Allergy and Immunology: An Annotated Bibliography of Recent Literature
Journal: Annals of Internal Medicine (1979)

The Literature of Medicine1 January 1979Allergy and Immunology: An Annotated Bibliography of Recent LiteratureReferences to Journal Articles and Other PapersALLERGY AND IMMUNOLOGY COMMITTEEALLERGY AND IMMUNOLOGY COMMITTEESearch for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-90-1-135 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptThis bibliography includes references to 95 recently published journal papers and similar sources that represent important developments in the concept and practice of internal medicine. It has been compiled by the Allergy and Immunology Committee as part of the Medical Knowledge Self-Assessment Program V Syllabus.In preparing this bibliography, the committee reviewed MEDLINE†bibliographic printouts of journal articles published since the beginning of 1975 and also drew on its own files. Consideration was given to availability of sources in libraries of hospitals and other clinical institutions in the United States and Canada; thus, only papers in English are listed, and...Basic Concepts of the Immune Response Mechanisms Directly Related to the immune Response IMMEDIATE HYPERSENSITIVITY CYTOTOXICITY-TISSUE BINDING IMMUNE COMPLEX DISEASE DELAYED HYPERSENSITIVITY Common Allergic Disorders Immunodeficiency Diseases Autoimmune Diseases Immunosuppression Drug Hypersensitivity Hypersensitivity Lung Disease Clinical Importance of the Major Histocompatibility Complex Tumor Immunology Diseases with Hypereosinophilia Miscellaneous1. CRADDOCKFEHRBRIGHAMKRONENBERGJACOB PJKRH: Complement and leukocyte-mediated pulmonary dysfunction in hemodialysis. N Engl J Med 296:169-114, 1977 Activation of the alternate complement pathway by contact with cellulose dialysis membranes is shown in vitro, and in patients on hemodialysis C5a causes aggregation of neutrophils with trapping in the pulmonary vasculature. This accounts for the neutropenia and acute pulmonary dysfunction that frequently accompany cellophane membrane hemodialysis. CrossrefMedlineGoogle Scholar2. DOHERTY P: Cellular immune response in virus infection. Arthritis Rheum (suppl) 2LS97-99, 1978 The killing of virus-infected target cells by cytotoxic T cells requires two specificity areas or signals: [1] viral recognition, and [2] shared expression of murine H2 genes regulating K and D region antigens but not the I region. This mechanism is also relevant to tumor immunity. Google Scholar3. MORETTAWEBBGROSSILYDYARDCOOPER LSCPM: Functional analysis of two human T-cell subpopulations: help and suppression of B-cell responses by T cells bearing receptors for IgM or IgG. J Exp Med 146:184-200, 1977 Different immunoregulatory capabilities have been shown to be characteristic of two distinct T cell subpopulations. This study is important in understanding immunoregulation by human lymphocyte populations. CrossrefMedlineGoogle Scholar4. MÜLLER-EBERHARD H: Chemistry and function of the complement system. Hosp Pract 12(8):33-43, 1977 Concise, up-to-date review of the structure and biologic functions of the proteins in the classical and alternate pathways and of the essential role of control mechanisms. CrossrefMedlineGoogle Scholar5. PARKER C: Control of lymphocyte function. N Engl J Med 295:1180-1186, 1976 Evidence of the positive and negative feedback control mechanisms regulating the activation of T and B lymphocytes is reviewed, with emphasis on intracellular events. CrossrefMedlineGoogle Scholar6. STASTNY P: Association of the B-cell alloantigen DRw4 with rheumatoid arthritis. N Engl J Med 298:869-871, 1978 The potential for rapid typing of D locus markers using antibody to B lymphocytes compared to the mixed lymphocyte culture reaction method is shown. The relative risk of developing seropositive classical rheumatoid arthritis for a person who is DRw4 positive is 6.1 times that for control subjects (those negative for DRw4). CrossrefMedlineGoogle Scholar7. HUNTVALENTINESOBOTKALICHTENSTEIN KMAL: Diagnosis of allergy to stinging insects by skin testing with Hymenoptera venoms. Ann Intern Med 85:56-59, 1976 Most patients with a history of anaphylactic reactions after Hymenoptera stings reacted to intradermal skin testing with venom, whereas only one of 150 skin tests in control patients was positive. LinkGoogle Scholar8. KAPLANGRAYSHAFFHORAKOVABEAVEN ALRZM: In vivo studies of mediator release in cold urticaria and cholinergic urticaria. J Allergy Clin Immunol 55:394-402, 1975 Histamine was found to be the major mediator of urticaria and hypotension in cold urticaria and in some cases of cholinergic urticaria. A single atypical patient with cholinergic urticaria had elevated plasma serotonin levels. CrossrefMedlineGoogle Scholar9. LEWISGOETZLWASSERMANVALONERUBINAUSTEN RESFRK: The release of four mediators of immediate hypersensitivity from human leukemic basophils. J Immunol 114:87-92, 1975 Human basophils contain the preformed mediators histamine, and eosinophil chemotactic factor, and generated slow-reacting substance of anaphylaxis and platelet aggregating factor. MedlineGoogle Scholar10. MATHISONARROYAVEBHATHUREWITZMARNELL DCKDD: Hypocomplementemia in chronic idiopathic urticaria. Ann Intern Med 86:534-538, 1977 Seventy-two consecutive patients with chronic urticaria were evaluated for hypocomplementemia; 10 had evidence of either classic or alternative mechanisms of complement activation, or both. Vasculitis with urticaria is not rare. LinkGoogle Scholar11. SOLLEYGLEICHJORDONSCHROETER GGRA: The late phase of the immediate wheal and flare skin reaction. Its dependence upon IgE antibodies. J Clin Invest 58:408-420, 1976 A late inflammatory response, 6 to 12 h after allergen challenge, is due to IgE antibody. This late phase, therefore, is not an Arthus reaction. CrossrefMedlineGoogle Scholar12. WASSERMANAUSTEN SK: Arylsulfatase B of human lung. Isolation, characterization, and interaction with slow-reacting substance of anaphylaxis. J Clin Invest 57:738-744, 1976 Human lung is shown to contain the enzyme arylsulfatase B. Because this material can inactivate slow-reacting substance of anaphylaxis, its presence at this site is of clinical importance. CrossrefMedlineGoogle Scholar13. WASSERMANGOETZLAUSTEN SEK: Inactivation of slow reacting substance of anaphylaxis by human eosinophil arylsulfatase. J Immunol 114:645-649, 1975 Human eosinophils contain arylsulfatase, an enzyme capable of cleaving slow-reacting substance of anaphylaxis. Specific eosinophil arrival activated by basophil release of eosinophil chemotactic factor of anaphylaxis may regulate the physiologic effects of slow-reacting substance of anaphylaxis. MedlineGoogle Scholar14. LOGUEKURLANDERPEPEDAVISSILBERMAN GRPWH: Antibody-dependent lymphocyte-mediated granulocyte cytotoxicity in man. Blood 51:97-108, 1978 Two patients with granulocytopenia associated with collagen vascular disease were shown to have IgG antibody-dependent, lymphocyte-mediated granulocytotoxicity. CrossrefMedlineGoogle Scholar15. GUPTAKUEPPERSDEREMEEHUSTONMCDUFFIE RFRKF: Pulmonary and extrapulmonary sarcoidosis in relation to circulating immune complexes. A quantification of immune complexes by two radioimmunoassays. Am Rev Respir Dis 116:261-266, 1977 Immune complexes were found in the sera of 27 of 53 patients with pulmonary sarcoidosis, suggesting a role of immune complexes in the pathogenesis of this disease, which is usually ascribed only to delayed-type hypersensitivity. MedlineGoogle Scholar16. LEVINSKYCAMERONSOOTHILL RJJ: Serum immune complexes and disease activity in lupus nephritis. Lancet 1:564-567, 1977 Immune complexes were found in sera of 21 of 29 patients with lupus nephritis, which correlated well with clinical disease activity. Effective treatment led to reduced levels of immune complexes. CrossrefMedlineGoogle Scholar17. SCHUR P: Immune complex assays: the state of the art (editorial). N Engl J Med 298:161-162, 1978 The burgeoning methods of assaying immune complexes are viewed in terms of clinical usefulness. CrossrefMedlineGoogle Scholar18. MCGEADYBUCKLEY SR: Depression of cell-mediated immunity in atopic eczema. J Allergy Clin Immunol 56:393-406, 1975 Twenty-one patients with atopic eczema were studied for immune competence by skin tests with Candida and streptokinasestreptodornase. A striking degree of anergy and elevated IgE levels correlated with the severity of the eczema. CrossrefMedlineGoogle Scholar19. OPELZSCHEER GM: Cutaneous sensitivity and in vitro responsiveness of lymphocytes in patients with disseminated coccidioidomycosis. J Infect Dis 132:250-255, 1975 Six patients with disseminated coccidioidomycosis did not show cutaneous reactivity to coccidioidin, whereas 68 of 74 has good skin reactions. Autologous plasma in two of the six patients suppressed lymphocyte function. CrossrefMedlineGoogle Scholar20. STEELEVINCENTHENSENFUCCILLOCHAPACANALES RMSDIL: Cellular immune responses to herpes simplex virus type 1 in recurrent herpes labialis: in vitro blastogenesis and cytotoxicity to infected cell lines. J Infect Dis 131:528-534, 1975 In 12 volunteers with recurrent herpes labialis, cell-mediated immunity was assessed by means of a proliferative assay and a cytotoxic assay. Patients had enhanced lymphocyte blastogenesis but decreased cytotoxicity. CrossrefMedlineGoogle Scholar21. DAVIESBUTCHERSALVAGGIO RBJ: Occupational asthma caused by low molecular weight chemical agents. J Allergy Clin Immunol 60:93-95, 1977 Concise editorial reviewing an important syndrome. CrossrefMedlineGoogle Scholar22. Drugs for Asthma. Med Lett Drugs Ther 20:69-72, 1978 An up-to-date concise description of drugs available for treatment of asthma. MedlineGoogle Scholar23. FISHROSENTHALBATRAMENKESSUMMERPERMUTTNORMAN JRGHWSP: Airway responses to methacholine in allergic and nonallergic subjects. Am Rev Respir Dis 113:579-586, 1976 Data suggest that asthmatics have hypersensitivity of large and small airways. Patients with hay fever have large airway sensitivity only. MedlineGoogle Scholar24. FRANKGELFANDATKINSON MJJ: Hereditary angioedema: the clinical syndrome and its management. Ann Intern Med 84:580-593, 1976. In-depth review of all aspects of hereditary angioedema. LinkGoogle Scholar25. GELFANDSHERINSALLINGFRANK JRDM: Treatment of hereditary angioedema with danazol. Reversal of clinical and biochemical abnormalities. N Engl J Med 295:1444-1448, 1976 The mechanism of action of androgens in the therapy of hereditary angioedema are demonstrated, and the use of danazol, a modified androgen with reduced virilization potential, is discussed. CrossrefMedlineGoogle Scholar26. KAPLAN A: Mediators of urticaria and angioedema. J Allergy Clin Immunol 60:324-332, 1977 An in-depth review of recent advances in knowledge of the pathogenesis of urticaria. CrossrefMedlineGoogle Scholar27. KELLYPATTERSONLIEBERMANMATHISONSTEVENSON JRPDD: Radiographic contrast media studies in highrisk patients. J Allergy Clin Immunol 62:181-184, 1978 Prospective but uncontrolled evaluation of diphenhydramine and prednisone in preventing anaphylactoid reactions in patients with a previous reaction. CrossrefMedlineGoogle Scholar28. LEVINSONSUMMERSLAWLEYEVANSFRANK ARTRM: Evaluation of the adverse effects of long-term hyposensitization. J Allergy Clin Immunol 62:109-114, 1978 A negative study, but the first systematic investigation of potential adverse effects of long-term immunotherapy. Immune complexes were absent in serum. CrossrefMedlineGoogle Scholar29. LICHTENSTEIN L: An evaluation of the role of immunotherapy in asthma. Am Rev Resp Dis 117:191-195, 1978 A critical review. MedlineGoogle Scholar30. LORENCHAIMIKLICHBARWISE MHDG: Comparison between simple nebulization and intermittent positive-pressure in asthmatic children with severe bronchospasm. Chest 72:145-147, 1977 No advantage was found for intermittent positive-pressure breathing in delivering bronchodilator aerosols, indicating that this common practice is a gross waste of expensive equipment. CrossrefMedlineGoogle Scholar31. MATHEWS K: Chronic urticaria revisited. J Allergy Clin Immunol 61:347-349, 1978 A concise editorial review of the pathogenesis of this common but poorly understood syndrome. CrossrefMedlineGoogle Scholar32. MINORDICKBAKEROUELLETTECOHENREED TEJJMC: Rhinovirus and influenza type A infections as precipitants of asthma. Am Rev Respir Dis 113:149-153, 1976 Infections with rhinoviruses, influenza A, and respiratory syncytial virus were noted to be strongly associated with flare-ups of asthma. MedlineGoogle Scholar33. NORMANLICHTENSTEIN PL: The clinical and immunologic specificity of immunotherapy. J Allergy Clin Immunol 61:370-377, 1978 A reconfirmation of the specificity of immunotherapy. CrossrefMedlineGoogle Scholar34. SCHATZPATTERSONKLONERFALK MRRJ: The prevalence of tuberculosis and positive tuberculin skin tests in a steroid-treated asthmatic population. Ann Intern Med 84:261-265, 1976 No evidence of tuberculosis was found in 132 corticosteroidtreated asthmatics. The authors conclude that routine isoniazid prophylaxis, regardless of tuberculin sensitivity, is not indicated. LinkGoogle Scholar35. SCOGGINSAHNPETTY CST: Status asthmaticus. A nine-year experience. JAMA 238:1158-1162, 1977 A retrospective analysis that also points out the hazards of mechanical ventilation. CrossrefMedlineGoogle Scholar36. SOTER N: Chronic urticaria as a manifestation of necrotizing venulitis. N Engl J Med 296:1440-1442, 1977 New insight into the relation between chronic urticaria and vasculitic syndromes and a brief review of related investigations. CrossrefMedlineGoogle Scholar37. STRAUSSMCFADDENINGRAMJAEGER RERJ: Enhancement of exercise-induced asthma by cold air. N Engl J Med 297:743-747, 1977 A clever experimental design shows the interaction of two common stimuli in producing asthmatic attacks. CrossrefMedlineGoogle Scholar38. TAYLOROHMANLOWELL WJF: Immunotherapy in cat-induced asthma. J Allergy Clin Immunol 61:283-287, 1978 A well-controlled study showing efficacy of treatment with cat antigen against inhalational challenge. CrossrefMedlineGoogle Scholar39. AGNELLO V: Association of systemic lupus erythematosus and SLE-like syndromes with hereditary and acquired complement deficiencies states. Arthritis Rheum (suppl) 21:S146-152, 1978 Review of the prominent association of autoimmune symptoms and inherited complement protein deficiencies. MedlineGoogle Scholar40. HONGSANTOSHAMSCHULTE-WISSERMANHOROWITZHSUWINKELSTEIN RMHSSJ: Reconstitution of B and T lymphocyte function in severe combined immunodeficiency disease after transplantation with thymic epithelium. Lancet 2:1270-1272, 1976 The success of thymic epithelial transplant in completely restoring immune function indicates that a thymic rather than a lymphoid stem cell defect was responsible for severe combined immunodeficiency in a 10-month-old girl with normal adenosine deaminase. CrossrefMedlineGoogle Scholar41. LAVINKIDSON MC: Lymphocytes in ataxia telangiectasia: implications of the DNA repair defect. Clin Immunol lmmunopathol 10:325-329, 1978 Defective DNA repair occurs on a genetic basis in ataxia telangiectasia and may cause the immunologic and central nervous system abnormalities and the increased malignancy associated with the syndrome. CrossrefMedlineGoogle Scholar42. POLMARSTERNSCHWARTZWETZLERCHASEHIRSCHHORN SRAEPR: Enzyme replacement therapy for adenosine deaminase deficiency and severe combined immunodeficiency. N Engl J Med 295:1337-1343, 1976 Treatment of an infant with adenosine deaminase deficiency and the autosomal recessive type of severe combined immunodeficiency disease with frozen, irradiated erythrocytes restored in-vitro lymphocyte responses. CrossrefMedlineGoogle Scholar43. VOGLERPEARLGATHINGSLAWTONCOOPER LEWAM: B lymphocyte precursors in bone-marrow in immunoglobulin deficiency diseases (letter). Lancet 2:376, 1976 The defect in X-linked hypoimmunoglobulinemia is postulated to be at the pre-B-lymphocyte stage of differentiation, based on the presence of lymphocytes with cytoplasmic IgM and not surface IgM. CrossrefMedlineGoogle Scholar44. WALDMANNBLAESEBRODERKRAKAUER TRSR: Disorders of suppressor immunoregulatory cells in the pathogenesis of immunodeficiency and autoimmunity. Ann Intern Med 88:226-238, 1978 Review of the evidence for excessive suppressor cell activity in the pathogenesis of immunoglobulin deficiency syndromes and for a decrease in suppressor cell activity in autoimmune diseases. LinkGoogle Scholar45. WILFERTBUCKLEYMOHANAKUMARGRIFFITHKATZWHISNANTEGGLESTONMOORETREADWELLOXMANROSEN CRTJSJPMEMF: Persistant and fatal central-nervous-system ECHOvirus infections in patients with agammaglobulinemia. N Engl J Med 296:1485-1489, 1977 Three of five patients with hypoimmunoglobulinemia and intact T cell response had a dermatomyositis-like syndrome in association with chronic central nervous system ECHO virus infection. Intact B cell function appears to be important in the elimination of central nervous system enterovirus infection. CrossrefMedlineGoogle Scholar46. BROWNSOLOMONBEALLTERASAKICHOPRAVAN HERLEWU JDGPIAS: Autoimmune thyroid diseases—Graves' and Hashimoto's. Ann Intern Med 88:379-391, 1978 Review of the pathogenic, immunologic, clinical, genetic, and therapeutic aspects of autoimmune thyroid diseases. LinkGoogle Scholar47. BUDMANSTEINBERG DA: Hematologic aspects of systemic lupus erythematosus. Current concepts. Ann Intern Med 86:220-229, 1977 Review of the numerous hematologic abnormalities seen in systemic lupus erythematosus. LinkGoogle Scholar48. DECKERSTEINBERGGERSHWINSEAMANKLIPPELPLOTZPAGET JAMWJPS: Systemic lupus erythematosus. Contrasts and comparisons. Ann Intern Med 82:391-404, 1975 A detailed discussion of possible pathogenic mechanisms, clinical features, and therapeutic approaches to systemic lupus erythematosus. LinkGoogle Scholar49. DRACHMAN D: Myasthenia gravis. N Engl J Med 298:136-142, 186-193, 1978 In-depth review of myasthenia gravis. CrossrefMedlineGoogle Scholar50. FAUCISTEINBERGHAYNESWHALEN AABG: Immunoregulatory abnormalities in systemic lupus erythematosus. J Immunol 121:1473-1479, 1978 A study and discussion of immunoregulatory abnormalities in 37 patients with systemic lupus erythematosus. MedlineGoogle Scholar51. FAUCIWOLFF AS: Wegener's granulomatosis and related disorders. DM 23:1-36, 1977 A clinicopathologic review of Wegener's granulomatosis and other related granulomatous vasculitides. CrossrefMedlineGoogle Scholar52. FAUCIHAYNESKATZ ABP: The spectrum of vasculitis. Clinical, pathologic, immunologic, and therapeutic considerations. Ann Intern Med 89:660-676, 1978 An extensive review of the clinical features and immunopathogenic mechanisms of the spectrum of the vasculitides is given. Certain modifications of classifications are proposed and outlines for therapeutic approaches to the different disorders are given. LinkGoogle Scholar53. MCDUFFIE F: Immune complexes in the rheumatic diseases. J Allergy Clin Immunol 62:37-43, 1978 A review of the immunopathogenic role of immune complexes in the rheumatic diseases, particularly systemic lupus erythematosus and rheumatoid arthritis. CrossrefMedlineGoogle Scholar54. MOUTSOPOULOSCHUSEDJOHNSONKNUDSENMANN HTABD: B lymphocyte antigens in sicca syndrome. Science 199:1441-1442, 1978 A study of the immunogenetic aspects of Sjögren's syndrome. CrossrefMedlineGoogle Scholar55. SILVERMANJOHNSONVAUGHANMCGLAMORY HJJJ: Altered lymphocyte reactivity in rheumatoid arthritis. Arthritis Rheum 19:509-515, 1976 Discussion of the lymphocyte abnormalities in rheumatoid arthritis. CrossrefMedlineGoogle Scholar56. WAGNER L: Immunosuppressive agents in lupus nephritis: a critical analysis. Medicine (Baltimore) 55:239-250, 1976 A detailed, critical review of therapeutic results using immunosuppressive regimens in systemic lupus erythematosus. CrossrefMedlineGoogle Scholar57. FAUCI A: Alternate-day corticosteroid therapy. Am J Med 64:729-731, 1978 Discussion of the advantages and disadvantages of alternate-day corticosteroid therapy. CrossrefMedlineGoogle Scholar58. FAUCIDALEBALOW ADJ: Glucocorticosteroid therapy: mechanisms of action and clinical considerations. Ann Intern Med 84:304-315, 1976 Review of the clinically relevant mechanisms of action of corticosteroids, particularly with regard to the diverse effects on circulating leukocytes. LinkGoogle Scholar59. GERSHWINGOETZLSTEINBERG MEA: Cyclophosphamide: use in practice. Ann Intern Med 80:531-540, 1974 Still current review of the mechanisms of action and clinical uses of cyclophosphamide. LinkGoogle Scholar60. LENTZBERGSTEINSTEFFESBROWNPREMMICHAELVERNIER RJMDKAR: Postpuberal evaluation of gonadal function following cyclophosphamide therapy before and during puberty. J Pediatr 91:385-394, 1977 Review of aspects of gonadal dysfunction encountered with chronic cyclophosphamide therapy. CrossrefMedlineGoogle Scholar61. OTTERNESSCHANG IY: Comparative study of cyclophosphamide, 6-mercaptopurine, azathioprine, and methotrexate. Relative effects on the humoral and the cellular immune response in the mouse. Clin Exp Immunol 26:346-354, 1976 Data on the relative effects of various cytotoxic agents on humoral versus cellular immune responses. MedlineGoogle Scholar62. SCHEINWINOKUR PS: Immunosuppressive and cytotoxic chemotherapy: long-term complications. Ann Intern Med 82:84-95, 1975 Review and discussion of the complications associated with commonly used cytotoxic agents. LinkGoogle Scholar63. TAUBDEUTSCH RV: Antilymphocytic serum. Pharmacol Ther(B) 28:89-111, 1977 Extensive review of the actions and clinical uses of antilymphocytic serum. Google Scholar64. ARNDTJICK KH: Rates of cutaneous reactions to drugs. A report from the Boston Collaborative Drug Surveillance Program. JAMA 235:918-923, 1976 Drug-specific quantitative data that can be used to evaluate the causes of drug-induced cutaneous lesions. CrossrefMedlineGoogle Scholar65. FELLNER M: Penicillin allergy 1976: a review of reactions, detection and current management. Int J Dermatol 15:497-504, 1976 Review of the state of the art. CrossrefMedlineGoogle Scholar66. GIRARDCATTINCUEVAS JSM: Immunological mechanisms and diagnostic tests in allergic drug reactions. Ann Clin Res 8:74-84, 1976 Preview of mechanisms of drug allergy, including an evaluation of currently available diagnostic tests. MedlineGoogle Scholar67. GREENROSENBLUMSWEET GAL: Evaluation of penicillin hypersensitivity: value of clinical history and skin testing with penicilloyl-polylysine and penicillin G. A cooperative prospective study of the penicillin study group of the American Academy of Allergy. J Allergy Clin Immunol 60:339-345, 1977 Cooperative, multicenter study in over 1000 patients of usefulness of penicillin skin testing in diagnosis of penicillin allergy. CrossrefMedlineGoogle Scholar68. PARKER C: Drug allergy. N Engl J Med 292:511-514, 732-736, 957-960, 1975 Review of causes, pathogenesis, diagnostic measures, and treatment of drug allergy. CrossrefMedlineGoogle Scholar69. ALLENBASTENWILLIAMSWOOLCOCK DAGA: Familial hypersensitivity pneumonitis. Am J Med 59:505-514, 1975 Seven of 19 members of two families had hypersensitivity pneumonitis. In the first family, both paternal haplotypes were associated with disease; in the second, disease developed in three of four members with one of the haplotypes. CrossrefMedlineGoogle Scholar70. FINKBANASZAKBARBORIAKHENSLEYKURUPSCANLONSCHLUETERSOSMANTHIEDEUNGER JEJGVGDAWG: Interstital lung disease due to contamination of forced air systems. Ann Intern Med 84:406-413, 1976 Eight patients with hypersensitivity pneumonitis developed illness related to contaminated forced-air heating or air conditioning systems. These patients probably represent a larger, undiagnosed population. LinkGoogle Scholar71. REYNOLDSFULMERKAZMIEROWSKIROBERTSFRANKCRYSTAL HJJWMR: Analysis of cellular and protein content of broncho-alveolar lavage fluid from patients with idiopathic pulmonary fibrosis and chronic hypersensitivity pneumonitis. J Clin Invest 59:165-175, 1977 Lavage fluid from patients with chronic hypersensitivity pneumonitis contains more IgM, fewer inflammatory cells, and a strikingly increased number of T lymphocytes when compared to fluids from patients with idiopathic fibrosis. CrossrefMedlineGoogle Scholar72. BILLINGHAM R: The feasibility of altering the immunogenicity of grafts. J Invest Dermatol 67:149-159, 1976 An approach to the problem of organ transplant rejection aims at altering antigenicity of graft rather than suppressing host response. CrossrefMedlineGoogle Scholar73. DOSSETORSINCLAIRSTILLER JNC: The genetics of HLA. Transplant Proc 10:309-312, 1978 Review of histocompatibility leukocyte antigen system, nomenclature, and genetics. MedlineGoogle Scholar74. LONDONDREWBLUMBERGGROSSMANLYONS WJBRP: Association of graft survival with host response to hepatitis B infection in patients with kidney transplants. N Engl J Med 296:241-244, 1977 Renal graft survival correlated with host response to hepatitis B infection. Antibody to hepatitis B surface antigen signaled early rejection, whereas hepatitis B surface antigen was associated with delayed rejection. CrossrefMedlineGoogle Scholar75. PETERSONOLSENKISSMEYER-NIELSENBOHMANHANSENHANSENSKOVSØLLING VTFSHEPK: Late failure of human renal transplants. An analysis of transplant disease and graft failure among 125 recipients surviving for one to eight years. Medicine (Baltimore) 54:45-71, 1975 Review of 125 cases featuring clinical, morphologic, and immunologic aspects of late rejection of renal allografts. CrossrefMedlineGoogle Scholar76. SCHALLEROMENN JG: The histocompatibility system and human disease. J Pediatr 88:913-925, 1976 Review of histocompatibility leukocyte antigen system and clinical disease associations; shows that histocompatibility leukocyte antigen typing is not diagnostic. CrossrefMedlineGoogle Scholar77. STORBPRENTICETHOMAS RRE: Marrow transplantation for treatment of aplastic anemia. An analysis of factors associated with graft rejection. N Engl J Med 296:61-66, 1977 Experience with 73 bone marrow transplants; analyses pretreatment of presensitized versus sensitized patients and correlates regimen with outcome. CrossrefMedlineGoogle Scholar78. THORSBYALBRECHTSENBERGHOLTZHIRSCHBERGSOLHEIM EDBHB: Identification and significance of products of the HLA-D region. Transplant Proc 10:313-318, 1978 Review of experimental and human data on the significance of the D histocompatibility leukocyte antigen (HLA-D) region to graft rejection. MedlineGoogle Scholar79. BRODEREDELSONLUTZNERNELSONMACDERMOTTDURMGOLDMANMEADEWALDMANN SRMDRMCBT: The Sézary syndrome. A malignant proliferation of helper T cells. J Clin Invest 58:1297-1306, 1976 Leukemic cells from patients with Sézary syndrome enhanced immunoglobulin production in vitro by normal B cells. They did not suppress immunoglobulin synthesis. Conclusion is that Sézary cells represent proliferation of cells programmed to function as helper T cells. CrossrefMedlineGoogle Scholar80. BRODERPOPLACKWHANG-PENGDURMGOLDMANMUULWALDMANN SDJMCLT: Characterization of a suppressor-cell leukemia. Evidence for the requirement of an interaction of two T cells in the development of human suppressor effector cells. N Engl J Med 298:66-72, 1978 In a patient with acute lymphoblastic leukemia neoplastic T cell plus normal T cell was required to suppress immunoglobulin production in vitro by normal or patient B cells. CrossrefMedlineGoogle Scholar81. Go V: Carcinoembryonic antigen. Clinical application. Cancer 37(suppl 1): 562-566, 1976 Reviews state of the art of carcinoembryonic antigen measurement and its use in clinical medicine. CrossrefMedlineGoogle Scholar82. GOODWINMESSNERBANKHURSTPEAKESAIKIWILLIAMS JRAGJR: Prostaglandin-producing suppressor cells in Hodgkin's disease. N Engl J Med 297:963-968, 1977 Macrophages may play suppressor role in the depressed cellularimmunity in patients with Hodgkin's disease. Macrophages produce prostaglandin E2, which suppresses in-vitro lymphocyte responses. CrossrefMedlineGoogle Scholar83. HERBERMAN R: Immunologic tests in the diagnosis of cancer. Am J Clin Pathol 68(suppl 5): 688-698, 1977 Review of immunodiagnosis of cancer focuses in-vitro immunologic assays and tumor-associated markers in the discrimination of neoplastic from nonneoplastic disease. MedlineGoogle Scholar84. MACKENZIEPAGLIERONI MT: Multiple myeloma: an immunologic profile. I. Peripheral blood studies. J Immunol 118:1864-1871, 1977 Discussion of lymphocyte function and surface markers or cells obtained from the blood of 74 patients with multiple myeloma (treated and untreated). MedlineGoogle Scholar85. WHITESIDEROWLANDS TD: T-cell and B-cell identification in the diagnosis of lymphoproliferative disease. A review. Am J Pathol 88:754-792, 1977 Review of markers present on T and B cells from patients with lymphoproliferative diseases. Correlates clinical outcome with T or B cell origin and certain markers. MedlineGoogle Scholar86. CHEWZIADYRAPHAELNELLENOAKLEY CGMMC: Primary restrictive cardiomyopathy. Non-tropical endomyocardial fibrosis and hypereosinophilic heart disease. Br Heart J 39:399-413, 1977 Study of 11 patients with restrictive cardiomyopathy and hypereosinophilia. CrossrefMedlineGoogle Scholar87. DALEHUBERTFAUCI DRA: Eosinophil kinetics in the hypereosinophilic syndrome. J Lab Clin Med 87:487-495, 1976 Study of the kinetics and circulating half-life of human eosinophils. MedlineGoogle Scholar88. GLEICH G: The eosinophil: new aspects of structure and function. J Allergy Clin Immunol 60:73-82, 1977 Detailed description of various structural and functional properties of eosinophils. CrossrefMedlineGoogle Scholar89. GOETZL E: Modulation of human eosinophil polymorphonuclear leukocyte migration and function. Am J Pathol 85:419-435, 1976 In-depth discussion of the factors regulating human eosinophil function. MedlineGoogle Scholar90. PARRILLOFAUCI JA: Human eosinophils. Purification and cytotoxic capability of eosinophils from patients with the hypereosinophilic syndrome. Blood 51:457-473, 1978 Description of the surface markers and several of the functional capabilities of human eosinophils. CrossrefMedlineGoogle Scholar91. PARRILLOFAUCIWOLFF JAS: Therapy of the hypereosinophilic syndrome. Ann Intern Med 89:167-172, 1978 Review of the literature on therapy of hypereosinophilic syndrome with a description of therapeutic responses to corticosteroids or hydroxyurea, or both, in 26 patients. LinkGoogle Scholar92. ZWEIMANSLOTTATKINS BRP: Factors in the tissue eosinophil responses in human immediate hypersensitivity reactions. Int Arch Allergy Appl Immunol 52:48-52, 1976 Discussion of the multiple factors in the migration of eosinophils into tissues in humans. CrossrefMedlineGoogle Scholar93. KELLYPATTERSONLIEBERMANMATHISONSTEVENSON JRPDD: Radiographic contrast media studies in high risk patients. J Allergy Clin Immunol 62:180-184, 1978 A double-blind controlled study of efficacy of pretreatment of patients at risk of reactions to intravenous contrast material with benadryl and prednisone; documents the value of pretreatment. CrossrefGoogle Scholar94. LIEBERMANSIEGLETAYLOR PRW: Anaphylactoid reactions to iodinated contrast material. J Allergy Clin Immunol 62:174-180, 1978 An in-depth review of the problem of untoward reactions to iodinated contrast material. CrossrefMedlineGoogle Scholar95. WITTERSOHMANWEIRRAYMONDLOWELL LJGLF: Insulin antibodies in the pathogenesis of insulin allergy and resistance. Am J Med 63:703-709, 1977 Discussion of the mechanism of insulin allergy and resistance and the interplay between the two. CrossrefMedlineGoogle Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: AMERICAN COLLEGE OF PHYSICIANS Philadelphia, Pennsylvania*Anthony S. Fauci, M.D., Chairman; William Franklin, M.D.; Fred S. Kantor, M.D.; Peter F. Kohler, M.D.; Phillip L. Lieberman, M.D.; and Ross E. Rocklin, M.D.†Computer literature-file of the National Library of Medicine, Bethesda, Maryland, consulted through the Regional Medical Library service Library of the College of Physicians of Philadelphia, with the technical assistance of Ruthanne Henner. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics 1 January 1979Volume 90, Issue 1Page: 135-139KeywordsAllergy and immunologyComputersLibraries ePublished: 1 December 2008 Issue Published: 1 January 1979 PDF downloadLoading ...