multicannulated O
Holstein O
steers O
( O
initial O
BW O
424 O
+ O
/ O
- O
16 O
kg O
) O
were O
used O
in O
a O
4 O
x O
4 O
Latin O
square O
to O
determine O
the O
influence O
of O
protein O
supplementation O
on O
forage O
intake O
, O
site O
and O
extent O
of O
digestion O
, O
and O
nutrient O
flow O
in O
steers O
consuming O
dormant O
bluestem O
- O
range O
forage O
( O
2 O
. O
3 O
% O
CP O
) O
. O

Patients O
with O
risk O
factors O
were O
compared O
with O
those O
without O
risk O
factors O
. O

The O
dermatoglyphic O
pattern O
demostrates O
a O
distal O
triradius O
and O
small O
whorl O
abnormalities O
. O

The O
term O
Kartagener O
syndrome O
applies O
to O
this O
syndrome O
when O
accompanied O
by O
infertility O
and O
dextrocardia O
or O
situs O
inversus O
. O

Finally O
, O
we O
tested O
whether O
activation O
by O
either O
of O
these O
factors O
is O
dependent O
on O
components O
of O
the O
SAGA B
complex I
. O

Eight O
of O
the O
17 O
seropositive O
patients O
failed O
to O
develop O
detectable O
hepatitis B
B I
surface I
antibody I
within O
three O
months O
of O
the O
third O
injection O
compared O
with O
only O
one O
of O
the O
18 O
seronegative O
patients O
( O
p O
less O
than O
0 O
. O
01 O
) O
. O

The O
antimicrobial O
of O
choice O
for O
initial O
prophylactic O
therapy O
among O
asymptomatic O
pregnant O
women O
exposed O
to O
Bacillus O
anthracis O
is O
ciprofloxacin O
, O
500 O
mg O
twice O
a O
day O
for O
60 O
days O
. O

Overexpression O
of O
eIF4E B
transforms O
cells O
, O
and O
mutations O
in O
eIF4E B
arrest O
cells O
in O
G O
, O
in O
cdc33 B
mutants I
. O

Benzyl O
alcohol O
administration O
in O
neonates O
. O

Repression O
of O
the O
herpes B
simplex I
virus I
1 I
alpha I
4 I
gene I
by O
its O
gene O
product O
( O
ICP4 B
) O
within O
the O
context O
of O
the O
viral O
genome O
is O
conditioned O
by O
the O
distance O
and O
stereoaxial O
alignment O
of O
the O
ICP4 B
DNA I
binding I
site I
relative O
to O
the O
TATA O
box O
. O

As O
anticipated O
, O
both O
intracellular O
p54 B
- O
SAPKbeta B
activation O
and O
Bcl B
- I
2 I
phosphorylation O
are O
blocked O
by O
co O
- O
transfection O
with O
the O
MAP B
kinase I
specific O
phosphatase O
MKP3 B
/ O
PYST1 B
. O

Relation O
between O
circulating O
immune O
complexes O
and O
serum O
ferritin B
in O
hemosiderosis O
of O
different O
etiologies O

PATIENT O
( O
S O
) O
: O
Two O
hundred O
thirty O
- O
eight O
patients O
with O
RPLs O
, O
48 O
patients O
with O
recurrent O
IVF O
- O
ET O
failure O
and O
179 O
nonpregnant O
and O
120 O
pregnant O
control O
group O
women O
. O

Many O
of O
the O
metabolic O
effects O
induced O
by O
thiazide O
diuretics O
, O
however O
, O
can O
be O
limited O
by O
the O
use O
of O
low O
doses O
. O

Teboroxime O
is O
a O
new O
boronic O
acid O
adduct O
of O
technetium O
dioxime O
( O
BATO O
) O
compound O
that O
demonstrates O
favorable O
characteristics O
in O
preliminary O
studies O
. O

The O
total O
area O
under O
the O
blood O
glucose O
curve O
over O
the O
initial O
value O
was O
not O
altered O
2 O
h O
after O
treatment O
with O
SH O
B O
209 O
AB O
or O
Neogynon O
. O

Treatment O
of O
excessive O
bleeding O
after O
cardiopulmonary O
bypass O
was O
based O
on O
an O
algorithm O
using O
point O
- O
of O
- O
care O
testing O
with O
whole O
blood O
prothrombin B
time O
, O
activated O
partial O
thromboplastin B
time O
, O
heparinase B
activated O
clotting O
time O
, O
and O
platelet O
count O
. O

In O
all O
cases O
, O
a O
large O
fraction O
of O
soluble O
Al O
became O
insoluble O
in O
the O
stomach O
after O
ingestion O
. O

To O
assess O
the O
safety O
of O
the O
angiotensin B
converting I
enzyme I
inhibitor O
enalapril O
a O
multicenter O
, O
open O
, O
randomized O
, O
prazosin O
- O
controlled O
trial O
was O
designed O
comparing O
the O
incidence O
and O
severity O
of O
symptomatic O
hypotension O
on O
the O
first O
day O
of O
treatment O
. O

Glucose O
may O
reduce O
slightly O
the O
pool O
of O
amino O
nitrogen O
available O
for O
albumin B
and O
urea O
synthesis O
by O
diverting O
some O
of O
the O
infused O
amino O
acids O
from O
protein O
synthesis O
by O
the O
liver O
to O
muscle O
. O

BACKGROUND O
. O

We O
have O
previously O
reported O
the O
first O
isolation O
of O
a O
c B
- I
myc I
- O
null O
cell O
line O
. O

Results O
demonstrated O
that O
for O
both O
S O
. O
europaea O
and O
A O
. O
prostrata O
, O
the O
total O
number O
of O
seeds O
that O
germinated O
decreased O
throughout O
the O
growing O
season O
. O

A O
nationwide O
record O
linkage O
of O
the O
Finnish O
Twin O
Cohort O
Study O
( O
FTCS O
) O
with O
the O
Hospital O
Discharge O
Registry O
and O
the O
Registry O
of O
Rights O
for O
Free O
medication O
is O
presented O
. O

Considering O
these O
two O
facts O
together O
, O
we O
propose O
that O
vasectomy O
may O
lead O
to O
decrease O
in O
the O
incidence O
of O
prostatic O
tumors O
- O
a O
disease O
that O
claims O
nearly O
22 O
, O
000 O
lives O
each O
year O
in O
the O
United O
States O
alone O
. O

Antibodies O
specific O
to O
the O
different O
members O
of O
the O
Jun B
and O
Fos B
family I
of O
transcription O
factors O
show O
that O
, O
in O
gel O
retardation O
assays O
, O
a O
Jun B
- I
like I
factor I
is O
a O
component O
of O
the O
H3 B
. I
2 I
specific I
complex I
. O

She O
had O
been O
diagnosed O
with O
severe O
aplastic O
anemia O
1 O
year O
previously O
and O
, O
while O
hospitalized O
, O
had O
received O
methyl O
prednisolone O
pulse O
therapy O
, O
which O
was O
not O
successful O
. O

The O
recombinant B
L I
/ I
F I
- I
transferase I
is O
as O
active O
as O
the O
previously O
purified O
wild O
type O
enzyme O
and O
contains O
no O
detectable O
RNA O
component O
. O

Cord O
formation O
in O
BACTEC O
7H12 O
medium O
for O
rapid O
, O
presumptive O
identification O
of O
Mycobacterium O
tuberculosis O
complex O
. O

Moreover O
, O
site O
- O
specific O
integration O
of O
the O
ITR B
- O
flanked O
DNA O
was O
also O
detected O
by O
PCR O
amplification O
of O
the O
ITR B
- O
aavs1 B
junction O
in O
transduced O
human O
fibroblasts O
. O

Oct B
- I
1 I
is O
a O
ubiquitously O
expressed O
regulatory O
gene O
of O
the O
POU B
domain I
family I
. O

In O
both O
study O
stages O
. O
post O
- O
exposure O
treatment O
consisted O
of O
five O
injections O
of O
vaccine O
[ O
( O
D O
) O
ays O
0 O
, O
3 O
, O
7 O
, O
14 O
, O
28 O
] O
, O
together O
with O
a O
dose O
of O
rabies B
immunoglobulin I
( O
RIG B
) O
of O
equine O
or O
human O
origin O
on O
D0 O
. O

( O
3 O
) O
With O
the O
exception O
of O
the O
anteroposterior O
direction O
of O
layers O
II O
- O
- O
III O
and O
the O
mediolateral O
direction O
of O
layer O
IV O
, O
the O
vertical O
conductivity O
of O
the O
cortex O
was O
always O
greater O
than O
either O
of O
the O
horizontal O
conductivities O
. O

Our O
study O
investigated O
the O
hypothesis O
that O
the O
combination O
of O
a O
high O
NaCl O
diet O
and O
social O
isolation O
stress O
would O
increase O
systolic O
blood O
pressure O
( O
SBP O
) O
and O
endogenous O
sodium B
pump I
ligands I
( O
SPL B
) O
, O
ouabainlike B
compound I
( O
OLC O
) O
, O
and O
marinobufagenin B
( O
MBG O
) O
. O

The O
relationship O
between O
polymorphonuclear O
granulocytes O
and O
cartilage O
destruction O
in O
rheumatoid O
arthritis O
. O

It O
is O
suggested O
that O
the O
loss O
of O
weight O
, O
the O
decrease O
in O
food O
intake O
and O
the O
increase O
in O
brain O
MOPEG O
- O
SO4 O
induced O
by O
cyanamide O
reflect O
possible O
anorectic O
properties O
of O
the O
drug O
. O

RESULTS O
: O
Twenty O
- O
two O
patients O
( O
30 O
% O
) O
showed O
ST O
- O
segment O
depression O
during O
AEM O
and O
34 O
( O
49 O
% O
) O
on O
ExT O
. O

The O
virus O
must O
progress O
past O
the O
immediate O
- O
early O
phase O
and O
express O
an O
early B
gene I
product I
( O
s O
) O
for O
activation O
of O
cyclin B
E I
expression O
. O

In O
CYP7A1 B
transgenic O
mice O
, O
livers O
contained O
approximately O
3 O
- O
fold O
more O
sterol B
response I
element I
- I
binding I
protein I
- I
2 I
mRNA I
. O

Adenocarcinoma O
and O
large O
cell O
carcinoma O
have O
a O
worse O
prognosis O
than O
squamous O
cell O
carcinoma O
in O
the O
T1N1 O
and O
T2N1 O
subsets O
. O

In O
group O
I O
during O
the O
pain O
free O
period O
26 O
of O
81 O
patients O
had O
positive O
thallium O
- O
201 O
scans O
, O
whereas O
20 O
patients O
had O
an O
abnormal O
ECG O
at O
that O
time O
; O
during O
angina O
18 O
patients O
had O
transient O
ECG O
changes O
. O

The O
prototype O
now O
structures O
oncology O
- O
related O
information O
available O
on O
the O
Internet O
and O
also O
places O
resources O
maintained O
locally O
at O
users O
' O
disposal O
. O

Binding O
of O
the O
TorR B
regulator I
to O
cis O
- O
acting O
direct O
repeats O
activates O
tor B
operon I
expression O
. O

For O
the O
most O
part O
, O
only O
aerobic O
microbial O
degradation O
systems O
have O
been O
reported O
so O
far O
. O

Asymptomatic O
ischaemia O
during O
daily O
life O
in O
stable O
coronary O
artery O
disease O
: O
relevant O
or O
redundant O
? O

This O
is O
a O
consensus O
casein B
kinase I
II I
( O
CKII B
) O
site O
and O
, O
using O
purified O
wild O
- O
type O
and O
mutant O
proteins O
, O
we O
show O
that O
it O
is O
the O
main O
CKII B
site I
in O
the O
body O
of O
the O
N O
- O
terminal O
complex O
- O
forming O
region O
. O

In O
contrast O
to O
Phase O
III O
, O
which O
has O
a O
single O
study O
design O
- O
- O
the O
controlled O
, O
randomized O
, O
double O
- O
blind O
study O
- O
- O
Phase O
IV O
requires O
different O
designs O
for O
each O
of O
the O
many O
different O
questions O
. O

Treatment O
with O
polyinosinic O
- O
polycytidylic O
acid O
to O
induce O
expression O
of O
Cre B
resulted O
in O
complete O
disruption O
of O
the O
Arnt B
gene I
and O
loss O
of O
ARNT B
messenger I
RNA I
( I
mRNA I
) I
expression O
in O
liver O
. O

Physical O
map O
of O
the O
genome O
of O
sonchus O
yellow O
net O
virus O
, O
a O
plant O
rhabdovirus O
with O
six O
genes O
and O
conserved O
gene O
junction O
sequences O
. O

Tubular O
dysfunction O
in O
renal O
lithiasis O
: O
cause O
or O
consequence O
? O
] O
To O
investigate O
whether O
overall O
tubular O
dysfunction O
is O
encountered O
in O
a O
particular O
subgroup O
of O
patients O
with O
urolithiasis O
, O
the O
following O
parameters O
of O
renal O
tubular O
function O
have O
been O
measured O
in O
fasting O
morning O
urine O
in O
124 O
male O
stone O
formers O
: O
excretion O
of O
lysozyme B
and O
gamma B
- I
glutamyl I
transpeptidase I
( O
gamma B
- I
GT I
) O
, O
fractional O
excretion O
( O
FE O
) O
or O
glucose O
, O
insulin B
, O
bicarbonate O
after O
an O
alkali O
load O
, O
and O
theoretical O
phosphate O
threshold O
( O
TmP O
/ O

GFR O
) O
. O

Interaction O
between O
the O
tobacco O
DNA O
- O
binding O
activity O
CBF B
and O
the O
cyt B
- I
1 I
promoter I
element I
of O
the O
Agrobacterium B
tumefaciens I
T I
- I
DNA I
gene I
T I
- I
CYT I
correlates O
with O
cyt B
- I
1 I
directed O
gene O
expression O
in O
multiple O
tobacco O
tissue O
types O
. O

As O
expected O
, O
the O
QOLS O
- O
N O
had O
a O
lower O
correlation O
with O
physical O
health O
( O
r O
= O
0 O
. O
24 O
, O
p O
< O
0 O
. O
000 O
) O
and O
self O
- O
reported O
symptoms O
( O
r O
= O
- O
0 O
. O
20 O
, O
p O
< O
0 O
. O
001 O
) O
, O
and O
a O
higher O
correlation O
with O
mental O
health O
( O
r O
= O
0 O
. O
52 O
, O
p O
< O
0 O
. O
000 O
) O
. O

The O
N1 O
- O
P2 O
amplitudes O
of O
the O
AEP O
decreased O
with O
increasing O
strength O
of O
muscular O
innervation O
. O

SN O
- O
38 O
rebound O
concentrations O
were O
observed O
in O
many O
courses O
at O
about O
0 O
. O
5 O
to O
1 O
hour O
following O
the O
end O
of O
the O
i O
. O
v O
. O
infusion O
, O
which O
is O
suggestive O
of O
enterohepatic O
recycling O
. O

Sequence O
analysis O
of O
AEBP2 B
cDNA I
revealed O
that O
it O
encodes O
a O
protein O
containing O
three O
Gli B
- O
Kruppel O
( O
Cys2 O
- O
His2 O
) O
- O
type O
zinc O
fingers O
. O

In O
the O
case O
of O
the O
pT181 O
plasmid O
, O
inactivation O
of O
the O
initiator O
RepC I
protein I
occurs O
by O
the O
attachment O
of O
an O
oligonucleotide O
to O
its O
active O
tyrosine O
residue O
. O

Xanthoepocin O
, O
a O
new O
antibiotic O
from O
Penicillium O
simplicissimum O
IFO5762 O
. O

Extracellular B
regulated I
kinases I
( I
ERK I
) I
1 I
and O
ERK2 B
are O
authentic O
substrates O
for O
the O
dual O
- O
specificity O
protein B
- I
tyrosine I
phosphatase I
VHR B
. O

Leukocytoclasic O
vasculitis O
during O
treatment O
with O
low O
- O
dose O
captopril O

H O
. O

Truncations O
from O
Hip B
' I
s I
N I
terminus I
resulted O
in O
an O
apparent O
loss O
of O
Hip B
homo O
- O
oligomerization O
, O
but O
these O
mutants O
retained O
association O
with O
hsp70 B
and O
were O
recovered O
in O
receptor O
complexes O
. O

The O
biological O
basis O
for O
the O
changes O
in O
CNAPs O
produced O
by O
CS2 O
is O
under O
investigation O
. O

A O
very O
large O
PC O
LAN O
as O
the O
basis O
for O
a O
hospital O
information O
system O
. O

This O
was O
tragically O
highlighted O
by O
the O
death O
of O
a O
17 O
year O
old O
dental O
nurse O
after O
taking O
a O
single O
1 O
g O
tablet O
of O
sodium O
nitrite O
. O

The O
human O
protein O
is O
encoded O
by O
a O
900 O
- O
base O
pair O
- O
long O
mRNA O
that O
is O
expressed O
in O
the O
glandular O
epithelial O
cells O
of O
the O
endometrium O
in O
a O
cyclic O
manner O
; O
in O
addition O
, O
it O
is O
found O
in O
the O
mucosal O
epithelial O
cells O
of O
the O
fallopian O
tubes O
. O

In O
this O
report O
, O
we O
describe O
a O
patient O
with O
renal O
cell O
carcinoma O
who O
experienced O
ventricular O
tachycardia O
while O
undergoing O
treatment O
with O
high O
- O
dose O
bolus O
IL B
- I
2 I
. O

Four O
antifungal O
agents O
were O
compared O
: O
amphotericin O
B O
, O
flucytosine O
, O
miconazole O
, O
and O
ketoconazole O
. O

The O
regulatory O
genes O
cnrYXH B
have O
been O
cloned O
, O
overexpressed O
, O
and O
purified O
in O
Escherichia O
coli O
. O

Examination O
of O
the O
Coomassie O
- O
stained O
gels O
revealed O
that O
the O
capsid O
- O
like O
particles O
composed O
of O
the O
N11 B
- O
VP1 B
protein O
did O
not O
contain O
any O
host B
- I
derived I
histones I
. O

Conversion O
of O
an O
NF B
- I
kappaB I
into O
a O
GABP B
binding I
site I
is O
likely O
to O
have O
occurred O
also O
during O
the O
worldwide O
spread O
of O
HIV O
- O
1 O
, O
as O
we O
noticed O
the O
same O
LTR O
modification O
in O
subtype O
E O
isolates O
from O
Thailand O
. O

Protein O
binding O
to O
UAS1MLS1 B
was O
observed O
with O
extracts O
from O
derepressed O
but O
not O
from O
repressed O
cells O
, O
and O
could O
be O
competed O
for O
by O
an O
excess O
of O
the O
unlabelled O
CSRE B
( O
ICL1 B
) O
sequence O
. O

We O
then O
isolated O
the O
full O
- O
length O
coding O
region O
of O
the O
human B
BACH1 I
gene I
using O
expressed O
sequence O
tags O
, O
reverse O
transcription O
- O
polymerase O
chain O
reaction O
and O
rapid O
amplification O
of O
cDNA O
ends O
. O

Authors O
present O
the O
anatomical O
variations O
of O
the O
course O
of O
the O
inferior O
mesenteric O
artery O
and O
its O
branches O
in O
62 O
human O
fetus O
. O

The O
7 B
. I
2 I
kb I
EcoRI I
fragment I
of O
AfMNPV O
was O
cloned O
and O
the O
nucleotide O
sequences O
of O
the O
polyhedrin B
coding I
region I
and O
its O
flanking O
regions O
were O
determined O
. O

1 O
. O

The O
increments O
in O
median O
plasma B
secretin I
concentration O
were O
1 O
. O
6 O
, O
3 O
. O
0 O
, O
and O
6 O
. O
4 O
pmol O
x O
1 O
( O
- O
1 O
) O
after O
secretin B
, O
125 O
, O
250 O
and O
500 O
fmol O
x O
kg O
- O
1 O
, O
and O
the O
corresponding O
15 O
- O
min O
bicarbonate O
output O
283 O
, O
442 O
, O
and O
1435 O
micromol O
, O
respectively O
. O

The O
resulting O
threshold O
elevation O
curves O
fell O
into O
a O
small O
number O
of O
distinct O
groups O
, O
suggesting O
the O
existence O
of O
discrete O
spatial O
frequency O
mechanisms O
in O
human O
central O
vision O
. O

Because O
c B
- I
myb I
encodes O
a O
transcriptional O
activator O
that O
functions O
via O
DNA O
binding O
, O
it O
is O
likely O
that O
c B
- I
myb I
exerts O
its O
biological O
activity O
by O
regulating O
the O
transcription O
of O
genes O
required O
for O
DNA O
synthesis O
and O
cell O
cycle O
progression O
. O

The O
rest O
of O
the O
3 O
' O
untranslated O
sequences O
were O
common O
to O
both O
cDNA O
clones O
. O

Coexpression O
of O
c B
- I
Rel I
in O
combination O
with O
ATF B
- I
1 I
, O
CREB2 B
, O
or O
ATF B
- I
1 I
/ O
CREB2 B
leads O
to O
synergistic O
transactivation O
of O
a O
CD28RE B
- O
TRE O
reporter O
plasmid O
in O
quiescent O
Jurkat O
T O
- O
cells O
. O

Conventional O
toxicological O
considerations O
suggest O
that O
the O
adverse O
health O
effects O
of O
any O
necessary O
increase O
in O
coal O
combustion O
effluents O
would O
be O
greatest O
per O
unit O
of O
coal O
in O
those O
areas O
which O
are O
most O
heavily O
populated O
and O
have O
the O
highest O
preexisting O
levels O
of O
the O
gas O
- O
aerosol O
complex O
. O

The O
genes O
on O
each O
chromosome O
involved O
in O
this O
translocation O
have O
been O
identified O
as O
the O
transcription O
factor O
- O
encoding O
genes O
PAX3 B
and O
FKHR B
. O

A O
search O
was O
conducted O
for O
suppressors O
of O
the O
inositol O
auxotrophic O
phenotype O
of O
the O
ino4 B
- I
8 I
mutant I
of O
yeast O
. O

Anesthesia O
was O
induced O
by O
droperidol O
0 O
. O
2 O
mg O
. O
kg O
- O
1 O
, O
fentanyl O
50 O
micrograms O
, O
thiamylal O
150 O
- O
250 O
mg O
, O
and O
succinylcholine O
1 O
mg O
. O
kg O
- O
1 O
, O
and O
maintained O
with O
nitrous O
oxide O
and O
O2 O
( O
67 O
: O
33 O
) O
supplemented O
with O
repeated O
i O
. O
v O
. O
doses O
of O
fentanyl O
( O
within O
15 O
micrograms O
. O
kg O
- O
1 O
) O
. O

Complexities O
in O
the O
host O
- O
pathogen O
interactions O
during O
actual O
infection O
with O
Gram O
- O
negative O
bacteria O
may O
account O
for O
the O
difficulties O
in O
demonstrating O
this O
phenomena O
in O
vivo O
. O

This O
novel O
TRPC7 B
gene I
could O
be O
a O
candidate O
gene O
for O
genetic O
disorders O
such O
as O
bipolar O
affective O
disorder O
, O
nonsyndromic O
hereditary O
deafness O
, O
Knobloch O
syndrome O
, O
and O
holoprosencephaly O
, O
which O
were O
mapped O
to O
this O
region O
. O

Paradoxically O
, O
loop O
3i O
from O
the O
M1Ach B
- I
muscarinic I
receptor I
also O
maximally O
inhibited O
GnRH B
agonist O
- O
stimulated O
cAMP O
accumulation O
and O
PRL B
release O
by O
40 O
% O
( O
both O
effects O
mediated O
through O
activation O
of O
the O
Gs B
protein I
) O
. O

We O
present O
the O
case O
of O
a O
patient O
who O
presented O
an O
unusually O
large O
mucoid O
cyst O
measuring O
38 O
x O
25 O
mm O
in O
an O
uncommon O
localization O
, O
the O
right O
maxillary O
. O

Acoustic O
- O
property O
measurements O
of O
the O
oxide O
superconductor O
BaPb1 O
- O
xBixO3 O
by O
the O
sphere O
- O
resonance O
method O
. O

In O
7 O
men O
and O
1 O
woman O
who O
died O
suddenly O
the O
functionally O
important O
areas O
of O
myocardium O
in O
the O
sino O
- O
auricular O
area O
and O
the O
subendocardial O
layers O
of O
left O
ventricle O
were O
obtained O
by O
necropsy O
no O
more O
than O
3 O
h O
after O
death O
, O
and O
then O
prepared O
for O
study O
by O
electron O
microscopy O
. O

IGF B
- I
I I
levels O
were O
measured O
in O
the O
CSF O
of O
11 O
children O
with O
autism O
( O
4 O
females O
, O
7 O
males O
; O
mean O
age O
3 O
. O
8 O
years O
, O
SD O
1 O
. O
1 O
) O
using O
a O
sensitive O
radioimmunoassay O
method O
and O
compared O
with O
levels O
in O
11 O
control O
participants O
( O
6 O
females O
, O
5 O
males O
; O
mean O
age O
3 O
. O
8 O
years O
) O
. O

In O
a O
subsequent O
phase O
- O
II O
clinical O
trial O
safety O
and O
efficacy O
of O
transrectal O
HIFU O
as O
a O
novel O
minimally O
invasive O
treatment O
modality O
for O
patients O
with O
symptomatic O
benign O
prostatic O
hyperplasia O
( O
BPH O
; O
n O
= O
102 O
) O
was O
determined O
. O

Three O
high O
- O
amylose O
rice O
varieties O
, O
IR42 O
, O
IR36 O
, O
and O
IR62 O
, O
with O
similar O
chemical O
composition O
including O
amylose O
content O
( O
26 O
. O
7 O
- O
27 O
. O
0 O
% O
) O
, O
were O
cooked O
under O
the O
same O
conditions O
and O
tested O
for O
in O
vitro O
digestibility O
as O
well O
as O
blood O
glucose O
and O
insulin B
responses O
in O
healthy O
human O
volunteers O
. O

Slowly O
adapting O
type O
I O
mechanoreceptor O
discharge O
as O
a O
function O
of O
dynamic O
force O
versus O
dynamic O
displacement O
of O
glabrous O
skin O
of O
raccoon O
and O
squirrel O
monkey O
hand O
. O

Using O
beta B
1 I
- I
AR I
- O
luciferase B
reporter O
recombinants O
, O
we O
have O
previously O
determined O
that O
important O
beta B
1 I
- I
AR I
genetic O
elements O
controlling O
expression O
within O
the O
C6 O
glioma O
cell O
line O
are O
contained O
within O
the O
region O
- O
396 O
to O
- O
299 O
, O
relative O
to O
the O
translational O
start O
site O
. O

FACS O
analysis O
demonstrated O
that O
MCs O
had O
reached O
middle O
to O
late O
G1 O
phase O
of O
cell O
cycle O
progression O
at O
this O
timepoint O
. O

Our O
case O
, O
however O
, O
had O
no O
histologic O
evidence O
of O
malignancy O
, O
no O
serum O
alpha B
fetoprotein I
at O
7 O
months O
of O
age O
, O
and O
no O
recurrence O
at O
1 O
years O
. O

Analysis O
of O
tobacco O
mRNA O
using O
the O
ACBF B
cDNA I
as O
probe O
showed O
that O
while O
ACBF B
mRNA I
was O
present O
in O
all O
tissues O
examined O
, O
the O
highest O
transcript O
accumulation O
occurred O
in O
stem O
tissues O
. O

Cotransfection O
experiments O
revealed O
that O
both O
hGATA B
- I
4 I
and O
PMA O
/ O
A23187 O
stimulation O
are O
necessary O
for O
the O
IL B
- I
5 I
promoter I
activation O
. O

Here O
we O
show O
that O
expression O
of O
KRAB B
domain I
suppresses O
in O
vivo O
the O
activating O
function O
of O
various O
defined O
activating O
transcription O
factors O
, O
and O
we O
demonstrate O
that O
the O
KRAB B
domain I
specifically O
silences O
the O
activity O
of O
promoters O
whose O
initiation O
is O
dependent O
on O
the O
presence O
of O
a O
TATA O
box O
. O

Adult O
- O
onset O
parkinsonism O
has O
appeared O
in O
several O
previously O
unaffected O
members O
in O
families O
with O
DRD O
suggesting O
that O
this O
may O
be O
an O
additional O
phenotypical O
expression O
of O
the O
disease O
. O

We O
tested O
the O
effects O
of O
antifungal O
drugs O
on O
adherence O
of O
Candida O
albicans O
in O
vitro O
. O

The O
signs O
of O
enterocolitis O
, O
pathological O
microbial O
flora O
in O
the O
stool O
and O
changes O
in O
the O
colon O
mucosa O
were O
absent O
. O

These O
results O
support O
an O
in O
vivo O
interaction O
between O
Nck B
and O
CKI B
- I
gamma2 I
and O
suggest O
that O
CKI B
- I
gamma2 I
could O
be O
involved O
in O
signaling O
pathways O
downstream O
of O
RTKs B
. O

Correlation O
of O
nitrogen O
, O
carbohydrate O
and O
lipid O
metabolic O
indices O
in O
rats O
cooled O
under O
conditions O
of O
an O
altered O
gaseous O
environment O

Reactivation O
of O
hepatitis O
B O
virus O
replication O
is O
a O
rare O
event O
during O
the O
natural O
history O
of O
chronic O
hepatitis O
B O
. O

A O
minimal O
region O
which O
stimulates O
origin O
function O
mapped O
to O
50 O
amino O
acids O
within O
the O
C O
- O
terminus O
of O
Abf1p B
. O

Cutaneous O
melanoma O
and O
bilateral O
retinoblastoma O
. O

Treatment O
of O
JEA2 O
cells O
with O
interleukin B
( I
IL I
) I
- I
7 I
induces O
CBLB B
phosphorylation O
as O
well O
. O

Phosphorylation O
by O
Cdc28 B
activates O
the O
Cdc20 B
- O
dependent O
activity O
of O
the O
anaphase O
- O
promoting O
complex O
. O

METHOD O
: O
After O
eligibility O
, O
patients O
were O
randomly O
allocated O
in O
one O
of O
the O
following O
groups O
: O
M O
= O
methylprednisolone O
30 O
mg O
. O
kg O
- O
1 O
over O
1 O
hour O
, O
followed O
by O
5 O
. O
4 O
mg O
. O
kg O
- O
1 O
. O
h O
- O
1 O
for O
23 O
hours O
, O
N O
= O
nimodipine O
0 O
. O
015 O
mg O
. O
kg O
- O
1 O
. O
h O
- O
1 O
over O
2 O
hours O
followed O
by O
0 O
. O
03 O
mg O
. O
kg O
- O
1 O
. O
h O
- O
1 O
for O
7 O
days O
, O
MN O
or O
P O
. O

The O
amount O
of O
overlap O
in O
VFI O
, O
EF O
, O
and O
RVF O
values O
among O
the O
clinical O
groups O
was O
considerable O
. O

After O
5 O
, O
850 O
C14 O
BP O
, O
the O
decrease O
in O
Acer O
stands O
could O
be O
attributed O
to O
fire O
as O
suggested O
by O
the O
strong O
increase O
in O
Betula O
and O
by O
the O
delayed O
expansion O
of O
Pinus O
cembra O
. O

Assays O
of O
APRE B
- O
luciferase B
reporter O
plasmids O
transfected O
into O
HepG2 O
cells O
show O
that O
a O
mutated O
APRE B
that O
binds O
only O
BPi B
functions O
as O
an O
IL B
- I
1 I
alpha I
inducible I
enhancer I
, O
whereas O
a O
mutated O
APRE B
that O
binds O
only O
BPc B
does O
not O
. O

RNA O
unwinding O
in O
U4 B
/ O
U6 B
snRNPs O
requires O
ATP O
hydrolysis O
and O
the O
DEIH B
- I
box I
splicing I
factor I
Brr2 I
. O

Several O
sequences O
resembling O
binding O
sites O
for O
liver O
- O
and O
fat O
body O
- O
specific O
transcription O
factors O
were O
identified O
within O
1 O
kb O
upstream O
and O
downstream O
of O
the O
gene O
. O

Northern O
blot O
analysis O
linked O
Plk B
expression O
to O
the O
proliferative O
activity O
of O
cells O
and O
tissues O
. O

It O
also O
showed O
which O
amino O
acid O
residues O
would O
contact O
an O
extended O
TATA O
box O
with O
a O
B O
recognition O
element O
, O
and O
evolutionary O
conservation O
of O
the O
TBP B
- O
TFB B
- O
DNA O
complex O
orientation O
between O
two O
archaeal O
organisms O
with O
widely O
different O
optimal O
temperature O
for O
growth O
( O
37 O
and O
100 O
degrees O
C O
) O
. O

Studies O
with O
and O
without O
attenuating O
media O
were O
performed O
, O
and O
myocardium O
- O
to O
- O
defect O
count O
ratios O
and O
defect O
sizes O
from O
dual O
- O
isotope O
SPECT O
images O
were O
compared O
to O
myocardium O
- O
to O
- O
defect O
count O
ratios O
and O
defect O
sizes O
from O
single O
- O
isotope O
( O
201Tl O
and O
99mTc O
) O
SPECT O
images O
. O

Comparison O
of O
this O
gene O
with O
the O
available O
databases O
reveals O
very O
significant O
homology O
to O
the O
ETS B
factor I
PE B
- I
1 I
and O
probable O
near O
- O
identity O
with O
the O
recently O
cloned O
factor O
ERF I
. O

Patients O
with O
Eales O
' O
disease O
, O
chorioretinitis O
, O
central O
serous O
retinopathy O
, O
or O
malignant O
choroidal O
melanoma O
were O
tested O
for O
HLA B
antigen I
deviation O
. O

Copyright O
1999 O
Academic O
Press O
. O

The O
1 O
. O
5 O
- O
Mb O
region O
was O
covered O
by O
137 O
cosmids O
with O
a O
minimum O
overlap O
set O
of O
52 O
cosmids O
assigned O
to O
17 O
bins O
and O
9 O
contigs O
. O

Similarly O
, O
beta B
adrenoceptors I
blockade O
has O
been O
shown O
to O
be O
of O
value O
in O
achieving O
continence O
in O
a O
small O
group O
of O
patients O
. O

Estrogen O
has O
positive O
effects O
on O
mood O
, O
sexual O
function O
, O
target O
end O
organs O
and O
cognitive O
function O
, O
and O
may O
play O
an O
important O
role O
in O
the O
etiology O
of O
Alzheimer O
' O
s O
Disease O
by O
acting O
to O
prevent O
amyloid O
plaque O
formation O
, O
oxidative O
stress O
, O
or O
deterioration O
of O
the O
cholinergic O
neurotransmitter O
system O
. O

These O
data O
also O
demonstrate O
that O
insulin B
and O
TPA O
activate O
MBP B
/ O
MAP2 B
kinase O
activity O
by O
de O
novo O
phosphorylation O
of O
threonine O
and O
tyrosine O
residues O
via O
a O
very O
similar O
pathway O
. O

Effect O
of O
capsaicin O
on O
hexobarbital O
- O
anesthetized O
rats O

Triethylene O
glycol O
. O

Such O
tetranucleotides O
are O
common O
in O
promoter O
regions O
, O
particularly O
in O
activating B
transcription I
factor I
/ O
cyclic B
AMP I
response I
element I
- I
binding I
protein I
( O
ATF B
/ O
CREB B
) O
and O
E O
- O
box O
motifs O
, O
suggesting O
that O
PIF B
may O
modulate O
the O
transcription O
of O
many O
genes O
. O

Red O
blood O
cell O
binding O
of O
99mTc O
- O
EC O
and O
131I O
- O
OIH O
was O
6 O
. O
1 O
% O
and O
20 O
% O
, O
respectively O
. O

The O
dominant O
inhibitory O
biological O
activity O
of O
the O
N O
terminus O
correlated O
with O
its O
ability O
to O
impair O
EGF B
- O
dependent O
activation O
of O
GTP B
- I
Ras I
and O
of O
MAP B
kinase I
, O
as O
well O
with O
the O
ability O
of O
endogenous B
Sos I
to O
form O
a O
stable O
complex O
with O
activated O
EGF B
receptors I
. O

Brainstem O
auditory O
evoked O
potential O
responses O
( O
BAEPs O
) O
were O
recorded O
from O
CZ O
- O
A1 O
and O
CZ O
- O
A2 O
scalp O
regions O
in O
23 O
hypertensive O
and O
14 O
normotensive O
subjects O
. O

SM B
binds O
RNA O
in O
vitro O
, O
suggesting O
that O
sequence O
- O
or O
structure O
- O
specific O
mRNA O
interactions O
might O
mediate O
SM B
specificity O
. O

The O
characterization O
of O
fly O
receptors O
with O
features O
similar O
to O
mammalian B
glycoprotein I
hormone I
receptors I
allows O
a O
better O
understanding O
of O
the O
evolution O
of O
this O
unique O
group O
of O
GPCRs B
and O
future O
elucidation O
of O
their O
ligand O
signaling O
mechanisms O
. O

Here O
we O
show O
that O
these O
sequences O
are O
essential O
for O
processing O
of O
U18 B
and O
snR38 B
snoRNAs I
and O
that O
they O
compensate O
for O
the O
lack O
of O
a O
canonical O
terminal O
stem O
. O

The O
use O
of O
aluminum O
hydroxide O
was O
associated O
with O
an O
increase O
in O
fecal O
fluoride O
excretion O
and O
a O
decrease O
in O
net O
absorption O
of O
fluoride O
regardless O
of O
the O
intake O
fluoride O
, O
calcium O
, O
phosphorus O
, O
or O
magnesium O
. O

In O
humans O
, O
there O
was O
a O
statistically O
significant O
decrease O
of O
TEWL O
on O
both O
forearm O
and O
thigh O
in O
CS O
- O
treated O
patients O
compared O
to O
non O
- O
CS O
- O
treated O
patients O
and O
controls O
( O
p O
less O
than O
0 O
. O
05 O
) O
. O

Kramer O
Hamburg O
1988 O
, O
ISBN O
: O
3 O
926952 O
07 O
5 O
) O
, O
the O
syndromatic O
day O
- O
to O
- O
day O
shifting O
of O
the O
psychopathological O
features O
with O
manic O
, O
depressive O
and O
several O
mixed O
states O
has O
a O
well O
- O
defined O
time O
structure O
, O
as O
in O
25 O
patients O
studied O
and O
reported O
on O
in O
this O
paper O
. O

Ras B
involvement O
in O
signal O
transduction O
by O
the O
serotonin B
5 I
- I
HT2B I
receptor I
. O

Analysis O
of O
exon O
2 O
of O
the O
factor B
IX I
gene I
revealed O
a O
C O
- O
- O
> O
T O
mutation O
in O
codon O
10 O
of O
the O
propeptide O
region O
, O
resulting O
in O
the O
substitution O
of O
alanine O
by O
valine O
. O

There O
was O
a O
direct O
proportional O
relationship O
between O
the O
spiral O
artery O
flow O
and O
the O
pressure O
difference O
between O
the O
spiral O
artery O
and O
the O
amniotic O
fluid O
; O
the O
pressure O
difference O
per O
flow O
( O
the O
resistance O
of O
the O
cotyledonary O
unit O
) O
was O
found O
to O
be O
1 O
- O
- O
3 O
mm O
Hg O
. O

ml O
- O
1 O
. O
min O
( O
8 O
- O
- O
24kPa O
. O
ml O
- O
1 O
. O
s O
) O
. O

Three O
putative O
ORFs O
have O
significant O
homology O
with O
known O
proteins O
: O
L0968 B
is O
a O
new O
member O
of O
the O
very O
large O
' B
seripauperins I
' I
family I
, O
comprising O
at O
least O
20 O
yeast O
members O
; O
L1313 B
is O
a O
new O
ABC B
transporter I
highly O
homologous O
to O
the O
yeast B
cadmium I
resistance I
protein I
Ycf1p I
and O
to O
the O
human B
multidrug I
resistance I
protein I
hMRP1 I
; O
the O
C O
- O
terminal O
part O
of O
L1325 B
present O
in O
our O
sequence O
is O
very O
homologous O
to O
the O
fruit B
fly I
abdominal I
segment I
formation I
protein I
Pumilio I
. O

The O
cells O
were O
densely O
ciliated O
, O
each O
cilium O
showing O
a O
typical O
9 O
+ O
2 O
fibrilar O
pattern O
. O

Immunofluorescence O
obtained O
using O
antibodies O
against O
alphaI B
SigmaI I
/ I
+ I
+ I
betaI I
SigmaI I
spectrin I
and O
Abl B
tyrosine I
kinase I
but O
not O
against O
alphaII B
/ I
betaII I
spectrin I
colocalized O
with O
the O
overexpressed O
green B
fluorescent I
protein I
- O
SH3 B
- I
binding I
protein I
. O

Extracellular B
protein I
kinase I
A I
as O
a O
cancer O
biomarker O
: O
its O
expression O
by O
tumor O
cells O
and O
reversal O
by O
a O
myristate O
- O
lacking O
Calpha B
and O
RIIbeta B
subunit I
overexpression O
. O

Detection O
of O
sinusoidal O
gratings O
by O
pattern O
- O
specific O
detectors O
: O
further O
evidence O
for O
the O
correlation O
principle O
in O
human O
vision O
. O

This O
B O
- O
box O
is O
not O
present O
in O
ETS1 B
, O
ETS2 B
, O
PEA3 B
or O
PU B
. I
1 I
and O
these O
proteins O
were O
unable O
to O
form O
ternary O
complexes O
with O
SRF B
and O
Egrl B
- O
SREs O
or O
c B
- I
fos I
SRE O
. O

MADS B
- I
box I
genes I
in O
plants O
are O
a O
diverse O
class O
of O
transcription O
factors O
that O
are O
involved O
in O
regulating O
developmental O
processes O
, O
particularly O
meristem O
and O
organ O
identity O
during O
floral O
development O
. O

Elevated O
systolic O
blood O
pressure O
and O
high O
postglucose O
serum B
insulin I
levels O
showed O
an O
independent O
, O
significant O
association O
with O
left O
ventricular O
mass O
in O
female O
diabetic O
subjects O
. O

This O
study O
investigated O
if O
signal O
- O
averaged O
low O
amplitude O
atrial O
potentials O
predict O
atrial O
fibrillation O
or O
flutter O
( O
AFF O
) O
. O

Thirty O
- O
five O
pts O
were O
transplanted O
for O
constitutional O
disease O
: O
Fanconi O
anemia O
( O
FA O
) O
( O
26 O
pts O
) O
, O
Dyskeratosis O
congenita O
( O
2 O
pts O
) O
, O
Blackfan O
- O
Diamond O
erythroblastopenia O
( O
2 O
pts O
) O
, O
Glanzmann O
thrombasthenia O
( O
1 O
pt O
) O
, O
osteopetrosis O
( O
1 O
pt O
) O
and O
Gaucher O
' O
s O
disease O
( O
1 O
pt O
) O
. O

Viral B
protein I
U I
( O
Vpu B
) O
is O
a O
protein O
encoded O
by O
human O
immunodeficiency O
virus O
type O
1 O
( O
HIV O
- O
1 O
) O
that O
promotes O
the O
degradation O
of O
the O
virus O
receptor O
, O
CD4 B
, O
and O
enhances O
the O
release O
of O
virus O
particles O
from O
cells O
. O

Because O
the O
variation O
in O
the O
carbohydrate O
content O
of O
human O
milk O
is O
very O
small O
, O
a O
more O
simple O
alternative O
approach O
would O
be O
to O
include O
only O
an O
average O
carbohydrate O
value O
for O
an O
estimate O
of O
energy O
content O
. O

Level O
II O
mosaicism O
occurred O
in O
0 O
. O
9 O
per O
cent O
of O
CVS O
mesenchyme O
and O
1 O
. O
5 O
per O
cent O
of O
amniotic O
fluid O
cultures O
and O
in O
general O
was O
not O
perceived O
to O
be O
of O
sufficient O
concern O
to O
warrant O
cytogenetic O
follow O
- O
up O
studies O
. O

Biol O
. O

IV O
. O

Sleeping O
position O
and O
sudden O
infant O
death O
syndrome O
( O
SIDS O
) O
: O
effect O
of O
an O
intervention O
programme O
to O
avoid O
prone O
sleeping O
. O

CSEn B
stimulated O
transcription O
of O
a O
variety O
of O
promoters O
, O
including O
the O
hCS B
, O
human B
growth I
hormone I
, O
thymidine B
kinase I
, O
and O
Rous B
sarcoma I
virus I
promoters I
, O
in O
human O
choriocarcinoma O
cell O
lines O
( O
BeWo O
and O
JEG O
- O
3 O
) O
but O
not O
HeLa O
cells O
or O
rat B
somatolactotrophes I
( O
GC B
) O
. O

Pretreatment O
of O
human O
skin O
with O
tRA O
inhibited O
UV O
induction O
of O
c B
- I
Jun I
protein I
and O
, O
consequently O
, O
AP B
- I
1 I
. O

c B
- I
Jun I
protein I
inhibition O
occurred O
via O
a O
posttranscriptional O
mechanism O
, O
since O
tRA B
did O
not O
inhibit O
UV O
induction O
of O
c B
- I
Jun I
mRNA I
. O

Thus O
, O
apoptosis O
in O
hematopoietic O
cells O
is O
the O
end O
result O
of O
a O
conflict O
between O
death O
and O
survival O
signals O
, O
rather O
than O
a O
simple O
death O
by O
default O
. O

Effect O
of O
age O
on O
glucose O
, O
reducing O
sugars O
and O
plasma B
insulin I
in O
blood O
of O
milk O
- O
fed O
calves O
. O

CONCLUSIONS O
: O
Results O
of O
this O
study O
suggest O
that O
both O
male O
and O
female O
soldiers O
who O
are O
diagnosed O
with O
chlamydia O
infections O
have O
relatively O
high O
risks O
of O
reinfection O
through O
their O
20s O
. O

During O
seed O
maturation O
, O
the O
transcriptional O
activity O
of O
napin B
genes I
is O
regulated O
by O
developmental O
signals O
involving O
the O
transcriptional O
activator O
ABI3 B
and O
abscisic O
acid O
( O
ABA O
) O
. O

DESIGN O
: O
Intradermally O
injected O
human O
squamous O
cell O
carcinoma O
cells O
were O
grown O
to O
40 O
to O
80 O
mm3 O
in O
athymic O
nude O
mice O
and O
irradiated O
with O
675 O
- O
nm O
light O
( O
75 O
J O
/ O
cm2 O
, O
75 O
mW O
/ O
cm2 O
) O
24 O
hours O
after O
the O
intraperitoneal O
injection O
of O
SiPc O
IV O
( O
1 O
. O
0 O
mg O
/ O
kg O
) O
. O

This O
results O
in O
a O
decreased O
secretion O
of O
immunoglobulin B
A I
, O
and O
, O
consequently O
, O
a O
decreased O
resistance O
against O
infections O
. O

The O
absolute O
CD4 B
count O
of O
patients O
with O
de O
novo O
IBD O
was O
210 O
- O
700 O
cells O
/ O
ml O
at O
the O
time O
of O
IBD O
. O

Categorisation O
of O
the O
different O
substructures O
within O
the O
sections O
allows O
them O
to O
be O
analysed O
and O
displayed O
separately O
. O

To O
study O
the O
mechanisms O
and O
the O
signaling O
pathways O
involved O
, O
IGF B
- I
1R I
promoter I
reporter I
constructs I
were O
transiently O
transfected O
in O
CHO O
- O
AT1 B
cells O
that O
overexpress O
angiotensin B
AT1 I
receptors I
. O

With O
this O
study O
, O
a O
total O
of O
13 O
operators O
for O
the O
glp B
regulon I
have O
been O
characterized O
. O

Natl O
. O

Coexpression O
of O
a O
dominant B
negative I
c I
- I
jun I
antagonized O
the O
ras B
- O
dependent O
stimulation O
of O
the O
92 B
- I
kDa I
gelatinase I
B I
promoter I
- O
driven O
CAT B
reporter I
. O

In O
two O
patients O
, O
a O
male O
homosexual O
and O
an O
hemophiliac O
, O
thrombocytopenia O
was O
associated O
with O
AIDS O
- O
related O
complex O
. O

However O
, O
LXRalpha B
inhibited O
binding O
of O
PPARalpha B
/ O
RXRalpha B
heterodimers O
to O
PPREs O
, O
and O
coexpression O
of O
LXRalpha B
in O
mammalian O
cells O
antagonized O
peroxisome O
proliferator O
signaling O
mediated O
by O
PPARalpha B
/ O
RXRalpha B
in O
vivo O
. O

The O
results O
can O
be O
summarized O
as O
follows O
. O

A O
case O
of O
heterotopic O
gastric O
mucosa O
( O
pyloric O
gland O
) O
in O
the O
wall O
of O
the O
gallbladder O
was O
reported O
. O

These O
findings O
suggest O
that O
the O
mechanism O
of O
ribosomal O
frameshifting O
at O
the O
PVM B
signal O
is O
different O
from O
the O
one O
described O
by O
the O
' O
simultaneous O
slippage O
' O
model O
in O
that O
only O
the O
string O
of O
four O
adenosine O
nucleotides O
represents O
the O
slippery O
sequence O
involved O
in O
a O
- O
1 O
P O
- O
site O
slippage O
. O

CTDK1 B
and O
CTDK2 B
also O
differ O
in O
their O
protein O
substrate O
specificity O
. O

This O
communication O
presents O
two O
patients O
with O
clinically O
massive O
PE O
of O
recent O
onset O
( O
confirmed O
by O
lung O
perfusion O
scans O
) O
who O
were O
successfully O
treated O
with O
a O
single O
i O
. O
v O
. O
dose O
of O
30 O
mg O
of O
anisoylated O
lys B
- I
plasminogen I
streptokinase I
activator I
complex I
( O
APSAC O
, O
comparable O
to O
1 O
, O
500 O
, O
000 O
U O
of O
streptokinase B
) O
followed O
by O
systemic O
heparinization O
for O
7 O
days O
. O

A O
Laboratory O
Manual O
, O
2nd O
ed O
. O

Engineering O
temperature O
- O
sensitive O
SH3 B
domains I
. O

A O
mean O
value O
for O
HbA1 B
greater O
than O
10 O
% O
was O
associated O
with O
an O
increased O
risk O
of O
progression O
of O
retinopathy O
and O
a O
mean O
value O
less O
than O
8 O
. O
7 O
% O
was O
associated O
with O
a O
diminished O
risk O
. O

Allergic O
field O
: O
how O
to O
detect O
it O
? O

At O
1 O
min O
postalfentanil O
, O
N2O O
caused O
significantly O
more O
rigidity O
than O
100 O
% O
O2 O
( O
p O
< O
0 O
. O
001 O
) O
. O

The O
preparative O
regimen O
consisted O
of O
fractionated O
total O
body O
irradiation O
( O
TBI O
) O
and O
cyclophosphamide O
( O
CY O
) O
. O

NGF B
also O
induced O
a O
synthetic O
promoter O
with O
repeated O
Sp1 B
sites I
linked O
to O
a O
core O
promoter O
, O
and O
a O
plasmid O
regulated O
by O
a O
chimeric O
transactivator O
in O
which O
the O
Gal4 B
DNA I
binding I
domain I
is O
fused O
to O
the O
Sp1 B
transactivation I
domain I
, O
indicating O
that O
this O
transactivation O
domain O
is O
regulated O
by O
NGF B
. O

The O
toxicity O
of O
copper O
to O
the O
collembolan O
Folsomia O
fimetaria O
L O
. O
was O
studied O
in O
soil O
incubated O
with O
copper O
sulfate O
for O
different O
periods O
before O
the O
introduction O
of O
collembolans O
, O
to O
assess O
the O
effect O
of O
aging O
of O
contamination O
on O
the O
toxicity O
of O
copper O
. O

The O
frequency O
of O
severe O
congestive O
heart O
failure O
( O
Killip O
class O
III O
or O
IV O
) O
at O
admission O
was O
significantly O
higher O
in O
patients O
with O
early O
presentation O
, O
than O
with O
late O
presentation O
( O
30 O
. O
7 O
% O
vs O
7 O
. O
6 O
% O
, O
p O
less O
than O
0 O
. O
05 O
) O
. O

Comparative O
ultrafiltration O
( O
UF O
) O
studies O
with O
BiGG O
and O
standard O
lactate O
( O
La O
) O
solutions O
in O
rabbits O
showed O
that O
net O
UF O
with O
La O
solution O
peaked O
at O
2 O
h O
and O
decreased O
significantly O
at O
4 O
h O
and O
6 O
h O
. O

The O
Giessener O
Tumordokumentationssystem O
( O
GTDS O
) O
is O
such O
a O
disease O
specific O
system O
. O

Measurement O
of O
protein O
in O
natural O
rubber O
latex O
. O

In O
cotransfection O
assays O
, O
a O
small O
fragment O
of O
Sin O
retaining O
the O
Src B
- O
SH3 B
- O
binding O
site O
and O
one O
tyrosine O
- O
containing O
motif O
induced O
c B
- I
Src I
activation O
as O
measured O
by O
a O
transcriptional O
reporter O
. O

The O
related O
tumor O
suppressor O
, O
p130 B
, O
also O
effects O
this O
function O
. O

p107 B
levels O
increase O
substantially O
as O
cells O
progress O
through O
S O
phase O
. O

p107 B
induction O
of O
E2F B
DNA O
binding O
was O
observed O
primarily O
in O
S O
phase O
cells O
coincident O
with O
the O
increase O
in O
p107 B
protein I
levels O
. O

Both O
flavoproteins O
are O
active O
as O
AhpC B
reductases I
and O
mediate O
electron O
transfer O
, O
resulting O
in O
the O
NADH O
- O
dependent O
reduction O
of O
hydrogen O
peroxide O
and O
cumene O
hydroperoxide O
. O

Transcriptional O
regulation O
of O
the O
murine B
k I
- I
fgf I
gene I
. O

Further O
, O
cells O
cotransfected O
with O
a O
UF B
promoter I
- O
luciferase B
( O
- O
1935UF O
- O
Luc B
) O
reporter O
gene O
and O
the O
BTEB B
expression O
vector O
had O
2 O
- O
fold O
higher O
Luc B
activity O
than O
those O
cotransfected O
with O
reporter O
gene O
and O
pcDNA O
- O
3 O
. O

Evasion O
of O
host O
immunity O
by O
Toxocara O
canis O
infective O
larvae O
is O
mediated O
by O
the O
nematode O
surface O
coat O
, O
which O
is O
shed O
in O
response O
to O
binding O
by O
host O
antibody O
molecules O
or O
effector O
cells O
. O

A O
Frank O
vectorcardiogram O
was O
recorded O
before O
and O
every O
15 O
- O
30 O
minutes O
for O
10 O
hours O
after O
the O
occlusion O
. O

In O
agreement O
with O
this O
, O
archaea O
appear O
to O
lack O
eIF5 B
, O
eIF2B B
and O
the O
lysine O
- O
rich O
binding O
domain O
for O
these O
factors O
in O
their O
eIF2beta B
homolog I
. O

Derepression O
of O
RAR B
signaling O
by O
expressing O
a O
dominant O
- O
negative O
corepressor O
resulted O
in O
embryos O
that O
exhibited O
phenotypes O
similar O
to O
those O
treated O
by O
RA O
. O

Chemiluminescence O
induced O
by O
opsonized O
zymosan O
increased O
significantly O
( O
P O
less O
than O
0 O
. O
05 O
) O
after O
treatment O
with O
bacterial O
extracts O
, O
whereas O
no O
significant O
changes O
were O
observed O
in O
the O
fMLP O
- O
stimulated O
PMNs O
. O

Lastly O
, O
several O
restriction O
length O
polymorphisms O
were O
identified O
and O
mapped O
within O
a O
1 O
kb O
region O
located O
immediately O
upstream O
from O
the O
JH B
cluster I
. O

Radiographic O
spatial O
frequencies O
essential O
to O
the O
diagnosis O
of O
incipient O
interproximal O
lesions O
. O

Promoter O
region O
of O
the O
human B
alpha I
2A I
adrenergic I
receptor I
gene I
. O

( O
1982 O
) O
. O

( O
99m O
) O
Tc O
Ethyl O
Cysteinate O
Dimer O
stable O
during O
6h O
has O
contributed O
to O
develop O
ictal O
studies O
to O
evaluate O
the O
location O
of O
partial O
seizure O
. O

LiF O
thermoluminescence O
dosimeters O
( O
TLD O
) O
in O
chip O
form O
were O
placed O
onto O
1 O
eyelid O
, O
the O
skin O
over O
the O
thyroid O
, O
and O
the O
patient O
' O
s O
clothes O
covering O
the O
region O
of O
breasts O
and O
ovaries O
of O
female O
patients O
and O
the O
testicles O
of O
male O
patients O
. O

Thus O
, O
the O
in O
vivo O
function O
of O
Gle2p B
is O
strictly O
coupled O
to O
the O
short B
GLEBS I
within O
Nup116p B
which O
links O
this O
putative O
mRNA O
transport O
factor O
to O
the O
nuclear O
pores O
. O

DNMT2 B
binds O
AdoHcy O
in O
the O
same O
conformation O
as O
confirmed O
m B
( I
5 I
) I
C I
MTases I
and O
, O
while O
DNMT2 B
shares O
all O
sequence O
and O
structural O
features O
with O
m B
( I
5 I
) I
C I
MTases I
, O
it O
has O
failed O
to O
demonstrate O
detectable O
transmethylase O
activity O
. O

The O
structure O
of O
the O
murine B
Dtk I
gene I
has O
been O
determined O
. O

In O
previous O
studies O
, O
we O
showed O
that O
the O
nuclear O
import O
of O
SREBP B
- I
2 I
occurs O
via O
the O
direct O
interaction O
of O
importin B
beta I
with O
the O
HLH O
- O
Zip O
domain O
. O

In O
the O
normal O
controls O
the O
plasma O
FPA B
level O
( O
mean O
+ O
/ O
- O
SD O
) O
was O
1 O
. O
43 O
+ O
/ O
- O
0 O
. O
46 O
ng O
/ O
ml O
. O

In O
this O
study O
we O
cloned O
the O
cDNA O
of O
AD1 B
Ag I
from O
a O
rat O
basophilic O
leukemia O
2H3 O
cDNA O
library O
. O

Ten O
patients O
( O
aged O
28 O
- O
76 O
years O
) O
with O
a O
terminal O
jejunostomy O
located O
within O
the O
first O
meter O
of O
jejunum O
were O
treated O
by O
infusion O
of O
an O
elemental O
diet O
into O
the O
distal O
small O
bowel O
( O
IEDDSB O
) O
. O

The O
procedure O
was O
technically O
successful O
in O
12 O
of O
14 O
( O
86 O
% O
) O
stenoses O
in O
the O
fibromuscular O
dysplasia O
subgroup O
but O
in O
only O
one O
of O
five O
( O
20 O
% O
) O
lesions O
in O
the O
neurofibromatosis O
subgroup O
. O

Conventional O
and O
digital O
radiography O
of O
the O
heart O
, O
aorta O
, O
and O
pulmonary O
vascularity O
. O

Regulation O
of O
the O
p21 B
- I
activated I
kinase I
( O
PAK B
) O
by O
a O
human B
Gbeta I
- I
like I
WD I
- I
repeat I
protein I
, O
hPIP1 B
. O

Other O
clinicopathological O
features O
have O
no O
definite O
influence O
in O
survival O
expectancy O
. O

These O
studies O
suggest O
that O
tandem O
alpha O
- O
helices O
located O
near O
the O
C O
- O
terminus O
of O
PC B
- I
TP I
facilitate O
membrane O
binding O
and O
extraction O
of O
phosphatidylcholines O
. O

The O
striking O
similarity O
in O
preference O
for O
mismatched O
and O
weakly O
paired O
nucleotides O
for O
binding O
and O
for O
excision O
suggests O
a O
functional O
relationship O
between O
binding O
and O
cleavage O
reactions O
. O

Interestingly O
, O
these O
" O
activation O
- O
defective O
" O
mutants O
segregated O
into O
two O
classes O
: O
1 O
) O
those O
that O
were O
unable O
to O
form O
dimers O
but O
that O
could O
still O
form O
higher O
order O
oligomers O
and O
transform O
cells O
, O
and O
2 O
) O
those O
that O
were O
defective O
for O
PDGF B
- I
R I
binding O
and O
were O
transformation O
- O
incompetent O
. O

APL O
patients O
showed O
a O
low O
, O
yet O
variable O
, O
level O
of O
JEM B
- I
1 I
mRNA I
in O
bone O
marrow O
. O

CONCLUSIONS O
: O
The O
results O
show O
that O
intranasal O
challenge O
with O
FLU O
induces O
changes O
in O
leukocyte O
histamine O
release O
, O
but O
not O
other O
systemic O
immune O
and O
inflammatory O
responses O
. O

We O
have O
shown O
previously O
that O
EGF B
receptor I
activation O
stimulates O
gastrin B
gene I
expression O
through O
a O
GC O
- O
rich O
element O
called O
gERE O
. O

We O
have O
earlier O
proposed O
that O
CG O
rich O
sequences O
resembling O
CpG O
islands O
, O
which O
are O
associated O
with O
many O
imprinted O
genes O
and O
often O
subject O
to O
parental O
- O
specific O
methylation O
, O
could O
act O
as O
a O
common O
imprinting O
element O
. O

The O
secondary O
end O
points O
included O
creatine B
kinase I
peak O
, O
ventricular O
fibrillation O
/ O
tachycardia O
within O
the O
first O
24 O
hours O
, O
death O
and O
congestive O
heart O
failure O
within O
the O
30 O
- O
day O
follow O
- O
up O
, O
and O
30 O
- O
day O
left O
ventricular O
ejection O
fraction O
. O

Prevention O
of O
gravid O
Rhesus O
isoimmunization O

All O
relapses O
occurred O
within O
the O
first O
year O
and O
were O
confined O
to O
patients O
with O
PS O
II O
disease O
and O
four O
or O
more O
sites O
of O
involvement O
. O

This O
demonstrates O
the O
presence O
of O
a O
PKC B
- O
dependent O
pathway O
which O
functions O
independently O
from O
Lck B
in O
MAP B
kinase I
activation O
. O

This O
interesting O
exon O
junction O
resulted O
in O
novel O
deduced O
amino O
terminal O
open O
reading O
frames O
, O
which O
are O
completely O
in O
- O
frame O
with O
sequences O
located O
further O
downstream O
. O

Cell O
lines O
expressing O
single O
amino O
acid O
substitutions O
of O
the O
carboxyl O
- O
terminal O
asparagine O
of O
CD2 B
are O
incapable O
of O
avidity O
regulation O
by O
TCR B
signaling O
, O
PMA O
treatment O
, O
or O
elevation O
of O
intracellular O
cAMP O
levels O
, O
demonstrating O
that O
each O
of O
these O
stimuli O
utilizes O
a O
common O
structural O
element O
for O
regulating O
CD2 B
avidity O
. O

12 O
per O
cent O
was O
observed O
with O
the O
use O
of O
tetracycline O
and O
4 O
- O
epianhydrotetracycline O
in O
doses O
of O
1000 O
and O
100gamma O
per O
embryo O
respectively O
. O

Present O
investigation O
was O
undertaken O
to O
elucidate O
what O
pharmacokinetic O
parameters O
in O
animal O
experiment O
could O
be O
of O
more O
predictable O
for O
human O
clinical O
trial O
of O
beta O
- O
blocking O
agents O
. O

The O
atpA B
reading I
frame I
ends O
with O
four O
tandem O
UGA O
codons O
which O
overlap O
four O
tandem O
AUG O
codons O
initiating O
an O
unidentified O
reading O
frame O
, O
orf214 O
. O

When O
transfected O
into O
HepG2 O
cells O
, O
which O
lack O
C B
/ I
EBP I
alpha I
, O
the O
mouse B
ob I
promoter I
was O
only O
weakly O
active O
. O

Vasodilator O
and O
hypotensive O
effects O
of O
the O
optical O
isomers O
of O
nicardipine O
( O
YC O
- O
93 O
) O
, O
a O
new O
Ca2 O
+ O
- O
antagonist O
. O

Between O
1988 O
and O
1995 O
, O
341 O
children O
with O
acute O
myeloid O
leukaemia O
( O
AML O
) O
were O
treated O
on O
the O
Medical O
Research O
Council O
Acute O
Myeloid O
Leukaemia O
Trial O
( O
MRC O
AML10 O
) O
. O

A O
heterologous O
promoter O
construct O
containing O
three O
repeats O
of O
a O
consensus O
Sp1 B
site I
, O
cloned O
upstream O
of O
a O
single O
copy O
of O
the O
ZII B
( O
CREB B
/ I
AP1 I
) O
element O
from O
the O
BZLF1 B
promoter I
linked O
to O
the O
beta B
- I
globin I
TATA O
box O
, O
exhibited O
phorbol O
ester O
inducibility O
. O

Nicotinic B
receptor I
antagonists O
mecamylamine O
( O
0 O
. O
5 O
and O
1 O
mg O
/ O
kg O
) O
and O
hexamethonium O
( O
5 O
and O
10 O
mg O
/ O
kg O
) O
reduced O
the O
response O
induced O
by O
nicotine O
( O
0 O
. O
25 O
mg O
/ O
kg O
) O
. O

Appl O
. O

Thus O
, O
the O
predicted O
M3 B
ORF I
is O
a O
functional O
gene O
that O
encodes O
an O
abundant O
secreted O
protein O
which O
is O
a O
candidate O
for O
interacting O
with O
host O
cellular O
receptors O
or O
cytokines O
. O

Protein O
metabolism O
disorders O
in O
burns O

The O
cumulative O
dose O
to O
nursing O
staff O
for O
the O
week O
after O
treatment O
was O
dependent O
on O
patient O
mobility O
and O
was O
estimated O
at O
0 O
. O
08 O
mSv O
for O
a O
self O
- O
caring O
patient O
to O
6 O
. O
3 O
mSv O
for O
a O
totally O
helpless O
patient O
( O
1840 O
MBq O
/ O
group O
A O
) O
. O

JCAHO O
to O
use O
ORYX O
data O
to O
detect O
sentinel O
events O
. O

We O
also O
show O
that O
psbH B
and O
psbT B
are O
transcribed O
from O
the O
upstream O
psbB B
gene I
promoter I
, O
and O
that O
the O
psbH B
mRNA I
has O
its O
own O
target O
sequence O
for O
Mbb1 B
function O
. O

To O
test O
whether O
Cd O
exposure O
would O
increase O
Ca O
release O
from O
bone O
during O
pregnancy O
and O
lactation O
in O
relation O
to O
the O
etiological O
mechanism O
of O
Itai O
- O
Itai O
disease O
, O
virgin O
female O
mice O
with O
45Ca O
prelabeled O
skeletons O
( O
15 O
microCi O
/ O
mouse O
) O
were O
subjected O
to O
one O
round O
of O
pregnancy O
/ O
lactation O
and O
were O
exposed O
to O
a O
Ca O
- O
deficient O
diet O
containing O
0 O
, O
5 O
, O
or O
25 O
ppm O
Cd O
or O
25 O
ppm O
Pb O
for O
32 O
days O
, O
from O
conception O
until O
Lactation O
Day O
14 O
. O

O5257 B
shows O
homology O
with O
the O
SAS2 B
protein I
and O
another O
hypothetical O
protein O
from O
yeast O
. O

Principles O
of O
thyroid O
hormone O
treatment O

Significance O
of O
esophagocardiac O
reflexes O
for O
inducing O
thoracic O
pain O

The O
fate O
of O
ochratoxin O
A O
during O
malting O
and O
brewing O
. O

METHODS O
: O
Precision O
and O
accuracy O
were O
measured O
using O
controls O
and O
method O
comparison O
studies O
. O

Messenger O
RNA O
for O
the O
Ad2 B
DNA I
binding I
protein I
: O
DNA O
sequences O
encoding O
the O
first O
leader O
and O
heterogenity O
at O
the O
mRNA O
5 O
' O
end O
. O

Consistent O
with O
this O
hypothesis O
, O
stimulation O
of O
A B
( I
2A I
) I
- I
R I
or O
PKA B
enhanced O
nuclear B
aPKC I
activity O
. O

Atrial O
dissociation O
. O

The O
gene O
sequence O
predicts O
a O
150 O
, O
825 O
mol O
wt O
apoprotein O
of O
1363 O
amino O
acids O
having O
an O
N O
- O
terminal O
hydrophobic O
signal O
sequence O
of O
17 O
amino O
acids O
, O
19 O
potential O
N O
- O
linked O
glycosylation O
sites O
, O
a O
hydrophobic O
anchor O
sequence O
of O
approximately O
17 O
amino O
acids O
near O
the O
C O
terminus O
, O
and O
a O
hydrophilic O
cysteine O
- O
rich O
C O
terminus O
of O
35 O
amino O
acids O
. O

However O
, O
depending O
upon O
the O
promoter O
context O
, O
we O
observed O
cooperative O
interactions O
between O
the O
two O
domains O
to O
confer O
high O
DNA O
- O
binding O
affinity O
and O
specificity O
. O

As O
a O
result O
, O
relative O
biological O
effects O
( O
RBE O
) O
of O
2 O
. O
3 O
- O
2 O
. O
7 O
was O
obtained O
from O
the O
straight O
line O
that O
was O
given O
by O
modifying O
by O
the O
method O
of O
least O
squares O
the O
curves O
of O
the O
frequency O
of O
anomalous O
fetuses O
in O
total O
implants O
and O
survived O
embryos O
irradiated O
from O
20 O
to O
120 O
rad O
by O
Cf O
- O
252 O
and O
from O
80 O
to O
220 O
rad O
by O
Co O
- O
60 O
on O
day O
8 O
of O
pregnancy O
. O

Samples O
( O
1 O
g O
) O
were O
extracted O
with O
0 O
. O
5 O
% O
potassium O
chloride O
( O
KCl O
) O
in O
70 O
% O
methanol O
( O
5 O
ml O
) O
and O
diluted O
subsequently O
to O
give O
two O
- O
fold O
to O
ten O
- O
fold O
step O
- O
wise O
dilutions O
in O
phosphate O
- O
buffered O
saline O
containing O
0 O
. O
05 O
% O
Tween O
20 O
and O
0 O
. O
2 O
% O
bovine B
serum I
albumin I
( O
PBS O
- O
T O
BSA I
) O
. O

To O
our O
knowledge O
108 O
cases O
of O
EAT O
treated O
by O
catheter O
ablation O
of O
the O
ectopic O
focus O
are O
reported O
in O
the O
literature O
with O
a O
success O
rate O
superior O
to O
90 O
% O
. O

Following O
immunization O
of O
transgenic O
mice O
, O
hybrid O
molecules O
were O
isolated O
from O
B O
cell O
DNA O
which O
contained O
the O
transgene O
recombined O
with O
the O
endogenous B
IgH I
locus I
. O

It O
produced O
extrapyramidal O
disturbances O
in O
nearly O
every O
subject O
, O
the O
most O
common O
being O
akathisia O
and O
the O
most O
severe O
, O
in O
the O
case O
of O
one O
individual O
, O
being O
acute O
dystonia O
. O

Northern O
blot O
analysis O
indicated O
that O
both O
human O
and O
murine O
genes O
encode O
approximately O
6 O
- O
kb O
transcripts O
. O

Transforming B
growth I
factor I
beta I
activates O
the O
promoter O
of O
cyclin B
- I
dependent I
kinase I
inhibitor O
p15INK4B B
through O
an O
Sp1 B
consensus O
site O
. O

Cox O
proportional O
hazard O
analysis O
identified O
dialysis O
modality O
and O
pre O
- O
dialysis O
UV O
of O
less O
than O
1 O
, O
000 O
ml O
/ O
m2 O
per O
24 O
h O
as O
the O
only O
significant O
risk O
factors O
for O
UV O
survival O
. O

The O
hyperpigmentation O
is O
linked O
to O
the O
presence O
of O
numerous O
single O
melanosomes O
and O
polymelanosomes O
in O
keratinocytes O
at O
all O
levels O
of O
the O
epidermis O
, O
and O
in O
dermal O
Factor B
XIIIa I
- O
positive O
dendrocytes O
. O

Possible O
or O
definite O
neglect O
or O
abuse O
before O
36 O
months O
of O
age O
was O
correlated O
with O
low O
DBH B
activity O
. O

Previously O
, O
we O
identified O
two O
human B
SMC I
family I
proteins I
, O
hCAP B
- I
C I
and O
hCAP B
- I
E I
, O
which O
form O
a O
heterodimeric O
complex O
( O
hCAP B
- I
C I
- O
hCAP B
- I
E I
) O
in O
the O
cell O
. O

The O
mouse B
alpha I
A I
- I
CRYBP1 I
gene I
specifies O
a O
2 O
, O
688 O
- O
amino O
acid O
protein O
with O
72 O
% O
amino O
acid O
identity O
to O
its O
human O
homologue O
, O
PRDII B
- I
BF1 I
. O

Methylation O
interference O
analysis O
established O
at O
single O
nucleotide O
resolution O
that O
purified O
recombinant B
Fos I
and O
Jun B
proteins I
bind O
in O
a O
sequence O
- O
specific O
manner O
to O
the O
AP B
- I
1 I
sites I
within O
the O
VDRE O
and O
OC O
box O
. O

These O
include O
urinalysis O
, O
chest O
X O
- O
P O
, O
ECG O
, O
hematological O
examinations O
( O
RBC O
, O
WBC O
, O
Ht O
, O
Hb B
, O
PLT O
and O
ESR O
) O
and O
biochemical O
tests O
( O
AST B
, O
ALT B
, O
ALP B
, O
gamma B
GTP I
, O
LDH B
, O
CPK B
, O
Chol O
, O
T O
- O
Bil O
, O
TP O
, O
Alb B
, O
TG O
, O
BUN O
, O
Cr O
, O
Glu O
, O
Na O
, O
K O
, O
Ca O
, O
P O
and O
CRP B
) O
. O

Both O
the O
inactive O
domain B
II I
P68 I
mutant I
and O
the O
deletion O
mutant O
lacking O
aa O
91 O
- O
243 O
were O
less O
inhibitory O
to O
growth O
in O
yeast O
due O
to O
the O
reduced O
ability O
to O
phosphorylate O
initiation B
factor I
2 I
alpha I
in O
vivo O
. O

All O
patients O
studied O
were O
pregnant O
women O
and O
their O
delivered O
children O
. O

Current O
body O
size O
( O
wt O
/ O
ht2 O
) O
was O
significantly O
associated O
with O
life O
- O
time O
weight O
dissatisfaction O
in O
both O
sexes O
( O
P O
less O
than O
0 O
. O
0005 O
) O
. O

Mad3 B
and O
Mad4 B
: O
novel O
Max B
- I
interacting I
transcriptional I
repressors I
that O
suppress O
c B
- I
myc I
dependent O
transformation O
and O
are O
expressed O
during O
neural O
and O
epidermal O
differentiation O
. O

CONCLUSIONS O
: O
Patients O
' O
attainment O
of O
TGB O
monotherapy O
was O
associated O
with O
their O
achievement O
of O
positive O
changes O
of O
varying O
degree O
on O
psychological O
tests O
. O

The O
pcaR B
regulatory I
locus I
has O
been O
found O
to O
be O
required O
for O
both O
induction O
of O
all O
of O
the O
genes O
within O
the O
pca B
regulon I
( O
pcaBDC B
, O
pcaIJ B
, O
and O
pcaF B
) O
and O
the O
chemotactic O
response O
of O
the O
bacteria O
to O
aromatic O
compounds O
. O

Furthermore O
, O
we O
have O
identified O
the O
major O
open O
reading O
frame O
( O
RF4 O
; O
2 O
. O
3 O
kb O
) O
as O
being O
essential O
for O
activation O
, O
and O
we O
have O
shown O
that O
the O
NF B
kappa I
B I
, O
SP1 B
, O
and O
TATA O
box O
motifs O
in O
the O
human B
immunodeficiency I
virus I
LTR I
are O
all O
required O
for O
full O
induction O
of O
the O
promoter O
by O
the O
HHV B
- I
6 I
- I
encoded I
transactivator I
. O

However O
, O
mutations O
of O
the O
C O
- O
rich O
element O
that O
disrupted O
a O
GC O
box O
located O
on O
the O
inverse O
strand O
eliminated O
PMA O
responsiveness O
and O
, O
in O
gel O
mobility O
shift O
assays O
, O
eliminated O
binding O
of O
Sp1 B
. O

TFIIB B
and O
VDR B
can O
also O
interact O
directly O
, O
and O
these O
factors O
synergize O
to O
mediate O
transactivation O
. O

Both O
algorithms O
have O
been O
implemented O
and O
applied O
to O
data O
simulated O
for O
a O
scanner O
with O
a O
large O
axial O
aperture O
( O
30 O
degrees O
) O
, O
and O
also O
to O
data O
acquired O
with O
the O
ECAT O
HR O
and O
the O
ECAT O
HR O
+ O
scanners O
. O

The O
well O
- O
trained O
group O
was O
measured O
once O
in O
the O
respiration O
chamber O
for O
36 O
h O
according O
to O
the O
same O
protocol O
. O

Samarium O
- O
153 O
for O
intravascular O
irradiation O
therapy O
with O
liquid O
- O
filled O
balloons O
to O
prevent O
restenosis O
: O
acute O
and O
long O
- O
term O
results O
in O
a O
hypercholesterolemic O
rabbit O
restenosis O
model O
. O

Only O
viable O
bacteria O
at O
a O
high O
concentration O
induced O
purulent O
otitis O
media O
, O
which O
was O
culture O
positive O
in O
58 O
% O
of O
the O
cases O
on O
day O
4 O
. O

Polyoma O
virus O
in O
urine O
during O
pregnancy O
. O

Transfer O
of O
F O
- O
like O
plasmids O
is O
regulated O
by O
the O
FinOP B
system O
, O
which O
controls O
the O
expression O
of O
traJ B
, O
a O
positive O
regulator O
of O
the O
transfer O
operon O
. O

PKCI B
- I
1 I
is O
a O
member O
of O
the O
HIT B
family I
of O
proteins O
, O
shown O
by O
sequence O
identity O
to O
be O
conserved O
in O
a O
broad O
range O
of O
organisms O
including O
mycoplasma O
, O
plants O
, O
and O
humans O
. O

Conversion O
of O
glucose O
phosphate O
- O
14C O
to O
glucose O
- O
14C O
in O
passage O
through O
human O
brain O
in O
vivo O
. O

A O
simple O
technique O
for O
obtaining O
ultra O
- O
thin O
sections O
of O
coccidian O
oocysts O
is O
reported O
. O

It O
was O
found O
that O
on O
the O
14th O
and O
21st O
day O
after O
cimetidine O
administration O
serum O
gastrin B
levels O
were O
significantly O
elevated O
. O

Dual O
signal O
peptides O
mediate O
the O
signal B
recognition I
particle I
/ O
Sec B
- O
independent O
insertion O
of O
a O
thylakoid O
membrane O
polyprotein O
, O
PsbY B
. O

The O
resulting O
score O
, O
which O
allowed O
the O
disease O
to O
be O
expressed O
as O
a O
continuous O
variable O
, O
was O
effectively O
utilized O
to O
see O
the O
correlations O
between O
the O
severity O
of O
coronary O
arterial O
disease O
( O
CAD O
) O
and O
individual O
risk O
factors O
/ O
risk O
markers O
. O

On O
guanidine O
and O
the O
treatment O
of O
botulism O
. O

Electronic O
structure O
of O
Si O
( O
111 O
) O
- O
NiSi2 O
( O
111 O
) O
A O
- O
type O
and O
B O
- O
type O
interfaces O
. O

In O
our O
case O
the O
development O
of O
the O
tumor O
at O
the O
posterior O
side O
of O
the O
prostate O
, O
the O
lack O
of O
PSA B
immunoreactivity O
and O
the O
presence O
of O
mucinous O
glands O
, O
sometimes O
" O
endocervical O
- O
like O
" O
, O
could O
suggest O
an O
origin O
from O
embryonic O
mullerian O
remnants O
in O
the O
prostatic O
utricle O
rather O
than O
urogenital O
sinus O
. O

Upon O
increasing O
the O
Marangoni O
number O
beyond O
this O
threshold O
, O
the O
initially O
stationary O
flow O
becomes O
quickly O
time O
dependent O
. O

The O
results O
showed O
differential O
activation O
of O
each O
parahippocampal O
region O
during O
verbal O
memory O
tasks O
in O
which O
the O
side O
activated O
shifted O
depending O
on O
the O
nature O
of O
the O
task O
employed O
; O
an O
increase O
in O
rCBF O
in O
the O
left O
parahippocampal O
gyrus O
was O
associated O
with O
retrieval O
strategy O
of O
non O
- O
matching O
, O
and O
an O
increase O
in O
rCBF O
in O
the O
right O
parahippocampal O
gyrus O
was O
associated O
with O
retrieval O
strategy O
of O
matching O
. O

Evolution O
of O
cytomegalovirus O
antibodies O
of O
maternal O
origin O
and O
acquired O
, O
throughout O
the O
first O
year O
of O
life O

Demonstration O
of O
changes O
in O
fetal O
liver O
erythropoiesis O
using O
echo O
- O
planar O
magnetic O
resonance O
imaging O
. O

The O
actual O
placement O
of O
the O
lesions O
was O
determined O
after O
mapping O
of O
the O
GPi O
by O
microrecording O
, O
using O
stimulation O
to O
identify O
the O
sensorimotor O
region O
and O
its O
somatotopic O
organization O
. O

Effect O
of O
prostaglandin O
E2 O
on O
experimental O
atherosclerosis O
. O

Both O
the O
arterial O
and O
venous O
thrombi O
were O
clearly O
discernible O
at O
2 O
to O
8 O
h O
after O
injection O
of O
99Tcm O
- O
SZ O
- O
51 O
. O

The O
effect O
of O
lung O
edema O
on O
pulmonary O
vasoactivity O
of O
furosemide O
. O

However O
, O
the O
malaria O
vectors O
Anopheles O
funestus O
and O
An O
. O
gambiae O
have O
both O
become O
more O
abundant O
during O
the O
1970s O
and O
1980s O
and O
sporozoite O
- O
positive O
specimens O
of O
both O
have O
been O
found O
. O

Soluble O
extracts O
from O
FGF B
- O
treated O
as O
compared O
with O
quiescent O
fibroblasts O
exhibited O
up O
to O
3 O
- O
fold O
higher O
kinase O
activity O
towards O
S6 B
in O
exogenously O
added O
rat B
liver I
40S I
ribosomes I
and O
a O
synthetic O
peptide O
, O
RRLSSLRA O
. O

In O
bell O
pepper O
, O
a O
gene O
encoding O
a O
major O
plastid O
- O
lipid O
associated O
protein O
is O
expressed O
as O
both O
partially O
and O
totally O
spliced O
transcripts O
( O
respectively O
PAP2 B
and O
PAP1 B
) O
. O

Inhibitory O
receptors O
are O
characterized O
by O
the O
presence O
of O
a O
characteristic O
sequence O
known O
as O
an O
immunoreceptor B
tyrosine I
- I
based I
inhibitory I
motif I
( O
ITIM B
) O
in O
their O
cytoplasmic O
tail O
. O

We O
conclude O
that O
LGCS O
is O
successful O
in O
treating O
esophageal O
varices O
in O
the O
setting O
of O
hyperdynamic O
portal O
circulation O
with O
acceptable O
liver O
function O
. O

Candidate O
factors O
have O
been O
identified O
by O
the O
observation O
that O
changes O
in O
glucocorticoid O
induction O
parameters O
in O
CV O
- O
1 O
cells O
could O
be O
reproduced O
by O
varying O
the O
cellular O
levels O
of O
coactivators O
[ O
transcriptional B
intermediary I
factor I
2 I
( O
TIF2 B
) O
, O
steroid B
receptor I
coactivator I
1 I
( O
SRC B
- I
1 I
) O
, O
and O
amplified B
in I
breast I
cancer I
1 I
( O
AIB1 B
) O
] O
, O
comodulator O
[ O
CREB B
- I
binding I
protein I
( O
CBP B
) O
] O
, O
or O
corepressor O
[ O
silencing B
mediator I
for I
retinoid I
and I
thyroid I
- I
hormone I
receptors I
( O
SMRT B
) O
] O
without O

concomitant O
increases O
in O
GR B
. O

Immunoblot O
analyses O
revealed O
that O
the O
loss O
of O
activator O
function O
coincides O
with O
selective O
removal O
of O
the O
C O
- O
terminal O
domain O
( O
CTD O
) O
- O
hyperphosphorylated O
RNAP B
IIO I
along O
with O
NC2 B
. O

The O
nucleotide O
sequence O
of O
the O
gene O
encoding O
this O
product O
was O
determined O
and O
the O
amino O
acid O
sequence O
deduced O
. O

At O
9 O
months O
postpartum O
, O
serum O
fT4 O
and O
fT3 O
levels O
were O
low O
normal O
( O
8 O
. O
0 O
and O
1 O
. O
7 O
pmol O
/ O
L O
, O
respectively O
) O
, O
but O
TSH B
was O
not O
raised O
( O
0 O
. O
4 O
mU O
/ O
L O
) O
. O

RESULTS O
: O
Three O
patients O
did O
not O
tolerate O
PPI O
medication O
and O
were O
managed O
by O
treatment O
with O
type O
2 O
histamine O
( O
H2 O
) O
blockers O
. O

It O
is O
considered O
important O
, O
therefore O
, O
when O
treating O
diabetic O
patients O
with O
peripheral O
neuropathy O
not O
to O
assume O
that O
the O
diabetes O
itself O
is O
the O
cause O
in O
all O
cases O
. O

TFIIIB B
assembles O
autonomously O
on O
the O
upstream O
promoter O
of O
the O
yeast B
U6 I
snRNA I
( O
SNR6 B
) O
gene O
in O
vitro O
, O
through O
the O
interaction O
of O
its O
TBP B
subunit I
with O
a O
consensus O
TATA O
box O
located O
at O
base O
pair O
- O
30 O
. O

Mutational O
analysis O
of O
the O
IME2 B
UAS I
reveals O
two O
critical O
sequence O
elements O
: O
a O
G O
+ O
C O
- O
rich O
sequence O
( O
called O
URS1 O
) O
, O
previously O
identified O
at O
many O
meiotic O
genes O
, O
and O
a O
newly O
described O
element O
, O
the O
T4C B
site O
, O
that O
we O
found O
at O
a O
subset O
of O
meiotic O
genes O
. O

Molecular O
analysis O
of O
the O
mannitol B
operon I
of I
Clostridium I
acetobutylicum I
encoding O
a O
phosphotransferase O
system O
and O
a O
putative O
PTS O
- O
modulated O
regulator O
. O

Our O
results O
show O
that O
a O
combination O
of O
Pfu B
exo I
( I
- I
) I
at O
the O
primer O
extension O
step O
and O
Taq B
at O
the O
PCR O
amplification O
step O
is O
ideal O
for O
in O
vivo O
DNA O
analysis O
and O
DNA O
damage O
mapping O
using O
LMPCR O
. O

Effect O
of O
TNF B
, O
IL B
- I
1 I
, O
and O
IL B
- I
6 I
on O
the O
proliferation O
of O
human O
Tenon O
' O
s O
capsule O
fibroblasts O
in O
tissue O
culture O
. O

Estimating O
the O
health O
care O
costs O
of O
smokers O
. O

Using O
lacZ B
- O
inducible O
T O
cells O
as O
a O
probe O
, O
we O
screened O
a O
splenic O
cDNA O
library O
in O
transiently O
transfected O
antigen O
- O
presenting O
cells O
( O
APCs O
) O
and O
isolated O
a O
cDNA O
clone O
that O
allowed O
expression O
of O
the O
appropriate O
peptide B
/ I
Kb I
MHC I
complex I
in O
APC O
. O

Endotoxemia O
and O
bacteremia O
in O
patients O
with O
sepsis O
syndrome O
in O
the O
intensive O
care O
unit O
. O

Early O
attempts O
for O
modifying O
growth O
functions O
to O
annual O
variations O
dating O
back O
up O
to O
2 O
decades O
are O
recalled O
together O
with O
examples O
for O
their O
application O
showing O
rather O
different O
degrees O
of O
approximation O
. O

A O
significant O
reduction O
in O
total O
cellular B
p27 I
protein I
levels O
and O
a O
moderate O
reduction O
in O
p27 B
mRNA I
are O
observed O
, O
but O
no O
changes O
in O
Cdk B
regulatory I
kinases I
and I
phosphatases I
occur O
. O

Genetic O
linkage O
mapping O
of O
the O
CYP11A1 B
gene I
encoding O
the O
cholesterol B
side I
- I
chain I
cleavage I
P450scc I
close O
to O
the O
CYP1A1 B
gene I
and O
D15S204 B
in O
the O
chromosome O
15q22 O
. O
33 O
- O
q23 O
region O
. O

Genetic O
and O
physiological O
experiments O
suggest O
a O
close O
relationship O
between O
cdc27 B
+ I
and O
the O
cdc2 B
+ I
gene I
, O
a O
key O
regulator O
of O
mitosis O
in O
yeast O
and O
also O
in O
higher O
eukaryotic O
cells O
. O

The O
study O
was O
performed O
in O
a O
class O
100 O
laminar O
flow O
clean O
bench O
in O
order O
to O
minimize O
particulate O
contamination O
from O
extraneous O
sources O
. O

The O
cloned O
rRNA O
operon O
was O
transcribed O
in O
vitro O
by O
using O
purified O
RNA B
polymerase I
of I
Escherichia I
coli I
. O

Of O
the O
CSF O
tested O
, O
24 O
stimulated O
oxygen O
consumption O
above O
our O
cut O
off O
, O
and O
8 O
did O
not O
( O
0 O
. O
84 O
+ O
/ O
- O
0 O
. O
34 O
, O
n O
= O
24 O
compared O
with O
the O
rate O
of O
0 O
. O
27 O
+ O
/ O
- O
0 O
. O
1 O
mumol O
/ O
min O
/ O
g O
dry O
wt O
, O
respectively O
; O
SD O
n O
= O
8 O
) O
at O
180 O
minutes O
. O

The O
sequence O
of O
the O
gene O
upstream O
to O
the O
cap O
site O
contains O
characteristic O
RNA B
polymerase I
II I
promoter I
- I
binding I
sites I
: O
a O
putative O
TATA O
box O
at O
position O
- O
29 O
and O
a O
Sp B
1 I
binding I
site I
( O
GGGGCGGAGA O
) O
at O
position O
- O
48 O
. O

The O
kinetics O
of O
[ O
14C O
] O
NG O
in O
the O
blood O
of O
the O
dosed O
animals O
was O
followed O
. O

These O
clones O
contain O
most O
of O
the O
exon O
of O
cyclin B
D2 I
except O
exon O
5 O
. O

One O
had O
a O
48 O
- O
hour O
cycle O
and O
the O
other O
a O
24 O
- O
hour O
cycle O
. O

This O
action O
could O
be O
antagonized O
by O
aminophylline O
, O
a O
competitive O
antagonist O
on O
P1 B
purinoceptors I
. O

Evidence O
for O
Gcr1p B
/ O
Gcr2p B
interaction O
has O
been O
presented O
earlier O
and O
is O
now O
supported O
by O
the O
isolation O
of O
mutations O
in O
Gcr1p B
suppressing O
gcr2 B
, O
as O
assessed O
by O
growth O
and O
enzyme O
assay O
. O

Keys O
can O
be O
used O
for O
identifying O
species O
from O
the O
European O
part O
of O
the O
USSR O
and O
north O
- O
eastern O
Kazakhstan O
. O

Promoter O
constructs O
containing O
mutations O
in O
the O
PTRE B
sequence O
that O
selectively O
abolished O
the O
binding O
of O
either O
one O
or O
both O
complexes O
exerted O
opposite O
effects O
on O
the O
transcriptional O
activity O
of O
trypsin B
promoters I
in O
A O
. O
gambiae O
and O
Aedes O
aegypti O
cell O
lines O
. O

The O
Rep78 B
protein I
of I
adeno I
- I
associated I
virus I
( I
AAV I
) O
contains O
amino O
acid O
sequence O
motifs O
common O
to O
rolling B
- I
circle I
replication I
( I
RCR I
) I
initiator I
proteins I
. O

Plasma B
prolactin I
in O
patients O
with O
colorectal O
cancer O
. O

Reality O
and O
clinical O
application O
of O
immunoglobulin B
preparations O

The O
extent O
of O
myocardial O
ischemia O
in O
the O
coronary O
sinus O
study O
was O
significantly O
correlated O
to O
the O
coronary O
diameters O
of O
the O
proximal O
and O
middle O
segments O
of O
the O
Ramus O
interventricularis O
anterior O
and O
the O
middle O
segment O
of O
the O
Ramus O
circumflexus O
( O
r O
= O
0 O
. O
87 O
, O
p O
< O
0 O
. O
02 O
) O
. O

The O
in O
- O
plane O
orientational O
anisotropy O
of O
the O
8CB O
films O
grown O
on O
unidirectionally O
photopolymerized O
PVCN O
substrates O
is O
considerably O
lower O
than O
the O
intrinsic O
surface O
orientational O
anisotropy O
of O
these O
substrates O
, O
which O
can O
explain O
the O
generally O
found O
weak O
surface O
anchoring O
of O
liquid O
crystals O
on O
PVCN O
alignment O
layers O
. O

Nuclear O
transcription O
assays O
confirmed O
that O
cys B
- I
3 I
+ I
was O
under O
sulfur O
- O
regulated O
transcriptional O
control O
and O
that O
cys B
- I
3 I
+ I
transcription O
was O
constitutive O
in O
sulfur B
controller I
( O
scon B
) O
- O
negative O
regulator O
mutants O
. O

The O
rifampicin O
resistance O
of O
uvsW B
- O
repressed O
replication O
suggests O
that O
it O
involves O
either O
tertiary O
initiation O
or O
some O
novel O
mode O
of O
initiation O
. O

Pharmacokinetics O
of O
skin O
penetration O
. O

Furthermore O
, O
the O
regulation O
conferred O
on O
a O
reporter O
gene O
in O
Drosophila O
by O
three O
closely O
related O
sequences O
demonstrates O
that O
even O
subtle O
sequence O
changes O
within O
an O
E O
box O
or O
flanking O
bases O
have O
dramatic O
consequences O
on O
the O
overall O
repertoire O
of O
proteins O
that O
can O
bind O
in O
vivo O
. O

Buspirone O
therapy O
for O
Type O
A O
behavior O
, O
hostility O
, O
and O
perceived O
stress O
in O
cardiac O
patients O
. O

Recently O
we O
have O
performed O
a O
detailed O
analysis O
of O
specific O
neuronal O
populations O
affected O
by O
the O
mutation O
which O
shed O
new O
light O
on O
the O
role O
of O
Krox B
- I
20 I
in O
the O
segmentation O
and O
on O
the O
physiological O
consequences O
of O
its O
inactivation O
. O

A O
species O
comparison O
of O
cDNA O
sequences O
and O
isolation O
of O
a O
genomic O
clone O
. O

The O
inhibition O
of O
monoamine B
oxidase I
( O
MAO B
) O
by O
tranylcypromine O
was O
studied O
in O
6 O
healthy O
volunteers O
given O
increasing O
doses O
of O
10 O
, O
15 O
, O
20 O
and O
25 O
mg O
/ O
day O
over O
a O
4 O
- O
week O
period O
. O

Deletion O
mutagenesis O
of O
the O
promoter O
indicated O
that O
a O
positive O
regulatory O
element O
( O
PRE O
) O
was O
likely O
to O
exist O
downstream O
of O
the O
UL94 B
mRNA I
start I
site I
, O
while O
a O
negative O
regulatory O
element O
( O
NRE O
) O
was O
present O
upstream O
of O
the O
TATA O
box O
. O

Expression O
of O
Helios B
was O
detected O
in O
the O
earliest O
hematopoietic O
sites O
of O
the O
embryo O
, O
in O
hematopoietic O
stem O
cells O
in O
the O
adult O
and O
was O
subsequently O
restricted O
to O
a O
subset O
of O
cells O
in O
the O
T O
cell O
lineage O
. O

Metapsychiatry O
is O
a O
term O
born O
of O
necessity O
to O
designate O
the O
important O
but O
hitherto O
unclassified O
interface O
between O
psychiatry O
and O
mysticism O
. O

Effects O
of O
negative O
pi O
mesons O
on O
mouse O
bone O
marrow O
cells O
. O

Using O
probes O
containing O
C B
/ I
EBP I
- I
binding I
sites I
from O
the O
iNOS B
gene I
revealed O
further O
binding O
of O
different O
complexes O
, O
all O
of O
which O
were O
strongly O
inducible O
by O
cAMP O
and O
to O
a O
lower O
extent O
also O
by O
IL B
- I
1beta I
. O

The O
Pi O
signals O
are O
conveyed O
to O
PHO8 B
by O
binding O
of O
PHO4 B
protein I
, O
a O
positive O
regulatory O
factor O
, O
to O
a O
promoter O
region O
of O
PHO8 B
( O
PHO8p B
) O
under O
the O
influence O
of O
the O
PHO B
regulatory O
circuit O
. O

Thus O
, O
the O
end O
of O
a O
linearized O
DNA O
fragment O
can O
initiate O
new O
DNA O
synthesis O
by O
BIR O
in O
which O
the O
newly O
synthesized O
DNA O
is O
displaced O
and O
subsequently O
forms O
circles O
by O
NHEJ O
. O

It O
would O
seem O
that O
caution O
should O
be O
exercised O
in O
using O
HPL O
values O
as O
an O
index O
of O
placental O
function O
in O
anaemic O
women O
. O

A O
case O
of O
acquired O
immune O
deficiency O
syndrome O
before O
1980 O
. O

These O
data O
suggest O
that O
multiple O
genetic O
recombination O
among O
bacteriophages O
with O
different O
immunities O
took O
place O
to O
generate O
the O
prophage O
VT1 O
- O
Sakai O
. O

The O
adenovirus O
E1B I
19 I
, I
000 I
- I
molecular I
- I
weight I
( I
19K I
) I
protein I
is O
a O
potent O
inhibitor O
of O
apoptosis O
and O
cooperates O
with O
E1A B
to O
transform O
primary O
rodent O
cells O
. O

Desensitization O
of O
the O
growth B
hormone I
- O
induced O
Janus B
kinase I
2 I
( O
Jak B
2 I
) O
/ O
signal B
transducer I
and I
activator I
of I
transcription I
5 I
( O
Stat5 B
) O
- O
signaling O
pathway O
requires O
protein O
synthesis O
and O
phospholipase B
C I
. O

In O
two O
separate O
studies O
, O
specimens O
of O
saliva O
from O
57 O
individuals O
over O
the O
age O
of O
65 O
years O
( O
mean O
age O
, O
76 O
. O
7 O
years O
) O
and O
37 O
persons O
under O
the O
age O
of O
40 O
years O
( O
mean O
age O
, O
28 O
. O
8 O
years O
) O
were O
examined O
for O
concentrations O
of O
IgA B
as O
functions O
of O
volume O
, O
total O
protein O
, O
and O
electrolyte O
conductivity O
; O
some O
were O
also O
tested O
for O
IgG B
and O
IgM B
content O
. O

E2A B
- O
HLF B
- O
mediated O
cell O
transformation O
requires O
both O
the O
trans O
- O
activation O
domains O
of O
E2A B
and O
the O
leucine O
zipper O
dimerization O
domain O
of O
HLF B
. O

Two O
adjacent O
, O
highly O
homologous O
endoglucanase I
genes I
, O
celD B
and O
celE B
from I
Fibrobacter I
succinogenes I
S85 I
, O
which O
were O
separated O
by O
an O
AT O
- O
rich O
223 O
- O
nucleotide O
intergenic O
region O
were O
characterized O
. O

The O
Seldinger O
technique O
for O
difficult O
transurethral O
catheterization O
: O
a O
gentle O
alternative O
to O
suprapubic O
puncture O
( O
Br O
J O
Surg O
2000 O
; O
87 O
: O
1729 O
- O
30 O
) O
. O

We O
screened O
an O
A O
. O
thaliana O
cDNA O
library O
, O
whose O
inserts O
are O
under O
the O
control O
of O
the O
galactose O
- O
inducible O
GAL10 B
promoter I
, O
for O
cDNAs O
which O
enabled O
YDH8 O
cells O
to O
grow O
at O
the O
restrictive O
temperature O
. O

We O
generated O
" O
signature O
" O
oligonucleotides O
from O
these O
CDR3s B
and O
probed O
PCR O
amplified O
V B
kappa I
products I
from O
the O
synovium O
and O
PBLs O
of O
the O
same O
patient O
, O
and O
from O
PBLs O
and O
spleen O
of O
individuals O
without O
rheumatic O
disease O
. O

One O
of O
the O
major O
transcripts O
encodes O
MEQ B
, O
a O
339 B
- I
amino I
- I
acid I
bZIP I
protein I
which O
is O
homologous O
to O
the O
Jun B
/ O
Fos B
family O
of O
transcription O
factors O
. O

It O
is O
concluded O
that O
the O
new O
class O
of O
competitive O
NMDA B
receptor I
antagonists O
, O
exemplified O
by O
CGP O
37849 O
, O
is O
the O
most O
promising O
candidate O
for O
clinical O
trials O
in O
anxiety O
disorders O
. O

An O
emended O
diagnosis O
of O
Tylocephalum O
is O
proposed O
excluding O
this O
feature O
, O
along O
with O
distribution O
of O
the O
testes O
in O
the O
preovarian O
field O
and O
circummedullary O
distribution O
of O
vitelline O
follicles O
. O

Induction O
of O
interferon B
- I
alpha I
( O
IFNalpha B
) O
gene O
expression O
in O
virus O
- O
infected O
cells O
requires O
phosphorylation O
- O
induced O
activation O
of O
the O
transcription O
factors O
IRF3 B
and O
IRF7 B
. O

METHODS O
: O
The O
prevalence O
of O
haemagglutination B
inhibiting I
( I
HI I
) I
antibodies I
to O
JE O
virus O
( O
JEV O
) O
, O
West O
Nile O
virus O
( O
WNV O
) O
and O
dengue O
- O
2 O
virus O
( O
DEN O
- O
2 O
) O
was O
detected O
by O
HI O
test O
and O
IgM B
antibody O
capture O
ELISA O
( O
MAC O
ELISA O
) O
was O
performed O
to O
determine O
recent O
infections O
with O
JE O
virus O
. O

Moreover O
, O
since O
the O
integrated O
transforming O
DNA O
was O
not O
altered O
or O
lost O
expression O
of O
the O
RbcS2 B
: O
: O
aadA B
: O
: O
RbcS2 B
gene I
( O
s I
) O
appears O
to O
be O
repressed O
. O

Donehower O
and O
H O
. O

A O
characteristic O
feature O
is O
their O
tendency O
to O
form O
homo O
- O
and O
/ O
or O
heterodimeric O
complexes O
. O

Further O
, O
more O
recent O
communications O
suggest O
that O
liver O
disease O
led O
to O
a O
differential O
alteration O
of O
the O
cytochrome B
P I
- I
450s I
with O
regard O
to O
protein O
content O
and O
activity O
. O

Apparent O
Buschke O
- O
Loewenstein O
tumour O
of O
the O
penis O
. O

This O
effect O
was O
stronger O
after O
multiple O
predoses O
of O
sertraline O
, O
when O
imipramine O
Cmax O
and O
AUC O
( O
0 O
- O
infinity O
) O
were O
increased O
by O
39 O
% O
and O
68 O
% O
, O
respectively O
. O

Mutations O
in O
nifR1 B
( O
ntrC B
) O
and O
nifR4 B
( O
rpoN B
, O
encoding O
sigma54 B
) O
had O
no O
influence O
on O
xdh B
gene I
expression O
. O

We O
therefore O
propose O
that O
PRR2 B
, O
and O
not O
PVR B
, O
is O
the O
true O
human B
homolog I
of I
MPH I
. O

Swarming O
of O
Moraxella O

Biochem O
. O

In O
contrast O
, O
vRel B
lacks O
a O
strong O
C O
- O
terminal O
gene O
activation O
function O
, O
since O
a O
LexA B
fusion I
protein I
containing O
C O
- O
terminal O
vRel B
sequences O
alone O
only O
weakly O
activated O
transcription O
. O

The O
crystal O
structure O
of O
the O
yeast B
Phe I
- I
tRNAPhe I
ternary O
complex O
with O
Thermus B
aquaticus I
EF I
- I
Tu I
- O
GDPNP B
( O
Phe B
- I
TC I
) O
has O
previously O
been O
determined O
as O
one O
representative O
of O
this O
general O
yet O
highly O
discriminating O
complex O
formation O
. O

Bovine O
anaplasmosis O
: O
susceptibility O
of O
seronegative O
cows O
from O
an O
infected O
herd O
to O
experimental O
infection O
with O
Anaplasma O
marginale O
. O

Because O
of O
gut O
fermentation O
, O
a O
substantial O
portion O
( O
16 O
- O
80 O
% O
) O
of O
N O
is O
absorbed O
as O
ammonia O
N O
( O
NH3N O
) O
. O

Quantitative O
measurement O
of O
antigamma B
globulin I
factors I
( O
rheumatoid B
factors I
) O
using O
laser O
- O
nephelometry O
in O
comparison O
with O
the O
latex O
agglutination O
test O
and O
the O
Waaler O
- O
Rose O
test O

The O
densities O
ranged O
between O
2 O
and O
3976 O
neurites O
/ O
mm2 O
skin O
surface O
, O
but O
the O
overlap O
between O
subjects O
and O
without O
PHN O
was O
small O
. O

Zoonotic O
areas O
in O
which O
HIV O
co O
- O
infection O
is O
common O
could O
also O
be O
at O
risk O
as O
sandflies O
can O
become O
infected O
from O
co O
- O
infected O
individuals O
. O

The O
determination O
of O
antithrombin B
III I
( O
AT B
III I
) O
and O
its O
clinical O
significance O

The O
kinetics O
of O
erythromycin O
( O
E O
. O
) O
was O
studied O
in O
16 O
patients O
with O
different O
degrees O
of O
impairment O
of O
renal O
function O
after O
a O
single O
intravenous O
dose O
. O

Base O
substitutions O
within O
this O
NFIL B
- I
6 I
site I
resulted O
in O
virtual O
elimination O
of O
LPS O
- O
induced O
IL B
- I
1 I
beta I
gene I
transcription O
. O

Mortality O
rates O
were O
not O
different O
( O
chi2 O
: O
0 O
. O
0298 O
, O
p O
> O
0 O
. O
5 O
) O
. O

The O
MALDI O
- O
TOF O
experiment O
, O
however O
, O
proved O
to O
be O
superior O
to O
the O
GC O
experiment O
, O
particularly O
with O
regard O
to O
baseline O
resolution O
of O
unsaturated O
acids O
. O

Modern O
aspects O
of O
shock O
- O
treatment O
in O
extensive O
burns O
( O
author O
' O
s O
transl O
) O

Of O
the O
private O
practices O
accredited O
in O
August O
1995 O
, O
40 O
. O
6 O
per O
cent O
( O
122 O
) O
participated O
. O

A O
Drosophila O
subobscura O
genomic O
fragment O
containing O
all O
the O
exons O
and O
the O
late O
and O
early O
promotors O
in O
the O
Sxl B
gene I
of I
D I
. I
melanogaster I
was O
isolated O
. O

Collectively O
, O
a O
unique O
mechanism O
involving O
NF B
- I
ATp I
appears O
to O
regulate O
the O
cell O
type O
- O
specific O
and O
activation O
- O
dependent O
expression O
of O
the O
SCM B
- I
1 I
genes I
. O

The O
sensitivity O
of O
central O
dopamine I
receptors I
was O
assessed O
with O
the O
growth B
hormone I
response O
to O
apomorphine O
application O
. O

Oral O
basal O
body O
temperature O
( O
BBT O
) O
recordings O
of O
46 O
women O
that O
conceived O
by O
donor O
insemination O
and O
who O
had O
midcycle O
monitoring O
of O
luteinising B
hormone I
( O
LH B
) O
were O
analysed O
to O
establish O
features O
associated O
with O
an O
optimal O
cycle O
. O

Our O
results O
admit O
that O
a O
ribosome O
scanning O
mechanism O
of O
the O
MP B
gene I
expression O
from O
I B
( I
2 I
) I
sgRNA I
operates O
concurrently O
. O

The O
three O
- O
dimensional O
structure O
of O
the O
E B
. I
coli I
CR I
domain I
indicates O
that O
this O
sequence O
conservation O
is O
likely O
to O
result O
in O
a O
conserved O
structural O
motif O
. O

Otosclerosis O
in O
monozygotic O
twins O
: O
clinical O
and O
genetical O
analysis O

A O
DNA O
fragment O
that O
complements O
the O
uvs B
- I
2 I
mutation I
was O
subcloned O
by O
monitoring O
its O
ability O
to O
transform O
the O
uvs B
- I
2 I
mutant I
to O
MMS O
resistance O
. O

The O
human B
lbc I
oncogene I
product I
is O
a O
guanine B
nucleotide I
exchange I
factor I
that O
specifically O
activates O
the O
Rho B
small I
GTP I
binding I
protein I
, O
thus O
resulting O
in O
biologically O
active O
, O
GTP O
- O
bound O
Rho B
, O
which O
in O
turn O
mediates O
actin B
cytoskeletal O
reorganization O
, O
gene O
transcription O
, O
and O
entry O
into O
the O
mitotic O
S O
phase O
. O

Insulin B
- O
stimulated O
autophosphorylation O
at O
specific O
sites O
in O
the O
tyrosine B
kinase I
domain I
of O
the O
receptor O
' O
s O
beta O
- I
subunit O
is O
correlated O
kinetically O
with O
activation O
of O
kinase O
- O
catalyzed O
phosphorylation O
of O
a O
model O
substrate O
( O
reduced O
and O
carboxyamidomethylated O
lysozyme B
; O
RCAM B
- I
lysozyme I
) O
. O

The O
critical O
obstacle O
in O
modeling O
psychiatric O
disorders O
has O
been O
limited O
information O
about O
their O
origin O
and O
underlying O
neural O
mechanisms O
. O

BACKGROUND O
: O
Allergic O
rhinoconjunctivitis O
is O
a O
common O
disorder O
, O
affecting O
> O
20 O
% O
of O
people O
of O
all O
socioeconomic O
strata O
. O

The O
results O
of O
these O
studies O
indicate O
that O
EHV B
- I
1 I
gp13 I
is O
the O
structural O
homolog O
of O
herpes B
simplex I
virus I
glycoprotein I
C I
and O
further O
suggest O
that O
the O
epitope O
- O
containing O
N O
- O
terminal O
amino O
acid O
sequences O
of O
the O
herpesvirus O
gC B
- I
like I
glycoproteins I
have O
undergone O
more O
extensive O
evolutionary O
divergence O
than O
the O
C O
- O
terminal O
sequences O
. O

Within O
the O
final O
11 O
amino O
acids O
there O
are O
5 O
aromatic O
, O
2 O
basic O
, O
and O
no O
acidic O
residues O
and O
it O
has O
been O
proposed O
that O
these O
residues O
stack O
with O
and O
electrostatically O
interact O
with O
the O
kinked O
DNA O
at O
the O
site O
of O
a O
pyrimidine O
dimer O
. O

By O
contrast O
, O
footprint O
II O
at O
position O
- O
125 O
is O
common O
to O
both O
HeLa O
and O
GH3 O
cell O
extracts O
and O
overlies O
a O
15 O
- O
base O
- O
pair O
sequence O
found O
in O
all O
members O
of O
the O
growth B
hormone I
gene I
family I
. O

Significance O
of O
the O
determination O
of O
vascular O
tonus O
in O
the O
differential O
diagnosis O
of O
rheumatic O
and O
arteriosclerotic O
lesions O
of O
the O
heart O
and O
vessels O
in O
middle O
- O
aged O
and O
elderly O
persons O
with O
fibrillation O
arrhythmia O

The O
gene O
responsible O
for O
multiple O
endocrine O
neoplasia O
type O
1 O
( O
MEN1 O
) O
, O
a O
heritable O
predisposition O
to O
endocrine O
tumours O
in O
man O
, O
has O
recently O
been O
identified O
. O

Toxicological O
effects O
of O
dietary O
Maillard O
reaction O
products O
in O
the O
rat O
. O

AST B
activity O
in O
PMNs O
was O
significantly O
low O
, O
approximately O
2 O
% O
of O
that O
observed O
in O
HPLFs O
. O

Specifically O
, O
we O
engineered O
two O
chimeras O
in O
which O
the O
N O
- O
terminal O
lobe O
of O
the O
SH1 B
domain I
of O
c B
- I
Abl I
was O
swapped O
with O
that O
of O
v B
- I
Src I
. O

Biochemical O
analyses O
revealed O
that O
the O
concentration O
of O
norepinephrine O
was O
reduced O
significantly O
in O
cortex O
- O
hippocampus O
and O
olfactory O
bulb O
but O
not O
in O
other O
regions O
, O
while O
dopamine O
and O
serotonin O
levels O
were O
not O
altered O
in O
any O
brain O
region O
examined O
. O

The O
cDNA O
contains O
a O
reading O
frame O
for O
a O
145 O
- O
amino O
- O
acid O
protein O
and O
it O
lacks O
the O
UGA O
codons O
, O
which O
have O
been O
found O
in O
the O
reading O
frame O
of O
the O
mouse B
MCS I
cDNA I
and O
have O
been O
presumed O
to O
encode O
the O
selenocysteine O
in O
the O
amino O
terminal O
of O
the O
deduced O
mouse O
amino O
acid O
sequence O
. O

These O
results O
suggest O
that O
repetitive O
GAA O
sequences O
enhance O
splicing O
by O
binding O
a O
protein O
complex O
containing O
a O
sequence O
- O
specific O
RNA O
binding O
protein O
and O
a O
general O
splicing O
activator O
that O
, O
in O
turn O
, O
recruit O
additional O
SR B
proteins I
. O

Letter O
: O
Dehydrated O
test O
strip O
for O
the O
detection O
of O
yeasts O
. O

Cell O
sloughing O
with O
proparacaine O
. O

Cytomegalovirus O
infection O
- O
- O
modern O
diagnosis O

Highly O
conserved O
regions O
called O
src B
homology I
2 I
and I
3 I
( O
SH2 B
and O
SH3 B
) O
, O
comprising O
amino O
acid O
residues O
88 O
to O
250 O
, O
are O
believed O
to O
modulate O
the O
protein B
- I
tyrosine I
kinase I
activity O
present O
in O
the O
carboxy O
- O
terminal O
halves O
of O
the O
src B
proteins I
. O

Interestingly O
, O
the O
NES B
mutant I
proteins I
appeared O
to O
have O
altered O
interactions O
with O
the O
splicing O
complex O
, O
binding O
more O
tightly O
to O
SC35 B
in O
co O
- O
immunoprecipitation O
assays O
. O

We O
propose O
2 O
separate O
PAI B
- I
1 I
inductory O
pathways O
for O
PMA O
and O
IL B
- I
1alpha I
in O
HepG2 O
, O
both O
involving O
protein B
tyrosine I
kinase I
activation O
; O
the O
serum O
- O
induced O
signaling O
pathway O
may O
( O
partially O
) O
overlap O
with O
the O
PMA B
- I
activated I
protein I
kinase I
C I
/ O
mitogen B
- I
activated I
protein I
kinase I
kinase I
pathway O
, O
leading O
to O
c B
- I
Jun I
homodimer I
binding O
to O
the O
PAI B
- I
1 I
TRE O
. O

We O
show O
that O
sae1 B
- I
1 I
and O
sae3 B
- I
1 I
mutations I
each O
confer O
a O
distinct O
defect O
in O
meiotic O
recombination O
. O

sae1 B
- I
1 I
produces O
recombinants O
but O
very O
slowly O
and O
ultimately O
to O
less O
than O
half O
the O
wild O
- O
type O
level O
; O
sae3 B
- I
1 I
makes O
persistent O
hyper O
- O
resected O
meiotic O
double O
- O
strand O
breaks O
and O
has O
a O
severe O
defect O
in O
formation O
of O
recombinants O
. O

G6620 O
is O
the O
3 O
' O
end O
of O
the O
MOL1 B
gene I
coding O
for O
a O
polypeptide O
similar O
to O
stress O
- O
inducible O
proteins O
from O
Fusarium O
; O
G6630 O
is O
the O
NAT2 B
gene I
which O
encodes O
a O
methionine B
N I
- I
acetyltransferase I
; O
G6635 O
is O
the O
RPL30B B
gene I
coding O
for O
the O
ribosomal B
protein I
L30 I
; O
G6658 O
is O
RSR1 B
encoding O
a O
ras B
- I
related I
protein I
; O
G6667 O
is O
CYS4 B
, O
the O
gene O
for O
cystathionine B
beta I
- I
synthase I

; O
G6670 O
is O
identical O
to O
ORF2 O
located O
close O
to O
CYS4 B
; O
G6673 O
is O
PEM1 B
/ O
CHO2 B
encoding O
a O
phosphatidylethanolamine B
methyltransferase I
; O
G7001 O
is O
the O
NSR1 B
gene I
coding O
for O
a O
nuclear O
signal O
recognition I
protein I
. O

Transcriptional O
analysis O
of O
this O
gene O
cluster O
was O
performed O
by O
detecting O
the O
presence O
of O
mRNAs O
spanning O
adjacent O
genes O
as O
well O
as O
by O
using O
a O
promoterless O
lacZ B
reporter I
gene I
fused O
to O
each O
of O
the O
seven O
genes O
contained O
in O
the O
tol B
- O
oprL B
locus O
. O

Serum O
IgG B
and O
IgA B
level O
were O
decreased O
. O

Histopathologically O
, O
( O
mice O
killed O
with O
high O
doses O
of O
GMC O
- O
II O
, O
given O
orally O
) O
there O
were O
diffuse O
hyperemia O
of O
the O
liver O
, O
parenchymal O
degeneration O
of O
the O
kidney O
tubuli O
epithelium O
, O
and O
edema O
and O
emphysema O
of O
the O
lungs O
. O

The O
contribution O
of O
reviewers O
and O
editors O
to O
the O
scientific O
work O
of O
the O
authors O

The O
effects O
of O
space O
radiation O
are O
partially O
known O
on O
astronauts O
, O
but O
much O
remains O
to O
be O
discovered O
. O

Therefore O
the O
effect O
of O
GAL11 B
on O
PGK B
transcription O
must O
be O
mediated O
at O
the O
PGK B
UAS I
, O
presumably O
as O
part O
of O
the O
activation O
complex O
. O

Wingless B
/ O
Wnt B
signaling O
directs O
cell O
- O
fate O
choices O
during O
embryonic O
development O
. O

RNase B
protection O
analysis O
reveals O
a O
10 O
- O
fold O
increase O
in O
the O
expression O
of O
SCD2 B
mRNA I
during O
3T3 O
- O
L1 O
preadipocyte O
differentiation O
. O

To O
determine O
whether O
transcription O
of O
orfX B
and O
vfr B
are O
controlled O
by O
the O
same O
mechanisms O
that O
control O
transcription O
of O
the O
region O
of O
the O
divergent O
ORF O
( O
dorf B
) O
and O
of O
crp B
, O
we O
compared O
the O
vfr B
- O
orfX B
and O
crp B
- O
dorf B
intergenic O
regions O
. O

As O
a O
member O
of O
the O
uncoupling B
protein I
family I
, O
UCP2 B
is O
ubiquitously O
expressed O
in O
rodents O
and O
humans O
, O
implicating O
a O
major O
role O
in O
thermogenesis O
. O

Copyright O
1998 O
Academic O
Press O
. O

Parental O
and O
vector O
- O
transfected O
MCF7 O
cells O
, O
which O
were O
sensitive O
to O
the O
growth O
- O
inhibitory O
effects O
of O
atRA O
, O
exhibited O
atRA O
- O
dependent O
retinoic B
acid I
receptor I
( O
RAR B
) O
transactivation O
and O
transrepression O
of O
12 O
- O
O O
- O
tetradecanoylphorbol O
- O
13 O
- O
acetate O
- O
induced O
AP B
- I
1 I
activity O
. O

The O
majority O
of O
the O
respondents O
( O
83 O
. O
4 O
% O
) O
considered O
that O
coitus O
should O
not O
be O
stopped O
during O
pregnancy O
. O

Based O
on O
the O
ratios O
of O
the O
areas O
under O
the O
concentration O
- O
time O
curves O
in O
CSF O
and O
serum O
, O
the O
overall O
penetration O
of O
rifampicin O
into O
CSF O
was O
0 O
. O
13 O
- O
0 O
. O
42 O
( O
median O
= O
0 O
. O
22 O
) O
. O

The O
differential O
diagnosis O
of O
mineral O
oil O
lipidosis O
in O
lymph O
nodes O
with O
reaction O
to O
radiopaque O
oils O
and O
Whipple O
' O
s O
disease O
is O
discussed O
. O

With O
rats O
fed O
on O
the O
Zn O
- O
sufficient O
diet O
, O
NaFe3 O
+ O
EDTA O
and O
Na2EDTA O
similarly O
increased O
the O
absorption O
, O
urinary O
excretion O
and O
retention O
of O
Zn O
but O
to O
a O
lesser O
extent O
. O

99Tcm O
- O
labelled O
albumin B
colloid O
, O
albures B
( O
radius O
250 O
nm O
) O
or O
nanocoll O
( O
radius O
25 O
nm O
) O
, O
or O
both O
were O
used O
as O
test O
substances O
to O
study O
the O
kinetics O
of O
vascular O
clearance O
after O
RES O
stimulation O
. O

Meq B
/ O
c B
- I
Jun I
heterodimers O
bind O
to O
an O
AP1 B
- I
like I
sequence I
in O
the O
meq B
promoter I
region I
with O
an O
affinity O
much O
greater O
than O
that O
of O
Meq B
/ O
Meq B
or O
c B
- I
Jun I
/ O
c B
- I
Jun I
homodimers O
. O

Moreover O
, O
the O
noncoordinate O
effects O
of O
FSK B
on O
PMA O
- O
stimulated O
MKK B
and O
MAPK B
activities O
indicates O
the O
presence O
of O
a O
additional O
distal O
cAMP O
- O
dependent O
inhibitory O
mechanisms O
. O

These O
findings O
indicated O
that O
p35 B
is O
a O
trans O
- O
dominant O
factor O
that O
facilitates O
AcMNPV O
growth O
in O
a O
cell O
line O
- O
specific O
manner O
. O

Pseudotumor O
form O
of O
pulmonary O
nocardia O
infection O
( O
Nocardia O
nova O
) O
in O
a O
renal O
transplant O
patient O

Hematological O
parameters O
( O
leukocyte O
, O
neutrophil O
and O
platelet O
counts O
) O
and O
liver O
function O
tests O
were O
determined O
by O
standard O
procedures O
. O

Karyo O
- O
and O
cytometric O
investigations O
of O
cortical O
layer O
V O
pyramidal O
neurons O
of O
albino O
rat O
after O
three O
various O
fixations O

Clinical O
experience O
gained O
from O
a O
combined O
study O
of O
252Cf O
brachytherapy O
by O
the O
staff O
- O
members O
of O
the O
Research O
Institute O
of O
Medical O
Radiology O
, O
USSR O
AMS O
, O
was O
generalized O
. O

The O
team O
physician O
and O
conditioning O
of O
athletes O
for O
sports O
: O
a O
consensus O
statement O
. O

To O
probe O
the O
inter O
- O
relationship O
between O
the O
virion O
- O
anchoring O
function O
and O
the O
oligomerization O
function O
, O
we O
constructed O
two O
serotype O
3 O
( O
T3 O
) O
sigma B
1 I
deletion O
mutants I
in O
SV40 O
expression O
vectors O
, O
one O
lacking O
the O
hydrophobic O
tail O
and O
the O
hinge O
, O
and O
the O
other O
lacking O
an O
adjacent O
region O
which O
constituted O
part O
of O
the O
coiled O
- O
coil O
. O

AIMS O
: O
The O
aim O
of O
this O
study O
was O
to O
assess O
static O
and O
dynamic O
bone O
changes O
in O
patients O
suffering O
from O
rickets O
. O

The O
law O
and O
the O
chiropractor O
. O

In O
contrast O
, O
overexpression O
of O
the O
DREB2A B
cDNA I
induced O
weak O
expression O
of O
the O
target O
genes O
under O
unstressed O
conditions O
and O
caused O
growth O
retardation O
of O
the O
transgenic O
plants O
. O

Our O
findings O
suggest O
that O
AIF O
may O
be O
a O
morphologic O
correlate O
of O
tumor O
regression O
following O
preoperative O
cytotoxic O
chemotherapy O
. O

Mean O
total O
homocysteine O
( O
tHcy O
) O
was O
21 O
. O
1 O
+ O
/ O
- O
9 O
. O
5 O
micromol O
/ O
L O
and O
median O
concentration O
was O
19 O
micromol O
/ O
L O
. O

Efficacy O
of O
probiotic O
feed O
additives O
: O
guidelines O
for O
the O
evaluation O
of O
the O
efficiency O
of O
microorganisms O
in O
dogs O
, O
cats O
, O
and O
horses O
] O
Probiotic O
microorganisms O
are O
frequently O
in O
use O
as O
feed O
additives O
for O
farm O
and O
pet O
animals O
. O

Lung O
transplantation O
is O
warranted O
for O
stable O
, O
ventilator O
- O
dependent O
recipients O
. O

Site O
- O
directed O
mutagenesis O
shows O
that O
sites O
throughout O
the O
IRE O
alter O
negative O
control O
and O
IRE B
- I
BP I
binding O
reflecting O
the O
fact O
that O
the O
footprint O
of O
the O
IRE B
- I
BP I
is O
over O
the O
entire O
IRE O
. O

An O
alternative O
approach O
to O
random O
integration O
of O
large O
DNA O
fragments O
into O
plants O
is O
to O
utilize O
one O
of O
several O
site O
- O
specific O
recombination O
( O
SSR O
) O
systems O
, O
such O
as O
Cre B
/ O
lox B
. O

Neonatal O
masculinization O
affects O
maternal O
behavior O
sensitivity O
in O
female O
rats O
. O

Recombinant B
mouse I
GSTT1 I
- I
1 I
was O
catalytically O
active O
towards O
1 O
, O
2 O
- O
epoxy O
- O
3 O
- O
( O
p O
- O
nitrophenoxy O
) O
propane O
, O
4 O
- O
nitrobenzyl O
chloride O
and O
dichloromethane O
. O

In O
addition O
, O
six O
other O
landmarks O
favored O
the O
xeroradiograph O
but O
not O
to O
the O
degree O
of O
statistical O
significance O
. O

Nonlethal O
sec71 B
- I
1 I
and O
sec72 B
- I
1 I
mutations I
eliminate O
proteins O
associated O
with O
the O
Sec63p B
- O
BiP B
complex O
from O
S O
. O
cerevisiae O
. O

Sequence O
and O
deletion O
analysis O
of O
the O
recombination B
enhancement I
gene I
( O
ref B
) O
of O
bacteriophage O
P1 O
: O
evidence O
for O
promoter O
- O
operator O
and O
attenuator O
- O
antiterminator O
control O
. O

A O
high O
titer O
of O
anti B
- I
HBc I
, O
thus O
suggested O
to O
be O
an O
indicator O
of O
persistent O
hepatitis O
B O
virus O
infection O
, O
was O
found O
rarely O
in O
seronegative O
patients O
with O
chronic O
hepatitis O
, O
non O
- O
alcoholic O
cirrhosis O
, O
or O
alcoholic O
liver O
diseases O
. O

Adaptability O
of O
Nippostrongylus O
brasiliensis O
( O
Travassos O
, O
1914 O
) O
to O
lead O
contamination O

Eph B
receptors I
and O
their O
membrane O
- O
associated O
ephrin B
ligands I
regulate O
cell O
- O
cell O
interactions O
during O
development O
. O

We O
discuss O
the O
possibility O
that O
DNA O
- O
protein O
interactions O
at O
homologous O
nucleotide O
sequences O
like O
those O
identified O
in O
PII B
are O
part O
of O
a O
regulatory O
gene O
cascade O
that O
participates O
in O
timing O
fla B
gene I
expression O
in O
the O
C O
. O
crescentus O
cell O
cycle O
. O

Postcontrast O
images O
were O
also O
acquired O
in O
the O
sagittal O
( O
six O
patients O
) O
and O
coronal O
( O
three O
patients O
) O
planes O
. O

Nature O
of O
the O
thermopower O
in O
bipolar O
semiconductors O
. O

To O
increase O
the O
number O
of O
circulating O
polymorphonuclear O
neutrophils O
, O
the O
patient O
was O
treated O
with O
recombinant B
granulocyte I
- I
macrophage I
colony I
- I
stimulating I
factor I
( O
GM B
- I
CSF I
) O
at O
a O
dose O
of O
2 O
micrograms O
protein O
/ O
kg O
bodyweight O
s O
. O
c O
. O
/ O
12 O
h O
. O

By O
using O
methanol O
- O
0 O
. O
15 O
M O
borate O
buffer O
of O
pH O
8 O
. O
0 O
, O
cate B
- I
chol I
- I
O I
- I
methyltransferase I
activity O
might O
be O
assayed O
. O

All O
patients O
were O
peritonitis O
- O
free O
at O
least O
6 O
weeks O
before O
each O
PET O
. O

The O
amino O
acid O
residues O
and O
subdomains O
important O
for O
DNA O
binding O
, O
hormone O
binding O
, O
dimerization O
, O
and O
transactivation O
are O
mostly O
conserved O
among O
all O
VDR B
species O
. O

Transcripts O
appear O
to O
be O
initiated O
from O
an O
upstream O
promoter O
, O
P1 O
, O
located O
in O
front O
of O
the O
tRNA B
( I
met1 I
) I
gene I
and O
from O
three O
internal O
promoters O
: O
P2 O
is O
located O
immediately O
in O
front O
of O
the O
tRNA B
( I
met2 I
) I
gene I
; O
PL10 O
is O
near O
the O
beginning O
of O
the O
L1 B
- O
L10 B
intergenic O
space O
, O
and O
PL12 B
is O
at O
the O
end O
of O
the O
L10 B
gene I
sequence I
. O

Based O
on O
the O
deduced O
amino O
acid O
sequence O
identity O
we O
designated O
GAPC1 B
and O
GAPC2 B
as O
group O
I O
( O
97 O
% O
identical O
) O
and O
GAPC3 B
and O
GAPC4 B
as O
group O
II O
( O
99 O
. O
4 O
% O
identical O
) O
. O

A O
case O
of O
functioning O
parathyroid O
carcinoma O
with O
hypereninemic O
hypertension O
. O

Finally O
, O
gel O
shift O
assays O
showed O
that O
ORF2 O
was O
able O
to O
bind O
to O
promoter O
fragment O
566 O
- O
888 O
. O

Critical O
review O
of O
general O
results O

Saturated O
fatty O
acids O
induce O
a O
1 O
. O
6 O
- O
fold O
increase O
in O
transcription O
activity O
, O
whereas O
a O
large O
family O
of O
unsaturated O
fatty O
acids O
repress O
OLE1 B
transcription O
as O
much O
as O
60 O
- O
fold O
. O

This O
polypeptide O
includes O
the O
first O
three O
zinc O
fingers O
of O
the O
TFIIIA B
DNA I
binding I
domain I
. O

An O
exception O
is O
the O
Bcl B
- I
6 I
gene I
, O
encoding O
a O
transcription O
factor O
, O
which O
was O
found O
to O
be O
mutated O
in O
normal O
human O
memory O
B O
cells O
. O

3 O
. O

A O
chest O
- O
X O
ray O
film O
showed O
infiltrative O
shadows O
in O
the O
right O
middle O
and O
lower O
lung O
fields O
, O
but O
a O
chest O
CT O
scan O
showed O
an O
abnormal O
lung O
density O
in O
the O
right O
lower O
lobe O
. O

In O
both O
liver O
and O
pancreatic O
islet O
, O
glucokinase B
is O
subject O
to O
inhibition O
by O
a O
regulatory O
protein O
( O
GCKR B
) O
. O

MRI O
measurements O
of O
the O
brain O
. O

Similarly O
, O
TAM O
- O
67 O
reverted O
the O
morphology O
of O
the O
AdoMetDC B
- I
antisense I
expressors I
. O

Of O
concern O
is O
the O
high O
, O
unexplained O
prevalence O
of O
BV O
among O
African O
- O
American O
women O
, O
who O
are O
also O
at O
extremely O
high O
risk O
for O
preterm O
birth O
. O

The O
use O
of O
bentonite O
in O
the O
compounding O
of O
hydrogels O

Cotransfection O
of O
expression O
plasmids O
encoding O
the O
protein B
kinase I
A I
inhibitor O
, O
or O
an O
inactive O
protein B
kinase I
A I
( O
PKA B
) O
catalytic B
beta I
subunit O
, O
inhibited O
both O
forskolin O
and O
PACAP B
activation O
of O
chromogranin B
A I
transcription O
, O
revealing O
that O
PACAP B
- O
induced O
trans O
- O
activation O
is O
highly O
dependent O
on O
PKA B
. O

GH B
induced O
rapid O
tyrosine O
phosphorylation O
of O
a O
protein O
at O
approximately O
93 O
kDa O
in O
normal O
fibroblasts O
, O
and O
Western O
blotting O
with O
STAT B
- I
specific I
antibodies I
revealed O
STAT5 B
activation O
( O
phosphorylation O
) O
by O
GH B
. O

The O
most O
valid O
method O
was O
the O
AXB O
bearing O
with O
the O
cephalometric O
clinical O
diagnosis O
a O
90 O
. O
91 O
% O
concordance O
. O

The O
cdc42W97R B
temperature O
- O
sensitive O
allele O
in O
S O
. O
cerevisiae O
displayed O
the O
same O
cell O
- O
division O
- O
cycle O
arrest O
phenotype O
( O
large O
, O
round O
unbudded O
cells O
) O
as O
the O
cdc42 B
- I
1ts I
mutant I
. O

The O
cyanobacterial O
phycobilisome I
is O
a O
large O
protein O
complex O
located O
on O
the O
photosynthetic O
membrane O
. O

One O
day O
after O
injection O
48 O
% O
of O
the O
injected O
activity O
was O
in O
the O
skeleton O
, O
9 O
. O
3 O
% O
in O
the O
liver O
, O
3 O
% O
in O
the O
kidneys O
and O
4 O
. O
4 O
% O
in O
the O
rest O
of O
the O
organs O
. O

OBJECTIVE O
: O
To O
assess O
laboratory O
practice O
in O
the O
examination O
of O
blood O
films O
for O
malarial O
parasites O
. O

BACKGROUND O
: O
A O
thorough O
understanding O
of O
malignant O
fibrous O
histiocytoma O
( O
MFH O
) O
, O
the O
most O
common O
subtype O
of O
soft O
tissue O
sarcoma O
, O
will O
lead O
to O
improved O
histologic O
- O
specific O
protocols O
. O

Sequence O
analyses O
revealed O
a O
partial O
941 O
bp O
cDNA O
that O
encoded O
a O
313 O
- O
amino O
- O
acid O
polypeptide O
. O

Furthermore O
, O
CBP B
/ O
p300 B
stimulated O
both O
TGF B
- I
beta I
- O
and O
Smad B
- O
induced O
transcription O
in O
a O
Smad4 B
/ O
DPC4 B
- O
dependent O
fashion O
. O

Sequences O
within O
and O
flanking O
hypersensitive O
sites O
3 O
and O
2 O
of O
the O
beta B
- I
globin I
locus I
control I
region I
required O
for O
synergistic O
versus O
additive O
interaction O
with O
the O
epsilon B
- I
globin I
gene I
promoter I
. O

Sequence O
analysis O
revealed O
100 O
% O
homology O
of O
all O
RA O
- O
derived O
PTEN B
fragments I
to O
those O
from O
normal O
SF O
as O
well O
as O
to O
the O
published O
GenBank O
sequence O
( O
accession O
number O
U93051 O
) O
. O

174 O
, O
233 O
- O
247 O
) O
and O
Serrate B
( O
Serrate1 B
and I
2 I
; O
Myat O
, O
A O
. O
, O
Henrique O
, O
D O
. O
, O
Ish O
- O
Horowicz O
, O
D O
. O
and O
Lewis O
, O
J O
. O
, O
1996 O
. O

Many O
retroviruses O
, O
including O
the O
human O
and O
simian O
immunodeficiency O
viruses O
, O
contain O
a O
leucine O
zipper O
- O
like O
repeat O
in O
a O
highly O
conserved O
region O
of O
the O
external O
domain O
of O
the O
transmembrane B
( I
TM I
) I
glycoprotein I
. O

Functional O
importance O
of O
a O
properly O
folded O
surface O
loop O
covering O
the O
catalytic O
center O
. O

The O
most O
frequent O
abnormality O
was O
GH B
deficiency O
( O
six O
cases O
) O
, O
followed O
by O
gonadotropin B
( O
four O
cases O
) O
, O
cortisol O
( O
four O
cases O
) O
, O
and O
TSH B
( O
one O
case O
) O
, O
whereas O
four O
patients O
showed O
high O
serum B
PRL I
values O
. O

Recombinant O
vaccinia O
viruses O
that O
express O
the O
bacteriophage B
T3 I
RNA I
polymerase I
( O
VV B
- I
T3pol I
) O
or O
the O
Escherichia B
coli I
lac I
repressor I
( O
VV B
- I
lacI I
) O
under O
control O
of O
the O
early B
- O
late I
vaccinia I
promoter I
P7 I
. I
5 I
were O
constructed O
. O

Internal O
controls O
over O
the O
volume O
of O
milk O
suckled O
do O
not O
appear O
until O
infant O
rats O
are O
about O
2 O
weeks O
of O
age O
at O
which O
time O
gastric O
distension O
, O
milk O
, O
systemic O
dehydration O
, O
and O
intestinal B
hormone I
cholecystokinin I
suppress O
milk O
intake O
derived O
through O
suckling O
. O

Shields O
needed O
for O
pacemakers O
. O

Concerning O
the O
practical O
approach O
in O
a O
clinical O
setting O
, O
it O
has O
to O
be O
pointed O
out O
that O
with O
these O
diseases O
a O
hepatitis O
C O
infection O
has O
to O
be O
considered O
and O
testing O
for O
hepatitis B
C I
antibodies I
and O
, O
if O
positive O
, O
hepatitis B
C I
- I
RNA I
is O
indicated O
. O

Confocal O
fluorescent O
microscopy O
analysis O
demonstrated O
that O
WT O
, O
VaI O
, O
and O
VaII O
all O
distribute O
equally O
to O
the O
cell O
surface O
while O
, O
as O
expected O
, O
a O
WT O
mutant O
lacking O
the O
two O
C O
- O
terminal O
valine O
residues O
does O
not O
. O

However O
, O
it O
is O
not O
easy O
to O
establish O
the O
diagnosis O
of O
thoracic O
MFH O
. O

In O
both O
cell O
types O
, O
insulin B
led O
to O
a O
dose O
- O
dependent O
increase O
in O
the O
association O
of O
tyrosine O
phosphorylated O
IRS B
- I
1 I
with O
the O
SH2 B
domain I
of O
the O
p85 B
regulatory I
subunit I
of O
PI B
- I
3 I
kinase I
, O
and O
also O
increased O
the O
amount O
of O
PI B
kinase I
activity O
detected O
in O
anti B
- I
IRS I
- I
1 I
immunoprecipitates O
. O

The O
" O
Ulm O
Zucker O
Uhr O
System O
" O
comprises O
a O
microdialysis O
probe O
, O
a O
biosensor O
( O
Glucosensor O
Unitec O
Ulm O
s O
. O
c O
. O
) O
, O
a O
sender O
transferring O
telemetrically O
the O
glucose O
concentrations O
, O
and O
a O
receiving O
indicator O
. O

The O
Xenopus B
annexin I
II I
heavy I
chain I
lacks O
the O
highly O
conserved O
tyrosine O
at O
position O
23 O
which O
is O
the O
site O
of O
src B
oncogene I
tyrosine B
kinase I
phosphorylation O
in O
the O
murine O
protein O
. O

No O
bafilomycin O
- O
sensitive O
ATPase B
is O
detected O
in O
a O
vacuolar O
fraction O
. O

Plasmids O
bearing O
the O
P O
. O
stipitis O
URA3 B
gene I
and O
ARS2 O
element O
produced O
more O
than O
30 O
, O
000 O
transformants O
per O
micrograms O
of O
plasmid O
DNA O
. O

Unfractionated O
heparin O
( O
UFH O
) O
given O
at O
a O
dose O
of O
0 O
. O
3 O
mg O
x O
kg O
- O
1 O
bolus O
+ O
0 O
. O
3 O
mg O
x O
kg O
- O
1 O
x O
h O
- O
1 O
infusion O
did O
not O
improve O
the O
incidence O
of O
reperfusion O
or O
lower O
the O
incidence O
of O
reocclusion O
. O

The O
scores O
for O
these O
three O
factors O
, O
which O
corresponded O
to O
recognizable O
dimensions O
of O
depressive O
illness O
, O
were O
then O
correlated O
with O
rCBF O
. O

Cloning O
, O
sequence O
analysis O
, O
and O
expression O
of O
the O
gene B
encoding I
formaldehyde I
dismutase I
from I
Pseudomonas I
putida I
F61 I
. O

However O
, O
at O
the O
time O
of O
salvage O
treatment O
, O
the O
mean O
serum B
PSA I
levels O
were O
9 O
. O
1 O
and O
1 O
. O
1 O
ng O
/ O
mL O
for O
the O
salvage O
RP O
and O
salvage O
RT O
groups O
, O
respectively O
( O
P O
= O
0 O
. O
0001 O
) O
. O

Protease O
digestion O
and O
alkaline O
extraction O
of O
microsomes O
containing O
labeled O
mutant O
proteins O
further O
showed O
that O
segment O
3 O
was O
sufficient O
for O
stable O
membrane O
anchoring O
of O
the O
glycoproteins O
, O
indicating O
that O
this O
segment O
may O
specify O
the O
transmembrane O
domain O
of O
the O
gB B
glycoprotein I
. O

The O
mean O
times O
to O
LNR O
in O
these O
groups O
were O
42 O
. O
0 O
months O
( O
range O
, O
3 O
. O
0 O
- O
194 O
. O
5 O
months O
) O
and O
49 O
. O
0 O
months O
( O
range O
, O
3 O
. O
6 O
- O
209 O
. O
0 O
months O
) O
respectively O
. O

A O
gene O
( O
FMR B
- I
1 I
) O
was O
identified O
within O
a O
four O
cosmid O
contig O
of O
YAC O
DNA O
that O
expresses O
a O
4 O
. O
8 O
kb O
message O
in O
human O
brain O
. O

Detailed O
comparison O
of O
the O
sequences O
of O
cp35 B
and O
human B
calpactin I
II I
shows O
that O
the O
only O
substantial O
sequence O
dissimilarity O
is O
a O
domain O
encoding O
amino O
acids O
between O
residues O
20 O
and O
40 O
which O
includes O
a O
tyrosine O
phosphorylation O
site O
in O
the O
human O
molecule O
, O
along O
with O
other O
residues O
of O
possible O
physiological O
significance O
. O

Exogenous O
heart O
stress O
raised O
log O
circulating O
NE O
concentration O
in O
proportion O
to O
the O
rise O
in O
heart O
rate O
at O
a O
given O
work O
load O
so O
that O
the O
usual O
relationship O
between O
these O
variables O
, O
previously O
observed O
during O
other O
stresses O
, O
was O
preserved O
. O

The O
implication O
of O
Rac B
and O
Cdc42 B
was O
analyzed O
in O
transient O
transfection O
experiments O
using O
either O
constitutively O
active O
( O
V12 O
) O
or O
dominant O
- O
interfering O
( O
N17 O
) O
mutants O
. O

Alternative O
immune B
globulin I
preparation O
when O
standard O
immune B
serum I
globulin I
is O
not O
available O
. O

Plasma B
insulin I
responses O
to O
glucose O
in O
femoral O
, O
hepatic O
, O
and O
pancreatic O
veins O
in O
dogs O
. O

The O
bioconversion O
of O
penicillin O
G O
, O
an O
inexpensive O
substrate O
, O
to O
the O
valuable O
intermediate O
for O
semisynthetic O
cephalosporin O
production O
, O
deacetoxycephalosporin O
G O
( O
DAOG O
) O
, O
had O
been O
recently O
shown O
to O
be O
increased O
by O
eliminating O
agitation O
and O
adding O
decane O
. O

Elevated O
potassium O
stimulation O
of O
GAL B
mRNA I
was O
completely O
blocked O
, O
but O
pituitary B
adenylyl I
cyclase I
- I
activating I
polypeptide I
and O
histamine O
stimulations O
were O
only O
partially O
blocked O
, O
by O
cycloheximide O
. O

In O
lyzozyme B
activity O
there O
was O
periodicity O
in O
three O
groups O
but O
not O
in O
the O
youngest O
foals O
. O

GBF1 B
and O
GBF2 B
mRNA I
is O
present O
in O
light O
and O
dark O
grown O
leaves O
as O
well O
as O
in O
roots O
. O

We O
now O
report O
identification O
and O
analysis O
of O
transcriptional O
activities O
mediated O
by O
three O
cis O
- O
acting O
sites O
within O
a O
90 O
- O
bp O
portion O
of O
the O
rDNA O
enhancer O
designated O
the O
modulator O
region O
. O

A O
porcine O
model O
of O
diffuse O
axonal O
injury O
, O
developed O
with O
information O
from O
these O
physical O
models O
and O
earlier O
in O
vitro O
tissue O
modeling O
studies O
, O
is O
used O
to O
correlate O
histologic O
and O
radiologic O
evidence O
of O
axonal O
injury O
to O
predicted O
regions O
of O
injury O
from O
the O
experimental O
and O
theoretical O
analysis O
. O

Sample O
size O
requirement O
for O
repeated O
measurements O
in O
continuous O
data O
. O

A O
transient O
induction O
of O
both O
c B
- I
fos I
and O
c B
- I
jun I
mRNAs I
by O
TPA O
was O
observed O
in O
both O
cell O
populations O
, O
together O
with O
an O
associated O
suppression O
of O
BSP B
mRNA I
in O
the O
fetal O
rat O
calvarial O
cells O
. O

Videoanalysis O
confirms O
the O
ballistic O
character O
of O
the O
initial O
phase O
of O
the O
reaching O
movement O
. O

The O
isomerization O
of O
the O
pre O
- O
existing O
closed O
complex O
to O
an O
open O
promoter O
form O
, O
as O
judged O
by O
the O
local O
denaturation O
of O
promoter O
DNA O
which O
rendered O
sequences O
from O
+ O
5 O
to O
- O
10 O
reactive O
towards O
KMnO4 O
, O
was O
shown O
to O
be O
fully O
dependent O
on O
NifA B
. O

HPV B
- I
16 I
MARs I
are O
context O
dependent O
transcriptional O
enhancers O
, O
and O
activated O
expression O
of O
HPV B
- I
16 I
oncogenes I
dependent O
on O
chromosomal O
integration O
may O
positively O
select O
tumorigenic O
cells O
during O
the O
multistep O
etiology O
of O
cervical O
cancer O
. O

Sociological O
analysis O
of O
the O
family O
. O

This O
enhancer O
element O
functioned O
in O
a O
position O
- O
and O
orientation O
- O
independent O
manner O
both O
on O
the O
Pax B
- O
QNR O
P0 B
promoter I
and O
the O
heterologous O
thymidine B
kinase I
promoter I
. O

These O
data O
suggest O
that O
overexpression O
of O
MT B
potentiates O
the O
growth O
of O
MCF7 O
cells O
, O
whereas O
downregulation O
of O
MT B
elicits O
antiproliferative O
effects O
. O

A O
multivariant O
study O
of O
pituitary O
adenoma O
, O
obtainment O
of O
two O
logistic O
regression O
equations O
as O
an O
auxiliary O
support O
in O
the O
diagnosis O
of O
these O
tumors O
. O

Bile O
salts O
, O
hormonal O
control O
, O
and O
the O
male O
disadvantage O
. O

Number O
of O
warm O
- O
units O
with O
higher O
rates O
of O
firing O
and O
greater O
thermal O
coefficients O
, O
comparable O
to O
those O
of O
warm O
- O
units O
in O
the O
adult O
, O
gradually O
increased O
with O
growth O
. O

Such O
unique O
recognition O
capabilities O
are O
generated O
with O
minimal O
alterations O
in O
the O
CDR3 B
loops O
of O
the O
TCR B
. O

To O
this O
effect O
, O
the O
present O
survey O
puts O
in O
evidence O
that O
the O
maximum O
delay O
of O
stream O
that O
guarantees O
the O
good O
dimensional O
stability O
of O
these O
class O
A O
alginates O
is O
of O
45 O
minutes O
in O
the O
hot O
and O
humid O
climatic O
tropical O
country O
conditions O
. O

Gene B
1 I
of O
the O
murine O
coronavirus O
, O
MHV O
- O
A59 O
, O
encodes O
approximately O
800 O
kDa O
of O
protein O
products O
within O
two O
overlapping O
open O
reading O
frames O
( O
ORFs O
1a O
and O
1b O
) O
. O

For O
this O
interaction O
, O
the O
C O
- O
terminal O
part O
of O
Sp1 B
and O
the O
N O
terminus O
of O
E2F1 B
, O
a O
domain O
also O
present O
in O
E2F2 B
and O
E2F3 B
but O
absent O
in O
E2F4 B
and O
E2F5 B
, O
were O
essential O
. O

Our O
results O
suggest O
a O
biological O
role O
for O
Elf B
- I
1 I
in O
the O
regulation O
of O
IgH B
gene I
expression O
, O
attribute O
a O
functional O
role O
for O
receptor O
- O
induced O
AP B
- I
1 I
proteins I
in O
B O
lymphocytes O
and O
provide O
evidence O
for O
a O
direct O
link O
between O
IgM B
receptor O
- O
mediated O
signalling O
and O
3 O
' O
enhancer O
activation O
. O

Oncogenic O
activation O
of O
RET B
is O
detected O
in O
human O
papillary O
thyroid O
tumours O
and O
in O
multiple O
endocrine O
neoplasia O
type O
2 O
syndromes O
. O

CD4 B
degradation O
mediated O
by O
Vpu B
does O
not O
require O
the O
ER O
chaperone O
calnexin B
and O
is O
dependent O
on O
an O
intact O
ubiquitin B
- O
conjugating O
system O
. O

The O
maternal B
par I
genes I
and O
the O
segregation O
of O
cell O
fate O
specification O
activities O
in O
early O
Caenorhabditis O
elegans O
embryos O
. O

These O
effects O
were O
antagonized O
by O
prior O
administration O
of O
1 O
- O
( O
2 O
- O
methoxyphenyl O
) O
- O
4 O
- O
[ O
- O
( O
2 O
- O
phthalimido O
) O
butyl O
] O
piperazine O
) O
( O
0 O
. O
5 O
mg O
/ O
kg O
, O
i O
. O
p O
. O
) O
. O

PURPOSE O
: O
To O
evaluate O
the O
effect O
of O
adding O
a O
pulsating O
bristle O
action O
to O
the O
established O
oscillating O
/ O
rotating O
action O
of O
the O
Braun O
Oral O
- O
B O
Ultra O
Plaque O
Remover O
( O
D9 O
) O
on O
plaque O
removal O
. O

Detailed O
analysis O
of O
the O
predicted O
amino O
acid O
sequence O
revealed O
sequence O
elements O
which O
are O
conserved O
in O
many O
DNA B
and I
RNA I
polymerases I
. O

Sub O
- O
inhibitory O
and O
post O
- O
antibiotic O
effects O
of O
spiramycin O
and O
erythromycin O
on O
Staphylococcus O
aureus O
. O

The O
CydDC B
system O
appears O
to O
be O
the O
first O
prokaryotic O
example O
of O
a O
heterodimeric O
ABC B
transport O
system O
in O
which O
each O
polypeptide O
contains O
both O
hydrophobic O
and O
ATP O
- O
binding O
domains O
. O

The O
single O
- O
copy O
U20 B
sequence I
is O
located O
on O
the O
same O
DNA O
strand O
as O
the O
nucleolin B
mRNA I
. O

Gel O
shift O
and O
southwestern O
experiments O
revealed O
nuclear O
proteins O
of O
43 O
kDa O
and O
30 O
kDa O
in O
GC O
and O
fish O
cells O
, O
respectively O
, O
that O
bind O
specifically O
to O
the O
tGH O
CRE O
, O
suggesting O
the O
involvement O
of O
CRE B
- I
binding I
- I
protein I
/ O
activating B
- I
transcription I
- I
factor I
- I
l I
- I
related I
peptides I
in O
cAMP O
response O
. O

The O
coding O
sequence O
of O
rat B
MEK I
kinase I
1 I
( O
MEKK1 B
) O
has O
been O
determined O
from O
multiple O
, O
independent O
cDNA O
clones O
. O

In O
response O
to O
the O
need O
for O
better O
quantitative O
estimates O
of O
the O
regional O
distribution O
of O
the O
active O
bone O
marrow O
organ O
in O
infants O
and O
children O
, O
a O
method O
using O
various O
anatomical O
data O
has O
been O
developed O
. O

We O
compared O
these O
responses O
for O
six O
hatchling O
and O
eight O
adult O
Chrysemys O
picta O
from O
an O
Ohio O
population O
. O

On O
the O
basis O
of O
experiments O
with O
mutant O
virus O
and O
transfection O
with O
isolated O
genes O
, O
the O
herpes B
simplex I
virus I
immediate I
- I
early I
gene I
product I
ICP4 B
is O
known O
to O
positively O
regulate O
the O
transcription O
of O
viral B
early O
and O
late O
genes O
and O
negatively O
regulate O
expression O
from O
its O
own O
promoter O
. O

A O
female O
infant O
with O
isolated O
noncompaction O
of O
ventricular O
myocardium O
who O
developed O
ventricular O
tachyarrhythmia O
is O
described O
. O

Recent O
, O
highly O
visible O
reviews O
on O
the O
subject O
have O
clearly O
pointed O
out O
that O
little O
is O
known O
about O
the O
cellular O
mechanisms O
through O
which O
inhaled O
asbestos O
fibers O
cause O
the O
well O
known O
, O
debilitating O
lung O
disease O
- O
asbestosis O
( O
i O
. O
e O
. O
interstitial O
pulmonary O
fibrosis O
) O
. O

The O
POU B
- I
specific I
domain I
of O
Pit B
- I
1 I
is O
essential O
for O
sequence O
- O
specific O
, O
high O
affinity O
DNA O
binding O
and O
DNA O
- O
dependent O
Pit B
- I
1 I
- O
Pit B
- I
1 I
interactions O
. O

Also O
, O
the O
placebo O
nondepressed O
patients O
had O
significantly O
better O
treatment O
outcome O
compared O
with O
the O
placebo O
depressed O
patients O
. O

Obtaining O
kidney O
cells O
from O
a O
beagle O
puppy O
( O
RPB O
- O
1 O
) O
and O
the O
establishment O
of O
a O
cryopreservation O
bank O

These O
results O
suggest O
an O
essential O
role O
of O
the O
binding O
of O
HNF B
- I
4 I
and O
/ O
or O
HNF B
- I
4 I
- I
related I
nuclear I
factors I
to O
the O
6 O
beta O
A O
- O
A O
site O
on O
the O
basal O
transcriptional O
activation O
of O
the O
CYP3A2 B
gene I
in O
liver O
cells O
. O

It O
is O
envisaged O
that O
the O
WHOQOL O
- O
BREF O
will O
be O
most O
useful O
in O
studies O
that O
require O
a O
brief O
assessment O
of O
quality O
of O
life O
, O
for O
example O
, O
in O
large O
epidemiological O
studies O
and O
clinical O
trials O
where O
quality O
of O
life O
is O
of O
interest O
. O

Restriction O
mapping O
showed O
that O
the O
two O
recombinant O
plasmids O
shared O
an O
EcoRI B
fragment I
of O
8 O
. O
9 O
kb O
. O

Usefulness O
of O
glucocorticoids O
in O
the O
treatment O
of O
congestive O
heart O
failure O

The O
chromosomal O
location O
of O
these O
YACs O
was O
verified O
using O
FISH O
, O
which O
also O
demonstrated O
their O
nonchimeric O
nature O
. O

After O
first O
strand O
cDNA O
synthesis O
from O
fetal O
brain O
mRNAs O
, O
short O
fragment O
cDNAs O
( O
sf O
- O
cDNAs O
) O
were O
synthesized O
with O
a O
two O
- O
step O
amplification O
system O
by O
use O
of O
our O
modified O
Degenerate O
Oligonucleotide O
Primed O
Shuttle O
Polymerase O
Chain O
Reaction O
( O
DOP O
- O
Shuttle O
- O
PCR O
) O
method O
. O

For O
evaluation O
of O
the O
model O
, O
simulations O
of O
physiological O
excitation O
and O
of O
pathologies O
( O
Wolff O
- O
Parkinson O
- O
White O
syndrome O
, O
complete O
AV O
- O
block O
, O
inferior O
wall O
ischaemia O
) O
were O
examined O
. O

Profilins B
IIa I
and I
IIb I
are O
also O
present O
in O
humans O
, O
suggesting O
that O
all O
mammals O
have O
three O
profilin B
isoforms I
. O

Using O
the O
yeast O
two O
- O
hybrid O
system O
, O
we O
have O
isolated O
an O
835 B
amino I
acid I
RING I
finger I
( I
C3HC4 I
zinc I
finger I
) I
protein I
, O
TIF1 B
beta I
( O
also O
named O
KAP B
- I
1 I
) O
, O
that O
specifically O
interacts O
with O
the O
KRAB B
domain I
of O
the O
human O
zinc B
finger I
factor I
KOX1 I
/ O
ZNF10 B
. O

The O
authors O
report O
about O
the O
exeresis O
of O
a O
pyelonephrictic O
left O
small O
kidney O
with O
laparoscopic O
surgery O
. O

New O
protein O
- O
free O
products O
for O
diet O
therapy O
in O
chronic O
renal O
insufficiency O

Unestablished O
quail O
myoblasts O
were O
infected O
with O
a O
retroviral O
vector O
encoding O
the O
oncogenic O
form O
of O
H B
- I
Ras I
in O
order O
to O
investigate O
the O
mechanism O
by O
which O
this O
oncoprotein O
interferes O
with O
terminal O
differentiation O
. O

Early O
detection O
and O
signs O
of O
hepatoangiosarcoma O
among O
vinyl O
chloride O
workers O
. O

The O
reduced O
expression O
caused O
by O
multimerization O
of O
UAS2 O
in O
the O
native O
promoter O
was O
observed O
only O
in O
the O
presence O
of O
ADR1 B
. O

VP4 B
differentially O
regulates O
TRAF2 B
signaling O
, O
disengaging O
JNK B
activation O
while O
directing O
NF B
- I
kappa I
B I
to O
effect O
rotavirus O
- O
specific O
cellular O
responses O
. O

On O
the O
3rd O
day O
after O
ischemia O
of O
the O
remained O
kidney O
for O
30 O
min O
, O
structural O
components O
of O
the O
walls O
of O
the O
glomerular O
arterioles O
and O
those O
of O
the O
filtration O
- O
reabsorption O
barrier O
undergo O
certain O
ultrastructural O
changes O
, O
that O
with O
time O
elapsed O
( O
7 O
, O
14 O
days O
) O
gradually O
pass O
away O
, O
and O
amount O
of O
cells O
with O
hypertrophic O
processes O
increases O
. O

The O
purified O
RAG1 B
protein I
overexpressed O
in O
E O
. O
coli O
exhibited O
the O
expected O
cleavage O
activity O
when O
combined O
with O
RAG2 B
purified O
from O
transfected O
293T O
cells O
. O

19 O
, O
4967 O
- O
4973 O
) O
. O

Preventing O
the O
heterosexual O
spread O
of O
HIV O
into O
this O
vulnerable O
population O
is O
a O
formidable O
public O
health O
challenge O
. O

Moreover O
, O
we O
show O
that O
GATA B
- I
1 I
and O
Sp1 B
synergize O
from O
a O
distance O
in O
constructs O
designed O
to O
mimic O
the O
architecture O
of O
globin B
locus I
control I
regions I
and O
downstream O
globin B
promoters I
. O

Superovulation O
with O
intrauterine O
insemination O
( O
SO O
- O
IUI O
) O
has O
been O
suggested O
as O
an O
alternative O
to O
gamete O
intrafallopian O
transfer O
( O
GIFT O
) O
, O
despite O
the O
absence O
of O
controlled O
or O
comparative O
trials O
. O

Mammalian O
chromosome O
ends O
contain O
long O
arrays O
of O
TTAGGG O
repeats O
that O
are O
complexed O
to O
a O
telomere O
specific O
protein O
, O
the O
TTAGGG B
repeat I
binding I
factor I
, O
TRF1 B
. O

Genotype O
- O
phenotype O
correlations O
in O
male O
patients O
with O
a O
partial O
nullisomy O
of O
the O
X O
chromosome O
have O
suggested O
that O
at O
least O
one O
locus O
involved O
in O
MRX B
is O
on O
Xp22 O
. O
3 O
. O

Mean O
disposition O
constants O
( O
+ O
/ O
- O
SD O
) O
were O
obtained O
from O
individualized O
fits O
( O
V1 O
: O
0 O
. O
398 O
+ O
/ O
- O
0 O
. O
336 O
LITER O
/ O
KG O
, O
Vdarea O
: O
2 O
. O
53 O
+ O
/ O
- O
0 O
. O
72 O
liter O
/ O
kg O
, O
alpha O
: O
0 O
. O
316 O
+ O
/ O
- O
0 O
. O
294 O
min O
- O
1 O
, O
beta O
: O
0 O
. O
00204 O
+ O
/ O
- O
0 O
. O
00262 O
min O
- O
1 O
, O
k2 O
: O
0 O
. O
0305 O
+ O
/ O
- O
0 O
. O
0101 O
min O
- O
1 O
) O
. O

The O
gene O
, O
from O
the O
initiator O
methionine O
to O
the O
polyadenylation O
site O
, O
is O
contained O
within O
13 O
244 O
basepairs O
and O
contains O
19 O
exons O
. O

In O
the O
unit O
housing O
of O
a O
compact O
cyclotron O
and O
positron O
emission O
CT O
( O
PET O
) O
, O
positron O
emitting O
gas O
such O
as O
15O O
, O
11C O
, O
C15O2 O
, O
C15O O
etc O
. O

is O
supplied O
from O
a O
cyclotron O
to O
a O
PET O
room O
through O
a O
transportation O
pipe O
with O
an O
appropriate O
shield O
to O
reduce O
positron O
annihilation O
radiation O
. O

Adhesion O
was O
inhibited O
by O
mAbs O
against O
the O
COOH O
- O
terminus O
and O
central O
cell O
binding O
domains O
of O
fibronectin B
, O
as O
well O
as O
by O
the O
corresponding O
CS1 B
and O
RGD O
peptides O
. O

These O
results O
were O
not O
obtained O
using O
either O
preimmune O
sera O
or O
antiserum O
specific O
for O
the O
luminal O
portion O
of O
the O
glycoprotein O
precursor O
. O

Cerebral O
blood O
flow O
was O
measured O
using O
the O
iv O
method O
of O
133 O
- O
Xe O
CBF O
determination O
and O
AVDO2 O
was O
measured O
using O
systemic O
arterial O
- O
jugular O
venous O
oxygen O
content O
differences O
. O

Activation O
of O
the O
mitogen B
- I
activated I
protein I
kinase I
( O
MAPK B
) O
pathway O
enhances O
long O
- O
range O
transactivation O
by O
the O
beta B
- I
globin I
locus I
control I
region I
( O
LCR B
) O
( O
W O
. O

In O
situ O
hybridization O
of O
whole O
- O
mount O
mouse O
embryos O
with O
Mex5 B
antisense I
RNA I
provide O
no O
evidence O
for O
the O
exclusion O
of O
Mex5 B
during O
embryonic O
development O
. O

In O
three O
other O
patients O
the O
electrophysiologic O
characteristics O
of O
atrioventricular O
conduction O
prevented O
a O
demonstration O
of O
these O
differences O
. O

Human O
psychophysical O
analysis O
of O
receptive O
field O
- O
like O
properties O
- O
- O
II O
. O

3 O
. O

New O
strategies O
are O
needed O
to O
prevent O
coronary O
rethrombosis O
in O
patients O
with O
minimal O
atherosclerosis O
after O
thrombolytic O
therapy O
for O
acute O
myocardial O
infarction O
. O

The O
Ick B
protein I
tyrosine I
kinase I
is O
not O
involved O
in O
antibody O
- O
mediated O
CD4 B
( O
CDR3 B
- I
loop I
) O
signal O
transduction O
that O
inhibits O
HIV O
- O
1 O
transcription O
. O

Programmed O
peritoneal O
lavage O
in O
suppurative O
complications O
of O
perforated O
ulcer O
of O
the O
stomach O

Deletion O
analysis O
of O
the O
7 B
. I
3 I
- I
kb I
GATA I
- I
2 I
promoter I
region I
revealed O
that O
a O
1 O
. O
1 O
- O
kb O
DNA O
sequence O
is O
critical O
for O
expression O
of O
GATA B
- I
2 I
in O
neurons O
. O

The O
model O
therefore O
predicts O
that O
1 O
) O
on O
- O
line O
calculation O
of O
airway O
dead O
space O
and O
end O
- O
expired O
lung O
volume O
can O
be O
made O
by O
the O
addition O
of O
an O
oxygen O
sine O
- O
wave O
perturbation O
component O
to O
the O
mean O
FIO2 O
; O
and O
( O
2 O
) O
QS O
/ O
QT O
can O
be O
measured O
from O
the O
resultant O
oxygen O
perturbation O
sine O
- O
wave O
amplitudes O
in O
the O
expired O
gas O
and O
in O
arterial O
and O
mixed O
- O
venous O
blood O
and O
is O
independent O
of O
the O
mean O
blood O
oxygen O
partial O
pressure O
and O
oxyhemoglobin O
saturation O
values O
. O

Consistent O
with O
this O
, O
rh5 B
expression O
in O
R8 O
disappears O
when O
R7 O
cells O
are O
absent O
( O
in O
sevenless O
mutant O
) O
. O

The O
fusion O
gene O
cassette O
was O
placed O
under O
the O
control O
of O
a O
vaccinia B
virus I
early I
promoter I
and O
cloned O
in O
a O
host O
- O
restricted O
fowlpox O
viral O
vector O
. O

Psoriasis O
on O
tumor O
. O

Fulminant O
hepatic O
failure O
in O
these O
cases O
could O
be O
characterized O
by O
: O
( O
1 O
) O
rapid O
decrease O
in O
serum B
alanine I
transaminase I
( O
ALT B
) O
level O
after O
discontinuation O
of O
ecarazine O
, O
( O
2 O
) O
prolonged O
jaundice O
despite O
discontinuation O
of O
ecarazine O
, O
( O
3 O
) O
high O
incidence O
of O
anti B
- I
nuclear I
antibody I
( O
ANA B
) O
( O
57 O
% O
) O
, O
and O
( O
4 O
) O
histological O
findings O
of O
extensive O
hepatocellular O
necrosis O
ranging O
from O
bridging O
necrosis O
to O
massive O
necrosis O
. O

These O
data O
suggest O
that O
the O
secreted O
, O
truncated O
receptor O
encoded O
by O
the O
2 B
. I
6 I
- I
kb I
c I
- I
erbB I
transcript I
can O
bind O
to O
TGF B
alpha I
and O
may O
play O
an O
important O
growth O
- O
regulatory O
function O
in O
vitro O
. O

Localization O
of O
the O
porcine O
enzyme O
in O
the O
endoplasmic O
reticulum O
is O
consistent O
with O
immuno O
- O
electron O
- O
microscopic O
studies O
using O
pig O
hepatocytes O
. O

A O
close O
homologue O
of O
the O
APK2a B
gene I
, O
named O
APK2b B
, O
was O
also O
isolated O
from O
the O
Arabidopsis O
cDNA O
library O
. O

Thus O
, O
the O
rates O
of O
a B
- I
factor I
receptor I
endocytosis O
and O
consequent O
vacuolar O
turnover O
depend O
on O
the O
available O
level O
of O
ubiquitin B
in O
the O
cell O
. O

Using O
the O
same O
approach O
we O
have O
shown O
that O
hFIRE B
binds O
the O
stimulatory O
proteins O
Sp1 B
and O
Sp3 B
in O
addition O
to O
CBF B
. O

Non O
- O
specific O
immunological O
abnormalities O
were O
detected O
in O
8 O
of O
the O
13 O
patients O
in O
whom O
they O
were O
looked O
for O
. O

Using O
a O
combined O
pharmacokinetic O
- O
pharmacodynamic O
model O
, O
the O
impact O
of O
various O
factors O
on O
the O
effective O
bioavailability O
and O
on O
its O
estimation O
, O
using O
the O
intravenous O
- O
to O
- O
oral O
dose O
ratio O
required O
to O
produce O
the O
same O
area O
under O
the O
response O
time O
curve O
after O
acute O
administration O
, O
are O
explored O
. O

CONCLUSIONS O
: O
The O
RRM O
is O
one O
of O
the O
most O
common O
and O
best O
characterized O
RNA O
- O
binding O
motifs O
. O

The O
cyclin B
- I
dependent I
kinase I
Cdk2 B
associates O
with O
cyclins B
A I
, I
D I
, I
and I
E I
and O
has O
been O
implicated O
in O
the O
control O
of O
the O
G1 O
to O
S O
phase O
transition O
in O
mammals O
. O

Mol O
. O

The O
amino O
- O
terminal O
gag B
- I
encoded I
region I
of O
P140gag B
- O
fps B
contains O
a O
phosphotyrosine O
residue O
in O
addition O
to O
normal O
gag B
phosphorylation O
sites O
. O

Rad18 B
is O
required O
for O
DNA O
repair O
and O
checkpoint O
responses O
in O
fission O
yeast O
. O

A O
less O
common O
and O
much O
more O
perplexing O
circumstance O
occurs O
when O
evaluating O
patients O
with O
sarcoidosis O
established O
for O
several O
years O
present O
with O
evidence O
of O
progressive O
or O
chronic O
pulmonary O
involvement O
and O
dyspnea O
. O

2 O
, O
4 O
- O
and O
2 O
, O
6 O
- O
toluenediamine O
in O
hydrolysed O
plasma O
and O
urine O
after O
test O
- O
chamber O
exposure O
of O
humans O
to O
2 O
, O
4 O
- O
and O
2 O
, O
6 O
- O
toluene O
diisocyanate O
. O

Further O
studies O
are O
warranted O
to O
determine O
whether O
these O
findings O
are O
idiosyncratic O
, O
coincidental O
, O
or O
a O
more O
general O
phenomenon O
. O

After O
1 O
day O
, O
none O
of O
the O
labeled O
erythrocytes O
were O
detected O
. O

This O
substrate O
was O
cleaved O
efficiently O
in O
trans O
by O
protease B
3C I
derived O
from O
another O
recombinant O
vaccinia O
virus O
expressing O
a O
3C B
precursor I
protein I
. O

The O
activity O
of O
gamma B
- I
glutamyl I
transpeptidase I
in O
the O
serum O
of O
problem O
drinkers O
. O

Patients O
with O
endocarditis O
or O
vascular O
infection O
were O
more O
frequently O
immunocompromised O
and O
older O
than O
those O
with O
acute O
Q O
fever O
. O

The O
effects O
of O
oxalate O
- O
containing O
products O
on O
the O
exposed O
dentine O
surface O
: O
an O
SEM O
investigation O
. O

The O
technique O
used O
by O
this O
system O
involves O
a O
transient O
, O
externally O
applied O
increase O
in O
resistance O
to O
breathing O
. O

We O
evaluated O
99mTc O
- O
ECD O
SPECT O
comparing O
with O
rCBF O
images O
obtained O
by O
PET O
in O
12 O
patients O
with O
spinocerebellar O
degeneration O
( O
SCD O
) O
. O

Monoclonal O
antibodies O
to O
the O
hapten O
phenyloxazolone O
were O
raised O
7 O
days O
after O
immunization O
in O
mice O
of O
six O
strains O
( O
BALB O
/ O
c O
, O
C57BL O
- O
Igha O
, O
DBA2 O
, O
RF O
, O
A O
/ O
J O
, O
and O
CE O
) O
. O

In O
particular O
, O
lacZ B
transcripts I
synthesised O
this O
way O
are O
highly O
unstable O
and O
yield O
little O
beta B
- I
galactosidase I
. O

Six O
cDNAs O
represent O
human O
homologs O
of O
genes O
known O
in O
other O
species O
, O
namely O
, O
mouse B
HSPE71 I
, O
Rat B
RhoGAP I
protein I
, O
S B
cerevisiae I
leucyl I
tRNA I
synthetase I
and O
S B
cerevisiae I
chromosome I
II I
ORF I
YBLO44W I
. O

The O
thalamic O
SD O
was O
regularly O
triggered O
by O
short O
( O
0 O
. O
02 O
- O
0 O
. O
05 O
s O
) O
high O
- O
frequency O
( O
200 O
- O
500 O
Hz O
) O
ES O
of O
the O
parietal O
cortical O
surface O
. O

All O
three O
SSV O
- O
transformed O
cells O
secreted O
v B
- I
sis I
gene I
product I
( O
p44 B
) O
. O

p44 B
was O
secreted O
but O
remained O
tightly O
associated O
with O
the O
cell O
surface O
. O

It O
is O
suggested O
that O
the O
mechanism O
of O
intestinal O
absorption O
of O
vitamin O
D O
and O
25 O
- O
hydroxy O
- O
vitamin O
D O
may O
differ O
in O
man O
, O
the O
absorption O
of O
25 O
- O
hydroxy O
- O
vitamin O
D O
possibly O
being O
less O
dependent O
on O
bile O
acids O
. O

To O
determine O
the O
role O
of O
elevated O
FAK B
expression O
in O
facilitating O
epidermal B
growth I
factor I
( O
EGF B
) O
- O
stimulated O
human O
adenocarcinoma O
( O
A549 O
) O
cell O
motility O
, O
antisense O
oligonucleotides O
were O
used O
to O
reduce O
FAK B
protein I
expression O
> O
75 O
% O
. O

It O
tends O
to O
be O
higher O
between O
frazioni O
of O
the O
same O
community O
than O
between O
communities O
. O

The O
BRSV B
L I
gene I
is O
6573 O
nt O
in O
length O
and O
the O
derived O
polypeptide O
has O
2162 O
aa O
. O

To O
further O
study O
the O
role O
of O
tTG B
in O
liver O
disease O
, O
we O
initiated O
investigations O
into O
the O
effect O
of O
a O
proinflammatory O
mediator O
, O
tumor B
necrosis I
factor I
( I
TNF I
) I
- I
alpha I
, O
on O
tTG B
activity O
in O
cultured O
liver O
cells O
. O

Availability O
of O
a O
less O
palatable O
diet O
( O
chow O
) O
following O
presentation O
of O
palatable O
diets O
will O
not O
result O
in O
diminished O
caloric O
intake O
, O
body O
weight O
, O
obesity O
and O
hyperinsulinemia O
. O

By O
contrast O
, O
CIS O
failed O
to O
affect O
the O
IL B
- I
9 I
response O
. O

METHODS O
AND O
RESULTS O
: O
With O
an O
array O
of O
112 O
unipole O
, O
epicardial O
maps O
of O
electrically O
induced O
AF O
in O
6 O
dogs O
( O
acute O
group O
) O
, O
self O
- O
sustained O
AF O
in O
6 O
dogs O
( O
chronic O
group O
) O
, O
and O
sinus O
rhythm O
and O
atrial O
pacing O
in O
3 O
dogs O
( O
control O
group O
) O
were O
analyzed O
before O
and O
after O
creating O
linear O
radiofrequency O
ablation O
lesions O
in O
both O
atria O
that O
eliminated O
the O
AF O
. O

In O
response O
to O
phosphorus O
limitation O
, O
the O
fungus O
Neurospora O
crassa O
synthesizes O
a O
number O
of O
enzymes O
that O
function O
to O
bring O
more O
phosphate O
into O
the O
cell O
. O

The O
effects O
of O
inoculum O
size O
, O
medium O
, O
temperature O
, O
and O
duration O
of O
growth O
on O
the O
in O
vitro O
susceptibility O
testing O
of O
Aspergillus O
fumigatus O
were O
investigated O
using O
broth O
micro O
- O
and O
macro O
- O
dilution O
techniques O
. O

We O
have O
analyzed O
the O
function O
of O
these O
putative O
SL O
structures O
in O
RNA O
translation O
by O
constructing O
chimeric O
chloramphenicol B
acetyltransferase I
( O
CAT B
) O
RNAs O
, O
flanked O
either O
by O
both O
5 O
' O
- O
and O
3 O
' O
- O
terminal O
sequence O
domains O
from O
the O
RV O
genome O
or O
several O
deletion O
derivatives O
of O
the O
same O
sequences O
. O

Periarteritis O
nodosa O
with O
ruptures O
of O
the O
renal O
vessels O
and O
the O
persistence O
of O
the O
hepatitis O
B O
and O
C O
viruses O
in O
the O
blood O

Despite O
the O
fact O
that O
biochemical O
estimates O
of O
bone O
turnover O
indicate O
that O
( O
short O
- O
term O
) O
administration O
of O
rhGH B
and O
IGF B
- I
I I
stimulates O
bone O
metabolism O
in O
non O
- O
osteoporotic O
older O
people O
, O
no O
significant O
changes O
have O
been O
observed O
in O
bone O
mineral O
density O
at O
the O
proximal O
femur O
. O

Compression O
of O
pulmonary O
artery O
and O
right O
ventricular O
outflow O
tract O
by O
aneurysm O
of O
ascending O
aorta O
. O

However O
, O
patients O
with O
DPX O
should O
be O
observed O
for O
the O
potential O
occurrence O
of O
an O
associated O
condition O
. O

Blood O
pressure O
was O
controlled O
long O
term O
( O
with O
/ O
without O
diuretics O
/ O
beta B
- I
adrenoreceptor I
blocking O
drugs O
) O
in O
sixteen O
out O
of O
nineteen O
patients O
with O
mild O
- O
moderate O
hypertension O
. O

However O
, O
FosB B
- I
L I
and O
FosB B
- I
S I
do O
not O
differ O
in O
all O
trans O
- O
regulatory O
properties O
: O
Trans O
- O
activation O
of O
a O
5x O
TRE B
- O
CAT B
reporter O
construct O
in O
HeLa O
and O
NIH O
- O
3T3 O
cells O
was O
found O
with O
both O
FosB B
forms O
. O

Mutations O
in O
glnB B
, O
nifR1 B
( O
ntrC B
) O
, O
and O
NifR4 B
( O
ntrA B
encoding O
sigma B
54 I
) O
had O
no O
influence O
on O
put B
gene I
expression O
. O

Disruption O
of O
INP53 B
, O
but O
not O
the O
related O
INP51 B
and O
INP52 B
genes I
, O
resulted O
in O
alpha B
- I
factor I
maturation O
defects O
and O
exacerbated O
alpha B
- I
factor I
maturation O
defects O
when O
combined O
with O
chc1 B
- I
521 I
. O

Impact O
of O
cytokine O
gene O
polymorphisms O
on O
outcomes O
of O
coronary O
artery O
bypass O
graft O
surgery O
. O

Regardless O
of O
muscle O
tensioning O
or O
marination O
treatments O
, O
aging O
of O
the O
carcass O
for O
24 O
h O
( O
T1 O
, O
T2 O
, O
and O
T3 O
) O
produced O
meats O
with O
lower O
shear O
values O
than O
those O
from O
hot O
- O
boned O
carcasses O
( O
T4 O
and O
T5 O
) O
. O

Blood O
and O
whole O
- O
brain O
mercury O
concentrations O
were O
determined O
in O
pups O
on O
PN O
0 O
( O
birth O
) O
and O
PN O
21 O
( O
weaning O
) O
. O

By O
subtractive O
and O
differential O
screening O
, O
we O
have O
cloned O
12 O
of O
these O
sequences O
, O
2 O
of O
which O
were O
c B
- I
fos I
and O
krox B
- I
24 I
. O

When O
stratifying O
FLM O
values O
< O
34 O
and O
> O
34 O
wks O
' O
gestation O
, O
again O
no O
difference O
was O
found O
in O
mean O
difference O
of O
L O
/ O
S O
and O
LB O
before O
and O
after O
34 O
wks O
. O

Cotransfection O
of O
a O
junB B
stimulated O
the O
basal O
activity O
of O
the O
alpha B
2 I
( I
I I
) I
collagen I
promoter I
93 O
- O
fold O
, O
respectively O
. O

Glutathione B
reductase I
( O
GR B
) O
was O
purified O
from O
the O
cyanobacterium O
Anabaena O
PCC O
7120 O
. O

Additional O
simulations O
account O
for O
interactions O
of O
spatial O
frequency O
with O
stimulus O
duration O
, O
effects O
of O
adaptation O
, O
and O
properties O
of O
residual O
traces O
, O
as O
opposed O
to O
visual O
persistence O
. O

Fractional O
Ca O
retention O
was O
measured O
from O
the O
72 O
- O
h O
postdose O
WBR O
divided O
by O
WBR O
at O
time O
0 O
. O

METHODS O
: O
The O
study O
population O
included O
70 O
patients O
with O
acute O
coronary O
syndromes O
( O
14 O
with O
recent O
acute O
myocardial O
infarction O
and O
56 O
with O
unstable O
angina O
pectoris O
) O
, O
105 O
patients O
with O
stable O
angina O
pectoris O
, O
and O
75 O
control O
subjects O
. O

Furthermore O
, O
rubella B
IgM I
antibody I
was O
never O
detected O
after O
immunization O
in O
women O
who O
were O
HAI B
- O
negative O
and O
LA O
- O
positive O
during O
pregnancy O
. O

Disease O
- O
free O
interval O
was O
greater O
in O
node O
- O
negative O
Lx O
patients O
for O
both O
T1 O
( O
P O
less O
than O
0 O
. O
007 O
) O
and O
T2 O
( O
P O
less O
than O
0 O
. O
05 O
) O
presentations O
. O

5 O
' O
- O
Deletion O
of O
the O
Stat B
- I
binding I
sites I
abolished O
all O
promoter O
- O
reporter O
activity O
in O
response O
to O
PRL B
. O

A O
review O
of O
156 O
patients O
younger O
than O
40 O
treated O
at O
our O
Department O
between O
1960 O
and O
1991 O
with O
transitional O
cell O
carcinoma O
of O
the O
bladder O
revealed O
that O
89 O
. O
1 O
% O
had O
superficial O
( O
Ta O
/ O
T1 O
) O
disease O
and O
the O
remaining O
10 O
. O
9 O
% O
presented O
with O
invasive O
disease O
. O

Plasma B
beta I
- I
thromboglobulin I
( O
beta B
- I
TG I
) O
, O
a O
platelet O
- O
specific O
protein O
, O
is O
a O
marker O
of O
intravascular O
platelet O
degranulation O
. O

John O
' O
s O
wort O

DESIGN O
, O
SETTING O
AND O
SUBJECTS O
: O
The O
analysis O
is O
based O
on O
data O
collected O
in O
the O
first O
two O
rounds O
of O
the O
nationally O
representative O
Ghana O
Living O
Standards O
Survey O
, O
held O
in O
1987 O
/ O
88 O
( O
GLSS O
- O
I O
) O
and O
1988 O
/ O
89 O
( O
GLSS O
- O
II O
) O
, O
with O
both O
surveys O
covering O
approximately O
3000 O
households O
. O

The O
DNA O
- O
binding O
proteins O
are O
without O
effect O
on O
the O
transcription O
of O
plasmids O
lacking O
binding O
sites O
or O
when O
the O
binding O
sites O
are O
located O
further O
upstream O
. O

The O
Gastrointestinal O
Tumor O
Study O
Group O
( O
GITSG O
) O
protocol O
GI O
- O
7175 O
randomized O
227 O
patients O
between O
1975 O
and O
1980 O
following O
complete O
surgical O
resection O
of O
stages O
B2 O
and O
C O
rectal O
adenocarcinoma O
to O
four O
treatment O
arms O
: O
( O
1 O
) O
no O
adjuvant O
therapy O
, O
( O
2 O
) O
chemotherapy O
only O
, O
( O
3 O
) O
radiotherapy O
only O
, O
and O
( O
4 O
) O
radiotherapy O
and O
chemotherapy O
( O
combined O
modality O
) O
. O

The O
RTI40 B
gene I
spans O
35 O
kilobase O
pairs O
; O
it O
contains O
6 O
exons O
and O
at O
least O
6 O
rat O
Identifier O
repetitive O
elements O
. O

The O
DNA O
element O
through O
which O
EBNA B
- I
3C I
activates O
the O
LMP B
- I
1 I
promoter I
includes O
a O
Spi B
- I
1 I
/ O
Spi B
- I
B I
binding O
site O
, O
previously O
characterized O
as O
an O
important O
EBNA B
- I
2 I
response I
element I
. O

DP B
- I
1 I
and O
DP B
- I
2 I
encode O
maternally O
stored O
transcripts O
that O
are O
expressed O
during O
early O
development O
. O

Furthermore O
, O
deletion O
of O
two O
potential O
Ste11 B
recognition I
sites I
in O
the O
fus1 B
promoter I
region I
abolished O
transcription O
, O
and O
expression O
could O
be O
restored O
when O
we O
inserted O
a O
different O
Ste11 B
site I
from O
the O
mat1 B
- I
P I
promoter I
. O

However O
, O
an O
analogous O
myristoylated O
peptide O
derived O
from O
c B
- I
Yes I
also O
has O
no O
inhibitory O
activity O
. O

The O
oral O
movements O
which O
did O
occur O
in O
the O
HAL O
- O
treated O
rats O
were O
slower O
than O
normal O
. O

Finally O
, O
we O
demonstrate O
that O
animals O
carrying O
a O
snf B
mutation I
that O
converts O
SNF B
from O
a O
bifunctional O
protein O
to O
a O
U1 B
snRNP I
- I
specific I
protein I
are O
viable O
. O

These O
sequence O
differences O
are O
reflected O
in O
differences O
in O
gene O
expression O
in O
three O
cell O
lines O
. O

This O
pathway O
has O
been O
reported O
to O
mediate O
heterodimer O
interactions O
with O
the O
proapoptotic O
regulator O
, O
Bad B
. O

Mutating O
bases O
- O
142 O
to O
- O
151 O
abolishes O
formation O
of O
complex O
VII O
and O
partially O
inhibits O
complex O
IV O
, O
suggesting O
that O
the O
proteins O
forming O
these O
complexes O
bind O
neighboring O
segments O
of O
DNA O
. O

The O
DFA O
slide O
prepared O
from O
the O
ThinPrep O
Test O
and O
the O
conventional O
DFA O
sample O
prepared O
from O
the O
endocervical O
swab O
were O
evaluated O
independently O
. O

Moclobemide O
is O
a O
well O
- O
tolerated O
alternative O
antidepressant O
, O
but O
there O
is O
a O
need O
for O
prospective O
controlled O
trials O
to O
evaluate O
its O
long O
- O
term O
efficacy O
. O

METHODS O
: O
Autocapture O
devices O
( O
Pacesetter O
Microny O
SR O
+ O
/ O
- O
and O
Regency O
SR O
+ O
/ O
- O
; O
Pacesetter O
, O
Solna O
, O
Sweden O
) O
and O
steroid O
- O
eluting O
epicardial O
pacing O
leads O
( O
Medtronic O
CapSure O
Epi O
10366 O
; O
Medtronic O
, O
Inc O
, O
Minneapolis O
, O
MN O
) O
were O
implanted O
in O
14 O
children O
. O

Hormonal O
replacement O
therapy O
for O
women O
with O
a O
personal O
history O
of O
breast O
cancer O
? O

Recently O
, O
we O
reported O
that O
SMRT B
also O
directly O
associates O
with O
LAZ3 B
( O
BCL B
- I
6 I
) O
, O
a O
POZ B
/ I
Zn I
finger I
transcriptional I
repressor I
involvedin O
the O
pathogenesis O
of O
non O
- O
Hodgkin O
lymphomas O
. O

Using O
reporter O
gene O
constructs O
driven O
by O
the O
CD4 B
promoter I
, O
we O
report O
that O
HHV O
- O
6 O
can O
efficiently O
transactivate O
such O
genetic O
elements O
. O

Characterization O
of O
a O
cDNA O
encoding O
CNN B
predicts O
a O
novel O
structural O
protein O
with O
three O
leucine O
zipper O
motifs O
and O
several O
coiled O
- O
coil O
domains O
exhibiting O
limited O
homology O
to O
the O
rod O
portion O
of O
myosin B
. O

A O
preformed O
SREBP B
- I
1a I
: O
[ O
32P O
] O
DNA O
complex O
bound O
specifically O
to O
membrane O
- O
immobilized O
GST B
- O
CBP B
fusion O
proteins O
that O
contained O
amino O
- O
terminal O
portions O
of O
CBP B
. O

Pyridostigmine O
pretreatment O
was O
supplemented O
by O
therapy O
with O
two O
doses O
of O
an O
antidotal O
combination O
( O
A O
, O
TM O
, O
B O
) O
consisting O
of O
0 O
. O
05 O
mg O
/ O
kg O
atropine O
, O
2 O
. O
24 O
mg O
/ O
kg O
TMB O
- O
4 O
, O
and O
0 O
. O
4 O
mg O
/ O
kg O
benactyzine O
which O
assured O
survival O
in O
five O
of O
six O
animals O
following O
three O
separate O
exposures O
to O
10 O
LD50 O
soman O
. O

Previous O
work O
established O
that O
the O
TAL1 B
proteins I
are O
phosphorylated O
exclusively O
on O
serine O
and O
identified O
Ser122 O
as O
a O
substrate O
for O
the O
mitogen B
- I
activated I
protein I
kinase I
ERK I
- I
1 I
. O

Rapid O
, O
sensitive O
gas O
chromatographic O
analysis O
of O
8 O
- O
methoxypsoralen O
in O
human O
plasma O
. O

If O
the O
size O
of O
the O
cystic O
liver O
- O
lesions O
excludes O
a O
curative O
operative O
treatment O
or O
if O
the O
patient O
refuses O
surgery O
, O
the O
indication O
for O
chemotherapy O
with O
mebendazole O
is O
given O
. O

When O
cotransfected O
with O
the O
HIV B
LTR I
CAT B
into O
CV O
- O
1 O
cells O
, O
both O
the O
pCD41 O
and O
pGD41 O
clones O
trans O
- O
activated O
the O
HIV B
LTR I
. O

The O
induction O
of O
the O
composite O
H B
- I
PK I
/ O
Tag B
and O
SV B
- I
PK I
/ O
Tag B
transgenes O
by O
a O
carbohydrate O
- O
rich O
diet O
in O
the O
liver O
was O
similar O
to O
that O
of O
the O
endogenous O
L I
- I
PK I
gene I
. O

Patients O
with O
advanced O
ovarian O
carcinoma O
, O
Stage O
III O
or O
IV O
( O
International O
Federation O
of O
Gynaecology O
and O
Obstetrics O
) O
, O
were O
randomized O
to O
primary O
chemotherapy O
with O
doxorubicin O
( O
Adriamycin O
) O
and O
cisplatin O
plus O
or O
minus O
hexamethylmelamine O
, O
and O
cyclophosphamide O
( O
CHAP O
) O
. O

Exit O
from O
mitosis O
in O
Drosophila O
syncytial O
embryos O
requires O
proteolysis O
and O
cyclin B
degradation O
, O
and O
is O
associated O
with O
localized O
dephosphorylation O
. O

Cough O
- O
CPR O
, O
a O
deep O
rhythmic O
forceful O
cough O
repeated O
30 O
- O
60 O
times O
per O
minute O
, O
can O
be O
an O
effective O
resuscitative O
technique O
during O
emergencies O
occurring O
in O
the O
cardiac O
catheterization O
laboratory O
. O

To O
understand O
this O
dramatic O
effect O
, O
we O
examined O
the O
localization O
of O
SR B
proteins I
and O
found O
that O
SC35 B
was O
shifted O
to O
the O
cytoplasm O
in O
sky1Delta O
yeast O
, O
although O
this O
phenomenon O
was O
not O
obvious O
with O
ASF B
/ O
SF2 B
, O
indicating O
that O
nuclear O
import O
of O
SR B
proteins I
may O
be O
differentially O
regulated O
by O
phosphorylation O
. O

Probiotics O
, O
prebiotics O
, O
vaccination O
, O
and O
acidification O
of O
drinking O
water O
were O
assessed O
as O
means O
of O
reducing O
Salmonella O
. O

On O
the O
contrary O
i O
. O
v O
. O
administration O
of O
calcitonin B
increased O
intrapyloric O
pressure O
via O
vagal O
nerves O
in O
atropinized O
or O
gallaminized O
animals O
. O

We O
conclude O
that O
in O
the O
presence O
of O
fatty O
acids O
in O
the O
medium O
transcription O
of O
SPS19 B
is O
directly O
regulated O
by O
both O
Pip2p B
- O
Oaf1p B
and O
Adr1p B
. O

Gas O
exchange O
was O
measured O
breath O
by O
breath O
. O

We O
found O
that O
in O
premature O
infants O
plasminogen B
levels O
are O
low O
; O
thus O
, O
defense O
against O
intra O
- O
alveolar O
fibrin B
deposition O
during O
birth O
trauma O
is O
reduced O
. O

Analysis O
of O
nt O
sequence O
was O
carried O
out O
on O
three O
out O
of O
the O
more O
than O
15 O
of O
these O
regions O
present O
in O
the O
mouse O
genome O
. O

Studies O
on O
the O
reaction O
of O
cytochrome B
c I
with O
corticosteroids O
. O

However O
, O
both O
RAR B
- O
and O
RXR B
- O
specific O
ligands O
increase O
CaSki B
number O
by O
> O
or O
= O
20 O
% O
. O

Also O
, O
in O
patients O
with O
MYD O
, O
there O
was O
a O
significant O
decrease O
in O
arterial O
PO2 O
from O
the O
seated O
posture O
to O
the O
supine O
posture O
, O
without O
a O
significant O
change O
in O
the O
arterial O
PCO2 O
. O

The O
primary O
structures O
of O
the O
human B
KB I
cell I
( I
FR I
- I
KB1 I
) I
folate I
receptor I
( O
FR B
) O
and O
of O
a O
human O
placental I
( I
FR I
- I
P2 I
) I
FR I
, O
proteins O
important O
in O
cellular O
accumulation O
of O
folates O
, O
have O
been O
deduced O
from O
cDNA O
sequences O
. O

The O
general O
concept O
of O
RARE O
- O
cleavage O
mapping O
as O
well O
as O
its O
applications O
and O
limitations O
are O
discussed O
. O

To O
further O
investigate O
the O
role O
of O
PKR B
in O
transcriptional O
signaling O
, O
we O
expressed O
the O
wild B
type I
human I
PKR I
and O
a O
catalytically O
inactive O
dominant B
negative I
PKR B
mutant I
in O
the O
murine O
pre O
- O
B O
lymphoma O
70Z O
/ O
3 O
cells O
. O

Selective O
upper O
abdominal O
sympathectomy O
increased O
basal O
acid O
output O
in O
rats O
but O
was O
without O
effect O
on O
stimulated O
acid O
output O
, O
serum B
gastrin I
concentration O
, O
and O
gastric B
mucosal I
histidine I
decarboxylase I
activity O
. O

Reasons O
are O
presented O
to O
support O
the O
contention O
that O
this O
increase O
is O
in O
part O
real O
. O

Patients O
received O
either O
sodium O
cephalothin O
or O
placebo O
intravenously O
before O
the O
procedure O
and O
for O
up O
to O
8 O
additional O
doses O
. O

In O
- O
111 O
- O
BLMC O
uptake O
was O
directly O
proportional O
to O
Ki B
- I
67 I
/ O
MIB B
- I
1 I
activity O
and O
number O
of O
mitoses O
in O
tumor O
tissue O
. O

Heating O
had O
no O
effect O
upon O
either O
force O
decline O
or O
slowing O
of O
relaxation O
during O
fatiguing O
contractions O
. O

Binding O
of O
transformed O
Ah B
receptor I
complex O
to O
a O
dioxin O
responsive O
transcriptional O
enhancer O
: O
evidence O
for O
two O
distinct O
heteromeric O
DNA O
- O
binding O
forms O
. O

Interestingly O
, O
VDR B
was O
able O
to O
bind O
to O
VDREs O
with O
high O
affinity O
and O
to O
fully O
activate O
transcription O
in O
intact O
yeast O
cells O
in O
the O
presence O
of O
the O
retinoid B
X I
receptor I
( O
RXR B
) O
. O

Thus O
, O
it O
appears O
that O
moxalactam O
is O
a O
reliable O
and O
useful O
antibiotic O
for O
the O
treatment O
of O
complicated O
urinary O
tract O
infections O
. O

A O
fine O
- O
structure O
radiation O
hybrid O
map O
for O
the O
region O
that O
extends O
from O
D8S520 B
( O
distal O
) O
to O
D8S1759 B
( O
proximal O
) O
was O
prepared O
, O
followed O
by O
construction O
of O
a O
single O
, O
integrated O
YAC O
/ O
BAC O
contig O
for O
the O
interval O
. O

The O
protein O
contains O
a O
SHAQKYF O
amino O
acid O
signature O
motif O
in O
the O
second O
myb B
- I
like I
repeat I
, O
which O
is O
highly O
conserved O
in O
a O
number O
of O
recently O
identified O
plant B
myb I
- I
related I
genes I
, O
thus O
defining O
a O
new O
class O
of O
plant O
DNA O
- O
binding O
proteins O
. O

IV O
. O

It O
has O
been O
shown O
that O
renin B
secretion O
is O
stimulated O
by O
endothelin B
, O
a O
recently O
discovered O
peptide O
with O
strong O
vasoconstrictive O
properties O
and O
stimulating O
effects O
on O
renin B
secretion O
. O

Here O
we O
describe O
the O
cloning O
and O
initial O
characterization O
of O
IPP B
, O
a O
novel O
human O
gene O
that O
predicts O
a O
kelch B
family I
protein I
homologous O
to O
the O
mouse B
Ipp I
gene I
, O
a O
previously O
described O
kelch B
family I
member O
. O

The O
encoded O
260 B
- I
amino I
- I
acid I
( I
aa I
) I
( I
H1 I
- I
I I
) I
and O
240 B
- I
aa I
( I
H1 I
- I
II I
) I
polypeptides I
possess O
the O
typical O
tripartite O
organization O
of O
animal B
H1 I
histones I
, O
with O
variable O
N O
- O
and O
C O
- O
terminal O
domains O
flanking O
a O
conserved O
' O
globular O
' O
DNA O
- O
binding O
domain O
. O

Instead O
, O
the O
large O
difference O
in O
ssDNA O
- O
binding O
affinities O
reflects O
the O
loss O
of O
hexamerization O
ability O
by O
uvsY B
, O
suggesting O
that O
a O
form O
of O
intrahexamer O
synergism O
or O
cooperativity O
between O
binding O
sites O
within O
the O
uvsY B
hexamer I
leads O
to O
its O
high O
observed O
affinity O
for O
ssDNA O
. O

No O
significant O
difference O
was O
seen O
found O
between O
the O
two O
groups O
in O
the O
degree O
of O
postoperative O
deterioration O
in O
cardiopulmonary O
function O
or O
in O
interleukin B
- I
6 I
levels O
. O

The O
PK B
domain I
showed O
pairwise O
in O
vitro O
binding O
interactions O
with O
the O
pseudokinase O
, O
HisRS B
, O
and O
C O
- O
term O
domains O
; O
additionally O
, O
the O
HisRS B
domain I
interacted O
with O
the O
C O
- O
term O
region O
. O

The O
effects O
of O
erythrosine O
( O
Red O
3 O
) O
, O
rose O
bengal O
B O
( O
Red O
105 O
) O
and O
thyroidectomy O
on O
the O
development O
of O
thyroid O
tumor O
were O
examined O
in O
male O
Wistar O
rats O
treated O
with O
N O
- O
bis O
( O
2 O
- O
hydroxypropyl O
) O
nitrosamine O
( O
DHPN O
) O
. O

The O
AmyI B
specific I
3 I
' I
- I
UTR I
is O
characterized O
by O
the O
absence O
of O
IR O
sequences O
and O
the O
presence O
of O
a O
putative O
' O
AATAAA O
' O
polyadenylation O
signal O
. O

To O
initiate O
our O
analysis O
of O
factors O
required O
for O
eIF B
- I
2 I
alpha I
expression O
, O
selected O
a O
CAP O
- O
proximal O
element O
shown O
by O
in O
vivo O
methylation O
protection O
analysis O
to O
bind O
a O
potential O
regulatory O
factor O
. O

Furthermore O
, O
addition O
of O
the O
recombinant B
NF I
- I
YA I
subunit I
restores O
NF B
- I
Y I
binding O
. O

Reversible O
pressure O
- O
induced O
amorphization O
in O
solid O
C70 O
: O
Raman O
and O
photoluminescence O
study O
. O

Alternative O
roles O
of O
these O
DNA O
motifs O
as O
activators O
of O
early O
mRNA O
transcription O
and O
as O
an O
initiator O
element O
for O
late O
mRNA O
transcription O
help O
explain O
how O
polyomavirus O
gene O
expression O
is O
regulated O
during O
lytic O
growth O
and O
provides O
a O
model O
for O
cellular O
transcription O
during O
development O
. O

Our O
results O
suggest O
that O
Sp1 B
binding I
site I
can O
function O
as O
a O
distinct O
TGF B
- I
beta I
responsive I
element I
for O
TGF B
- I
beta I
mediated O
promoter O
expression O
and O
Sp1 B
per O
se O
can O
mediate O
this O
response O
. O

Effect O
of O
bromocriptine O
and O
metoclopramide O
on O
serum B
prolactin I
levels O
in O
patients O
with O
amyotrophic O
lateral O
sclerosis O
. O

Phase O
I O
study O
of O
5 O
- O
fluorouracil O
and O
leucovorin O
by O
a O
14 O
- O
day O
circadian O
infusion O
in O
metastatic O
adenocarcinoma O
patients O
. O

The O
results O
of O
this O
study O
demonstrate O
that O
release O
of O
a O
single O
pulley O
after O
repair O
of O
the O
tendons O
in O
this O
area O
improved O
gliding O
excursions O
of O
the O
tendons O
and O
reduced O
resistance O
to O
motion O
of O
the O
repaired O
tendons O
, O
and O
provide O
support O
for O
partial O
A2 O
pulley O
incision O
after O
repair O
of O
the O
tendons O
in O
the O
area O
of O
the O
pulley O
. O

Heart O
rate O
and O
oxygen O
consumption O
increased O
75 O
+ O
/ O
- O
4 O
bpm O
and O
26 O
. O
3 O
+ O
/ O
- O
1 O
. O
4 O
ml O
O2 O
/ O
kg O
x O
min O
( O
- O
1 O
) O
from O
supine O
resting O
values O
, O
respectively O
. O

To O
evaluate O
the O
effect O
of O
selective O
thromboxane O
A2 O
blockade O
on O
clinical O
findings O
and O
platelet O
reactivity O
in O
refractory O
unstable O
angina O
, O
OKY O
- O
046 O
( O
600 O
mg O
/ O
day O
, O
p O
. O
o O
. O
) O
was O
administered O
to O
another O
14 O
patients O
with O
refractory O
unstable O
angina O
in O
addition O
to O
the O
conventional O
therapy O
. O

Prolongation O
of O
prothrombin B
time O
was O
not O
due O
to O
depletion O
of O
vitamin B
K I
- I
dependent I
coagulation I
factors I
or O
manifest O
fibrinolysis O
, O
but O
due O
to O
the O
presence O
of O
circulating O
fibrinogen B
fibrinmonomer B
- O
FDP B
complexes O
. O

Effect O
of O
tetracycline O
on O
the O
glycogen O
- O
producing O
function O
of O
the O
liver O
and O
intestinal O
microflora O
in O
white O
mice O

A O
case O
report O
. O

Mutation O
of O
K14 O
in O
H3 B
, O
which O
serves O
as O
the O
major O
target O
of O
recombinant B
Gcn5p I
acetylation O
in O
vitro O
, O
confers O
a O
strong O
, O
synthetic O
growth O
defect O
in O
gcn5 B
cells O
. O

On O
the O
other O
hand O
, O
V3 O
" O
values O
were O
not O
significantly O
correlated O
with O
the O
degree O
of O
tremor O
, O
seborrhea O
, O
and O
duration O
of O
the O
illness O
. O

All O
patients O
whose O
PSA B
levels O
reached O
0 O
. O
1 O
ng O
. O
/ O
ml O
. O

In O
addition O
, O
we O
found O
that O
the O
transactivation O
mediated O
by O
the O
p68c B
- I
ets I
- I
1 I
pr I
p55erg B
through O
the O
Polyomavirus B
enhancer I
sequence I
is O
specifically O
inhibited O
by O
the O
p46kDaPax B
- I
QNR I
in O
transient O
transfection O
assay O
. O

Treatment O
with O
ACE B
inhibitors O
after O
acute O
myocardial O
infarction O

Conversely O
, O
as O
a O
GAL B
- I
4 I
chimera I
, O
the O
isolated O
LAZ3 B
/ O
BCL6 B
BTB O
/ O
POZ O
domain O
appears O
nearly O
as O
efficient O
as O
the O
entire O
protein O
at O
inducing O
transcriptional O
repression O
. O

A O
new O
myocyte B
- I
specific I
enhancer I
- I
binding I
factor I
that O
recognizes O
a O
conserved O
element O
associated O
with O
multiple O
muscle O
- O
specific O
genes O
. O

Diclofenac O
sodium O
: O
blood O
concentration O
of O
the O
slow O
- O
release O
form O
and O
influence O
on O
the O
metabolism O
of O
kallikrein B
. O

The O
LS B
gene I
promoter I
sequence I
has O
homology O
with O
Escherichia O
coli O
promoter O
sequences O
; O
its O
terminator O
sequence O
is O
capable O
of O
forming O
a O
stem O
- O
and O
- O
loop O
structure O
. O

We O
demonstrate O
that O
the O
recently O
proposed O
synthetic O
imaging O
technique O
[ O
J O
. O

Overexpression O
of O
either O
Sp1 B
or O
phosphorylated O
CREB B
transactivated O
the O
mCgA B
promoter I
dose O
dependently O
, O
while O
coexpression O
of O
both O
transcription O
factors O
resulted O
in O
an O
additive O
mCgA B
promoter I
response O
. O

mCgA B
- O
92 O
to O
- O
64 O
bp O
, O
comprising O
the O
Sp1 B
/ O
Egr B
- I
1 I
site O
and O
the O
CRE O
motif O
, O
conferred O
gastrin B
responsiveness O
to O
a O
heterologous O
thymidine B
kinase I
promoter I
system O
, O
and O
therefore O
functions O
as O
a O
" O
true O
" O
enhancer O
element O
. O

Together O
with O
the O
up O
- O
regulation O
of O
the O
cAMP B
response I
element I
modulator I
protein I
( O
CREM B
) O
mRNA O
and O
protein O
levels O
demonstrated O
previously O
in O
CREB B
mutant I
mice O
, O
we O
suggest O
that O
the O
up O
- O
regulation O
of O
CREB B
beta I
may O
also O
contribute O
to O
compensation O
within O
the O
CREB B
/ O
ATF B
family O
of O
transcription O
factors O
, O
when O
CREB B
delta I
and O
CREB B
alpha I
are O
absent O
. O

The O
data O
obtained O
with O
cellular O
RNA O
from O
HepG2 O
cells O
demonstrated O
that O
transcription O
is O
initiated O
891 O
bases O
upstream O
of O
the O
translation O
- O
start O
site O
and O
that O
the O
polyadenylation O
site O
is O
located O
550 O
bases O
downstream O
of O
the O
stop O
codon O
. O

Finally O
, O
there O
is O
now O
appropriate O
recognition O
of O
the O
pivotal O
role O
of O
BP O
reduction O
in O
forestalling O
pressure O
- O
related O
cardiovascular O
complications O
, O
even O
among O
high O
- O
risk O
persons O
with O
diabetes O
mellitus O
and O
renal O
insufficiency O
. O

Individual O
and O
combined O
effects O
of O
fumonisin O
B1 O
present O
in O
Fusarium O
moniliforme O
culture O
material O
and O
diacetoxyscirpenol O
or O
ochratoxin O
A O
in O
turkey O
poults O
. O

It O
is O
hypothesized O
this O
occurs O
through O
antagonism O
of O
PML B
/ O
RAR B
alpha I
actions O
in O
these O
leukemic O
cells O
. O

13 O
: O
961 O
- O
969 O
, O
1993 O
) O
suggested O
that O
T B
antigen I
could O
mediate O
transcriptional O
activation O
through O
interaction O
with O
the O
TATA B
- I
binding I
protein I
, O
as O
well O
as O
upstream B
bound I
transcription I
factors I
. O

The O
Grb2 B
SH3 B
( I
C I
) I
binding O
region O
of O
Gab1 B
has O
significant O
homology O
to O
a O
region O
of O
the O
adapter B
protein I
SLP I
- I
76 I
. O

A O
phase O
II O
study O
of O
interferon B
- I
alpha I
, O
interleukin B
- I
2 I
and O
5 O
- O
fluorouracil O
in O
advanced O
renal O
carcinoma O
: O
clinical O
data O
and O
laboratory O
evidence O
of O
protease O
activation O
. O

Activation O
of O
the O
cytotactin B
promoter I
by O
the O
homeobox B
- I
containing I
gene I
Evx I
- I
1 I
. O

CONCLUSION O
/ O
INTERPRETATION O
: O
We O
found O
an O
increased O
risk O
of O
hip O
fracture O
in O
women O
younger O
than O
75 O
years O
with O
Type O
I O
diabetes O
or O
with O
Type O
II O
diabetes O
of O
long O
duration O
. O

Maternal O
behavior O
was O
assessed O
using O
observational O
techniques O
. O

Rat B
p8 I
mRNA I
was O
discovered O
because O
of O
its O
strong O
activation O
in O
pancreas O
during O
the O
acute O
phase O
of O
pancreatitis O
. O

LV O
compliance O
was O
determined O
from O
the O
slope O
of O
the O
LV O
end O
- O
diastolic O
pressure O
( O
LVEDP O
) O
vs O
. O

The O
practice O
of O
measuring O
of O
AChE B
levels O
in O
acute O
poisoning O
is O
limited O
. O

Vasodilation O
in O
congestive O
heart O
failure O
is O
an O
established O
therapeutic O
principal O
. O

The O
collicular O
labels O
found O
after O
injections O
within O
the O
MST O
area O
exhibited O
their O
distribution O
over O
the O
deep O
SC O
subdivision O
, O
whereas O
they O
spared O
all O
the O
superficial O
layers O
but O
the O
deep O
part O
of O
the O
SO O
. O

When O
furA O
alone O
was O
introduced O
into O
the O
Delta O
( I
furA I
- I
katG I
) O
mutant O
, O
survival O
in O
mouse O
lungs O
was O
moderately O
increased O
, O
suggesting O
that O
FurA O
could O
regulate O
genes O
, O
other O
than O
katG B
, O
that O
are O
involved O
in O
pathogenesis O
. O

The O
effects O
of O
cefazolin O
, O
given O
into O
the O
III O
cerebral O
ventricle O
at O
different O
doses O
were O
studied O
on O
GABA O
content O
, O
GAD B
and O
GABA B
- I
T I
in O
the O
brain O
- O
stem O
of O
young O
chickens O
( O
Gallus O
domesticus O
) O
. O

Subclones O
of O
R15 O
which O
reverted O
to O
kappa B
light I
chain I
production O
contained O
genomic O
deletions O
of O
R15ns B
and O
/ O
or O
the O
surrounding O
intron O
. O

The O
disorders O
are O
accompanied O
by O
consistent O
, O
but O
similarly O
reversible O
, O
electroencephalographic O
changes O
. O

The O
delayed O
activation O
of O
the O
prostaglandin B
G I
/ I
H I
synthase I
- I
2 I
promoter I
in O
bovine O
granulosa O
cells O
is O
associated O
with O
down O
- O
regulation O
of O
truncated B
upstream I
stimulatory I
factor I
- I
2 I
. O

The O
authors O
recommend O
in O
cases O
with O
an O
elevated O
transaminase O
serum O
activity O
more O
frequent O
check O
- O
up O
examinations O
to O
avoid O
missing O
of O
a O
relapse O
, O
and O
to O
examine O
repeatedly O
IgM B
anti I
- I
HAV I
as O
in O
protracted O
forms O
of O
hepatitis O
IgM B
anti I
- I
HAV I
may O
persist O
even O
when O
the O
transaminase O
activity O
is O
normal O
. O

Mean O
latency O
periods O
were O
29 O
. O
6 O
among O
insulators O
, O
35 O
. O
4 O
among O
dock O
workers O
, O
43 O
. O
7 O
in O
a O
heterogeneous O
group O
defined O
as O
various O
, O
46 O
. O
4 O
in O
non O
- O
shipbuilding O
industry O
workers O
, O
49 O
. O
4 O
in O
shipyard O
workers O
, O
51 O
. O
7 O
among O
women O
with O
a O
history O
of O
domestic O
exposure O
to O
asbestos O
, O
and O
56 O
. O
2 O
in O
people O
employed O
in O
maritime O
trades O
. O

The O
strategy O
uses O
RNA B
ligase I
to O
add O
DNA O
oligonucleotide O
priming O
sites O
to O
the O
RNA O
for O
subsequent O
reverse O
transcription O
and O
PCR O
( O
RNA B
ligase I
, O
reverse O
transcription O
- O
PCR O
, O
or O
RL O
/ O
RT O
/ O
PCR O
) O
. O

We O
analyzed O
the O
effects O
of O
light O
on O
tubulin B
mRNA I
abundance O
in O
Arabidopsis O
seedlings O
using O
RNA O
gel O
blot O
hybridizations O
and O
gene O
- O
specific O
probes O
. O

More O
important O
, O
ligand O
- O
activated O
insulin B
and O
IGF B
- I
I I
receptors I
phosphorylate O
SHC B
proteins I
in O
vitro O
, O
indicating O
that O
SHC B
proteins I
could O
be O
direct O
substrates O
for O
insulin B
and O
IGF B
- I
I I
receptors I
. O

This O
chimera O
transduced O
IL B
- I
4 I
- O
specific O
signals O
in O
response O
to O
IL B
- I
2 I
binding O
and O
dramatically O
enhanced O
type O
2 O
responses O
( O
IL B
- I
4 I
, O
IL B
- I
5 I
, O
and O
immunoglobulin B
E I
production O
) O
upon O
in O
vitro O
TCR B
stimulation O
or O
in O
vivo O
antigen O
challenge O
. O

A O
distinct O
5 O
' O
- O
sequence O
in O
clone O
hMIWC2 O
suggested O
an O
alternative O
upstream O
transcription O
initiation O
site O
. O

Two O
human B
beta I
- I
1 I
, I
6 I
- I
N I
- I
acetylglucosaminyltransferases I
forming O
the O
core O
2 O
O O
- O
glycan O
branch O
, O
C2GnT B
and O
the O
I B
antigen I
, O
IGnT B
, O
are O
homologous O
to O
each O
other O
in O
three O
regions O
of O
the O
catalytic O
domain O
( O
A O
, O
B O
, O
C O
) O
and O
their O
genes O
reside O
at O
the O
same O
locus O
, O
chromosome O
9 O
, O
band O
q21 O
( O
Bierhuizen O
, O
M O
. O
F O
. O
A O
. O
, O
Mattei O
, O
M O
. O
- O
G O
. O
and O
Fukuda O
, O
M O
. O
, O
Genes O
Dev O
. O
, O
7 O
, O

468 O
- O
478 O
, O
1993 O
) O
. O

Significantly O
higher O
levels O
of O
vitamin O
C O
and O
catalase B
activity O
were O
found O
in O
vegetarians O
( O
C O
- O
63 O
. O
6 O
and O
86 O
. O
5 O
mumol O
/ O
l O
; O
CAT B
- O
1497 O
and O
1313 O
U O
/ O
ml O
for O
males O
and O
females O
, O
respectively O
) O
when O
compared O
to O
nonvegetarians O
( O
C O
- O
41 O
. O
3 O
and O
54 O
. O
4 O
mumol O
/ O
l O
; O
CAT B
- O
1192 O
and O
1086 O
U O
/ O
ml O
) O
. O

Cutting O
the O
intraorbital O
nerves O
produced O
a O
temporary O
retrieval O
impairment O
that O
was O
indistinguishable O
from O
that O
produced O
by O
intramystacial O
lidocaine O
injection O
. O

Developmental O
regulation O
of O
expression O
of O
the O
malate B
synthase I
gene I
in O
transgenic O
plants O
. O

In O
contrast O
, O
no O
change O
in O
the O
level O
of O
either O
p53 B
or O
activation O
of O
mdm2 B
protein I
by O
p53 B
was O
observed O
in O
hamster O
UV61 O
cells O
after O
UV O
exposure O
. O

On O
day O
1 O
at O
3500 O
m O
, O
RI O
showed O
a O
significant O
fall O
in O
body O
weight O
( O
BW O
) O
with O
respect O
to O
SL O
but O
AI O
maintained O
it O
. O

Transcriptional O
activation O
of O
the O
proopiomelanocortin B
gene I
by O
cyclic B
AMP I
- I
responsive I
element I
binding I
protein I
. O

The O
NF1 B
family I
members I
and O
HNF4 B
interacted O
with O
overlapping O
sequences O
of O
the O
L O
- O
II O
element O
, O
wherein O
the O
5 O
' O
half O
- O
site O
was O
more O
critical O
for O
NF1 B
binding O
, O
and O
the O
3 O
' O
site O
was O
more O
important O
for O
HNF4 B
binding O
. O

RAP B
has O
been O
shown O
to O
be O
a O
useful O
vaccine O
target O
site O
, O
and O
RIP B
and O
inhibitory B
AIPs I
as O
therapeutic O
molecules O
to O
prevent O
and O
suppress O
S O
. O
aureus O
infections O
. O

After O
institution O
of O
insulin B
treatment O
, O
diabetic O
control O
was O
improved O
as O
demonstrated O
by O
decreasing O
levels O
of O
HbA1 B
. O

The O
mouse B
lactoferrin I
gene I
promoter I
was O
active O
in O
human O
endometrium O
carcinoma O
RL O
95 O
- O
2 O
cells O
and O
in O
rat O
glioma O
C6 O
cells O
. O

The O
1 O
- O
year O
and O
2 O
- O
year O
survival O
rates O
for O
the O
patients O
with O
N0 O
- O
3 O
significantly O
exceeded O
those O
of O
N4 O
patients O
. O

Measurement O
of O
the O
spectral O
sensitivity O
and O
the O
ERG O
can O
thus O
help O
in O
the O
diagnosis O
of O
these O
three O
hereditary O
diseases O
. O

As O
a O
quantitative O
index O
of O
cochlear O
function O
, O
2f1 O
- O
f2 O
distortion O
- O
product O
otoacoustic O
emissions O
( O
DPOAEs O
) O
were O
monitored O
systematically O
over O
time O
in O
three O
groups O
of O
rabbits O
, O
with O
each O
group O
experiencing O
a O
unique O
paradigm O
that O
incorporated O
repeated O
exposure O
to O
the O
low O
- O
frequency O
tone O
. O

The O
subjects O
' O
lungs O
were O
ventilated O
with O
N2O O
in O
O2 O
( O
FIO2 O
0 O
. O
3 O
) O
to O
the O
end O
- O
tidal O
CO2 O
present O
before O
anesthesia O
, O
and O
then O
CBF O
was O
measured O
using O
intravenous O
133Xe O
and O
ten O
scintillation O
counters O
, O
five O
over O
each O
cerebral O
hemisphere O
. O

This O
indicates O
changes O
in O
postvaccination O
allergy O
to O
BCG O
. O

Analysis O
of O
1 O
Mb O
of O
published O
sequence O
from O
the O
region O
of O
conserved O
synteny O
on O
human O
chromosome O
5q31 O
- O
q33 O
identified O
45 O
gene O
candidates O
, O
including O
35 O
expressed O
genes O
in O
the O
human B
IL I
- I
4 I
cytokine I
gene I
cluster I
. O

There O
is O
no O
indication O
from O
these O
experiments O
that O
linker B
histones I
bind O
fundamentally O
differently O
to O
5 B
S I
and O
bulk O
nucleosomes O
. O

Km O
values O
of O
the O
uncoupled O
enzymes O
IIGlc B
for O
glucose O
ranged O
from O
0 O
. O
5 O
to O
2 O
. O
5 O
mM O
, O
2 O
orders O
of O
magnitude O
higher O
than O
the O
value O
of O
normal O
IIGlc B
. O

Cytochrome B
bd I
biosynthesis O
in O
Escherichia O
coli O
: O
the O
sequences O
of O
the O
cydC B
and O
cydD B
genes I
suggest O
that O
they O
encode O
the O
components O
of O
an O
ABC B
membrane I
transporter I
. O

In O
contrast O
, O
inhibition O
of O
MAPK B
activity O
by O
MAPK B
kinase I
inhibitor O
( O
PD O
98059 O
) O
or O
by O
overexpression O
of O
kinase O
- O
deficient O
MAPKs B
activated O
basal O
and O
GnRH B
- I
A I
- O
stimulated O
GnRHR B
- O
Luc B
activity O
. O

The O
influence O
of O
gas O
composition O
in O
the O
air O
cell O
on O
pipping O
and O
liver O
metabolism O
in O
embryonic O
chicks O
. O

Historically O
, O
Cyps B
were O
first O
identified O
by O
their O
ability O
to O
bind O
the O
immunosuppressive O
agent O
cyclosporin O
A O
( O
CsA O
) O
with O
high O
affinity O
; O
they O
later O
were O
found O
to O
have O
peptidyl B
- I
prolyl I
cis I
- I
trans I
isomerase I
( O
PPIase B
) O
activity O
, O
which O
catalyzes O
the O
folding O
of O
oligopeptides O
at O
proline O
- O
peptide O
bonds O
in O
vitro O
and O
may O
be O
important O
for O
protein O
folding O
in O
vivo O
. O

Thus O
, O
oxyR B
mutants I
are O
locked O
on O
for O
Ag43 B
expression O
, O
whereas O
dam B
mutants I
are O
locked O
off O
for O
Ag43 B
expression O
. O

A O
rare O
tRNA B
- I
Arg I
( I
CCU I
) I
that O
regulates O
Ty1 B
element I
ribosomal O
frameshifting O
is O
essential O
for O
Ty1 B
retrotransposition O
in O
Saccharomyces O
cerevisiae O
. O

Several O
35 O
- O
mm O
slides O
of O
dystrophin B
- O
, O
laminin B
- O
, O
and O
concanavalin B
A I
( O
ConA B
) O
- O
stained O
muscle O
sections O
were O
used O
to O
calculate O
myofiber O
cross O
- O
sectional O
areas O
and O
to O
compare O
different O
techniques O
and O
settings O
of O
an O
image O
capture O
system O
. O

In O
addition O
, O
both O
inhibitors O
blocked O
phosphatidylcholine O
hydrolysis O
and O
protein B
kinase I
C I
translocation O
. O

New O
, O
selective O
catechol B
- I
O I
- I
methyltransferase I
inhibitors O
as O
therapeutic O
agents O
in O
Parkinson O
' O
s O
disease O
. O

Furthermore O
, O
the O
social O
learning O
theory O
of O
depression O
, O
developed O
by O
Lewinsohn O
, O
is O
described O
. O

Laser O
soldering O
with O
exogenous O
fibrinogen B
is O
feasible O
without O
topical O
administration O
of O
additional O
clotting O
agents O
, O
significantly O
improves O
the O
bursting O
strength O
of O
primary O
laser O
welded O
anastomoses O
, O
and O
appears O
to O
result O
from O
urokinase B
- O
resistant O
fibrinogen B
cross O
- O
linking O
. O

Intensity O
variations O
from O
the O
variable O
TR O
were O
removed O
, O
and O
the O
data O
were O
evaluated O
for O
correlation O
with O
the O
lateralized O
stimulus O
. O

Routine O
imaging O
modalities O
revealed O
a O
total O
of O
79 O
metastases O
. O

We O
have O
previously O
reported O
that O
analgesic O
doses O
of O
morphine O
accelerate O
mortality O
of O
rats O
exposed O
to O
hemorrhage O
( O
Feuerstein O
and O
Siren O
: O
Circ O
Shock O
19 O
: O
293 O
- O
300 O
, O
1986 O
) O
. O

We O
describe O
a O
novel O
stat B
- I
related I
factor I
, O
p93 B
, O
that O
is O
found O
in O
EGF B
- O
treated O
A431 O
cell O
extracts O
but O
appears O
to O
be O
absent O
in O
bovine B
fibroblast I
growth I
factor I
( O
bFGF B
) O
, O
IFN B
- I
gamma I
, O
tumor B
necrosis I
factor I
- I
alpha I
( O
TNF B
- I
alpha I
) O
, O
and O
untreated O
cells O
. O

p93 B
appears O
to O
be O
antigenically O
related O
to O
stat91 B
. O

p185c B
- O
neu B
+ O
, O
EGFr B
+ I
( I
M1 I
) O
, O
and O
p185c B
- I
neu B
- O
kinase O
inactive O
, O
EGFr B
+ I
( O
NEN757 B
) O
expressing O
cells O
undergo O
different O
mitotic O
responses O
to O
EGF B
. O

In O
addition O
, O
Fc B
epsilon I
R1 I
cross O
- O
linking O
activates O
PI B
3 I
- I
kinase I
. O

When O
single O
- O
point O
mutation O
was O
introduced O
to O
each O
GC O
box O
, O
EBS O
, O
and O
GT O
box O
in O
PFP9a20 B
, O
at O
least O
3 O
- O
fold O
less O
CAT B
activity O
was O
observed O
in O
CTLL O
- O
R8 O
cells O
. O

Ribosome O
association O
of O
GCN2 B
protein I
kinase I
, O
a O
translational O
activator O
of O
the O
GCN4 B
gene I
of I
Saccharomyces I
cerevisiae I
. O

Treatment O
with O
H7 O
did O
not O
affect O
IL B
- I
4R I
- O
mediated O
immediate O
signaling O
events O
such O
as O
tyrosine O
phosphorylation O
of O
Jak1 B
, O
Jak3 B
, O
insulin B
receptor I
substrate I
( I
IRS I
) I
- I
1 I
and O
IRS B
- I
2 I
, O
or O
tyrosine O
phosphorylation O
and O
DNA O
binding O
of O
Stat6 B
. O

Thus O
, O
STK1 B
is O
most O
likely O
the O
human B
homologue I
of I
MO15 I
. O

BACKGROUND O
: O
Recent O
iterative O
methods O
for O
sequence O
alignment O
have O
indicated O
that O
the O
380 O
kDa O
motor O
unit O
of O
dynein B
belongs O
to O
the O
AAA B
class I
of O
chaperone B
- I
like I
ATPases I
. O

When O
the O
VBP B
- I
1 I
protein I
was O
solely O
expressed O
, O
it O
located O
to O
the O
cytoplasm O
and O
did O
not O
localize O
to O
the O
nucleus O
. O

Consistent O
with O
other O
experiments O
with O
these O
compounds O
, O
cocaine O
was O
the O
most O
potent O
of O
the O
group O
. O

The O
significance O
of O
these O
changes O
in O
relation O
to O
the O
control O
of O
phosphorus O
balance O
in O
ruminants O
is O
discussed O
. O

Transfection O
assays O
using O
the O
first O
600 O
bp O
of O
the O
upstream O
nucleotide O
sequences O
indicated O
that O
a O
region O
from O
- O
75 O
to O
- O
120 O
was O
necessary O
for O
the O
ALDH2 B
gene I
expression O
, O
and O
especially O
NF B
- I
Y I
/ O
CP1 B
binding O
site O
from O
- O
92 O
to O
- O
96 O
( O
CCAAT O
box O
) O
is O
important O
in O
the O
expression O
of O
the O
gene O
. O

This O
distinct O
biochemical O
difference O
between O
STAT5A B
and O
STAT5B B
was O
confirmed O
with O
purified O
activated O
STAT5 B
recombinant I
proteins I
. O

The O
results O
show O
that O
in O
uninfected O
NIH O
- O
3T3 O
cells O
Avarol O
( O
i O
) O
causes O
a O
50 O
% O
reduction O
of O
the O
growth O
rate O
only O
at O
the O
high O
concentration O
of O
29 O
. O
6 O
microM O
and O
( O
ii O
) O
is O
accumulated O
in O
the O
cytoplasm O
close O
to O
the O
nucleus O
. O

Blood B
coagulation I
Factor I
X I
and O
its O
activated O
form O
Factor B
Xa I
play O
an O
essential O
role O
in O
the O
midphase O
of O
the O
clotting O
cascade O
. O

The O
exon O
sequences O
determined O
from O
genomic O
DNA O
sequencing O
showed O
some O
differences O
when O
compared O
to O
the O
published O
rat B
GLUT2 I
cDNA I
. O

Intramolecular O
interaction O
is O
believed O
to O
result O
in O
the O
formation O
of O
MxA B
monomers I
, O
whereas O
intermolecular O
interaction O
may O
induce O
the O
formation O
of O
large O
MxA B
oligomers I
. O

The O
psbA B
gene I
encodes O
the O
D1 B
protein I
of I
photosystem I
II I
, O
which O
is O
synthesized O
at O
very O
high O
rates O
in O
the O
light O
in O
order O
to O
replace O
photodamaged O
protein O
. O

The O
three O
more O
slower O
migrating O
Stat5B B
bands I
observed O
in O
response O
to O
GH B
contain O
phosphorylated O
tyrosyl O
residues O
. O

The O
maximum O
levels O
of O
reporter O
proteins O
attained O
in O
transformed O
cells O
after O
prolonged O
induction O
represented O
from O
1 O
% O
to O
7 O
% O
of O
total O
cellular O
protein O
. O

Specifically O
, O
we O
demonstrate O
that O
this O
25 O
- O
base O
pair O
region O
mediates O
the O
up O
- O
regulatory O
effect O
of O
TGF B
- I
beta I
on O
COL1A2 B
promoter I
activity O
and O
allows O
antagonistic O
activity O
of O
TNF B
- I
alpha I
on O
the O
TGF B
- I
beta I
effect O
. O

Sin4 B
and O
Srb10 B
, O
components O
of O
specific O
RNA B
polymerase I
II I
sub O
- O
complexes O
that O
are O
required O
for O
Ssn6 B
- O
Tup1 B
repression O
activity O
, O
are O
found O
to O
be O
required O
for O
Sfl1 B
repression O
function O
. O

Translation O
of O
mok B
is O
tightly O
regulated O
by O
Sok B
RNA I
, O
and O
Sok B
RNA I
thus O
regulates O
hok B
translation O
indirectly O
through O
mok B
. O

Depending O
on O
the O
patient O
' O
s O
body O
position O
telemetrically O
measured O
CSF O
- O
pressures O
varied O
between O
- O
20 O
cmH2O O
in O
erect O
and O
15 O
cmH2O O
in O
supine O
position O
. O

One O
- O
hour O
O3 O
, O
NO2 O
, O
and O
SO2 O
personal O
exposures O
were O
measured O
using O
samplers O
developed O
in O
our O
laboratory O
, O
while O
short O
- O
term O
PM2 O
. O
5 O
, O
CO O
, O
and O
VOCs O
exposures O
were O
measured O
using O
currently O
available O
monitors O
. O

These O
Tlr B
family I
members I
, O
unlike O
others O
reported O
to O
date O
, O
were O
identified O
within O
a O
genomic O
database O
. O

We O
hypothesized O
that O
subjects O
would O
demonstrate O
PKAR O
during O
both O
hopping O
and O
stepping O
, O
adding O
support O
to O
the O
hypothesis O
that O
PKAR O
is O
a O
centrally O
mediated O
adaptation O
of O
general O
locomotor O
trajectory O
that O
is O
not O
specific O
to O
the O
form O
of O
locomotion O
used O
while O
on O
the O
rotating O
disk O
. O

In O
this O
study O
, O
transcriptional O
fusions O
were O
constructed O
between O
the O
xcpP B
and O
xcpR B
genes I
and O
the O
lacZ B
reporter I
. O

To O
explore O
the O
mechanism O
of O
insulin B
receptor I
phosphorylation O
we O
have O
used O
NIH3T3 O
cells O
transfected O
with O
two O
receptor O
constructs O
: O
one O
encoding O
a O
chimeric O
receptor O
composed O
of O
the O
extracellular O
domain O
of O
the O
human B
EGF I
receptor I
and O
the O
cytosolic O
domain O
of O
the O
human B
insulin I
receptor I
beta I
- I
subunit I
, O
and O
a O
second O
construct O
encoding O
a O
kinase O
- O
defiecient O
human B
insulin I
receptor I
. O

It O
is O
pathogenetically O
proved O
to O
use O
antithrombin B
III I
concentrate O
preparations O
with O
anti O
- O
and O
dysaggregatory O
properties O
, O
fibronectin B
preparations O
, O
trasylol O
or O
its O
analogs O
during O
complex O
preoperative O
preparation O
of O
patients O
. O

Stimulation O
of O
the O
endosteal O
bone O
formation O
rate O
was O
mainly O
impaired O
in O
D O
- O
depleted O
rats O
, O
resulting O
in O
trabecular O
bone O
loss O
, O
which O
, O
in O
- O
D O
mother O
rats O
, O
was O
associated O
with O
decreased O
bone O
ash O
and O
total O
bone O
calcium O
. O

Hematopoietic B
progenitor I
kinase I
1 I
( O
HPK1 B
) O
is O
a O
member O
of O
the O
mitogen B
- I
activated I
protein I
kinase I
kinase I
kinase I
kinase I
( O
MAP4K B
) O
family O
and O
an O
upstream O
activator O
of O
the O
c B
- I
Jun I
N I
- I
terminal I
kinase I
( O
JNK B
) O
signaling O
cascade O
. O

We O
have O
used O
the O
chloramphenicol B
acetyltransferase I
( O
CAT B
) O
reporter O
gene O
system O
to O
study O
the O
effect O
of O
T O
on O
the O
long O
terminal O
repeats O
( O
LTRs O
) O
of O
a O
large O
family O
of O
human O
endogenous O
retrovirus O
- O
like O
sequences O
, O
RTVL O
- O
H O
. O

To O
investigate O
this O
possibility O
, O
we O
conducted O
split O
- O
visual O
- O
field O
studies O
in O
which O
we O
manipulated O
stimulus O
sets O
, O
recognition O
task O
, O
and O
exposure O
duration O
. O

In O
conclusion O
, O
ANH O
had O
no O
negative O
effects O
on O
the O
endocrine O
stress O
response O
during O
orthopedic O
surgery O
under O
epidural O
anesthesia O
. O

Finally O
, O
the O
conservation O
of O
the O
two O
residues O
most O
sensitive O
to O
mutations O
( O
Y949 O
and O
Y953 O
) O
in O
TM11 O
, O
and O
in O
the O
homologous O
TM5 O
, O
of O
all O
mammalian B
P I
- I
gps I
and O
also O
in O
other O
ABC B
transporters I
, O
suggests O
that O
these O
residues O
and O
domains O
may O
play O
an O
important O
role O
in O
structural O
as O
well O
as O
mechanistic O
aspects O
common O
to O
this O
family O
of O
proteins O
. O

Based O
on O
the O
requirement O
for O
CREM B
/ O
ICER B
and O
Rad6B B
proteins I
in O
spermatogenesis O
, O
we O
determined O
expression O
of O
Cdc34 B
, O
Rad6B B
, O
CREM B
/ O
ICER B
isoforms O
, O
and O
the O
Skp1 B
- O
Cullin B
- O
F B
- I
box I
ubiquitin B
protein O
ligase O
subunits O
Cul B
- I
1 I
and O
Cul B
- I
2 I
, O
which O
are O
associated O
with O
Cdc34 B
activity O
during O
murine O
testicular O
development O
. O

The O
enigma O
of O
aging O
bone O
loss O
. O

Transcription B
factor I
IIIC I
( O
TFIIIC B
) O
is O
required O
for O
the O
assembly O
of O
a O
preinitiation O
complex O
on O
5S B
RNA I
, O
tRNA O
, O
and O
adenovirus B
VA I
RNA I
genes I
and O
contains O
two O
separable O
components O
, O
TFIIIC1 B
and O
TFIIIC2 B
. O

Furthermore O
, O
we O
show O
that O
Nck B
- I
2 I
is O
capable O
of O
recognizing O
several O
key O
components O
of O
growth B
factor I
receptor I
kinase I
- O
signaling O
pathways O
including O
EGF B
receptors I
, O
PDGF B
receptor I
- I
beta I
, O
and O
IRS B
- I
1 I
. O

These O
two O
enhancer O
elements O
also O
enhanced O
transcription O
when O
fused O
separately O
to O
the O
basal O
promoter O
region O
of O
the O
chicken B
vimentin I
gene I
. O

When O
they O
were O
exposed O
to O
intermittent O
schedules O
of O
punishment O
( O
fixed O
- O
interval O
[ O
FI O
] O
120 O
s O
or O
FI O
300 O
s O
) O
, O
SIB O
for O
all O
but O
1 O
of O
the O
participants O
increased O
to O
levels O
similar O
to O
those O
observed O
during O
baseline O
. O

Deep O
bite O
CL O
II O
Div O
. O

Patients O
with O
binocular O
pregeniculate O
visual O
loss O
, O
patients O
with O
balanced O
binocular O
pregeniculate O
loss O
without O
RAPD O
, O
and O
patients O
with O
monocular O
pregeniculate O
visual O
loss O
had O
significantly O
larger O
pupils O
than O
age O
- O
matched O
controls O
. O

Protein O
sequencing O
, O
electrophoretic O
mobility O
shift O
assay O
, O
and O
immunoblot O
analyses O
identify O
p54 B
and O
p47 B
/ I
48 I
as O
members O
of O
the O
hepatocyte B
nuclear I
factor I
3 I
( O
HNF3 B
[ O
forkhead B
] O
) O
family O
of O
transcription O
factors O
. O

p54 B
belongs O
to O
the O
subfamily O
of O
HNF3 B
beta I
proteins I
, O
while O
p47 B
/ I
48 I
binding O
activity O
includes O
HNF3 B
gamma I
. O

In O
this O
cross O
- O
over O
controlled O
study O
, O
five O
male O
volunteers O
donated O
one O
unit O
of O
red O
cells O
by O
MCS O
and O
one O
unit O
of O
whole O
blood O
by O
the O
conventional O
manual O
method O
, O
3 O
months O
apart O
. O

Promoter O
activity O
was O
assayed O
by O
transient O
transfection O
of O
luciferase B
reporter I
constructs I
containing O
nested O
deletions O
of O
the O
upstream O
sequence O
in O
the O
human O
RPE O
cell O
lines O
ARPE19 O
and O
D407 O
, O
as O
well O
as O
in O
the O
SK O
- O
Mel O
- O
28 O
and O
HeLa O
cell O
lines O
. O

This O
model O
system O
exploits O
the O
polyomavirus O
late O
transcription O
termination O
and O
polyadenylation O
signals O
, O
which O
are O
sufficiently O
weak O
to O
allow O
the O
production O
of O
many O
multigenome O
- O
length O
primary O
transcripts O
with O
repeating O
introns O
, O
exons O
, O
and O
poly O
( O
A O
) O
sites O
. O

Magnification O
- O
corrected O
planimetry O
of O
the O
parapapillary O
region O
was O
performed O
according O
to O
Littmann O
' O
s O
method O
in O
312 O
unselected O
eyes O
with O
chronic O
primary O
open O
- O
angle O
glaucoma O
and O
in O
125 O
normal O
eyes O
of O
an O
age O
- O
and O
refraction O
- O
matched O
control O
group O
using O
optic O
disk O
photographs O
. O

Although O
the O
elevated O
signalling O
is O
eliminated O
by O
deletion O
of O
Ste20p B
( O
or O
components O
downstream O
of O
Ste20p B
) O
, O
the O
growth O
and O
morphological O
abnormalities O
of O
cells O
lacking O
Akr1p B
are O
not O
rescued O
by O
deletion O
of O
any O
of O
the O
known O
pheromone O
response O
pathway O
components O
. O

Southern O
hybridization O
of O
genomic O
DNA O
from O
WT O
and O
Lp O
/ O
Lp O
embryos O
failed O
to O
identify O
specific O
rearrangements O
at O
or O
near O
the O
Nhlh1 B
locus I
, O
and O
Northern O
RNA O
blotting O
and O
RT O
- O
PCR O
evaluation O
of O
Nhlh1 B
mRNA I
expression O
indicated O
that O
both O
the O
levels O
and O
types O
of O
Nhlh1 B
mRNAs I
produced O
in O
WT O
and O
Lp O
/ O
Lp O
embryos O
were O
indistinguishable O
. O

In O
addition O
, O
mrp17 B
- I
1 I
, O
in O
combination O
with O
some O
mutations O
affecting O
another O
mitochondrial B
ribosomal I
protein I
, O
caused O
a O
synthetic O
defective O
phenotype O
. O

Expression O
, O
characterization O
, O
and O
genomic O
structure O
of O
carp B
JAK1 I
kinase I
gene I
. O

Significantly O
, O
the O
murine B
P I
- I
selectin I
gene I
had O
several O
features O
not O
found O
in O
the O
human O
gene O
. O

Fluorescent O
in O
situ O
hybridization O
( O
FISH O
) O
analysis O
showed O
that O
the O
novel O
regions O
involved O
in O
the O
NFKB2 B
rearrangement O
originated O
from O
chromosome O
7q34 O
, O
thus O
implying O
the O
occurrence O
of O
a O
t O
( O
7 O
; O
10 O
) O
( O
q34 O
; O
q24 O
) O
reciprocal O
chromosomal O
translocation O
. O

Transient O
- O
expression O
assay O
analysis O
of O
subclones O
of O
pPstI B
- I
G I
localized O
the O
trans O
- O
active O
factor O
to O
a O
3 O
. I
0 O
- I
kilobase I
XbaI I
fragment I
. O

MGF B
is O
a O
novel O
member O
of O
the O
cytokine O
- O
regulated O
transcription O
factor O
gene O
family O
. O

This O
is O
the O
first O
report O
that O
the O
PH B
domain I
of O
an O
IRS B
protein I
can O
function O
in O
a O
dominant O
negative O
manner O
to O
inhibit O
insulin B
signaling O
. O

A O
29 O
. O
425 O
kb O
segment O
on O
the O
left O
arm O
of O
yeast O
chromosome O
XV O
contains O
more O
than O
twice O
as O
many O
unknown O
as O
known O
open O
reading O
frames O
. O

This O
result O
, O
together O
with O
the O
fact O
that O
unrearranged O
V B
kappa I
genes I
are O
transcriptionally O
silent O
, O
suggests O
that O
structural O
features O
of O
both O
the O
V B
kappa I
and O
C B
kappa I
loci I
contribute O
to O
the O
overall O
transcriptional O
efficiency O
of O
a O
rearranged O
V B
kappa I
- I
C I
kappa I
gene O
. O

The O
first O
270 O
bp O
of O
the O
promoter O
region O
were O
sequenced O
and O
found O
to O
contain O
a O
CATAA O
box O
rather O
than O
a O
TATAA O
box O
and O
several O
DNA O
motifs O
found O
in O
activation O
genes O
. O

C O
. O
glabrata O
cells O
containing O
the O
CBF1 B
gene I
under O
the O
influence O
of O
a O
shutdown O
promoter O
( O
tetO B
- I
ScHOP I
) O
arrested O
their O
growth O
after O
5 O
h O
of O
cultivation O
in O
the O
presence O
of O
the O
reactive O
drug O
doxycycline O
. O

SF1 B
/ O
mBBP B
utilizes O
a O
" B
maxi I
- I
K I
homology I
" I
( O
maxi B
- I
KH I
) O
domain O
for O
recognition O
of O
the O
single O
- O
stranded O
BPS O
and O
requires O
a O
cooperative O
interaction O
with O
splicing B
factor I
U2AF65 I
bound O
to O
an O
adjacent O
polypyrimidine O
tract O
( O
PPT O
) O
for O
high O
- O
affinity O
binding O
. O

An O
X B
- I
linked I
zinc I
finger I
gene I
mapping O
to O
Xq21 O
. O
1 O
- O
q21 O
. O
3 O
closely O
related O
to O
ZFX B
and O
ZFY B
: O
possible O
origins O
from O
a O
common O
ancestral O
gene O
. O

Validity O
of O
the O
short O
- O
form O
QIF O
was O
assessed O
by O
correlation O
with O
motor O
scores O
and O
using O
analysis O
of O
variance O
by O
motor O
levels O
and O
motor O
score O
groupings O
. O

Juvenile O
angiofibromas O
. O

Comment O
on O
" O
Critical O
behavior O
of O
a O
binary O
mixture O
of O
protein O
and O
salt O
water O
" O

The O
pineal O
hormone O
melatonin O
( O
MLT O
) O
is O
able O
to O
exert O
an O
oncostatic O
action O
. O

In O
addition O
, O
there O
was O
an O
increase O
in O
the O
amount O
of O
p120 B
Ras B
- O
specific I
GTPase B
- I
activating I
protein I
( O
GAP B
) O
and O
GAP B
- O
associated O
p190 B
. O

Additional O
transection O
of O
both O
ADN O
eliminated O
the O
remaining O
sigmoidal O
correlation O
between O
MAP O
and O
RSNA O
with O
further O
increases O
in O
resting O
MAP O
and O
RSNA O
. O

Pregnancy O
was O
ensued O
in O
4 O
cases O
in O
group O
I O
. O

The O
1 O
. O
3 O
- O
kb O
DNA O
, O
when O
placed O
upstream O
of O
the O
chloramphenicol B
acetyltransferase I
gene I
, O
was O
shown O
to O
be O
functionally O
active O
. O

Controversy O
exists O
regarding O
both O
the O
natural O
life O
cycle O
for O
this O
parasite O
as O
well O
as O
the O
species O
identity O
of O
opossum O
Sarcocystis O
. O

The O
mean O
was O
66 O
cells O
/ O
mm2 O
in O
the O
laser O
group O
and O
63 O
. O
7 O
cells O
/ O
mm2 O
in O
the O
control O
group O
. O

Prazosin O
in O
the O
treatment O
of O
high O
blood O
pressure O

The O
5 O
- O
HT3 O
antagonist O
ondansetron O
reduced O
alcohol O
intake O
in O
both O
the O
medium O
and O
high O
alcohol O
preferring O
rats O
at O
doses O
between O
0 O
. O
01 O
and O
0 O
. O
16 O
mg O
/ O
kg O
. O

Oral O
hypoglycemic O
agents O

1 O
. O

In O
this O
system O
, O
enhancers O
act O
primarily O
to O
increase O
the O
probability O
of O
rapid O
and O
efficient O
transcription O
complex O
formation O
and O
initiation O
. O

The O
relationship O
between O
donor O
status O
for O
antibody O
to O
hepatitis B
B I
core I
antigen I
and O
the O
occurrence O
of O
non O
- O
A O
, O
non O
- O
B O
posttransfusion O
hepatitis O
in O
the O
recipient O
was O
prospectively O
studied O
in O
112 O
patients O
undergoing O
open O
- O
heart O
surgery O
who O
were O
followed O
for O
6 O
. O
5 O
months O
after O
surgery O
. O

By O
sequencing O
of O
exonuclease B
III I
deletion O
clones O
an O
open O
reading O
frame O
of O
405 O
nucleotides O
was O
found O
coding O
for O
a O
protein O
of O
135 O
amino O
acids O
with O
a O
molecular O
mass O
of O
15 O
kDa O
. O

The O
pepI B
gene I
was O
overexpressed O
in O
Escherichia O
coli O
. O

Treatment O
of O
steroid O
resistant O
rejection O
following O
renal O
transplantation O
: O
benefits O
and O
risks O
of O
OKT3 B
therapy O
. O

The O
level O
of O
transcription O
generated O
by O
all O
of O
these O
activators O
is O
greater O
than O
the O
sum O
of O
the O
levels O
generated O
by O
individual O
factors O
, O
a O
phenomenon O
designated O
transcriptional O
synergy O
. O

Conservation O
of O
this O
zinc O
finger O
motif O
from O
yeast O
to O
mouse O
and O
human O
implies O
functional O
importance O
. O

This O
nuclear O
/ O
organellar O
gene O
transfer O
event O
is O
strikingly O
similar O
to O
the O
experimentally O
accessible O
process O
of O
nuclear O
integration O
of O
introduced O
heterologous O
DNA O
. O

RESULTS O
: O
In O
the O
test O
data O
set O
, O
the O
single O
- O
sample O
model O
was O
confirmed O
to O
give O
excellent O
estimation O
of O
the O
AUC O
: O
AUC O
( O
mg O
/ O
ml O
x O
min O
) O
= O
0 O
. O
93 O
x O
C3h O
+ O
0 O
. O
47 O
( O
MPE O
% O
= O
4 O
. O
4 O
% O
, O
RMSE O
% O
= O
8 O
. O
9 O
% O
) O
. O

SH3A B
competes O
with O
the O
SH3 B
domains I
of O
Grb2 B
in O
binding O
to O
mSos1 B
, O
and O
the O
intersectin O
- O
mSos1 B
complex O
can O
be O
separated O
from O
Grb2 B
by O
sucrose O
gradient O
centrifugation O
. O

Comparison O
between O
the O
Ricketts O
and O
Bimler O
therapeutic O
technics O
in O
the O
treatment O
of O
cases O
of O
class O
II O
, O
division O
I O
mmalocclusion O

This O
resulted O
in O
a O
shift O
of O
C1 O
/ O
C2 O
, O
so O
that O
the O
effect O
of O
collagen B
was O
more O
pronounced O
( O
maximal O
increase O
of O
C1 O
/ O
C2 O
= O
134 O
% O
) O
than O
ADP O
( O
maximal O
increase O
of O
C1 O
/ O
C2 O
= O
79 O
% O
) O
. O

We O
conclude O
that O
exercise O
- O
induced O
ST O
- O
segment O
elevation O
in O
patients O
without O
a O
history O
of O
myocardial O
infarction O
or O
left O
ventricular O
aneurysm O
is O
caused O
by O
coronary O
spasm O
of O
a O
major O
coronary O
vessel O
. O

Xnr3 B
is O
transcriptionally O
activated O
by O
wnt B
signaling O
during O
gastrulation O
in O
the O
Xenopus O
embryo O
. O

For O
economical O
reasons O
, O
we O
could O
not O
send O
a O
questionnaire O
to O
all O
the O
35 O
, O
779 O
individuals O
, O
but O
based O
the O
investigation O
on O
a O
SRS O
of O
4 O
, O
000 O
men O
, O
post O
- O
stratified O
in O
a O
high O
- O
risk O
and O
a O
low O
- O
risk O
group O
. O

Several O
waveforms O
of O
both O
the O
somatosensory O
( O
N20 O
, O
P O
/ O
N O
30 O
, O
and O
P200 O
) O
and O
the O
brainstem O
auditory O
- O
evoked O
response O
( O
I O
, O
III O
, O
IV O
, O
and O
V O
) O
demonstrated O
shorter O
( O
p O
less O
than O
0 O
. O
05 O
) O
latencies O
in O
growth O
- O
retarded O
fetuses O
relative O
to O
normal O
- O
sized O
fetuses O
. O

Effect O
of O
choline O
magnesium O
trisalicylate O
on O
prostacyclin O
production O
by O
isolated O
vascular O
tissue O
of O
the O
rat O
. O

The O
sequences O
of O
two O
previously O
known O
tail B
genes I
, O
R O
and O
S O
, O
of O
the O
temperate O
bacteriophage O
P2 O
and O
the O
sequence O
of O
an O
additional O
open O
reading O
frame O
( O
orf O
- O
30 O
) O
located O
between O
S O
and O
V O
, O
were O
determined O
. O

The O
high O
prevalence O
of O
atrial O
septal O
defect O
in O
tetralogy O
of O
Fallot O
is O
cited O
as O
a O
possible O
analogy O
because O
right O
ventricular O
pressure O
is O
high O
and O
right O
ventricular O
compliance O
is O
low O
from O
birth O
. O

The O
stress O
/ O
BHV O
- O
1 O
model O
resulted O
in O
a O
mild O
respiratory O
infection O
in O
all O
calves O
with O
no O
difference O
observed O
between O
treatment O
groups O
. O

Solution O
of O
the O
Boltzmann O
equation O
in O
a O
random O
magnetic O
field O
. O

GH B
signals O
by O
interacting O
with O
GH B
receptor I
( O
GHR B
) O
. O

Ultraviolet O
crosslinking O
experiments O
using O
element O
B O
revealed O
the O
specific O
binding O
of O
two O
proteins O
of O
approximately O
43 O
and O
80 O
kDa O
. O

RESULTS O
: O
At O
short O
dwell O
times O
, O
uncorrected O
( O
whole O
plasma O
) O
D O
/ O
P O
and O
Cp O
of O
urea O
values O
were O
higher O
than O
the O
corrected O
( O
plasma O
water O
) O
values O
by O
5 O
. O
7 O
% O
- O
5 O
. O
9 O
% O
. O

The O
recovery O
of O
xanthomegnin O
added O
to O
corn O
samples O
at O
levels O
of O
0 O
. O
75 O
- O
- O
9 O
. O
6 O
mg O
/ O
kg O
averaged O
41 O
% O
with O
a O
coefficient O
of O
variation O
of O
25 O
% O
. O

The O
extraordinary O
high O
substrate O
specificity O
of O
rPulA B
together O
with O
its O
thermal O
stability O
makes O
this O
enzyme O
a O
good O
candidate O
for O
biotechnological O
applications O
in O
the O
starch O
- O
processing O
industry O
. O

A O
relationship O
between O
the O
immunological O
changes O
and O
the O
disease O
stage O
was O
discovered O
: O
the O
index O
of O
the O
3H O
- O
thymidine O
incorporation O
was O
found O
to O
be O
67 O
. O
9 O
at O
the O
stage O
of O
disease O
exacerbation O
, O
and O
208 O
. O
8 O
at O
the O
stage O
of O
remission O
; O
the O
complement O
titre O
reached O
, O
respectively O
, O
46 O
. O
64 O
+ O
/ O
- O
5 O
. O
28 O
and O
112 O
. O
0 O
+ O
/ O
- O
6 O
. O
0 O
units O
per O
ml O
. O

Sodium O
dodecylsulfate O
polyacrylamide O
gel O
electrophoresis O
( O
SDS O
- O
PAGE O
) O
showed O
two O
bands O
of O
about O
the O
same O
intensity O
with O
apparent O
molecular O
masses O
of O
24 O
. O
5 O
and O
22 O
. O
5 O
kDa O
. O

Consistent O
with O
an O
effect O
on O
transcription O
, O
p21 B
was O
localized O
in O
nuclei O
of O
transfected O
cells O
. O

Analysis O
of O
pseudo B
VH I
gene I
segment I
recombination O
products O
reveals O
no O
bias O
in O
D B
gene I
segment O
reading O
frame O
utilization O
. O

Plasma B
prolactin I
concentrations O
during O
incubation O
, O
25 O
. O
8 O
+ O
/ O
- O
2 O
. O
3 O
ng O
/ O
ml O
, O
decreased O
to O
baseline O
levels O
, O
10 O
. O
8 O
+ O
/ O
- O
1 O
. O
9 O
ng O
/ O
ml O
, O
within O
24 O
hr O
after O
nestbox O
removal O
. O

After O
30 O
weeks O
of O
amifostine O
therapy O
, O
the O
morphology O
of O
the O
MDS O
switched O
to O
a O
chronic O
myelomonocytic O
leukemia O
( O
CMML O
) O
- O
like O
appearance O
, O
with O
continuously O
increasing O
leukocytes O
, O
so O
that O
we O
discontinued O
amifostine O
therapy O
for O
1 O
month O
to O
exclude O
a O
possible O
side O
effect O
of O
amifostine O
. O

Heart O
disease O
. O

A O
new O
B O
- O
cell O
- O
specific O
enhancer O
element O
has O
been O
identified O
3 O
' O
of O
E4 B
and O
the O
octamerlike O
motifs O
in O
the O
human B
immunoglobulin I
heavy I
- I
chain I
gene I
enhancer I
. O

CONCLUSIONS O
: O
This O
initial O
experience O
indicates O
that O
there O
is O
short O
- O
to O
middle O
- O
term O
efficiency O
and O
safety O
when O
using O
GKS O
to O
treat O
MTLE O
. O

Two O
cases O
of O
Paraquat O
poisoning O
of O
anticonservative O
origin O
are O
described O
. O

Genetic O
mutation O
or O
loss O
of O
activin B
/ O
transforming B
growth I
factor I
- I
beta I
( I
TGFbeta I
) I
receptor I
function O
has O
been O
shown O
in O
human O
lymphoid O
, O
breast O
, O
and O
colorectal O
tumors O
as O
well O
as O
Hep2B O
and O
Mv1Lu O
cell O
lines O
. O

A O
poly O
( O
ortho O
ester O
) O
designed O
for O
combined O
ocular O
delivery O
of O
dexamethasone O
sodium O
phosphate O
and O
5 O
- O
fluorouracil O
: O
subconjunctival O
tolerance O
and O
in O
vitro O
release O
. O

We O
now O
describe O
a O
HepG2 B
cell I
casein B
kinase I
II I
beta I
subunit I
cDNA I
of O
2 O
. O
57 O
kb O
containing O
96 O
bases O
of O
5 O
' O
untranslated O
sequence O
, O
645 O
bases O
of O
open O
reading O
frame O
, O
and O
1832 O
bases O
of O
3 O
' O
untranslated O
sequence O
with O
two O
polyadenylation O
consensus O
signal O
sequences O
and O
two O
poly O
( O
A O
) O
stretches O
. O

Brome B
mosaic I
virus I
polymerase I
- I
like I
protein I
2a I
is O
directed O
to O
the O
endoplasmic O
reticulum O
by O
helicase B
- I
like I
viral I
protein I
1a I
. O

Diagnostic O
importance O
of O
determining O
leucine B
aminotransferase I
activity O
in O
acute O
pancreatitis O

Therapeutic O
iodine O
125 O
for O
hyperthyroidism O
: O
evidence O
for O
a O
special O
radiobiological O
effect O
on O
the O
follicular O
cell O
. O

Maintenance O
metabolizable O
- O
energy O
( O
ME O
) O
requirement O
averaged O
31 O
. O
0 O
kcal O
/ O
kg O
body O
wt O
. O

This O
finding O
is O
of O
importance O
because O
it O
completes O
an O
explanation O
for O
central O
near O
- O
location O
errors O
in O
the O
partial O
- O
report O
bar O
- O
probe O
task O
. O

A O
DNA O
binding O
protein O
was O
identified O
which O
binds O
to O
two O
novel O
target O
- O
like O
sequences O
: O
( O
i O
) O
at O
the O
5 O
' O
flanking O
site O
of O
the O
breakpoint O
junction O
of O
chromosome O
8 O
in O
a O
patient O
with O
T O
- O
acute O
lymphoblastic O
leukemia O
( O
ALL O
) O
carrying O
the O
t O
( O
8 O
; O
14 O
) O
( O
q24 O
; O
q11 O
) O
rearrangement O
and O
( O
ii O
) O
on O
chromosome O
1 O
in O
three O
of O
five O
T O
- O
ALL O
patients O
with O
the O
t O
( O
1 O
; O
14 O
) O
( O
p32 O
; O
q11 O
) O
rearrangement O
. O

Nedocromil O
sodium O
shifted O
the O
severity O
of O
the O
early O
allergic O
reaction O
( O
EAR O
) O
from O
mean O
- O
34 O
. O
8 O
% O
to O
- O
6 O
. O
9 O
% O
and O
inhibited O
the O
later O
allergic O
reaction O
( O
LAR O
) O
from O
- O
30 O
. O
5 O
% O
to O
+ O
0 O
. O
4 O
% O
( O
p O
less O
than O
0 O
. O
005 O
) O
. O

The O
Mer1 B
protein I
contains O
the O
KH O
motif O
found O
in O
some O
RNA O
- O
binding O
proteins O
, O
and O
RNA O
gel O
mobility O
shift O
assays O
demonstrate O
that O
Mer1 B
binds O
specifically O
to O
MER2 B
RNA I
. O

Results O
obtained O
from O
the O
tritium O
test O
and O
direct O
chemical O
analysis O
were O
compared O
. O

We O
found O
that O
in O
anesthetized O
, O
paralyzed O
cats O
, O
the O
visual O
evoked O
potential O
( O
VEP O
) O
was O
dependent O
only O
on O
magnitude O
of O
delta O
C O
at O
each O
pattern O
transition O
, O
and O
was O
independent O
of O
the O
starting O
or O
ending O
contrast O
level O
. O

Experiment O
B O
: O
Clinical O
, O
biochemical O
, O
and O
histological O
variables O
were O
measured O
over O
a O
12 O
- O
day O
period O
after O
the O
zymosan O
had O
been O
given O
. O

Direct O
saturation O
analysis O

From O
this O
asymmetry O
, O
we O
suggested O
previously O
that O
GCN4 B
interacts O
with O
nonequivalent O
and O
possibly O
overlapping O
half O
- O
sites O
( O
ATGAC O
and O
ATGAG O
) O
that O
have O
different O
affinities O
. O

Furthermore O
the O
aldosterone O
stimulating O
effect O
of O
low O
sodium O
diet O
( O
17 O
children O
) O
, O
severe O
and O
prolonged O
vomiting O
( O
19 O
children O
) O
and O
synthetic B
ACTH I
( O
10 O
children O
) O
has O
been O
studied O
by O
our O
modified O
method O
. O

A O
case O
control O
study O
( O
1 O
: O
2 O
) O
of O
182 O
pairs O
of O
Hepatitis O
A O
was O
carried O
out O
in O
Weng O
- O
ling O
Zhe O
- O
jiang O
during O
April O
1988 O
. O

The O
mutant O
strain O
carries O
three O
tandem O
copies O
of O
the O
18 O
bp O
sequence O
that O
is O
duplicated O
in O
the O
amdI66 B
mutation I
. O

Although O
cDNA O
encoding O
a O
human B
phagocyte I
formyl I
peptide I
- I
binding I
protein I
has O
been O
reported O
recently O
( O
Boulay O
, O
F O
. O
, O
Tardif O
, O
M O
. O
, O
Brouchon O
, O
L O
. O
, O
and O
Vignais O
, O
P O
. O

In O
NIH O
3T3 O
fibroblasts O
the O
high O
affinity O
IL B
- I
2R I
bearing O
a O
deletion O
of O
a O
region O
rich O
in O
acidic O
amino O
acids O
( O
the O
" O
acidic O
" O
region O
) O
in O
the O
IL B
- I
2R I
beta I
- I
chain I
failed O
to O
induce O
the O
tyrosine O
phosphorylation O
of O
MAP B
kinase I
as O
well O
as O
the O
expression O
of O
the O
all O
three O
nuclear O
proto O
- O
oncogenes O
. O

A O
small O
molecule O
ILK B
inhibitor O
suppresses O
the O
phosphorylation O
of O
PKB B
at O
the O
Ser O
- O
473 O
but O
not O
the O
Thr O
- O
308 O
site O
in O
the O
PTEN B
mutant I
cells O
. O

The O
avian O
carcinoma O
virus O
MH2 O
contains O
a O
hybrid O
gene O
delta B
gag I
- O
mht B
with O
a O
contiguous O
open O
reading O
frame O
of O
2682 O
base O
pairs O
as O
well O
as O
v B
- I
myc I
and O
avian O
helper O
virus O
- O
related O
sequences O
. O
delta B
gag I
is O
a O
partial O
retroviral B
core I
protein I
gene I
while O
v B
- I
mht I
and O
v B
- I
myc I
are O
cell O
- O
drived O
sequences O
. O

The O
results O
in O
the O
untreated O
patients O
demonstrate O
the O
primary O
importance O
of O
bulk O
reduction O
at O
initial O
laparotomy O
. O

Of O
3841 O
serum O
samples O
from O
sows O
received O
from O
the O
Tennessee O
State O
Diagnostic O
Laboratory O
in O
1991 O
- O
1992 O
, O
1130 O
were O
positive O
for O
Toxoplasma O
gondii O
antibody O
. O

These O
findings O
also O
demonstrate O
that O
unless O
excluded O
by O
other O
factors O
, O
the O
C B
proteins I
are O
likely O
to O
be O
located O
along O
the O
length O
of O
nascent O
transcripts O
. O

In O
this O
group O
of O
patients O
, O
the O
mean O
LH B
( O
9 O
. O
3 O
+ O
/ O
- O
5 O
. O
9 O
IU O
/ O
l O
) O
and O
sex B
- I
hormone I
binding I
globulin I
( O
SHBG B
) O
( O
54 O
. O
5 O
+ O
/ O
- O
22 O
. O
9 O
nmol O
/ O
l O
) O
concentrations O
were O
significantly O
greater O
than O
those O
of O
five O
normal O
control O
subjects O
( O
4 O
. O
7 O
+ O
/ O
- O
1 O
. O
11 O
IU O
/ O
l O
and O
26 O
. O
0 O
+ O
/ O
- O
7 O
. O
0 O
nmol O
/ O
l O
respectively O
) O
. O

We O
have O
now O
completed O
the O
primary O
structure O
of O
fibrillin B
, O
elucidated O
the O
exon O
/ O
intron O
organization O
of O
the O
gene O
and O
derived O
a O
physical O
map O
of O
the O
genetic O
locus O
. O

The O
BCL B
- I
6 I
POZ O
domain O
and O
other O
POZ O
domains O
interact O
with O
the O
co O
- O
repressors O
N B
- I
CoR I
and O
SMRT B
. O

Cytokine O
inducibility O
of O
VCAM1 B
in O
endothelial O
cells O
utilizes O
the O
interaction O
of O
heterodimeric O
p50 B
/ O
p65 B
proteins O
with O
IRF B
- I
1 I
. O

In O
particular O
, O
MF O
males O
receiving O
either O
a O
5 O
. O
0 O
- O
mg O
/ O
kg O
CDP O
dose O
or O
a O
3 O
. O
0 O
- O
mg O
/ O
kg O
RO O
dose O
explored O
the O
object O
more O
often O
than O
MM O
males O
. O
( O
ABSTRACT O
TRUNCATED O
AT O
250 O
WORDS O
) O

To O
gain O
additional O
insights O
into O
this O
novel O
apoptotic O
checkpoint O
, O
we O
have O
now O
characterized O
the O
mouse B
survivin I
locus I
. O

Both O
proteins O
share O
sequence O
similarity O
with O
the O
myelin B
- I
associated I
glycoprotein I
, O
an O
adhesion O
molecule O
of O
oligodendrocytes O
and O
Schwann O
cells O
that O
has O
been O
implicated O
in O
the O
process O
of O
myelination O
, O
raising O
the O
important O
question O
of O
whether O
myelin B
- I
associated I
glycoprotein I
is O
also O
a O
sialic B
acid I
- I
binding I
protein I
. O

Localized O
Bicaudal B
- I
C I
RNA I
encodes O
a O
protein O
containing O
a O
KH B
domain O
, O
the O
RNA O
binding O
motif O
of O
FMR1 B
. O

To O
facilitate O
the O
characterization O
of O
NF B
- I
E2 I
functions O
in O
human O
cells O
, O
we O
isolated O
cDNAs O
encoding O
two O
members O
of O
the O
small B
Maf I
family I
, O
MafK B
and O
MafG B
. O

Indigenous O
microbial O
flora O
and O
the O
large O
intestine O
in O
tadpoles O
. O

Copyright O
1998 O
The O
Association O
for O
the O
Study O
of O
Animal O
Behaviour O
. O

In O
contrast O
to O
the O
restricted O
tissue O
expression O
of O
gonadotropin B
and O
TSH B
receptors I
in O
gonads O
and O
thyroid O
, O
respectively O
, O
LGR4 B
is O
expressed O
in O
diverse O
tissues O
including O
ovary O
, O
testis O
, O
adrenal O
, O
placenta O
, O
thymus O
, O
spinal O
cord O
, O
and O
thyroid O
, O
whereas O
LGR5 B
is O
found O
in O
muscle O
, O
placenta O
, O
spinal O
cord O
, O
and O
brain O
. O

Not O
only O
will O
it O
substantially O
reduce O
incident O
cases O
of O
hepatitis O
B O
for O
the O
next O
decade O
, O
it O
will O
also O
provide O
a O
framework O
for O
the O
successful O
introduction O
of O
future O
adolescent O
vaccine O
initiatives O
in O
Australia O
. O

A O
new O
technique O
for O
the O
identification O
of O
phases O
contained O
within O
a O
polymer O
blend O
is O
described O
in O
this O
paper O
. O

Radioactively O
labeled O
microspheres O
were O
used O
to O
determine O
and O
compare O
the O
hemodynamic O
effects O
of O
sodium O
nitroprusside O
( O
SNP O
) O
, O
nitroglycerin O
( O
NTG O
) O
, O
and O
deep O
enflurane O
anesthesia O
on O
oral O
tissues O
during O
controlled O
hypotension O
when O
compared O
with O
controls O
. O

Their O
central O
projections O
were O
indistinguishable O
from O
those O
of O
control O
axons O
in O
all O
four O
trigeminal O
subnuclei O
. O

The O
JNK B
/ O
SAPK B
activator O
mixed B
lineage I
kinase I
3 I
( O
MLK3 B
) O
transforms O
NIH O
3T3 O
cells O
in O
a O
MEK B
- O
dependent O
fashion O
. O

The O
decision O
support O
system O
uses O
the O
network O
at O
its O
core O
and O
helps O
not O
only O
in O
reaching O
a O
diagnosis O
but O
also O
in O
finding O
the O
optimal O
way O
to O
reach O
that O
diagnosis O
. O

In O
contrast O
to O
the O
effect O
on O
DNA O
replication O
, O
the O
L13V O
substitution O
in O
large B
T I
antigen I
did O
not O
prevent O
complex O
formation O
with O
Hsc70 B
and O
the O
Rb B
protein I
. O

To O
explore O
the O
effect O
of O
persistent O
cardiomegaly O
on O
cardiovascular O
function O
, O
groups O
of O
newborn O
rats O
inhaled O
up O
to O
500 O
ppm O
CO O
for O
33 O
days O
, O
after O
which O
development O
continued O
in O
ambient O
air O
. O

Bandwidth O
of O
auditory O
units O
in O
the O
chick O
forebrain O
( O
field O
L O
/ O
Hv O
complex O
) O
was O
measured O
with O
isointensity O
tone O
stimuli O
. O

Members O
of O
the O
steroid B
/ I
hormone I
nuclear I
receptor I
superfamily I
regulate O
target O
gene O
transcription O
via O
recognition O
and O
association O
with O
specific O
cis O
- O
acting O
sequences O
of O
DNA O
, O
called O
hormone O
response O
elements O
( O
HREs O
) O
. O

Indeed O
, O
DEX O
also O
enhanced O
the O
RA O
- O
dependent O
increase O
in O
RARbeta B
mRNA I
in O
a O
cycloheximide O
- O
sensitive O
manner O
. O

The O
highest O
concentration O
of O
respirable O
mineral O
fibres O
was O
found O
during O
the O
overhaul O
of O
a O
truck O
with O
asbestos O
insulation O
; O
respirable O
fibre O
concentration O
reached O
the O
value O
of O
5 O
f O
/ O
cm3 O
, O
and O
total O
dust O
concentration O
- O
the O
value O
of O
about O
80 O
mg O
/ O
m3 O
. O

Avian B
EMILIN I
was O
extracted O
from O
19 O
- O
day O
- O
old O
embryonic O
chick O
aortas O
and O
associated O
blood O
vessels O
and O
purified O
by O
ion O
- O
exchange O
chromatography O
and O
gel O
filtration O
. O

CYP51P1 B
is O
96 O
. O
5 O
% O
identical O
to O
the O
human B
CYP51 I
coding I
sequence I
and O
is O
not O
interrupted O
with O
introns O
but O
has O
six O
in O
- O
frame O
stop O
codons O
resulting O
from O
point O
mutations O
. O

We O
report O
here O
that O
AChE B
activity O
tends O
to O
decrease O
in O
individuals O
sampled O
in O
tanks O
at O
a O
salinity O
of O
30 O
per O
thousand O
as O
temperature O
increases O
. O

Three O
RasV12 B
mutants I
( O
S35 O
, O
G37 O
, O
and O
C40 O
) O
which O
differ O
by O
their O
ability O
to O
bind O
to O
Ras B
effectors O
( O
Raf B
, O
Ral B
- I
GEFs I
, O
and O
the O
p110 B
subunit I
of O
PI B
3 I
- I
kinase I
, O
respectively O
) O
were O
able O
to O
induce O
sustained O
NR O
cell O
proliferation O
, O
although O
none O
of O
these O
mutants O
was O
reported O
to O
transform O
NIH O
3T3 O
cells O
. O

Mating O
response O
, O
ovulation O
rate O
, O
follicle O
and O
corpus O
luteum O
size O
, O
gestation O
length O
, O
pregnancy O
rate O
, O
lambing O
rate O
, O
and O
lamb O
birth O
weight O
were O
recorded O
. O

Membrane O
extraction O
has O
been O
interfaced O
with O
gas O
chromatography O
and O
mass O
spectroscopy O
for O
the O
analysis O
of O
volatile O
organics O
in O
water O
. O

A O
significant O
difference O
in O
the O
distribution O
of O
antibodies O
to O
thyroglobulin B
and O
thyroid B
peroxidase I
was O
found O
in O
subgroup O
2 O
. O

Here O
, O
we O
characterize O
TCR B
/ O
CD48 B
and O
TCR B
/ O
CD28 B
costimulation O
in O
T O
cells O
expressing O
Lck B
Src B
homology I
3 I
( O
SH3 B
) O
mutants O
. O

High O
expression O
of O
p51A B
( O
TAp63gamma B
) O
in O
the O
skeletal O
muscle O
tissue O
drove O
us O
to O
investigate O
a O
differentiation O
- O
inducible O
myoblastic O
cell O
line O
which O
showed O
increased O
p51A B
expression O
after O
differentiation O
induction O
. O

The O
rate O
for O
Arm O
III O
( O
CAF O
therapy O
) O
was O
61 O
. O
5 O
% O
( O
32 O
/ O
52 O
) O
, O
a O
little O
higher O
than O
that O
for O
the O
other O
two O
. O

These O
results O
induce O
that O
the O
putative O
TATA O
box O
and O
initiator O
are O
not O
involved O
in O
the O
promoter O
activity O
, O
and O
that O
the O
vitronectin B
promoter I
lacks O
the O
TATA O
box O
, O
initiator O
and O
GC O
box O
. O

Using O
the O
interaction O
- O
trap O
assay O
to O
identify O
candidate O
proteins O
that O
bind O
the O
cytoplasmic O
region O
of O
the O
LAR B
transmembrane I
protein I
tyrosine I
phosphatase I
( O
PT B
- I
Pase I
) O
, O
we O
isolated O
a O
cDNA O
encoding O
a O
2861 O
- O
amino O
acid O
protein O
termed O
Trio B
that O
contains O
three O
enzyme O
domains O
: O
two O
functional O
GEF B
domains I
and O
a O
protein B
serine I
/ I
threonine I
kinase I
( O
PSK B
) O
domain O
. O

We O
have O
isolated O
two O
H19 B
cDNAs I
( O
AP B
and O
ES O
) O
that O
contain O
this O
ORF4 O
and O
correspond O
to O
incomplete O
copies O
of O
the O
unique O
2 B
. I
3 I
kb I
H19 I
RNA I
. O

The O
combined O
in O
vitro O
effects O
of O
ethanol O
wih O
methaqualone O
, O
phenobarbital O
, O
pyrazole O
or O
disulfiram O
were O
studied O
using O
rat B
brain I
microsomal I
NA I
- I
K I
- I
ATPase I
. O

The O
related O
adhesion O
focal B
tyrosine I
kinase I
is O
tyrosine O
- O
phosphorylated O
after O
beta1 B
- I
integrin I
stimulation O
in O
B O
cells O
and O
binds O
to O
p130cas B
. O

Earlier O
reports O
have O
localized O
mutations O
which O
affect O
the O
processing O
and O
transport O
of O
herpes B
simplex I
virus I
1 I
glycoproteins I
to O
a O
region O
located O
between O
the O
genes B
specifying O
glycoprotein B
B I
and O
the O
major O
viral O
DNA O
- O
binding O
protein O
( O
beta B
8 I
) O
. O

These O
results O
indicate O
that O
the O
2127 O
- O
bp O
cDNA O
encodes O
a O
functional O
feline B
L I
/ I
B I
/ I
K I
- I
type I
ALP I
expressed O
on O
cell O
surfaces O
via O
phosphatidylinositol O
- O
glycan O
linkage O
. O

Our O
experience O
suggests O
that O
T2 O
weighting O
is O
adequate O
and O
is O
the O
method O
of O
choice O
for O
the O
early O
recognition O
of O
necrosis O
of O
the O
lunate O
. O

Analysis O
of O
a O
Het B
- I
mutation I
in O
Anabaena O
sp O
. O
strain O
PCC O
7120 O
implicates O
a O
secondary O
metabolite O
in O
the O
regulation O
of O
heterocyst O
spacing O
. O

Here O
we O
report O
that O
components O
of O
the O
Ras B
/ O
Raf B
/ O
MAPK B
pathway O
are O
constitutively O
activated O
in O
these O
lck B
- O
transformed O
immature O
thymoblasts O
. O

p56 B
( O
lck B
) O
utilizes O
Shc B
and O
Grb2 B
adaptors I
to O
mediate O
activation O
of O
p21 B
( O
ras B
) O
in O
the O
thymoblast O
lines O
by O
promoting O
tyrosine O
phosphorylation O
of O
the O
Shc B
protein I
and O
constitutive O
interaction O
between O
Shc B
and O
Grb2 B
. O

The O
rec8 B
( I
+ I
) I
, O
rec10 B
( I
+ I
) I
, O
and O
rec11 B
( I
+ I
) I
genes I
of O
the O
fission O
yeast O
Schizosaccharomyces O
pombe O
exhibit O
similar O
specificities O
for O
meiotic O
recombination O
and O
rec8 B
( I
+ I
) I
is O
required O
for O
sister O
chromatid O
cohesion O
and O
homolog O
pairing O
. O

Based O
on O
the O
results O
of O
this O
analysis O
, O
we O
also O
predict O
that O
the O
budding B
yeast I
arsenate I
resistance I
protein I
Acr2 I
and O
the O
ORF O
Ygr203w B
encode O
protein O
phosphatases O
with O
catalytic O
properties O
similar O
to O
that O
of O
the O
Cdc25 B
family I
. O

At O
onset O
of O
first O
post O
- O
weaning O
estrus O
, O
sows O
received O
either O
an O
intravulval O
injection O
of O
3 O
. O
75 O
mg O
of O
prostaglandin O
analogue O
( O
PGF O
) O
or O
, O
served O
as O
a O
non O
- O
injected O
control O
( O
CON O
) O
. O

We O
concluded O
that O
HFV O
can O
achieve O
values O
of O
CO2 O
elimination O
close O
to O
the O
estimated O
metabolic O
CO2 O
production O
in O
normal O
unintubated O
subjects O
over O
short O
periods O
of O
time O
. O

The O
PHO5 B
gene I
is O
activated O
by O
the O
Pho4p B
transcription I
factor I
, O
which O
itself O
is O
negatively O
regulated O
through O
phosphorylation O
by O
the O
products O
of O
PHO80 B
and O
PHO85 B
. O

Although O
the O
only O
authoritative O
way O
to O
determine O
the O
effect O
of O
diet O
on O
urinary O
pH O
is O
a O
feeding O
trial O
, O
there O
is O
no O
universally O
accepted O
protocol O
for O
measuring O
urinary O
pH O
. O

Analysis O
of O
the O
DNA O
sequence O
upstream O
of O
the O
narQ B
gene I
, O
which O
encodes O
the O
second O
nitrate O
- O
responsive O
sensor O
- O
transmitter O
protein O
in O
Escherichia O
coli O
, O
revealed O
an O
open O
reading O
frame O
( O
ORF O
) O
whose O
product O
shows O
a O
high O
degree O
of O
similarity O
to O
a O
number O
of O
iron B
- I
sulfur I
proteins I
as O
well O
as O
to O
the O
beta O
subunit O
of O
glutamate B
synthase I
( O
gltD B
) O
of O
E O
. O
coli O
. O

Endothelial O
purine O
content O
. O

We O
suggest O
that O
the O
responses O
to O
types B
I I
and I
II I
collagen I
were O
caused O
by O
a O
cross O
- O
reaction O
with O
type B
III I
collagen I
peptides I
. O

Possibilities O
and O
limints O
of O
immunofluorescence O
in O
the O
laboratory O
diagnosis O
of O
rabies O

The O
5 O
' O
half O
of O
the O
EWS B
gene I
has O
recently O
been O
described O
to O
be O
fused O
to O
the O
3 O
' O
regions O
of O
genes O
encoding O
the O
DNA O
- O
binding O
domain O
of O
several O
transcriptional O
regulators O
, O
including O
ATF1 B
, O
FLI B
- I
1 I
, O
and O
ERG B
, O
in O
several O
human O
tumors O
. O

PDGF B
stimulated O
CDK2 B
activity O
in O
mesangial O
cells O
and O
decreased O
the O
level O
of O
p27 B
( O
kip1 B
) O
cyclin B
kinase O
inhibitor O
protein O
. O

Recombinant B
apoA I
- I
I I
protein I
recovered O
from O
the O
soluble O
fraction O
of O
the O
bacterial O
cell O
pellet O
was O
purified O
to O
greater O
than O
95 O
% O
homogeneity O
by O
reversed O
- O
phase O
high O
- O
performance O
liquid O
chromatography O
. O

The O
influence O
of O
different O
temperatures O
between O
13 O
degrees O
C O
and O
45 O
degrees O
C O
on O
coagulation O
factors O
in O
vitro O
was O
studied O
by O
measuring O
clotting O
time O
with O
the O
recalcification O
time O
, O
partial O
thromboplastin B
time O
( O
PTT O
) O
, O
and O
thromboplastin B
time O
test O
. O

On O
average O
, O
rCMRglc O
values O
were O
23 O
% O
below O
control O
values O
for O
all O
regions O
studied O
, O
with O
the O
greatest O
differences O
in O
posterior O
brain O
regions O
( O
visual O
association O
cortex O
, O
primary O
visual O
cortex O
, O
and O
parietal O
cortex O
) O
and O
thalamus O
. O

Thus O
, O
transgene O
expression O
directed O
by O
both O
the O
human O
and I
mouse I
Rb I
promoters I
is O
restricted O
to O
a O
subset O
of O
tissues O
in O
which O
Rb B
is O
normally O
expressed O
during O
embryogenesis O
. O

Thus O
, O
Tax1 B
activates O
CArG O
- O
mediated O
transcription O
without O
mitogenic O
signals O
through O
interaction O
with O
a O
CArG B
- I
binding I
factor I
, O
p67SRF B
. O

In O
experiment O
1 O
, O
subjects O
were O
required O
to O
discriminate O
male O
from O
female O
faces O
and O
no O
hemispheric O
asymmetries O
were O
found O
. O

Membrane O
depolarization O
is O
a O
critical O
element O
of O
neuronal O
signaling O
. O

The O
transcription O
start O
site O
was O
localized O
224 O
bp O
upstream O
the O
ATG O
codon O
by O
RNase B
protection O
and O
primer O
extension O
mapping O
. O

All O
the O
cognate O
gene O
clones O
were O
constructed O
, O
using O
either O
PCR O
products O
amplified O
from O
genomic O
DNA O
, O
or O
gap O
- O
repair O
. O

Rising O
antibody O
titres O
to O
the O
astrovirus O
particles O
were O
demonstrated O
in O
one O
child O
, O
and O
IgM B
was O
also O
demonstrated O
in O
this O
patient O
' O
s O
serum O
. O

Effect O
of O
isobarin O
on O
electrocardiographic O
indices O
in O
hypertensive O
disease O

Although O
RAD17 B
, O
RAD24 B
and O
MEC3 B
are O
not O
required O
for O
cell O
cycle O
arrest O
when O
S O
phase O
is O
inhibited O
by O
hydroxyurea O
( O
HU O
) O
, O
they O
do O
contribute O
to O
the O
viability O
of O
yeast O
cells O
grown O
in O
the O
presence O
of O
HU O
, O
possibly O
because O
they O
are O
required O
for O
the O
repair O
of O
HU O
- O
induced O
DNA O
damage O
. O

Long O
chain O
acyl O
- O
CoA O
esters O
are O
important O
intermediates O
in O
degradation O
and O
synthesis O
of O
fatty O
acids O
, O
as O
well O
as O
having O
important O
functions O
in O
regulation O
of O
intermediary O
metabolism O
and O
gene O
expression O
. O

PTP1 B
- O
lacZ B
studies O
indicate O
that O
PTP1 B
is O
spatially O
localized O
to O
prestalk O
and O
anterior O
- O
like O
cell O
types O
. O

When O
translated O
in O
- O
frame O
with O
PKC B
zeta I
, O
a O
stop O
codon O
is O
located O
28 O
amino O
acids O
towards O
the O
N O
- O
terminus O
of O
the O
divergence O
point O
and O
the O
intervening O
sequence O
lacks O
an O
expected O
initiating O
methionine O
. O
psi B
PKC I
zeta I
is O
non O
- O
functional O
in O
terms O
of O
protein O
synthesis O
since O
Western O
blotting O
with O
an O
antibody O
directed O
against O
the O
C O
- O
terminus O
of O
PKC B
zeta I
failed O
to O
reveal O
a O
protein O
smaller O
than O
PKC B
zeta I
, O
and O
synthetic B
psi I
PKC I
zeta I
RNA I
failed O
to O
support O
protein O
synthesis O
in O
a O
translation O
system O
in O
vitro O
. O

W O
. O
, O
and O
Touster O
, O
O O
. O

Electrically O
induced O
undulations O
and O
their O
competition O
with O
electrically O
induced O
convection O
in O
cholesteric O
liquid O
crystals O
. O

The O
sequence O
immediately O
upstream O
of O
the O
translation O
start O
site O
was O
G O
+ O
C O
rich O
( O
greater O
than O
75 O
% O
) O
and O
contained O
a O
consensus O
CCAAT O
sequence O
despite O
the O
absence O
of O
a O
TATA O
box O
. O

These O
conditions O
may O
determine O
an O
increase O
in O
the O
level O
of O
indoor O
pollutants O
( O
tobacco O
smoke O
, O
gases O
produced O
by O
cooling O
processes O
etc O
. O
) O
and O
of O
allergens O
derived O
from O
mites O
, O
domestic O
animals O
and O
cockroaches O
. O

Nevertheless O
, O
lower O
doses O
which O
alone O
were O
lacking O
in O
activity O
( O
100 O
- O
250 O
mg O
/ O
kg O
B1 O
and O
B6 O
, O
1 O
- O
2 O
. O
5 O
mg O
/ O
kg O
B12 O
p O
. O
o O
. O
) O
dose O
- O
dependently O
potentiated O
the O
antinociceptive O
of O
diclofenac O
. O

For O
O3 O
, O
the O
correlation O
between O
personal O
exposures O
and O
ambient O
levels O
was O
weakest O
in O
the O
winter O
for O
residential O
microenvironments O
( O
rs O
= O
0 O
. O
05 O
, O
p O
> O
0 O
. O
05 O
) O
, O
and O
was O
strongest O
in O
the O
summer O
for O
outdoor O
near O
- O
roadway O
microenvironments O
( O
rs O
= O
0 O
. O
91 O
, O
p O
< O
0 O
. O
05 O
) O
. O

Changes O
in O
water O
and O
electrolyte O
content O
of O
the O
brain O
and O
edema O
formation O
after O
acute O
, O
drug O
- O
induced O
hypertension O
were O
studied O
in O
albino O
rabbits O
. O

Like O
Rev B
- O
Erb B
, O
BD73 B
binds O
as O
a O
monomer O
to O
a O
DNA O
sequence O
which O
consists O
of O
a O
specific O
A O
/ O
T O
- O
rich O
sequence O
upstream O
of O
the O
consensus O
hexameric O
half O
- O
site O
specified O
by O
the O
P O
box O
of O
the O
DNA O
- O
binding O
domain O
. O

In O
contrast O
, O
the O
holo B
- I
dTFIIA I
( I
L I
/ I
S I
) I
binds O
TBP B
with O
high O
affinity O
. O

Overexpression O
of O
wild O
type O
and O
SeCys O
/ O
Cys O
mutant O
of O
human B
thioredoxin I
reductase I
in O
E O
. O
coli O
: O
the O
role O
of O
selenocysteine O
in O
the O
catalytic O
activity O
. O

Biopsy O
of O
the O
lesion O
revealed O
granulomatous O
inflammation O
and O
numerous O
septate O
hyphae O
. O

Western O
and O
immunocytochemical O
analysis O
implied O
that O
PREB B
accumulates O
specifically O
in O
GH3 O
cell O
nuclei O
. O

Two O
genes O
from O
the O
family O
encoding O
mouse B
ribosomal I
protein I
S16 I
were O
cloned O
, O
sequenced O
, O
and O
analyzed O
. O

A O
novel O
serine B
kinase I
activated O
by O
rac1 B
/ O
CDC42Hs B
- O
dependent O
autophosphorylation O
is O
related O
to O
PAK65 B
and O
STE20 B
. O

The O
deduced O
gene O
product O
was O
found O
to O
have O
significant O
sequence O
similarity O
to O
the O
yeast B
and I
prokaryotic I
RNA I
polymerase I
subunits I
involved O
with O
subunit O
assembly O
. O

Maximum O
overexpression O
of O
holoenzyme O
activity O
was O
achieved O
by O
the O
inclusion O
in O
such O
plasmids O
of O
Salmonella B
typhimurium I
cysG I
, O
which O
encodes O
a O
uroporphyrinogen B
III I
methyltransferase I
required O
for O
the O
synthesis O
of O
siroheme O
, O
a O
cofactor O
for O
the O
hemoprotein O
. O

Overexpression O
of O
SNURF B
in O
cultured O
mammalian O
cells O
enhanced O
not O
only O
androgen O
, O
glucocorticoid B
, O
and O
progesterone I
receptor I
- O
dependent O
transactivation O
but O
also O
basal O
transcription O
from O
steroid O
- O
regulated O
promoters O
. O

The O
human O
protein O
binds O
most O
strongly O
to O
the O
SH3 B
domain I
from O
the O
abl B
proto I
- I
oncogene I
. O

Suprapubic O
and O
intravesical O
methods O

These O
data O
show O
that O
tyrosine O
phosphorylation O
by O
FES B
affects O
the O
interaction O
of O
BCR B
with O
multiple O
signaling O
partners O
and O
suggest O
a O
general O
role O
for O
BCR B
in O
non B
- I
receptor I
protein I
- I
tyrosine I
kinase I
regulation O
and O
signal O
transduction O
. O

We O
have O
shown O
that O
the O
human O
cell O
MRC O
closely O
resembles O
the O
murine O
cell O
MRC O
, O
in O
both O
its O
protein O
composition O
and O
its O
fractionation O
and O
chromatographic O
profile O
. O

From O
1977 O
to O
1985 O
, O
42 O
patients O
with O
squamous O
cell O
carcinoma O
of O
the O
anal O
canal O
were O
treated O
with O
mitomycin O
C O
( O
15 O
mg O
/ O
m2 O
) O
and O
5 O
- O
fluorouracil O
( O
750 O
mg O
/ O
m2 O
) O
on O
day O
1 O
, O
5 O
- O
FU O
( O
750 O
mg O
/ O
m2 O
/ O
d O
) O
alone O
on O
days O
2 O
to O
5 O
, O
and O
radiation O
therapy O
( O
3000 O
cGy O
) O
on O
days O
7 O
to O
28 O
. O

For O
any O
given O
age O
, O
high O
- O
stressed O
plantaris O
tendons O
were O
of O
a O
higher O
fatigue O
quality O
than O
low O
- O
stressed O
extensor O
tendons O
. O

In O
addition O
to O
TAK1 B
, O
TGF B
- I
beta I
also O
stimulated O
JNK B
activity O
. O

Foamy O
viruses O
( O
FVs O
) O
express O
the O
Gag B
protein I
as O
a O
precursor O
with O
a O
molecular O
mass O
of O
74 O
kDa O
( O
pr74 B
) O
from O
which O
a O
70 O
- O
kDa O
protein O
( O
p70 B
) O
is O
cleaved O
by O
the O
viral B
protease I
. O

Our O
results O
suggest O
that O
at O
least O
some O
chloroplast O
- O
like O
tRNA O
genes O
in O
wheat O
mtDNA O
are O
transcribed O
, O
with O
transcripts O
undergoing O
processing O
, O
post O
- O
transcriptional O
modification O
and O
3 O
' O
- O
CCA O
addition O
, O
to O
produce O
mature O
tRNAs O
that O
may O
participate O
in O
mitochondrial O
protein O
synthesis O
. O

If O
, O
however O
, O
myocardial O
stunning O
is O
severe O
, O
and O
it O
involves O
large O
parts O
of O
the O
LV O
and O
thus O
impairs O
global O
LV O
function O
, O
it O
can O
be O
reversed O
with O
inotropic O
agents O
and O
procedures O
. O

268 O
, O
6858 O
- O
6861 O
) O
. O

The O
occurrence O
of O
the O
different O
types O
of O
TCRB B
rearrangement I
patterns O
has O
implications O
for O
PCR O
- O
based O
clonality O
assessment O
and O
for O
PCR O
- O
based O
detection O
of O
minimal O
residual O
disease O
via O
TCRB B
gene I
analysis O
. O

Under O
control O
conditions O
, O
pretreatment O
with O
lithium O
during O
7 O
days O
did O
not O
modify O
the O
hyperlocomotion O
produced O
by O
d O
- O
amphetamine O
. O

Six O
patients O
underwent O
an O
electrophysiologic O
study O
. O

Rats O
on O
the O
multiple O
oral O
dosage O
regimen O
were O
given O
unlabelled O
HMCF O
in O
their O
drinking O
water O
for O
13 O
days O
before O
the O
administration O
of O
a O
bolus O
dose O
of O
[ O
14C O
] O
HMCF O
on O
day O
14 O
. O

After O
multivariate O
analysis O
, O
TWA O
correlated O
with O
age O
( O
P O
= O
0 O
. O
02 O
) O
and O
LV O
function O
( O
P O
= O
0 O
. O
002 O
) O
and O
occurred O
more O
often O
in O
patients O
after O
nonanterior O
MI O
( O
P O
= O
0 O
. O
03 O
) O
. O

No O
patient O
demonstrated O
a O
decrease O
in O
bone O
marrow O
fibrosis O
as O
determined O
by O
serial O
procollagen B
( O
PC B
III I
) O
serum O
level O
analysis O
. O

Asthma O
diagnoses O
were O
made O
according O
to O
recommended O
National O
Asthma O
Expert O
Panel O
Guidelines O
. O

Charcot O
joints O
. O

Idealized O
versus O
realized O
overall O
treatment O
times O
. O

Non O
- O
suppressed O
thyroidal O
radioactive O
iodine O
uptake O
( O
RAIU O
) O
in O
thyrotoxic O
phase O
in O
a O
case O
of O
subacute O
thyroiditis O
with O
thyroid B
- I
stimulating I
antibodies I
( O
TSAb B
) O
. O

Ultra O
- O
energy O
( O
UHE O
) O
imaging O
is O
usually O
performed O
in O
simultaneous O
F O
- O
18 O
FDG O
/ O
Tc O
- O
99m O
MIBI O
studies O
. O

Bilateral O
renal O
oncocytoma O
in O
a O
Greyhound O
dog O
. O

Uncoupling O
gene O
activity O
from O
chromatin O
structure O
: O
promoter O
mutations O
can O
inactivate O
transcription O
of O
the O
yeast B
HSP82 I
gene I
without O
eliminating O
nucleosome O
- O
free O
regions O
. O

Ectopic O
expression O
of O
cyclin B
D1 I
in O
progestin O
- O
inhibited O
cells O
led O
to O
the O
reappearance O
of O
the O
120 O
- O
kDa O
active O
form O
of O
cyclin B
E I
- O
Cdk2 B
preceding O
the O
resumption O
of O
cell O
cycle O
progression O
. O

Unlike O
these O
contaminant O
- O
responsive O
T O
cells O
, O
those O
that O
are O
truly O
specific O
for O
natural O
AChR B
epitopes I
appear O
less O
heterogeneous O
and O
therefore O
more O
suitable O
targets O
for O
selective O
immunotherapy O
. O

Because O
the O
wa B
- I
2 I
mutation I
was O
mapped O
previously O
to O
the O
vicinity O
of O
the O
EGF B
/ I
TGF I
- I
alpha I
receptor I
( O
EGFR B
) O
gene O
on O
mouse O
chromosome O
11 O
, O
we O
hypothesized O
that O
the O
wa B
- I
2 I
phenotype O
might O
result O
from O
a O
defect O
in O
either O
the O
expression O
or O
activity O
of O
EGFR B
, O
or O
both O
. O

Region O
I O
includes O
the O
two O
cysteine O
- O
cysteine O
zinc O
fingers O
that O
comprise O
a O
DNA O
- O
binding O
domain O
which O
typifies O
all O
members O
of O
the O
superfamily O
. O

Southern O
zoo O
blot O
analysis O
indicated O
that O
ZNF236 B
is O
conserved O
in O
the O
genomes O
of O
all O
mammalian O
species O
tested O
, O
but O
not O
in O
yeast O
. O

RNA O
determinants O
required O
for O
L4 B
- O
mediated O
attenuation O
control O
of O
the O
S10 B
r I
- I
protein I
operon I
of I
Escherichia I
coli I
. O

Spodoptera O
frugiperda O
( O
Sf9 O
) O
cells O
have O
proved O
a O
suitable O
cell O
system O
in O
which O
to O
study O
this O
association O
and O
to O
produce O
recombinant B
CR3 I
, O
and O
we O
show O
here O
that O
another O
lepidopteran O
cell O
line O
, O
Trichoplusia O
niTN O
- O
5B1 O
- O
4 O
( O
High O
- O
Five O
) O
cells O
, O
allows O
the O
recovery O
of O
large O
amounts O
of O
functional B
recombinant I
CR3 I
. O

INTERVENTIONS O
: O
Postoperative O
follow O
- O
up O
consisted O
of O
serial O
determination O
of O
different O
biochemical O
markers O
( O
CK B
, O
CK B
- I
MB I
, O
cTnI B
) O
, O
ECGs O
, O
and O
echocardiography O
. O

At O
the O
first O
phase O
( O
1 O
- O
43 O
ms O
for O
the O
VMN O
and O
1 O
- O
10 O
ms O
for O
the O
LN O
) O
the O
hypothalamic O
- O
cortical O
responses O
completely O
inhibited O
the O
formation O
of O
the O
VC O
response O
to O
the O
light O
stimulus O
. O

6 O
, O
320 O
cells O
/ O
mm3 O
, O
respectively O
; O
P O
less O
than O
. O
005 O
) O
; O
twelve O
( O
86 O
% O
) O
of O
the O
14 O
inmates O
who O
developed O
AIDS O
had O
counts O
of O
less O
than O
5 O
, O
000 O
cells O
/ O
mm3 O
, O
compared O
with O
only O
six O
( O
14 O
% O
) O
of O
the O
42 O
controls O
( O
P O
less O
than O
. O
00001 O
) O
. O

There O
is O
, O
however O
, O
uncertainty O
as O
to O
how O
results O
obtained O
in O
recent O
experiments O
scale O
up O
to O
landscape O
and O
regional O
levels O
and O
generalize O
across O
ecosystem O
types O
and O
processes O
. O

The O
facilitation O
started O
preceding O
the O
onset O
of O
electromyographic O
activity O
of O
the O
masseter O
muscle O
. O

The O
recruitment O
of O
constitutively O
phosphorylated O
p185 B
( O
neu B
) O
and O
the O
activated O
mitogenic O
pathway O
proteins O
to O
this O
membrane O
- O
microfilament O
interaction O
site O
provides O
a O
physical O
model O
for O
integrating O
the O
assembly O
of O
the O
mitogenic O
pathway O
with O
the O
transmission O
of O
growth O
factor O
signal O
to O
the O
cytoskeleton O
. O

Deletion O
of O
the O
region O
from O
amino O
acid O
residues O
2 O
- O
67 O
in O
E1A B
, O
which O
has O
been O
postulated O
to O
interact O
with O
p300 B
/ O
CBP B
, O
also O
abolished O
the O
inhibitory O
effect O
of O
E1A B
, O
whereas O
deletion O
of O
the O
region O
from O
residues O
120 O
to O
140 O
had O
no O
effect O
. O

Quantitative O
Tl O
- O
201 O
analysis O
after O
stress O
has O
also O
shown O
viable O
myocardium O
in O
most O
mild O
to O
moderate O
( O
51 O
% O
to O
85 O
% O
of O
normal O
uptake O
) O
irreversible O
Tl O
- O
201 O
defects O
. O

The O
point O
mutation O
Asp O
- O
316 O
- O
- O
> O
Asn O
in O
the O
C O
- O
terminus O
of O
p38 B
, O
analogous O
to O
the O
ERK2 B
( O
extracellular B
- I
signal I
- I
regulated I
kinase I
2 I
) O
sevenmaker O
mutation O
, O
dramatically O
decreases O
its O
binding O
to O
MKP B
- I
1 I
and O
substantially O
compromises O
its O
stimulatory O
effect O
on O
the O
catalytic O
activity O
of O
this O
phosphatase O
. O

J O
. O

Causal O
modeling O
combines O
theory O
and O
research O
, O
and O
because O
the O
interpretation O
of O
data O
is O
possible O
only O
within O
the O
context O
of O
the O
proposed O
theory O
, O
it O
offers O
an O
important O
method O
for O
advancing O
the O
science O
while O
maintaining O
the O
specificity O
of O
the O
practice O
. O

In O
one O
trial O
, O
116 O
subjects O
with O
transfusion O
- O
related O
chronic O
hepatitis O
C O
were O
treated O
with O
lymphoblastoid B
interferon I
( O
5 O
MU O
/ O
m2 O
three O
times O
a O
week O
for O
2 O
mo O
, O
then O
3 O
MU O
/ O
m2 O
three O
times O
a O
week O
for O
4 O
or O
10 O
mo O
) O
. O

A O
derived O
RA O
- O
resistant O
line O
, O
NT2 O
/ O
D1 O
- O
R1 O
, O
is O
deficient O
in O
this O
activity O
and O
is O
co O
- O
resistant O
to O
cisplatin O
. O

BCR B
- O
ABL B
elicits O
transformation O
of O
both O
fibroblast O
and O
hematopoietic O
cells O
and O
blocks O
apoptosis O
following O
cytokine O
deprivation O
in O
various O
factor O
- O
dependent O
cells O
. O

Aggregation O
in O
washed O
platelets O
from O
rats O
with O
diabetes O
was O
enhanced O
. O

Sequence O
analysis O
of O
this O
region O
showed O
it O
contained O
five O
potential O
copies O
of O
the O
sterol O
regulatory O
element O
( O
SRE O
- O
1 O
) O
( O
Smith O
, O
J O
. O
R O
. O
, O
Osborne O
, O
T O
. O
F O
. O
, O
Brown O
, O
M O
. O
S O
. O
, O
Goldstein O
, O
J O
. O
L O
. O
, O
and O
Gil O
, O
G O
. O

Mutations O
in O
Saccharomyces O
cerevisiae O
that O
block O
meiotic O
prophase O
chromosome O
metabolism O
and O
confer O
cell O
cycle O
arrest O
at O
pachytene O
identify O
two O
new O
meiosis O
- O
specific O
genes O
SAE1 B
and O
SAE3 B
. O

Isolation O
and O
characterization O
of O
human O
orthologs O
of O
yeast O
CCR4 B
- O
NOT B
complex O
subunits O
. O

Thus O
, O
the O
i B
- I
leader I
protein I
is O
a O
viral O
gene O
product O
of O
unknown O
function O
and O
high O
stability O
that O
is O
made O
in O
large O
quantities O
at O
intermediate O
times O
of O
productive O
infection O
. O
( O
ABSTRACT O
TRUNCATED O
AT O
400 O
WORDS O
) O

CONCLUSIONS O
: O
Latanoprost O
administered O
once O
daily O
was O
significantly O
more O
effective O
in O
reducing O
IOP O
compared O
with O
unoprostone O
administered O
twice O
daily O
in O
patients O
with O
POAG O
and O
OH O
. O

The O
molecular O
dynamics O
simulations O
provide O
insight O
into O
the O
conformational O
flexibility O
of O
these O
analogs O
. O

In O
alfalfa O
, O
the O
insecticides O
caused O
significant O
mortality O
to O
most O
of O
the O
insects O
evaluated O
. O

Therefore O
, O
we O
have O
reevaluated O
the O
age O
- O
related O
changes O
in O
serum B
leptin I
levels O
and O
their O
relationship O
with O
adiposity O
and O
androgen O
levels O
in O
a O
large O
group O
of O
community O
dwelling O
men O
. O

Injections O
of O
PD O
solutions O
with O
13 O
. O
6 O
, O
22 O
. O
7 O
, O
and O
38 O
. O
6 O
g O
/ O
liter O
of O
glucose O
reduced O
the O
ingestion O
of O
sucrose O
by O
12 O
. O
4 O
% O
, O
23 O
. O
6 O
% O
and O
36 O
. O
1 O
% O
, O
respectively O
, O
but O
did O
not O
affect O
the O
ingestion O
of O
protein O
. O

It O
is O
recommended O
that O
area O
correction O
be O
attempted O
in O
bioequivalence O
studies O
of O
drugs O
where O
high O
intrasubject O
variability O
in O
clearance O
is O
known O
or O
suspected O
. O

Expression O
of O
the O
E2 B
protein I
resulted O
in O
rapid O
repression O
of O
HPV B
E6 I
and O
E7 B
expression O
, O
followed O
approximately O
12 O
h O
later O
by O
profound O
inhibition O
of O
cellular O
DNA O
synthesis O
. O

Plasma O
renin I
activity O
in O
end O
- O
stage O
kidney O
disease O
. O

Immunoblotting O
with O
antiphosphotyrosine B
antibody I
showed O
that O
many O
yeast O
proteins O
, O
including O
the O
p34CDC28 B
kinase I
, O
became O
phosphorylated O
at O
tyrosine O
in O
cells O
expressing O
v B
- I
src I
. O

Renal O
cell O
carcinoma O
induced O
Coombs O
negative O
autoimmune O
hemolytic O
anemia O
and O
severe O
thrombocytopenia O
responsive O
to O
nephrectomy O
. O

Pretreatment O
of O
rats O
with O
different O
dose O
levels O
of O
CCl4 O
resulted O
in O
a O
prolongation O
of O
TMO O
half O
- O
life O
, O
and O
increase O
of O
the O
area O
under O
the O
curve O
( O
AUC O
) O
, O
and O
a O
decrease O
of O
clearance O
, O
but O
the O
apparent O
volume O
of O
distribution O
( O
Vd O
) O
was O
not O
significantly O
decreased O
. O

In O
cells O
co O
- O
expressing O
high O
levels O
of O
the O
p38 B
( I
MAPK I
) I
kinase I
( O
MKK3 B
) O
together O
with O
the O
p38 B
( O
MAPK B
) O
, O
a O
significant O
inhibition O
of O
mitogen O
- O
induced O
cyclin B
D1 I
expression O
was O
observed O
. O

In O
patients O
autografted O
in O
CR1 O
, O
the O
transplant O
- O
related O
mortality O
( O
TRM O
) O
was O
15 O
+ O
/ O
- O
4 O
% O
, O
the O
relapse O
incidence O
( O
RI O
) O
was O
58 O
+ O
/ O
- O
5 O
% O
, O
the O
leukaemia O
- O
free O
survival O
( O
LFS O
) O
was O
36 O
+ O
/ O
- O
5 O
% O
and O
the O
overall O
survival O
was O
47 O
+ O
/ O
- O
5 O
% O
at O
3 O
years O
. O

Administration O
of O
FR O
34235 O
reduced O
aortic O
blood O
pressure O
and O
increased O
cardiac O
output O
in O
anesthetized O
dogs O
with O
an O
ameroid O
- O
induced O
coronary O
artery O
occlusion O
. O

The O
primary O
conditions O
for O
acceptable O
hospital O
use O
were O
that O
exhaled O
- O
volume O
monitoring O
be O
performed O
for O
all O
patients O
and O
that O
O2 O
monitoring O
be O
performed O
both O
when O
setting O
FIO2 O
and O
continuously O
when O
FIO2 O
levels O
are O
critical O
. O

The O
RHO1 B
gene I
encodes O
a O
homolog O
of O
the O
mammalian B
RhoA I
small I
GTP I
- I
binding I
protein I
in O
the O
yeast O
Saccharomyces O
cerevisiae O
. O

As O
regards O
the O
short O
stature O
, O
they O
have O
proved O
that O
the O
syndrome O
is O
related O
to O
low O
levels O
of O
somatomedin B
C I
( O
SmC B
) O
. O

2 O
. O

Menstrual O
and O
lunar O
cycles O
. O

HBO O
had O
marked O
effects O
on O
these O
enzymes O
: O
lung O
SOD B
increased O
( O
guinea O
pigs O
47 O
% O
, O
rats O
88 O
% O
) O
and O
CAT B
and O
GSHPx B
activities O
decreased O
( O
33 O
% O
) O
in O
brain O
and O
lung O
. O

Smectic O
- O
A O
ordering O
at O
a O
liquid O
- O
vapor O
interface O
. O

Phylogenetic O
analysis O
of O
yeast O
, O
invertebrate O
, O
and O
multiple O
mammalian B
isoforms I
of O
SNF4 B
shows O
that O
the O
gene O
duplication O
likely O
occurred O
early O
in O
the O
metazoan O
lineage O
, O
as O
the O
protein O
products O
of O
the O
different O
loci O
are O
relatively O
divergent O
. O

Hard O
methacrylic O
polymers O
. O

Thus O
the O
Hi O
- O
RARE O
represents O
a O
new O
type O
of O
RA O
response O
element O
with O
a O
role O
in O
the O
modulation O
of O
the O
expression O
of O
MHC B
class I
1 I
family I
genes I
. O

We O
found O
multiple O
transcription O
start O
sites O
located O
within O
a O
15 O
- O
base O
pair O
region O
, O
205 O
base O
pairs O
upstream O
of O
the O
translation O
start O
codon O
. O

During O
the O
last O
three O
days O
of O
the O
study O
, O
mean O
urine O
osmolality O
( O
Uosm O
) O
and O
free O
water O
reabsorption O
( O
TCH2O O
) O
increased O
significantly O
: O
[ O
formula O
: O
see O
text O
] O
. O

The O
structural O
intestinal O
defects O
are O
presumed O
to O
be O
the O
result O
of O
defective O
collagen B
synthesis O
in O
these O
hereditary O
connective O
tissue O
disorders O
. O

Rev O
. O

Although O
indomethacin O
is O
useful O
for O
examining O
the O
role O
of O
cyclooxygenase B
products O
in O
asthmatic O
responses O
, O
it O
should O
not O
be O
considered O
in O
the O
treatment O
of O
asthma O
. O

Incidence O
of O
beta O
- O
thalassemia O
carriers O
and O
those O
deficient O
in O
erythrocyte O
glucose B
- I
6 I
- I
phosphate I
dehydrogenase I
in O
the O
greater O
Buenos O
Aires O
area O

Both O
v B
- I
Myb I
and O
c B
- I
Myb I
bind O
specifically O
to O
delta B
E3 I
. O

Anatomical O
considerations O
in O
transsphenoidal O
hypophysectomy O
. O

The O
salt O
sensitivity O
of O
the O
interaction O
indicated O
that O
two O
ion O
pairs O
are O
involved O
in O
the O
association O
of O
Zn2 O
+ O
( O
NC B
- I
F1 I
) O
with O
polynucleotide O
, O
whereas O
one O
ion O
pair O
is O
found O
in O
the O
metal O
- O
free O
peptide O
- O
nucleic O
acid O
complex O
. O

Investigation O
on O
the O
asymmetrical O
induced O
yields O
in O
90Sr O
- O
90Y O
- O
beta O
- O
irradiated O
D O
- O
and O
L O
- O
alanines O
. O

An O
hcr1 B
null I
mutant I
was O
viable O
, O
but O
showed O
slight O
reduction O
of O
growth O
when O
compared O
with O
the O
wild O
- O
type O
strain O
. O

Serum O
levels O
of O
total O
and O
specific O
immunoglobulin B
E I
( O
IgE B
) O
have O
been O
determined O
by O
radioimmunoassays O
in O
sixty O
- O
nine O
allergic O
subjects O
. O

IgE B
- I
binding I
proteins I
of O
Psocoptera O
were O
determined O
by O
immunoblotting O
experiments O
. O

We O
also O
show O
that O
the O
products O
of O
both O
the O
GIY B
- O
YIG B
ORF O
and O
the O
non O
- O
canonical O
LAGLI O
- O
DADG O
- O
GIY O
- O
YIG O
ORF O
, O
which O
is O
generated O
by O
its O
integration O
, O
have O
endonuclease O
activities O
which O
recognize O
and O
cut O
the O
insertion O
site O
of O
the O
optional O
sequence O
. O

Further O
, O
we O
find O
that O
the O
BMI1 B
protein I
can O
also O
interact O
with O
itself O
. O

Indeed O
, O
ectopic O
expression O
of O
MOX4 B
in O
aerobic O
cells O
resulted O
in O
partially O
constitutive O
expression O
of O
DAN1 B
. O

We O
measured O
electromyograms O
( O
EMG O
) O
of O
the O
alae O
nasi O
to O
determine O
the O
relationship O
between O
their O
activity O
and O
timing O
to O
contraction O
of O
the O
rib O
cage O
muscles O
and O
diaphragm O
during O
obstructive O
apnea O
in O
nine O
patients O
. O

Hybridization O
was O
detected O
between O
polyadenylated O
H B
chain I
mRNA I
, O
isolated O
from O
the O
majority O
of O
the O
hybridomas O
, O
and O
the O
VH B
probe O
. O

Identification O
of O
a O
putative O
infC B
- O
rpmI B
- O
rplT B
operon O
flanked O
by O
long O
inverted O
repeats O
in O
Mycoplasma O
fermentans O
( O
incognitus O
strain O
) O
. O

The O
standardized O
incidence O
in O
Europeans O
has O
risen O
significantly O
to O
4 O
. O
7 O
/ O
10 O
( O
5 O
) O
/ O
year O
from O
3 O
. O
4 O
/ O
10 O
( O
5 O
) O
/ O
year O
in O
the O
1970s O
( O
chi O
2 O
= O
8 O
. O
1 O
, O
p O
less O
than O
0 O
. O
005 O
) O
. O

Neurological O
toxicity O
occurred O
in O
8 O
/ O
219 O
patients O
treated O
with O
fludarabine O
( O
FAMP O
) O
, O
30 O
mg O
/ O
m2 O
per O
day O
and O
cytosine O
arabinoside O
( O
Ara O
- O
C O
) O
, O
0 O
. O
5 O
g O
/ O
m2 O
per O
hour O
for O
2 O
- O
6 O
hours O
for O
5 O
days O
, O
for O
new O
or O
relapsed O
acute O
leukemia O
or O
myelodysplasia O
. O

Evaluation O
of O
gamma B
- I
glutamyl I
transpeptidase I
in O
myocardial O
infarction O
. O

Loss O
of O
pRb B
or O
p107 B
binding O
results O
in O
the O
loss O
of O
transforming O
activity O
. O

The O
analysis O
of O
results O
suggests O
that O
seroprevalence O
of O
Lyme O
borreliosis O
in O
dogs O
of O
the O
Kosice O
region O
is O
not O
negligible O
. O

Peak O
insulin B
response O
to O
glucose O
infusion O
declined O
from O
Groups O
1 O
to O
3 O
( O
51 O
+ O
/ O
- O
14 O
, O
42 O
. O
4 O
+ O
/ O
- O
31 O
, O
20 O
. O
4 O
+ O
/ O
- O
6 O
. O
8 O
microU O
/ O
ml O
, O
respectively O
) O
with O
Group O
3 O
exhibiting O
a O
significantly O
decreased O
mean O
peak O
level O
compared O
to O
the O
other O
groups O
. O

Iris O
binding O
and O
bioavailability O
. O

The O
influence O
of O
a O
high O
n O
- O
3 O
PUFA O
intake O
on O
measures O
of O
lipid O
peroxidation O
has O
been O
equivocal O
. O

The O
proliferative O
potential O
of O
the O
pilocytic O
astrocytoma O
: O
the O
relation O
between O
MIB B
- I
1 I
labeling O
and O
clinical O
and O
neuro O
- O
radiological O
follow O
- O
up O
. O

RESULTS O
: O
Here O
, O
we O
report O
the O
identification O
of O
two O
major O
and O
novel O
Shc B
tyrosine I
phosphorylation I
sites I
, O
Y239 O
and O
Y240 O
. O

Dominant B
- I
negative I
upf1 I
mutations I
were O
isolated O
following O
in O
vitro O
mutagenesis O
of O
a O
plasmid O
containing O
the O
UPF1 B
gene I
. O

The O
tensile O
strengths O
of O
an O
adhesive O
luting O
resin O
' O
Panavia O
EX O
' O
to O
the O
bovine O
dentin O
decreased O
significantly O
by O
the O
zinc O
iontophoresis O
. O

We O
previously O
identified O
a O
highly O
conserved O
L O
- O
X O
- O
X O
- O
L O
- O
F O
sequence O
near O
the O
C O
terminus O
of O
the O
p6 B
domain I
of O
the O
Gag B
precursor I
as O
the O
major O
virion O
association O
motif O
for O
HIV B
- I
1 I
Vpr I
. O

Papillomavirus O
type O
16 O
oncogenes O
downregulate O
expression O
of O
interferon B
- O
responsive I
genes I
and O
upregulate O
proliferation O
- O
associated O
and O
NF B
- I
kappaB I
- I
responsive I
genes I
in O
cervical O
keratinocytes O
. O

The O
A3B3 B
domain I
had O
a O
mass O
of O
34 O
, O
462 O
, O
with O
a O
glycosylation O
mass O
of O
14 O
, O
900 O
, O
in O
good O
agreement O
with O
seven O
N O
- O
linked O
glycosylation O
sites O
of O
average O
mass O
2100 O
. O

We O
found O
that O
the O
frequency O
spectrum O
of O
physiologic O
chest O
sounds O
is O
contained O
entirely O
within O
that O
of O
jet O
rotocraft O
noise O
. O

The O
human B
CA11 I
gene I
appears O
to O
be O
located O
between O
the O
secretor B
type I
alpha I
( I
1 I
, I
2 I
) I
- I
fucosyltransferase I
gene I
cluster O
( O
FUT1 B
- O
FUT2 B
- O
FUT2P B
) O
and O
the O
D B
- I
site I
binding I
protein I
gene I
( O
DBP B
) O
on O
chromosome O
19q13 O
. O
3 O
. O

An O
anoerxiant O
, O
mazindol O
suppresses O
food O
intake O
by O
1 O
) O
stimulating O
beta B
- I
adrenergic I
receptors I
, O
2 O
) O
inhibiting O
the O
feeding O
center O
and O
, O
3 O
) O
stimulating O
the O
satiety O
center O
in O
the O
hypothalamus O
. O

These O
segments O
may O
represent O
pseudogenes O
. O

The O
fusion O
of O
AML1 B
to O
MDS1 B
is O
in O
frame O
, O
and O
adds O
127 O
codons O
to O
the O
interrupted O
AML1 B
. O

In O
this O
study O
, O
the O
effect O
of O
the O
antibiotic O
lasalocid O
on O
the O
rumen O
anaerobic O
fungus O
Neocallimastix O
frontalis O
RK O
21 O
was O
examined O
. O

WBC O
differential O
( O
SEG O
, O
BAND O
, O
LYMPH O
, O
MONO O
, O
EOSINO O
, O
BASO O
) O
usually O
showed O
log O
- O
normal O
distribution O
. O

A O
model O
for O
oligomerization O
- O
dependent O
subunit O
folding O
. O

Chromosome O
- O
specific O
STSs O
for O
27 O
telomeres O
were O
identified O
from O
the O
196 O
TYACs O
. O

Second O
, O
overexpression O
of O
RIM11 B
can O
suppress O
an O
ime1 B
missense O
mutation O
( O
ime1 B
- I
L321F I
) O
but O
not O
an O
ime1 B
deletion O
. O

Calculation O
of O
energy O
levels O
, O
E1 O
transition O
amplitudes O
, O
and O
parity O
violation O
in O
francium O
. O

All O
except O
the O
last O
two O
interventions O
are O
physical O
treatments O
that O
create O
a O
wound O
- O
tissue O
environment O
conducive O
to O
healing O
. O

This O
50 O
- O
kDa O
protein O
contains O
two O
SH2 B
domains I
and O
an O
inter O
- I
SH2 I
domain O
of O
p85alpha B
, O
but O
the O
SH3 B
and O
bcr B
homology I
domains I
of O
p85alpha B
were O
replaced O
by O
a O
unique O
6 O
- O
amino O
acid O
sequence O
. O

Hydropathy O
analysis O
of O
deduced O
A4 B
amino I
acid I
sequence I
revealed O
four O
putative O
membrane O
- O
spanning O
alpha O
- O
helices O
. O

A O
clinical O
double O
- O
blind O
trial O
of O
topical O
miconazole O
and O
clotrimazole O
against O
superficial O
fungal O
infections O
and O
erythrasma O
. O

Studies O
on O
low O
- O
dose O
oral O
contraceptives O
: O
cervical O
mucus O
and O
plasma O
hormone O
changes O
in O
relation O
to O
circulating O
D O
- O
norgestrel O
and O
17alpha O
ethynyl O
- O
estradiol O
concentrations O
. O

Herewith O
C O
. O
psittaci O
could O
be O
diagnosed O
in O
all O
positive O
samples O
. O

The O
more O
sustained O
effect O
of O
cyclofenil O
on O
prolactin B
secretion O
with O
a O
reduced O
frequency O
of O
relapse O
, O
and O
the O
lower O
oestradiol O
level O
, O
which O
might O
indicate O
a O
reduced O
risk O
of O
thromboembolism O
, O
suggest O
that O
this O
drug O
has O
some O
advantage O
over O
bromocriptine O
in O
the O
inhibition O
of O
postpartum O
lactation O
. O

Response O
surface O
methodology O
was O
employed O
to O
describe O
the O
relationship O
between O
soman O
- O
induced O
incapacitation O
and O
the O
ATR O
/ O
DZ O
or O
ATR O
/ O
SCP O
dosages O
. O

The O
brains O
were O
bisected O
and O
T1 O
and O
T2 O
relaxation O
times O
were O
determined O
for O
the O
right O
and O
left O
hemispheres O
by O
MR O
spectroscopy O
immediately O
after O
dissection O
. O

Simulations O
of O
velocities O
, O
vorticity O
, O
pressure O
and O
certain O
stress O
values O
were O
developed O
by O
a O
computer O
and O
displayed O
for O
man O
- O
machine O
interaction O
. O

In O
the O
present O
study O
, O
we O
report O
the O
entire O
genomic O
structure O
of O
KCNQ1 B
, O
which O
consists O
of O
19 O
exons O
spanning O
400 O
kb O
on O
chromosome O
11p15 O
. O
5 O
. O

Northern O
( O
RNA O
) O
blot O
hybridization O
analysis O
with O
the O
p68c B
- I
ets I
- I
1 I
- I
specific I
sequence I
and O
RNase B
protection O
experiments O
showed O
that O
the O
corresponding O
mRNA O
was O
expressed O
in O
normal O
chicken O
spleen O
and O
not O
in O
normal O
chicken O
thymus O
or O
in O
various O
T O
lymphoid O
cell O
lines O
. O

To O
investigate O
this O
issue O
, O
we O
used O
a O
recognition O
task O
in O
which O
two O
strings O
of O
letters O
are O
presented O
sequentially O
. O

Kettin B
is O
a O
large O
modular O
protein O
associated O
with O
thin O
filaments O
in O
the O
Z O
- O
disc O
region O
of O
insect O
muscles O
. O

Enzymatic O
and O
chemical O
structure O
probing O
revealed O
mainly O
the O
conserved O
terminal O
part O
( O
termed O
3 O
' O
C O
) O
of O
the O
DI9c B
3 I
' I
UTR I
containing O
distinctive O
RNA O
motifs O
, O
i O
. O
e O
. O
, O
a O
stable O
stem O
- O
loop O
, O
SL O
I O
, O
near O
the O
RNA O
3 O
' O
terminus O
and O
a O
considerably O
less O
stable O
stem O
- O
loop O
, O
SL O
II O
, O
that O
forms O
the O
5 O
' O
portion O
of O
3 O
' O
C O
. O

This O
review O
chronicles O
the O
characteristics O
of O
deliberate O
and O
accidental O
mass O
poisonings O
that O
occurred O
in O
World O
Wars O
I O
and O
II O
, O
in O
Bhopal O
, O
and O
in O
other O
historical O
cases O
up O
to O
and O
including O
modern O
wars O
. O

The O
effects O
of O
4 O
weeks O
of O
cyclosporin O
A O
( O
7 O
mg O
/ O
kg O
per O
day O
) O
( O
CyA O
) O
on O
the O
survival O
of O
vascularized O
osteochondral O
grafts O
between O
rat O
strains O
[ O
DA O
( O
donor O
) O
and O
Lewis O
( O
recipient O
) O
] O
and O
the O
presence O
and O
significance O
of O
host O
immune O
tolerance O
and O
graft O
antigen O
modulation O
after O
cessation O
of O
immunosuppression O
have O
been O
examined O
. O

By O
incorporating O
a O
stent O
with O
posts O
that O
pivot O
about O
their O
base O
, O
such O
that O
a O
10 O
% O
expansion O
at O
the O
commissures O
is O
realized O
, O
we O
were O
able O
to O
reduce O
the O
compressive O
commissural O
stressing O
from O
250 O
to O
150 O
kPa O
. O

In O
group O
1 O
, O
mean O
z O
- O
score O
for O
GFR O
was O
- O
0 O
. O
27 O
( O
94 O
. O
6 O
% O
of O
normal O
) O
and O
in O
group O
2 O
mean O
z O
- O
score O
was O
- O
1 O
. O
51 O
( O
72 O
. O
7 O
% O
of O
normal O
for O
two O
kidneys O
) O
( O
P O
= O
0 O
. O
022 O
, O
Mann O
- O
Whitney O
U O
- O
test O
) O
. O

The O
cooperation O
between O
health O
and O
hospital O
services O

The O
free O
fraction O
of O
amitriptyline O
( O
AT O
) O
, O
measured O
by O
equilibrium O
dialysis O
in O
plasma O
from O
29 O
AT O
- O
treated O
depressed O
patients O
, O
was O
5 O
. O
4 O
- O
9 O
. O
8 O
% O
( O
mean O
7 O
. O
7 O
% O
) O
, O
which O
was O
the O
same O
as O
the O
values O
in O
26 O
healthy O
controls O
( O
4 O
. O
9 O
- O
9 O
. O
6 O
% O
, O
mean O
7 O
. O
6 O
% O
) O
. O

Different O
effects O
of O
reoxygenation O
on O
the O
electrical O
activity O
of O
ventricular O
muscle O
. O

Metastatic O
tumours O
of O
bone O
in O
Nigerians O
. O

However O
, O
microinjection O
of O
reconstituted O
mRNPs O
into O
Xenopus O
oocytes O
demonstrates O
that O
although O
translational O
repression O
occurs O
in O
the O
absence O
of O
consensus O
RNA O
binding O
sequences O
for O
FRGY2 B
, O
the O
presence O
of O
FRGY2 B
recognition I
elements I
within O
mRNA O
potentiates O
translational O
repression O
. O

Any O
respectative O
material O
( O
class O
0 O
) O
was O
not O
obtained O
from O
334 O
patients O
( O
4 O
. O
2 O
% O
) O
, O
class O
I O
- O
II O
findings O
belonged O
to O
6m141 O
patients O
( O
76 O
. O
7 O
% O
) O
, O
950 O
patients O
( O
11 O
. O
9 O
% O
) O
were O
classified O
as O
class O
HII O
and O
IV O
, O
578 O
patients O
( O
7 O
. O
2 O
% O
) O
as O
class O
V O
. O

However O
, O
whether O
this O
was O
associated O
with O
spleen O
removal O
after O
adjustment O
for O
risk O
factors O
was O
not O
determined O
. O

Virgin O
, O
P O
. O

IL B
- I
6 I
was O
detected O
in O
the O
urine O
of O
52 O
% O
of O
children O
with O
pyelonephritis O
compared O
with O
15 O
% O
of O
other O
children O
( O
P O
< O
0 O
. O
001 O
) O
. O

We O
have O
determined O
the O
complete O
cDNA O
sequence O
of O
rat B
plectin I
from O
a O
number O
of O
well O
- O
characterized O
overlapping O
lambda O
gt11 O
clones O
. O

SETTING O
- O
- O
Hospital O
laboratories O
in O
the O
United O
States O
submitting O
pneumococcal O
isolates O
to O
the O
CDC O
between O
October O
1 O
, O
1991 O
, O
and O
September O
30 O
, O
1992 O
. O

The O
terminal O
a O
sequence O
of O
the O
herpes O
simplex O
virus O
genome O
contains O
the O
promoter O
of O
a O
gene O
located O
in O
the O
repeat O
sequences O
of O
the O
L B
component I
. O

In O
contrast O
to O
p62 B
( O
dok B
) O
, O
DOKL B
lacks O
YxxP O
motifs O
in O
the O
C O
terminus O
and O
does O
not O
bind O
to O
Ras B
GTPase I
- I
activating I
protein I
( O
RasGAP B
) O
upon O
phosphorylation O
. O

These O
observations O
demonstrate O
that O
KSR B
is O
capable O
of O
uncoupling O
the O
MAP B
kinase I
activation O
from O
its O
target O
phosphorylation O
, O
and O
thus O
provide O
a O
novel O
mechanism O
for O
modulating O
the O
Ras B
- O
MAP B
kinase I
signaling O
pathway O
. O

G B
- I
CSF I
( O
10 O
microg O
/ O
kg O
) O
was O
started O
on O
day O
+ O
1 O
and O
all O
patients O
engrafted O
within O
a O
median O
day O
number O
of O
12 O
( O
range O
, O
10 O
- O
22 O
) O
until O
leukocytes O
> O
1 O
. O
0 O
x O
10 O
( O
9 O
) O
/ O
l O
and O
a O
median O
day O
number O
of O
56 O
( O
range O
, O
10 O
- O
180 O
) O
until O
platelets O
> O
20 O
. O
0 O
x O
10 O
( O
9 O
) O
/ O
l O
( O
ie O
platelet O
transfusion O
independence O
) O
. O

A O
higher O
percentage O
of O
children O
with O
cerebral O
malaria O
( O
40 O
per O
cent O
) O
than O
with O
non O
- O
cerebral O
malaria O
( O
29 O
per O
cent O
) O
or O
controls O
( O
20 O
per O
cent O
) O
also O
had O
either O
serum O
ferritin B
< O
100 O
micrograms O
/ O
l O
and O
inflammation O
or O
sTfR B
> O
7 O
. O
3 O
mg O
/ O
l O
or O
both O
. O

During O
the O
burst O
period O
( O
approximately O
5 O
- O
10 O
min O
) O
, O
local O
blood O
H2O2 O
concentrations O
and O
xanthine B
oxidase I
activities O
were O
highly O
correlated O
( O
r O
= O
0 O
. O
999 O
) O
. O

Previous O
studies O
have O
demonstrated O
that O
the O
21 O
- O
or O
the O
72 O
- O
bp O
repeat O
transcriptional O
control O
elements O
enhance O
the O
efficiency O
of O
SV40 O
DNA O
replication O
in O
vivo O
, O
provided O
either O
of O
these O
repeats O
is O
located O
near O
the O
end O
of O
the O
core O
replication O
origin O
containing O
the O
17 O
- O
bp O
A O
+ O
T O
- O
containing O
sequence O
. O

UV O
cross O
- O
linking O
and O
Southwestern O
analysis O
suggested O
that O
KTP1 B
is O
a O
DNA O
- O
binding O
protein O
clearly O
distinct O
from O
AP2 B
, O
and O
this O
protein O
may O
be O
responsible O
for O
the O
basal O
keratinocyte O
- O
specific O
expression O
of O
the O
BPAG1 B
gene I
. O

The O
model O
of O
the O
complex O
provides O
a O
rationale O
for O
conserved O
ITAM B
residues I
, O
substrate O
specificity O
, O
and O
suggests O
that O
substrate O
binds O
only O
the O
active O
conformation O
of O
the O
Src B
family O
tyrosine B
kinase I
, O
unlike O
the O
ATP O
cofactor O
, O
which O
can O
bind O
the O
inactive O
form O
. O

Here O
we O
demonstrate O
that O
a O
region O
of O
high O
homology O
between O
the O
members O
of O
this O
class O
of O
proteins O
contains O
a O
type O
A O
nucleotide O
binding O
site O
consensus O
sequence O
which O
has O
ATPase O
activity O
and O
is O
sufficient O
to O
bind O
DNA O
containing O
specific O
mismatched O
residues O
. O

Copyright O
1999 O
Academic O
Press O
. O

Nickel O
release O
from O
tools O
on O
the O
Swedish O
market O
. O

S O
- O
phase O
- O
promoting O
cyclin B
- I
dependent I
kinases I
( O
Cdks B
) O
and O
the O
kinase O
Dbf4 B
- O
Cdc7 B
then O
act O
to O
initiate O
replication O
. O

NF B
- I
kappaB I
only O
partially O
mediates O
Epstein B
- I
Barr I
virus I
latent I
membrane I
protein I
1 I
activation O
of O
B O
cells O
. O

Purified O
fER B
exhibited O
a O
distribution O
constant O
( O
KD O
) O
for O
17 O
beta O
- O
estradiol O
of O
0 O
. O
45 O
nM O
. O

The O
long O
terminal O
repeat O
is O
590 O
bp O
in O
length O
, O
with O
the O
U3 O
region O
containing O
consensus O
sequences O
likely O
to O
be O
involved O
in O
viral O
gene O
expression O
. O

An O
incorrect O
response O
on O
the O
previous O
trial O
enhanced O
the O
accuracy O
in O
the O
current O
trial O
only O
in O
the O
left O
visual O
field O
( O
LVF O
) O
. O

The O
peptides O
are O
generated O
in O
the O
cytosol O
, O
then O
translocated O
across O
the O
membrane O
of O
the O
endoplasmic O
reticulum O
by O
the O
transporter B
associated I
with I
antigen I
processing I
( O
TAP B
) O
. O

It O
competes O
with O
the O
calcium O
ion O
which O
brings O
about O
inhibition O
of O
myosine B
kinase I
, O
and O
therefore O
a O
drop O
in O
phosphorylated O
myosine B
. O

Comparison O
with O
the O
data O
of O
clinical O
development O
and O
hemodynamic O
results O

Sera O
collected O
in O
1973 O
- O
1975 O
from O
3053 O
residents O
of O
12 O
selected O
Alaskan O
Eskimo O
villages O
were O
tested O
for O
evidence O
of O
hepatitis O
B O
virus O
infection O
. O

Injection O
of O
horseradish B
peroxidase I
( O
HRP B
) O
into O
the O
cerebellar O
flocculus O
of O
the O
rat O
was O
employed O
to O
identify O
neurons O
in O
the O
abducens O
nucleus O
that O
project O
to O
the O
flocculus O
. O

To O
assay O
for O
sequences O
that O
could O
potentially O
regulate O
Xenopus B
proenkephalin I
expression O
, O
we O
transfected O
constructs O
that O
contained O
upstream O
genomic O
sequences O
linked O
to O
the O
CAT B
reporter I
gene I
into O
various O
eukaryotic O
cell O
lines O
. O

This O
suggests O
that O
the O
ratio O
of O
IL B
- I
6 I
to O
IL B
- I
10 I
may O
be O
used O
to O
predict O
the O
injury O
severity O
after O
trauma O
. O

UVB O
- O
induced O
association O
of O
tumor B
necrosis I
factor I
( I
TNF I
) I
receptor I
1 I
/ O
TNF B
receptor I
- I
associated I
factor I
- I
2 I
mediates O
activation O
of O
Rel B
proteins I
. O

A O
major O
feature O
of O
this O
genomic O
sequence O
is O
the O
presence O
of O
a O
first O
intron O
( O
Il O
) O
, O
215 O
bp O
long O
, O
located O
48 O
bp O
downstream O
of O
the O
translation O
start O
ATG O
codon O
. O

Experimental O
studies O
and O
clinical O
application O
of O
plasma O
ACTH B
radioimmunoassay O
kit O
without O
extraction O
process O

In O
the O
cervical O
enlargement O
they O
were O
located O
in O
the O
middle O
part O
of O
lamina O
VII O
and O
in O
lamina O
VIII O
, O
accounting O
for O
about O
25 O
% O
of O
the O
total O
labeled O
neurons O
. O

Nature O
and O
incidence O
of O
conditioned O
responses O
in O
a O
methadone O
population O
: O
a O
comparison O
of O
laboratory O
, O
clinic O
, O
and O
naturalistic O
settings O
. O

While O
some O
still O
automatically O
refuse O
all O
patients O
with O
positive O
mediastinoscopy O
, O
most O
authors O
still O
remain O
very O
interventionistic O
for O
N2 O
patients O
selected O
on O
very O
accurate O
criteria O
that O
are O
analyzed O
above O
. O

Our O
previous O
studies O
demonstrated O
that O
the O
promyelocytic B
leukemia I
gene I
, O
PML B
which O
involved O
in O
the O
15 O
; O
17 O
translocation O
in O
acute O
promyelocytic O
leukemia O
( O
APL O
) O
is O
a O
growth O
and O
transformation O
suppressor O
. O

A O
sharp O
transition O
from O
a O
random O
chaotic O
state O
to O
a O
correlated O
turbulent O
state O
of O
finite O
coherence O
time O
is O
found O
when O
the O
Rayleigh O
number O
becomes O
larger O
than O
a O
critical O
value O
Ra O
( O
c O
) O
approximately O
equal O
to O
5 O
x O
10 O
( O
7 O
) O
. O

Our O
data O
define O
Ile O
( O
16 O
) O
, O
Val O
( O
18 O
) O
, O
Val O
( O
31 O
) O
, O
and O
Ile O
( O
33 O
) O
as O
crucial O
for O
protein B
S I
binding O
, O
with O
secondary O
effects O
from O
Leu O
( O
38 O
) O
and O
Val O
( O
39 O
) O
. O

In O
the O
yeast O
two O
- O
hybrid O
assays O
, O
PNRC B
interacted O
with O
the O
orphan O
receptors O
SF1 B
and O
ERRalpha1 B
in O
a O
ligand O
- O
independent O
manner O
. O

Nuclear O
export O
of O
proteins O
in O
plants O
: O
AtXPO1 B
is O
the O
export O
receptor O
for O
leucine O
- O
rich O
nuclear O
export O
signals O
in O
Arabidopsis O
thaliana O
. O

Thus O
, O
CR2 B
possesses O
an O
essential O
function O
besides O
pRb B
- O
binding O
. O

From O
the O
autopsy O
findings O
, O
in O
5 O
out O
of O
the O
remaining O
23 O
expired O
patients O
, O
Legionella O
pneumonia O
seemed O
to O
be O
successfully O
treated O
, O
but O
other O
disease O
or O
other O
bacterial O
pneumonia O
put O
an O
end O
to O
the O
patients O
. O

Nitroglycerin O
( O
glyceryl O
trinitrate O
) O
as O
a O
monoamine B
oxidase I
inhibitor O
. O

This O
high O
dose O
5 O
- O
FU O
+ O
Act O
- O
D O
protocol O
was O
effective O
for O
drug O
resistant O
cases O
with O
MEA O
protocol O
. O

With O
stimulation O
beyond O
the O
theta O
- O
range O
three O
phenomena O
occurred O
: O
shift O
of O
the O
burst O
frequencies O
to O
higher O
or O
lower O
harmonics O
of O
stimulation O
frequencies O
; O
complex O
interactions O
of O
basic O
background O
frequency O
with O
rhythm O
of O
stimulation O
( O
" O
beating O
" O
) O
; O
return O
to O
background O
theta O
- O
burst O
frequency O
in O
spite O
of O
continuing O
stimulation O
( O
" O
escape O
" O
) O
. O

CONCLUSIONS O
: O
Transpulmonary O
stable O
air O
microbubbles O
bound O
to O
a O
galactose O
carrier O
represent O
a O
useful O
and O
safe O
contrast O
agent O
in O
case O
of O
an O
insufficient O
native O
signal O
in O
transcranial O
Doppler O
investigation O
. O

K562 O
erythroleukemia O
cells O
stably O
transfected O
with O
constructs O
containing O
the O
human O
Agamma B
- I
globin I
promoter O
linked O
to O
an O
enhanced B
green I
fluorescent I
protein I
( O
EGFP B
) O
reporter O
, O
with O
or O
without O
HS2 B
, O
were O
analyzed O
for O
EGFP B
expression O
by O
flow O
cytometry O
. O

Gastrin B
stimulates O
transcription O
of O
the O
human B
histidine I
decarboxylase I
( O
HDC B
) O
gene O
through O
binding O
to O
the O
G B
- I
protein I
- I
coupled I
cholecystokinin I
- I
B I
/ I
gastrin I
receptor I
. O

Here O
, O
we O
report O
the O
isolation O
of O
a O
new O
spindle O
checkpoint O
gene O
, O
mph1 B
( O
Mps1p B
- I
like I
pombe I
homolog I
) O
, O
in O
the O
fission O
yeast O
Schizosaccharomyces O
pombe O
, O
that O
is O
required O
for O
checkpoint O
activation O
in O
response O
to O
spindle O
defects O
. O

mph1 B
functions O
upstream O
of O
mad2 B
, O
a O
previously O
characterized O
component O
of O
the O
spindle O
checkpoint O
. O

Hepatocyte B
growth I
factor I
( O
HGF B
) O
is O
an O
inducible O
cytokine O
that O
is O
essential O
for O
the O
normal O
growth O
and O
development O
of O
various O
tissues O
, O
such O
as O
the O
liver O
. O

However O
, O
all O
pathophysiologic O
assessments O
reported O
to O
date O
have O
been O
carried O
out O
during O
or O
immediately O
after O
IVOX O
utilization O
. O

Reduced O
concentrations O
of O
antithrombin B
III I
, O
plasminogen B
, O
( B
free I
) I
protein I
S I
, O
and O
protein B
C I
were O
found O
in O
some O
patients O
but O
were O
not O
associated O
with O
either O
thrombosis O
or O
lupus O
anticoagulant O
. O

The O
first O
decision O
in O
management O
is O
to O
consider O
cardioversion O
which O
can O
be O
achieved O
in O
suitable O
cases O
electrically O
, O
or O
pharmacologically O
with O
a O
class O
Ic O
antiarrhythmic O
drug O
like O
flecainide O
or O
propafenone O
. O

Structure O
and O
chromosomal O
localization O
of O
the O
human O
gene O
of O
the O
phosphotyrosyl B
phosphatase I
activator I
( O
PTPA B
) O
of O
protein B
phosphatase I
2A I
. O

R O
of O
the O
chest O
wall O
was O
maximum O
( O
35 O
. O
6 O
cmH2O O
. O
L O
- O
1 O
. O
sec O
- O
1 O
+ O
/ O
- O
2 O
. O
2 O
SE O
) O
at O
0 O
. O
2 O
Hz O
- O
10 O
ml O
and O
decreased O
with O
increasing O
f O
and O
VT O
, O
although O
the O
VT O
effect O
diminished O
at O
the O
higher O
f O
. O

Since O
1988 O
Modulit O
- O
SZ O
- O
20 O
has O
been O
tested O
for O
localization O
of O
nephroliths O
and O
ureteroliths O
. O

JCAHO O
moves O
to O
rewrite O
many O
rules O
to O
lessen O
hospitals O
' O
regulatory O
burden O
. O

These O
transcripts O
directed O
the O
synthesis O
of O
three O
proteins O
: O
the O
virus B
trans I
- I
activator I
protein I
( O
EIAV B
Tat I
) O
encoded O
by O
ORF O
S1 O
, O
a O
protein O
of O
unknown O
function O
encoded O
by O
ORF O
S2 O
, O
and O
the O
virus B
envelope I
glycoprotein I
. O

However O
, O
no O
significant O
difference O
in O
either O
the O
Valsalva O
ratio O
or O
delta O
SBP O
was O
found O
between O
diabetic O
patients O
and O
controls O
. O

Transmural O
metabolic O
heterogeneity O
at O
high O
cardiac O
work O
states O
. O

We O
therefore O
investigated O
whether O
the O
promoter O
activity O
of O
the O
mouse B
TRH I
gene I
is O
directly O
regulated O
by O
RA O
using O
a O
transient O
transfection O
assay O
into O
CV O
- O
1 O
cells O
. O

Spinal O
injuries O
in O
a O
multiple O
trauma O
patient O
. O

The O
unusual O
properties O
of O
TRAC B
activity O
and O
its O
relationship O
, O
if O
any O
, O
with O
the O
enigmatic O
Ku B
protein I
, O
are O
discussed O
. O

Statistical O
aspects O
of O
the O
relation O
of O
the O
radioprotective O
action O
of O
mercaptoethylamine O
derivatives O
and O
analogs O
to O
their O
electron O
parameters O

Cerebellar O
functional O
compensation O
after O
its O
complete O
destruction O
by O
a O
neoplasm O

The O
RNA O
, O
whether O
antisense O
, O
ribozyme O
, O
or O
RNA O
aptamer O
, O
must O
be O
efficiently O
transcribed O
, O
stabilized O
against O
rapid O
degradation O
, O
folded O
correctly O
, O
and O
directed O
to O
the O
part O
of O
the O
cell O
where O
it O
can O
be O
most O
effective O
. O

We O
have O
isolated O
and O
determined O
the O
complete O
nucleotide O
sequence O
of O
another O
operon O
, O
cpeCD B
, O
that O
encodes O
the O
linker O
proteins O
associated O
with O
phycoerythrin O
hexamers O
in O
the O
phycobilisome O
. O

A B
. I
thaliana I
cystathionine I
beta I
- I
lyase I
exhibits O
22 O
% O
sequence O
identity O
with O
the O
E O
. O
coli O
corresponding O
enzyme O
and O
contains O
a O
70 O
amino O
acid O
N O
- O
terminal O
additional O
sequence O
compared O
with O
the O
bacterial O
protein O
. O

The O
enzyme O
ACC B
oxidase I
catalyses O
the O
last O
step O
of O
ethylene O
biosynthesis O
in O
plants O
, O
converting O
1 O
- O
aminocyclopropane O
- O
1 O
- O
carboxylic O
acid O
( O
ACC O
) O
to O
ethylene O
. O

A O
99m O
Tc O
bone O
scan O
showed O
a O
focal O
area O
of O
an O
increased O
uptake O
at O
the O
site O
of O
the O
mass O
below O
the O
calcaneus O
. O

Renal O
function O
was O
evaluated O
in O
a O
group O
of O
24 O
patients O
( O
21 O
men O
and O
3 O
women O
, O
mean O
age O
47 O
years O
) O
who O
had O
survived O
the O
malignant O
phase O
of O
hypertension O
during O
the O
period O
of O
1969 O
- O
1979 O
. O

The O
need O
for O
such O
guidelines O
is O
clear O
on O
both O
clinical O
and O
scientific O
grounds O
. O

When O
expressed O
in O
thymidine B
kinase I
- O
deficient O
L O
cells O
or O
3T3 O
cells O
, O
C B
/ I
EBPalpha I
is O
detected O
in O
a O
protein O
complex O
which O
binds O
to O
the O
E2F B
binding I
sites I
found O
in O
the O
promoters O
of O
the O
genes O
for O
E2F B
- I
1 I
and O
dihydrofolate B
reductase I
( O
DHFR B
) O
. O

Members O
of O
the O
CRE B
- I
binding I
protein I
( O
CREB B
) O
/ O
activating B
transcription I
factor I
transcription I
family I
as O
well O
as O
members O
of O
the O
Jun B
/ O
Fos B
family O
have O
been O
shown O
to O
bind O
to O
this O
sequence O
. O

Csk B
( O
C B
- I
terminal I
Src I
kinase I
) O
is O
a O
protein B
tyrosine I
kinase I
that O
phosphorylates O
Src B
family I
member O
C O
- O
terminal O
tails O
, O
resulting O
in O
downregulation O
of O
Src B
family I
members I
. O

Role O
of O
double B
- I
stranded I
RNA I
- I
dependent I
protein I
kinase I
in O
mediating O
hypersensitivity O
of O
Fanconi O
anemia I
complementation O
group O
C O
cells O
to O
interferon B
gamma I
, O
tumor B
necrosis I
factor I
- I
alpha I
, O
and O
double O
- O
stranded O
RNA O
. O

In O
clinical O
stroke O
cardiovascular O
abnormalities O
are O
frequently O
neglected O
although O
they O
occur O
more O
often O
than O
it O
is O
generally O
assumed O
. O

In O
the O
presence O
of O
high O
concentrations O
( O
propofol O
100 O
microg O
mL O
- O
1 O
, O
thiopental O
50 O
microg O
mL O
- O
1 O
) O
marked O
relaxation O
was O
observed O
( O
propofol O
- O
32 O
% O
up O
to O
- O
49 O
% O
, O
P O
< O
0 O
, O
05 O
; O
thiopental O
- O
23 O
% O
up O
to O
- O
67 O
% O
, O
P O
< O
0 O
. O
05 O
) O
. O

Our O
results O
suggest O
a O
dose O
- O
dependent O
activity O
of O
lysine B
vasopressin I
on O
the O
EEG O
, O
which O
might O
be O
similar O
to O
that O
observed O
in O
animal O
and O
man O
after O
administration O
of O
nicotine O
. O

Mutations O
of O
the O
human B
PTEN I
gene I
. O

Guanylyl B
cyclase I
C I
, O
the O
apparent O
guanylin B
receptor I
, O
was O
found O
in O
abundant O
amounts O
in O
the O
intestine O
by O
Northern O
analysis O
, O
and O
by O
the O
polymerase O
chain O
reaction O
or O
cDNA O
cloning O
it O
was O
also O
found O
in O
adrenal O
gland O
, O
airway O
epithelial O
cells O
, O
brain O
, O
and O
olfactory O
and O
tracheal O
mucosa O
. O

In O
the O
present O
study O
we O
have O
used O
Southern O
blot O
analysis O
to O
demonstrate O
that O
TSG6 B
is O
a O
single O
- O
copy O
gene O
in O
the O
human O
and O
murine O
species O
. O

Reaction O
of O
aromatic O
/ O
heterocyclic O
sulfonamides O
possessing O
free O
amino O
, O
imino O
or O
hydrazino O
moieties O
with O
7 O
- O
chloro O
- O
4 O
- O
chloromethylcoumarin O
afforded O
a O
series O
of O
N O
- O
[ O
( O
7 O
- O
chloro O
- O
4 O
- O
coumarinyl O
) O
- O
methyl O
] O
- O
derivatives O
which O
showed O
effective O
inhibition O
of O
three O
carbonic B
anhydrase I
( O
CA B
) O
isozymes O
. O

As O
well O
, O
variation O
in O
capillary O
size O
and O
density O
was O
apparent O
between O
ventricles O
in O
the O
fetal O
and O
neonatal O
periods O
. O

The O
Polycomb B
group I
proteins I
are O
involved O
in O
maintenance O
of O
the O
silenced O
state O
of O
several O
developmentally O
regulated O
genes O
. O

Uncertainty O
exists O
about O
the O
function O
of O
these O
potential O
isoforms O
of O
the O
Cbfa1 B
gene I
. O

When O
using O
the O
high O
- O
impedance O
testing O
device O
, O
the O
post O
shock O
rhythm O
was O
initially O
displayed O
as O
' O
no O
signal O
' O
( O
Hewlett O
Packard O
XL O
) O
or O
' O
asystole O
' O
( O
Physio O
- O
Control O
LIFEPAK O
9 O
) O
. O

Human B
CD38 I
, O
a O
leukocyte O
receptor O
and O
ectoenzyme O
, O
is O
a O
member O
of O
a O
novel O
eukaryotic O
gene O
family O
of O
nicotinamide B
adenine I
dinucleotide I
+ I
- I
converting I
enzymes I
: O
extensive O
structural O
homology O
with O
the O
genes O
for O
murine B
bone I
marrow I
stromal I
cell I
antigen I
1 I
and O
aplysian B
ADP I
- I
ribosyl I
cyclase I
. O

It O
is O
noteworthy O
that O
under O
more O
physiological O
conditions O
mimicking O
the O
cellular O
GDP O
/ O
GTP O
ratio O
, O
Raf B
enhances O
the O
GEF B
- O
stimulated O
GDP O
/ O
GTP O
exchange O
on O
Ha B
- I
Ras I
, O
in O
agreement O
with O
the O
sequestration O
of O
Ras B
. I
GTP I
by O
Raf B
. O

When O
O O
- O
2A O
/ O
c B
- I
myc I
cells O
underwent O
terminal O
differentiation O
APprog O
complexes O
were O
lost O
and O
conventional O
AP B
- I
1 I
DNA O
- O
binding O
activity O
became O
evident O
, O
particularly O
in O
astrocytes O
. O

From O
the O
deduced O
amino O
acid O
sequence O
, O
a O
molecular O
weight O
of O
38 O
, O
549 O
was O
calculated O
for O
the O
ADH B
subunit I
. O

The O
higher O
level O
of O
calories O
was O
attained O
by O
an O
increased O
intake O
of O
carbohydrates O
. O

A O
simple O
algorithm O
fitting O
the O
data O
for O
the O
moving O
192Ir O
source O
is O
proposed O
. O

TAF B
- I
1 I
activates O
transcription O
constitutively O
and O
was O
mapped O
to O
the O
first O
32 O
amino O
acids O
of O
the O
A O
- O
region O
. O

For O
the O
first O
, O
short O
- O
term O
( O
concurrent O
) O
TAL O
, O
two O
different O
- O
flavored O
stimuli O
were O
presented O
at O
the O
same O
time O
, O
one O
associated O
with O
simultaneous O
intragastric O
administration O
of O
an O
aversive O
product O
, O
hypertonic O
NaCl O
, O
and O
the O
other O
with O
saline O
. O

The O
great O
cardiac O
vein O
flow O
became O
zero O
due O
to O
the O
cardiac O
arrest O
and O
remained O
at O
zero O
for O
a O
moment O
( O
dead O
time O
) O
after O
the O
initiation O
of O
reperfusion O
. O

INTERNET O
is O
literally O
a O
world O
of O
data O
, O
dialogue O
, O
and O
discourse O
on O
any O
topic O
imaginable O
, O
right O
at O
your O
fingertips O
. O

Attention O
is O
devoted O
in O
particular O
to O
the O
action O
of O
thermoregulating O
nervous O
and O
hormonal O
mechanisms O
. O

The O
mammalian O
UPR O
is O
mediated O
by O
the O
cis O
- O
acting O
ER O
stress O
response O
element O
consisting O
of O
19 O
nt O
( O
CCAATN O
( O
9 O
) O
CCACG O
) O
, O
the O
CCACG O
part O
of O
which O
is O
considered O
to O
provide O
specificity O
. O

The O
formation O
of O
a O
D O
- O
loop O
is O
an O
essential O
step O
in O
DNA O
double O
- O
strand O
break O
repair O
through O
recombination O
. O

The O
cDNA O
is O
predicted O
to O
encode O
a O
polypeptide O
of O
298 O
amino O
acids O
that O
is O
homologous O
to O
the O
Y B
- I
box I
( I
inverted I
CCAAT I
) I
family I
of I
DNA I
- I
binding I
transcription I
factors I
. O

Toward O
this O
end O
, O
we O
used O
two O
cultured O
colon O
cancer O
cell O
lines O
; O
one O
( O
RKO O
) O
has O
a O
transcriptionally O
activated O
u B
- I
PAR I
gene I
, O
and O
the O
other O
( O
GEO O
) O
overexpresses O
the O
receptor O
only O
after O
phorbol O
ester O
treatment O
. O

Site O
- O
directed O
mutagenesis O
of O
the O
conserved O
cysteine O
residue O
( O
tom1C3235A B
) O
in O
the O
hect B
- I
domain I
, O
supposed O
to O
be O
necessary O
for O
thioester O
- O
bond O
formation O
with O
ubiquitin B
, O
abolished O
the O
gene O
function O
. O

The O
two O
yeast B
pheromone I
receptors I
, O
the O
a B
and I
alpha I
- I
factor I
receptors I
, O
share O
many O
functional O
similarities O
: O
both O
G B
protein I
- I
coupled I
receptors I
couple O
to O
the O
same O
downstream O
signal O
transduction O
pathway O
, O
and O
both O
receptors O
undergo O
feedback O
regulation O
involving O
increased O
phosphorylation O
on O
their O
C O
- O
terminal O
domains O
in O
response O
to O
ligand O
challenge O
. O

Moreover O
, O
the O
formation O
of O
the O
complex O
between O
IclR B
and O
the O
operator O
/ O
promoter O
region O
has O
been O
found O
to O
be O
impaired O
by O
phosphoenol O
pyruvate O
but O
insensitive O
to O
acetate O
, O
acetyl O
- O
CoA O
, O
pyruvate O
, O
and O
oxaloacetate O
. O

After O
enzymatic O
hydrolysis O
of O
the O
water O
soluble O
fraction O
, O
it O
is O
shown O
that O
28 O
% O
is O
again O
extractable O
by O
DCM O
of O
which O
57 O
% O
consists O
of O
unchanged O
SC4453 O
which O
is O
therefore O
the O
main O
partner O
of O
conjugated O
products O
. O

The O
expression O
pattern O
of O
the O
reporter O
gene O
was O
compared O
to O
that O
of O
the O
endogenous B
PDGFbeta I
r I
gene I
. O

The O
two O
SH3 B
domains I
are O
separated O
by O
a O
54 O
amino O
acid O
linker O
region O
, O
whose O
length O
is O
highly O
conserved O
in O
xenopus O
, O
chicken O
, O
and O
mamalian O
Crk I
II I
proteins I
. O

This O
complex O
was O
eliminated O
by O
mutation O
of O
either O
half O
- O
site O
, O
and O
it O
was O
supershifted O
by O
antibodies O
against O
chicken B
ovalbumin I
upstream I
promoter I
- I
transcription I
factor I
( O
COUP B
- I
TF I
) O
. O

The O
fz B
- I
1 I
and O
fz B
- I
2 I
genes I
are O
widely O
expressed O
in O
rat O
tissues O
with O
the O
highest O
steady O
- O
state O
levels O
of O
mRNA O
in O
kidney O
, O
liver O
, O
heart O
, O
uterus O
, O
and O
ovary O
. O

fz B
- I
1 I
and I
- I
2 I
mRNA I
levels O
were O
greater O
in O
neonatal O
than O
in O
corresponding O
adult O
tissues O
. O

These O
results O
indicate O
that O
the O
changes O
in O
the O
patient O
' O
s O
thyroid O
scan O
and O
RAIU O
were O
attributable O
to O
the O
presence O
of O
TSAb O
. O

The O
methods O
used O
were O
the O
AOAC O
method O
of O
dilution O
and O
heating O
; O
the O
addition O
of O
mercuric O
chloride O
, O
charcoal O
, O
and O
Carrez O
reagents O
at O
2 O
different O
pH O
values O
; O
and O
direct O
analysis O
of O
sample O
supernatants O
with O
no O
treatment O
( O
control O
) O
. O

Cytochemical O
and O
ultrastructural O
observations O
on O
the O
tegument O
of O
the O
trematode O
Megalodiscus O
temperatus O
. O

In O
this O
report O
, O
we O
establish O
that O
the O
heteromeric B
CCAAT I
- I
binding I
factor I
CBF B
is O
the O
major O
component O
of O
the O
C1 B
- I
binding I
factor I
( O
C1F B
) O
in O
human O
cells O
. O

A O
40 O
- O
fold O
stimulation O
of O
chloramphenicol B
acetyltransferase I
activity O
mediated O
by O
four O
tandem O
repeats O
of O
the O
SNE O
could O
be O
induced O
within O
2 O
h O
( O
and O
up O
to O
250 O
- O
fold O
within O
6 O
h O
) O
after O
addition O
of O
TPA O
in O
DNA O
- O
transfected O
U O
- O
937 O
cells O
, O
indicating O
that O
the O
stimulation O
appeared O
likely O
to O
be O
a O
true O
protein B
kinase I
C I
- O
mediated O
signal O
transduction O
event O
rather O
than O
a O
differentiation O
response O
. O

This O
increased O
frequency O
of O
infection O
has O
been O
accompanied O
by O
significant O
excess O
mortality O
. O

Deletion O
mutants O
of O
the O
human B
CHOP I
promoter I
identify O
a O
region O
comprising O
nucleotides O
- O
75 O
to O
- O
104 O
required O
for O
both O
constitutive O
and O
ER O
- O
stress O
- O
inducible O
expression O
. O

The O
duration O
of O
PGE2 O
and O
PGF2 O
alpha O
elevations O
as O
well O
as O
the O
peak O
values O
were O
influenced O
by O
day O
of O
the O
cycle O
. O

We O
propose O
that O
the O
cDNA O
encodes O
an O
apical O
plasma O
membrane O
protein O
that O
plays O
a O
role O
in O
the O
functional O
expression O
of O
the O
amiloride O
- O
sensitive O
epithelial B
sodium I
channel I
. O

Electrophysiology O
and O
pharmacology O
of O
epicardial O
, O
endocardial O
, O
and O
M O
cells O
. O

In O
contrast O
, O
an O
N B
- I
terminal I
RNAP I
mutant I
that O
has O
a O
decreased O
capacity O
to O
bind O
single O
- O
stranded O
RNA O
forms O
halted O
complexes O
with O
much O
lower O
levels O
of O
stability O
than O
the O
WT O
enzyme O
, O
and O
these O
complexes O
are O
not O
stabilized O
by O
the O
presence O
of O
the O
NT O
strand O
. O

A O
cephalothoracophagus O
disymmetros O
buffalo O
calf O
. O

A O
mouse O
brain O
cDNA O
library O
in O
lambdagt11 O
was O
screened O
for O
this O
association O
, O
and O
two O
positive O
clones O
encoding O
tyrosine B
phosphatase I
SH B
- I
PTP2 I
were O
isolated O
. O

Disorganization O
scores O
of O
the O
hypertrophy O
zone O
and O
trabecular O
bone O
were O
low O
, O
approaching O
normal O
( O
P O
< O
0 O
. O
05 O
) O
, O
for O
turkey O
poults O
fed O
on O
diets O
with O
phytase B
supplementation O
, O
and O
tibial O
abnormality O
scores O
were O
linearly O
decreased O
( O
P O
< O
0 O
. O
001 O
) O
as O
nP O
levels O
increased O
( O
zero O
score O
is O
considered O
normal O
) O
. O

Repression O
was O
strictly O
dependent O
on O
the O
presence O
of O
upstream O
Oshox1 B
binding I
sites I
in O
the O
reporter O
gene O
constructs O
and O
a O
function O
of O
the O
N O
- O
terminal O
region O
of O
Oshox1 B
, O
preceding O
the O
homeodomain B
. O

Ondansetron O
is O
thus O
an O
effective O
first O
- O
line O
antiemetic O
in O
children O
undergoing O
chemotherapy O
, O
radiotherapy O
and O
surgery O
. O

To O
elucidate O
the O
molecular O
mechanism O
by O
which O
the O
Ras B
signaling O
pathway O
activates O
a O
cell O
- O
type O
- O
specific O
gene O
, O
we O
have O
used O
the O
pituitary B
- I
specific I
rat I
prolactin I
( O
rPRL B
) O
promoter O
as O
a O
target O
of O
oncogenic B
Ras I
and O
Raf B
in O
GH4 O
rat O
pituitary O
cells O
. O

The O
human B
RFC I
- I
1 I
gene I
differs O
from O
the O
mouse O
and O
hamster O
genes O
both O
in O
terms O
of O
the O
total O
number O
of O
exons O
and O
in O
regard O
to O
alternatives O
of O
exon O
1 O
which O
encode O
5 O
' O
end O
heterogeneity O
. O

Type O
C O
units O
summate O
the O
responses O
to O
the O
two O
- O
tone O
stimulus O
, O
and O
show O
little O
or O
no O
inhibitory O
influences O
. O

HGF B
treatment O
increased O
cyclin B
A I
, O
cyclin B
G1 I
and O
nuclear B
transcriptional I
factor I
( O
NFkappaB I
) O
protein O
expression O
. O

Since O
blood O
serotonin O
is O
primarily O
produced O
peripherally O
, O
these O
results O
suggest O
that O
some O
aspect O
of O
peripheral O
serotonin O
metabolism O
is O
abnormal O
in O
major O
depression O
. O

Although O
several O
large O
waterborne O
outbreaks O
occurred O
during O
the O
past O
decade O
, O
most O
were O
in O
small O
communities O
. O

Cytogenetic O
structures O
showed O
a O
mixte O
karyotype O
: O
mosaicim O
46 O
XX O
/ O
46 O
XY O
with O
a O
ratio O
of O
a O
80 O
/ O
20 O
. O

The O
1 O
. O
6 O
- O
kb O
cDNA O
( O
CBF B
- I
A I
) O
encoding O
285 O
amino O
acids O
( O
aa O
) O
was O
obtained O
, O
and O
a O
beta B
- I
galactosidase I
fusion I
protein I
, O
bacterially O
produced O
from O
the O
cDNA O
, O
bound O
to O
DNA O
fragments O
containing O
several O
CArG O
boxes O
. O

Mutant B
M I
proteins I
were O
tested O
for O
their O
ability O
to O
complement O
growth O
of O
the O
temperature O
- O
sensitive O
M B
protein I
mutant I
virus O
tsO23 O
at O
the O
nonpermissive O
temperature O
. O

In O
this O
paper O
we O
review O
evidence O
on O
smoking O
and O
lung O
cancer O
among O
Latinos O
, O
including O
findings O
from O
several O
unpublished O
studies O
and O
technical O
reports O
. O

The O
yjr041c O
delta O
haploid O
cells O
gave O
rise O
to O
microcolonies O
comprising O
about O
20 O
to O
50 O
cells O
. O

Despite O
these O
high O
initial O
rates O
of O
transcription O
, O
of O
all O
the O
promoter O
constructs O
only O
LAT B
- O
LTR O
was O
able O
to O
remain O
transcriptionally O
active O
after O
the O
establishment O
of O
a O
latent O
state O
. O

Two O
mutants O
, O
mapping O
at O
the O
HindIII B
site I
( O
between O
the O
consensus O
sequences O
) O
of O
the O
pSC101 B
tetA B
promoter I
, O
were O
studied O
: O
MA2 O
corresponds O
to O
a O
4 O
bp O
deletion O
between O
positions O
- O
12 O
and O
- O
15 O
; O
B30 O
bears O
a O
44 O
bp O
insertion O
C O
( O
TA O
) O
21 O
G O
at O
the O
HindIII B
site I
. O

We O
concluded O
that O
these O
results O
support O
the O
feasibility O
and O
usefulness O
of O
N O
- O
of O
- O
1 O
RCT O
in O
rheumatology O
practice O
. O

Sci O
. O

Effects O
of O
timepidium O
bromide O
( O
TB O
; O
anticholinergic O
agent O
) O
, O
acetylcholine O
( O
ACh O
) O
and O
neostigmine O
( O
Neost O
) O
on O
gastric O
and O
duodenal O
blood O
flow O
distribution O
were O
studied O
by O
the O
use O
of O
131I O
- O
labeled O
macroaggregated B
human I
serum I
albumin I
( O
MAA B
) O
in O
rabbits O
. O

These O
extrachromosomal O
copies O
can O
be O
isolated O
as O
covalently O
closed O
molecules O
with O
lengths O
around O
3mu O
. O

Comparison O
of O
the O
footprint O
patterns O
of O
the O
mouse O
and O
human B
HPRT I
genes I
demonstrated O
that O
the O
in O
vivo O
binding O
of O
regulatory O
proteins O
between O
these O
species O
is O
generally O
conserved O
but O
not O
identical O
. O

Here O
, O
we O
describe O
the O
three O
- O
dimensional O
structure O
of O
the O
enzyme O
from O
Escherichia O
colidetermined O
and O
refined O
to O
2 O
. O
0 O
A O
resolution O
. O

Our O
Northern O
and O
Southern O
blot O
analyses O
of O
meningiomas O
clearly O
suggest O
the O
CLH B
- I
22 I
gene I
may O
be O
involved O
in O
the O
tumor O
development O
and O
can O
be O
considered O
as O
a O
candidate O
for O
a O
tumor O
suppressor O
. O

Plasmodium O
vivax O
malaria O
. O

This O
set O
of O
genetic O
and O
physical O
interactions O
suggests O
a O
role O
for O
yeast O
RNA O
- O
binding O
proteins O
in O
transcriptional O
regulation O
. O

POPULATION O
AND O
METHODS O
: O
105 O
patients O
were O
followed O
prospectively O
( O
male O
: O
87 O
% O
; O
age O
: O
56 O
+ O
/ O
- O
10 O
) O
; O
clinical O
evaluation O
was O
performed O
in O
the O
1st O
, O
3rd O
and O
6th O
month O
, O
SEKG O
in O
the O
4th O
month O
and O
recatheterization O
for O
angiographic O
control O
in O
the O
6th O
month O
. O

All O
other O
cysts O
showed O
a O
unilocular O
, O
purely O
cystic O
pattern O
, O
with O
homogeneous O
fluids O
, O
although O
the O
T2 O
relaxation O
times O
of O
four O
lesions O
overlapped O
those O
of O
odontogenic O
keratocysts O
. O

Genetic O
diagnosis O
of O
respiratory O
diseases O

The O
c B
- I
Jun I
delta I
- I
domain I
inhibits O
neuroendocrine O
promoter O
activity O
in O
a O
DNA O
sequence O
- O
and O
pituitary O
- O
specific O
manner O
. O

Cross O
- O
sectional O
studies O
continue O
to O
dominate O
. O

The O
role O
of O
the O
medical O
facilities O
in O
the O
prognosis O
of O
the O
patient O
with O
tetanus O
specially O
the O
importance O
of O
considering O
at O
the O
same O
time O
the O
severity O
of O
the O
disease O
and O
the O
characteristics O
of O
the O
therapy O
deserve O
further O
study O
in O
order O
to O
contribute O
to O
the O
development O
of O
the O
medical O
assistance O
to O
the O
patients O
with O
tetanus O
. O

Differences O
were O
not O
found O
in O
colons O
by O
SEM O
. O

Increasing O
the O
height O
of O
the O
agar O
column O
overlying O
the O
SN O
fabric O
diminished O
the O
inhibitory O
effect O
of O
SN O
on O
microbial O
growth O
. O

Neither O
raw O
nor O
retrograded O
resistant O
starch O
lowers O
fasting O
serum O
cholesterol O
concentrations O
in O
healthy O
normolipidemic O
subjects O
. O

( O
5 O
) O
. O

Hepatitis O
B O
markers O
in O
Lancashire O
police O
officers O
. O

In O
the O
other O
two O
groups O
( O
NAAR O
and O
PLA O
) O
, O
no O
variation O
in O
this O
serum O
protease O
was O
observed O
. O

Maximum O
likelihood O
estimation O
in O
covariance O
structure O
analysis O
with O
truncated O
data O
. O

A O
single O
dose O
of O
Cef O
was O
administered O
intravenously O
to O
11 O
patients O
with O
autoimmune O
diseases O
and O
varying O
degrees O
of O
renal O
impairment O
( O
Group O
I O
CLCR O
less O
than O
50 O
ml O
/ O
min O
, O
Group O
II O
CLCR O
greater O
than O
50 O
ml O
/ O
min O
) O
. O

The O
liver O
- O
specific O
sequence O
contains O
unique O
consensus O
sites O
for O
phosphorylation O
by O
cyclic B
AMP I
- I
dependent I
protein I
kinase I
and O
by O
protein B
kinase I
C I
. O

Effect O
of O
gentamycin O
on O
the O
kidney O
functional O
state O
in O
experimental O
pyelonephritis O

They O
represent O
a O
physiologically O
more O
meaningful O
pattern O
than O
the O
arithmetic O
mean O
with O
standard O
deviation O
. O

The O
Valpha B
and O
Vbeta B
domains I
have O
the O
canonical O
features O
of O
known O
teleost O
and I
mammalian I
TCR I
V I
domains I
, O
including O
conserved O
residues O
in O
the O
beginning O
of O
FR2 B
and O
at O
the O
end O
of O
FR3 B
. O

Whether O
this O
lack O
of O
preferential O
repair O
has O
to O
be O
explained O
by O
a O
defect O
in O
repair O
or O
in O
general O
transcription O
is O
unclear O
at O
present O
. O

This O
study O
examines O
LEIBNIZ O
' O
idea O
of O
Veterinary O
medicine O
in O
a O
biographical O
context O
. O

Dipetalonema O
( O
Acanthocheilonema O
) O
didelphis O
sp O
. O

n O
. O

However O
, O
the O
MCMI O
- O
III O
group O
profile O
was O
significantly O
lower O
in O
magnitude O
compared O
with O
the O
MCMI O
- O
II O
. O

While O
there O
is O
growing O
evidence O
that O
perception O
and O
imagery O
share O
common O
neural O
substrates O
, O
the O
fact O
that O
D O
. O
F O
. O
shows O
intact O
visual O
imagery O
in O
the O
face O
of O
a O
massive O
perceptual O
deficit O
in O
form O
vision O
challenges O
recent O
suggestions O
that O
these O
two O
psychological O
processes O
share O
common O
input O
pathways O
in O
early O
vision O
. O

An O
ethnic O
analysis O
was O
made O
of O
8947 O
cases O
of O
primary O
central O
nervous O
system O
( O
CNS O
) O
tumors O
seen O
at O
the O
Armed O
Forces O
Institute O
of O
Pathology O
( O
AFIP O
) O
, O
Washington O
, O
DC O
, O
from O
1971 O
to O
1985 O
. O

The O
exact O
function O
of O
IP B
- I
30 I
is O
not O
yet O
known O
, O
but O
it O
may O
play O
a O
role O
in O
gamma B
- I
interferon I
mediated O
immune O
reactions O
. O

The O
levels O
of O
products O
expressed O
from O
these O
chimeric O
cDNAs O
in O
COS O
cells O
were O
assessed O
by O
activity O
assay O
and O
by O
metabolic O
labeling O
of O
the O
proteins O
followed O
by O
immunoprecipitation O
and O
SDS O
- O
PAGE O
. O

In O
this O
report O
, O
we O
demonstrate O
that O
FRS2 B
forms O
a O
complex O
with O
the O
N B
- I
terminal I
SH2 I
domain I
of O
the O
protein B
tyrosine I
phosphatase I
Shp2 B
in O
response O
to O
FGF B
stimulation O
. O

It O
is O
concluded O
that O
the O
drinking O
ampules O
of O
VP O
- O
16 O
- O
213 O
can O
be O
replaced O
with O
the O
new O
oral O
capsules O
with O
a O
recommended O
initial O
dose O
of O
100 O
- O
130 O
mg O
/ O
m2 O
given O
in O
5 O
- O
day O
courses O
every O
21 O
- O
28 O
days O
. O

The O
H5 B
mutants I
were O
: O
DH5 O
( O
all O
amino O
acids O
in O
D O
configuration O
) O
and O
H5F B
( O
where O
all O
His O
are O
replaced O
by O
Phe O
at O
positions O
3 O
, O
7 O
, O
8 O
, O
15 O
, O
18 O
, O
19 O
, O
21 O
) O
. O

Although O
the O
number O
of O
kits O
has O
increased O
, O
the O
precision O
has O
improved O
, O
showing O
that O
more O
robust O
methods O
are O
now O
employed O
. O

These O
results O
establish O
that O
the O
DNA O
fragment O
we O
have O
isolated O
contains O
the O
human B
gp130 I
promoter I
and O
that O
interleukin B
- I
6 I
type I
cytokines I
may O
influence O
the O
activity O
of O
this O
promoter O
via O
activated O
STATs B
. O

When O
targeted O
to O
the O
GAL1 B
promoter I
by O
fusing O
with O
the O
Gal4p B
DNA I
- I
binding I
domain I
, O
Sko1p B
acts O
as O
an O
Ssn6 B
/ O
Tup1p B
- O
dependent O
repressor O
regulated O
by O
osmotic O
stress O
. O

The O
unexpected O
prevalence O
of O
c B
- I
myc I
and O
alpha B
- I
tubulin I
in O
the O
S O
- O
phase O
library O
is O
supported O
by O
Northern O
analysis O
of O
RNA O
from O
phase O
- O
synchronous O
cells O
. O

Growth O
factors O
promote O
cell O
survival O
through O
phosphorylation O
of O
Bad B
, O
resulting O
in O
its O
dissociation O
from O
Bcl B
- I
2 I
and O
Bcl B
- I
x I
( I
L I
) I
and O
its O
association O
with O
14 B
- I
3 I
- I
3tau I
. O

A O
representative O
group O
of O
patients O
undergoing O
open O
radical O
nephrectomy O
for O
clinical O
T1 O
, O
T2 O
lesions O
was O
also O
identified O
. O

By O
applying O
a O
sufficiently O
strong O
crusher O
gradient O
in O
the O
EPI O
pulse O
sequence O
, O
the O
temporal O
variation O
induced O
by O
SSFP O
disturbance O
can O
be O
suppressed O
due O
to O
diffusion O
. O

However O
, O
in O
each O
case O
in O
which O
either O
the O
DQ B
alpha I
- I
or O
DQ B
beta I
- I
chain I
was O
exchanged O
, O
major O
alterations O
or O
reversals O
of O
this O
pattern O
of O
interaction O
were O
observed O
. O

Only O
the O
native O
structure O
of O
phosphorylated O
ERK B
was O
recognized O
by O
VHR B
and O
was O
inactivated O
with O
a O
second O
- O
order O
rate O
constant O
of O
40 O
, O
000 O
M O
- O
1 O
s O
- O
1 O
. O

From O
1998 O
to O
1999 O
, O
a O
large O
number O
of O
community O
- O
acquired O
respiratory O
tract O
isolates O
of O
Streptococcus O
pneumoniae O
( O
n O
= O
566 O
) O
, O
Haemophilus O
influenzae O
( O
n O
= O
513 O
) O
and O
Moraxella O
catarrhalis O
( O
n O
= O
228 O
) O
were O
collected O
from O
15 O
centres O
in O
Australia O
, O
Hong O
Kong O
, O
Japan O
, O
China O
, O
the O
Philippines O
, O
Singapore O
, O
South O
Africa O
and O
Taiwan O
through O
the O
SENTRY O
Antimicrobial O
Surveillance O
Program O
. O

Diethyldithiocarbamate O
and O
amphetamine O
stereotype O
behavior O
. O

We O
used O
an O
immobilized O
template O
- O
based O
assay O
to O
examine O
transcription O
termination O
by O
VA1 B
, O
7SL B
, O
and O
Alu B
class O
III O
templates O
and O
the O
role O
of O
transcript O
release O
in O
the O
pol B
III I
terminator O
- O
dependent O
inhibition O
of O
processing O
of O
B1 B
- I
Alu I
transcripts I
. O

A O
novel O
zinc B
finger I
- I
containing I
RNA I
- I
binding I
protein I
conserved O
from O
fruitflies O
to O
humans O
. O

Although O
IL B
- I
6 I
can O
activate O
ERK B
- I
1 I
in O
HepG2 O
cells O
, O
STAT3 B
transactivation O
and O
Ser O
( O
727 O
) O
phosphorylation O
were O
not O
reduced O
by O
using O
the O
MAP B
kinase I
/ I
ERK I
kinase I
( O
MEK B
) O
inhibitor O
PD98059 O
or O
by O
overexpression O
of O
dominant B
- I
negative I
Raf I
. O

6 O
h O
after O
ingestion O
of O
aspirin O
on O
day O
1 O
to O
day O
14 O
, O
both O
TX O
and O
PGI2 O
levels O
also O
significantly O
decreased O
( O
p O
< O
0 O
. O
0001 O
) O
. O

Tritiated O
1 O
, O
24 O
, O
25 O
- O
trihydroxyvitamin O
D3 O
was O
synthesized O
biologically O
and O
used O
as O
tracer O
to O
monitor O
the O
recovery O
of O
endogenous O
metabolite O
during O
isolation O
from O
serum O
. O

Joint O
ultrasonography O
demonstrating O
thickening O
of O
synoviae O
and O
tendons O
has O
become O
a O
useful O
non O
- O
invasive O
diagnostic O
tool O
, O
although O
it O
is O
not O
specific O
for O
beta B
2 I
- I
M I
amyloidosis O
, O
and O
results O
depend O
on O
observer O
experience O
. O

Immunofluorescence O
tests O
for O
Borrelia O
burgdorferi O
serum O
antibodies O
had O
positive O
results O
, O
but O
G O
- O
penicillin O
treatment O
was O
ineffective O
. O

The O
size O
and O
the O
predicted O
N O
- O
terminal O
sequence O
of O
the O
mouse O
protein O
were O
confirmed O
experimentally O
. O

Nuclear O
runoff O
assays O
show O
that O
complementation O
by O
HPV16 B
E7 I
restores O
the O
ability O
of O
the O
E1A B
mutants I
to O
stimulate O
early B
gene I
expression O
at O
the O
level O
of O
transcription O
. O

Yet O
IL B
- I
2 I
and O
IFN B
- I
gamma I
production O
are O
independently O
regulated O
, O
as O
demonstrated O
by O
their O
differential O
expression O
in O
certain O
T O
cell O
subsets O
, O
suggesting O
that O
the O
regulatory O
elements O
in O
these O
two O
genes O
must O
differ O
. O

We O
conclude O
that O
a O
0 O
. O
25 O
mg O
dose O
of O
triazolam O
does O
not O
effectively O
counteract O
a O
posture O
- O
induced O
sleep O
disturbance O
, O
but O
induces O
changes O
in O
the O
EEG O
spectra O
which O
are O
typical O
for O
benzodiazepine B
receptor I
agonists O
. O

Anisotropy O
of O
Hc2 O
and O
the O
breadth O
of O
the O
resistive O
transition O
of O
polycrystalline O
YBa2Cu O

Microscopic O
examination O
of O
these O
colonies O
showed O
a O
high O
percentage O
of O
histiocytes O
identical O
to O
those O
seen O
in O
the O
patient O
' O
s O
bone O
marrow O
. O

Functional O
uncoupling O
of O
the O
Janus B
kinase I
3 I
- O
Stat5 B
pathway O
in O
malignant O
growth O
of O
human O
T O
cell O
leukemia O
virus O
type O
1 O
- O
transformed O
human O
T O
cells O
. O

The O
herpesvirus B
entry I
mediator I
C I
( O
HveC B
) O
, O
previously O
known O
as O
poliovirus B
receptor I
- I
related I
protein I
1 I
( O
PRR1 B
) O
, O
and O
the O
herpesvirus B
Ig I
- I
like I
receptor I
( O
HIgR B
) O
are O
the O
bona O
fide O
receptors O
employed O
by O
herpes O
simplex O
virus O
- O
1 O
and O
- O
2 O
( O
HSV O
- O
1 O
and O
- O
2 O
) O
for O
entry O
into O
the O
human O
cell O
lines O
most O
frequently O
used O
in O
HSV O
studies O
. O

4 O
- O
Hydroxyproline O
assays O
were O
performed O
to O
determine O
collagen B
content O
. O

The O
combination O
of O
ICP47 B
and O
US11 B
rendered O
fibroblasts O
negative O
for O
surface O
class B
I I
MHC I
and O
allowed O
a O
class B
I I
MHC I
- O
low O
population O
of O
T O
cells O
to O
be O
sorted O
by O
flow O
cytometry O
. O

Although O
IRS B
- I
1 I
, I
- I
2 I
, I
and I
- I
4 I
are O
similar O
in O
overall O
structure O
, O
IRS B
- I
3 I
is O
approximately O
50 O
% O
shorter O
and O
differs O
with O
respect O
to O
sites O
of O
tyrosine O
phosphorylation O
. O

The O
interaction O
of O
different O
protein O
systems O
with O
microtubules O
is O
a O
critical O
step O
in O
the O
cellular O
function O
of O
these O
organelles O
. O

In O
addition O
, O
we O
mapped O
a O
putative O
hnRNP B
A1 I
binding I
site I
in O
U5 B
RNA I
and O
demonstrated O
that O
p40CRS B
( O
hnRNP B
A1 I
) O
binding O
to O
that O
site O
correlates O
with O
CRS B
function O
. O

Sailer O
, O
K O
. O

Copyright O
2000 O
Academic O
Press O
. O

This O
element O
was O
required O
for O
both O
basal O
and O
activated O
expression O
and O
almost O
certainly O
functions O
as O
a O
TATA B
- I
binding I
protein I
interaction I
site I
. O

In O
HepG2 O
cells O
, O
SREBP B
- I
1c I
mRNA I
and O
precursor O
protein O
levels O
were O
induced O
by O
treatment O
with O
22 O
( O
R O
) O
- O
hydroxycholesterol O
and O
9 O
- O
cis O
- O
retinoic O
acid O
, O
confirming O
that O
endogenous O
LXR B
- O
RXR B
activation O
can O
induce O
endogenous B
SREBP I
- I
1c I
expression O
. O

Univariate O
and O
multivariate O
analysis O
was O
used O
to O
calculate O
the O
5 O
- O
year O
survival O
probabilities O
with O
respect O
to O
the O
following O
variables O
: O
age O
( O
< O
or O
= O
65 O
, O
> O
65 O
) O
, O
sex O
, O
depth O
of O
invasion O
( O
mucosal O
, O
submucosal O
) O
tumor O
location O
( O
upper O
, O
middle O
and O
lower O
third O
) O
, O
gross O
appearance O
( O
type O
I O
, O
type O
II O
and O
type O
III O
) O
, O
size O
( O
< O
or O
= O
1 O
. O
5 O
cm O
, O
> O
1 O
. O
5 O
cm O
) O
, O
presence O
or O
absence O
of O
lymph O
node O
metastasis O
, O
histological O
type O
( O
intestinal O
, O
diffuse O
) O
, O
extent O
of O

lymphadenectomy O
( O
limited O
or O
extended O
) O
, O
and O
type O
of O
gastrectomy O
( O
total O
or O
distal O
subtotal O
) O
. O

This O
integration O
results O
in O
the O
production O
of O
MMTV B
/ I
mdr3 I
fusion O
transcripts O
that O
originate O
from O
the O
antisense O
5 B
' I
LTR I
of O
the O
provirus O
. O

In O
32 O
of O
them O
the O
results O
of O
an O
exercise O
performance O
test O
based O
on O
heart O
rate O
response O
to O
submaximal O
exercise O
( O
VO2 O
, O
170 O
[ O
bpm O
] O
) O
was O
compared O
with O
another O
index O
of O
physical O
performance O
capacity O
, O
which O
is O
independent O
from O
heart O
rate O
: O
the O
ventilatory O
threshold O
. O

Sequence O
comparisons O
reveal O
that O
the O
P B
. I
woesei I
GTP I
- I
binding I
protein I
is O
strikingly O
related O
in O
sequence O
to O
eubacterial B
FtsZ I
and O
is O
marginally O
more O
similar O
to O
eukaryotic B
tubulins I
than O
are O
bacterial B
FtsZ I
proteins I
. O

Four O
of O
15 O
patients O
with O
R2 O
had O
rheumatoid O
arthritis O
and O
in O
two O
of O
these O
four O
cases O
the O
antibody O
was O
of O
the O
IgA B
class O
. O

Phosphate O
compounds O
in O
isolated O
, O
perfused O
hearts O
during O
pH O
variation O
due O
to O
changes O
in O
extracellular O
PCO2 O
and O
bicarbonate O

Vav B
and O
Dbl B
are O
members O
of O
a O
novel O
class O
of O
oncogene O
proteins O
that O
share O
significant O
sequence O
identity O
in O
a O
approximately O
250 O
- O
amino O
- O
acid O
domain O
, O
designated O
the O
Dbl B
homology I
domain I
. O

Serum O
AFP I
is O
a O
useful O
tumor O
marker O
for O
monitoring O
the O
results O
of O
therapy O
. O

The O
possible O
equivalency O
of O
a O
short O
form O
and O
the O
long O
form O
of O
the O
Multiple O
Affect O
Adjective O
Check O
List O
- O
Revised O
( O
MAACL O
- O
R O
; O
Zuckerman O
& O
Lubin O
, O
1985 O
) O
was O
studied O
by O
correlating O
both O
forms O
of O
the O
MAACL O
- O
R O
with O
the O
State O
Trait O
Personality O
Inventory O
( O
Spielberger O
, O
1995 O
) O
; O
the O
Affect O
Balance O
Scale O
( O
Bradburn O
, O
1969 O
) O
; O
and O
the O
Sensation O
Seeking O
Scale O
( O
Zuckerman O
, O
1978 O
) O
. O

The O
data O
show O
that O
processing O
time O
as O
a O
function O
of O
intensity O
is O
modified O
not O
only O
at O
the O
retina O
but O
also O
at O
later O
processing O
sites O
. O

Identification O
of O
masked O
toxins O
in O
the O
exotoxin B
complex I
of I
Staphylococcus I
pyogenes I
und O
Cl I
. I
perfringens I
; O
significance O
of O
their O
existence O
for O
clarifying O
of O
the O
masking O
mechanism O

Unlike O
LANA1 B
, O
LANA2 B
does O
not O
elicit O
a O
serologic O
response O
from O
patients O
with O
KS O
, O
PEL O
, O
or O
CD O
as O
measured O
by O
Western O
blot O
hybridization O
. O

However O
, O
this O
posttranslational O
modification O
of O
the O
gar2 B
protein I
does O
not O
appear O
to O
be O
essential O
for O
normal O
production O
of O
18S B
rRNA I
. O

Here O
, O
we O
analyzed O
the O
interaction O
of O
CYT B
- I
18 I
with O
a O
small O
RNA O
( O
P4 B
- O
P6 B
RNA O
) O
corresponding O
to O
the O
isolated O
P4 B
- O
P6 B
domain O
of O
the O
N B
. I
crassa I
mitochondrial I
large I
subunit I
ribosomal I
RNA I
intron I
. O

The O
deduced O
amino O
acid O
sequence O
of O
PutR B
( O
154 I
amino I
acid I
residues I
) I
showed O
homology O
to O
the O
small O
regulatory I
proteins I
Lrp I
, O
BkdR B
, O
and O
AsnC B
. O

When O
yeast O
cells O
reach O
a O
critical O
size O
in O
late O
G1 O
they O
simultaneously O
start O
budding O
, O
initiate O
DNA O
synthesis O
, O
and O
activate O
transcription O
of O
a O
set O
of O
genes O
that O
includes O
G1 B
cyclins I
CLN1 B
, O
CLN2 B
, O
and O
many O
DNA O
synthesis O
genes O
. O

Functional O
PET O
scanning O
in O
the O
preoperative O
assessment O
of O
cerebral O
arteriovenous O
malformations O
. O

In O
addition O
, O
269 O
mice O
from O
a O
( O
Mus O
spretus O
x O
C57BL O
/ O
6J O
) O
F1 O
x O
C57BL O
/ O
6J O
interspecific O
backcross O
were O
also O
used O
to O
order O
marker O
loci O
and O
calculate O
intergene O
distances O
for O
this O
region O
. O

Accordingly O
, O
superpeptide O
antigenic O
material O
was O
readily O
detected O
by O
immunoblotting O
cell O
extracts O
and O
enriched O
in O
vacuolar O
preparations O
of O
PrA B
deficient O
mutant O
cells O
. O

Blockade O
of O
T O
cell O
activation O
using O
a O
surface O
- O
linked O
single O
- O
chain O
antibody O
to O
CTLA B
- I
4 I
( O
CD152 B
) O
. O

Water O
pair O
and O
three O
- O
body O
potential O
of O
spectroscopic O
quality O
from O
Ab O
initio O
calculations O

4 O
) O
Mutation O
of O
the O
three O
extracellular O
cysteine O
residues O
of O
GFKAR B
beta I
indicated O
that O
the O
two O
conserved O
cysteine O
residues O
( O
C305 O
and O
C385 O
) O
, O
located O
between O
two O
transmembrane O
segments O
, O
form O
a O
solvent O
- O
accessible O
disulfide O
bond O
. O

In O
this O
work O
, O
we O
have O
used O
Xenopus O
oocyte O
maturation O
as O
a O
read O
- O
out O
for O
examining O
the O
ability O
of O
the O
neu B
tyrosine I
kinase I
( O
p185neu B
) O
to O
participate O
with O
the O
epidermal B
growth I
factor I
( I
EGF I
) I
receptor I
in O
a O
common O
signal O
transduction O
pathway O
. O

The O
best O
- O
studied O
neurotrophin O
, O
nerve B
growth I
factor I
( O
NGF B
) O
, O
is O
a O
major O
survival O
factor O
in O
sympathetic O
and O
sensory O
neurons O
and O
promotes O
differentiation O
in O
a O
well O
- O
studied O
model O
system O
, O
PC12 O
cells O
. O

In O
contrast O
, O
MAPK B
activation O
stimulated O
by O
the O
Gq B
- I
coupled I
alpha I
1B I
AR I
or O
M1 B
muscarinic I
cholinergic I
receptor I
is O
unaffected O
by O
expression O
of O
beta B
ARKct I
or O
RasN17 B
expression O
or O
by O
PTK B
inhibitors O
, O
but O
is O
blocked O
by O
expression O
of O
N B
delta I
Raf I
or O
by O
PKC B
depletion O
. O

Transcriptional O
analysis O
indicates O
that O
human B
TBP I
functions O
poorly O
at O
promoters O
recognized O
by O
RNA B
polymerases I
I I
and I
III I
and O
at O
RNA B
Pol I
II I
promoters I
lacking O
a O
conventional O
TATA O
element O
. O

The O
steady O
- O
state O
levels O
of O
Cdk2 B
, O
Cdk4 B
and O
Cyclin B
A I
proteins I
were O
unaffected O
by O
HPR B
, O
while O
those O
of O
Cyclin B
D1 I
were O
significantly O
reduced O
in O
all O
three O
cell O
lines O
. O

Mapping O
the O
5 O
' O
and O
3 O
' O
termini O
of O
TED B
RNAs I
indicated O
that O
the O
LTRs O
have O
a O
retroviral O
U3 B
- O
R O
- O
U5 B
structural O
organization O
that O
is O
capable O
of O
directing O
the O
synthesis O
of O
transcripts O
that O
represent O
potential O
substrates O
for O
reverse O
transcription O
and O
intermediates O
in O
transposition O
. O

Human O
forearm O
was O
used O
as O
a O
dummy O
ear O
and O
the O
electrode O
HN O
- O
5 O
was O
fixed O
thereon O
, O
and O
a O
sound O
stimulus O
of O
90dBnHL O
was O
delivered O
by O
the O
earphone O
( O
NC O
- O
3 O
) O
. O

These O
proteins O
were O
detected O
by O
an O
antibody O
which O
recognizes O
the O
N O
- O
terminus O
of O
SRK3 B
and O
, O
in O
an O
F2 O
progeny O
segregating O
for O
the O
S3 O
haplotype O
, O
were O
only O
expressed O
in O
plants O
possessing O
the O
S3 O
haplotype O
. O

The O
blood O
pressures O
in O
43 O
subjects O
were O
compared O
during O
rapid O
weight O
loss O
and O
at O
identical O
weights O
during O
post O
- O
fast O
weight O
gain O
( O
Study O
A O
) O
. O

The O
animals O
were O
followed O
over O
a O
1 O
- O
to O
6 O
- O
h O
posttraumatic O
course O
, O
and O
processed O
for O
the O
LM O
and O
TEM O
visualization O
of O
HRP B
. O

Total O
body O
water O
and O
distribution O
space O
of O
Na24 O
in O
women O
with O
various O
body O
weight O

Three O
experiments O
using O
20 O
microM O
2 O
- O
( O
hydroxyamino O
) O
- O
1 O
- O
methyl O
- O
6 O
- O
phenylimidazo O
[ O
4 O
, O
5 O
- O
b O
] O
pyridine O
( O
N O
- O
OH O
- O
PhIP O
) O
were O
performed O
to O
induce O
mutations O
in O
the O
dihydrofolate B
reductase I
( O
DHFR B
) O
gene O
of O
a O
hemizygous O
Chinese O
hamster O
ovary O
( O
CHO O
) O
cell O
line O
( O
UA21 O
) O
. O

This O
is O
mediated O
by O
vascular O
remodeling O
, O
an O
active O
process O
that O
results O
in O
a O
change O
in O
the O
geometry O
of O
the O
blood O
vessel O
. O

Our O
results O
separate O
these O
factors O
into O
four O
regulatory O
classes O
: O
( O
i O
) O
constitutive O
factors O
, O
such O
as O
Oct B
- I
1 I
and O
probably O
Sp1 B
, O
that O
are O
expressed O
in O
thymocytes O
at O
all O
stages O
; O
( O
ii O
) O
inducible O
factors O
, O
such O
as O
NF B
- I
kappa I
B I
and O
complexes O
binding O
to O
the O
region O
of O
a O
CD28 B
response I
element I
, O
that O
can O
be O
activated O
in O
all O
thymocytes O
, O
including O
those O
cells O
( O
CD4 B
+ O
CD8 B
+ O
TcRlow B
) O
that O
can O
undergo O
selection O
; O
( O
iii O
) O
inducible O
factors O
, O
such O
as O
NF B
- I
AT I
and O
AP B
- I
1 I
, O
that O
can O
be O
activated O

in O
mature O
( O
CD4 B
+ O
CD8 B
- O
TcRhigh B
) O
and O
immature O
( O
CD4 B
- O
CD8 B
- O
TcR B
- O
) O
thymocytes O
alike O
but O
not O
in O
the O
transitional O
stages O
when O
the O
cells O
( O
CD4 B
+ O
CD8 B
+ O
TcRlow B
) O
are O
subject O
to O
selection O
; O
and O
( O
iv O
) O
a O
factor O
containing O
CREB B
, O
which O
can O
be O
activated O
in O
thymocytes O
of O
all O
developmental O
stages O
by O
culture O
but O
does O
not O
require O
specific O
induction O
. O

Thus O
, O
22 O
% O
of O
MDS O
responded O
to O
oral O
PLAC O
. O

Posterior O
osteophytes O
around O
the O
posterior O
edges O
of O
vertebral O
body O
resulted O
in O
intervertebral O
insufficiency O
, O
and O
corresponded O
to O
hypertrophic O
spur O
in O
osteochondrosis O
. O

They O
showed O
moderate O
in O
vitro O
antifungal O
activity O
against O
a O
wide O
variety O
of O
fungi O
and O
yeasts O
including O
clinically O
important O
pathogens O
, O
and O
were O
highly O
effective O
in O
systemic O
infection O
with O
Candida O
albicans O
in O
mice O
after O
iv O
and O
im O
administrations O
. O

Of O
these O
four O
PP2C B
genes I
, O
the O
expression O
of O
the O
PP2Cbeta B
gene I
has O
been O
reported O
to O
be O
tissue O
- O
specific O
and O
development O
- O
dependent O
. O

We O
also O
show O
that O
in O
fusions O
with O
the O
DNA O
binding O
domain O
of O
GAL4 B
, O
full O
activity O
requires O
the O
entire O
BHV B
- I
alpha I
TIF I
, O
although O
both O
amino O
and O
carboxyl O
termini O
display O
some O
activity O
on O
their O
own O
. O

The O
induction O
by O
dexamethasone O
of O
rat B
liver I
CYP3A1 I
differs O
from O
classical O
glucocorticoid O
gene O
regulation O
in O
part O
because O
both O
glucocorticoids O
and O
antiglucocorticoids O
such O
as O
pregnenolone O
16 O
alpha O
- O
carbonitrile O
( O
PCN O
) O
induce O
CYP3A1 B
through O
transcriptional O
gene O
activation O
. O

Moreover O
, O
antibody O
binding O
to O
the O
same O
two O
determinants O
was O
also O
inhibited O
when O
ZAP B
- I
70 I
or O
the O
SH2 B
domains I
bound O
to O
the O
zeta B
chain I
or O
to O
a O
2pY B
- O
ITAM I
. O

This O
surface O
in O
specimens O
from O
unfertilized O
eggs O
is O
almost O
flat O
. O

Fetal O
transplants O
rescue O
axial O
muscle O
representations O
in O
M1 O
cortex O
of O
neonatally O
transected O
rats O
that O
develop O
weight O
support O
. O

We O
examined O
the O
contribution O
of O
the O
M O
- O
MuLV O
enhancers O
to O
the O
transcriptional O
activity O
and O
pathogenesis O
of O
M O
- O
MuLV O
by O
constructing O
LTRs O
containing O
heterologous O
enhancer O
elements O
. O

There O
were O
no O
major O
postoperative O
complications O
and O
at O
follow O
- O
up O
of O
the O
13 O
patients O
at O
10 O
to O
26 O
months O
after O
operation O
the O
results O
were O
satisfactory O
. O

The O
immunoreactive B
antithrombin I
III I
increased O
significantly O
while O
the O
increase O
in O
antithrombin B
III I
activity O
was O
insignificant O
in O
20 O
male O
Caucasian O
Danes O
upon O
3 O
weeks O
supplementation O
of O
the O
diet O
with O
10 O
ml O
daily O
of O
a O
cod O
liver O
oil O
concentrate O
. O

Northern O
blot O
analysis O
confirmed O
that O
ADAMTS B
- I
1 I
was O
up O
- O
regulated O
in O
both O
metaphyseal O
( O
14 O
- O
to O
35 O
- O
fold O
) O
and O
diaphyseal O
( O
4 O
. O
2 O
- O
fold O
) O
bone O
1 O
h O
after O
PTH B
- I
( I
1 I
- I
38 I
) I
injection O
and O
returned O
to O
control O
levels O
by O
24 O
h O
. O

The O
transcriptional B
transactivator I
( O
Tas B
) O
of O
simian O
foamy O
virus O
type O
1 O
strongly O
augments O
gene O
expression O
directed O
by O
both O
the O
promoter O
in O
the O
viral B
long I
terminal O
repeat O
and O
the O
newly O
discovered O
internal O
promoter O
located O
within O
the O
env B
gene I
. O

In O
the O
other O
two O
cases O
, O
fluoxetine O
adjunct O
produced O
benefits O
with O
no O
additional O
side O
effects O
. O

Deletion O
of O
this O
consensus O
element O
from O
the O
spo6 B
promoter I
abolished O
the O
transcription O
of O
spo6 B
+ I
and O
resulted O
in O
a O
sporulation O
deficiency O
. O

Chem O
. O

Mutations O
in O
the O
beta B
- I
galactosidase I
gene I
result O
in O
the O
lysosomal O
storage O
disorders O
GM1 O
- O
gangliosidosis O
and O
Morquio O
B O
syndrome O
. O

Results O
indicated O
that O
hobbies O
/ O
leisure O
activities O
, O
mobility O
, O
transport O
, O
social O
relationships O
, O
working O
capacity O
, O
energy O
and O
doing O
things O
slower O
were O
aspects O
relevant O
to O
IPF O
patients O
' O
QOL O
. O

We O
have O
generated O
transgenic O
fly O
strains O
with O
mutations O
affecting O
specific O
TRA B
- I
2 I
isoforms I
to O
investigate O
their O
individual O
roles O
in O
regulating O
the O
alternative O
processing O
of O
doublesex B
, O
exuperantia B
and O
tra B
- I
2 I
pre I
- I
mRNA I
. O

The O
IR4 B
ORF I
exhibits O
significant O
homology O
to O
the O
immediate B
- I
early I
gene I
US1 I
( O
ICP22 B
) O
of O
herpes O
simplex O
virus O
type O
1 O
and O
to O
the O
ICP22 B
homologs I
of O
varicella O
- I
zoster I
virus I
( I
ORF63 I
) O
, O
pseudorabies O
virus O
( O
RSp40 O
) O
, O
and O
equine B
herpesvirus I
4 I
( I
ORF4 I
) I
. O

These O
studies O
define O
a O
class O
of O
DNA O
elements O
with O
a O
mode O
of O
action O
that O
has O
not O
been O
heretofore O
described O
. O

We O
were O
able O
to O
demonstrate O
a O
significant O
statistical O
interaction O
effect O
between O
gender O
and O
globulin B
protein O
concentration O
on O
Vu O
( O
p O
= O
0 O
. O
022 O
) O
. O

Chest O
X O
- O
P O
revealed O
an O
abnormal O
shadow O
in O
the O
left O
upper O
lobe O
. O

From O
the O
frozen O
specimen O
, O
a O
large O
number O
of O
sections O
with O
good O
morphologic O
characteristics O
may O
be O
produced O
. O

These O
include O
not O
only O
antioxidants O
, O
N B
- I
methyl I
- I
D I
- I
aspartate I
receptor I
antagonists O
, O
inhibitors O
of O
glutamate O
release O
, O
calcium O
channel O
blockers O
, O
polyamine O
antagonists O
, O
and O
nitric B
oxide I
synthase I
inhibitors O
, O
but O
cannabinoids O
, O
aspirin O
, O
melatonin O
, O
and O
vitamin O
B O
- O
12 O
. O

A O
series O
of O
mutations O
were O
constructed O
within O
the O
E2 B
open I
reading I
frame I
which O
delete O
various O
regions O
of O
the O
conserved O
DNA O
binding O
and O
transactivation O
domains O
as O
well O
as O
the O
internal O
hinge O
region O
. O

CrkII B
and O
CrkL B
constitutively O
formed O
complex O
with O
the O
guanine B
nucleotide I
exchange I
factor I
C3G B
, O
in O
unstimulated O
as O
well O
as O
PDGF B
- O
stimulated O
cells O
. O

In O
further O
support O
of O
the O
biological O
significance O
of O
these O
DNA O
- O
protein O
interactions O
, O
the O
IL2RA B
oligonucleotide I
from O
- O
291 O
to O
- O
245 O
proved O
to O
be O
sufficient O
in O
either O
orientation O
to O
confer O
PMA O
inducibility O
to O
the O
mitogen O
- O
insensitive O
thymidine B
kinase I
gene I
promoter I
in O
Jurkat O
cells O
. O

The O
plasma O
steroid O
levels O
in O
the O
CAPD O
patients O
after O
each O
infusion O
rate O
were O
equal O
to O
or O
greater O
than O
the O
levels O
in O
normal O
subjects O
. O

With O
BCPP O
protocol O
, O
12 O
achieved O
a O
complete O
response O
, O
11 O
evidenced O
a O
75 O
% O
response O
, O
and O
7 O
displayed O
a O
50 O
% O
response O
. O

The O
RPC31 B
gene I
encoding O
the O
C31 B
subunit I
of I
Saccharomyces B
cerevisiae I
RNA I
polymerase I
C I
( I
III I
) I
has O
been O
isolated O
, O
starting O
from O
a O
C O
- O
terminal O
fragment O
cloned O
on O
a O
lambda O
gt11 O
library O
. O

Electrophoretic O
mobility O
shift O
assays O
demonstrated O
that O
sequences O
within O
this O
region O
bind O
members O
of O
the O
Sp1 B
family I
of I
transcription I
factors I
. O

The O
phosphatase O
active O
site O
is O
located O
within O
residues O
367 O
- O
374 O
. O

After O
at O
least O
one O
year O
follow O
- O
up O
, O
argon O
laser O
showed O
a O
statistically O
significant O
effect O
( O
p O
less O
of O
0 O
, O
01 O
; O
Kolmogorow O
- O
Smirnov O
test O
) O
on O
the O
stabilization O
or O
the O
amelioration O
of O
visual O
acuity O
compared O
with O
the O
non O
treated O
group O
. O

Further O
studies O
aim O
at O
characterizing O
the O
trans O
- O
acting O
factors O
that O
mediate O
the O
spatial O
and O
temporal O
expression O
of O
Hox B
genes I
in O
the O
developing O
embryo O
. O

Contraindications O
to O
adenosine O
and O
drug O
interactions O
are O
noted O
in O
this O
article O
. O

As O
to O
the O
side O
effects O
of O
SISO O
, O
cylindruria O
with O
aggravation O
of O
microscopic O
hematuria O
and O
elevations O
of O
GOT B
, O
GPT B
and O
A1 O
- O
P O
were O
observed O
each O
one O
of O
them O
, O
respectively O
. O

Serum O
C3 B
was O
measured O
in O
1 O
, O
282 O
school O
children O
with O
abnormal O
urinary O
findings O
between O
1980 O
and O
1997 O
. O

Two O
measures O
of O
sensory O
inhibition O
were O
employed O
, O
S2 O
/ O
S1 O
x O
100 O
amplitude O
ratio O
and O
S1 O
- O
S2 O
amplitude O
difference O
. O

Computer O
analysis O
revealed O
that O
the O
human B
protein I
P1 I
sequence I
corresponding O
to O
amino O
acid O
residues O
within O
the O
N O
- O
terminal O
RNA O
binding O
domain O
displays O
high O
homology O
( O
greater O
than O
54 O
% O
identity O
; O
61 O
to O
94 O
% O
similarity O
) O
with O
two O
animal O
virus O
proteins O
which O
possess O
RNA O
binding O
activity O
( O
vaccinia B
virus I
E3L I
; O
rotavirus B
VP2 I
) O
and O
two O
proteins O
of O
unknown O
function O
( O
murine B
TIK I
; O
rotavirus B
NS34 I
) O
, O
but O
which O
are O
likely O
RNA O
binding O
proteins O
. O

Determinants O
of O
recurrent O
ischaemia O
and O
revascularisation O
procedures O
after O
thrombolysis O
with O
recombinant O
tissue B
plasminogen I
activator I
in O
primary O
coronary O
occlusion O
. O

For O
large O
breasts O
( O
> O
70 O
mm O
) O
the O
use O
of O
X O
- O
ray O
sets O
such O
as O
the O
IGE O
DMR O
, O
which O
automatically O
select O
the O
beam O
quality O
for O
each O
breast O
, O
resulted O
in O
lower O
doses O
compared O
with O
sets O
using O
manual O
tube O
potential O
selection O
. O

Experimental O
exposure O
of O
poults O
with O
each O
of O
two O
strains O
representing O
the O
rarely O
reported O
capsular O
group O
B O
indicated O
that O
both O
were O
virulent O
. O

Computer O
system O
trains O
employees O
to O
meet O
JCAHO O
safety O
ED O
requirements O
. O

Finally O
, O
transfection O
of O
Grb10 B
genes I
with O
specific O
mutations O
in O
their O
SH2 B
domains I
induces O
apoptosis O
in O
HTC B
- I
IR I
and O
COS O
- O
7 O
cells O
. O

Comparison O
of O
dose O
volume O
histograms O
revealed O
that O
the O
6 O
- O
field O
plan O
spared O
relatively O
more O
heart O
whereas O
the O
8 O
- O
field O
plan O
spared O
relatively O
more O
lung O
. O

We O
have O
demonstrated O
that O
a O
putative O
HOX B
cofactor I
, O
PBX1A B
, O
participates O
in O
cooperative O
DNA O
binding O
with O
HOXA B
- I
1 I
and O
the O
Deformed B
group I
protein I
HOXD B
- I
4 I
. O

The O
CHL B
1 I
( O
CTF B
1 I
) O
gene O
product O
of O
Saccharomyces O
cerevisiae O
is O
important O
for O
chromosome O
transmission O
and O
normal O
cell O
cycle O
progression O
in O
G2 O
/ O
M O
. O

Heterotrimeric B
guanine I
nucleotide I
- I
binding I
proteins I
( O
G B
- I
proteins I
) O
function O
as O
signal O
transducers O
for O
a O
variety O
of O
hormone O
- O
coupled O
enzyme O
and O
ion O
transport O
systems O
in O
eukaryotic O
cells O
. O

In O
contrast O
, O
polypropylene O
glycol O
, O
PG O
- O
C O
, O
and O
corn O
oil O
all O
removed O
68 O
- O
86 O
% O
of O
the O
MDI O
in O
the O
first O
h O
, O
74 O
- O
79 O
% O
at O
4 O
h O
, O
and O
72 O
- O
86 O
% O
at O
8 O
h O
. O

DDD O
( O
R O
) O
pacing O
with O
automatic O
mode O
switch O
in O
patients O
with O
paroxysmal O
atrial O
fibrillation O
following O
AV O
nodal O
ablation O
. O

There O
was O
a O
highly O
significant O
decrease O
in O
serum B
eosinophil I
cationic I
protein I
in O
patients O
with O
elevated O
AGA O
. O

Sequence O
analysis O
revealed O
that O
Hv B
- I
p68 I
belongs O
to O
the O
large O
family O
of O
FAD B
- I
dependent I
glucose I
methanol I
choline I
oxidoreductases I
and O
that O
it O
shares O
significant O
sequence O
identity O
( O
> O
67 O
% O
) O
with O
the O
alcohol B
oxidases I
of O
the O
methylotrophic O
yeasts O
. O

It O
is O
required O
for O
correct O
expression O
of O
both O
genes O
. O

In O
the O
HIV B
LTR I
, O
this O
region O
has O
been O
demonstrated O
to O
have O
both O
positive O
and O
negative O
regulatory O
effects O
on O
HIV O
gene O
expression O
. O

A O
comparison O
between O
attenuation O
- O
corrected O
and O
- O
uncorrected O
transmission O
- O
emission O
SPECT O
images O
obtained O
with O
Tl O
- O
201 O
in O
CAD O
patients O

Accumulation O
of O
amines O
in O
the O
isolated O
perfused O
rabbit O
lung O
. O

Since O
it O
is O
often O
necessary O
to O
resort O
to O
thoracotomy O
as O
a O
final O
step O
in O
making O
such O
a O
diagnosis O
, O
we O
have O
sought O
a O
procedure O
that O
is O
simpler O
while O
capable O
of O
providing O
the O
same O
information O
. O

Both O
control O
and O
Mn O
- O
exposed O
quail O
accumulated O
5 O
to O
10 O
times O
more O
Mn O
in O
their O
livers O
than O
similarly O
treated O
rodents O
. O

Small B
t I
also O
inhibited O
the O
dephosphorylation O
of O
cAMP B
- I
dependent I
protein I
kinase I
( O
PKA B
) O
- O
phosphorylated O
CREB B
in O
rat O
liver O
nuclear O
extracts O
. O

A O
retrospective O
analysis O
of O
data O
accumulated O
over O
a O
21 O
- O
month O
period O
, O
from O
December O
1989 O
to O
September O
1991 O
, O
regarding O
investigational O
use O
of O
a O
308 O
nanometer O
Xenon O
Chloride O
Excimer O
Laser O
Coronary O
Angioplasty O
( O
ELCA O
) O
system O
( O
Advanced O
Interventional O
Systems O
, O
Inc O
. O
, O
Irvine O
, O
CA O
) O
was O
performed O
. O

Presently O
, O
we O
made O
chain O
pair O
switch O
, O
chimeric O
, O
and O
site B
mutant I
gamma I
delta I
TCRs I
and O
transfected O
them O
into O
TCR B
- I
mutant O
Jurkat O
T O
cells O
to O
examine O
the O
effects O
of O
changing O
the O
TCR B
gamma I
junctional I
region I
sequences I
on O
reactivity O
to O
prenyl O
pyrophosphate O
Ags O
. O

Fos B
/ O
Jun B
bound O
to O
an O
unacetylated O
nucleosome O
with O
only O
a O
4 O
- O
to O
5 O
- O
fold O
reduction O
in O
DNA O
binding O
affinity O
compared O
with O
naked O
DNA O
. O

Activated O
Ki B
- I
Ras I
suppresses O
12 O
- O
O O
- O
tetradecanoylphorbol O
- O
13 O
- O
acetate O
- O
induced O
activation O
of O
the O
c B
- I
Jun I
NH2 I
- I
terminal I
kinase I
pathway O
in O
human O
colon O
cancer O
cells O
. O

Electromobility O
shift O
analysis O
using O
oligonucleotides O
encompassing O
the O
proximal O
, O
distal O
, O
and O
BED B
/ O
AP B
- I
1 I
- O
binding O
regions O
failed O
to O
demonstrate O
selective O
transactivation O
after O
CD2 B
signaling O
of O
LPMC O
. O

The O
present O
report O
shows O
that O
a O
cis O
- O
acting O
element O
between O
- O
189 O
and O
- O
175 O
bp O
, O
which O
binds O
thyroid B
transcription I
factor I
- I
1 I
( O
TTF B
- I
1 I
) O
, O
is O
involved O
in O
both O
activities O
. O

Increased O
platelet B
- I
derived I
growth I
factor I
production O
and O
intimal O
thickening O
during O
healing O
of O
Dacron O
grafts O
in O
a O
canine O
model O
. O

Despite O
their O
high O
degree O
of O
sequence O
similarity O
, O
all O
five O
RFC B
genes I
are O
essential O
for O
cell O
proliferation O
in O
S O
. O
cerevisiae O
. O

Moreover O
, O
we O
show O
that O
ectopic O
expression O
of O
the O
E2A B
protein I
E12 B
in O
this O
macrophage O
line O
results O
in O
the O
induction O
of O
many O
B O
lineage O
genes O
, O
including O
EBF B
, O
IL7Ralpha B
, O
lambda5 B
, O
and O
Rag B
- I
1 I
, O
and O
the O
ability O
to O
induce O
kappa B
light I
chain I
in O
response O
to O
mitogen O
. O

Also O
, O
anti B
- I
HIV I
gp120 I
and O
anti B
- I
CD4 I
crosslinking O
induced O
a O
10 O
- O
15 O
- O
fold O
increase O
in O
levels O
of O
both O
PI B
3 I
- I
and I
PI I
4 I
- I
kinase I
activity O
in O
anti B
- I
CD4 I
precipitates O
. O

The O
differences O
of O
percentages O
for O
HIV O
- O
1 O
infection O
in O
intravenous O
drug O
users O
( O
IVDU O
) O
were O
67 O
. O
7 O
% O
and O
in O
non O
IVDU O
were O
3 O
. O
8 O
% O
with O
a O
significant O
statistical O
difference O
( O
chi O
2 O
= O
557 O
. O
5 O
; O
p O
< O
0 O
. O
0001 O
) O
. O

One O
of O
the O
ORFs O
in O
the O
Methanococcus O
jannaschii O
genome O
possesses O
high O
similarity O
to O
the O
M O
. O
aeolicus O
ilvB B
, O
indicating O
that O
it O
is O
an O
authentic O
AHAS B
. O

More O
than O
80 O
% O
appropriate O
lever O
responding O
was O
established O
after O
27 O
, O
38 O
and O
44 O
daily O
training O
sessions O
with O
DN O
- O
2327 O
, O
diazepam O
and O
PTZ O
, O
respectively O
, O
as O
the O
training O
drug O
. O

We O
have O
used O
nuclear O
magnetic O
resonance O
( O
NMR O
) O
to O
obtain O
the O
structure O
of O
an O
RNA O
" O
kissing O
" O
hairpin O
complex O
formed O
between O
the O
HIV B
- I
2 I
TAR I
hairpin I
loop O
and O
a O
hairpin O
with O
a O
complementary O
loop O
sequence O
. O

RESULTS O
: O
In O
men O
, O
weight O
/ O
height2 O
met O
the O
criteria O
for O
a O
satisfactory O
index O
in O
that O
there O
was O
a O
very O
strong O
correlation O
with O
triceps O
skinfold O
, O
and O
a O
negligible O
correlation O
with O
height O
. O

PRP20 B
is O
related O
, O
both O
in O
structure O
and O
function O
, O
to O
the O
RCC1 B
gene I
of O
mammals O
and O
the O
PIM1 B
gene I
of O
Schizosaccharomyces O
pombe O
, O
both O
of O
which O
appear O
to O
regulate O
entry O
into O
mitosis O
and O
chromosome O
condensation O
. O

No O
consensus O
binding O
sequence O
could O
be O
discerned O
in O
these O
fragments O
and O
bound O
factor O
is O
in O
rapid O
equilibrium O
with O
unbound O
. O

A O
selective O
transcriptional O
induction O
system O
for O
mammalian O
cells O
based O
on O
Gal4 B
- O
estrogen B
receptor I
fusion O
proteins O
. O

Kinase O
renaturation O
tests O
designed O
to O
detect O
reactivated O
protein O
kinases O
after O
electrophoresis O
in O
sodium O
dodecyl O
sulfate O
- O
polyacrylamide O
gels O
revealed O
the O
presence O
of O
a O
60 O
- O
kDa O
kinase O
in O
the O
washed O
immunoprecipitate O
obtained O
from O
liver O
cytosol O
using O
anti B
- I
AHR I
antibody I
( O
IgG B
) O
and O
protein B
A I
/ I
G I
/ O
agarose O
beads O
but O
not O
when O
a O
nonspecific O
IgG B
was O
used O
instead O
of O
anti B
- I
AHR I
antibody I
. O

Immunoblotting O
using O
a O
phosphospecific O
anti B
- I
Src I
( I
416Y I
) I
antibody I
showed O
a O
ceramide O
- O
induced O
increase O
in O
pp60 B
( O
src B
) O
tyrosine O
phosphorylation O
. O

In O
contrast O
, O
dominant B
negative I
Rac I
( O
N17Rac1 B
) O
inhibited O
JNK B
activation O
by O
Galpha12 B
in O
HEK293 O
cells O
as O
well O
as O
three O
other O
cell O
lines O
. O

Metabolic O
studies O
of O
tetrabenazine O
, O
a O
psychotropic O
drug O
in O
animals O
and O
man O
. O

Reading O
disability O
in O
twins O
. O

These O
results O
suggest O
that O
1a B
, O
which O
also O
interacts O
independently O
with O
the O
ER O
and O
viral O
RNA O
, O
is O
a O
key O
organizer O
of O
RNA O
replication O
complex O
assembly O
. O

HrpE B
is O
similar O
to O
YscL B
of O
Yersinia O
spp O
. O

Effect O
of O
a O
high O
sugar O
intake O
on O
some O
metabolic O
and O
regulatory O
indicators O
in O
young O
men O
. O

Production O
of O
a O
chicken O
meat O
infusion O
broth O
suitable O
for O
the O
mass O
production O
of O
a O
Haemophilus O
gallinarum O
bacterin O
. O

The O
results O
indicate O
that O
expression O
of O
proU B
in O
E O
. O
coli O
is O
directed O
from O
two O
promoters O
, O
one O
( O
P2 O
) O
characterized O
earlier O
by O
other O
workers O
with O
the O
start O
site O
of O
transcription O
60 O
nucleotides O
upstream O
of O
the O
initiation O
codon O
of O
the O
first O
structural O
gene O
( O
proV B
) O
, O
and O
the O
other O
( O
P1 O
) O
situated O
250 O
nucleotides O
upstream O
of O
proV B
. O

The O
maximal O
rise O
in O
plasma O
adrenaline O
was O
of O
similar O
magnitude O
in O
all O
three O
groups O
but O
a O
lower O
plasma O
glucose O
was O
required O
to O
stimulate O
this O
hormonal O
response O
in O
the O
' O
unaware O
' O
patients O
, O
in O
whom O
the O
plasma O
adrenaline O
concentration O
was O
lower O
at O
the O
time O
of O
the O
reaction O
. O

Through O
interactions O
with O
the O
pre O
- O
mRNA O
and O
the O
CTD O
domain O
of O
the O
Polymerase B
II I
, O
SR B
proteins I
have O
been O
shown O
to O
regulate O
alternative O
splicing O
. O

One O
child O
, O
recently O
fed O
, O
vomited O
a O
small O
amount O
of O
breast O
milk O
after O
a O
short O
period O
of O
crying O
and O
apparently O
had O
a O
laryngospasm O
, O
shown O
by O
a O
sudden O
drop O
in O
the O
PtcO2 O
level O
without O
any O
other O
signs O
of O
discomfort O
. O

Changes O
in O
the O
size O
and O
organisation O
of O
the O
brain O
in O
man O
and O
his O
ancestors O
. O

Only O
Staphylococcus O
xylosus O
SX63 O
was O
detected O
as O
a O
strain O
with O
high O
adherence O
activity O
. O

It O
also O
produces O
an O
NH2 O
terminus O
that O
corresponds O
to O
an O
equivalent O
NH2 O
terminus O
on O
the O
processed O
matrix O
form O
of O
the O
similar O
alpha1 B
( I
XI I
) I
chain I
, O
thus O
suggesting O
physiological O
significance O
. O

Transmission O
of O
mutans O
streptococci O
to O
infants O
following O
short O
term O
application O
of O
an O
iodine O
- O
NaF O
solution O
to O
mothers O
' O
dentition O
. O

Expression O
of O
Msx B
- I
1 I
and O
Msx B
- I
2 I
has O
been O
studied O
during O
development O
of O
the O
osteoblast O
phenotype O
, O
but O
the O
role O
of O
Dlx B
in O
this O
context O
and O
in O
the O
regulation O
of O
bone O
- O
expressed O
genes O
is O
unknown O
. O

Scalp O
blood O
flow O
was O
recorded O
by O
a O
laser O
Doppler O
flow O
sensor O
that O
was O
incorporated O
in O
the O
transcutaneous O
PO2 O
electrode O
. O

DESIGN O
: O
Activating B
protein I
- I
1 I
( O
AP B
- I
1 I
) O
and O
Tat B
- O
induced O
transcription O
were O
assessed O
using O
Jun B
and O
hybrid O
Tat B
/ O
Jun B
- O
expression O
plasmids O
and O
reporter O
gene O
constructs O
which O
contained O
AP B
- I
1 I
binding I
sites I
upstream O
of O
the O
rat B
prolactin I
TATAA I
element I
or O
an O
HIV B
- I
1 I
LTR I
construct I
in O
which O
AP B
- I
1 I
binding I
sites I
replaced O
the O
TAR O
element O
. O

Mycobacterium O
tuberculosis O
inhibits O
IFN B
- I
gamma I
transcriptional O
responses O
without O
inhibiting O
activation O
of O
STAT1 B
. O

Rabbit B
KCC1 I
( O
rbKCC1 B
) O
and O
rat B
KCC1 I
( O
rtKCC1 B
) O
were O
cloned O
by O
screening O
rabbit O
kidney O
and O
rat O
brain O
cDNA O
libraries O
using O
homologous O
cDNA O
probes O
. O

However O
, O
using O
wet O
weight O
/ O
dry O
weight O
methodology O
, O
we O
found O
that O
administration O
of O
U74006F O
significantly O
reduced O
water O
content O
in O
the O
right O
hippocampus O
( O
contralateral O
to O
the O
site O
of O
injury O
) O
compared O
to O
saline O
- O
treated O
animals O
( O
p O
less O
than O
0 O
. O
05 O
) O
. O

GH B
stimulated O
the O
activity O
of O
promoter O
fragments O
with O
5 O
' O
- O
ends O
between O
nucleotide O
( O
nt O
) O
- O
2001 O
and O
nt O
- O
653 O
by O
1 O
. O
9 O
- O
to O
2 O
. O
7 O
- O
fold O
. O

We O
have O
recently O
shown O
that O
induced O
expression O
of O
a O
CDK B
target O
site O
- O
deficient O
mutant O
, O
Op18 B
- O
S25A O
, O
S38A O
, O
blocks O
human O
cell O
lines O
during O
G2 O
/ O
M O
transition O
. O

A O
protein O
which O
promotes O
DNA O
strand O
transfer O
between O
linear O
double O
- O
stranded O
M13mp19 B
DNA O
and O
single O
- O
stranded O
viral B
M13mp19 I
DNA I
has O
been O
isolated O
from O
recA B
- I
E I
. I
coli I
. O

The O
brains O
were O
processed O
according O
to O
the O
tetramethylbenzidine O
( O
TMB O
) O
protocol O
of O
Mesulam O
( O
' O
78 O
) O
and O
studied O
with O
darkfield O
microscopy O
. O

Seizure O
recurrence O
( O
HR O
1 O
. O
30 O
; O
95 O
% O
CI O
= O
0 O
. O
84 O
, O
2 O
. O
01 O
) O
and O
antiepileptic O
drug O
treatment O
( O
HR O
0 O
. O
97 O
; O
95 O
% O
CI O
= O
0 O
. O
67 O
, O
1 O
. O
38 O
) O
did O
not O
influence O
mortality O
rate O
. O

One O
day O
postoperatively O
, O
the O
mononuclear B
leukocyte I
beta I
2 I
- I
receptor I
density O
decreased O
maximally O
by O
45 O
+ O
/ O
- O
11 O
% O
in O
the O
enflurane O
patients O
, O
and O
by O
53 O
+ O
/ O
- O
6 O
% O
in O
the O
neurolept O
patients O
. O

The O
serum O
levels O
of O
cortisol O
and O
prolactin B
after O
nasal O
instillation O
of O
a O
suspension O
of O
vaginal O
exudate O
showed O
lower O
values O
than O
in O
control O
conditions O
( O
nasal O
instillation O
of O
saline O
) O
. O

IgE B
antibody I
to O
twelve O
common O
food O
and O
inhalant O
allergens O
was O
measured O
by O
enzyme O
- O
linked O
immunosorbent O
assay O
( O
ELISA O
) O
in O
the O
sera O
of O
thirteen O
atopic O
patients O
with O
one O
or O
more O
allergic O
disorders O
( O
asthma O
occurring O
in O
eleven O
; O
rhinitis O
in O
ten O
; O
eczema O
in O
six O
; O
urticaria O
in O
four O
; O
mouth O
and O
gastro O
- O
intestinal O
symptoms O
in O
six O
) O
, O
of O
twelve O
non O
- O
atopic O
patients O
with O
various O
clinical O
symptoms O
( O
asthma O
in O
four O
; O
rhinitis O
in O
four O
; O
eczema O
in O
one O
; O
urticaria O
in O

two O
; O
mouth O
and O
gastro O
- O
intestinal O
symptoms O
in O
four O
) O
and O
sixteen O
cord O
blood O
sera O
. O

Innervation O
of O
reticular O
papillae O
in O
sheep O
and O
goats O
. O

Biochemical O
status O
was O
examined O
using O
erythrocyte O
enzyme O
function O
and O
blood O
vitamin O
levels O
. O

Sexual O
adventurism O
, O
high O
- O
risk O
behavior O
, O
and O
human O
immunodeficiency O
virus O
- O
1 O
seroconversion O
among O
the O
Chicago O
MACS O
- O
CCS O
cohort O
, O
1984 O
to O
1992 O
. O

Pendulin B
, O
a O
Drosophila O
protein O
with O
cell O
cycle O
- O
dependent O
nuclear O
localization O
, O
is O
required O
for O
normal O
cell O
proliferation O
. O

Paradoxically O
there O
is O
a O
decrease O
in O
the O
level O
of O
soluble O
A B
beta I
secreted O
from O
the O
cells O
. O

Signaling O
through O
mitogen B
- I
activated I
protein I
kinase I
and O
Rac B
/ O
Rho B
does O
not O
duplicate O
the O
effects O
of O
activated O
Ras B
on O
skeletal O
myogenesis O
. O

Internalization O
of O
prolactin B
receptor I
and O
prolactin B
in O
transfected O
cells O
does O
not O
involve O
nuclear O
translocation O
. O

Using O
mammalian O
expression O
vectors O
( O
pRSV O
- O
neo B
and O
pSV2 O
- O
neo B
) O
, O
antisense O
constructs O
for O
perforin B
and O
granzyme B
B I
were O
independently O
electroporated O
into O
YT O
- O
INDY O
, O
a O
human B
non I
- I
MHC I
- O
restricted O
, O
IL B
- I
2 I
- O
independent O
, O
cytotoxic O
lymphocyte O
. O

When O
expressed O
in O
Escherichia O
coli O
, O
SH B
- I
PTP2 I
displays O
tyrosine B
- I
specific I
phosphatase I
activity O
. O

The O
structure O
elucidation O
of O
the O
iridoid O
compounds O
2 O
and O
3 O
are O
discussed O
in O
detail O
. O

The O
results O
of O
a O
study O
to O
investigate O
the O
effectiveness O
of O
a O
propolis O
- O
containing O
mouthrinse O
in O
the O
inhibition O
of O
de O
novo O
plaque O
formation O
are O
presented O
. O

Effects O
of O
shosaikoto O
, O
an O
oriental O
herbal O
medicinal O
mixture O
, O
on O
restraint O
- O
stressed O
mice O
. O

The O
region O
surrounding O
the O
ZNF35 B
zinc I
finger I
protein I
gene I
on O
3p21 O
is O
of O
particular O
interest O
, O
as O
this O
region O
of O
chromosome O
3 O
is O
frequently O
involved O
in O
rearrangements O
and O
/ O
or O
deletions O
associated O
with O
various O
human O
tumors O
including O
lung O
and O
renal O
carcinoma O
. O

In O
the O
promoters O
of O
many O
immediate B
early I
genes I
, O
including O
c B
- I
fos I
, O
CArG O
DNA O
regulatory O
elements O
mediate O
basal O
constituitive O
expression O
and O
rapid O
and O
transient O
serum O
induction O
. O

The O
biological O
activity O
of O
these O
mutants O
was O
tested O
by O
determining O
their O
capacity O
to O
( O
i O
) O
reconstitute O
RNA B
polymerase I
activity O
in O
vivo O
by O
cotransfection O
with O
proteins B
NP I
, O
PB1 B
, O
and O
PA B
and O
a O
virion O
- O
like O
RNA O
encoding O
the O
cat B
gene I
into O
vaccinia O
virus O
T7 O
- O
infected O
COS O
- O
1 O
cells O
and O
( O
ii O
) O
complete O
with O
the O
wild O
- O
type I
PB2 I
activity O
. O

Transient O
thyrotoxicosis O
in O
primary O
anti O
- O
phospholipid O
syndrome O
. O

We O
were O
unable O
to O
construct O
chvI B
or O
chvG B
insertion I
mutants I
of O
R O
. O
meliloti O
, O
whereas O
mutants O
carrying O
insertions O
outside O
of O
these O
genes O
were O
readily O
obtained O
. O

Western O
blot O
analyses O
reveals O
that O
cJun B
and O
the O
C B
/ I
EBP I
family I
member O
C B
/ I
EBP I
- I
beta I
are O
physiologically O
relevant O
transcription O
factors O
whose O
expression O
corresponds O
with O
mda B
- I
7 I
mRNA I
expression O
. O

In O
isolated O
olfactory O
cilia O
certain O
odorants O
elicit O
a O
rapid O
and O
transient O
cAMP O
response O
, O
terminated O
by O
a O
concerted O
process O
which O
requires O
the O
action O
of O
two O
protein O
kinases O
, O
protein B
kinase I
A I
( O
PKA B
) O
and O
a O
receptor B
- I
specific I
kinase I
( O
GRK3 B
) O
( O
Schleicher O
, O
S O
. O
, O
Boekhoff O
, O
I O
. O

Acetyl B
- I
CoA I
carboxylase I
from I
yeast I
is O
an O
essential O
enzyme O
and O
is O
regulated O
by O
factors O
that O
control O
phospholipid O
metabolism O
. O

To O
investigate O
the O
role O
of O
this O
domain O
in O
the O
incorporation O
of O
the O
SIV B
Env I
into O
virions O
, O
we O
generated O
a O
series O
of O
SIV B
Env I
mutants I
carrying O
small O
in O
- O
frame O
deletions O
within O
the O
cytoplasmic O
domain O
. O

Computer O
analysis O
, O
simple O
correlation O
hypothesis O
, O
and O
distribution O
method O
with O
1 O
degree O
of O
freedom O
revealed O
significant O
correlation O
between O
vertical O
root O
fracture O
and O
the O
technique O
of O
instrumentation O
and O
obturation O
of O
the O
canal O
( O
p O
= O
0 O
. O
025 O
) O
, O
the O
length O
of O
the O
post O
( O
p O
= O
0 O
. O
05 O
) O
, O
and O
the O
existence O
of O
the O
post O
( O
p O
= O
0 O
. O
05 O
) O
. O

Minute B
virus I
of I
mice I
NS1 B
, O
an O
83 O
- O
kDa O
mainly O
nuclear O
phosphoprotein O
, O
is O
the O
only O
viral O
nonstructural O
protein O
required O
in O
all O
cell O
types O
and O
it O
is O
involved O
in O
multiple O
processes O
necessary O
for O
virus O
propagation O
. O

The O
TDx O
assay O
for O
cyclosporine O
and O
its O
metabolites O
in O
blood O
samples O
compared O
with O
HPLC O
and O
RIA O
methods O
. O

However O
, O
the O
sequence O
of O
mRAR B
- I
beta I
3 I
region I
A I
differs O
from O
that O
of O
mRAR B
- I
beta I
1 I
by O
an O
additional O
27 O
C O
- O
terminal O
amino O
acids O
encoded O
in O
an O
81 O
nucleotide O
- O
long O
putative O
exon O
which O
is O
spliced O
in O
between O
the O
exons O
encoding O
the O
A O
and O
B O
regions O
of O
mRAR B
- I
beta I
1 I
. O

We O
propose O
that O
GCD7 B
and O
GCD2 B
play O
important O
roles O
in O
the O
regulatory O
interaction O
between O
eIF B
- I
2 I
and O
eIF B
- I
2B I
and O
that O
the O
suppressor O
mutations O
we O
isolated O
in O
these O
genes O
decrease O
the O
susceptibility O
of O
eIF B
- I
2B I
to O
the O
inhibitory O
effects O
of O
phosphorylated O
eIF B
- I
2 I
without O
impairing O
the O
essential O
catalytic O
function O
of O
eIF B
- I
2B I
in O
translation O
initiation O
. O

The O
3 O
' O
region O
of O
the O
transcript O
contained O
a O
fully O
conserved O
, O
correctly O
spliced O
TCR B
alpha I
C I
region I
which O
was O
polyadenylated O
at O
the O
3 O
' O
end O
. O

These O
data O
demonstrate O
that O
CCTalpha B
can O
be O
regulated O
by O
lipids O
by O
two O
independent O
domains O
: O
( O
i O
) O
the O
three O
amphipathic O
alpha O
- O
helical O
repeats O
that O
interact O
with O
both O
neutral O
and O
anionic O
lipid O
mixtures O
and O
( O
ii O
) O
the O
last O
57 O
residues O
that O
interact O
with O
anionic O
lipids O
. O

Sorting O
information O
to O
the O
chloroplastic O
inner O
envelope O
is O
contained O
in O
an O
NH2 O
- O
proximal O
part O
of O
mature B
IEP110 I
( I
110N I
) I
. O

Most O
of O
these O
elements O
are O
adjacent O
to O
type O
X O
telomeric O
repeats O
, O
and O
regions O
flanking O
four O
of O
five O
characterized O
S O
. O
paradoxus O
insertions O
carry O
autonomously O
replicating O
sequences O
. O

From O
1981 O
to O
1989 O
, O
a O
total O
of O
26 O
women O
with O
locally O
or O
regionally O
advanced O
carcinoma O
of O
the O
uterine O
cervix O
were O
treated O
with O
radiotherapy O
( O
RT O
) O
and O
pelvic O
regional O
hyperthermia O
( O
HT O
) O
, O
in O
the O
Divisions O
of O
Radiation O
Oncology O
at O
the O
University O
of O
Utah O
Medical O
Center O
( O
UU O
) O
and O
the O
Kenneth O
Norris O
Jr O
Cancer O
Hospital O
of O
the O
University O
of O
Southern O
California O
( O
USC O
) O
. O

The O
ARE O
is O
loosely O
defined O
as O
the O
five O
- O
nucleotide O
sequence O
AUUUA O
embedded O
in O
a O
uracil O
- O
rich O
region O
. O

Lack O
of O
changes O
in O
milk O
production O
and O
milk O
composition O
suggest O
that O
acute O
increases O
in O
somatotropin B
with O
concomitant O
increases O
in O
insulin B
are O
not O
sufficient O
to O
stimulate O
synthesis O
of O
milk O
and O
milk O
components O
by O
cows O
during O
established O
lactation O
. O

The O
material O
then O
was O
cut O
with O
a O
diamond O
saw O
into O
sheets O
of O
8 O
x O
10 O
x O
3 O
mm O
, O
and O
the O
upper O
surface O
was O
polished O
by O
colloidal O
SiO2 O
and O
/ O
or O
covered O
with O
a O
carbon O
- O
titanium O
( O
C O
: O
Ti O
) O
layer O
( O
3 O
. O
3 O
microm O
) O
using O
the O
plasma O
- O
enhanced O
physical O
vapor O
deposition O
method O
. O

Iomazenil O
- O
SPECT O
revealed O
a O
highly O
significant O
increase O
in O
the O
benzodiazepine B
receptor I
uptake O
in O
all O
studied O
cortical O
regions O
except O
temporal O
cortices O
. O

Both O
in O
the O
Federal O
Republic O
of O
Germany O
and O
in O
some O
neighbouring O
countries O
the O
epizootic O
situation O
of O
Aujeszky O
' O
s O
disease O
has O
been O
unsatisfactory O
for O
a O
long O
time O
, O
especially O
in O
areas O
with O
a O
high O
pig O
density O
. O

Since O
RIP140 B
generally O
down O
- O
regulates O
receptor O
activity O
in O
mammalian O
cells O
and O
specifically O
down O
- O
regulates O
coactivation O
mediated O
by O
SRC B
- I
1 I
, O
we O
propose O
a O
model O
in O
which O
RIP140 B
indirectly O
regulates O
nuclear O
receptor O
AF O
- O
2 O
activity O
by O
competition O
for O
coactivators O
such O
as O
SRC B
- I
1 I
. O

DIC O
and O
low O
grade O
fibrinolysis O
may O
account O
for O
the O
coagulation O
abnormalities O
of O
the O
syndrome O
. O

As O
in O
the O
case O
of O
the O
chicken O
homologue O
, O
there O
are O
two O
hPR B
forms O
, O
A O
and O
B O
, O
which O
originate O
from O
translational O
initiation O
at O
AUG2 O
( O
codon O
165 O
) O
and O
AUG1 O
, O
respectively O
. O

Fluctuations O
of O
the O
measured O
ECF O
energy O
- O
related O
substances O
corresponded O
to O
various O
clinical O
events O
presumably O
involving O
hypoxia O
/ O
ischemia O
. O

Using O
lasers O
in O
diabetic O
wound O
healing O
. O

Furthermore O
, O
the O
zincon O
- O
loaded O
resin O
was O
applied O
to O
the O
selective O
concentration O
of O
trace O
amounts O
of O
chalcophile O
elements O
in O
natural O
water O
samples O
prior O
to O
neutron O
activation O
analysis O
. O

In O
cells O
stably O
transfected O
with O
G B
alpha I
i I
- I
2 I
or O
G B
alpha I
i I
- I
3 I
gene I
5 O
' O
- O
flanking O
sequences O
fused O
to O
firefly B
luciferase I
cDNA I
reporter I
, O
temporal O
10 O
- O
15 O
- O
fold O
transcriptional O
activation O
of O
both O
genes O
occurred O
before O
cellular O
polarization O
. O

CONCLUSIONS O
: O
These O
results O
suggest O
that O
histamine B
- I
2 I
receptor I
activation O
mechanisms O
may O
not O
be O
involved O
in O
postoperative O
IL B
- I
6 I
synthesis O
. O

In O
resume O
response O
to O
treatment O
with O
usual O
regimens O
, O
doxycycline O
plus O
streptomycin O
of O
doxycycline O
rifampicin O
is O
good O
, O
being O
however O
time O
elapsed O
until O
pain O
ceases O
of O
mean O
length O
in O
hospital O
stay O
shorter O
in O
the O
group O
receiving O
doxycycline O
plus O
streptomycin O
. O

Fibrosis O
of O
the O
adjacent O
myocardium O
was O
seen O
in O
five O
cases O
. O

Purified O
preparations O
of O
all O
four O
AGT B
mutants I
showed O
an O
approximately O
similar O
degree O
( O
74 O
- O
120 O
- O
fold O
) O
of O
reduction O
in O
the O
rate O
of O
reaction O
with O
O6 O
- O
benzylguanine O
. O

Significant O
reductions O
were O
noted O
in O
vertical O
GRFs O
per O
newton O
of O
body O
weight O
exerted O
at O
10 O
% O
( O
P O
= O
0 O
. O
0009 O
) O
and O
20 O
% O
( O
P O
= O
0 O
. O
0383 O
) O
of O
stance O
phase O
and O
in O
anteroposterior O
GRFs O
exerted O
at O
10 O
% O
( O
P O
= O
0 O
. O
0009 O
) O
and O
50 O
% O
( O
P O
= O
0 O
. O
0033 O
) O
of O
stance O
phase O
when O
ambulation O
was O
compared O
with O
and O
without O
the O
orthotic O
device O
. O

Identification O
of O
a O
68 B
- I
kilodalton I
nuclear I
ATP I
- I
binding I
phosphoprotein I
encoded O
by O
bovine O
papillomavirus O
type O
1 O
. O

It O
is O
suggested O
that O
biliary O
secretion O
of O
both O
TBZ O
and O
FBZ O
and O
their O
metabolites O
may O
contribute O
to O
this O
recycling O
. O

During O
chordotonal O
organ O
development O
, O
the O
3 O
' O
enhancer O
directs O
expression O
in O
proneural O
clusters O
; O
whereas O
successive O
modular O
enhancers O
located O
in O
the O
5 O
' O
region O
drive O
tissue O
- O
specific O
expression O
in O
chordotonal O
organ O
precursors O
in O
the O
embryo O
and O
larval O
leg O
, O
wing O
and O
antennal O
imaginal O
discs O
. O

The O
use O
of O
self O
- O
measured O
blood O
pressure O
determinations O
in O
assessing O
dynamics O
of O
drug O
compliance O
in O
a O
study O
with O
amlodipine O
once O
a O
day O
, O
morning O
versus O
evening O
. O

Nucleocapsid O
structure O
and O
thermostability O
of O
the O
virion O
, O
nucleocapsid O
and O
polymerase O
complex O
. O

A O
vascular O
component O
to O
impotence O
was O
shown O
to O
be O
common O
in O
those O
with O
neurological O
impairment O
, O
and O
may O
alter O
management O
. O

RESULTS O
- O
- O
The O
maximal O
early O
asthmatic O
response O
after O
allergen O
with O
placebo O
treatment O
was O
18 O
. O
4 O
% O
( O
SE O
4 O
. O
4 O
% O
) O
and O
with O
WEB O
2086 O
18 O
. O
9 O
% O
( O
4 O
. O
4 O
% O
) O
. O

Column O
chromatographic O
determination O
of O
polymyxin O
B O
sulfate O
. O

Usefulness O
of O
computerized O
tomography O
in O
hemodialysis O
patients O
. O

The O
aim O
of O
our O
study O
was O
to O
determine O
whether O
exercise O
training O
can O
augment O
left O
ventricular O
diastolic O
filling O
at O
rest O
and O
during O
exercise O
in O
patients O
with O
ischemic O
cardiomyopathy O
and O
whether O
any O
correlation O
exists O
between O
changes O
in O
diastolic O
filling O
and O
changes O
in O
exercise O
tolerance O
. O

On O
the O
pharmacology O
of O
9 O
, O
10 O
- O
dihydro O
- O
10 O
- O
( O
1 O
- O
methyl O
- O
4 O
- O
piperidylidene O
) O
- O
9 O
- O
anthrol O
( O
WA O
335 O
) O
, O
a O
histamine O
and O
serotonin O
antagonist O

METHODOLOGY O
: O
A O
case O
control O
study O
of O
survivors O
with O
gestational O
age O
( O
GA O
) O
< O
28 O
weeks O
or O
birthweight O
( O
BW O
) O
< O
1000 O
g O
using O
data O
collected O
prospectively O
in O
our O
Neonatal O
Intensive O
Care O
Unit O
database O
. O

There O
are O
two O
regulatory O
regions O
between O
the O
US3 B
and O
the O
US6 B
transcription I
units I
. O

Database O
searches O
revealed O
a O
50 O
- O
63 O
% O
similarity O
and O
34 O
- O
42 O
% O
identity O
with O
several O
families O
of O
serine B
proteases I
, O
in O
particular O
the O
trypsin B
- I
like I
proteases I
, O
members O
of O
the O
glandular B
kallikrein I
family I
( O
including O
prostate B
- I
specific I
antigen I
, O
nerve B
growth I
factor I
gamma I
, O
and O
epidermal B
growth I
factor I
- I
binding I
protein I
) O
and O
the O
activators O
for O
the O
kringle B
family I
proteins I
( O
including O
the O
human B
tissue I
plasminogen I
activator I
and O
human B
hepatocyte I
growth I
factor I
activator I
) O
. O

Manifestation O
of O
superfluidity O
in O
an O
evolving O
bose O
- O
einstein O
condensed O
Gas O
We O
study O
the O
generation O
of O
excitations O
due O
to O
an O
" O
impurity O
" O
( O
static O
perturbation O
) O
placed O
into O
an O
oscillating O
Bose O
- O
Einstein O
condensed O
gas O
in O
the O
time O
- O
dependent O
trapping O
field O
. O

4 O
. O
7 O
+ O
/ O
- O
0 O
. O
6 O
micromol O
. O

kg O
- O
1 O
. O

min O
- O
1 O
) O
but O
was O
not O
changed O
by O
training O
. O

We O
established O
that O
, O
in O
unstimulated O
lymphocytes O
, O
the O
Src B
homology I
2 I
( O
SH2 B
) O
and O
SH3 B
domain O
- O
containing O
protein O
Grb2 B
and O
the O
p85 B
subunit I
of O
phosphatidylinositol B
3 I
- I
kinase I
, O
associate O
constitutively O
with O
Cbl B
via O
their O
SH3 B
domains I
. O

Zta B
stimulated O
the O
HAT B
activity O
of O
CBP B
that O
had O
been O
partially O
purified O
or O
immunoprecipitated O
from O
mammalian O
cells O
as O
well O
as O
from O
affinity O
- O
purified O
, O
baculovirus O
expressed O
CBP B
. O

Increased O
gliadin B
antibody I
levels O
were O
found O
more O
frequently O
in O
patients O
with O
subtotal O
villous O
atrophy O
( O
9 O
out O
of O
17 O
patients O
, O
or O
53 O
% O
; O
p O
less O
than O
0 O
. O
05 O
) O
than O
in O
patients O
with O
partial O
villous O
atrophy O
( O
2 O
out O
of O
13 O
patients O
, O
or O
15 O
% O
) O
or O
normal O
villous O
appearance O
( O
2 O
out O
of O
10 O
patients O
, O
or O
20 O
% O
) O
. O

However O
, O
the O
optimum O
compressive O
strength O
reached O
values O
up O
to O
40 O
% O
higher O
than O
CC O
- O
free O
samples O
. O

Expression O
of O
the O
Hox B
genes I
egl B
- I
5 I
and O
mab B
- I
5 I
is O
reduced O
in O
lin B
- I
49 I
and O
lin B
- I
59 I
mutants I
, O
suggesting O
lin B
- I
49 I
and O
lin B
- I
59 I
regulate O
HOM B
- I
C I
gene I
expression O
in O
C O
. O
elegans O
as O
the O
trx B
- I
G I
genes I
do O
in O
Drosophila O
. O

lin B
- I
49 I
and O
lin B
- I
59 I
transgenes I
are O
expressed O
widely O
throughout O
C O
. O
elegans O
animals O
. O

Interleukin B
- I
1 I
increased O
the O
noradrenaline O
release O
. O

Magnetic O
field O
influence O
on O
central O
nervous O
system O
function O
. O

3 O
patients O
with O
acute O
leukaemia O
, O
HLA B
antibodies I
and O
thrombocytopenia O
refractory O
to O
random O
donor O
platelet O
transfusions O
were O
treated O
with O
high O
- O
dose O
i O
. O
v O
. O
immunoglobulin B
. O

Two O
genes O
( O
ptsI B
and O
ptsA B
) O
that O
encode O
homologues O
of O
the O
energy B
coupling I
Enzyme I
I I
of O
the O
phosphoenolpyruvate B
- I
dependent I
sugar I
- I
transporting I
phosphotransferase I
system O
( O
PTS O
) O
have O
previously O
been O
identified O
on O
the O
Escherichia O
coli O
chromosome O
. O

Eight O
of O
these O
suppressors O
for O
into O
two O
complementation O
groups O
, O
designated O
KCS1 B
and O
KCS2 B
. O

These O
results O
point O
towards O
post O
- O
translational O
steric O
and O
/ O
or O
allosteric O
control O
of O
EKLF B
function O
that O
may O
be O
important O
not O
just O
for O
its O
DNA O
binding O
ability O
, O
but O
also O
for O
its O
potential O
to O
interact O
with O
other O
proteins O
that O
fully O
establish O
the O
correct O
stereospecific O
array O
leading O
to O
efficient O
switching O
of O
beta B
- I
globin I
transcription O
during O
development O
. O

Oxalates O
and O
the O
digestive O
tract O

However O
, O
the O
addition O
of O
intravenous O
injection O
of O
FPFD O
101 O
resulted O
in O
a O
marked O
improvement O
in O
their O
symptoms O
. O

Western O
blot O
experiments O
detected O
SMBP B
as O
a O
70 O
kDa O
protein O
that O
may O
be O
further O
cleaved O
into O
an O
active O
34 O
kDa O
N O
- O
terminal O
polypeptide O
. O

The O
aetiology O
and O
management O
of O
diabetic O
impotence O
is O
well O
- O
documented O
; O
the O
effects O
of O
diabetes O
on O
female O
sexuality O
are O
not O
so O
clear O
. O

Treatment O
of O
diabetes O
and O
retinal O
complications O

The O
activated O
glucocorticoid B
receptor I
forms O
a O
complex O
with O
Stat5 B
and O
enhances O
Stat5 B
- O
mediated O
transcriptional O
induction O
. O

Taken O
together O
, O
these O
findings O
demonstrate O
that O
ZNF76 B
and O
ZNF143 B
are O
two O
members O
of O
a O
same O
family O
of O
transactivator O
proteins O
. O

For O
example O
, O
the O
presence O
of O
two O
related O
genes O
, O
b O
and O
b B
' I
, O
in O
the O
cyanobacterium O
suggests O
that O
its O
ATP B
synthase I
is O
a O
complex O
of O
nine O
polypeptides O
, O
and O
that O
it O
may O
have O
single O
copies O
of O
related O
b B
and O
b B
' I
proteins O
rather O
than O
two O
copies O
of O
identical O
b O
subunits O
as O
found O
in O
the O
E O
. O
coli O
enzyme O
. O
4 I
+ O
off O

Antiserum O
raised O
against O
rat B
liver I
S6 I
kinase I
specifically O
immunoprecipitates O
the O
purified O
32P O
- O
labeled O
H4 O
hepatoma O
insulin B
- O
stimulated O
S6 B
kinase I
. O

CRKL B
, O
an O
SH2 B
- O
SH3 B
- O
SH3 B
adapter O
protein O
, O
is O
one O
of O
the O
major O
tyrosine O
phosphoproteins O
detected O
in O
primary O
leukemic O
neutrophils O
from O
patients O
with O
CML O
. O

KU O
- O
1257 O
also O
significantly O
accelerated O
the O
healing O
of O
acetic O
acid O
- O
induced O
duodenal O
ulcers O
as O
well O
as O
famotidine O
and O
roxatidine O
acetate O
. O

To O
further O
identify O
the O
residues O
responsible O
for O
the O
activity O
, O
we O
isolated O
the O
mutant O
viruses O
that O
were O
not O
neutralized O
with O
the O
soluble O
form O
of O
MHV B
receptor I
proteins I
, O
since O
such O
mutants O
were O
expected O
to O
have O
mutations O
in O
amino O
acids O
responsible O
for O
receptor O
- O
binding O
activity O
. O

It O
is O
suggested O
that O
death O
of O
S O
. O
mansoni O
cercariae O
during O
penetration O
of O
mammalian O
host O
skin O
is O
probably O
due O
to O
exhaustion O
of O
their O
energy O
reserves O
. O

Especially O
if O
the O
number O
of O
dimensions O
of O
the O
samples O
is O
large O
, O
it O
can O
be O
said O
that O
BPN O
is O
better O
than O
k O
- O
NN O
in O
classification O
ability O
. O

In O
contrast O
, O
inactivation O
of O
the O
Sir4p B
- I
interacting I
protein I
Rap1p B
reduces O
partitioning O
by O
a O
LexA B
- O
Sir4p B
fusion O
. O

Nine O
individuals O
who O
were O
stable O
after O
1 O
month O
of O
therapy O
at O
low O
dosage O
were O
randomized O
to O
a O
further O
month O
of O
therapy O
at O
low O
or O
high O
dosage O
, O
during O
which O
one O
of O
four O
at O
high O
dosage O
had O
a O
partial O
response O
, O
and O
none O
of O
five O
at O
low O
dosage O
manifested O
response O
. O

BACKGROUND O
: O
Murine B
Nramp I
is O
a O
candidate O
for O
the O
macrophage O
resistance O
gene O
Ity B
/ O
Lsh B
/ O
Bcg B
. O

In O
cases O
in O
which O
it O
was O
identified O
, O
insulitis O
affected O
23 O
% O
of O
islets O
containing O
insulin B
, O
but O
affected O
only O
1 O
% O
of O
islets O
which O
were O
insulin B
deficient O
, O
thus O
supporting O
the O
concept O
that O
insulitis O
represents O
an O
immunologically O
mediated O
destruction O
of O
insulin B
secreting O
B O
cells O
. O

( O
4 O
) O
Expression O
in O
different O
sites O
in O
the O
central O
nervous O
system O
is O
driven O
by O
separable O
elements O
widely O
dispersed O
throughout O
8 O
kb O
3 O
' O
of O
the O
gene O
. O

To O
investigate O
possible O
roles O
for O
HCP B
during O
late O
erythroid O
differentiation O
, O
effects O
of O
manipulating O
HCP B
expression O
or O
recruitment O
on O
EPO B
- O
induced O
hemoglobinization O
in O
erythroleukemic O
SKT6 O
cells O
have O
been O
investigated O
. O

The O
other O
end O
of O
this O
cluster O
contained O
the O
human B
type I
I I
cytokeratin I
K20 I
and O
K12 B
gene I
loci I
. O

An O
11 O
. O
5 O
- O
kb O
intron O
is O
found O
at O
the O
end O
of O
transmembrane O
6 O
, O
and O
the O
rest O
of O
the O
ORF O
is O
in O
exon O
3 O
. O

Are O
there O
any O
lessons O
to O
be O
learnt O
? O

Eighteen O
patients O
with O
advanced O
epidermoid O
carcinoma O
of O
the O
head O
and O
neck O
were O
entered O
into O
a O
phase O
II O
trial O
of O
N O
- O
Methylformamide O
( O
NMF O
) O
, O
800 O
mg O
/ O
M2 O
IV O
daily O
for O
5 O
days O
every O
4 O
weeks O
. O

We O
provided O
evidence O
that O
CaM B
kinase I
II I
played O
a O
role O
in O
regulating O
neurite O
outgrowth O
and O
growth O
cone O
motility O
in O
these O
cells O
, O
and O
that O
the O
autophosphorylation O
is O
essential O
for O
the O
kinase O
to O
sufficiently O
exert O
its O
cellular O
function O
in O
vivo O
[ O
Y O
. O

However O
, O
in O
transient O
transfection O
assays O
, O
a O
truncation O
of O
as O
little O
as O
15 O
Nurr1 B
COOH I
- O
terminal I
amino O
acids O
diminished O
transcriptional O
activation O
of O
B1A B
- O
thymidine B
kinase I
- O
chloramphenicol B
acetyltransferase I
reporter O
. O

All O
sows O
nursed O
nine O
pigs O
. O

In O
the O
second O
group O
the O
untreated O
fellow O
eye O
was O
microscopically O
intact O
, O
but O
was O
shown O
( O
as O
the O
treated O
eye O
) O
to O
alter O
the O
concentration O
of O
uronic O
acid O
in O
different O
parts O
of O
the O
sclera O
. O

Effects O
were O
studied O
of O
extremely O
high O
- O
frequency O
electromagnetic O
radiation O
( O
EHF O
EMR O
) O
on O
indices O
for O
the O
immune O
and O
endocrine O
systems O
in O
a O
series O
of O
48 O
patients O
presenting O
with O
hyperplastic O
processes O
in O
endometrium O
. O

Characterization O
of O
human B
activating I
transcription I
factor I
4 I
, O
a O
transcriptional O
activator O
that O
interacts O
with O
multiple O
domains O
of O
cAMP B
- I
responsive I
element I
- I
binding I
protein I
( I
CREB I
) I
- I
binding I
protein I
. O

Effect O
of O
a O
constant O
magnetic O
field O
on O
different O
models O
of O
carcinogenesis O

In O
contrast O
to O
the O
reciprocal O
effects O
of O
Gab1 B
and O
Gab2 B
in O
mediating O
Elk B
- I
1 I
induction O
, O
these O
two O
molecules O
have O
a O
similar O
function O
in O
extracellular B
signal I
- I
regulated I
kinase I
activation O
induced O
by O
either O
oncogenic B
Ras I
or O
growth O
factor O
stimulation O
. O

This O
result O
is O
consistent O
with O
the O
previous O
observation O
that O
expression O
of O
the O
hsp70 B
genes I
in O
T O
. O
brucei O
is O
mainly O
controlled O
at O
the O
posttranscriptional O
level O
. O

A O
definition O
of O
bias O
founded O
on O
the O
concept O
of O
the O
study O
base O
. O

Chromaffin O
cells O
transfected O
with O
a O
plasmid O
with O
the O
entire O
coding O
sequence O
of O
c B
- I
Rabphilin3a I
inserted O
in O
the O
antisense O
orientation O
inhibited O
secretion O
of O
co O
- O
expressed O
GH B
by O
approximately O
30 O
% O
. O

Using O
a O
set O
of O
sera O
for O
which O
full O
chlamydial O
micro O
- O
immunofluorescence O
results O
suggested O
a O
clear O
diagnosis O
, O
we O
have O
evaluated O
the O
Chlamydia O
Spot O
- O
IF O
test O
( O
bioMerieux O
) O
, O
which O
allows O
a O
comparison O
of O
titres O
to O
Chlamydia O
trachomatis O
and O
C B
. I
psittaci I
antigens I
. O

An O
environmental O
investigation O
found O
evidence O
of O
suboptimal O
food O
storage O
and O
cooking O
temperatures O
at O
Restaurant O
A O
; O
cross O
contamination O
of O
foods O
may O
have O
contributed O
to O
the O
low O
attributable O
risk O
identified O
for O
chile O
rellenos O
. O

Evidence O
from O
four O
areas O
- O
- O
sensory O
deprivation O
, O
enriched O
environments O
, O
nervous O
system O
plasticity O
, O
and O
sensitive O
periods O
of O
neurodevelopment O
- O
- O
suggests O
that O
sensory O
stimulation O
programs O
are O
potentially O
beneficial O
for O
STR O
patients O
. O

In O
each O
study O
group O
, O
elevated O
levels O
of O
serum O
I B
were O
observed O
. O

With O
repetitive O
intermittent O
exercise O
, O
gradual O
increases O
in O
blood O
lactate O
concentration O
[ O
( O
LA O
] O
b O
) O
occurred O
, O
whereas O
its O
rate O
of O
accumulation O
( O
delta O
[ O
LA O
] O
b O
) O
decreased O
. O

A O
physical O
and O
genetic O
map O
covering O
the O
entire O
RP12 B
candidate I
gene I
region I
was O
constructed O
. O

The O
single O
copy O
flotillin B
- I
1 I
gene I
is O
located O
at O
6p21 O
. O
3 O
in O
the O
MHC B
class I
I I
region I
and O
consists O
of O
13 O
exons O
over O
15 O
kb O
. O

An O
atypical O
form O
of O
juvenile O
myasthenia O
gravis O
associated O
with O
severe O
emaciation O
, O
muscle O
atrophy O
, O
ophthalmoplegia O
, O
bulbar O
signs O
and O
joint O
contracture O

The O
in O
vivo O
response O
of O
the O
PacC B
- I
like I
decamers I
to O
external O
pH O
was O
dependent O
on O
the O
status O
of O
the O
pH O
- O
regulated O
activator O
YlRim101p B
, O
which O
is O
homologous O
to O
the O
A B
. I
nidulans I
PacC I
regulator I
. O

Electron O
microscopic O
visualization O
of O
the O
general O
population O
of O
active O
genes O
in O
flies O
overexpressing O
hnRNP B
proteins I
also O
indicated O
that O
the O
great O
majority O
of O
genes O
seemed O
normal O
in O
terms O
of O
cotranscriptional O
RNA O
processing O
events O
, O
although O
there O
were O
a O
few O
abnormalities O
consistent O
with O
rare O
exon O
- O
skipping O
events O
. O

Having O
demonstrated O
proper O
localization O
of O
GFP B
- O
calmodulin B
in O
budding O
yeast O
, O
we O
examined O
the O
localization O
of O
a O
fusion O
between O
GFP B
and O
calmodulin B
( O
GFP B
- O
Camlp B
) O
in O
fission O
yeast O
, O
where O
calmodulin B
had O
not O
been O
localized O
by O
any O
method O
. O

Sera O
from O
33 O
newborn O
infants O
with O
gestational O
ages O
ranging O
from O
27 O
to O
41 O
weeks O
were O
tested O
by O
radioimmunoassay O
for O
IgG B
antibodies I
to O
surface O
antigens O
of O
group O
B O
streptococci O
( O
GBS O
) O
types O
Ia O
, O
Ib O
, O
II O
and O
III O
. O

In O
frozen O
sections O
the O
spots O
appear O
as O
blue O
labelled O
neurones O
with O
a O
light O
microscope O
, O
or O
as O
a O
brilliant O
red O
neurones O
surrounded O
by O
reddish O
tissue O
with O
a O
fluorescence O
microscope O
. O

The O
percentage O
distribution O
of O
types O
of O
fixation O
disparity O
curves O
was O
found O
to O
be O
similar O
to O
some O
previous O
studies O
, O
with O
a O
higher O
prevalence O
of O
Type O
I O
curve O
( O
64 O
. O
3 O
% O
) O
, O
followed O
by O
Type O
II O
( O
28 O
. O
6 O
% O
) O
and O
Type O
III O
( O
7 O
. O
1 O
% O
) O
curves O
. O

The O
BUR O
was O
set O
at O
20 O
/ O
min O
and O
the O
IT O
at O
. O
5 O
seconds O
. O

These O
data O
are O
discussed O
in O
the O
context O
of O
the O
pathogenesis O
and O
differential O
diagnosis O
of O
EEC O
. O

Seminal B
prolactin I
levels O
were O
significantly O
elevated O
in O
all O
infertile O
males O
. O

Northern O
blot O
analysis O
identified O
retina O
- O
specific O
transcripts O
of O
3 O
. O
0 O
kb O
for O
rod B
G I
alpha I
t I
and O
2 O
. O
6 O
kb O
for O
cone B
G I
alpha I
t I
. O

In O
the O
univariate O
models O
of O
either O
fixed O
or O
time O
dependent O
covariates O
, O
many O
variables O
were O
significantly O
associated O
with O
risk O
of O
progression O
to O
AIDS O
( O
T4 O
cell O
count O
, O
T4 O
/ O
T8 O
ratio O
, O
blastogenic O
responses O
to O
phytohemagglutinin B
, O
concanavalin B
A I
, O
and O
pokeweed B
mitogen I
, O
serum B
IgA I
, O
appearance O
of O
p24 B
antigen I
, O
and O
the O
development O
of O
oral O
hairy O
leukoplakia O
, O
thrush O
, O
or O
herpes O
zoster O
) O
. O

Adenovirus O
- O
mediated O
gene O
transfer O
of O
MMAC1 B
/ O
PTEN B
to O
glioblastoma O
cells O
inhibits O
S O
phase O
entry O
by O
the O
recruitment O
of O
p27Kip1 B
into O
cyclin B
E I
/ O
CDK2 B
complexes O
. O

Cost O
analysis O
and O
the O
HSA O
: O
a O
framework O
. O

These O
cells O
also O
showed O
an O
increase O
in O
cyclin B
A I
and O
cyclin B
A I
- I
and I
E I
- I
associated I
kinase I
activities O
characteristic O
of O
S O
phase O
induction O
. O

Characterization O
of O
a O
tonB B
- O
phoA B
gene O
fusion O
suggests O
that O
the O
amino O
- O
terminal O
41 O
amino O
acids O
of O
TonB B
are O
sufficient O
to O
promote O
export O
of O
the O
fusion O
protein O
and O
presumably O
TonB B
as O
well O
. O

Calcium O
bilirubinate O
crystals O
were O
seen O
in O
gallbladder O
bile O
or O
wall O
scrapings O
of O
7 O
of O
8 O
TPN O
animals O
but O
in O
none O
of O
the O
controls O
( O
P O
less O
than O
0 O
. O
001 O
) O
. O

This O
domain O
encompasses O
the O
region O
of O
ARP B
- I
1 I
/ O
COUP B
- I
TFII I
corresponding O
to O
helices O
3 O
to O
12 O
in O
the O
recently O
published O
crystal O
structure O
of O
other O
members O
of O
the O
nuclear B
receptor I
superfamily I
. O

Different O
clinical O
variants O
of O
the O
empty O
sella O
turcica O
syndrome O
are O
demonstrated O
, O
including O
variants O
resultant O
from O
substitution O
therapy O
of O
thyroid O
hypofunction O
, O
from O
dopamine O
agonist O
therapy O
for O
hyperprolactinemic O
hypogonadism O
, O
from O
radiotherapy O
of O
hypophyseal O
adenoma O
. O

Both O
genes O
ARO3 B
and O
ARO4 B
are O
strongly O
regulated O
under O
the O
general O
control O
regulatory O
system O
. O

Polar O
effects O
of O
transposon O
insertions O
demonstrated O
that O
all O
of O
these O
mRNAs O
arose O
from O
a O
single O
promoter O
region O
, O
where O
transcription O
initiated O
80 O
bp O
5 O
' O
to O
nifH B
. O

J O
. O
, O
& O
Olefsky O
, O
J O
. O

Importantly O
, O
a O
single O
base O
change O
in O
the O
fifth O
position O
of O
the O
c B
- I
fos I
sequence I
( O
GGTCTnnnAGACC O
to O
GGTCA O
/ O
GnnnAGACC O
) O
produced O
an O
element O
that O
bound O
the O
estrogen B
receptor I
and O
conferred O
estrogen O
- O
dependent O
transcriptional O
activation O
of O
a O
reporter O
gene O
. O

Measuring O
the O
dim O
light O
melatonin O
onset O
( O
DLMO O
) O
is O
a O
useful O
and O
practical O
way O
to O
assess O
circadian O
phase O
position O
in O
humans O
. O

Co O
- O
expression O
of O
a O
gag B
- O
TRalpha B
fusion O
protein O
in O
AEV O
- O
transformed O
cells O
and O
addition O
of O
ligand O
derepresses O
CAII B
transcription O
. O

The O
IB2 B
gene I
( O
HGMW O
- O
approved O
symbol O
MAPK8IP2 B
) O
maps O
to O
human O
chromosome O
22q13 O
and O
contains O
10 O
coding O
exons O
. O

Transcription O
start O
sites O
of O
sal B
and O
salR B
genes I
were O
determined O
to O
lie O
30 O
- O
and O
24 O
- O
bp O
upstream O
of O
the O
respective O
initiation O
codons O
and O
separated O
from O
each O
other O
by O
78 O
nucleotides O
. O

1 O
. O
23 O
) O
as O
a O
PPARalpha B
- I
interacting I
protein I
. O

Here O
we O
demonstrate O
that O
the O
T B
cell I
antigen I
receptor I
zeta I
- I
chain I
- I
associated I
ZAP I
- I
70 I
kinase I
and O
T B
cell I
antigen I
receptor I
zeta I
- I
chain I
immunoreceptor O
tyrosine O
- O
based O
activation O
motifs O
are O
essential O
for O
the O
membrane O
recruitment O
of O
SOS B
and O
Vav B
. O

We O
carried O
out O
two O
experiments O
to O
investigate O
this O
problem O
. O

When O
linked O
to O
the O
herpes B
simplex I
thymidine I
kinase I
minimal I
promoter I
, O
this O
fragment O
acts O
as O
an O
enhancing O
element O
in O
Rcho O
but O
not O
GC O
cells O
. O

A O
mammalian O
protein O
called O
RFX B
or O
NF B
- I
X I
binds O
to O
the O
X O
box O
( O
or O
X1 O
box O
) O
in O
the O
promoters O
of O
a O
number O
of O
major B
histocompatibility I
( I
MHC I
) I
class I
II I
genes I
. O

Site O
- O
directed O
mutagenesis O
was O
conducted O
to O
investigate O
the O
functional O
significance O
of O
this O
element O
and O
the O
3 O
' O
minus O
- O
strand O
terminal O
sequence O
" O
3 O
' O
- O
OH O
- O
CCCUAU O
, O
" O
which O
contains O
the O
minus O
- O
strand O
3 O
' O
- O
end O
sequence O
" O
3 O
' O
- O
OH O
- O
CC O
( O
1 O
- O
2 O
) O
( O
A O
/ O
U O
) O
( O
A O
/ O
U O
) O
( O
A O
/ O
U O
) O
" O
found O
in O
all O
carmovirus O
RNAs O
. O

The O
incidence O
of O
the O
variability O
of O
the O
free O
PSA B
/ O
total O
PSA B
ratio O
on O
the O
early O
diagnosis O
of O
prostate O
cancer O

VBP B
and O
a1 B
/ I
EBP I
could O
mediate O
the O
high O
rates O
of O
ALV O
and O
RSV B
LTR I
- O
enhanced O
transcription O
in O
bursal O
lymphoma O
cells O
and O
many O
other O
cell O
types O
. O

In O
the O
second O
, O
37 O
students O
were O
given O
trimethoprim O
/ O
sulphamethoxazole O
( O
160 O
mg O
TMP O
- O
SMX O
800 O
mg O
) O
, O
38 O
TMP O
( O
200 O
mg O
) O
, O
and O
in O
35 O
a O
placebo O
was O
given O
b O
. O
i O
. O
d O
. O
for O
five O
days O
. O

Developmental O
slot O
blots O
demonstrate O
that O
mRNAs O
corresponding O
to O
the O
three O
LHM B
cDNAs I
are O
transcribed O
from O
prophase O
of O
meiosis O
I O
to O
the O
uninucleate O
microspore O
stage O
, O
while O
Northern O
analysis O
reveals O
these O
tapetally O
expressed O
cDNAs O
to O
correspond O
with O
transcripts O
of O
some O
500 O
bp O
. O

One O
hundred O
men O
who O
received O
alpha O
- O
tocopherol O
were O
matched O
on O
age O
, O
study O
center O
, O
and O
length O
of O
time O
between O
blood O
draws O
to O
100 O
men O
who O
received O
a O
placebo O
. O

The O
contribution O
of O
residues O
outside O
the O
Ras B
binding I
domain I
of O
Raf B
( O
RafRBD B
) O
to O
Ras B
- O
Raf B
interaction O
and O
Ras B
- O
dependent O
Raf B
activation O
has O
remained O
unresolved O
. O

Other O
recommendations O
for O
enhancing O
the O
safety O
of O
potent O
antiplatelet O
agents O
in O
a O
variety O
of O
clinical O
situations O
are O
provided O
. O

RESULTS O
- O
- O
261 O
patients O
were O
enrolled O
into O
this O
study O
; O
of O
these O
, O
138 O
( O
53 O
% O
) O
patients O
attended O
for O
repeat O
colposcopy O
and O
cytology O
at O
one O
year O
. O

Changes O
in O
the O
greater O
omentum O
of O
mice O
of O
different O
strains O
after O
intraperitoneal O
immunization O
with O
sheep O
erythrocytes O
. O

In O
this O
study O
, O
we O
sought O
to O
determine O
the O
specific O
effect O
of O
HIV B
protease I
inhibitors O
on O
patient O
weight O
. O

However O
massive O
necrosis O
of O
the O
renal O
tubules O
had O
been O
found O
with O
more O
than O
15mg O
of O
DSM O
administration O
. O

The O
characteristics O
of O
a O
simple O
apparatus O
for O
clinical O
use O
to O
measure O
DEOAEs O
are O
described O
together O
with O
typical O
examples O
of O
emissions O
. O

To O
obtain O
information O
on O
the O
functional O
role O
of O
the O
Rac1 B
/ O
p38 B
/ O
MAPKAPK B
- I
2 I
pathway O
in O
RA O
signaling O
, O
the O
effects O
of O
pharmacological O
inhibition O
of O
p38 B
on O
RA O
- O
induced O
gene O
transcription O
and O
cell O
differentiation O
were O
determined O
. O

The O
cost O
and O
effectiveness O
of O
nurse O
education O
- O
- O
1 O
. O

The O
ATF B
/ O
CRE O
site O
is O
also O
essential O
for O
CD4 B
promoter I
activation O
by O
forskolin O
, O
an O
activator O
of O
adenylate B
cyclase I
. O

YLL031c B
is O
an O
essential O
gene O
. O

Restricted O
expression O
of O
a O
novel O
murine B
atonal I
- I
related I
bHLH I
protein I
in O
undifferentiated O
neural O
precursors O
. O

Since O
the O
early O
1900s O
, O
a O
genetic O
hypothesis O
has O
been O
suggested O
. O

We O
report O
the O
genomic O
organization O
of O
the O
mouse O
orphan I
receptor I
related I
to I
tyrosine I
kinases I
( O
Ryk B
) O
, O
a O
structurally O
unclassified O
member O
of O
the O
growth B
factor I
receptor I
family I
. O

In O
contrast O
, O
T229E B
- I
p70s6k I
migrated O
more O
slowly O
in O
SDS O
- O
polyacrylamide O
gels O
, O
but O
demonstrated O
partial O
kinase O
activity O
( O
approximately O
20 O
% O
compared O
with O
the O
wild O
type O
) O
. O

The O
structure O
and O
expression O
of O
the O
murine O
gene I
encoding I
granulocyte I
- I
macrophage I
colony I
stimulating I
factor I
: O
evidence O
for O
utilisation O
of O
alternative O
promoters O
. O

Thus O
, O
the O
search O
for O
genetic O
and O
acquired O
susceptibility O
to O
nontuberculous O
mycobacteria O
is O
also O
a O
search O
for O
susceptibility O
factors O
for O
MTB O
as O
well O
as O
an O
opportunity O
to O
recognize O
endogenous O
pathways O
that O
can O
be O
exploited O
therapeutically O
. O

Liver O
disease O
and O
HCV O
infection O
after O
transplantation O
of O
organs O
from O
hepatitis B
C I
antibody I
positive O
donors O
. O

After O
dialysis O
online O
, O
lactate O
was O
converted O
by O
means O
of O
lactate B
oxidase I
immobilized O
to O
porous O
glass O
, O
and O
the O
depletion O
of O
oxygen O
was O
registered O
by O
means O
of O
a O
Clark O
electrode O
. O

The O
deduced O
amino O
- O
acid O
sequence O
of O
the O
mature O
enzyme O
showed O
very O
low O
homology O
( O
< O
20 O
. O
4 O
% O
identity O
) O
to O
those O
of O
known O
pectinolytic O
enzymes O
in O
the O
large O
pectate B
lyase I
superfamily I
( O
the O
polysaccharide B
lyase I
family I
1 I
) O
. O

Incubation O
of O
HeLa O
cell O
cytoplasmic O
extracts O
with O
a O
purified O
recombinant O
glutathione B
S I
- I
transferase I
- O
raf B
fusion O
protein O
in O
the O
presence O
of O
ATP O
released O
active O
NF B
- I
kappa I
B I
that O
could O
be O
detected O
by O
electrophoretic O
gel O
mobility O
shift O
assay O
. O

Despite O
this O
loss O
, O
YEpFAS1 B
is O
still O
able O
to O
complement O
a O
fas1 B
mutation I
at O
the O
enoyl B
reductase I
domain I
. O

Cross O
- O
species O
characterization O
of O
the O
promoter O
region O
of O
the O
cystic B
fibrosis I
transmembrane I
conductance I
regulator I
gene I
reveals O
multiple O
levels O
of O
regulation O
. O

Pretreatment O
with O
lipid O
X O
was O
not O
necessary O
to O
improve O
survival O
: O
16 O
of O
17 O
( O
94 O
% O
) O
infected O
and O
visibly O
ill O
animals O
that O
received O
lipid O
X O
and O
ticarcillin O
6 O
h O
after O
thigh O
inoculation O
survived O
versus O
30 O
of O
44 O
( O
68 O
% O
) O
control O
animals O
treated O
with O
ticarcillin O
alone O
( O
P O
less O
than O
0 O
. O
0001 O
) O
. O

The O
predicted O
vav B
oncogene I
protein I
sequence O
exhibits O
several O
motifs O
reminiscent O
of O
transcriptional O
factors O
. O

The O
pharmacokinetics O
were O
best O
described O
by O
a O
two O
- O
compartment O
open O
model O
giving O
distribution O
half O
- O
lives O
of O
0 O
. O
31 O
h O
and O
1 O
. O
53 O
h O
, O
and O
elimination O
half O
- O
lives O
of O
69 O
. O
7 O
h O
and O
60 O
. O
3 O
h O
for O
oxolinic O
acid O
and O
oxytetracycline O
, O
respectively O
. O

The O
predictive O
value O
of O
the O
remotely O
sensed O
map O
based O
on O
NDVI O
data O
appears O
to O
be O
better O
than O
that O
from O
forecast O
indices O
based O
only O
on O
climatic O
data O
. O

It O
has O
been O
confirmed O
in O
animal O
experiments O
that O
the O
number O
of O
microspheres O
in O
a O
myocardial O
sample O
approximately O
follows O
a O
Poisson O
distribution O
, O
under O
adequate O
experimental O
conditions O
. O

Hepatitis B
B I
immune I
globulins I
and O
HIV O
antibodies O
. O

This O
gene O
is O
divided O
into O
six O
exons O
spanning O
about O
3kb O
, O
and O
encodes O
a O
175 O
amino O
acid O
protein O
with O
40 O
- O
52 O
% O
identity O
with O
the O
other O
five O
mouse O
Reg I
( O
regenerating B
gene I
product I
) O
proteins O
. O

The O
p63 B
sequence I
also O
features O
an O
acidic O
domain O
characteristic O
of O
transcriptional O
activation O
factors O
, O
as O
well O
as O
sequence O
blocks O
displaying O
limited O
similarity O
to O
positionally O
equivalent O
regions O
in O
sigma B
factors I
from O
eubacteria O
related O
to O
mitochondria O
. O

In O
contrast O
, O
a O
similar O
fusion O
protein O
( O
hGH B
- O
LDLR B
- O
DAF17 B
, O
abbreviated O
HLD B
) O
containing O
a O
fragment O
of O
the O
serine O
/ O
threonine O
- O
rich O
domain O
of O
the O
LDL B
receptor I
( O
LDLR B
) O
in O
place O
of O
the O
DAF B
- I
derived I
serine I
/ O
threonine I
- I
rich I
sequences I
, O
does O
not O
become O
GPI O
anchored O
. O

Thus O
, O
the O
pathway O
to O
Fc B
epsilonRI I
- O
mediated O
MEK B
/ O
ERK B
and O
ERK B
activation O
can O
apparently O
bypass O
Ras B
and O
Raf B
- I
1 I
. O

To O
assess O
whether O
thallium O
- O
201 O
thallous O
chloride O
( O
Tl O
) O
can O
detect O
childhood O
tumors O
and O
whether O
diagnostic O
effectiveness O
improves O
with O
combined O
blood O
flow O
imaging O
, O
28 O
children O
( O
1 O
. O
0 O
- O
18 O
. O
6 O
years O
) O
were O
studied O
using O
single O
photon O
emission O
computed O
tomography O
( O
SPECT O
) O
: O
Tl O
( O
1 O
. O
3 O
- O
1 O
. O
8 O
mCi O
intravenously O
) O
, O
followed O
in O
13 O
of O
the O
patients O
by O
technetium O
- O
99m O
- O
hexamethylpropyleneamine O
oxime O
( O
99mTc O
- O
HMPAO O
; O
8 O
- O
18 O
mCi O

intravenously O
) O
. O

Ftz B
, O
on O
the O
other O
hand O
, O
influences O
Ftz B
- I
F1 I
activity O
by O
interacting O
with O
its O
AF O
- O
2 O
domain O
in O
a O
manner O
that O
mimics O
a O
nuclear O
receptor O
coactivator O
. O

Successive O
5 O
' O
deletions O
of O
the O
[ O
- O
326 O
; O
+ O
20 O
] I
type I
II I
sPLA2 I
promoter I
indicated O
that O
the O
region O
upstream O
of O
position O
- O
195 O
inhibits O
the O
transcription O
activity O
sixfold O
in O
HepG2 O
cells O
but O
not O
in O
HeLa O
cells O
. O

Forty O
- O
five O
patients O
with O
recent O
- O
onset O
hyperthyroidism O
( O
< O
12 O
weeks O
) O
were O
sex O
and O
menopause O
stratified O
and O
randomly O
allocated O
to O
treatment O
with O
carbimazole O
( O
Neotomizol O
) O
, O
carbimazole O
plus O
low O
dose O
CT O
( O
Calsynar O
; O
100 O
IU O
/ O
day O
, O
2 O
days O
/ O
week O
) O
, O
or O
carbimazole O
plus O
high O
dose O
CT O
( O
Calsynar O
; O
100 O
IU O
/ O
day O
, O
14 O
days O
/ O
month O
) O
. O

The O
cloning O
of O
CWH43 B
showed O
that O
it O
corresponds O
to O
YCR017c B
and O
encodes O
a O
protein O
with O
14 O
- O
16 O
transmembrane O
segments O
containing O
several O
putative O
phosphorylation O
and O
glycosylation O
sites O
. O

However O
, O
strains O
in O
which O
both O
of O
the O
two O
most O
highly O
related O
genes O
, O
DdPIK1 B
and O
DdPIK2 B
, O
were O
disrupted O
showed O
both O
growth O
and O
developmental O
defects O
, O
while O
double O
knockouts O
of O
DdPIK1 B
and O
DdPIK3 B
and O
DdPIK2 B
and O
DdPIK3 B
appear O
to O
be O
lethal O
. O

This O
mechanism O
seems O
to O
be O
involved O
in O
VIP B
- O
induced O
PRL B
gene I
regulation O
. O

Competition O
and O
methylation O
interference O
assays O
showed O
that O
the O
binding O
site O
for O
the O
novel O
factor O
was O
limited O
to O
nucleotides O
in O
the O
3 O
' O
half O
of O
the O
kappa B
B I
site I
. O

These O
properties O
prompted O
us O
to O
investigate O
Ets B
- I
1 I
expression O
in O
32 O
human O
astroglial O
tumors O
of O
WHO O
grades O
I O
- O
IV O
and O
to O
correlate O
the O
data O
with O
the O
expression O
pattern O
of O
VEGF B
, O
Flt B
- I
1 I
, O
and O
KDR B
. O

Mean O
concentration O
of O
insulin B
in O
maternal O
plasma O
across O
all O
sampling O
times O
was O
higher O
in O
ethanol O
treated O
animals O
. O

The O
strongly O
DNA O
binding O
p50 B
subunit I
showed O
only O
very O
weak O
, O
if O
any O
, O
induction O
of O
gene O
expression O
. O

The O
results O
obtained O
demonstrate O
the O
applicability O
of O
countercurrent O
chromatography O
to O
the O
determination O
of O
ultratrace O
elements O
. O

E1A B
+ O
cHa B
- I
ras I
transformed O
rat O
embryo O
fibroblast O
cells O
are O
characterized O
by O
high O
and O
constitutive O
DNA O
binding O
activities O
of O
AP B
- I
1 I
dimers I
with O
significantly O
altered O
composition O
. O

Minor O
changes O
within O
the O
transmembrane O
domains O
( O
TMs O
) O
sometimes O
produced O
major O
effects O
and O
more O
drastic O
changes O
in O
the O
TMs O
ablated O
surface O
expression O
entirely O
. O

Nuclear O
localization O
of O
this O
SNF2 B
- I
like I
putative O
helicase B
is O
dependent O
on O
a O
nuclear O
localization O
sequence O
located O
in O
the O
NH2 O
- O
terminal O
region O
. O

The O
feasibility O
of O
creating O
CR1 B
/ O
Ig B
chimeras O
makes O
possible O
a O
new O
strategy O
of O
targeting O
complement O
inhibition O
by O
the O
use O
of O
Ig B
fusion O
partners O
having O
particular O
antigenic O
specificities O
. O

Morphological O
- O
functional O
status O
of O
the O
edentulous O
mandible O
and O
selection O
of O
the O
most O
effective O
method O
for O
functional O
impression O
- O
taking O

They O
are O
both O
insensitive O
to O
inhibitors O
of O
serine B
and I
aspartyl I
proteinases I
but O
are O
sensitive O
to O
sulfhydryl O
reagents O
and O
0 O
. O
5 O
mM O
ZnCl2 O
. O

The O
tau O
v O
at O
rest O
was O
4 O
. O
06 O
+ O
/ O
- O
2 O
. O
16 O
s O
and O
decreased O
to O
2 O
. O
44 O
+ O
/ O
- O
1 O
. O
07 O
s O
( O
P O
< O
0 O
. O
05 O
) O
at O
1 O
Hz O
and O
to O
1 O
. O
81 O
+ O
/ O
- O
0 O
. O
4 O
s O
at O
5 O
Hz O
. O

The O
authors O
investigate O
in O
a O
group O
of O
211 O
children O
aged O
2 O
- O
6 O
years O
, O
incl O
. O
in O
particular O
a O
group O
of O
33 O
2 O
- O
year O
- O
old O
ones O
, O
in O
1990 O
and O
checked O
in O
1991 O
at O
two O
sites O
in O
Bratislava O
- O
- O
a O
town O
with O
a O
varying O
concentration O
of O
NOX O
, O
SO2 O
, O
dustiness O
and O
dust O
fallout O
- O
- O
their O
influence O
on O
the O
incidence O
, O
type O
and O
course O
of O
relapsing O
respiratory O
disease O
. O

However O
, O
no O
statistical O
difference O
in O
the O
Ca O
/ O
P O
ratio O
, O
carbon O
concentration O
and O
surface O
energy O
were O
observed O
with O
different O
heat O
treatments O
. O

Using O
in O
vivo O
genomic O
dimethyl O
sulfate O
and O
KMnO4 O
footprinting O
, O
we O
showed O
that O
the O
promoter O
region O
is O
differentially O
protected O
, O
depending O
upon O
which O
holoenzyme O
is O
bound O
. O

Here O
we O
show O
that O
inhibition O
occurs O
with O
a O
NS1 B
- O
Rev B
chimera O
in O
which O
the O
78 O
amino O
- O
terminal O
amino O
acids O
of O
the O
NS1A B
protein I
comprising O
its O
entire O
RNA O
- O
binding O
domain O
is O
deleted O
, O
thereby O
establishing O
that O
this O
carboxyl O
portion O
of O
the O
NS1A B
protein I
can O
function O
as O
an O
independent O
effector O
domain O
. O

Overall O
, O
it O
appears O
that O
prenatal O
exposure O
to O
androgen O
- O
based O
synthetic O
progestin O
exerted O
a O
masculinizing O
and O
/ O
or O
defeminizing O
influence O
on O
human O
behavioral O
development O
, O
whereas O
prenatal O
exposure O
to O
natural O
progesterone O
and O
progesterone O
- O
based O
synthetic O
progestin O
had O
a O
feminizing O
and O
/ O
or O
demasculinizing O
influence O
, O
particularly O
among O
female O
subjects O
. O

The O
testes O
of O
8 O
specimens O
of O
Triturus O
marmoratus O
were O
collected O
during O
each O
month O
of O
1987 O
and O
processed O
for O
electron O
microscopy O
and O
light O
microscopy O
demonstration O
of O
testosterone O
( O
T O
) O
following O
the O
ABC B
( O
avidin B
- I
biotin I
peroxidase I
complex I
) O
method O
. O

MTX O
therapy O
decreased O
Ga O
- O
67 O
uptake O
in O
liver O
, O
tumor O
, O
and O
muscle O
. O

In O
stage O
I O
, O
histochemistry O
for O
copper O
was O
positive O
in O
11 O
out O
of O
21 O
cases O
: O
6 O
cases O
were O
T O
+ O
; O
1 O
case O
R O
+ O
and O
2 O
cases O
O O
+ O
; O
2 O
cases O
were O
T O
+ O
, O
R O
+ O
, O
O O
+ O
. O

Encapsulation O
of O
sodium O
fluorescein O
for O
dye O
release O
studies O
. O

Primer O
extension O
analysis O
and O
subcloning O
of O
the O
virJ B
- O
phoA B
fusion O
indicate O
that O
the O
acetosyringone O
- O
inducible O
promoter O
lies O
directly O
upstream O
of O
the O
virJ B
structural I
gene I
. O

To O
isolate O
genes O
that O
contain O
zinc O
finger O
motifs O
, O
a O
human O
brain O
cDNA O
library O
was O
screened O
with O
an O
oligonucleotide O
complementary O
to O
the O
conserved O
" O
linker O
" O
sequence O
between O
adjacent O
zinc O
fingers O
. O

Percentage O
incremental O
18 O
- O
OHB O
responses O
to O
metoclopramide O
were O
greater O
( O
P O
less O
than O
0 O
. O
01 O
) O
in O
the O
subjects O
after O
5 O
days O
on O
a O
200 O
- O
meq O
sodium O
intake O
than O
after O
5 O
days O
on O
a O
10 O
- O
meq O
sodium O
intake O
. O

Using O
in O
vivo O
screening O
, O
we O
first O
identified O
a O
gene O
that O
appeared O
to O
interfere O
with O
the O
His O
- O
Asp O
phosphorelay O
between O
the O
HPt B
domain I
and O
the O
receiver O
domain O
of O
OmpR B
, O
provided O
that O
the O
gene O
product O
was O
expressed O
through O
a O
multicopy O
plasmid O
. O

The O
non O
- I
major I
histocompatibility I
complex I
( O
MHC B
) O
- O
encoded O
CD1 B
family O
has O
recently O
emerged O
as O
a O
new O
antigen O
- O
presenting O
system O
that O
is O
distinct O
from O
either O
MHC B
class I
I I
or O
class B
II I
molecules I
. O

The O
predicted O
amino O
acid O
sequence O
of O
Pst1p B
possessed O
high O
sequence O
homology O
with O
the O
Sin3 B
family I
of O
proteins O
, O
known O
for O
their O
interaction O
with O
histone B
deacetylases I
. O

Quinethazone O

Studies O
on O
nitrogen O
- O
containing O
heterocyclic O
compounds O
. O

Removal O
of O
ink4a B
dramatically O
reduces O
the O
lymphoid O
and O
neurological O
defects O
seen O
in O
bmi B
- I
1 I
- O
deficient O
mice O
, O
indicating O
that O
ink4a B
is O
a O
critical O
in O
vivo O
target O
for O
Bmi B
- I
1 I
. O

We O
found O
that O
RXR B
and O
VDR B
transactivated O
selectively O
from O
VDRE O
- O
linked O
templates O
exclusively O
as O
a O
heterodimeric O
complex O
, O
since O
neither O
receptor O
alone O
enhanced O
transcription O
in O
vitro O
. O

METHODS O
: O
We O
performed O
nasal O
mupirocin O
treatment O
on O
10 O
infants O
who O
were O
MRSA O
- O
positive O
either O
in O
the O
nose O
or O
the O
pharynx O
and O
evaluated O
the O
effect O
of O
mupirocin O
on O
the O
eradication O
of O
MRSA O
. O

RESULTS O
: O
Of O
10 O
, O
709 O
, O
701 O
chemistry O
specimens O
submitted O
to O
the O
participating O
laboratories O
during O
the O
data O
collection O
period O
, O
37 O
, O
208 O
( O
0 O
. O
35 O
% O
) O
were O
rejected O
prior O
to O
testing O
. O

Early O
complement O
components O
, O
C1q B
and O
C4 B
, O
and O
IgA B
secretory I
piece I
were O
absent O
. O

Administration O
of O
SnCl2 O
and O
ZnSO4 O
( O
group O
V O
) O
resulted O
in O
a O
decrease O
of O
ALA B
- I
D I
activity O
and O
in O
an O
increase O
in O
CP B
- I
U I
. O

Using O
a O
dominant O
negative O
form O
of O
Sp3 B
and O
transcriptional O
activation O
assays O
in O
Schneider O
SL O
- O
2 O
insect O
cells O
, O
it O
was O
confirmed O
that O
ERalpha B
- O
Sp3 B
interactions O
define O
a O
pathway O
for O
E2 O
- O
mediated O
inhibition O
of O
gene O
expression O
, O
and O
this O
represents O
a O
new O
mechanism O
for O
decreased O
gene O
expression O
by O
E2 O
. O

From O
the O
supposition O
that O
there O
exists O
a O
possible O
connection O
between O
certain O
psychopathological O
symptoms O
, O
and O
/ O
or O
syndromes O
( O
e O
. O
g O
. O
, O
hallucinations O
) O
and O
regional O
cerebral O
dysfunction O
, O
patients O
suffering O
from O
auditory O
and O
tactile O
hallucinations O
were O
investigated O
, O
in O
a O
symptom O
- O
oriented O
study O
, O
using O
the O
method O
of O
technetium O
- O
99m O
- O
Hexamethyl O
- O
propylenamine O
Oxime O
( O
99m O
- O
Tc O
- O
HMPAO O
) O
- O
Single O
Photon O
Emission O
Computed O
Tomography O
( O
SPECT O
) O
and O
compared O
with O
normal O
controls O

. O

The O
lack O
of O
chronically O
depressed O
LCBF O
( O
after O
1 O
day O
) O
may O
be O
related O
to O
the O
quick O
structural O
repair O
of O
endothelium O
. O

In O
this O
study O
, O
to O
elucidate O
the O
possible O
mechanism O
of O
action O
of O
EM O
, O
we O
examined O
the O
effect O
of O
the O
drug O
on O
the O
expression O
of O
neutrophil O
adhesion O
molecules O
such O
as O
L B
- I
selectin I
and O
Mac B
- I
1 I
. O

Tissue O
expansion O
was O
used O
successfully O
to O
prepare O
adequate O
soft O
tissue O
for O
closure O
following O
a O
difficult O
clubfoot O
correction O
. O

The O
transcription O
start O
site O
was O
identified O
by O
primer O
extension O
analysis O
and O
over O
800 O
bp O
of O
5 O
' O
flanking O
DNA O
was O
sequenced O
. O

The O
reporter O
gene O
with O
two O
copies O
of O
the O
wild O
- O
type O
Repeat O
2 O
+ O
3 O
sequence O
was O
transcribed O
actively O
in O
sterol O
- O
deprived O
cells O
and O
was O
repressed O
by O
more O
than O
80 O
% O
when O
sterols O
were O
present O
. O

Changes O
in O
tension O
were O
monitored O
isometrically O
on O
spiral O
strips O
of O
freshly O
obtained O
bovine O
basilar O
arteries O
. O

A O
1 O
. O
5 O
- O
month O
- O
old O
boy O
with O
Sandifer O
' O
s O
syndrome O
is O
described O
. O

However O
, O
PC12 O
cell O
clones O
which O
expressed O
p33rsu B
- I
1 I
at O
an O
increased O
level O
in O
a O
regulatable O
fashion O
in O
response O
to O
dexamethasone O
were O
isolated O
. O

In O
this O
study O
, O
we O
verified O
that O
PI B
3 I
- I
kinase I
associates O
with O
the O
CD4 B
: O
p56lck B
complex O
as O
judged O
by O
the O
presence O
of O
PI O
3 O
- O
phosphate O
generated O
from O
anti B
- I
CD4 I
immunoprecipitates O
and O
detected O
by O
high O
- O
pressure O
liquid O
chromatographic O
analysis O
. O

The O
clinical O
picture O
, O
the O
light O
and O
electron O
microscopic O
appearance O
, O
and O
the O
histochemical O
findings O
are O
described O
in O
six O
cases O
of O
fibroma O
of O
tendon O
sheath O
. O

However O
, O
their O
stability O
was O
low O
as O
already O
reported O
for O
the O
ARS O
in O
S O
. O
occidentalis O
. O

In O
this O
study O
, O
a O
site O
of O
PDGF B
- O
induced O
tyrosine O
phosphorylation O
was O
mapped O
to O
Tyr O
138 O
in O
the O
SH3 B
domain I
; O
Tyr O
138 O
is O
exposed O
on O
the O
SH3 B
peptide I
binding O
surface O
. O

The O
results O
of O
this O
study O
suggest O
that O
in O
patients O
with O
syndrome O
X O
, O
myocardial O
ischemia O
frequently O
develops O
during O
daily O
life O
; O
silent O
ischemia O
is O
an O
important O
component O
of O
this O
syndrome O
; O
and O
increased O
oxygen O
demand O
in O
the O
presence O
of O
impaired O
coronary O
vasodilatory O
capacity O
is O
not O
the O
only O
cause O
of O
myocardial O
ischemia O
. O

We O
also O
identified O
a O
DNA O
fragment O
, O
10 O
. O
7 O
kbp O
upstream O
from O
the O
first O
coding O
exon O
of O
human B
aFGF I
, O
whose O
sequence O
is O
conserved O
in O
both O
the O
primate O
and O
rodent O
genomes O
. O

The O
false O
positive O
fraction O
( O
FPF O
) O
decreased O
significantly O
if O
the O
count O
density O
increased O
; O
no O
difference O
in O
FPF O
was O
found O
for O
a O
change O
in O
lesion O
tracer O
concentration O
. O

These O
events O
were O
only O
weakly O
stimulated O
by O
the O
activated O
PDGF B
alpha I
- I
receptor I
. O

Human B
plastin I
genes I
. O

Buserelin O
offers O
an O
effective O
alternative O
medical O
treatment O
of O
carcinoma O
of O
the O
prostate O
and O
, O
apart O
from O
impotence O
, O
does O
not O
have O
the O
side O
effects O
of O
oestrogens O
. O

C B
/ I
EBP I
delta I
( O
NFIL B
- I
6 I
beta I
) O
was O
not O
detected O
in O
complexes O
utilizing O
extracts O
from O
the O
IL B
- I
1 I
nonproducing O
T O
cell O
line O
. O

Reduced O
risk O
of O
upper O
gastrointestinal O
ulcer O
complications O
with O
celecoxib O
, O
a O
novel O
COX B
- I
2 I
inhibitor O
. O

Moreover O
, O
our O
recent O
findings O
on O
the O
Tc52 B
encoding I
gene I
underline O
the O
interest O
of O
genetic O
manipulation O
of O
T O
. O
cruzi O
, O
not O
only O
making O
it O
possible O
to O
use O
more O
closely O
an O
in O
vitro O
approach O
to O
find O
out O
how O
genes O
function O
, O
but O
also O
to O
obtain O
' O
attenuated O
' O
strains O
that O
could O
be O
used O
in O
the O
development O
of O
vaccinal O
strategies O
. O

Infra O
- O
red O
spectroscopy O
of O
tissues O
in O
the O
700 O
- O
400 O
cm O
- O
1 O
region O
. O

YDL003w B
( O
also O
termed O
MCD1 B
) O
is O
a O
homologue B
of I
Schizosaccharomyces I
pombe I
rad21 I
, O
an O
essential O
gene O
implicated O
in O
DNA O
double O
- O
strand O
break O
repair O
and O
nuclear O
organization O
in O
fission O
yeast O
. O

The O
recent O
concept O
of O
a O
bioartificial O
organ O
is O
an O
attempt O
to O
avoid O
such O
disadvantages O
. O

The O
objectives O
of O
this O
study O
were O
1 O
) O
to O
quantify O
the O
comparative O
structural O
static O
and O
fatigue O
properties O
of O
the O
pointe O
shoe O
toe O
box O
, O
and O
2 O
) O
to O
evaluate O
the O
preferred O
shoe O
characteristics O
as O
determined O
by O
a O
survey O
of O
local O
dancers O
. O

In O
the O
postoperative O
patient O
with O
tetralogy O
of O
Fallot O
with O
symptomatic O
ventricular O
arrhythmias O
, O
it O
is O
concluded O
that O
electrophysiologic O
study O
is O
useful O
in O
reproducing O
clinical O
episodes O
of O
VT O
and O
in O
selecting O
effective O
antiarrhythmic O
medication O
; O
a O
small O
number O
of O
patients O
with O
ventricular O
premature O
complexes O
alone O
will O
have O
inducible O
sustained O
VT O
during O
electrophysiologic O
study O
; O
prognosis O
of O
these O
patients O
may O
be O
improved O
by O
treatment O
that O
results O
in O
prevention O
of O
VT O
induction O
; O
and O
in O
patients O
with O
right O
ventricular O
hypertension O
, O

VT O
is O
likely O
to O
be O
refractory O
to O
drug O
treatment O
. O

Indications O
for O
therapy O
using O
MAO B
inhibitors O

The O
absorbance O
of O
the O
eluent O
was O
monitored O
at O
254 O
nm O
. O

Here O
, O
we O
describe O
the O
results O
of O
a O
detailed O
study O
aimed O
at O
identifying O
the O
gene O
or O
genes O
responsible O
for O
the O
rapid O
growth O
- O
arrest O
response O
obtained O
with O
human O
chromosome O
- O
9 O
. O

Footprints O
on O
the O
HPV O
18 O
enhancer O
show O
five O
protected O
regions O
with O
homologies O
to O
NF1 B
, O
AP1 B
and O
EFII B
transcription I
factor I
binding I
motifs I
. O

Insulin B
binding O
to O
the O
alpha O
- O
subunit O
of O
its O
receptor O
stimulates O
the O
receptor B
tyrosine I
kinase I
to O
phosphorylate O
the O
beta O
- O
subunit O
and O
several O
endogenous O
protein O
substrates O
, O
including O
pp120 B
/ O
HA4 B
, O
a O
liver O
- O
specific O
plasma I
membrane I
glycoprotein I
of O
M O
( O
r O
) O
20 O
, O
000 O
. O

Motilin B
serum O
levels O
were O
measured O
for O
one O
hour O
. O

The O
role O
of O
phosphorylation O
on O
receptor O
desensitization O
was O
assessed O
using O
receptor O
mutants O
expressed O
in O
COS O
cells O
or O
Chinese O
hamster O
lung O
fibroblasts O
. O

First O
aid O
. O

Improvement O
of O
some O
pharmaceutical O
properties O
of O
clofibrate O
by O
cyclodextrin B
complexation O
. O

The O
detection O
limits O
obtained O
by O
using O
the O
mixed O
eluent O
were O
0 O
. O
05 O
- O
0 O
. O
13 O
mg O
/ O
L O
, O
several O
times O
lower O
than O
those O
obtained O
by O
using O
single O
KHPh O
eluent O
. O

Early O
diagnosis O
and O
early O
surgical O
division O
of O
the O
biliary O
tract O
and O
pancreatic O
duct O
is O
recommended O
for O
children O
with O
AAPBD O
. O

We O
have O
analyzed O
herein O
the O
roles O
that O
four O
different O
PIAS B
proteins I
( O
ARIP3 B
/ O
PIASx B
alpha I
, O
Miz1 B
/ O
PIASx B
beta I
, O
GBP B
/ O
PIAS1 B
, O
and O
PIAS3 B
) O
play O
in O
the O
regulation O
of O
steroid B
receptor I
- O
or O
STAT B
- O
mediated O
transcriptional O
activation O
. O

Comparison O
of O
the O
predicted O
amino O
acid O
sequence O
with O
that O
of O
the O
Drosophila O
virilis O
gene O
shows O
that O
several O
blocks O
of O
amino O
acid O
sequence O
have O
been O
very O
highly O
conserved O
. O

Analyses O
focused O
on O
two O
platyrrhine O
( O
New O
World O
monkey O
) O
species O
: O
the O
common O
marmoset O
( O
Callithrix O
jacchus O
) O
and O
the O
brown O
capuchin O
monkey O
( O
Cebus O
apella O
) O
, O
each O
of O
which O
has O
paired O
, O
non O
- O
allelic O
gamma O
loci O
( O
5 O
' O
- O
gamma B
1 I
- O
gamma B
2 I
- O
3 O
' O
) O
. O

A O
phase O
II O
clinical O
trial O
on O
MDS O
was O
conducted O
in O
a O
cooperative O
study O
with O
orally O
administrable O
ara O
- O
C O
analogue O
, O
PLAC O
, O
which O
is O
resistant O
to O
cytidine B
deaminase I
and O
had O
shown O
an O
anti O
- O
tumor O
activity O
on O
various O
experimental O
tumors O
by O
oral O
route O
. O

A O
decrease O
was O
also O
found O
of O
the O
levels O
of O
total O
cholesterol O
and O
LDL B
- I
cholesterol I
. O

Previously O
, O
we O
had O
shown O
that O
interleukin B
8 I
( O
IL B
- I
8 I
) O
is O
constitutively O
expressed O
in O
HTLV O
- O
I O
- O
infected O
cells O
and O
in O
cells O
transiently O
expressing O
Tax B
. O

Unlike O
JNK B
activation O
, O
ERK B
activation O
could O
not O
be O
mapped O
to O
specific O
reovirus O
gene O
segments O
, O
suggesting O
that O
ERK B
activation O
and O
JNK B
activation O
are O
triggered O
by O
different O
events O
during O
virus O
- O
host O
cell O
interaction O
. O

Despite O
the O
same O
decreases O
or O
increases O
in O
MAP O
, O
RSNA O
was O
attenuated O
after O
15 O
and O
30 O
min O
of O
propofol O
infusion O
in O
both O
groups O
compared O
with O
control O
( O
P O
< O
0 O
. O
05 O
) O
. O

To O
address O
this O
question O
with O
respect O
to O
skeletal O
muscle O
, O
we O
have O
examined O
the O
effects O
of O
the O
Providence B
mutation I
in O
cultured O
muscle O
cells O
, O
after O
adoptive O
gene O
transfer O
to O
adult O
mice O
, O
and O
in O
two O
infants O
homozygous O
for O
spectrin B
Providence I
. O

It O
appears O
to O
be O
useful O
in O
untreated O
patients O
with O
stage O
I O
or O
II O
NHL O
but O
no O
definite O
radiographic O
abnormalities O
, O
or O
with O
abnormal O
radiographs O
but O
no O
extrathoracic O
spread O
, O
and O
in O
treated O
patients O
with O
questionable O
radiographs O
. O

B B
and O
C1 B
fusions I
with O
yeast B
GAL4 I
DNA O
- O
binding O
and O
transcriptional O
activation O
domains O
were O
also O
found O
to O
interact O
when O
synthesized O
and O
assayed O
in O
yeast O
. O

Because O
of O
the O
data O
, O
we O
suggest O
that O
LAC9 B
contacts O
positions O
6 O
, O
7 O
, O
and O
8 O
, O
both O
plus O
and O
minus O
, O
of O
the O
UAS O
, O
which O
are O
separated O
by O
more O
than O
one O
turn O
of O
the O
DNA O
helix O
, O
and O
twists O
part O
way O
around O
the O
DNA O
, O
thus O
protecting O
the O
broad O
region O
of O
the O
minor O
groove O
between O
the O
major O
- O
groove O
contacts O
. O

On O
the O
optic O
tentacle O
- O
gonadal O
axis O
in O
the O
control O
of O
the O
male O
- O
phase O
ovotestis O
in O
the O
slug O
( O
Ariolimax O
californicus O
) O
. O

Substitution O
of O
cholinergic O
drugs O
in O
the O
treatment O
of O
Alzheimer O
type O
dementia O
( O
AD O
/ O
SDAT O
) O
has O
hitherto O
not O
been O
very O
successful O
. O

Here O
we O
show O
that O
the O
nucleotide O
in O
the O
middle O
of O
the O
anticodon O
( O
i O
. O
e O
. O
, O
psi O
35 O
) O
also O
contributes O
to O
the O
suppressor O
efficiency O
displayed O
by O
cytoplasmic B
tRNA I
( I
Tyr I
) I
. O

The O
root O
- O
mean O
- O
square O
deviation O
of O
bond O
lengths O
from O
the O
ideal O
values O
is O
0 O
. O
02 O
A O
. O

Jet O
lag O
and O
melatonin O
. O

In O
addition O
, O
these O
results O
suggest O
that O
the O
interactions O
, O
and O
consequently O
the O
mechanisms O
, O
governing O
transcriptional O
activation O
by O
CTF B
are O
distinct O
from O
those O
mediating O
DNA O
replication O
. O

A O
TAAATA O
sequence O
is O
present O
26 O
nt O
upstream O
from O
the O
major O
CAP O
site O
, O
and O
within O
the O
5 O
' O
- O
flanking O
region O
there O
are O
several O
putative O
transcription O
factor O
binding O
sites O
. O

It O
was O
found O
that O
both O
versions O
of O
the O
brief O
SL O
& O
amp O
; O
shy O
; O
ASIA O
had O
similar O
correlations O
to O
the O
target O
variables O
as O
the O
full O
scale O
SL O
& O
amp O
; O
shy O
; O
ASIA O
. O

The O
reconstituted O
enzyme O
binds O
DNA O
with O
an O
affinity O
that O
is O
approximately O
20 O
- O
fold O
lower O
than O
that O
of O
the O
intact O
topo70 B
. O

Treatment O
with O
KP O
- O
45 O
preparation O
usually O
resulted O
in O
partial O
regression O
of O
tumor O
growth O
, O
accompanied O
by O
improvement O
of O
the O
clinical O
state O
of O
these O
patients O
, O
as O
well O
as O
reappearance O
of O
normal O
values O
of O
blood O
picture O
and O
biochemical O
parameters O
. O

Istanbul O
, O
Turkey O
. O

We O
propose O
that O
optimal O
transcription O
of O
the O
gD B
gene I
depends O
on O
the O
interaction O
of O
ICP4 B
with O
multiple O
binding O
sites O
across O
the O
gene O
and O
cellular O
factors O
that O
recognize O
specific O
sequence O
elements O
in O
the O
promoter O
. O

Recently O
, O
we O
and O
other O
laboratories O
have O
identified O
a O
family O
of O
caveolin B
- I
related I
proteins I
; O
caveolin B
has O
been O
re O
- O
termed O
caveolin B
- I
1 I
. O

Role O
of O
zinc B
- I
finger I
proteins I
Sp1 B
and O
zif268 B
/ O
egr B
- I
1 I
in O
transcriptional O
regulation O
of O
the O
human B
synaptobrevin I
II I
gene I
. O

The O
human B
Fc I
gamma I
receptor I
gene I
Fc B
gamma I
RIIA I
is O
expressed O
in O
platelets O
, O
neutrophils O
, O
monocytes O
and O
macrophages O
. O

This O
dip O
also O
occurred O
during O
prednisone O
and O
vitamin O
D O
treatment O
, O
but O
did O
not O
occur O
when O
calcium O
was O
added O
to O
prednisone O
, O
although O
the O
baseline O
value O
was O
lower O
at O
the O
start O
of O
combined O
treatment O
with O
prednisone O
and O
calcium O
. O

E O
. O
granulosus O
infection O
has O
been O
confirmed O
in O
25 O
patients O
( O
20 O
. O
6 O
% O
) O
, O
in O
16 O
cases O
by O
finding O
parasite O
protoscoleces O
or O
hooks O
and O
in O
nine O
cases O
by O
detection O
of O
an O
antigen O
specific O
for O
E O
. O
granulosus O
, O
antigen B
5 I
( O
Ag5 B
) O
. O

The O
effect O
of O
stimulation O
of O
the O
entopeduncular O
nucleus O
( O
EP O
) O
on O
the O
jaw O
- O
opening O
reflex O
( O
JOR O
) O
was O
studied O
in O
the O
cat O
anesthetized O
with O
sodium O
pentobarbital O
. O

A O
Premenstrual O
Syndrome O
, O
the O
late O
luteal O
phase O
disorder O
of O
DSM O
- O
IIIR O
criteria O
, O
was O
identified O
in O
6 O
% O
of O
the O
women O
. O

First O
, O
mutations O
in O
the O
IFNgamma B
- I
activated I
sequence I
( O
GAS B
) O
, O
either O
multimerized O
or O
in O
the O
context O
of O
the O
1 B
. I
7 I
- I
kb I
IRF I
- I
1 I
promoter I
, O
failed O
to O
mediate O
a O
PRL B
response O
, O
showing O
that O
the O
IRF B
- I
1 I
GAS I
is O
a O
target O
of O
PRL B
signaling O
. O

025 O
) O
. O

RESULTS O
: O
PET O
revealed O
wide O
variations O
in O
CBF O
between O
regions O
during O
isoflurane O
anaesthesia O
, O
particularly O
in O
comparison O
with O
propofol O
anaesthesia O
, O
while O
rCMRO2 O
decreased O
globally O
in O
a O
dose O
- O
dependent O
manner O
during O
both O
isoflurane O
and O
propofol O
anaesthesia O
. O

This O
selective O
squelching O
activity O
suggests O
that O
GRIM B
can O
interact O
with O
an O
essential O
component O
of O
the O
RNA B
polymerase I
II I
transcription O
machinery O
. O

This O
is O
the O
first O
description O
of O
an O
integron B
located O
on O
a O
composite O
transposon O
. O

The O
nine O
subjects O
were O
treated O
in O
random O
sequence O
with O
cimetidine O
0 O
- O
8 O
- O
1 O
- O
0 O
g O
on O
one O
day O
and O
placebo O
capsules O
on O
the O
other O
. O

The O
protein O
was O
overexpressed O
in O
Escherichia O
coli O
and O
purified O
to O
homogeneity O
. O

Identification O
and O
characterization O
of O
a O
yeast O
gene O
encoding O
the O
U2 B
small I
nuclear I
ribonucleoprotein I
particle I
B I
" I
protein I
. O

A O
360 O
- O
bp O
DNA O
fragment O
located O
over O
500 O
bp O
upstream O
from O
the O
cfl B
transcriptional I
start I
site I
was O
used O
in O
DNase B
I I
protection O
assays O
to O
define O
the O
specific O
bases O
bound O
by O
CorR B
. O

Taking O
into O
account O
certain O
well O
defined O
conditions O
( O
frequent O
feedings O
, O
no O
supplementary O
feeding O
before O
4 O
- O
6 O
months O
, O
method O
only O
to O
be O
used O
in O
the O
absence O
of O
menstruation O
) O
, O
LAM O
can O
be O
relied O
on O
for O
contraceptive O
protection O
for O
up O
to O
1 O
year O
post O
partum O
. O

Nramp2 B
contains O
a O
classical O
iron O
responsive O
element O
in O
the O
3 O
' O
untranslated O
region O
that O
confers O
iron O
dependent O
mRNA O
stabilization O
. O

Likewise O
, O
epidural O
morphine O
does O
not O
modify O
the O
intraoperative O
metabolic O
and O
hormonal O
responses O
. O

CONCLUSIONS O
- O
- O
Postlumbar O
puncture O
headache O
may O
be O
mediated O
by O
the O
release O
of O
substance B
P I
triggered O
by O
lumbar O
puncture O
, O
in O
patients O
predisposed O
to O
headache O
by O
a O
hypersensitivity O
to O
substance B
P I
. O

Continuous O
arterial O
blood O
sampling O
method O
based O
on O
the O
microsphere O
model O
was O
used O
as O
a O
quantitative O
rCBF O
measurement O
. O

A O
mutated B
ABF1 I
site I
that O
displays O
a O
very O
low O
affinity O
for O
ABF1 B
does O
not O
functionally O
replace O
the O
ILV1 B
REB1 B
site I
. O

Given O
a O
rapid O
induction O
of O
Egr B
- I
1 I
on O
stimulation O
with O
growth O
factors O
or O
injury O
, O
these O
findings O
may O
represent O
at O
least O
one O
of O
the O
molecular O
mechanisms O
underlying O
phenotypic O
modulation O
of O
smooth O
muscles O
after O
vascular O
injury O
. O

A O
nationwide O
survey O
uncovered O
a O
tenfold O
variation O
between O
counties O
in O
the O
prescribing O
of O
IFNB B
. O

In O
this O
first O
- O
person O
account O
, O
the O
author O
chronicles O
her O
experience O
of O
being O
mentally O
ill O
and O
homeless O
in O
New O
York O
and O
New O
Jersey O
in O
the O
early O
1980s O
. O

Rectal O
temperatures O
after O
the O
walk O
were O
greater O
( O
P O
< O
. O
001 O
) O
in O
native O
Simmental O
( O
39 O
. O
87 O
+ O
/ O
- O
. O
05 O
degrees O
C O
) O
than O
in O
Bos O
indicus O
( O
39 O
. O
46 O
+ O
/ O
- O
. O
05 O
degrees O
C O
) O
. O

Dominant O
- O
negative O
mutations O
in O
the O
G B
- I
protein I
- I
coupled I
alpha I
- I
factor I
receptor I
map O
to O
the O
extracellular O
ends O
of O
the O
transmembrane O
segments O
. O

Such O
simple O
injuries O
may O
however O
be O
accompanied O
by O
far O
reaching O
consequences O
. O

This O
is O
the O
first O
report O
which O
clearly O
proved O
CEA B
synthesis O
in O
the O
cells O
of O
HCC O
. O

By O
screening O
a O
human O
testis O
cDNA O
library O
with O
a O
probe O
containing O
the O
mouse O
PEA3 B
ETS B
domain O
, O
we O
isolated O
a O
2 O
. O
2 O
kb O
clone O
containing O
a O
510 O
AA O
open O
reading O
frame O
. O

AU O
- O
rich O
elements O
in O
the O
3 O
' O
- O
untranslated O
region O
of O
a O
new O
mucin B
- I
type I
gene I
family I
of O
Trypanosoma O
cruzi O
confers O
mRNA O
instability O
and O
modulates O
translation O
efficiency O
. O

These O
findings O
suggest O
that O
IL B
- I
1 I
- O
stimulated O
, O
Rho B
- O
dependent O
cytoskeletal O
reorganization O
may O
cluster O
signaling O
molecules O
in O
specific O
architectures O
that O
are O
necessary O
for O
persistent O
cell O
activation O
in O
chronic O
inflammatory O
disease O
. O

Changes O
in O
testicular O
testosterone O
and O
acid B
and I
alkaline I
phosphatase I
activity O
in O
testis O
and O
accessory O
sex O
organs O
after O
induction O
of O
varicocele O
in O
Noble O
rats O
. O

M O
. O
, O
Apps O
, O
D O
. O

These O
data O
suggest O
that O
weight O
gain O
or O
increased O
caloric O
intake O
, O
in O
contrast O
to O
its O
large O
effect O
on O
peripheral O
thyroid O
function O
, O
has O
relatively O
little O
effect O
on O
CNS B
TRH I
activity O
. O

Alfalfa O
phosphorus O
release O
kinetics O
showed O
high O
bacterial O
phosphorus O
contamination O
. O

The O
quantitative O
analysis O
of O
1 O
- O
alpha O
- O
acetylmethadol O
and O
its O
principal O
metabolites O
in O
biological O
specimens O
by O
gas O
chromatography O
- O
chemical O
ionization O
- O
multiple O
ion O
monitoring O
mass O
spectrometry O
. O

Clinical O
evidence O
provides O
tentative O
suggestions O
on O
( O
a O
) O
possible O
additional O
risk O
of O
cigarette O
smoking O
( O
b O
) O
avoidance O
of O
venography O
( O
c O
) O
avoidance O
of O
varicose O
vein O
surgery O
. O

Effects O
of O
prolonged O
inhibition O
of O
labour O
pains O
with O
Th O
1165a O
and O
Isoptin O
on O
the O
heart O
, O
circulation O
, O
organ O
- O
and O
metabolic O
parameters O
of O
the O
mother O

Skytthe O
, O
and O
K O
. O

In O
the O
Saccharomyces B
cerevisiae I
actin I
intron I
a O
silent O
branch O
point O
- O
like O
sequence O
( O
UACUAAG O
) O
is O
located O
7 O
nt O
upstream O
of O
the O
canonical O
sequence O
. O

The O
requirement O
for O
an O
essential O
interaction O
between O
NPH B
I I
and O
H4L B
provides O
an O
explanation O
for O
the O
observed O
restriction O
of O
transcription O
termination O
to O
early O
viral O
genes O
. O

After O
birth O
, O
there O
was O
a O
significantly O
higher O
increase O
of O
IgA B
and O
IgM B
anti I
- I
M I
. I
leprae I
antibody I
activity O
in O
sera O
taken O
3 O
- O
6 O
months O
after O
birth O
from O
babies O
of O
Group O
1 O
compared O
to O
Group O
2 O
, O
but O
the O
IgA B
and O
IgM B
activity O
in O
sera O
taken O
after O
6 O
months O
of O
age O
showed O
the O
same O
increase O
in O
the O
two O
groups O
. O

The O
relation O
between O
the O
changes O
of O
bone O
density O
and O
the O
level O
of O
blood O
calcium O
, O
blood O
phosphorus O
and O
alkaline B
phosphatase I
( O
AKP B
) O
as O
well O
as O
the O
metabolism O
of O
vitamin O
D O
was O
studied O
. O

These O
results O
demonstrate O
the O
extensive O
homology O
in O
sequence O
among O
light B
chains I
of O
IgM B
kappa I
autoantibodies I
and O
indicate O
that O
a O
particular O
V B
kappa I
germ I
line I
gene I
, O
kappa B
IIIb I
, O
is O
expressed O
as O
a O
phylogenetic O
response O
to O
certain O
self O
antigens O
or O
as O
part O
of O
a O
selection O
process O
by O
which O
these O
autoimmune O
responses O
are O
regulated O
. O

In O
addition O
, O
the O
findings O
confirmed O
previous O
studies O
which O
showed O
that O
monkeys O
with O
total O
bilateral O
striatectomies O
could O
require O
the O
ability O
to O
execute O
accurate O
visually O
guided O
reaches O
. O

Comparison O
of O
the O
sequences O
of O
the O
alpha B
- I
like I
DNA I
polymerases I
including O
E B
. I
coli I
DNA I
polymerase I
II I
showed O
that O
there O
were O
nine O
highly O
conserved O
regions O
, O
and O
we O
constructed O
an O
unrooted O
phylogenetic O
tree O
of O
the O
DNA B
polymerases I
based O
on O
the O
differences O
in O
these O
conserved O
regions O
. O

Before O
and O
6 O
and O
12 O
months O
after O
H O
. O
pylori O
eradication O
the O
patients O
were O
evaluated O
for O
fasting O
gastrinemia B
and O
pepsinogen B
I I
, O
basal O
and O
peak O
acid O
output O
, O
and O
detailed O
histological O
assessment O
including O
the O
ECL O
cell O
proliferative O
patterns O
. O

Four O
experiments O
examined O
the O
role O
of O
the O
cholinergic O
projections O
from O
the O
septum O
and O
vertical O
limb O
nucleus O
of O
the O
diagonal O
band O
of O
Broca O
( O
VDB O
) O
in O
acquisition O
and O
performance O
of O
a O
conditional O
visual O
discrimination O
. O

trans O
- O
activation O
of O
the O
HIV B
- I
1 I
LTR I
by O
the O
HIV B
- I
1 I
Tat I
and O
HTLV B
- I
I I
Tax I
proteins I
is O
mediated O
by O
different O
cis O
- O
acting O
sequences O
. O

Defective O
concanavalin B
A I
- O
induced O
suppression O
in O
bancroftian O
filariasis O
. O

For O
more O
detailed O
mapping O
, O
we O
constructed O
mouse O
A9 O
cells O
containing O
STFs O
derived O
from O
human O
chromosome O
2 O
tagged O
with O
pSTneo O
at O
different O
regions O
in O
2q31 O
- O
qter O
. O

Left O
ventricular O
insufficiency O
requires O
two O
SP O
/ O
patient O
, O
with O
good O
results O
in O
approximately O
81 O
p O
. O
cent O
. O

However O
, O
there O
are O
some O
cows O
that O
fail O
to O
produce O
adequate O
1 O
, O
25 O
- O
dihydroxyvitamin O
D O
at O
the O
onset O
of O
lactation O
. O

Acidic O
carboxyl O
- O
terminal O
domain O
of O
gene B
2 I
. I
5 I
protein I
of I
bacteriophage I
T7 I
is O
essential O
for O
protein O
- O
protein O
interactions O
. O

This O
result O
, O
together O
with O
the O
fact O
that O
unrearranged O
V B
kappa I
genes I
are O
transcriptionally O
silent O
, O
suggests O
that O
structural O
features O
of O
both O
the O
V B
kappa I
and O
C B
kappa I
loci I
contribute O
to O
the O
overall O
transcriptional O
efficiency O
of O
a O
rearranged O
V B
kappa I
- I
C I
kappa I
gene O
. O

A O
total O
of O
10 O
, O
763 O
men O
and O
3 O
, O
118 O
women O
has O
been O
studied O
, O
of O
whom O
97 O
% O
and O
89 O
% O
, O
respectively O
, O
have O
been O
traced O
and O
36 O
% O
and O
16 O
% O
have O
died O
. O

NSD1 B
contains O
a O
SET B
domain I
and O
multiple O
PHD B
fingers I
. O

EMG O
- O
based O
measures O
of O
fatigue O
during O
a O
repetitive O
squat O
exercise O
. O

We O
have O
introduced O
the O
same O
change O
in O
a O
yeast B
tRNA I
( I
Trp I
) I
gene I
and O
demonstrated O
that O
the O
tRNA O
acts O
as O
an O
efficient O
amber O
suppressor O
in O
vivo O
. O

This O
evaluation O
examines O
several O
analytical O
and O
cost O
parameters O
for O
five O
representative O
assays O
: O
IgG B
, O
IgA B
, O
IgM B
, O
phenytoin O
, O
and O
phenobarbital O
. O

These O
results O
suggest O
that O
UvrA B
is O
involved O
in O
triplet O
repeat O
instability O
in O
cells O
. O

Among O
women O
, O
plasma O
factor B
VII I
: O
Ag O
was O
inversely O
associated O
with O
income O
. O

A O
novel O
protein B
kinase I
C I
( O
PKC B
) O
- O
interacting O
protein O
was O
identified O
by O
the O
yeast O
two O
- O
hybrid O
screening O
using O
the O
regulatory O
domain O
of O
PKC B
beta I
I I
as O
a O
bait O
. O

CONCLUSIONS O
: O
The O
number O
of O
Spanish O
articles O
published O
in O
Anesthesiology O
, O
BJA O
, O
and O
Anesth O
Analg O
is O
low O
, O
although O
the O
comparison O
of O
our O
productivity O
with O
that O
of O
other O
EU O
countries O
in O
function O
of O
GNPpc O
places O
us O
in O
an O
intermediate O
position O
. O

One O
of O
the O
repeating O
domains O
( O
Yheb1 B
) O
, O
consisting O
of O
67 O
amino O
acids O
, O
was O
cloned O
from O
the O
E O
. O
coli O
chromosome O
and O
purified O
by O
metal O
chelating O
chromatography O
. O

Phosphorylation O
at O
Ser O
( O
670 O
) O
impairs O
the O
ability O
of O
GRK2 B
to O
phosphorylate O
both O
soluble O
and O
membrane O
- O
incorporated O
receptor O
substrates O
and O
dramatically O
attenuates O
Gbetagamma B
- O
mediated O
activation O
of O
this O
enzyme O
. O

METHODS O
: O
CD4 B
cell O
counts O
and O
interview O
data O
were O
recorded O
in O
a O
survey O
of O
136 O
adult O
patients O
attending O
a O
community O
hospital O
- O
based O
HIV O
outpatient O
clinic O
. O

Biol O
. O

Inhibition O
of O
endogenous B
Cdk2 I
by O
dominant B
negative I
Cdk2 I
attenuated O
phosphorylation O
of O
Thr447 O
, O
Thr490 O
and O
Thr497 O
, O
but O
had O
no O
effect O
upon O
Ser581 O
modification O
. O

A O
rabbit O
antiserum O
raised O
against O
a O
unique O
( O
V3 O
domain O
) O
bacterially O
expressed O
PKC B
theta I
fragment I
immunoprecipitated O
specifically O
an O
82 O
- O
kDa O
protein O
from O
Jurkat O
cell O
lysates O
. O

Histological O
examination O
revealed O
variable O
loss O
of O
motor O
neurons O
limited O
to O
the O
injection O
site O
. O

This O
is O
the O
most O
northern O
and O
, O
at O
the O
same O
time O
largest O
, O
Q O
fever O
epidemic O
recorded O
in O
Germany O
over O
the O
last O
28 O
years O
. O

Symptoms O
, O
skin O
- O
prick O
tests O
( O
SPT O
) O
with O
environmental O
allergens O
and O
Pt O
salt O
, O
total O
serum B
IgE I
, O
lung O
function O
, O
and O
bronchial O
hyperresponsiveness O
were O
assessed O
by O
standard O
procedures O
. O

Binding O
sites O
were O
mapped O
for O
each O
factor O
. O

Complementary O
DNA O
libraries O
from O
liver O
and O
ovary O
of O
an O
immature O
female O
channel O
catfish O
were O
screened O
with O
a O
homologous B
ERalpha I
cDNA I
probe O
. O

The O
binding O
of O
PTB B
was O
antagonistic O
to O
the O
binding O
of O
U2AF B
to O
the O
enhancer O
- O
located O
pyrimidine O
tract O
. O

VLA B
- I
4 I
integrin I
cross O
- O
linking O
on O
human O
monocytic O
THP O
- O
1 O
cells O
induces O
tissue B
factor I
expression O
by O
a O
mechanism O
involving O
mitogen B
- I
activated I
protein I
kinase I
. O

However O
, O
studies O
using O
the O
A20 B
promoter I
demonstrated O
that O
LMP1 B
transcriptionally O
activates O
the O
A20 B
gene I
through O
cis O
- O
acting O
kappa B
B I
sites I
. O

Southern O
blot O
analysis O
revealed O
that O
CCaMK B
is O
encoded O
by O
a O
single O
gene O
. O

Surgical O
methods O
in O
the O
treatment O
of O
rhinophyma O
. O

Phenazopyridine O
- O
induced O
hemolytic O
anemia O
in O
a O
patient O
with O
G6PD B
deficiency O
. O

Finally O
, O
we O
demonstrated O
that O
although O
the O
DNA O
- O
binding O
domain O
of O
c B
- I
myb I
is O
required O
for O
both O
the O
differentiation O
block O
and O
the O
shift O
in O
cell O
cycle O
after O
PMA O
treatment O
, O
phosphorylation O
by O
casein B
kinase I
II I
and O
mitogen B
- I
activated I
protein I
kinase I
at O
positions O
11 O
and O
12 O
or O
532 O
of O
c B
- I
myb I
, O
respectively O
, O
are O
not O
. O

The O
activation O
of O
NF B
- I
kappaB I
- O
dependent O
reporter O
gene O
transcription O
by O
E1A B
was O
potently O
suppressed O
upon O
coexpression O
of O
the O
E1B B
19 I
- I
kDa I
protein I
( O
19K O
) O
. O

Our O
results O
showed O
that O
direct O
enantiomeric O
separation O
of O
mephenytoin O
was O
obtained O
by O
using O
a O
chiral O
capillary O
column O
, O
the O
retention O
times O
for O
S O
- O
and O
R O
- O
mephenytoin O
were O
25 O
. O
5 O
and O
26 O
. O
2 O
min O
respectively O
, O
with O
a O
detection O
limit O
less O
than O
50 O
ng O
/ O
ml O
of O
mephenytoin O
. O

The O
myeloid B
zinc I
finger I
gene I
1 I
( O
MZF1 B
) O
encodes O
a O
C B
( I
2 I
) I
H I
( I
2 I
) I
zinc I
finger I
transcription I
factor I
that O
regulates O
granulopoiesis O
and O
may O
have O
a O
regulatory O
role O
in O
cellular O
proliferation O
and O
oncogenesis O
. O

Snails O
infected O
with O
three O
or O
five O
miracidia O
produced O
more O
metacercariae O
than O
those O
infected O
with O
a O
single O
miracidium O
. O

We O
conclude O
that O
GRF1 B
and O
GRF2 B
can O
form O
homo O
- O
and O
hetero O
- O
oligomers O
via O
their O
DH O
domains O
, O
that O
mutational O
inactivation O
of O
oligomer O
formation O
by O
GRF1 B
is O
associated O
with O
impaired O
biological O
and O
signaling O
activities O
, O
and O
that O
in O
293T O
cells O
GRF1 B
mediates O
at O
least O
two O
pathways O
for O
Raf B
activation O
: O
one O
a O
constitutive O
signal O
that O
is O
mainly O
Ras B
- O
dependent O
, O
and O
one O
an O
ionomycin O
- O
induced O
signal O
that O
cooperates O
with O
the O
constitutive O
signal O
without O
further O
augmenting O
the O
level O
of O
GTP B
- I
Ras I

. O

For O
the O
target O
stimuli O
, O
P3 O
amplitude O
and O
latency O
were O
negatively O
correlated O
and O
most O
tightly O
coupled O
over O
the O
frontal O
- O
central O
and O
right O
medial O
/ O
lateral O
recording O
sites O
. O

Mitoxantrone O
plus O
ara O
- O
C O
is O
an O
active O
combination O
with O
great O
promise O
for O
the O
therapy O
of O
previously O
untreated O
patients O
with O
AML O
. O

In O
the O
renal O
cortex O
, O
spleen O
, O
distal O
colon O
, O
ileum O
, O
gallbladder O
, O
and O
stomach O
body O
, O
regional O
O2 O
delivery O
was O
significantly O
decreased O
with O
hemodilution O
to O
20 O
% O
in O
both O
groups O
. O

( O
A O
short O
account O
of O
Karl O
Touton O
' O
s O
life O
is O
also O
included O
. O
) O
After O
reviewing O
the O
historical O
development O
of O
the O
concept O
of O
giant O
cells O
and O
current O
interpretations O
of O
their O
nature O
, O
the O
unitarian O
view O
of O
polykaryons O
now O
favored O
by O
workers O
in O
the O
field O
is O
extended O
to O
include O
also O
the O
" O
xanthelasmatic O
giant O
cell O
" O
of O
Touton O
, O
whose O
characteristic O
appearance O
is O
determined O
merely O
by O
the O
presence O
of O
demonstrable O
lipid O
in O
the O
cytoplasm O
. O

Chronic O
active O
hepatitis O
and O
pregnancy O
. O

Influenza O
virus O
- O
induced O
NF B
- I
kappaB I
- O
dependent O
gene O
expression O
is O
mediated O
by O
overexpression O
of O
viral O
proteins O
and O
involves O
oxidative O
radicals O
and O
activation O
of O
IkappaB B
kinase I
. O

These O
observed O
changes O
were O
not O
found O
when O
assessing O
NPY B
level O
in O
plasma O
fluid O
. O

Reversible O
topology O
of O
a O
bifunctional O
transmembrane O
protein O
depends O
upon O
the O
charge O
balance O
around O
its O
transmembrane O
domain O
. O

Protein B
kinase I
C I
( O
PKC B
) O
activation O
after O
treatment O
of O
human O
neuroblastoma O
SK O
- O
N O
- O
BE O
( O
2 O
) O
C O
cells O
with O
phorbol O
12 O
- O
myristate O
13 O
- O
acetate O
( O
PMA O
) O
was O
found O
to O
enhance O
transcription O
of O
the O
human B
dopamine I
beta I
- I
hydroxylase I
( O
DBH B
) O
in O
those O
cells O
. O

The O
second O
( O
control O
) O
group O
was O
represented O
by O
cases O
diagnosed O
as O
HSIL O
by O
cytology O
. O

The O
illuminator O
uses O
a O
grain O
- O
of O
- O
wheat O
light O
bulb O
and O
an O
adjustable O
bulb O
holder O
fashioned O
from O
bent O
paper O
clips O
. O

Bedbug O
( O
Cimex O
lectularius O
) O
samples O
adult O
and O
nymphs O
either O
engorged O
or O
starved O
from O
Central O
Security O
Forces O
sleeping O
wards O
, O
laboratory O
animal O
house O
and O
control O
samples O
from O
laboratory O
reared O
colonies O
were O
ground O
and O
subjected O
to O
ELISA O
test O
of O
hepatitis B
B I
surface I
antigen I
together O
with O
276 O
serum O
samples O
from O
the O
recruits O
slept O
in O
those O
wards O
. O

The O
genomic O
organization O
in O
this O
region O
is O
similar O
to O
the O
neural O
- O
restricted O
family O
members O
, O
Hel B
- I
N1 I
( O
ELAVL2 B
) O
and O
mHuD B
( O
Elavl4 B
) O
. O

Synergistic O
induction O
of O
apoptosis O
in O
human O
leukemia O
cells O
( O
U937 O
) O
exposed O
to O
bryostatin B
1 I
and O
the O
proteasome O
inhibitor O
lactacystin O
involves O
dysregulation O
of O
the O
PKC B
/ O
MAPK B
cascade O
. O

With O
the O
help O
of O
partially O
overlapping O
parenchymatous O
sutures O
, O
different O
renal O
segments O
were O
devitalized O
. O

Promoter O
elements O
and O
transcriptional O
control O
of O
the O
mouse B
acetylcholinesterase I
gene I
. O

The O
average O
cumulative O
UVA O
dose O
was O
65 O
J O
/ O
cm2 O
; O
40 O
patients O
had O
received O
more O
than O
200 O
J O
/ O
cm2 O
. O

Malformations O
of O
the O
regenerating O
optic O
tectum O
of O
larvae O
of O
the O
Egyptian O
toad O
, O
Bufo O
regularis O
Reuss O
. O

These O
morphological O
changes O
are O
associated O
with O
a O
marked O
induction O
of O
P450arom B
gene I
expression O
. O

The O
immunoprofile O
of O
60 O
adult O
patients O
of O
pulmonary O
tuberculosis O
was O
studied O
and O
compared O
with O
22 O
normal O
controls O
. O

Central O
- O
venous O
- O
catheter O
- O
related O
infection O
is O
readily O
diagnosed O
by O
comparing O
simultaneous O
quantitative O
cultures O
of O
blood O
samples O
obtained O
via O
the O
catheter O
and O
a O
peripheral O
vein O
. O

The O
role O
of O
haptoglobin B
in O
asphyxia O
mechanism O
( O
II O
) O
- O
- O
Acute O
drowning O
and O
hanging O

Processing O
MES O
data O
requires O
large O
bit O
manipulations O
and O
heavy O
memory O
storage O
requirements O
. O

Thus O
, O
BCR B
ligation O
initiates O
a O
PI B
3 I
- I
kinase I
/ O
Akt B
/ O
GSK B
- I
3 I
signaling O
pathway O
. O

cDNA O
cloning O
and O
transcriptional O
properties O
of O
a O
novel O
GC B
box I
- I
binding I
protein I
, O
BTEB2 B
. O

This O
activity O
creates O
a O
4 O
- O
bp O
staggered O
cut O
with O
3 O
' O
OH O
overhangs O
within O
a O
specific O
sequence O
of O
the O
21S B
rRNA I
gene I
of O
omega O
- O
strains O
. O

The O
two O
ORFs O
had O
significant O
similarity O
with O
the O
putative O
replication O
protein O
from O
plasmid O
pTiK12 O
of O
Thiobacillus O
intermedius O
and O
other O
CoIE2 O
- O
related O
plasmids O
. O

In O
contrast O
SF B
, O
but O
not O
GM B
- I
CSF I
or O
IL B
- I
3 I
, O
induced O
tyrosine O
phosphorylation O
of O
phospholipase B
C I
- I
gamma I
( O
PLC B
- I
gamma I
) O
. O

Phase O
I O
clinical O
trial O
with O
a O
combination O
of O
methotrexate O
and O
mitomycin O
C O
. O

Replacement O
of O
Val20 O
- O
Leu21 O
with O
Ala O
- O
Ala O
produced O
a O
dimeric O
RII B
beta I
protein I
that O
binds O
AKAP75 B
approximately O
4 O
% O
as O
avidly O
as O
wild B
- I
type I
RII I
beta I
. O

The O
temporal O
expression O
of O
CRAlBP B
is O
similar O
to O
IRBP B
while O
bFGF B
is O
not O
expressed O
until O
after O
photoreceptor O
differentiation O
is O
complete O
. O

I O
. O

In O
T O
cells O
these O
two O
signal O
pathways O
are O
critical O
for O
interleukin B
- I
2 I
production O
. O

Replacement O
of O
a O
valine O
( O
V68 O
) O
in O
the O
ETS B
domain I
of O
SAP1a B
by O
aspartic O
acid O
( O
as O
found O
in O
c B
- I
Ets I
- I
1 I
, O
Elk B
- I
1 I
, O
and O
Net B
) O
enhanced O
ternary O
complex O
formation O
by O
more O
than O
60 O
- O
fold O
. O

In O
order O
to O
investigate O
the O
effect O
of O
the O
local O
direct O
action O
of O
danazol O
upon O
endometriosis O
, O
intravaginal O
and O
intrauterine O
application O
were O
tried O
. O

The O
NF B
- I
kappaB I
/ O
Rel B
transcription O
factors O
participate O
in O
the O
activation O
of O
immune O
system O
regulatory O
genes O
and O
viral B
early I
genes I
including O
the O
human B
immunodeficiency I
virus I
type I
1 I
long I
terminal I
repeat I
. O

A O
total O
of O
4 O
, O
916 O
children O
were O
examined O
and O
the O
questionnaire O
satisfactorily O
completed O
by O
a O
relative O
or O
guardian O
( O
usually O
the O
mother O
) O
of O
4 O
, O
651 O
. O

cDNA O
cloning O
reveals O
two O
mouse B
beta5 I
integrin I
transcripts I
distinct O
in O
cytoplasmic O
domains O
as O
a O
result O
of O
alternative O
splicing O
. O

Earlier O
nonoptical O
methods O
probably O
did O
not O
include O
the O
portion O
of O
the O
film O
that O
contained O
mucus O
. O

OBJECTIVE O
: O
To O
summarise O
quantitatively O
the O
association O
between O
moderate O
alcohol O
intake O
and O
biological O
markers O
of O
risk O
of O
coronary O
heart O
disease O
and O
to O
predict O
how O
these O
changes O
would O
lower O
the O
risk O
. O

Like O
other O
known O
GCD B
genes I
, O
GCD14 B
and O
GCD15 B
are O
therefore O
probably O
required O
for O
general O
translation O
initiation O
in O
addition O
to O
their O
roles O
in O
GCN4 B
- O
specific O
translational O
control O
. O

The O
presence O
of O
highly O
correlated O
frequencies O
provided O
strong O
evidence O
of O
a O
medium O
- O
frequency O
pattern O
generator O
which O
may O
remain O
operative O
beyond O
the O
neonatal O
period O
. O

This O
gene O
is O
named O
PKNOX1 B
by O
the O
Human O
Nomenclature O
Committee O
. O

Differentiation O
of O
porcine O
mycoplasma O
strains O
by O
means O
of O
a O
simple O
paper O
chromatography O
test O
. O

HIV O
- O
2 O
in O
Spain O
. O

No O
effect O
of O
4 O
beta O
- O
phorbol O
12 O
- O
myristate O
13 O
- O
acetate O
was O
observed O
on O
the O
expression O
of O
hnRNP B
H I
. O

Patients O
commonly O
considered O
the O
external O
frame O
cumbersome O
. O

D O
. O

The O
atf1 B
+ I
gene I
of O
Schizosaccharomyces O
pombe O
encodes O
a O
bZIP B
transcription I
factor I
with O
strong O
homology O
to O
the O
mammalian B
factor I
ATF I
- I
2 I
. O

The O
effects O
of O
different O
sources O
of O
commercial O
rations O
on O
the O
humoral O
immune O
response O
of O
broilers O
to O
Newcastle O
disease O
vaccination O
. O

Multiple O
attempts O
aimed O
at O
disruption O
of O
the O
chromosomal B
fabG I
gene I
were O
unsuccessful O
. O

Primary O
TJRs O
were O
clinically O
evaluated O
for O
degree O
of O
osteoarthrosis O
. O

Radiation O
sensitivity O
of O
rats O
irradiated O
in O
a O
condition O
of O
hypoxia O

The O
activity O
of O
plasminogen B
activator I
has O
been O
measured O
in O
tissue O
sections O
with O
the O
aid O
of O
a O
modified O
Todd O
technique O
. O

The O
complexity O
of O
some O
of O
the O
reports O
of O
VACURG O
studies O
has O
led O
some O
to O
believe O
that O
this O
group O
was O
opposed O
to O
estrogens O
, O
but O
that O
is O
not O
true O
. O

Thus O
, O
the O
mutants O
are O
deficient O
in O
production O
of O
the O
O B
- I
antigen I
polymerase I
and O
were O
termed O
rfc B
mutants I
. O

Up O
to O
1988 O
, O
145 O
cases O
had O
been O
reported O
in O
literature O
and O
32 O
cases O
in O
our O
country O
. O

Focal O
glomerulosclerosis O
- O
like O
disease O
with O
nephrotic O
syndrome O
in O
a O
horse O
. O

The O
antihypertensive O
action O
corresponds O
to O
those O
of O
established O
diuretics O
. O

Interconversions O
between O
haloperidol O
and O
reduced O
haloperidol O
in O
schizophrenic O
patients O
and O
guinea O
pigs O
: O
a O
steady O
- O
state O
study O
. O

The O
frequency O
of O
eating O
dark O
- O
meat O
fish O
was O
positively O
associated O
with O
serum O
eicosapentaenoic O
acid O
and O
docosahexaenoic O
acid O
, O
omega3 O
fatty O
acids O
, O
and O
inversely O
associated O
with O
serum O
stearic O
acid O
and O
linoleic O
acid O
. O

Effect O
of O
food O
on O
hemodynamics O
. O

The O
D443N O
substitution O
is O
an O
activating O
mutation O
that O
increases O
the O
activity O
of O
SCAP B
and O
renders O
it O
resistant O
to O
inhibition O
by O
25 O
- O
hydroxycholesterol O
. O

Central O
( O
heart O
rate O
, O
VE O
, O
VO2 O
) O
or O
local O
( O
muscle O
and O
blood O
lactate O
, O
adenosine O
triphosphate O
, O
creatine B
phosphokinase I
, O
glycogen O
) O
cues O
highlighted O
in O
these O
studies O
demonstrate O
both O
the O
complexity O
of O
effort O
perception O
, O
and O
the O
need O
for O
better O
understanding O
of O
the O
physiological O
components O
upon O
which O
it O
is O
based O
. O

The O
gene O
is O
predicted O
to O
encode O
an O
880 O
- O
amino O
- O
acid O
protein O
which O
contains O
two O
C2H2 O
zinc O
fingers O
, O
a O
nuclear O
localization O
sequence O
and O
two O
transcriptional O
activation O
domains O
. O

This O
approach O
allowed O
examination O
of O
baseline O
factors O
as O
well O
as O
the O
effect O
of O
change O
in O
mileage O
, O
the O
occurrence O
of O
a O
musculoskeletal O
problem O
, O
or O
the O
occurrence O
of O
another O
health O
problem O
on O
the O
rate O
of O
dropout O
. O

Microalbuminuria O
: O
a O
marker O
of O
systemic O
disease O
. O

Regulation O
of O
activating B
transcription I
factor I
- I
1 I
and O
the O
cAMP B
response I
element I
- I
binding I
protein I
by O
Ca2 B
+ I
/ I
calmodulin I
- I
dependent I
protein I
kinases I
type I
I I
, I
II I
, I
and I
IV I
. O

Coronary O
arteries O
were O
found O
in O
the O
biopsy O
tissue O
of O
30 O
( O
76 O
% O
) O
of O
the O
39 O
patients O
who O
formed O
the O
study O
group O
. O

Serum B
amyloid I
A I
( O
SAA B
) O
is O
a O
plasma O
protein O
which O
has O
been O
associated O
with O
several O
diseases O
, O
including O
amyloidosis O
, O
arthritis O
, O
and O
atherosclerosis O
, O
and O
its O
abnormal O
expression O
, O
particularly O
in O
nonhepatic O
cells O
, O
is O
implicated O
in O
the O
pathogenesis O
of O
these O
diseases O
. O

Phenacetin O
nephritis O

We O
have O
been O
developing O
a O
digital O
fluoroscopic O
imaging O
system O
to O
replace O
the O
portal O
films O
that O
are O
currently O
used O
to O
verify O
patient O
positioning O
during O
radiotherapy O
treatments O
. O

The O
human B
p18 I
gene I
spans O
at O
least O
7 O
. O
5 O
kb O
and O
is O
composed O
of O
three O
exons O
, O
two O
of O
which O
encode O
the O
p18 B
protein I
. O

Serum O
creatinine B
kinase I
activity O
was O
7 O
, O
800 O
- O
17 O
, O
500 O
U O
/ O
l O
, O
making O
acute O
muscle O
damage O
likely O
, O
probably O
rhabdomyolysis O
. O

These O
results O
suggest O
that O
termination O
is O
a O
highly O
specific O
event O
, O
and O
not O
merely O
a O
consequence O
of O
decreased O
stability O
of O
the O
EC O
. O

Like O
Mdm2 B
, O
MdmX B
is O
able O
to O
bind O
p53 B
and O
inhibit O
p53 B
transactivation O
; O
however O
, O
the O
ability O
of O
MdmX B
to O
degrade O
p53 B
has O
yet O
to O
be O
examined O
. O

PURPOSE O
: O
Second O
malignant O
neoplasms O
( O
SMNs O
) O
are O
a O
rare O
occurrence O
after O
the O
successful O
treatment O
of O
childhood O
cancer O
. O

Recent O
geological O
studies O
focusing O
on O
the O
Davie O
Fracture O
Zone O
of O
the O
Mozambique O
Channel O
offer O
a O
resolution O
to O
this O
situation O
, O
by O
suggesting O
the O
presence O
of O
a O
land O
- O
bridge O
from O
the O
mid O
- O
Eocene O
to O
the O
early O
Miocene O
, O
an O
interval O
that O
matches O
the O
ages O
of O
Madagascar O
' O
s O
mammalian O
groups O
. O

The O
nucleotide O
sequence O
of O
a O
PCR O
- O
amplified O
SMT3A B
genomic I
DNA I
fragment I
was O
found O
to O
be O
identical O
to O
that O
of O
SMT3A B
cDNA I
, O
indicating O
the O
absence O
of O
intron O
( O
s O
) O
in O
its O
protein O
coding O
region O
. O

Mutant B
p53 I
DNA O
clones O
from O
human O
colon O
carcinomas O
cooperate O
with O
ras B
in O
transforming O
primary O
rat O
cells O
: O
a O
comparison O
of O
the O
" O
hot O
spot O
" O
mutant O
phenotypes O
. O

Gas O
chromatographic O
analysis O
of O
headspace O
samples O
of O
alkalinized O
vaginal O
discharges O
indicated O
the O
presence O
of O
trimethylamine O
in O
all O
11 O
samples O
with O
the O
fishy O
odor O
but O
not O
in O
the O
other O
samples O
. O

Standing O
values O
: O
placebo O
114 O
/ O
79 O
; O
BHT O
- O
933 O
92 O
/ O
67 O
. O

The O
Ixodes O
fauna O
in O
the O
Michourin O
region O
, O
where O
the O
pastures O
in O
1971 O
were O
grazed O
only O
by O
sheep O
, O
shows O
the O
following O
trends O
: O
H O
. O
punctata O
and O
R O
. O
turanicus O
ticks O
, O
which O
are O
preferably O
parasitising O
in O
sheep O
, O
increase O
their O
relative O
share O
. O

The O
SEC27 B
gene I
was O
cloned O
, O
and O
the O
sequence O
predicts O
a O
99 O
. O
4 O
- O
kDa O
protein O
with O
45 O
% O
sequence O
identity O
to O
mammalian B
beta I
' I
- I
COP I
. O

Studies O
of O
fetal O
maturity O

Fetal O
arterial O
and O
sagittal O
sinus O
pH O
, O
base O
excess O
, O
po2 O
, O
and O
oxygen O
saturation O
decreased O
, O
and O
hydrogen O
ion O
concentrations O
and O
pco2 O
increased O
during O
asphyxia O
alone O
and O
asphyxia O
plus O
isoflurane O
- O
oxygen O
. O

Therefore O
, O
it O
appears O
that O
GCN2 B
protein I
kinase I
function O
is O
stimulated O
posttranslationally O
in O
amino O
acid O
- O
starved O
cells O
. O

Although O
paclitaxel O
alone O
failed O
to O
induce O
p38 B
MAPK I
activation O
, O
subsequent O
( O
but O
not O
prior O
) O
exposure O
to O
PD98059 O
induced O
a O
dramatic O
increase O
in O
p38 B
MAPK I
phosphorylation O
. O

In O
3 O
of O
the O
5 O
patients O
with O
AVNRT O
and O
AJR O
, O
junctional O
beats O
served O
as O
a O
trigger O
for O
reentry O
. O

The O
three O
respective O
time O
control O
groups O
( O
C5 O
, O
C10 O
, O
and O
C15 O
) O
were O
injected O
with O
saline O
solution O
. O

Sequence O
analysis O
of O
cDNA O
clones O
encoding O
human O
gamma O
revealed O
an O
open O
reading O
frame O
predicting O
a O
protein O
of O
299 O
amino O
acids O
( O
approximately O
32 O
kDa O
) O
, O
half O
the O
size O
of O
the O
bovine O
gamma O
subunit O
. O

A O
positive O
FTA B
- I
ABS I
- O
19S B
- I
IgM I
test O
indicates O
the O
necessity O
of O
treatment O
. O

Our O
data O
suggest O
that O
some O
defects O
observed O
in O
BS O
, O
WS O
or O
RTS O
are O
the O
consequence O
of O
unrestrained O
recombination O
. O

The O
promoter O
for O
HMG B
- I
CoA I
synthase I
contains O
two O
binding O
sites O
for O
the O
sterol B
regulatory I
element I
- I
binding I
proteins I
( O
SREBPs B
) O
. O

These O
data O
indicate O
that O
chromatic O
processing O
in O
areas O
V1 O
and O
V2 O
is O
normal O
after O
V4 O
lesions O
and O
, O
together O
with O
the O
behavioural O
evidence O
, O
that O
these O
areas O
are O
sufficient O
for O
some O
basic O
aspects O
of O
colour O
perception O
. O

The O
curse O
of O
the O
Pharaohs O
. O

Covariates O
were O
lower O
extremity O
range O
of O
motion O
and O
sensory O
status O
. O

Characterization O
and O
targeted O
disruption O
of O
murine B
Nup50 I
, O
a O
p27 B
( O
Kip1 B
) O
- O
interacting O
component O
of O
the O
nuclear O
pore O
complex O
. O

p27 B
( O
Kip1 B
) O
is O
a O
member O
of O
the O
Cip B
- O
Kip B
family O
of O
cyclin B
- I
dependent I
kinase I
( O
Cdk B
) O
inhibitors O
that O
binds O
to O
cyclin B
- O
Cdk B
complexes O
and O
inhibits O
their O
catalytic O
activity O
in O
response O
to O
antiproliferative O
stimuli O
. O

p27 B
( O
Kip1 B
) O
is O
regulated O
by O
several O
posttranscriptional O
mechanisms O
, O
including O
subcellular O
localization O
. O

Therefore O
, O
the O
Ogg1 B
protein I
is O
a O
eukaryotic O
DNA B
glycosylase I
/ I
AP I
lyase I
. O

One O
patient O
had O
chronic O
dysphoria O
and O
one O
encephalopatia O
toxica O
. O

Alveoli O
- O
capillary O
diffusion O
. O

Mutations O
of O
SIN4 B
, O
ROX3 B
, O
SRB8 B
, O
SRB9 B
, O
SRB10 B
, O
SRB11 B
, O
and O
two O
novel O
genes O
, O
NUT1 B
and O
NUT2 B
, O
relieve O
the O
requirement O
of O
Swi4p B
for O
expression O
of O
this O
reporter O
. O

The O
software O
is O
in O
BASIC O
adapted O
for O
the O
Hewlett O
- O
Packard O
calculators O
HP O
- O
9845B O
and O
HP O
- O
85A O
. O

For O
Thermus O
strain O
ZO5 I
dihydroorotase B
( O
DHOase B
; O
encoded O
by O
pyrC B
) O
, O
the O
highest O
similarity O
scores O
( O
about O
40 O
% O
identity O
) O
were O
obtained O
with O
DHOases B
from O
B O
. O
caldolyticus O
and O
Bacillus O
subtilis O
. O

Precision O
of O
data O
from O
models O
of O
sodium O
kinetics O
in O
hemodialysis O

These O
data O
suggest O
that O
the O
fibrinogen B
gamma I
chain I
region I
Gly190 I
- I
Val202 I
functions O
as O
a O
minimal O
recognition O
sequence O
for O
the O
leukocyte O
integrin O
CD11b B
/ O
CD18 B
. O

Attenuated O
serum O
cortisol O
responses O
were O
found O
in O
six O
of O
the O
patients O
despite O
a O
normal O
ACTH B
test O
. O

We O
recently O
described O
the O
isolation O
and O
characterization O
of O
nontoxic O
PAP B
mutants I
, O
NT123 O
- O
2 O
, O
which O
has O
a O
point O
mutation O
( O
E176V O
) O
in O
the O
active O
site O
that O
abolishes O
enzymatic O
activity O
, O
and O
NT124 O
- O
3 O
, O
which O
has O
a O
nonsense O
mutation O
that O
results O
in O
deletion O
of O
the O
C O
- O
terminal O
25 O
aa O
( O
W237Stop O
) O
. O

Toxicity O
of O
benzoyl O
chloride O
( O
2 O
, O
4 O
, O
6 O
- O
trichlorophenyl O
) O
hydrazone O
( O
Banamite O
) O
and O
potential O
metabolites O
to O
twospotted O
spider O
mites O
and O
potency O
as O
inhibitors O
of O
rat B
liver I
monoamine I
oxidase I
. O

The O
screen O
identifies O
mutants O
whose O
growth O
depends O
on O
high O
levels O
of O
expression O
of O
that O
gene O
. O

The O
genes O
expressed O
during O
the O
embryonic O
( O
zeta B
) O
or O
fetal O
and O
adult O
stage O
( O
alpha B
2 I
and O
alpha B
1 I
) O
can O
be O
modified O
by O
point O
mutations O
which O
affect O
either O
the O
processing O
- O
translation O
of O
mRNA O
or O
make O
the O
polypeptide O
chains O
extremely O
unstable O
. O

In O
a O
study O
in O
Singapore O
, O
Chinese O
, O
Malay O
and O
Indian O
patients O
with O
pulmonary O
tuberculosis O
received O
2 O
months O
of O
daily O
treatment O
with O
streptomycin O
, O
isoniazid O
, O
rifampicin O
, O
and O
pyrazinamide O
followed O
by O
daily O
isoniazid O
, O
and O
rifampicin O
either O
with O
pyrazinamide O
( O
SHRZ O
/ O
HRZ O
) O
or O
without O
it O
( O
SHRZ O
/ O
HR O
) O
, O
allocated O
at O
random O
. O

Kaposi O
' O
s O
sarcoma O
- O
associated O
herpesvirus B
/ I
human I
herpesvirus I
- I
8 I
ORF50 I
gene I
product I
contains O
a O
potent O
C O
- O
terminal O
activation O
domain O
which O
activates O
gene O
expression O
via O
a O
specific O
target O
sequence O
. O

NSAIDS O
and O
the O
microcirculation O
of O
the O
stomach O
. O

The O
efficiency O
of O
transfections O
was O
normalized O
relative O
to O
the O
net O
amount O
of O
CAT B
plasmid O
actually O
transfected O
into O
recipient O
cells O
, O
determined O
by O
a O
modified O
Southern O
hybridization O
procedure O
. O

Genomic O
Southern O
blot O
analysis O
suggests O
that O
the O
human O
haploid O
genome O
contains O
a O
single O
Gs B
alpha I
gene I
. O

Evaluation O
of O
the O
effects O
of O
chelation O
therapy O
with O
time O
following O
strontium O
exposure O
to O
mice O
. O

In O
the O
present O
study O
we O
found O
that O
CD34 B
downregulation O
during O
hematopoiesis O
occured O
at O
the O
level O
of O
transcriptional O
initiation O
. O

Allergen O
- O
stimulated O
levels O
of O
PGD2 O
were O
1274 O
+ O
/ O
- O
565 O
versus O
1468 O
+ O
/ O
- O
679 O
after O
prednisone O
; O
likewise O
, O
allergen O
- O
stimulated O
5 O
- O
HETE O
levels O
were O
95 O
+ O
/ O
- O
21 O
versus O
82 O
+ O
/ O
- O
21 O
; O
those O
of O
LTE4 O
were O
54 O
+ O
/ O
- O
20 O
versus O
91 O
+ O
/ O
- O
51 O
; O
and O
those O
of O
15 O
- O
HETE O
were O
63 O
+ O
/ O
- O
19 O
versus O
60 O
+ O
/ O
- O
25 O
. O

Foreign O
DNA O
inserts O
contained O
within O
the O
fowlpox O
virus O
sequence O
were O
transferred O
to O
the O
viral O
genome O
by O
homologous O
recombination O
occurring O
in O
cells O
infected O
with O
a O
fowlpox O
virus O
temperature O
- O
sensitive O
mutant O
and O
transfected O
with O
both O
wild O
- O
type O
viral O
DNA O
and O
plasmid O
DNA O
. O

Although O
there O
is O
evidence O
that O
cone O
ratios O
do O
determine O
colour O
appearance O
under O
many O
conditions O
, O
the O
site O
or O
sites O
of O
their O
computation O
is O
unknown O
. O

Furthermore O
, O
we O
demonstrate O
that O
in O
myeloid O
and O
B O
cell O
extracts O
, O
PU B
. I
1 I
forms O
a O
novel O
, O
specific O
, O
more O
slowly O
migrating O
complex O
( O
PU B
- I
SF I
) O
when O
binding O
the O
GM B
- I
CSF I
receptor I
alpha I
promoter I
PU B
. I
1 I
site I
. O

Several O
components O
in O
cytokine O
signaling O
remain O
unidentified O
. O

This O
is O
the O
first O
report O
demonstrating O
specificity O
at O
the O
level O
of O
the O
energy O
coupling O
proteins O
of O
the O
PTS O
. O

In O
contrast O
, O
stem O
- O
loop O
structures O
2 O
, O
3 O
, O
and O
4 O
of O
the O
3 O
' O
domain O
showed O
great O
variations O
in O
size O
, O
sequence O
, O
and O
structure O
. O

A O
spectrophotometric O
method O
for O
the O
microdetermination O
of O
periodate O
. O

Result O
were O
as O
followed O
: O
1 O
) O
Early O
cancer O
- O
like O
advanced O
cancers O
were O
consisted O
of O
133 O
lesions O
, O
of O
which O
128 O
lesions O
( O
96 O
. O
2 O
% O
) O
had O
peptic O
ulcer O
in O
cancerous O
lesion O
( O
73 O
lesions O
were O
active O
stage O
and O
55 O
lesions O
were O
scarring O
stage O
) O
. O

Effect O
of O
hyperaemia O
on O
thallium O
- O
201 O
redistribution O
in O
normal O
canine O
myocardium O
. O

Immunoblotting O
showed O
that O
both O
the O
( O
1 O
- O
404 O
) O
- O
peptide O
and O
( O
1 O
- O
506 O
) O
- O
peptide O
are O
recognized O
by O
95J10 O
, O
a O
monoclonal O
antibody O
that O
was O
previously O
raised O
against O
perlecan B
- I
( I
24 I
- I
404 I
) I
- I
peptide I
expressed O
in O
Escherichia O
coli O
. O

The O
anesthesiologist O
and O
the O
cardiac O
surgeon O
. O

To O
gain O
further O
insights O
into O
these O
events O
, O
the O
porcine B
G I
alpha I
i I
- I
3 I
gene I
minimal I
promoter I
was O
characterized O
and O
found O
67 O
base O
pairs O
upstream O
from O
the O
major O
transcription O
start O
site O
. O

FDP O
- O
1 O
( O
200 O
micrograms O
. O
kg O
- O
1 O
) O
of O
intracoronary O
injection O
increased O
collateral O
flow O
, O
reduced O
systemic O
artery O
resistance O
, O
and O
coronary O
systolic O
pressure O
in O
infarcted O
region O
and O
the O
area O
of O
infarction O
decreased O
from O
28 O
. O
9 O
+ O
/ O
- O
1 O
. O
3 O
% O
to O
15 O
. O
3 O
+ O
/ O
- O
1 O
. O
2 O
% O
( O
P O
< O
0 O
. O
01 O
, O
n O
= O
6 O
) O
. O

Pathophysiologic O
mechanisms O
underlying O
spatial O
disorientation O
in O
patients O
with O
Alzheimer O
' O
s O
disease O
. O

Many O
human O
malignant O
cells O
lack O
methylthioadenosine B
phosphorylase I
( O
MTAP B
) O
enzyme O
activity O
. O

Following O
40 O
min O
" O
ischemia O
" O
, O
preparations O
treated O
with O
PC O
recovered O
from O
transmural O
conduction O
block O
more O
rapidly O
( O
PC1 O
group O
, O
4 O
min O
, O
P O
less O
than O
0 O
. O
05 O
; O
PC2 O
group O
, O
23 O
min O
, O
ns O
) O
, O
compared O
to O
control O
. O
( O
ABSTRACT O
TRUNCATED O
AT O
250 O
WORDS O
) O

The O
novel O
gene O
spans O
16 O
. O
7 O
kb O
of O
genomic O
sequence O
and O
it O
is O
formed O
of O
11 O
exons O
and O
10 O
intervening O
introns O
. O

G B
- I
box I
binding I
factors I
( O
GBFs B
) O
constitute O
a O
family O
of O
plant O
DNA O
- O
binding O
proteins O
that O
bind O
to O
the O
G O
- O
box O
motif O
, O
a O
regulatory O
cis O
element O
present O
in O
many O
plant O
genes O
with O
a O
palindromic O
DNA O
motif O
of O
CACGTG O
. O

CONCLUSION O
: O
In O
an O
acute O
canine O
model O
of O
progressive O
LCX O
coronary O
stenosis O
, O
CFR O
was O
impaired O
in O
both O
ischemic O
and O
remote O
nonischemic O
regions O
in O
association O
with O
mild O
to O
moderate O
ischemic O
- O
induced O
regional O
myocardial O
dysfunction O
. O

A O
third O
- O
order O
polynominal O
fit O
showed O
a O
good O
correlation O
for O
the O
parameter O
Q O
* O
and O
MTT O
, O
whereas O
T O
and O
SImax O
were O
found O
to O
have O
a O
poor O
correlation O
. O

Late O
results O
in O
the O
treatment O
of O
urotuberculosis O

Changes O
in O
the O
frequency O
of O
X O
- O
chromatin O
in O
women O
under O
Prednisone O
therapy O

Other O
toxicities O
attributed O
to O
CPT O
- O
11 O
included O
dehydration O
, O
nausea O
, O
vomiting O
, O
and O
asthenia O
. O

Proceedings O
: O
Influence O
of O
halothane O
on O
mortality O
from O
murine O
hepatitis O
virus O
( O
MHV3 O
) O
. O

These O
results O
extend O
our O
knowledge O
of O
this O
SNF2 B
- I
like I
family I
member I
and O
suggest O
a O
role O
for O
PASG B
in O
leukemogenesis O
. O

The O
difference O
between O
the O
results O
in O
the O
high O
dose O
AHLG O
and O
in O
the O
control O
group O
was O
significant O
( O
p O
less O
than O
0 O
. O
05 O
) O
. O

Results O
of O
this O
study O
indicate O
that O
when O
amniotic O
fluid O
samples O
are O
stored O
frozen O
and O
strict O
quality O
control O
is O
maintained O
in O
analytic O
procedures O
, O
only O
minimal O
changes O
occur O
in O
phospholipid O
concentrations O
over O
12 O
months O
. O

Retinoic O
acid O
, O
which O
inhibits O
the O
progression O
of O
certain O
cancers O
, O
repressed O
v B
- I
src I
- O
induced O
MMP B
- I
1 I
transcription O
. O

The O
pattern O
of O
net O
RNA O
expression O
in O
adult O
mouse O
tissues O
is O
different O
. O

Immunoprecipitation O
of O
spinophilin B
or O
neurabin B
from O
crude O
brain O
extracts O
selectively O
coprecipitated O
PP1gamma B
( I
1 I
) I
over O
PP1beta B
. O

The O
authors O
discussed O
their O
experience O
in O
investigating O
20 O
patients O
with O
maxillary O
malignant O
tumors O
using O
routine O
x O
- O
ray O
studies O
and O
MR O
- O
tomography O
, O
and O
13 O
patients O
, O
investigated O
in O
the O
same O
way O
plus O
CT O
. O

Reduction O
of O
p53 B
protein I
was O
detected O
after O
1 O
day O
of O
OM O
treatment O
and O
reached O
maximal O
suppression O
of O
10 O
- O
20 O
% O
of O
control O
after O
3 O
days O
in O
H3922 O
and O
40 O
% O
of O
control O
after O
4 O
days O
in O
MCF O
- O
7 O
cells O
. O

Association O
of O
Syk B
protein B
tyrosine I
kinase I
( O
PTK B
) O
with O
LMP2A B
occurs O
at O
the O
two O
tyrosines O
of O
the O
LMP2A B
immunoreceptor I
tyrosine I
- I
based I
activation I
motif I
, O
and O
it O
is O
hypothesized O
that O
Lyn B
PTK B
associates O
with O
the O
YEEA O
amino O
acid O
motif O
at O
LMP2A B
tyrosine I
112 I
( O
Y112 O
) O
. O

The O
treatment O
with O
glucocorticoids O
and O
immunosuppressants O
resulted O
in O
a O
more O
noticeable O
decrease O
in O
the O
levels O
of O
T3 O
and O
E B
- I
RCF I
. O

Four O
of O
these O
fibroblast O
clones O
were O
infected O
with O
a O
retrovirus O
containing O
the O
cDNA B
encoding O
myoD B
and O
a O
puromycin O
selection O
marker O
. O

Serum O
estriol O
levels O
during O
beta B
- I
receptor I
agonist O
infusion O
in O
the O
third O
trimester O
of O
pregnancy O
. O

In O
this O
study O
, O
we O
investigated O
the O
mechanism O
by O
which O
Tax B
activates O
gene O
expression O
in O
conjunction O
with O
members O
of O
the O
ATF B
/ O
CREB B
family O
. O

Four O
of O
12 O
patients O
( O
33 O
% O
) O
were O
initially O
diagnosed O
as O
having O
a O
benign O
disease O
( O
false O
- O
negatives O
) O
. O

However O
, O
it O
was O
observed O
that O
storage O
time O
affected O
significantly O
the O
biochemical O
changes O
occurring O
in O
the O
constituents O
of O
carrots O
exposed O
to O
gamma O
- O
irradiation O
. O

Suppression O
of O
the O
cka1 B
delta I
cka2 B
- I
8 I
mutant I
phenotype O
occurs O
by O
interaction O
of O
CKB1 B
with O
the O
defective O
, O
cka2 B
- I
8 I
- I
encoded I
, I
catalytic I
subunit O
. O

The O
median O
cumulative O
incidence O
rates O
were O
found O
to O
be O
341 O
per O
million O
for O
males O
and O
296 O
per O
million O
for O
females O
. O

Thomas O
, O
S O
. O
S O
. O

Similarly O
, O
antisense O
TGF B
- I
beta I
type I
II I
receptor I
oligodeoxynucleotides O
( O
40 O
microM O
) O
resulted O
in O
a O
58 O
% O
increase O
in O
branching O
, O
compared O
to O
scrambled O
and O
mismatched O
sequence O
controls O
, O
while O
TGF B
- I
beta I
, O
type B
II I
receptor I
mRNA I
and O
its O
protein O
expression O
levels O
were O
suppressed O
by O
95 O
and O
84 O
% O
, O
respectively O
. O

Secretion O
of O
cuticle O
over O
and O
between O
the O
domed O
apical O
surfaces O
of O
these O
cells O
leads O
to O
a O
honeycomb O
- O
like O
structure O
and O
gives O
the O
superficial O
wart O
- O
like O
phenotype O
of O
mitotic O
clones O
on O
the O
adult O
. O

Taken O
together O
, O
these O
results O
indicate O
that O
ICSBP B
and O
PU B
. I
1 I
are O
critical O
elements O
for O
IL B
- I
18 I
gene I
expression O
. O

Based O
on O
these O
results O
, O
it O
has O
been O
concluded O
that O
an O
IPFSA O
can O
promote O
the O
healing O
process O
in O
the O
avascular O
zone O
of O
a O
torn O
meniscus O
in O
rabbits O
and O
that O
systemic O
vascularity O
to O
the O
synovium O
or O
the O
meniscus O
is O
not O
essential O
for O
healing O
to O
occur O
. O

NASA O
CR O
- O
1223 O
. O

The O
magnitude O
of O
all O
four O
parameters O
increased O
in O
an O
exponential O
fashion O
with O
increasing O
sperm O
number O
up O
to O
400 O
x O
10 O
( O
6 O
) O
per O
ejaculate O
. O

We O
studied O
about O
the O
histological O
findings O
of O
the O
kidneys O
including O
of O
the O
infiltrated O
macrophages O
in O
one O
hour O
post O
- O
transplantation O
kidney O
biopsies O
( O
one O
hour O
biopsies O
) O
and O
re O
- O
biopsies O
on O
11 O
patients O
. O

An O
additional O
stimulation O
of O
D1 B
receptors I
by O
giving O
SKF38393 O
30 O
min O
later O
produced O
an O
almost O
continuous O
pattern O
of O
jaw O
openings O
but O
less O
closure O
movements O
from O
the O
rest O
position O
, O
and O
the O
openings O
were O
accompanied O
by O
frequent O
tongue O
protrusions O
. O

( O
control O
hearts O
) O
. O

The O
( O
0 O
0 O
2 O
) O
diffraction O
of O
the O
synthesized O
HAp B
crystals O
on O
a O
Col O
fiber O
showed O
an O
around O
60 O
arching O
angle O
, O
while O
that O
on O
a O
ChS O
fiber O
showed O
just O
around O
10 O
degrees O
. O

During O
4 O
% O
O2 O
ventilation O
, O
baseline O
pulmonary O
vascular O
resistance O
( O
PVR O
) O
and O
the O
dilator O
response O
to O
both O
ACh O
and O
SNP O
were O
greater O
in O
2D O
lungs O
. O

To O
determine O
the O
role O
of O
the O
transmembrane O
region O
of O
HN B
on O
fusion O
- O
promoting O
activity O
, O
mutant B
HN I
proteins I
were O
expressed O
and O
their O
biological O
activities O
examined O
. O

To O
be O
a O
nurse O
. O

Transient O
transfection O
assays O
indicated O
that O
the O
( O
- O
4551 O
) O
UCP1 B
- O
CAT B
construct O
, O
containing O
the O
5 O
' O
- O
regulatory O
region O
of O
the O
rat B
ucp I
- I
1 I
gene I
, O
was O
activated O
by O
PPARalpha B
co O
- O
transfection O
in O
a O
dose O
- O
dependent O
manner O
and O
this O
activation O
was O
potentiated O
by O
Wy O
14 O
, O
643 O
and O
retinoid B
X I
receptor I
alpha I
. O

Synthesis O
of O
2 O
- O
( O
2 O
' O
- O
thiazolyl O
) O
alkanebenzimidazoles O
- O
- O
potential O
anthelmintics O

Human B
CR2 I
has O
a O
restricted O
cellular O
distribution O
, O
being O
expressed O
on O
B O
lymphocytes O
, O
dendritic O
cells O
of O
the O
spleen O
, O
pharyngeal O
epithelial O
cells O
, O
and O
at O
low O
levels O
on O
some O
T O
lymphocytes O
. O

ADR1 B
in O
extracts O
made O
from O
glucose O
- O
repressed O
and O
- O
derepressed O
cells O
bound O
UAS1 O
DNA O
with O
similar O
affinities O
despite O
having O
greatly O
different O
abilities O
to O
activate O
ADH2 B
gene I
expression O
in O
vivo O
. O

Furthermore O
, O
agonist O
action O
at O
a O
5 B
- I
HT1 I
receptor I
and O
antagonist O
action O
at O
5 B
- I
HT2 I
receptors I
both O
appear O
to O
contribute O
to O
antidepressant O
- O
like O
activity O
on O
the O
DRL O
72 O
- O
s O
schedule O
. O

MsEPV B
genes I
with O
putative O
functions O
in O
prevention O
and O
repair O
of O
DNA O
damage O
include O
a O
complete O
base O
excision O
repair O
pathway O
( O
uracil B
DNA I
glycosylase I
, O
AP B
endonuclease I
, O
DNA B
polymerase I
beta I
, O
and O
an O
NAD B
+ I
- I
dependent I
DNA I
ligase I
) O
, O
a O
photoreactivation O
repair O
pathway O
( O
cyclobutane B
pyrimidine I
dimer I
photolyase I
) O
, O
a O
LINE B
- I
type I
reverse I
transcriptase I
, O
and O
a O
mutT B
homologue I
. O

These O
three O
groups O
were O
further O
examined O
with O
regard O
to O
the O
modulation O
of O
discharge O
rate O
i O
) O
during O
motor O
activity O
in O
W O
, O
ii O
) O
at O
the O
transitional O
phase O
from O
SS O
to O
paradoxical O
sleep O
( O
PS O
) O
, O
iii O
) O
during O
rapid O
eye O
movements O
( O
REMs O
) O
in O
PS O
, O
and O
iv O
) O
following O
electrical O
stimulation O
of O
the O
midbrain O
reticular O
formation O
( O
MRF O
) O
. O

Likewise O
, O
the O
consensus O
signal O
believed O
to O
be O
involved O
in O
terminating O
VV B
early I
gene I
transcription O
, O
TTTTTNT O
, O
was O
evident O
at O
the O
3 O
' O
- O
boundary O
of O
both O
the O
N2 B
and O
M1 B
ORFs I
suggesting O
that O
these O
genes O
may O
be O
VV B
early I
genes I
. O

This O
radiolabeled O
, O
900 O
- O
bp O
amplicon O
was O
used O
as O
a O
hybridization O
probe O
to O
screen O
a O
cDNA O
library O
constructed O
from O
poly O
( O
A O
) O
( O
+ O
) O
RNA O
isolated O
from O
induced O
Taxus O
cells O
, O
from O
which O
a O
full B
- I
length I
transacetylase I
sequence I
was O
obtained O
. O

Low O
temperature O
( O
8 O
degrees O
C O
) O
impairs O
secretory O
activity O
, O
again O
independently O
of O
the O
photoperiod O
selected O
. O

Left O
ventricle O
to O
coronary O
sinus O
fistula O
. O

In O
this O
report O
we O
describe O
the O
structure O
and O
DNA O
sequence O
of O
transcriptional O
control O
regions O
from O
both O
human O
and O
Wistar B
- I
rat I
single I
copy I
EGF I
genes I
and O
their O
functional O
analysis O
in O
epithelial O
cell O
cultures O
. O

The O
magnesium O
content O
in O
leukocytes O
could O
not O
be O
estimated O
because O
of O
interaction O
between O
the O
ion O
and O
the O
Percoll O
( O
R O
) O
media O
. O

The O
sequence O
of O
three O
of O
them O
revealed O
pure O
CAG O
stretches O
. O

Furthermore O
, O
mutations O
in O
element O
II O
result O
in O
changes O
in O
the O
RNA O
structure O
near O
element O
III O
, O
consistent O
with O
a O
long O
- O
range O
interaction O
that O
may O
promote O
translation O
. O

Although O
the O
QT O
duration O
was O
higher O
( O
p O
= O
0 O
. O
0001 O
) O
at O
night O
, O
the O
beat O
- O
to O
- O
beat O
variability O
of O
this O
interval O
was O
lower O
: O
in O
the O
time O
domain O
( O
decreased O
standard O
deviation O
, O
p O
= O
0 O
. O
0005 O
) O
, O
in O
the O
frequency O
domain O
( O
decreased O
low O
- O
frequency O
power O
of O
the O
spectra O
, O
p O
= O
0 O
. O
004 O
) O
, O
and O
the O
chaotic O
domain O
( O
tighter O
clustering O
of O
the O
points O
in O
the O
Poincare O
plots O
) O
. O

The O
biological O
functions O
of O
rat B
surfactant I
protein I
A I
( O
SP B
- I
A I
) O
, O
an O
oligomer O
composed O
of O
18 O
polypeptide O
subunits O
derived O
from O
a O
single O
gene O
, O
are O
dependent O
on O
intact O
disulfide O
bonds O
. O

A O
V1 B
mutant I
generated O
in O
vivo O
with O
11 O
of O
the O
14 O
N O
- O
terminal O
amino O
acids O
altered O
, O
was O
viable O
and O
produced O
symptoms O
typical O
of O
a O
wild O
- O
type O
infection O
. O

This O
seems O
to O
take O
place O
in O
the O
same O
nuclear O
compartment O
as O
normal O
cis O
splicing O
and O
proceeds O
through O
Y O
- O
branched O
intermediates O
analogous O
to O
the O
lariats O
formed O
in O
cis O
splicing O
. O

We O
have O
demonstrated O
that O
the O
specific O
determination O
and O
identification O
of O
plasma O
FbDP O
alone O
is O
not O
sufficient O
to O
follow O
the O
effectiveness O
of O
thrombolytic O
therapy O
. O

Chronic O
, O
recurrent O
multifocal O
osteomyelitis O
. O

Maximum O
baroreceptor O
activity O
measured O
at O
140 O
mm O
Hg O
and O
the O
slope O
of O
the O
pressure O
- O
activity O
curve O
were O
reduced O
in O
atherosclerotic O
( O
n O
= O
15 O
) O
compared O
with O
normal O
( O
n O
= O
13 O
) O
rabbits O
( O
425 O
+ O
/ O
- O
34 O
versus O
721 O
+ O
/ O
- O
30 O
spikes O
per O
second O
and O
6 O
. O
2 O
+ O
/ O
- O
0 O
. O
6 O
versus O
10 O
. O
8 O
+ O
/ O
- O
0 O
. O
8 O
spikes O
per O
second O
per O
mm O
Hg O
, O
respectively O
, O
P O
< O
. O
05 O
) O
. O

A O
pathogenetic O
relation O
with O
the O
Eisenmenger O
' O
s O
syndrome O
is O
discussed O
. O

Coronary O
vasoconstriction O
caused O
by O
endothelin B
- I
1 I
is O
enhanced O
by O
ischemia O
- O
reperfusion O
and O
by O
norepinephrine O
present O
in O
concentrations O
typically O
observed O
after O
neonatal O
cardiopulmonary O
bypass O
. O

Located O
between O
the O
3 O
' O
trans O
- O
spliced O
leader O
acceptor O
site O
and O
the O
translational O
initiation O
codon O
of O
the O
GP63 B
gene I
is O
an O
area O
of O
conserved O
hexanucleotide O
direct O
repeats O
( O
CTCGCC O
) O
which O
vary O
in O
number O
according O
to O
species O
. O

An O
association O
was O
found O
between O
zafirlukast O
plasma O
concentrations O
and O
increases O
in O
PC8SRaw O
10 O
h O
after O
treatment O
( O
p O
= O
0 O
. O
001 O
) O
. O

Insulin B
- O
secreting O
islet O
- O
cell O
tumours O
of O
the O
pancreas O
. O

Disruption O
and O
reconstruction O
: O
narrative O
insights O
into O
the O
experience O
of O
family O
members O
caring O
for O
a O
relative O
diagnosed O
with O
serious O
mental O
illness O
. O

The O
results O
suggest O
that O
the O
posterior O
wall O
is O
more O
compliant O
than O
the O
anterior O
wall O
( O
that O
is O
, O
for O
a O
given O
difference O
in O
transmural O
pressure O
, O
the O
local O
segment O
length O
change O
of O
the O
posterior O
wall O
was O
greater O
) O
. O

We O
have O
previously O
demonstrated O
that O
pyrene O
in O
diesel O
- O
exhaust O
particles O
( O
DEP O
) O
has O
an O
adjuvant O
activity O
on O
immunoglobulin B
E I
( O
IgE B
) O
antibody O
production O
in O
mice O
immunized O
with O
Japanese B
cedar I
pollen I
allergen I
( O
JCPA B
) O
or O
ovalbumin B
( O
OA B
) O
intraperitoneally O
. O

Partial O
characterization O
of O
the O
CCAAT O
box O
in O
the O
promoter O
of O
the O
hLGFBP B
- I
1 I
gene I
: O
interaction O
with O
negatively O
acting O
transcription O
factors O
in O
decidualized O
human O
endometrial O
stromal O
cells O
. O

We O
conclude O
that O
transcription O
of O
both O
SCD1 B
and O
SCD2 B
genes I
is O
responsive O
to O
cellular O
sterol O
levels O
and O
to O
the O
levels O
of O
nuclear O
SREBP B
/ O
ADD1 B
and O
that O
transcriptional O
induction O
requires O
three O
spatially O
conserved O
cis O
elements O
, O
that O
bind O
SREBP B
and O
NF B
- I
Y I
. O

Three O
transcriptional O
control O
elements O
within O
300 O
base O
pairs O
of O
the O
5 O
' O
- O
flanking O
region O
of O
the O
rat B
glucagon I
gene I
interact O
with O
regulatory O
cellular O
proteins O
and O
direct O
transcription O
only O
in O
glucagon B
- O
producing O
islet O
cells O
. O

A O
role O
for O
phosphatidylinositol B
3 I
- I
kinase I
in O
the O
regulation O
of O
beta B
1 I
integrin I
activity O
by O
the O
CD2 B
antigen I
. O

Relationship O
between O
age O
- O
associated O
endocrine O
deficiencies O
and O
muscle O
function O
in O
elderly O
women O
: O
a O
cross O
- O
sectional O
study O
. O

In O
contrast O
, O
the O
sequences O
essential O
for O
the O
transcription O
of O
EFT1 B
were O
localized O
to O
the O
region O
between O
the O
start O
ATG O
and O
the O
stop O
codon O
of O
the O
VPS17 B
gene I
that O
terminates O
267 O
nt O
upstream O
on O
the O
same O
strand O
. O

Exposure O
of O
raft O
cultures O
to O
activators O
of O
protein B
kinase I
C I
, O
such O
as O
phorbol O
esters O
, O
results O
in O
the O
further O
induction O
of O
late O
gene O
expression O
as O
well O
as O
virion O
assembly O
. O

A O
case O
of O
2 O
cancers O
of O
the O
colon O

The O
" O
Gregg O
phenomenon O
" O
implies O
that O
myocardial O
function O
and O
oxygen O
consumption O
( O
MVO2 O
) O
increase O
when O
coronary O
perfusion O
is O
enhanced O
within O
or O
above O
the O
autoregulatory O
range O
. O

We O
have O
mapped O
the O
specific O
domains O
within O
each O
of O
the O
proteins O
responsible O
for O
this O
interaction O
. O

In O
each O
of O
the O
studied O
HPV O
types O
, O
the O
two O
CDP B
/ O
Cut B
binding O
sites O
of O
PSM B
overlap O
with O
the O
known O
or O
presumed O
binding O
sites O
of O
the O
replication B
initiator I
protein I
E1 I
. O

Reduced O
cell O
numbers O
72 O
h O
after O
insonation O
were O
recorded O
when O
the O
cells O
were O
insonated O
at O
an O
antinode O
but O
not O
when O
they O
were O
at O
a O
node O
. O

Hydroxyl O
radical O
footprinting O
of O
DNA O
complexes O
of O
the O
ets B
domain I
of O
PU B
. I
1 I
and O
its O
comparison O
to O
the O
crystal O
structure O
. O

ATF B
is O
formulated O
from O
the O
prothrombin B
time O
( O
PT O
) O
, O
fibrinogen B
transformation O
rate O
( O
FTR O
) O
( O
a O
representation O
of O
thrombin B
activity O
) O
, O
and O
a O
consideration O
of O
the O
fibrinogen B
( O
FBG B
) O
content O
of O
blood O
plasma O
. O

( O
LH B
P O
< O
0 O
. O
05 O
, O
LH B
/ O
FSH B
P O
< O
0 O
. O
01 O
) O
. O

Inhibition O
of O
Raf B
- I
1 I
signaling O
by O
a O
monoclonal O
antibody O
, O
which O
interferes O
with O
Raf B
- I
1 I
activation O
and O
with O
Mek B
substrate O
binding O
. O

Phenotypic O
and O
molecular O
analyses O
of O
patients O
with O
partial O
chromosome O
21 O
monosomy O
enabled O
us O
to O
define O
a O
region O
, O
spanning O
2 O
. O
4 O
Mb O
between O
D21S190 B
and O
D21S226 B
, O
associated O
with O
arthrogryposis O
, O
mental O
retardation O
, O
hypertonia O
, O
and O
several O
facial O
anomalies O
. O

The O
classic O
sterol O
regulatory O
cis O
element O
( O
sre O
- O
1 O
) O
in O
the O
LDL B
receptor I
promoter I
mediates O
sterol B
regulatory I
element I
binding I
protein I
( O
SREBP B
) O
- O
binding O
and O
the O
effects O
of O
insulin B
and O
platelet B
derived I
growth I
factor I
( O
PDGF B
) O
. O

The O
Ca2 O
+ O
concentration O
in O
rats O
serum O
after O
intragastrically O
fenarimol O
administration O
( O
doses O
1 O
/ O
40 O
, O
1 O
/ O
20 O
and O
1 O
/ O
10 O
LD50 O
) O
during O
5 O
days O
was O
investigated O
. O

In O
Cos O
7 O
cells O
transfected O
with O
oCRF1var B
, O
cAMP O
accumulation O
was O
only O
observed O
at O
the O
highest O
concentration O
of O
oCRF B
utilized O
( O
100 O
nM O
) O
. O

Pyknotic O
osteocytes O
increased O
at O
12 O
h O
( O
p O
less O
than O
0 O
. O
01 O
) O
and O
2 O
days O
( O
p O
less O
than O
0 O
. O
001 O
) O
of O
ischemia O
. O

The O
right O
cornea O
of O
rats O
was O
cauterized O
by O
drops O
of O
1 O
. O
5 O
N O
HC1 O
over O
30 O
s O
; O
the O
left O
one O
served O
as O
a O
control O
. O

The O
isoproterenol O
infusion O
was O
adjusted O
so O
that O
heart O
rate O
( O
HR O
) O
was O
at O
least O
30 O
beats O
/ O
min O
greater O
than O
baseline O
. O

This O
is O
probably O
because O
, O
in O
vivo O
, O
some O
bacteria O
( O
perhaps O
dormant O
forms O
) O
are O
not O
entirely O
dependent O
upon O
urease B
for O
survival O
. O

The O
system O
consists O
of O
a O
microcomputer O
and O
six O
laboratory O
analyzers O
: O
a O
blood O
gas O
analyzer O
, O
a O
flame O
photometer O
, O
a O
plasma O
osmotic O
pressure O
meter O
, O
a O
chloride O
ion O
titrator O
, O
a O
blood O
sugar O
analyzer O
, O
and O
a O
hemoglobin B
concentration O
and O
saturation O
meter O
. O

Comparing O
the O
outcome O
of O
the O
group O
having O
histologically O
normal O
biopsies O
with O
the O
group O
having O
nonspecifically O
abnormal O
ones O
it O
is O
concluded O
that O
frontal O
biopsy O
is O
not O
of O
such O
high O
prognostic O
value O
as O
has O
been O
claimed O
in O
previous O
reports O
. O

He O
presented O
with O
left O
lower O
abdominal O
pain O
and O
hematoma O
after O
a O
fall O
. O

Fusion O
of O
mouse O
CD8 B
+ I
class B
I I
MHC I
- O
restricted O
T O
cells O
with O
the O
BW5147 O
thymoma O
invariably O
yields O
CD8 B
- I
hybridomas O
in O
which O
RNA O
transcribed O
from O
the O
CD8 B
alpha I
( O
Lyt B
- I
2 I
) O
gene O
is O
undetectable O
. O

Peak O
plasma O
ACTH B
responses O
were O
not O
significantly O
different O
between O
control O
and O
patient O
groups O
. O

For O
the O
P B
transcript I
from O
phage O
with O
the O
G O
( O
- O
) O
orientation O
, O
full O
termination O
activity O
required O
both O
the O
region O
containing O
the O
stem O
- O
loop O
structure O
and O
upstream O
sequences O
. O

Development O
of O
neural O
control O
of O
alimentary O
motor O
function O
in O
the O
vertebrates O
. O

Nb2 O
- O
11C O
cells O
expressed O
three O
PNR B
- I
related I
mRNA I
transcripts I
of O
2 O
. O
5 O
, O
3 O
. O
0 O
and O
> O
10 O
kb O
. O

These O
results O
suggest O
that O
the O
Reg1 B
- O
Glc7 B
phosphatase O
is O
a O
cytoplasmic O
component O
of O
the O
machinery O
responsible O
for O
returning O
Snf1 B
kinase I
activity O
to O
its O
basal O
level O
and O
reestablishing O
glucose O
repression O
. O

However O
, O
the O
blend O
of O
Metanil O
yellow O
and O
Orange O
II O
( O
1 O
: O
1 O
) O
resulted O
in O
the O
D1 O
50 O
. O
value O
of O
288 O
min O
. O

The O
majority O
of O
these O
cells O
were O
also O
OKT4 B
positive O
( O
helper O
/ O
inducer O
T O
lymphocytes O
) O
, O
while O
the O
minority O
were O
OKT8 B
positive O
( O
suppressor O
/ O
cytotoxic O
T O
lymphocytes O
) O
. O

AIMS O
: O
We O
postulated O
that O
perhexiline O
, O
a O
novel O
anti O
- O
ischaemic O
agent O
with O
an O
oxygen O
- O
sparing O
metabolic O
effect O
in O
the O
myocardium O
( O
via O
inhibition O
of O
carnitine B
palmitoyltransferase I
- I
1 I
) O
and O
no O
adverse O
haemodynamic O
effects O
, O
may O
improve O
symptomatic O
status O
in O
elderly O
patients O
with O
severe O
aortic O
stenosis O
. O

The O
IFNAR B
chain I
interacts O
with O
both O
IFN B
- I
alpha I
2 I
and O
IFN B
- I
beta I
, O
as O
demonstrated O
by O
cross O
- O
linking O
. O

To O
clone O
cDNAs O
for O
the O
beta O
subunit O
of O
rabbit B
eIF I
- I
2B I
, O
amino O
acid O
sequence O
data O
was O
first O
obtained O
and O
used O
to O
design O
redundant O
oligonucleotide O
primers O
for O
use O
in O
PCR O
. O

We O
report O
the O
cloning O
of O
the O
ade1 B
gene I
on O
a O
4 O
. O
4 O
kb O
Sau3A O
insert O
in O
the O
yeast O
shuttle O
vector O
pWH5 O
. O

Nimodipine O
( O
5 O
micrograms O
/ O
kg O
) O
followed O
by O
infusion O
of O
0 O
. O
75 O
microgram O
/ O
kg O
/ O
min O
lowered O
the O
blood O
pressure O
by O
10 O
% O
in O
both O
normotensive O
and O
hypertensive O
rats O
; O
the O
same O
dose O
schedule O
of O
nifedipine O
did O
not O
lower O
MAP O
. O

Results O
of O
these O
in O
vitro O
studies O
may O
be O
relevant O
to O
the O
pathogenesis O
of O
alveolitis O
in O
organic O
dust O
- O
induced O
lung O
diseases O
. O

The O
odds O
of O
atopy O
( O
defined O
as O
a O
positive O
test O
for O
at O
least O
one O
of O
the O
antigens O
) O
were O
5 O
times O
higher O
( O
odds O
ratio O
= O
7 O
. O
0 O
; O
95 O
% O
confidence O
interval O
= O
1 O
. O
6 O
- O
31 O
. O
1 O
% O
; O
p O
= O
0 O
. O
01 O
) O
in O
the O
uninfected O
group O
, O
after O
taking O
into O
account O
the O
potential O
influence O
of O
gender O
and O
age O
. O

A O
selectable O
marker O
for O
transformation O
was O
constructed O
by O
transcriptional O
fusion O
of O
a O
Ustilago O
maydis I
heat I
shock I
gene I
promoter I
with O
the O
hygromycin B
B I
phosphotransferase I
gene I
of O
Escherichia O
coli O
. O

The O
structure O
of O
the O
mAR B
ORF I
was O
confirmed O
by O
sequence O
analysis O
of O
mAR B
cDNA I
fragments I
, O
which O
were O
obtained O
by O
PCR O
amplification O
of O
mouse O
testis O
cDNA O
, O
using O
mAR B
specific O
primers O
. O

The O
aim O
of O
this O
study O
was O
to O
determine O
the O
response O
properties O
of O
the O
human O
visual O
cortex O
to O
chromatic O
stimuli O
using O
magnetoencephalography O
( O
MEG O
) O
. O

The O
role O
of O
NUT1 B
and O
regulation O
of O
nitrogen O
metabolism O
in O
the O
disease O
process O
was O
evaluated O
by O
pathogenicity O
assays O
. O

These O
three O
phases O
of O
cyclin B
E I
association O
with O
chromatin O
may O
facilitate O
the O
diverse O
activities O
of O
cyclin B
E I
- O
- O
Cdk2 B
in O
initiating O
replication O
, O
blocking O
rereplication O
, O
and O
allowing O
resetting O
of O
origins O
after O
mitosis O
. O

No O
IORT O
related O
mortality O
has O
been O
observed O
. O

Dermatophagoides O
farinae O
- O
sensitized O
lymphocytes O
in O
asthmatic O
children O
as O
evaluated O
by O
interleukin B
- I
2 I
( O
IL B
- I
2 I
) O
production O
and O
acquisition O
of O
IL B
- I
2 I
responsiveness O

Enterotoxin O
production O
by O
staphylococci O
isolated O
from O
foods O
in O
France O
. O

Measurements O
in O
rats O
given O
a O
dose O
of O
propranolol O
that O
partially O
inhibited O
the O
NA O
- O
induced O
increase O
in O
Q O
to O
IBAT O
indicated O
that O
the O
decline O
in O
ENAIBAT O
was O
attributable O
primarily O
to O
the O
increase O
in O
Q O
rather O
than O
to O
increasing O
saturation O
of O
uptake O
mechanisms O
. O

Plasma B
LH I
and O
progesterone O
levels O
after O
single O
or O
multiple O
injections O
of O
synthetic B
LH I
- I
RH I
in O
anoestrous O
ewes O
and O
comparison O
with O
levels O
during O
the O
oestrous O
cycle O
. O

MATERIAL O
AND O
METHODS O
: O

But O
, O
when O
mated O
males O
built O
nests O
in O
the O
absence O
of O
young O
, O
T O
levels O
were O
higher O
than O
in O
all O
other O
conditions O
, O
though O
only O
for O
early O
broods O
. O

Primers O
were O
developed O
for O
specific O
amplification O
of O
9804 O
gene O
fragments O
. O

The O
promyelocytic B
leukemia I
protein I
interacts O
with O
Sp1 B
and O
inhibits O
its O
transactivation O
of O
the O
epidermal B
growth I
factor I
receptor I
promoter I
. O

The O
mutants O
retained O
the O
parental O
ability O
to O
participate O
in O
intergeneric O
coaggregation O
with O
human O
oral O
actinomyces O
, O
indicating O
the O
specificity O
of O
the O
mutation O
in O
altering O
intrageneric O
coaggregations O
. O

The O
Saccharomyces O
cerevisiae O
targets O
of O
rapamycin O
, O
TOR1 B
and O
TOR2 B
, O
signal O
activation O
of O
cell O
growth O
in O
response O
to O
nutrient O
availability O
. O

Biology O
of O
Anopheles O
quadrimaculatus O
under O
field O
conditions O
in O
central O
Florida O
. O

Immunogenicity O
tested O
in O
experiments O
on O
guinea O
pigs O
and O
rabbits O
was O
not O
reduced O
and O
corresponded O
to O
that O
of O
the O
reference O
STI O
- O
1 O
vaccine O
. O

The O
Thrombolysis O
in O
Myocardial O
Infarction O
( O
TIMI O
) O
Phase O
II O
Trial O
randomized O
3 O
, O
339 O
patients O
to O
either O
an O
invasive O
( O
INV O
, O
n O
= O
1 O
, O
681 O
) O
or O
a O
conservative O
( O
CON O
, O
n O
= O
1 O
, O
658 O
) O
strategy O
after O
intravenous O
recombinant B
tissue I
- I
type I
plasminogen I
activator I
( O
rt B
- I
PA I
) O
for O
acute O
myocardial O
infarction O
. O

In O
addition O
, O
mutations O
within O
the O
Ubx B
unit I
exons I
are O
known O
and O
most O
of O
these O
behave O
as O
null O
alleles O
. O

Taken O
together O
, O
these O
data O
suggest O
that O
the O
Smad3 B
/ O
Smad4 B
complex O
has O
at O
least O
two O
separable O
nuclear O
functions O
: O
it O
forms O
a O
rapid O
, O
yet O
transient O
sequence O
- O
specific O
DNA O
binding O
complex O
, O
and O
it O
potentiates O
AP1 B
- O
dependent O
transcriptional O
activation O
. O

When O
phentermine O
was O
mixed O
with O
other O
unlabeled O
reversible O
MAO B
inhibitors O
( O
e O
. O
g O
. O
pseudoephedrine O
, O
ephedrine O
, O
norephedrine O
; O
estradiol O
benzoate O
) O
, O
the O
degree O
of O
MAO B
inhibition O
was O
additive O
. O

Here O
we O
report O
that O
point O
mutations O
of O
the O
Abd B
- I
B I
gene I
in O
trans O
suppress O
the O
Fab B
- I
7 I
phenotype O
in O
a O
pairing O
- O
dependent O
manner O
and O
thus O
represent O
a O
type O
of O
transvection O
. O

4 O
382 O
new O
mothers O
were O
examined O
retrospectively O
with O
the O
enzyme O
- O
linked O
immunosorbent O
assay O
( O
ELISA O
) O
for O
IgG B
activity O
to O
cytomegalovirus O
( O
CMV O
) O
during O
pregnancy O
. O

Also O
, O
residues O
273 O
to O
287 O
, O
which O
are O
identical O
in O
sequence O
to O
a O
15 O
- O
amino O
- O
acid O
segment O
near O
the O
carboxy O
terminus O
of O
the O
simian B
foamy I
virus I
transcriptional I
activator I
Taf I
, O
can O
independently O
enhance O
the O
activity O
of O
the O
minimal O
activator O
region O
. O

An O
analogue O
of O
calcium O
, O
strontium O
chloride O
Sr O
89 O
is O
rapidly O
cleared O
from O
the O
blood O
after O
i O
. O
v O
. O
injection O
. O

The O
effect O
of O
pneumoperitoneum O
on O
dissemination O
and O
scar O
implantation O
of O
intra O
- O
abdominal O
tumor O
cells O
. O

In O
two O
of O
these O
nine O
patients O
, O
a O
decrease O
in O
dyskinesia O
score O
was O
observed O
without O
a O
concomitant O
worsening O
of O
parkinsonian O
symptoms O
, O
whereas O
in O
the O
remaining O
seven O
, O
full O
parkinsonian O
akinesia O
followed O
THDL O
administration O
. O

Alterations O
in O
TCR B
- O
MHC B
contacts O
subsequent O
to O
cross O
- O
recognition O
of O
class B
I I
MHC I
and O
singly O
substituted O
peptide O
variants O
. O

Two O
patients O
with O
Ewing O
' O
s O
sarcoma O
relapsed O
( O
1 O
patient O
with O
both O
local O
and O
distant O
failure O
) O
at O
26 O
and O
58 O
months O
and O
were O
again O
rendered O
disease O
- O
free O
with O
surgery O
, O
total O
body O
irradiation O
and O
further O
chemotherapy O
. O

The O
cloning O
of O
human B
apo I
B I
- I
100 I
has O
provided O
new O
insights O
into O
the O
structure O
and O
physicochemical O
properties O
of O
apo B
B I
- I
100 I
and O
will O
facilitate O
studies O
on O
the O
factors O
modulating O
apo B
B I
- I
100 I
biosynthesis O
and O
the O
expression O
of O
the O
apo B
B I
- I
100 I
gene I
in O
patients O
with O
dyslipoproteinemias O
. O

Angiographic O
follow O
- O
up O
was O
obtained O
in O
92 O
% O
of O
patients O
with O
a O
primary O
success O
of O
angioplasty O
. O

Acceptability O
of O
home O
monitoring O
as O
an O
aid O
to O
conception O
. O

Repeated O
interferon B
- I
beta I
( O
IFN B
- I
beta I
) I
silencer I
B I
motifs O
and O
a O
lysozyme B
silencer I
1 I
motif I
have O
been O
found O
in O
the O
CD95 B
gene I
at O
approximately O
- O
1 O
, O
600 O
and O
- O
1 O
, O
100 O
, O
respectively O
. O

Action O
against O
Vibrio O
cholerae O
O1 O
Tox B
+ I
of O
chemical O
products O
used O
in O
the O
lemon O
production O
. O

Patients O
with O
the O
chronic O
photosensitivity O
dermatitis O
with O
actinic O
reticuloid O
syndrome O
have O
high O
total O
serum B
IgE I
concentrations O
. O

The O
sequence O
of O
the O
25 O
- O
kDa O
chain O
of O
the O
alpha O
subunit O
was O
found O
in O
a O
cDNA O
clone O
, O
and O
the O
amino O
acid O
sequence O
deduced O
from O
the O
cDNA O
establishes O
the O
complete O
amino O
acid O
sequence O
of O
the O
25 O
- O
kDa O
chain O
. O

Characterization O
of O
the O
chromosome O
19 O
breakpoint O
region O
revealed O
that O
the O
transcription O
factor O
- O
encoding O
USF2 I
gene I
is O
affected O
. O

Tax B
and O
the O
active O
Tax B
mutants I
were O
able O
to O
abrogate O
the O
G1 O
arrest O
and O
apoptosis O
induced O
by O
p53 B
, O
and O
this O
effect O
does O
not O
correlate O
with O
an O
altered O
localization O
of O
nuclear B
p53 I
or O
with O
the O
disruption O
of O
p53 B
- I
DNA I
complexes I
. O

When O
patients O
were O
admitted O
within O
one O
week O
of O
the O
onset O
of O
dark O
urine O
, O
45 O
% O
were O
found O
to O
be O
shedding O
HAV O
, O
whereas O
only O
11 O
% O
of O
specimens O
obtained O
from O
patients O
admitted O
during O
the O
second O
week O
contained O
virus O
. O

Biochemical O
studies O
have O
identified O
a O
cellular O
non O
- O
P O
element O
- O
encoded O
DNA O
binding O
protein O
, O
termed O
the O
inverted B
repeat I
binding I
protein I
( O
IRBP B
) O
, O
that O
specifically O
interacts O
with O
the O
outer O
half O
of O
the O
31 O
- O
bp O
terminal O
inverted O
repeats O
. O

Threefold O
risks O
were O
observed O
for O
men O
with O
the O
highest O
level O
of O
intensity O
and O
for O
those O
with O
the O
highest O
probability O
of O
EMF O
exposure O
, O
although O
women O
with O
heavy O
EMF O
exposure O
did O
not O
experience O
increased O
risk O
. O

In O
the O
spontaneous O
abortion O
group O
, O
the O
levels O
of O
PAPP B
- I
A I
were O
significantly O
lower O
than O
in O
normal O
pregnancy O
but O
higher O
than O
in O
non O
- O
pregnant O
controls O
. O

We O
report O
the O
sequence O
of O
a O
7941 O
bp O
DNA O
fragment O
from O
the O
left O
arm O
of O
chromosome O
VII O
of O
Saccharomyces O
cerevisiae O
which O
contains O
four O
open O
reading O
frames O
( O
ORFs O
) O
of O
greater O
than O
100 O
amino O
acid O
residues O
. O

A O
4 B
. I
0 I
- I
kb I
Nlk I
message I
is O
also O
present O
during O
embryogenesis O
, O
detectable O
at O
day O
E10 O
. O

Promoting O
fat O
utilisation O
by O
reducing O
the O
carbohydrate O
- O
fat O
ratio O
of O
the O
TPN O
reduces O
free O
radical O
activity O
to O
a O
similar O
extent O
as O
fat O
exclusion O
. O

In O
cells O
infected O
with O
a O
recombinant O
vaccinia O
virus O
expressing O
HCV O
structural O
proteins O
( O
core B
, O
E1 B
, O
and O
E2 B
) O
, O
DDX3 B
and O
core B
colocalized O
in O
distinct O
spots O
in O
the O
perinuclear O
region O
of O
the O
cytoplasm O
. O

Mutating O
this O
sequence O
eliminated O
complex O
formation O
and O
markedly O
reduced O
basal O
and O
cAMP O
- O
dependent O
promoter O
activity O
of O
transfected O
reporter O
genes O
. O

The O
lowest O
PaCO2 O
values O
were O
significantly O
lower O
in O
MCE O
than O
in O
the O
F O
, O
W O
, O
and O
D O
( O
F O
+ O
W O
+ O
D O
) O
group O
or O
controls O
. O

The O
translational O
product O
of O
UL26 B
. I
5 I
is O
infected B
- I
cell I
protein I
35c I
, O
d O
( O
ICP35c B
, I
d I
) O
( O
F O
. O

The O
mouse B
tapasin I
gene I
was O
mapped O
about O
70 O
kilobases O
from O
H2 B
- I
K I
at O
the O
centromeric O
end O
of O
the O
mouse B
MHC I
. O

Due O
to O
significant O
homology O
to O
the O
C O
- O
terminus O
of O
the O
Mutator B
transposase I
this O
alternative O
gene O
product O
was O
named O
Trap B
( O
transposon B
- I
associated I
protein I
) O
. O

Nonetheless O
, O
the O
majority O
were O
located O
within O
three O
short O
sequences O
at O
the O
N O
terminus O
, O
middle O
, O
and O
C O
terminus O
that O
are O
phylogenetically O
conserved O
among O
all O
known O
eubacterial O
and O
chloroplast O
versions O
of O
this O
protein O
. O

The O
c B
- I
raf I
- I
1 I
proto I
- I
oncogene I
is O
the O
cellular B
homologue I
of I
v I
- I
raf I
, O
the O
oncogene O
of O
the O
acutely O
transforming O
retrovirus O
3611 O
- O
MSV O
. O

Using O
gel O
retardation O
analysis O
, O
four O
binding O
sites O
for O
Rap1p B
have O
been O
identified O
within O
the O
promoter O
of O
the O
RAP1 B
gene I
. O

In O
general O
, O
the O
scallop O
extract O
potentiated O
phototactic O
suppression O
. O

Alcohol O
abuse O
and O
treatment O
resistance O
in O
skin O
disease O
. O

The O
mean O
serum O
CDT B
increased O
from O
8 O
. O
5 O
( O
SD O
2 O
. O
2 O
) O
U O
/ O
l O
to O
16 O
. O
6 O
( O
SD O
7 O
. O
2 O
) O
U O
/ O
l O
( O
P O
< O
0 O
. O
001 O
) O
. O

Subsequent O
analysis O
revealed O
that O
MyD88 B
possesses O
a O
unique O
modular O
structure O
, O
which O
consists O
of O
an O
N O
- O
terminal O
" O
death O
domain O
, O
" O
similar O
to O
the O
intracellular O
segments O
of O
TNF B
receptor I
1 I
and O
Fas B
, O
and O
a O
C O
- O
terminal O
region O
related O
to O
the O
cytoplasmic O
domains O
of O
the O
Drosophila B
morphogen I
Toll I
and O
vertebrate B
interleukin I
- I
1 I
receptors I
. O

RT O
- O
PCR O
analysis O
of O
FGFR B
- I
3 I
mRNA I
showed O
the O
presence O
of O
a O
splice O
variant O
in O
which O
exons O
7 O
and O
8 O
are O
deleted O
. O

In O
human O
myxoid O
liposarcoma O
, O
a O
chromosomal O
rearrangement O
leads O
to O
fusion O
of O
the O
growth O
- O
arresting O
and O
DNA I
- I
damage I
- I
inducible I
transcription I
factor I
CHOP I
( O
GADD153 B
) O
to O
a O
peptide O
fragment O
encoded O
by O
the O
TLS B
gene I
. O

They O
also O
show O
that O
M O
. O
malmoense O
can O
be O
isolated O
from O
the O
environment O
which O
may O
be O
the O
source O
of O
the O
infection O
. O

The O
monkeys O
had O
been O
administered O
a O
common O
lead O
isotope O
" O
mix O
" O
at O
the O
rate O
of O
about O
1300 O
micrograms O
Pb O
/ O
kg O
body O
wt O
/ O
day O
from O
age O
10 O
months O
until O
the O
start O
of O
the O
study O
. O

We O
previously O
described O
the O
upregulation O
of O
the O
MT2 B
antigen I
during O
urodele O
limb O
regeneration O
and O
characterized O
the O
MT2 B
antigen I
as O
a O
310 B
- I
to I
325 I
- I
kDa I
chondroitin I
- I
sulfated I
glycoprotein I
with O
a O
core O
protein O
of O
285 O
- O
300 O
kDa O
. O

Analysis O
of O
the O
MCV B
- I
1 I
RPO1 I
revealed O
high O
amino O
acid O
homologies O
to O
the O
corresponding O
polypeptides O
of O
vaccinia O
and O
variola O
virus O
. O

The O
effects O
of O
dopamine O
and O
of O
dopamine B
D2 I
receptor I
blocker O
haloperidol O
on O
the O
activity O
of O
carotid O
chemoreceptors O
were O
studied O
in O
24 O
anesthetized O
, O
paralyzed O
and O
artificially O
ventilated O
newborn O
kittens O
aged O
0 O
- O
17 O
days O
. O

Fifty O
- O
seven O
per O
cent O
of O
the O
primary O
patients O
reporting O
mild O
symptoms O
had O
abnormal O
levels O
of O
leucocytes O
in O
their O
CSF O
. O

When O
proper O
controls O
were O
used O
, O
the O
psychophysical O
data O
and O
computer O
simulation O
gave O
remarkably O
comparable O
results O
. O

Patients O
were O
treated O
with O
either O
4 O
. O
8 O
million O
units O
of O
procaine O
penicillin O
with O
1 O
g O
probenecid O
, O
3 O
. O
5 O
or O
4 O
. O
5 O
g O
of O
ampicillin O
with O
1 O
g O
probenecid O
, O
or O
9 O
. O
5 O
g O
of O
tetracycline O
given O
over O
4 O
days O
. O

The O
results O
of O
this O
study O
indicate O
that O
the O
Improved O
Crest O
Complete O
with O
longer O
rippled O
outer O
bristles O
provided O
significantly O
superior O
( O
p O
< O
0 O
. O
05 O
) O
interproximal O
penetration O
overall O
and O
at O
the O
gumline O
than O
the O
Colgate O
Total O
and O
Oral O
- O
B O
Advantage O
brushes O
. O

Cardiac O
NE O
spillover O
was O
higher O
( O
P O
< O
. O
05 O
) O
at O
baseline O
in O
the O
patient O
group O
than O
in O
healthy O
subjects O
, O
whereas O
renal O
and O
whole O
- O
body O
NE O
spillovers O
were O
similar O
between O
the O
study O
groups O
. O

The O
activity O
of O
platelet B
factor I
4 I
in O
plasma O
of O
healthy O
and O
high O
risk O
newborns O
. O

Plasma O
renin I
activity O
in O
essential O
arterial O
hypertension O
. O

A O
trend O
was O
observed O
toward O
increased O
graft O
half O
- O
life O
in O
era O
2 O
( O
p O
= O
NS O
) O
. O

The O
proteins O
encoded O
by O
these O
genes O
, O
called O
GF14 B
proteins I
, O
participate O
in O
protein O
/ O
DNA O
complexes O
and O
show O
more O
than O
60 O
% O
identity O
with O
a O
highly O
conserved O
, O
widely O
distributed O
protein O
family O
, O
collectively O
referred O
to O
as O
14 B
- I
3 I
- I
3 I
proteins I
. O

The O
p58 B
( I
PITSLRE B
beta I
1 I
) O
protein O
kinase O
( O
PK B
) O
is O
a O
member O
of O
a O
large O
supergene O
family O
related O
to O
the O
master O
mitotic O
protein O
kinase O
, O
p34cdc2 B
. O

Maximal O
exercise O
duration O
and O
peak O
oxygen O
consumption O
were O
not O
changed O
. O

In O
contrast O
, O
NF B
- I
kappa I
B I
activation O
was O
observed O
in O
these O
cells O
after O
PMA O
stimulation O
. O

PtdIns O
( O
4 O
, O
5 O
) O
P2 O
synthesis O
, O
on O
the O
other O
hand O
, O
is O
only O
moderately O
affected O
even O
in O
fab1Delta B
mutants I
. O

A O
simple O
procedure O
for O
accurate O
quantitation O
of O
factor B
VIII I
inhibitors I
. O

We O
found O
that O
48 O
% O
of O
the O
patients O
had O
their O
thoracolumbar O
blood O
supply O
based O
on O
two O
anterior O
radiculospinal O
arteries O
the O
lowest O
of O
which O
was O
located O
at O
, O
or O
lower O
than O
, O
T12 O
, O
and O
the O
second O
and O
higher O
one O
between O
T6 O
and O
T10 O
. O

Low O
serum B
C3 I
values O
were O
observed O
in O
all O
11 O
children O
at O
some O
stage O
of O
their O
illness O
. O

The O
plants O
were O
then O
brought O
to O
our O
Houston O
laboratory O
where O
they O
were O
measured O
and O
analyzed O
for O
lignin O
and O
protein O
content O
and O
for O
phenylalanine B
ammonia I
- I
lyase I
( O
PAL B
) O
and O
peroxidase B
activities O
. O

Pax B
- I
6 I
constructs I
lacking O
the O
C O
- O
terminal O
activation O
domain O
repressed O
betaB1 B
- I
crystallin I
promoter I
activity O
as O
effectively O
as O
the O
full O
- O
length O
protein O
, O
but O
the O
PD O
alone O
or O
Pax B
- I
6 I
( I
5a I
) I
, O
a O
splice O
variant O
with O
an O
altered O
PD O
affecting O
its O
DNA O
binding O
specificity O
, O
did O
not O
. O

A O
regression O
analysis O
showed O
that O
86 O
% O
of O
the O
variance O
in O
weight O
gain O
was O
predicted O
by O
two O
leading O
indicators O
in O
the O
middle O
phase O
of O
treatment O
. O

OBJECTIVE O
: O
Respiratory O
- O
related O
electromyographic O
( O
EMG O
) O
activity O
of O
the O
superior O
pharyngeal O
constrictor O
( O
SPC O
) O
muscle O
was O
analyzed O
during O
the O
early O
stage O
of O
forced O
breathing O
. O

The O
predicted O
amino O
acid O
sequence O
contained O
all O
consensus O
regions O
for O
S B
- I
adenosyl I
methionine I
methyltransferases I
and O
presented O
26 O
% O
identity O
with O
Saccharomyces B
cerevisiae I
DHHB I
- I
methyltransferase I
and O
38 O
% O
identity O
with O
the O
rat O
protein O
, O
as O
well O
as O
with O
a O
bacterial O
( O
Escherichia O
coli O
and O
Salmonella O
typhimurium O
) O
methyltransferase O
encoded O
by O
the O
UBIG B
gene I
. O

Letter O
: O
Ballistocardiography O
. O

The O
introduction O
of O
FWD1 B
into O
cells O
significantly O
promotes O
ubiquitination O
and O
degradation O
of O
IkappaBalpha B
in O
concert O
with O
IkappaB B
kinases I
, O
resulting O
in O
nuclear O
translocation O
of O
NF B
- I
kappaB I
. O

Fosinopril O
decreased O
blood O
pressure O
from O
174 O
/ O
101 O
mm O
Hg O
to O
149 O
/ O
88 O
mm O
Hg O
in O
patients O
with O
diastolic O
hypertension O
and O
from O
182 O
/ O
86 O
mm O
Hg O
to O
151 O
/ O
80 O
mm O
Hg O
in O
patients O
with O
ISH O
. O

Although O
CB O
- O
4 O
isolates O
were O
less O
resistant O
to O
chlorine O
than O
CB O
- O
5 O
isolates O
, O
after O
1 O
, O
000 O
min O
of O
contact O
0 O
. O
01 O
% O
of O
the O
input O
virus O
was O
still O
infectious O
. O

The O
treatment O
of O
Sudeck O
' O
s O
atrophy O
in O
the O
upper O
limb O
by O
sympathetic O
blockade O
. O

To O
assess O
the O
effect O
on O
thrombus O
growth O
, O
we O
determined O
the O
accretion O
of O
125I O
- O
labeled O
fibrinogen B
onto O
autologous O
non O
- O
radioactive O
thrombi O
preformed O
in O
the O
jugular O
veins O
of O
rabbits O
. O

Thallium O
- O
201 O
scintigraphy O
after O
dipyridamole O
infusion O
with O
low O
level O
exercise O
. O

A O
kinetic O
model O
for O
99mTc O
- O
DMSA O
in O
the O
rat O
. O

Crude O
drugs O
from O
aquatic O
plants O
. O

Yet O
testosterone O
was O
predominant O
in O
the O
plasma O
of O
the O
Type O
II O
male O
morph O
which O
neither O
courts O
females O
nor O
nests O
, O
but O
instead O
parasitizes O
Type O
I O
males O
with O
sneak O
or O
satellite O
spawning O
tactics O
. O

Comparison O
of O
the O
therapeutic O
effectiveness O
of O
membrane O
and O
centrifugation O
plasmapheresis O

Amino O
- O
terminal O
polymorphisms O
of O
the O
human B
beta I
2 I
- I
adrenergic I
receptor I
impart O
distinct O
agonist O
- O
promoted O
regulatory O
properties O
. O

Hence O
, O
it O
is O
perhaps O
not O
necessary O
to O
perform O
spinal O
puncture O
if O
the O
only O
purpose O
is O
to O
determine O
the O
CSF O
/ O
serum O
ratio O
. O

Two O
plasmids O
, O
one O
containing O
the O
amino O
terminus O
of O
P B
fused O
to O
the O
DNA O
- O
binding O
domain O
of O
the O
yeast O
transactivator O
, O
GAL4 B
, O
and O
the O
other O
containing O
the O
amino O
terminus O
of O
NP B
fused O
to O
the O
herpesvirus O
transactivator I
, O
VP16 B
, O
were O
transfected O
in O
COS O
- O
1 O
cells O
along O
with O
a O
chloramphenicol B
acetyltransferase I
( O
CAT B
) O
reporter O
plasmid O
containing O
GAL4 B
DNA I
- I
binding I
sites I
. O

Biochemical O
experiments O
and O
genome O
sequencing O
have O
shown O
that O
, O
despite O
the O
prokaryotic O
cell O
and O
genome O
organization O
, O
basal O
transcriptional O
elements O
of O
members O
of O
the O
domain O
Archaea O
( O
i O
. O
e O
. O
, O
TATA O
box O
- O
like O
sequences O
, O
RNA B
polymerase I
, O
and O
transcription O
factors O
TBP B
, O
TFIIB B
, O
and O
TFIIS B
) O
are O
of O
the O
eukaryotic O
type O
. O

Humoral O
immunity O
is O
altered O
with O
the O
drop O
in O
IgG B
levels O
. O

For O
the O
second O
phase O
, O
it O
was O
12 O
. O
5 O
wk O
for O
RIR O
and O
10 O
for O
WL O
males O
, O
whereas O
it O
was O
5 O
wk O
for O
RIR O
and O
6 O
for O
WL O
females O
. O

Modified O
Grocott O
' O
s O
methenamine O
silver O
nitrate O
method O
for O
quick O
staining O
of O
Pneumocystis O
carinii O
. O

Additionally O
, O
VAHS O
is O
often O
associated O
with O
fatal O
infectious O
mononucleosis O
( O
IM O
) O
. O

RESULTS O
: O
In O
patients O
with O
necrotising O
fasciitis O
the O
arterial O
PO2 O
rose O
about O
7 O
- O
fold O
whereas O
the O
arterial O
PCO2 O
increased O
only O
slightly O
during O
exposure O
to O
2 O
. O
5 O
absolute O
atmospheres O
( O
ATA O
) O
of O
oxygen O
. O

We O
report O
on O
a O
case O
of O
a O
43 O
- O
old O
- O
male O
with O
a O
bronchogenic O
cyst O
in O
the O
distal O
esophagus O
, O
which O
was O
misdiagnosed O
as O
a O
malignant O
esophageal O
tumor O
based O
on O
preoperative O
imaging O
and O
high O
levels O
of O
the O
tumor O
markers O
CA B
19 I
- I
9 I
and O
CA B
125 I
. O

100 O
and O
51 O
p O
. O

A O
plasma O
cell O
- O
rich O
infiltrate O
was O
present O
in O
the O
connective O
tissue O
cores O
of O
the O
papillae O
. O

During O
normoxic O
exercise O
, O
at O
a O
mean O
O2 O
uptake O
( O
VO2 O
) O
of O
4 O
. O
0 O
l O
/ O
min O
, O
almitrine O
increased O
arterial O
PO2 O
( O
PaO2 O
) O
( O
P O
< O
0 O
. O
05 O
) O
, O
SaO2 O
( O
P O
< O
0 O
. O
01 O
) O
, O
and O
VE O
( O
P O
< O
0 O
. O
05 O
) O
and O
decreased O
arterial O
PCO2 O
( O
P O
< O
0 O
. O
01 O
) O
, O
without O
affecting O
pulmonary O
hemodynamics O
or O
ventilation O
- O
perfusion O
distributions O
. O

Gene B
GPR I
51 I
was O
localized O
by O
radiation O
hybrid O
mapping O
to O
chromosome O
9 O
, O
4 O
. O
81 O
cR O
from O
the O
WI B
- I
8684 I
marker O
, O
and O
proximal O
to O
the O
hereditary B
sensory I
neuropathy I
type I
1 I
locus I
. O

We O
conclude O
that O
the O
outcome O
of O
patients O
with O
uterine O
MMMT O
is O
mainly O
influenced O
by O
the O
initial O
stage O
and O
the O
type O
of O
epithelial O
component O
. O

The O
expression O
of O
the O
leukocyte B
EL I
- I
246 I
antigen I
was O
regulated O
in O
the O
same O
manner O
as O
L B
- I
selectin I
and O
EL B
- I
246 I
recognized O
anti B
- I
L I
- I
selectin I
mAb I
affinity O
- O
purified O
antigen O
in O
SDS O
/ O
PAGE O
Western O
blot O
analysis O
. O

Other O
Fischer O
- O
344 O
rats O
were O
also O
exposed O
for O
1 O
hr O
to O
0 O
. O
0 O
or O
to O
0 O
. O
6 O
ppm O
O3 O
. O

A O
computerized O
database O
of O
veterans O
discharged O
from O
the O
military O
after O
1967 O
was O
selected O
as O
the O
source O
because O
it O
contains O
about O
50 O
% O
of O
the O
total O
Vietnam O
era O
veteran O
population O
, O
is O
reasonably O
unbiased O
, O
and O
provides O
a O
feasible O
method O
for O
identifying O
twins O
. O

To O
examine O
the O
functional O
relationship O
between O
distinct O
cis O
- O
active O
elements O
within O
the O
distal O
enhancer O
region O
of O
the O
rat B
PRL I
gene I
, O
we O
have O
used O
deletional O
and O
mutational O
analysis O
of O
that O
region O
in O
transient O
transfection O
studies O
in O
GH3 O
pituitary O
tumor O
cells O
. O

The O
entire O
coding O
region O
of O
an O
ovine B
endometrial I
oxytocin I
receptor I
( O
OTR B
) O
cDNA O
was O
generated O
by O
PCR O
, O
subcloned O
into O
the O
SV40 B
major I
late I
promoter I
expression O
vector O
pSVLJ O
and O
transiently O
expressed O
in O
Cos O
- O
7 O
cells O
. O

Alcohol O
and O
stomach O
diseases O

These O
operons O
encode O
subunits O
of O
photosystems B
I I
( O
psa B
) O
and O
II I
( O
psb B
) O
, O
the O
cytochrome B
bGf I
complex I
( O
pet B
) O
, O
the O
plastid B
NAD I
( I
P I
) I
H I
dehydrogenase I
( O
ndh B
) O
, O
and O
the O
unidentified O
open O
reading O
frame I
ycf9 I
. O

The O
phosphatidylglycerol B
/ I
phosphatidylinositol I
transfer I
protein I
( O
PG B
/ I
PI I
- I
TP I
) O
is O
a O
new O
and O
original O
phospholipid B
transfer I
protein I
( O
PLTP B
) O
isolated O
from O
the O
Deuteromycete O
, O
Aspergillus O
oryzae O
. O

Transcriptional O
modulation O
of O
the O
anti B
- I
apoptotic I
protein I
BCL I
- I
XL I
by O
the O
paired B
box I
transcription I
factors I
PAX3 B
and O
PAX3 B
/ O
FKHR B
. O

The O
level O
of O
expression O
of O
the O
PDE B
protein I
was O
monitored O
by O
immunoblot O
analysis O
using O
two O
specific O
cAMP B
- I
PDE I
polyclonal I
antibodies I
and O
by O
measuring O
the O
PDE B
activity O
. O

Blotting O
analysis O
of O
intestinal O
RNA O
and O
hybridization O
of O
the O
blots O
with O
carboxy B
apoB I
cDNA I
probes O
produced O
a O
single O
15 O
- O
kb O
hybridization O
band O
whereas O
hybridization O
with O
amino O
terminal O
probes O
produced O
two O
hybridization O
bands O
of O
15 O
and O
8 O
kb O
. O

Since O
UmuD B
protein I
is O
proteolytically O
processed O
to O
an O
active O
form O
( O
UmuD B
* I
) O
in O
a O
RecA B
* I
- O
dependent O
fashion O
, O
and O
MucA B
shares O
extensive O
amino O
acid O
homology O
with O
UmuD B
, O
we O
examined O
whether O
MucA B
is O
similarly O
processed O
in O
the O
cell O
, O
using O
antiserum O
against O
a O
LacZ B
' I
- I
' I
MucA I
fusion O
protein O
. O

Influence O
of O
long O
- O
term O
zimelidine O
treatment O
on O
LSD O
- O
induced O
behavioural O
effects O
. O

We O
have O
determined O
the O
molecular O
organization O
and O
transcription O
start O
points O
( O
tsp O
) O
for O
the O
murine O
gene O
( O
TK B
) O
encoding O
thymidine B
kinase I
. O

Together O
with O
a O
number O
of O
phase O
II O
trials O
, O
phase O
I O
trials O
utilizing O
escalating O
doses O
of O
carboplatin O
and O
paclitaxel O
with O
growth O
factor O
or O
growth O
factor O
and O
blood O
stem O
- O
cell O
support O
have O
shown O
that O
substantial O
increases O
in O
dose O
intensity O
can O
be O
achieved O
. O

It O
is O
an O
untested O
argument O
that O
conventional O
beta B
- I
adrenoceptor I
antagonists O
possess O
unwanted O
metabolic O
effects O
that O
may O
counter O
some O
of O
their O
potential O
cardiac O
benefits O
. O

Genetic O
polymorphisms O
in O
the O
5 O
' O
- O
flanking O
region O
change O
transcriptional O
regulation O
of O
the O
human B
cytochrome I
P450IIE1 I
gene I
. O

Although O
the O
occurrence O
of O
any O
ventricular O
ectopic O
activity O
, O
as O
detected O
by O
either O
or O
both O
methods O
, O
was O
common O
, O
the O
incidence O
was O
significantly O
higher O
( O
P O
less O
than O
0 O
. O
001 O
) O
in O
patients O
with O
coronary O
heart O
disease O
( O
86 O
percent O
; O
77 O
/ O
90 O
) O
, O
as O
compared O
to O
that O
in O
normal O
subjects O
( O
40 O
percent O
; O
12 O
/ O
30 O
) O
. O

Increased O
trabecular O
spacing O
, O
such O
as O
it O
occurs O
in O
osteoporosis O
, O
reduces O
the O
spatial O
field O
inhomogeneity O
and O
thus O
prolongs O
T2 O
* O
, O
which O
has O
been O
shown O
both O
in O
vitro O
and O
in O
vivo O
. O

Healthy O
preterm O
infants O
of O
gestational O
age O
26 O
- O
29 O
weeks O
showed O
a O
' O
mature O
' O
pattern O
of O
permeability O
at O
birth O
, O
followed O
by O
a O
temporary O
period O
of O
enhanced O
permeability O
after O
3 O
- O
4 O
weeks O
of O
life O
. O

These O
studies O
reveal O
that O
CREM B
, O
a O
tissue O
- O
specific O
factor O
, O
is O
expressed O
and O
regulated O
by O
gonadotropins B
in O
the O
ovary O
, O
that O
the O
predominant O
CREM B
transcripts I
encode O
the O
repressor B
protein I
ICER I
, O
and O
that O
ICER B
is O
capable O
of O
inhibiting O
cAMP O
- O
induced O
expression O
of O
the O
inhibin B
alpha I
- I
subunit I
gene I
. O

The O
Brassica B
BTH1 I
gene I
may O
correspond O
to O
the O
Arabidopsis B
TH I
- I
1 I
gene I
. O

The O
same O
pattern O
of O
firing O
was O
seen O
with O
saccades O
in O
light O
and O
in O
complete O
darkness O
. O

While O
these O
could O
be O
unusual O
cases O
of O
chronic O
non O
- O
A O
, O
non O
- O
B O
hepatitis O
, O
this O
can O
be O
only O
speculation O
until O
a O
serologic O
test O
for O
non O
- O
A O
, O
non O
- O
B O
hepatitis O
becomes O
available O
. O

Acanthamoeba B
myosin I
I I
heavy I
chain I
kinase I
( O
MIHCK B
) O
phosphorylates O
the O
heavy O
chains O
of O
amoeba B
myosins I
I I
, O
increasing O
their O
actin B
- O
activated O
ATPase B
activities O
. O

Mutation O
of O
the O
HNF3 B
element I
significantly O
reduced O
promoter O
activity O
in O
HepG2 O
cells O
, O
whereas O
this O
element O
in O
isolation O
conferred O
HNF3beta B
responsiveness O
to O
a O
heterologous O
promoter O
. O

The O
mitochondrial B
tyrosyl I
- I
tRNA I
synthetase I
of I
Podospora I
anserina I
is O
a O
bifunctional O
enzyme O
active O
in O
protein O
synthesis O
and O
RNA O
splicing O
. O

The O
findings O
in O
the O
human O
multiple O
myeloma O
cell O
lines O
represent O
the O
first O
examples O
of O
B O
cells O
with O
downregulated O
PU B
. I
1 I
expression O
and O
apparently O
contradict O
observations O
in O
the O
murine O
system O
in O
which O
PU B
. I
1 I
is O
expressed O
and O
active O
in O
plasmacytoma O
cell O
lines O
. O

The O
traW B
gene I
of O
the O
Escherichia B
coli I
K I
- I
12 I
sex I
factor I
, I
F I
, O
encodes O
one O
of O
the O
numerous O
proteins O
required O
for O
conjugative O
transfer O
of O
this O
plasmid O
. O

Mibefradil O
( O
Ro O
40 O
- O
5967 O
) O
is O
a O
novel O
nondihydropyridine O
calcium O
antagonist O
. O

However O
, O
technical O
advances O
have O
shown O
the O
limitations O
of O
these O
tests O
as O
tests O
for O
IgM B
can O
be O
positive O
because O
of O
residual O
specific O
IgM B
or O
even O
in O
subjects O
free O
of O
acute O
infection O
due O
to O
the O
existence O
of O
natural O
interfering O
IgM B
. O

Carcinogenic O
action O
of O
groundnut O
meal O
containing O
aflatoxin O
in O
rats O
. O

( O
1991 O
) O
EMBO O
J O
. O

We O
have O
utilized O
transient O
transfections O
, O
mutation O
analysis O
, O
electromobility O
gel O
- O
shifts O
, O
and O
immunoblot O
analysis O
to O
test O
the O
hypothesis O
that O
expression O
of O
the O
CTalpha B
gene I
is O
controlled O
in O
part O
by O
the O
binding O
of O
three O
trans O
- O
acting O
nuclear O
factors O
, O
Sp1 B
, O
Sp2 B
, O
and O
Sp3 B
. O

AMPK B
is O
a O
heterotrimer O
composed O
of O
a O
catalytic O
subunit O
( O
alpha O
) O
and O
two O
regulatory O
subunits O
( O
beta O
and O
gamma O
) O
. O

The O
evolutionary O
significance O
of O
the O
similarities O
of O
intron O
secondary O
structures O
and O
open O
reading O
frames O
of O
the O
ND3 B
, I
4 I
and O
ATPase B
6 I
genes I
is O
discussed O
, O
including O
the O
possible O
separate O
evolution O
of O
structural O
and O
coding O
sequences O
. O

Antinuclear O
antibodies O
, O
rheumatoid B
factor I
and O
C B
- I
reactive I
protein I
in O
serum O
of O
normal O
women O
using O
oral O
contraceptives O
. O

The O
destruction O
of O
the O
mesometrium O
did O
not O
lengthen O
the O
oestrous O
cycle O
. O

Concentrations O
of O
platelet O
nitrite O
and O
total O
nitrate O
/ O
nitrite O
were O
determined O
using O
simple O
and O
sensitive O
nitrate O
/ O
nitrite O
fluorometric O
assay O
techniques O
. O

Power O
deposition O
by O
inverse O
- O
bremsstrahlung O
is O
modeled O
with O
a O
scheme O
based O
on O
Gaussian O
quadrature O
to O
accommodate O
a O
deposition O
rate O
whose O
spatial O
variation O
is O
highly O
nonuniform O
. O

The O
sulfur O
species O
could O
be O
separated O
within O
less O
than O
4 O
min O
by O
CZE O
with O
a O
pyromellitic O
acid O
electrolyte O
at O
pH O
3 O
. O
5 O
to O
5 O
. O
0 O
. O

We O
propose O
that O
unc B
- I
37 I
may O
be O
regulated O
by O
unc B
- I
4 I
. O

Adenovirus O
infection O
of O
hepatoma O
cells O
inhibited O
transcription O
of O
the O
phosphoenolpyruvate B
carboxykinase I
( I
GTP I
) I
( O
EC B
4 I
. I
1 I
. I
1 I
. I
32 I
) O
( O
PEPCK B
) O
gene O
and O
virtually O
eliminated O
transcription O
of O
a O
chimeric O
gene O
which O
contained O
the O
PEPCK B
promoter I
linked O
to O
the O
structural O
gene O
for O
chloramphenicol B
acetyltransferase I
( O
CAT B
) O
. O

After O
discharge O
, O
he O
was O
finally O
given O
a O
diagnosis O
of O
PCH O
because O
a O
Donath O
- O
Landsteiner O
test O
was O
positive O
. O

The O
second O
intron O
is O
the O
smallest O
of O
all O
the O
introns O
( O
116 O
bp O
) O
. O

Expression O
of O
the O
gene O
encoding O
transcription O
factor O
cyclic I
adenosine I
3 I
' I
, I
5 I
' I
- I
monophosphate I
( I
cAMP I
) I
response I
element I
- I
binding I
protein I
( O
CREB B
) O
: O
regulation O
by O
follicle B
- I
stimulating I
hormone I
- O
induced O
cAMP O
signaling O
in O
primary O
rat O
Sertoli O
cells O
. O

Efficacy O
was O
evaluated O
in O
24 O
patients O
, O
of O
whom O
eight O
received O
RD O
and O
16 O
SD O
. O

A O
silica O
- O
based O
, O
fluoride O
- O
free O
placebo O
containing O
NaTPP O
, O
and O
a O
NaF O
- O
containing O
silica O
- O
based O
USP O
reference O
standard O
toothpaste O
were O
used O
as O
negative O
and O
positive O
control O
toothpastes O
, O
respectively O
. O

The O
overall O
response O
rate O
including O
CR O
and O
PR O
was O
23 O
. O
5 O
% O
( O
4 O
/ O
17 O
) O
. O

High O
and O
pathological O
PAI B
- I
1 I
levels O
before O
and O
after O
the O
VO O
test O
were O
consistent O
with O
a O
defective O
fibrinolytic O
potential O
due O
to O
the O
inhibitory O
effect O
of O
PAI B
- I
1 I
on O
plasminogen B
activation O
. O

During O
hexamethylene O
bisactamide O
( O
HMBA O
) O
- O
induced O
differentiation O
of O
murine O
erythroleukemia O
( O
MEL O
) O
cells O
erythroid O
genes O
are O
transcriptionally O
activated O
while O
c B
- I
myb I
and O
several O
other O
nuclear O
proto O
- O
oncogenes O
are O
down O
- O
regulated O
. O

The O
recovery O
of O
adults O
schistosomes O
by O
extracorporeal O
filtration O
. O

Torsion O
of O
the O
contralateral O
testis O
5 O
years O
after O
orchiopexy O
. O

Frequency O
of O
recurrence O
of O
fibroids O
after O
myomectomy O
has O
been O
evaluated O
in O
145 O
women O
( O
median O
age O
38 O
years O
, O
range O
21 O
- O
52 O
) O
who O
underwent O
myomectomy O
. O

Choline B
acetyltransferase I
immunohistochemistry O
combined O
with O
the O
retrograde O
transport O
of O
horseradish B
peroxidase I
showed O
that O
the O
reticular O
and O
mediodorsal O
thalamic O
nuclei O
of O
the O
cat O
receive O
an O
important O
input O
from O
cholinergic O
and O
non O
- O
cholinergic O
neurons O
of O
substantia O
innominata O
and O
adjacent O
structures O
in O
the O
basal O
forebrain O
. O

( O
i O
) O
The O
chimeric O
gene O
consisting O
of O
the O
coding O
and O
5 O
' O
nontranslated O
leader O
regions O
of O
the O
TK B
gene I
fused O
to O
portions O
of O
the O
domain O
of O
alpha B
gene I
0 I
extending O
largely O
upstream O
from O
the O
site O
of O
initiation O
of O
transcription O
of O
alpha B
gene I
0 I
was O
regulated O
in O
the O
same O
fashion O
as O
the O
alpha B
4 I
- I
and I
alpha I
27 I
- I
TK I
chimeras I
. O

Ethanol O
preference O
in O
strains O
of O
rats O
selectively O
bred O
for O
behavioral O
characteristics O
. O

For O
the O
albino O
, O
both O
the O
ST1 O
and O
ST2 O
spatial O
responses O
peak O
at O
around O
0 O
. O
3 O
cycles O
deg O
- O
1 O
, O
and O
both O
curves O
are O
displaced O
considerably O
to O
the O
low O
spatial O
frequency O
side O
of O
the O
normal O
ST2 O
spatial O
response O
. O

Nuclear O
proteins O
bound O
the O
cad B
+ O
55 O
/ O
+ O
75 O
element O
in O
a O
cell O
cycle O
- O
dependent O
manner O
in O
electromobility O
shift O
assays O
; O
antibodies O
specific O
to O
USF B
and O
Max B
blocked O
the O
DNA O
- O
binding O
activity O
of O
different O
growth O
- O
regulated O
protein O
- O
DNA O
complexes O
. O

Thus O
, O
an O
unmodified O
threonine O
at O
position O
169 O
in O
Cdc28 B
is O
important O
for O
interaction O
with O
G1 B
cyclins I
. O

Additional O
genetic O
analyses O
described O
herein O
suggest O
that O
Skb1 B
is O
a O
component O
of O
the O
morphology O
control O
branch O
of O
the O
Ras B
signaling O
cascade O
in O
S O
. O
pombe O
and O
that O
it O
positively O
modulates O
Shk1 B
function O
. O

Recent O
studies O
have O
concentrated O
on O
the O
methods O
of O
preparation O
of O
coffee O
, O
which O
vary O
from O
country O
to O
country O
. O

From O
a O
chromosomal O
cosmid O
library O
of O
Streptomyces O
argillaceus O
, O
a O
Mtm O
producer O
, O
a O
clone O
( O
cosAR7 O
) O
was O
isolated O
by O
homology O
to O
the O
actI B
/ I
III I
region I
of O
S O
. O
coelicolor O
and O
the O
strDEM B
genes I
of O
S O
. O
griseus O
. O

In O
this O
report O
, O
we O
investigated O
whether O
the O
cleavage O
of O
the O
RRKR O
motif O
of O
MT B
- I
MMP1 I
by O
Golgi O
- O
associated I
furin I
is O
analogous O
to O
a O
similar O
enzyme O
activation O
mechanism O
observed O
with O
stromelysin B
- I
3 I
. O

Coexpression O
of O
C B
/ I
EBP I
alpha I
altered O
the O
cell O
specificity O
. O

A O
preterm O
formula O
with O
a O
traditional O
corn O
oil O
/ O
MCT O
blend O
containing O
38 O
% O
MCTs O
( O
MCT O
group O
) O
was O
compared O
to O
a O
new O
fat O
blend O
, O
designed O
to O
resemble O
human O
milk O
more O
, O
containing O
6 O
% O
MCTs O
( O
LCT O
group O
) O
. O

Thirty O
- O
three O
small O
glottic O
carcinomas O
( O
T1 O
and O
small O
T2 O
; O
UICC O
, O
1978 O
) O
were O
examined O
by O
malignancy O
grading O
using O
the O
8 O
- O
factor O
system O
proposed O
by O
Jakobsson O
et O
al O
. O

Phenylalanine O
and O
tyrosine O
levels O
were O
higher O
in O
those O
who O
received O
Vamin O
9 O
glucose O
but O
55 O
% O
of O
infants O
given O
Vamin O
Infant O
had O
tyrosine O
levels O
below O
the O
lower O
limit O
of O
the O
target O
range O
. O

We O
sought O
to O
test O
whether O
a O
prolonged O
infusion O
of O
magnesium O
sulfate O
( O
MgSO O
( O
4 O
) O
; O
40 O
mmol O
/ O
24 O
hours O
) O
would O
normalize O
QT O
interval O
variability O
in O
patients O
with O
compensated O
heart O
failure O
. O

Escherichia O
coli O
strains O
carrying O
recA730 B
( O
or O
other O
recA B
* I
alleles I
) O
exhibit O
dramatic O
increases O
in O
SOS O
- O
dependent O
spontaneous O
mutator O
activity O
. O

Outlook O
in O
oral O
and O
cutaneous O
Kaposi O
' O
s O
sarcoma O
. O

We O
report O
the O
complete O
nucleotide O
sequence O
of O
the O
camC B
gene I
along O
with O
155 O
base O
pairs O
of O
5 O
' O
and O
175 O
base O
pairs O
of O
3 O
' O
flanking O
sequence O
. O

However O
, O
it O
is O
only O
loosely O
associated O
, O
or O
not O
associated O
, O
with O
viral O
particles O
. O

gp170 B
is O
generated O
by O
an O
alternatively O
spliced O
Env B
mRNA I
using O
a O
splice O
donor O
and O
splice O
acceptor O
pair O
localized O
within O
the O
env B
open I
reading I
frame I
( O
ORF O
) O
, O
which O
is O
normally O
used O
to O
generate O
Bell B
and O
Bet B
transcripts I
derived O
from O
the O
internal O
promoter O
within O
the O
env B
ORF I
. O

gp170 B
is O
expressed O
at O
a O
level O
30 O
to O
50 O
% O
of O
the O
Env B
precursor I
gp130 B
. O

In O
marked O
contrast O
to O
the O
previously O
published O
human B
CD6 I
sequence I
, O
the O
mouse O
sequence O
predicts O
a O
long O
cytoplasmic O
tail O
that O
is O
not O
closely O
related O
to O
other O
proteins O
and O
possesses O
two O
proline O
- O
rich O
motifs O
containing O
the O
SH3 B
- I
domain I
binding I
consensus I
sequence I
, O
three O
protein B
kinase I
C I
phosphorylation O
site O
motifs O
, O
nine O
casein B
kinase I
- I
2 I
phosphorylation I
site I
motifs I
, O
and O
a O
serine O
- O
threonine O
- O
rich O
motif O
repeated O
three O
times O
. O

ET B
- I
1 I
limited O
the O
electrocardiographic O
evidence O
of O
subendocardial O
ischemia O
and O
attenuated O
contractile O
dysfunction O
compared O
with O
mechanical O
stenosis O
at O
the O
same O
coronary O
flows O
, O
even O
though O
lactate O
flux O
was O
similar O
. O

Our O
results O
are O
consistent O
with O
TCOF1 B
mutations O
leading O
to O
the O
Treacher O
Collins O
syndrome O
phenotype O
. O

Experimental O
production O
of O
a O
syndrome O
analogous O
to O
hydramnios O
in O
the O
rat O
fetus O

Total O
cross O
sections O
for O
electron O
scattering O
by O
CO2 O
molecules O
in O
the O
energy O
range O
400 O
- O
5000 O
eV O
. O

Thus O
, O
the O
cognitive O
performance O
of O
elderly O
subjects O
could O
be O
trained O
to O
a O
large O
extent O
. O

Partial O
albinism O
and O
immunodeficiency O
: O
ultrastructural O
study O
of O
haemophagocytosis O
and O
bone O
marrow O
erythroblasts O
in O
one O
case O
. O

Residues O
that O
affect O
PLCbeta B
and O
adenylyl B
cyclase I
II I
activity O
are O
found O
on O
opposite O
sides O
of O
the O
central O
tunnel O
, O
suggesting O
that O
PLC B
and O
adenylyl B
cyclase I
, O
like O
the O
alpha O
subunit O
, O
make O
many O
contacts O
on O
the O
top O
surface O
. O

The O
dynamics O
of O
protozoa O
were O
studied O
in O
two O
groups O
of O
rumen O
- O
fistulated O
cattle O
fed O
on O
a O
basal O
diet O
of O
molasses O
ad O
lib O
. O
, O
with O
oaten O
chaff O
given O
at O
6 O
or O
18 O
g O
/ O
kg O
live O
weight O
. O

The O
CBS B
- I
1 I
gene I
was O
mapped O
to O
human O
chromosome O
10p12 O
between O
markers O
WI O
- O
8535 O
and O
WI O
- O
4724 O
, O
and O
is O
tightly O
linked O
to O
the O
two O
STRP O
markers O
of O
D10S1789 B
and O
D10S550 B
. O

Blood O
ammonia O
concentration O
was O
significantly O
higher O
in O
males O
at O
70 O
, O
80 O
and O
90 O
% O
of O
VO2 O
peak O
. O

Value O
of O
bone O
scintigraphy O
with O
Tc99MDP O
in O
the O
early O
diagnosis O
of O
mobilization O
of O
the O
total O
hip O
prosthesis O

Youssoufian O
, O
A O
. O

The O
dipper O
( O
RONF O
of O
BP O
( O
or O
HR O
) O
> O
or O
= O
10 O
% O
2 O
. O

In O
the O
present O
study O
, O
we O
have O
investigated O
the O
effects O
of O
SDZ O
ENA O
713 O
on O
spatial O
learning O
deficits O
in O
aged O
rats O
. O

MATERIALS O
AND O
METHODS O
: O
A O
seven O
- O
wavelength O
frequency O
- O
domain O
photon O
migration O
probe O
was O
used O
to O
perform O
noninvasive O
NIR O
measurements O
in O
the O
breasts O
of O
28 O
healthy O
women O
, O
both O
pre O
- O
and O
postmenopausal O
, O
aged O
18 O
- O
64 O
years O
. O

YACs O
, O
BACs O
, O
cosmids O
, O
and O
STSs O
are O
defined O
to O
aid O
in O
further O
study O
of O
this O
gene O
. O

HCVR O
- O
L O
significantly O
increased O
dVAS O
/ O
dPCO2 O
to O
4 O
. O
9 O
+ O
/ O
- O
0 O
. O
7 O
mm O
/ O
Torr O
compared O
to O
HCVR O
- O
S O
( O
p O
< O
0 O
. O
05 O
) O
. O

The O
MEF B
- I
2 I
proteins I
are O
a O
family O
of O
transcriptional O
activators O
that O
have O
been O
detected O
in O
a O
wide O
variety O
of O
cell O
types O
. O

The O
ferric B
uptake I
regulation I
( I
fur I
) I
gene I
product I
participates O
in O
regulating O
expression O
of O
the O
manganese B
- I
and I
iron I
- I
containing I
superoxide I
dismutase I
genes I
of I
Escherichia I
coli I
. O

To O
date O
, O
6 O
mammalian B
GRKs I
have O
been O
identified O
by O
molecular O
cloning O
. O

[ O
( O
OP O
) O
2Cu O
] O
+ O
also O
detected O
protections O
in O
the O
C O
alpha O
- O
helix O
, O
the O
interdomain O
hinge O
, O
and O
beta O
- O
strands O
2 O
- O
7 O
. O

The O
aim O
of O
the O
present O
study O
was O
to O
investigate O
the O
influence O
of O
long O
term O
intravenous O
administration O
of O
naftidrofuryl O
( O
Dusodril O
- O
Lipha O
Arzn O
) O
twice O
daily O
in O
a O
dose O
of O
200 O
mg O
in O
continuous O
, O
4 O
- O
hour O
infusion O
in O
500 O
ml O
0 O
. O
9 O
% O
NaCl O
to O
the O
patients O
suffering O
from O
a O
peripheral O
arterial O
occlusive O
disease O
( O
PAOD O
) O
in O
a O
clinical O
condition O
with O
special O
attention O
paid O
to O
transcutaneous O
partial O
oxygen O
pressure O
measurements O
( O
tcPO2 O
) O
and O
rheographic O
parameters O
. O

Vertebrate B
U6 I
small I
nuclear I
RNA I
( I
snRNA I
) I
loci I
exemplify O
a O
novel O
class O
of O
polymerase B
III I
- I
transcribed I
genes I
that O
lack O
an O
intragenic O
control O
region O
( O
ICR O
) O
. O

Elements O
of O
the O
hAT B
transposon I
family I
, O
such O
as O
the O
maize B
activator I
( O
Ac B
) O
, O
have O
been O
discovered O
in O
a O
large O
number O
of O
eukaryotic O
species O
. O

Judge O
Christian O
Byk O
renders O
service O
to O
the O
Steering O
Committee O
on O
Bioethics O
of O
the O
Council O
of O
Europe O
( O
CDBI O
) O
by O
proposing O
a O
draft O
of O
the O
protocol O
destined O
to O
fill O
in O
a O
gap O
in O
international O
law O
on O
the O
status O
of O
the O
human O
embryo O
. O

Correlation O
coefficients O
between O
SF O
thickness O
and O
NIR O
optical O
density O
readings O
at O
940 O
nm O
( O
OD1 O
) O
and O
950 O
nm O
( O
OD2 O
) O
wavelengths O
ranged O
from O
r O
= O
- O
0 O
. O
30 O
( O
subscapula O
) O
to O
r O
= O
- O
0 O
. O
67 O
( O
biceps O
) O
for O
OD1 O
and O
r O
= O
- O
0 O
. O
39 O
( O
axilla O
) O
to O
r O
= O
- O
0 O
. O
68 O
( O
biceps O
) O
for O
OD2 O
. O

We O
found O
both O
the O
v B
- I
Ras I
- O
and O
PC B
- I
PLC I
- O
transformed O
cells O
to O
be O
insensitive O
to O
stimulation O
with O
platelet B
- I
derived I
growth I
factor I
( O
PDGF B
) O
. O

Analysis O
of O
domain O
deletion O
mutants O
demonstrated O
strong O
synergy O
between O
the O
RRM O
and O
a O
central O
degenerate O
RRM O
repeat O
in O
binding O
to O
RNA O
. O

Sequence O
analysis O
of O
the O
catfish O
JH B
- I
CH I
intron O
suggests O
that O
several O
sequences O
are O
present O
which O
appear O
similar O
to O
important O
transcriptional O
regulatory O
elements O
found O
within O
JH B
- O
CH B
introns O
of O
higher O
vertebrates O
. O

The O
patient O
groups O
consisted O
of O
31 O
with O
dilated O
cardiomyopathy O
, O
22 O
with O
hypertrophic O
cardiomyopathy O
, O
38 O
with O
myocardial O
infarction O
, O
15 O
with O
angina O
pectoris O
and O
26 O
with O
rheumatic O
valvular O
disease O
. O

100 O
. O

However O
, O
the O
pre3 B
- I
2 I
mutation O
strengthened O
phenotypes O
induced O
by O
other O
20 B
S I
proteasomal I
mutations O
, O
indicating O
that O
the O
peptidylglutamyl O
peptide O
- O
hydrolyzing O
activity O
has O
to O
fulfill O
some O
rescue O
functions O
. O

Whereas O
Sprague O
- O
Dawleys O
displayed O
lights O
- O
off O
and O
lights O
- O
on O
peaks O
of O
ingestive O
activity O
, O
only O
a O
minority O
of O
Fisher O
- O
344s O
displayed O
a O
consistent O
lights O
- O
on O
peak O
of O
ingestive O
activity O
. O

RESULTS O
: O
In O
76 O
trials O
, O
5 O
, O
351 O
patients O
received O
24 O
different O
regimens O
of O
droperidol O
. O

The O
tro B
operon I
is O
flanked O
by O
a O
Holliday B
structure I
DNA I
helicase I
homolog I
( O
upstream O
) O
and O
two O
ORFs O
representing O
a O
purine B
nucleoside I
phosphorylase I
homolog I
and O
tpp15 B
, O
a O
previously O
characterized O
gene O
encoding O
a O
membrane O
lipoprotein O
( O
downstream O
) O
. O

We O
report O
nine O
consecutive O
cases O
of O
ACB O
, O
which O
occurred O
in O
five O
males O
and O
four O
females O
and O
were O
detected O
in O
11 O
, O
159 O
routine O
spiral O
CT O
examinations O
of O
the O
chest O
, O
performed O
between O
1994 O
and O
1998 O
. O

In O
order O
to O
search O
for O
mutations O
in O
the O
multicopy O
RBM B
genes I
that O
might O
be O
associated O
with O
male O
infertility O
, O
we O
have O
used O
sequence O
data O
from O
the O
reported O
cDNA O
clone O
to O
determine O
the O
intron O
exon O
boundaries O
of O
the O
YRRM B
1 I
gene I
. O

98 O
, O
93 O
- O
98 O
) O
. O

Studies O
on O
the O
excretion O
of O
selenium O
in O
urine O
and O
feces O
and O
distribution O
in O
the O
organs O
of O
rats O
by O
a O
method O
of O
radioactivity O
measurement O

The O
use O
of O
the O
two O
pharmacokinetic O
parameters O
, O
t1 O
/ O
2 O
and O
Cltp O
, O
as O
indices O
of O
drug O
elimination O
ability O
are O
discussed O
. O

Gastric O
secretory O
inhibition O
induced O
by O
three O
methyl O
analogs O
of O
prostaglandin O
E2 O
administered O
intragastrically O
to O
man O
. O

By O
RT O
- O
PCR O
, O
different O
levels O
of O
E75 B
expression O
can O
be O
detected O
in O
the O
epidermis O
, O
nerve O
cord O
and O
the O
eyestalk O
of O
early O
pre O
- O
molt O
shrimp O
. O

The O
vicinity O
of O
large O
industrial O
emissions O
of O
lead O
, O
where O
mean O
Pb O
- O
B O
concentrations O
were O
usually O
twice O
as O
high O
as O
in O
rural O
areas O
of O
the O
Katowice O
voivodship O
. O

Forty O
- O
five O
per O
cent O
of O
the O
dose O
was O
excreted O
as O
methyldopa O
as O
opposed O
to O
18 O
% O
normally O
seen O
after O
oral O
methyldopa O
dosages O
. O

No O
cleavage O
of O
gp B
130 I
was O
observed O
in O
analogous O
pulse O
- O
chase O
radiolabelling O
of O
Ad B
- I
gB I
- O
infected O
human O
fibroblasts O
, O
even O
though O
these O
cells O
are O
permissive O
for O
HCMV O
replication O
and O
can O
process O
the O
native O
gB B
molecule I
. O

A O
phylogenetic O
comparison O
of O
32 O
species O
showed O
minor O
differences O
in O
the O
apoB B
mRNA I
sequence I
, O
and O
the O
apoB B
mRNA I
from O
31 O
species O
was O
robustly O
edited O
in O
vitro O
. O

The O
other O
five O
from O
within O
the O
SRO O
may O
provide O
an O
entrance O
point O
for O
the O
cloning O
of O
candidate O
genes O
for O
neuroblastoma O
. O

Sugar O
analysis O
was O
performed O
on O
alpha B
- I
TM I
to O
investigate O
a O
possible O
biosynthetic O
mechanism O
for O
part O
- O
time O
PGs O
. O

Pediatric O
patients O
- O
- O
handle O
with O
care O
. O

These O
include O
the O
genes O
, O
undefined B
1 I
( O
UD1 B
) O
, O
UD2 B
, O
and O
UD3 B
, O
each O
coding O
for O
proteins O
of O
unknown O
function O
, O
the O
ken B
gene I
encoding O
a O
new O
Kruppel B
- I
like I
putative I
transcription I
factor I
, O
the O
fly O
homologues O
of O
the O
mammalian B
mitochondrial I
trifunctional I
enzyme I
( O
thiolase B
) O
, O
and O
the O
TAR B
DNA I
- I
binding I
protein I
- I
43 I
( O
TBPH B
) O
, O
the O
first O
nonvertebrate O
member O
of O
the O
transmembrane B
4 I
superfamily I
( O
TM4SF B
) O
gene O
, O
a O
new O
homeodomain B
gene I
, O
and O
a O
gene O
coding O
for O

a O
putative O
nuclear B
binding I
protein I
( O
PNBP B
) O
that O
is O
homologous O
to O
maleless B
, O
and O
a O
Copia B
- I
like I
element I
. O

TRAF2 B
has O
previously O
been O
demonstrated O
to O
activate O
both O
transcription B
factor I
nuclear I
factor I
kappaB I
( O
NFkappaB B
) O
and O
the O
c B
- I
Jun I
N I
- I
terminal I
kinase I
/ O
stress B
- I
activated I
protein I
kinase I
( O
JNK B
/ O
SAPK B
) O
pathway O
, O
which O
in O
turn O
stimulates O
transcription B
factor I
activating I
protein I
1 I
( O
AP1 B
) O
mainly O
via O
phosphorylation O
of O
the O
c B
- I
Jun I
component O
. O

A O
procaryotic O
regulatory O
factor O
with O
a O
histone B
H1 I
- I
like I
carboxy I
- I
terminal I
domain I
: O
clonal O
variation O
of O
repeats O
within O
algP B
, O
a O
gene O
involved O
in O
regulation O
of O
mucoidy O
in O
Pseudomonas O
aeruginosa O
. O

The O
host O
range O
( O
HR O
) O
of O
poliovirus O
is O
thought O
to O
be O
primarily O
determined O
by O
a O
cell O
surface O
molecule O
that O
functions O
as O
poliovirus B
receptor I
( O
PVR B
) O
, O
since O
it O
has O
been O
shown O
that O
transgenic O
mice O
are O
made O
poliovirus O
sensitive O
by O
introducing O
the O
human B
PVR I
gene I
into O
the O
genome O
. O

Human B
alcohol I
dehydrogenase I
( O
ADH B
) O
exists O
as O
a O
heterogeneous O
group O
of O
isozymes O
capable O
of O
oxidizing O
a O
wide O
variety O
of O
aliphatic O
and O
aromatic O
alcohols O
. O

The O
control O
of O
human B
aromatase I
expression O
is O
complex O
in O
that O
several O
promoters O
drive O
aromatase B
expression O
in O
a O
tissue O
- O
specific O
manner O
. O

In O
addition O
, O
renal O
sympathetic O
activity O
was O
measured O
in O
a O
separate O
group O
of O
rats O
. O

This O
incompatibility O
phenotype O
requires O
the O
global O
transcriptional O
repressor O
, O
KorB B
, O
and O
the O
target O
for O
incC B
- O
mediated O
incompatibility O
is O
a O
KorB B
- I
binding I
site I
( O
O B
( I
B I
) O
) O
. O

Bodily O
referents O
and O
the O
experience O
of O
affect O
. O

The O
effect O
of O
the O
negative O
regulatory O
element O
is O
negated O
by O
the O
viral B
IE2 I
protein I
( O
L O
. O

RXRalpha B
/ O
RARalpha B
heterodimers O
and O
HNF B
- I
4 I
homodimers I
bind O
to O
DR B
- I
1 I
motifs I
on O
elements O
B O
and O
I4 O
, O
respectively O
. O

Evidence O
for O
involvement O
of O
proteins B
HU I
and O
RpoS B
in O
transcription O
of O
the O
osmoresponsive O
proU B
operon I
in I
Escherichia I
coli I
. O

However O
, O
a O
lesser O
extent O
effect O
was O
noticed O
in O
nonpregnant O
than O
seen O
in O
pregnant O
rats O
. O

Morphine O
administration O
acutely O
reduced O
plasma O
clearance O
of O
sulfobromophthalein O
( O
BSP O
) O
in O
mice O
and O
increased O
hepatic O
retention O
of O
this O
dye O
. O

Buchstein O
, O
L O
. O
- O
L O
. O

Intraspinal O
and O
mediastinal O
foregut O
cyst O
compressing O
the O
spinal O
cord O
. O

The O
nutraceuticals O
of O
specific O
vitamins O
, O
minerals O
, O
phytoestrogens O
, O
and O
essential O
fatty O
acid O
supplementations O
are O
a O
vital O
component O
of O
the O
risk O
reduction O
health O
program O
. O

Characterization O
of O
a O
unique O
protein O
component O
of O
yeast B
RNase I
MRP I
: O
an O
RNA O
- O
binding O
protein O
with O
a O
zinc O
- O
cluster O
domain O
. O

The O
present O
study O
indicates O
that O
the O
high O
- O
dose O
5 O
- O
fluorouracil O
regimen O
shows O
weak O
activity O
in O
advanced O
pancreatic O
cancer O
which O
seems O
comparable O
to O
gemcitabine O
. O

Like O
the O
nuclear O
envelope O
, O
the O
intranuclear O
double O
membrane O
lamellae O
enclosed O
a O
defined O
cisterna O
that O
was O
interrupted O
by O
pores O
but O
, O
unlike O
the O
nuclear O
envelope O
pores O
, O
they O
lacked O
NPCs O
. O

The O
22 O
- O
d O
orbital O
flight O
of O
rats O
onboard O
the O
Cosmos O
- O
605 O
biosatellite O
was O
followed O
by O
inhibition O
of O
erythroblastosis O
, O
alteration O
in O
the O
morphology O
of O
megakaryocytes O
, O
and O
stimulation O
of O
myelopoiesis O
. O

An O
OD O
650 O
greater O
than O
0 O
. O
15 O
appears O
to O
be O
a O
rapid O
, O
reliable O
indicator O
of O
fetal O
lung O
maturity O
. O

The O
role O
of O
PORT O
in O
the O
treatment O
of O
N2 O
tumours O
is O
not O
clear O
and O
may O
justify O
further O
research O
. O

We O
concluded O
that O
RXR B
- I
gamma I
induced O
terminal O
differentiation O
in O
SCC O
lines O
, O
suggesting O
a O
potential O
tumor O
suppressor O
function O
for O
this O
transcription O
factor O
. O

Phosphorylated O
tyrosine O
residues O
were O
subsequently O
identified O
by O
sequencing O
the O
separated O
phosphopeptides O
by O
matrix O
assisted O
laser O
desorption O
ionization O
mass O
spectrometry O
( O
MALDI O
- O
MS O
) O
and O
Edman O
degradation O
. O

The O
infection O
of O
cells O
with O
Moloney O
murine O
leukemia O
virus O
( O
M O
- O
MuLV O
) O
causes O
an O
increase O
in O
specific O
cellular O
gene O
products O
, O
including O
the O
major B
histocompatibility I
complex I
( I
MHC I
) I
class I
I I
antigens I
. O

These O
data O
demonstrate O
that O
oleosin B
gene I
transcription O
is O
regulated O
in O
a O
tissue O
- O
specific O
and O
temporally O
regulated O
manner O
and O
clearly O
indicate O
that O
oleosin B
protein I
expression O
is O
co O
- O
ordinated O
primarily O
at O
the O
transcriptional O
level O
. O

Myocardial O
technetium O
- O
99m O
- O
teboroxime O
uptake O
during O
adenosine O
- O
induced O
hyperemia O
in O
dogs O
with O
either O
a O
critical O
or O
mild O
coronary O
stenosis O
: O
comparison O
to O
thallium O
- O
201 O
and O
regional O
blood O
flow O
. O

UICC O
criteria O
) O
for O
VM O
and O
35 O
% O
for O
VE O
( O
not O
significant O
) O
. O

Although O
immunoglobulin B
and O
CRP B
concentration O
increased O
, O
anemia O
obviously O
improved O
with O
hemoglobin B
levels O
increasing O
from O
4 O
. O
8 O
g O
/ O
dl O
to O
8 O
. O
5 O
g O
/ O
dl O
without O
any O
side O
effects O
. O

Diff O
- O
Quik O
stain O
for O
Tzanck O
smears O
. O

Experience O
in O
using O
the O
Romashka O
- O
1 O
laser O
surgical O
unit O
in O
treating O
suppurative O
wounds O

At O
D28 O
, O
149 O
of O
386 O
patients O
( O
49 O
% O
) O
had O
had O
episodes O
of O
automatic O
mode O
switch O
prompted O
by O
atrial O
arrhythmias O
. O

Together O
, O
these O
results O
implicate O
Fal1p B
in O
the O
18S B
rRNA I
maturation O
pathway O
rather O
than O
in O
translation O
initiation O
. O

Most O
other O
differences O
between O
these O
two O
Leporipoxviruses O
are O
located O
in O
the O
telomeres O
. O

We O
challenge O
each O
of O
55 O
consecutive O
ragweed O
( O
RW O
) O
- O
allergic O
patients O
with O
hay O
fever O
and O
with O
graded O
increasing O
doses O
of O
ragweed O
extract O
to O
investigate O
the O
frequency O
and O
relationship O
between O
the O
early O
( O
ER O
) O
, O
late O
( O
LPR O
) O
, O
and O
rechallenge O
reactions O
( O
RCRs O
) O
to O
nasal O
challenge O
. O

The O
expression O
of O
CAPL B
, O
a O
second O
protein O
involved O
in O
calcium O
metabolism O
, O
was O
only O
moderately O
elevated O
in O
the O
doxorubicin O
- O
resistant O
cells O
. O

The O
central O
region O
of O
the O
sarcomere O
, O
coincident O
with O
the O
M O
line O
, O
was O
selectively O
labeled O
with O
antibodies O
to O
the O
short O
C O
- O
terminal O
form O
. O

Despite O
this O
dependency O
, O
however O
, O
a O
B B
. I
japonicum I
fixK I
mutant I
did O
not O
have O
the O
phenotypic O
characteristics O
of O
B B
. I
japonicum I
fixL I
and O
fixJ B
mutants I
: O
the O
fixK B
mutant I
was O
neither O
Fix B
- I
in O
symbiosis O
with O
soybean O
plants O
nor O
defective O
in O
anaerobic O
respiration O
with O
nitrate O
as O
the O
terminal O
electron O
acceptor O
. O

ANL O
- O
7535 O
. O

The O
QCS O
appears O
comparable O
with O
the O
MMSE O
and O
is O
quicker O
to O
administer O
. O

We O
used O
high O
- O
resolution O
ultrasound O
to O
characterize O
postprandial O
antral O
excursion O
characteristics O
in O
15 O
healthy O
volunteers O
. O

Copyright O
1998 O
Academic O
Press O
. O

Human B
PDNP3 I
is O
expressed O
in O
glioma O
cells O
, O
prostate O
, O
and O
uterus O
, O
but O
not O
in O
the O
alimentary O
tract O
. O

Epithelial O
cells O
had O
abnormal O
and O
accelerated O
exfoliation O
which O
resulted O
in O
multifocal O
epithelial O
defects O
. O

Tight O
- O
binding O
band O
structure O
calculations O
for O
beta O
- O
MNX O
( O
M O
= O
Zr O
, O
X O
= O
Cl O
, O
Br O
; O
M O
= O
Hf O
, O
X O
= O
Cl O
) O
, O
ZrCl O
, O
and O
Y O
( O
2 O
) O
C O
( O
2 O
) O
Br O
( O
2 O
) O
are O
reported O
. O

Screening O
of O
a O
human O
foetal O
brain O
genomic O
DNA O
library O
allowed O
us O
to O
isolate O
an O
EcoRI B
- O
EcoRI B
fragment O
containing O
6 O
kb O
of O
the O
5 O
' O
- O
flanking O
region O
, O
the O
open O
reading O
frame O
and O
4 O
kb O
of O
the O
3 O
' O
- O
flanking O
region O
of O
the O
alpha2C4 B
gene I
. O

The O
psychosomatic O
approach O
to O
temporomandibular O
arthrosis O

The O
rate O
of O
decrease O
of O
PRA O
after O
REM O
onset O
closely O
approximates O
the O
most O
recent O
estimations O
of O
PRA O
half O
- O
life O
, O
which O
suggests O
that O
REM O
onset O
is O
associated O
with O
a O
virtual O
cessation O
in O
renin B
production O
. O

Similarly O
, O
interfering O
with O
MEKK B
, O
which O
lies O
upstream O
of O
JNK1 B
, O
using O
a O
dominant O
negative O
expression O
vector O
reduced O
MMP B
- I
9 I
promoter I
activity O
over O
the O
same O
concentration O
range O
which O
repressed O
the O
AP B
- I
1 I
- O
thymidine B
kinase I
CAT B
reporter O
construct O
. O

There O
was O
no O
difference O
in O
serum O
albumin B
and O
transferrin B
levels O
, O
but O
serum B
prealbumin I
levels O
in O
the O
group O
fed O
early O
were O
more O
desirable O
than O
those O
of O
the O
control O
group O
( O
from O
15 O
. O
8 O
+ O
/ O
- O
2 O
. O
5 O
mg O
/ O
dl O
to O
28 O
. O
9 O
+ O
/ O
- O
3 O
. O
8 O
mg O
/ O
dl O
vs O
from O
18 O
. O
0 O
+ O
/ O
- O
2 O
. O
0 O
mg O
/ O
dl O
to O
25 O
. O
9 O
+ O
/ O
- O
3 O
. O
9 O
mg O
/ O
dl O
) O
. O

The O
repeats O
are O
highly O
conserved O
both O
within O
a O
given O
element O
as O
well O
as O
between O
different O
members O
of O
the O
family O
( O
less O
than O
10 O
% O
divergence O
) O
. O

Utility O
of O
thallium O
- O
201 O
scintigraphy O
in O
detecting O
right O
ventricular O
dysfunction O
in O
pulmonary O
embolism O
. O

CONCLUSIONS O
: O
The O
system O
developed O
in O
this O
study O
can O
be O
used O
as O
a O
method O
to O
detect O
air O
- O
trapping O
during O
TGI O
. O

The O
carboxyl O
- O
terminal O
75 O
amino O
acids O
of O
the O
two O
proteins O
contain O
the O
bHLH O
motif O
and O
differ O
from O
each O
other O
by O
only O
three O
conservative O
amino O
acid O
changes O
, O
while O
the O
amino O
- O
terminal O
portions O
are O
markedly O
divergent O
from O
each O
other O
. O

In O
a O
rat O
model O
of O
SAH O
, O
we O
assessed O
BBB O
changes O
by O
means O
of O
the O
quantitative O
[ O
14C O
] O
- O
alpha O
- O
aminoisobutyric O
acid O
technique O
. O

Pollinosis O
in O
the O
U O
. O
A O
. O
R O
. O

Northern O
blot O
analysis O
of O
enoyl B
- I
ACP I
reductase I
mRNA I
steady O
- O
state O
levels O
during O
seed O
development O
suggests O
that O
the O
increase O
in O
enzyme O
activity O
during O
the O
phase O
of O
storage O
lipid O
accumulation O
is O
regulated O
at O
the O
level O
of O
gene O
expression O
. O

Time O
between O
tests O
( O
usually O
less O
than O
one O
year O
) O
did O
not O
affect O
the O
correlations O
, O
but O
MMPI O
response O
- O
set O
variables O
( O
L O
, O
F O
, O
K O
, O
F O
- O
K O
) O
did O
. O

The O
differences O
in O
binding O
( O
Kdapp O
) O
, O
incorporation O
, O
and O
extension O
kinetics O
of O
8 O
- O
oxo O
- O
dGTP O
compared O
to O
normal O
dNTP O
incorporation O
at O
template O
8 O
- O
oxo O
- O
G O
adducts O
indicate O
that O
polymerase O
fidelity O
does O
not O
depend O
solely O
upon O
the O
overall O
geometry O
of O
Watson O
- O
Crick O
base O
pairs O
and O
reflects O
the O
asymmetry O
of O
the O
enzyme O
active O
site O
. O

This O
constant O
expression O
profile O
, O
coupled O
with O
the O
observation O
that O
over O
- O
expression O
of O
mSin3A B
does O
not O
augment O
the O
anti B
- I
Myc I
activity O
of O
Mxi1 B
- I
SR I
in O
the O
rat O
embryo O
fibroblast O
( O
REF O
) O
transformation O
assay O
, O
suggests O
that O
mSin3A B
is O
not O
a O
limiting O
factor O
in O
the O
regulation O
of O
Myc B
superfamily I
function O
. O

The O
sequences O
at O
- O
10 O
and O
- O
35 O
relative O
to O
the O
transcriptional O
starting O
site O
showed O
55 O
% O
homology O
with O
the O
consensus O
sequences O
of O
the O
Escherichia B
coli I
sigma I
70 I
- I
type I
promoter I
. O

The O
results O
were O
subjected O
to O
analysis O
of O
23 O
autopsies O
carried O
out O
in O
children O
dying O
of O
intoxication O
with O
Amanita O
phalloides O
. O

METHODS O
: O
The O
Die O
Deutsche O
Diabetes O
Dialyse O
Studie O
is O
a O
prospective O
randomized O
placebo O
- O
controlled O
trial O
that O
tests O
the O
hypothesis O
that O
atorvastatin O
, O
a O
hydroxymethyl B
- I
glutaryl I
coenzyme I
A I
reductase I
inhibitor O
, O
decreases O
the O
rate O
of O
cardiovascular O
mortality O
and O
of O
nonfatal O
myocardial O
infarction O
in O
patients O
with O
type O
2 O
diabetes O
who O
have O
been O
on O
hemodialysis O
treatment O
for O
no O
more O
than O
two O
years O
. O

There O
was O
no O
significant O
correlation O
between O
serum O
amiodarone O
or O
desethylamiodarone O
levels O
and O
dosage O
of O
amiodarone O
. O

The O
effect O
of O
monaural O
middle O
ear O
destruction O
on O
postnatal O
development O
of O
mouse O
inferior O
colliculus O
. O

This O
unusual O
behaviour O
may O
be O
explained O
by O
the O
18 B
amino I
acid I
- I
long I
CDR I
- I
H3 I
and O
could O
be O
of O
value O
in O
the O
design O
of O
' O
single O
domain O
' O
antibodies O
. O

A O
definitive O
answer O
to O
the O
question O
must O
come O
from O
large O
scale O
mortality O
studies O
of O
patients O
in O
whom O
the O
risk O
/ O
benefit O
ratio O
of O
thrombolysis O
is O
not O
unacceptably O
high O
, O
in O
whom O
electrocardiographic O
criteria O
of O
infarction O
are O
unequivocal O
, O
and O
in O
whom O
treatment O
can O
be O
initiated O
early O
after O
the O
onset O
of O
symptoms O
with O
regimens O
that O
will O
induce O
not O
only O
early O
recanalization O
, O
but O
also O
sustained O
patency O
in O
infarct O
- O
related O
arteries O
. O

Parameters O
of O
sperm O
quality O
were O
evaluated O
before O
and O
after O
freezing O
/ O
thawing O
. O

It O
has O
also O
been O
previously O
demonstrated O
that O
LPS O
treatment O
of O
splenic O
B O
cells O
from O
athymic O
mice O
results O
in O
a O
decrease O
in O
steady O
state O
mRNA O
encoding O
the O
A B
alpha I
class I
II I
protein I
. O

Amongst O
53 O
" O
inoperable O
" O
( O
T4 O
, O
N0 O
, O
N1 O
, O
T O
. O
, O
N2 O
, O
N3 O
) O
cases O
, O
5 O
( O
10 O
% O
) O
had O
a O
positive O
scan O
and O
5 O
a O
doubtful O
scan O
. O

Chemical O
structure O
of O
antibiotic O
SF O
- O
837 O
. O

Aspirin O
therapy O
in O
the O
rheumatic O
diseases O
. O

Is O
conventional O
sperm O
analysis O
of O
any O
use O
? O

The O
computerised O
records O
of O
867 O
couples O
were O
used O
to O
investigate O
the O
prognostic O
significance O
of O
semen O
volume O
, O
motility O
, O
density O
and O
morphology O
. O

It O
was O
observed O
that O
muscle O
infarction O
in O
the O
IPC O
( O
24 O
+ O
/ O
- O
2 O
% O
) O
and O
preischemic O
LMK O
( O
21 O
+ O
/ O
- O
2 O
% O
) O
groups O
were O
smaller O
( O
P O
< O
0 O
. O
05 O
) O
than O
that O
in O
the O
control O
( O
42 O
+ O
/ O
- O
2 O
% O
) O
. O

High O
- O
affinity O
binding O
was O
also O
observed O
with O
recombinant B
SH2 I
domains I
from O
v B
- I
src I
and O
v B
- I
fps I
, O
raising O
the O
possibility O
of O
protein O
- O
protein O
interactions O
between O
various O
members O
of O
the O
cytoplasmic B
PTK I
family I
. O

Mutational O
analysis O
of O
the O
putative O
effector O
domain O
of O
the O
GTP O
- O
binding O
Ypt1 I
protein I
in O
yeast O
suggests O
specific O
regulation O
by O
a O
novel O
GAP B
activity O
. O

The O
interaction O
obtained O
with O
DBM O
scores O
was O
further O
qualified O
by O
a O
three O
- O
way O
interaction O
that O
limited O
this O
pattern O
to O
participants O
scoring O
higher O
on O
self O
- O
deception O
. O

The O
broad O
range O
protein B
tyrosine I
kinase I
inhibitor O
genistein O
and O
the O
phosphatidylinositol B
3 I
- I
kinase I
inhibitors O
wortmannin O
and O
LY O
294002 O
( O
2 O
- O
( O
4 O
- O
morpholinyl O
) O
- O
8 O
- O
phenyl O
- O
4H O
- O
1 O
- O
benzopyran O
- O
4 O
- O
one O
) O
also O
blocked O
adenosine B
A3 I
receptor I
stimulation O
of O
p42 B
/ O
p44 B
MAPK O
. O

Surprisingly O
, O
although O
Mist1 B
binds O
to O
E O
- O
boxes O
in O
vivo O
, O
the O
Mist1 B
protein I
lacks O
a O
functional O
transcription O
activation O
domain O
. O

As O
exogenously O
expressed O
PKR B
can O
form O
heterodimers O
with O
endogenous O
PKR B
, O
the O
results O
obtained O
on O
the O
functional O
characterization O
of O
mutant O
forms O
of O
PKR B
have O
been O
taken O
with O
caution O
. O

Orlistat O
is O
a O
specific O
lipase B
inhibitor O
that O
impairs O
fat O
absorption O
, O
thereby O
reducing O
fat O
uptake O
. O

The O
metalloproteinase O
mediating O
Met B
cleavage O
was O
specifically O
inhibited O
by O
the O
tissue B
inhibitor I
of I
metalloproteinases I
( I
TIMP I
) I
- I
3 I
, O
but O
not O
by O
TIMP B
- I
1 I
or O
TIMP B
- I
2 I
. O

This O
demonstrates O
a O
relative O
independence O
of O
the O
given O
synchronizer O
of O
rhythmicity O
at O
a O
tissue O
level O
from O
any O
influence O
of O
the O
higher O
regulatory O
center O
. O

It O
is O
also O
conceivable O
that O
elevated O
insulin B
levels O
may O
cause O
hypertriglyceridaemia O
and O
possibly O
other O
abnormalities O
of O
lipid O
metabolism O
. O

Flicker O
thresholds O
were O
measured O
from O
1 O
to O
40 O
Hz O
with O
a O
vertical O
- O
line O
target O
used O
for O
the O
asynchrony O
thresholds O
. O

The O
clr1 B
locus I
regulates O
the O
expression O
of O
the O
cryptic O
mating B
- I
type I
loci I
of O
fission O
yeast O
. O

Use O
of O
ELISAs O
for O
the O
diagnosis O
of O
canine O
atopy O
. O

A O
clinical O
study O
. O

METHODS O
. O

Instead O
, O
HBx B
is O
shown O
to O
activate O
the O
cyclin B
A I
promoter I
, O
induce O
cyclin B
A I
- O
cyclin B
- O
dependent I
kinase I
2 I
complexes O
, O
and O
promote O
cycling O
of O
growth O
- O
arrested O
cells O
into O
G1 O
through O
a O
pathway O
involving O
activation O
of O
Src B
tyrosine I
kinases I
. O

Growth B
hormone I
exerts O
its O
effects O
on O
the O
ovarian O
follicular O
cycle O
directly O
or O
by O
local O
production O
of O
insulin B
- I
like I
growth I
factor I
1 I
( O
IGF B
- I
1 I
) O
. O

The O
aim O
of O
this O
study O
was O
to O
evaluate O
the O
impact O
of O
the O
ESRD O
treatment O
modality O
on O
the O
quality O
of O
life O
in O
patients O
with O
Type O
I O
( O
insulin B
- O
dependent O
) O
diabetes O
mellitus O
. O

The O
subjects O
were O
started O
on O
indomethacin O
25 O
mg O
thrice O
daily O
. O

A O
Buddhist O
view O
of O
abortion O
. O

The O
arterial O
blood O
pressure O
was O
measured O
by O
using O
bloody O
method O
in O
anesthizied O
animals O
. O

After O
15 O
min O
of O
hemorrhage O
the O
cardiovascular O
parameters O
were O
the O
same O
in O
fed O
and O
starved O
animals O
, O
but O
at O
45 O
min O
striking O
differences O
were O
observed O
. O

Decidual B
/ I
trophoblast I
prolactin I
- I
related I
protein I
: O
characterization O
of O
gene O
structure O
and O
cell O
- O
specific O
expression O
. O

Fluorescence O
in O
situ O
hybridization O
was O
used O
to O
investigate O
the O
physical O
distribution O
and O
revealed O
that O
both O
retrotransposon O
families O
are O
present O
on O
all O
sugar O
beet O
chromosomes O
and O
largely O
excluded O
from O
chromosomal O
regions O
harbouring O
the O
18S B
- O
5 B
. I
8S I
- O
25S B
rRNA O
genes O
. O

Plasma O
NE O
and O
E O
increased O
to O
significantly O
higher O
values O
after O
15 O
min O
in O
the O
young O
subjects O
: O
1 O
. O
68 O
+ O
/ O
- O
0 O
. O
18 O
vs O
. O

An O
insertion O
sequence O
element O
( O
IS1170 O
) O
was O
identified O
upstream O
of O
the O
nimC B
gene I
. O

The O
organelles O
synthesize O
their O
own O
set O
of O
Fe B
/ I
S I
proteins I
, O
and O
they O
initiate O
the O
generation O
of O
extramitochondrial O
Fe B
/ I
S I
proteins I
. O

Most O
significantly O
, O
the O
cyclin B
E I
- O
Cdk2 B
complex O
is O
maximally O
active O
at O
the O
G1 O
/ O
S O
transition O
, O
and O
overexpression O
of O
cyclin B
E I
decreases O
the O
time O
it O
takes O
the O
cell O
to O
complete O
G1 O
and O
enter O
S O
phase O
. O

A O
genomic O
clone O
of O
the O
chicken B
osteopontin I
- I
encoding I
gene I
( O
opn B
) O
was O
isolated O
and O
found O
to O
be O
organized O
as O
follows O
: O
an O
untranslated O
5 O
' O
exon O
; O
a O
signal O
peptide O
; O
a O
recognition O
sequence O
for O
phosphorylation O
by O
casein B
kinase I
II I
; O
a O
domain O
containing O
a O
possible O
O O
- O
linkage O
site O
for O
glycosylation O
; O
a O
second O
casein B
kinase I
II I
phosphorylation I
site I
; O
an O
exon O
containing O
three O
functional O
regions O
, O
the O
poly O
- O
Asp O
sequence O
of O
seven O
consecutive O
Asp O
residues O
, O
the O
RGD O
integrin B
recognition I
site I
and O
a O
potential O
N O

- O
linkage O
site O
for O
glycosylation O
; O
and O
a O
large O
C O
- O
terminal O
exon O
which O
also O
contains O
a O
potential O
N O
- O
linkage O
site O
for O
glycosylation O
. O

However O
, O
neither O
SF B
- I
1 I
nor O
NGF B
- I
IB I
alone O
, O
binding O
as O
monomers O
, O
increases O
transcription O
. O

Satisfactory O
correlation O
was O
also O
obtained O
between O
the O
in O
vivo O
and O
the O
in O
vitro O
results O
. O

Structural O
organization O
of O
the O
human B
Elk1 I
gene I
and O
its O
processed O
pseudogene B
Elk2 I
. O

After O
cumulative O
occlusions O
of O
15 O
, O
30 O
, O
45 O
, O
and O
90 O
min O
, O
transmural O
needle O
biopsies O
were O
taken O
from O
the O
ischemic O
area O
to O
be O
analyzed O
for O
adenine O
nucleotides O
, O
nucleosides O
, O
creatine O
phosphate O
, O
and O
ultrastructural O
changes O
. O

The O
prevalence O
of O
ocular O
/ O
adnexal O
SCC O
was O
significantly O
greater O
for O
all O
hair O
colors O
when O
compared O
with O
bay O
, O
brown O
, O
or O
black O
( O
P O
less O
than O
0 O
. O
01 O
) O
. O

Seven O
patients O
were O
without O
a O
syndromic O
diagnosis O
. O

In O
treatments O
R1 O
and O
R2 O
, O
feed O
quality O
was O
restricted O
by O
withholding O
concentrates O
for O
3 O
and O
4 O
. O
5 O
mo O
, O
respectively O
. O

In O
addition O
, O
while O
GCN1 B
is O
required O
in O
vivo O
for O
phosphorylation O
of O
eIF B
- I
2 I
alpha I
by O
GCN2 B
, O
cell O
extracts O
from O
gcn1 B
delta I
strains O
contained O
wild O
- O
type O
levels O
of O
GCN2 B
eIF B
- I
2 I
alpha I
- I
kinase I
activity O
. O

SCOF B
- I
1 I
localized O
to O
the O
nucleus O
but O
did O
not O
bind O
directly O
to O
either O
C O
- O
repeat O
/ O
dehydration O
( O
CRT O
/ O
DRE O
) O
or O
ABA O
responsive O
element O
( O
ABRE O
) O
, O
cis O
- O
acting O
DNA O
regulatory O
elements O
present O
in O
COR B
gene I
promoters I
. O

On O
the O
basis O
of O
these O
findings O
we O
suggest O
that O
the O
acceptor O
peptide O
binds O
the O
transferase O
in O
a O
beta O
- O
like O
conformation O
and O
that O
penultimate O
residue O
side O
chain O
steric O
interactions O
may O
play O
a O
role O
in O
determining O
extent O
that O
a O
given O
Ser O
or O
Thr O
is O
glycosylated O
. O

The O
concentration O
of O
vitamin O
B1 O
, O
B2 O
and O
B6 O
are O
found O
to O
be O
9 O
. O
96 O
, O
9 O
. O
92 O
and O
3 O
. O
01 O
mg O
, O
respectively O
in O
240 O
mg O
of O
capsule O
powder O
of O
a O
standard O
company O
( O
name O
has O
not O
been O
disclosed O
due O
to O
secrecy O
purpose O
) O
. O

Polymorphisms O
in O
the O
CCR5 B
genes I
of O
African O
green O
monkeys O
and O
mice O
implicate O
specific O
amino O
acids O
in O
infections O
by O
simian O
and O
human O
immunodeficiency O
viruses O
. O

Furthermore O
, O
we O
detect O
hCoch B
- I
5B2 I
on O
three O
overlapping O
YACs O
, O
two O
of O
which O
also O
contain O
one O
of O
the O
markers O
linked O
to O
DFNA9 B
. O

mCoch B
- I
5B2 I
was O
genetically O
mapped O
in O
the O
mouse O
to O
chromosome O
12 O
, O
in O
a O
region O
of O
homologous O
synteny O
with O
human O
14q11 O
. O
2 O
- O
q13 O
, O
which O
contains O
the O
asp1 B
( O
audiogenic B
seizure I
prone I
) O
locus O
in O
the O
mouse O
. O

Absence O
of O
nitroso O
formation O
from O
( O
14C O
) O
methomyl O
and O
sodium O
nitrite O
under O
simulated O
stomach O
conditions O
. O

Although O
no O
detectable O
phenotypes O
are O
associated O
with O
a O
disruption O
allele O
of O
ABP1 B
, O
mutations O
that O
create O
a O
requirement O
for O
this O
protein O
have O
now O
been O
isolated O
in O
the O
previously O
identified O
gene B
SAC6 I
and O
in O
two O
new O
genes O
, O
SLA1 B
and O
SLA2 B
. O

The O
local O
median O
scores O
( O
ranges O
) O
of O
the O
above O
tests O
were O
as O
follows O
- O
AMT O
: O
9 O
( O
6 O
- O
10 O
) O
; O
CMMS O
: O
25 O
( O
16 O
- O
28 O
) O
; O
SBT O
: O
2 O
( O
0 O
- O
10 O
) O
; O
WL O
- O
i O
: O
17 O
( O
8 O
- O
27 O
) O
; O
WL O
- O
d O
: O
5 O
( O
0 O
- O
10 O
) O
; O
WL O
- O
r O
: O
9 O
( O
1 O
- O
10 O
) O
; O
ST O
: O
13 O
( O
6 O
- O
25 O
) O
; O
BNT O
: O
14 O
( O
10 O
- O
15 O
) O
; O
CPT O
: O
1 O
( O
0 O
- O
3 O
) O
; O
BDT O
: O
19 O
( O
0 O
- O
42 O
) O
; O
OAT O
: O

20 O
( O
3 O
- O
33 O
) O
. O

Antigen O
dose O
- O
response O
curves O
were O
drawn O
, O
and O
the O
cumulative O
dose O
required O
for O
a O
35 O
% O
reduction O
in O
specific O
airway O
conductance O
was O
calculated O
and O
designated O
Provocation O
Dose O
( O
PD35 O
) O
. O

Cross O
- O
talk O
between O
transcription O
factors I
NF I
- I
kappa I
B I
and O
C B
/ I
EBP I
in O
the O
transcriptional O
regulation O
of O
genes O
. O

Elective O
replacement O
of O
the O
aortic O
root O
in O
Marfan O
' O
s O
syndrome O
. O

The O
detergent O
- O
solubilized O
complex O
oxidizes O
caldariella O
quinol O
at O
high O
rates O
and O
is O
completely O
inhibited O
by O
cyanide O
and O
by O
quinolone O
analogs O
, O
potent O
inhibitors O
of O
quinol B
oxidases I
. O

We O
have O
characterized O
a O
new O
cDNA O
, O
33k O
- O
6 O
, O
potentially O
encoding O
a O
tobacco O
33 B
kDa I
chloroplast I
RNP I
( O
cp33 B
) O
homologue O
. O

The O
regions O
encoding O
the O
mature O
proteinases O
were O
cloned O
into O
an O
expression O
vector O
and O
recombinant O
protein O
produced O
in O
Escherichia O
coli O
. O

Furthermore O
, O
CL100 B
suppresses O
the O
[ B
val12 I
] I
ras I
- O
induced O
activation O
of O
MAP B
kinase I
in O
a O
cell O
- O
free O
system O
from O
Xenopus O
oocytes O
. O

The O
Xenopus B
homeobox I
gene I
twin B
mediates O
Wnt B
induction O
of O
goosecoid B
in O
establishment O
of O
Spemann O
' O
s O
organizer O
. O

Between O
1988 O
and O
1994 O
, O
data O
from O
3 O
large O
sites O
revealed O
a O
3 O
- O
5 O
fold O
increase O
in O
the O
prevalence O
of O
antidepressant O
( O
ATD O
) O
treatment O
for O
U O
. O
S O
. O
youths O
aged O
2 O
- O
19 O
years O
. O

The O
vaccinia B
virus I
D6R I
open I
reading I
frame I
encodes O
the O
small O
subunit O
of O
the O
heterodimeric O
vaccinia B
virus I
early I
transcription I
factor I
( O
VETF B
) O
that O
activates O
transcription O
of O
early B
genes I
in O
vitro O
. O

The O
Sensititre O
MIC O
panels O
containing O
meropenem O
offer O
a O
convenient O
and O
valid O
alternative O
to O
the O
NCCLS O
reference O
method O
for O
the O
susceptibility O
testing O
of O
potential O
pathogens O
likely O
to O
be O
recovered O
from O
mixed O
infections O
. O

Recognition O
of O
DNA O
by O
single O
- O
chain O
derivatives O
of O
the O
phage B
434 I
repressor I
: O
high O
affinity O
binding O
depends O
on O
both O
the O
contacted O
and O
non O
- O
contacted O
base O
pairs O
. O

The O
paper O
calls O
for O
standardized O
question O
formats O
and O
a O
centralized O
body O
to O
monitor O
changes O
in O
these O
parameters O
of O
sexual O
behaviour O
. O

We O
show O
that O
GCNF B
binds O
to O
one O
of O
the O
two O
DRO O
sequences O
in O
the O
Prm1 B
promoter I
, O
and O
to O
the O
DRO O
sequence O
in O
the O
Prm2 B
promoter I
in O
a O
specific O
manner O
. O

139La O
NQR O
relaxation O
and O
microSR O
study O
of O
Zn O
- O
doping O
effects O
in O
La2CuO4 O
. O

The O
data O
also O
suggest O
that O
gamma B
CACCC I
box I
binding I
factors I
mediate O
LCR B
- I
gamma I
interactions O
which O
normally O
enhance O
gamma B
- I
globin I
and O
suppress O
beta B
- I
globin I
gene I
expression O
in O
fetal O
erythroid O
cells O
. O

However O
, O
the O
in O
vivo O
attachment O
strength O
of O
the O
CSTi O
- O
2 O
coating O
was O
comparable O
both O
to O
CSTi O
- O
1 O
and O
to O
an O
HA O
- O
coated O
control O
after O
8 O
weeks O
. O

Octamer O
recognition O
is O
mediated O
by O
the O
POU B
domain I
, O
a O
conserved O
structural O
motif O
which O
- O
- O
like O
the O
zinc O
finger O
and O
leucine O
zipper O
- O
- O
defines O
a O
family O
of O
related O
transcription O
factors O
. O

All O
testosterone O
treatments O
raised O
plasma O
testosterone O
concentrations O
above O
control O
and O
pretreatment O
levels O
( O
testosterone O
and O
synovex O
+ O
testosterone O
> O
synovex O
> O
control O
; O
all O
P O
< O
0 O
. O
05 O
) O
. O

Lifestyle O
characteristics O
were O
ascertained O
by O
a O
self O
- O
administered O
questionnaire O
. O

The O
major O
promoter O
element O
of O
the O
Xenopus B
laevis I
5S I
RNA I
gene I
is O
located O
within O
the O
transcribed O
region O
of O
the O
gene O
and O
forms O
the O
binding O
site O
for O
the O
transcription B
initiation I
factor I
TFIIIA I
. O

During O
each O
step O
of O
the O
pulse O
infusion O
the O
osteocalcin B
responses O
to O
changes O
in O
CaI O
in O
general O
were O
parallel O
to O
the O
changes O
in O
PTH B
( O
r O
= O
0 O
. O
462 O
; O
P O
= O
0 O
. O
02 O
) O
and O
were O
inversely O
correlated O
to O
CaI O
( O
r O
= O
- O
0 O
. O
562 O
; O
P O
= O
0 O
. O
003 O
) O
. O

The O
gamma B
- I
GCS I
gene I
is O
expressed O
ubiquitously O
and O
induced O
coordinately O
with O
NAD B
( I
P I
) I
H I
: I
quinone I
oxidoreductase I
( I
1 I
) I
( O
NQO1 B
) O
and O
glutathione B
S I
- I
transferase I
Ya I
( O
GST B
Ya I
) O
in O
response O
to O
xenobiotics O
and O
antioxidants O
. O

Interestingly O
, O
the O
expression O
of O
Id4 B
in O
Sertoli O
cells O
is O
only O
detectable O
after O
stimulation O
with O
FSH B
or O
cAMP O
. O

We O
determined O
their O
N O
- O
terminal O
amino O
acid O
sequence O
and O
found O
that O
these O
polypeptides O
were O
CotT B
, O
YeeK O
, O
YxeE O
, O
CotF B
, O
YrbA B
( O
31 O
and O
45 O
kDa O
) O
, O
and O
SpoIVA B
, O
respectively O
. O

The O
effect O
of O
betamethasone O
on O
duodenal O
calcium O
absorption O
and O
1 O
, O
25 O
- O
dihydroxy O
vitamin O
D3 O
production O
in O
the O
chick O
. O

Furthermore O
, O
it O
is O
also O
established O
that O
serine O
phosphorylation O
of O
STAT5a B
transactivation I
domain I
, O
via O
the O
MAPK B
pathway O
, O
is O
a O
means O
of O
modifying O
GH B
- O
induced O
transcriptional O
activation O
. O

ES O
cells O
in O
which O
the O
gene O
for O
the O
erythroid B
transcription I
factor I
GATA I
- I
1 I
has O
been O
disrupted O
fail O
to O
produce O
mature O
erythroid O
cells O
either O
in O
vivo O
or O
in O
vitro O
. O

Two O
conserved O
motifs O
, O
W89RKRRY94 O
and O
P156KKIKP161 O
, O
seemed O
to O
act O
as O
nuclear O
addressing O
signals O
. O

It O
is O
concluded O
that O
the O
predominant O
action O
of O
LSD O
on O
the O
female O
copulatory O
response O
is O
not O
mediated O
by O
increased O
dopamine B
receptor I
activity O
but O
that O
the O
LSD O
effect O
might O
be O
modulated O
by O
decreased O
dopaminergic O
activity O
. O

Therefore O
, O
in O
VDR B
- O
mediated O
transcriptional O
activation O
, O
1 O
, O
25 O
( O
OH O
) O
2D3 O
binding O
to O
VDR B
alters O
the O
conformation O
of O
the O
ligand O
binding O
domain O
such O
that O
it O
: O
( O
i O
) O
engages O
in O
strong O
heterodimerization O
with O
RXR B
to O
facilitate O
VDRE O
binding O
, O
( O
ii O
) O
influences O
the O
RXR B
ligand I
binding I
domain I
such O
that O
it O
is O
resistant O
to O
the O
binding O
of O
9 O
- O
cis O
RA O
but O
active O
in O
recruiting O
coactivator B
to O
its O
AF O
- O
2 O
and O
( O
iii O
) O
presents O
the O
AF O
- O
2 O
region O
in O
VDR B
for O
coactivator B
association O
. O

In O
patients O
, O
750 O
mL O
of O
saline O
lowered O
DL O
( O
CO O
) O
( O
- O
8 O
% O
, O
P O
< O
0 O
. O
01 O
versus O
baseline O
) O
, O
D O
( O
M O
) O
( O
- O
10 O
% O
, O
P O
< O
0 O
. O
01 O
versus O
baseline O
) O
, O
aldosterone O
( O
- O
29 O
% O
, O
P O
< O
0 O
. O
01 O
versus O
baseline O
) O
, O
renin B
( O
- O
52 O
% O
, O
P O
< O
0 O
. O
01 O
versus O
baseline O
) O
, O
and O
hematocrit O
( O
- O
6 O
% O
, O
P O
< O
0 O
. O
05 O
versus O
baseline O
) O
and O
increased O
V O
( O
C O
) O
( O
20 O
% O
, O
P O
< O
0 O
. O
01 O
versus O
baseline O
) O
, O
without O
changing O
rap O

and O
wpp O
. O

We O
have O
previously O
identified O
LIT1 B
, O
a O
paternally O
expressed O
antisense O
RNA O
within O
the O
KvLQT1 B
locus I
through O
a O
positional O
screening O
approach O
using O
human O
monochromosomal O
hybrids O
. O

A O
series O
of O
deletion O
mutants O
was O
expressed O
transiently O
in O
two O
human O
hepatocytes O
, O
HepG2 O
and O
PLC O
. O

The O
predicted O
amino O
acid O
sequence O
of O
the O
FlaB2 B
polypeptide I
was O
92 O
% O
identical O
to O
that O
of O
T B
. I
pallidum I
FlaB2 I
, O
with O
a O
76 O
% O
identity O
at O
the O
nucleotide O
level O
. O

Comparative O
analyses O
indicate O
that O
the O
5 O
' O
- O
peripheral O
domain O
exhibits O
a O
75 O
- O
bp O
length O
polymorphism O
near O
sequences O
associated O
with O
the O
termination O
of O
the O
H O
- O
strand O
replication O
. O

The O
survival O
of O
patients O
with O
a O
short O
bowel O
, O
even O
if O
they O
need O
long O
- O
term O
parenteral O
nutrition O
, O
is O
good O
. O

Thus O
, O
cross O
- O
resistance O
between O
fluoroquinolones O
was O
shown O
for O
both O
enterococci O
and O
MRSA O
. O

The O
5 O
' O
ends O
of O
the O
P B
and I
O I
gene I
mRNAs I
are O
separated O
by O
109 O
nucleotide O
pairs O
in O
the O
DNA O
template O
. O

Using O
community O
development O
approaches O
. O

Biol O
. O

We O
have O
cloned O
the O
cDNA O
for O
the O
SLBP B
from O
humans O
, O
mice O
, O
and O
frogs O
, O
using O
the O
recently O
developed O
yeast O
three O
- O
hybrid O
system O
. O

Along O
with O
these O
species O
, O
F O
. O
graminearum O
group O
2 O
( O
ZON O
, O
DON O
and O
/ O
or O
3AcDON O
or O
15AcDON O
) O
; O
F O
. O
chlamydosporum O
; O
F O
. O
acuminatum O
( O
type O
- O
A O
trichothecene O
derivatives O
) O
; O
and O
F O
. O
semitectum O
were O
often O
found O
to O
be O
associated O
. O

TGF B
- I
beta1 I
induced O
phosphorylation O
of O
Smad2 B
and O
Smad3 B
in O
Mv1Lu O
mink O
lung O
epithelial O
cells O
. O

There O
is O
little O
supportive O
evidence O
that O
ACE B
inhibitors O
( O
captopril O
or O
enalapril O
) O
are O
teratogenic O
. O

Thus O
, O
the O
potential O
induction O
of O
this O
gene O
rearrangement O
by O
E1A B
gene I
therapy O
is O
unlikely O
to O
be O
clinically O
significant O
in O
the O
treatment O
of O
advanced O
malignant O
disease O
. O

Although O
the O
cDNAs O
of O
HMG B
- I
14 I
and O
HMG B
- I
17 I
do O
not O
cross O
- O
hybridize O
, O
they O
have O
several O
similar O
structural O
features O
: O
the O
open O
reading O
frame O
comprises O
only O
23 O
% O
of O
the O
transcripts O
, O
the O
5 O
' O
- O
untranslated O
region O
is O
extremely O
GC O
rich O
whereas O
the O
3 O
' O
- O
untranslated O
region O
is O
unusually O
long O
and O
AT O
rich O
. O

Previously O
, O
we O
reported O
the O
identification O
of O
a O
23 O
- O
kD O
protein O
that O
interacts O
with O
zyxin B
in O
vitro O
( O
Sadler O
et O
al O
. O
, O
1992 O
) O
. O

If O
vascular O
induction O
between O
a O
vascular O
carrier O
and O
the O
selected O
jejunal O
segment O
is O
done O
as O
a O
kind O
of O
flap O
prefabrication O
, O
the O
jejunal O
interposition O
flap O
can O
be O
used O
without O
the O
need O
for O
complex O
microsurgery O
. O

In O
gel O
mobility O
shift O
assays O
, O
high O
binding O
activities O
of O
ATPC B
- I
2 I
and O
low O
binding O
activities O
of O
CBF B
were O
observed O
with O
nuclear O
extracts O
from O
tissue O
with O
low O
AtpC B
expression O
levels O
, O
and O
the O
opposite O
was O
observed O
with O
extracts O
from O
tissues O
with O
high O
AtpC B
expression O
levels O
. O

Activation O
of O
tyrosine B
kinase I
- I
containing I
receptors I
, O
heterotrimeric B
G I
proteins I
, O
and O
protein B
kinase I
C I
enhanced O
the O
activities O
of O
both O
MEK B
isoforms I
in O
293 O
and O
PC12 O
cells O
. O

The O
Fugu B
rubripes I
( O
Fugu B
) O
gene O
fragment O
was O
used O
to O
isolate O
the O
GH B
gene I
from O
a O
Fugu O
genomic O
library O
. O

An O
in O
vitro O
' O
glass O
stomach O
' O
( O
containing O
various O
volumes O
of O
hydrochloric O
acid O
[ O
HCl O
] O
and O
ammonium O
chloride O
[ O
NH4Cl O
] O
) O
was O
used O
to O
evaluate O
the O
means O
of O
increasing O
' O
gastric O
' O
pH O
to O
that O
of O
the O
NH4 O
+ O
- O
- O
> O
NH3 O
transition O
that O
occurs O
significantly O
at O
pH O
9 O
. O
24 O
. O

Acyclovir O
( O
3 O
% O
ointment O
) O
used O
topically O
one O
to O
five O
times O
a O
day O
on O
acute O
ocular O
HSV O
infection O
gave O
beneficial O
results O
as O
measured O
by O
a O
reduction O
in O
corneal O
involvement O
, O
conjunctivitis O
, O
iritis O
, O
and O
corneal O
clouding O
. O

We O
have O
documented O
previously O
that O
glucocorticoid O
hormones O
modulate O
the O
posttranslational O
localization O
of O
cell O
surface O
mouse O
mammary I
tumor I
virus I
( I
MMTV I
) I
glycoproteins O
in O
the O
viral O
- O
infected O
M1 O
. O
54 O
rat O
HTC O
hepatoma O
cell O
line O
. O

Partial O
proteolysis O
of O
cell O
surface O
- O
iodinated O
B B
- I
G I
molecules I
generates O
extremely O
similar O
patterns O
of O
spots O
, O
both O
within O
and O
between O
haplotypes O
. O

SETTING O
: O
University O
research O
laboratory O
. O

There O
was O
a O
57 O
% O
reduction O
in O
IGF B
IR I
mRNA I
levels O
in O
clone O
CA9 O
after O
confluence O
compared O
with O
clone O
ME10 O
. O

Oligomers O
corresponding O
to O
the O
region O
of O
the O
mlc B
- I
1 I
/ I
3 I
enhancer I
, O
which O
encompasses O
this O
conserved O
sequence O
, O
bound O
MEF B
- I
2 I
and O
competed O
for O
its O
binding O
to O
the O
mck B
enhancer I
. O

Similar O
tissue O
- O
specific O
patterns O
are O
observed O
with O
a O
fusion O
between O
the O
caufliflower B
mosaic I
virus I
35S I
RNA I
promotor I
and O
the O
GUS B
gene I
. O

This O
difference O
between O
SP B
- I
1 I
and O
SP B
- I
2 I
peptides I
was O
not O
due O
to O
their O
differential O
uptake O
by O
cell O
, O
since O
approximately O
100 O
times O
more O
SP B
- I
2 I
peptide I
could O
be O
found O
in O
cytoplasmic O
extracts O
than O
SP B
- I
1 I
peptide I
in O
experiments O
using O
radiolabeled O
peptides O
. O

One O
function O
of O
p53 B
is O
in O
regulating O
cell O
cycle O
check O
- O
point O
control O
after O
DNA O
damage O
. O

We O
used O
event O
- O
related O
functional O
magnetic O
resonance O
imaging O
( O
fMRI O
) O
during O
a O
cued O
spatial O
- O
attention O
task O
to O
dissociate O
brain O
activity O
related O
to O
attentional O
control O
from O
that O
related O
to O
selective O
processing O
of O
target O
stimuli O
. O

BACKGROUND O
: O
The O
extract O
of O
medicinal O
plants O
containing O
curcumin O
is O
traditionally O
believed O
to O
have O
a O
positive O
contraction O
effect O
on O
the O
human O
gall O
- O
bladder O
. O

Apoptosis O
triggered O
through O
Fas B
and O
F B
/ I
F I
was O
inhibited O
by O
coexpression O
of O
CrmA B
and O
p35 B
, O
but O
not O
Bcl B
- I
xL I
. O

The O
product O
of O
one O
gene O
, O
Bv80 B
, O
has O
a O
single O
divergent O
copy O
of O
a O
sequence O
of O
149 O
amino O
acids O
( O
approx O
. O

The O
1031 O
- O
bp O
wild B
- I
type I
HIT1 I
DNA I
which O
contained O
an O
open O
reading O
frame O
encoding O
a O
protein O
of O
164 O
amino O
acids O
and O
the O
AGG B
arginine I
tRNA I
gene I
complemented O
all O
hit1 B
- I
1 I
mutant I
phenotypes O
, O
indicating O
that O
the O
mutant O
phenotypes O
were O
caused O
by O
the O
deletion O
of O
these O
genes O
. O

We O
found O
a O
prevalence O
of O
eating O
disorders O
of O
5 O
. O
9 O
% O
( O
lifetime O
prevalence O
of O
10 O
% O
) O
, O
irrespective O
of O
gender O
and O
type O
of O
diabetes O
; O
4 O
. O
1 O
% O
of O
the O
whole O
sample O
reported O
intentional O
insulin B
undertreatment O
or O
omission O
. O

Tilisolol O
and O
carboxymethylcellulose O
sodium O
salt O
( O
CMC O
) O
were O
used O
as O
the O
model O
ophthalmic O
drug O
and O
viscous O
polymer O
, O
respectively O
. O

These O
results O
are O
discussed O
in O
the O
context O
of O
a O
model O
of O
the O
myogenic O
lineage O
that O
is O
based O
on O
the O
expression O
of O
desmin B
. O

We O
identified O
two O
potential O
phosphorylation O
sites O
at O
serine49 O
and O
serine133 O
, O
both O
of O
which O
seem O
to O
be O
necessary O
for O
Myf B
- I
5 I
activity O
. O

We O
constructed O
a O
series O
of O
chimeric O
genes O
containing O
part O
of O
the O
first O
exon O
and O
increasingly O
longer O
5 O
' O
flanking O
sequences O
of O
the O
ODC B
gene I
fused O
to O
either O
bacterial B
chloramphenicol I
acetyltransferase I
( O
CAT B
) O
or O
luciferase B
reporter I
genes I
. O

CVO2 O
saturation O
is O
a O
reliable O
and O
sensitive O
method O
for O
detecting O
blood O
loss O
. O

The O
gene O
encoding O
the O
Neisseria B
lactamica I
III I
DNA I
methyltransferase I
( O
M B
. I
NlaIII I
) O
which O
recognizes O
the O
sequence O
CATG O
has O
been O
cloned O
and O
expressed O
in O
Escherichia O
coli O
. O

This O
latter O
complex O
reacts O
with O
an O
antibody O
to O
serum B
response I
factor I
( O
SRF B
) O
and O
exhibits O
the O
same O
binding O
characteristics O
as O
purified B
SRF I
. O

The O
second O
patient O
was O
successfully O
treated O
with O
intravenous O
prostaglandin O
F2 O
alpha O
( O
PGF2 O
alpha O
) O
. O

Alignment O
of O
the O
rat B
and I
human I
WIN I
cDNAs I
and O
their O
comparison O
with O
mouse O
genomic O
sequence O
revealed O
that O
the O
WIN B
DNA I
binding I
domain I
is O
encoded O
by O
four O
exons O
, O
two O
of O
which O
( O
exons O
4 O
and O
6 O
) O
are O
alternatively O
spliced O
to O
generate O
at O
least O
three O
classes O
of O
mRNA O
transcripts O
. O

In O
a O
pilot O
study O
in O
which O
two O
dogs O
were O
given O
parenteral O
rifampin O
before O
and O
after O
operation O
and O
grafts O
were O
preclotted O
with O
blood O
that O
contained O
rifampin O
, O
it O
was O
suggested O
that O
there O
was O
a O
slight O
increase O
in O
inhibition O
at O
24 O
hours O
( O
93 O
% O
) O
. O

A O
flow O
probe O
for O
direct O
measurement O
of O
blood O
flow O
of O
the O
mitral O
valve O
was O
devised O
. O

Here O
, O
we O
analyzed O
the O
interaction O
of O
the O
Lactococcus B
lactis I
Ll I
. I
LtrB I
group I
II I
intron I
endonuclease I
with O
its O
DNA O
target O
site O
by O
DNA O
footprinting O
and O
modification O
- O
interference O
approaches O
. O

Clinical O
trials O
bring O
informations O
for O
pharmacology O
and O
epidemiology O
. O

In O
several O
steps O
we O
converted O
the O
retinoid O
specific O
response O
element O
of O
the O
human B
retinoic I
acid I
receptor I
beta I
promoter I
into O
the O
vitamin O
D O
/ O
retinoic O
acid O
response O
element O
of O
the O
human B
osteocalcin I
promoter I
. O

The O
immediate O
5 O
' O
- O
flanking O
region O
was O
GC O
- O
rich O
with O
3 O
SP B
- I
1 I
- I
like I
and O
2 O
AP B
- I
2 I
sites I
identified O
in O
close O
proximity O
to O
the O
transcription O
start O
site O
. O

Akt B
- O
dependent O
antiapoptotic O
action O
of O
insulin B
is O
sensitive O
to O
farnesyltransferase B
inhibitor O
. O

For O
all O
samples O
, O
the O
sensitivity O
of O
the O
blast O
flag O
on O
the O
CD3500 O
was O
85 O
% O
with O
a O
specificity O
of O
91 O
% O
( O
STKS O
: O
sensitivity O
, O
75 O
% O
; O
specificity O
, O
85 O
% O
) O
; O
the O
sensitivity O
of O
the O
CD3500 B
IG B
flag O
was O
72 O
% O
with O
a O
specificity O
of O
76 O
% O
( O
STKS O
: O
sensitivity O
, O
75 O
% O
: O
specificity O
, O
73 O
% O
) O
; O
and O
the O
sensitivity O
of O
the O
NRBC O
flag O
was O
43 O
% O
with O
a O
specificity O
of O
94 O
% O
( O
STKS O
sensitivity O
, O
37 O
% O
; O
specificity O
, O
88 O
% O
) O
. O

Control O
of O
ovulation O
in O
mares O
in O
the O
early O
breeding O
season O
with O
ovarian O
steroids O
and O
prostaglandin O
. O

Reasons O
are O
given O
for O
suggesting O
that O
tiapamil O
warrants O
further O
clinical O
evaluation O
, O
after O
which O
it O
may O
join O
the O
more O
established O
calcium O
antagonists O
as O
a O
valuable O
therapeutic O
agent O
. O

The O
central O
venous O
O2 O
saturation O
test O
in O
acute O
pulmonary O
fat O
embolism O
. O

Two O
of O
the O
proteins O
UPRF B
- I
1 I
and O
UPRF B
- I
2 I
( O
which O
is O
apparently O
a O
proteolytic O
degradation O
product O
of O
UPRF B
- I
1 I
) O
bind O
inefficiently O
to O
mutant O
versions O
of O
the O
UPR O
that O
are O
unable O
to O
confer O
responsiveness O
to O
unfolded O
proteins O
to O
the O
( B
CYC1 I
) I
promoter I
. O

Identification O
of O
domains O
mediating O
transcription O
activation O
, O
repression O
, O
and O
inhibition O
in O
the O
paired B
- I
related I
homeobox I
protein I
, O
Prx2 B
( O
S8 I
) O
. O

METHODS O
: O
A O
spatial O
counting O
approach O
in O
which O
the O
location O
of O
each O
and O
every O
PN O
and O
NP O
in O
the O
stratum O
pyramidale O
of O
sectors O
CA1 O
- O
4 O
was O
applied O
to O
11 O
normal O
control O
( O
CONs O
) O
and O
10 O
SZs O
matched O
for O
age O
and O
postmortem O
interval O
, O
as O
well O
as O
4 O
manic O
depressive O
( O
MD O
) O
subjects O
matched O
for O
age O
. O

Analytical O
scanning O
isoelectrofocusing O
. O

Upstream B
stimulatory I
factor I
USF B
is O
a O
human O
transcriptional O
activation O
factor O
, O
which O
uses O
a O
basic O
/ O
helix O
- O
loop O
- O
helix O
/ O
leucin O
zipper O
( O
b O
/ O
HLH O
/ O
Z O
) O
motif O
to O
homodimerize O
and O
recognize O
specific O
sequences O
in O
the O
promoter O
region O
of O
both O
nuclear O
and O
viral O
genes O
transcribed O
by O
RNA B
polymerase I
II I
. O

43 O
% O
, O
6 O
/ O
14 O
; O
P O
= O
0 O
. O
013 O
) O
, O
whereas O
, O
in O
RCC O
samples O
with O
VHL B
methylation O
or O
mutation O
, O
the O
frequency O
of O
3p14 B
- O
p21 B
LOH O
did O
not O
differ O
from O
that O
of O
sp25 B
- O
p26 B
( O
72 O
% O
, O
18 O
/ O
25 O
vs O
. O

Sequences O
homologous O
to O
the O
consensus O
TUF B
- I
binding I
UAS I
boxes I
are O
present O
in O
the O
5 O
' O
flanking O
regions O
of O
both O
genes O
. O

The O
analysis O
predicted O
that O
progressive O
increases O
in O
serum O
creatinine O
or O
aspartate B
transaminase I
activity O
will O
result O
in O
only O
a O
50 O
% O
reduction O
of O
clearance O
. O

RESULTS O
: O
Although O
low O
levels O
of O
vIRF B
- I
2 I
mRNAs I
can O
be O
detected O
in O
the O
HHV O
- O
8 O
- O
positive O
BCBL O
- O
1 O
tumor O
cell O
line O
, O
12 O
- O
0 O
- O
tetradecanoylphorbol O
- O
13 O
- O
acetate O
( O
TPA O
) O
treatment O
does O
not O
stimulate O
expression O
of O
vIRF B
- I
2 I
gene I
together O
with O
primary O
lytic O
cycle O
genes O
. O

Central O
projections O
of O
gastric O
afferent O
vagal O
inputs O
. O

DNA O
sequence O
of O
the O
UL6 B
to O
UL20 B
genes I
of O
infectious O
laryngotracheitis O
virus O
and O
characterization O
of O
the O
UL10 B
gene I
product I
as O
a O
nonglycosylated O
and O
nonessential O
virion O
protein O
. O

Coimmunoprecipitation O
analysis O
suggests O
that O
these O
residues O
likely O
contribute O
to O
the O
multimerization O
function O
required O
for O
homomeric O
complex O
formation O
or O
heteromeric O
complex O
formation O
with O
p50 B
in O
that O
no O
association O
of O
p65 B
delta I
with O
itself O
or O
with O
p50 B
was O
evident O
. O

Cytostatic O
activity O
of O
some O
essential O
oils O
against O
HEp O
- O
2 O
cells O
. O

A O
yeast O
mitochondrial O
translation O
initiation O
codon O
mutation O
affecting O
the O
gene O
for O
cytochrome B
oxidase I
subunit I
III I
( O
COX3 B
) O
was O
partially O
suppressed O
by O
a O
spontaneous O
nuclear O
mutation O
. O

Only O
the O
27 O
- O
kDa O
protein O
was O
detected O
after O
N B
- I
glycosidase I
treatment O
, O
indicating O
that O
PLP B
- I
H I
is O
a O
glycoprotein O
. O

Moreover O
, O
we O
studied O
the O
relationship O
between O
initial O
percentage O
of O
bone O
marrow O
plasma O
cells O
and O
prognosis O
. O

These O
results O
demonstrate O
that O
GGTase B
- I
I I
inhibitors O
arrest O
cells O
in O
G0 O
/ O
G1 O
and O
induce O
accumulation O
of O
p21WAF B
in O
a O
p53 B
- O
independent O
manner O
and O
that O
FTase B
inhibitors O
can O
interfere O
with O
cell O
cycle O
events O
by O
a O
mechanism O
that O
involves O
neither O
p21WAF B
nor O
p27KIP B
. O

We O
report O
here O
on O
an O
eight O
- O
year O
- O
old O
boy O
who O
first O
developed O
acute O
intravascular O
hemolysis O
following O
therapy O
with O
amphotericin O
B O
( O
AmB O
) O
and O
subsequently O
a O
delayed O
hemolytic O
transfusion O
reaction O
due O
to O
alloantibodies O
. O

The O
temporal O
sequence O
of O
organ O
failure O
was O
lung O
, O
clotting O
system O
, O
kidney O
, O
and O
liver O
. O

The O
potential O
biological O
significance O
of O
the O
identified O
structural O
motif O
shared O
by O
the O
human B
CCR5 I
, O
CCR3 B
, O
CCR2B B
and O
other O
GPCRs B
is O
discussed O
. O

Analysis O
of O
rat O
brain O
, O
heart O
, O
lung O
, O
liver O
, O
kidney O
and O
skeletal O
muscle O
revealed O
psi B
PKC I
zeta I
mRNA I
only O
in O
brain O
. O

The O
papers O
provide O
a O
broad O
perspective O
of O
MPAs O
and O
include O
social O
, O
economic O
, O
cultural O
, O
biological O
and O
statistical O
components O
. O

Management O
of O
patients O
with O
Ewing O
' O
s O
sarcoma O
has O
been O
discussed O
with O
reference O
to O
the O
need O
to O
achieve O
an O
excellent O
local O
result O
from O
radiation O
therapy O
now O
that O
patients O
are O
experiencing O
long O
- O
term O
survival O
. O

Furthermore O
, O
the O
rapid O
light O
- O
mediated O
increase O
of O
CPRF B
proteins I
was O
insensitive O
to O
transcriptional O
inhibitors O
, O
suggesting O
that O
a O
post O
- O
transcriptional O
mechanism O
controls O
CPRF B
accumulation O
. O

In O
those O
patients O
who O
before O
transplantation O
were O
serological O
negative O
for O
CMV O
but O
had O
received O
organs O
from O
CMV B
- I
IgG I
positive O
donors O
, O
the O
incidence O
of O
the O
disease O
was O
highest O
. O

Since O
1985 O
the O
combination O
of O
chlorambucil O
( O
10 O
mg O
daily O
, O
initially O
for O
six O
weeks O
, O
then O
alternating O
fortnights O
for O
12 O
weeks O
) O
and O
interferon B
- I
alpha I
2b I
( O
Schering I
- I
Plough O
; O
2 O
x O
10 O
( O
6 O
) O
U O
/ O
m2 O
three O
times O
weekly O
by O
subcutaneous O
injection O
for O
18 O
weeks O
) O
has O
been O
compared O
in O
a O
randomised O
trial O
with O
chlorambucil O
alone O
in O
previously O
untreated O
patients O
with O
stage O
III O
or O
IV O
follicular O
lymphoma O
. O

The O
BDU O
neurons O
and O
the O
ALM O
touch O
neurons O
are O
lineal O
sister O
cells O
in O
the O
AB O
. O
a O
lineage O
and O
the O
VA O
and O
VB O
motor O
neurons O
are O
lineal O
sister O
cells O
in O
the O
AB O
. O
p O
lineage O
. O

We O
compared O
retrospectively O
the O
efficacy O
of O
granulocyte B
colony I
stimulating I
factor I
( O
G B
- I
CSF I
) O
alone O
with O
chemotherapy O
plus O
G B
- I
CSF I
in O
mobilizing O
CD34 B
- O
positive O
cells O
in O
patients O
with O
malignant O
lymphoma O
. O

Serum O
concentrations O
of O
hormones O
in O
patients O
classified O
as O
C O
were O
characteristically O
and O
inclined O
to O
be O
hypergonadotropic O
and O
hypoestrogenic O
. O

Cervical O
CT O
revealed O
an O
irregular O
low O
density O
at O
the O
periphery O
of O
the O
cervical O
vertebra O
from O
the O
C2 O
to O
C4 O
level O
. O

Incorporation O
of O
this O
information O
in O
the O
data O
brought O
about O
a O
revised O
five O
- O
year O
survival O
estimate O
of O
55 O
percent O
. O

These O
motifs O
, O
and O
their O
cognate O
transcription O
factors O
, O
appear O
to O
act O
in O
concert O
, O
with O
partial O
redundancy O
, O
so O
that O
discrete O
mutations O
were O
only O
partially O
effective O
in O
reducing O
transcriptional O
activity O
. O

We O
now O
show O
that O
purified O
recombinant O
c B
- I
sis I
/ O
PDGF B
can O
induce O
this O
binding O
activity O
which O
we O
have O
termed O
SIF B
, O
for O
sis B
- I
inducible I
factor I
. O

Statistical O
conservation O
laws O
in O
turbulent O
transport O
. O

Expression O
of O
the O
bacteriophage B
T4 I
DNA I
terminase I
genes I
16 I
and I
17 I
yields O
multiple O
proteins O
. O

Erdheim O
- O
Chester O
disease O
: O
a O
distinct O
lipoidosis O
or O
part O
of O
the O
spectrum O
of O
histiocytosis O
? O

Erdheim O
- O
Chester O
disease O
has O
always O
been O
considered O
a O
distinct O
lipoidosis O
based O
on O
clinical O
and O
radiographic O
criteria O
. O

Self O
- O
consistent O
calculation O
of O
the O
surface O
electronic O
structure O
of O
the O
( O
1 O
x O
2 O
) O
reconstructed O
Au O
( O
110 O
) O
surface O
. O

The O
plasmid O
was O
found O
to O
confer O
drug O
resistance O
specifically O
to O
LMB O
. O

Two O
pharmacokinetic O
models O
were O
compared O
. O

A O
higher O
median O
proportion O
of O
spirochaetes O
was O
observed O
in O
sites O
after O
diagnosis O
of O
loss O
of O
attachment O
. O

Ocular O
injuries O
during O
general O
anesthesia O
. O

Of O
81 O
girls O
cured O
from O
the O
bacteriuria O
24 O
recurred O
, O
5 O
with O
a O
symptomatic O
pyelonephritis O
and O
3 O
with O
cystitis O
. O

The O
performances O
of O
two O
tests O
, O
the O
ProstAsure O
index O
and O
the O
percent O
free O
PSA B
test O
, O
were O
evaluated O
in O
detecting O
cancer O
. O

The O
protein O
encoded O
by O
the O
NF2 B
gene I
has O
a O
similarity O
to O
ezrin B
, O
radixin B
and O
moesin B
( O
ERM B
) O
proteins O
that O
link O
membrane O
proteins O
to O
the O
cytoskeleton O
. O

PATIENTS O
- O
- O
125 O
women O
consulting O
the O
general O
practitioner O
for O
symptomatic O
bacterial O
vaginosis O
. O

In O
the O
healing O
tendons O
, O
the O
rate O
of O
collagen B
synthesis O
decreased O
and O
the O
rate O
of O
noncollagen O
protein I
synthesis O
remained O
unchanged O
. O

From O
our O
own O
investigations O
on O
phantoms O
, O
the O
smallest O
signal O
- O
to O
- O
noise O
ratio O
( O
delta O
S O
min O
/ O
delta O
R O
) O
was O
obtained O
which O
determines O
the O
minimal O
contrast O
, O
given O
the O
film O
properties O
( O
film O
response O
S O
, O
film O
gradient O
gamma O
) O
and O
the O
amount O
of O
scatter O
. O

However O
, O
one O
activity O
binds O
preferentially O
to O
G O
+ O
C O
- O
rich O
promoters O
, O
and O
another O
activity O
appears O
to O
bind O
preferentially O
to O
only O
two O
of O
the O
promoters O
tested O
. O

Cardiovascular O
effects O
in O
man O
of O
intravenous O
prizidilol O
hydrochloride O
( O
SK O
& O
F O
92657 O
) O
; O
a O
new O
antihypertensive O
agent O
. O

From O
the O
Centers O
for O
Disease O
Control O
and O
Prevention O
. O

Expression O
of O
a O
cDNA O
clone O
in O
yeast O
containing O
a O
large O
portion O
of O
the O
open O
reading O
frame O
produced O
cAMP B
PDEase I
activity O
identical O
in O
properties O
to O
the O
Drosophila O
enzyme O
affected O
by O
the O
dnc B
mutation O
. O

FdsR B
purified O
to O
homogeneity O
after O
overexpression O
of O
fdsR B
in O
E O
. O
coli O
is O
a O
130 O
kDa O
homotetramer O
binding O
to O
the O
fds B
control I
region I
located O
between O
the O
fdsR B
and O
fdsG B
genes I
. O

ZNS O
, O
in O
the O
mmolar O
range O
, O
scavenged O
hydroxyl O
and O
nitric O
oxide O
radicals O
in O
a O
dose O
- O
dependent O
manner O
. O

The O
orthologs O
of O
the O
Y B
( I
1 I
) I
and O
Y B
( I
2 I
) I
subtypes I
display O
high O
amino O
acid O
sequence O
identities O
between O
pig O
, O
human O
, O
and O
mouse O
( O
92 O
% O
- O
94 O
% O
) O
, O
whereas O
the O
Y B
( I
4 I
) I
, O
Y B
( I
5 I
) I
, O
and O
y B
( I
6 I
) I
subtypes O
display O
lower O
identities O
( O
76 O
% O
- O
87 O
% O
) O
. O

Binding O
of O
ZAP B
- I
70 I
to O
phospho B
- I
TAMs I
is O
notable O
for O
the O
high O
level O
of O
cooperativity O
between O
the O
two O
SH2 B
domains I
, O
which O
individually O
demonstrate O
low O
affinity O
interaction O
with O
the O
ligand O
. O

The O
benzo O
- O
pyrone O
drug O
Venalot O
had O
a O
considerable O
effect O
in O
reducing O
the O
protein O
concentration O
in O
the O
air O
spaces O
and O
the O
interstitial O
tissue O
, O
and O
of O
the O
oedema O
in O
the O
latter O
. O

Radiation O
was O
delivered O
in O
a O
single O
4 O
- O
mJ O
pulse O
of O
200 O
microseconds O
duration O
by O
means O
of O
an O
articulated O
zirconium O
fluoride O
optical O
fibre O
and O
a O
320 O
- O
microns O
quartz O
- O
fibre O
tip O
. O

Perception O
of O
sweetness O
cannot O
be O
used O
to O
accurately O
meter O
the O
metabolizable O
energy O
or O
nutritive O
value O
of O
a O
food O
. O

The O
leader O
peptide O
was O
hypothesized O
to O
inhibit O
ribosome O
release O
at O
the O
tnaC B
stop O
codon O
, O
thereby O
blocking O
Rho B
' O
s O
access O
to O
the O
transcript O
. O

Response O
properties O
of O
single O
units O
in O
the O
accessory O
optic O
system O
of O
the O
dark O
- O
reared O
cat O
. O

Excretion O
of O
radiopharmaceuticals O
into O
breast O
milk O
. O

Influence O
of O
huanglian O
used O
in O
combination O
with O
huangqin O
and O
gancao O
on O
the O
erythrocytic O
osmotic O
fragilitas O
of O
experimental O
glucose B
- I
6 I
- I
phosphate I
dehydrogenase I
deficiency O
in O
rats O
] O
OBJECTIVE O
: O
To O
study O
the O
influence O
of O
common O
combination O
of O
Huanglian O
( O
Coptis O
chinensis O
) O
on O
the O
erythrocytic O
osmotic O
fragilitas O
of O
glucose B
- I
6 I
- I
phosphate I
dehydrogenase I
( O
G6PD B
) O
deficiency O
in O
rats O
. O

The O
leukotriene B
receptor I
antagonist O
zafirlukast O
inhibits O
sulfur O
dioxide O
- O
induced O
bronchoconstriction O
in O
patients O
with O
asthma O
. O

This O
sequence O
was O
sufficient O
to O
confer O
p53 B
- O
dependent O
activation O
to O
a O
heterologous O
promoter O
and O
p53 B
was O
capable O
of O
binding O
to O
this O
sequence O
as O
assessed O
by O
gel O
shift O
analysis O
. O

The O
embryology O
of O
the O
vertebral O
column O
is O
reviewed O
briefly O
, O
and O
the O
origin O
of O
each O
anomaly O
of O
the O
C2 O
vertebra O
is O
traced O
to O
one O
of O
the O
two O
early O
stages O
in O
development O
as O
follows O
: O
1 O
) O
the O
formation O
of O
the O
mesenchymal O
vertebra O
or O
2 O
) O
its O
induction O
to O
cartilage O
. O

Two O
of O
the O
three O
groups O
were O
submitted O
to O
two O
different O
levels O
of O
hypoxia O
( O
FiO2 O
= O
0 O
. O
05 O
, O
group O
F5 O
and O
FiO2 O
= O
0 O
. O
1 O
, O
group O
F10 O
) O
and O
the O
third O
to O
normoxia O
( O
FiO2 O
= O
0 O
. O
21 O
, O
group O
F21 O
) O
in O
a O
thermoneutral O
and O
controlled O
environment O
. O

Parathyroid B
hormone I
responses O
of O
cyclic O
AMP O
- O
, O
serum O
- O
and O
phorbol O
ester O
- O
responsive O
reporter O
genes O
in O
osteoblast O
- O
like O
UMR O
- O
106 O
cells O
. O

Our O
results O
strongly O
suggest O
that O
activation O
of O
the O
beta O
origin O
by O
a O
distant O
replication O
enhancer O
element O
requires O
a O
small O
target O
sequence O
essential O
for O
initiator O
protein O
- O
mediated O
DNA O
looping O
. O

HNF4alpha7 B
mRNA I
is O
also O
found O
in O
totipotent O
embryonic O
stem O
cells O
. O

Penicillamine O
- O
induced O
myasthenia O
gravis O
associated O
with O
antibodies O
to O
acetylcholine B
receptor I
. O

Magnetic O
force O
and O
torque O
at O
1 O
. O
5 O
T O
did O
not O
dislodge O
the O
GF O
or O
result O
in O
perforation O
of O
canine O
IVC O
by O
the O
GF O
. O

A O
. O

LCCH1 B
was O
identical O
to O
the O
Rdl B
gene I
, O
a O
known O
GABA B
receptor I
subunit I
gene I
from O
D O
. O
melanogaster O
, O
whereas O
LCCH2 B
and O
LCCH3 B
were O
novel O
D O
. O
melanogaster O
sequences O
that O
exhibited O
structural O
similarity O
to O
other O
members O
of O
the O
ligand B
- I
gated I
chloride I
channel I
gene I
family I
. O

A O
significant O
increase O
of O
gastric O
mucosal O
permeability O
was O
observed O
in O
six O
normal O
human O
subjects O
after O
instillation O
of O
ethanol O
( O
20 O
percent O
v O
/ O
v O
) O
. O

Using O
a O
simulated O
cough O
machine O
, O
we O
analyzed O
the O
effect O
of O
adding O
tensio O
- O
active O
liquids O
as O
sol O
phase O
simulant O
on O
the O
clearance O
of O
gel O
mucus O
simulant O
by O
cough O
. O

First O
, O
the O
amino O
- O
terminal O
regulatory O
arm O
of O
the O
GDI B
binds O
to O
the O
switch O
I O
and O
II O
domains O
of O
Cdc42 B
leading O
to O
the O
inhibition O
of O
both O
GDP O
dissociation O
and O
GTP O
hydrolysis O
. O

By O
a O
combination O
of O
alternative O
promoter O
usage O
and O
exon O
splicing O
, O
each O
ROR B
gene I
generates O
several O
isoforms O
that O
differ O
only O
in O
their O
amino O
terminus O
. O

Electrophoretic O
mobility O
- O
shift O
assays O
showed O
a O
rapid O
induction O
of O
nuclear O
proteins O
that O
bound O
to O
the O
consensus O
kappa B
B I
motif I
. O

Analysis O
of O
larger O
cDNA O
clones O
demonstrates O
that O
there O
are O
at O
least O
two O
isoforms O
of O
RIP14 B
that O
differ O
in O
the O
N O
- O
terminal O
( O
A O
and O
B O
) O
and O
hinge O
( O
D O
) O
domains O
. O

Multiple O
copies O
of O
this O
binding O
site O
are O
found O
in O
the O
viral O
genomes O
. O

During O
subsequent O
CS O
- O
alone O
trials O
, O
the O
responses O
of O
( O
+ O
) O
MK O
- O
801 O
- O
injected O
mice O
were O
extinguished O
as O
easily O
as O
those O
of O
saline O
- O
injected O
mice O
. O

In O
contrast O
, O
the O
tail O
deviated O
ipsilaterally O
in O
cats O
with O
a O
hemisection O
, O
and O
the O
tail O
was O
ventroflexed O
in O
cats O
with O
a O
transection O
. O

The O
major O
open O
reading O
frame O
of O
the O
cDNA O
predicts O
a O
protein O
of O
59 O
kD O
, O
with O
a O
leucine O
zipper O
situated O
adjacent O
to O
an O
myc B
- I
related I
motif I
that O
has O
been O
proposed O
to O
assume O
a O
helix O
- O
loop O
- O
helix O
structure O
. O

This O
residue O
, O
although O
located O
55 O
rather O
than O
35 O
residues O
NH2 O
- O
terminal O
of O
the O
essential O
glutamate O
, O
is O
undoubtedly O
the O
analog O
of O
the O
second O
aspartate O
of O
the O
Asp O
- O
Asp O
- O
35 O
- O
Glu O
motif O
found O
in O
other O
family O
members O
. O

In O
addition O
to O
loss O
of O
digit O
identity O
and O
varying O
degrees O
of O
polydactyly O
, O
proximal O
skeletal O
elements O
are O
severely O
shortened O
in O
Xt O
; O
ld B
double O
homozygous O
limbs O
. O

The O
PEP4 B
gene I
was O
isolated O
from O
a O
genomic O
DNA O
library O
by O
complementation O
of O
the O
pep4 B
- I
3 I
mutation O
. O

A O
gene O
with O
a O
high O
degree O
of O
sequence O
identity O
with O
mammalian B
ADP I
- I
ribosylation I
factors I
( O
ARFs B
) O
, O
believed O
to O
be O
important O
in O
vesicular O
transport O
, O
was O
identified O
. O

Off O
- O
shell O
effect O
in O
Rydberg O
- O
atom O
- O
alkali O
- O
metal O
- O
atom O
scattering O
. O

Short O
- O
term O
growth O
: O
rhythms O
, O
chaos O
, O
or O
noise O
? O

Association O
between O
diaphragm O
use O
and O
asymptomatic O
bacteriuria O
. O

The O
effect O
of O
iron O
on O
the O
toxigenicity O
of O
Vibrio O
cholerae O
. O

The O
Tat B
protein I
coded O
by O
human O
immunodeficiency O
virus O
( O
HIV O
) O
is O
a O
strong O
activator O
of O
viral O
gene O
expression O
from O
the O
long O
terminal O
repeat O
( O
LTR O
) O
. O

Is O
it O
time O
for O
a O
wine O
trial O
? O

The O
average O
coefficient O
of O
variation O
between O
assays O
for O
gas O
chromatography O
mass O
spectrometry O
was O
5 O
. O
8 O
% O
and O
for O
radioimmunoassay O
was O
12 O
. O
3 O
% O
, O
while O
the O
average O
coefficient O
of O
variation O
within O
assays O
for O
gas O
chromatography O
mass O
spectrometry O
was O
4 O
. O
9 O
% O
and O
for O
radioimmunoassay O
6 O
. O
9 O
% O
. O

Macrophages O
: O
modulators O
of O
immunity O
. O

The O
NH2 O
- O
terminal O
fragments O
were O
tested O
for O
susceptibility O
to O
modification O
with O
Nalpha O
- O
( O
3 O
- O
maleimidylpropionyl O
) O
biocytin O
, O
which O
attaches O
a O
biotin O
group O
to O
cysteine O
sulfhydryls O
. O

Production O
of O
VT O
was O
also O
examined O
retrospectively O
in O
32 O
E O
. O
coli O
strains O
of O
serotype O
O26 O
: O
H11 O
isolated O
from O
children O
with O
diarrhoea O
; O
eight O
( O
25 O
% O
) O
of O
them O
produced O
moderate O
to O
high O
levels O
of O
verotoxin B
1 I
despite O
several O
years O
storage O
in O
vitro O
. O

Serum O
pituitary O
and O
steroid O
hormone O
levels O
in O
the O
adult O
male O
: O
one O
value O
is O
as O
good O
as O
the O
mean O
of O
three O
. O

The O
sections O
were O
stained O
with O
anti B
- I
polymorphonuclear I
leukocyte I
antibody I
, O
the O
endothelial O
marker O
factor B
VIII I
- I
related I
antigen I
, O
and O
with O
hematoxylin O
and O
eosin O
. O

Mapping O
of O
the O
region O
in O
KAP B
- I
1 I
required O
for O
HP1 B
interaction O
showed O
that O
amino O
acid O
substitutions O
which O
abolish O
HP1 B
binding O
in O
vitro O
reduce O
KAP B
- I
1 I
mediated O
repression O
in O
vivo O
. O

The O
flanking O
open O
reading O
frames O
in O
pARGC2 B
showed O
no O
homologies O
to O
arginine B
biosynthetic I
genes O
. O

Promoter O
activity O
of O
the O
proliferating B
- I
cell I
nuclear I
antigen I
gene I
is O
associated O
with O
inducible O
CRE B
- I
binding I
proteins I
in O
interleukin B
2 I
- O
stimulated O
T O
lymphocytes O
. O

METHODS O
: O
Levels O
of O
sIL B
- I
2R I
were O
measured O
simultaneously O
in O
plasma O
and O
pleural O
fluid O
in O
111 O
patients O
with O
pleural O
effusions O
of O
unknown O
causes O
. O

The O
GIS O
software O
program O
, O
ArcView O
, O
was O
used O
for O
spatial O
analysis O
and O
distance O
calculations O
. O

chi2 O
Tests O
were O
used O
to O
compare O
the O
distribution O
of O
the O
characteristics O
between O
intersection O
and O
midblock O
collisions O
. O

A O
phase O
II O
study O
was O
initiated O
to O
evaluate O
the O
effect O
of O
a O
combination O
of O
the O
pure O
L O
- O
isomer O
of O
LV O
and O
5 O
- O
FU O
along O
with O
epirubicin O
in O
patients O
with O
advanced O
pancreatic O
cancer O
. O

RESULTS O
: O
After O
3 O
weeks O
of O
therapy O
, O
the O
patient O
had O
an O
increase O
in O
platelet O
count O
from O
a O
baseline O
of O
5 O
, O
000 O
to O
8 O
, O
000 O
/ O
microliter O
to O
a O
maximal O
level O
of O
33 O
, O
000 O
/ O
microliter O
. O

The O
specificity O
of O
the O
transcriptional O
activation O
by O
ISGF3 B
is O
mediated O
by O
specific O
elements O
termed O
IFN B
- I
stimulatory I
response I
element I
( O
ISRE B
) O
located O
in O
the O
promoter O
region O
of O
IFN B
- O
inducible O
genes O
. O

It O
appears O
that O
the O
K O
- O
ABC O
is O
a O
relatively O
nonbiased O
test O
suitable O
for O
the O
evaluation O
of O
both O
gifted O
and O
nongifted O
children O
regardless O
of O
race O
or O
gender O
. O

In O
another O
patient O
, O
IRPF O
recurred O
in O
the O
ureter O
of O
a O
living O
related O
renal O
graft O
6 O
months O
after O
transplantation O
. O

These O
mutants O
, O
assayed O
by O
injection O
into O
Xenopus O
oocyte O
nuclei O
, O
delimit O
the O
promoter O
to O
36 O
bp O
of O
DNA O
upstream O
of O
the O
cap O
site O
and O
73 O
bp O
of O
the O
untranslated O
mRNA O
leader O
. O

Upstream O
tissue B
inhibitor I
of I
metalloproteinases I
- I
1 I
( I
TIMP B
- I
1 I
) I
element I
- I
1 I
, O
a O
novel O
and O
essential O
regulatory O
DNA O
motif O
in O
the O
human B
TIMP I
- I
1 I
gene I
promoter I
, O
directly O
interacts O
with O
a O
30 O
- O
kDa O
nuclear O
protein O
. O

The O
results O
of O
these O
experiments O
indicated O
that O
charcoal O
, O
imbiber O
beads O
, O
and O
the O
anion O
exchanges O
resins O
, O
Dowex O
XFS O
- O
4022 O
and O
Dowex O
SBR O
- O
C1 O
, O
would O
not O
be O
useful O
agents O
for O
increasing O
the O
amount O
of O
dieldrin O
eliminated O
via O
feces O
( O
droppings O
) O
of O
chickens O
. O

The O
receptor O
for O
CSF B
- I
1 I
, O
c B
- I
Fms I
, O
is O
expressed O
in O
osteoclasts O
, O
possesses O
intrinsic O
tyrosine B
- I
kinase I
activity O
, O
and O
signals O
via O
rapid O
phosphorylation O
of O
selected O
proteins O
. O

Adaptive O
effects O
of O
repeated O
immersion O
exposure O
on O
the O
human O
body O

To O
this O
end O
we O
generated O
a O
detailed O
physical O
map O
of O
the O
genomic O
region O
spanning O
between O
sequence O
- O
tagged O
site O
markers O
D16S518 B
and O
D16S516 B
. O

Spinophilin B
, O
a O
novel O
protein B
phosphatase I
1 I
binding I
protein I
localized O
to O
dendritic O
spines O
. O

We O
have O
found O
that O
pro B
- I
IGF I
- I
2 I
can O
initially O
form O
two O
disulfide O
isomers O
that O
undergo O
rearrangement O
to O
a O
single O
conformation O
in O
vivo O
. O

While O
working O
on O
the O
toxicity O
of O
war O
gases O
. O

he O
formulated O
' O
Haber O
' O
s O
rule O
' O
, O
also O
known O
as O
C O
x O
T O
= O
constant O
in O
order O
to O
characterize O
the O
toxicity O
of O
an O
inhalant O
. O

The O
existence O
of O
a O
TFIIIB B
assembly O
pathway O
leading O
to O
the O
faithful O
transcription O
of O
natural O
eukaryotic O
tRNA O
genes O
in O
the O
absence O
of O
TFIIIC B
provides O
novel O
insights O
into O
the O
functional O
flexibility O
of O
the O
eukaryotic O
tRNA O
gene O
transcription O
machinery O
and O
on O
its O
evolution O
from O
an O
ancestral O
RNA B
polymerase I
III I
system O
relying O
on O
upstream O
, O
TATA O
- O
centered O
control O
elements O
. O

Ectopic O
Xbra B
can O
induce O
Xegr B
- I
1 I
transcription O
by O
an O
indirect O
mechanism O
that O
appears O
to O
operate O
via O
primary O
activation O
of O
fibroblast B
growth I
factor I
secretion O
. O

A O
rapid O
mixing O
device O
for O
quasielastic O
light O
scattering O
studies O
of O
reacting O
systems O
. O

The O
major O
transcription O
start O
site O
, O
designated O
as O
+ O
1 O
, O
was O
determined O
by O
RACE O
( O
rapid O
amplification O
of O
cDNA O
ends O
) O
analysis O
of O
human O
liver O
cDNA O
and O
was O
found O
to O
be O
located O
50 O
bp O
upstream O
from O
the O
translation O
start O
site O
. O

These O
aa O
residues O
correspond O
to O
codons O
59 O
and O
108 O
in O
the O
P B
. I
falciparum I
DHFR I
active I
site I
in O
which O
similar O
aa O
substitutions O
( O
Cys O
Arg O
- O
59 O
and O
Ser O
Asn O
- O
108 O
) O
are O
associated O
with O
pyrimethamine O
resistance O
. O

Of O
all O
YASR O
syndromes O
, O
the O
highest O
stability O
was O
for O
the O
Anxious O
/ O
Depressed O
scale O
. O

Here O
we O
investigate O
the O
mechanism O
of O
TPA O
induction O
of O
FGF B
- I
BP I
gene I
expression O
in O
the O
human O
ME O
- O
180 O
SCC O
cell O
line O
. O

Between O
the O
RNA O
initiation O
site O
and O
the O
coding O
region O
is O
a O
GC O
- O
rich O
hairpin O
structure O
followed O
by O
8 O
T O
residues O
that O
looks O
like O
a O
rho B
- O
independent O
transcription O
terminator O
. O

Conversely O
the O
rate O
of O
bioavailability O
was O
significantly O
different O
, O
because O
from O
the O
Reference O
patch O
the O
release O
rate O
is O
fast O
in O
the O
first O
24 O
h O
, O
leading O
to O
an O
E2 O
peak O
at O
8 O
h O
and O
to O
a O
Cmax O
in O
average O
at O
23 O
h O
. O

3 O
) O
The O
progress O
of O
fibrinolysis O
was O
observed O
by O
serial O
collection O
of O
middle O
ear O
fluid O
in O
the O
same O
side O
ear O
of O
5 O
cases O
out O
of O
17 O
cases O
having O
much O
activity O
for O
fibrin B
degradation O
, O
and O
lowering O
of O
fibrinolytic O
activity O
was O
revealed O
in O
4 O
out O
of O
these O
5 O
cases O
as O
the O
result O
. O

Thus O
G17 B
targets O
the O
c B
- I
fos I
promoter I
CArG O
sequence O
via O
Rho B
A I
- O
dependent O
pathways O
, O
and O
Rho B
A I
appears O
to O
play O
an O
important O
role O
in O
the O
regulation O
of O
the O
trophic O
action O
of O
G17 B
. O

The O
MIC90 O
of O
ABK O
against O
coagulase B
type I
IV I
strains O
was O
rather O
high O
, O
12 O
. O
5 O
micrograms O
/ O
ml O
. O

In O
each O
patient O
, O
MBF O
was O
quantified O
in O
the O
three O
major O
vascular O
territories O
: O
the O
left O
anterior O
descending O
and O
left O
circumflex O
coronary O
artery O
territories O
and O
the O
right O
coronary O
artery O
( O
control O
region O
) O
territory O
. O

These O
results O
demonstrate O
that O
m2 B
receptors I
couple O
to O
both O
G B
alpha I
i2 I
and O
G B
alpha I
i3 I
in O
vivo O
and O
that O
this O
interaction O
is O
integral O
to O
both O
positive O
and O
negative O
regulatory O
pathways O
leading O
to O
activation O
of O
PLC B
and O
desensitization O
of O
receptor O
signaling O
. O

Platelet O
count O
and O
plasma O
volume O
were O
significantly O
higher O
in O
group O
A O
than O
in O
group O
B O
throughout O
pregnancy O
. O

The O
fru B
gene I
has O
seven O
exons O
spanning O
15 O
- O
kb O
encoding O
two O
transcripts O
with O
ORFs O
of O
841 O
and O
695 O
amino O
acids O
. O

HC B
- I
toxin I
exerts O
a O
potent O
cytostatic O
effect O
on O
plant O
and O
animal O
cells O
by O
inhibiting O
histone B
deacetylase I
. O

The O
human B
RIL I
gene I
: O
mapping O
to O
human O
chromosome O
5q31 O
. O
1 O
, O
genomic O
organization O
and O
alternative O
transcripts O
. O

The O
delta O
mean O
VAF O
and O
delta O
mean O
SABP O
indicated O
varied O
mean O
values O
of O
VAF O
and O
SABP O
, O
respectively O
. O

Azoospermia O
was O
graded O
in O
the O
following O
way O
: O
adequate O
spermatozoa O
( O
A1 O
) O
, O
low O
, O
scanty O
or O
rare O
spermatozoa O
( O
A2 O
) O
, O
spermatid O
arrest O
( O
B1 O
) O
, O
spermatocyte O
arrest O
( O
B2 O
) O
, O
Sertoli O
cell O
- O
only O
pattern O
( O
C O
) O
and O
sclerosis O
( O
D O
) O
. O