tbuckley/cabot-search huggingface dataset:
COMPARATIVE ANATOMY EXHIBITS IN WASHINGTON.
Journal: JAMA (1897)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Recurrent Scarlatina.
Authors: H. M. Byall
Journal: JAMA (1896)

Montpelier, Ohio, April 29, 1896. <h3>To the Editor:</h3> —The following is to me a unique case, and may prove so to many of the brethren. I therefore desire to report it. Was called some time in January of this year to see a boy 10 years of age, suffering from well defined scarlatina. It ran the usual course, desquamation being complete even to the finger and toe nails. Four weeks ago, being the last of March, saw him again, having scarlatina; case pursued usual course, desquamation as before. He had been up and around about a week, when a third attack seized him; desquamation is at the present time going on. The exfoliation is so complete and followed so soon after the second attack, that patient is rendered extremely sensitive, in some situations blood oozing through the skin. Five years ago, according to the parents, the boy had an attack
Nitrate Methemoglobinemia
Authors: Harold Marcus, Joy R. Joffe
Journal: New England Journal of Medicine (1949)

MORE than forty years have passed since the first report of a fatal case of methemoglobinemia secondary to bismuth subnitrate ingestion.1 Fifteen years ago Roe2 reviewed the subject and reemphasized the hazards attending its use. Nevertheless, during the past four years 3 more cases have been reported.3 4 5 A recent experience with a newborn infant has prompted us to review the general subject of nitrate methemoglobinemia, with emphasis on the causes, the types encountered, the mechanism involved and the rationale of present-day therapy.Thirty cases of methemoglobinemia secondary to bismuth subnitrate, ammonium nitrate and nitrates in well water have been reported. . . .
Request for Abstracts
Journal: JAMA (1967)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
PEDIATRICS
Authors: James M. Sutherland
Journal: JAMA (1966)

Nelson's<i>Textbook of Pediatrics</i>(ed 8, Saunder, 1964, $18) is the most appealing pediatrics text, with<i>Pediatrics</i>by L. E. Holt, R. McIntosh, and H. L. Barnett (ed 13, Appleton, 1962, $18) running a close second. Although many of the other available texts have features to recommend them, these two remain the favorites of practitioners, students, and teachers. The yearly<i>Yearbook of Pediatrics</i>, superbly edited by Sydney Gellis (Year Book, 1965, $8.50) and brightened by lively discussions, provides the best single means of keeping informed on details of reasonably current papers. Certain miscellaneous texts deserve special mention. The posthumous edition of Wilkins' text (<i>The Diagnosis and Treatment of Endocrine Disorders in Childhood and Adolescence</i>, ed 3, Thomas, 1965, $26.50), written with the editorial assistance of Blizzard and Migeon, updates this excellent standard work.<i>Pediatric Procedures</i>by W. T. Hughes, Jr. (Saunders, 1964, $7.50) is the first reference on this subject,
Santo Domingo
Authors: Arthur L. Springer
Journal: JAMA (1962)

<h3>To the Editor:—</h3> With reference to your comment (<i>JAMA</i><b>178</b>:440 [Oct. 28] 1961) regarding "Trepanation," I would like to call your attention to the fact that there is no such place as San Domingo. The expression "San Domingo" does not occur in the Spanish language. The University of Santo Tomás is in Santo Domingo.
Schweizerische Medizinische Wochenschrift, Basel
Journal: JAMA (1970)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Bacterial Shock.
Authors: Abraham I. Braude
Journal: Annals of Internal Medicine (1964)

s1 October 1964Bacterial Shock.Abraham I. Braude, M.D.Abraham I. Braude, M.D.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-61-4-808_2 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptThe clinical features of bacterial shock often deviate from the classic picture of shock because of the peculiar effects of each microorganism or its toxin. Such deviations from classic shock take many forms, including the hot, dry, and flushed appearance often evident early in septicemia, especially that due to gram-negative organisms; the unusual mental alertness that characterizes patients with clostridial septicemia; and the bradycardia sometimes seen in myocarditis. Although most reports dealing with the problem of shock in infections are concerned with cases of demonstrated bacteremia and especially those due to gram-negative bacilli, intravascular bacterial multiplication is not a prerequisite... This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAuthors: Abraham I. Braude, M.D.Affiliations: Pittsburgh, Pa. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics 1 October 1964Volume 61, Issue 4Page: 808-808KeywordsBacteremiaBradycardiaGram negative bacterial infectionsMyocarditisSepsisShockToxins ePublished: 1 December 2008 Issue Published: 1 October 1964 PDF downloadLoading ...
Inhalation of Diesel Engine Exhaust Affects Spermatogenesis in Growing Male Rats
Authors: Nobue Watanabe, Yoji Oonuki
Journal: Environmental Health Perspectives (1999)

We conducted experiments to determine whether diesel engine exhaust affects reproductive endocrine function in growing rats. The rats were assigned to three groups: a group exposed to total diesel engine exhaust containing 5.63 mg/m3 particulate matter, 4.10 ppm nitrogen dioxide, and 8.10 ppm nitrogen oxide; a group exposed to filtered exhaust without particulate matter; and a group exposed to clean air. Dosing experiments were performed for 3 months beginning at birth (6 hr/day for 5 days/week). Serum levels of testosterone and estradiol were significantly higher in animals exposed to total diesel exhaust and filtered exhaust (p &lt; 0.05 for each group) as compared to the controls. Follicle-stimulating hormone was significantly decreased in the two groups exposed to diesel exhaust as compared to the control group (p &lt; 0.05). Luteinizing hormone was significantly decreased in the total exhaust-exposed group as compared to the control and filtered groups (p &lt; 0.05). Although testis weight did not show any significant difference among the groups, sperm production and activity of testicular hyaluronidase were significantly reduced in both exhaust-exposed groups as compared to the control group. Histological examination showed decreased numbers of step 18 and 19 spermatids in stage VI, VII, and VIII tubules in the testes of both diesel exhaust-exposed groups. This study suggests that diesel exhaust stimulates hormonal secretion of the adrenal cortex, depresses gonadotropin-releasing-hormone, and inhibits spermatogenesis in rats. Because these effects were not inhibited by filtration, the gaseous phase of the exhaust appears to be more responsible than particulate matter for disrupting the endocrine system.
Cumulative List of United States Adopted Names
Journal: JAMA (1968)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
The Pulmonary Macrophage
Authors: Drummond H. Bowden
Journal: Environmental Health Perspectives (1976)

An overview of the pulmonary macrophage is provided, with particular emphasis on the origin of this cell and the adaptive mechanisms whereby the macrophagic system is able to respond to increased inhalant loads of organic and inorganic pollutants. Evidence is presented which favors an hematopoietic origin for the alveolar macrophage with a monocytic transportation compartment in the blood and an interstitial cell compartment in the lung in which cellular division and maturation may occur. Through the simple mechanism of increased cellular turnover this system of mononuclear phagocytes rapidly adapts to most inhalant challenges. In addition to its primary tasks phagocyte and destroyer of microorganisms the macrophage plays a pivotal role in the genesis of silicotic fibrosis, and it is possible that similar mechanisms may hold for a variety of cryptogenic fibroses. Paradoxically, destruction of collagen by the dual mechanisms of phagocytosis and the secretion of lytic enzymes may also occur. The relevance of this secretory function of the macrophage to the pathogenesis of destructive diseases of the lung such as emphysema remains to be determined.
Electricity in the Diseases of Women, with Special Reference to the Application of Strong Currents.
Authors: None F. H. M.
Journal: JAMA (1889)

Taken as a whole the work done by Dr. Massey in his "Electricity in the Diseases of Women," is very satisfactory, and helps to fill a deeply-felt need of the practitioner, viz: a modern scientific work on electricity. The rapid development and recent scientific demonstrations of this subject have rendered former authorities almost absolute, and as yet comparatively nothing has arisen to take their place. While Dr. Massey's book covers but a limited portion of this vast subject, it still is in most respects, scientific and will prove of assistance to the practitioner who is desirous of employing electricity as a therapeutic agent. But little space is occupied by the consideration of the physics of electricity proper, but the detailed experiments given in chapters III and IV are well conceived, and will do much toward clearing up this intricate subject to the minds of the uninitiated, and they should be
Abstract 1810: LncCGU1 and AFP serve as important prognostic marker in non-hepatitis B non-hepatitis C hepatocellular carcinoma
Authors: Kwang-Huei Lin, Yang-Hsiang Lin, Yu-Chin Liu
Journal: Cancer Research (2019)

Abstract Background: Thyroid hormone (T3) and its receptor (TR) are involved in cell metabolism and cancer progression. Hypothyroidism is a significantly elevated risk factor for hepatocellular carcinoma (HCC). Alpha-fetoprotein (AFP) is increased in the majority of patients with HCC and may be useful in the diagnosis and follow-up of cases of HCC. The relationship between T3/TR and AFP levels in HCC is still unclear. Design: We used microarrays to compare expression profiles of lncRNAs in HepG2-TRα1 cells treated with/without T3 and HCC specimens. The profiling results were validated using qRT-PCR. The overall survival and recurrence-free survival were analyzed using Kaplan-Meier method and further confirmed by online available datasets. Results: In the current study, long non-coding RNA CGU1 was downregulated by T3/TR in vitro. Notably, α-fetoprotein (AFP) mRNA levels were downregulated by T3/TR through repression of CGU1. Notably, AFP mRNA is strongly positively correlated with CGU1 and unfavorable prognosis in non-hepatitis B/non-hepatitis C HCC (NBNC-HCC) group patients. Conclusions: our data collectively suggest that T3/TR/ CGU1/AFP act as a potential prognostic marker in NBNC-HCC. Citation Format: Kwang-Huei Lin, Yang-Hsiang Lin, Yu-Chin Liu. LncCGU1 and AFP serve as important prognostic marker in non-hepatitis B non-hepatitis C hepatocellular carcinoma [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2019; 2019 Mar 29-Apr 3; Atlanta, GA. Philadelphia (PA): AACR; Cancer Res 2019;79(13 Suppl):Abstract nr 1810.
Annales d'Oculistique, Paris
Journal: JAMA (1966)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Neural Circuitry Engaged During Unsuccessful Motor Inhibition in Pediatric Bipolar Disorder
Authors: Ellen Leibenluft, Brendan A. Rich, Deborah T. Vinton, Eric E. Nelson, Stephen J. Fromm, Lisa H. Berghorst, Paramjit Joshi, Adelaide Robb, Russell J. Schachar, Daniel P. Dickstein, Erin B. McClure, Daniel S. Pine
Journal: American Journal of Psychiatry (2007)

Objective: Deficits in motor inhibition may contribute to impulsivity and irritability in children with bipolar disorder. Studies of the neural circuitry engaged during failed motor inhibition in pediatric bipolar disorder may increase our understanding of the pathophysiology of the illness. The authors tested the hypothesis that children with bipolar disorder and comparison subjects would differ in ventral prefrontal cortex, striatal, and anterior cingulate activation during unsuccessful motor inhibition. They also compared activation in medicated versus unmedicated children with bipolar disorder and in children with bipolar disorder and attention deficit hyperactivity disorder (ADHD) versus those with bipolar disorder without ADHD. Method: The authors conducted an event-related functional magnetic resonance imaging study comparing neural activation in children with bipolar disorder and healthy comparison subjects while they performed a motor inhibition task. The study group included 26 children with bipolar disorder (13 unmedicated and 15 with ADHD) and 17 comparison subjects matched by age, gender, and IQ. Results: On failed inhibitory trials, comparison subjects showed greater bilateral striatal and right ventral prefrontal cortex activation than did patients. These deficits were present in unmedicated patients, but the role of ADHD in mediating them was unclear. Conclusions: In relation to comparison subjects, children with bipolar disorder may have deficits in their ability to engage striatal structures and the right ventral prefrontal cortex during unsuccessful inhibition. Further research should ascertain the contribution of ADHD to these deficits and the role that such deficits may play in the emotional and behavioral dysregulation characteristic of bipolar disorder.
The Board of Trustees
Journal: JAMA (1984)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Toxic Agoraphobia
Authors: Thomas L. Kurt
Journal: Annals of Internal Medicine (1990)

Letters and Corrections1 February 1990Toxic AgoraphobiaThomas L. Kurt, MD, MPH, Timothy J. Sullivan III, MDThomas L. Kurt, MD, MPH, Timothy J. Sullivan III, MDAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-112-3-231 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptTo the Editor: Kahn and Letz's recent editorial (1) on clinical ecology noted that the American College of Physician's position paper and studies by Brodsky, Stewart, and Terr suggest that clinical ecology methods can lead to intense fear of injury from various chemicals or foods associated with increased symptoms or to withdrawal from normal human activities. Our independent medical assessments of significant numbers of clinical ecology patients strongly support this concept.In addition to instructions for avoidance of exposure to chemicals deemed offensive, clinical ecology patients often are given ethanol injections or intravenous sodium bicarbonate to "neutralize" their reactions. Detoxification...References1. KahnLetz EG. Clinical ecology: environmental medicine or unsubstantiated theory? [Editorial]. Ann Intern Med. 1989;111:104-5. LinkGoogle Scholar2. Sheehan D. Panic attacks and phobias. N Engl J Med. 1982;307:156-8. CrossrefMedlineGoogle Scholar3. . Diagnostic Criteria from DSM-III-R. Washington, D.C.: American Psychiatric Association; 1987. Google Scholar4. Rea W. v. The Aetna Life Insurance Company and the Prudential Insurance Companies of America. Civil Action No. 2-84-0219-H. U.S. Dist. Ct., N. Dist. Texas, Dallas Div. Feb. 25 1985. Google Scholar5. SchickeleRhodes SD. Re: Motion in limine regarding A Levin. Civil Action No. C-451843. Sup. Ct. Arizona, Maricopa County. Jul. 16 1986. Google Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics Cited ByRepression of let‐7b‐5p prevents the development of multifidus muscle dysfunction by promoting vitamin D accumulation via upregulation of electron transfer flavoprotein alpha subunit in a rat model of multifidus muscle injuryMultiple Chemical Sensitivities–A Syndrome of Pseudotoxicity Manifest as Exposure Perceived SymptomsEditorial Commentary: Multiple Chemical Sensitivities–Public Policy 1 February 1990Volume 112, Issue 3Page: 231-232KeywordsBicarbonatesDetoxificationEcologyEthanolFearFoodSodium Issue Published: 1 February 1990 PDF DownloadLoading ...