tbuckley/cabot-search huggingface dataset:
MADRID
Journal: JAMA (1933)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
The Child Guidance Approach to Juvenile Delinquency
Journal: JAMA (1952)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Correction
Journal: BMJ (1959)

redness of the nose denotes a chronic " boozer" is, however, not well founded.It is well known that many people who are extremely temperate may have a red nose and that this feature is frequently absent in chronic alcoholics.In hepatic cirrhosis, whether due to alcohol or to other causes, the presence of spider naevi and telangiectasia of the vessels of the face may cause the so-called " hepatic complexion," of which redness of the nose is one feature.
INFECTION AND IMMUNITY IN CHRONIC LYMPHOCYTIC LEUKEMIA
Journal: JAMA (1960)

Shaw and co-workers<sup>1</sup>present a study of the immunologic response of patients with chronic lymphocytic leukemia to bacterial infection. In their experience the absolute granulocyte counts, the polymorphonuclear response to infection, and the phagocytic function of the polymorphonuclear cells were found to be normal in these patients. However, a decreased or even absent circulating antibody response to antigenic stimulation was observed. These findings pointed to a failure of the immunologic response. In a group of 18 patients with chronic lymphocytic leukemia admitted to the Clinical Center at Bethesda, Md., 10 patients, according to Shaw, had multiple bacterial infections and five of these died. Hypogammaglobulinemia in this group appeared to be directly related to the incidence and severity of these infections. A study of the serum γ-globulin concentrations and circulating antibody production was undertaken with regard to bacterial infection. In a later group of 42 patients with chronic lymphocytic leukemia
Echovirus 9 meningoencephalitis in infants and children
Authors: R. E. Haynes
Journal: JAMA (1969)

Echovirus 9 meningoencephalitis in 196 children was confirmed by the isolation of the virus from the cerebrospinal fluid of each patient. Although the illness was usually mild, one patient died. The cerebrospinal fluid (CSF) leukocyte counts were unusually high in some patients, and CSF protein content was over 100 mg/100 cc in nine patients. The glucose content of all specimens of cerebrospinal fluid was normal. Echovirus 9 was recovered from 57% of the CSF specimens of patients with a clinical diagnosis of aseptic meningoencephalitis who were studied between May 19, 1968, and Sept 30, 1968. In patients who had specimens from multiple sites available for culture, 76 yielded echovirus 9 from one or more sites, and of these 76 patients, 66 (87%) had virus recovered from a CSF specimen.
Low-density lipoprotein density and atherosclerosis. Unraveling the connection
Authors: A. H. Slyper
Journal: JAMA (1994)

<h3>Objective.</h3> —To provide an overview of the relationship between low-density lipoprotein (LDL) density and coronary artery disease and to examine this relationship in terms of lipid peroxidation and the atherogenic remnant hypothesis. <h3>Data Sources.</h3> —English-language studies and reviews pertaining to LDL composition and size were identified through a MEDLINE search and reference citations. <h3>Study Selection.</h3> —Clinical studies on the following topics were critically reviewed: (1) the association between LDL density and coronary artery disease, (2) the relationship between LDL density and the levels and composition of other lipoproteins, and (3) the influence of environmental and genetic factors on LDL density. <h3>Data Synthesis.</h3> —Low-density lipoprotein is a heterogeneous lipoprotein containing many subpopulations identifiable on the basis of density and size. There is an increased prevalence of small, dense LDL particles in coronary artery disease and conditions commonly associated with atherogenesis, such as non—insulin-dependent diabetes mellitus and familial combined hyperlipidemia. Exercise, visceral fat accumulation, and diet influence LDL density. In comparison with buoyant LDL, dense LDL is associated with a more atherogenic type of lipoprotein profile. An important relationship exists between the triglyceride level and the formation of LDL subpopulations. <h3>Conclusion.</h3> —Unraveling the connection between dense LDL and atherogenesis should provide important insights into the basic mechanisms underlying atherogenesis. (<i>JAMA</i>. 1994;272:305-308)
Essays for Students.
Journal: JAMA (1899)

These essays or lectures were prepared for students, and are more or less elementary. There are four essays, the subjects being "Strangulated Hernia," "Cancer of the Breast," "Some Run Over Cases," and "Elements of Aural Surgery." The three former are made up of reports of cases, and the lessons to be deduced from them. The last, which takes up nearly half the pages of the book, is the most important of the series, and covers practically the whole range of aural disease.
Psychiatric problems in obese children followed up for 10 years
Authors: A. M. Schindler
Journal: JAMA (1991)

<h3>To the Editor.—</h3> The report by Epstein et al,<sup>1</sup>"Ten-Year Follow-up of Behavioral, Family-Based Treatment for Obese Children," notes long-term positive results with that mode of treatment applied to 76 selected patients: obese children with intact families, no history of psychiatric contact, at least one obese parent, and one parent willing to attend treatment meetings. Noted by the authors, but otherwise overlooked in the discussion of the outcome of this study, were the psychopathological effects that occurred among these 76 supposedly psychologically healthy children. Near the beginning of the study, one of the 76 children was dropped because of "unreported psychiatric problems." During the second 5 years, six of the 67 children who were still eligible for the study developed "significant psychiatric problems that required hospitalization and long-term medication use." Three children had manic-depressive illness, two had major depression with suicide attempts, and one developed an eating disorder. Finally,
Osteosclerotic myeloma
Authors: A. R. Rodriguez
Journal: JAMA (1976)

A 49-year-old man with an osteosclerotic lesion of a thoracic vertebra had peripheral neuropathy, lymphadenopathy, and elevated serum alkaline phosphatase and monoclonal immunoglobulin levels in the cerebrospinal fluid. These findings, not usually seen in "classical" myeloma, have been noted in different combinations in the osteosclerotic variety. (<i>JAMA</i>236:1872-1874, 1976)
Information on treatment INDs as they become available to the practitioner
Authors: F. E. Young
Journal: JAMA (1988)

The Food and Drug Administration has initiated a new system for making available certain investigational drugs—for the treatment of desperately ill patients for whom there is no effective alternative therapy—prior to their approval for marketing. This system and the process involved in identifying and designating those drugs as "Treatment INDs" have been described previously.<sup>1</sup>As these drugs for certain patients with serious or life-threatening diseases become more available under the Treatment IND process, it is important for the Food and Drug Administration to directly inform the practitioner about the drugs and about the treatment protocols under which they may be used. Most important for physicians who may want to pursue use of one of these drugs for their patients is the need to provide and publicize a contact (eg, a telephone source) for further information on each drug, especially information on matters such as patient selection criteria and where
Bronchiolitis
Authors: D. Isaacs
Journal: BMJ (1995)

plasty in myocardial infarction trial no retinal haemorrhages were seen in 148 diabetic patients, 7% of whom had documented retinopathy.'2Patients with diabetes have a poor outcome after myo- cardial infarction, yet-as for some other groups with a poor prognosis-they are less likely to receive thrombolytic treat- ment.2We believe that in the light of the proved benefits of thrombolysis in patients with acute myocardial infarction the treatment should not be withheld on the basis of the existence of diabetic retinopathy.4Any increase in risk seems to be small.Those who support the empowerment of patients might also suggest that this is a decision in which it is appropriate for the patient to have a voice.
Corrections
Authors: M Eng, F Ross, L Eng, C Rowntree, W Scott, M Toronto, C Eng, L Lond, G Thompson, St Mary's Hospital, C Walters, B Middleton, W Wiley, J Leech, J Rolmes, J Nichol- Son, E Collen, H Boxwell, H O', R May, T Clampett, M Farquharson, M Greenwood
Journal: BMJ (1903)
Lidocaine intramuscularly in acute myocardial infarction
Authors: V. Bernstein
Journal: JAMA (1972)

Two hundred milligrams of lidocaine hydrochloride was given intramuscularly for controlling ventricular premature beats in 40 patients continuously monitored in an intensive care unit. In 14 patients, serum levels were determined by gas chromatographic technique. The treatment abolished premature ventricular beats in 30 of the 40 patients. Eight of the failures had persistent premature ventricular beats over a long period before admission to hospital. Serum levels and clinical results showed that effective drug concentrations were reached between 15 to 30 minutes after injection and that therapeutic levels were maintained for at least 60 minutes. It is suggested that lidocaine should be given intramuscularly to patients suffering an acute myocardial infarction before they are moved to hospital to prevent serious ventricular arrhythmias during the prehospital phase.
Fetal Alcohol Syndrome--Alaska, Arizona, Colorado, and New York, 1995-1997
Journal: JAMA (2002)

Fetal alcohol syndrome (FAS) is caused by maternal alcohol use during pregnancy and is one of the leading causes of preventable birth defects and developmental disabilities in the United States. FAS is diagnosed on the basis of a combination of growth deficiency (pre- or postnatal), central nervous system (CNS) dysfunction, facial dysmorphology, and maternal alcohol use during pregnancy. Estimates of the prevalence of FAS vary from 0.2 to 1.0 per 1,000 live-born infants. This variation is due, in part, to the small size of the populations studied, varying case definitions, and different surveillance methods. In addition, differences have been noted among racial/ethnic populations. To monitor the occurrence of FAS, CDC collaborated with five states (Alaska, Arizona, Colorado, New York, and Wisconsin*) to develop the Fetal Alcohol Syndrome Surveillance Network (FASSNet). This report summarizes the results of an analysis of FASSNet data on children born during 1995-1997, which indicate that FAS rates in Alaska, Arizona, Colorado, and New York ranged from 0.3 to 1.5 per 1,000 live-born infants and were highest for black and American Indian/Alaska Native populations. This study demonstrates that FASSNet is a useful tool that enables health care professionals to monitor the occurrence of FAS and to evaluate the impact of prevention, education, and intervention efforts.
Lunch
Authors: Christine Y. Todd
Journal: Annals of Internal Medicine (2019)

On Being a Doctor21 May 2019LunchChristine Y. Todd, MDChristine Y. Todd, MDSouthern Illinois University School of Medicine, Springfield, Illinois (C.Y.T.)Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/M18-2609 Audio Reading - “Lunch” Audio. Virginia Hood, MD, reads “Lunch” by Christine Y. Todd, MD Your browser does not support the audio element. Audio player progress bar Step backward in current audio track Play current audio trackPause current audio track Step forward in current audio track Mute current audio trackUnmute current audio track 00:00/ SectionsAboutFull TextPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail Yesterday I ate lunch in a way that reminded me of a very specific time and place.When I was an intern, I spent October on the oncology ward. It was housed in an old building attached to the main hospital by a series of walkways and tunnels. It contained an assembly of horrors, patients who had reached depths and crescendoes of sickness I had never encountered before. You could hear honest-to-God wailing. None of the admissions would end up with a short 3-day stay. If you were admitted to oncology, you were in a world of hurt. Every story ... Author, Article, and Disclosure InformationAffiliations: Southern Illinois University School of Medicine, Springfield, Illinois (C.Y.T.)Corresponding Author: Christine Y. Todd, MD, Southern Illinois University School of Medicine, 913 North Rutledge, Suite 1100, Springfield, IL 62702; e-mail, [email protected]edu. PreviousarticleNextarticle Advertisement Audio Reading - “Lunch” Audio. Virginia Hood, MD, reads “Lunch” by Christine Y. Todd, MD Your browser does not support the audio element. Audio player progress bar Step backward in current audio track Play current audio trackPause current audio track Step forward in current audio track Mute current audio trackUnmute current audio track 00:00/ FiguresReferencesRelatedDetails Metrics 21 May 2019Volume 170, Issue 10Page: 730KeywordsBehaviorBreast cancerChemotherapyEatingFeversInterleukinsPatientsPharmacistsResidencyWalking ePublished: 21 May 2019 Issue Published: 21 May 2019 Copyright & PermissionsCopyright © 2019 by American College of Physicians. All Rights Reserved.PDF downloadLoading ...
Presidential Families Support Breast Cancer Awareness Month
Journal: JNCI Journal of the National Cancer Institute (1991)

Presidential Families Support Breast Cancer Awareness Month Get access JNCI: Journal of the National Cancer Institute, Volume 83, Issue 20, 16 October 1991, Page 1449, https://doi.org/10.1093/jnci/83.20.1449-a Published: 16 October 1991
Cost-Effective Treatment of Candida Esophagitis
Authors: Loren Laine
Journal: Annals of Internal Medicine (1993)

Letters15 May 1993Cost-Effective Treatment of Candida EsophagitisLoren Laine, MDLoren Laine, MDSearch for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-118-10-199305150-00016 SectionsAboutFull TextPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail IN RESPONSE:We strongly agree that cost should always be considered when prescribing a medication. Our study, however, like most randomized, controlled trials, was designed to evaluate the safety and efficacy of two agents, fluconazole and ketoconazole, for candida esophagitis. We did not conclude that either agent was the treatment of choice. We feel that the reader can use the results of our study in combination with other considerations such as local drug costs and individual patient characteristics (for example, previous antifungal therapy) to make decisions about treatment.A formal decision analysis incorporating cost could be extremely helpful in the ...REFERENCE1. Laine L, Dretler RH, Conteas CN, Tuazon C, Koster FM, Sattler F, et al. Fluconazole compared with ketoconazole for the treatment of candida esophagitis in AIDS: a randomized trial. Ann Intern Med. 1992; 117:655-60. Google Scholar Author, Article, and Disclosure InformationAffiliations: PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics 15 May 1993Volume 118, Issue 10Page: 824-825KeywordsAntifungalsCandidaDecision analysisDrug therapyEndoscopyHIV infectionsImmune deficiencyReproducibilitySafetySafety studies ePublished: 15 August 2000 Issue Published: 15 May 1993 Copyright & PermissionsCopyright © 1993 by American College of Physicians. All Rights Reserved.PDF downloadLoading ...
Congestive Heart Failure
Authors: Samuel Vaisrub
Journal: JAMA (1967)

Like the last chapter of a tragic novel, cardiac failure cannot be fully understood without the knowledge of preceding events. As a terminal phase of most cardiac disorders, it cannot be lifted out of the general context of heart disease. A book devoted to heart-failure must touch on many aspects of heart disease and, in the process, run the risk of sacrificing depth for breadth. This sacrifice has not been successfully avoided in Myerson and Pastor's monograph. Some subjects are given superficial treatment. In the paragraph on chemical factors in vasomotor control, we are told that "serotonin, 5-hydroxytryptophane, is a peripheral vasodilator"—the entire complex subject of serotonin effects on the circulation dismissed in one short sentence. Controversial statements are made without supporting evidence. Acetylcholine is stated to have a positive inotropic action on the ventricular myocardium, but no evidence is presented for what is, at best, a debatable concept. Potassium
The Cleveland Museum of Health
Journal: JAMA (2015)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Journals JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
The effects of thermal stimulation on the ultrastructure of the human atrichial sweat gland. I. The fundus
Authors: I. MONTGOMERY, D.McEWAN JENKINSON, H.Y. ELDER, D. CZARNECKI, R.M. MacKIE
Journal: British Journal of Dermatology (1984)

Ultrastructural examination of sweat glands from the human loin before and during heat-induced activity indicated that the sweat is formed from the contents of disrupted cells as well as from the products of secretion. The principal secretory processes appear to be fluid transport and exocytosis of vesicles. However, configurations suggesting microapocrine secretion were also observed. It is concluded that the mechanisms involved in sweat production in man are fundamentally similar to those in animals and the terms ‘apocrine’ and ‘eccrine’ should be discarded. The myocpithelial cells which were contracted at the onset of sweating appeared to be under less tension after 3 h of continuous activity.
Correction: Antiretroviral Therapy and HIV Shedding
Journal: Annals of Internal Medicine (2001)

Corrections1 May 2001Correction: Antiretroviral Therapy and HIV SheddingFREESearch for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-134-9_Part_1-200105010-00028 SectionsAboutVisual AbstractPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail In an article on the effect of antiretroviral treatment on HIV shedding in semen (1), the third sentence of the first paragraph on page 282 (under the heading “Effect of Therapy on HIV Shedding”) should read “Six months after introduction of therapy, HIV RNA was detected in 21 (25%) semen samples and 38 (44%) blood samples,” not “… in 29 (33%) semen samples and 33 (38%) blood samples.”Reference1. Barroso PF, Schechter M, Gupta P, Melo MF, Vieira M, Murta FC, et al . Effect of antiretroviral therapy on HIV shedding in semen. Ann Intern Med. 2000;133:280-4. [PMID: 00010929169] LinkGoogle Scholar Comments0 CommentsSign In to Submit A Comment Author, Article, and Disclosure InformationAffiliations: PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetailsSee AlsoEffect of Antiretroviral Therapy on HIV Shedding in Semen Paulo F. Barroso , Mauro Schechter , Phalguni Gupta , Maria F. Melo , Marcello Vieira , Fernanda C. Murta , Yeda Souza , and Lee H. Harrison Metrics 1 May 2001Volume 134, Issue 9_Part_1Page: 799KeywordsAntiretroviral therapyAntiretroviralsBloodHIVRNASemen ePublished: 1 May 2001 Issue Published: 1 May 2001 Copyright & PermissionsCopyright © 2001 by American College of Physicians. All Rights Reserved.PDF downloadLoading ...