tbuckley/cabot-search huggingface dataset:
THE SUPPLEMENTARY SCHOOL LUNCH
Journal: JAMA (1923)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Deaths
Journal: JAMA (1967)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Influence of Post-Developmental Cadmium on Caries and Cariostasis by Fluoride
Authors: T. R. Shearer, J. L. Britton, D. J. DeSart
Journal: Environmental Health Perspectives (1980)

Uptake of cadmium into molar enamel and dentin from rats receiving cadmium in their drinking water was markedly elevated and proportional to the amount of cadmium in the drinking water. Post-developmental cadmium did not influence caries development or alter the cariostatic effectiveness of fluoridated drinking water. The data indicate that the absolute concentration of cadmium in teeth is not predictive or cadmium-induced caries and that the critical period for caries promotion by cadmium may be during the developmental rather than the post-developmental period of tooth formation.
Tucatinib Impresses in Breast Cancer
Journal: Cancer Discovery (2019)

Recent results suggest that tucatinib, when combined with trastuzumab and capecitabine, is effective in patients with locally advanced inoperable or metastatic HER2-positive breast cancer who have received prior therapies: In a phase II trial, the drug extended overall survival and progression-free survival compared with trastuzumab and capecitabine alone, and improved progression-free survival in patients with brain metastases.
Planning the boost in early-stage breast carcinoma: Comparison of two methods.
Authors: V. Krakobsky, D. Gwirc
Journal: Journal of Clinical Oncology (2011)

126 Background: When it comes to breast-conserving surgery the boost volume becomes a critical target because it has the highest risk of residual cancer cells. At our institution the most common methods to contour the tumor bed are: the scar-based planning guided by fluoroscopy (2DP) and the one that relies on the visualization of the seroma under CT (3DP). We evaluated the accuracy of the breast boost volume treated with clinical mark-up compared with the volume delineated with CT planning. Additionally, we evaluated whether or not the lumpectomy scar is a suitable landmark to define the tumor bed. Methods: 20 patients diagnosed with breast cancer stage I-IIA underwent tumor bed boost planning using two techniques: 2DP and 3DP. All the information concerning to the 2DP was reproduced in a radiotherapy planning system. Statistical analysis: the match pair t-test and the coefficient of variation were used to test for an association among the two planning techniques for tumor bed coverage. The D95, D90, V15, volume, medium lung dose and geographic miss were analyzed. Pearson correlation analysis was used to assess the relationship between CVS, date of surgery, age and previous chemotherapy treatment. Other items taken into account were the match between the scar and the seroma, and how good the visualization of the seroma was by using the CVS. Results: 2DP was inaccurate: the mean D95, D90 and V15 were 3.12 Gy, 4.6 Gy and 29% respectively (variation coefficient: 135%, 107% and 65% respectively). The difference between the mean 2DP D95 and 3DP D95 was 11 Gy (95% CI, 9 Gy to 13 Gy, p&lt; 0.001). The difference between the mean 2DP V15 and 3DP V15 was 59% (95% CI, 49% to 69%, p &lt; 0.001).The percentage of geographic miss was: 80% (95% CI, 63% to 97%). All the seromas were finely visualized There was no correlation between CVS, age, prior chemotherapy and date of surgery. The volumes and the medium lung dose were larger for the 3D method: 373.1 cm 3 vs. 248.8 cm 3 and 0.27Gy vs. 0.14 Gy respectively. Only 45% of the lumpectomy scars matched the seroma. Conclusions: CT-based planning allows for optimal tumor bed coverage compared with scar-based planning which carries a significant risk of missing the target. The lumpectomy scar is often a poor indicator of the tumor bed.
Books Received
Journal: JAMA (1906)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Researchers amass abortion data
Authors: M. F. Goldsmith
Journal: JAMA (1989)

UNLIKE CHARGES for many other medical services, the amount women pay for an abortion has not kept pace with cost-of-living increases in the economy. It apparently does not cost much more today than in 1986, when the average charge to patients for a first-trimester abortion was $209 at an abortion clinic, $235 at other medical clinics, and $272 in the office of a physician (usually an obstetrician-gynecologist). The reasons: competition among providers and more efficient performance. That is just one of the facts surfacing in a survey begun this spring by the Alan Guttmacher Institute, New York, NY. The institute, an independent nonprofit corporation for research, policy analysis, and public education regarding reproductive health, will complete and publish its survey of the status of abortion in the United States in approximately 6 months. Investigators taking part in this 10th survey regarding this subject want to determine where abortions are available,
Direct measurement of lead in bone. A promising biomarker
Authors: P. J. Landrigan
Journal: JAMA (1994)

Chronic excessive exposure to lead is widespread in industrialized societies. In the United States, an estimated 3 million young children have a blood lead level of 0.50 μmol/L (10 μg/dL) or more, the level considered by the Centers for Disease Control and Prevention to indicate increased absorption.<sup>1</sup>Also, more than 1.4 million industrial workers have the potential for chronic exposure through occupations such as stained glass manufacturing, battery making, and bridge demolishing.<sup>2</sup> See also p 197. Despite the extent of this exposure, great gaps exist in our knowledge of the chronic toxicity of lead.<sup>3</sup>For example, we do not know the level of cumulative exposure in adults that is associated with chronic dysfunction of the central or peripheral nervous systems. We do not know whether chronic exposure is associated with motor neuron disease, parkinsonism, or other chronic neurological illnesses. We do not know the fraction of hypertension
Bullous amyloidosis
Authors: T. RUZICKA, C. SCHMOECKEL, J. RING, R.P. LINKE, O. BRAUN-FALCO
Journal: British Journal of Dermatology (1985)

A patient with a 12-year history of a relapsing bullous dermatosis is presented. Unusual clinical features included urticarial erythema, conspicuous mottled hyper- and depigmentation, lichenification and ichthyosiform hyperkeratosis. Serum immunoglobulin E levels were elevated. Histological examination showing deposits of amyloid in the uppermost dermis confirmed the diagnosis of bullous amyloidosis. Ultrastructurally, blister formation occurred at the level of the lamina lucida. The amyloid did not react with a panel of antibodies directed against amyloid fibril proteins. No underlying systemic disease was found. The bullous eruption responded to prednisolone therapy.
Current status of propranolol hydrochloride (inderal)
Journal: JAMA (1973)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Trends in Health Programs for Workers in Industry
Journal: JAMA (1963)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Ivy Poisoning.
Authors: Emil King
Journal: JAMA (1907)

Fulda, Minn., Sept. 4, 1907. <h3>To the Editor:</h3> —An abstract of several articles on "Efficient Treatment of Ivy Poisoning" was published inThe Journal, Aug. 31, 1907, page 789. Dr. Syme advises against the use of alcohol in this affection. I, on the contrary, have satisfied myself, after considerable experience with this affection, that no other remedy is so efficient if properly used. Many years ago Prof. Theo. G. Wormley, I believe, suggested the use of hyposulphite of soda. I, therefore, prescribe a 1 per cent, solution of this salt in 95 per cent, alcohol. When a patient with ivy poisoning comes to me within forty-eight hours after the eruption appears, I wash the affected part thoroughly and repeatedly with the above mixture, directing the patient to continue its use in the same manner every four hours. Following this plan, I have often cured these patients in two days. Severe
Mathematical Model to Predict Individual Survival for Patients With Renal Cell Carcinoma
Authors: A. Zisman
Journal: Journal of Clinical Oncology (2002)

PURPOSE: To develop a multivariate model and mathematical formula capable of calculating personalized survival for renal cell carcinoma (RCC) patients with clinically available variables. PATIENTS AND METHODS: A total of 477 patients out of 661 undergoing nephrectomy at the University of California Los Angeles between 1989 and 1999 were eligible for evaluation and formed the analyzed cohort for this retrospective study. Time to death was the primary end point assessed. Univariate analysis for 14 to 20 variables was conducted, followed by a multivariate Cox analysis. The variables that provided independent information as to the time of death for metastatic and nonmetastatic patients were coded and incorporated into a function based on the Nadas equation principle. RESULTS: For nonmetastatic patients, the significant variables in the multivariate analysis were Fuhrman's grade and Eastern Cooperative Oncology Group performance status. For the metastatic patients, Fuhrman's grade, 1997 classification T stage, number of symptoms, nodal involvement, and immunotherapy were independent predictors for survival. These variables, based on the Cox multivariate regression model, were implanted into an exponential Nadas equation. The expected survival predicted by use of the Nadas equations faithfully describes the actual survival based on Kaplan-Meier curves. CONCLUSION: We have developed mathematical equations for estimating survival after radical nephrectomy for RCC. The resulting formulas are capable of better tailoring survival estimates for a specific patient and are based on widely accepted clinical prognostic variables. On validation with external data, this type of representation can be used as a tool for the determination of personalized prognosis and may be useful for patient education and counseling.
Mountain accidents.
Authors: N. F. Kirkman, M. K. Hartley
Journal: BMJ (1968)

cleared relapsed and required dapsone again, and of the three patients whose dapsone requirements had fallen two needed an increased dose in order to control the skin lesions.The changes that occurred in the macroscopic appearances of the small intestine, in the heights of the intestinal villi and of the surface epithelial cells, in faecal fat excretion, and in serum and red cell folate levels as a result of the diet, and their reversal on reintroducing gluten, were all compatible with gluten sensitivity.These recent findings will be reported in full elsewhere, but to our mind they demonstrate that in a number of patients the skin disorder is influenced by a gluten-free diet, and that there is a direct relationship between the gut and the skin in dermatitis herpeti- formis.The fact that in some patients the skin lesions do not improve with a gluten-free diet does not negate the fact that they do improve in others.It was apparent from our studies that improvement in the skin disorder in response to the gluten-free diet may take a considerable time.Failure of other workers to observe improvement in the skin condition on gluten withdrawal may be due to: inadequate length of time on the gluten-free diet; failure of the patient to keep strictly to the diet ; and, since the enteropathy in idiopathic steator- rhoea or in dermatitis herpetiformis does not always respond to a gluten-free diet, pre-sumably, therefore, by analogy, the skin lesions cannot be expected to respond in all patients with dermatitis herpetiformis.Since idiopathic steatorrhoea is known to have a genetic basis7 it is likely that the gluten sensitive enteropathy in dermatitis herpetiformis will also have a genetic basis; nevertheless this does not exclude the possibility that a similar or related acquired defect is responsible for both lesions.-Weare, etc.,
A randomized controlled trial of IFN?+ketoprofen or IFN?+ribavirin in HCV chronic active hepatitis (CAH) non-responder to IFN? alone . Patologia Medica I and (1)Anatomia Patologica and (2)Laboratorio Centralizzato, Bologna; (3)Clinica Medica, Universit� Cattolica Roma; Ricerca in Medicina, Bologna; Italy
Journal: Hepatology (1995)
Incorrect Units in Table.
Journal: JAMA (1995)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Respiratory monitoring
Authors: D. W. Bethune, J. M. Collis
Journal: BMJ (1967)

<h3>Summary:</h3> Increasing detection of unruptured intracranial aneurysms, catastrophic outcomes from subarachnoid hemorrhage, and risks and cost of treatment necessitate defining objective predictive parameters of aneurysm rupture risk. Image-based computational fluid dynamics models have suggested associations between hemodynamics and intracranial aneurysm rupture, albeit with conflicting findings regarding wall shear stress. We propose that the “high-versus-low wall shear stress” controversy is a manifestation of the complexity of aneurysm pathophysiology, and both high and low wall shear stress can drive intracranial aneurysm growth and rupture. Low wall shear stress and high oscillatory shear index trigger an inflammatory-cell-mediated pathway, which could be associated with the growth and rupture of large, atherosclerotic aneurysm phenotypes, while high wall shear stress combined with a positive wall shear stress gradient trigger a mural-cell-mediated pathway, which could be associated with the growth and rupture of small or secondary bleb aneurysm phenotypes. This hypothesis correlates disparate intracranial aneurysm pathophysiology with the results of computational fluid dynamics in search of more reliable risk predictors.
THE TRUDEAU SANATORIUM, 1885-1925
Journal: JAMA (1925)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal