Assertion: authentic prion | /r/HasProperty | patient
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Source sentences:
text: Notably, while patients with different PRNP missense mutations that produce full-length mutant PrP (for example P102L) would be expected to be capable of co-propagating both authentic prions (infectious PrP rods) and alternative transmissible PrP amyloid assemblies, patients with different PRNP stop mutations which produce C-terminally truncated PrP devoid of N-linked glycans (for example Y163X; Mead et al., 2013) may only be capable of propagating transmissible PrP amyloids giving rise to distinct disease phenotypes (Mead and Reilly, 2015).