tbuckley/cabot-search huggingface dataset:
FDG PET/CT in the Diagnosis and After Sunitinib Follow-up of Synchronous Base of Tongue and Thyroid Metastases From Renal Cell Carcinoma
Authors: Srinivas Ananth Kumar, Bhagwant Rai Mittal, Suraj Kumar, Harmandeep Singh, Rajender Kumar, Amanjit Bal
Journal: Clinical Nuclear Medicine (2024)

Renal cell carcinoma (RCC) is a leading cause of mortality among genitourinary malignancies with limited therapeutic options. The hematogenous route, lymphatic spread, and direct invasion have been documented in RCC. Usually, metastases are regional lymph nodes, lungs, bone, liver, adrenal glands, contralateral kidney, and brain. Metastases to the rare sites such as skin, breast, head and neck were documented in the literature. In the present case, we describe the synchronous metastases to the base of the tongue and thyroid gland in RCC and the response to sunitinib therapy on 18F-FDG PET/CT.
Studying the stability of polymer nanoparticles by size exclusion chromatography of radioactive polymers
Authors: Amrita Dikpati, Vanessa Dos Passos Maio, Ece Ates, Karine Greffard, Nicolas Bertrand
Journal: Journal of Controlled Release (2024)

The properties of nanomedicines will influence how they can deliver drugs to patients reproducibly and effectively. For conventional pharmaceutical products, Chemistry, Manufacturing and Control (CMC) documents require monitoring stability and storage conditions. For nanomedicines, studying these important considerations is hindered by a lack of appropriate methods. In this paper, we show how combining radiolabelling with size exclusion chromatography, using a method called SERP (for Size Exclusion of Radioactive Polymers), can inform on the in vitro degradation of polymer nanoparticles. Using nanoparticles composed of biodegradable poly(lactic acid) (PLA) and poly(lactic-co-glycolic acid) (PLGA), we show that SERP is more sensitive than dynamic light scattering (DLS) and nanoparticle tracking analysis (NTA) to detect degradation. We also demonstrate that the properties of the polymer composition and the nature of the aqueous buffer affect nanoparticle degradation. Importantly, we show that minute changes in stability that cannot be detected by DLS and NTA impact the pharmacokinetic of nanoparticles injected in vivo. We believe that SERP might prove a valuable method to document and understand the pharmaceutical quality of polymer nanoparticles.
Under threat: the International AIDS Society–Lancet Commission on Health and Human Rights
Authors: Chris Beyrer, Adeeba Kamarulzaman, Michael Isbell, Joseph Amon, Stefan Baral, Mary T Bassett, Javier Cepeda, Harriet Deacon, Lorraine Dean, Lilianne Fan, Rita Giacaman, Carolyn Gomes, Sofia Gruskin, Ravi Goyal, Sandra Hsu Hnin Mon, Samer Jabbour, Michel Kazatchkine, Kasoka Kasoka, Carrie Lyons, Allan Maleche, Natasha Martin, Martin McKee, Vera Paiva, Lucy Platt, Dainius Puras, Robert Schooley, Gerson Smoger, Lucy Stackpool-Moore, Peter Vickerman, Josephine G Walker, Leonard Rubenstein
Journal: The Lancet (2024)
Pembrolizumab or placebo with chemoradiotherapy followed by pembrolizumab or placebo for newly diagnosed, high-risk, locally advanced cervical cancer (ENGOT-cx11/GOG-3047/KEYNOTE-A18): a randomised, double-blind, phase 3 clinical trial
Authors: Domenica Lorusso, Yang Xiang, Kosei Hasegawa, Giovanni Scambia, Manuel Leiva, Pier Ramos-Elias, Alejandro Acevedo, Vladyslav Sukhin, Noelle Cloven, Andrea J Pereira de Santana Gomes, Fernando Contreras Mejía, Ari Reiss, Ali Ayhan, Jung-Yun Lee, Valeriya Saevets, Flora Zagouri, Lucy Gilbert, Jalid Sehouli, Ekkasit Tharavichitkul, Kristina Lindemann, Roberta Lazzari, Chih-Long Chang, Rudolf Lampé, Hong Zhu, Ana Oaknin, Melissa Christiaens, Stephan Polterauer, Tomoka Usami, Kan Li, Karin Yamada, Sarper Toker, Stephen M Keefe, Sandro Pignata, Linda R Duska
Journal: The Lancet (2024)
Interaction of SMAC with a survivin-derived peptide alters essential cancer hallmarks: Tumor growth, inflammation, and immunosuppression
Authors: Manikandan Santhanam, Swaroop Kumar Pandey, Anna Shteinfer-Kuzmine, Avijit Paul, Nur Abusiam, Ran Zalk, Varda Shoshan-Barmatz
Journal: Molecular Therapy (2024)

SMAC/Diablo is known as a pro-apoptotic mitochondrial protein released into the cytosol in response to apoptotic signals. We recently reported SMAC overexpression in cancers as essential for cell proliferation and tumor growth due to non-apoptotic functions including phospholipid synthesis regulation. These functions may be associated with its interactions with partner proteins. Using a peptide array with 768 peptides derived from 11 selected SMAC-interacting proteins, we identified SMAC-interacting sequences. These SMAC-binding sequences were produced as cell penetrating peptides targeted to the cytosol, mitochondria, or nucleus, inhibiting cell proliferation and inducing apoptosis in several cell lines. For in-vivo study, a survivin/BIRC5-derived peptide was selected due to its overexpression in many cancers, and involvement in mitosis, apoptosis, autophagy, cell proliferation, inflammation, and immune responses, as a target for cancer therapy. Specifically, a SMAC-targeting survivin/BIRC5-derived peptide, given intratumorally or intravenously, strongly inhibited lung tumor growth, cell proliferation, angiogenesis, and inflammation, induced apoptosis, and remodeled the tumor microenvironment. The peptide promoted tumor infiltration of CD8+ cells and increased cell-intrinsic PD-1/PD-L1 expression, resulting in cancer cell self-destruction and increased tumor cell death, preserving immune cells. Thus, targeting the interaction between the multifunctional proteins SMAC and survivin represents a innovative therapeutic cancer paradigm.
Timing of radiotherapy (RT) after radical prostatectomy (RP): long-term outcomes in the RADICALS-RT trial (NCT00541047)
Authors: C.C. Parker, P.M. Petersen, A.D. Cook, N.W. Clarke, C. Catton, W.R. Cross, H. Kynaston, W.R. Parulekar, R.A. Persad, F. Saad, L. Bower, G.C. Durkan, J. Logue, C. Maniatis, D. Noor, H. Payne, J. Anderson, A.K. Bahl, F. Bashir, D.M. Bottomley, K. Brasso, L. Capaldi, C. Chung, P.W. Cooke, J.F. Donohue, B. Eddy, C.M. Heath, A. Henderson, A. Henry, R. Jaganathan, H. Jakobsen, N.D. James, J. Joseph, K. Lees, J. Lester, H. Lindberg, A. Makar, S.L. Morris, N. Oommen, P. Ostler, L. Owen, P. Patel, A. Pope, R. Popert, R. Raman, V. Ramani, A. Røder, I. Sayers, M. Simms, V. Srinivasan, S. Sundaram, K.L. Tarver, A. Tran, P. Wells, J. Wilson, A.M. Zarkar, M.K.B. Parmar, M.R. Sydes
Journal: Annals of Oncology (2024)

The optimal timing of radiotherapy (RT) after radical prostatectomy for prostate cancer has been uncertain. RADICALS-RT compared efficacy and safety of adjuvant RT versus an observation policy with salvage RT for prostate-specific antigen (PSA) failure.
Polyploidisation pleiotropically buffers ageing in hepatocytes
Authors: Kelvin Yin, Maren Büttner, Ioannis K. Deligiannis, Mateusz Strzelecki, Liwei Zhang, Carlos Talavera-López, Fabian Theis, Duncan T. Odom, Celia P. Martinez-Jimenez
Journal: Journal of Hepatology (2024)

Background & Aims Polyploidy in hepatocytes has been proposed as a genetic mechanism to buffer against transcriptional dysregulation. Here, we aim to demonstrate the role of polyploidy in modulating gene regulatory networks in hepatocytes during ageing. Methods We performed single-nucleus RNA-sequencing in hepatocyte nuclei of different ploidy levels isolated from young and old wild-type mice. Changes in the gene expression and regulatory network were compared to three independent haploinsufficient strains for HNF4A, CEBPA or CTCF, representing non-deleterious perturbations. Phenotypic characteristics of the liver section were additionally evaluated histologically, whereas the genomic allele composition of hepatocytes was analysed by BaseScope. Results We observed that ageing in wild-type mice results in nuclei polyploidy and marked increase in steatosis. Haploinsufficiency of liver-specific master regulators (HFN4A or CEBPA) results in the enrichment of hepatocytes with tetraploid nuclei at a young age, affecting the genomic regulatory network, and dramatically suppressing ageing-related steatosis tissue-wide. Notably, these phenotypes are not the result of subtle disruption to liver-specific transcriptional networks, since haploinsufficiency in CTCF insulator protein resulted in the same phenotype. Further quantification of genotypes of tetraploid hepatocytes in young and old HFN4A haploinsufficient mice revealed that during ageing, tetraploid hepatocytes lead to the selection of wild-type alleles, restoring non-deleterious genetic perturbation. ConclusionsOur results suggest a model whereby polyploidisation leads to fundamentally different cell states. Polyploid conversion enables pleiotropic buffering against age-related decline via non-random allelic segregation to restore a wild-type genome.
Change in Age profile of Respiratory Syncytial Virus disease over the course of annual epidemics: a multi-national study
Authors: Saverio Caini, Jean-Sebastien Casalegno, Ana Paula Rodrigues, Vernon Lee, Cheryl Cohen, Q.Sue Huang, Alfredo Bruno Caicedo, Anne Teirlinck, Raquel Guiomar, Li Wei Ang, Jocelyn Moyes, Tim Wood, Doménica de Mora, Mathieu Bangert, Rolf Kramer, Lisa Staadegaard, Susanne Heemskerk, Jojanneke van Summeren, Adam Meijer, John Paget
Journal: Journal of Infection (2024)

We aimed to study whether the percentwise age distribution of RSV cases changes over time during annual epidemics.
Loss of stress sensor GADD45A promotes stem cell activity and ferroptosis resistance in LGR4/HOXA9-dependent AML
Authors: Nunki Hassan, Hangyu Yi, Bilal Malik, Lucie Gaspard-Boulinc, Saumya E. Samaraweera, Debora A. Casolari, Janith Seneviratne, Anushree Balachandran, Tracy Chew, Alastair Duly, Daniel R. Carter, Belamy B. Cheung, Murray Norris, Michelle Haber, Maria Kavallaris, Glenn M. Marshall, Xu Dong Zhang, Tao Liu, Jianlong Wang, Dan A. Liebermann, Richard J. D’Andrea, Jenny Y. Wang
Journal: Blood (2024)

Abstract The overall prognosis of acute myeloid leukemia (AML) remains dismal, largely because of the inability of current therapies to kill leukemia stem cells (LSCs) with intrinsic resistance. Loss of the stress sensor growth arrest and DNA damage-inducible 45 alpha (GADD45A) is implicated in poor clinical outcomes, but its role in LSCs and AML pathogenesis is unknown. Here, we define GADD45A as a key downstream target of G protein-coupled receptor (LGR)4 pathway and discover a regulatory role for GADD45A loss in promoting leukemia-initiating activity and oxidative resistance in LGR4/HOXA9-dependent AML, a poor prognosis subset of leukemia. Knockout of GADD45A enhances AML progression in murine and patient-derived xenograft (PDX) mouse models. Deletion of GADD45A induces substantial mutations, increases LSC self-renewal and stemness in vivo, and reduces levels of reactive oxygen species (ROS), accompanied by a decreased response to ROS-associated genotoxic agents (eg, ferroptosis inducer RSL3) and acquisition of an increasingly aggressive phenotype on serial transplantation in mice. Our single-cell cellular indexing of transcriptomes and epitopes by sequencing analysis on patient-derived LSCs in PDX mice and subsequent functional studies in murine LSCs and primary AML patient cells show that loss of GADD45A is associated with resistance to ferroptosis (an iron-dependent oxidative cell death caused by ROS accumulation) through aberrant activation of antioxidant pathways related to iron and ROS detoxification, such as FTH1 and PRDX1, upregulation of which correlates with unfavorable outcomes in patients with AML. These results reveal a therapy resistance mechanism contributing to poor prognosis and support a role for GADD45A loss as a critical step for leukemia-initiating activity and as a target to overcome resistance in aggressive leukemia.
IL-18-secreting multiantigen targeting CAR T cells eliminate antigen-low myeloma in an immunocompetent mouse model
Authors: Brandon D. Ng, Adhithi Rajagopalan, Anastasia I. Kousa, Jacob S. Fischman, Sophia Chen, Alyssa Massa, Harold K. Elias, Dylan Manuele, Michael Galiano, Andri L. Lemarquis, Alexander P. Boardman, Susan DeWolf, Jonah Pierce, Bjarne Bogen, Scott E. James, Marcel R. M. van den Brink
Journal: Blood (2024)
Wiskott-Aldrich syndrome: a study of 577 patients defines the genotype as a biomarker for disease severity and survival
Authors: Tanja C. Vallée, Jannik S. Glasmacher, Hannes Buchner, Peter D. Arkwright, Uta Behrends, Anastasia Bondarenko, Michael J. Browning, David Buchbinder, Alessandro Cattoni, Liudmyla Chernyshova, Peter Ciznar, Theresa Cole, Wojciech Czogała, Gregor Dueckers, John David M. Edgar, Fatih Erbey, Anders Fasth, Francesca Ferrua, Renata Formankova, Eleonora Gambineri, Andrew R. Gennery, Frederick D. Goldman, Luis I. Gonzalez-Granado, Carsten Heilmann, Tarja Heiskanen-Kosma, Hanna Juntti, Leena Kainulainen, Hirokazu Kanegane, Neslihan E. Karaca, Sara S. Kilic, Christoph Klein, Sylwia Kołtan, Irina Kondratenko, Isabelle Meyts, Gulnara M. Nasrullayeva, Lucia D. Notarangelo, Srdjan Pasic, Isabelle Pellier, Claudio Pignata, Siraj Misbah, Ansgar Schulz, Gesmar R. Segundo, Anna Shcherbina, Mary Slatter, Robert Sokolic, Pere Soler-Palacin, Polina Stepensky, Joris M. van Montfrans, Samppa Ryhänen, Beata Wolska-Kuśnierz, John B. Ziegler, Xiaodong Zhao, Alessandro Aiuti, Hans D. Ochs, Michael H. Albert
Journal: Blood (2024)
Reply to Chen et al.: Reexamining COPD in Bronchiectasis: Elucidating Overdiagnosis and Outcomes from EMBARC's ROSE Criteria
Authors: James D. Chalmers, Anthony De Soyza, Stefano Aliberti, Eva Polverino
Journal: American Journal of Respiratory and Critical Care Medicine (2024)

"Reply to Chen et al.: Reexamining COPD in Bronchiectasis: Elucidating Overdiagnosis and Outcomes from EMBARC's ROSE Criteria." American Journal of Respiratory and Critical Care Medicine, 0(ja), pp.
Effects of Food Insecurity on Hepatic Steatosis and Fibrosis in People With HIV
Authors: Ani Kardashian, Audrey Lloyd, Eduardo Vilar-Gomez, Susanna Naggie, Mark S. Sulkowski, Tinsay Woreta, Jordan E. Lake, Holly Crandall, Rohit Loomba, Laura A. Wilson, Richard K. Sterling, Sonya Heath, Samer Gawrieh, Naga P. Chalasani, Jennifer C. Price, Susanna Naggie, Anna Mae Diehl, Mariko Kopping, Rebecca Mangus, Naga Chalasani, Kaelea Cheek, Holly Crandall, Oscar W. Cummings, Nicole Garcia, Samer Gawrieh, Montreca Releford, Eduardo Vilar-Gomez, Mark Sulkowski, Sherilyn Brinkley, Daniel Coleman, Lee Gilman, Sara Mekhael, Juhi Moon, Lauren Stelmash, Quintara Williams, Tinsay Woreta, Sonya L. Heath, Paul A. Goepfert, Meagan Gray, Olivia Hogue, Audrey Lloyd, Heather Logan, Kristen Spraggins, William Tingle, Latasha Vereen, Rohit Loomba, Veeral Ajmera, Edward Cachay, Vanessa Cervantes, Christie Hernandez, Egbert Madamba, Lisa Richards, Kaleb Tesfai, Monica Tincopa, Jennifer Price, Rosaura Camberos, Yesenia Laguardia, Annie Luetkemeyer, Jeffrey McKinney, Brittlyn Pearlman, Sophie Stern, Jordan E. Lake, Marisel Negret Hernandez, Anoma Somasunderam, Richard Sterling, Megan Devore, Daniel Nixon
Journal: Clinical Gastroenterology and Hepatology (2024)
Genome-wide Analysis Identifies Nuclear Factor 1C as a Novel Transcription Factor and Potential Therapeutic Target in Small Cell Lung Cancer
Authors: Vivek Shukla, Haitao Wang, Lyuba Varticovski, Songjoon Baek, Ruihong Wang, Xinwei Wu, Frank Echtenkamp, Frank Villa-Hernandez, Katherine P. Prothro, Sudheer K. Gara, Mary R. Zhang, Stephanie Shiffka, Razi Raziuddin, Leonard M. Neckers, W. Marston Linehan, Haobin Chen, Gordon L. Hager, David S. Schrump
Journal: Journal of Thoracic Oncology (2024)
JNJ-73763989 and bersacapavir treatment in nucleos(t)ide analogue-suppressed patients with chronic hepatitis B: REEF-2
Authors: Kosh Agarwal, Maria Buti, Florian van Bömmel, Pietro Lampertico, Ewa Janczewska, Marc Bourliere, Thomas Vanwolleghem, Oliver Lenz, Thierry Verbinnen, Thomas N. Kakuda, Cristiana Mayer, John Jezorwski, Daniel Muenz, Maria Beumont, Ronald Kalmeijer, Michael Biermer, Isabelle Lonjon-Domanec
Journal: Journal of Hepatology (2024)

Functional cure for chronic hepatitis B (CHB) requires finite treatment. Two agents under investigation with the goal of achieving functional cure are the small-interfering RNA JNJ-73763989 (JNJ-3989) and the capsid assembly modulator JNJ-56136379 (JNJ-6379; bersacapavir).
Prevalence of the Cardiovascular-Kidney-Metabolic Syndrome in the United States
Authors: Abdul Mannan Khan Minhas, Roy O. Mathew, Laurence S. Sperling, Vijay Nambi, Salim S. Virani, Sankar D. Navaneethan, Michael D. Shapiro, Dmitry Abramov
Journal: Journal of the American College of Cardiology (2024)
UNRAVELING THE DYNAMIC PROGRESSION: LYME CARDITIS TRANSFORMING FROM FIRST -DEGREE AV BLOCK TO COMPLETE HEART BLOCK IN MERE HOURS
Authors: Mohammad Ibrar, Hamza Saber, Muhammad Hassan Shakir, Salman Abdul Basit, Nadia Jamil, Aamir Makda, Muhammad Waqas, Douglas Klamp
Journal: Journal of the American College of Cardiology (2024)
EXCLUSION OF LEFT ATRIAL APPENDAGE THROMBUS WITH DOBUTAMINE PRIOR TO WATCHMAN FLX IMPLANT IN THE PRESENCE OF DENSE SPONTANEOUS ECHO CONTRAST
Authors: Mohammad Hashmi, Daniel Ice, Richard C. Kovach, Pedram Kazemian, Mihir Odak, Steven Douedi, Chunguang Chen
Journal: Journal of the American College of Cardiology (2024)
TRENDS AND DISPARITIES IN VALVULAR HEART DISEASE MORTALITY IN THE UNITED STATES FROM 1999 TO 2020
Authors: Min Choon Tan, Yong Hao Yeo, Boon Jian San, Addi Suleiman, Justin Zon-Ern Lee, Arka Chatterjee, Kristen Sell-Dottin, John P. Sweeney, F. David Fortuin, Kwan S. Lee
Journal: Journal of the American College of Cardiology (2024)
Blocking senescence and tolerogenic function of dendritic cells induced by γδ Treg cells enhances tumor-specific immunity for cancer immunotherapy
Authors: Fusheng Si, Xia Liu, Yan Tao, Yuanqin Zhang, Feiya Ma, Eddy C Hsueh, Sidharth V Puram, Guangyong Peng
Journal: Journal for ImmunoTherapy of Cancer (2024)

Background Regulatory T (Treg) cells are a key component in maintaining the suppressive tumor microenvironment and immune suppression in different types of cancers. A precise understanding of the molecular mechanisms used by Treg cells for immune suppression is critical for the development of effective strategies for cancer immunotherapy. Methods Senescence development and tolerogenic functions of dendritic cells (DCs) induced by breast cancer tumor-derived γδ Treg cells were fully characterized using real-time PCR, flow cytometry, western blot, and functional assays. Loss-of-function strategies with pharmacological inhibitor and/or neutralizing antibody were used to identify the potential molecule(s) and pathway(s) involved in DC senescence and dysfunction induced by Treg cells. Impaired tumor antigen HER2-specific recognition and immune response of senescent DCs induced by γδ Treg cells were explored in vitro and in vivo in humanized mouse models. In addition, the DC-based HER2 tumor vaccine immunotherapy in breast cancer models was performed to explore the enhanced antitumor immunity via prevention of DC senescence through blockages of STAT3 and programmed death-ligand 1 (PD-L1) signaling. Results We showed that tumor-derived γδ Treg cells promote the development of senescence in DCs with tolerogenic functions in breast cancer. Senescent DCs induced by γδ Treg cells suppress Th1 and Th17 cell differentiation but promote the development of Treg cells. In addition, we demonstrated that PD-L1 and STAT3 signaling pathways are critical and involved in senescence induction in DCs mediated by tumor-derived γδ Treg cells. Importantly, our complementary in vivo studies further demonstrated that blockages of PD-L1 and/or STAT3 signaling can prevent γδ Treg-induced senescence and reverse tolerogenic functions in DCs, resulting in enhanced HER2 tumor-specific immune responses and immunotherapy efficacy in human breast cancer models. Conclusions These studies not only dissect the suppressive mechanism mediated by tumor-derived γδ Treg cells on DCs in the tumor microenvironment but also provide novel strategies to prevent senescence and dysfunction in DCs and enhance antitumor efficacy mediated by tumor-specific T cells for cancer immunotherapy.
CARDIAC FIBROBLAST ACTIVATION AFTER ACUTE CORONARY SYNDROME DETECTED BY 99MTCIFAP SPECT, MULTIMODALITY APPROACH AND FOLLOW-UP
Authors: Jesus Hernandez, Erick Alexanderson Rosas, Estela Isabel Carvajal Juarez, Guillermina Ferro-Flores, CLARA LETICIA SANTOS CUEVAS, Myrna Luna-Gutiérrez, Gabriela Meléndez Ramírez, Joel Vargas Ahumada, Salvador Hernández Sandoval, Nadia Canseco León, Aloha Meave, CECILIA Ortiz Moreno, Elia Ortiz, Francisco Osvaldo Garcia-Perez, Juan Carlos Vazquez Fuentes, Francisco Joel Serrato Palencia, Eder Jonathan Amaro Palomo
Journal: Journal of the American College of Cardiology (2024)
EFFECTS OF SACUBITRIL/VALSARTAN IN PATIENTS WITH HEART FAILURE ACROSS THE SPECTRUM OF KIDNEY RISK
Authors: Safia Chatur, Brian Claggett, Finnian Mc Causland, Milton Packer, Akshay S. Desai, Jean L. Rouleau, Michael R. Zile, Martin Lefkowitz, John J.V. McMurray, Scott D. Solomon, Muthiah Vaduganathan
Journal: Journal of the American College of Cardiology (2024)
ANTI-HMGCR ASSOCIATED IMMUNE MEDIATED NECROTIZING MYOPATHY: A RARE CASE AND TREATMENT INSIGHTS
Authors: Abhijith Paladugula, Dipsa Shastri, Aditya Thakkar, Koushik Sanku, Muhammad Abdur Raafey, Ashley Blaske
Journal: Journal of the American College of Cardiology (2024)
THE DUAL DILEMMA: TRANSIENT CONSTRICTIVE PERICARDITIS AND COEXISTENT CHRONIC PLEURAL EFFUSION
Authors: Aqieda Bayat, Gauranga Mahalwar, Alveena Batool Syed, Abdel Hadi El Hajjar, Pradeep V. Kandula, Ankit Agrawal, Tiffany Dong, Tom Kai Ming Wang, Allan L. Klein
Journal: Journal of the American College of Cardiology (2024)
IS AIRCRAFT NOISE HARMFUL FOR THE HEART?
Authors: Constantin-Cristian Topriceanu, Xiangpu Gong, Mit Shah, Katie Eminson, Glory O Atilola, Nishi Chaturvedi, Calvin Jephcote, Kathryn Adams, Marta Blangiardo, John Gulliver, Alex Rowlands, Declan O'Regan, Anna Hansell, Gabriella Captur
Journal: Journal of the American College of Cardiology (2024)
“UNRAVELING VASOSPASTIC ANGINA: A CASE OF DIAGNOSTIC INTRICACIES”
Authors: Muhammad Umair Rana, Saad Ur Rahman, Muhammad Hasib Khalil, Talha Bin Farooq, Krishna Prasad Kurpad, Uday B. Kanakadandi, Alina Brener
Journal: Journal of the American College of Cardiology (2024)
ANTERIOR STEMI IN A 25-YEARS-OLD WITH COGAN SYNDROME
Authors: Andrew Takla, Mohamed Eid, Akshay Joshi, Moghniuddin Mohammed, Fahad Eid, Mostafa Zaalouk, Mostafa Elbanna, Mostafa Najim, Kirolus Sourial, Scott Clinton Feitell
Journal: Journal of the American College of Cardiology (2024)
CLUSTER ANALYSIS OF SLEEP-DISORDERED BREATHING PHENOTYPES IN PATIENTS WITH PULMONARY ARTERIAL HYPERTENSION TO PREDICT CLINICAL WORSENING OUTCOMES
Authors: Zhihua Huang, Anqi DUAN, Qin Luo, Zhihui Zhao, Qing Zhao, Yi Zhang, Li Xin, Sicheng Zhang, Luyang Gao, Zhihong Liu
Journal: Journal of the American College of Cardiology (2024)