tbuckley/cabot-search huggingface dataset:
Tubal Fluid Fertilization Achieved
Journal: JAMA (1965)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Officials of the Association 1970-1971
Journal: JAMA (1970)

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Immunologic mechanisms in multiple sclerosis. Exacerbation by type A hepatitis and skin test antigens
Authors: R. L. Owen
Journal: JAMA (1980)

A 29-year-old man with quiescent multiple sclerosis had hemiparesis during the prodrome of serologically confirmed type A hepatitis. After neurological symptoms and acute hepatitis had abated, hemiparesis again developed when skin tests were applied. While he was receiving prednisone, skin test inflammation and neurological signs cleared. One year later, lymphocyte stimulation, which had been noticeably elevated during his exacerbations, returned to low normal. Both hepatitis A and skin test antigens produced immunostimulation that resulted in exacerbation of multiple sclerosis. (<i>JAMA</i>244:2307-2309, 1980)
Diagnosis related groups in Europe: moving towards transparency, efficiency, and quality in hospitals?
Authors: R. Busse, A. Geissler, A. Aaviksoo, F. Cots, U. Hakkinen, C. Kobel, C. Mateus, Z. Or, J. O'Reilly, L. Serden, A. Street, S. S. Tan, W. Quentin
Journal: BMJ (2013)

Hospitals in most European countries are paid on the basis of diagnosis related groups. <b>Reinhard Busse and colleagues</b> find much variation within and between systems and argue that they could be improved if countries learnt from each other
Archives of Environmental Health, Chicago
Journal: JAMA (1965)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Service Costs of Caring for Adolescents With Mental Illness in a Rural Community, 1993–2000
Authors: E. Jane Costello, William Copeland, Alexander Cowell, Gordon Keeler
Journal: American Journal of Psychiatry (2007)

Objective: Costs of treating child psychiatric disorders fall on educational, primary care, juvenile justice, and social service agencies as well as on psychiatric services. The authors estimated multiagency mental health costs by integrating service unit costs with utilization rates in an 11-county area. Using psychiatric diagnoses made independently of service use records, the authors calculated costs across agencies as well as the extent of unmet need for psychiatric care. Method: Annual parent and child reports were used to measure mental health care needs and units of service across 21 types of settings for the population-based Great Smoky Mountain Study sample of 1,420 adolescents from ages 13 to 16. Unit costs for services were generated from information from service providers and records. The authors calculated costs overall, costs by type of service, and costs by diagnosis. Results: Average annual costs per adolescent treated were $3,146. Juvenile justice and inpatient/residential facilities accounted for well over half of the total costs. Costs for youths with two or more diagnoses were twice as much as costs of those with a single disorder. Among adolescents with service needs, 66.9% received no services. Public health insurance was associated with higher rates of specialty mental health care than either private insurance or no insurance. Conclusions: Annual costs across all services were three to four times greater than recent health insurance estimates alone. Many costs for adolescents with mental health problems were borne by agencies not designed primarily to provide psychiatric or psychological services. Only one in three adolescents needing psychiatric care received any mental health services.
Supplemental Oxygen and Risk of Surgical Wound Infection
Authors: Ritesh Agarwal
Journal: JAMA (2006)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Relapsing Fever
Authors: Michael D. Malison
Journal: JAMA (1979)

RELAPSING fever is an uncommonly diagnosed disease in this country (12 cases reported in 1977).<sup>1</sup>The few cases reported each year mainly occur in the western United States, and only recently has a case been documented that originated east of the Mississippi.<sup>2</sup> Two forms of relapsing fever are recognized, louse-borne (epidemic) and tick-borne (endemic). Both are caused by spirochetes of the genus<i>Borrelia</i>and have similar symptoms. The louse-borne variety requires a human reservoir and is common in many other parts of the world. The only case of louse-borne relapsing fever reported in this century in the United States was in a recent Ethiopian immigrant,<sup>3</sup>but tick-borne relapsing fever has many endemic foci in North America, largely because of the prevalence of animal reservoirs. The following two cases appeared in a two-month period at the same hospital. Both fortuitously were diagnosed by the laboratory technicians, who noticed
Approaches for Exposure Characterization and Data Needs for Hazardous Waste Site Assessment
Authors: Jed M. Waldman
Journal: Environmental Health Perspectives (1995)

This article provides an understanding of the approaches for determining exposure and dose to populations in the vicinity of hazardous waste sites.A review of the federal legislation and jurisdiction for assessments is provided, and the approaches of the U.S. Environmental Protection Agency and the Agency for Toxic Substances and Disease Registry are compared.These methods strive to aid in the evaluation of public health impacts of contaminants that were, are, or may be released to the community, and they are concerned with various aspects of the contaminant fate, human contact, and toxic response for chemicals of concern.Such approaches have been designed for generic contamination scenarios, but they aim to be applicable to a wide range of chemicals and sites in the real world.Along with any modeling framework for exposure and dose characterization, detailed information or real data are requisite for the completion of any site-specific assessment.What kinds of data are needed and where they may be found are also discussed.A comprehensive framework for exposure characterization, recently proposed by Georgopoulos and Lioy, is outlined.The framework is one employing the following elements: chemodynamic analyses of sources and receptors; characterization of the target population; toxicokinetic/toxicodynamic analyses; uncertainty/error analyses; and evaluation of the characterization performance.-Environ Health Perspect 1 03(Suppl 1 ):99-104 (1995) Key words: exposure assessment, hazardous waste sites, superfund, public health * enable evaluation of exposure trends.Their primary weaknesses are that they * do not characterize the full range of exposure or all routes of potential exposures; * provide data that are not true measures of exposure; * provide inadequate information on sensitive populations; * measure only a limited number of pollutants; * do not include pollutant mixtures; * only consider limited health end points; * offer data of inconsistent quality; * are inaccessible to users;Environmental Health Perspectives * are expensive to use and maintain; * provide information that is not current.The ideal exposure database would be able to: * provide exposures for individuals; * delineate distribution of population; * characterize most highly exposed sub- populations; * measure total exposure; * apportion information, by source; * provide longitudinal data on exposure trends; * incorporate a strong public health basis for measurements; * measure cross-media transport; * track environmental fate; * allow archiving of biological and envi- ronmental samples; * provide linkages to other databases; * offer high accessibility; * provide flexibility to increase the num- ber of pollutants measured.
PRODUCTION OF BILE PIGMENT WITHOUT INTERVENTION OF THE LIVER
Journal: JAMA (1922)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Reply to Carsten Stephan, Henning Cammann, and Klaus Jung's Letter to the Editor re: Scott A. Tomlins, John R. Day, Robert J. Lonigro, et al. Urine TMPRSS2:ERG Plus PCA3 for Individualized Prostate Cancer Risk Assessment. Eur Urol. In press. http://dx.doi.org/10.1016/j.eururo.2015.04.039
Authors: Scott A. Tomlins, Jack Groskopf, Arul M. Chinnaiyan
Journal: European Urology (2015)
Resolving repression
Authors: Steven M. Smith
Journal: Behavioral and Brain Sciences (2006)

The feuding factions of the memory wars, that is, those concerned with the validity of recovered memories versus those concerned with false memories, are unified by Erdelyi's theory of repression. Evidence shows suppression, inhibition, and retrieval blocking can have profound yet reversible effects on a memory's accessibility, and deserve as prominent a role in the recovered memory debate as evidence of false memories. Erdelyi's theory shows that both inhibitory and elaborative processes cooperate to keep unwanted memories out of consciousness.
NATIONAL PLANNING FOR CONTINUING MEDICAL EDUCATION
Journal: JAMA (1963)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Brain Death
Authors: Kristin Walter
Journal: JAMA (2020)

David M. Greer, MD, MA; Sam D. Shemie, MD; Ariane Lewis, MD; Sylvia Torrance, BSc; Panayiotis Varelas, MD; Fernando D. Goldenberg, MD; James L. Bernat, MD; Michael Souter, MBChB; Mehmet Akif Topcuoglu, MD; Anne W. Alexandrov, PhD; Marie Baldisseri, MD, MPH; Thomas Bleck, MD; Giuseppe Citerio, MD; Rosanne Dawson, LLB; Arnold Hoppe, MD; Stephen Jacobe, MD, MHL; Alex Manara, MBBCh; Thomas A. Nakagawa, MD; Thaddeus Mason Pope, JD, PhD; William Silvester, MD; David Thomson, MD; Hussain Al Rahma, MD; Rafael Badenes, MD, PhD; Andrew J. Baker, MD; Vladimir Cerny, MD, PhD; Cherylee Chang, MD; Tiffany R. Chang, MD; Elena Gnedovskaya, MD, PhD; Moon-Ku Han, MD; Stephen Honeybul, MD; Edgar Jimenez, MD; Yasuhiro Kuroda, MD, PhD; Gang Liu, MD, PhD; Uzzwal Kumar Mallick, MD; Victoria Marquevich, MD; Jorge Mejia-Mantilla, MD, MSc; Michael Piradov, MD, PhD; Sarah Quayyum, JD, LLM; Gentle Sunder Shrestha, MD; Ying-ying Su, MD, PhD; Shelly D. Timmons, MD; Jeanne Teitelbaum, MD; Walter Videtta, MD; Kapil Zirpe, MD; Gene Sung, MD, MPH
Author Index Vol. 65, 1996
Authors: Giovanni A. Fava, Antonella Montanari, Clare Bradley, P. Bech, C. Gudex, Fuad Lechin, Bertha van der Dijs, Marcel E. Lechin, Fuad Lechin, Bertha van der Dijs, Marcel E. Lechin, Fuad Lechin, Bertha van der Dijs, Marcel E. Lechin, B. Buddeberg-Fischer, R. Bernet, J. Schmid, C. Buddeberg, Maido Uusküla, K. Staehr Johansen
Journal: Psychotherapy and Psychosomatics (1996)
THÉ CARE AND FEEDING OF INFANTS
Journal: JAMA (1923)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Section 35.—Tuberculous Arthritis
Journal: JAMA (1973)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Abstract 19282: Transient Bone Morphogenic Protein Antagonism Directs Differentiation of Ipscs Into the Cardiac Neural Crest and Ckit+ Myocardial Progenitor Lineages
Authors: Konstantinos E Hatzistergos, Lauro M Takeuchi, Dieter Saur, Barbara Seidler, Susan M Dymecki, Jia Jia Mai, Rosemeier K Takeuchi, Wayne Balkan, Joshua M Hare
Journal: Circulation (2015)

Introduction: The signaling pathways that govern cKit+ cardiac progenitor cell (CPC) differentiation into cardiomyocytes (CMs) are unknown. Some studies suggest an essential role in cardiomyogenesis, others suggest a minimal CPC contribution. We studied if it is a non-permissive cardiac milieu that minimizes the generation of CMs from CPCs. Hypothesis: Transient BMP antagonism directs the generation of cardiomyocytes from CPCs. Methods: We lineage-traced CPCs using a novel dual-recombinase responsive indicator mice (cKitCreERT2;Wnt1::Flpe;RC::Fela) and iPSCs derived from cKitCreERT2;IRG (iPSCKit) mice. Results: Intersectional genetic fate-mapping of cKitCreERT2;Wnt1:: Flpe;RC::Fela embryos supported that cKit marks Wnt1-expressing cardiac neural crest (CNC) progenitors, emerging at ~E9.5 and contributing a limited number of cardiomyocytes (n=3). We lineage-traced CPCs during stage-specific cardiogenic differentiation of iPSCKit. Ascorbate treatment promoted differentiation of iPSCKit-derived embryoid bodies (EBs) into Nkx2.5+ myocardium, 45.5%±6.7% of which co-expressed the Cre-reporter EGFP (n=154 EBs; 12 preps), suggesting that CPCs encompass fully competent cardiomyogenic progenitors. Noggin (or Dorsomorphin), a BMP antagonist transiently expressed in the heart at E7.5-E8.5 but not during CNC invasion, directed the differentiation of iPSCkit-EBs into Mesp1+/Isl1+/Nkx2.5+ cardiac mesoderm progenitors (p≤0.0001). The same signaling pathway subsequently directed EBs into the cKit+/Wnt1+/Pax3+/Mitf-H+/Isl1+/Nkx2.5+ CNC lineage (p≤0.0001), while suppressing the generation of WT1+/Tbx18+ epicardium (p&lt;0.05). Stage-specific induction of Cre-recombination delineated that iPSCkit-derived CPCs encompass Mesp1–/cKit+/Nkx2.5+ CNC progenitors, which contributed EGFP+ CNC derivatives, including Nkx2-5+ cardiomyocytes, to 60.7%±7.3% of spontaneously beating EBs (n=147 EBs; 12 preps). Conclusions: Our data show that CPCkit are fully competent CNC-derived cardiomyogenic progenitors, whose differentiation to cardiomyocytes is minimized by a latent Noggin-mediated signaling pathway. Therapeutically exploiting CPCkit, provides an important strategy for maximizing myocardial regeneration.
Vanishing Vertebra
Authors: LAURENCE MABILLE, NATHALIE BERENGER, JEAN-DENIS LAREDO, DANIEL KUNTZ, OLIVIER MUNDLER
Journal: Clinical Nuclear Medicine (1997)

The authors describe a patient who had Gorham-Stout syndrome involving the lumbar spine. This syndrome, which also is called vanishing bone disease or idiopathic massive osteolysis, is a rare and generally unifocal disorder, seen in young adults (about 140 patients reported). Involvement of the lumbar spine is not frequent (3 patients reported) and has a worse prognosis because of late neurological complications. The pathogenesis is incompletely known. The diagnosis is made with biopsy (bony lymphangiomatous changes on microscopy). The clinical evolution is unpredictable. Complications depend on the affected bones (neurological or pulmonary).