 For involving intercellular calcium dynamics in finite cell clusters, we observe the window-broadening of the oscillation-frequency spectrum under increasing cell numbers, which hence raises the possibility of synchronized oscillations for vasomotions. Our analyses indicate that the rhythm of SMC cluster could (1) correspond to significant enhancements of signal communications among remote cells, (2) respond to calcium sparks against transient stimulations for driving follow-up globally-oscillating modes, and (3) characterize the globally-oscillating modes via frog-leap (non-molecular-diffusion) calcium waves across inhomogeneous SMCs.
<coherence>  For involving intercellular calcium dynamics in finite cell clusters, we observe the window-broadening of the oscillation-frequency spectrum under increasing cell numbers, which hence raises the possibility of synchronized oscillations for vasomotions. Our analyses indicate that the rhythm of SMC cluster could (1) correspond to significant enhancements of signal communications among remote cells, (2) respond to calcium sparks against transient stimulations for driving follow-up globally-oscillating modes, and (3) characterize the globally-oscillating modes via frog-leap (non-molecular-diffusion) calcium waves across inhomogeneous SMCs.
 For involving intercellular calcium dynamics in finite cell clusters, our analyses indicate that the rhythm of SMC cluster could (1) correspond to significant enhancements of signal communications among remote cells, (2) respond to calcium sparks against transient stimulations for driving follow-up globally-oscillating modes, and (3) characterize the globally-oscillating modes via frog-leap (non-molecular-diffusion) calcium waves across inhomogeneous SMCs.