tbuckley/cabot-search huggingface dataset:
Misplaced Comma, Etc.
Journal: JAMA (1981)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
"A NEW METHOD OF TREATMENT FOR VARICOSE ULCERS OF THE LEG"—"ADHESIVE STRIPS FOR ANAL EXPOSURE IN SURGICAL PROCEDURES"
Journal: JAMA (1922)

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THE HEMATOZOA OF MALARIAL FEVERS.
Journal: JAMA (1896)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Hallucinogenic Drug Reaction—MDA
Authors: Chauncey D. Leake
Journal: JAMA (1972)

<h3>To the Editor.—</h3> In their report on near fatal reaction to ingestion of the hallucinogenic drug methylenedioxyamphetamine (MDA), Richards and Borgstedt (<b>218</b>:1826, 1971) state that they could find little information upon it. Gordon Alles (1901-1963), who first made and developed amphetamine, reported quite fully on structure and action of phenethylamines with discussion of MDA<i>Neuropharmacology: Transactions of the Fourth Conference</i>. (Abramson HA, Macy J Jr [eds]: New York, Foundation Press, 1959, pp 181-268). In this, Alles says that on injection of MDA into small animals it was determined that its toxicity is not significantly different from the lethal dosage of phenethylamine or of amphetamine. He fully described the effects on himself of taking a total divided dose by mouth of 1.4 mg/kg in two hours. These effects included visual and auditory distortions, with a tendency to neck-muscle tension, pupil dilatation, and the peculiar phenomenon of seeing himself hit a
Session summaries and discussion*
Authors: J Cotruvo
Journal: Environmental Health Perspectives (1982)

J. A. COTRUVO: In the course of this conference we have had sessions devoted to the toxicology, mechanisms of action, chemistry, epidemiology, and clinical experience associated with drinking water disinfectants.Each of the session chairper- sons will, in turn, summarize the discussions in each group.D. COURI (Mechanisms): I will limit my remarks to the session which dealt with direct effects of disinfectants in studies involving cats, rats, goats, monkeys, and man.There appears to be a common toxicity which emerges among these species which is referable to the oxidative capacities of the Cl compound disinfectants.These oxidative agents produce effects which ultimately impair red blood cell functions.This was demonstrated in the chemical, morphological, and physiological data.For example, the decrease in red blood cell levels of intermediates such as the decrease in red blood cell glutathione produced hemolysis and morphological alterations of red cells in each of the species mentioned (except man).There are at least subtle hints that the erythrocytic function in man may in fact be under some circumstances and treatments vulnerable in the same direction.It is important to emphasize that definitive effects occurred at very high doses, much higher than we would expect to see from water disinfection.Few, if any effects were observed at the low doses, i.e., at 1-5 or 10 parts per million, which we would expect to be used in water treatment plants.It is likely that a unifying hypothesis on the mechanism of action could be derived from the original reports of Heffernan and others at the USEPA (HERL) laboratory, where it was shown that very high levels of hydrogen peroxide could be generated in the red cell after treatment of animals with chlo- rite.It is well known that hydrogen peroxide produces damage to cellular elements.However, the biological system has the capability of compen- sating for these oxidative results.Therefore,
Researchers Find Evidence of a New Gene for Late-Onset Alzheimer Disease
Journal: JAMA (1997)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Abstract 1596: Genome-wide gene-environmental interaction and random survival forest analyses: Insulin resistance and breast cancer risk
Authors: Su Yon Jung, Herbert Yu, Matteo Pellegrini, Jeanette Papp, Eric Sobel, Zuo-Feng Zhang
Journal: Cancer Research (2019)

Abstract Purpose: Obesity–insulin connections have been considered potential risk factors for postmenopausal breast cancer, and the association between insulin resistance (IR) genotypes and phenotypes can be modified by obesity-lifestyle factors, affecting breast cancer risk. We explored the role of IR in those pathways at the genome-wide level. With identified IR genetic factors and selected lifestyles, we further generated risk profiles for breast cancer risk. Methods: Using large-scale cohort data from postmenopausal women in the Women’s Health Initiative Database for Genotypes and Phenotypes Study, our previous genome-wide association gene–behavior interaction study identified 58 loci for associations with IR phenotypes (homeostatic model assessment–IR, hyperglycemia, and hyperinsulinemia). We evaluated those single-nucleotide polymorphisms (SNPs) and additional 31 lifestyles in relation to breast cancer risk by conducting a two-stage multimodal random survival forest analysis. Results: We identified the most predictive genetic and lifestyle variables in overall and subgroup analyses (stratified by body mass index [BMI], exercise, and dietary fat intake). Two SNPs (LINC00460 rs17254590 and MKLN1 rs117911989), endogenous and exogenous factors related to lifetime cumulative exposure to estrogen, BMI, and dietary alcohol intake were the most common influential factors across the analyses. Individual SNPs did not have significant associations with breast cancer, but the combined SNPs and lifestyles synergistically increased the risk of breast cancer in a dose-dependent manner. In subgroup analysis, we further stratified women by the two strongest variables (durations of oral contraceptive [OC] and opposed estrogen use). Shorter-duration OC and ever-opposed estrogen users with both risks of genotypes and lifestyles had greater risk (2.5 times and 6.3 times, respectively) than their counterparts without risks in both. This indicates the combined effect of SNPs and lifestyles and the joint effect with OC use and opposed estrogen on breast cancer risk. Conclusions: Our findings may contribute to more accurate prediction of breast cancer and suggest intervention strategies for those women with genetic risk to reduce their breast cancer risk. Citation Format: Su Yon Jung, Herbert Yu, Matteo Pellegrini, Jeanette Papp, Eric Sobel, Zuo-Feng Zhang. Genome-wide gene-environmental interaction and random survival forest analyses: Insulin resistance and breast cancer risk [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2019; 2019 Mar 29-Apr 3; Atlanta, GA. Philadelphia (PA): AACR; Cancer Res 2019;79(13 Suppl):Abstract nr 1596.
Error in Open Access Status
Authors: John Hutton
Journal: JAMA Pediatrics (2020)

Li Ming Wen, MD, MMed, PhD; Chris Rissel, PhD; Huilan Xu, MBiostat, PhD; Sarah Taki, PhD; Limin Buchanan, PhD; Karen Bedford, MPH; Philayrath Phongsavan, PhD; Louise A. Baur, MBBS, PhD
Epistaxis
Authors: Hadi Seikaly
Journal: New England Journal of Medicine (2021)

Initial management of epistaxis involves digital compression of the lower third of the nose for 15 to 20 minutes. Anterior bleeding not controlled with compression can usually be controlled with topical vasoconstrictors, tranexamic acid, cautery, or anterior nasal packing. If epistaxis continues, posterior packing, arterial ligation, or embolization may be used.
Rethinking Treatment for Patients With Diabetes and Cardiovascular Disease
Authors: M. Mitka
Journal: JAMA (2002)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Early warning skin signs in AIDS and persistent generalized lymphadenopathy (PGLS)
Authors: M.F. Muhlemann, M.G. Anderson, F.J. Paradinas, P.R. Key, B.A. Evans, I.M. Murray Lyon, J.J. Cream
Journal: British Journal of Dermatology (1985)

Cutaneous manifestations of AIDS include Kaposi's sarcoma, chronic genital herpes simplex, herpes zoster, mucocutaneous candidiasis, fungal infection, pyoderma and seborrhoeic dermatitis. We have consistently seen distinctive patterns of skin disease in male homosexuals with AIDS, PGLS and in a high‐risk group who initially had neither condition. The prevalence of skin disease was assessed in nine patients with AIDS, 15 with PGLS, four in the high‐risk group and in 40 male homosexual patients acting as controls. Three unusual patterns of skin disease were seen in patients with AIDS, PGLS and high‐risk patients that were not seen in the control groups. A florid neck and beard impetigo was present in seven patients; a chronic acneiform folliculitis in ten and extensive fungus infection in nine. Acneiform folliculitis began on the face, back and chest with follicular papulopustules that resembled acne vulgaris. Progression occurred with involvement of the perianal area, buttocks and thighs, often on a background of erythema and xerosis. An axillary folliculitis was present in eight patients. Histology showed a severe chronic lymphohistiocytic perifoUicular inflamma tion. Staphylococcus aureus was isolated occasionally from the AF and always from the impetigo. Relapse following treatment was common. There was extensive infection of the nails, feet, groins and trunk with Trichophyton rubrum. Other skin diseases noted were Kaposi's sarcoma, herpes simplex, herpes zoster, molluscum contagiosum, warts, candidiasis, vasculitis, eczema, seborrhoeic dermatitis, leukonychia and leukoplakia. In contrast the control group had no distinctive skin signs. Tinea pedis was present in four and genital warts in five patients. In male homosexuals we now view with concern the development of chronic acneiform folliculitis, neck and beard impetigo and intractable fungus infection.
Genetic and morphological findings in progressive familial intrahepatic cholestasis (Byler disease [PFIC-1] and Byler syndrome): Evidence for heterogeneity
Authors: L Bull
Journal: Hepatology (1997)

Byler disease (ByD) is an autosomal recessive disorder in which cholestasis of onset in infancy leads to hepatic fibrosis and death. Children who have a clinically similar disorder, but are not members of the Amish kindred in which ByD was described, are said to have Byler syndrome (ByS). Controversy exists as to whether ByD and ByS (subtypes of progressive familial intrahepatic cholestasis [PFIC]) represent one clinicopathological entity. The gene for ByD has been mapped to a 19-cM region of 18q21-q22. PFIC caused by a lesion in this region, including ByD, can be designated PFIC-1. Examination of haplotypes in siblings with ByS in two unrelated non-Amish families showed that the gene(s) responsible for their disorder(s) did not lie in the PFIC-1 candidate region. On light microscopy and transmission electron microscopy (TEM), liver tissue differed between Amish children with PFIC-1, who had coarsely granular bile and at presentation had bland intracanalicular cholestasis, and the children with ByS in the two non-Amish families, who had amorphous or finely filamentous bile and at presentation had neonatal hepatitis. Bile acid composition of bile also differed: In the Amish children with PFIC-1 and in one ByS family, the proportional concentration of chenodeoxycholic acid (CDCA) in bile was low compared with normal bile; in the other ByS family, it was only slightly reduced. Genetic analysis and light microscopy and TEM of liver may help distinguish PFIC-1 from other forms of ByS.
Notes on Diseases Treated by Medical Gymnastics and Massage.
Journal: JAMA (1922)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Abstracts for Society of Nuclear Medicine and Molecular Imaging, Greater New York and New England Chapters, 28th Annual Northeast Regional Scientific Meeting, Stamford, CT
Journal: Clinical Nuclear Medicine (2014)

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Leads from the MMWR. Drinking and driving and binge drinking in selected states, 1982 and 1985--the Behavioral Risk Factor Surveys
Journal: JAMA (1987)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Mast cells contribute to synovial inflammation in non-psoriatic and psoriatic spondyloarthritis
Authors: T. Noordenbos, N. Yeremenko, T. Cantaert, C. Teitsma, J. Canete, M. van de Sande, P. P. Tak, D. Baeten
Journal: Annals of the Rheumatic Diseases (2010)
Hypothyroidism in polymyalgia rheumatica and giant cell arteritis
Authors: G S Rai, P Wiseman, K Stewart
Journal: BMJ (1990)

Hypothyroidism in polymyalgia rheumatica and giant cell arteritis SIR,-The paper by Dr B Dasgupta and colleagues showing no association between polymyalgia rheumatica or giant cell arteritis and hypothyroidism in 77 patients' agreed with our experience.After reading of such an association,2 we measured the thyroid function of patients being followed up for these conditions in our rheumatology clinic.
Part of Navy Psychiatric Screening Process Questioned
Journal: JAMA (1964)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Bioinstructive Naringin‐Loaded Micelles for Guiding Stem Cell Osteodifferentiation
Authors: Pedro Lavrador, Vítor M. Gaspar, João F. Mano
Journal: Advanced Healthcare Materials (2019)

Adv. Healthcare Mater. 2018, 7, 1800890 In the original version of this article, the images in Figure 4D were incorrect. The correct figure is shown below. This mistake was due to errors in the assembly of the original figure, and do not affect the conclusions published in this work. The authors apologize for any confusion caused.