tbuckley/cabot-search huggingface dataset:
Ancestors
Authors: Carol Snyder Halberstadt
Journal: JAMA (2018)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Diabetic Ketoacidosis
Authors: Clifford W. Zwillich
Journal: JAMA (1970)

<h3>To the Editor:—</h3> I read with great interest the article "Severe Diabetic Ketoacidosis" (<b>210:</b>1082, 1969). The authors suggest that the first intravenous administration should contain "approximately 150 mEq of sodium," and later state that hyperosmolality is common in ketoacidosis. They suggest that the hyperosmotic state be considered after the initial 2,000 cc is already infused. The hyperosmotic state of ketoacidosis should be treated from the onset of therapy as it is not an uncommon cause of early death in ketoacidosis. This can easily be accomplished by using 0.45% sodium chloride solution with 44 mEq sodium bicarbonate added. This would make a solution with about 120 mEq/liter of sodium and would begin immediately to correct the hyperosmotic state. The article later suggests starting potassium replacement more vigorously if the patient has a known history of azotemia because "potassium levels are already decreased at the time of admission." Treatment of patients
Medical Specialization
Authors: G. G. Liddle
Journal: JAMA (1968)

<h3>To the Editor:—</h3> I am dismayed and embarrassed to have to report to your readers the lax attitude in medicine toward and by its specialists. Other fields have marched forward, in step with progress, while medicine has dillied and dallied with the principles of Empedocles and Hippocrates, finally emerging with Gilbertus Anglicus, clinging to medieval specialties such as surgery, obstetrics, and diseases of children. Reading my favorite medical journal, (second only to your esteemed periodical) the<i>New York Times Sunday Juice</i>, I discovered titles for business specialists far more advanced than those in medicine. A commercial firm sought a "Genius in Software Consulting." Could they have wanted a cardiologist or a brain surgeon? I don't think so, but what more poetic delirium for a medical specialty have you in<i>your</i>backyard? My point is clear. Medicine is retarded. Where are our Senior Programming Systems Analysts? Where are medicine's Procurement Technique
特应性皮炎和教育程度
Authors: J. Smirnova, L.B. Kobyletzki, M. Lindberg, Å. Svensson, S.M. Langan, S. Montgomery
Journal: British Journal of Dermatology (2019)

特应性皮炎 (AD, 通常称为湿疹) 是一种影响多达 20% 儿童的常见皮肤病。但是, 对于此病的某些长期影响, 如教育程度 (即一个人在学校、大学或学院中达到的教育水平), 我们的了解尚不完整。AD 通常是可见的, 会导致瘙痒和 (有时的) 失眠,因此可能影响一个人的教育。在此研究中使用瑞典注册数据比较了有和无 AD 青少年男性的教育水平,还比较了认知功能 (让我们完成任务的脑力过程) 和抗压能力 (应对压力的能力)。这项研究纳入了 23 4,715 名在 1969 至 1976 年间接受了兵役评估的 17‐20 岁男性: 他们的评估涉及大量的生理和心理检查, 包括一项书面认知功能测试。共有 1,673 人 (0.7%) 被诊断为 AD。令人惊讶的是,AD 与教育程度或认知功能水平下降无关。事实上观察到教育和认知功能水平轻微增高,这似乎可通过 AD 男性父母的社会经济地位更高,因此导致教育表现提升有关。但是, AD 患者应对压力的能力似乎较弱, 已证实这与成年期后期中不利的心理和生理健康结局有关。
Heredity and Eugenics.
Journal: JAMA (1913)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Hyperleptinemia in chronic heart failure relates to growth hormone binding protein in male patients with and without cardiac cachexia Wolfram Doehner, Mathias Rauchhaus, Karl Egerer, Claus D. Pflaum, Christian J. Strasburger, Constantinos H. Davos, Andrew J. S. Coats, Stefan D. Anker. Clinical Cardiology, NHLI, London, United Kingdom, Franz Volhard Klinik at MDC, Charit�, Berlin, Germany
Journal: Journal of the American College of Cardiology (2001)
Surgical Diagnosis. Volume I and Volume II.
Journal: JAMA (1931)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Abstract 4311: Mechanisms responsible for the inflammatory polarization of monocytes by subclinical low-dose LPS
Authors: Kisha Pradhan
Journal: Cancer Research (2019)

Abstract Dynamic polarizations of innate leukocytes such as monocytes and macrophages have been increasingly recognized to play key roles in the pathogenesis and/or treatment of human disease. However, mechanisms responsible for distinct polarizations of monocytes are not well understood. Our previous studies indicate that monocytes can be persistently polarized into a low-grade inflammatory state by subclinical super-low dose LPS (bacterial lipopolysaccharide). In this current study, we further characterized the unique polarization of monocytes by super-low dose LPS and its underlying mechanisms. We observed that super-low dose LPS preferentially induced the expression of inflammatory mediators such as MCP-1 while potently suppressed the expression of homeostatic mediators such as Catalase and FPN. Super-low dose LPS activated the inflammatory signaling molecules such as p38, CamKII, and NFkB. In contrast, super-low dose LPS suppressed the basal activation of immune suppressing molecules such as AMPK and SIRT3. Taken together, our data reveal novel insights regarding the persistent polarization of inflammatory monocyte, an emerging key player in various human diseases including cancer. Citation Format: Kisha Pradhan. Mechanisms responsible for the inflammatory polarization of monocytes by subclinical low-dose LPS [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2019; 2019 Mar 29-Apr 3; Atlanta, GA. Philadelphia (PA): AACR; Cancer Res 2019;79(13 Suppl):Abstract nr 4311.
Randomised phase 3 trial of enzalutamide in first line androgen deprivation therapy for metastatic prostate cancer: the ANZUP ENZAMET Trial (ANZUP 1304).
Authors: Ian D. Davis, Martin R. Stockler, Andrew James Martin, Vittorio Marchesin, Olwyn Deignan, Ray McDermott, Wendy R. Parulekar, Scott A. North, Barbara Graham, Anne Poh Long, Felicia T Roncolato, Sonia Yip, Wendy Hague, Carlo Dazo, Xanthi Coskinas, Christopher Sweeney
Journal: Journal of Clinical Oncology (2015)

TPS5077 Background: Androgen deprivation therapy (ADT) with a luteinising hormone releasing hormone analogue (LHRHA) or surgical castration, either alone or combined with conventional non-steroidal anti-androgen (NSAA), is widely used as initial treatment for hormone-naive, metastatic prostate cancer (PC). Meta-analysis of RCTs showed a 3% absolute improvement in 5 year survival with the addition of NSAA to ADT. Residual, low level androgen receptor (AR) signalling or agonist activity from conventional NSAA may provide a stimulatory signal to hormone-sensitive PC cells. We hypothesize that early use of enzalutamide, a more potent and effective androgen receptor blocker, will reduce residual AR signalling, and improve survival. The aim is to determine the effectiveness of ADT + enzalutamide versus ADT + conventional NSAA, as 1st line endocrine therapy for M1 PC. Methods: DESIGN Open label, randomised, stratified, 2-arm, intergroup, phase 3 trial including ANZ, Canada, UK and USA. ELIGIBILITY Metastatic PC starting 1stline ADT. STRATIFICATION Volume of disease, anti-resorptive therapy, comorbidities, early docetaxel use, study site. ENDPOINTS Overall survival (primary), PSA progression free survival (PFS), clinical PFS, health related quality of life, adverse events, cost-effectiveness. SAMPLE SIZE 1100 participants recruited over 2 years + 3.5 years minimum f/up for 80% power to detect a 25% reduction in the hazard of death assuming an OS rate at 3 years of 65% in control group. TREATMENT LHRHA or surgical castration plus either enzalutamide 160mg daily orally, or conventional oral NSAA until disease progression or prohibitive toxicity. ASSESSMENTS Baseline, days 29 and 85 then 12 weekly until clinical progression; imaging prior to randomisation and on progression (PSA and clinical). Tertiary correlative objectives include identification of prognostic/predictive biomarkers from archival tumour tissue and fasting bloods collected at baseline, week 24 and progression (PSA and clinical). Email: enzamet@ctc.usyd.edu.au Website: http://www.anzup.org.au/ Clinical trial information: ACTRN12614000110684.
A comparison of stenting and conventional coronary angioplasty: Long-term survival and rehospitalization in the Department of Veterans Affairs Medical Centers Charles Maynard, Nathan R. Every, Steven M. Wright, James L. Ritchie. Department of Veterans Affairs Puget Sound Health Care System, Seattle, WA, Massachusetts Veterans Epidemiology Research Information Center, Boston, MA
Journal: Journal of the American College of Cardiology (2001)
The Nature of the Mutagenicity and Carcinogenicity of Nitrated, Aromatic Compounds in the Environment
Authors: Hiroshi Tokiwa, Reiko Nakagawa, Kazumi Horikawa, Akito Ohkubo
Journal: Environmental Health Perspectives (1987)

Gaseous substances such as nitrogen dioxide (NO2) and sulfur dioxide (SO2) stimulate the process of nitration of polycyclic aromatic hydrocarbons, and the transformation products display a broad spectrum of mutagenicity, genotoxicity, and carcinogenicity. Bacterial mutation by nitroarenes is specific. Tetracyclic nitroarenes are thought to be the most mutagenic compounds in the Salmonella test system, and some are carcinogenic in rats and mice. Furthermore, it was found that the mutational nitroarenes produced mostly DNA damage, which is subject to recombination repair in the rec assay system using Bacillus subtilis. Nitroarenes in the environment seem to be ubiquitous; the majority of the compounds are emitted directly from diesel emissions, kerosene heaters, and gas and liquefied-gas burners or heaters. In nitroarenes induced during incomplete combustion, nitropyrene and nitrofluoranthene derivatives are the most important mutagens/carcinogens for determining the chronic toxicity of nitroarenes overall.
Abstract 5331: Gene Transfer Of The Peripheral Type Benzodiazepine Receptor Alters The Spatio-temporal Dynamics Of The Mitochondrial Membrane Potential (ΔΨ <sub>M</sub> ) Across The Intact Heart
Authors: Alexander R Lyon, Jaeho Park, Lifan Liang, Roger J Hajjar, Fadi G Akar
Journal: Circulation (2008)

The peripheral type benzodiazepine receptor (mBZR) is a key mediator of mitochondrial dysfunction during oxidative stress. In isolated cardiomyocytes agonists of the mBZR cause ΔΨ m depolarization, KATP channel activation, and subsequent loss of cellular excitability. However, the spatiotemporal dynamics of ΔΨ m in the intact heart and their relationship to arrhythmias are unknown. Here, we investigate the mechanistic link between mBZR and ΔΨ m directly using adenoviral mediated gene transfer of the mBZR encoding gene (PKBS) in rat myocardium. Methods : Optical imaging was performed at a subcellular resolution in ex vivo perfused hearts from AdPKBS (n=5), AdGFP (n=2), and normal (n=4) rats. Normalized ΔΨ m was measured using a novel quantitative technique of TMRM imaging during global ischemia (7.5 min) followed by reperfusion. Mean and SD of baseline corrected &amp; normalized ΔΨ m across 6400 sites were compared between groups. Results : In sharp contrast to normal and GFP groups, 4 of 5 PKBS hearts exhibited a paradoxical increase in ΔΨ m during ischemia (Fig A ) as ΔΨ m was significantly (p&lt;0.05) greater in PKBS (+0.18±0.07) compared to GFP (−0.62±0.21) and normal hearts (−0.17±0.16) following 7.5min of ischemia. A transient overshoot in ΔΨ m observed in all groups during early (&lt;30sec) reperfusion was greatest in PKBS hearts (Fig A ). Finally, ΔΨ m heterogeneity indexed by SD was markedly greater in PKBS compared to GFP and normal hearts during ischemia (p&lt;0.01) and reperfusion (p&lt;0.01) (Fig B ). Conclusion : PKBS alters spatiotemporal dynamics of ΔΨ m and increases ΔΨ m heterogeneity across the intact heart. Modulating PKBS expression may be a novel therapeutic strategy for arrhythmias. Figure 1. Influence of PKBS gene transfer on myocardial ΔΨm during ischemia and reperfusion. Mean myocardial ΔΨm ( A ) and spatial Δψm heteroogeneity ( B ) during ischemia and reperfusion.
Abstract 5543: The P2Y <sub>2</sub> Nucleotide Receptor Contributes To Intimal Lesion Growth And Regulates Lymphotoxin-α Secretion
Authors: Gary Weisman, Ningpu Yu, Cansu Agca, Rikka Shivaji, Ying Wan, Agca Yuksel, Cheikh Seye
Journal: Circulation (2008)

Inflammatory processes play a crucial role in atherosclerotic lesion growth. A number of pro-inflammatory molecules have been implicated in the pathogenesis of atherosclerosis, including lymphotoxin-α (LTA). Our research has shown that activation of a G protein-coupled P2Y 2 nucleotide receptor (P2Y 2 R) expressed in vascular cells mediates inflammatory responses. To examine the role of the P2Y 2 R in lesion growth, we developed P2Y 2 R over-expressing transgenic rats. Collar-induced injury to the carotid artery of P2Y 2 R transgenic rats caused a dramatic increase in intimal lesion growth and significant macrophage accumulation, which nearly blocked the arterial lumen. Immunohistochemical staining showed that LTA and galectin-2 (gal-2) were abundantly expressed in smooth muscle cells (SMC) and macrophages in carotid lesions of P2Y 2 R transgenic rats. We also identified galectin-2 as a P2Y 2 R binding partner using the yeast two-hybrid system and a co-immunoprecipitation assay. P2Y 2 R agonist, UTP, stimulated gal-2 mRNA expression in rat carotid SMC. Transient transfection of SMC with a genomic fragment including the rat galectin (LGALS2) promoter incorporated into a luciferase (pGL-3) reporter vector showed that UTP markedly increased LGALS2 promoter activity in a dose-dependent manner. Moreover, UTP induced LTA secretion in cultured aortic SMC from wild type, but not P2Y 2 R −/− mice. Adenoviral expression of the full length P2Y 2 R in SMC from P2Y 2 R −/− mice fully restored UTP-induced LTA secretion. However, expression of a mutant P2Y 2 R that does not bind filamin A, an actin-binding protein that interacts with the P2Y 2 R, only partially restored UTP-induced LTA secretion in P2Y 2 R −/− SMC. Gal-2 siRNA partially inhibited LTA release into medium of cultured SMC expressing the P2Y 2 R. In contrast, gal-2 siRNA abolished UTP-induced LTA secretion in SMC isolated from P2Y 2 R −/− mice expressing the filamin A binding mutant P2Y 2 R. These results indicate that P2Y 2 Rs regulate gal-2-dependent LTA secretion in SMC via a filamin A-dependent mechanism that likely contributes to vascular inflammation.
An appraisal of narrowband (TL-01) UVB phototherapy. British Photodermatology Group Workshop Report (April 1996)
Authors: D. BILSLAND, R. DAWE, B.L. DIFFEY, P. FARR, J. FERGUSON, S. GEORGE, N.K. GIBBS, C. GREEN, J. MCGREGOR, H. WEELDEN, N.J. WAINWRIGHT, A.R. YOUNG
Journal: British Journal of Dermatology (1997)
Integrative Medicine
Journal: JAMA (2005)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Balloon occlusion of the internal spermatic vein for the treatment of varicoceles
Authors: P. C. Walsh
Journal: JAMA (1981)

Varicocele is one of the most common causes of male infertility. We have developed a technique for the treatment of this disorder in which occlusion of the internal spermatic vein is accomplished percutaneously using a detachable balloon. Twenty-one varicoceles in 20 patients have been treated successfully without complication. This procedure can be performed easily, without general anesthesia or surgical incision, on an outpatient basis. (<i>JAMA</i>1981;246:1701-1702)
Nosocomial Infections in Intensive Care Units
Authors: Ida Salvo
Journal: JAMA (1996)

<h3>To the Editor.</h3> —The European Prevalence of Infection in Intensive Care (EPIC) study by Dr Vincent and colleagues<sup>1</sup>is, to our knowledge, the first study in which prevalence of intensive care unit (ICU) infections is estimated in more than 10 000 patients of 1417 ICUs in 17 European countries. This study provides an estimate of the magnitude of the problem of infections in ICUs as well as the sites and bacteria involved, using the 1-day prevalence techniques. A high variability of the prevalence of ICU-acquired infections among countries, from a minimum of 9.7% in Switzerland to a maximum of 31.6% in Italy, results from this study. Despite the many caveats expressed by the authors, those differences appear as an indicator of the efficacy of infection control and quality of care in the different countries. From this point of view, the high prevalence of infections in Italian ICUs created alarm
Highlights
Journal: JAMA (2014)

Tadalafil to Prevent ED After Prostate Cancer Radiotherapy 1300Erectile dysfunction (ED) is common after radiotherapy for prostate cancer.In a randomized trial that enrolled 242 men with intact erectile function who were to receive radiotherapy for prostate cancer, Pisansky and colleagues assessed whether daily treatment with the phosphodiesterase-5 inhibitor tadalafil-initiated at the onset of radiotherapy and continued for 24 weeks-could preserve erectile function.The authors report that compared with placebo, daily use of tadalafil did not result in improved off-drug, spontaneous erectile function assessed 28 to 30 weeks after starting radiotherapy.
Alzheimer's/apo E link grows stronger
Authors: P. Cotton
Journal: JAMA (1994)

AN EPIDEMIOLOGIC finding that many said did not make sense is proving to be more than a statistical fluke. Skepticism greeted reports last year of a strong association between Alzheimer's disease and the genes for apolipoprotein E (apo E), which were thought to be important only in cholesterol metabolism (<i>JAMA</i>. 1994;271:89-91). Critics scoffed that apo E could not play a role in neuronal death because it was never seen in neurons. But during this past year, dozens of studies around the world have confirmed that the apo E4 allele confers the greatest risk of developing late-onset Alzheimer's, the relatively rare<i>E2</i>allele seems to be protective in most but not all ethnic groups, and risk with the most common<i>E3</i>allele lies in the middle. One new study suggests that the<i>E4</i>allele is the strongest predictor of which patients with mild memory problems will go on to develop Alzheimer's
Physicians, Clergymen, and the Hospitalized Patient
Journal: JAMA (1967)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Intensive therapy with cyclophosphamide, carmustine, etoposide +/- cisplatin, and autologous bone marrow transplantation for Hodgkin's disease in first relapse after combination chemotherapy [see comments]
Authors: DE Reece, JM Connors, JJ Spinelli, MJ Barnett, RN Fairey, HG Klingemann, SH Nantel, S O'Reilly, JD Shepherd, HJ Sutherland
Journal: Blood (1994)