tbuckley/cabot-search huggingface dataset:
Not Needed
Authors: Donna Pucciani
Journal: JAMA (2009)

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2-deoxy-D-glucose encapsulated PLGA nanoparticles suppress hepatocellular carcinoma through cytotoxic effect and activation of antitumor immunity
Authors: Kyo Sasaki, Sohji Nishina, Akira Yamauchi, Kotaro Fukuda, Yuichi Hara, Keisuke Hino
Journal: Journal of Hepatology (2020)
Tenth Annual Symposium of the Lineberger Cancer Research Center: Damaged DNA—Its Structure and Recognition
Journal: JNCI Journal of the National Cancer Institute (1986)

Tenth Annual Symposium of the Lineberger Cancer Research Center: Damaged DNA—Its Structure and Recognition JNCI: Journal of the National Cancer Institute, Volume 76, Issue 2, February 1986, Page 361, https://doi.org/10.1093/jnci/76.2.361 Published: 01 February 1986
The Unipolar Electrocardiogram: A Clinical Interpretation.
Journal: JAMA (1952)

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Pernicious anemia in Indians
Authors: M. L. Sievers
Journal: JAMA (1970)

<h3>To the Editor.—</h3> In regard to the recently published letter on "Pernicious Anemia in a Full Blooded Indian" (<b>212</b>:1378, 1970) Dr. Richard V. Lee erroneously concluded that pernicious anemia among American Indians is infrequent. The basis for his opinion was that only one recent case report was discovered. In 1960, Hampton reported six full-blooded Indians with pernicious anemia and referred to five other cases in Indians in Oklahoma.<sup>1</sup>Because pernicious anemia is not an unusual event, since that date most physicians with extensive experience among Indians have not considered that separate case reports were warranted. During my 12 years as Chief of Medicine at the Public Health Service, Phoenix Indian Medical Center, I have observed at least 17 full-blooded American Indians from various tribes (mostly southwestern ones) with unequivocal pernicious anemia. I have one such patient on my ward at this time. A similar frequency of pernicious anemia
NONTHRIVING INFANTS— A FAMILY PROBLEM
Journal: JAMA (1966)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Response
Authors: R. L. STOLFI, L. M. STOLFI, R. C. SAWYER, D. S. MARTIN
Journal: JNCI Journal of the National Cancer Institute (1988)

Journal Article Response Get access ROBERT L. STOLFI, ROBERT L. STOLFI Department of Surgery-Research, Catholic Medical Center of Brooklyn and Queens, Inc.89-15 Woodhaven Blvd., Woodhaven, NY 11421 Search for other works by this author on: Oxford Academic PubMed Google Scholar LESLIE M. STOLFI, LESLIE M. STOLFI Department of Surgery-Research, Catholic Medical Center of Brooklyn and Queens, Inc.89-15 Woodhaven Blvd., Woodhaven, NY 11421 Search for other works by this author on: Oxford Academic PubMed Google Scholar ROBERT C. SAWYER, ROBERT C. SAWYER Department of Surgery-Research, Catholic Medical Center of Brooklyn and Queens, Inc.89-15 Woodhaven Blvd., Woodhaven, NY 11421 Search for other works by this author on: Oxford Academic PubMed Google Scholar DANIEL S. MARTIN DANIEL S. MARTIN Department of Surgery-Research, Catholic Medical Center of Brooklyn and Queens, Inc.89-15 Woodhaven Blvd., Woodhaven, NY 11421 Search for other works by this author on: Oxford Academic PubMed Google Scholar JNCI: Journal of the National Cancer Institute, Volume 80, Issue 10, 20 July 1988, Page 778, https://doi.org/10.1093/jnci/80.10.778-a Published: 20 July 1988
Abstract P155: Low Socioeconomic Status Relates to Short-term Mortality Risk After Acute Myocardial Infarction, Especially in Men: Results From a Nationwide Study
Authors: Louise van Oeffelen, Charles Agyemang, Carla Koopman, Michiel Bots, Karien Stronks, Ilonca Vaartjes
Journal: Circulation (2012)

Introduction Previous studies show poorer short-term prognosis after an acute myocardial infarction (AMI) in subjects with a low socioeconomic status (SES). Yet, the magnitude of these relations may differ by age and sex. Data on these issues are however scarce. Methods A nationwide Dutch cohort of first AMI patients between January 1 st 1998 and December 31 st 2007 was identified through linkage of national registers. SES was defined as the standardized disposable income on household level in 1997. For every SES quintile, age- and sex- specific short-term mortality rates were quantified. Logistic regression models were used to estimate differences between SES quintiles in out-of-hospital mortality and 28-day case-fatality. Results We identified 70.368 first AMI patients with income data available, of which 55.860 were men and 14.508 were women. There were strong inverse associations between SES and both short-term mortality outcomes when comparing the lowest with the highest income quintile (out-of-hospital mortality: Odds Ratio (OR) 1.26; 95% Confidence Interval (95% CI) 1.18–1.34), 28-day case-fatality: OR 1.26; 95% CI 1.15–1.37). For men graded relations were found across quintiles of SES, whereas for women only differences between the lowest and the highest quintile were seen. These relations remained consistent across all age categories, except for women below 55 years of age. Conclusion The results from our nationwide study show an increased risk of short-term mortality after a first AMI in subjects with a low SES of all ages, which is most pronounced in men.
Effectiveness of antibacterial soaps in a healthy population
Authors: A. R. MacKenzie
Journal: JAMA (1970)

An antibacterial bar soap containing 1.5% of a mixture of hexachlorophene and triclocarban was used exclusively during a six-month period at the US Naval Academy to determine its ability to reduce the incidence of superficial cutaneous bacterial infections. Comparison was made with a bar soap identical, except in color, that did not contain the antibacterial ingredients. The incidence of superficial cutaneous bacterial infections was decreased a statistically significant 44% in the group using the antibacterial soap. No instances of irritation, photosensitivity, or other untoward reactions were noted among the 1,201 users of either bar soap.
ECMO/PPHN
Authors: G. A. Pearson, D. Macrae, Y. Merzel, A. Lev, Yosef G. Bar, M. Halbertal, A. Lorber, P. E. Sigston, A. P. Goldman, J. Keating, R. Crook, D. J. Macrae, J. N. van den Anker, R. C. Schoemaker, M. Vogel, W. Goessens, D. Tibboel, M. Schindler, E. Cutz, B. Duffy, A. C. Bryan
Journal: Intensive Care Medicine (1996)
AUTHORS' REPLY
Authors: A. Webb, D Bilton
Journal: Thorax (1991)

Relation of urinary cotinine concentrations to cigarette smoking and to exposure to other people's smoke Dr Simon Thompson and his colleagues measured urinary cotinine in 184 self repor- ted non-smokers (May 1990;45:356-61).Taking 10-30% of average smokers' concen- trations to indicate occasional smoking and over 30% to indicate regular smoking, they found two (1-1%) occasional smokers and no regular smokers among their non-smokers.
Aid for Pharmaceutical Research.
Journal: JAMA (1922)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Invasive fungal diseases in haematopoietic cell transplant recipients and in patients with acute myeloid leukaemia or myelodysplasia in Brazil
Authors: Marcio Nucci, Marcia Garnica, Ana B. Gloria, Denise S. Lehugeur, Viviane C. H. Dias, Leonardo C. Palma, Paola Cappellano, Kleber Y. Fertrin, Fabianne Carlesse, Belinda Simões, Maria D. Bergamasco, Clóvis A. Cunha, Adriana Seber, Maria P. D. Ribeiro, Flavio Queiroz-Telles, Maria L. M. Lee, Maria L. Chauffaille, Lucia Silla, Carmino A de Souza, Arnaldo L Colombo
Journal: Clinical Microbiology and Infection (2012)

<h2>Abstract</h2> Invasive fungal disease (IFD) shows distinct regional incidence patterns and epidemiological features depending on the geographic region. We conducted a prospective survey in eight centres in Brazil from May 2007 to July 2009. All haematopoietic cell transplant (HCT) recipients and patients with acute myeloid leukaemia (AML) or myelodysplasia (MDS) were followed from admission until 1 year (HCT) or end of consolidation therapy (AML/MDS). The 12-month cumulative incidence (CI) of proven or probable IFD was calculated, and curves were compared using the Grey test. Among 237 AML/MDS patients and 700 HCT recipients (378 allogeneic, 322 autologous), the 1-year CI of IFD in AML/MDS, allogeneic HCT and autologous HCT was 18.7%, 11.3% and 1.9% (p <0.001), respectively. Fusariosis (23 episodes), aspergillosis (20 episodes) and candidiasis (11 episodes) were the most frequent IFD. The 1-year CI of aspergillosis and fusariosis in AML/MDS, allogeneic HCT and autologous HCT were 13.4%, 2.3% and 0% (p <0.001), and 5.2%, 3.8% and 0.6% (p 0.01), respectively. The 6-week probability of survival was 53%, and was lower in cases of fusariosis (41%). We observed a high burden of IFD and a high incidence and mortality for fusariosis in this first multicentre epidemiological study of IFD in haematological patients in Brazil.
Environmental Lead Toxicity: Nutrition As a Component of Intervention
Authors: Kathryn R. Mahaffey
Journal: Environmental Health Perspectives (1990)

The influence of nutritional status on susceptibility to the toxicity of lead is discussed. Emphasis is given to dietary factors of substantial clinical importance. Subtle changes in susceptibility are difficult to evaluate under conditions of overwhelming lead exposure. It is clear that subtle effects of lead exposure on neurobehavioral and cognitive development are a major concern. The role of nutrition is considered to be an adjunct to reduction of environmental lead exposure, which is the primary means of reducing adverse health effects of lead. Nutrition should be evaluated as a component of strategies to address this broad societal issue.
Error in Acknowledgment.
Journal: JAMA (1993)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
ALCOHOLISM. A DISEASE.
Authors: A. ENFIELD
Journal: JAMA (1992)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Projects demonstrate how to involve physicians in care of workers at risk of health impairment
Authors: C. A. Raymond
Journal: JAMA (1988)

FROM 1980 TO 1984, the Washington, DC—based Workers' Institute for Safety and Health (a private nonprofit educational and technical resource center for labor unions), with funds from the National Cancer Institute, initiated three demonstration projects to assess means to identify, notify, screen, and educate workers exposed to various toxic substances (<i>J Occup Med</i>1986;28:719-727). Each project, which continues to monitor workers and collect data, elicited the cooperation and participation of community-based physicians. At a New York Academy of Sciences meeting on Occupational Health in the 1990s, one project investigator, Knut Ringen, DPH, discussed the projects in light of new national efforts to notify workers at risk, including national notification legislation recently defeated in the Senate, and an ongoing National Institute for Occupational Safety and Health (NIOSH) program to notify members of its cohort studies of occupational disease risks. The Augusta, Ga, project identified 1385 workers at risk for bladder cancer
Immunization status of children of employees in a large corporation
Authors: J. E. Fielding
Journal: JAMA (1994)

<h3>Objective.</h3> —To assess immunization levels for children of employees of a large corporation. <h3>Design.</h3> —A mail survey of a random sample of employees on the immunization history of one child per family. <h3>Setting.</h3> —US employees of Johnson &amp; Johnson. <h3>Participants.</h3> —1500 employees with children born between 1984 and 1991. <h3>Main Outcome Measures.</h3> —Coverage rates for recommended vaccines at different ages up to 6 years. <h3>Main Results.</h3> —Only 45.2% and 55.3% of the study children at ages 2 and 6 years were current for all recommended immunizations (65.1% and 70.3%, respectively, excluding the<i>Haemophilus influenzae</i>type b vaccine). Using the minimum standard required by many states for school entry, the coverage level at age 6 years was 90.4%. Factors associated with higher immunization rates at age 2 years were the corporate health plan (choices), higher pay level, greater parental formal education, white race, and knowing when to initiate immunization. Lower immunization rates at age 2 years were associated with delayed receipt of the first dose of diphtheria, tetanus, and pertussis vaccine, use of city or county clinics, employee-reported barriers of difficulty leaving work, and provider access problems, but not cost of services. After adjusting for the effects of other variables through logistic regression, race, pay level, and plan choice were no longer significant. Modeling with the remaining variables predicted rates of adequate immunization at age 2 years from 15% to 81%. <h3>Conclusion.</h3> —Even in this relatively affluent group with good insurance (including immunizations), preschool immunization rates did not reach public health goals. Changing modifiable factors, such as knowing when to initiate immunization, enabling parents to leave work more easily, and improving provider access, might improve preschool immunization rates. (<i>JAMA</i>. 1994;271:525-530)
Quackery persists
Journal: JAMA (1972)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Reply:
Authors: Neal D. Freedman, Teresa M. Curto, James E. Everhart
Journal: Hepatology (2009)

We thank Drs. Purnak and Ozaslan for their interest in our article.1 First, we agree that measurement of coffee intake by food frequency questionnaire has limitations. Food frequency questionnaires are useful tools, but are subject to misclassification. Furthermore, coffee has many constituents, the concentrations of which vary by preparation and serving method. We were not able to examine preparation and serving methods in our study. Nevertheless, significant differences in liver disease progression between self-reported coffee drinkers and nondrinkers suggest a role for coffee constituents in liver disease prevention. Future work is needed to establish this association and to identify active constituents of coffee. Second, in our study and other previous studies, smokers reported drinking more coffee than nonsmokers. We divided coffee intake into the number of cups consumed per day, and the effect of coffee was independent of either lifetime or current smoking status. When we stratified by current smoking status, the effect of consuming the same number of cups of coffee did not differ between smokers and nonsmokers.1 Third, Drs. Purnak and Ozaslan suggest an interesting mechanism that might link coffee consumption with liver disease progression, namely, that the caffeine in coffee induces cytochrome P450 1A2, which may affect response to antiviral treatment. Though the potential effect of coffee intake on virological response was outside the scope of our article, we did note that the effect of coffee on study outcomes was similar in both individuals receiving long-term treatment with low-dose peginterferon alfa-2a and those who did not. These results suggest that coffee did not exert its effect through modulation of an effect of this treatment. Neal D. Freedman*, Teresa M. Curto , James E. Everhart , * Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Rockville, MD, New England Research Institutes, Watertown, MA, Division of Digestive Diseases and Nutrition, National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health, Department of Health and Human Services, Bethesda, MD.
The prognosis of familial hypertrophic cardiomyopathy caused by myosin binding protein-C Glu451 Gln mutation is more unfavorable than that of InsG791 mutation Hideshi Niimura, Ryuichiro Anan, Linda L. Bachinski, Barry J. Maron, Shinichi Minagoe, Robert Roberts, J G. Seidman, Christine E. Seidman, Chuwa Tei. Kagoshima University, Kagoshima, Japan, Harvard Medical School, Boston, MA
Journal: Journal of the American College of Cardiology (2001)