tbuckley/cabot-search huggingface dataset:
Pulmonary venous circulating tumor cell dissemination before tumor resection and disease relapse
Authors: Francesca Chemi, Dominic G. Rothwell, Nicholas McGranahan, Sakshi Gulati, Chris Abbosh, Simon P. Pearce, Cong Zhou, Gareth A. Wilson, Mariam Jamal-Hanjani, Nicolai Birkbak, Jackie Pierce, Chang Sik Kim, Saba Ferdous, Deborah J. Burt, Daniel Slane-Tan, Fabio Gomes, David Moore, Rajesh Shah, Maise Al Bakir, Crispin Hiley, Selvaraju Veeriah, Yvonne Summers, Philip Crosbie, Sophia Ward, Barbara Mesquita, Marek Dynowski, Dhruva Biswas, Jonathan Tugwood, Fiona Blackhall, Crispin Miller, Allan Hackshaw, Ged Brady, Charles Swanton, Caroline Dive
Journal: Nature Medicine (2019)
Microsatellite instability status in metastatic colorectal cancer and effect of immune checkpoint inhibitors on survival in MSI-high metastatic colorectal cancer
Authors: W. Okamoto, Y. Nakamura, M. Shiozawa, Y. Komatsu, T. Denda, H. Hara, Y. Kagawa, Y. Narita, H. Kawakami, T. Esaki, T. Nishina, N. Izawa, K. Ando, T. Moriwaki, T. Kato, F. Nagashima, T. Satoh, S. Nomura, T. Yoshino, K. Akagi
Journal: Annals of Oncology (2019)
Initial results of a phase II study of nivolumab and ipilimumab in metastatic adrenal tumours
Authors: M.T. Campbell, W. Xie, A.Y. Shah, M.A. Habra, C. Jimenez, A.M. Venkatesan, G. Bubley, R.R. McKay, T.K. Choueiri, B.A. McGregor, K.L. Killbridge, A. Mortazavi, M.A. Bilen
Journal: Annals of Oncology (2019)
Hierarchy in Somatic Mutations Detected in Circulating and Tissue-Resident Follicular Lymphoma Precursors before Clinical Diagnosis
Authors: Joseph G Schroers-Martin, Florian Scherer, Joanne Soo, David M. Kurtz, Chih Long Liu, Henning Stehr, Agnès Bru, Yasodha Natkunam, Maximilian Diehn, Bertrand Nadel, Sandrine Roulland, Ash A. Alizadeh
Journal: Blood (2017)
Gastrointestinal Tract Dysbiosis Enhances Distal Tumor Progression through Suppression of Leukocyte Trafficking
Authors: Samir V. Jenkins, Michael S. Robeson, Robert J. Griffin, Charles M. Quick, Eric R. Siegel, Martin J. Cannon, Kieng B. Vang, Ruud P.M. Dings
Journal: Cancer Research (2019)

The overall use of antibiotics has increased significantly in recent years. Besides fighting infections, antibiotics also alter the gut microbiota. Commensal bacteria in the gastrointestinal tract are crucial to maintain immune homeostasis, and microbial imbalance or dysbiosis affects disease susceptibility and progression. We hypothesized that antibiotic-induced dysbiosis of the gut microbiota would suppress cytokine profiles in the host, thereby leading to changes in the tumor microenvironment. The induced dysbiosis was characterized by alterations in bacterial abundance, composition, and diversity in our animal models. On the host side, antibiotic-induced dysbiosis caused elongated small intestines and ceca, and B16-F10 melanoma and Lewis lung carcinoma progressed more quickly than in control mice. Mechanistic studies revealed that this progression was mediated by suppressed TNFα levels, both locally and systemically, resulting in reduced expression of tumor endothelial adhesion molecules, particularly intercellular adhesion molecule-1 (ICAM-1) and a subsequent decrease in the number of activated and effector CD8+ T cells in the tumor. However, suppression of ICAM-1 or its binding site, the alpha subunit of lymphocyte function-associated antigen-1, was not seen in the spleen or thymus during dysbiosis. TNFα supplementation in dysbiotic mice was able to increase ICAM-1 expression and leukocyte trafficking into the tumor. Overall, these results demonstrate the importance of commensal bacteria in supporting anticancer immune surveillance, define an important role of tumor endothelial cells within this process, and suggest adverse consequences of antibiotics on cancer control. SIGNIFICANCE: Antibiotic-induced dysbiosis enhances distal tumor progression by altering host cytokine levels, resulting in suppression of tumor endothelial adhesion molecules and activated and effector CD8+ T cells in the tumor.
FUSAFE individual patient data meta-analysis (MA) to assess the performance of dihydropyrimidine dehydrogenase (DPD) gene polymorphisms for predicting grade 4-5 fluoropyrimidine (FP) toxicity
Authors: M.-C. Etienne-Grimaldi, N. Cozic, J.-C. Boyer, V. Boige, R.B. Diasio, J. Taieb, D. Meulendijks, C. Palles, U. Zanger, M. Deenen, C. Largiader, M. Boisdron-Celle, A. Marinaki, B. Jennings, E. Gross, F. Thomas, M.-A. Loriot, G Le Teuff, J.-P. Pignon
Journal: Annals of Oncology (2019)
Quantitative interactomics in primary T cells unveils TCR signal diversification extent and dynamics
Authors: Guillaume Voisinne, Kristof Kersse, Karima Chaoui, Liaoxun Lu, Julie Chaix, Lichen Zhang, Marisa Goncalves Menoita, Laura Girard, Youcef Ounoughene, Hui Wang, Odile Burlet-Schiltz, Hervé Luche, Frédéric Fiore, Marie Malissen, Anne Gonzalez de Peredo, Yinming Liang, Romain Roncagalli, Bernard Malissen
Journal: Nature Immunology (2019)
Gut microbiota shape ‘inflamm-ageing’ cytokines and account for age-dependent decline in DNA damage repair
Authors: Avital Guedj, Yael Volman, Anat Geiger-Maor, Julia Bolik, Neele Schumacher, Sven Künzel, John F Baines, Yuval Nevo, Sharona Elgavish, Eithan Galun, Hagai Amsalem, Dirk Schmidt-Arras, Jacob Rachmilewitz
Journal: Gut (2019)

Objective Failing to properly repair damaged DNA drives the ageing process. Furthermore, age-related inflammation contributes to the manifestation of ageing. Recently, we demonstrated that the efficiency of repair of diethylnitrosamine (DEN)-induced double-strand breaks (DSBs) rapidly declines with age. We therefore hypothesised that with age, the decline in DNA damage repair stems from age-related inflammation. Design We used DEN-induced DNA damage in mouse livers and compared the efficiency of their resolution in different ages and following various permutations aimed at manipulating the liver age-related inflammation. Results We found that age-related deregulation of innate immunity was linked to altered gut microbiota. Consequently, antibiotic treatment, MyD88 ablation or germ-free mice had reduced cytokine expression and improved DSBs rejoining in 6-month-old mice. In contrast, feeding young mice with a high-fat diet enhanced inflammation and facilitated the decline in DSBs repair. This latter effect was reversed by antibiotic treatment. Kupffer cell replenishment or their inactivation with gadolinium chloride reduced proinflammatory cytokine expression and reversed the decline in DSBs repair. The addition of proinflammatory cytokines ablated DSBs rejoining mediated by macrophage-derived heparin-binding epidermal growth factor-like growth factor. Conclusions Taken together, our results reveal a previously unrecognised link between commensal bacteria-induced inflammation that results in age-dependent decline in DNA damage repair. Importantly, the present study support the notion of a cell non-autonomous mechanism for age-related decline in DNA damage repair that is based on the presence of ‘inflamm-ageing’ cytokines in the tissue microenvironment, rather than an intrinsic cellular deficiency in the DNA repair machinery.
Punctate Keratotic Papules on the Palm
Authors: Megan Brinkworth Vissing, Leila Tolaymat, Jason Sluzevich
Journal: JAMA Dermatology (2019)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA Dermatology HomeNew OnlineCurrent IssueFor Authors Podcast Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA Dermatology journal
Improved survival with secondary surgery in a reference center after a first R1 or R2 resection in soft tissue sarcoma (STS) of the limbs or trunk wall: An analysis 10931 patients (pts) in NETSARC
Authors: F. Gouin, E. Stoeckle, S. Bonvalot, C. Honoré, A. Decanter, P. Anract, E. Bompas, G. Ferron, F. Sirveaux, A. Rochwerger, S. Carrere, A Di Marco, M. Ropars, L Le Nail, O. Marco, J. Guiramand, A. Italiano, N. Penel, A Le Cesne, J.-Y. Blay
Journal: Annals of Oncology (2019)
Abstract #1484: Cellular compartmentalization of BTG1 in Glucocorticoid-evoked apoptosis of human leukemic CCRF-CEM cells
Authors: Mia Wibowo, Rheem Medh
Journal: Cancer Research (2009)

Our goal is to characterize key early modulators of apoptosis to facilitate the treatment of diseases involving dysfunctional apoptosis mechanism such as cancer and autoimmune disease. Glucocorticoids (GC) are steroid hormones that induce apoptosis in thymocytes and therefore are a viable treatment option for leukemia. Two clones of human leukemia CCRF-CEM cells, the GC resistant CEM-C1-15 and GC sensitive CEM-C7-14 are useful models to study pathways for GC-evoked apoptosis. Apoptosis in these cells is preceded by G1 cell cycle arrest and up regulation of several genes, including the antiproliferative gene btg1. BTG1 has been previously shown to arrest the cell cycle in G1 phase through a mechanism that requires nuclear translocation. Interestingly, GC resistant CEM-C1-15 cells express higher basal BTG1 transcript and protein levels, although they are refractory to GC-induced G1 arrest and apoptosis. In this study, we are investigating if nuclear translocation of BTG1 is a requirement for its anti-proliferative activity in GC-evoked apoptosis and whether a failure to translocate to the nucleus can explain the GC-resistance of CEM-C1-15 cells. Cytoplasmic and nuclear proteins from GC-treated cells are resolved by Western blot to compare the localization of BTG1 in the sensitive and resistant cell lines. Immunocytochemical visualization of BTG1 using an anti-BTG1 primary antibody and a Texas Red-conjugated secondary antibody is being done by confocal microscopy. Localization of ectopically expressed BTG1 is being investigated using transiently transfected GFP-BTG1. We have been successful in obtaining clean nuclear and cytoplasmic protein fractions for Western blotting, and data suggest higher nuclear localization of BTG1 protein in CEM-C7-14 cells stimulated to undergo apoptosis, compared to resistant CEM-C1-15 cells. Immunocytochemical localization of BTG1 shows distribution in both cytoplasmic and nuclear compartments even after GC stimulation of CEM-C7-14 cells. Further studies are ongoing to clarify the role of GC-induced nuclear translocation of BTG1 in evoking an apoptotic response. Acknowledgements: Funding for this project is provided by the Academic Research Enhancement Award (AREA) #R15CA122613-01A1 from the NIH. MW is a MARC U*STAR undergraduate research scholar.  Citation Information: In: Proc Am Assoc Cancer Res; 2009 Apr 18-22; Denver, CO. Philadelphia (PA): AACR; 2009. Abstract nr 1484.
5α-Reductase Inhibitor Use in Patients With Prostate Cancer
Authors: Ian M. Thompson, Catherine M. Tangen, Barnett Kramer
Journal: JAMA Internal Medicine (2019)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA Internal Medicine HomeNew OnlineCurrent IssueFor Authors Podcast Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA Internal Medicine journal
Impact of lung metastasis on overall survival (OS) in the phase III SELECT study with lenvatinib (LEN) in patients (pts) with radioiodine refractory differentiated thyroid cancer (RR-DTC)
Authors: M. Tahara, N. Kiyota, A.O. Hoff, C. Badiu, T.K. Owonikoko, C.E. Dutcus, T. Suzuki, M. Ren, S. Misir, L.J. Wirth
Journal: Annals of Oncology (2019)
Gene Expression Profiling (GEP) As a Prognostic Indicator of Multiple Myeloma, Irrespective of Disease Stage, Prior Treatment and Prior Transplant - a Single Center Experience
Authors: Miguel Gonzalez Velez, Tracy Andrews, Narjust Duma, Ning Dong, Badal Thakkar, Giselle Suero, Joshua R Richter, David H. Vesole, David Siegel, Noa Biran
Journal: Blood (2017)
Thermotolerance-induced goblet cell activity confers protection in post-operative gut barrier dysfunction
Authors: R Ali, T Farrell
Journal: Endoscopy (2006)

Aims: The aims of this research is to examine the hypothesis that the gut-barrier dysfunction which results from ischaemia-reperfusion injury and bowel-handling is prevented or decreased by prior induction of thermotolerance and whether the protective effect of thermotolerance may be due to phenomena other than heat shock protein.
Perioperative chemotherapy of oxaliplatin combined with S-1 (SOX) versus postoperative chemotherapy of SOX or oxaliplatin with capecitabine (XELOX) in locally advanced gastric adenocarcinoma with D2 gastrectomy: A randomized phase III trial (RESOLVE trial)
Authors: J. Ji, L. Shen, Z. Li, X. Zhang, H. Liang, Y. Xue, Y. Wang, Z. Zhou, J. Yu, L. Chen, Y. Du, G. Li, G. Xiao, D. Wu, Y. Zhou, C. Dang, Y. He, Z. Zhang, Y. Sun, Y. Li
Journal: Annals of Oncology (2019)
Induction of IgG<sub>2</sub> and IgG<sub>4</sub> B‐cell memory following sublingual immunotherapy for ryegrass pollen allergy
Authors: Jorn J. Heeringa, Craig I. McKenzie, Nirupama Varese, Mark Hew, Amy T. C. M. Bakx, Pei M. Aui, Jennifer M. Rolland, Robyn E. O’Hehir, Menno C. van Zelm
Journal: Allergy (2019)

While treatment for atopic rhinitis is aimed mostly to relieve symptoms, only allergen-specific immunotherapy (AIT) is targeted to modify the natural history of allergic diseases. This results in sustained clinical tolerance, even when treatment has stopped. The immunomodulatory effects of AIT are attributed mainly to increased regulatory T-cell function and increased allergen-specific IgG4 , yet little is known about the effect on the memory B-cell compartment.We aimed to examine the effects of AIT on the IgE- and IgG subclass-expressing memory B cells.We recruited 29 patients with atopic seasonal rhinoconjunctivitis and performed a longitudinal analysis of the peripheral immune compartment before, during, and after sublingual immunotherapy (SLIT) for allergy to temperate grass pollen, predominantly to ryegrass pollen (RGP; Lolium perenne). Using flow cytometry on peripheral blood mononuclear cells and serum immunoassays, we analyzed the effects of a 4 months preseasonal treatment regimen comprising two or three courses in consecutive years on circulating IgE+ and IgG+ memory B cells and allergen-specific Ig levels.SLIT increased RGP-specific serum IgG2 and IgG4 , as well as the frequencies of IgG2+ and IgG4+ memory B cells, whereas no effect was observed on the IgE+ memory B-cell compartment. Furthermore, SLIT enhanced proportions of regulatory T cells specific to RGP. These changes were associated with clinical improvement.Our data provide evidence for immunological effects of SLIT on B-cell memory. Skewing responses toward IgG2 and IgG4 subclasses might be a mechanism to suppress IgE-mediated allergic responses.
Updated analysis of a phase I trial of afatinib (Afa) and bevacizumab (Bev) in chemo-naïve patients (pts) with advanced non-small cell lung cancer (NSCLC) harboring EGFR-mutations: OLCSG1404
Authors: T. Ninomiya, N. Nogami, T. Kozuki, D. Harada, T. Kubo, K. Ohashi, S. Kuyama, K. Kudo, A. Bessho, N. Fujimoto, K. Aoe, T. Shibayama, D. Minami, K. Sugimoto, N. Ochi, N. Takigawa, K. Hotta, K. Kiura
Journal: Annals of Oncology (2019)
Serum biomarkers of bone metabolism in metastatic castration-resistant prostate cancer (mCRPC) patients (pts) treated with radium-223 (Ra223): Results from a prospective multicentre study
Authors: N Romero Laorden, R Lozano Mejorada, A Hernández Jorge, N. Lainez, E Almagro Casado, O Fernández Calvo, S. Hernando, A Fernández Freire, E Gonzalez Billalabeitia, I. Chirivella, R García Domínguez, U Anido Herranz, F J Vazquez Mazon, S. Ros, A. Montesa, G.A. De Velasco Oria de Rueda, R Morales Barrera, D. Lorente, E. Castro, D Olmos Hidalgo
Journal: Annals of Oncology (2019)
Defining a SUV decrease cut-off in PET/CT response monitoring after one cycle of preoperative breast cancer chemotherapy
Authors: M. Kubeczko, K. Świderska, A. d’Amico, M. di Pietro, O. Chrabański, A. Polakiewicz-Gilowska, M. Mianowska-Malec, W. Bal, A. Badora-Rybicka, A. Leśniak, A. Pasierbek, A. Kuś, R. Tarnawski, B. Bobek-Billewicz, M. Jarzab
Journal: Annals of Oncology (2019)
Abstract 17297: Identification of Putative Fibrous Plaque Marker Proteins by Unsupervised Deconvolution of Heterogeneous Vascular Proteomes
Authors: Sarah J Parker, LuLu Chen, Qin Fu, Vidya Venkatraman, Ronald Holewinski, Georgia Saylor, Chunhong Mao, Richard Vander Heide, Yue Wang, David Herrington, Jennifer van Eyk
Journal: Circulation (2017)

Accurate risk stratification of individuals for appropriate prioritization of therapeutic intervention remains a significant clinical challenge for early atherosclerosis management. Circulating bio...
Abstract P152: Cardiometabolic Risk Factors in Adolescent Blood Donors
Authors: James Keeton, Stephen J Eason, Merlyn Sayers, Colby Ayers, Maria O Gore
Journal: Circulation (2017)

Objective: Cardiometabolic risk factors have been extensively studied in adults, but to a lesser extent in adolescents. We assessed potential cardiometabolic risk factors in a large cohort of adolescent blood donors. Methods: Glycated hemoglobin (HbA 1c ), blood pressure (BP), and total cholesterol were measured in 10,756 blood donors aged 16-19 years at school blood drives conducted by Carter BloodCare, a large North Texas blood center. Borderline values were defined as HbA 1c 5.7-6.4%, BP (systolic/diastolic) 120-139/80-89 mm Hg, and total cholesterol 170-199 mg/dL. Elevated values were defined as HbA 1c ≥6.5%, BP ≥140/90, and total cholesterol ≥200 mg/dL. Subjects were classified into one of three subcohorts: (A) no borderline or elevated values (“healthy” subcohort); (B) one borderline value; (C) either two borderline values or one elevated value. The subcohorts were further stratified as shown in the Table. Results: Of the 10,756 blood donors, 35.2% had one borderline cardiometabolic risk factor, and 17.9% had either two borderline or one elevated risk factor. There were more girls than boys in the “healthy” subcohort (p&lt;0.0001). Girls had a higher prevalence of borderline or elevated total cholesterol (p&lt;0.0001), whereas boys had higher prevalence of borderline or elevated BP (p&lt;0.0001). Other differences between subcohorts are summarized in the Table. Conclusion: More than half of adolescents in this study had at least one cardiometabolic risk factor that was either borderline or elevated. Blood donation programs can serve as highly efficient and cost-effective gateways for cardiometabolic risk screening in adolescents, with potential for the development of targeted interventions aimed at promoting healthy behaviors early in life, specifically among those at increased risk.
Outcome of 98 patients with epithelioid sarcoma treated in curative intent: A retrospective study from the French Sarcoma Group (GSF-GETO)
Authors: M. Pedrono, O. Mir, L. Chaltiel, M. Brahmi, A. Italiano, G. Decanter, P Boudou Rouquette, M. Ropars, E. Bompas, N. Firmin, N. Isambert, T. Valentin, F. Duffaud, J. Gantzer, A. Thyss, C. Guillemet, J.-Y. Blay, A Le Cesne, C.M. Chevreau, C. Perrin
Journal: Annals of Oncology (2019)
Phase I dose escalation study of a selective androgen receptor modulator RAD140 in estrogen receptor positive (ER+), HER2 negative (HER2-) breast cancer (BC)
Authors: E. Hamilton, N. Vidula, C. Ma, P. LoRusso, R.G. Bagley, Z. Yu, M. Annett, A. Weitzman, M.G. Conlan, A. Weise
Journal: Annals of Oncology (2019)
Disease monitoring of EGFR mutation-positive NSCLC patients via circulating tumour DNA
Authors: W.F. Hsu, H.-L. Ho, C.L. Chiang, Y. Jiang, S.M. Karwowska, R. Yerram, K. Sharma, A. Sharma, S. Scudder, C.-M. Tsai, J. Palma, C.-H. Chiu, T.-Y. Chou
Journal: Annals of Oncology (2019)
GI101, a novel triple-targeting bispecific CD80-IgG4-IL2variant fusion protein, elicits synergistic anti-tumour effects in preclinical models
Authors: J.C. Park, K.H. Pyo, Y.J. Song, W.J. Lee, J. Kim, H.N. Ji, S.S. Park, Y.J. Koh, K. Lee, B. Cho, B.-H. Chun, Y.M. Oh, B.-G. Yang, S.Y. Nam, M.H. Jang
Journal: Annals of Oncology (2019)
Investigation of the CXCL12-CXCR4/CXCR7 Axis in Multiple Myeloma (MM): Adhesion Molecules As Novel Targets of the Potent Proteasome Inhibitor Carfilzomib
Authors: Anna Simon, Dagmar Wider, Johannes Waldschmidt, Marie Follo, Sarah Decker, Ralph Wäsch, Monika Engelhardt
Journal: Blood (2014)
Prognostic and predictive factors from the phase III CELESTIAL trial of cabozantinib (C) versus placebo (P) in previously treated advanced hepatocellular carcinoma (aHCC)
Authors: T. Meyer, R.K. Kelley, M. Mangeshkar, A.-L. Cheng, A.B. El-Khoueiry, G.K. Abou-Alfa
Journal: Annals of Oncology (2019)
Biliary tract cancers in Portuguese families with BRCA gene mutation: A retrospective study
Authors: P. Pereira, I. Oliveira, S. Fernandes, A. Clara, I. Miguel, S. Bento, S. Fragoso, S. Santos, P. Rodrigues, J. Parreira, F. Rodrigues, I. Silva, P. Louro, F. Vaz, A. Luis
Journal: Annals of Oncology (2019)
Geriatric parameters predict both disease-related and patient-reported outcomes in older patients with breast cancer
Authors: W. van der Plas-Krijgsman, A. de Boer, P. de Jong, E. Bastiaannet, F. van den Bos, G.-J. Liefers, J.E.A. Portielje, N. De Glas
Journal: Annals of Oncology (2019)