['As an essential trace element selenium plays a significant role in many physiological functions of the organs. It is found within muscles as selenocystein in selenoprotein N, which is involved in redox-modulated calcium homeostasis and in protection against oxidative stress.', 'The effects of two different selenium compounds (selenate and NanoSe in 0.5 and 5\xa0ppm concentration for two weeks) on muscle properties of mice were examined by measuring ', 'While the grip force did not change, 5\xa0ppm selenium diets significantly increased the speed of voluntary running and the daily distance covered. Both forms of selenium increased significantly the amplitude of single twitches in EDL and SOL muscle in a concentration dependent manner. Selenate increased fatigue resistance in SOL. The amplitude of the calcium transients evoked by KCl depolarization increased significantly from the control of 343\u2009±\u200944 nM to 671\u2009±\u200951 nM in the presence of 0.5\xa0ppm selenate in FDB fibers. In parallel, the rate of calcium release during short depolarizations increased significantly from 28.4\u2009±\u20092.2 to 45.5\u2009±\u20093.8 and 52.1\u2009±\u20091.9\xa0μM/ms in the presence of 0.5\xa0ppm NanoSe and selenate, respectively. In 0.5\xa0ppm concentration both selenium compounds increased significantly the selenoprotein N expression only in EDL muscle.'] Does dietary selenium augment sarcoplasmic calcium release and mechanical performance in mice?