tbuckley/cabot-search huggingface dataset:
SYMPATHECTOMY FOR THROMBOANGIITIS OBLITERANS
Journal: JAMA (1935)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Response to Letter Regarding Articles, “Long-Term Cardiovascular Mortality After Procedure-Related or Spontaneous Myocardial Infarction in Patients With Non-ST-Segment Elevation Acute Coronary Syndrome: A Collaborative Analysis of Individual Patient Data From the FRISC II, ICTUS, and RITA-3 Trials (FIR)” and “American College of Cardiology/American Heart Association/European Society of Cardiology/World Heart Federation Universal Definition of Myocardial Infarction Classification System and the …
Authors: Marc P. Bonaca, Stephen D. Wiviott, Eugene Braunwald, Sabina A. Murphy, Christian T. Ruff, Elliott M. Antman, David A. Morrow
Journal: Circulation (2012)
Jargon Confuses The Physician
Journal: JAMA (1966)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
New Excerpta Medica Section on Cancer
Journal: JNCI Journal of the National Cancer Institute (1953)

Journal Article New Excerpta Medica Section on Cancer Get access JNCI: Journal of the National Cancer Institute, Volume 14, Issue 2, October 1953, Page 455, https://doi.org/10.1093/jnci/14.2.455 Published: 01 October 1953
Response
Authors: R. T. SKEEL
Journal: JNCI Journal of the National Cancer Institute (1989)

Journal Article Response Get access ROLAND T. SKEEL ROLAND T. SKEEL Division of Hematology/Oncology Medical College of OhioP.O. Box 10008 Toledo, OH 43699 Search for other works by this author on: Oxford Academic PubMed Google Scholar JNCI: Journal of the National Cancer Institute, Volume 81, Issue 14, 19 July 1989, Page 1107, https://doi.org/10.1093/jnci/81.14.1107 Published: 19 July 1989
JAMA 100 Years Ago
Journal: JAMA (1987)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
LOEFFLER'S NEW METHOD OF PRODUCING ANTIBODIES.
Journal: JAMA (1905)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Screening for Renovascular Hypertension
Authors: David G. Martinez
Journal: Annals of Internal Medicine (1993)

Letters1 June 1993Screening for Renovascular HypertensionDavid G. Martinez, MDDavid G. Martinez, MDSearch for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-118-11-199306010-00016 SectionsAboutFull TextPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail TO THE EDITOR:Mann and Pickering [1] correctly recommend screening for renovascular hypertension only in patients with a moderate or high suspicion of the disease [1]. Other determining factors for screening, however, include the patient's willingness to have treatment if renovascular disease is found and the physician's willingness to pursue treatment based on the patient's risk profile and ease of hypertension control.In the best of hands, surgical mortality for repair of atheromatous renovascular disease is 2% [2] and may approach 9.3% [3]. Many of these patients have coronary artery disease or peripheral vascular disease, use tobacco, or have chronic ...References1. Mann SJ, Pickering TG. Detection of renovascular hypertension. State of the art: 1992. Ann Intern Med. 1992; 117:845-53. Google Scholar2. Novick AC, Ziegelbaum M, Vidt DG. Trends in surgical revascularization for renal artery disease: ten years' experience. JAMA. 1987; 257:498-501. Google Scholar3. Foster JH, Dean RH, Pinkerton JA, Rhamy RK. Ten years' experience with the surgical management of renovascular hypertension. Ann Surg. 1973; 177:755-66. Google Scholar Author, Article, and Disclosure InformationAffiliations: PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics 1 June 1993Volume 118, Issue 11Page: 905-906KeywordsChronic obstructive pulmonary diseaseDiureticsHypertensionMortalityMotivationPeripheral vascular diseaseStable coronary artery diseaseSurgical repair Issue Published: 1 June 1993 CopyrightCopyright © 1993 by American College of Physicians. All Rights Reserved.PDF DownloadLoading ...
Unusual expression of mRNA typical of Philadelphia positive acute lymphoblastic leukemia detected in chronic myeloid leukemia
Authors: Judy A. Kirk, Jerald Radich, Scott Edmands, Arthur Lee, Donald R. VanDevanter, Jo Anna Reems, Eileen M. Bryant
Journal: American Journal of Hematology (1996)

The Philadelphia chromosome (Ph) is found in both chronic myeloid leukemia (CML) and acute lymphoblastic leukemia (ALL). The Ph translocation, t(9;22)(q34;q11), can disrupt the BCR gene on chromosome 22 in one to two areas called the major (Mbcr1) and minor (mbcr1) breakpoint cluster regions. In CML the breakpoint has been mapped almost exclusively to Mbcr1, whereas in Ph positive ALL both Mbcr1 and the upstream mbcr1 breakpoints have been described. In this communication we describe an unusual patient with typical chronic phase Ph positive CML and evidence of the uncharacteristic mbcr1 breakpoint, predicting expression of the ALL-type p190 fusion protein. Fluorescence in situ hybridization demonstrated BCR gene rearrangement, the reverse transcription polymerase chain reaction detected the BCR-ABL fusion mRNA characteristic of the mbcr1 breakpoint, and failed to detect BCR-ABL mRNA characteristic of the Mbcr1 breakpoint. Southern blot analysis revealed no rearrangement in Mbcr1, and direct sequencing of the PCR product confirmed it to be the ALL-type mbcr1 fusion mRNA with the first exon of the BCR gene fused to ABL exon a2. This case differs from the previously reported cases of "p190" CML in that the patient presented without abnormal hematopoietic features other than those found in typical CML and provides further evidence that the p190 mRNA is not sufficient to cause an acute rather than chronic leukemia. © 1996 Wiley-Liss, Inc.
MORE DEATHS FROM WOOD ALCOHOL.
Journal: JAMA (1904)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Effects of Different Insulin Regimes on Postprandial Myocardial Perfusion Defects in Type 2 Diabetic Patients
Authors: R. Scognamiglio
Journal: Diabetes Care (2006)

OBJECTIVE—Postprandial glycemia is an independent risk factor for cardiovascular disease that is more powerful than fasting glycemia and determines myocardial perfusion defects in type 2 diabetes. We examined the efficacy of two different insulin regimes (regular insulin and insulin analog) in controlling postprandial hyperglycemia and in preventing myocardial perfusion abnormalities.
Availability of Curricular Materials About Vaccines, Vaccine-Preventable Diseases, and Vaccination Practices
Journal: JAMA (1999)

DEATHS FROM INFECTIOUS DISEASES HAVEdeclined markedly in the United States during the 20th century.This decline contributed to a sharp drop in infant and child mortality 1,2 and to the 29.2-year increase in life expectancy. 2 In 1900, 30.4% of all deaths occurred among children aged Ͻ5 years; in 1997, that percentage was only 1.4%.In 1900, the three leading causes of death were pneumonia, tuberculosis (TB), and diarrhea and enteritis, which (together with diphtheria) caused one third of all deaths.Of these deaths, 40% were among children aged Ͻ5 years. 1 In 1997, heart disease and cancers accounted for 54.7% of all deaths, with 4.5% attributable to pneumonia, influenza, and human immunodeficiency virus (HIV) infection. 2Despite this overall progress, one of the most devastating epidemics in human history occurred during the 20th century: the 1918 influenza pandemic that resulted in 20 million deaths, including 500,000 in the United States, in less than1 year-more than have died in as short a time during any war or famine in the world. 3 HIV infection, first recognized in 1981, has caused a pandemic that is still in progress, affecting 33 million people and causing an estimated 13.9 million deaths. 4These episodes illustrate the volatility of infectious disease death rates and the unpredictability of disease emergence.Public health action to control infectious diseases in the 20th century is based on the 19th century discovery of microorganisms as the cause of many serious diseases (e.g., cholera and TB).Disease control resulted from improvements in sanitation and hygiene, the discovery of antibiotics, and the imple-mentation of universal childhood vaccination programs.Scientific and technologic advances played a major role in each of these areas and are the foundation for today's disease surveillance and control systems.Scientific findings also have contributed to a new understanding of the evolving relation between humans and microbes.
Gonadal liberation
Authors: R. S. Sanford
Journal: JAMA (1971)

The privates are becoming more public! Television touts Feminine Hygiene Spray for "that special problem." The female body has now become acceptable matter for open discussion and viewing, even by the most prudish. Public service announcements recommend a Pap smear or suggest that once a month, while bathing, a woman should palpate her breasts as instructed by her physician. But the male body is still taboo. Cancer of the testis and penis, two of the most obvious and deforming of male neoplasms (responsible for up to 3% of all male cancers,<sup>1-4</sup>18% to 20% in an uncircumcized population<sup>5</sup>) and usually attacking those in the prime of life, are never mentioned in public. Can you imagine the public service ad: "Look your penis over by pulling back the foreskin. Feel your testicles once per month to check their size and firmness." These lesions are "picked up" by wives, girl
Cardioversion of paroxysmal arrhythmias
Authors: R. E. Kleiger
Journal: JAMA (1970)

Dr. Michael Cebrik, Junior Assistant Resident in Medicine, Jewish Hospital of St. Louis and Assistant in Medicine, Washington University School of Medicine: The first patient is a 68-year-old white woman who was admitted to Jewish Hospital for the second time on Aug 31, 1969, with a chief complaint of palpitations and a rapid heart rate. In September 1968, the patient had been hospitalized for similar complaints, at which time she was found to have atrial flutter with a 2:1 atrioventricular block and a ventricular rate of 140 beats per minute. Right bundle branch block, which had been present prior to this admission, was noted. The patient was treated with digoxin and warfarin sodium, and quinidine sulfate was added later. The cardiac rhythm varied from atrial flutter to atrial fibrillation with a ventricular response greater than 100 beats per minute. The quinidine effected a conversion to normal sinus rhythm and the
UCLA Sponsors Courses Abroad
Journal: JAMA (1964)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Transplantation/Digestive tract
Authors: P. Durand, E. Jacquemin, C. Chardot, F. Iserin, B. Dousset, D. Devictor, Irwin Weiss, Cherif Erian, Marvin Ament, Jorge Vargas, Sue McDiarmid, Judith Brill, C. Le Pommelet, D. Debray, J. Sasbón, M. Centeno, E. Entin, M. Acerenza, M. Ciocca, J. Goñi, G. Bianco, G. Weller, O. Imventarza, F. Ruza, C. Calvo, P. G. Dorao, D. Filgueiras, E. Alvarez, M. A. Delgado, J. Casado Flores, E. Mora, Teresa M. A. García, A. Martínez Azagra, A. Serrano, E. Ibiza, A. Abengochea, V. Modesto, B. Abengochea, J. Arago, R. Sanchis, R. Varas, E. Garcia, P. D. Booker, D. P. Prosser, R. Franks, Kiel Dang Phuong, Hai Le Thanh
Journal: Intensive Care Medicine (1996)
Hormone replacement: gynecologists consider the heart of the matter
Authors: C. A. Raymond
Journal: JAMA (1987)

CARDIOVASCULAR DISEASE is apparently superseding osteoporosis and breast cancer as the current focus of attention in the controversial matter of providing estrogen replacement therapy for menopausal women. A study published this summer (<i>Circulation</i>1987;75:1102-1109) of 2270 women suggests that those receiving unopposed estrogen have approximately one third the risk of death from cardiovascular disease of an age-matched group not taking the hormone. This study is one of the largest to specifically address the cardioprotective value of estrogen replacement. Yet it leaves open the question of the risks and benefits of the estrogen-progestin combination that is more typically prescribed. The growing importance of cardiovascular considerations in estrogen replacement therapy was underscored by several investigators who spoke at the annual meeting of the American College of Obstetricians and Gynecologists (ACOG) in Las Vegas. At least one of them, Lila E. Nachtigall, MD, associate professor of obstetrics and gynecology at New York University
<i>A Contribution to the Study of Diseases of the Circulatory System in the Insane</i>. (The Essay to which the ten guinea prize of the British Medico-Psychological Association was awarded). By T. Duncan Greenlees, M. B., Edin., Assistant Medical Officer to the Cumberland and Westmoreland Asylum, Garlands, Carlisle
Journal: American Journal of Psychiatry (1886)
Biochemical Mechanisms in Asbestos Toxicity
Authors: Q. Rahman, B. Das, P. N. Viswanathan
Journal: Environmental Health Perspectives (1983)

The alarming hazardous nature of asbestos makes it the foremost among toxic fugitive dusts. The biochemical mechanisms responsible for the diverse biological effects of asbestos, such as fibrosis, asbestos bodies, pleural plaques, respiratory difficulty, cancer, and cytotoxicity, are being studied in this laboratory. As asbestosis progresses in guinea pigs, along with reticulum formation, lysosomal enzymes are released from membrane-bound latent state to active free form, initiating degradative changes. Considerable alterations take place in the pulmonary metabolic machinery. Mitochondria in lung cells were found to be important loci for the toxic effect of asbestos. A profile of mitochondrial activity, in control and asbestotic animals, revealed specific enzymic changes such as increased cytochrome c oxidase during the disease. The functional organization of mitochondria was also altered, since the organelles from asbestotic lungs were swollen as measured by spectrophotometry. Glutamate dehydrogenase activity of mitochondria became exposed in asbestosis. The maleate dehydrogenase shunt which is involved in transport of the redox potential across the membrane was enhanced in cytosol and mitochondria. The involvement of microsomal enzymes in asbestosis was indicated by alterations in glucose-6-phosphatase and tyrosine transaminase and aniline hydroxylase. Changes in the biotransformational capacity of lung, due to asbestos, could be an important aspect in toxicity, especially the carcinogenic effect. Considerable alterations were encountered in the levels of different phospholipids and in mucopolysaccharide constituents. On the basis of the above, the molecular mechanisms in asbestos toxicity are explained as an integrated model. Interactions of dust constituents with those of membranes and the ensuing metabolic adjustments are thus important in the etiology of asbestosis.
Spontaneous air space enlargement in the lungs of mice lacking tissue inhibitor of metalloproteinases-3 (TIMP-3)
Authors: Kevin J. Leco, Paul Waterhouse, Otto H. Sanchez, Katrina L.M. Gowing, A. Robin Poole, Andrew Wakeham, Tak W. Mak, Rama Khokha
Journal: Journal of Clinical Investigation (2001)
Tuberculosis
Authors: Donald S. King
Journal: New England Journal of Medicine (1955)

THE year 1954 will stand out as one of special importance in man's fight to rid his body of infection by the tubercle bacillus. In that year the National Tuberculosis Association celebrated the fiftieth anniversary of its founding, and tuberculosis workers over the world took note of the tenth anniversary of the availability of streptomycin, the first "drug" ever to prove effective in the treatment of tuberculosis.Since the 1952 progress report emphasis has been given to many other interesting points. Outstanding among these are the annual meetings on the chemotherapy of tuberculosis in which the Veterans Administration, the Army . . .
Bougienage is effective therapy for most benign esophageal strictures
Authors: F. L. Lanza
Journal: JAMA (1978)

Esophageal stricture is the most frequent serious complication of reflux esophagitis. Bougienage was used as the primary therapy of benign esophageal stricture in 92 patients, followed by institution of a medical antireflux program. Response to treatment was excellent, and 78% of patients required two or fewer subsequent dilations. Twenty patients were followed up for two years or longer, and 75% did not require more than one dilation annually. Primary therapy for most patients with peptic stenosis of the esophagus is recommended to be dilation; operative therapy should be reserved for those patients in whom a malignant neoplasm is suspected or those in whom frequent and rapid stricture recurrence makes medical therapy impractical. (<i>JAMA</i>240:844-847, 1978)