tbuckley/cabot-search huggingface dataset:
Simplified Diabetic Manual With 163 International Recipes (American, Jewish, French, German, Italian, Armenian, etc.)
Journal: JAMA (1941)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
DIFFERENTIATION IN DIABETES.
Journal: JAMA (1900)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Books Received
Journal: JNCI Journal of the National Cancer Institute (1966)

Books Received Get access Basic physiology and anatomy. By Norman B. Taylor. 648 p., G. P. Putnam's Sons, 200 Madison Avenue, New York, N.Y., 1965. Text-aidtm for basic physiology and anatomy. By Richard H. Miller. 182 p., G. P. Putnam's Sons, 200 Madison Avenue, New York, N.Y., 1965. Biochemical approaches to cancer. By Eric Reid. 198 p., $3.95. Pergamon Press Inc.44-01, 21st Street, Long Island City, New York 11101, 1965. Management of the diabetic patient. By Elaine P. Ralli. 207 p., $7.00. G. P. Putnam's Sons, 200 Madison Avenue, New York, N.Y., 1965. La tubercolosi polmonare. By Fortunio G., Carlizza L., and Beggi D.. Edizioni “Omnia Medica,” via S. Michele degli Scazzi, N. 73, Pisa, Italy, 146 p., Price: Lire 1.800, 1965. Hospital policy decisions: Process & action. By Arthur B. Moss, Wayne G. BroehlJr., Robert H. Guest, and John W. HennesseyJr., 332 p., $8.50. G. P. Putnam's Sons, 200 Madison Avenue, New York, N.Y., 1966. Primary hepatoma. Edited by Walter J. Burdette. 150 p., University of Utah Press, Press Road and Campus Drive, Bldg. 301, Salt Lake City, Utah 84112, 1965. Canadian Cancer Conference: Proceedings of the Sixth Canadian Cancer Conference, Honey Harbour, Ontario, 1964. Edited by Begg W.R., Leblond P.C., Noble L.R., Rossiter J.R., Taylor M.R., and Wallace C.A., 525 p., $15.00. Pergamon Press, 44-01, 21st Street, Long Island City, New York 11101, 1966. Il problema della coscienza in neuropsichiatria. By Raffaello Vizioli and Clelia Bietti. 216 p., Price: Lire 3.000. Edizioni “Omnia Medica,” via S. Michele degli Scazzi, N. 73, Pisa, Italy, 1966. The Swedish Cancer Society Year Book 1963–1965. Edited by Hilding Bergstrand. 509 p., Almquist & Wiksell, Stockholm, Sweden, 1966. JNCI: Journal of the National Cancer Institute, Volume 37, Issue 2, August 1966, Page 255, https://doi.org/10.1093/jnci/37.2.255 Published: 01 August 1966
Thrombomodulin and induced tissue factor expression on monocytes as markers of diabetic microangiopathy: A prospective study on hemostasis and lipoproteins in insulin‐dependent diabetes mellitus
Authors: Jordi L. Reverter, Joan C. Reverter, Dolors Tàssies, Ferran Rius, Joan Monteagudo, Joan Rubiés‐Prat, Ginés Escolar, Antoni Ordinas, Anna Sanmartí
Journal: American Journal of Hematology (1997)

Vascular complications are the main cause of morbidity in diabetes mellitus. To evaluate lipoprotein and hemostatic parameters and their relationship with clinically detectable microangiopathy, we studied 58 insulin-dependent diabetes mellitus patients and 60 controls matched for age, sex, and body mass index. Thirteen patients presented clinically detectable microangiopathy (8 retinopathy and 5 both retinopathy and microalbuminuria). A cross-sectional study of lipid profile, coagulation parameters, and a flow-cytometric evaluation of tissue factor expression in normal monocytes induced by patient plasma were performed. Patients were re-evaluated for microangiopathy in a 3-year median follow-up. Patients showed triglyceride enrichment in low (P = 0.00002) and high density lipoproteins (P = 0.004) and increased levels of D-dimer (P < 0.00001), prothrombin fragment 1 + 2 (P < 0.00001), and thrombin-antithrombin III complex (P = 0.0001). Patients with clinically detectable microangiopathy had increased type 1 plasminogen activator inhibitor (P = 0.00001), thrombomodulin (P = 0.02), and induced monocyte tissue factor expression (P < 0.00001). Nine patients developed clinically detectable microangiopathy in the follow-up and the only predictive variable was increased induced tissue factor expression. In conclusion, in these patients elevated thrombin and fibrin generation reflects a hypercoagulable state but clinically detectable microangiopathy seems related to endothelial cell injury markers and to increased induced tissue factor expression on monocytes. Am. J. Hematol. 56:93–99, 1997. © 1997 Wiley-Liss, Inc.
The Caregiving Environments Provided to Children by Depressed Mothers With or Without an Antisocial History
Authors: Julia Kim-Cohen, Avshalom Caspi, Michael Rutter, Mónica Polo Tomás, Terrie E. Moffitt
Journal: American Journal of Psychiatry (2006)

Objective: Many depressed women have a history of antisocial behavior, but research into maternal depression has not ascertained if this has implications for children of depressed mothers. This study compared the developmental outcomes in and caregiving environments provided to children by depressed mothers with or without an antisocial history. Method: In the Environmental Risk Longitudinal Twin Study, a nationally representative study of 1,106 families, mothers were administered the Diagnostic Interview Schedule for Major Depressive Disorder and interviewed about their lifetime history of antisocial personality disorder symptoms. Mothers and teachers provided information regarding the children's behavior problems at 5 and 7 years of age. The authors assessed the quality of the caregiving environment through maternal reports and interviewer observations. Results: Compared with children of mothers with depression only, the children of depressed and antisocial mothers had significantly higher levels of antisocial behavior and rates of DSM-IV conduct disorder, even after the authors controlled for numbers of symptoms and chronicity of maternal major depressive disorder. The children of depressed and antisocial mothers were at an elevated risk of experiencing multiple caregiving abuses, including physical maltreatment, high levels of maternal hostility, and exposure to domestic violence. Conclusions: If one ignores the common co-occurrence of an antisocial history in depressed mothers, it may obscure the significantly elevated risks in children's development. Clinicians treating women's depression should be aware that children of depressed and antisocial mothers constitute a group at extremely high risk for early-onset psychopathology.
Congenital Adrenal Hyperplasia Secondary to 17-Hydroxylase Deficiency Presenting as Amenorrhea, Hypokalemia, Hypertension, and Cystic Ovaries.
Authors: Sanford R. Mallin
Journal: Annals of Internal Medicine (1968)

s1 May 1968Congenital Adrenal Hyperplasia Secondary to 17-Hydroxylase Deficiency Presenting as Amenorrhea, Hypokalemia, Hypertension, and Cystic Ovaries.Sanford R. Mallin, M.D. (Associate)Sanford R. Mallin, M.D. (Associate)Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-68-5-1144_2 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptTwo sisters, 16 and 17 years old, were seen initially because of primary amenorrhea and absence of sexual development. In a subsequent 18-month period each girl presented with an acute surgical abdomen, and a hemorrhagic twisted cystic ovary was removed from each girl. At the time of surgery in each case, the "unaffected" ovary was enlarged, firm, polycystic, and sclerotic, and a biopsy showed diminished ovarian stroma with occasional primordial follicles and small follicular cysts but no further maturation. The younger sister subsequently developed a second acute abdomen, and her remaining ovary showed identical twisted, hemorrhagic cysts and was removed.... This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAuthors: Sanford R. Mallin, M.D. (Associate)Affiliations: Milwaukee, Wisconsin PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics 1 May 1968Volume 68, Issue 5Page: 1144-1144KeywordsBiopsyCongenital adrenal hyperplasiaHypertensionOvariesSurgery ePublished: 1 December 2008 Issue Published: 1 May 1968 PDF downloadLoading ...
Specimen Says
Authors: Julie Dunlop
Journal: JAMA (2013)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Abstract P186: IL-10 Mitigates Adverse Cardiac Remodeling and Improves Left Ventricle Function in Pressure Overload--Induced Hypertrophy
Authors: Suresh K Verma, Prasanna Krishnamurthy, Neha Senger, Melissa Thal, Eneda Hoxha, Erin Lambers, Tina Thorne, Gangjian Qin, Douglas W Losordo, Raj Kishore
Journal: Circulation Research (2011)

Background: Hypertrophy associated heart failure remain the leading cause of death worldwide. Inflammatory cytokines play critical role in adverse cardiac remodeling and heart failure. Therefore, approaches geared towards inhibiting cardiac inflammation may provide therapeutic benefits. Thus, we tested the hypothesis that anti-inflammatory cytokine, IL-10, therapy might inhibit pressure overload-induced cardiac remodeling. Methods and Results: Cardiac hypertrophy was induced in Wild-type (WT) and IL-10-knockout (KO) mice by isoproterenol (ISO; 45mg/kg b.wt./day, 14 days) infusion through mini-osmotic pumps. Control groups mice received saline. ISO-induced increase in heart weight/tibia length ratio in WT was further increased in KO mice. ISO-mediated cardiac dysfunction (%EF and %FS) in WT mice were exaggerated in KO mice. KO mice displayed higher expression of fetal genes (ANP and BNP) and inflammatory cytokines (TNFα and IL-1β). ISO-treated KO mice had notably higher fibrosis and apoptosis than the WT. Interestingly, systemic recombinant IL-10 administration markedly attenuated effects of ISO and improved left ventricle function, reversed fetal gene expression, fibrosis and apoptosis both in WT and in KO mice. To further understand the mechanism of IL-10 protection, NRCM and H9C2 cells were treated with ISO (10µM) and/or IL-10 (20ng/mL), in vitro . ISO significantly increased the mRNA expression of ANP and BNP as well as of inflammatory cytokines which was markedly reduced by IL-10. ISO treatment also induced the activation of p38, ERK1/2 MAP kinases and of NFkB while it inhibited STAT-3 phosphorylation. Interestingly, co-treatment with IL-10 suppressed p38 and ERK1/2 phosphorylation while enhancing STAT-3 phosphorylation. Interestingly, ISO-induced nuclear translocation of NFkB was mimicked by STAT3 specific inhibitor cucurbitacin, suggesting STAT3 as the downstream target of IL-10 effects. Conclusion: Our studies suggest that IL-10 treatment not only inhibits the progression but also reverses the pressure overload-induced adverse cardiac remodeling. Ongoing investigations will further provide a better understanding on the mechanistic and therapeutic aspects of IL-10 on hypertrophic remodeling and heart failure.
Author Corrections
Authors: Stanley Leong, David Byrd, Alistair Cochran, Alexander Eggermont, Kelly Mcmasters, Martin Mihm, Keith Flaherty, Donald Morton, K Sondak, H Lee
Journal: Journal of Clinical Oncology (2011)

In the sidebar and in the last sentence of the Introduction section, the QUASAR trial number was given as ISRCTN82375366, whereas it should have been given as ISRCTN82375386.The following acknowledgement was inadvertently omitted: "WethankEmmaTinkler-Hundalforhersignificantcontributionsto the preparation of clinical trial material used within the study."
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Journal: JAMA (1992)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Journals JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2024 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Marine -3 Polyunsaturated Fatty Acid Intake is Associated with Lower Risk of MSI-High CRC
Authors: Asad Umar, Ellen Richmond, Barnett Kramer, Andrew Chan
Journal: JNCI Journal of the National Cancer Institute (2015)

Marine ϖ-3 Polyunsaturated Fatty Acid Intake is Associated with Lower Risk of MSI-High CRCHigh intake of marine ϖ-3 polyunsaturated fatty acids (PUFAs) is associated with lower risk of microsatellite instable (MSI) colorectal cancers (CRCs) but not microsatellite stable (MSS) CRCs, according to a new study published March 25 in the JNCI: Journal of the National Cancer Institute.Both development of CRC and microsatellite instability (MSI) are associated with chronic inflammation.Marine ϖ-3 PUFAs have anti-inflammatory and antineoplastic activity, but the evidence for a role of marine ϖ-3 PUFAs in CRC has been inconsistent.To investigate the relationship between marine ϖ-3 PUFAs by MSI status, Mingyang Song, Ph.D., from the Departments of Nutrition and Epidemiology at the Harvard T.H. Chan School of Public Health in Boston, MA, and colleagues, analyzed data from 1125 CRC cases from the Nurses' Health Study and Health Professionals Follow-up Study.Although dietary ϖ-3 PUFA intake was not associated with CRC overall, high intake was associated with a lower risk of MSI-high tumors but not MSS tumors.The authors conclude, "Our findings generate hypotheses for the potential anticancer activity of ω-3 PUFAs and may have clinical implications for the potential of using marine ω-3 PUFAs in prevention of CRC.
Food "Poisoning" Due to Sodium Nicotinate: Report of an Outbreak and Review of the Literature
Journal: JAMA (1963)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Complications associated with implantable cardioverter defibrillator implantation: Single chamber versus atrioventricular devices Ralph J. Verdino, Andrea M. Russo, Dusan Z. Kocovic, Behzad B. Pavri, Henry H. Hsia, Vickas V. Patel, Manisha Ashar, David J. Callans, Francis E. Marchlinski. University of Pennsylvania Health System, Philadelphia, PA
Journal: Journal of the American College of Cardiology (2001)
Film Reviews
Journal: JAMA (1963)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Journals JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2024 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Definition and prognostic relevance of the salve grading and staging system for alcohol-related liver disease
Authors: Carolin Lackner, Dina Tiniakos, Annette Gouw, Peter Schirmacher, Valérie Paradis, Hans-Peter Dienes, Luigi Maria Terracciano, Davies Susan, Maria Guido, Viviane Gnemmi, Rosa Miguel, Rudolf E. Stauber, Helmut Denk
Journal: Journal of Hepatology (2020)
New guidelines for the management of adult lower respiratory tract infections
Authors: M. Woodhead
Journal: European Respiratory Journal (2011)

<b>1.</b> A 40-year-old man was admitted with acute hepato-renal failure after a paracetamol overdose. Blood pressure was consistently 170/100. He complained of headache and on day 6 developed sudden bilateral visual loss. Brain MRI (Fig. 1) showed increased FLAIR signal in the occipital and parietal lobes. Seizures then developed. After treatment his central vision recovered over 2 weeks, leaving a minor inferior quadrantinopia and residual contralateral parenchymal damage on repeat MRI (Fig. 2). What is the neurological syndrome? What is the treatment priority? Name five settings, or causes. <i>Andrew Chancellor, Tauranga, New Zealand</i> <b>2.</b> A late-middle-aged retired seaman gave a five and a half year history of progressive wasting and weakness. Six years earlier he had noticed distal leg weakness which had progressed rapidly, and six months later he had bilateral foot drop. Three years later he had reported progressive arm weakness. By the time of presentation he was confined
Letter: Hyperthyroid crisis
Authors: William L. Green
Journal: JAMA (1975)

<h3>To the Editor.—</h3> Retrospective analysis of reported cases of myxedema coma leads to the conclusion that vigorous therapy with thyroid hormones favors survival. To derive optimum dosage schedules, our approach, patterned after that of Holvey et al,<sup>1</sup>has been to estimate how much hormone would be needed to bring body hormone stores and plasma levels to normal, and then give somewhat less than this estimated total replacement.<sup>2</sup>Compared to this approach, Newmark et al (230:884, 1974) recommend doses of liothyronine sodium (50μg initially, then 25μg every eight hours) that are too large and doses of levothyroxine sodium (200μg intravenously or 250μg orally initially, then 50μg intravenously or 75μg orally every eight hours) that are too small. <h3>Treatment With Liothyronine.—</h3> The extrathyroidal pool of triiodothyronine (T<sub>3</sub>) in normal individuals is about 50μg. Thus, an initial dose of 50μg of liothyronine sodium would provide sudden total replacement, leading to