tbuckley/cabot-search huggingface dataset:
Can you alter your heart disease risk?
Authors: R. S. Johnson
Journal: JAMA (1981)

Testing of the hypothesis that people can prevent heart disease by changing their life-styles has yielded promising but inconclusive results, said attendees at the American Heart Association's (AHA) recent annual conference on cardiovascular disease epidemiology in Washington, DC. New community intervention trials are scheduled for the 1980s. Many investigators firmly believe that such things as a high-fat diet, cigarette smoking, and high blood pressure (BP) lead to heart disease. But others question whether these are reversible causes of heart disease and whether universal public health policies can be made or only selective recommendations. Newer findings suggest that it is important to moderate or balance some risk factors. The healthy state may lie in between two extremes, over a certain threshold, or even in cultural tradition. In brief, reports at the conference dealt with the following: Diet. People with low serum cholesterol, low body weight, or who eat a low-fat diet
One year outcome after cardiac transplantation is not compromised by elevated pretransplant panel reactive antibody levels in patients with ventricular assist devices
Authors: S. Trehan, S.H. McKellar, J. Marks, D.A. Bull, J.C. Stringham, D.O. Taylor, D. Ronlund, T.C. Fuller, J.W. Long
Journal: Journal of the American College of Cardiology (1998)
Comparison of the diagnostic quality of aspiration and core biopsy needles for transjugular liver biopsy
Authors: Georg Semmler, Judith Stift, Katharina Wöran, Benedikt Simbrunner, Bernhard Scheiner, Philipp Schwabl, Rafael Paternostro, Matthias Pinter, Albert Stättermayer, Tobias Meischl, Andrea Beer, Michael Trauner, Mattias Mandorfer, Thomas Reiberger
Journal: Journal of Hepatology (2020)
The Arg389Gly polymorphism of human $beta;1-Adrenergic receptor is a new genetic marker for acute myocardial infarction Chikao Iwai, Hozuka Akita, Kenji Kanazawa, Nobuyuki Shiga, Masahiro Terashima, Yasuaki Matsuda, Eiji Takai, Yoshitomo Miyamoto, Teishi Kajiya, Takatoshi Hayashi, Mitsuhiro Yokoyama. The 1st Dept. of Internal Medicine, Kobe University School of Medicine, Kobe, Japan, Dept. of Cardiology, Himeji Cardiovascular Center, Himeji, Japan
Journal: Journal of the American College of Cardiology (2001)
The Two Types of Right Aortic Arch
Journal: JAMA (1963)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
THE SPIRIT OF REVOLT
Journal: JAMA (1921)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Occupational Exposure of Workers to 1,3-Butadiene
Authors: John M. Fajen, Dennis R. Roberts, Leslie J. Ungers, E. Radha Krishnan
Journal: Environmental Health Perspectives (1990)

Researchers from the National Institute for Occupational Safety and Health (NIOSH) conducted an extent-of-exposure study of the 1,3-butadiene monomer, polymer, and end-user industries to determine the size of the exposed workforce, evaluate control technologies and personal protective equipment programs, and assess occupational exposure to 1,3-butadiene. A new analytical method was developed for 1,3-butadiene that increased the sensitivity and selectivity of the previous NIOSH method. The new method is sensitive to 0.2 microgram per 1,3-butadiene sample. Walk-through surveys were conducted in 11 monomer, 17 polymer, and 2 end-user plants. In-depth industrial hygiene surveys were conducted at 4 monomer, 5 polymer, and 2 end-user plants. Airborne exposure concentrations of 1,3-butadiene were determined using personal sampling for each job category. A total of 692 full shift and short-term personnel and 259 area air samples were examined for the presence of 1,3-butadiene. Sample results indicated that all worker exposures were well below the current OSHA PEL of 1000 ppm. Exposures ranged from less than 0.006 ppm to 374 ppm. The average exposure for all samples was less than 2 ppm. The present American Conference of Governmental Industrial Hygienists (ACGIH) threshold limit value for 1,3-butadiene is 10 ppm. To reduce the potential for occupational exposure, it is recommended that quality control sampling be conducted using a closed loop system. Also all process pumps should be retrofitted with dual mechanical seals, magnetic gauges should be used in loading and unloading rail cars, and engineering controls should be designed for safely voiding quality control cylinders.
Barbiturate Toxicity
Authors: Russell S. Fisher
Journal: New England Journal of Medicine (1949)

BARBITURATE TOXICITYThe rapid and accurate quantitation of barbiturates in blood and tissue has been made feasible in the past three years by the development of methods utilizing the ultraviolet spectrophotometer.1 , 2 These require only 5 to 10 cc. of specimen, and it is possible for the first time to determine the blood barbiturate concentration of a comatose patient in the emergency room as well as that of a dead person whose residual concentration is very low.Correlation of Barbiturate Level with State of ConsciousnessPreliminary studies of the concentration of pentothal in the blood during surgical anesthesia3 show that consciousness . . .
Foreign Objects Left in the Patient
Journal: JAMA (1966)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Manuscripts Approved for JNCI, December 1, 1966, to January 1, 1967
Journal: JNCI Journal of the National Cancer Institute (1967)

Journal Article Manuscripts Approved for JNCI, December 1, 1966, to January 1, 1967 Get access JNCI: Journal of the National Cancer Institute, Volume 38, Issue 2, February 1967, Page 249, https://doi.org/10.1093/jnci/38.2.249 Published: 01 February 1967
IDENTIFYING THE MANUFACTURER
Journal: JAMA (2012)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Artificial insemination with fresh or frozen semen. A comparative study
Authors: E. Steinberger
Journal: JAMA (1973)

A comparison of fertilizing capacity was made between fresh semen and semen stored in liquid nitrogen. A total of 107 attempts at conception were made in 74 women. Fresh semen produced 35 conceptions in 48 attempts; frozen semen resulted in 36 pregnancies in 59 attempts. The 36 women who failed to conceive were found to have a high incidence of polycystic ovarian disease. The 71 conceptions resulted in 59 live births, 36 boys and 23 girls, 11 early spontaneous abortions, and 1 tubal pregnancy.
Chronic Lymphocytic Leukemia Associated with Autoimmune Hemolytic Anemia Is Characterized by a Distinctive miRNA Signature,
Authors: Gerardo Ferrer, Alfons Navarro, Kate E Hodgson, Dolors Colomer, Marta Aymerich, Tycho Baumann, Arturo Pereira, Mariano Monzó, Carol Moreno, Emili Montserrat
Journal: Blood (2011)
Swallowed Partial Dentures
Authors: Syed Hashmi, John Walter, Wendy Smith, Sergios Latis
Journal: Journal of the Royal Society of Medicine (2004)

Swallowed or inhaled partial dentures can present a diagnostic challenge. Three new cases are described, one of them near-fatal because of vascular erosion and haemorrhage. The published work points to the importance of good design and proper maintenance. The key to early recognition is awareness of the hazard by denture-wearers, carers and clinicians.
Characterization of initiated cells in &lt;I&gt;N&lt;/I&gt;-methylnitrosourea–induced carcinogenesis of the CNS in the adult rat
Authors: Demetrius M. Kokkinakis, Mark L. Watson, Lawrence S. Honig, Elisabeth J. Rushing, Bruce E. Mickey, S. Clifford Schold
Journal: Neuro-Oncology (2001)

A P R I L 2 0 0 1 99 Glial tumors may originate from the malignant transformation of multipotent glial progenitor cells, but tools to study malignant transformation leading to gliomas are limited by the lack of biological systems that represent early stages of this disease in adult animals.In order to characterize the initiated cells that give rise to gliomas, we have employed the N-methylnitrosourea (MNU) model for induction of brain tumors in adult rats (Rushing et al., 1998).Speci cally, we have isolated and cultured transformed (premalignant) cells from normal-appearing brains of rats exposed to MNU for 10 weeks and from histologically abnormal brains of rats exposed to MNU for 15 weeks.We compared them with cells cultured from control animals under identical conditions.Cultured cells were classi ed according to their morphology, immunophenotype, karyotype, proliferation capacity, and tumorigenicity in athymic mice.Cultures from untreated normal rat brains grew as monolayers and had normal karyotypes (42 X,Y), epithelioid morphology, and slow proliferative capacity (doubling time >120 h).In contrast, cultured cells from brains of MNU-exposed animals had karyotypes that ranged from normal to highly aneuploid.Aneuploid lines grew rapidly in multilayers (doubling time <24 h), had differentiated astrocytic or oligodendroglial morphology and immunohistochemical staining pro le, and yielded tumors in athymic mice.Initiated cells with minor chromosomal aberrations assumed mixed bipolar or tripolar morphologies in high density cultures, proliferated rapidly, but showed contact inhibition and failed to induce tumors when injected s.c. in athymic mice.In general, lines showing no evidence of chromosomal aberrations had the most epithelioid morphology, proliferated slowly (doubling time >72 h), and retained strict contact growth inhibition.The presumed undifferentiated glial progenitor cells in culture from either control or MNU-treated rats variably expressed markers such as vimentin, nestin, and NG2 proteoglycan, and they weakly expressed the mature astrocytic or oligoden-
Dr. Wagner and Colleagues Reply
Authors: K. D. WAGNER, R. A. KOWATCH, G. J. EMSLIE, R. L. FINDLING, T. E. WILENS, K. McCAGUE, J. D'SOUZA, A. WAMIL, R. B. LEHMAN, D. BERV, D. LINDEN
Journal: American Journal of Psychiatry (2006)
Pregnancy and abortion in women with essential thrombocythemia
Authors: Anna Candoni, Renato Fanin, Teresa Michelutti, Domenico Russo, Angela Michelutti
Journal: American Journal of Hematology (2002)

Intravenous anti-D is often used in the treatment of autoimmune thrombocytopenic purpura (AITP), but little is known about its mechanisms of action. To investigate anti-D's potential in vivo mechanism(s) of action, a small group (N = 7) of children with chronic AITP was studied. The children initially received either 25 or 50 μg/kg of WinRho-SD in a four-cycle cross-over trial, and peripheral blood samples from the first and third cycles were assessed for cytokine levels at pre-treatment, 3 hr, 1 day, and 8 days post-treatment. Results showed that platelet counts significantly increased in all the children by day 8 post-treatment. Analysis of serum by ELISA showed that there was a significant but transient rise in both pro- and anti-inflammatory cytokine/chemokine levels (e.g., IL1RA, IL6, GM-CSF, MCP-1α, TNF-α and MCP-1) by 3 hr post-treatment in both cycles which returned to baseline levels by 8 days post-treatment. These results suggest that anti-D administration may initially activate the RES in the form of cytokine/chemokine secretion, which is subsequently followed by an increase in platelet counts. It is possible that the induced cytokine/chemokine storm may have an effect on several physiological processes such as those mediating either adverse effects or potentially RES phagocytic activity. Am. J. Hematol. 69:225-227, 2002. © 2002 Wiley-Liss, Inc.
Motherhood, Culture
Authors: Wendy Kline
Journal: JAMA (2006)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Journals JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2024 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Corrections
Journal: BMJ (1953)

ANY QUESTIONS? MEDICALJOnRNALpopulation so long as their pneumoconiosis does not become complicated by tuberculosis, when the prognosis is much worse.The more rapid the onset of pneumoconiosis, and usually, therefore, the younger the man, the greater the lia- bility of conversion at some time to silico-tuberculosis and the worse the prognosis.Cases with " massive fibrosis " have a worse prognosis, and cases with open tuberculosis seldom live more than two years.The liability to develop open tuberculosis appears to be much greater in cases of pure silicosis than in the various forms of mixed dust pneumo- coniosis such as those of coal-workers and foundry workers.This is shown by the much higher tuberculosis death rates that have prevailed in the pure silica risk industries than in coal-mining and foundries.Other forms of pneumoconiosis than silicosis-for example, asbestosis, aluminosis, beryllosis -do not appear to predispose to tuberculosis.The frequency with which tuberculosis is found to com- plicate pneumoconiosis depends upon the diagnostic criteria and upon the material used for the study.For instance, in post-mortem examinations. of coal-miners Rogers' found tuberculosis in 75.8 % of cases with massive fibrosis, Gooding' found it in 26% of all cases.Theodos and
CERUMINOSIS OBTURANS.
Authors: LEWIS S. SOMERS
Journal: JAMA (1897)

The ceruminous glands of the external auditory canal secrete small quantities of a semi-solid, light yellow substance, called cerumen or wax. The secretion is produced in such small amounts as to be unnoticed and by the action of the lower jaw is propelled toward the exterior of the canal, entangling small foreign substances and to a certain extent protecting the deeper and more delicate structures. If for any cause the secretion of cerumen is increased in amount or by osseous or other changes, the configuration of the canal become altered; this outward movement is restricted and the cerumen being retained, undergoes change producing the condition known as ceruminosis obturans, or more commonly called impacted cerumen or wax plug. The size and density of the cerumen mass varies with the secretory activity of the glands and the length of time which it remains in the auditory canal. The increase in size
Adult Leukemias and Diagnostic X-Rays
Journal: JAMA (1963)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Correction
Authors: Jill Buyon, A Brucato, D Friedman
Journal: Annals of the Rheumatic Diseases (2007)

What's in a name?Bertsias, and colleagues have provided an extensive report of the European League Against Rheumatism (EULAR) recommendations for systemic lupus erythaematosus (SLE). 1 While clearly a minor point in an excellent, well balanced clinical manifesto for doctors managing individuals with SLE, in the section on pregnancy outcomes atrioventricular (AV) block is referred to as ''foetal heart block'', qualified in parenthesis as ''formerly congenital heart block''.The issue of a universally accepted definition of ''congenital'' heart block may be somewhat controversial. 2 It is readily acknowledged that the initial term applied to the clinical condition of atrioventricular block, ''complete congenital heart block (CCHB)'', was too restrictive.Increased awareness of this disease and improved in utero echocardiographic techniques revealed that less advanced degrees of block were also associated with maternal anti-SSA/Ro-SSB/La antibodies and that the degree of block could progress and regress. 3 4As greater numbers of cases were identified, it also became evident that the heart block in the absence of serious structural abnormalities was identified most often between 18-24 weeks of gestation. 3 Heart block detected after birth is generally not associated with maternal autoantibodies. 2 5Furthermore, cardiologists often define ''congenital'' blocks as detected after birth. 6 That said, the adjective ''congenital'' seems appropriate since it literally describes a condition as ''existing at or dating from birth... acquired during development in the uterus...'' 7 while foetal connotes ''pertaining to an unborn foetus''.The descriptor should emphasise the time of identification or onset of a condition rather than ascribing to the age status during which the condition is diagnosed, which makes sense as noted by the following example.A teenager carries the diagnosis of a heart block identified during gestation but which has persisted throughout his life.He is no longer a foetus, and thus does not have foetal heart block, but rather a condition diagnosed at or before birth, hence congenital heart block.Perhaps an appropriate compromise, acceptable for several specialists involved in the field, based on timing of diagnosis might be to define it as ''congenital if an AV block is diagnosed in utero, at birth, or within the neonatal period (0-27 days after birth)''. 2In the case of a foetal diagnosis of incomplete AV block that resolves by birth, this could be referred to as ''resolved'' congenital heart block.Surely it is the condition itself we need to fix and not its name, whichas it stood-was technically correct.