Carbon monoxide (CO) released from its pharmacological donor prevents esophageal mucosa against experimental reflux esophagitis. Possible involvement of annexin 1, prostaglandin E2, endogenous hydrogen sulfide producing enzymes and selected vasoactive mechanisms
Authors: Marcin Magierowski, Dagmara Wójcik, Aleksandra Danielak, Robert Pajdo, Edyta Korbut, Dominik Bakalarz, Zbigniew Sliwowski, Grzegorz Ginter, Anna Chmura, Slawomir Kwiecien, Tomasz Brzozowski
Published in: 27th United European Gastroenterology Week 2019
Publication type: conference