Rheumatology: An Annotated Bibliography of Recent Literature
Authors: RHEUMATOLOGY COMMITTEE
Journal: Annals of Internal Medicine (1979)

The Literature of Medicine1 December 1979Rheumatology: An Annotated Bibliography of Recent LiteratureReferences to Journal Articles and Other PapersRHEUMATOLOGY COMMITTEERHEUMATOLOGY COMMITTEESearch for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-91-6-932 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptThis bibliography includes references to 89 recently published journal papers and similar sources that represent important developments in the concept and practice of internal medicine. It has been compiled by the Rheumatology Committee as a part of the Medical Knowledge Self-Assessment Program V Syllabus.In preparing this bibliography, the committee reviewed MEDLINE†bibliographic printouts of journal articles published since the beginning of 1976 and also drew on sources in libraries of hospitals and other clinical institutions in the United States and Canada; thus, only papers in English are listed, and most are from American journals....Connective Tissue Clinical Pharmacology of Drugs for the Rheumatic Diseases Rheumatoid Arthritis Systemic Lupus Erythematosus Scleroderma (Systemic Sclerosis) Polymyositis, Dermatomyositis, and Overlapping Conditions Osteoarthritis Avascular (Aseptic) Necrosis, Osteonecrosis Histocompatibility Antigens and Arthritis Ankylosing Spondylitis Arthritis Associated with Intestinal Bypass Procedures The Vasculitides Crystal-Induced Synovitis and Other Arthritic Disorders Infectious Arthritis Osteoporosis and Osteomalacia (Osteopenia) Paget's Disease1. FESSLERFESSLER JL. Biosynthesis of procollagen. Annu Rev Biochem. 1978;47:129-62. A medium-length review of the biological and pathologic implications of the synthesis of collagens as precursors, called procollagens, that are enzymatically cleaved to form collagens outside the cell. CrossrefMedlineGoogle Scholar2. HASCALL V. Interaction of cartilage proteoglycans with hyaluronic acid. J Supramol Struct. 1977;7:101-20. Thorough discussion of the structure, biosynthesis, and macromolecular interactions of proteoglycans, with emphasis on cartilage. CrossrefMedlineGoogle Scholar3. MILLER E: Comparison of basement membrane collagens with interstitial collagens. In: KEFALIDES NA, ed. Biology and Chemistry of Basement Membranes. New York: Academic Press; 1978:265-78. Concise statement that basement membrane collagens are a family of proteins different from the interstitial collagens. Google Scholar4. JAFFE I. D-penicillamine. Bull Rheum Dis. 1977-78;28:948-52. Management practices are described, with a description of side-effects, their timing, and appropriate responses to their appearance. Use of the drug in rheumatoid arthritis is emphasized. D-penicillamine for progressive systemic sclerosis is said to have little effect on the visceral features and is considered investigational at best. MedlineGoogle Scholar5. MCMICHAELSASAZUKIMCDEVITTPAYNE ATHR: Increased frequency of HLA-Cw3 and HLA-Dw4 in rheumatoid arthritis. Arthritis Rheum. 1978;20:1037-42. HLA-Dw4 was found in 36% of patients with rheumatoid arthritis, compared with 13% of controls. Cw3 is in linkage disequilibrium with Dw4. A good study that extends Stastny's observations. CrossrefGoogle Scholar6. HARTMARMION HB. Rubella virus and rheumatoid arthritis. Ann Rheum Dis. 1977;36:3-12. The authors present data that do not support the hypothesis that a persistent infection with rubella virus initiates the rheumatoid process. References provide a good review of viral studies in this disease. CrossrefMedlineGoogle Scholar7. ALSPAUGHTAN ME. Serum antibody in rheumatoid arthritis reactive with a cell-associated antigen: demonstration by precipitation and immunofluorescence. Arthritis Rheum. 1976;19:711-9. A soluble antigen (rheumatoid arthritis nuclear antigen) from a lymphoblastoid cell line precipitated with antibody in sera of approximately 60% of patients with rheumatoid arthritis (seropositive) and Sjögren's syndrome, but in only 8% of controls. Later data have shown that the antigen is induced by Epstein-Barr virus infection. CrossrefMedlineGoogle Scholar8. BANKHURSTHUSBYWILLIAMS AGR. Predominance of T cells in the lymphocytic infiltrates of synovial tissues in rheumatoid arthritis. Arthritis Rheum. 1976;19:555-62. Seventy five percent to 90% of lymphocytes in rheumatoid synovial tissue were found to be T cells using cell-specific antibodies. A review of earlier data is included. CrossrefMedlineGoogle Scholar9. BENNETT J. The infectious etiology of rheumatoid arthritis: new considerations. Arthritis Rheum. 1978;21:531-8. Provides direction for future research into possible infectious causes of rheumatoid arthritis. CrossrefMedlineGoogle Scholar10. DAYERRUSSELLKRANE JRS. Collagenase production by rheumatoid synovial cells: stimulation by a human lymphocyte factor. Science 1977;195:181-3. Human peripheral blood mononuclear cells in culture release a factor that stimulates rheumatoid synovial cells to produce collagenase. These data help link the inflammatory and the proliferative lesion in rheumatoid arthritis. CrossrefMedlineGoogle Scholar11. ENGLEMANSPONZILLIBATEYRAMCHARANMCDEVITT EEMSH. Mixed lymphocyte reaction in healthy women with rheumatoid factor: lack of association with HLA-Dw4. Arthritis Rheum. 1978;21:690-3. This study, using mixed lymphocyte culture techniques, found HLA-Dw4 in only three of 24 healthy women who had circulating rheumatoid factor, whereas approximately 50% of patients with rheumatoid arthritis have HLA-Dw4. CrossrefMedlineGoogle Scholar12. GELMANWARD MJ. Septic arthritis: a complication of rheumatoid arthritis. Radiology. 1977;122:17-24. Study emphasizing the problems of septic arthritis super-imposed on rheumatoid disease suggests radiographic criteria to help make the diagnosis. CrossrefMedlineGoogle Scholar13. GRAYGOTTLIEB RN. Hand flexor tenosynovitis in rheumatoid arthritis: prevalence, distribution, and associated rheumatic features. Arthritis Rheum. 1977;20:1003-8. Tenosynovitis was noted in 55% of hand tendons in rheumatoid patients. This problem, rather than joint disease, may cause hand pain and dysfunction in significant numbers of rheumatoid patients. CrossrefMedlineGoogle Scholar14. HUTCHINSONDAVISJAYSON RPM. Thrombocytosis in rheumatoid arthritis. Ann Rheum Dis. 1976;35:138-42. Thrombocytosis in rheumatoid arthritis is associated with increased disease severity and a low level of hemoglobin. The mechanism appears to be a compensatory increase in platelet production in face of active intravascular coagulation. CrossrefMedlineGoogle Scholar15. MACDONALDCRAWFORDKLIPPELZVAIFLERO'ROURKE WMJNR. Echocardiographic assessment of cardiac structure and function in patients with rheumatoid arthritis. Am J Med. 1977;63:890-6. With sophisticated echocardiography, pericardial effusion was found in 31% of patients with rheumatoid arthritis, almost all of whom were asymptomatic. CrossrefMedlineGoogle Scholar16. MCADAMO'HANLANBLUESTONEPEARSON LMRC. Relapsing polychondritis: prospective study of 23 patients and a review of the literature. Medicine (Baltimore). 1976;55:193-215. Excellent prospective study of 23 patients with polychondritis followed at one institution. Diagnostic criteria are proposed and a differential diagnosis developed. CrossrefMedlineGoogle Scholar17. OHNOCOOKE OT. Electron microscopic morphology of immunoglobulin aggregates and their interactions in rheumatoid articular collagenous tissues. Arthritis Rheum. 1978;21:516-27. Some data, a speculative discussion, and up-to-date references on the possible role played by deposition of immune complexes in avascular connective tissue. CrossrefMedlineGoogle Scholar18. RUNGEPINALSLOURIETOMAR LRSR. Treatment of rheumatoid arthritis with levamisole: a controlled trial. Arthritis Rheum. 1977;20:1445-8. Careful study suggesting that this antihelminthic "immunostimulatory" drug has efficacy in rheumatoid arthritis. CrossrefMedlineGoogle Scholar19. STASTNY P. Association of the B-cell alloantigen DRw4 with rheumatoid arthritis. N Engl J Med. 1978;298:869-71. By serologic methods, B-lymphocytes from patients with rheumatoid arthritis were typed for alloantigens. HLA-DRw4 was found in 70% of patients and 28% of controls. This assay is easier and less expensive than determination of HLA-D antigens by mixed lymphocyte culture techniques. CrossrefMedlineGoogle Scholar20. TRENTHAMDYNESIUSROCKLINDAVID DRRJ. Cellular sensitivity to collagen in rheumatoid arthritis. N Engl J Med. 1978;299:327-32. Mononuclear cells from 35% to 40% of 50 patients with definite or classical rheumatoid arthritis produced (in vitro) leukocyte inhibitory factor in response to type II or III collagen, or both, but not type I. The hypothesis that in rheumatoid arthritis collagen may function as an autoantigen is reviewed. CrossrefMedlineGoogle Scholar21. WERBMAINARDIVATERHARRIS ZCCE. Endogenous activation of latent collagenase by rheumatoid synovial cells: evidence for a role of plasminogen activator. N Engl J Med. 1977;296:1017-23. Rheumatoid synovial cells in culture were shown to release latent collagenase that, although protected from forming complexes with α2-macroglobulin, could bind to substrate. Plasmin, produced by synovial cell plasminogen activator, was put forth as a probable in vivo activator of latent collagenase. CrossrefMedlineGoogle Scholar22. ZUBLERNYDEGGERPERRIN RUL. Circulating and intra-articular immune complexes in patients with rheumatoid arthritis; correlation of 125I-Clq binding activity with clinical and biological features of the disease. J Clin Invest. 1976;57:1308-19. A sensitive radiolabeled (125I) Clq binding assay was used to quantitate immune-complex-like material in synovial fluid and serum of patients with both seropositive and seronegative rheumatoid arthritis. See also reference 4. CrossrefMedlineGoogle Scholar23. BALDWINGLUCKLOWENSTEINGALLO DMJG. Lupus nephritis: clinical course as related to morphologic forms and their transitions. Am J Med. 1977;62:12-30. Study of 88 patients with lupus nephritis followed by biopsy that tends to emphasize the relation between histologic classification and clinical outcome and the benefits of early therapy. CrossrefMedlineGoogle Scholar24. DONADIOHOLLEYFERGUSONILSTRUP JKRD. Treatment of diffuse proliferative lupus nephritis with prednisone and combined prednisone and cyclophosphamide. N Engl J Med. 1978;299:1151-5. Four-year follow-up study comparing prednisone with prednisone plus cyclophosphamide. Although the rate of recurrence of lupus nephritis was higher in the steroid-only group, the proportion in each group who were alive with stable or improved renal function was similar. CrossrefMedlineGoogle Scholar25. FEINGLASSARNETTDORSCHZIZICSTEVENS EFCTM. Neuropsychiatric manifestations of systemic lupus erythematosus: diagnosis, clinical spectrum, and relationship to other features of the disease. Medicine (Baltimore). 1976;55:323-39. Study of 120 patients with systemic lupus erythematosus, of whom 37% had neuropsychiatric manifestations secondary to the disease. Although psychiatric illnesses were common, only two patients were believed to have steroid-induced psychosis. CrossrefMedlineGoogle Scholar26. LAMBERTDIXONZUBLER PFR. A WHO collaborative study for the evaluation of eighteen methods for detecting immune complexes in serum. J Clin Lab Immunol. 1978;1:1-15. The ability of certain tests to detect preferentially certain kinds of immune complexes associated with rheumatic diseases is apparent. CrossrefGoogle Scholar27. REINERTSENKLIPPELJOHNSON JJA. B-lymphocyte alloantigens associated with systemic lupus erythematosus. N Engl J Med. 1978;299:515-8. A study comparing the D-related B-cell alloantigens in patients with systemic lupus erythematosus and controls. Significant increases in DRw2 and DRw3 were observed in patients with the disease. CrossrefMedlineGoogle Scholar28. ROSSENHERSHSHARP REJ. Effect of plasma exchange on circulating immune complexes and antibody formation in patients treated with cyclophosphamide and prednisone. Am J Med. 1977;63:674-82. Careful study of the results of plasmapheresis in two patients, one with systemic lupus erythematosus and a second with a disease resembling Goodpasture's disease. CrossrefMedlineGoogle Scholar29. RUDDY S. Complement, rheumatic diseases and the major histocompatibility complex. Clin Rheum Dis. 1977;3:215-37. Review of deficiencies of complement proteins, their linkage to the major histocompatibility complex, and the ways in which they appear to predispose to rheumatic diseases including glomerulonephritis and systemic lupus erythematosus-like syndromes. CrossrefGoogle Scholar30. SHARPIRVINMAY GWC. Association of antibodies to ribonucleoprotein and Sm antigens with mixed connective-tissue disease, systemic lupus erythematosus and other rheumatic diseases. N Engl J Med. 1976;295:1149-54. The clinical and laboratory characteristics of patients with antibodies to two of the constituents of extractable nuclear antigen are defined. Antibodies to ribonuclear protein are associated with the overlap or mixed connective tissue disease syndrome and antibodies to the Sm antigens, with systemic lupus erythematosus. CrossrefMedlineGoogle Scholar31. TALBOTT J. Acute dermatomyositis-polymyositis and malignancy. Semin Arthritis Rheum. 1977;6:305-60. Thorough review of the clinical, laboratory, and histologic features of the acute or fulminant form of polymyositis with skin involvement. The association of malignancy with florid dermatomyositis that is rapidly progressive and relentless is emphasized. CrossrefMedlineGoogle Scholar32. URMANROTHFIELD JN. Corticosteroid treatment in systemic lupus erythematosus: survival studies. JAMA. 1977;238:2272-6. Documentation of improved survival in two large groups of patients, one treated between 1957 and 1968 and the second between 1968 and 1976. Improved survival may be related to better understanding of the disease, use of newer antibiotics, or use of serologic data as a guide to therapy. CrossrefMedlineGoogle Scholar33. ZVAIFLERBLUESTEIN NH. Lymphocytotoxic antibody activity in cryoprecipitates from serum of patients with SLE. Arthritis Rheum. 1976;19:844-50. Cytotoxic activity against normal human peripheral blood lymphocytes is selectively concentrated in cryoglobulins from the serum of patients with lupus erythematosus. Such lymphocytotoxic activity does not, however, appear to relate to clinical manifestations of the disease. CrossrefMedlineGoogle Scholar34. BUCKINGHAMPRINCERODNANTAYLOR RRGF. Increased collagen accumulation in dermal fibroblast cultures from patients with progressive systemic sclerosis (scleroderma). J Lab Clin Med. 1978;92:5-21. Confirmation of the enhanced synthesis of collagen by scleroderma skin cells and explanation of the discordance of previous studies by technical differences. MedlineGoogle Scholar35. BULKLEYRIDOLFISALYERHUTCHINS BRWG. Myocardial lesions of progressive systemic sclerosis: a cause of cardiac dysfunction. Circulation. 1976;53:483-90. Extensive review of myocardial lesions in scleroderma, concluding that, in the heart as elsewhere, fibrosis follows intermittent vascular occlusion. CrossrefMedlineGoogle Scholar36. CLEMENTSFURSTCAMPION PDD. Muscle disease in progressive systemic sclerosis: diagnostic and therapeutic considerations. Arthritis Rheum. 1978;21:62-71. Careful description of the several types of muscle disease in scleroderma, emphasizing that the commonest type is not responsive to glucocorticoids. CrossrefMedlineGoogle Scholar37. FLEISCHMAJERGAYMEIGELPERLISH RSWJ. Collagen in the cellular and fibrotic stages of scleroderma. Arthritis Rheum. 1978;21:418-28. Careful histologic description of early (cellular) and late (fibrotic) changes showing that collagen deposition is similar here to that of other sclerotic disorders, such as cirrhosis, with type III collagen deposited early and type I, late. CrossrefMedlineGoogle Scholar38. KONDORABINRODNAN HBG. Cutaneous antigen-stimulating lymphokine production by lymphocytes of patients with progressive systemic sclerosis (scleroderma). J Clin Invest. 1976;58:1388-94. The demonstration that peripheral blood mononuclear cells from scleroderma patients are sensitized to a skin antigen of unknown composition warrants re-examination of the role of cell-mediated immunity in scleroderma. CrossrefMedlineGoogle Scholar39. KOVALCHIKGUGGENHEIMSILVERMANROBERTSONSTEIGERWALD MSMJJ. The kidney in progressive systemic sclerosis: a prospective study. Ann Intern Med. 1978;89:881-7. The first indication that subclinical renal vascular lesions in scleroderma may be detected noninvasively, by renin response to a cold pressor test. LinkGoogle Scholar40. LAMRICANATIKHANKUSHNER MEMI. Reversal of severe renal failure in systemic sclerosis. Ann Intern Med. 1978;89:642-3. Unique case report is which the patient's renal function returned after 11 months of hemodialysis, suggesting that all renal scleroderma is not irreversible. LinkGoogle Scholar41. LAWLEYPECKMOUTSOPOULOSGRATWOHLDEISSEROTH TGHAA. Scleroderma, Sjögren-like syndrome, and chronic graft-versus-host disease. Ann Intern Med. 1977;87:707-9. Scleroderma-like changes in marrow-transplanted leukemia patients with chronic graft-versus-host disease add fuel to attempts to identify immunologically a prescleroderma state. LinkGoogle Scholar42. MARICQSPENCER-GREENLEROY HGE. Skin capillary abnormalities as indicators of organ involvement in scleroderma (systemic sclerosis), Raynaud's syndrome and dermatomyositis. Am J Med. 1976;61:862-70. First demonstration of microvascular (capillary) abnormalities in the same patients who have visceral involvement, supporting the use of capillary observations to predict visceral scleroderma. CrossrefMedlineGoogle Scholar43. RODNANLIPINSKILUKSICK GEJ. Skin thickness and collagen content in progressive systemic sclerosis and localized scleroderma. Arthritis Rheum. 1979. In press. A simple technique, the weight of a standard skin punch biopsy, might prove useful in objectively evaluating the role of therapies on the fibrotic features of scleroderma. CrossrefMedlineGoogle Scholar44. ROWELL N. The prognosis of systemic sclerosis. Br J Dermatol. 1976;95:57-60. Report of extensive experience with scleroderma highlighting the lack of distinguishing features between proposed subsets of the disease such as the CREST syndrome. CrossrefMedlineGoogle Scholar45. KASSANTHOMASMOUTSOPOULOS STH. Increased risk of lymphoma in sicca syndrome. Ann Intern Med. 1978;89:888-92. Report of seven cases of non-Hodgkin's lymphoma developing in 136 women followed up for biopsy-proven Sjögren's syndrome during a mean of 8.1 years. The incidence was 44 times the observed rate in an age- and sex-matched population. Parotid enlargement, sometimes radiation treated, appeared to predispose to the development of lymphoma. LinkGoogle Scholar46. MOUTSOPOULOSCHUSEDMANN HTD. Sjögren's syndrome (sicca syndrome); current issues. Ann Intern Med. 1980;92. In press. Thorough discussion on clinical, genetic, and immunologic features of Sjögren's syndrome. Differences between "primary" Sjögren's syndrome, an entity in itself, and "secondary" sicca syndrome, found to accompany conditions such as systemic lupus erythematosus and rheumatoid arthritis, are emphasized. Google Scholar47. OLSENNIELSEN NS. Prevalence of primary Raynaud phenomena in young females. Scand J Clin Lab Invest. 1978;38:761-4. Fifteen of 67 healthy young women were classified by questionnaire as having Raynaud's phenomenon. All eight studied showed digital artery closure or near closure on experimental exposure to local cold. A history of white or "dead" fingers on exposure was considered more pertinent to the diagnosis than frequent cold or bluish fingers. CrossrefMedlineGoogle Scholar48. SHARP G. Mixed connective tissue disease. Bull Rheum Dis. 1974-75;25:828-31. Concise description of the clinical and laboratory features of this syndrome, including the frequency of various clinical manifestations in 100 patients with high titers of antibody to ribonucleoprotein, then described as hemagglutinating antibodies to ribonuclease-sensitive extractable nuclear antigen. Polyarthritis or polyarthralgia, Raynaud's phenomenon, disordered esophageal function, decreased pulmonary diffusion, and swollen hands were each observed in at least two thirds of the patients. MedlineGoogle Scholar49. HOWELLMOSKOWITZ DR. Introduction: symposium on osteoarthritis: a brief review of research and investigations. Arthritis Rheum. 1977;20(suppl):S96-103. Detailed review of current concepts of pathology, pathogenesis, and research directions in osteoarthritis. Basic concepts of the disease process are well outlined. Google Scholar50. MOSKOWITZ R. Cartilage and osteoarthritis: current concepts. J Rheumatol. 1977;4:329-31. Editorial. Succinct but clear introduction to current knowledge relating to basic cartilage biochemistry and physiology. MedlineGoogle Scholar51. RESNICKSHAPIROWIESNERNIWAYAMAUTSINGERSHAUL DRKGPS. Diffuse idiopathic skeletal hyperostosis (DISH) (Ankylosing hyperostosis of Forestier and Rotes-Querol). Semin Arthritis Rheum. 1978;7:153-87. Classic review and redefinition of radiologic and clinical manifestations of ankylosing hyperostosis, now renamed diffuse idiopathic skeletal hyperostosis. Provides details of differential diagnosis of clinical syndromes that have to be differentiated. CrossrefMedlineGoogle Scholar52. SOLOMON L. Patterns of osteoarthritis of the hip. J Bone Joint Surg [Br.] 1976;58B:176-83. Excellent description of basic concepts of osteoarthritis introduced by its effects on one joint. Concepts of pathogenesis, particularly differentiating primary and secondary forms of the disease, are provided. CrossrefGoogle Scholar53. IBELSALFREYHUFFERWEIL LAWR. Aseptic necrosis of bone following renal transplantation: experience in 194 transplant recipients and review of the literature. Medicine (Baltimore). 1978;57:25-45. Excellent review of experience with aseptic necrosis in renal transplant patients. Pathogenic concepts of avascular necrosis in general are provided. CrossrefMedlineGoogle Scholar54. LINDEN B. Osteochondritis dissecans of the femoral condyles: a long-term follow-up study. J Bone Joint Surg. [Am]. 1977;59A:769-76. Careful, detailed study of patients with this disorder. The clinical features of the disease are well-outlined and concepts of diagnosis and complications reviewed on the basis of the author's extensive experience. CrossrefGoogle Scholar55. LOTKEECKERALAVI PMA. Painful knees in older patients: radionuclide diagnosis of possible osteonecrosis with spontaneous resolution. J Bone Joint Surg [Am]. 1977;59A:617-21. This paper, reporting a study of a not uncommon disorder, notes the importance of the clinical history and the use of bone scans in diagnosis. CrossrefGoogle Scholar56. WOODROW J. Histocompatibility antigens and rheumatic diseases. Semin Arthritis Rheum. 1977;6:257-76. Excellent review of histocompatibility antigens and rheumatic diseases that describes in detail the HLA system and outlines its relation to various spondyloarthropathies. CrossrefMedlineGoogle Scholar57. WRIGHT V. Seronegative polyarthritis: a unified concept. Arthritis Rheum. 1978;21:619-733. Clinical study of the interrelations of the various seronegative polyarthropathies by one of the leading investigators in this field. Unifying characteristics of the diseases and interrelations to the HLA system are given. CrossrefMedlineGoogle Scholar58. CALINPORTAFRIESSCHURMAN AJJD. Clinical history as a screening test for ankylosing spondylitis. JAMA. 1977;237:2613-4. This paper underscores the accuracy of the clinical history as a sensitive and specific screening test in the diagnosis and differentiation of back pain due to ankylosing spondylitis. CrossrefMedlineGoogle Scholar59. KHANKUSHNERBRAUN MIW. Comparison of clinical features in HLA-B27 positive and negative patients with ankylosing spondylitis. Arthritis Rheum. 1977;20:909-12. Study of clinical features of ankylosing spondylitis related to the presence of HLA-B27. Interesting concepts of the relation of anterior uveitis are suggested. CrossrefMedlineGoogle Scholar60. NAMEYMCINTYREBUSELEROY TJME. Nucleography studies of axial spondarthritides: I. Quantitative sacroiliac scintigraphy in early HLA-B27-associated sacroiliitis. Arthritis Rheum. 1977;20:1058-64. Sacroiliac scintigraphy when properly used is a sensitive indicator of early inflammatory disease of the sacroiliac joints. Detailed information on methodology and areas to be avoided are given. CrossrefMedlineGoogle Scholar61. RADFORDDOLLSMITH ERP. Mortality among patients with ankylosing spondylitis not given x-ray therapy. N Engl J Med. 1977;297:572-6. Provocative study of mortality rates in patients with ankylosing spondylitis. The disease may not be as benign as previously believed. CrossrefMedlineGoogle Scholar62. ROSEESPINOZAOSTERLAND ELC. Intestinal bypass arthritis: association with circulating immune complexes and HLA B27. J Rheumatol. 1977;4:129-34. Interesting study of the possible causal role of immune complexes in this interesting disorder, based on studies of cases seen by the authors. MedlineGoogle Scholar63. GILLIAMSMILEY JJ. Cutaneous necrotizing vasculitis and related disorders. Ann Allergy. 1976;37:328-39. Illuminating review of the vasculitides, with particular emphasis on the distinctive features to be noted in the skin. A useful discussion of classification is accompanied by a diagram showing the type and depth in the skin of the particular vessels involved. MedlineGoogle Scholar64. COHENHIRSCHHORNHOROWITZ ARS. Deoxyadenosine triphosphate as a potentially toxic metabolite in adenosine deaminase deficiency. Proc Natl Acad Sci USA. 1978;75:472-6. Demonstration that patients with adenosine deaminase deficiency accumulate deoxyadenosine triphosphate in erythrocytes, providing an important clue to the cause of the immunodeficiency characteristic of this disease. CrossrefMedlineGoogle Scholar65. DIEPPECROCKERHUSKISSONWILLOUGHBY PPED. Apatite deposition disease: a new arthropathy. Lancet. 1976;1:266-8. The original description of hydroxyapatite deposition disease in six patients with an osteoarthritis-like syndrome. CrossrefMedlineGoogle Scholar66. EDITORIAL. Reflex sympathetic dystrophy syndrome. Lancet. 1976:1:1226-7. Brief but comprehensive review of the cause, pathogenesis, evaluation, and treatment of the reflex sympathetic dystrophy syndrome. MedlineGoogle Scholar67. FOX I. Hypouricaemic agents in the treatment of gout. Clin Rheum Dis. 1977;3:145-58. Summary of current recommendations on use of drugs to control hyperuricemia. CrossrefGoogle Scholar68. GILBERT M. Musculoskeletal manifestations of hemophilia. Mt Sinai J Med NY. 1977;44:339-58. A review of the pathophysiology and management of hemarthrosis in this disease. MedlineGoogle Scholar69. GRACCHIOLONAMEROWCAMPIONBLUESTONE ANDR. Peripheral nerve entrapments. West J Med. 1977;127:299-313. Good review of peripheral nerve entrapment syndromes, including pathophysiology, electrodiagnosis, pathogenesis, and management. MedlineGoogle Scholar70. GRAYGOTTLIEB RN. Rheumatic disorders associated with diabetes mellitus: literature review. Semin Arthritis Rheum. 1976;6:19-34. Extensive assessment of causes of musculoskeletal disorders in patients with diabetes mellitus. CrossrefMedlineGoogle Scholar71. HOLMES E. Pathogenesis of hyperuricaemia in primary gout. Clin Rheum Dis. 1977;3:3-23. Complete review of the causes of hyperuricemia including recent advances in our understanding of regulation of purine bio synthesis de novo. CrossrefGoogle Scholar72. MCCARTY D. Calcium pyrophosphate dihydrate crystal deposition disease (pseudogout syndrome)—clinical aspects. Clin Rheum Dis. 1977;3:61-89. Review of the author's total experience with the disease. CrossrefGoogle Scholar73. MITCHELLMEJIASDADDONAKELLEY BEPW. Purinogenic immunodeficiency diseases: selective toxicity of deoxyribonucleosides for T cells. Proc Natl Acad Sci USA. 1978;75:5011-4. First evidence that the accumulation of adenosine triphosphate and guanosine triphosphate could account for the nearly selective loss of T cells in patients with adenosine deaminase deficiency and purine nucleoside phosphorylase deficiency, respectively. CrossrefMedlineGoogle Scholar74. M'SEFFARFORNASIERFOX AVI. Arthropathy as the major clinical indicator of occult iron storage disease. JAMA. 1977;238:1825-8. Emphasizes diagnosis of hemochromatosis arthritis based on the characteristics of the arthritis in patients without other clinical findings suggestive of excessive iron storage. CrossrefMedlineGoogle Scholar75. O'DUFFY J. Clinical studies of acute pseudogout attacks: comments on prevalence, predispositions and treatment. Arthritis Rheum. 1976;19:349-52. Review of the Mayo Clinic experience with calcium pyrophosphate deposition disease, emphasizing the possible role of a sudden decrease in serum calcium in the precipitation of the acute attack. CrossrefMedlineGoogle Scholar76. RAVIDROBSONKEDAR MMI. Prolonged colchicine treatment in four patients with amyloidosis. Ann Intern Med. 1977;87:568-70. Demonstration of improvement with colchicine treatment in the nephrotic syndrome in three patients with familial Mediterranean fever and in renal function in one patient with primary amyloidosis. LinkGoogle Scholar77. SCHEINBERG M. Immunology of amyloid disease: a review. Semin Arthritis Rheum. 1977;7:133-40. Review of amyloid disease with special reference to immunochemistry, the amyloid-related serum component, cellular immunology, and experimental models. CrossrefMedlineGoogle Scholar78. SCHUMACHER H. Articular manifestations of hypertrophic pulmonary osteoarthropathy in bronchogenic carcinoma. Arthritis Rheum. 1976;19:629-36. Description of clinical pattern of arthritis in eight patients with bronchogenic carcinoma who presented with painful joint effusions. Electron-dense deposits in synovial vessel walls suggested circulating materials in pathogenesis. CrossrefMedlineGoogle Scholar79. SCHUMACHERDORWARTBONDALAVIMILLER HBJAW. Chronic synovitis with early cartilage destruction in sickle cell disease. Ann Rheum Dis. 1977;36:413-9. Report of two cases of destructive arthritis in patients with sickle cell disease, with review of causes of musculoskeletal symptoms in these patients. CrossrefMedlineGoogle Scholar80. BAYERCHOWLOUIEGUZE AAJL. Sternoarticular pyoarthrosis due to gram-negative bacilli. Arch Intern Med. 1977;137:1036-40. Infectious arthritis in this unusual site was found to be due to Pseudomonas aeruginosa in six of eight patients. All cases occurred in intravenous heroin abusers. CrossrefMedlineGoogle Scholar81. DONOVANCHAPMANHARRINGTONNAGEL TMKD. Serratia arthritis: report of seven cases. J Bone Joint Surg [Am]. 1976;58A:1009-11. The frequency of Serratia marcescens septic arthritis is increasing, especially in the drug-addict population. Aggressive treatment with combination antibiotic agents and surgical débridement is recommended. CrossrefGoogle Scholar82. GOLDENBERGCOHEN DA. Acute infectious arthritis: a review of patients with non-gonococcal joint infections (with emphasis on therapy and prognosis). Am J Med. 1976;60:369-77. Review of the clinical course of a large series of patients with acute nongonococcal septic arthritis, emphasizing the increasing prevalence of gram-negative infections. Rapid initiation of treatment is vital. CrossrefMedlineGoogle Scholar83. HANDSFIELDWIESNERHOLMES HPK. Treatment of the gonococcal arthritic-dermatitis syndrome. Ann Intern Med. 1976;84:661-7. High-dose intravenous penicillin G followed by oral ampicillin, or oral ampicillin alone, is the preferred treatment for uncomplicated disseminated gonococcal infection. Purulent synovial effusions require inpatient hospital evaluation and therapy. LinkGoogle Scholar84. HOTICEKAPLAN GAS. Septic bursitis in the prepatellar and olecranon bursae: an analysis of 25 cases. Ann Intern Med. 1978;89:21-7. Septic bursitis of the prepatellar and olecranon areas is primarily due to penicillin-resistant Staphylococcus aureus, appears to be traumatically induced, and responds readily to appropriate antibiotics and drainage. LinkGoogle Scholar85. MITCHELLBROOKESTEVENSONBUCHANAN WPRW. Septic arthritis in patients with rheumatoid disease: a still underdiagnosed complication. J Rheumatol. 1976;3:124-33. Superimposed septic arthritis is a potentially devastating but poorly recognized complication of rheumatoid arthritis. Many factors that encourage susceptibility to infection in rheumatoid patients are reviewed. MedlineGoogle Scholar86. NEWMAN J. Review of septic arthritis throughout the antibiotic era. Ann Rheum Dis. 1976;35:198-205. Review of a 30-year experience with septic arthritis showed little change in incidence during the past 20 years or in distribution of organisms. Poor prognosis relates to delay in diagnosis. CrossrefMedlineGoogle Scholar87. STEEREMALAWISTAHARDINRUDDYASKENASEANDIMAN ASJSPW. Erythema chronicum migrans and Lyme arthritis: the enlarging clinical spectrum. Ann Intern Med. 1977;86:685-98. Description of a new rheumatic syndrome that includes a specific rash, inflammatory arthritis, systemic symptoms, and occasionally neurologic abnormalities or myocardial conduction abnormalities. An arthropod-transmitted virus infection is suspected as the cause. LinkGoogle Scholar88. HAHNHAHN TB. Osteopenia in patients with rheumatic diseases: principles of diagnosis and therapy. Semin Arthritis Rheum. 1976;6:165-88. Classic review article on diagnosis, pathogenesis, and current concepts of therapy of the various disorders included in this area of study. The paper is clearly written and exhaustive without being tedious, and should be read by anyone interested in the field. CrossrefMedlineGoogle Scholar89. KHAIRIALTMANDEROSAZIMMERMANSCHENKJOHNSTON MRGJRC. Sodium etidronate in the treatment of Paget's disease of bone: a study of long-term results. Ann Intern Med. 1977;87:656-63. Excellent study of the use of a promising therapeutic agent in management of symptomatic Paget's disease that includes guidelines for diagnosis, follow-up, and therapy. LinkGoogle Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: MEDICAL KNOWLEDGE SELF-ASSESSMENT PROGRAM V AMERICAN COLLEGE OF PHYSICIANS Philadelphia, Pennsylvania*Edward D. Harris, Jr. M.D., Chairman; John L. Decker, M.D.; William N. Kelley, M.D.; E. Carwile LeRoy, MD.; Roland W. Moskowitz, M.D.; Allen R. Myers, M.D.; and Shaun Ruddy, M.D.†Computer literature-file of the National Library of Medicine, Bethesda, Maryland, consulted through the Regional Medical Library service Library of the College of Physicians of Philadelphia, with the technical assistance of Ruthanne Henner. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics 1 December 1979Volume 91, Issue 6Page: 932-937KeywordsComputersLibrariesRheumatology Issue Published: 1 December 1979 PDF downloadLoading ...