tbuckley/cabot-search huggingface dataset:
THE INDEX.
Journal: JAMA (1899)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Loss
Authors: Mary E. Martin
Journal: JAMA (2008)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Water Quality Problems in Nogales, Sonora
Authors: Roberto A. Sanchez
Journal: Environmental Health Perspectives (1995)

This article presents the results of a transboundary water quality monitoring program at the two Nogales area in the Arizona-Sonora border region.The program was carried out jointly in 1990 by U.S. and Mexican institutions.The results show pollution problems due to deficiencies in Nogales, Sonora municipal sewerage system, causing not only sewage spills in several parts of the city but also creating occasional transboundary problems.The results also showed potential illegal dumping of industrial hazardous waste (VOCs) into Nogales' municipal sewerage system.All of the organic compounds found in the sewage samples are solvents frequently used by the border industry.Occasional brakes of pipes spill the pollutants into the Nogales Wash, a water stream that runs parallel to Nogales' main sewerage line.Samples of the municipal water system showed no traces of pollutants.However, two rounds of samples detected concentrations of VOCs in wells used to supply water by trucks to low income neighbor- hoods in Nogales, Sonora.Ironically, the pollution detected in these wells has a greater impact in low income groups of the city that pay three to four times more per liter of water they consume, than the rest of the inhabitants with clean water from the municipal system.
Optimisation and qualification of tumour mutational burden (TMB) by targeted next-generation sequencing (TNGS) as a clinically applicable biomarker in hepatocellular carcinoma (HCC)
Authors: Ching Ngar Wong, Kathy Dominy, Francesco Mauri, Takahiro Kaneko, Persephone Du Parcq, Jamshid Khorasad, Pierluigi Toniutto, Robert D. Goldin, Claudio Avellini, David J. Pinato
Journal: Journal of Hepatology (2020)
Treatment IND/Group C Distribution for Advanced Metastatic Ovarian Cancer
Journal: JAMA (1992)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
BAALC-Associated Mir-3151 Is An Independent Prognostic Factor In Younger Patients With Intermediate-Risk Cytogenetic Acute Myeloid Leukemia
Authors: Marina Díaz-Beyá, Alfons Navarro, Marta Pratcorona, Salut Brunet, Josep F Nomdedeu, Josep-Maria Ribera, Mar Tormo, Rafael F. Duarte, Olga Salamero, David Gallardo, Lourdes Escoda, Meritxell Nomdedeu, Ruth M Risueño, Montserrat Hoyos, Francisco Cervantes, Jorge Sierra, Mariano Monzó, Jordi Esteve
Journal: Blood (2013)
Retiring may predispose to fatal heart attack
Authors: E. R. Gonzalez
Journal: JAMA (1980)

Clinicians have long suspected that retirement is detrimental to the health of some patients. Now, investigators at Harvard Medical School's Channing Laboratory in Boston suggest that it may lead to fatal coronary heart disease. Charles H. Hennekens, MD, and associates stumbled on this finding in the course of a study originally designed to test the hypothesis that coffee consumption is a risk factor for coronary heart disease. Coffee drinkers, they found, were not at greater risk of dying of heart attacks, but retirees were. The team selected their subjects via a weekly review of death certificates for married white men, aged 30 to 70, in 1973 and 1974. All of the men had died of coronary heart disease within 24 hours after symptoms of it developed. They were matched by age (within one decade) and neighborhood of residence to an equal number of living control subjects chosen through systematic household
ANTISEPSIS IN OBSTETRICS.
Journal: JAMA (1894)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Initiative Seeks Answers to Cancer Disparities
Authors: M. Mitka
Journal: JAMA (2000)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Hidradenitis suppurativa and electrocardiographic changes: a cross‐sectional population study
Authors: C.R. Juhl, I.M. Miller, G.B. Jemec, J.K. Kanters, C. Ellervik
Journal: British Journal of Dermatology (2018)

Hidradenitis Suppurativa (HS) is a chronic skin condition that usually develops after puberty. It causes recurrent painful boils in the armpits, groins, genital region and other areas with skin folds. Beyond the debilitating skin problems, the disease is associated with several factors that increase risk of cardiovascular disease (smoking, obesity, metabolic syndrome and systemic inflammation) and recently, higher risk of developing cardiovascular disease. The aim of this study from Denmark was to investigate this possible cardiovascular burden associated with HS influence on the heart's electrical activity. To fulfil this aim, several electrocardiographic parameters were compared between HS patients and a control group. The HS patients were identified using a questionnaire which also was used to divide the HS patients into severity groups (mild, moderate and severe). Overall 404 HS patients and 19,001 controls were identified in the population. The heart's electrical activity parameters investigated were heart rate, and measurements called PR‐interval, QRS‐duration, JTcF‐ and QTcF‐interval. The analyses performed in this study took into account age, sex, smoking and body mass index. The main finding of this study was a higher heart rate for the patients with severe HS compared to the control group. Other findings were a shorter QRS‐duration (the depolarization of the ventricles of the heart) for the patients with mild HS, a result which needs further investigation, and no difference for the parameters PR‐interval, JTcF‐interval and QTcF‐interval. The conclusion of this study is that severe HS is associated with a higher resting heart rate. Given that resting heart rate is associated with increased mortality, and that HS patients have increased risk of cardiovascular issues, this finding is potentially important, easily testable and allows for treatment.
II. USE OF VACCINES AND SERUMS IN GONORRHEAL URETHRITIS AND ITS COMPLICATIONS
Journal: JAMA (1921)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Addendum for Euro Surveill. 2017;22(8)
Authors:  Eurosurveillance Editorial Team
Journal: Eurosurveillance (2017)

Preview this article: Addendum for Euro Surveill. 2017;22(8), Page 1 of 1 < Previous page | Next page > /docserver/preview/fulltext/eurosurveillance/22/40/eurosurv-22-40-5-1.gif
Prinzmetal's angina. Provocative test and current therapy
Authors: R. C. Curry
Journal: JAMA (1978)

ALMOST 18 years have passed since Prinzmetal and his colleagues<sup>1</sup>originally described a variant form of angina pectoris characterized by cyclic attacks of chest pain occurring at rest and associated with transient ST-segment elevation. Prinzmetal postulated that these attacks were due to spasm of a major coronary artery. Recently, a number of investigators have reported coronary artery spasm occurring during spontaneous episodes of variant angina confirming Prinzmetal's hypothesis. Since spontaneous attacks of variant angina may be infrequent, specific tests for provoking coronary artery spasm in variant angina would be helpful in the diagnosis and evaluation of therapy in patients with this disorder. This case of a patient with classic Prinzmetal's angina illustrates the clinical presentation, current pharmacologic therapy, angiographic changes associated with pain episodes, an old but revived provocative test, and a new therapeutic agent for treatment of this condition. <h3>Report of a Case</h3> A 40-year-old woman was awakened at
The Test
Authors: Kimberly R. Myers
Journal: Annals of Internal Medicine (2015)

Ad Libitum2 June 2015The TestKimberly R. Myers, PhDKimberly R. Myers, PhDFrom Hershey, Pennsylvania.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/M15-0293 Audio Reading - “The Test” Audio. Michael A. LaCombe, MD, Annals Associate Editor, reads "The Test," by K.R. Myers. Your browser does not support the audio element. Audio player progress bar Step backward in current audio track Play current audio trackPause current audio track Step forward in current audio track Mute current audio trackUnmute current audio track 00:00/ SectionsAboutFull TextPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail in the waiting roomI test the waters of your absence,sounding the dark edgesof its ultimate depth.Imagining day by daythe primal smell fadingfrom your clothes, dimminglike my dream of you:my head in your lap, yours in mine.Listening over and over, tryingto hear that song exactly as you'd doneits rises and falls,transcendencein a minor key.Picturing places finallyempty of you:a bridge, a path, a doorand all the world gonehollow.Nowup from murky bottomssurfacing into sunlightand soundthe grace of your returningand these words my gift to you,coming home.Kimberly R. Myers, PhDHershey, Pennsylvania Author, Article, and Disclosure InformationAffiliations: From Hershey, Pennsylvania.Current Author Address: Kimberly R. Myers, PhD; e-mail, [email protected]edu. PreviousarticleNextarticle Advertisement Audio Reading - “The Test” Audio. Michael A. LaCombe, MD, Annals Associate Editor, reads "The Test," by K.R. Myers. Your browser does not support the audio element. Audio player progress bar Step backward in current audio track Play current audio trackPause current audio track Step forward in current audio track Mute current audio trackUnmute current audio track 00:00/ FiguresReferencesRelatedDetails Metrics 2 June 2015Volume 162, Issue 11Page: 800KeywordsFallsSmell ePublished: 2 June 2015 Issue Published: 2 June 2015 Copyright & PermissionsCopyright © 2015 by American College of Physicians. All Rights Reserved.PDF downloadLoading ...
Synopsis of Midwifery and Gynaecology.
Journal: JAMA (1925)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Increased nitric oxide synthase activity as a cause of mitochondrial dysfunction in rat hepatocytes: roles for tumor necrosis factor alph
Authors: I Kurose
Journal: Hepatology (1996)

Kupffer cells have been implicated in playing an important role in the pathogenesis of endotoxemia-associated liver injury. The present study was designed to investigate whether Kupffer cell-derived mediators alter the mitochondrial oxidative phosphorylation of hepatocytes in the endotoxemic condition. Liver cells were isolated from male Wistar rats. Oxidative phosphorylation was monitored as the fluorescence of rhodamine 123 (Rh123), which is the fluorescent cationic dye used to indicate mitochondrial energy synthesis. Two hours after coculture of hepatocytes with lipopolysaccharide (LPS)-pretreated Kupffer cells, a marked decrease in hepatocyte rhodamine 123 fluorescence was observed. The hepatocyte mitochondrial dysfunction was attenuated by the addition of either NG-monomethyl-L-arginine (L-NMMA), an inhibitor of nitric oxide (NO) synthesis, or aminoguanidine, an inducible-type of NO synthase inhibitor, to the culture medium of cocultures, to the pretreatment of LPS-activated Kupffer cells with antisense oligodeoxynucleotides against iNOS messenger RNA (mRNA), or to tumor necrosis factor alpha (TNF-alpha) mRNA. Four hours after the coculture, hepatocyte Rh123 fluorescence further decreased, and an iNOS induction as well as an increased NO production were observed in hepatocytes that were cocultured with LPS-pretreated Kupffer cells. The membrane barrier dysfunction of hepatocytes, indicated by propidium iodide staining, was also induced by a 4-hour coculture with LPS-pretreated Kupffer cells. These late-phase changes were inhibited either by the pretreatment of hepatocytes with antisense oligodeoxynucleotides against iNOS mRNA or by treatments that are effective in the early phase (within 2 hours). Incubation with recombinant rat TNF-alpha decreased hepatocyte Rh123 fluorescence within 2 hours. Thus, the present study suggests that NO and TNF-alpha released from LPS-pretreated Kupffer cells directly inhibit the hepatocyte mitochondrial function in the early phase, and then NO synthesized by TNF-alpha-induced hepatocyte iNOS causes lethal hepatocyte injury, characterized by diminished mitochondrial energization and membrane barrier function in the late phase. (Hepatology 1996 Nov;24(5):1185-92)
Occupational exposure to silica and end-stage renal disease
Authors: R. A. Wilke
Journal: JAMA (1997)

<h3>To the Editor.</h3> —The analysis by Dr Calvert and colleagues<sup>1</sup>demonstrated an association between chronic occupational exposure to silica and the development of end-stage renal disease (ESRD). Based on a standardized incidence ratio (SIR) of 1.37 (95% confidence interval [CI], 0.68-2.46), the authors reported that the overall risk for ESRD was significantly elevated in gold miners who had worked underground for at least 1 year. Although this study clearly illustrates the utility of the ESRD Program Management and Medical Information System, 1 limitation of the data base is that it only identifies individuals with renal disease once they have reached end stage. As the authors point out, the exact nature of the pathogenetic link between silica exposure and renal dysfunction will only be elucidated by studies using a more moderate end point, such as the development of chronic renal insufficiency. Interestingly, Calvert et al<sup>1</sup>showed that the risk for