tbuckley/cabot-search huggingface dataset:
RISK OF THROMBOEMBOLIC EVENTS IN PATIENTS WITH HIGH CHA2DS2-VASC SCORE UNDERGOING CATHETER ABLATION OF NON-PAROXYSMAL ATRIAL FIBRILLATION WITH/WITHOUT LEFT ATRIAL APPENDAGE ELECTRICAL ISOLATION: A PROPENSITY SCORE MATCHED COMPARISON
Authors: Domenico Giovanni Della Rocca, Chintan Trivedi, Luigi Di Biase, Sanghamitra Mohanty, Qiong Chen, Omer Gedikli, Alfredo Chauca Tapia, Amin Al-Ahmad, John Burkhardt, Gerald Gallinghouse, Javier Sanchez, Rodney Horton, Sharan Sharma, Dhanunjaya Lakkireddy, Andrea Natale
Journal: Journal of the American College of Cardiology (2019)
POINT-OF-CARE SALT-SENSITIVITY TESTING IN POST-MENOPAUSAL WOMEN TO IDENTIFY RISK FOR HEART FAILURE WITH PRESERVED EJECTION FRACTION
Authors: Scott L. Hummel, Carrie A. Karvonen-Gutierrez, Michelle M. Hood, Ana Baylin, Elizabeth Jackson, Sioban D. Harlow
Journal: Journal of the American College of Cardiology (2019)
Sodium-Glucose Cotransporter 2 Inhibitors and Amputations
Authors: Jennifer N. Clements
Journal: JAMA Internal Medicine (2019)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA Internal Medicine HomeNew OnlineCurrent IssueFor Authors Podcast Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA Internal Medicine journal
Axl kinase drives immune checkpoint and chemokine signalling pathways in lung adenocarcinomas
Authors: Yoko Tsukita, Naoya Fujino, Eisaku Miyauchi, Ryoko Saito, Fumiyoshi Fujishima, Koji Itakura, Yorihiko Kyogoku, Koji Okutomo, Mitsuhiro Yamada, Tatsuma Okazaki, Hisatoshi Sugiura, Akira Inoue, Yoshinori Okada, Masakazu Ichinose
Journal: Molecular Cancer (2019)

Axl receptor tyrosine kinase is involved in the growth and metastasis and is an indicator of poor prognosis in several cancers including lung cancers. Although a mitogen-activated protein kinase (MAPK) pathway and an epithelial-to-mesenchymal transition (EMT) program are critical, molecular mechanisms underlying the Axl-driven cancer progression have not been fully elucidated. We aimed to identify molecules up-regulated by Axl kinase in lung adenocarcinomas. Through the global gene expression analysis and the functional annotation clustering, we found that AXL expression positively correlated with mRNA expressions of immune checkpoint molecules and chemokine receptors in non-small-cell lung cancers. Validation cohorts including our biobank confirmed that the AXL expression significantly correlated with expression of genes encoding programmed death-ligand1 (PD-L1) and CXC chemokine receptor 6 (CXCR6) in lung adenocarcinoma, especially in epidermal growth factor receptor (EGFR) mutation-positive adenocarcinoma. Pharmacological inhibition of Axl kinase activity decreased mRNA expressions of PD-L1 and CXCR6 in EGFR mutation-positive cell lines. Our data indicates the novel role of Axl kinase as a driver of immune checkpoint molecules and chemokine signalling pathways in the progression of lung adenocarcinomas. This study also highlights the necessity of clinical trials in order to test the efficacy of Axl kinase inhibition in the Axl-highly expressing subset of lung adenocarcinomas.
ADVERSE EVENTS AND MODES OF FAILURE RELATED TO THE IMPELLA PERCUTANEOUS LEFT VENTRICULAR ASSIST DEVICES: A RETROSPECTIVE ANALYSIS OF THE MAUDE DATABASE
Authors: Nauman Khalid, Toby Rogers, Evan Shlofmitz, Yuefeng Chen, Jaffar Khan, Anees Musallam, Micaela Iantorno, Ron Waksman
Journal: Journal of the American College of Cardiology (2019)
THE ASSOCIATION OF SLEEP APNEA AND EXERCISE CAPACITY WITH LONG-TERM MAJOR CARDIOVASCULAR EVENTS
Authors: Maria Irene Barillas Lara, Jose Medina-Inojosa, Bhanuprakash Kolla, Joshua Smith, Amanda Bonikowske, Thomas Allison, Thomas Olson, Francisco Lopez-Jimenez, Virend Somers, Sean Caples, Meghna P. Mansukhani
Journal: Journal of the American College of Cardiology (2019)
PREDICTORS OF APPROPRIATE IMPLANTABLE CARDIOVERTER DEFIBRILLATOR THERAPY IN PATIENTS WITH ARRHYTHMOGENIC RIGHT VENTRICULAR DYSPLASIA/CARDIOMYOPATHY
Authors: Sajya Singh, Aaron Thomas, Sue Casey, Katelyn Storey, Miranda Kunz, Allison Berg, William Katsiyiannis, Raed Abdelhadi, Mosi Bennett, Jay Sengupta
Journal: Journal of the American College of Cardiology (2019)
Abstract 497: Rtf1 Regulates Cardiac Development and Function
Authors: Jaunian Chen, Adam D Langenbacher, Fei Lu, Vincent Ren, Yibin Wang, Chen Gao
Journal: Circulation Research (2018)

Highly regulated transcriptional control of cardiac gene networks is critical for heart development and function. Rtf1 is a critical regulator of multiple transcription processes including elongation and co-transcriptional histone modification. Rtf1 ablation in zebrafish and mice eliminates the cardiac progenitor population, producing embryos without hearts, and knockdown of Rtf1 in neonatal rat ventricular myocytes decreases the expression of sarcomere component genes, suggesting that Rtf1 is indispensable for both establishing the myocardial lineage and maintaining normal physiological homeostasis in differentiated cardiomyocytes. To explore the function of Rtf1 in the adult heart, we generated an inducible knockout of Rtf1 in the adult myocardium. Loss of Rtf1 function in adult cardiomyocytes results in left ventricular systolic dysfunction and pathological features of heart failure. The Rtf1 deficient hearts exhibit a gene expression profile characteristic of heart failure, and display disrupted myofibrils and cell-cell junctions. Intriguingly, and in line with Rtf1’s established role in promoting histone H2B ubiquitination, diminished H2BK120 ubiquitination was also observed in Rtf1 deficient adult hearts. Collectively, our findings show that Rtf1 and Rtf1 mediated H2BK120 ubiquitination are essential epigenetic regulators of both myocardial lineage determination during development as well as maintaining cellular homeostasis, integrity and function in mature cardiomyocytes.
Abstract 20994: Sedentary Behavior Independently Predicts Mortality in the Pacemaker Population
Authors: Katie E Cohen, Sudhi Tyagi, Ridhima Kapoor, Marcie Berger, Judy Fox, Scott Strath, James Roth, James Oujiri, Jason Rubenstein, Nicholas Kluge, Michael Widlansky
Journal: Circulation (2017)

Introduction: Embedded accelerometers offer a novel method of measuring physical activity levels in the pacemaker population. In an initial cohort of 96 subjects with pacemakers, we previously showed sedentary behavior significantly correlates with decreased survival. We hypothesize that physical activity of &lt; 2 hours/day correlates with mortality independently of the traditional cardiovascular risk factors in an expanded cohort. Methods: Records of 273 individuals with de novo Medtronic pacemaker implantation at a single center between January 1, 2005 and December 31, 2012 were reviewed. An accelerometer embedded in these devices measures activity level as above or below a 70 steps/minute threshold and reports the average hours/day of activity exceeding this threshold. Activity levels abstracted from pacemaker interrogations were averaged over a 1 year period following a 6-month blanking period after implantation. Among this cohort, 48.7% were categorized as having failed to reach the activity threshold for ≥2 hours/day (n=140). Survival was compared between groups by Kaplan-Meier estimate and Cox proportional hazards models. Multivariable logistic regression examined predictors of survival. Results: The average age of the study cohort was 71±13 years with CAD prevalent in 34.1% of subjects. Average follow-up time was 3.67±2.43 years. Death occurred in 40 subjects (14.7%). In unadjusted analysis, death was nearly 5 times more likely in the inactive group than the active group [OR 4.81, 95% CI (2.13-10.88), P&lt;0.001]. A multivariate model revealed that &lt; 2 hours of daily activity remained an independent predictor of death [OR 4.39, 95% CI (1.90-10.12), P=0.001] along with age [OR 1.04, 95% CI (1.01-1.08), P=0.02] while sex [OR 0.09, P=0.74] and CAD [OR 0.54, P=0.08] were not predictors. Conclusion: Sedentary behavior as measured by an implanted pacemaker correlates with increased mortality independently of traditional cardiovascular risk factors.
ADRENOMEDULLIN ACTIVITY AS AN ALTERNATE MECHANISM OF ACTION IN ARNI THERAPY
Authors: Henrike Arfsten, Georg Goliasch, Philipp Bartko, Anna Cho, Johannes Novak, Helmuth Haslacher, Guido Strunk, Joachim Struck, Martin Hülsmann, Noemi Pavo
Journal: Journal of the American College of Cardiology (2019)
A PROPENSITY MATCHED ANALYSIS OF INVASIVE VERSUS CONSERVATIVE MANAGEMENT OF ELDERLY PATIENTS WITH NON-ST ELEVATION MYOCARDIAL INFARCTION (SENIOR-NSTEMI STUDY)
Authors: Amit Kaura, Abdulrahim Mulla, Vasileios Panoulas, Glampson Benjamin, Jim Davies, Kerrie Woods, Joe Omigie, Anoop D. Shah, Keith Channon, Jonathan N. Weber, Mark R. Thursz, Paul Elliott, Harry Hemingway, Bryan Williams, Folkert W. Asselbergs, Michael O'Sullivan, Rajesh Kharbanda, Graham M. Lord, Narbeh Melikian, Riyaz Patel, Divaka Perera, Ajay Shah, Darrel Francis, Jamil Mayet
Journal: Journal of the American College of Cardiology (2019)
Adiponectin has anti-malignant potential in hepatocellular carcinoma (HCC) by activation of AMPK in HepG2 cells
Authors: Neeraj Saxena, Songbai Lin, Xiaokun Ding, Dipali Sharma, Frank Anania
Journal: Cancer Research (2007)

2049  Hepatocellular carcinoma (HCC) has a high relative-risk association with obesity. Recent studies have established that obesity is an endocrine disorder mediated by hormones secreted from white adipose tissue (WAT). Adiponectin, an adipocytokine secreted by WAT, proposed to have therapeutic potential, is inversely associated with obesity. Importantly, low levels of serum adiponectin are associated with higher relative-risk of HCC. The Purpose of the present study was to elucidate the potential mechanism whereby adiponectin negatively modulates the growth of HCC. Methods: The HCC cell line HepG2 was used for these experiments. Both mRNA transcripts and protein expression of both adiponectin receptors, AdipoR1 and AdipoR2, were detected by RT-PCR and immunoblot analysis, respectively. Adiponectin dose and time studies were conducted to determine cell viability by XTT-reduction and cell proliferation by BrdU incorporation. Metastatic properties, including the effect of adiponectin on cell migration, were determined, in part, by Electric Cell-substrate Impedance Sensing (ECIS). Adiponectin signal-transduction was assessed by immunoblot assay for phosphorylated 5’-AMP-activated protein kinase (AMPK), AMPK, PCNA, Cyclin D1, p21, Caspase-3 and cleaved caspase-3. Results: AdipoR1 and AdipoR2 transcripts and respective proteins were identified in HepG2 cells. Optimization from dose and time course studies revealed that either globular or full-length adiponectin treatment [10µg/ml] reduced HepG2 cell proliferation by 50% and increased apoptosis by 6-fold as compared to serum-free (SF) treatment (p
ADULTS AND ADOLESCENTS WITH CONGENITAL HEART DISEASE: WHO ARE WE LOSING TO FOLLOW UP?
Authors: J.D. Serfas, Toi Spates, Alfred D'Ottavio, Tracy Spears, Elizabeth Ciociola, Karen Chiswell, Manuela Wiedemeier, Richard Krasuski, Alex R. Kemper, Timothy M. Hoffman, Michael Walsh, Charlie Sang, Karl F. Welke, Jennifer Li
Journal: Journal of the American College of Cardiology (2019)
COMPARISON OF BLEEDING RISK PREDICTION MODELS WITH DUAL ANTI-PLATELET THERAPY TREATMENT AMONG PATIENTS WITH ACUTE CORONARY SYNDROMES TREATED MEDICALLY WITHOUT REVASCULARIZATION
Authors: Guillaume Marquis-Gravel, Megan Neely, Marco Valgimigli, Francesco Costa, David Van Klaveren, Deepak L. Bhatt, Rituparna Altner, Paul Armstrong, Keith Fox, Harvey White, E. Magnus Ohman, Matthew Roe
Journal: Journal of the American College of Cardiology (2019)
Dapagliflozin and Cardiovascular Outcomes in Patients With Type 2 Diabetes Mellitus and Previous Myocardial Infarction
Authors: Remo H.M. Furtado, Marc P. Bonaca, Itamar Raz, Thomas A. Zelniker, Ofri Mosenzon, Avivit Cahn, Julia Kuder, Sabina A. Murphy, Deepak L. Bhatt, Lawrence A. Leiter, Darren K. McGuire, John P.H. Wilding, Christian T. Ruff, Jose C. Nicolau, Ingrid A.M. Gause-Nilsson, Martin Fredriksson, Anna Maria Langkilde, Marc S. Sabatine, Stephen D. Wiviott
Journal: Circulation (2019)

Background: Sodium glucose transporter-2 inhibitors reduce the risk of major adverse cardiovascular events (MACE) in patients with type 2 diabetes mellitus and a history of atherosclerotic cardiovascular disease. Because of their baseline risk, patients with previous myocardial infarction (MI) may derive even greater benefit from sodium glucose transporter-2 inhibitor therapy. Methods: DECLARE-TIMI 58 (Dapagliflozin Effect on Cardiovascular Events–Thrombolysis in Myocardial Infarction 58) randomized 17 160 patients with type 2 diabetes mellitus and either established atherosclerotic cardiovascular disease (n=6974) or multiple risk factors (n=10 186) to dapagliflozin versus placebo. The 2 primary end points were composite of MACE (cardiovascular death, MI, or ischemic stroke) and the composite of cardiovascular death or hospitalization for heart failure. Those with previous MI (n=3584) made up a prespecified subgroup of interest. Results: In patients with previous MI (n=3584), dapagliflozin reduced the relative risk of MACE by 16% and the absolute risk by 2.6% (15.2% versus 17.8%; hazard ratio [HR], 0.84; 95% CI, 0.72–0.99; P =0.039), whereas there was no effect in patients without previous MI (7.1% versus 7.1%; HR, 1.00; 95% CI, 0.88–1.13; P =0.97; P for interaction for relative difference=0.11; P for interaction for absolute risk difference=0.048), including in patients with established atherosclerotic cardiovascular disease but no history of MI (12.6% versus 12.8%; HR, 0.98; 95% CI, 0.81–1.19). There seemed to be a greater benefit for MACE within 2 years after the last acute event ( P for interaction trend=0.007). The relative risk reductions in cardiovascular death/hospitalization for heart failure were more similar, but the absolute risk reductions tended to be greater: 1.9% (8.6% versus 10.5%; HR, 0.81; 95% CI, 0.65–1.00; P =0.046) and 0.6% (3.9% versus 4.5%; HR, 0.85; 95% CI, 0.72–1.00; P =0.055) in patients with and without previous MI, respectively ( P interaction for relative difference=0.69; P interaction for absolute risk difference=0.010). Conclusions: Patients with type 2 diabetes mellitus and previous MI are at high risk of MACE and cardiovascular death/hospitalization for heart failure. Dapagliflozin appears to robustly reduce the risk of both composite outcomes in these patients. Future studies should aim to confirm the large clinical benefits with sodium glucose transporter-2 inhibitors we observed in patients with previous MI. Clinical Trial Registration: URL: https://www.clinicaltrials.gov . Unique identifier: NCT01730534.
Herd protection or herding cats?
Authors: Mark van der Linden, Cynthia G Whitney
Journal: Thorax (2019)

In this issue of Thorax , Hanquet et al 1 present results of surveillance for invasive pneumococcal disease (IPD) among adults aged 65 years and older in 10 European countries during the 5 years 2011–2015 following introduction of second-generation pneumococcal conjugate vaccines for infants that covered 10 (PCV10) or 13 (PCV13) of the more than 90 pneumococcal serotypes. They also compared the incidence rates to the pre-PCV10/13 year 2009. A total of 56 194 cases of disease are reported. Since the authors are quantifying the effects of PCVs rolled out as part of infant immunisation programmes throughout Europe around 2010, the trends described are entirely indirect, or herd, effects. In other words, the authors describe how well pneumococcal conjugate vaccine programmes are interrupting pneumococcal transmission from children, and therefore, preventing disease in vulnerable older adults. The key questions the authors address are whether herd protection effects differ depending on the vaccine used in childhood immunisation programmes (PCV10 or PCV13) and whether vaccination of the elderly themselves with PCV13 has value, considering the reduction in circulating vaccine-serotype pneumococci and herd protection.

Given the high number of pneumococcal serotypes that have been described, trying to prevent overall disease using vaccines that target a relatively small number of serotypes could end up being like herding cats; disease caused by some serotypes will move in the right direction, and disease caused by others may not. Most countries …
SAFETY OF CARVEDILOL IN MANAGEMENT OF HEART FAILURE AMONG COCAINE USERS
Authors: Raza M. Alvi, Dahlia Banerji, Noor Tariq, Malek Z.O. Hassan, Magid Awadalla, Lili Zhang, Connor P. Mulligan, Adam Rokicki, Maryam Afshar, Tomas G. Neilan
Journal: Journal of the American College of Cardiology (2019)
ADOPTION OF THE CORONARY ARTERY DISEASE: REPORTING AND DATA SYSTEM™ RESULTS IN HIGHER RATES OF APPROPRIATE ASPIRIN AND STATIN INITIATION REGARDLESS OF ORDERING PROVIDER
Authors: Robert Hull, Jeremy Berger, Joshua Boster, Michael Williams, Alec Sharp, Emilio Fentanes, Christopher Maroules, Ricardo Cury, Dustin Thomas
Journal: Journal of the American College of Cardiology (2019)
TRENDS AND OUTCOMES OF ECMO SUPPORT IN CARDIOGENIC SHOCK DUE TO ACUTE MYOCARDIAL INFARCTION: ANALYSIS OF NATIONWIDE INPATIENT SAMPLE
Authors: Moghniuddin Mohammed, Mohamed Elkhashab, Venkat Lakshmi Kishan Vuddanda, Juwairiya Arshi, Nicholas Haglund, Travis Abicht, Andrew Sauer, Zubair Shah
Journal: Journal of the American College of Cardiology (2019)
British Association of Dermatologists' guidelines for the management of Stevens–Johnson syndrome/toxic epidermal necrolysis in children and young people, 2018
Authors: T. McPherson, L.S. Exton, S. Biswas, D. Creamer, P. Dziewulski, L. Newell, K.L. Tabor, G.N. Wali, G. Walker, R. Walker, S. Walker, A.E. Young, M.F. Mohd Mustapa, R. Murphy, N.J. Levell, P.M. McHenry, T.A. Leslie, S. Wakelin, R.Y.P. Hunasehally, M. Cork, G.A. Johnston, N. Chiang, F.S. Worsnop, P. Rakvit, A. Salim, B. McDonald, S.L. Chua, D. Buckley, G. Petrof, F. Hussain, A. Bardhan, N. Callachand, T. Flavell, A.A. Salad
Journal: British Journal of Dermatology (2019)

Linked Comment:Lee. Br J Dermatol 2019; 181:10–11. Plain language summary available online
Abstract 443: α <sub>1</sub> -adrenergic Receptor Agonists Activate Akt Signalling via the Insulin Receptor Family of Receptors
Authors: Angela Clerk, Daniel N Meijles, Kerry A Rostron, Spencer Shaw, Joshua J Cull, Stephen J Fuller, Peter H Sugden
Journal: Circulation Research (2018)

Introduction: Akt signalling was delineated in the context of insulin stimulation and is cardioprotective. Akt is activated by insulin or insulin-like growth factor 1 (IGF1), acting through their receptors (IRs, IGF1Rs). Other receptors including α 1 -adrenergic receptors (α 1 ARs) activate Akt, but the mechanism is unclear. Hypothesis: We hypothesise that α 1 -ARs activate Akt via insulin receptor family receptors (InsRFs). Our aims were to establish if this is so, and if linsitinib [a highly selective inhibitor of InsRFs developed for cancer] affects cardiac responses to α 1 -AR agonists. Methods: Rat neonatal cardiomyocytes were exposed to or adult rat hearts perfused with α 1 AR agonists [50 nM A61603; 100 μM phenylephrine (PE)] with/without linsitinib. Effects on signalling were assessed by immunoblotting. Protein synthesis was assessed using 3 H-Phe. Effects of linsitinib (2 mg/kg/d) on cardiac function/hypertrophy induced by PE (40 mg/kg/d, 4 d) in mice (male C57Bl6; 10 wks) in vivo were determined. Echocardiography was used to assess cardiac function and pulmonary/aortic flow. Results: We identified a third InsRF in heart, the insulin receptor-related receptor (IRR). IRRs are alkali-sensitive and required for pH regulation in kidney. Alkaline pH (pH 9.0) activated IRRs and Akt, and increased protein synthesis in perfused hearts, being activated in a complex with IR/IGF1Rs. α 1 ARs activated Akt in cardiomyocytes and perfused hearts and increased protein synthesis. Linsitinib inhibited activation of Akt and the increase in protein synthesis induced by alkaline pH or α 1 AR agonists. Thus, α 1 ARs signal via InsRFs to Akt and protein synthesis. In perfused hearts, linsitinib enhanced the increase in developed pressure induced by A61603 and, in vivo, linsitinib enhanced PE-induced left ventricular (LV) hypertrophy with increased LV wall thickness and decreased internal diameter. Aortic flow was compromised in the presence of PE/linsitinib. Conclusions: Our data demonstrate a novel signalling paradigm in which α 1 -ARs signal through one or more InsRFs in cardiomyocytes to activate Akt and increase protein synthesis. However, functionally, the InsRF signal appears to mitigate some of the hypertrophic effects of α 1 -AR signalling in the whole heart.
The Importance of Prenatal Diagnosis By Chorionic Villous Sampling in Detecting and Preventing Beta Haemoglobinopathies in a Resource Poor Setting: A Report from Eastern Part of India
Authors: Ankita Sen, Rajib De, Tuphan Kanti Dolai, Prakas Kumar Mandal, Shyamali Dutta, Shuvra Neel Baul, Prantar Chakrabarti
Journal: Blood (2018)
THE PREDICTIVE VALUE OF HEMOCONCENTRATION AND HEMOGLOBIN LEVEL DURING THE TREATMENT OF ACUTE DECOMPENSATED HEART FAILURE
Authors: Hiroyuki Kono, Takeshi Kitai, Kitae Kim, Natsuhiko Ehara, Atsushi Kobori, Makoto Kinoshita, Shuichiro Kaji, Yutaka Furukawa
Journal: Journal of the American College of Cardiology (2019)
LONG-TERM FOLLOW-UP OF REDUCED EJECTION FRACTION HEART FAILURE PATIENTS RECEIVING AUTONOMIC REGULATION THERAPY IN THE ANTHEM-HF PILOT STUDY
Authors: Rajendra K. Premchand, Kamal Sharma, Sanjay Mittal, Rufino Monteiro, Imad Libbus, Lorenzo DiCarlo, Jeffrey Ardell, Badri Amurthur, Bruce Kenknight, Inderjit Anand
Journal: Journal of the American College of Cardiology (2019)
PROGNOSTIC IMPACT OF PRIOR RADIATION EXPOSURE IN CANCER SURVIVORS WITH SEVERE AORTIC STENOSIS UNDERGOING TRANSCATHETER AORTIC VALVE REPLACEMENT
Authors: Nikhil Agrawal, Sharma Kattel, Vasvi Singh, Ankita Kapoor, Ashutosh Sharma, Vijay Iyer, Umesh Sharma
Journal: Journal of the American College of Cardiology (2019)