tbuckley/cabot-search huggingface dataset:
IDENTIFYING THE CORONARY PRONE
Journal: JAMA (1968)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Blood Lead Levels—United States, 1988-1991
Journal: JAMA (1994)

Since the late 1970s, ongoing contamination of the U.S. environment by lead has been substantially reduced as major uses of lead in house paint, gasoline, water-distribution systems, and food cans have been eliminated or reduced (1). During the 1980s, blood lead data from both selected populations and convenience samples indicated a continuation of the decline in blood lead levels (BLLs) (2) observed during 1976-1980 during the Second National Health and Nutrition Examination Survey (NHANES II) (3). However, research during the past two decades has demonstrated adverse health effects at BLLs previously considered to be safe (1). This report summarizes estimates of BLLs in the U.S. population from Phase 1 of the Third National Health and Nutrition Examination Survey (NHANES III), compares these estimates to those from NHANES II, and examines demographic patterns of BLLs among children aged 1-5 years (4,5).
Evaluation of carcinoma of the uterine cervix before radiotherapy
Authors: J. S. Cooper
Journal: JAMA (1979)

In 1974 the Patterns of Care Study began a prospective nationwide survey of radiotherapeutic cancer care in the United States. Cancer of the uterine cervix was one of the diseases surveyed. Pretreatment evaluation data were collected from 170 randomly chosen radiation therapy facilities. The frequency of use of tests in workup varied substantially according, for example, to the severity of disease and the availability of tests. For carcinoma of the uterine cervix, we found that the following tests were used routinely: physical examination, including taking a history; complete blood cell count; serum chemistry analysis; chest roentgenogram; and intravenous pyelogram. Frequent but not routine tests included dilation and curettage and evaluation of the involvement of the bladder and rectum. Other tests were used infrequently. (<i>JAMA</i>242:1996-1997, 1979)
Estimation of splanchnic blood flow by the Fick principle in man and problems in the use of indocyanine green
Authors: A. Uusaro, E. Ruokonen, J. Takala
Journal: Cardiovascular Research (1995)

Journal Article Estimation of splanchnic blood flow by the Fick principle in man and problems in the use of indocyanine green Get access A. Uusaro, A. Uusaro Critical Care Research Program, Department of Intensive Care, Kuopio University Hospital, FIN-70210 Kuopio, Finland Search for other works by this author on: Oxford Academic PubMed Google Scholar E. Ruokonen, E. Ruokonen Critical Care Research Program, Department of Intensive Care, Kuopio University Hospital, FIN-70210 Kuopio, Finland Search for other works by this author on: Oxford Academic PubMed Google Scholar J. Takala J. Takala * Critical Care Research Program, Department of Intensive Care, Kuopio University Hospital, FIN-70210 Kuopio, Finland *Corresponding author. Tel. (+358-71) 173311; Fax (+358-71) 173443. Search for other works by this author on: Oxford Academic PubMed Google Scholar Cardiovascular Research, Volume 30, Issue 1, July 1995, Pages 106–112, https://doi.org/10.1016/S0008-6363(95)00007-0 Published: 01 July 1995 Article history Received: 24 November 1994 Accepted: 13 March 1995 Published: 01 July 1995
Enhancement of Influenza Virus Infections by Secalonic Acid D
Authors: Deborah S. Fleischhacker, Thomas G. Akers, Stephen P. Katz, Muriel S. Palmgren
Journal: Environmental Health Perspectives (1986)

Secalonic acid D (SAD), a hepatotoxic, teratogenic, and slightly mutagenic metabolite of Penicillium oxalicum has been identified as a natural contaminant of grain dust. Secalonic acid D was administered intraperitoneally to male ICR mice that were exposed to influenza virus aerosols 5 days earlier. The mortality rate was significantly higher (p less than 0.001) in mice subjected to both influenza and SAD than those subjected to influenza alone. Virus titers in lung tissue samples at selected time intervals appeared similar for both influenza and influenza-SAD treated groups of mice for 9 days after exposure to the virus. After 9 days, influenza-SAD treated mice appeared to have higher virus titers. No difference in the pathological progression of pneumonia was discernible between these two groups of mice. The influenza-SAD group, in addition to pneumonia, exhibited severe hepatic necrosis characteristic of SAD administration. Mice infected with influenza virus followed by administration of SAD responded with significantly lower (p less than 0.05) antibody titers to influenza virus than mice exposed to influenza virus alone.
Q Fever Linked With Xenotourism
Journal: JAMA (2015)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Garlic as a Treatment for High Cholesterol
Journal: Annals of Internal Medicine (2000)

Summaries for Patients19 September 2000Garlic as a Treatment for High CholesterolSearch for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-133-6-200009190-00004 SectionsAboutFull TextPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail What is the problem and what is known about it so far?Cholesterol is a type of fat. High blood cholesterol levels are associated with an increased risk for heart disease, stroke, and other circulation problems. High cholesterol levels can be treated with a low-fat diet, exercise, or drugs. Some previous studies have suggested that large amounts of garlic in the diet can also lower cholesterol levels. Many of those studies were small, however, which makes them hard to interpret, and the size of the reduction in cholesterol that can be achieved with garlic is uncertain.Why did the researchers ... Author, Article, and Disclosure InformationAffiliations: The summary below is from the full report titled “Garlic for Treating Hypercholesterolemia. A Meta-Analysis of Randomized Clinical Trials.” It is in the 19 September 2000 issue of Annals of Internal Medicine (volume 133, pages 420-429). The authors are C Stevinson, MH Pittler, and E Ernst.Summaries for Patients are a service provided by Annals to help patients better understand the complicated and often mystifying language of modern medicine.Summaries for Patients are presented for informational purposes only. These summaries are not a substitute for advice from your own medical provider. If you have questions about this material, or need medical advice about your own health or situation, please contact your physician. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetailsSee AlsoGarlic for Treating Hypercholesterolemia Clare Stevinson , Max H. Pittler , and Edzard Ernst Metrics 19 September 2000Volume 133, Issue 6Page: I-31KeywordsCardiovascular therapyCholesterolDietDrugsExerciseFatsHypercholesterolemiaStatistical methodsStroke ePublished: 19 September 2000 Issue Published: 19 September 2000 Copyright & PermissionsCopyright © 2000 by American College of Physicians. All Rights Reserved.PDF downloadLoading ...
Abstract 2498: Minor Myocardial Damage and LV Dysfunction in Patients with Duchenne Muscular Dystrophy-The Preventive Efficacy of Carvedilol on Plasma Cardiac Troponin I-
Authors: Takao Nishizawa, Fumihiko Yasuma, Motoko Sakai, Satoshi Kuru, Seigo Kimura, Rei Shibata, Taiki Ohashi, Akiko Noda, Hideo Izawa, Kohzo Nagata, Mitsuhiro Yokota, Masaaki Konagaya, Toyoaki Murohara
Journal: Circulation (2007)

Background: Cardiac dysfunction is one of the major prognostic factors in patients with Duchenne muscular dystrophy (DMD). Minor myocardial damage assessed by plasma cardiac troponin I (cTnI) is often observed in patients with DMD. However, it is unclear that how the minor myocardial damage occurs to which patient with DMD and how it relates to LV dysfunction. Therefore, we assessed the hypothesis that minor myocardial damage is associated with LV dysfunction, the evaluation of plasma cTnI helps the prediction of LV functional deterioration, and carvedilol prevents elevation of plasma cTnI in patients with DMD. Methods: Plasma cTnI were repeatedly (every 3 months) measured for 2 years and LV function was assessed by echocardiography in 58 patients with DMD. Carvedilol (2.5–5 mg/day) was orally administered for a year to the patients who have shown plasma cTnI elevation (positive cTnI, cut off 0.06ng/mL). Results: There were 3 differential groups regarding the progression rate of LV systolic dysfunction, i.e. rapid (19% of total, LVEF &lt; 50% in their 10 th ), slow (50% of total, LVEF &gt; 50% in their 20 –30 th ), and unchanged group (31% of total, LVEF &lt; 50% in their 20 –30 th ). The episode of positive cTnI was observed in 27 (46%) of total patients with DMD. LVEF was lower in patients with positive cTnI than that with negative cTnI (42 ± 2 vs. 52 ± 2%, p &lt; 0.05). Positive cTnI was observed in all patients in rapid group, 84% of patients in slow group, and only 6% of patients in unchanged group. Fourteen differential dystrophin gene mutations were recognized in 48 patients but they were not associated with those 3 differential groups or patients with positive cTnI. Administration of carvedilol in 13 patients (LVEF 40 ± 3) decreased the cTnI detection rate (from 44 ± 5% to 26 ± 10%, p &lt; 0.05), while it was unchanged in 14 patients (LVEF 41 ± 3) without carvedilol treatment (from 44 ± 7% to 39 ± 6%) during same observation period. Conclusions: The elevation of plasma cTnI was associated with LV systolic dysfunction in patients with DMD. The prediction of LV dysfunction in patients with DMD may be feasible with combinations of age, LVEF, and plasma cTnI elevation. Carvedilol could be a new therapeutic strategy to prevent minor myocardial damage in patients with DMD.
COURT-MARTIAL OF A NAVAL MEDICAL OFFICER.
Journal: JAMA (1995)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Application and Evaluation of Portable Field Instruments for Measuring Forced Expiratory Volume of Children and Adults in Environmental Health Surveys
Authors: Robert M. Burton, Walter M. Kozel, Robert L. Penley, George H. Ward, Robert S. Chapman
Journal: Environmental Health Perspectives (1974)

In support of Health Effects Research Studies, pulmonary function tests are periodically administered to a large number of children. The ventilatory performance of these children is being evaluated by measuring the 0.75-sec forced expiratory volume (FEV0.75) with a waterless mechanical volume spirometer used in conjunction with an electronic timing unit. During a 1-yr testing period, operation with the volume spirometer and the EPA designed electronic timing unit proved to be highly successful. The volume spirometer was found to be more advantageous in conducting tests at remote field stations than the water spirometer and other electronic instruments which measure flow rate with a transducer element. The volume spirometer is lightweight, easy to operate, and has the capability of easy and accurate field calibration when used in conjunction with the electronic timing unit. Presently the volume spirometer and EPA designed electronic timing package are employed in all Community Health and Surveillance System (CHESS) pulmonary function testing studies.
Familiality of Postpartum Depression in Unipolar Disorder: Results of a Family Study
Authors: Liz Forty, Lisa Jones, Stuart Macgregor, Sian Caesar, Caroline Cooper, Andrea Hough, Laura Dean, Subodh Dave, Anne Farmer, Peter McGuffin, Shyama Brewster, Nick Craddock, Ian Jones
Journal: American Journal of Psychiatry (2006)

Objective: The authors previously reported strong evidence for familial aggregation of postpartum (puerperal) psychotic episodes in women with bipolar disorder. The authors here examine whether vulnerability to postpartum triggering of depressive episodes aggregates in families and assess how this aggregation varies with the definition of postpartum onset. Method: Postpartum depression occurrence was studied in the female members of 120 sibling pairs recruited at a site within an international multicenter study of sibling pairs with recurrent unipolar depression. Employing a range of definitions of postpartum onset, the authors examined concordance for postpartum episode status between sisters. Results: Episodes of depression with onset within 4 weeks of delivery clustered in families, but there was no significant evidence of familial clustering of broadly defined postpartum depression (onset within 6 months). Among women with a family history of narrowly defined postpartum episodes, 42% experienced depression following their first delivery, whereas only 15% of women with no such family history experienced depression following first delivery. The evidence for familiality maximized with a postpartum onset definition of 6–8 weeks. Conclusions: These results implicate familial factors in susceptibility to the triggering of narrowly defined postpartum depressive episodes in women with recurrent major depression. They suggest that a postnatal onset definition of within 6–8 weeks of delivery may be optimal in studies of the triggering of depressive illness by childbirth.
Medical Maps
Authors: David O. Freedman
Journal: JAMA (2006)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Biochem &amp;amp; Biophys Res Commun, New York
Journal: JAMA (1969)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Remember
Authors: Julia Kreger
Journal: Annals of Internal Medicine (2013)

Ad LibitumSeptember 3, 2013RememberJulia Kreger, BAJulia Kreger, BAFrom Albuquerque, New Mexico.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-159-5-201309030-00016 Audio Reading - “Remember” Audio. Michael A. LaCombe, MD, Annals Associate Editor, reads "Remember," by J. Kreger. Your browser does not support the audio element. Audio player progress bar Step backward in current audio track Play current audio trackPause current audio track Step forward in current audio track Mute current audio trackUnmute current audio track 00:00/ SectionsAboutFull TextPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail Lean in, come closer,Hear me this,Listen to the words my eyes tellIn their glisten of heavy silence and the weakness in these clenched fists.Lean in, listen closely,Pause a second by my bed,As you attend to the sounds of my heart, catch the words of my story that dance between the lubs, and the dubs,That try to break free the chains of these medsLean in, look harder,Behind the yellow of my skin, or the blood in my veins that returns so slowly as you press,Watching,Waiting,ImpatientlyStill pumps ... Author, Article, and Disclosure InformationAffiliations: From Albuquerque, New Mexico.Current Author Address: Julia Kreger, BA; e-mail, [email protected]. PreviousarticleNextarticle Advertisement Audio Reading - “Remember” Audio. Michael A. LaCombe, MD, Annals Associate Editor, reads "Remember," by J. Kreger. Your browser does not support the audio element. Audio player progress bar Step backward in current audio track Play current audio trackPause current audio track Step forward in current audio track Mute current audio trackUnmute current audio track 00:00/ FiguresReferencesRelatedDetails Metrics September 3, 2013Volume 159, Issue 5Page: 369KeywordsBloodHeartLiverVeins ePublished: 3 September 2013 Issue Published: September 3, 2013 Copyright & PermissionsCopyright © 2013 by American College of Physicians. All Rights Reserved.PDF downloadLoading ...
Evidence-based medicine. A new approach to teaching the practice of medicine. Evidence-Based Medicine Working Group
Authors: Gordon Guyatt, John Cairns, David Churchill, Deborah Cook, Brian Haynes, Jack Hirsh, Jan Irvine, Mark Levine, Mitchell Levine, Jim Nishikawa, David Sackett, Patrick Brill-Edwards, Hertzel Gerstein, Jim Gibson, Roman Jaeschke, Anthony Kerigan, Alan Neville, Akbar Panju, Allan Detsky, Murray Enkin, Pamela Frid, Martha Gerrity, Andreas Laupacis, Valerie Lawrence, Joel Menard, Virginia Moyer, Cynthia Mulrow, Paul Links, Andrew Oxman, Jack Sinclair, Peter Tugwell
Journal: JAMA (1992)

A NEW paradigm for medical practice is emerging. Evidence-based medicine de-emphasizes intuition, unsystematic clinical experience, and pathophysiologic rationale as sufficient grounds for clinical decision making and stresses the examination of evidence from clinical research. Evidence-based medicine requires new skills of the physician, including efficient literature searching and the application of formal rules of evidence evaluating the clinical literature. An important goal of our medical residency program is to educate physicians in the practice of evidence-based medicine. Strategies include a weekly, formal academic half-day for residents, devoted to learning the necessary skills; recruitment into teaching roles of physicians who practice evidence-based medicine; sharing among faculty of approaches to teaching evidence-based medicine; and providing faculty with feedback on their performance as role models and teachers of evidence-based medicine. The influence of evidencebased medicine on clinical practice and medical education is increasing. <h3>CLINICAL SCENARIO</h3> A junior medical resident working in a teaching hospital
Multicenter performance evaluation of a new approach to myocardial perfusion SPECT attenuation and scatter correction with resolution recovery Timothy M. Bateman, Lynne L. Johnson, Peter L. Tilkemeier, Ami E. Iskandrian, William P. Follansbee, Gary V. Heller, Daniel S. Berman, Samia M. Arram, Ernest V. Garcia. Mid America Heart Institute, Kansas City, MO, Emory University, Atlanta, GA
Journal: Journal of the American College of Cardiology (2001)