tbuckley/cabot-search huggingface dataset:
PHONOCARDIOGRAPHY AND ITS CLINICAL CORRELATION
Authors: H. Arenberg
Journal: Annals of Internal Medicine (1941)

Article1 March 1941PHONOCARDIOGRAPHY AND ITS CLINICAL CORRELATIONH. ARENBERG, M.D.H. ARENBERG, M.D.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-14-9-1607 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptEver since the discovery of the heart sounds by Harvey in 1626, there must have been a desire for their graphic representation on the printed page. For, according to Orías and Braun-Menéndez1the skeptics of that day denied the existence of the heart sounds even after Harvey's classical description of the same. The refusal to recognize Harvey's discovery went so far as for one authority to state that such perfect auditory acuity as to hear heart sounds could exist only in London and nowhere else in the world. Visual demonstration would certainly have helped then to convince doubters of facts,...References1. ORÍASBRAUN-MENÉNDEZ OE: The heart sounds in normal and pathological conditions, 1939, Oxford University Press, London. Google Scholar2. EINTHOVEN W: Die Registrierung der Menschlichen Herztöne mittels des Saitengalvanometers, Pflüger's Arch., 1907, cxvii, 461. CrossrefGoogle Scholar3. FRANK O: Die Theorie der Segmentkolbenkapsel, Ztschr. f. Biol., 1913, lix, 526-530. Google Scholar4. WIGGERS CJ: Physiology in health and disease, Third Edition, 1939, Lea and Febiger, Philadelphia. Google Scholar5. BOYERECKSTEINWIGGERS NHRWCJ: Characteristics of normal heart sounds recorded by direct methods, Am. Heart Jr., 1940, xix, 257-274. CrossrefGoogle Scholar6. MCKEE MH: Heart sounds in normal children, Am. Heart Jr., 1938, xvi, 79-87. CrossrefGoogle Scholar7. LOCKHART ML: Stethograph, Am. Heart Jr., 1938, xvi, 72-78. CrossrefGoogle Scholar8. WOLFERTHMARGOLIES CCA: Gallop rhythm and physiological third heart sound; characteristics of sounds, classification, comparative incidence of various types and differential diagnosis, Am. Heart Jr., 1933, viii, 441-461. CrossrefGoogle Scholar9. O'FARRELL PT: What is gallop rhythm? Irish Jr. Med. Sci., 1939, xxxiii, 729-739. CrossrefGoogle Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: New York, N. Y.*Received for publication November 9, 1940.From the U. S. Public Health Service. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics Cited byAortic valve insufficiency in arterial hypertension∗Phonocardiographic differentiation of vibratory (functional) murmurs from those of valvular insufficiency: Further observations and application to the diagnosis of rheumatic heart diseaseThe graphic registration of basal diastolic murmurs 1 March 1941Volume 14, Issue 9Page: 1607-1616KeywordsHeartMedical services ePublished: 1 December 2008 Issue Published: 1 March 1941 PDF downloadLoading ...
Abstract 3387: Hypoxia-induced Vascular Repair by Progenitor Cells Requires the Nadph Oxidase Nox2
Authors: Katrin Schröder, Andreas Kohen, Alexandra Aicher, Ralf P Brandes
Journal: Circulation (2008)

Endothelial progenitor cells (EPC) promote vascular repair but the impact of systemic hypoxia on EPC function is unclear. Hypoxia is a strong stimulus for angiogenesis and affects reactive-oxygen species (ROS) production and signalling. We studied the role of hypoxia for EPC mobilization and vascular repair and the contribution of the NADPH oxidase Nox2, the most important source of ROS in endothelial cells, in this process. Murine EPCs were defined as linage negative and sca-1/flk-1 positive and measured by FACS from whole blood. Vascular repair was induced by the carotid artery wire injury model. Hypoxia (10%, 96hours) induced mobilisation of EPC and improved vascular repair after wire injury in wild-type mice but not in NADPH oxidase knockout mice (Nox2y/−). Hypoxia induces the formation of erythropoietin (EPO), which might contribute to EPC mobilization. EPO production in response to hypoxia however was enhanced in Nox2y/− as compared to WT mice. Importantly, EPO, which induced pronounced EPC mobilization in WT mice, failed to do so in Nox2−/− mice. These data suggest that EPO signalling is defective in Nox2y/− mice. Indeed, in EPCs from WT-mice, EPO induced ROS production and proliferation and both responses were missing in EPCs from Nox2y/− mice. Transplantation of bone marrow from Nox2y/− mice into WT mice resulted in an attenuated mobilization in response to hypoxia and EPO, whereas the transplantation of WT bone marrow into Nox2y/− mice restored the normal phenotype . This demonstrates that a failure of bone marrow stem cells to generate ROS in response to EPO underly the mobilization defect. We conclude that Nox2-derived ROS are required for EPO-signalling in stem cells and that hypoxia-induced EPC mobilisation and vascular repair requires the NADPH oxidase Nox2.
Abstract 14136: Aortic Annulus Dimension Assessment by Computed Tomography for Transcatheter Aortic Valve Implantation - Differences Between Diastole and Systole
Authors: Angela G Bertaso, Dennis Wong, Gary Liew, Mike Cunnington, Viji Thomson, James D Richardson, Brett Lorraine, George Kourlis, Di Leech, Matthew I Worthley, Stephen G Worthley
Journal: Circulation (2011)

Background: Accurate assessment of aortic annular dimensions is critical for successful transcatheter aortic valve implantation (TAVI). Annular dimensions are conventionally measured at end-diastol...
Abstract 1823: Inhibition of Histone Deacetylases Accelerates Unloading-Induced Cardiac Atrophy
Authors: Andrew G Blagg, Beverly A Rothermel, Joseph A Hill
Journal: Circulation (2008)

Background: Mechanical unloading of the diseased ventricle has emerged as an important treatment strategy in advanced heart failure. In rare instances, dramatic improvements in ventricular performance are seen, yet little is known regarding underlying mechanisms. Histone deacetylases (HDACs) are implicated in the control of cell growth and remodeling. Recent studies have demonstrated that HDAC inhibition (HDACi) suppresses pathological cardiac hypertrophy and attenuates re-induction of the fetal gene program. We hypothesized that HDACi would accelerate unloading-induced cardiac atrophy. Methods: Mechanical unloading of murine hearts was accomplished by heterotopic transplantation into the abdomen of same-strain recipients (7 days). Two days prior to surgery, mice were randomized to daily sq injections of either trichostatin A (TSA, 1 mg/kg) or vehicle. Left ventricular (LV) cardiomyocyte area was measured from H&amp;E sections (100 myocytes/heart), and LV transcript abundance was measured by quantitative real-time RT-PCR. Native hearts from recipient mice served as controls. Results: Consistent with a role for HDACs in cardiac atrophy, the protein abundance of HDAC-1 (54%; p&lt;0.05), -2 (52%; p&lt;0.01) and -3 (40%; p&lt;0.05) were significantly increased in untreated donor hearts. Unloading-induced cardiomyocyte atrophy was accelerated in hearts transplanted into TSA-treated mice (25±4% atrophy, TSA vs 13±1% atrophy, vehicle; p&lt;0.05). Remarkably, TSA treatment did not blunt (p=NS) the dramatic (72-fold, p&lt;0.05) up-regulation of fetal β MHC seen in vehicle-treated hearts, despite significant increases in the rate of atrophy development. The cyclin dependent kinase inhibitors (CDKIs) p21(Cip1/Waf1) and p27(Kip1), which are known to negatively regulate cardiac hypertrophy, were each increased (p21, 45%, p&lt;0.1; p27, 31%, p&lt;0.1) in TSA-treated hearts relative to control. Conclusions: HDAC inhibition elicits a bona fide cardiomyocyte atrophy response during ventricular unloading in association with up-regulation of CDKI transcript expression. HDAC inhibition, however, does not blunt re-activation of the fetal gene program. These findings may have important implications for heart failure patients undergoing LV assist device support. This research has received full or partial funding support from the American Heart Association, AHA South Central Affiliate (Arkansas, New Mexico, Oklahoma &amp; Texas).
Abstract 23: Overexpression Of Tbx20 In Adult Cardiomyocytes Promotes Cardiomyocyte Proliferation And Improves Cardiac Function Post Myocardial Infarction
Authors: Fu-li Xiang, Katherine Yutzey
Journal: Circulation Research (2014)

Background: Adult mammalian cardiomyocytes (CM) have the potential to proliferate, but this is not sufficient to compensate for the massive loss of functional CMs after myocardial infarction (MI), which remains a leading cause of death in the US. During embryonic heart development, the transcription factor Tbx20 is required for CM proliferation, and Tbx20 overexpression promotes fetal characteristics in adult CMs when initiated before birth in mice. We hypothesize that Tbx20 overexpression (Tbx20OE), when induced in adult CMs after injury, improves cardiac function and repair via dedifferentiation of CMs, thus promoting cell cycle re-entry and repair in mice post-MI. Methods and Results: αMHCMerCreMer (STG) and the inducible cardiomyocyte-specific Tbx20 transgenic (αMHCMerCreMer/CAG-CAT-Tbx20, DTG) mice were subjected to MI or sham surgeries. Tbx20OE was induced 3 days post-surgery via tamoxifen to specifically target cardiac repair post-MI. In sham-operated mice, no difference in cardiac function or morphology was observed between DTG and STG groups. However, more proliferating CMs as labeled by Ki67 were found in DTG sham myocardium compared to STG. Expression of cyclin D1, E1 (cell cycle markers) and IGF1 mRNA was increased, while p21 (cell cycle inhibitor) and Meis1 (negative regulator of proliferation) were decreased, in DTG sham hearts compared to STG controls. In mice subjected to MI, cardiac function, as measured by echocardiography, was significantly improved, and the infarct scar size was smaller (58.1% vs 38.3%) in the DTG group compared to STG controls 2 and 4 weeks post-MI. Myocardial hypertrophy determined by heart to body weight ratio and myocyte diameter was also significantly reduced in DTG heart compared to STG 4 weeks post-MI. Thus, induction of Tbx20OE post-MI injury leads to improved cardiac performance, decreased scar size, and decreased maladaptive cardiac remodeling. Ongoing studies will determine if proliferation indices (Ki67, pHH3, aurora kinase B) and cytokinesis of CM post-MI are increased in myocardium and isolated adult cardiomyocytes with Tbx20OE. Conclusions: Tbx20OE in adult CM activates cell proliferation markers and also improves cardiac function and repair in mice when induced post-MI.
Pharmaco-economic evaluation of 2 induction regimens prior to surgery for NSCLC
Authors: Anne-Pascale Meert, David Lewin, Thierry Berghmans, Marianne Paesmans, Jean-Jacques Lafitte, Arnaud Scherpereel, Delphine Gourcerol, Thierry Bosschaerts, Nathalie Leclercq, Jean-Paul Sculier
Journal: Journal of Thoracic Oncology (2008)
Abstract 4108: Myocardial Injury Can Be Identified by Magnetic Resonance Imaging in the Course of Ablation Procedures for Atrial Fibrillation
Authors: Anil-Martin Sinha, Guido Ritscher, Christian Mahnkopf, Robert S Oakes, Troy J Badger, Martin Schmidt, Harald Marschang, Edward V Dibella, Nassir F Marrouche, Johannes Brachmann
Journal: Circulation (2008)

Background: Pulmonary vein antrum isolation (PVAI) is regarded as an effective therapy in patients with atrial fibrillation (AF). Extension and location of ablation lesions often remain unclear during the procedure. We report a new approach on visualization of myocardial injury using magnet resonance imaging (MRI) during PVAI procedure. Methods: Patients who underwent PVAI, received MRI before and at the terminal phase of PVAI. Delayed enhancement (DE-) MRI sequences were applied, and maximum intensity projections (MIP) obtained. Myocardial injury size was then measured on manually segmented 3D images by a computer algorithm using dynamic thresholding. Results: 30 patients received PVAI from January 2008 to May 2008. In a subset of 15 patients, MRI was performed before and during the procedure. Using DE-MRI, the average lesion to healthy myocardium ratio was 7.56±1.27% during PVAI. The figure shows an example of MIP of a DE-MRI scan in 2D (A,B) and 3D segmentation (C,D) in a anterior view pre (A,C), and during PVAI (B,D). Myocardial injury is identifiable as white tissue around PV single ostia (full arrows) and common trunk (dashed arrows). Conclusions: Using MRI is feasible in the course of ablation procedures. In PVAI patients, DE-MRI allowed identification of location and extension of myocardial injury. Therefore, this new MRI approach might improve ablation techniques, and thus long-term success of PVAI.
Abstract 15186: Clinical Features of Congenital Long QT Syndrome in Pre-elementary-age Children
Authors: Hisaki Makimoto, Minoru Horie, Satoshi Ogawa, Yoshifusa Aizawa, Tohru Ohe, Kengo F Kusano, Masakazu Yamagishi, Naomasa Makita, Toshihiro Tanaka, Takeru Makiyama, Masao Yoshinaga, Nobuhisa Hagiwara, Naokata Sumitomo, Yuko Yamada, Hideo Okamura, Takashi Noda, Kazuhiro Satomi, Takeshi Aiba, Naohiko Aihara, Shiro Kamakura, Yoshihiro Miyamoto, Wataru Shimizu
Journal: Circulation (2010)

Methods: Symptomatic genotyped 346 LQTS type 1, 2, and 3 patients (156, 160, and 30 individuals, respectively) were divided into 2 groups; 72 patients (Group A; 42 men, 56 probands) who experienced...
A Clear Cell Variant of Papillary Thyroid Microcarcinoma With Lung, Bone, and Soft Tissue Metastases
Authors: Bulent Yazici, Yesim Ertan, Aylin Oral, Aysegül Akgün
Journal: Clinical Nuclear Medicine (2015)

A 56-year-old woman presented with a mass lesion on the right occipital bone underwent total resection of the tumor. An adenocarcinoma with immunostaining positive for thyroid transcription factor-1 and thyroglobulin was found. An ultrasound/thyroid scan detected a hot nodule of 9 mm in the right lobe. Fine needle biopsy revealed the similar histological findings with the previous bone resection material. Then, a total thyroidectomy was performed. Histopathologic examination revealed clear cell variant of papillary thyroid microcarcinoma. She received 7.4 GBq of I. On posttherapy scan, metastatic focuses were seen in the left lung and soft tissue between the left paravertebral muscles.
The Hematopoietic Cell Transplant-Co-Morbidity Index (HCT-CI) Predicts Outcomes After T Cell Depleted (TCD) Allogeneic HCT For AML and MDS
Authors: Ravin Ratan, Izaskun Ceberio, Patrick Hilden, Sean M. Devlin, Molly A Malloy, Juliet N Barker, Hugo Castro-Malaspina, Ann A. Jakubowski, Guenther Koehne, Esperanza B Papadopoulos, Doris M Ponce, Craig S. Sauter, Marcel R.M. van den Brink, James W. Young, Richard J. O'Reilly, Sergio Giralt, Jenna D Goldberg, Miguel-Angel Perales
Journal: Blood (2013)
Variation in Body Composition Determines Long-Term Blood Pressure Changes in Pre-Hypertension
Authors: Marcello Ricardo Paulista Markus, Jan Stritzke, Ulrike Siewert, Wolfgang Lieb, Andreas Luchner, Angela Döring, Ulrich Keil, Hans-Werner Hense, Heribert Schunkert
Journal: Journal of the American College of Cardiology (2010)
Abstract 3705: Acute Intravenous Infusion of CXL-1020, a Nitroxyl Donor, Improves Left Ventricular Function in Dogs With Advanced Heart Failure
Authors: Mengjun Wang, Itamar Ilsar, Alice Jiang, Michael S Sabbah, Ramesh C Gupta, T D Cowart, Reza Mazhari, Hani N Sabbah
Journal: Circulation (2009)
Efficacy, PK and Safety Results of Clinical Study Of Recombinant Fusion Protein Linking Coagulation Factor IX With Albumin (rIX-FP) In Previously Treated Patients With Hemophilia B
Authors: Aaron Lubetsky, Toshko Lissitchkov, Elena Santagostino, Gantcho Jotov, Tami Barazani-Brutman, Christine Voigt, Tina Moises, Iris C. Jacobs, Uri Martinowitz
Journal: Blood (2013)
ARTERIOLAR DISEASE OF THE HEART
Authors: James R. Lisa, Chester R. Brown
Journal: Annals of Internal Medicine (1941)

Case Reports1 May 1941ARTERIOLAR DISEASE OF THE HEARTJAMES R. LISA, M.D., F.A.C.P., CHESTER R. BROWN, M.D.JAMES R. LISA, M.D., F.A.C.P., CHESTER R. BROWN, M.D.Author, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-14-11-2147 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptArteriolar disease of the heart is relatively rare. In a large autopsy service it is occasionally found with the more severe grades of coronary arteriosclerosis. Plaut and Kramer1 have reported a case of hyaline degeneration with extensive myomalacia and relatively intact coronary arteries. This report deals with a case of severe cardiac failure and complete heart block, with widespread medial hypertrophy of the arterioles of the heart, minimal changes of the larger coronary arteries, a diffuse thickening of the parietal endocardium, intact valves and microscopic necroses of the myocardial fibers.CASE REPORTThe patient was a white man, 41 years...References1. PLAUTKRAMER AML: Arteriolar disease of the heart, Arch. Path., 1936, xxiii, 393. Google Scholar2. WHITE PD: Heart disease, 1935, Macmillan Co., New York, p. 673. Google Scholar3. HUNT JH: The Raynaud phenomena: a critical review, Quart. Jr. Med., 1936, xxix, 399. Google Scholar4. LISA JR: Pathological changes in the heart in sudden cardiac death, ANN. INT. MED., 1938-39, xii, 1968. Google Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: New York, N. Y.*Received for publication May 2, 1939.From the Pathological Laboratories, City Hospital, Welfare Island, and Lincoln Hospital, Dept. of Hospitals, New York, N. Y. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics Cited bySmall Vessel DiseaseIntramyocardial microarteriopathyMYOCARDIAC CHANGES IN EXPERIMENTAL RENAL FAILURE-A LIGHT AND ELECTRON MICROSCOPIC STUDY*Arteriolar Disease of the Heart 1 May 1941Volume 14, Issue 11Page: 2147-2156KeywordsArteriolesAutopsyCardiovascular therapyCoronary arteriesEndocardiumHeart diseasesHeart failureNecrosisStable coronary artery disease Issue Published: 1 May 1941 PDF downloadLoading ...
BBC is accused of bias in charging Indian government with suppressing report on child malnutrition
Authors: Sanjeet Bagcchi
Journal: BMJ (2015)

Health experts in India have criticised the BBC over a recent report that claimed that a nutrition survey of 100 000 children in India was being suppressed to spare embarrassment to India’s prime minister, Narendra Modi.1

The BBC noted that the Rapid Survey of Children , which measured and weighed nearly 100 000 children and interviewed over 200 000 people in India’s 29 states, had been due for publication in October 2014 but that it still remained a “secret.” The BBC quoted Jean Dreze, a development economist, as saying that the delay in publishing the results was …
THE SIGNIFICANCE OF MORTALITY STATISTICS IN MEDICAL RESEARCH: AN ANALYSIS OF 1,000 DEATHS AT THE PHILADELPHIA GENERAL HOSPITAL
Authors: S O WAIFE, P F LUCCHESI, B SIGMOND
Journal: Annals of Internal Medicine (1952)

ExcerptStatistics collected in large hospitals are the basis for a great many studies, some of which are valuable from a medical standpoint, while others draw conclusions which have at best only a surface accuracy.Often these reports are based upon analyses of autopsy data, because the information is more complete, detailed and consistent than may be obtained from case histories. If, for example, one examines autopsy reports of patients who have died from a particular disease and assembles some unique data with respect to these patients, one often asks, "How far can I generalize from the autopsy reports of one...
Focal Atrial Tachycardia Originating From the Non-Coronary Aortic Sinus
Authors: Feifan Ouyang, Jian Ma, Siew Yen Ho, Dietmar Bänsch, Boris Schmidt, Sabine Ernst, Karl-Heinz Kuck, Shaowen Liu, He Huang, Min Chen, Julian Chun, Yunlong Xia, Kazuhiro Satomi, Huimin Chu, Shu Zhang, Matthias Antz
Journal: Journal of the American College of Cardiology (2006)
Abstract 13646: Quantification of Longitudinal, Radial, and Circumferential Left Ventricular Deformation by 3 Dimensional Speckle Tracking Echocardiography
Authors: Akira Yamada, Sushil A Luis, Shayan Tai, Vicki Speranza, Anthony Benjamin, Jonathan Sherman, Amelia Barthelmess, Lisa Hourigan, Luke Haseler, Darryl Burstow, Jonathan Chan
Journal: Circulation (2012)

Background: Three-dimensional speckle tracking echocardiography (3D-STE) is a novel technique to assess left ventricular (LV) regional deformation. The study was designed to validate segmental LV s...
Abstract 2461: The Improved Outcome with AngioJetTM Thrombectomy Catheter During Primary Stenting in Acute Myocardial Infarction Patients with High-Grade Thrombus
Authors: Annapoorna S Kini, Pedro R Moreno, Angelica M Mares, Michael C Kim, Samin K Sharma
Journal: Circulation (2006)

Background - Primary stenting during acute myocardial infarction (AMI) with large intracoro-nary thrombus is often complicated by slow-flow/no-reflow due to distal embolization of intracoronary thr...
PULMONARY CHANGES RELATED TO CARDIOSPASM
Authors: R A LAKE
Journal: Annals of Internal Medicine (1951)

Article1 September 1951PULMONARY CHANGES RELATED TO CARDIOSPASMRICHARD A. LAKE, M.D.RICHARD A. LAKE, M.D.Author, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-35-3-593 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptPast medical literature is only sporadically documented with instances of pulmonary findings associated with cardiospasm. Aspiration of esophageal overflow, however, has long been recognized as a source of certain types of pulmonary disease, usually secondary to such causes as esophageal neoplasm, caustic stricture of the esophagus, pulsion diverticulum, esophageal encroachment from extrinsic tumors, foreign body in the esophagus, and postoperative emesis. The source of chronic or recurrent pulmonary disease of unknown origin, however, is seldom associated with the well-recognized clinical entity of cardiospasm.1In 1938 Sampson2found in the files of Peter Bent Brigham Hospital only one case in which...Bibliography1. Bird-Acosta I: Pulmonary suppuration secondary to cardiospasm, Am. J. Roentgenol. 52: 481-486, 1944. Google Scholar2. Sampson DA: Lung abscess due to esophageal overflow, New England J. Med. 219: 982-985, 1938. CrossrefGoogle Scholar3. Freedman LM: The etiology of lung abscess, New England J. Med. 218: 663-668, 1938. CrossrefGoogle Scholar4. GrayJankelson WIR: Cardiospasm as a cause of pneumonitis, New England J. Med. 231: 522-525, 1944. CrossrefGoogle Scholar5. HawesSoule LEAB: Pulmonary changes in cardiospasm, Am. J. Roentgenol. 53: 124-128, 1945. Google Scholar6. Dirkse PR: Aspiration pneumonitis associated with chronic obstructive lesions of the esophagus, Univ. Hosp. Bull., Ann Arbor 2: 21, 1936. Google Scholar7. Eisenstein A: Beitrage zur Radiologie der Speiserohre, Fortschr. a. d. Geb. d. Röntgenstrahlen 21: 381-405, 1913-14. Google Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: Long Beach, California*Received for publication April 10, 1950.From the Department of Internal Medicine, University Hospital, University of Michigan, Ann Arbor, Michigan. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics Cited byCardiac achalasia in childhoodKomplikationen beim sogenannten KardiospasmusAchalasia in children as a cause of recurrent pulmonary diseaseIncidence of Chronic Pulmonary Disease Among Patients with Esophageal DisordersÜber den Kardiospasmus und seine BehandlungRöntgenologische Fehldiagnosen bei Oesophagusdilatation und Kardiospasmus 1 September 1951Volume 35, Issue 3Page: 593-599KeywordsCardiospasmEsophageal cancerEsophagusHospital medicinePulmonary diseases Issue Published: 1 September 1951 PDF downloadLoading ...