Pulmonary Disease: An Annotated Bibliography of Recent Literature
Authors: PULMONARY DISEASES COMMITTEE
Journal: Annals of Internal Medicine (1983)

The Literature of Medicine1 June 1983Pulmonary Disease: An Annotated Bibliography of Recent LiteratureReferences to Journal Articles and Other PapersPULMONARY DISEASES COMMITTEEPULMONARY DISEASES COMMITTEESearch for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-98-6-1033 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptThis bibliography includes references to 101 recently published journal papers and similar sources that represent important developments in the concept and practice of internal medicine. It has been compiled by the Pulmonary Diseases Committee as part of the Medical Knowledge Self-Assessment Program VI Syllabus.In preparing this bibliography, the committee reviewed MEDLINE†bibliographic printouts of journal articles published since the beginning of 1978 and also drew on sources in libraries of hospitals and other clinical institutions in the United States and Canada; thus, only papers in English are listed, and most are from American journals....Clinical Evaluation of Pulmonary Function Diagnostic Procedures Immunology of the Lung Chronic Obstructive Pulmonary Disease Asthma Upper Airway Obstruction Sleep Disorders Infectious Diseases of the Respiratory Tract Occupational Lung Diseases Pulmonary Vascular Diseases Respiratory Failure Genetic Defects Lung Neoplasms1. WANNER A. Interpretation of pulmonary function tests. In: SACKNER MA, ed. Diagnostic Techniques in Pulmonary Disease. Part 1. New York and Basel: Marcel Dekker, Inc. ; 1980:353-426. A complete review of the clinical applications of pulmonary function testing in adults. Patterns of pulmonary dysfunction are used to arrive at a physiologic classification of lung disease. Google Scholar2. HUNNINGHAKEGADEKKAWANAMIFERRANSCRYSTAL GJOVR. Inflammatory and immune processes in the human lung in health and disease: evaluation by bronchoalveolar lavage. Am J Pathol. 1979;97:149-206. Bronchoalveolar lavage was used to evaluate inflammatory and immune processes of the lung. This technique may yield valuable information about pathogenesis, prognosis, and response to thereapy in inflammatory lung disease. MedlineGoogle Scholar3. MINTZERMALAVENEIMANMICHAELISVANECKOSANDERS RSHLRJ. Computed vs. conventional tomography in evaluation of primary and secondary pulmonary neoplasms. Radiology. 1979;132:653-9. Points out the superiority of computerized chest tomography over conventional chest tomography in the radiographic evaluation of the mediastinum. CrossrefMedlineGoogle Scholar4. MUHMBROWNCROWESHEEDYHATTERYSTEPHENS JLJPRD. Comparison of whole lung tomography and computed tomography for detecting pulmonary nodules. AJR 1978;131:981-4. Indicates that in patients with known malignancy, not all additional nodules detected by computed tomography of the chest are malignant. CrossrefMedlineGoogle Scholar5. OLDENBURGNEWHOUSE FM. Thoracoscopy: a safe, accurate diagnostic procedure using the rigid thoracoscope and local anesthesia. Chest. 1979;75:45-50. Describes the authors' experience with thoracoscopy in 32 patients with pleural effusion. The procedure was safe, and yielded an accurate diagnosis in 28 patients. CrossrefMedlineGoogle Scholar6. ROTHHUCHONARNOUXSTANISLAS-LEGUERNMARSACCHRETIEN CGAGJJ. Bronchoalveolar cells in advanced pulmonary sarcoidosis. Am Rev Respir Dis. 1981;124:9-12. Suggests that an increased number of neutrophils (versus lymphocytes) in bronchoalveolar lavage fluid reflects the development of fibrosis in patients with advanced sarcoidosis, an observation of prognostic significance. MedlineGoogle Scholar7. RUDDHASLAMTURNER-WARWICK RPM. Cryptogenic fibrosin alveolitis: relationships of pulmonary physiology and bronchoalveolar lavage to response to treatment and prognosis. Am Rev Respir Dis. 1981;124:1-8. Suggests that a predominance of lymphocytes in bronchoalveolar lavage fluid predicts responsiveness to glucocorticosteroid treatment in patients with idiopathic pulmonary fibrosis. MedlineGoogle Scholar8. SCHANERCHANGDOPPMANCONKLEFLYEROSENBERG EAJDMS. Comparison of computed and conventional whole lung tomography in detecting pulmonary nodules: a prospective radiologie-pathologie study. AJR. 1978;131:51-4. In 25 patients with malignancy, computed chest tomography was more sensitive than conventional chest tomography in detecting pulmonary nodules. However, 60% of additional nodules detected by computed tomography turned out to be benign granulomas. CrossrefMedlineGoogle Scholar9. ZAVALASCHOELL DJ. Ultrathin needle aspiration of the lung in infectious and malignant disease. Am Rev Respir Dis. 1981;123:125-31. Ultrathin needle aspiration of the lung in patients with pulmonary malignancy or infection has an 87% sensitivity and 100% specificity. MedlineGoogle Scholar10. DANIELE R. Immune defenses of the lung. In: FISHMAN AP, ed. Pulmonary Diseases and Disorders. New York: McGraw-Hill Book Co. ; 1980:624-32. A current summary of the structure and function of the lung's immune system. Summarizes what is currently known about the lung's handling of inhaled antigens, the role of the alveolar macrophage in antigen processing, and current concepts of the secretory immune system. Google Scholar11. CARPJANOFF HA. Possible mechanisms of emphysema in smokers: in vitro suppression of serum elastase-inhibitory capacity by fresh cigarette smoke and its prevention by antioxidants. Am Rev Respir Dis. 1978;118:617-21. Evidence indicates that multiple factors can tip the protease-antiprotease balance in the lung in favor of protease—and thereby promote elastin destruction. Oxidants in cigarette smoke are found to inactivate antiprotease. MedlineGoogle Scholar12. CUGELLFISH DJ eds. Beta2 adrenergic agents and other drugs in reversible airway disease. Chest. 1978;73:913-1022. A group of articles discussing most of the agents used for the reversal or prevention of bronchospasm. CrossrefGoogle Scholar13. DEMARCOWYNNEBLOCKBOYSENTAASAN FJAPV. Oxygen desaturation during sleep as a determinant of the "blue and bloated" syndrome. Chest. 1981;79:621-5. Another finding that may help to explain why there are "pink puffers" and "blue bloaters." CrossrefMedlineGoogle Scholar14. DI SANT'AGNESEDAVIS PP. Cystic fibrosis in adults: 75 cases and a review of 232 cases in the literature. Am J Med. 1979;66:121-32. Patients with cystic fibrosis are surviving in increasing numbers into adulthood. In some instances, the diagnosis is not made during childhood. CrossrefMedlineGoogle Scholar15. HUDSONPIERSON LD. Comprehensive respiratory care for patients with chronic obstructive pulmonary disease. Med Clin North Am. 1981;65(3):629-45. A detailed review of what is involved in the comprehensive care of patients with chronic obstructive pulmonary disease, plus data indicating that programs of this type have a positive impact on the lives of such patients. MedlineGoogle Scholar16. KACHEL R. Left ventricular function in chronic obstructive pulmonary disease. Chest. 1978;74:286-90. Concludes that left ventricular function is normal in most patients with chronic obstructive pulmonary disease; when it is not, other causes are present. Left heart failure, although uncommon, is difficult to detect in these patients. CrossrefMedlineGoogle Scholar17. MCCOMBSLOWELLOHMAN RFJ. Myths, morbidity, and mortality in asthma. JAMA. 1979;242:1521-4. A brief review of certain misconceptions regarding asthma that can influence morbidity and mortality. CrossrefMedlineGoogle Scholar18. MCFADDENFELDMAN EN. Asthma: pathophysiology and clinical correlates. Med Clin North Am. 1977;61:1229-38. A discussion of the pathophysiology of asthma and its relation to clinical findings. CrossrefMedlineGoogle Scholar19. MOSERBOKINSKYSAVAGEARCHIBALDHANSEN KGRCP. Results of a comprehensive rehabilitation program: physiologic and functional effects on patients with chronic obstructive pulmonary disease. Arch Intern Med. 1980;140:1596-601. Improved exercise capability and performance of activities of daily living follow participation by patients with chronic obstructive pulmonary disease in comprehensive rehabilitation. CrossrefMedlineGoogle Scholar20. . Continuous or nocturnal oxygen therapy in hypoxemic chronic obstructive lung disease: a clinical trial. Ann Intern Med. 1980;93:391-8. A multicenter study indicating that in patients with chronic obstructive lung disease who are hypoxemic, continuous oxygen therapy leads to a lower mortality than does oxygen therapy given 12 hours per day. The basis for the difference is not clear. LinkGoogle Scholar21. PETTYZWILLICH TC eds. The 22nd Aspen Lung Conference: chronic obstructive pulmonary disease. Chest. 1980;77(Suppl.):249-330. Multiple papers in this supplement review aspects of pathogenesis, diagnosis, natural history, and management. MedlineGoogle Scholar22. TIMMSKVALEANTHONISENBOYLENCUGELLPETTYWILLIAMS RPNCDTG. Selection of patients with chronic obstructive pulmonary disease for long-term oxygen therapy. JAMA. 1981;245:2514-5. A report that provides criteria for identifying patients with chronic obstructive pulmonary disease who should receive long-term oxygen therapy. Many patients (45%) who seem to need oxygen when first seen are found not to be candidates a month later. CrossrefMedlineGoogle Scholar23. TRAVERCLINEBURROWS GMB. Predictors of mortality in chronic obstructive pulmonary disease: a 15-year follow-up study. Am Rev Respir Dis. 1979;119:895-902. Wide variability in prognosis exists. The forced exploratory volume in 1 second (FEV1) after administration of bronchodilator was the best prognostic indicator. MedlineGoogle Scholar24. BOUSHEYHOLTZMANSHELLERNADEL HMJJ. Bronchial hyperreactivity. Am Rev Respir Dis. 1980;121:389-413. A complete review of the basic and clinical aspects of airway hyperresponsiveness, a typical feature of bronchial asthma. MedlineGoogle Scholar25. DEALMCFADDENINGRAMSTRAUSSJAEGER EERRJ. Role of respiratory heat exchange in production of exercise-induced asthma. J Appl Physiol. 1979;46:467-75. Indicates that exercise-induced asthma is mediated by respiratory heat loss. CrossrefMedlineGoogle Scholar26. JACKSONPOWELLWANDELLBENTLEYDORR JJMJR. Cimetidine decreases theophylline clearance. Am Rev Respir Dis. 1981;123:615-7. A study of drug interaction between theophylline and Cimetidine. MedlineGoogle Scholar27. MCFADDEN E. Aerosolized bronchodilators and steroids in the treatment of airway obstruction in adults. Am Rev Respir Dis. 1980;122(5 Pt 2):89-96. A review of the different types of aerosolized bronchodilators and topical glucocorticosteroids. Indications for and efficacy and adverse effects of these agents in the treatment of bronchial obstruction are critically assessed. CrossrefMedlineGoogle Scholar28. TOOGOOD J. Aspirin intolerance, asthma, prostaglandins, and cromolyn sodium [Editorial]. Chest. 1977;72:135-7. A brief review of the possible interactions between mediator release and prostaglandin effects in asthmatic patients with aspirin intolerance. CrossrefMedlineGoogle Scholar29. WEISS E. Status Asthmaticus. Baltimore: University Park Press; 1978:201-53. Useful reviews of the pharmacokinetics and clinical applications of theophylline preparations, beta-adrenergic agents, and glucocorticosteroids. Google Scholar30. WILLIAMS M. Beclomethasone diproprionate. Ann Intern Med. 1981;95:464-7. A critical review of the role of the aerosol glucocorticosteroid beclomethasone diproprionate in patients with airways disease. LinkGoogle Scholar31. ACRESKRYGER JM. Clinical significance of pulmonary function tests: upper airway obstruction. Chest. 1981;80:207-11. A clear description of the physiologic diagnosis of upper airway obstruction with special attention to the flow-volume loop. CrossrefMedlineGoogle Scholar32. KRYGERBODEANTICANTHONISEN MFRN. Diagnosis of obstruction of the upper and central airways. Am J Med. 1976;61:85-93. A classic description of the flow-volume loop as a diagnostic aid in patients with upper airway obstruction. CrossrefMedlineGoogle Scholar33. BLOCKWYNEEBOYSENLINDSEYMARTINCANTOR AJPSCB. Menopause, medroxyprogesterone and breathing during sleep. Am J Med. 1981;70:506-10. Disturbed breathing patterns in sleep are seen more often in postmenopausal and premenopausal women, giving support to the use of medroxyprogesterone for at least some types of sleep apnea. CrossrefMedlineGoogle Scholar34. CHERNIACK N. Respiratory dysrhythmias during sleep. N Engl J Med. 1981;305:325-30. Basic review of respiratory rhythm disorders occurring during sleep by a widely renowned investigator of the control mechanisms of respiration. CrossrefMedlineGoogle Scholar35. GUILLEMINAULTSIMMONSMOTTA CFJ. Obstructive sleep apnea syndrome and tracheostomy: long-term follow-up experience. Arch Intern Med. 1981;141:985-8. Review of long-term follow-up of tracheostomy for obstructive sleep apnea, indicating the benefits and sometimes troublesome complications of maintaining a tracheostomy. CrossrefMedlineGoogle Scholar36. LITTNERMCGINTYARAND MDD. Determinants of oxygen desaturation in the course of ventilation during sleep in chronic obstructive pulmonary disease. Am Rev Respir Dis. 1980;122:849-57. Periodic respirations during sleep are possibly more important in causing periods of significant hypoxemia than is the severity of airway obstruction measured on pulmonary function studies while the patient is awake. MedlineGoogle Scholar37. BELLSTERNPANTZERSINHADELUCA NPETH. Evidence that increased circulating α 25-dihydroxyvitamin D is the probable cause for abnormal calcium metabolism in sarcoidosis. J Clin Invest. 1979;64:218-25. A detailed study exploring the mechanisms of hypercalcemia and hypercalciuria in sarcoidosis. CrossrefMedlineGoogle Scholar38. CARRINGTONGAENSLERCOUTUFITZGERALDGUPTA CERMR. Natural history and treated course of usual and desquamative interstitial pneumonia. N Engl J Med. 1978;298:801-9. A long-term prospective study describing the differences between the clinical and pathologic features of desquamative interstitial pneumonitis and usual interstitial pneumonitis; emphasizes natural history, survival, and response to therapy. CrossrefMedlineGoogle Scholar39. CRYSTALGADEKFERRANSFULMERLINEHUNNINGHAKE RJVJBG. Interstitial lung disease: current concepts of pathogenesis, staging and therapy. Am J Med. 1981;70:542-68. A review emphasizing the pathogenesis of the various interstitial lung diseases with respect to approaches for diagnosis, staging, assessment of disease activity, and the use of therapy. CrossrefMedlineGoogle Scholar40. DANIELEDAUBERROSSMAN RJM. Immunologic abnormalities in sarcoidosis. Ann Intern Med. 1980;92:406-16. A review describing current understanding of the immunologic abnormalities of sarcoidosis with relevance to diagnosis and management. LinkGoogle Scholar41. DANIELEHENSONFANTONEWARDDREISIN RPJPR. Immune complex injury of the lung. Am Rev Respir Dis. 1981;124:738-55. Reviews our current understanding of the immunologic aspects of lung disease, especially with respect to immune complex formation. MedlineGoogle Scholar42. DEREMEEROHRBACH RM. Serum angiotensin-converting enzyme activity in evaluating the clinical course of sarcoidosis. Ann Intern Med. 1980;92:361-5. Addresses the strengths and limitations in the use of serum angiotensin-converting enzyme in assessing disease activity and in following the course and progression of disease in sarcoidosis. LinkGoogle Scholar43. FAUCIHAYNESCOSTAKATZWOLFF ABJPS. Lymphomatoid granulomatosis: prospective clinical and therapeutic experiences over 10 years. N Engl J Med. 1982;306:68-74. An important long-term prospective study evaluating clinical and therapeutic experience in lymphomatoid granulomatosis. The emergence of lymphoid tumors in untreated patients and the use of cyclophosphamide in bringing about remission are described. CrossrefMedlineGoogle Scholar44. FAUCIHAYNESKATZ ABP. The spectrum of vasculitis: clinical, pathologic, immunologic, and therapeutic considerations. Ann Intern Med. 1978;89:660-76. Reviews the clinical features and immunopathogenic mechanisms in the vasculitides; also gives practical approaches in assessing disease activity and therapy. LinkGoogle Scholar45. HUNNINGHAKEFAUCI GA. Pulmonary involvement in the collagen vascular diseases. Am Rev Respir Dis. 1979;119:471-503. A review concerning pulmonary involvement in collagen vascular diseases. Pathology and immunopathogenesis are outlined for each disorder; the bibliography is up-to-date and complete. MedlineGoogle Scholar46. ROSSMANDAUBER MJ. Sarcoidosis: assessment of inflammatory activity. In: FISHMAN AP, eds. Pulmonary Diseases and Disorders. New York: McGraw-Hill Book Co. ; 1982:193-304. Reviews newer methods for the assessment of inflammatory activity in sarcoidosis, including the use of gallium scanning, bronchoalveolar lavage, and serum angiotensinconverting enzyme activity. Google Scholar47. WALLGAENSLERCARRINGTONHAYES CECJ. Comparison of transbronchial and open biopsies in chronic infiltrative lung disease. Am Rev Respir Dis. 1981;123:280-5. Emphasizes that open lung biopsy instead of transbronchial biopsy is often required to obtain enough tissue for a specific diagnosis and assessment of the histologic pattern. MedlineGoogle Scholar48. WEISSMUGGIA RF. Cytotoxic drug-induced pulmonary disease: update 1980. Am J Med. 1980;68:259-66. A review of cytotoxic-induced pulmonary drug reactions. Contains several important practical considerations concerning the use of these drugs (for example, the additive toxic effects of other drugs, radiation, and oxygen therapy). CrossrefMedlineGoogle Scholar49. BARTLETT J. Anaerobic bacterial pneumonitis. Am Rev Respir Dis. 1979;119:19-23. Clinical features of patients with anaerobic pulmonary infections are compared with patients having pneumococcal pneumonia. MedlineGoogle Scholar50. BAYER A. Fungal pneumonias; pulmonary coccidioidal syndromes (Part 1): primary and progressive primary coccidioidal pneumonias—diagnostic, therapeutic, and prognostic considerations. (Part 2): miliary, nodular, and cavitary pulmonary coccidioidomycosis; chemotherapeutic and surgical considerations. Chest. 1981;79:575-83, 686-91. Two-part series covering epidemiology, mycology, clinical aspects, and management of Coccidioides immitis infection. MedlineGoogle Scholar51. EDELSTEINMEYERFINEGOLD PRS. Laboratory diagnosis of Legionnaire's disease. Am Rev Respir Dis. 1980;121:317-27. Sensitivity and specificity of various diagnostic methods was evaluated in 32 patients with nosocomial Legionnaire's disease. MedlineGoogle Scholar52. GLASSROTHROBINSSNIDER JAD. Tuberculosis in the 1980s. N Engl J Med. 1980;302:1441-50. A review of epidemiology, diagnosis, and treatment of tuberculosis in the 1980s, with an especially useful discussion of treatment and prophylaxis. CrossrefMedlineGoogle Scholar53. GLIMPBAYER RA. Fungal pneumonias: Part 3. Allergic bronchopulmonary aspergillosis. Chest. 1981;80:85-94. Pathophysiology, clinical features, diagnosis, and treatment of allergic bronchopulmonary aspergillosis. CrossrefMedlineGoogle Scholar54. GOODWINSHAPIROTHURMANTHURMANDES PREZ RJGSR. Disseminated histoplasmosis: clinical and pathologic correlations. Medicine. 1980;59:1-33. Comprehensive review of Histoplasma capsulatum infection, detailing the initial invasion of tissue, clinical manifestations, and diagnostic studies used to detect the disease. Treatment is discussed briefly. CrossrefMedlineGoogle Scholar55. HERBERTBAYER PA. Fungal pneumonia (Part 4): invasive pulmonary aspergillosis. Chest. 1981;80:220-5. Pathogenesis, histopathology, clinical features, diagnosis, and therapy of invasive pulmonary aspergillosis. CrossrefMedlineGoogle Scholar56. . Ann Intern Med. 1979;90:491-714. Comprehensive review of Legionnaire's disease dealing with the clinical illness, its pathology, epidemiology, microbiology, and diagnostic methods. Google Scholar57. KERKERINGDUMASHADOMY TRS. The evolution of pulmonary cryptococcosis: clinical implications from a study of 41 patients with and without compromising host factors. Ann Intern Med. 1981;94:611-6. Dissemination of Cryptococcus neoformans infection to the central nervous system occurred in 28 of 34 immunocompromised patients. Dissemination did not occur in six of seven normal hosts. Recommendations for therapy are stated. LinkGoogle Scholar58. LERNER A. The gram-negative bacillary pneumonias. DM 1980;27:1-56. Typical features of Pseudomonas eruginosa, Escherichia coli, Enterobacter, Proteus, Haemophilus, and anaerobic pulmonary infections are described. Diagnostic methods for early recognition of these infections and appropriate antibiotic therapy are discussed. MedlineGoogle Scholar59. NARAGIMCDONNELL SG. Hematogenous staphylococcal pneumonia secondary to soft tissue infection. Chest. 1981;79:173-5. Clinical presentation of ten previously healthy patients with staphylococcal pneumonia after soft tissue infection. CrossrefMedlineGoogle Scholar60. SAROSIDAVIES GS. Blastomycosis. Am Rev Respir Dis. 1979;120:911-38. Infection with Blastomyces dermatitides is comprehensively reviewed. Microbiologic and immunologic aspects are discussed; clinical presentations, diagnosis, and treatment are stressed. MedlineGoogle Scholar61. SCHWARTZ J. Pneumococcal vaccine: clinical efficacy and effectiveness. Ann Intern Med. 1982;96:208-20. The currently available pneumococcal vaccine protects against serotypes responsible for 80% of pneumococcal bacteremia in the United States. High risk, immunocompetent patients should be vaccinated. Inconclusive results have been obtained in other patient groups. LinkGoogle Scholar62. WALLACEMUSHERMARTIN RDR. Hemophilus influenzae pneumonia in adults. Am J Med. 1978;64:87-93. In 41 adults with Haemophilus influenzae pneumonia, bacteremia was common in those over 50 years old. Multisegmental infiltrates were common and pleural effusions occurred in 50% of patients. Mortality was associated with severe underlying disease. CrossrefMedlineGoogle Scholar63. WANGPATTERSONROBERTSGHORY JRMA. The management of allergic bronchopulmonary aspergillosis. Am Rev Respir Dis. 1979;120:87-92. Evaluation of clinical course and plan for drug therapy of patients with allergic bronchopulmonary aspergillosis. MedlineGoogle Scholar64. WINSTONLAUGALEYOUNG DDRL. Trimethoprim-sulfamethoxazole for treatment of Pneumocystis carinii pneumonia. Ann Intern Med. 1980;92:762-9. Intravenous trimethoprim-sulfamethoxazole cured 64% of all patients with Pneumocystis carinii pneumonia and 78% of those treated for 4 or more days. Oral therapy is also highly effective. Drug regimens are recommended. LinkGoogle Scholar65. AISNERWIERNIK JP eds. Asbestos-related neoplasms. Semin Oncol. 1981;8:241-346. Focuses on malignant mesothelioma. Topics include the role of asbestos in producing cytologic changes; pathologic appearance of the tumor; natural history of the disease; and the role of surgery, radiation, therapy, and chemotherapy. MedlineGoogle Scholar66. BERNSTEIN I. Occupational asthma. Clin Chest Med. 1981;2:255-72. Pathophysiologic factors in occupationally induced asthma are discussed. IgE-mediated bronchospasm, reflex and inflammatory bronchoconstriction, pharmacologic agonists, and inorganic as well as organic allergens are cited as causes. Diagnosis and evaluation of affected workers are described. CrossrefMedlineGoogle Scholar67. BRAUNDOPICOTSIATISHORVATHDICKIERANKIN SGAEHJ. Farmer's lung disease: long-term clinical and physiologic outcome. Am Rev Respir Dis. 1979;119:185-91. Recurrent attacks of extrinsic allergic alveolitis are the most important factor in determining long-term effects of farmer's lung disease. MedlineGoogle Scholar68. MORGANLAPPSEATON WLD. Respiratory disability in coal miners. JAMA. 1980;243:2401-4. Observations on pulmonary impairment in coal miners in relation to legislation on black lung disease. CrossrefMedlineGoogle Scholar69. PREGER L. Asbestos Related Disease. New York: Grune & Stratton; 1978:1:288. Practical evaluation of the health effects of asbestos exposure, including clinical assessment, epidemiology, radiologic manifestations, asbestos-related neoplasms, and medical problems. Google Scholar70. REISERLAST KJ. Silicosis and fibrogenesis: fact and artifact. Toxicology. 1979;13:51-72. Review of current knowledge regarding fibrogenic mechanisms in silica exposure using in-vivo and in-vitro observations. CrossrefMedlineGoogle Scholar71. SPRINCEKANAREKWEBERCHAMBERLINKAZEMI NDARH. Reversible respiratory disease in beryllium workers. Am Rev Respir Dis. 1978;117:1011-7. Air sampling for beryllium content combined with physiologic and radiologic assessment showed that diminution in ambient beryllium levels led to improved pulmonary status in 13 of 20 workers over a 3-year period. MedlineGoogle Scholar72. DANTZKERBOWER DJ. Partial reversibility of chronic pulmonary hypertension caused by pulmonary thromboembolic disease. Am Rev Respir Dis. 1981;124:129-31. Indicates that pulmonary vasodilating agents may reduce pulmonary arterial pressure even in some patients with thromboembolic pulmonary hypertension. The changes are modest. MedlineGoogle Scholar73. EDWARDSEDWARDS WJ. Clinical primary pulmonary hypertension: three pathologic types. Circulation. 1977;56:884-8. Concise review of the pathology of three forms of pulmonary hypertension that clinically may be difficult to distinguish: plexogenic pulmonary arteriopathy, recurrent pulmonary embolism, and pulmonary veno-occlusive disease. CrossrefMedlineGoogle Scholar74. ENSON Y. Pulmonary heart disease: relation of pulmonary hypertension to lung structure and function. Bull NY Acad Med. 1977;53:551-66. A review of the primary mechanisms responsible for increases in pulmonary failure. Provides especially useful data regarding the relation of changes in lung volumes associated with restrictive disease to pulmonary hemodynamics. MedlineGoogle Scholar75. HONEYCOTTERDAVIESDENISON MLND. Clinical and haemodynamic effects of diazoxide in primary pulmonary hypertension. Thorax. 1980;35:269-76. Diazoxide is one of many drugs being used to treat primary pulmonary hypertension. As with all the drugs, individual reponses vary widely to diazoxide, and some reponses may be deleterious. CrossrefMedlineGoogle Scholar76. HULLHIRSHPOWERSTURPIEWALKER RJPAI. Combined use of leg scanning and impedance plethysmography in suspected venous thrombosis: an alternative to venography. N Engl J Med. 1977;296:1497-500. Indicates that the combination of radiofibrinogen-impedance tests is sufficiently sensitive and specific to make contrast venography unnecessary in most patients suspected of having lower extremity deep venous thrombosis. CrossrefMedlineGoogle Scholar77. MOSER K. Pulmonary embolism. Am Rev Respir Dis. 1977;115:829-52. A review of the pathophysiology, natural history, diagnosis, and treatment of venous thrombosis and pulmonary embolism. MedlineGoogle Scholar78. MOSER K ed. Pulmonary Vascular Diseases. New York: Marcel Dekker; 1979:1-706. A comprehensive volume dealing with all aspects of pulmonary vascular diseases. Google Scholar79. MOSERLEMOINE KJ. Is embolic risk conditioned by location of deep venous thrombosis? Ann Intern Med. 1981;94:439-44. A report suggesting that pulmonary embolism occurs commonly from deep venous thrombi that involve above knee veins; rarely, from thrombi confined to below-knee veins. LinkGoogle Scholar80. . Am J Med. 1980;69:127-34. A review of many of the knowns and unknowns regarding pulmonary hypertension. CrossrefMedlineGoogle Scholar81. STAUB N. Pulmonary edema. Physiol Rev. 1974;54:678-811. An excellent review of the factors that can promote pulmonary edema and the sequence in which fluid accumulates in the lung. CrossrefMedlineGoogle Scholar82. STEINWILLISDEMETS PPD. History and physical examination in acute pulmonary embolism in patients without preexisting cardiac or pulmonary disease. Am J Cardiol. 1981;47:218-23. A review of the findings in a subgroup of patients with massive or submassive embolism. A constellation of findings can suggest this diagnosis, but findings are not specific. CrossrefMedlineGoogle Scholar83. Urokinase-Streptokinase Embolism Trial: Phase 2 results. JAMA. 1974;229:1606-13. A large study using thrombolytic agents in submassive and massive pulmonary embolism shows no reduction in mortality or ultimate degree of embolic resolution by scan and increased bleeding with thrombolytic agents versus heparin. CrossrefMedlineGoogle Scholar84. BOYSENBLOCKWYNNEHUNTFLICK PAJLM. Nocturnal pulmonary hypertension in patients with chronic obstructive pulmonary disease. Chest. 1979;76:536-42. Description of nocturnal hypoxemia associated with transient pulmonary hypertension, both of which are prevented by oxygen administration. CrossrefMedlineGoogle Scholar85. DANTZKERBROOKDEHARTLYNCHWEG DCPJJ. Ventilation-perfusion distributions in adult respiratory distress syndrome. Am Rev Respir Dis. 1979;120:1039-52. A thorough description of gas exchange in adult respiratory distress syndrome. Shunting or alveoli with very low ventilation perfusion ratios were responsible for observed hypoxemia. Positive end-expiratory pressure improved arterial oxygenation by decreasing perfusion of unventilated lung. MedlineGoogle Scholar86. ELLIOTTMORRISCENGIZ CAM. Pulmonary function and exercise gas exchange in survivors of adult respiratory distress syndrome. Am Rev Respir Dis. 1981;123:492-5. Pulmonary function was studied in survivors of adult respiratory distress syndrome. Six months afterward no abnormalties of vital capacity were noted, but the diffusing capacity was moderately reduced. The alveolar-arterial oxygen difference was normal at rest in most patients but was increased in the majority during exercise. MedlineGoogle Scholar87. GILBERTASHUTOSHAUCHINCLOSSRANAPEPPI RKJSD. Prospective study of controlled oxygen therapy: poor prognosis of patients with asynchronous breathing. Chest. 1977;71:456-62. Good exposition of the importance of synchronous use of thoracic and abdominal muscles in avoiding mechanical ventilation during acute respiratory failure in patients with chronic obstructive pulmonary disease. CrossrefMedlineGoogle Scholar88. LUCEPIERSONHUDSON JDL. Intermittent mandatory ventilation. Chest. 1981;79:678-85. Excellent review of the current state of intermittent mandatory ventilation with a balanced discussion of data supporting or contradicting its claimed superiority over other modes of ventilation. CrossrefMedlineGoogle Scholar89. . Long term domiciliary oxygen therapy in chronic hypoxic cor pulmonale complicating chronic bronchitis and emphysema. Lancet. 1981;1:681-6. Controlled, randomized study of the improvement in survival resulting from oxygen therapy in hypoxemic patients with chronic obstructive pulmonary disease. MedlineGoogle Scholar90. SHAPIROCANEHARRISONSTEINER BRRM. Changes in intrapulmonary shunting with administration of 100 percent oxygen. Chest. 1980;77:138-41. The conventional means of measuring intrapulmonary shunting with 100% oxygen in respiratory failure actually increases shunting. CrossrefMedlineGoogle Scholar91. TIMMSKVALEANTHONISENBOYLENCUGELLPETTYWILLIAMS RPNCDTG. Selection of patients with chronic obstructive pulmonary disease for long-term oxygen therapy. JAMA. 1981;245:2514-5. After 1 month of intensive treatment, 45% of hypoxemic patients initially selected for long-term oxygen therapy improved sufficiently to allow discontinuation of oxygen. Low-flow (1 to 3 L/min) oxygen delivered by nasal prongs reversed hypoxemia in 95% of stable patients. CrossrefMedlineGoogle Scholar92. AFZELIUS B. "Immotile-cilia" syndrome and ciliary abnormalities induced by infection and injury (Editorial). Am Rev Respir Dis. 1981;124:107-9. A complete review of the ciliary dyskinetic syndromes. Discusses the approaches for diagnosing ciliary abnormalities and emphasizes acquired ciliary abnormalities induced by infection and injury, and contains a fairly complete and up-to-date bibliography. MedlineGoogle Scholar93. ALIMOUNTAINEWERJOHNSTONHAYNIE MCMDT. Predicting loss of pulmonary function after pulmonary resection for bronchogenic carcinoma. Chest. 1980;77:337-42. Describes the preoperative use of lung scans with spirometry to predict respiratory tolerance for surgery and prediction of loss of function from resection. CrossrefMedlineGoogle Scholar94. BOYSENHARRISBLOCKOLSEN PJAG. Prospective evaluation of pneumonectomy using perfusion scanning: follow-up beyond one year. Chest. 1981;80:163-6. A 1- to 5-year postoperative follow-up of patients having resection based on a formula of forced exploratory volume in 1 second or maximum voluntary ventilation standards for postoperative functional survival. CrossrefMedlineGoogle Scholar95. MITTMANBRUDERMAN CI. Lung cancer: to operate or not? Am Rev Respir Dis. 1977;116:477-96. The basic arguments for operative versus nonoperative treatment of lung cancer are summarized concisely in a debate form. CrossrefMedlineGoogle Scholar96. ROSENOWCARR ED. Bronchogenic carcinoma. CA. 1979;29:233-45. A concise summary of the pathologic classification and clinical correlation of the various types of lung cancer. Google Scholar97. WILLIAMSPAIROLERODAVIS DPC. Survival of patients surgically treated for stage I lung cancer. J Thorac Cardiovasc Surg. 1981;82:70-6. Resection of asymptomatic and especially peripheral cancers without nodal spread may result in 5-year survival of 70% (or greater) of patients. CrossrefMedlineGoogle Scholar98. WOOLNERFONTANASANDERSONMILLERMUHMTAYLORUHLENHAPP LRDWJWM. Mayo lung project: evaluation of lung cancer screening through December 1979. Mayo Clin Proc. 1979;56:544-55. Lung cancer screening is still in the investigative stage regarding improvement of life expectancy. However, screening has been shown to detect a number of asymptomatic cancers in which 5-year survival after resection is quite high. Google Scholar99. LUCECULVER JB. Respiratory muscle function in health and disease. Chest. 1982:81:82-90. Respiratory muscle dysfunction is reviewed as it occurs in chronic obstructive lung disease, asthma, chest wall deformity, obesity, neuromuscular disease, and diaphragmatic paralysis. A section on respiratory muscle training is included. CrossrefMedlineGoogle Scholar100. MACKLEM P. Respiratory muscles: the vital pump. Chest. 1980:78:753-8. A concise review of respiratory muscle function with evaluation of factors affecting efficiency of the system. CrossrefMedlineGoogle Scholar101. HOOPERBEECHLERJOHNSON RCM. Radioisotope scanning in the initial staging of bronchogenic carcinoma. Am Rev Respir Dis. 1978;118:279-86. In patients with bronchogenic carcinoma, liver, brain, and bone scans were used to identify metastatic disease. In the absence of clinical signs, liver and brain scans were uniformly negative whereas only 4% of bone scans were true-positive. MedlineGoogle Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: AMERICAN COLLEGE OF PHYSICIANS Philadelphia, Pennsylvania*Marvin A. Sackner, M.D. Chairman; Ronald P. Daniele, M.D., Paul E. Epstein, M.D., Robert A. Klocke, M.D., R. Drew Miller, M.D., Kenneth M. Moser, M.D., and Adam Wanner, M.D.†Computer literature-file of the National Library of Medicine, Bethesda, Maryland, consulted through the Regional Medical Library service of the College of Physicians of Philadelphia. 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