tbuckley/cabot-search huggingface dataset:
Mortality trends in men and women with diabetes, 1971 to 2000. Commentary
Authors: Edward W. Gregg, Qiuping Gu, Yiling J. Cheng, K. M. Venkat Narayan, Catherine C. Cowie, Nanette K. Wenger
Journal: Annals of Internal Medicine (2007)

Background: Whether mortality rates among diabetic adults or excess mortality associated with diabetes in the United States has declined in recent decades is not known. Objective: To examine whether all-cause and cardiovascular disease mortality rates have declined among the U.S. population with and without self-reported diabetes. Design: Comparison of 3 consecutive, nationally representative cohorts. Setting: Population-based health surveys (National Health and Nutrition Examination Surveys I, II, and III) with mortality follow-up assessment. Patients: Survey participants age 35 to 74 years with and without diabetes. Measurements: Diabetes was determined by self-report for each survey (1971-1975, 1976-1980, and 1988-1994), and mortality rates were determined through 1986, 1992, and 2000 for the 3 surveys, respectively. Results: Among diabetic men, the all-cause mortality rate decreased by 18.2 annual deaths per 1000 persons (from 42.6 to 24.4 annual deaths per 1000 persons; P = 0.03) between 1971 to 1986 and 1988 to 2000, accompanying decreases in the nondiabetic population. Trends for cardiovascular disease mortality paralleled those of all-cause mortality, with 26.4 annual deaths per 1000 persons in 1971 to 1986 and 12.8 annual deaths per 1000 persons in 1988 to 2000 (P = 0.06). Among women with diabetes, however, neither all-cause nor cardiovascular disease mortality declined between 1971 to 1986 and 1988 to 2000, and the all-cause mortality rate difference between diabetic and nondiabetic women more than doubled (from a difference of 8.3 to 18.2 annual deaths per 1000 persons). The difference in all-cause mortality rates by sex among people with diabetes in 1971 to 1986 were essentially eliminated in 1988 to 2000. Limitations: Diabetes was assessed by self-report, and statistical power to examine the factors explaining mortality trends was limited. Conclusions: Progress in reducing mortality rates among persons with diabetes has been limited to men. Diabetes continues to greatly increase the risk for death, particularly among women.
短暂性意识丧失——初步评估、诊断及专家治疗：NICE指南概要
Authors: 许实, 王翠娣
Journal: BMJ (2011)

短暂性意识丧失是一种自发的意识丧失，可完全恢复，有1／4至1／2的人群在他们一生当中某些时候曾出现过”。导致短暂性意识丧失的原因很多，包括心血管疾病（从心律失常至血管迷走性晕厥）、癫痫及心因性发作。但这些病因常不能及早、高效、准确地被诊断出，
High risk behaviour is common in prisons in Berlin
Authors: R Muller, K Stark, I Guggenmoos-Holzmann
Journal: BMJ (1995)

EDITOR,—The issue of 4 February provides important further evidence that injecting drug users in prisons are at high risk of acquiring parenterally transmitted diseases. In a recent study among 612 injecting drug users in Berlin 418 reported a history of imprisonment.1 Of these, 202 continued to inject while in prison and 152 of these started sharing needles during their imprisonment. Needle sharing in prison proved to be the most important risk factor for HIV infection as well as a major risk factor for infection with hepatitis B and C viruses (adjusted odds ratio 10.0, 1.7, and 9.8 respectively). …
Abstract 1386: Association of the Extent of Perceived Mental Stress and Physical Inactivity, and the Extent of Coronary Artery Disease Diagnosed by Computed Tomography
Authors: Vahid Nabavi, Naser Ahmadi, Anila Saeed, Fereshteh Hajsadeghi, Ferdinand Flores, Souraya Sourayanezhad, Helen Mendoza, Ramin Ebrahimi, Ahmad Ahmdi, Susan Levenstein, Matthew Budoff
Journal: Circulation (2009)

Background: Psychological stress and lack of physical activity is known to have a negative effect on the health and well-being of coronary artery disease (CAD) patients. Current study evaluates the association between psychological stress, physical activity and subclinical atherosclerosis measured by coronary artery calcification (CAC). Methods: Two hundred and forty subjects (age 60±9 years, 27% female) underwent CAC. Perceived mental stress (PMS) was studied using perceived stress questionnaire (PSQ, Levenstein, 1995). Physical activity status (MET) was measured using Danish physical activity questionnaire (DPAQ, Aadahl, 2003). Regression analysis was utilized for comparison of CAC, PMS and MET. The severity of CAC was CAC=0, 1≤CAC≤99, 100≤CAC≤399 and CAC≥400. PMS Score was calculated based on general perceived mental stress during the last two years. Total amount of MET was calculated based on an average weekday activity. Results: PMS score increased substantially form CAC=0 to 1≤CAC≤99 to 100≤CAC≤399 ...
Abstract 4683: Risk Stratification in Hypertrophic Cardiomyopathy: Evidence that Syncope is a Marker for Sudden Death
Authors: Paolo Spirito, Camillo Autore, Claudio Rapezzi, Paola Bernabo, Robert Badagliacca, Martin S Maron, Sergio Bongioanni, Fabio Coccolo, N. A Estes, Caterina S Barilla, Elena Biagini, Giovanni Quarta, Maria R Conte, Paolo Bruzzi, Barry J Maron
Journal: Circulation (2008)

Risk stratification and identification of those patients with hypertrophic cardiomyopathy (HCM) at the highest risk for sudden death is a major issue for patient management, given the effective option of the implantable defibrillator in this disease. For example, the prognostic significance of syncope has not been investigated systematically in large HCM cohorts, and therefore treatment strategies related to this symptom have been based largely on intuition and experience. We assessed the relationship between syncope and sudden death in 1511 consecutive HCM patients. Unexplained (n=153) or neurally-mediated (n=52) syncope occurred in 205 (14%) of patients. Over a 5.6±5.2 year follow-up, 74 patients died suddenly. Relative risk of sudden death was 1.78 (95% CI 0.88 –3.51; p=0.08) in patients with unexplained syncope, and 0.91 (95% CI 0.00 –3.83; p=1.0) in those with neurally-mediated syncope, compared to patients without syncope. However, the time interval between unexplained syncope and initial patient evaluation proved to have a strong impact on sudden death risk. Patients with recent syncope (≤6 months from initial evaluation) showed a 5-fold increase in risk compared to patients without syncope (adjusted HR=4.89; CI 2.19 –10.94; p=0.006), a relationship maintained throughout all age groups (&lt;18, 18 –39 and ≥40 years). Conversely, adult patients (18 –39 and ≥40 years) with remote syncope (&gt;6 months from evaluation) showed no increase in sudden death rate (4.8; 95% CI 0.1–26.7 and 3.2; 95% CI 0.1–17.8 per 1000 person-years, respectively), compared to patients of similar age without syncope (11.8; 95% CI 7.6 –17.6 and 5.7%; 95% CI 3.8 – 8.3 per 1000 person-years). Left ventricular outflow obstruction was not associated with sudden death risk (p=0.29). In this large HCM cohort, unexplained syncope proved to be a risk factor for sudden death. In addition, the temporal proximity of syncopal events to patient evaluation is of clinical relevance. Recent unexplained syncope is associated with an increased risk for sudden death in all age groups, while remote syncopal events do not convey increased risk to adult patients.
Prognostic Impact of p190 and p210 Co-Expression at Diagnosis in Chronic Myeloid Leukemia (CML) Patients Treated with Imatinib
Authors: Gioacchino Catania, Federico Monaco, Massimo Pini, Maria Teresa Corsetti, Michela Salvio, Nicol Trincheri, Francesco Zallio, Daniela Pietrasanta, Anna Baraldi, Depaoli Lorella, Salvi Flavia, Marco Ladetto
Journal: Blood (2014)
Multicolor Spectral Karyotyping Identifies New Recurring Breakpoints and Translocations in Multiple Myeloma
Authors: Pulivarthi H. Rao, Juan C. Cigudosa, Yi Ning, Marı́a J. Calasanz, Shinsuke Iida, Shinichi Tagawa, Joseph Michaeli, Bernard Klein, Riccardo Dalla-Favera, Suresh C. Jhanwar, Thomas Ried, R.S.K. Chaganti
Journal: Blood (1998)
Abstract 14176: Frequency and Clinical Correlates of ST-segment Elevation on the Electrocardiogram at Presentation in Apical Ballooning Syndrome (Takotsubo/Stress Cardiomyopathy)
Authors: Kamran A Ahmed, Malini Madhavan, Abhiram Prasad
Journal: Circulation (2011)

Background: Apical ballooning syndrome (ABS) is characterized by transient left ventricular systolic dysfunction that often mimics an acute myocardial infarction. The aim of this study was to asses...
Abstract 15553: A Dietary Intervention Rich in Antioxidants Reduces Inflammation and Oxidative Stress
Authors: Martha J Biddle, Terry A Lennie, Gia Mudd-Martin, Alison Bailey, Michael John Novak, Baretta Casey, Misook Chung, Debra Moser
Journal: Circulation (2013)

Background/Objectives: Inflammation and oxidative stress contribute to the pathophysiology of CVD. Antioxidants found in food products may slow the progression of CVD by inhibiting damaging inflamm...
Abstract 3468: Beneficial Effects of Trimetazidine on Right Ventricular Function and Myocardial Metabolism in Right Ventricular Hypertrophy Induced by Pulmonary Artery Banding
Authors: Yong-Hu Fang, Lin Piao, Peter Toth, Dalia Urboniene, Jalees Rehman, Stephen L Archer
Journal: Circulation (2009)
Abstract 2688: Persistent Risk of Appropriate ICD Therapy for Up to 7 Years in Patients with Ischemic Cardiomyopathy and Low Ejection Fraction
Authors: Alawi A Alsheikh-Ali, Airley E Fish, N. A. Mark Estes, Mark S Link
Journal: Circulation (2006)

BACKGROUND: In clinical trials of implantable cardioverter-defibrillators (ICD) for primary prevention of sudden death in patients with ischemic cardiomyopathy (ICMP) and left ventricular ejection ...
乳腺癌术后等待放疗的时间 尽量减少放疗延迟可以改善预后，因此需要投资和计划
Authors: Ruth, Jack, 王燕
Journal: BMJ (2010)

一些国家对乳腺癌术后等待放疗的时间长短对复发的影响进行了研究，其中部分研究结果显示二者有相关性，其余研究则认为二者没有联系。而近期一个系统性回顾发现，如果保乳手术与术后放疗的时间间隔超过8—12周，且期间没有其他治疗，局部复发率将增加。但由于该文章所纳入的研究中等待放疗的时限长短不一，因此难以得出确切的结论。
Abstract 9899: Wnt11-Pretreated Bone Marrow Mesenchymal Stem Cells Induce Superior Cardiac Repair After a Reperfused Myocardial Infarction
Authors: Guangming Cheng, Yu-Ting Xuan, Carrie M Quinn, Santosh K Sanganalmath, Magdy Girgis, Ou-Li Wang, Yanqing Zhu, Yanjuan Yang, Michael P Flaherty, Shelley Lietzau-Mourer, Ewa K Zuba-Surma, Robert J Vincent, Dong Xu, Arash Davani, Buddhadeb Dawn
Journal: Circulation (2011)

Wnt11, a member of the evolutionarily conserved Wnt family of glycoproteins, plays important roles in cellular migration, proliferation, and differentiation during embryogenesis. However, the impact of Wnt11 signaling in cardiac repair after an acute myocardial infarction (MI) remains largely unknown. Based on the ability of Wnt11 to induce cardiomyogenic differentiation in adult cells, we hypothesized that pretreatment of bone marrow mesenchymal stem cells (MSCs) with Wnt11 would improve the outcomes of infarct repair. In vitro, Wnt11 pretreatment upregulated the expression of antiapoptotic and angiogenic molecules in MSCs. Wnt11 also increased the expression of Nkx2.5 (Figure), a marker of cardiac commitment. To test the efficacy of Wnt11 pretreatment on MSC-induced cardiac repair in vivo, MSCs were cultured in presence of Wnt11-secreting 293 cells (on an insert) for 5 days and transplanted intramyocardially after a reperfused MI. Age-matched male wild-type (C57BL/6J) mice underwent a 30-min coronary occlusion followed by reperfusion, and 48 h later received intramyocardial injection of vehicle, untreated MSCs (1 million in 30 microL), or Wnt11-pretreated MSCs (1 million in 30 microL). Echocardiography was performed 4 d prior to coronary occlusion/reperfusion (BSL1) and at 48 h (BSL2), 21 d, and 35 d after cell injection. After 35 d, compared with mice that received vehicle or untreated MSCs, mice injected with Wnt11-pretreated MSCs exhibited greater LV ejection fraction (Figure) and smaller LV end-diastolic volume, indicating that Wnt11 pretreatment transforms MSCs into superior vehicles of infarct repair. We conclude that intramyocardial transplantation of Wnt11-pretreated MSCs after a reperfused MI results in improved myocardial repair. These results underscore that pretreatment with Wnt11 may potentially enhance the outcomes of therapeutic infarct repair in humans.
Abstract 3180: The Relationship Between Spontaneous and Iatrogenic Hypoglycemia and Mortality in Patients Hospitalized with Acute Myocardial Infarction
Authors: Mikhail Kosiborod, Silvio E Inzucchi, Abhinav Goyal, Harlan M Krumholz, Lan Xiao, Frederick A Masouid, John A Spertus
Journal: Circulation (2008)
Abstract 16745: Usefulness of Analysis of Transthoracic Impedance Signals for Accurate Diagnosis of Sleep Disordered Breathing in Patients with Permanent Pacemaker Implantation
Authors: Shoichi Miyamoto, Masatoshi Fujita, Takao Ishimura, Moriaki Inoko, Tetsuya Haruna, Toshiaki Izumi, Takao Katoh, Eisaku Nakane, Yosiaki Saji, Koji U eyama, Ryuji Nohara
Journal: Circulation (2011)

Purpose: There are several reports regarding an excessively high prevalence of undiagnosed sleep disordered breathing (SDB) in patients with permanent pacemaker implantation. Some programs for pacemakers utilize transthoracic impedance changes between the generator and the electrode tip to derive minute ventilation as a sensor for rate adaptation. We investigated whether this system can detect sleep-related breathing disorders. Methods: We investigated 39 patients (19 male, 20 female, 78±13 (SD) years) who underwent permanent pacemaker implantation for sick sinus syndrome and advanced or complete atrio-ventricular nodal block. Body mass index of these patients was 23±4 kg/m 2 . Chronic heart failure was observed in 10 (26%) of 39 patients. We measured the number of the transthoracic impedance signals remaining unchanged for more than 10 seconds with use of the pacemaker minute ventilation sensor (SORIN), and compared with the results of polysomnography (Morpheus C, Teijin). By using polysomnography, we calculated the number of apnea/hypopnea episode per hour (apnea/hypopnea index: AHI). We defined the number of respiratory cycles more than 10 seconds of unchanged transthoracic impedance signals as the respiratory disturbance index (RDI). Receiver operating characteristic (ROC) analysis was performed on the RDI for the diagnosis of moderate to severe SDB (AHI &gt;20). Results: Moderate to severe SDB was diagnosed in 18 (46%) of 39 patients. The area under the ROC curve was 0.87 (95% CI 0.75 to 0.99). Given a cutoff value of 210/hour for the RDI, the RDI can significantly (p&lt;0.05) discriminate between patients with and without moderate to severe SDB, and showed 72.2% sensitivity and 90.5% specificity for the diagnosis. The RDI was well correlated with the AHI in patients with moderate to severe SDB (n=15, r=0.771, p=0.0004). Conclusions: Detection of apnea/hypopnea episode by pacemaker minute ventilation sensor is feasible and accurate for diagnosing SDB in patients with permanent pacemaker implantation.
Abstract 17199: Cost-Effectiveness of Fondaparinux versus Enoxaparin in Non-st-Elevation Acute Coronary Syndrome in Canada. Oasis-5
Authors: Greta Lozano-Ortega, Robyn Kendall, Mark Sculpher, Jorge Ross Terres, Reid Robson, Adrian Levy
Journal: Circulation (2010)

Purpose: To estimate the lifetime cost-effectiveness of fondaparinux compared to enoxaparin for non-ST-elevation acute coronary syndrome (NSTE-ACS) patients in a Canadian hospital setting. Methods: An event-based decision analytic model was constructed using clinical and resource use data from OASIS-5, a randomised trial of 20,078 patients from 41 countries. A public payer perspective in the hospital setting was adopted. The probabilities of death, non-fatal myocardial infarction (MI), non-fatal stroke, and major and minor bleeds over a period of 180 days, for both treatment strategies, were estimated using a set of risk equations derived from OASIS-5 data. Resource use data from the trial were valued using Canadian costs. A cost regression model was developed to estimate the mean cost of managing the clinical events over the 180 day period. Annual costs of long-term care for ACS patients were added after 180 days until death. Long-term survival was incorporated using Canadian life tables with further adj...
Abstract 17006: Genistein Rescues Pulmonary Hypertension Induced Right Ventricular Failure by Inhibiting the Proliferation of Pulmonary Artery Smooth Muscle Cells and Reversing Cardiac Hypertrophy
Authors: Rangarajan D Nadadur, Humann Matori, Soban Umar, Andrea Iorga, Rod Partow-Navid, Mansoureh Eghbali
Journal: Circulation (2011)

INTRODUCTION Pulmonary Hypertension (PH) induced Right Ventricular Failure (RVF) is a major cause of morbidity and mortality. PH is characterized by thickening of pulmonary vasculature and gradually increasing pulmonary arterial pressure, resulting in RV hypertrophy and failure. We have previously shown that genistein (Gen), a soy isoflavone, is able to rescue PH induced RVF. Here we explore whether inhibition of pulmonary smooth muscle cell proliferation as well as reversal of hypertrophy are the mechanisms involved in the rescue. METHODS To induce PH, male rats received a single injection of Monocrotaline (60mg/kg, s.c.) or saline as control (CTRL n=7). At day 21, when rats had developed severe PH, Gen therapy was begun (1 mg/kg/day, s.c.) to one group (n=8) until day 30. Some were left untreated to develop severe RVF (n=7). In vitro, proliferation of human pulmonary artery smooth muscle cells (HPASMCs) was measured with or without Gen by MTT assay. Hypertrophy of cardiomyocytes was measured by incubating neonatal rat ventricular myocytes (NRVMs) with pro-hypertrophic phenylephrine (PE, 10uM) with and without Gen. P&lt;0.05 were considered significant. Values are given mean±SE. RESULTS Gen was able to reverse depressed RV Ejection Fraction (RVEF=65.67±1.08% vs 28.76±0.79% in RVF, p&lt;0.05). The RVF group was characterized by increased pulmonary arteriolar medial thickness (2.55±0.09 vs 1.00±0.05 in CTRL, p&lt;0.05) as well as significant hypertrophy of the RV (RV/(LV+IVS)=0.69±0.08 vs 0.25±0.03 in CTRL, p&lt;0.05). Gen was able to reverse arteriolar thickening and RV hypertrophy (thickening=1.09±0.04; RV/(LV+IVS)=0.35±0.03. all p&lt;0.05 vs RVF). Gen was able to inhibit the proliferation of HPASMCs (0.47±0.07 vs 1.00±0.12 in CTRL, p&lt;0.05). Gen treatment of NRVMs was also able to reverse cardiomyocyte hypertrophy (0.71±0.06 vs 2.45±0.16 in PE alone, normalized to untreated cell size, p&lt;0.05). CONCLUSION Genistein therapy rescues depressed RV Ejection Fraction and reverses pulmonary arteriolar medial thickness as well as RV hypertrophy in PH-induced RVF rats. Genistein inhibits the proliferation of HPASMCs and reverses the hypertrophy of NRVMs in vitro. These provide a possible physiological mechanism for the rescue of PH induced RVF by Genistein therapy.
Abstract 2246: Coronary Arterial Positive Remodeling Detected by MRI: The Multi-Ethnic Study of Atherosclerosis (MESA)
Authors: Cuilian Miao, Shaoguang Chen, Robson Macedo, Shenghan Lai, Kiang Liu, Debiao Li, Bruce A Wasserman, Jens Vogel-Clausen, Joao A Lima, David A Bluemke
Journal: Circulation (2008)

The purpose of this study was to determine the ability of magnetic resonance imaging (MRI) to detect early changes of coronary atherosclerotic remodeling. Positive remodeling is associated with plaque vulnerability and rupture. 179 subjects (90 men, age 61±9 yrs) free of clinical cardiovascular disease underwent cross-sectional coronary wall MRI using a black blood technique. Outer contour area (“vessel size”), lumen area, and mean vessel wall thickness were obtained. Linear regression was used to determine the correlations of mean wall thickness with outer contour area or lumen area before and after adjustment for body size parameters. The outer contour area increased with increasing mean wall thickness (p&lt;0.0001). Lumen area also increased, but at a smaller rate than outer contour area (p&lt;0.0001). When men and women were examined separately (Figure ), the relationships continued to be significant (P&lt;0.0001 and 0.05 respectively) with no differences in these relationships between men and women. When vessel size was normalized by LV size or body mass index, the relationships remained significant, and the relative slopes were similar to that in the non-normalized model. The positive correlations between outer contour area and vessel wall thickness remained significant for each coronary bed (right, left main, left anterior descending) individually.
Abstract 12697: Comparison of 1-Year Myocardial Perfusions Pattern by Randomized Treatment in Patients with Diabetes and Coronary Artery Disease and Relation to Clinical Outcomes: Results from the BARI 2D Trial
Authors: Leslee J Shaw, Manuel Cerqueira, Maria M Brooks, Veronica V Sansing, George A Beller, Rodica Pop-Busui, Raymond Taillefer, Bernard R Chaitman, Raymond J Gibbons, Ami E Iskandrian
Journal: Circulation (2010)

Background: For patients with type 2 diabetes and stable coronary artery disease, the Bypass Angioplasty Revascularization Investigation 2 Diabetes (BARI 2D) trial demonstrated similar long term cl...
Abstract 231: Upregulation of Tenascin-C Expression in Rheumatic Heart Valve Disease by Synergistic TNF-a and Shear Stress via RhoA/ROCK Signaling Pathway
Authors: Xufeng Wei, Wei Yi, Yu Chen, Yang Sun, Wayne Bond Lau, Zhenxiao Jin, Tao Chen, Xiaochao Dong, Xin-Liang Ma, Hongbing Wang, Dinghua Yi
Journal: Circulation (2009)
941. Development of Cell Penetration Domain and Cell Killing Domain Genes for Treatment of Cancer
Authors: Sean M. Sullivan, Harm J. Knot, Ron Mandel, J. P. Jarajapu, Jacqueline Baltunis
Journal: Molecular Therapy (2004)

Fluorescein labeled peptides derived from HIV-Tat (Tat) and Drosophila antennaepedia homeodomain (ANT) were able to intracellularly label two human glioblastoma cell lines (A172 and T98G) and one rat glioma cell line (RG2). The Tat peptide localized to the cell nuclei whereas the ANT sequence was distributed evenly throughout the cytoplasm. A 14 amino acid sequence derived from P14, a cell cycle suppression protein, was added to the C-terminus of each peptide. Increasing the size of the peptide did not inhibit cell entry and did not change fluorescent staining patterns of cells. It did kill each cell line within 1 hr after adding 10 uM of peptide. Tat, ANT and Exon1 peptides by themselves had no effect on cell viability. A dose response curve yielded an IC50 concentration of 9 uM for the Tat-Exon1. Reversing the domain order reduced the IC50 to 4 uM. Uptake studies showed that addition of Exon1 sequence to the Tat sequence reduced the rate of uptake by half. Reversing the domain order, i.e., Exon-1-Tat, restored the rate of uptake to that observed by Tat alone. To gain a better understanding of the behavior of these peptides in a three dimensional cell system, cell penetration and killing were tested in rat cerebral artery organ cultures. These vessels are maintained under physiological pressure and blood flow. Intraluminal administration of the fluorescent peptides produced cell-staining patterns similar to that observed in the in vitro brain tumor cell lines, with cell staining restricted to the endothelium. Adventitial administration of the Tat and ANT peptides yielded smooth muscle cell labeling of the nucleus and cytoplasm, respectively. No labeling of the endothelium was observed. Addition of the cell-killing domain did not inhibit the ability of the peptides to penetrate into the endothelium; however, response to vasoconstriction was diminished where as smooth muscle cell response was not affected. The Tat-ExonI sequence has been cloned into an expression cassette with an upstream signal sequence and a down stream HA epitope tag. Western blots showed that all three tumor cell lines expressed the peptide. Transfection results further showed a direct correlate between transfection efficiency and cell death. Experiments are directed toward proving the bystander effect mediated by the cell penetration domain in an in vivo rat brain tumor model.
Prognostic Factor, Including Relative Dose Intensity, For Adult T-Cell Leukemia/Lymphoma In Clinical Practice
Authors: Ayako Kamiunten, Akira Kitanaka, Yoko Kubuki, Tomonori Hidaka, Kotaro Shide, Takuro Kameda, Masaaki Sekine, Kiyoshi Yamashita, Hitoshi Matsuoka, Hiroshi Kawano, Takanori Toyama, Seiichi Satou, Kouichi Maeda, Junzo Ishizaki, Kazuya Shimoda
Journal: Blood (2013)