Andaman cobra
The Andaman cobra or Andaman spitting cobra ("Naja sagittifera") is a species of cobra endemic to the Andaman Islands of India. The name of this cobra comes from the Islands itself. It is venomous and also has the ability to spit its venom, although not as fairly well as other Asian spitting cobras. Taxonomy. "Naja sagittifera" is classified under the genus "Naja" of the family Elapidae. It was first described by Frank Wall, a British physician and herpetologist in 1913. The generic name "Naja" is a Latinisation of the Sanskrit word "" (), meaning "cobra". The specific epithet "sagittifera" is  Latin and means "arrow-bearing". The Caspian cobra ("Naja oxiana") and Monocled cobra ("Naja kaouthia") have been demonstrated to be sister clades to "Naja sagittifera". Noticeably, despite population separation caused by the Hindu Kush mountains, "N. oxiana" demonstrates a single evolutionary lineage, suggesting historic rapid range expansion. "N. oxiana" is a non-spitter unlike "N. kaouthia" and "N. sagittifera". Description. The Andaman cobra is medium to large sized snake in length, is a heavy bodied snake with long cervical ribs capable of expansion to form a hood when threatened, it may also spit its venom into the eyes, although not  as accurately as the true spitting cobras. Body is compressed dorsoventrally and sub-cylindrical posteriorly. The average length of these snakes is about , but they can grow to about regularly, although the longest specimen found measured . Adults come in three color patterns: a) Uniform glossy black above, with indistinct black monocle hood mark. Glossy dark-gray below. Head black, with pale white patches on shields. b) Light brown or creamish above, with indistinct black lines round the body and tail and indistinct black monocle hood mark, with brown centre. Pale brown below. Head light brown, spotted with black. c) Uniform  grayish above, with distinct black narrow, irregular cross-bars on the body and tail and distinct black monocle hood mark, with gray centre. Grayish below. Head black, with pale white patches on shields. Juveniles have broad black cross-band on throat or underside of neck. Juveniles are glossy black above with a series of narrow, white, irregular cross-bars on body and tail, which show 'A' shape marks when seen from the side. There is a distinct white monocle mark, with black centre. Monocle mark becomes black with the age, also a broad white band below monocle mark disappears with  age. Black below. Head black, with some shields blueish-white. Scalation. Head broad, slightly distinct from neck. Eye medium, with round pupil; nostrils large; frontal small; no loreal; 1 preocular in contact with posterior nasal; 3 postoculars; temporals 2+1 or 2+2. Scales smooth, in 27-29 : 21-23 : 15-17 rows; supralabials 7 ( 3rd and 4th in contact with the eye ), 7th longest, elongated; cuneates 1 on each side; infralabials 8, first 4 touching the first pair of genials; ventrals 172–184; subcaudals 60–64, paired; anal entire. Distribution. This species is endemic to Andaman and Little Andaman  Islands. Venom. Using WHO-recommended protocols, venom potencies of this species and its congener from mainland India ("Naja naja") were evaluated in a murine model of envenoming. The results indicated that the of "N. sagittifera" venom to be 0.475 mg/kg via intravenous injection. Like "N. kaouthia" and "N. atra", this isn't a "true spitting cobra". It may, in rare instances, eject venom. If venom gets into the eye, it can cause huge amount of pain and blindness if left untreated. Cobras causing both extensive local effects, ± flaccid paralysis, such as Naja kaouthia, generally cause a painful bite , with progressive swelling and if necrosis develops, then there is often discolouration of the skin and/or blistering first. This may progress to full thickness skin necrosis over 3-7 days. Such wounds may be extensive, sometimes involve underlying tissues and be difficult to heal. There is a potential for both secondary infection and long term morbidity. Squamous cell carcinoma can develop in such long-term sores. In addition to these unpleasant local effects, there may be systemic symptoms, such as headache, nausea, vomiting, abdominal pain and less commonly, evidence of mild, sometimes moderate to severe flaccid paralysis. This may develop  within a few hours or be delayed >12 hrs. Ptosis is usually the first sign, followed by ophthalmoplegia, then if it progresses, dysarthria, dysphagia, poor tongue extrusion, drooling, limb weakness, lastly respiratory paralysis. Relative rates of necrosis versus paralysis for "Naja kaouthia" vary between studies, but it appears necrosis will develop in about 10-40% of cases, while paralysis occurs in >50% of cases. Infection of the bite area is also common, as high as 58% of cases. Dry bites constitute roughly 20-40% of all bites.