tbuckley/cabot-search huggingface dataset:
Abstract 4146113: Clinical, Sociodemographic, Lifestyle, and Genetic Factors Associated with Worsening Heart Failure: Evidence from the UK Biobank
Authors: Taehyun Hwang, Pil-sung Yang, EUNSUN JANG, Daehoon Kim, Hee Tae Yu, Tae-Hoon Kim, Jae-Sun Uhm, Jung-Hoon Sung, Hui-Nam Pak, Moon-Hyoung Lee, BOYOUNG JOUNG
Journal: Circulation (2024)

Background: Heart failure (HF) is the leading cause of hospitalization and death. Hypothesis: This study examined the associations and clinical utilities of genetic, sociodemographic, lifestyle, laboratory, and clinical risk factors for worsening HF (WHF). Methods: Data were from 4,913 UK Biobank middle-aged adults with established HF at enrolment. WHF comprised HF admission, visit events of the emergency department due to acute congestive HF, and cardiovascular death. We performed Cox proportional hazard regression analyses to investigate the associations related to WHF after enrolment utilizing 24 variables encompassing sociodemographic, clinical, lifestyle, and laboratory data. To assess the impact of genetic factors, we additionally adjusted the polygenic risk score (PRS) for coronary artery disease (CAD). Results: Over a median follow-up of 11.5 [10.9-12.1] years, 411 (8.4%) WHF events occurred. Among the 24 variables, statistically significant hazard ratios were observed for 12 variables, with hypertension (2.20, 1.64-2.94), AF (1.99, 1.58-2.49), coronary heart disease (1.90, 1.51-2.40), and current smoking (1.77, 1.29-2.44), showing the highest hazard ratios, while female gender (0.47, 0.36-0.62) exhibited the smallest hazard ratio. After adjusting for CAD PRS, CAD PRS (HR 1.16, 1.05-1.28) showed significance in relation to WHF. In the WHF prediction model, the C index was 0.834 (0.816-0.851) with clinical variables, and it marginally increased to 0.847 (0.830-0.864) when all factors, including the genetic factor, were applied. Conclusions: Sociodemographic, clinical, laboratory, lifestyle, and genetic factors are each associated with worsening HF with male gender, hypertension, AF, CHD, diabetes, chronic obstructive pulmonary disease, low eGFR, low hemoglobin, high lipoprotein, cigarette smoking, no regular physical activity, and CAD PRS.
Abstract 4146163: Statewide Burden of Ischemic Heart Disease Attributable to High Temperature in India and its Burden from 1990-2021: A Benchmarking Systematic Analysis for the Global Burden of Disease Study 2021
Authors: Tapan Giri, Anaswara gopi, Deval Surana, Danish Ahmed, Ashwinikumar Shandilya, Mohit Lakkimsetti, Saif Syed, VISHRANT AMIN, Rabia Islam, Paritaben Bhalodia, JUHI PATEL, HARDIK DESAI, kamal sharma, Kshitija Manishbhai Thakkar
Journal: Circulation (2024)

Introduction: Ischemic Heart Disease (IHD) is the primary cause of death and disability among all cardiovascular diseases in India. As global warming escalates, the role of environmental changes, particularly high temperatures, has become a major concern. This study uniquely focuses on high temperatures (HT) as a significant environmental risk factor for IHD, providing the first estimates of its impact over the last three decades in India, including the initial two years of the COVID-19 pandemic. Method: Using the global burden of disease 2021 standardized methodology, we estimated deaths, disability-adjusted life years (DALYs), and years of life lost (YLLs) due to IHD attributable to HT in India by age, sex, year and location from 1990-2021. The results were presented in absolute counts and age-standardized rate (per 100,000 person-years). Results: From 1990 to 2021, the total number of deaths due to IHD attributable to HT in India rose from 10,874 (95% uncertainty interval: 2,320-19,061) to 40,245 (13,140-68,121). The age-standardized mortality rate (ASMR) saw a total percentage change (TPC) of 42% (19%-178%), and the DALYs rate, (ASDALR) increased by 33% (11%-156%) during this period. Uttar Pradesh recorded the highest total number of deaths at 6,202, followed by Gujarat with 4,821. The highest death counts were in the 70-74 age group, totalling 5,704 (1,888-9,578), and the highest DALYs were in the 55-59 age group at 132,936 (42,040-227,650) in 2021. In terms of gender, males exhibited a consistently higher burden over the last three decades, with a TPC in deaths of 56% for males compared to 30% for females, and DALYs of 44% for males compared to 21% for females. Conclusion: In 2021, deaths due to IHD attributable to HT accounted for 2.46% of all CVD-related deaths in India. The rapid urbanization, industrialization, and increasing pollution in India require urgent action from a public health policy perspective. Stakeholders, including government agencies and environmental groups, must collaborate to develop policies that address these issues effectively, aiming to reduce the health impact of environmental risks and enhance cardiovascular health across the nation.
Preclinical evaluation of the CD38‐targeting engineered toxin body MT‐0169 against multiple myeloma
Authors: Wassilis S. C. Bruins, Rosa Rentenaar, John Newcomb, Wenrou Zheng, Ruud W. J. Ruiter, Thomas Baardemans, Eric Poma, Chris Moore, Garrett L. Robinson, Anya Lublinsky, Yuhong Zhang, Sakeena Syed, Michael Milhollen, Ajeeta B. Dash, Niels W. C. J. van de Donk, Richard W. J. Groen, Sonja Zweegman, Tuna Mutis
Journal: HemaSphere (2024)

Abstract Despite significant progress in the treatment of multiple myeloma (MM), relapsed/refractory patients urgently require more effective therapies. We here describe the discovery, mechanism of action, and preclinical anti‐MM activity of engineered toxin body MT‐0169, a next‐generation immunotoxin comprising a CD38‐specific antibody fragment linked to a de‐immunized Shiga‐like toxin A subunit (SLTA) payload. We show that specific binding of MT‐0169 to CD38 on MM cell lines triggers rapid internalization of SLTA, causing cell death via irreversible ribosome inhibition, protein synthesis blockade, and caspase 3/7 activation. In co‐culture experiments, bone marrow mesenchymal stromal cells did not induce drug resistance against MT‐0169. In the preclinical setting, MT‐0169 effectively lysed primary MM cells from newly diagnosed and heavily pretreated MM patients, including those refractory to daratumumab, with minimal toxicity against nonmalignant hematopoietic cells. MM cell lysis showed a significant correlation with their CD38 expression levels but not with cytogenetic risk, tumor load, or number of prior lines of therapy. Finally, MT‐0169 showed efficient in vivo anti‐MM activity in various mouse xenograft models, including one in which MM cells are grown in a humanized bone marrow‐like niche. These findings support clinical investigation of MT‐0169 in relapsed/refractory MM patients, including those refractory to CD38‐targeting immunotherapies.
Single-cell transcriptomic landscape deciphers olfactory neuroblastoma subtypes and intra-tumoral heterogeneity
Authors: Jingyi Yang, Xiaole Song, Huankang Zhang, Quan Liu, Ruoyan Wei, Luo Guo, Cuncun Yuan, Fu Chen, Kai Xue, Yuting Lai, Li Wang, Junfeng Shi, Chengle Zhou, Juan Wang, Yingxuan Yu, Qibing Mei, Li Hu, Huan Wang, Chen Zhang, Qianqian Zhang, Houyong Li, Ye Gu, Weidong Zhao, Huapeng Yu, Jingjing Wang, Zhuofu Liu, Han Li, Shixing Zheng, Juan Liu, Lu Yang, Wanpeng Li, Rui Xu, Jiani Chen, Yumin Zhou, Xiankui Cheng, Yiqun Yu, Dehui Wang, Xicai Sun, Hongmeng Yu
Journal: Nature Cancer (2024)
Developmental trajectories of atopic dermatitis with multi-omics approaches in the infant gut: COCOA birth cohort
Authors: Eun Lee, Jeong-Hyun Kim, So-Yeon Lee, Si Hyeon Lee, Yoon Mee Park, Hea Young Oh, Jeonghun Yeom, Hee-Sung Ahn, Hyun Ju Yoo, Bong-Soo Kim, Sun Mi Yun, Eom Ji Choi, Kun Baek Song, Min Jee Park, Kangmo Ahn, Kyung Won Kim, Youn Ho Shin, Dong In Suh, Joo Young Song, Soo-Jong Hong
Journal: Journal of Allergy and Clinical Immunology (2024)
BTK and MMP9 regulate NLRP3 inflammasome-dependent cytokine and NET responses in primary neutrophils.
Authors: Vinicius N C Leal, Francesca Bork, Maria Mateo Tortola, Juli-Christin von Guilleaume, Carsten L Greve, Stefanie Bugl, Bettina Danker, Bodo Grimbacher, Alessandra Pontillo, Alexander N R Weber
Journal: Journal of Allergy and Clinical Immunology (2024)

Inflammation is a double-edged state of immune activation required to resolve threats harmful to the host but can also cause severe collateral damage. Polymorphonuclear neutrophils (PMN) the primary leukocyte population in humans, mediate inflammation through the release of cytokines and neutrophil extracellular traps (NETs). Whilst the pathophysiological importance of NETs is unequivocal, the multiple molecular pathways driving NET release are not fully defined. Recently, NET release was linked to the NLRP3 inflammasome which is regulated by Bruton's tyrosine kinase in macrophages.
Large Scale Ultrafast Manufacturing of Wireless Soft Bioelectronics Enabled by Autonomous Robot Arm Printing Assisted by a Computer Vision‐Enabled Guidance System for Personalized Wound Healing
Authors: Jihyun Kim, Seol‐Ha Jeong, Brendan Craig Thibault, Javier Alejandro Lozano Soto, Hiroyuki Tetsuka, Surya Varchasvi Devaraj, Estefania Riestra, Yeongseok Jang, Jeong Wook Seo, Rafael Alejandro Cornejo Rodríguez, Lucia L. Huang, Yuhan Lee, Ioana Preda, Sameer Sonkusale, Lance Fiondella, Jungmok Seo, Lorenzo Pirrami, Su Ryon Shin
Journal: Advanced Healthcare Materials (2024)

A Customized wound patch for Advanced tissue Regeneration with Electric field (CARE), featuring an autonomous robot arm printing system guided by a computer vision-enabled guidance system for fast image recognition is introduced. CARE addresses the growing demand for flexible, stretchable, and wireless adhesive bioelectronics tailored for electrotherapy, which is suitable for rapid adaptation to individual patients and practical implementation in a comfortable design. The visual guidance system integrating a 6-axis robot arm enables scans from multiple angles to provide a 3D map of complex and curved wounds. The size of electrodes and the geometries of power-receiving coil are essential components of the CARE and are determined by a MATLAB simulation, ensuring efficient wireless power transfer. Three heterogeneous inks possessing different rheological behaviors can be extruded and printed sequentially on the flexible substrates, supporting fast manufacturing of large customized bioelectronic patches. CARE can stimulate wounds up to 10 mm in depth with an electric field strength of 88.8 mV mm
Enhanced Hemodynamics of Anisometric TPMS Topology Reduce Blood Clotting in 3D Printed Blood Contactors
Authors: Lukas T. Hirschwald, Franziska Hagemann, Maik Biermann, Paul Hanßen, Patrick Hoffmann, Tim Höhs, Florian Neuhaus, Maerthe Theresa Tillmann, Petar Peric, Maximilian Wattenberg, Maik Stille, Tamara Fechter, Alexander Theißen, Patrick Winnersbach, Kai P. Barbian, Sebastian V. Jansen, Ulrich Steinseifer, Bettina Wiegmann, Rolf Rossaint, Matthias Wessling, Christian Bleilevens, John Linkhorst
Journal: Advanced Healthcare Materials (2024)

Artificial organs, such as extracorporeal membrane oxygenators, dialyzers, and hemoadsorber cartridges, face persistent challenges related to the flow distribution within the cartridge. This uneven flow distribution leads to clot formation and inefficient mass transfer over the device's functional surface. In this work, a comprehensive methodology is presented for precisely integrating triply periodic minimal surfaces (TPMS) into module housings and question whether the internal surface topology determining the flow distribution affects blood coagulation. Three module types are compared with different internal topologies: tubular, isometric, and anisometric TPMS. First, this study includes a computational fluid dynamics (CFD) simulation of the internal hemodynamics, validated through experimental residence time distributions (RTD). Blood tests using human whole blood and subsequent visualization of blood clots by computed tomography, allow the quantification of structure-induced blood clotting. The results indicate that TPMS topologies, particularly anisometric ones, serve as effective flow distributors and significantly reduce and delay blood clotting compared to conventional tubular geometries. For these novel TPMS modules, the inner surfaces can be activated chemically or functionalized to function as a selective adsorption site or biocatalytic surface or made of a permeable material to facilitate mass transfer.
Effect of nicotinamide riboside on airway inflammation in COPD: a randomized, placebo-controlled trial
Authors: Kristoffer L Norheim, Michael Ben Ezra, Indra Heckenbach, Louise Munkholm Andreasson, Lise Lotte Eriksen, Nanna Dyhre-Petersen, Mads Vargas Damgaard, Magnus Berglind, Luca Pricolo, Dayle Sampson, Ryan W Dellinger, Asger Sverrild, Jonas T Treebak, Sisse Bolm Ditlev, Celeste Porsbjerg, Morten Scheibye-Knudsen
Journal: Nature Aging (2024)

Chronic obstructive pulmonary disease (COPD) is a progressive, incurable disease associated with smoking and advanced age, ranking as the third leading cause of death worldwide. DNA damage and loss of the central metabolite nicotinamide adenine dinucleotide (NAD+) may contribute to both aging and COPD, presenting a potential avenue for interventions. In this randomized, double-blind, placebo-controlled clinical trial, we treated patients with stable COPD (n = 40) with the NAD+ precursor nicotinamide riboside (NR) for 6 weeks and followed-up 12 weeks later. The primary outcome was change in sputum interleukin-8 (IL-8) from baseline to week 6. The estimated treatment difference between NR and placebo in IL-8 after 6 weeks was −52.6% (95% confidence interval (CI): −75.7% to −7.6%; P = 0.030). This effect persisted until the follow-up 12 weeks after the end of treatment (−63.7%: 95% CI −85.7% to −7.8%; P = 0.034). For secondary outcomes, NR treatment increased NAD+ levels by more than twofold in whole blood, whereas IL-6 levels in plasma remained unchanged. In exploratory analyses, treatment with NR showed indications of upregulated gene pathways related to genomic integrity in the airways and reduced epigenetic aging, possibly through a reduction in cellular senescence. These exploratory analyses need to be confirmed in future trials. ClinicalTrials.gov identifier: NCT04990869 . Drivers of physiological aging are also linked to the etiology of chronic obstructive pulmonary disease (COPD), including inflammation and senescence, both influenced by nicotinamide adenine dinucleotide (NAD+) metabolism. Norheim et al. performed a randomized controlled trial in patients with COPD, testing whether boosting NAD+ levels reduces airway inflammation.
Multimodal evaluation of network activity and optogenetic interventions in human hippocampal slices
Authors: John P Andrews, Jinghui Geng, Kateryna Voitiuk, Matthew A T Elliott, David Shin, Ash Robbins, Alex Spaeth, Albert Wang, Lin Li, Daniel Solis, Matthew G Keefe, Jessica L Sevetson, Julio A Rivera de Jesús, Kevin C Donohue, H Hanh Larson, Drew Ehrlich, Kurtis I Auguste, Sofie Salama, Vikaas Sohal, Tal Sharf, David Haussler, Cathryn R Cadwell, David V Schaffer, Edward F Chang, Mircea Teodorescu, Tomasz Jan Nowakowski
Journal: Nature Neuroscience (2024)
Humoral and cellular responses to a fifth bivalent SARS-CoV-2 vaccine dose in patients with immune-mediated inflammatory diseases on tumour necrosis factor inhibitors: a prospective cohort study
Authors: Hilde S Ørbo, Taissa de Matos Kasahara, Asia-Sophia Wolf, Kristin H Bjørlykke, Joseph Sexton, Ingrid Jyssum, Anne T Tveter, Guri Solum, Ingrid Fadum Kjønstad, Sabin Bhandari, Ingrid E Christensen, Tore K Kvien, Andreas Lind, Hassen Kared, Jørgen Jahnsen, Espen A Haavardsholm, Ludvig A Munthe, Sella A Provan, John T Vaage, Siri Mjaaland, Silje Watterdal Syversen, Kristin K Jørgensen, Gunnveig Grødeland, Guro Løvik Goll
Journal: The Lancet Regional Health - Europe (2024)
Gene expression signature-defined necroinflammation is not associated with sex-biased survival or bevacizumab benefit in glioblastoma
Authors: Johannes Weller, Emre Kocakavuk, Barbara Pregler, Thomas Zeyen, Niklas Schäfer, Anna-Laura Potthoff, Matthias Schneider, Ulrich Herrlinger
Journal: Neuro-Oncology (2024)

Journal Article Corrected proof Gene expression signature-defined necroinflammation is not associated with sex-biased survival or bevacizumab benefit in glioblastoma Get access Johannes Weller, Johannes Weller Department of Vascular Neurology, Center of Neurology, University Hospital Bonn, Bonn, GermanyCenter of Integrated Oncology (CIO ABCD), University Hospital Bonn, Bonn, GermanyDepartment of Neurooncology, Center of Neurology, University Hospital Bonn, Bonn, Germany Corresponding Author: Johannes Weller, MD, Department of Neurooncology, University Hospital Bonn, Venusberg Campus 1, 53121 Bonn, Germany (Johannes.weller@ukbonn.de). https://orcid.org/0000-0001-5818-5392 Search for other works by this author on: Oxford Academic PubMed Google Scholar Emre Kocakavuk, Emre Kocakavuk Department of Hematology and Stem Cell Transplantation, West German Cancer Center, University Hospital Essen, Essen, Germany Search for other works by this author on: Oxford Academic PubMed Google Scholar Barbara Pregler, Barbara Pregler Department of Neurosurgery, University Hospital Bonn, Bonn, Germany Search for other works by this author on: Oxford Academic PubMed Google Scholar Thomas Zeyen, Thomas Zeyen Center of Integrated Oncology (CIO ABCD), University Hospital Bonn, Bonn, GermanyDepartment of Neurooncology, Center of Neurology, University Hospital Bonn, Bonn, Germany https://orcid.org/0009-0006-2919-8622 Search for other works by this author on: Oxford Academic PubMed Google Scholar Niklas Schäfer, Niklas Schäfer Center of Integrated Oncology (CIO ABCD), University Hospital Bonn, Bonn, GermanyDepartment of Neurooncology, Center of Neurology, University Hospital Bonn, Bonn, Germany Search for other works by this author on: Oxford Academic PubMed Google Scholar Anna-Laura Potthoff, Anna-Laura Potthoff Department of Neurosurgery, University Hospital Bonn, Bonn, Germany https://orcid.org/0000-0002-5710-6557 Search for other works by this author on: Oxford Academic PubMed Google Scholar Matthias Schneider, Matthias Schneider Department of Neurosurgery, University Hospital Bonn, Bonn, Germany https://orcid.org/0000-0002-6025-7479 Search for other works by this author on: Oxford Academic PubMed Google Scholar Ulrich Herrlinger Ulrich Herrlinger Center of Integrated Oncology (CIO ABCD), University Hospital Bonn, Bonn, GermanyDepartment of Neurooncology, Center of Neurology, University Hospital Bonn, Bonn, Germany Search for other works by this author on: Oxford Academic PubMed Google Scholar Neuro-Oncology, noae216, https://doi.org/10.1093/neuonc/noae216 Published: 15 November 2024 Article history Corrected and typeset: 15 November 2024 Published: 15 November 2024
Video introduction: Special issue on alcohol use disorder and liver disease
Authors: Michael R. Lucey
Journal: Hepatology (2024)

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Fellows’ Corner
Authors: Maya Deeb
Journal: Hepatology (2024)

Division of Liver Diseases, Department of Medicine, Recanati/Miller Transplantation Institute, Icahn School of Medicine at Mount Sinai, New York, New York, USA Abbreviations: AAH, alcohol-associated hepatitis; ALCHAIN, ALCoholic Hepatitis Artificial Intelligence; ALD, alcohol-associated liver disease; MetALD, metabolic and alcohol-associated liver disease. Correspondence Maya Deeb, Division of Liver Diseases, Department of Medicine, Recanati/Miller Transplantation Institute, Icahn School of Medicine at Mount Sinai, The Mount Sinai Hospital, 1 Gustave L. Levy Place, New York, NY 10029, USA. Email: [email protected]
Systolic Blood Pressure and Pulse Pressure in Heart Failure: Pooled Participant-Level Analysis of Four Trials
Authors: Henri Lu, Toru Kondo, Brian L. Claggett, Muthiah Vaduganathan, Brendon L. Neuen, Iris E. Beldhuis, Pardeep S. Jhund, Finnian R. Mc Causland, Inder S. Anand, Marc A. Pfeffer, Bertram Pitt, Faiez Zannad, Michael R. Zile, John J.V. McMurray, Scott D. Solomon, Akshay S. Desai
Journal: Journal of the American College of Cardiology (2024)
Author Correction: Duvelisib plus romidepsin in relapsed/refractory T cell lymphomas: a phase 1b/2a trial
Authors: Steven M Horwitz, Ajit J Nirmal, Jahan Rahman, Ran Xu, Esther Drill, Natasha Galasso, Nivetha Ganesan, Theresa Davey, Helen Hancock, Leslie Perez, Catherine Maccaro, Alexandra Bahgat, Evan Marzouk, Elizabeth Cathcart, Alison Moskowitz, Ariela Noy, Anita Kumar, Eric Jacobsen, David C Fisher, Neha Mehta-Shah, Youn H Kim, Michael Khodadoust, Nikita Kotlov, Anastasia Nikitina, Olga Kudryashova, Valeria Zubareva, Ksenia Zornikova, Nara Shin, Maria Sorokina, Sandrine Degryse, Ekaterina Postovalova, Aleksander Bagaev, Kinga Hosszu, Devin McAvoy, Jaap J Boelens, Wenchao Wu, Zoe Ciantra, Jackson W Appelt, Christopher Trevisani, Sam Amaka, David M Weinstock, Santosha A Vardhana
Journal: Nature Medicine (2024)
P2Y12 Inhibitor Pretreatment in Non–ST-Segment Elevation Acute Coronary Syndrome
Authors: Hiroki A Ueyama, Kevin F Kennedy, Jennifer A Rymer, Alexander T Sandhu, Toshiki Kuno, Frederick A Masoudi, John A Spertus, Shun Kohsaka
Journal: Journal of the American College of Cardiology (2024)
Association of Histologic Findings With Long-Term Outcomes in Symptomatic Obstructive Hypertrophic Cardiomyopathy Patients Undergoing Surgical Myectomy
Authors: Shada Jadam, Andrew Gaballa, Alaa Alashi, Bo Xu, Maran Thamilarasan, E Rene Rodriguez, Carmela D Tan, Susan Ospina, Nicholas Smedira, Zoran B Popovic, Milind Y Desai
Journal: JACC Heart Failure (2024)
Treating Older Patients in Cardiogenic Shock with a Microaxial Flow Pump: Is it DANGERous?
Authors: Anika Klein, Rasmus P Beske, Christian Hassager, Lisette O Jensen, Hans Eiskjær, Norman Mangner, Axel Linke, Amin Polzin, P Christian Schulze, Carsten Skurk, Peter Nordbeck, Peter Clemmensen, Vasileios Panoulas, Sebastian Zimmer, Andreas Schäfer, Nikos Werner, Thomas Engstøm, Lene Holmvang, Anders Junker, Henrik Schmidt, Christian J Terkelsen, Jacob E Møller
Journal: Journal of the American College of Cardiology (2024)
Oxidized Phospholipids and Calcific Aortic Valvular Disease
Authors: Harpreet S Bhatia, Marc R Dweck, Neil Craig, Romain Capoulade, Philippe Pibarot, Patrick J Trainor, Seamus P Whelton, Rishi Rikhi, Karita C F Lidani, Wendy S Post, Michael Y Tsai, Michael H Criqui, Michael D Shapiro, Matthew J Budoff, Andrew P DeFilippis, George Thanassoulis, Sotirios Tsimikas
Journal: Journal of the American College of Cardiology (2024)
Intensive lifestyle intervention, cardiac biomarkers, and cardiovascular outcomes in diabetes: LookAHEAD cardiac biomarker ancillary study
Authors: Kershaw V Patel, Zainali Chunawala, Subodh Verma, Matthew W Segar, Katelyn R Garcia, Chiadi E Ndumele, Thomas J Wang, James L Januzzi, Antoni Bayes-Genis, Javed Butler, Carolyn S P Lam, Christie M Ballantyne, James A de Lemos, Alain G Bertoni, Mark Espeland, Ambarish Pandey
Journal: Journal of the American College of Cardiology (2024)
Biomimetic fabricated tubular graft in situ immobilized with peptides to restore the vascular structure and regulate the inflammation homeostasis through gastrodin coating
Authors: Shiwei Niu, Fan Li, Zhi Lin, Bo Chen, Lechun Lyu, Yongliang Jiang, Wanting Li, Jianlin Jiao, Yi Han, Di Lu, Lin Sun
Journal: Bioactive Materials (2024)