tbuckley/cabot-search huggingface dataset:
From JAMA’s Daily News Site
Journal: JAMA (2014)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Abstract 1191: Elevated Concentration of Pentraxin 3 is Associated With Coronary Plaque Vulnerability
Authors: Takeshi Soeki, Toshiyuki Niki, Kunihiko Koshiba, Kenya Kusunose, Koji Yamaguchi, Tomohito Kawano, Hirotsugu Yamada, Tetsuzo Wakatsuki
Journal: Circulation (2007)

Background: Inflammation is a critical contributing factor to the development and the progression of atherosclerosis. Pentraxin 3 (PTX3), which has C-terminal sequence homology with the classic pentraxin C-reactive protein (CRP), is produced abundantly from atherosclerotic lesion while CRP is mainly produced in the liver. In this study, we investigated whether plasma level of PTX3 might be a sensitive marker for the severity of coronary artery disease by coronary angiography and vulnerable plaque by multidedector-row CT. Methods: We measured the concentration of PTX3 in peripheral and coronary sinus plasma of 36 patients with angina pectoris (AP) and 18 control subjects who had no coronary artery stenosis on angiography. Then, we determined the correlation between the plasma PTX3 concentration and the Gensini score as an index of severity of coronary atherosclerosis or the CT density of plaque as a quantitative index of plaque vulnerability. The plasma concentration of PTX3 was measured by enzyme-linked immunosorbent assay. Results: There was no significant difference of PTX3 concentrations in peripheral and coronary sinus plasma between patients with AP and control subjects (peripheral plasma, 2.6±1.1 vs 2.0±0.8 ng/ml; coronary sinus plasma, 2.8±1.2 vs 2.0±0.8 ng/ml). The concentration of PTX3 in coronary sinus but not peripheral plasma correlated with Gensini score (r=0.47, p&lt;0.05 and r=0.34, NS, respectively). Interestingly, there was significantly negative correlation between plasma PTX3 concentration and the CT density (r=−0.71, p&lt;0.01). On the other hand, plasma high-sensitive CRP concentration was not correlated with the CT density. Conclusion: PTX3 might reflect coronary plaque vulnerability that leads to plaque rupture more specifically than CRP.
The compromised host and infection. I. Pneumocystis carinii pneumonia
Authors: J. Ruskin
Journal: JAMA (1967)

Pneumocystis pneumonia occurred in five adults. Each of them was receiving immunosuppressive drug therapy for underlying lymphoma or systemic lupus erythematosus at the time pneumonitis became evident. Premortem diagnosis of infection was made in three of the patients by open-lung biopsy, and two were treated with specific therapy. Two other patients hospitalized in the same room showed signs of Pneumocystis infection within one week of each other. This experience and data from studies of pneumocystosis within families and in European nurseries suggest that contagion may play a role in acquisition of the infection, although experimental animal models and other reported cases in man imply that activation of already latent infection with Pneumocystis accounts for overt disease. A review of the literature acquaints the reader with problems in diagnosis, treatment, and epidemiology of the disease.
The HLA Ligandome Landscape of Chronic Myeloid Leukemia Delineates Novel T-Cell Epitopes for Immunotherapy
Authors: Tatjana Bilich, Annika Nelde, Leon Bichmann, Helmut R. Salih, Daniel Johannes Kowalewski, Heiko Schuster, Ana Marcu, Marian C. Neidert, Maren Lübke, Jonas Rieth, Mirle Schemionek, Tim H Brümmendorf, Vladan Vucinic, Dietger Niederwieser, Melanie Märklin, Janet K. Peper, Malte Roerden, Reinhild Klein, Oliver Kohlbacher, Lothar Kanz, Hans-Georg Rammensee, Stefan Stevanovic, Juliane S. Walz
Journal: Blood (2018)
RACHITIS
Authors: J. W. VAN DERSLICE
Journal: JAMA (1910)

Rachitis is a dystrophy with which the pediatrician comes into contact with a frequency which breeds contempt; on the other hand there are a certain number of physicians to whom the condition of rachitis is sufficient explanation for any and all pathologic conditions. To what may be attributed this great variation of opinion among medical men? It is undoubtedly due to the fact that there is no definite limitation in the symptomatology of this condition. The definition of rachitis as set down in most of the text-books is the following: Rachitis is a condition of malnutrition affecting all structures and systems of the body with characteristic defects of nutrition, chiefly manifested in the bones. It is this last phrase of the definition that has caused the many errors in the use of the name. Rachitis is a dystrophy, therefore a constitutional disorder, and though the chief macroscopic and known microscopic
Bureau of Investigation
Journal: JAMA (1929)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Methods and Problems of Medical Education. (Sixteenth Series.)
Journal: JAMA (1930)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Interpreting hospital mortality data. The role of clinical risk adjustment
Authors: S. F. Jencks
Journal: JAMA (1988)

This study uses national Medicare data as well as data that were abstracted to calibrate the Medicare Mortality Predictor System to assess the usefulness of a risk adjustment system in interpreting hospital mortality rates. The majority of variation in annual hospital death rates for the four conditions studied (stroke, pneumonia, myocardial infarction, and congestive heart failure) is chance variability that results from the relatively small numbers of patients treated in most hospitals in a year. For hospitals in the highest and lowest quartiles of observed death rates, the difference between observed rates and those predicted by the Medicare Mortality Predictor System is not quite one third smaller than the difference between observed rates and unadjusted national rates. Risk adjustment methods do not show whether the unexplained difference in mortality rates results from differences in effectiveness of care or unmeasured differences in patient risk at the time of admission. Risk-adjusted mortality rates, therefore, should be supplemented by review of the actual care rendered before conclusions are drawn regarding effectiveness of care. (<i>JAMA</i>1988;260:3611-3616)
MEDICAL WRITING
Journal: JAMA (1967)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Omission
Journal: JAMA (1987)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
From the NIH
Journal: JAMA (1977)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Blood pressure, fitness, and fatness in 5- and 6-year-old children
Authors: B. Gutin
Journal: JAMA (1990)

Cross-sectional relations among blood pressure (BP), aerobic fitness, body fatness, and fat patterning were studied in 216 primarily Hispanic inner-city 5-and 6-year-olds. Fitness was measured with a submaximal treadmill test, and fatness was measured with five skin folds. Diastolic BP was inversely related to fitness in the boys and girls, and positively related to fatness for the boys. Systolic BP was positively related to fatness for the boys and girls. Using multiple regression and including parental BPs, fatness explained significant proportions of the variance in systolic BP for both the boys and girls and in diastolic BP for the boys. There were tendencies for central skin folds to explain more of the variation in BP than peripheral skin folds only for the boys. Fitness and fatness were inversely related for the boys and girls. Thus, at 5 and 6 years of age children exhibit some of the same risk factors for cardiovascular disease seen in adults. (<i>JAMA</i>. 1990;264:1123-1127)
Asthma gets worse in women pregnant with girls
Journal: BMJ (1998)

No link exists between exposure to benzodiazepines in pregnancy and cleft palateConflicting evidence exists about whether first trimester exposure to benzodiazepines increases the risk of major malformations or oral cleft.In their metaanalysis of cohort trials Dolovich et al (p 839) found that fetal exposure to benzodiazepines was not associated with major malformations or oral cleft, but a smaller pooled analysis on the basis of data from casecontrol studies found a significantly increased risk for major malformations or oral cleft.Even if the "worst case scenario" is assumed, benzodiazepines do not seem to be major human teratogens.
Correction to: Low-dose decitabine enhances the effect of PD-1 blockade in colorectal cancer with microsatellite stability by re-modulating the tumor microenvironment
Authors: Ganjun Yu, Yanfeng Wu, Wenying Wang, Jia Xu, Xiaoping Lv, Xuetao Cao, Tao Wan
Journal: Cellular and Molecular Immunology (2019)
Endoscopie opératoire des voies urinaires.
Journal: JAMA (1924)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Small Sacrifices
Authors: C L Joseph
Journal: Annals of Internal Medicine (1994)

On Being a Doctor15 July 1994Small SacrificesCarol L. Joseph, MDCarol L. Joseph, MDVeterans Affairs Medical Center; Portland, OregonSearch for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-121-2-199407150-00012 SectionsAboutFull TextPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail They are young and eager. A little anxious at being confronted with a real patient, performing their first real physical examination. Their white coats shine as they plumb the mysteries of stethoscopes, otoscopes, and sphygmomanometers.One of the joys of being a doctor is teaching these students the art of medicine. My favorite assignment is helping the second-year medical students learn to interview and examine patients. It is also one of the few chances I have to hear about medical education from the students' point of view.My last two students seemed much like the others, two young women, bright, ... Author, Article, and Disclosure InformationAuthors: Carol L. Joseph, MDAffiliations: Veterans Affairs Medical Center; Portland, Oregon PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics 15 July 1994Volume 121, Issue 2 Page: 143 Keywords Emotions Instinct Medical education Pregnancy ePublished: 15 August 2000 Issue Published: 15 July 1994 Copyright & PermissionsCopyright © 1994 by American College of Physicians. All Rights Reserved.PDF downloadLoading ...
Evaluating quality of patient care. A strategy relating outcome and process assessment
Authors: J. W. Williamson
Journal: JAMA (1971)

One of the most important questions to be asked of any health care system is the following:<i>Who</i>needs to learn<i>what</i>to most improve health status of the population receiving care? Previous studies have demonstrated (1) that there is an important relationship between patient care assessment and education that might provide a framework for answering this question<sup>1</sup>and (2) that systematic application of this approach requires a priority list of health problems to be studied.<sup>2</sup> The first study dealt with physician response to apparently unexpected abnormalities in three routine screening tests in which it was found that physicians made no apparent response of any kind to approximately two thirds of the test abnormalities. In solving this problem, it was learned that systematic investigation is needed to identify education objectives that specify the individual who needs to learn, as well as the goals to be achieved in the
Autoimmune Neutropenia
Authors: Laurence A. Boxer, Mortimer S. Greenberg, Grace J. Boxer, Thomas P. Stossel
Journal: New England Journal of Medicine (1975)

Serologic tests for antineutrophil antibodies were used to determine if autoantibodies cause neutropenia. The serums of five patients with idiopathic neutropeniaopsonized normal neutrophils, causing them to be ingested by rabbit macrophages or else to activate glucose oxidation rates of other normal neutrophils by at least twice the rate of controls. Some of the serums inhibited the ability of normal neutrophils to ingest by 62 to 56 per cent. At splenectomy in two of the patients splenic macrophages contained ingested neutrophils, suggesting that the opsonic activity of the serum demonstrated in vitro had pathogenetic importance. In two adults, and possibly in an infant, corticosteroids raised the neutrophil count, although antibody activity remained in the serum of the adults. The findings indicate that autoantibodies are the basis of some cases of idiopathic neutropenia, and that they act by promoting the clearance of neutrophils by mononuclear phagocytes.
Current Comment
Journal: JAMA (1946)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Panic disorder
Authors: D S Samarasinghe
Journal: BMJ (1983)

<h3>Objective</h3> The aim of this study was to identify possible correlations between nailfold microangiopathy severity, finger dermal thickness (DT) and fingertip blood perfusion (FBP) in systemic sclerosis (SSc) patients. <h3>Methods</h3> Fifty-seven SSc patients and 37 healthy subjects were enrolled. All patients were evaluated by nailfold videocapillaroscopy (NVC) to classify and score the severity of microangiopathy. Both modified Rodnan skin score (mRss) and skin high-frequency ultrasound were used to detect finger DT. Laser Doppler flowmetry (LDF) was employed to detect FBP. <h3>Results</h3> A positive correlation was found between nailfold microvascular damage severity and both ultrasound-DT (p=0.028) and mRss values (p&lt;0.0001). In particular, both ultrasound-DT and mRss were found progressively higher in patients with ‘Early’, ‘Active’ or ‘Late’ NVC pattern of microangiopathy. A negative correlation was observed between nailfold microvascular damage severity and FBP (p&lt;0.0001), showing the lowest FBP of the patients with more advanced NVC patterns. A negative correlation was observed between FBP, and both ultrasound-DT (p=0.007) and mRss values (p=0.0002). SSc patients showed a higher ultrasound-DT at the level of the fingers, as well as a lower FBP than healthy subjects (p&lt;0.0001). <h3>Conclusions</h3> This study demonstrates a relationship between nailfold microangiopathy severity, DT and FBP in SSc patients.
Plasticity Of Sensory And Motor Maps In Adult Mammals
Authors: J. Kaas
Journal: Annual Review of Neuroscience (1991)

The brain's default mode network consists of discrete, bilateral and symmetrical cortical areas, in the medial and lateral parietal, medial prefrontal, and medial and lateral temporal cortices of the human, nonhuman primate, cat, and rodent brains. Its ...Read More