tbuckley/cabot-search huggingface dataset:
Epigenetic Reader BRD4 (Bromodomain-Containing Protein 4) Governs Nucleus-Encoded Mitochondrial Transcriptome to Regulate Cardiac Function
Authors: Soo Young Kim, Xin Zhang, Gabriele G. Schiattarella, Francisco Altamirano, Thais A.R. Ramos, Kristin M. French, Nan Jiang, Pamela A. Szweda, Bret M. Evers, Herman I. May, Xiang Luo, Hongliang Li, Luke I. Szweda, Vinicius Maracaja-Coutinho, Sergio Lavandero, Thomas G. Gillette, Joseph A. Hill
Journal: Circulation (2020)

BET (bromodomain and extraterminal) epigenetic reader proteins, in particular BRD4 (bromodomain-containing protein 4), have emerged as potential therapeutic targets in a number of pathological conditions, including cancer and cardiovascular disease. Small-molecule BET protein inhibitors such as JQ1 have demonstrated efficacy in reversing cardiac hypertrophy and heart failure in preclinical models. Yet, genetic studies elucidating the biology of BET proteins in the heart have not been conducted to validate pharmacological findings and to unveil potential pharmacological side effects.
Annals Graphic Medicine - Dr. Mom: 2015 Versus 2020
Authors: Grace E. Farris
Journal: Annals of Internal Medicine (2020)

Web Exclusives3 November 2020Annals Graphic Medicine - Dr. Mom: 2015 Versus 2020FREEGrace E. Farris, MDGrace E. Farris, MDUniversity of Texas, Austin, Texas (G.E.F.)Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/G20-0102 SectionsAboutVisual AbstractPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail Download figure Download PowerPoint Comments0 CommentsSign In to Submit A Comment Author, Article, and Disclosure InformationAffiliations: University of Texas, Austin, Texas (G.E.F.)Disclosures: The author has disclosed no conflicts of interest. The form can be viewed at www.acponline.org/authors/icmje/ConflictOfInterestForms.do?msNum=G18-0042.Author/Illustrator Information: Grace E. Farris, MD, is an assistant professor of medicine at Dell Medical School at the University of Texas in Austin, Texas. You can view her work at her website: www.farrisgrace.com.About Dr. Mom: In Dr. Mom, physician and cartoonist Grace Farris examines work–life balance while mothering and doctoring. Dr. Mom appears monthly in Annals Graphic Medicine. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics 3 November 2020Volume 173, Issue 9Page: W145-W146 ePublished: 3 November 2020 Issue Published: 3 November 2020 Copyright & PermissionsCopyright © 2020 by American College of Physicians. All Rights Reserved.PDF downloadLoading ...
Administration of KER-047, a Novel ALK2 Inhibitor, Elicited Robust and Sustained Increases in Serum Iron in Healthy Participants
Authors: Claudia Ordonez, Snyder Ben, Rachel Barger, Timna Serino, Claire C Tseng, Christopher Rovaldi, Jenn Lachey, Jasbir Seehra
Journal: Blood (2020)
Abstract MP114: Cardiotropic Adenovirus Increases Arrhythmia Susceptibility During Acute Infection
Authors: Rachel L Padget, D. Ryan King, Michael D North, Mira N Tanenbaum, Patrick J Calhoun, Gregory S HOEKER, Steven Poelzing, James W Smyth
Journal: Circulation Research (2020)

Myocarditis is responsible for up to 42% of sudden cardiac death in young adults yet mechanisms underlying virally-induced arrhythmia remain elusive. Adenovirus is a leading etiological agent of myocarditis but due to species-specificity, in vivo models are limited. Altered cardiac gap junction function underlies sudden cardiac death and given that gap junctions also propagate antiviral immune responses, we hypothesized adenovirus would target intercellular coupling, leading to arrhythmogenesis. Employing a recently described strain of mouse adenovirus, MAdV-3, we investigated cardiotropism, arrhythmia susceptibility, and molecular mechanisms of a virally-induced arrhythmogenic substrate. Adult C57BL/6 mice were inoculated with MAdV-3 and infection allowed to progress for one week. Viral genomes were detected by qPCR to measure tissue tropism together with whole animal histopathology. Echo- and electrocardiography were used to measure cardiac function and electrical activity. Viral effects on transcription and junctional protein/ion channel expression and localization were quantified using RT-qPCR, confocal microscopy, and western blotting. Finally, ex vivo optical mapping was employed to test conduction defects and susceptibility to arrhythmia. We find MAdV-3 viral genomes specifically enriched in heart tissue, confirming cardiotropism. Immune cell infiltration was not detected in the heart and no cardiomyopathy was apparent by echocardiography. RR interval widening, however, does occur in MAdV-3 infected mice with reductions in cardiac ion channel and gap junction mRNA transcripts. Optical mapping experiments show that conduction velocity is altered in acutely infected hearts and norepinephrine challenge uncovered electrical impairment presenting as atrioventricular dissociation with increased premature ventricular contraction burden. Our data demonstrate that MAdV-3 is cardiotropic and acute infection result in changes in cardiac electrical activity prior to development of cardiomyopathy or a detectable immune response. MAdV-3 infection is a novel model for adenoviral myocarditis, providing insights into viral subversion of cardiac intercellular coupling and mechanisms of arrhythmogenesis.
Largest Cohort Study of Gaucher Disease Type 3 from a Single Center in Egypt Spanning Two Decades
Authors: Amal El-Beshlawy, Khaled Mohamed Abd El-Azim Eid, Amina AbdelSalam, Fadwa Said Mohamed, Yasmeen M.M. Selim, Nadeen Gebril, Ekram Fateen, Pramod K. Mistry
Journal: Blood (2020)
Early Clinical Results of a Novel Anti-CD20 Chimeric Antigen Receptor (CAR)-T Cell Therapy for B-Cell NHL Patients Who Are Relapsed/Resistant Following CD19 CAR-T Therapy
Authors: Shiguang Ye, Jiaqi Huang, Shigui Zhu, Xin Yao, Lili Zhou, Judy Zhu, Chengxiao Zheng, Shiyi Chen, Liping Lan, Shicheng Chen, Yutian Wei, Wangqin Shu, Chen Ye, Dan Zhang, Rirfei Wang, Yi Hong, Jiaqiang Ren, Li Zhang, Wenjun Zhang, Xiaochen Tang, Junbang Wang, Jie Liu, Michael Humphries, Ping Li, Yihong Yao, Aibin Liang
Journal: Blood (2020)
A Real-World Assessment of Hepatic Dysfunction Among Chronic Phase Chronic Myeloid Leukemia (CP-CML) Patients Receiving First-Line (1L) Tyrosine Kinase Inhibitors (TKIs) in the United States
Authors: Kathryn Kolibaba, Jie Zhou, Scott Justin Keating, Jamyia Clark, John Brokars, Arianna Kee, Ronda Copher, Brian Stwalley, Elias Jabbour
Journal: Blood (2020)
Prevalence of Inherited Predisposition Syndromes in Young Patients with Acute Myeloid Leukemia and Aberrant Karyotype
Authors: Martin Kirschner, Benjamin Rolles, Martina Crysandt, Christoph Röllig, Friedrich Stolzel, Martin Bornhäuser, Carsten Müller-Tidow, Kim Kricheldorf, Margherita Vieri, Matthias Begemann, Angela Maurer, Jens Panse, Susanne Isfort, Marcin Wlodarski, Sushree Sangita Sahoo, Tim H Brümmendorf, Edgar Jost, Fabian Beier
Journal: Blood (2020)
Standardization of <sup>18</sup>F-FDG–PET/CT According to Deauville Criteria for Metabolic Complete Response Definition in Newly Diagnosed Multiple Myeloma
Authors: Elena Zamagni, Cristina Nanni, Luca Dozza, Thomas Carlier, Clément Bailly, Paola Tacchetti, Annibale Versari, Stephane Chauvie, Andrea Gallamini, Barbara Gamberi, Denis Caillot, Francesca Patriarca, Margaret Macro, Mario Boccadoro, Laurent Garderet, Simona Barbato, Stefano Fanti, Aurore Perrot, Francesca Gay, Peter Sonneveld, Lionel Karlin, Michele Cavo, Caroline Bodet-Milin, Philippe Moreau, Françoise Kraeber-Bodéré
Journal: Journal of Clinical Oncology (2020)

18F-Fluorodeoxyglucose (FDG) positron emission tomography (PET)/computed tomography (CT) is currently the standard technique to define minimal residual disease (MRD) status outside the bone marrow (BM) in patients with multiple myeloma (MM). This study aimed to define criteria for PET complete metabolic response after therapy, jointly analyzing a subgroup of newly diagnosed transplantation-eligible patients with MM enrolled in two independent European randomized phase III trials (IFM/DFCI2009 and EMN02/HO95).Two hundred twenty-eight patients were observed for a median of 62.9 months. By study design, PET/CT scans were performed at baseline and before starting maintenance (premaintenance [PM]). The five-point Deauville scale (DS) was applied to describe BM (BM score [BMS]) and focal lesion (FL; FL score [FS]) uptake and tested a posteriori in uni- and multivariable analyses for their impact on clinical outcomes.At baseline, 78% of patients had FLs (11% extramedullary), 80% with an FS ≥ 4. All patients had BM diffuse uptake (35.5% with BMS ≥ 4). At PM, 31% of patients had visually detectable FLs (2% extramedullary), 24% and 67.7% of them with an FS of 3 and ≥ 4, respectively. At PM, 98% of patients retained residual BM diffuse uptake, which was significantly lower than at baseline (mainly between BMS 2 and 3, BMS was ≥ 4 in only 8.7% of patients). By both uni- and multivariable analysis, FS and BMS < 4 were associated with prolonged progression-free survival (PFS) and overall survival (OS) at PM (OS: hazard ratio [HR], 0.6 and 0.47, respectively; PFS: HR, 0.36 and 0.24, respectively).FL and BM FDG uptake lower than the liver background after therapy was an independent predictor for improved PFS and OS and can be proposed as the standardized criterion of PET complete metabolic response, confirming the value of the DS for patients with MM.
<i>In situ</i> tumor-triggered subcellular precise delivery of multi-drugs for enhanced chemo-photothermal-starvation combination antitumor therapy
Authors: Xinglu Jiang, Xiaobo Fan, Rui Zhang, Wei Xu, Hailu Wu, Fengfeng Zhao, Han Xiao, Chen Zhang, Chenggui Zhao, Guoqiu Wu
Journal: Theranostics (2020)

Drug combination therapy for cancer treatment exerts a more potent antitumor effect. The targeted delivery and release of multiple drugs in a patient's body thus presents a more effective treatment approach, warranting further research.
Polyreactivity and Somatic Hypermutation Analysis Reveals the Innate B Cell Origin of Human PF4/Heparin Reactive Antibodies
Authors: Wen Zhu, Lu Zhou, Ting Zhao, Yongwei Zheng, Mei Yu, Jianhui Wei, Anand Padmanabhan, Richard H. Aster, Demin Wang, Renren Wen
Journal: Blood (2020)
Thalidomide Plus Prednisone and Methotrexate for Symptomatic Large Granular Lymphocyte Leukemia: A Prospective, Single-Center, Pilot Study
Authors: Shuhua Yi, Jun Du, Ying Yu, Yang Jiao, Yi Wang, Huijun Wang, Tingyu Wang, Rui Lv, Wei Liu, Wenjie Xiong, Huimin Liu, Wenyang Huang, Dehui Zou, Gang An, Yaozhong Zhao, Jianxiang Wang, Lugui Qiu
Journal: Blood (2020)
A Phase II Study to Investigate the Efficacy and Safety of Eltrombopag in Combination with Dexamethasone As First-Line Treatment in Adult Patients with Newly Diagnosed Primary ITP (XPAG-ITP)
Authors: Axel Matzdorff, Mascha Binder, Falk Nimmerjahn, Oliver Meyer, Mathias J. Rummel, Tamara Tesanovic, Tanja Sauer
Journal: Blood (2020)
High Doses of Targeted Radiation with Anti-CD45 Iodine (131I) Apamistamab [Iomab-B] Do Not Correlate with Incidence of Mucositis, Febrile Neutropenia or Sepsis in the Prospective, Randomized Phase 3 Sierra Trial for Patients with Relapsed or Refractory Acute Myeloid Leukemia
Authors: Boglarka Gyurkocza, Rajneesh Nath, Hannah Choe, Stuart Seropian, Patrick J. Stiff, Sunil Abhyankar, Edward Agura, Mark Litzow, Benjamin K. Tomlinson, George L. Chen, Parameswaran Hari, Johnnie Orozco, Zaid S. Al-Kadhimi, Camille Abboud, Koen Van Besien, Mitchell Sabloff, Margarida Magalhaes Magalhaes-Silverman, James M. Foran, Michael W. Schuster, Partow Kebriaei, Moshe Levy, Hillard M Lazarus, Sergio A. Giralt, Qing Liang, Mark S. Berger, Vijay Reddy, John M. Pagel
Journal: Blood (2020)
Treatment of Severe, Drug-Refractory Viral Infections with Allogeneic, Off-the-Shelf Multi-Virus Specific T Cells in Patients Following HSCT: Results from a Phase 2 Study
Authors: Ifigeneia Tzannou, Ayumi Watanabe, Anastasia Papadopoulou, Manik Kuvalekar, Swati Naik, Premal Lulla, Robert A. Krance, Carlos A. Ramos, Helen E. Heslop, Ann M. Leen, Bilal Omer
Journal: Blood (2020)
老人ホーム入居者の肺炎予防および生存改善に対する23価肺炎球菌ワクチンの有効性:二重盲検,無作為化,プラセボ対照試験
Authors: Maruyama Takaya, Taguchi Osamu, Niederman Michael S, Morser John, Kobayashi Hiroyasu, Kobayashi Tetsu, D’Alessandro-Gabazza Corina, Nakayama Sei, Nishikubo Kimiaki, Noguchi Takashi, Takei Yoshiyuki, Gabazza Esteban C
Journal: BMJ (2010)
Fatigue, However Measured, Continues to Refine Prognosis in Higher Risk MDS: An MDS-CAN Study
Authors: Irina Amitai, Michelle Geddes, Nancy Zhu, Mary-Margaret Keating, Mitchell Sabloff, Grace Christou, Brian Leber, Dina Khalaf, Heather A. Leitch, Eve St-Hilaire, Nicholas Finn, April Shamy, Karen W.L. Yee, John M. Storring, Thomas J. Nevill, Robert Delage, Mohamed Elemary, Versha Banerji, Lisa Chodirker, Lee Mozessohn, Anne Parmentier, Mohammed Siddiqui, Alexandre Mamedov, Liying Zhang, Rena J. Buckstein
Journal: Blood (2020)
Characterisation of gas exchange in COPD with dissolved-phase hyperpolarised xenon-129 MRI
Authors: Lukasz Myc, Kun Qing, Mu He, Nicholas Tustison, Zixuan Lin, Ani W Manichaikul, James Patrie, Joanne Cassani, Roselove N Nunoo-Asare, Yong Huang, Zaid Obaida, Sina Tafti, Alan M Ropp, Grady Wilson Miller, Jaime Mata, Talissa Altes, John Mugler, Y Michael Shim
Journal: Thorax (2020)

To investigate whether hyperpolarised xenon-129 MRI (HXeMRI) enables regional and physiological resolution of diffusing capacity limitations in chronic obstructive pulmonary disease (COPD), we evaluated 34 COPD subjects and 11 healthy volunteers. We report significant correlations between airflow abnormality quantified by HXeMRI and per cent predicted forced expiratory volume in 1 s; HXeMRI gas transfer capacity to red blood cells and carbon monoxide diffusion capacity (%DLCO); and HXeMRI gas transfer capacity to interstitium and per cent emphysema quantified by multidetector chest CT. We further demonstrate the capability of HXeMRI to distinguish varying pathology underlying COPD in subjects with low %DLCO and minimal emphysema.
Mechanisms of Resistance and Determinants of Response of the GPRC5D-Targeting T-Cell Redirecting Bispecific Antibody JNJ-7564 in Multiple Myeloma
Authors: Christie P.M. Verkleij, Marloes Broekmans, Amy Wong, Sonja Zweegman, Raluca Verona, Homer Adams, Tuna Mutis, Niels W.C.J. van de Donk
Journal: Blood (2020)
Abstract 548: Cellular Contributions to Neonatal Cardiac Regeneration
Authors: Adwiteeya Misra, Pearl J Quijada, Ryan Burke, Ronald Dirkx, Eric M Small
Journal: Circulation Research (2020)

While the neonatal mouse heart possesses a remarkable ability to regenerate up to a week after birth, the adult mammalian heart is susceptible to irreversible scar formation that impedes cardiac function. Such a scar is formed by the precocious deposition of extracellular matrix (ECM) by resident cardiac fibroblasts (CFs). Unlike their adult counterparts, neonatal CFs in the regenerative widow may have a unique phenotype that contributes to cardiac regeneration and scar resolution. Indeed, the neonatal cardiac ECM secreted by CFs is reported to stimulate regeneration, yet the underlying mechanisms of CF-mediated cardiac repair in the neonate has not been examined. Here, we present a strategy to establish the role of tissue resident CFs in mouse neonatal cardiac regeneration through selective cell depletion and RNA sequencing. Through the initial analysis of published RNA sequencing data, we identified an enrichment of pro-regenerative molecules such as amphiregulin (Areg) during the neonatal regenerative window. Areg, an epidermal growth factor ligand, has been shown to paradoxically stimulate both cardiac repair and pathological fibrosis after adult cardiac injury. To assess its impact on the neonatal cardiac injury response, we developed an adeno-associated virus serotype 9 with the complete coding sequence of mouse Areg (AAV9:Areg) controlled by the cardiomyocyte-specific cardiac troponin T promoter. In vivo , AAV9:Areg treated mice accumulate BrdU+ (proliferative) non-myocytes adjacent to Areg-expressing cardiomyocytes. Ongoing studies are aimed at evaluating the contribution of CFs to neonatal cardiac repair, including whether Areg-dependent cellular changes impact CFs in the neonatal regenerative window in a manner distinct from that in the adult injury response.
Type of TP53 Mutations Affects Subclonal Configuration and Selection Pressure for Acquisition of Additional Hits in Contralateral Alleles
Authors: Carmelo Gurnari, Vera Adema, Hassan Awada, Simona Pagliuca, Cassandra M Kerr, Sunisa Kongkiatkamon, Thomas Laframboise, Valeria Visconte, Maria Teresa Voso, Mikkael A. Sekeres, Hetty E. Carraway, Torsten Haferlach, Jaroslaw P. Maciejewski
Journal: Blood (2020)
Abstract 357: DNA Methylation Profiles of African American Val122Ile-Transthyretin Mutation Carriers Reveals Genes Involved in Amyloidosis Regulation
Authors: Gita Pathak, Frank Wendt, Antonella De Lillo, Yari Nunez, Aranyak Goswami, Flavio De Angelis, Maria Fuciarelli, Henry Kranzler, Joel Gelernter, Renato Polimanti
Journal: Circulation Research (2020)

The Transthyretin ( TTR ) Val122Ile mutation causes a rare life-threatening disorder attributable to amyloid deposition. This mutation is mainly present in people of African descent; carriers have an increased risk of congestive heart failure and several other non-cardiac phenotypes such as carpal tunnel syndrome, peripheral edema, and arthroplasty. Cardiac disease in Val122Ile carriers may not depend solely on this mutation and other uninvestigated factors could contribute to clinical heterogeneity. One possible mechanism is through DNA methylation, the addition of a methyl group on CG-dinucleotides, which can result in altered gene expression. We investigated methylation changes contributing to heart disease in Val122Ile carriers to identify non-TTR regulatory mechanisms. We investigated 96 Val122Ile carriers of genetically-confirmed African descent using the Illumina EPIC array, which covers 850,000 methylation sites across the genome. We found changes in five methylated sites associated with heart disease. These map to FAM129B, SKI, WDR27, GLS , and an intergenic site near RP11-550A5.2 (p=1.6 to 4.6e-8), and a methylated region containing KCNA6 and GALNT3 (p=1.1e-12). Weighted methylated sites mapped to PPI network analysis identified ABCA1 gene (p=0.001). We also found six cis-mQTLs associated with the FAM129B CpG site (p=4.1e-24 to 2.8e-14). We replicated two of the aforementioned CpG sites near RP11-550A5.2 (p=0.021) and in FAM129B (p=0.016) at nominal significance in a case-control analysis of confirmed cases of TTR amyloidosis. GLS encodes glutaminase, which catalyzes the conversion of glutamine to glutamate. Its expression is increased in amyloid-beta neurons and neurofibrillary tangles. ABCA1 regulates cholesterol transport and interacts with APOA1 and APP ; its dysregulation increases amyloid deposition. Increasing FAM129B expression improves the clearance of amyloid deposits and rescues hippocampal neurons from apoptosis. The SKI expression modulates TGF-beta , resulting in cardiac fibrosis. Of note, SKI and FAM129B together are involved in the regularion of various muscle tissues. Collectively, these findings suggest that a complex amyloid-related gene circuit could explain diverse symptoms in Val122Ile carriers.
The Anterior Cingulate Cortex Predicts Future States to Mediate Model-Based Action Selection
Authors: Thomas Akam, Ines Rodrigues-Vaz, Ivo Marcelo, Xiangyu Zhang, Michael Pereira, Rodrigo Freire Oliveira, Peter Dayan, Rui M. Costa
Journal: Neuron (2020)

Behavioral control is not unitary. It comprises parallel systems, model based and model free, that respectively generate flexible and habitual behaviors. Model-based decisions use predictions of the specific consequences of actions, but how these are implemented in the brain is poorly understood. We used calcium imaging and optogenetics in a sequential decision task for mice to show that the anterior cingulate cortex (ACC) predicts the state that actions will lead to, not simply whether they are good or bad, and monitors whether outcomes match these predictions. ACC represents the complete state space of the task, with reward signals that depend strongly on the state where reward is obtained but minimally on the preceding choice. Accordingly, ACC is necessary only for updating model-based strategies, not for basic reward-driven action reinforcement. These results reveal that ACC is a critical node in model-based control, with a specific role in predicting future states given chosen actions.
Outcomes in Patients with Hematologic Malignancies Infected with Sars-Cov-2: The Northwestern University Experience
Authors: Carlos Galvez, Imran Nizamuddin, Emily Tuchman, Peter Doukas, Neha Reddy, Jessica K. Altman, Jane N. Winter, Leo I. Gordon, Dylan Barth, Nicole Hodgins, Barbara Pro, Shuo Ma, Shira Dinner, Olga Frankfurt, Jayesh Mehta, Seema Singhal, Kehinde Adekola, Jonathan Moreira, Sajal D Tanna, Firas Wehbe, Reem Karmali
Journal: Blood (2020)
Efficacy and Safety of a Combination of Thrombopoietin Receptor Agonist with an Immunosuppressant Therapy for the Management of Multirefractory Adult ITP: Results from a Retrospective, Multicenter, Observational Study
Authors: Etienne Crickx, Guillaume Moulis, Marc Ruivard, Bouchra Asli, Jean Pierre Marolleau, Mikael Ebbo, Etienne Riviere, Sylvain Audia, Louis Terriou, Marc Michel, Bertrand Godeau, Matthieu Mahevas
Journal: Blood (2020)
Hemorrhagic Cystitis in Patients Undergoing Allogeneic Hematopoietic Cell Transplant with Post Transplant Cyclophosphamide As GvHD Prophylaxis
Authors: Dat Ngo, Stephanie Mac, Dongyun Yang, Saloomeh Mokhtari, Jason Chen, Haris Ali, Shukaib Arslan, Amandeep Salhotra, Thai Cao, Nicole Karras, Ibrahim Aldoss, Pkoller Koller, Andrew S. Artz, Ahmed Aribi, Karamjeet S. Sandhu, Chatchada Karanes, Samer Khaled, Vinod A. Pullarkat, Anthony S. Stein, Guido Marcucci, Eileen P. Smith, Stephen J. Forman, David S Snyder, Ryotaro Nakamura, Monzr M. Al Malki
Journal: Blood (2020)
Inpatient Outcomes and 60-Day Unplanned Readmissions after Hip Arthroplasty in Sickle Cell Patients with Avascular Necrosis
Authors: Prasanth Lingamaneni, Krishna Moturi, Sheeba Ba Aqeel, Shristi Upadhyay Banskota, Binav Baral, Maryam Zia
Journal: Blood (2020)
Accurate Prediction of Gene Mutations with Flow Cytometry Immune-Phenotyping By Machine Learning Algorithm
Authors: Bor-Sheng Ko, Yu-Fen Wang, Jeng-Lin Li, Hsin-An Hou, Wen-Chien Chou, Hwei-Fang Tien, Ting-Yu Chang, Chi-Chun Lee
Journal: Blood (2020)