 I show that these variations are strongly correlated with the levels of the ACE2 protein in the lungsbut surprisingly, despite ACE2 is the viral receptor, higher levels lead to lower fatality. This behaviour is consistent with a previous mathematical model that predicts that the speed of viral progression in URLanism has a maximum and then declines with the receptor level.
<meaning-changed>  I show that these variations are strongly correlated with the levels of the ACE2 protein in the lungsbut surprisingly, despite ACE2 is the viral receptor, higher levels lead to lower fatality. This behaviour is consistent with a previous mathematical model that predicts that the speed of viral progression in URLanism has a maximum and then declines with the receptor level.
 I show that these variations are strongly correlated with the levels of the ACE2 protein in the lungsbut surprisingly, despite ACE2 is the level of the viral receptor protein ACE2 in rat lungs, which is consistent with the still limited and apparently contradictory data on human ACE2. Surprisingly, lower levels of the receptor correlate with higher fatality. However, a previous mathematical model that predicts that the speed of viral progression in URLanism has a maximum and then declines with the receptor level.