tbuckley/cabot-search huggingface dataset:
Tumor cell-activated “Sustainable ROS Generator” with homogeneous intratumoral distribution property for improved anti-tumor therapy
Authors: Junjie Liu, Xiu Zhao, Weimin Nie, Yue Yang, Chengcheng Wu, Wei Liu, Kaixiang Zhang, Zhenzhong Zhang, Jinjin Shi
Journal: Theranostics (2020)

Photodynamic therapy (PDT) holds a number of advantages for tumor therapy. However, its therapeutic efficiency is limited by non-sustainable reactive oxygen species (ROS) generation and heterogeneous distribution of photosensitizer (PS) in tumor. Herein, a "Sustainable ROS Generator" (SRG) is developed for efficient antitumor therapy. Methods: SRG was prepared by encapsulating small-sized Mn3O4-Ce6 nanoparticles (MC) into dendritic mesoporous silica nanoparticles (DMSNs) and then enveloped with hyaluronic acid (HA). Due to the high concentration of HAase in tumor tissue, the small-sized MC could be released from DMSNs and homogeneously distributed in whole tumor. Then, the released MC would be uptaken by tumor cells and degraded by high levels of intracellular glutathione (GSH), disrupting intracellular redox homeostasis. More importantly, the released Ce6 could efficiently generate singlet oxygen (1O2) under laser irradiation until the tissue oxygen was exhausted, and the manganese ion (Mn2+) generated by degraded MC would then convert the low toxic by-product (H2O2) of PDT to the most harmful ROS (·OH) for sustainable and recyclable ROS generation. Results: MC could be homogeneously distributed in whole tumor and significantly reduced the level of intracellular GSH. At 2 h after PDT, obvious intracellular ROS production was still observed. Moreover, during oxygen recovery in tumor tissue, ·OH could be continuously produced, and the nanosystem could induce 82% of cell death comparing with 30% of cell death induced by free Ce6. For in vivo PDT, SRG achieved a complete inhibition on tumor growth. Conclusion: Based on these findings, we conclude that the designed SRG could induce sustainable ROS generation, homogeneous intratumoral distribution and intracellular redox homeostasis disruption, presenting an efficient strategy for enhanced ROS-mediated anti-tumor therapy.
Jo Ann McLaughlin
Authors: Ian Clegg, Lesley Hewson, Anne Taylor
Journal: BMJ (2020)

Jo Ann McLaughlin was born in Rainhill, Merseyside, to Millie and Jim, an American. The family moved out to the USA when Jo was 2 years old; they lived in Rhode Island and Washington. With her mother unable to settle, Jo and her younger sister, Sue, returned to Widnes when Jo was 5. Joined by their father a year later, the family moved to Surrey and three years later to Swindon. When Jo was 12, they all moved back to Merseyside, where, to her delight, Jo’s youngest sister, Jayne, was born. Jo had found the many school moves hard, but she was now able to enjoy …
Abstract 317: Endoplasmic Reticulum Stress Alters the Localization of the Prolyl Hydroxylase Ogfod1 in Adult Mouse Cardiomyocytes
Authors: Michael Harris, Leslie Kennedy, Junhui Sun, Matthew Cockman, Peter Ratcliffe, Elizabeth Murphy
Journal: Circulation Research (2020)

Prolyl hydroxylation is a post-translational modification that is known to regulate several key cell functions including translation and protein stability. Enzymes that catalyze prolyl hydroxylation belong to the 2-oxoglutarate- and iron-dependent oxygenase (2OGDO) family. A newly-identified member of the 2OGDO enzyme family, 2-oxoglutarate- and iron-dependent oxygenase domain-containing protein 1 (Ogfod1), catalyzes prolyl hydroxylation of ribosomal protein s23 (Rps23), a component of the 40S ribosome. To establish an in vivo role for Ogfod1, we ablated Ogfod1 in mice and subjected isolated perfused hearts to ischemia and reperfusion and found Ogfod1 ablation to be cardioprotective. The mechanisms by which these changes occur are currently unknown, so we investigated Ogfod1 regulation and mechanisms of cardioprotection. Previous work has shown that Ogfod1-mediated Rps23 prolyl hydroxylation occurs in the nucleus, where ribosomes are assembled. Additionally, Ogfod1 localizes to cytoplasmic stress granules in transformed cells exposed to endoplasmic reticulum (ER) stress. Based on these studies, we tested the hypothesis that Ogfod1 localization, and subsequently its function, change in response to stress. We induced ER stress by treating isolated adult cardiomyocytes with thapsigargin, and monitored Ogfod1 localization using immunofluorescence. Thapsigargin inhibits sarco/endoplasmic reticulum Ca 2+ ATPase (SERCA) which activates the unfolded protein response. In the absence of thapsigargin, Ogfod1 localized to the nucleus, peri-nuclear space, cytoplasm, and cell membrane. After stress induction, Ogfod1 nuclear- and perinuclear-localization decreased. This suggests that in murine adult cardiomyocytes subjected to ER stress, Ogfod1 is exported from the nucleus, potentially as a mechanism for down-regulating its activity. As Ogfod1 ablation has been found to be cardioprotective in mice, understanding Ogfod1 localization and regulation in the cardiomyocyte stress response may provide mechanistic insight that will be useful in developing treatments for stressors such as heart failure.
Rituximab and Cyclophosphamide Based Regimens for Treatment of Waldensterom Macroglobulemia, Lessons from Clinical Literature: A Systemic Review
Authors: Qasim Khurshid, Abdul Jabbar Dar, Muhammad Ali Mirza, Muhaddis Ejaz Ahmad, Sobia Aamir, Richi Kashyap, Syed Maaz Abdullah, Muhammad Saad Farooqi, Israr Khan, Arshia Akbar, Waqas Khan, Muhammad Tayyeb, Sundas Ali, Saad Javed, Fazeel Qamar, Faiz Anwer
Journal: Blood (2020)
Outcomes in COVID-19 Patients on Treatment Dose Anti-Coagulation Compared to Prophylactic Dose Anti-Coagulation
Authors: Rich Kodama, Amardeep Kalsi, Angelica Singh, Nivetha Vishnuvardhan, Neil Nimkar, Sonal Paul, Soeb Usta, Farah Ashraf, Anita Mazloom, Abeer Ashfaq, Nisha Patel, Chathula Kumarasinghe, Uqba Khan, Peter Gregos, Jeremy Weingarten, Eshan Patel
Journal: Blood (2020)
Warranty Period of a Calcium Score of Zero
Authors: Omar Dzaye, Zeina A. Dardari, Miguel Cainzos-Achirica, Ron Blankstein, Arthur S. Agatston, Matthias Duebgen, Joseph Yeboah, Moyses Szklo, Matthew J. Budoff, Joao A.C. Lima, Roger S. Blumenthal, Khurram Nasir, Michael J. Blaha
Journal: JACC. Cardiovascular imaging (2020)
Efficacy and Cost Analysis of Eltrombopag in Thrombocytopenia and Poor Graft Function Post Allogeneic Hematopoietic Cell Transplantation in a Canadian Centre - a Prospective Observational Study
Authors: RAM V Nampoothiri, Lina Ho, Cassandra McEwan, Ivan Pasic, Zeyad Al-Shaibani, Wilson Lam, Arjun Law, Fotios Michelis, Dennis Dong Hwan Kim, Armin Gerbitz, Auro Viswabandya, Jeffrey H. Lipton, Jonas Mattsson, Rajat Kumar
Journal: Blood (2020)
Acute Airway Obstruction in 4 Pediatric Patients with Congenital Plasminogen Deficiency (C-PLGD) Treated with Intravenous Plasminogen (Human) Replacement Therapy Under an Expanded Access Protocol
Authors: Charles Nakar, Neelam Thukral, Heather L. McDaniel, Joseph M Parker, Diane Trybul, Amy D Shapiro
Journal: Blood (2020)
The Prognostic Power of Gene Expression Profiling with Cytogentics and Routinely Acquired Serum Markers: SKY92 Combined with Revised ISS
Authors: Rowan Kuiper, Mark van Duin, Martin H Van Vliet, Erik H Van Beers, Berna Berna Beverloo, Sonja Zweegman, Tom Cupedo, Annemiek Broijl, Pieter Sonneveld
Journal: Blood (2020)
The modern view on the role of glyprolines by metabolic syndrome
Authors: Nikolai F. Myasoedov, Lyudmila A. Lyapina, Lyudmila A. Andreeva, Marina E. Grigorieva, Tamara Y. Obergan, Tatiana A. Shubina
Journal: Medicinal Research Reviews (2020)

Abstract The analysis of the literature data and own experimental studies on the effect of glyproline peptides on fat, carbohydrate metabolism, and hemostasis system when modeling metabolic syndrome in animals (rats) was carried out. Violations of fat and carbohydrate metabolism are characterized by hypercoagulation, increased glucose levels, dyslipidemia, and decreased rheological properties of blood. In this condition, the arginine‐ and lysine‐containing glyprolines (PGP, PRP, RPGP, KKRRPGP, RKKRPGP, and KRRKPGP) at multiple (7 days) intranasal introduction had a complex effect on the hemostatic parameters, increasing antiplatelet, anticoagulant, and fibrinolytic activity of blood plasma. All the studied drugs also showed normoglycemic and normolipidemic effects and led to a slowdown or decrease in body weight growth. The analysis of the presented material allows us to speak about the prospects of using hit compounds as protective therapeutic agents. In the case of violations of fat and carbohydrate metabolism, regulatory glyprolines protect the body by displaying antithrombotic, hypolipidemic, and hypoglycemic properties.
Evolutionary Action Score Identifies a Subset of TP53 Mutated Myelodysplastic Syndrome with Favorable Prognosis
Authors: Rashmi Kanagal-Shamanna, Guillermo Montalban Bravo, Panagiotis Katsonis, Koji Sasaki, Caleb Class, Christopher B. Benton, Elias Jabbour, Kelly S. Chien, Rajyalakshmi Luthra, Carlos E. Bueso-Ramos, Tapan M. Kadia, Michael Andreeff, Nicholas J. Short, Naval Daver, Mark Routbort, Joseph D. Khoury, Keyur Patel, Irene Ganan-Gomez, Yue Wei, Gautam Borthakur, Farhad Ravandi, Kim-Anh Do, Kelly A. Soltysiak, Olivier Lichtarge, L. Jeffrey Medeiros, Hagop M. Kantarjian, Guillermo Garcia-Manero
Journal: Blood (2020)
Light Chain Deposition Disease: First Analysis of an International Study in 359 Patients
Authors: Paolo Milani, Nelson Leung, Efstathios Kastritis, Stefan Schönland, Ute Hegenbart, Frank Bridoux, Florent Joly, Sascha A Tuchman, Victor Jimenez-Zepeda, Sriram Ravichandran, Holly Lee, Tamara Berno, Giampaolo Merlini, Giovanni Palladini, Ashutosh D. Wechalekar
Journal: Blood (2020)
<i>ATRA Augments BCMA Expression on Myeloma Cells and Enhances Recognition By BCMA-CAR T-Cells</i>
Authors: Estefania Garcia-Guerrero, Luis Gerardo Rodríguez-Lobato, Sophia Danhof, Belén Sierro-Martínez, Ralph Goetz, Markus Sauer, Jose A. Perez-Simon, Hermann Einsele, Michael Hudecek, Sabrina Prommersbe
Journal: Blood (2020)
A National Collaborative Multicenter Phase II Study for Potential Safety Efficacy of Convalescent Plasma to Treat Severe COVID-19 Patients
Authors: Hani Al Hashmi, Deia Alawami, Syed Ziauddin A Zaidi, Nawal F Alshehry, Hazza A. Alzahrani, Abdul Rehman Z Zaidi, Ahmed Al Sagheir, Osamah Khojah, Ahmed Alaskar, Noura AlMozain, Rehab Al-Ansari, Mona Alfaraj, Ghada ElGohary, Afra Dayel, Ahmed Bahrani, Arwa Nabhan Abdulhamid, Hind Alhumaidan, Mohammed Albalawi, Alia AlFraedhi, Jawaher Al-Otaibi, Rania Abdallah
Journal: Blood (2020)
Abstract 550: FOG2 Modulates a TBX5-Dependent Gene Regulatory Network for Cardiac Rhythm Homeostasis
Authors: Sonja Lazarevic, Michael Broman, Rangarajan Nadadur, Jeff Steimle, Brigitte Laforest, Carlos Perez-Cervantes, Stefan R Mazurek, Kaitlyn M Shen, Margaret Gadek, Kohta Ikegami, Xinan Yang, Christopher Weber, Eric Svensson, Ivan P Moskowitz
Journal: Circulation Research (2020)

Atrial fibrillation (AF) is the most common cardiac arrhythmia. Recent work has implicated numerous transcription factors in AF risk, including ZFPM2 (FOG2), GATA4, and TBX5. FOG2 is overexpressed in human heart failure, a major AF risk factor. We found that genetic inducible physiologic overexpression of FOG2 in the adult mouse heart caused spontaneous atrial fibrillation. Single cell electrophysiology revealed action potential prolongation and inappropriate depolarizations in FOG2-OE atrial cardiomyocytes. RNA-seq of the FOG2-OE left atrium prior to AF onset demonstrated that FOG2 suppresses a network of calcium cycling genes, providing a possible mechanism for disrupted cardiomyocyte electrophysiology. FOG2 ChIP-seq demonstrated genomic localization only at locations co-occupied by GATA4. Unexpectedly, we observed little overlap between genes activated by GATA4 and repressed by FOG2. Instead, we observed highly significant overlap between genes repressed by FOG2 and activated by TBX5. To identify the FOG2-dependent atrial gene regulatory network, we performed differential deep sequencing of ncRNAs. Comparison of atrial FOG2-dependent non-coding transcripts with previously performed TBX5-dependent ncRNA profiling indicated that TBX5 activated and FOG2 repressed a shared atrial rhythm gene regulatory network. Integration of FOG2, GATA4, and TBX5 ChIP-seq revealed that FOG2 only affected ncRNA transcription, indicative of enhancer activity changes, at locations co-occupied by TBX5. TBX5-dependent activation of specific regulatory elements for calcium handling genes was abolished by FOG2 in vitro . The genomic TBX5/FOG2 interaction predicted a genetic interaction in-vivo , in which cardiac conduction and arrhythmia abnormalities caused by Tbx5 haploinsufficiency were rescued by FOG2 haploinsufficiency. Non-coding RNA profiling thereby predicted a novel functional TF interaction between FOG2 and TBX5. This work reveals a specific genomic model of atrial rhythm control in which FOG2 is recruited to GATA4 and TBX5 bound locations to modulate a TBX5-dependent atrial gene regulatory network for calcium handling and cardiac rhythm homeostasis.
Abstract 232: PDE5A Inhibition in Burn-induced Cardiac Dysfunction
Authors: Jake J Wen, Ravi S Radhakrishnan
Journal: Circulation Research (2020)

Severe burn injury initiates a feedback cycle of inflammation, fibrosis and oxidative stress and others that may contribute to heart dysfunction via the PDE5A-cGMP-PKG pathway. Sildenafil (SIL) has been shown decrease immune adverse events in multiple cardiac diseases by inhibition of PDE5A. In this study, using Nano LC MS/MS strategy to identify differentially expressed proteins, we will determined the pathological importance of PDE5A-dependent injury in burn-related heart dysfunction using our well-established rat model with 4 groups: sham; sham/SIL (sham with sildenafil injection); 24 hpb (60% total body surface area scald burn and harvested at 24 hours post burn) and 24 hpb/SIL (24 hpb with SIL immediately after burn). We performed proteomics, Vevo 2100 ECG, qPCR, histology and ELISA to test our hypothesis. Our results show Sildenafil 1) inhibited burn-induced PDE5A mRNA, 2) increased cGMP and PKG activity leading to recover of PKG down-regulated genes (IRAG, PLB, RGS2, RhoA and MYTP), 3) decreased the ROS level (H2O2), oxidatively modified adducts (malonyldialdehyde (MDA), carbonyls), fibrotic gene expression (ANP, BNP, COL1A2, COL3A2, aSMA and αsk-Actin), inflammation-related gene expression (RELA, IL-18 and TGF-β) from burned rats (BRs) and 4) preserved Left-ventricular heart function (CO, EF, SV, LVvol at systolic, LVPW at diastolic and FS) and restored the oxidant/antioxidant balance (total antioxidant, total SOD activity and Cu,ZnSOD activity) in BRs. We demonstrate that the PDE5A-cGMP-PKG pathway mediates burn-induced heart dysfunction and will provide the theoretic foundation to guide pre-clinical translational study of Sildenafil in burned patients.
Evaluation of a Deep Learning–Derived Quantitative Retinopathy of Prematurity Severity Scale
Authors: J. Peter Campbell, Sang Jin Kim, James M. Brown, Susan Ostmo, R. V. Paul Chan, Jayashree Kalpathy-Cramer, Michael F. Chiang, Michael F. Chiang, Susan Ostmo, Sang Jin Kim, Kemal Sonmez, Robert Schelonka, J. Peter Campbell, R.V. Paul Chan, Karyn Jonas, Jason Horowitz, Osode Coki, Cheryl-Ann Eccles, Leora Sarna, Anton Orlin, Audina Berrocal, Catherin Negron, Kimberly Denser, Kristi Cumming, Tammy Osentoski, Tammy Check, Mary Zajechowski, Thomas Lee, Aaron Nagiel, Evan Kruger, Kathryn McGovern, Charles Simmons, Raghu Murthy, Sharon Galvis, None Jerome Rotter MD, Ida Chen, Xiaohui Li, Kent Taylor, Kaye Roll, Jayashree Kalpathy-Cramer, Deniz Erdogmus, Stratis Ioannidis, Maria Ana Martinez-Castellanos, Samantha Salinas-Longoria, Rafael Romero, Andrea Arriola, Francisco Olguin-Manriquez, Miroslava Meraz-Gutierrez, Carlos M. Dulanto-Reinoso, Cristina Montero-Mendoza
Journal: Ophthalmology (2020)
Use of Convalescent Plasma Therapy in Severe Coronavirus Disease 2019: The Yale-New Haven Health System Experience
Authors: Sabrina L. Browning, Michael Gormally, Neima Briggs, Muyi Li, Alan Zakko, Maudry Laurent-Rolle, Joseph Ladines-Lim, Alyssa Morrison, Prakash Kandel, Mohammad Khan, Gavin X. McLeod, Gregory K. Buller, Kent A. Owusu, Miriam M. Treggiari, Jeanne E. Hendrickson, Christopher A. Tormey, Mahalia S. Desruisseaux
Journal: Blood (2020)
FLT3 Inhibitors Induce Instability of p53 By Mir-181 Mediated Downregulation of Deubiquitinase YOD1 in Acute Myeloid Leukemia
Authors: Han Zhong Pei, Yao Guo, Bo Lu, Zhiguang Chang, Dengyang Zhang, Liuting Yu, Yuming Zhao, Suk-Hwan Baek, Yun Chen, Zhizhuang Joe Zhao
Journal: Blood (2020)
Transfer Learning for Identifying Morphological Heterogeneity of Neutrophils Nuclei in Hematological Diseases Based on Nuclei Semantic Segmentations of Bone Marrow Smear
Authors: Hui Shao, Wen Gao, Qi Zhang, Jun Li, Di Zhou, Lintao Bi, Yuansong BAI, Zhangzhen Shi
Journal: Blood (2020)
Concomitant Occurrence of Polyclonal Hematopoiesis and Cell-Autonomous Megakaryopoiesis in Triple-Negative Essential Thrombocythemia
Authors: Tadaaki Inano, Marito Araki, Soji Morishita, Misa Imai, Yoshihiko Kihara, Maho Okuda, Masafumi Ito, Satoshi Osaga, Yinjie Yang, Yoko Edahiro, Tomonori Ochiai, Kyohei Misawa, Yasutaka Fukuda, Akimichi Ohsaka, Norio Komatsu
Journal: Blood (2020)
A Phase 1 Study of the Combination of MG4101, <i>Ex Vivo</i>-Expanded Allogeneic NK Cells and Rituximab for Relapsed or Refractory Non-Hodgkin Lymphoma
Authors: Dok Hyun Yoon, Youngil Koh, Heesook Park, Yu kyeong Hwang, Won Seog Kim
Journal: Blood (2020)
Safety and Efficacy of Blinatumomab As Monotherapy Vs Combination Therapy in the Treatment of Relapsed/Refractory Adult ALL: A Systematic Review
Authors: Usman Ali Akbar, Anam Khan, Muhammad Ashar Ali, Atif Irfan Khan, Sobia Aamir, Muhammad Talha Liaquat, Saad Ur Rahman, Anum Waqar, Muhammad Ammar Yaser, Faiz Anwer
Journal: Blood (2020)
Repetitive Elements Trigger RIG-I-like Receptor Signaling that Regulates the Emergence of Hematopoietic Stem and Progenitor Cells
Authors: Stylianos Lefkopoulos, Aikaterini Polyzou, Marta Derecka, Veronica Bergo, Thomas Clapes, Pierre Cauchy, Carolina Jerez-Longres, Megumi Onishi-Seebacher, Na Yin, Natalia-Adriana Martagon-Calderón, Kathryn S. Potts, Lhéanna Klaeylé, Feng Liu, Teresa V. Bowman, Thomas Jenuwein, Maria Caterina Mione, Eirini Trompouki
Journal: Immunity (2020)
An Experimental Test of Adaptive Introgression in Locally Adapted Populations of Splash Pool Copepods
Authors: Joanna S Griffiths, Yasmeen Kawji, Morgan W Kelly
Journal: Molecular Biology and Evolution (2020)

As species struggle to keep pace with the rapidly warming climate, adaptive introgression of beneficial alleles from closely related species or populations provides a possible avenue for rapid adaptation. We investigate the potential for adaptive introgression in the copepod, Tigriopus californicus, by hybridizing two populations with divergent heat tolerance limits. We subjected hybrids to strong heat selection for 15 generations followed by whole-genome resequencing. Utilizing a hybridize evolve and resequence (HER) technique, we can identify loci responding to heat selection via a change in allele frequency. We successfully increased the heat tolerance (measured as LT50) in selected lines, which was coupled with higher frequencies of alleles from the southern (heat tolerant) population. These repeatable changes in allele frequencies occurred on all 12 chromosomes across all independent selected lines, providing evidence that heat tolerance is polygenic. These loci contained genes with lower protein-coding sequence divergence than the genome-wide average, indicating that these loci are highly conserved between the two populations. In addition, these loci were enriched in genes that changed expression patterns between selected and control lines in response to a nonlethal heat shock. Therefore, we hypothesize that the mechanism of heat tolerance divergence is explained by differential gene expression of highly conserved genes. The HER approach offers a unique solution to identifying genetic variants contributing to polygenic traits, especially variants that might be missed through other population genomic approaches.
Prevalence of Genetic Abnormalities in Patients with Multiple Myeloma and Its Clinical Relevance in a Developing Country like India
Authors: Aditi Shah, Nataraj KS, Sundareshan T S, Shilpa Prabhu, Bharath RAM S, Hamza Yusuf Dalal, Shiva Kumar Komaravelli, Sharat Damodar
Journal: Blood (2020)
NUTM1-Rearranged Infant and Pediatric B Cell Precursor Acute Lymphoblastic Leukemia: A Good Prognostic Subtype Identified in a Collaborative International Study
Authors: Judith M. Boer, Maria Grazia Valsecchi, Femke M. Hormann, Zeljko Antic, Marketa Zaliova, Claire Schwab, Giovanni Cazzaniga, Chloé Arfeuille, Hélène Cavé, Andishe Attarbaschi, Sabine Strehl, Gabriele Escherich, Toshihiko Imamura, Kentaro Ohki, Tanja A. Gruber, Rosemary Sutton, Agata Pastorczak, Tim Lammens, Frederic Lambert, Chi Kong Li, Enrique Carrillo de Santa Pau, Steve Hoffmann, Anja Möricke, Christine J. Harrison, Monique L. Den Boer, Paola De Lorenzo, Ronald W. Stam, Anke K. Bergmann, Rob Pieters
Journal: Blood (2020)
Pembrolizumab with R-CHOP in Previously Untreated Diffuse Large B-Cell Lymphoma: Long Term Follow up and Analysis of the Mechanism of Pdl-1 Tumor Expression
Authors: Stephen D. Smith, Jonathan R. Fromm, Min Fang, Brian G. Till, Mazyar Shadman, Ryan C. Lynch, Andrew J. Cowan, Qian Vicky Wu, Jenna Voutsinas, Heather A. Rasmussen, Katie Blue, Chaitra S. Ujjani, Andrei R Shustov, Ryan D. Cassaday, Ajay K. Gopal
Journal: Blood (2020)
Venetoclax Re-Treatment of Chronic Lymphocytic Leukemia (CLL) Patients after a Previous Venetoclax-Based Regimen
Authors: Meghan C. Thompson, John N. Allan, Kavita Sail, Beenish S. Manzoor, Jeffrey J. Pu, Paul M. Barr, Catherine C. Coombs, Stephen J. Schuster, Alan Skarbnik, Joanna M Rhodes, Jacqueline C. Barrientos, Lindsey E Roeker, Lori A. Leslie, Manali Kamdar, Michael Y. Choi, Martin Simkovic, Frederick Lansigan, Brittany Jane Hale, Andrew D Zelenetz, Alison J. Moskowitz, Kurt S. Bantilan, Celina J. Komari, Andre H. Goy, Tatyana A. Feldman, Richard R. Furman, Anthony R. Mato
Journal: Blood (2020)