Rheumatology: An Annotated Bibliography of Recent Literature
Journal: Annals of Internal Medicine (1976)

The Literature of Medicine1 August 1976Rheumatology: An Annotated Bibliography of Recent LiteratureReferences to Journal Articles and Other PapersRHEUMATOLOGY COMMITTEE, MEDICAL-KNOWLEDGE SELF-ASSESSMENT PROGRAM IV, AMERICAN COLLEGE OF PHYSICIANSRHEUMATOLOGY COMMITTEESearch for more papers by this author, MEDICAL-KNOWLEDGE SELF-ASSESSMENT PROGRAM IVSearch for more papers by this author, AMERICAN COLLEGE OF PHYSICIANSSearch for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-85-2-264 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptThis bibliography includes references to 98 recently published journal papers and similar sources that represent recent developments important in the concepts and practice of internal medicine. It has been compiled as a part of the work of the Rheumatology Committee toward the preparation of the Medical Knowledge Self-Assessment Program IV Syllabus.In preparing this bibliography, the committee reviewed MEDLINE†bibliographic printouts of journal articles published since the beginning of 1972 and also drew on their own files. Papers were selected with a view to their likely availability in libraries of hospitals and other clinical institutions in the United...Connective Tissue Immunologic Mechanisms Rheumatoid Arthritis Juvenile Rheumatoid Arthritis Systemic Lupus Erythematosus Scleroderma (Systemic Sclerosis) Dermatomyositis-Polymyositis Sjögren's Syndrome Amyloid Giant Cell Arteritis and Polymyalgia Vasculitides Ankylosing Spondylitis, Reiter's Syndrome, Psoriatic Arthritis, and other HLA-B27-associated Disorders Rheumatic Fever Degenerative Joint Disease Infectious Arthritis Gout and Pseudogout Bone and Metabolic Disease Yersinia Arthritis1. LEROY E: Collagens and human disease. Bull Rheum Dis 25:778-785, 1974 A brief review of collagen in the context of understanding human disease. MedlineGoogle Scholar2. UITTOLICHTENSTEIN JJ: Defects in the biochemistry of collagen in diseases of connective tissue. J Invest Dermatol 66:59-79, 1976 Up to date information on collagen biosynthesis and interpretation of heritable disorders of connective tissue in terms of synthetic mechanisms. CrossrefMedlineGoogle Scholar3. BROUETCLAUVELDANON JJF: Biologic and clinical significance of cryoglobulins. Am J Med 57:775-788, 1974 An impressive series of about 80 well-classified cases of cryoglobulinemia with clinically useful associations. Increasingly, cryoglobulins are being found in patients with signs and symptoms of vasculitis, hyperviscosity, or urticaria. CrossrefMedlineGoogle Scholar4. FEARONAUSTEN DK: Inhibition of complement-derived enzymes. Ann NY Acad Sci 256:441-450, 1975 A good review of diseases associated with aberrant proteins that control complement. It is well-written, understandable and shows how important homeostasis is in this group of enzymes. CrossrefMedlineGoogle Scholar5. RUDDYFEARONAUSTEN SDK: Depressed synovial fluid levels of properdin and properdin factor B in patients with rheumatoid arthritis. Arthritis Rheum 18:289-295, 1975 This group has determined all of the complement proteins and activities in synovial fluid for which techniques are available. Their previous papers are referenced here. CrossrefMedlineGoogle Scholar6. GOLDSTEIN I: Clinical applications of complement measurements in rheumatic diseases. Am J Med Sci 269:172-182, 1975 This is the single best paper on the clinical application of complement knowledge. It is useful, up to date, and gives a good idea of both the practical value of complement measurements and the excitement in this rapidly expanding area of research. CrossrefMedlineGoogle Scholar7. OSTERLANDESPINOZAPARKER CLL: Inherited C2 deficiency and systemic lupus erythematosus: studies on a family. Ann Intern Med 82:323-328, 1975 A patient with absent C2 who had clinical systemic lupus erythematosus is described, with a review of the literature on complement deficiency studies and an excellent discussion of the pathogenetic implications of the clinical and serologic findings. LinkGoogle Scholar8. KOHLER P: Inherited complement deficiencies and systemic lupus erythematosus: an immunogenetic puzzle (editorial). Ann Intern Med 82:420-421, 1975 A brief review and discussion of the implications of the association between absent complement components, lupus erythematosus, and transplantation antigens. LinkGoogle Scholar9. VAUGHAN J: Rheumatologic disorders due to immune complexes. Postgrad Med 54:129-136, 1973 This is an outstanding statement of current clinical thinking about vasculitis and deals more generally and specifically with all vasculitic problems and their causes, in so far as they are known, in the collagen diseases and related syndromes. CrossrefMedlineGoogle Scholar10. THEOFILOPOULOSWILSONDIXON ACF: The Raji cell radioimmune assay for detecting immune complexes in human sera. J Clin Invest 57:169-182, 1976 A new, highly sensitive, and quantitative method for detecting soluble immune complexes has been used to study patients with autoimmune, vasculitic, and neoplastic diseases. This method and others recently developed will aid in elucidating pathogenetic mechanisms in these disorders. CrossrefMedlineGoogle Scholar11. KOHLER P: Clinical immune complex disease. Medicine (Baltimore) 52:419-429, 1973 An excellent discussion of immune complex diseases, emphasizing systemic lupus erythematosus and hepatitis-B virus infection. CrossrefMedlineGoogle Scholar12. TALAL N: Autoimmunity and lymphoid malignancy in New Zealand black mice. Prog Clin Immunol 2:101-120, 1974 The spontaneous appearance of autoimmunity, lymphoid malignancy, and monoclonal macroglobulinemia in NZB mice is discussed in relation to systemic lupus erythematosus, Sjögren's syndrome and Waldenstrom's macroglobulinemia. Emphasis is on T lymphocyte regulation of immune reactivity. MedlineGoogle Scholar13. ZAWADZKIEDWARDS ZG: Nonmyelomatous monoclonal immunoglobulinemia. Prog Clin Immunol 1:105-156, 1972 A thorough review of the varied clinical situations in which monoclonal immunoglobulins arise in the absence of overt myeloma. Many of these patients have features of autoimmunity. MedlineGoogle Scholar14. SCHUR P: Complement in lupus. Clin Rheum Dis 1:519-543, 1975 This review is clinically oriented; although the value of following complement levels as a guide to therapy is discussed, the exceptions are considered. There is also a succinct guide to the biology of the complement system and the known genetic defects. CrossrefGoogle Scholar15. WANDSLAMONTMANN JJB: Arthritis associated with intestinal-bypass procedure for morbid obesity—complement activation and characterization of circulating cryoproteins. N Engl J Med 294:121-124, 1976 Arthritis may develop after intestinal resection for obesity. The data suggest that circulating immune complexes containing bacterial antigen, antibody, and complement are involved in the pathogenesis. CrossrefMedlineGoogle Scholar16. JOHNSONVAUGHNHENCH JJP: Rheumatoid arthritis 1970-1972. Ann Intern Med 78:937-953, 1973 An up-to-date review of the specific joint manifestations and the systemic complications of rheumatoid arthritis. LinkGoogle Scholar17. PEARSONAMSTUTZBLUESTONE CHR: Diagnosis and treatment of erosive rheumatoid arthritis and other forms of joint destruction. Ann Intern Med 82:241-256, 1975 A panel discussion of the approach to crippling disease and pain in the patient with arthritis. The surgical therapeutic procedures available are outlined, with their complications and indications. LinkGoogle Scholar18. ZVAIFLER N: Rheumatoid synovitis. An extravascular immune complex disease. Arthritis Rheum 17:297-305, 1974 A brief synopsis of current information on the role of immune complexes and complement activation in the pathogenesis of rheumatoid arthritis. CrossrefMedlineGoogle Scholar19. HARRISKRANE ES: Collagenases. N Engl J Med 291:557-563, 605-609, 652-661, 1974 A review of the mechanisms whereby collagen is destroyed. The enzymatic mechanisms are interpreted in terms of disease states and physiologic processes. CrossrefMedlineGoogle Scholar20. THEOFILOPOULOSBURTONBOYLOSPALLUTO AGJ: IgM rheumatoid factor and low molecular weight IgM. An association with vasculitis. Arthritis Rheum 17:272-284, 1974 The pattern of serologic abnormalities is different in patients with rheumatoid vasculitis than in rheumatoid patients without overt vasculitis. The prevalence of IgG rheumatoid factor and 7S IgM is higher in patients with rheumatoid vasculitis. CrossrefMedlineGoogle Scholar21. SCHALLERWEDGWOOD JR: Juvenile rheumatoid arthritis: a review. Pediatrics 50:940-953, 1972 The various types of juvenile rheumatoid arthritis are well described. Ankylosing spondylitis may initially and for several years be difficult to separate from juvenile rheumatoid arthritis. MedlineGoogle Scholar22. BUJAKAPTEKARDECKER JRJ: Juvenile rheumatoid arthritis presenting in the adult as fever of unknown origin. Medicine (Baltimore) 52:431-444, 1973 Still's disease, a form of juvenile rheumatoid arthritis with an acute systemic onset, may also occur in young adults. This diagnosis is a consideration in patients presenting with fever of unknown origin. CrossrefMedlineGoogle Scholar23. RUDNICKIRUDERMANSCULL RME: Clinical features and serologic abnormalities in juvenile rheumatoid arthritis. Arthritis Rheum 17:1007-1015, 1974 An excellent succinct review of the clinical features and serologic abnormalities of this multifaceted disease. Patients' serums were tested for rheumatoid factor, complement, immunoglobulin levels, antinuclear antibodies, and DNA binding. CrossrefMedlineGoogle Scholar24. ATHREYAMOSERCECIL BGH: Aspirin-induced hepatotoxicity in juvenile rheumatoid arthritis. Arthritis Rheum 18:347-352, 1975. In a prospective study, 22 of 34 children with juvenile rheumatoid arthritis developed abnormal liver function tests within 1 to 2 weeks of initiating therapy. Liver function tests returned to normal on stopping aspirin in six patients and became normal in 12 despite continuing therapy. CrossrefMedlineGoogle Scholar25. NOTMANKURATATAN DNE: Profiles of antinuclear antibodies in systemic rheumatic diseases. Ann Intern Med 83:464-469, 1975 An excellent review that dissects the prevalence of specific antinuclear antibodies and titer differences in rheumatic diseases. High titers of anti-DNA were limited to lupus erythematosus whereas high titers of anti-ribonucleoprotein are present in diseases other than mixed connective tissue disease. LinkGoogle Scholar26. BRUNNERDAVIS CJ: Immune mechanisms in the pathogenesis of systemic lupus erythematosus. Bull Rheum Dis 26:854-861, 1975-76 A concise review of the current understanding of immune mechanisms in systemic lupus erythematosus which includes animal models of lupus erythematosus, drug-induced lupus erythematosus, and humoral, cellular, and genetic factors. MedlineGoogle Scholar27. GHOSE M: Pericardial tamponade. A presenting manifestation of procainamide-induced lupus erythematosus. Am J Med 58:581-585, 1975 The fourth reported case of pericardial tamponade in a patient with drug-induced lupus erythematosus. This life-threatening manifestation of drug-induced lupus erythematosus requires prompt initiation of corticosteroid treatment. CrossrefMedlineGoogle Scholar28. BULKLEYROBERTS BW: The heart in systemic lupus erythematosus and the changes induced in it by corticosteroid therapy. A study of 36 necropsy patients. Am J Med 58:243-264, 1975 An important review of clinical and pathologic abnormalities of the heart in corticosteroid-treated patients emphasizing the presence of coronary atherosclerotic plaques in patients treated for more than 1 year. Increasing numbers of reports are describing myocardial infarction in such patients. CrossrefMedlineGoogle Scholar29. GROSSMANCALLERAMECONDEMI JMJ: Skin immunofluorescence studies on lupus erythematosus and other anti-nuclear antibody-positive diseases. Ann Intern Med 80:496-500, 1974 The diagnostic value of immunofluorescence studies of the deposition of immunoglobulins and C3 in the dermal-epidermal junction of normal-appearing skin from patients with lupus erythematosus is described. Clinical disease activity and dermal-epidermal junction staining are related. LinkGoogle Scholar30. RICKHOYER ML: Haemostatic disorders in systemic lupus erythematosus. Clin Rheum Dis 1:583-596, 1975 An excellent review of the mechanisms of the hemostatic disorders emphasizing problems related to deficiency or inhibition of blood coagulation proteins, thrombocytopenia, and defective platelet function. CrossrefGoogle Scholar31. KLIPPELZVAIFLER JN: Neuropsychiatric abnormalities in systemic lupus erythematosus. Clin Rheum Dis 1:621-638, 1975 A concise review that describes the clinical manifestations of central nervous system lupus erythematosus; the functional disorders seen in these patients as a reaction to their debilitating chronic disease; and the pathophysiology, diagnosis, and treatment. CrossrefGoogle Scholar32. BALDWINGALLO DG: Lupus nephritis. Clin Rheum Dis 1:639-663, 1975 The clinical and pathologic manifestations of focal proliferative, diffuse proliferative, and membranous lupus nephritis are reviewed, with information on mesangial lupus nephritis and the end-stage kidney with glomerular sclerosis. CrossrefGoogle Scholar33. DECKER J: Cytotoxic agents in the management of systemic lupus erythematosus. Clin Rheum Dis 1:665-677, 1975 A thoughtful review of the literature through 1975 on management with cytotoxic agents. The detailed analysis of the five published controlled trials of cytotoxic drugs in lupus nephritis should be considered before using these agents. CrossrefGoogle Scholar34. HADLERGERWINFRANK NRM: The fourth component of complement in the cerebrospinal fluid in systemic lupus erythematosus. Arthritis Rheum 16:507-521, 1973 A single determination of C4 in cerebrospinal fluid does not discriminate between the patients with and without CNS involvement. Serial determinations in four patients with acute neurologic illness showed depressed levels when compared with those at other times. CrossrefMedlineGoogle Scholar35. SOLOWAYSUMMERSKILLBAGGENSTOSS RWA: "Lupoid" hepatitis, a nonentity in the spectrum of chronic active liver disease. Gastroenterology 63:458-465, 1972 A positive lupus erythematosus cell test indicated more active and severe liver disease but had no significance in relation to sex, age, or associated disease of "autoimmune" type. "Lupoid" hepatitis does not appear to be an entity with a specific cause or prognosis. CrossrefMedlineGoogle Scholar36. BLOMGRENCONDEMIVAUGHN SJJ: Procainamide-induced lupus erythematosus—clinical and laboratory observations. Am J Med 52:338-348, 1972 Forty-four patients with symptomatic procainamide-induced lupus erythematosus in whom laboratory and clinical features differentiated them from those with the naturally occurring disease. CrossrefMedlineGoogle Scholar37. BRENTJENSSEPULVEDABALIAH JMT: Interstitial immune complex nephritis in patients with systemic lupus erythematosus. Kidney Int 7:342-350, 1975 Interstitial nephritis is described in renal tissue from 30 patients out of 45. Immunoglobulins, or complement, or both in peritubular capillaries, interstitium, and tubular basement membranes distinguished renal tissue from patients with lupus erythematosus from tissue from patients with idopathic membranous nephropathy and minimal glomerular disease. CrossrefMedlineGoogle Scholar38. SHARP G: Mixed connective tissue disease—overlap syndromes. Clin Rheum Dis 1:561-572, 1975 The most recent review of the syndrome originally described by the author in 1972 in which recent studies on the antibody to ribonucleoprotein and its relation to the clinical syndrome are well described. CrossrefGoogle Scholar39. PHILLIPS P: The role of viruses in systemic lupus erythematosus. Clin Rheum Dis 1:505-518, 1975 A thoughtful review of the current information available on the role of viruses in systemic lupus erythematosus. CrossrefGoogle Scholar40. DEHORATIUSPILLARISETTYMESSNER RRR: Anti-nucleic acid antibodies in systemic lupus erythematosus patients and their families—incidence and correlation with lymphocytotoxic antibodies. J Clin Invest 56:1149-1154, 1975 These investigators, who previously showed that antilymphocyte antibodies occur in approximately 80% of patients and in 50% of their relatives, now show that these antibodies correlate with antinucleic acid antibodies. Genetic relation and contact with proband relate to the prevalence in family members; these antibodies may reflect a response to a virus. CrossrefMedlineGoogle Scholar41. BULKLEYRIDOLFISALYER BRW: Myocardial lesions of progressive systemic sclerosis. A cause of cardiac dysfunction. Circulation 53:483-490, 1976 A careful pathologic study of the clinical significance and pathogenetic mechanisms of myocardial necrosis and fibrosis in scleroderma. CrossrefMedlineGoogle Scholar42. LEROY E: Increased collagen synthesis by scleroderma skin fibroblasts in vitro. A possible defect in the regulation or activation of the scleroderma fibroblast. J Clin Invest 54:880-889, 1974 Further studies of the scleroderma fibroblast in culture, showing an enhanced proportion of overall protein synthesis committed to collagen production. CrossrefMedlineGoogle Scholar43. MARICQLEROY HE: Patterns of finger capillary abnormalities in connective tissue diseases by "wide-field" microscopy. Arthritis Rheum 16:619-628, 1973 Defines three patterns of nailfold capillary changes in subjects with [1] rheumatoid arthritis, [2] systemic lupus erythematosus, and [3] Raynaud's syndrome, scleroderma, and dermatomyositis-polymyositis. This work emphasizes the similarities in the disorders grouped in [3] above, at least at the microcirculatory level. CrossrefMedlineGoogle Scholar44. CANNONHASSARCASE PMD: The relationship of hypertension and renal failure in scleroderma (progressive systemic sclerosis) to structural and functional abnormalities of the renal cortical circulation. Medicine (Baltimore) 53:1-46, 1974 An extensive evaluation of the kidney in scleroderma from a clinical, radiographic, histopathologic, and renovascular point of view. CrossrefMedlineGoogle Scholar45. CAMPBELLLeROY PE: Pathogenesis of systemic sclerosis: a vascular hypothesis. Semin Arthritis Rheum 4:351-368, 1975 An attempt to synthesize the various concepts and features of scleroderma and related disorders. CrossrefMedlineGoogle Scholar46. WALTON J: Polymyositis: new light on pathogenesis and treatment. Proc Aust Assoc Neurol 9:1-7, 1973 This is an authoritative review of research in viral and autoimmune pathogenesis. The suggestion is strong that cellular mediated hypersensitivity against muscle is the major abnormality. MedlineGoogle Scholar47. ARNETTWHELTONZIZIC FJT: Methotrexate therapy in polymyositis. Ann Rheum Dis 32:536-546, 1973 A reminder of the pulmonary complications of methotrexate with suggestions for differential diagnosis. CrossrefMedlineGoogle Scholar48. METZGERBOHANGOLDBERG AAL: Polymyositis and dermatomyositis: combined methotrexate and corticosteroid therapy. Ann Intern Med 81:182-189, 1974 A considered review with new data discussing the concept of steroid resistance and the use of methotrexate. LinkGoogle Scholar49. BOHANPETER AJ: Polymyositis and dermatomyositis. N Engl J Med 292:344-347, 403-407, 1975 This is an interesting and important critique of criteria for diagnosis, differential diagnosis, response to treatment, and the varieties of polymyositis. Critical references are included. CrossrefMedlineGoogle Scholar50. MASONGUMPELGOLDING AJP: Sjögren's syndrome—a clinical review. Semin Arthritis Rheum 2:301-331, 1973 A careful review of the clinical spectrum of the sicca syndrome and its associations. CrossrefMedlineGoogle Scholar51. ANDERSONTALAL LN: The spectrum of benign to malignant lymphoproliferation in Sjögren's syndrome. Clin Exp Immunol 10:199-221, 1971 Illustrative clinical presentations support the concept that Sjögren's syndrome can develop into a systemic lymphoproliferative disorder with features of lymphoma or pseudolymphoma. The relation to immue deficiency and autoimmunity is discussed. Google Scholar52. TALALSMITHDIXON NLJ: Recent clinical and experimental developments in Sjögren's syndrome. Western J Med 122:50-58, 1975 The oral, ocular, and systemic complications of Sjögren's syndrome are discussed in relation to a patient with renal and pulmonary lymphoid infiltrates. MedlineGoogle Scholar53. MICHALSKIDANIELSTALAL JTN: Beta2 microglobulin and lymphocytic infiltration in Sjögren's syndrome. N Engl J Med 293:1228-1231, 1975 The clinical and immunologic significance of elevated serum beta2-microglobulin concentration is discussed in relation to renal and lymphoproliferative complications of Sjögren's syndrome. CrossrefMedlineGoogle Scholar54. ALSPAUGHTAN ME: Antibodies to cellular antigens in Sjögren's syndrome. J Clin Invest 55:1067-1073, 1975 Different precipitating antibodies reacting with soluble cellular antigens correlate with the presence or absence of rheumatoid arthritis in Sjögren's syndrome. CrossrefMedlineGoogle Scholar55. FRANKLIN E: Amyloidosis. Bull Rheum Dis 26:832-836, 1976 A comprehensive and compact review of clinical and conceptual aspects of systemic amyloidosis, including recent new immunologic information. Google Scholar56. WIERNICK P: Amyloid joint disease. Medicine (Baltimore) 51:465-479, 1972 A detailed review of the world literature and a case report of joint disease with amyloid deposits in the synovium and periarticular tissues. CrossrefMedlineGoogle Scholar57. GORDONPRUZANSKIOGRYZLO DWM: Amyloid arthritis simulating rheumatoid disease in five patients with multiple myeloma. Am J Med 55:142-154, 1973 A report of five cases of multiple myeloma presenting with a rheumatoidlike polyarthritis caused by amyloid deposition in the synovium. Arthrocentesis showed a noninflammatory fluid containing amyloid fragments. CrossrefMedlineGoogle Scholar58. FAUCHALDRYGVOLDØYSTESE POB: Temporal arteritis and polymyalgia rheumatica: clinical and biopsy findings. Ann Intern Med 77:845-852, 1972 This report on 94 patients with one or both of these diagnoses includes instructive observations during a follow-up period averaging 4.5 years. LinkGoogle Scholar59. FRIEDMANKINKAID-SMITH AP: Arteritis with impaired renal function. Perspect Nephrol Hypertens 2:1047-1056, 1973 In 21 patients with necrotizing vasculitis and proliferative or necrotizing glomerular changes, the results of transplantation and treatment with anticoagulants were favorable and overall retrospective results seemed better than the general experience. Google Scholar60. BLOMGREN S: Erythema nodosum. Semin Arthritis Rheum 4:1-24, 1974 An excellent and comprehensive review of this disorder. CrossrefGoogle Scholar61. WOLFFFAUCIHORN SAR: Wegener's granulomatosis. Ann Intern Med 81:513-525, 1974 A large collection of cases with up-to-date information on the immunopathology, immunology, and, most importantly, the response to cyclophosphamide treatment. LinkGoogle Scholar62. PATTERSONIRONSKELLY RJJ: Pulmonary infiltrates with eosinophilia. J Allergy Clin Immunol 53: 245-255, 1974 A well-planned and helpful clinical review of the differential diagnosis of polyarteritis, bronchopulmonary allergic aspergillosis, and eosinophilic leukemia with general remarks on the syndrome of pulmonary infiltrates and eosinophilia, a clinically difficult area. CrossrefMedlineGoogle Scholar63. SERGENTLOCKSHINCHRISTIAN JMC: Vasculitis with hepatitis B antigenemia: long-term observations in nine patients. Medicine (Baltimore) 55:1-18, 1976 A careful clinical assessment of a group of patients with vasculitis associated with hepatitis B antigenemia; the necrotizing vasculitis associated with hepatitis B is clinically identical to vasculitis in patients without the antigen except for the presence of liver disease. CrossrefMedlineGoogle Scholar64. BREWERTONJAMES DD: The histocompatibility antigen (HL-A 27) and disease. Semin Arthritis Rheum 4: 191-207, 1975 A very high prevalence of HLA-B27 antigen was found in ankylosing spondylitis, Reiter's syndrome, spondylarthritis associated with inflammatory bowel disease or psoriasis, and arthritis with Yersinia or Salmonella infection. CrossrefMedlineGoogle Scholar65. CALINFRIES AJ: Striking prevalence of ankylosing spondylitis in "healthy" W27-positive males and females. N Engl J Med 293:835-839, 1975 Twenty per cent of apparently normal persons with the HLA-B27 met criteria for ankylosing spondylitis; both sexes were equally affected, indicating that, in a mild form, more women have this disease than previously appreciated. CrossrefMedlineGoogle Scholar66. ARNETTMCCLUSKYSCHACTER FOB: Incomplete Reiter's syndrome: discriminating features and HL-A W27 in diagnosis. Ann Intern Med 84:8-12, 1976 A group of young men with oligoarticular and asymmetric arthritis, but without other features of Reiter's syndrome, were HLA-B27 positive. Persons who are positive for HLA-B27 develop a characteristic arthritis in response to certain infectious agents or other stimuli. LinkGoogle Scholar67. MOLLHASLOCKMACRAE JII: Associations between ankylosing spondylitis, psoriatic arthritis, Reiter's disease, the intestinal arthopathies, and Behçet's syndrome. Medicine (Baltimore) 53:343-364, 1974 An excellent discussion of the clinical similarities among these seronegative spondyloarthritides, which should no longer be considered as atypical forms of rheumatoid arthritis. CrossrefMedlineGoogle Scholar68. CHAJEKFAINARU TM: Behçet's disease. Report of 41 cases and a review of the literature. Medicine (Baltimore) 54:179-196, 1975 Thrombophlebitis, arthritis and neurologic abnormalities occur frequently enough in Behçet's syndrome to be included among the manifestations of this syndrome, which was originally described as a classic triad of oral ulcers, genital ulcers, and eye inflammation. CrossrefMedlineGoogle Scholar69. GOOD A: Reiter's disease: a review with special attention to cardiovascular and neurologic sequellae. Semin Arthritis Rheum 3:253-286, 1974 An excellent clinical review of Reiter's disease. Cardiac and neurologic abnormalities of this disease are described in detail. CrossrefMedlineGoogle Scholar70. MOLLWRIGHT JV: Psoriatic arthritis. Semin Arthritis Rheum 3:55-78, 1973 A well-written review of psoriatic arthritis that clearly defines this disease as a separate clinical entity. The authors divide psoriatic arthritis into five clinical patterns. CrossrefMedlineGoogle Scholar71. LEVINEDOBBINS MW: Joint changes in Whipple's disease. Semin Arthritis Rheum 3:79-93, 1973 The joint manifestations of this rare disease are thoroughly discussed. Arthralgias and arthritis may precede bowel manifestations by several years. CrossrefMedlineGoogle Scholar72. KRAUSE R: Prevention of streptococcal sequelae by penicillin prophylaxis: a reassessment. J Infect Dis 131: 592-601, 1975 This special article discusses current trends in rheumatic fever (primarily diagnosis, treatment, and the changing clinical picture) and uncertainties regarding penicillin prophylaxis, at home and through the world. CrossrefMedlineGoogle Scholar73. GORDIS L: Effectiveness of comprehensive-care programs in preventing rheumatic fever. N Engl J Med 289:331-335, 1973 An outstanding epidemiologic survey showing the value of comprehensive programs in low socioeconomic areas. The problems associated with prophylaxis and prevention of rheumatic fever are highlighted. CrossrefMedlineGoogle Scholar74. WANNAMAKER L: The chain that links the heart to the throat. Circulation 48:9-18, 1973 A very good review of current hypotheses as to why streptococcal pharyngitis and not streptococcal pyoderma might cause rheumatic fever. This is an interesting enigma; the strain of streptococcus does not make the difference. CrossrefMedlineGoogle Scholar75. GRESHAMRATHEY GU: Osteoarthritis in knees of aged persons. Relationship between roentgenographic and clinical manifestations. JAMA 233:168-170, 1975 This study of the knee joints of 75 elderly persons showed a close correlation between specific clinical abnormalities and X-ray findings, as well as absence of symptoms or disability in some persons with roentgen abnormalities. CrossrefMedlineGoogle Scholar76. MANKIN H: The reaction of articular cartilage to injury and osteoarthritis. N Engl J Med 291:1285-1292, 1335-1340, 1974 A review of the normal stucture, biochemistry, and metabolic activity of cartilage. The second part describes the histologic, biochemical, and metabolic abnormalities in osteoarthritis. CrossrefMedlineGoogle Scholar77. BRANDTCATHCARTCOHEN KEA: Gonococcal arthritis. Clinical features correlated with blood, synovial fluid and genitourinary cultures. Arthritis Rheum 17:503-510, 1974 Gonococcal arthritis has often been divided into a bacteremic form and a septic-joint type. This study found no clinical differences between patients with positive blood cultures and those with only positive synovial fluid cultures. CrossrefMedlineGoogle Scholar78. GOLDENBERGBRANDTCATHCART DKE: Acute arthritis caused by Gram-negative bacilli: a clinical characterization. Medicine (Baltimore) 53:197-208, 1974 Excellent paper on Gram-negative septic arthritis points out the high mortality and the severe joint damage that often occur. CrossrefMedlineGoogle Scholar79. GOLDENBERGBRANDTCOHEN DKA: Treatment of septic arthritis. Comparison of needle aspiration and surgery as initial modes of joint drainage. Arthritis Rheum 18:83-90, 1975 Whether to drain a septic joint by needle aspiration surgically or medically has been debated for years. In this study most septic joints except hips were adequately treated with antibiotics and repeated needle aspirations. CrossrefMedlineGoogle Scholar80. WANDSMANNALPERT JEE: The pathogenesis of arthritis associated with acute hepatitis-B surface antigen-positive hepatitis. J Clin Invest 55:930-936, 1975 This paper describes the characterization of immune complexes recovered in cryoprecipitates of serum from patients with arthritis associated with hepatitis-B antigen. Concepts of pathogenesis are discussed. CrossrefMedlineGoogle Scholar81. DUFFYLIDSKYSHARP JMJ: Polyarthritis, polyarteritis and hepatitis B. Medicine (Baltimore) 55:19-37, 1976 Two rheumatic syndromes are associated with hepatitis B infection: transient polyarthritis sometimes associated with rash, and multisystem disease often associated with arteritis or glomerulonephritis. CrossrefMedlineGoogle Scholar82. SCHUMACHERGALL HE: Arthritis in acute hepatitis and chronic active hepatitis. Pathology of the synovial membrane with evidence for the presence of Australia antigen in synovial membranes. Am J Med 57:655-664, 1974 This paper describes in histologic detail the synovium in two patients. The scarcity of lymphocytes suggested that cell-mediated immunity was not playing a significant role in the arthritis. CrossrefMedlineGoogle Scholar83. GUTMAN A: Medical management of gout. Postgrad Med 51:61-66, 1972 Gout is the most manageable but most often mismanaged rheumatic disease. This review tells the internist everything he needs to know to treat (or not treat) gout and hyperuricemia wisely. CrossrefMedlineGoogle Scholar84. MCCARTY D: Mechanisms of the crystal deposition diseases—gout and pseudogout (editorial). Ann Intern Med 78:767-771, 1973 Editorial by a major investigator in pseudogout describes briefly and lucidly the current knowledge, ignorance, and hypotheses concerning the synovial inflammation due to crystals of sodium urate and calcium pyrophosphate. LinkGoogle Scholar85. BECKERMEYERSEEGMILLER MLJ: Gout with purine overproduction due to increased phosphoribosyl-pyrophosphate synthetase activity. Am J Med 55:232-242, 1973 This important paper describes an hereditary enzyme abnormality leading to increased purine production in gout, the first example of disease stemming from overactivity of an enzyme rather than deficiency. CrossrefMedlineGoogle Scholar86. STEELE T: Urate secretion in man: the pyrazinamide suppression test. Ann Intern Med 79:734-737, 1973 An overview of the hypothetical models of urate transport by the renal tubules. Appropriate discussion for the general internist. LinkGoogle Scholar87. RAMSDELLKELLEY CW: The clinical significance of hypouricemia. Ann Intern Med 78:239-242, 1973 Of 6000 automated serum urate measurements, 1% were less than 2 mg/dl. Various patients and the reasons for their hypouricemia are intelligently discussed. LinkGoogle Scholar88. WYNGAARDEN J: Metabolic defects of primary hyperuricemia and gout. Am J Med 56:651-664, 1974 An excellent and readable review of gout as a syndrome of multiple pathogenesis rather than as a single disease entity. The paper's clarity reflects experience, thought, and perspective. CrossrefMedlineGoogle Scholar89. HADLERFRANCKBRESS NWN: Acute polyarticular gout. Am J Med 56:715-719, 1974 This retrospective review describes polyarticular gout, which is not so rare and may pose a diagnostic problem in patients on urate-lowering drugs. CrossrefMedlineGoogle Scholar90. MANKIN H: Rickets, osteomalacia, and renal osteodystrophy. J Bone Joint Surg (Am) 56A: 101-128, 352-386, 1974 A review of the clinical problem of rickets and osteomalacia and an exhaustive list of references to the various disorders that may be present with these abnormalities. CrossrefGoogle Scholar91. AVIOLIHADDAD LJ: Vitamin D: current concepts. Metabolism 22:507-531, 1973 Although progress in this field is rapid, this article provides a good interpretation of vitamin D metabolism with respect to clinical disorders. CrossrefMedlineGoogle Scholar92. DEQUEKER J: Bone and ageing. Ann Rheum Dis 34:100-115, 1975 A scholarly review of the mechanisms of bone loss that accompany ageing, with approaches to management. CrossrefMedlineGoogle Scholar93. BORDIERPECHETHESSE PMP: Response of adult patients with osteomalacia to treatment with crystalline 1 alpha-hydroxy-vitamin D3. N Engl J Med 291:866-871, 1974 An early report of the success of a synthetic, hydroxylated derivative on osteomalacia in man. CrossrefMedlineGoogle Scholar94. FRANCKBRESSSINGER WNF: Rheumatic manifestations of Paget's disease of bone. Am J Med 56: 592-603, 1974 A clinical description of Paget's disease with respect to the problems that a rheumatologist should consider. CrossrefMedlineGoogle Scholar95. SINGER F: Treatment of Paget's disease of bone. Postgrad Med 57:117-120, 1975 A brief review with adequate references to the current approach using drugs to treat patients with Paget's disease of bone. CrossrefMedlineGoogle Scholar96. ROONEYBALLANTYNEBUCHANAN PDW: Disorders of the locomotor system associated with abnormalities of lipid metabolism and the lipoidoses. Clin Rheum Dis 1: 163-193, 1975 This paper reviews the association between lipid abnormalities and rheumatic manifestations. Arthralgias and arthritis occur in patients with both type II and type IV hyperlipoproteinemia. CrossrefGoogle Scholar97. AHOAHVONENLASSUS KPA: HL-A 27 in reactive arthritis: a study of yersinia arthritis and Reiter's disease. Arthritis Rheum 17:521-526, 1974 Arthritis after Yersinia infection was shown to occur predominantly in those persons with the HLA-B27 antigen. The frequency of HLA-B27 antigen in patients with yersinia arthritis was approximately the same as in those with Reiter's syndrome. CrossrefMedlineGoogle Scholar98. LEINOKALLIOMAKI RJ: Yersiniosis as an internal disease. Ann Intern Med 81:458-461, 1974 Yersinia infection causes a problem in the differential diagnosis of several types of arthritis, rheumatic fever in particular, because there are fever, arthritis, and occasionally electrocardiographic changes. This diagnosis is primarily serologic. LinkGoogle Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: Philadelphia, Pennsylvania*E. Carwile LeRoy, M.D., F.A.C.P., Chairman; Bruce C. Gilliland, M.D.; L. Andrew Healey, M.D., F.A.C.P.; Stephen M. Krane, M.D., F.A.C.P.; Naomi F. Rothfield, M.D.; Robert M. Stroud, M.D., F.A.C.P.; and Norman Talal, M.D.†Computer literature-file of the National Library of Medicine, Bethesda, Maryland, consulted through the Regional Medical Library service Library of the College of Physicians of Philadelphia, with the technical assistance of Ruthanne Henner. 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