tbuckley/cabot-search huggingface dataset:
Leads from the MMWR. Human T-lymphotropic virus type III/lymphadenopathy-associated virus: agent summary statement
Journal: JAMA (1986)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Hormonal and Neurogenic Cardiovascular Disorders: Endocrine and Neuro-Endocrine Factors in Pathogenesis and Treatment.
Journal: JAMA (1953)

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Revue Médicale de la Suisse Romande, Lausanne
Journal: JAMA (1966)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Insulin and the kidney
Journal: JAMA (1971)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
C<scp>OLOR</scp>V<scp>ISION</scp>
Authors: Karl R. Gegenfurtner, Daniel C. Kiper
Journal: Annual Review of Neuroscience (2003)

▪ Abstract Color vision starts with the absorption of light in the retinal cone photoreceptors, which transduce electromagnetic energy into electrical voltages. These voltages are transformed into action potentials by a complicated network of cells in the retina. The information is sent to the visual cortex via the lateral geniculate nucleus (LGN) in three separate color-opponent channels that have been characterized psychophysically, physiologically, and computationally. The properties of cells in the retina and LGN account for a surprisingly large body of psychophysical literature. This suggests that several fundamental computations involved in color perception occur at early levels of processing. In the cortex, information from the three retino-geniculate channels is combined to enable perception of a large variety of different hues. Furthermore, recent evidence suggests that color analysis and coding cannot be separated from the analysis and coding of other visual attributes such as form and motion. Though there are some brain areas that are more sensitive to color than others, color vision emerges through the combined activity of neurons in many different areas.
The Ingelfinger rule
Authors: L. D. Grouse
Journal: JAMA (1981)

At last spring's national clinical meetings, Jesse Roth, MD, president of the American Society for Clinical Investigation, discussed a study he had carried out. Roth found that few society members were Nobel laureates, despite the fact that US scientists have monopolized Nobel prizes in medicine and physiology since World War II. That the society, supposedly the US scientist-internist's hall of fame, had shown such limited ability in recognizing scientific genius was most surprising. He concluded that the society ought to recruit more diverse persons for its membership if the organization wished truly to represent excellence in US medical science. In my opinion, the most important conclusion from Roth's study is that it is difficult to know in advance which scientific work will have the greatest importance. If this premier group of US investigators has limited success in picking great scientists, one must conclude that editors of medical journals and their
Industrial Fermentations.
Journal: JAMA (1926)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Summary of a Workshop on Immunosuppression in the Management of Type I Diabetes Mellitus (IDDM)
Journal: New England Journal of Medicine (1983)

Immunotherapputic interventions are now being used in the treatment of a number of diseases thought to be of immune origin or to have a major component involving the immune system. These diseases include various vasculitides, rheumatoid arthritis, multiple sclerosis, glomerulonephritis, and a large number of hematologic disorders. Recent evidence suggests that insulin-dependent (Type I, juvenile-onset) diabetes mellitus (IDDM) may have an important autoimmune component in most, if not all, cases,1 possibly triggered by a viral infection. This evidence includes round-cell infiltration in the endocrine pancreas, circulating islet-cell antibodies as measured by several different techniques, an association with certain HLA alleles . . .
Asbestosis-Related Years of Potential Life Lost Before Age 65 Years—United States, 1968-2005
Journal: JAMA (2009)

Exposure to asbestos fibers can cause asbestosis and other diseases after a latency of 10-40 years from initial exposure to onset of illness. Asbestos still is used in the United States (approximately 2,200 metric tons in 2006) in certain products manufactured domestically. In addition, an undocumented amount of asbestos continues to be imported in products manufactured elsewhere, and a substantial amount of asbestos remains in existing buildings and manufactured products. An estimated 1.3 million construction and general industry workers in the United States potentially are exposed to asbestos each year, mainly from manipulation of asbestos during renovation or demolition activities. Also, although asbestos ore is no longer mined in the United States, some U.S. mine workers might remain at risk for exposure to asbestos contained in other ores. To characterize trends in premature mortality attributed to asbestosis in the United States, CDC analyzed annual underlying cause-of-death data for 1968-2005, the most recent years for which data were available. This report describes the results of that analysis, which indicated that annual years of potential life lost before age 65 years (YPLL) attributed to asbestosis increased 64%, from an average of 146.0 YPLL per year during 1968-1972 to 239.6 per year during 2001-2005 (regression trend for the 5-year moving average, p<0.001), for an overall total of 7,267 YPLL (mean per decedent: 6.2) over the entire period. These results demonstrate that asbestosis-attributable YPLL continue to occur and that efforts to prevent, track, and eliminate asbestosis need to be maintained.
Kidney
Authors: H. P. Schuster, K. F. Bodmann, P. M. Suter, M. Hemmer, R. Clemens, R. Robert, J. Rouffineau, K. Dhoste, F. Malin, O. Pourrat, J. M. Péchier, B. Levy, P. Burtin, C. Dopff, P. M. Mertes, J. P. Villemot, Ph. Mezzarobba, M. M. Agostini, Ph. Loirat, D. Kleinknecht, F. Brivet, P. Landais, A. Mercatello, A. Hadj-Aïssa, S. Négrier, B. Allaouchiche, B. Coronel, N. Pozet, J. F. Moskovtchenko, T. Philip
Journal: Intensive Care Medicine (1992)
Migraine--research.
Authors: J N Blau
Journal: BMJ (1971)

<h3>Abstract</h3> <h3>Background</h3> Many SARS-CoV-2 variants of concern have emerged since the Covid-19 outburst, notably the lineages detected in the UK, South Africa, and Brazil. Their increased transmissibility and higher viral load put them in the spotlight. Much has been investigated on the ability of those new variants to evade antibody recognition. However, not enough attention has been given to pre-existing and induced SARS-CoV-2-specific CD8+ T cell responses during the natural course of infection by new lineages. <h3>Methods</h3> In this work, we investigated the SARS-CoV-2-specific CD8+ T cell epitopes from the main variants of concern and the potential of associated mutations to trigger or hinder CD8+ T-cells response. We also estimated the population’s coverage of these different lineages, considering peptide binding predictions to class I HLA alleles from 29 countries to investigate differences in the fraction of individuals expected to respond to a given epitope set from new and previous lineages. <h3>Results</h3> We observed a lower populational coverage for 20B/S.484K (P.2 lineage) in contrast to an increased coverage found for 20H/501Y.V2 (B.1.351 Lineage) and 20J/501Y.V3 (P.1 lineage) compared to a reference lineage. Moreover, mutations such as Spike N501Y and Nucleocapsid T205I were predicted to have an overall higher affinity through HLA-I than the reference sequence. <h3>Conclusions</h3> In summary, the data in this work provided evidence for the existence of potentially immunogenic and conserved epitopes across new SARS-CoV-2 variants, but also highlights the reduced populational’s coverage for the Brazilian lineage P.2, suggesting its potential to evade from CD8+ T-cell responses. Our results also may guide efforts to characterize and validate relevant peptides to trigger CD8+ T-cell responses, and design new universal T-cell-inducing vaccine candidates that minimize detrimental effects of viral diversification and at the same time induce responses to a broad human population.
Folic Acid.--Part II
Authors: J. F. Wilkinson
Journal: BMJ (1948)

ANAEMIA OF PREGNANCY BRTIRNSH form of a small tasteless tablet and because of the rapid clinical and haematological improvement which results even in cases refractory to parenteral liver therapy.Since the duration of treatment with folic acid is limited to a few weeks, the danger of producing subacute comibined degen- eration of the cord is greatly reduced as compared with Addisonian pernicious anaemia.Further time must elapse, however, before it can be stated confidently that neuro- logical features will not occur in patients with pernicious anaemia of pregnancy receiving folic acid.As many cases of pernicious anaemia of pregnancy are suffering from a dual deficiency of iron and the factor required for maintenance of normoblastic blood formation it is desirable that both deficiencies be corrected simul- taneously.While our patients were receiving folic acid iron was deliberately withheld in order to simplify the interpretation of the therapeutic response.(Anahaemin is a refined liver extract, and " plexan " and i" campolon " are crude extracts.)Summary Three cases of pernicious anaemia of pregnancy and one case of Addisonian pernicious anaemia complicated by pregnancy responded excellently to treatment with folic acid.Three of the cases had failed to respond to the parenteral injection of liver extracts prior to the administration of folic acid.We wish to thank Prof. Kellar, Dr. Fahmy, and others of our colleagues in the
"PHYSICAL EXAMINATION OF FOOD HANDLERS"
Journal: JAMA (1922)

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Incidence of Megaloblastic Anemia after Subtotal Gastrectomy
Authors: Lloyd D. MacLean
Journal: New England Journal of Medicine (1957)

MEGALOBLASTIC anemia indistinguishable from pernicious anemia is a late but inevitable sequela of total gastrectomy and probably of proximal subtotal gastrectomy with esophagoantrostomy.1 2 3 This conclusion was reached with the use of the absorption of radioactive vitamin B12 as an assay of intrinsic factor in the gastrointestinal tract and the determination of the incidence of megaloblastosis after total gastrectomy when all patients undergoing this operation at one institution were followed for more than three years. Megaloblastic anemia has been reported after distal subtotal gastrectomy, but the true incidence has never been investigated. Anemia after subtotal gastrectomy is common (as high . . .
Influenza vaccine
Journal: JAMA (1971)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Lincoln, Kennedy, and the autopsy
Authors: M. S. Micozzi
Journal: JAMA (1992)

Presidential assassinations leave a deep scar on our collective memory and consciousness as a nation. Somehow, the American public in 1992 seems more comfortable with its knowledge of the circumstances of the death of our first assassinated President, Abraham Lincoln, investigated in the mid-1860s, than it does with that of our most recent, John F. Kennedy, investigated in the mid-1960s. See also p 2794. When Lincoln was shot at Ford's Theatre in Washington, DC, on the evening of Good Friday, April 14, 1865, no US President had been assassinated before, and, coupled with the simultaneous bloody attack on Secretary of State William Seward and his family at his home and the attempt on Vice President Andrew Johnson, no one knew quite what to do. Ambulance service did not then exist in Washington and cases like that of the wounded President were generally attended in the home and not the hospital.
COLLEGE OF PHYSICIANS OF PHILADELPHIA.SECTION ON ORTHOPEDIC SURGERY.
Journal: JAMA (1894)

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