The late-time relaxation dynamics is analyzed for a general contagion model. In this model, nodes are either active or failed. Active nodes can fail either ``spontaneously'' at any time or ``externally'' if their neighborhoods are sufficiently damaged. Failed nodes may always recover spontaneously. At late times, the breaking of time-translation-invariance is a necessary condition for physical aging. Indeed, we observe that time-translational invariance is lost for initial conditions that lie between the basins of attraction of the model's two stable stationary states. Based on corresponding mean-field predictions, we characterize the observed model behavior in terms of a phase diagram spanned by the fractions of spontaneously and externally failed nodes. For the square lattice, the phases in which the dynamics approaches one of the two stable stationary states are not linearly separable due to spatial correlation effects. Our results provide new insights into aging and relaxation phenomena that are observable in a model of general contagion processes.
We analyze the late-time relaxation dynamics for a general contagion model. In this model, nodes are either active or failed. Active nodes can fail either "spontaneously" at any time or "externally" if their neighborhoods are sufficiently damaged. Failed nodes may always recover spontaneously. At late times, the breaking of time-translation-invariance is a necessary condition for physical aging. We observe that time-translational invariance is lost for initial conditions that lie between the basins of attraction of the model's two stable stationary states. Based on corresponding mean-field predictions, we characterize the observed model behavior in terms of a phase diagram spanned by the fractions of spontaneously and externally failed nodes. For the square lattice, the phases in which the dynamics approaches one of the two stable stationary states are not linearly separable due to spatial correlation effects. Our results provide new insights into aging and relaxation phenomena that are observable in a model of social contagion processes.
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