tbuckley/cabot-search huggingface dataset:
Abstract 9109: Patient-Specific Ipsc-Derived Cardiac Fibroblasts Phenotypes in Lmna Cardiomyopathy
Authors: Jiajia Yang, Mariana Argenziano, Mariana Burgos Angulo, Thomas V McDonald
Journal: Circulation (2021)

Introduction: Mutations in LMNA (encoding lamin A/C) are an important cause of familial Dilated Cardiomyopathy (DCM), characterized by systolic ventricular dysfunction and prominent dysrhythmias. However, little is known about how diverse mutations in LMNA can vary in molecular pathways leading to DCM other than affecting cardiomyocytes (CMs). Induced pluripotent stem cells (iPSCs) retain the genetic identity of the individual donor and enable the generation of patient-specific cells to enhance Precision Medicine. We generated CMs and cardiac fibroblasts (CFs) from patient-specific iPSCs harboring various LMNA mutations to compare their cellular phenotypes. Hypothesis: We hypothesize that different mutation locations within LMNA lead to DCM by affecting both CMs and CFs. Methods: iPSCs from seven DCM patients with heterozygous mutations in LMNA (M1I, R216C/R399H, R216C#1, R216C#2, R335Q, R377H and R541C) were differentiated into CMs and CFs. Nuclear morphology, sarcomere structure, molecular signatures, cell migration rate, and RNA-sequencing were analyzed in CMs and CFs. Results: Immunostaining of lamin A/C displayed no discernable nuclear deformity in LMNA -mutated CMs, whereas CFs showed dramatic nuclear morphologic defects of varying degrees between patient cell lines. Staining of troponin T revealed pronounced sarcomere disarray due to LMNA mutation. LMNA -patient lines exhibited dysregulated expression of connexin 43, MMP1, collagen 1 and 3, and fibronectin 1 in CFs assessed by qPCR and immunoblot, and reduced cell migration rate detected by wound healing assay. RNA-sequencing of variant R377H demonstrated differentially expressed genes in both CMs (up:1500, down:2630, padj&lt;0.05) and CFs (up:491, down:783, padj&lt;0.05). LMNA R377H CMs showed altered Gene Ontology terms including MAPK pathway, cardiac muscle contraction and calcium signaling pathway. Enrichment analysis of R377H CFs highlighted extracellular matrix constituent and organization pathway. Conclusions: CFs exhibit mutation-specific molecular and cellular pathophysiology in LMNA related DCM. Further functional studies may uncover novel therapeutic targets in cardiac fibroblasts for the treatment of laminopathies.
Incorporating Structural Competency Into Dermatologic Curricula—Reply
Authors: Jenna C. Lester, Susan C. Taylor
Journal: JAMA Dermatology (2021)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA Dermatology HomeNew OnlineCurrent IssueFor Authors Podcast Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA Dermatology journal
Chronic Lymphocytic Leukemia (CLL) Clonal Growth Rate Is Slower Following Venetoclax-Rituximab (VenR): Results from a Minimal Residual Disease (MRD) Model from the Randomized Phase 3 Murano Trial
Authors: Arnon P. Kater, Tong Lu, Anton W. Langerak, Clemens Mellink, Brenda Chyla, Jenny Qun Wu, Michelle Boyer, Marcus Lefebure, Yanwen Jiang, John F. Seymour
Journal: Blood (2021)
Targeting Metabolic Alterations in CLL Microenvironment; Inhibition of Glutamine Import Attenuates Venetoclax Resistance
Authors: Zhenghao Chen, Helga Simon-Molas, Gaspard Cretenet, Beatriz Valle-Argos, Francesco Forconi, Bauke V Schomakers, Michel van Weeghel, Dean Bryant, Jaco A. C. Van Bruggen, Fleur Peters, Gerritje J. W. van der Windt, Arnon P. Kater, Graham Packham, Eric Eldering
Journal: Blood (2021)
Neutrophil and platelet increases with luspatercept in lower-risk MDS: secondary endpoints from the MEDALIST trial
Authors: Guillermo Garcia-Manero, Ghulam J. Mufti, Pierre Fenaux, Rena Buckstein, Valeria Santini, María Díez-Campelo, Carlo Finelli, Osman Ilhan, Mikkael A. Sekeres, Amer M. Zeidan, Rodrigo Ito, Jennie Zhang, Anita Rampersad, Daniel Sinsimer, Jay T. Backstrom, Uwe Platzbecker, Rami S. Komrokji
Journal: Blood (2021)
Efficacy and Safety of Recombinant Factor IX-FIAV and Bypassing Agents in Thrombin Generation Analyses in Hemophilia A Patient Plasma: The FIVITAS Study
Authors: Lorenzo G.R. Romano, Viola J.F. Strijbis, Ka Lei Cheung, Ying Poi Liu, Andrew C. McCreary, Frank W.G. Leebeek, Mettine H.A. Bos
Journal: Blood (2021)
A very rare case of submucosal invasive duodenal adenocarcinoma in a patient with a history of radiation therapy
Authors: Pierre Lafeuille, Jérôme Rivory, Thomas Lambin, Paul Bonniaud, Thierry Ponchon, Florian Rostain, Mathieu Pioche
Journal: Endoscopy (2021)

Adenocarcinomas of the small intestine are rare compared with other gastrointestinal carcinomas. They are mostly found situated in the duodenum. Despite an increasing incidence of duodenal carcinomas in recent decades, most such lesions are either intramucosal or deep invasive carcinomas; intermediate invasive cancers in the submucosa are extremely rare, having been described only in a few cases in the Japanese language [1] [2]. Because of the rarity of their incidence, the exact risk of lymph node recurrence for these submucosal tumors is unknown. Therefore, the importance of R0 resection by endoscopic submucosal dissection to obtain a perfect histological assessment is debatable as submucosal invasion usually leads to salvage surgery being performed. In our patient, who had a past history of radiation therapy, endoscopic submucosal dissection was performed ([Fig. 1]; [Video 1]), and histological assessment revealed a well-differentiated adenocarcinoma invading the submucosa to a depth of 250 µm ([Fig. 2]), without any evidence of lymphovascular invasion or tumor budding.
Impact of COVID-19 Pandemic on Global Unrelated Stem Cell Donations in 2020 - Report from World Marrow Donor Association
Authors: Monique Jöris, Stefanie Bernas, Alexander H. Schmidt, Jay Feinberg, Nicoletta Sacchi, Heidi Elmoazzen, Fatma Oguz, Danielli Oliveira, Kuo-Liang Yang, Soraya Moomivand, Seied Asadullah Mousavi, Helder Trindade, Juliana Villa López, Mirjam Fechter, Guldane Seval Cengiz, Thaneya Jeyarajah, Steven Devine, Bronwen E. Shaw, Stephen J Forman, Lydia Foeken
Journal: Blood (2021)
Abstract 9217: Characterizing Barriers to Fetal Cardiac Care Within the Greater Chicago Area
Authors: Joyce L Woo, Rupali Gandhi, Matthew M Davis, Angira Patel, Lynn Yee, Joyce T Johnson
Journal: Circulation (2021)

Introduction: Prenatal diagnosis (PND) is key in the treatment of congenital heart disease (CHD), the most common birth defect. National PND rates remain only ~50%, yet barriers to PND are region-specific. We aimed to identify sociodemographic barriers to CHD PND within the Greater Chicago Area. Methods: Infants &lt; 12 months of age with CHD that could be diagnosed by fetal echocardiography, who received cardiac surgery between 2016 and 2020, were identified from the Society of Thoracic Surgeons (STS) database at Lurie Children’s Hospital. Sociodemographic variables of maternal-infant dyads were collected from STS and electronic medical records. ZIP codes were linked to US Census Bureau data. Multivariable logistic regression and mediation analysis were used to analyze relationships between sociodemographic variables and prenatal diagnosis. Results: In total, 558 dyads met inclusion criteria. Of these, 58% had PND. In unadjusted analysis, those with private insurance were 1.6 times likelier to have PND than those with public insurance (95% CI 1.1-2.2, Table 1). However, in adjusted analysis (Table 1), payor status was an effect modifier of the associations between critical CHD status (public vs. private payor OR 11.4 vs 3.1) and maternal ZIP within a far suburb (public vs. private payor OR 15.9 vs. 0.7) on PND. There were no differences in PND by maternal race or ethnicity. In a secondary mediation analysis, the indirect effect of receiving transportation aid mediated 54% (p=0.005) of the total effect between maternal public insurance status and PND. Conclusions: Maternal payor status is strongly associated with CHD PND, and modifies the relationship between geographic factors, critical CHD, and PND in the Greater Chicago area. For those residing in far suburbs, PND is paramount for access to surgical care. Transportation aid lies in the causal pathway between payor status and PND. Future study will identify modifiable factors to improve access to CHD PND at the state level.
Long-Term Renal Outcomes in Hereditary TTP Patients: Data from the International Hereditary TTP Registry
Authors: Erika Tarasco, Ralph Wendt, Anne Sophie von Krogh, Sibylle Tschumi, James N. George, Masanori Matsumoto, Paul Knöbl, Reinhard Schneppenheim, Bernhard Lämmle, other members of Hereditary TTP Registry Steering Committee, Johanna A. Kremer Hovinga Strebel
Journal: Blood (2021)
Non-Viral <i>Sleeping Beauty</i> Transposon Engineered CD19-CAR-NK Cells Show a Safe Genomic Integration Profile and High Antileukemic Efficiency
Authors: Tobias Bexte, Lacramioara Botezatu, Csaba Miskey, Julia Campe, Lisa Marie Reindl, Veronika Gebel, Winfried S Wels, Michael Hudecek, Zoltan Ivics, Evelyn Ullrich
Journal: Blood (2021)
Homeoprotein SIX1 compromises antitumor immunity through TGF-β-mediated regulation of collagens
Authors: Wancheng Liu, Meiling Gao, Lili Li, Yu Chen, Huimin Fan, Qiaomei Cai, Yueyue Shi, Chaohu Pan, Junxiao Liu, Lucy S. Cheng, Heng Yang, Genhong Cheng
Journal: Cellular and Molecular Immunology (2021)

Abstract The tumor microenvironment (TME), including infiltrated immune cells, is known to play an important role in tumor growth; however, the mechanisms underlying tumor immunogenicity have not been fully elucidated. Here, we discovered an unexpected role for the transcription factor SIX1 in regulating the tumor immune microenvironment. Based on analyses of patient datasets, we found that SIX1 was upregulated in human tumor tissues and that its expression levels were negatively correlated with immune cell infiltration in the TME and the overall survival rates of cancer patients. Deletion of Six1 in cancer cells significantly reduced tumor growth in an immune-dependent manner with enhanced antitumor immunity in the TME. Mechanistically, SIX1 was required for the expression of multiple collagen genes via the TGFBR2-dependent Smad2/3 activation pathway, and collagen deposition in the TME hampered immune cell infiltration and activation. Thus, our study uncovers a crucial role for SIX1 in modulating tumor immunogenicity and provides proof-of-concept evidence for targeting SIX1 in cancer immunotherapy.
Publisher Correction: Outcome of older (≥70 years) APL patients frontline treated with or without arsenic trioxide—an International Collaborative Study
Authors: Sabine Kayser, Ramy Rahmé, David Martínez-Cuadrón, Gabriel Ghiaur, Xavier Thomas, Marta Sobas, Agnes Guerci-Bresler, Ana Garrido, Arnaud Pigneux, Cristina Gil, Emmanuel Raffoux, Mar Tormo, Norbert Vey, Javier de la Serna, Olga Salamero, Eva Lengfelder, Mark J. Levis, Pierre Fenaux, Miguel A. Sanz, Uwe Platzbecker, Richard F. Schlenk, Lionel Adès, Pau Montesinos
Journal: Leukemia (2021)
Abstract 9337: Right Ventricular's Strain Behavior in Patients Treated With Chemotherapy / Radiotherapy in a Patient Cohort
Authors: Grisel Canahuate, Amparo Taveras Hiraldo, Eleana Rivera, Saray Perez, miguel arias
Journal: Circulation (2021)

Introduction: Is well known the usefulness of left ventricle (LV) longitudinal global strain (GLS) in patients receiving antineoplastic therapy but studies suggested a similar behavior of the right ventricle (RV) free wall longitudinal strain (FWLS). Still, many controversies exists that limit its evaluation, so this study is a contribution to this need for practicality. Objective: To assess the behavior and usefulness of RV FWLS in patients treated with chemotherapy and / or radiotherapy and its relationship with LV GLS. Method: A data collection protocol of demographic variables and personal history was applied to patients diagnosed with cancer treated with chemotherapy and/or radiotherapy who underwent an echocardiogram with strain at our center from November 2020 to March 2021. Echocardiogram performed accordingly with American Society of Echocardiography guidelines standards, including a LV GLS and a RV FWLS. The data was analyzed in the SPSS. Results: A total of 84 patients collected. Women 89%, mean age 59 ±11 years, 73% breast cancer. Hypertension 54%, 27% obesity, 23% dyslipidemia, 19% were diabetic and 11% smokers. Received concomitantly chemotherapy and radiotherapy 46% and 48% treatment was more than a year ago. Echocardiographic findings: mean LV ejection fraction (EF) 64±7 %, with LV GLS being reduced in 82% (44% being less than -15%). Similarly, 74% of RV FWLS was reduced, but only 15% of the S wave of the RV by tissue Doppler was abnormal, 7% of TAPSE was reduced, and 6% of fractional area change (FAC). The Pearson correlation coefficient between LV GLS and RV FWLS was 0.463 with a p value of &lt;.001. Conclusion: Our study suggests that RV FWLS has a similar behavior as LV GLS and is a better predictor of RV dysfunction than conventional parameters and should be incorporated in echocardiographic evaluation of patients receiving antineoplastic therapy.
Abstract 9448: The Effect of Diabetes on Cardiovascular Events After Coronary Artery Bypass Grafting versus Drug-Eluting Stents for Left Main Coronary Artery Disease: A Meta-Analysis
Authors: Marc-Andre d'Entremont, Ryuichiro Yagi, Soziema J Salia, Shuqi Zhang, Lamyaa Shaban, Yakubu Bene-Alhasan, Stefania Papatheodorou, Etienne L Couture, Thao Huynh, Michel Nguyen, Rikuta Hamaya
Journal: Circulation (2021)

Background: The optimal method of coronary revascularization for diabetes mellitus (DM) patients with left main coronary artery disease (LMCAD) is controversial in the drug-eluting stent (DES) era. We performed a systematic review and meta-analysis comparing DES-based percutaneous coronary intervention (PCI) to coronary artery bypass grafting (CABG) for LMCAD in DM patients and tested for potential effect measure modification (EMM) by diabetes on major adverse cardiovascular and cerebrovascular events (MACCE). Methods and Results: We included all randomized controlled trials (RCTs) and observational studies comparing CABG to DES-based PCI including DM patients with LMCAD published up to March 1, 2021. We completed separate random-effects meta-analyses for four RCTs (4,356 patients, mean follow-up of 4.9 years) and six observational studies (9,360 patients, mean follow-up of 5.2 years). DM and non-DM patients were at increased risk of the composite endpoint of all-cause mortality, myocardial infarction, stroke, and unplanned revascularization when comparing CABG to DES-based PCI (p-value for interaction = 0.70) (Figure 1, DM =1, Non-DM =0). In observational studies, there was no difference between DES-based PCI and CABG for all-cause mortality in patients with DM (Figure 2). Conclusion: DES-based PCI was associated with an increased risk of MACCE compared to CABG in LMCAD patients irrespectively of DM status. We did not observe significant EMM by DM status. Considering these data, heart teams could consider DM as one of the many components in the clinical decision-making process, but not as a primary deciding factor between DES and CABG for LMCAD.
Immune Profiling of Diagnostic DLBCL Biopsies Dramatically Improves upon Cell-of-Origin Risk Stratification
Authors: Michael Nissen, Xuehai Wang, Clementine Sarkozy, Aixiang Jiang, Daisuke Ennishi, Tomohiro Aoki, Stacy Hung, Elizabeth Chavez, Jeffrey W Craig, Pedro Farinha, Graham W. Slack, David W. Scott, Christian Steidl, Andrew P. Weng
Journal: Blood (2021)
Quantification of Sepsis Model Alerts in 24 US Hospitals Before and During the COVID-19 Pandemic
Authors: Andrew Wong, Jie Cao, Patrick G. Lyons, Sayon Dutta, Vincent J. Major, Erkin Ötleş, Karandeep Singh
Journal: JAMA Network Open (2021)

This descriptive study evaluates the association between nursing reports of sepsis overalerting and alert volume by quantifying the number of alerts generated by the Epic Sepsis Model at 24 US hospitals before and during the COVID-19 pandemic.
Real-World Management of Patients with Newly Diagnosed Acute Myeloid Leukemia Treated with Venetoclax-Based Regimens: Results from the AML Real World Evidence (ARC) Initiative
Authors: Pankit Vachhani, Ofir Wolach, Jacqueline S. Garcia, Joshua F. Zeidner, Chetasi Talati, Daniel A. Pollyea, Catherine Lai, Yakir Moshe, Sameem Abedin, David Lavie, Tsila Zuckerman, Marin Xavier, Sangmin Lee, Evan Chen, Wesleigh Edwards, Cat N. Bui, Anders Svensson, Steve Kye, Rebecca Burne, Jessica Maitland, Esprit Ma, Melissa Montez, Moshe Grunspan, Aaron D Goldberg
Journal: Blood (2021)
Validating a Machine Learning Grading System for Acute Gvhd. a Study on Behalf of the EBMT Transplant Complications Working Party
Authors: Amin T. Turki, Christophe Peczynski, Olaf Penack, Helene Schoemans, Gerard Socie, Christian Reinhardt, Nathalie Fegueux, Didier Blaise, Nicolaus Kröger, Stephan Mielke, Jan J. Cornelissen, Patrice Chevallier, Ibrahim Yakoub-Agha, Edouard Forcade, Grzegorz W Basak, Zinaida Perić
Journal: Blood (2021)
TERT Rare Variants in Myeloid Neoplasia: Lack of Clinical Impact or Role as Risk Alleles
Authors: Carmelo Gurnari, Adam Wahida, Simona Pagliuca, Tariq Kewan, Waled Bahaj, Minako Mori, Laila Terkawi, Ishani Pandit, Misam Zawit, Hetty E. Carraway, Torsten Haferlach, Jaroslaw P. Maciejewski, Valeria Visconte
Journal: Blood (2021)
Incorporating Structural Competency Into Dermatologic Curricula
Authors: Erica Ogwumike, Donald A. Glass, Rebecca Vasquez
Journal: JAMA Dermatology (2021)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA Dermatology HomeNew OnlineCurrent IssueFor Authors Podcast Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA Dermatology journal
Abstract 12948: Both Electronic and Regular Cigarettes Induce Transcriptional Changes in iPS-Derived Cardiomyocytes
Authors: Hesham Basma, Brian D Lowes, Kajari Dhar, Fang Qiu
Journal: Circulation (2021)

Introduction: Cigarette smoking is an important etiology for cardiac diseases and electronic cigarettes are increasing in popularity. In the current study, we sought to assess differential gene expression in iPS-cardiomyocytes after exposure to conventional cigarette smoke extract (CSE) and electronic cigarette extracts (ECE) utilizing direct digital mRNA detection of heart failure targeted genes. Hypothesis: Methods: Human cardiomyocytes derived from iPSCs were acutely exposed for two consecutive days with 2.5% of CSE and ECE. RNAs were isolated and NanoString analysis was used to compare gene expression between groups for 126 heart failure specific genes. NanoString mRNA raw count data were preprocessed and normalized using nSolver 4.0 Analysis SoftwareNano-string of customized target panel was run for mRNA expression of cardiomyocytes with direct exposure to CSE (N=3) and ECE (N=3) and was compared to control (N=3). At least a 1.5-fold change in gene expression with FDR adjusted p&lt;0.05 was considered as significant. Results: Five genes were significantly differentially expressed cardiomyocytes exposed to CSE vs control (CETP, ATRLN1, ENOS, C10ORF88 and ERF). In ECE exposed cells, five genes were also significantly differentially expressed compared to control (PDK4, CAV3, PER1, MYH6 and ERF). ERF gene is a transcription factor and protooncogene involved in development, apoptosis, and the regulation of telomerase was significantly overexpressed in cells exposed to both CSE and ECE. Despite different nicotine concentrations in ECE and CSE, no significant gene differences between CSE and ECE were observed. Conclusions: ECE and CSE exposure alters transcriptional profiles in iPS-cardiomyocytes compared to controls in heart failure targeted genes. These genes are involved in transcription and circadian regulation, metabolism and contractile proteins. There were no significant differences in transcriptional profiles between ECE and CSE exposed cells.
IL7R Targeting Using OSE-127 Shows Robust Anti-Leukemic Activity in B-Cell Precursor Acute Lymphoblastic Leukemia Xenografts
Authors: Lennart Lenk, Irène Baccelli, Dorothee Winterberg, Frédérique Corallo, Fotini Vogiatzi, Lea Spory, Martin Schrappe, Gunnar Cario, Anna Laqua, Monika Brüggemann, Nicolas Poirier, Denis Martin Schewe
Journal: Blood (2021)
Shared and Distinct Genetic Features in Human and Canine B-Cell Lymphomas
Authors: Tiana Hillman, Matthew Cheung, Bruno M. Grande, Kevin R Bushell, Sarah E. Arthur, Miguel Alcaide, Nicole Thomas, Kostiantyn Dreval, Jovanveer Shoker, Krishanna Campbell, Stephanie Wong, Ryan D Morin, Krysta M. Coyle
Journal: Blood (2021)
A Phase 1 Dose Escalation Study of Protein Arginine Methyltransferase 5 (PRMT5) Inhibitor PRT543 in Patients with Myeloid Malignancies
Authors: Manish R. Patel, Varun Monga, Shekeab Jauhari, Don Stevens, Lucia Masarova, Meredith McKean, David Mauro, John Viscusi, Peggy Scherle, Neha Bhagwat, Andrew Moore, William Sun, Rachel Chiaverelli, Eric Mintah, Rajneesh Nath, Eytan M. Stein, Neil Palmisiano, Grerk Sutamtewagul, Srdan Verstovsek
Journal: Blood (2021)
Exploring Alternative Dosing Regimens of Single-Agent Belantamab Mafodotin on Safety and Efficacy in Patients with Relapsed or Refractory Multiple Myeloma: DREAMM-14
Authors: Malin Hultcrantz, David Kleinman, Pavandeep Ghataorhe, Astrid McKeown, Wei He, Thomas Ling, Roxanne C Jewell, Joseph Brunner, Julie Byrne, Laurie Eliason, Emma Scott, Joanna Opalinska
Journal: Blood (2021)