 For the noticeable agonist-dependent types in mesenteric SMCs, we demonstrate the flow of signaling pathways associated with intracellular calcium oscillationsand find the in-phase (out-phase) tendency among cation-cation (cation-anion), which implies the maximization of charge oscillations. For involving intercellular calcium dynamics in finite cell clusters, we observe the window-broadening of the oscillation-frequency spectrum under increasing cell numbers, which hence raises the possibility of synchronized oscillations for vasomotions.
<clarity>  For the noticeable agonist-dependent types in mesenteric SMCs, we demonstrate the flow of signaling pathways associated with intracellular calcium oscillationsand find the in-phase (out-phase) tendency among cation-cation (cation-anion), which implies the maximization of charge oscillations. For involving intercellular calcium dynamics in finite cell clusters, we observe the window-broadening of the oscillation-frequency spectrum under increasing cell numbers, which hence raises the possibility of synchronized oscillations for vasomotions.
 For the noticeable agonist-dependent types in mesenteric SMCs, we demonstrate properties of synchronization among complex signals related to calcium oscillations, and show different correlation relations between calcium and voltage signals for various synchronization and resonance conditions. For practical cell clusters, we observe the window-broadening of the oscillation-frequency spectrum under increasing cell numbers, which hence raises the possibility of synchronized oscillations for vasomotions.