tbuckley/cabot-search huggingface dataset:
Adapting to the Effects of Climate Change in the Practice of Dermatology—A Call to Action
Authors: Sarah J. Coates, Timothy H. McCalmont, Mary L. Williams
Journal: JAMA Dermatology (2019)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA Dermatology HomeNew OnlineCurrent IssueFor Authors Podcast Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA Dermatology journal
Abstract 557: Exosome Biogenesis/Uptake Mediates Cardioprotection by Globular Adiponectin but Not High Molecular Weight Adiponectin
Authors: Yajing Wang, Lu Gan, Dina Xie, Dajun Zhao, Erhe Gao, Walter J. Koch, Xinliang Ma
Journal: Circulation Research (2018)

Objectives: Adiponectin, a multifunction adipokine with established cardioprotective effect, presents in high molecular weight (HMW) and globular isotype (gAPN). Recent study demonstrated that HMW cardioprotection depends on T-Cadherin (TCad). However, whether TCad also mediates the cardioprotective effects of gAPN, an isotype with much greater biological activity and wildly utilized in metabolic/cardiovascular investigations, remains unclear. Methods and results: Adult male WT, AdipoR1KO and TCadKO mice were subjected to MI, treated with vehicle or gAPN, and followed for 4 weeks. Knockout of AdipoR1, but not Tcad, abolished the cardioprotective effects of gAPN observed in WT mice, indicating that AdipoR1 but not Tcad mediates gAPN cardioprotection. Unexpectedly, we observed that gAPN cardioprotection was significantly attenuated when animals were pre-treated with GW4869, suggesting exosome involvement. To further delineate whether gAPN regulates exosome biogenesis and/or uptake and which stage is mediated by AdipoR1, cardiomyocytes were isolated from adult AdipoR1KO and TCadKO mice. Effects of gAPN and HMW upon exosome generation and uptake were evaluated. Surprisingly, cardiomyocyte exosome production was dramatically increased (8-fold) when cells were treated with gAPN but not with HMW (at 5-fold concentration of gAPN). Moreover, gAPN increases cardiomyocyte exosome production in a receptor-independent fashion and is achieved by regulating ESCRT-multivesicular body (MVB) pathway. However, gAPN stimulated exosome uptake and cellular protection observed in WT cardiomyocytes was abolished in AdipoR1KO-derived but no TCadKO-derived cardiomyocytes. Conclusions: These data demonstrated that gAPN and HMW cardioprotection exhibits different receptor dependence and is mediated by distinct mechanisms. HMW protects heart via binding with TCad. However, gAPN has a unique ability, promoting exosome biogenesis in a receptor-independent fashion and stimulating exosome uptake and cardioprotection in an AdipoR1-dependent manner. These results indicate that gAPN is superior to HMW in concerning cardioprotection against ischemia heart injury, and is a promising therapeutic strategy in patients with coronary artery disease.
Abstract 423: Cardiomyocyte-Specific Epidermal Growth Factor Receptor Deletion Differentially Alters Cardiac Contractility and Remodeling During Physiologic versus Pathologic Development
Authors: Shuchi Guo, Jonathan Lambert, Jianliang Song, Rhonda Carter, Ana Maria Lucchese, Erhe Gao, Joseph Cheung, John Elrod, Douglas Tilley
Journal: Circulation Research (2018)

Epidermal Growth Factor Receptor (EGFR), a member of the Erbb receptor tyrosine kinase family, is essential to the development of multiple tissue and organs. Due to the embryonic lethality of global EGFR deletion, the impact of EGFR signaling in the adult heart, normally or in response to pathological stimuli, has not been well-explored. Using recently attained mice with floxed EGFR alleles crossed with αMHC-Cre mice, we have generated a cardiomyocyte-specific constitutive EGFR knockout mouse model (CM-EGFR KO) to address its role in the heart. Compared to their wild-type (WT) littermate controls, CM-EGFR-KO mice displayed age-related development of cardiac dysfunction and remodeling, occurring between 7 and 9 weeks of age, as monitored via echocardiography and immunohistochemistry analyses. Although contractile responsiveness to β-adrenergic receptor stimulation was unaffected by EGFR deletion, RNASeq analysis of CM-EGFR-KO hearts within this timeframe revealed alterations in myofilament protein expression profiles. Additionally, there was enhanced cardiomyocyte death in the 7-9 week window of development, which persisted to 10 months of age, at which time the CM-EGFR KO mice had progressively worsened cardiac function, dilation and cardiomyocyte loss. To investigate the impact of cardiomyocyte-specific EGFR deletion on the response to pathologic stress, we challenged 12-14 week old WT and CM-EGFR KO mice with chronic isoproterenol infusion. Notably, while chronic isoproterenol treatment diminished cardiac function and induced hypertrophic remodeling in WT mouse hearts, CM-EGFR-KO mouse hearts displayed progressively improved contractility and ablated hypertrophy over time, suggesting that EGFR differentially regulates cardiac function and remodeling processes during physiological versus pathological development.
IN HOSPITAL OUTCOMES OF TRANSCATHETER AORTIC VALVE REPLACEMENT COMPARED WITH SURGICAL VALVE REPLACEMENT IN PATIENTS WITH CANCER: A NATIONAL INPATIENT SAMPLE DATABASE STUDY
Authors: Ezequiel Munoz, Xavier Andrade, Juan Del Cid, Jose Sleiman, Andres Mendez, Miguel Salazar, Christian Torres, Alok Uprety, Vatsala Katiyar, Cezar Iliescu, Tochi Okwuosa, John H. Stroger
Journal: Journal of the American College of Cardiology (2019)
Bubble-Wrapping Halloween—Reply
Authors: John A. Staples, Candace Yip, Donald A. Redelmeier
Journal: JAMA Pediatrics (2019)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA Pediatrics HomeNew OnlineCurrent IssueFor Authors Podcast Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA Pediatrics journal
Letter to the Editor: Characterization of CD169+ Macrophages in Liver Regeneration: Tissue Distribution and Fate?
Authors: Yeni Ait‐Ahmed, Adrien Guillot, Fouad Lafdil
Journal: Hepatology (2019)

Potential conflict of interest: Nothing to report. To the Editor: We read this article by Behnke et al. recently published in Hepatology with great interest.1 This study demonstrated the role of B cells in favoring liver regeneration, notably by maintaining splenic CD169+ macrophages and hepatic IL‐6 expression. This is of great importance to us, because we believe there is still a lot to investigate to unravel the full scope of the immune mechanisms implicated in the tremendous liver regenerative abilities. Behnke et al. showed that splenectomy or genetic B‐cell depletion resulted in reduced numbers of splenic CD169+ macrophages. In another experiment, Behnke et al. used CD169‐DTR transgenic mice in which CD169+ cells are depleted following diphtheria toxin injection. After partial hepatectomy, diphtheria injected CD169‐DTR mice had a lower survival rate, decreased liver/body weight ratio, and Ki67 staining reflecting a diminished regenerative response, as well as lower hepatic IL‐6 expression, as compared to control mice. However, we came across some imprecisions while reading the article. Although, as stated by the authors, spleen‐resident macrophages are CD169+, other macrophage populations express CD169 (including liver‐tissue–resident Kupffer cells and bone‐marrow–derived monocytes).2 Interestingly, Gupta et al. showed that tissue‐resident macrophages are effectively ablated in the spleen and liver from diphtheria toxin‐treated CD169‐DTR mice. It is also well known that macrophage depletion dramatically alters liver regeneration after partial hepatectomy.3 The confusion about CD169+ macrophage tissue distribution remains in the rest of the study, as the authors have not characterized hepatic macrophage populations in their models. This analysis would certainly strengthen the authors’ conclusions. Overall, we are enthusiastic about the authors’ findings on innate and adaptive immune responses in liver regeneration. Thus, we are looking forward to getting greater precision regarding the fate of liver macrophages throughout tissue regeneration in these models.
Novel influenza A(H1N2) seasonal reassortant identified in a patient sample, Sweden, January 2019
Authors: Åsa Wiman, Theresa Enkirch, AnnaSara Carnahan, Blenda Böttiger, Tove Samuelsson Hagey, Per Hagstam, Rosmarie Fält, Mia Brytting
Journal: Eurosurveillance (2019)

In January 2019, a human seasonal reassortant influenza A(H1N2) virus with a novel 7:1 genetic constellation was identified in a 68-year-old female patient with suspected pneumonia. The virus harboured A(H3N2) neuraminidase and remaining genes from A(H1N1)pdm09. The patient recovered after severe illness. No additional cases have been detected. This is the second identified A(H1N2) seasonal reassortant in a human in Europe within 1 year; a previous case was detected in the Netherlands in March 2018.
SAFETY AND EFFECTIVENESS OF PHARMACOLOGICAL CONVERSION AND DIRECT CURRENT CARDIOVERSION IN PERSISTENT ATRIAL FIBRILLATION: RESULTS OF RANDOMIZED TRIAL
Authors: Nikolay Mironov, Vladislav Vlodzyanovsky, Yulia Yuricheva, Sergey Sokolov, Sergey Golitsyn, Leonid Rosenstraukh, Eugeny Chazov
Journal: Journal of the American College of Cardiology (2019)
B-Cell Maturation Antigen (BCMA)-Specific, CentyrinTM-Based, PiggyBacTM-Transposed CAR-T Memory Stem Cells Are Effective Against p53-/- and Patient-Derived Multiple Myeloma Tumors
Authors: Rohit Mathur, Burton Earle Barnett, David Hermanson, Jin He, Zheng Zhang, Srinivas Rengarajan, Rebecca Codde, Xinxin Wang, Yening Tan, Christopher Martin, Jenessa Smith, Hans C. Lee, Elisabet E. Manasanch, Sheeba K. Thomas, Krina K. Patel, Donna M. Weber, R. Eric Davis, Robert Z. Orlowski, Eric M Ostertag, Devon Shedlock, Jing Yang, Sattva S. Neelapu
Journal: Blood (2017)
Interstitial fluid pressure (IFP) and metastasis in a panel of tumor models
Authors: Sarah J. Lunt, Tuula Mk Kalliomaki, Mike Milosevic, Richard P. Hill
Journal: Cancer Research (2006)

1036  Introduction: High tumor IFP has been linked to poor drug uptake in patients, and there is strong evidence that high IFP is associated with spread of disease in cervix cancer patients treated with radiotherapy. To date, studies of IFP have focused on genetically identical tumor models, often grown sub-cutaneously, although evidence suggests that orthotopic models may be more clinically relevant. In this study we compared IFP in an array of murine and xenograft models, spontaneously arising or grown both intramuscularly (i/m) and orthotopically. Methods: KHT-C murine fibrosarcoma, or CaSki, Me180 and SiHa human cervix carcinoma cells were injected i/m into the hind legs of C3H or SCID mice. In addition, i/m tumors were used as donors to establish orthotopic cervix tumors from the CaSki, Me180 or SiHa models. Polyoma middle-T (PyMT) transgenic mice generated spontaneous mammary tumors, which were transplanted orthotopically into the 4th mammary gland of wild-type or SCID mice. IFP was measured at multiple points in all tumors using the wick-in-needle method. Spontaneous metastasis formation in the lungs or lymph nodes was assessed in all models. Results: All models showed intertumor heterogeneity in IFP values; KHT-C i/m: 2-42 mmHg, CaSki: i/m 6-63, cervix -2-17 mmHg, Me180: i/m 17-38, cervix 5-9 mmHg, SiHa: i/m 20-56, cervix 2-26 mmHg, PyMT: spontaneous 2-20 and transplanted 2-22 mmHg. The PyMT mice develop multiple tumors and there was significant variation between tumors in the same mouse (5-20 mmHg). Similarly, there was variation in IFP between two tumors transplanted into the same recipient and there was no correlation between donor and recipient IFP in the cervix carcinoma models or the PyMT model. No significant correlation was apparent between IFP values and metastatic burden in any of the tumor models. Furthermore, exposure of KHT-C and CaSki tumor cells to simulated high pressure in vitro prior to i/v injection in vivo did not significantly enhance experimental metastases presentation. Conclusions: The data show consistent intertumor heterogeneity in IFP in all models, irrespective of site, although median IFP values varied for the different tumors and different sites of growth. In particular, orthotopic transplantation of the cervix carcinoma cell lines gave tumors with lower IFP values than for tumors grown i/m. In addition, there was a lack of correlation in IFP values between recipient and donor tumors, indicative of individual tumor development irrespective of previous molecular interactions. Consistent with this, IFP measurement of different tumors in the same mouse using the PyMT spontaneous tumor model showed a high degree of variation. Whether this reflects differences in vascular density and permeability in the tumors is under investigation. Despite a broad range of IFP values in an assortment of different tumors, no correlation was observed between IFP and metastatic dissemination.
A CORONARY CONUNDRUM: PAPILLARY MUSCLE RUPTURE AND ISCHEMIC MITRAL REGURGITATION SECONDARY TO THROMBOEMBOLISM IN ANTIPHOSPHOLIPID SYNDROME
Authors: Amar Shere, Pradyumna Agasthi, Farouk Mookadam, Reza Arsanjani
Journal: Journal of the American College of Cardiology (2019)
Induction of Death Receptors for TRAIL, a Cytotoxic Factor for GVL Effect, by Oncogenic Fusion E2A-HLF Derived from t(17;19)-Positive Acute Lymphoblastic Leukemia.
Authors: Xiaochun Zhang, Takeshi Inukai, Koshi Akahane, Kinuko Hirose, Itaru Kuroda, Hiroko Honna, Kumiko Goi, Keiko Kagami, Toshiya Inaba, Hidemitsu Kurosawa, Hiroaki Goto, Mikiya Endo, Hideo Yagita, A. Thomas Look, Kanji Sugita
Journal: Blood (2007)
The Interleukin-1 Axis and Risk of Death in Patients With Acutely Decompensated Heart Failure
Authors: Domingo A. Pascual-Figal, Antoni Bayes-Genis, Maria C. Asensio-Lopez, Alvaro Hernández-Vicente, Iris Garrido-Bravo, Francisco Pastor-Perez, Javier Díez, Borja Ibáñez, Antonio Lax
Journal: Journal of the American College of Cardiology (2019)
Antibiotic overconsumption and resistance in Turkey
Authors: B. Isler, Ş. Keske, M. Aksoy, Ö.K. Azap, M. Yilmaz, S.Ş. Yavuz, G. Aygün, E. Tigen, H. Akalın, A. Azap, Ö. Ergönül
Journal: Clinical Microbiology and Infection (2019)
THE ASSOCIATION OF VASCULAR DYSFUNCTION AND PLATELET REACTIVITY IS REGULATED BY THE INTERACTION BETWEEN ANTIDIABETIC AND ANTIPLATELET TREATMENT IN PATIENTS WITH TYPE 2 DIABETES MELLITUS
Authors: Gerasimos Siasos, Panagiota Stampouloglou, Evanthia Bletsa, Vasiliki Tsigkou, Konstantinos Mourouzis, Nikolaos Gouliopoulos, Marina Zaromytidou, Savvas Mazaris, Evangelos Oikonomou, Alexios Antonopoulos, Sotirios Tsalamandris, Georgia Vogiatzi, Anastasia Thanopoulou, Andromahi Vryonidou, Stavroula A. Paschou, Marianna Politou, Nikolaos Tentolouris, Dimitris Tousoulis
Journal: Journal of the American College of Cardiology (2019)
RETRIEVAL OF UNDEPLOYED STENT FROM THE LEFT MAIN DURING A STEMI USING A BALLOON AND A SNARE
Authors: Sahitya Posimreddy, Bader Abu Ghalyoun, Muhammed Shittu, Zaid Altheeb, Habib Habib, Biren Patel, Mirette Habib, Fayez Shamoon
Journal: Journal of the American College of Cardiology (2019)
DIAGNOSTIC PERFORMANCE OF ANGIOGRAPHY-BASED FRACTIONAL FLOW RESERVE IN SUBGROUPS: REPORT FROM THE FAST-FFR STUDY
Authors: Yuhei Kobayashi, Carlos Collet, Stephan Achenbach, Thomas Engstrom, Abid Assali, Richard Shlofmitz, Allen Jeremias, Stephane Fournier, Ajay Kirtane, Ran Kornowski, Gabriel Greenberg, Rami Jubeh, Daniel Kolansky, Thomas McAndrew, Ovidiu Dressler, Akiko Maehara, Mitsuaki Matsumura, Martin Leon, Bernard De Bruyne, William Fearon
Journal: Journal of the American College of Cardiology (2019)
EXERCISE TREADMILL TESTING HAS LOW UTILITY IN THE EVALUATION OF CORONARY MICROVASCULAR DISEASE
Authors: Diana M. Lopez, Sanjay Divakaran, Ankur Gupta, Navkaranbir Bajaj, Michael Osborne, Wunan Zhou, Jon Hainer, Josh Klein, Courtney Bibbo, Hicham Skali, Ron Blankstein, Sharmila Dorbala, Viviany R. Taqueti, Marcelo Di Carli
Journal: Journal of the American College of Cardiology (2019)
Epigenetic Profile of Treg-like Cells Induced By Mesenchymal Stem Cells in Vitro Resembles That of Natural Treg
Authors: Rita I Azevedo, Ekaterina Minskaia, Cláudia Lobato da Silva, Ana Fernandes Platzgummer, Ana I.S. Vieira, Joaquim M.S. Cabral, Joao F Lacerda
Journal: Blood (2018)
INTRACLASS CORRELATIONS OF VOLTAGE, FRACTIONATED ELECTROGRAMS, AND DOMINANT FREQUENCY IN PATIENTS WHERE LOCALIZED ABLATION TERMINATED PERSISTENT ATRIAL FIBRILLATION
Authors: Albert J. Rogers, Nosheen Moosvi, Abhinav Singh, Mahmood Alhusseini, Tina Baykaner, Paul Clopton, Wouter-Jan Rappel, Paul Wang, Sanjiv Narayan
Journal: Journal of the American College of Cardiology (2019)
EXAMINING PRESCRIPTION DRUG ADHERENCE AND LONG-TERM OUTCOMES IN PATIENTS MANAGED MEDICALLY FOR ACUTE CORONARY SYNDROME
Authors: Razi Khan, Padma Kaul, Sanjidatul Islam, Anamaria Savu, Sean Van Diepen, Kevin Bainey, Robert Welsh, Akshay Bagai, Andrew Yan, Shaun Goodman
Journal: Journal of the American College of Cardiology (2019)
COMPARISON OF ECHOCARDIOGRAPHIC AND FLUOROSCOPIC SIZING OF THE LEFT ATRIAL APPENDAGE OSTIUM PRIOR TO PERCUTANEOUS LEFT ATRIAL APPENDAGE CLOSURE
Authors: Soroosh Kiani, Akshar V. Patel, Mikhael El Chami, Anshul Patel, David Delurgio, Michael Hoskins
Journal: Journal of the American College of Cardiology (2019)
LATE ONSET HYPERTROPHIC CARDIOMYOPATHY: UNUSUAL CAUSE OF SYNCOPE IN ELDERLY
Authors: Omar Al-Taweel, Loui Rejjal, Ahmad Al-Taweel, Asif Sewani, Masood Ahmad, Luba Frank, Mohamed Faher Almahmoud, George Carayannopoulos
Journal: Journal of the American College of Cardiology (2019)
DISCORDANCE OF LIPID RICH PLAQUE DIAGNOSIS BETWEEN OPTICAL COHERENCE TOMOGRAPHY AND INTRAVASCULAR ULTRASOUND
Authors: Tetsumin Lee, Gary Mintz, Eisuke Usui, Masahiro Hoshino, Yoshihisa Kanaji, Tadashi Murai, Taishi Yonetsu, Tsunekazu Kakuta, Akiko Maehara
Journal: Journal of the American College of Cardiology (2019)
COMPARISON OF TWO RADIAL HEMOSTASIS TECHNIQUES, CATECHOL CONJUGATED CHITOSAN-BASED PAD (INNOSEAL) WITH COMPRESSION BAND: A RANDOMIZED CONTROLLED TRIAL
Authors: Saadia Sattar, Saba Aijaz, Zohaib Akhter, Chang Kim, Moon Sue Lee, Rehan Malik, Asad Pathan
Journal: Journal of the American College of Cardiology (2019)
Abstract 204: Temporal Assessment of Human iPSC-Derived Cardiomyocytes to Assess the Maturity for Cardiac Repair Applications
Authors: Naresh Kumar, Julie A Dougherty, Muhamad Mergaye, Mark G Angelos, Mahmood Khan
Journal: Circulation Research (2018)

Cardiovascular disease is the major cause of heart failure following myocardial infarction (MI), which leads to death in acute condition. In the recent year, stem cell therapy has shown a great potential to repair the damaged heart tissue following an MI. Both adult and embryonic stem cells (ES) have potential to repair and regenerate tissue to compensate cell death during MI. Apart from ethical concern, there are several advantages of using human iPSC cardiomyocytes (hiPSC-CMs) over human ES. Most important, the hiPSC-CMs provides autologous cells source which one side eliminate the possibility of cellular rejection while on the other side enhance the possibility of its survival in damaged cardiac tissue. However, the lack of maturity in hiPSC-CMs is a major challenge to replicate and/or replace the adult cardiomyocytes with mature cardiomyocytes at the site of MI. Temporal assessment of cardiac maturity markers and structural gene in hiPSC-CMs cultured in vitro for up to four weeks and comparison with non-failing human heart (NF). The wk-4 hiPSC-CMs has shown an increase in cardiac transcription factor (2-fold, NKX-2.5); Myosin genes (2-3 fold, MYH7, MYHL2); cardiac troponin-T (3-4 fold, TNNI3 and TNNT3); K + channel genes (20-fold, KCNJ2); Ca 2+ related genes (10-fold, CASQ2); Gap-junction genes (2-fold, GJA1) and Na + Channel gene (5-fold, SCN5A). Further, TEM showed that the myofibrils in the wk-4 cardiomyocytes were more compact, aligned and organized. The Z and I-band and specifically M-band were also prominent in the wk-4 sarcomeres. Furthermore, the maturity of hiPSC-CMs was compared with the NF human heart. This study showed a time-course assessment of cardiomyocytes morphology, structural and expression of cardiac maturity genes in hiPSC-CMs cultured for up to four weeks in vitro . Based on the study of the cardiac transcription factor, Na-K channel-related gene and TEM analysis, wk-4 cardiomyocytes showed more maturity than week-1 cardiomyocytes. Studies are ongoing to compare the contractility and action potential of hiPSC-CMs. Finally, the mature hiPSC-CMs will provide autologous cell source and have a significant impact on the functional outcome of patients with acute MI.
EFFICACY OF DUAL ANTI-PLATELET THERAPY IN CORONARY ARTERY BYPASS GRAFT PATIENTS WITH STABLE ISCHEMIC HEART DISEASE
Authors: Iftikhar A. Ch (Chaudhry), Naeem Tahirkheli, Faisal Latif, Ahmad Usmani, Mashal Tahirkheli, Faris Amil, Sabrina Chaudry, Ananya Bhaktaram, Aldon Whitehead, Hunter Weitzel, Raja Ullah, Abdul Qadar, Pei-Tzu Wu, Haris Ijaz, Jeffrey Garrett
Journal: Journal of the American College of Cardiology (2019)
PREVALENCE OF TRIGGERS FROM NON-PULMONARY VEIN SITES IN PATIENTS WITH PRIOR CORONARY ARTERY BYPASS GRAFTING OR MAZE PROCEDURE UNDERGOING CATHETER ABLATION FOR ATRIAL FIBRILLATION
Authors: Sanghamitra Mohanty, Chintan Trivedi, Domenico Della Rocca, Qiong Chen, Omer Gedikli, Carola Gianni, Alfredo C. Tapia, Sharan Sharma, John Burkhardt, Javier Sanchez, Gerald Gallinghouse, Amin Al-Ahmad, Rodney Horton, Dhanunjaya Lakkireddy, Luigi di Biase, Andrea Natale
Journal: Journal of the American College of Cardiology (2019)
INTERACTION OF SMOKING STATUS ON THE ASSOCIATION OF BODY MASS INDEX WITH MAJOR ADVERSE LIMB OUTCOMES IN PERIPHERAL ARTERY DISEASE INSIGHTS FROM THE REACH REGISTRY
Authors: Poghni Peri-Okonny, Philip Jones, Elizabeth Magnuson, John A. Spertus, Vittal Hijjaji, Ali Malik, Kaijun Wang, Philippe Steg, Deepak L. Bhatt, Kim Smolderen
Journal: Journal of the American College of Cardiology (2019)
EARLY STRUCTURAL VALVE DETERIORATION OF A BALLOON EXPANDABLE EDWARD SAPIEN VALVE TREATED WITH SELF EXPANDABLE EVOLUT-R USING TRANSCATHETER VALVE IN VALVE TECHNIQUE
Authors: Abdulwahab Hritani, Imaad Razzaque, Amanda Kirby, Suhail Allaqaband, Bijoy K. Khandheria, Daniel P. O'Hair, Renuka Jain, Tanvir Bajwa
Journal: Journal of the American College of Cardiology (2019)