RPTOR
PMID 16183647.
“Nutrients suppress phosphatidylinositol 3-kinase/Akt signaling via raptor-dependent mTOR-mediated insulin receptor substrate 1 phosphorylation”. Mol. Cell. Biol. 26 (1): 63–76. (2006). doi:10.1128/MCB.26.1.63-76.2006. PMC 1317643. PMID 16354680. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1317643/.
“Turnover of the active fraction of IRS1 involves raptor-mTOR- and S6K1-dependent serine phosphorylation in cell culture models of tuberous sclerosis”. Mol. Cell. Biol. 26 (17): 6425–34. (2006). doi:10.1128/MCB.01254-05. PMC 1592824. PMID 16914728. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1592824/.
“Dissociation of raptor from mTOR is a mechanism of rapamycin-induced inhibition of mTOR function”. Genes Cells 9 (4): 359–66. (2004). doi:10.1111/j.1356-9597.2004.00727.x. PMID 15066126.
“Rictor, a novel binding partner of mTOR, defines a rapamycin-insensitive and raptor-independent pathway that regulates the cytoskeleton”. Curr. Biol. 14 (14): 1296–302. (2004). doi:10.1016/j.cub.2004.06.054. PMID 15268862.
“Large-scale characterization of HeLa cell nuclear phosphoproteins”. Proc. Natl. Acad. Sci. U.S.A. 101 (33): 12130–5. (2004). Bibcode: 2004PNAS..10112130B. doi:10.1073/pnas.0404720101. PMC 514446. PMID 15302935. https://www.ncbi.nlm.nih.