tbuckley/cabot-search huggingface dataset:
Abstract 330: Systemic Blocking Exosome Formation/Release Improves Ischemic Hindlimb Repair in Diabetic db/db Mice
Authors: Zhongjian Cheng, Venkata Naga Srikanth Garikipati, Maria Cimini, Yan Tang, Chunlin Wang, May Trungcao, Yujia Ye, Cindy Benedict, Vandana Mallaredy, Walter Koch, Suresh K Verma, David Goukassian, Raj Kishore
Journal: Circulation Research (2018)

Background: Recent studies demonstrated that plasma exosomes from diabetic patients and animals lose their ability to maintain physiological properties of cardiomyocytes. Exosome inhibitor GW 4869 (GW) tended to improve heart function in diabetic mice with myocardial infarction. We tested the hypotheses that systemic blocking exosome synthesis/formation improves ischemic hindlimb repair in diabetes. Methods and Results: Unilateral hindlimb ischemia (HLI) surgery was conducted by ligation of left femoral artery in 12-week male db/db and db/+ mice which were administrated with either GW (2 mg/kg body weight, i.p) or vehicle for 4 weeks starting from one week before HLI. Blood perfusion in ischemic hindlimbs was examined by Laser Doppler at pre-HLI, and 0, 3, 7, 14 and 21 days post-HLI. Plasma exosomes were isolated by standard ultracentrifugation method and counted by NanoSight. GW increased blood concentration of CD31 + /Sca-1 + cell 3-day post-HLI and decreased necrosis and loss of toe/toenail; improved blood flow; enhanced capillary/arterial density determined by CD31 and SMA-α staining and decreased fibrosis in the muscles of ischemic hindlimbs in db/db mice 21-day post-HLI. Plasma exosomes from db/db mice significantly impaired human cardiac microvascular endothelial cells (HCMVECs) tube formation and migration. Mechanistically, hepatocyte growth factor (HGF) expression was decreased in bone marrow-derived endothelial progenitor cells (EPCs) from db/db mice and HCMVECs treated with diabetic plasma exosomes. Administration of GW increased HGF level in diabetic EPCs. Furthermore, diabetic plasma exosomes decreased H3K4me3 (active maker of transcription) whereas increased H3K27me3 (suppressive marker of transcription) at HGF promoter. Finally, treatment of HCMEVs with exogenous HGF rescued diabetic exosomes impaired tube formation of HCMVEC. Conclusions: Systemic inhibition of exosome synthesis/formation by GW increased ischemic limb repair in diabetic db/db mice, at least partially, via histone methylation mediated suppression of HGF. Our findings suggest that therapeutic targeting of systemic dysfunctional exosomes could represent a new avenue for therapeutics of ischemic tissue injury in patients with diabetes.
THE ASSOCIATION BETWEEN URIC ACID LEVELS AND CARDIORENAL OUTCOMES AND DEATH IN PATIENTS WITH TYPE 2 DIABETES: A SUB-ANALYSIS OF EMPA-REG OUTCOME
Authors: Subodh Verma, Qiuhe Ji, Deepak L. Bhatt, C. David Mazer, Mohammed Al-Omran, Silvio E. Inzucchi, Christoph Wanner, Anne Pernille Ofstad, Isabella Zwiener, Jyothis T. George, Bernard Zinman, David Fitchett
Journal: Journal of the American College of Cardiology (2019)
TRENDS IN HOSPITALIZATIONS AND IN-HOSPITAL OUTCOMES OF CARDIAC SARCOIDOSIS IN THE UNITED STATES FROM 2002 - 2014: A POPULATION-BASED STUDY
Authors: Mohamad Soud, Yasser Al-Khadra, Homam Moussa Pacha, Fahed Darmoch, M. Chadi Alraies
Journal: Journal of the American College of Cardiology (2019)
ADMISSION-TO-DISCHARGE WEIGHT CHANGE AND 30-DAY POST-DISCHARGE OUTCOMES IN OLDER PATIENTS WITH HEART FAILURE WITH PRESERVED AND REDUCED EJECTION FRACTION (HFPEF AND HFREF)
Authors: Gauravpal S. Gill, Awais Malik, Matt Nazari, Cherinne Arundel, Steven Singh, Gurusher Panjrath, Prakash Deedwania, Ioannis Kanonidis, Charles Faselis, Gregg C. Fonarow, Ali Ahmed
Journal: Journal of the American College of Cardiology (2019)
THE EFFECT OF ULTRASOUND TARGETED MICROBUBBLES DESTRUCTION MEDIATED ANGIOPOIETIN 1 GENE TRANSFECTION ON LEFT VENTRICULAR STRUCTURAL AND SYMPATHETIC REMODELING IN MYOCARDIAL INFARCTION CANINES
Authors: Sheng Cao, Tuantuan Tan, Qing Deng, Nan Jiang, Wei Hu, Bo Hu, Jin-Ling Chen, Qing Zhou
Journal: Journal of the American College of Cardiology (2019)
Abstract 252: Downregulated Striated Muscle Preferentially Expressed Protein Kinase Enhances Susceptibility to Post-Operative Atrial Fibrillation
Authors: Hannah Campbell, Li Ni, Julia Reynolds, Katherina Alsina, Tina Veleva, Issam Abu-Taha, Ann Quick, Zachary Donoviel, Na Li, Dobromir Dobrev, Xander Wehrens
Journal: Circulation Research (2018)

Post-operative atrial fibrillation (poAF) is the most common perioperative arrhythmia associated with increased hospital morbidity and mortality. It is known that atrial myocyte Ca 2+ handling changes can play a crucial role in AF pathogenesis, although the specific mechanisms involved in poAF have not been elucidated. Our lab has previously identified Striated Muscle Preferentially Expressed Protein Kinase (SPEG) as a binding partner of the major cardiac Ca 2+ release channel RyR2 and its negative regulator JPH2, which are critical for cardiac Ca 2+ homeostasis. We hypothesized that decreased SPEG levels may create an arrhythmogenic substrate promoting poAF development. SPEG protein and mRNA levels were assessed using Western blotting and qRT-PCR, respectively. To establish a mouse model of poAF, we performed thoracotomy in C57Bl/6J mice followed by rapid atrial pacing three days post-operatively to assess for poAF susceptibility. To test for a cause-effect relationship between decreased SPEG and AF susceptibility, we used a cardiac specific tamoxifen-inducible SPEG conditional knockout (cKO) mouse. Rapid atrial pacing was performed in SPEG cKO mice two weeks post-tamoxifen injection prior to onset of heart failure as assessed by echocardiography. In human atrial samples from patients with poAF we found 41±9% lower SPEGβ mRNA expression (n=7, p=0.04) compared to patients with normal sinus rhythm (NSR, n=8), while SPEGα expression was unchanged. Similarly, SPEGβ protein was 45±7% lower in poAF vs NSR patients (n=6, p=0.04). In contrast, SPEGβ mRNA expression was not decreased in patients with persistent or paroxysmal AF. Similar to poAF patients, our novel mouse model exhibited 90±4% lower SPEGβ protein levels (n=4, p=0.03) compared to mice with NSR (n=4) after rapid atrial pacing three days post-thoracotomy. Furthermore, SPEG cKO mice had increased AF duration after rapid atrial pacing (19.43 ± 15.16 s, n=3) compared to controls (0.27 ± 0.27 s, n=6, p=0.01), suggesting that loss of SPEG is sufficient to drive AF. In conclusion, we discovered reduced SPEGβ mRNA and protein in both humans and mice with poAF and established that a loss of SPEG might causally contribute to poAF pathogenesis.
PRESENCE OF ATRIAL FIBRILLATION IS ASSOCIATED WITH INCREASED MORTALITY AND MORBIDITY IN PATIENTS WITH TAKOTSUBO CARDIOMYOPATHY: AN INSIGHT FROM MOST RECENT NATIONAL INPATIENT DATABASE (2016)
Authors: Dhrubajyoti Bandyopadhyay, Vivek Amit Modi, Faris Haddadin, Adrija Hajra, Hassan Beydoun, Alba Munoz Estrella, Karan Sud, Rishi Shrivastav, Eyal Herzog
Journal: Journal of the American College of Cardiology (2019)
COMPARING CARVEDILOL WITH METOPROLOL SUCCINATE FOR ALL-CAUSE MORTALITY IN PATIENTS WITH HEART FAILURE WITH REDUCED EJECTION FRACTION: A SYSTEMATIC REVIEW AND META-ANALYSIS
Authors: Paul A. Gurbel, Amit Rout, Kevin Bliden, Udaya Tantry, Ankit Vyas, Rahul Chaudhary
Journal: Journal of the American College of Cardiology (2019)
Diverticular disease: picking pockets and population biobanks
Authors: Rinse K Weersma, Miles Parkes
Journal: Gut (2019)

The last decade has seen an explosion in genome-wide association studies (GWAS) in many diseases. With these—and its worth reminding ourselves that this is the primary goal of GWAS—have come valuable insights into pathogenic pathways. Some have been predictable (eg, variants affecting antigen presentation, co-stimulation and T-cell responses in immune-mediated diseases) and others, unexpected (eg, autophagy in Crohn’s disease). Until recently, such studies were performed in disease cohorts specifically ascertained for the purpose, with clear evidence that the larger the cohort the more loci will be found.1 

Such disease-specific cohorts with well-defined phenotypes can be assembled where there are engaged clinical communities, but may be tricky to ascertain for more general traits. Into this category falls diverticular disease. Diverticulosis affects up to 50% of the population but was poorly studied at a genetic level until recently, despite clear evidence for its relatively high heritability.2 3 In Gut , Schafmayer et al report on a large-scale analysis using clinical and genetic data from the UK Biobank. This population cohort totals 500 000 individuals, among whom ~32 000 have a recorded diagnosis of diverticular disease according to the international classification of disease (ICD) 9/10 coding. The authors went on to replicate their findings in a hospital-based case–control cohort.4 Interestingly, a similar approach was recently deployed by Maguire et al , using the same biobank ‘discovery’ dataset and a separate independent hospital-based registry as replication cohort.5 The earlier study identified 39 susceptibility loci for diverticular disease and showed that candidate genes reside in plausible biological pathways involved …
VARIABILITY IN GLYCATED HEMOGLOBIN VALUES AND CARDIOVASCULAR EVENTS IN PATIENTS WITH TYPE 2 DIABETES AND MULTIVESSEL CORONARY ARTERY DISEASE
Authors: Paulo Rezende, Whady Hueb, Mark Hlatky, Rosa Garcia, Cibele Garzillo, Thiago Scudeler, Gustavo André Boeing Boros, Fernando Faglioni Ribas, Anderson Roberto Dallazen, Desiderio Favarato, Jose Ramires, Roberto Kalil-Filho
Journal: Journal of the American College of Cardiology (2019)
MOTION COMPENSATION IMPROVES TEST-RETEST REPRODUCIBILITY OF CORONARY 18F-SODIUM FLUORIDE POSITRON EMISSION TOMOGRAPHY
Authors: Martin Lyngby Lassen, Jacek Kwiecinski, Sebastien Cadet, Damini Dey, Guido Germano, Philip Adamson, Alastair Moss, Marc Dweck, David Newby, Daniel Berman, Piotr Slomka
Journal: Journal of the American College of Cardiology (2019)
THE EFFECT OF PLATELET INHIBITON BY CANGRELOR AMONG OBESE PATIENTS UNDERGOING CORONARY STENTING: INSIGHTS FROM THE CHAMPION TRIALS
Authors: Ben Peterson, Robert Harrington, Gregg Stone, Philippe Gabriel Steg, C. Michael Gibson, Christian Hamm, Matthew Price, Renato Lopes, Sergio Leonardi, Jayne Prats, Efthymios Deliargyris, Kenneth Mahaffey, Harvey White, Deepak L. Bhatt
Journal: Journal of the American College of Cardiology (2019)
Abstract P2-03-05: S100A7 enhances triple negative breast cancer growth and metastasis by cross-talk with resistin
Authors: S Mishra, DK Ahiwar, RK Shukla, N Chatterjee, R Ganju
Journal: Cancer Research (2019)

Abstract Triple negative breast cancer (TNBC) represents ˜20% of all breast cancer cases and is highly refractory to existing therapies. Although, a number of putative functions have been proposed for S100A7, but its crosstalk with resistin in TNBC pathogenesis remains to be defined. S100A7/RAGE signaling has been shown to play an important role in inflammation-mediated breast cancer pathogenesis. S100A7/psoriasin, a member of the epidermal differentiation complex, is widely overexpressed in invasive estrogen receptor (ER)α-negative breast cancers. Thus, the objective of this study is to explore the cross-talk between S100A7 and resistin in TNBC cells. In this study, we observed that the treatment or overexpression of S100A7 increased the expression of resistin, cPLA2 and FASN expressions in TNBC cells. The overexpression/downregulation of S100A7 with exogenous resistin in TNBC cells also showed the marked changes in their migrating, sphere forming and colony forming abilities. The treatment of resistin also increases the effect of S100A7-mediated expansion of breast cancer stem cells (BCSCs) by increasing the ALDH activity and CD44+CD24- subpopulations. The in-vivo effects of resistin on breast tumor growth and metastasis were also analyzed by using MDA-MB-231 vector and S100A7 overexpressed MDA-MB-468 vector and S100A7 knock-down cells by using Xenograft NOD/SCID Il2rg null (NSG) mice model. Here, we have found that the treatment of resistin increased the tumor growth and metastases mediated by S100A7 by modulating the different myeloid cells populations. We have also shown that the S100A7 overexpressed MDA-MB-231 cells were much more sensitive to FASN and cPLA2 inhibitors as compared to vector control cells. The in-silico analyses also showed the clinical significance of S100A7 and resistin in TNBC pathogenesis. Collectively, these findings suggest the novel cross-talk in between S100A7 and resistin in TNBC pathogenesis and how resistin, cPLA2 and FASN could be used as potential therapeutic targets to inhibit the S100A7-mediated TNBC progression and metastasis. Citation Format: Mishra S, Ahiwar DK, Shukla RK, Chatterjee N, Ganju R. S100A7 enhances triple negative breast cancer growth and metastasis by cross-talk with resistin [abstract]. In: Proceedings of the 2018 San Antonio Breast Cancer Symposium; 2018 Dec 4-8; San Antonio, TX. Philadelphia (PA): AACR; Cancer Res 2019;79(4 Suppl):Abstract nr P2-03-05.
RELATIVE EFFECTS OF THE HOSPITAL READMISSIONS REDUCTION PROGRAM FOR CONGESTIVE HEART FAILURE ON HOSPITALS THAT SERVE POORER PATIENTS
Authors: Jason H. Wasfy, Vijeta Bhambhani, Emma Healy, Christine Choirat, Francesca Dominici, Changyu Shen, Yun Wang, Robert Yeh
Journal: Journal of the American College of Cardiology (2019)
LOWER RIGHT VENTRICULAR PRESSURE IS ASSOCIATED WITH INCREASED MORTALITY OR TRANSPLANT IN INFANTS WITH PULMONARY ATRESIA WITH INTACT VENTRICULAR SEPTUM
Authors: Zachary Spigel, Shaine Morris, Athar Qureshi, Rodrigo Zea-Vera, Kristen Sexson, Christopher Caldarone, Jeffrey Heinle, E. Dean Mckenzie, Ziyad Binsalamah, Michiaki Imamura, Iki Adachi
Journal: Journal of the American College of Cardiology (2019)
CHARACTERIZATION OF IMMUNE CHECKPOINT INHIBITOR-RELATED CARDIOTOXICITY IN LUNG CANCER PATIENTS FROM A RURAL SETTING IN EASTERN NORTH CAROLINA
Authors: Melissa Y.Y. Moey, Anna Tomdio, Justin D. McCallen, Lauren Vaughan, Rafeh Naqash, Cynthia Cherry, Blase Carabello, Paul R. Walker
Journal: Journal of the American College of Cardiology (2019)
EFFICACY AND SAFETY OF LONG-TERM EVOLOCUMAB USE IN ASIAN VERSUS OTHER SUBJECTS: THE FOURIER TRIAL
Authors: Anthony C. Keech, Peter Sever, Lixin Jiang, Atsushi Hirayama, Chen Lu, Leslie Tay, Prakash Deedwania, Chung-Wah Siu, Armando Lira Pineda, Donghoon Choi, Min-Ji Charng, John Amerena, Wan Azman Wan Ahmad, Vijay Kumar Chopra, Terje Pedersen, Terje Pedersen, Robert Giugliano, Marc Sabatine
Journal: Journal of the American College of Cardiology (2019)
HEMODYNAMIC IMPACT OF ISOLATED AORTIC VALVE REPLACEMENT IN PATIENTS WITH SEVERE AORTIC STENOSIS AND MITRAL STENOSIS: A DOPPLER ECHOCARDIOGRAPHIC STUDY
Authors: Nahoko Kato, Mizuho Hoshina, Sorin Pislaru, Cristina Pislaru, Ratnasari Padang, William Miranda, Kentaro Shibayama, Hiroyuki Watanabe, Vuyisile Nkomo, Patricia Pellikka
Journal: Journal of the American College of Cardiology (2019)
IMPACT OF 2017 ACC/AHA GUIDELINES ON PREVALENCE OF HYPERTENSION AMONG INDIAN ADULTS: RESULTS FROM A NATIONALLY REPRESENTATIVE SURVEY
Authors: Kartik Gupta, Sivasubramanian Ramakrishnan, Geever Zachariah Arimbur, Shivkumar Jallepalli, Rishi Sethi, Dharmendra Jain, Neil Bardoloi, Santhosh Satheesh, Sandeep Bansal, Ranjit Nath, Sanjay Tyagi, P.P. Mohanan, K. Venugopal, Gurpreet Singh Wander, Satish Gupta, Nagendra Senguttuvan, Debabrata Roy, Satyanarayan Routray, Bateswar Singh, Mrinal Das, Soumitra Kumar, Pradip Deb, Hirday Chopra, Ashok Seth
Journal: Journal of the American College of Cardiology (2019)
<i>Notes from the Field:</i> Tetanus in an Unvaccinated Child — Oregon, 2017
Authors: Judith A. Guzman-Cottrill, Christina Lancioni, Carl Eriksson, Yoon-Jae Cho, Juventila Liko
Journal: MMWR Morbidity and Mortality Weekly Report (2019)

In 2017, a boy aged 6 years who had received no immunizations sustained a forehead laceration while playing outdoors on a farm; the wound was cleaned and sutured at home. Six days later, he had episodes of crying, jaw clenching, and involuntary upper extremity muscle spasms, followed by arching of the neck and back (opisthotonus) and generalized spasticity.
Abstract 553: Inhibition of Microrna-21 Prevents Myocardial Remodelling and Dysfunction in a Pig Model of Ischemia/reperfusion Injury
Authors: Deepak Prabhu Ramanujam, Rabea Hinkel, Veronika Kazcmarek, Andrea Howe, Katharina Klett, Anne Dueck, Thomas Thum, Karl-Ludwig Laugwitz, Christian Kupatt, Stefan Engelhardt
Journal: Circulation Research (2018)

Background: Upregulation of microRNA-21 (miR-21) has been associated with myocardial disease and its systemic inhibition was effective in reducing cardiac fibrosis and dysfunction in rodent models. However, the translation to human disease is challenging, as systemic application of miR inhibitors does not achieve adequate delivery in most organs including the heart. Here, we sought to test the applicability and therapeutic efficacy of local, catheter-based delivery of antimiR-21 in a large animal model of heart failure. Methods and results: Pigs underwent percutaneous occlusion of the left coronary artery for 60 minutes and were followed up for 33 days. A synthetic inhibitor against miR-21 (LNA-antimiR-21, 10 mg per application) was applied locally by intracoronary infusion at days 5 and 19 after the injury. AntimiR-21 effectively suppressed the I/R-induced increase of miR-21. At 33 days after I/R, LNA-21-treated hearts exhibited reduced cardiac fibrosis and hypertrophy, and improved cardiac function (ejection fraction: I/R control 22±1% vs. I/R LNA-21 33±2%). RNA sequencing of ischemic myocardium revealed a significant enrichment for miR-21 targets in anti-miR-21 treated hearts. Mechanistically, antimiR-21 interfered with a number of signalling pathways involved in myocardial remodelling including the MAPK-ERK signalling cascade. Conclusion: Taken together, our study provides the first evidence for the feasibility and therapeutic efficacy of miR-21 inhibition in a large animal model of myocardial ischemia. AntimiR-21 prevented the structural and functional consequences of ischemic myocardial damage.
LONG-TERM CLINICAL OUTCOMES OF ORBITAL ATHERECTOMY PRECEDED BY OCT-GUIDED STENT IMPLANTATION
Authors: Masaru Ishida, Gary Mintz, Yuqi Fan, Alec Goldberg, Evan Shlofmitz, Mitsuaki Matsumura, Allen Jeremias, Ziad Ali, Ajay Kirtane, Jeffrey Moses, Richard Shlofmitz, Akiko Maehara
Journal: Journal of the American College of Cardiology (2019)
CHARACTERIZATION OF ATRIAL FLUTTER AFTER PULMONARY VEIN ISOLATION BY CRYOBALLOON ABLATION
Authors: Jayson Baman, Rachel Kaplan, Celso Diaz, Graham Peigh, Aakash Bavishi, Amar Trivedi, Mark Shen, Prasongchai Sattayaprasert, Jeremiah Wasserlauf, Alexandru Chicos, Rishi Arora, Susan Kim, Albert Lin, Nishant Verma, Bradley Knight, Rod Passman
Journal: Journal of the American College of Cardiology (2019)
Abstract 06: Body Mass Index Growth Trajectories During Childhood and Adult Obesity Risk
Authors: Tao Zhang, Bo Xi, Chunhong Li, Lydia Bazzano, Jiang He, Paul Whelton, Wei Chen, Shengxu Li
Journal: Circulation (2017)

Background: Obesity measures track from childhood to adulthood. However, information is limited regarding the relationship between body mass index (BMI) growth trajectories during childhood and adult obesity risk. We hypothesize that BMI growth rates at different childhood age windows have influences on adult obesity, independent of absolute BMI values. Methods: The longitudinal study cohort consisted of 2,732 adults (1,772 whites and 960 blacks; 1,226 males) who had BMI measured at least 4 times from childhood (4-19 years) to adulthood (20-51 years). A random-effects mixed model was used to construct growth curves of BMI from childhood to adulthood in race-sex groups. Model-estimated linear growth rates (kg/m 2 /year) in BMI at different childhood ages, calculated by using the first derivatives of the growth curves, were linked to adult obesity (BMI&gt;=30) in multivariable regression models, adjusted for adult age, race, sex, adult smoking and alcohol drinking, and childhood BMI levels. Results: BMI from childhood to adulthood fit cubic growth curves; linear and nonlinear curve parameters differed significantly between race-sex groups. BMI levels showed race and sex differences from age 15 years onwards. Obese adults had significantly higher BMI levels than non-obese adults from childhood to adulthood. Differences in linear and nonlinear slope parameters of BMI between obese and non-obese groups were all significant (p&lt;0.001). The association patterns of childhood BMI growth trajectories with adult obesity were similar in all race-sex groups. In the total sample, the association between childhood BMI growth rate and adult obesity was peaked at age 17 (odds ratio=5.7 and 95% confidence interval=4.7-6.8) as shown in the figure. Conclusions: These observations indicate that adult obesity originates in early life with different longitudinal BMI trajectory profiles. Puberty is a crucial period for the development of obesity in later life, which has implications for obesity prevention beginning in childhood.
CARDIAC IMPLANTABLE ELECTRONIC DEVICES AND 6MV RADIOTHERAPY: A PROSPECTIVE, RANDOMIZED, BLINDED, IN-VITRO EVALUATION
Authors: Enrico Di Girolamo, Massimiliano Faustino, Nanda Furia, Marianna Appignani, Clelia Di Carlo, Domenico Genovesi, Maria Daniela Falco
Journal: Journal of the American College of Cardiology (2019)
ACUTE AND SUSTAINED EFFECTS OF PLANT-BASED BIOEQUIVALENT INORGANIC NITRATE SUPPLEMENT TABLETS ON BLOOD PRESSURE REDUCTION IN PRE-HYPERTENSIVES MEN AND WOMEN
Authors: Lavanya Cherukuri, Chandana Shekar, Dong Li, Divya Birudaraju, Sajad Hamal, Ferdinand Flores, Sion Roy, William Sotka, Shawn J. Green, Matthew Budoff
Journal: Journal of the American College of Cardiology (2019)
TCTAP A-094 Clinical Outcomes of Patients Received Lower Dose of Non-vitamin K Antagonist Oral Anticoagulants as a Medical Management for Acute Pulmonary Embolism
Authors: Jin Sup Park, Jiyeon Shin, Han Cheol Lee, Kwang Soo Cha, Taek Jong Hong
Journal: Journal of the American College of Cardiology (2019)
COMPUTER DEEP LEARNING FOR AUTOMATIC IDENTIFICATION OF ECHOCARDIOGRAPHIC VIEWS APPLIED FOR RHEUMATIC HEART DISEASE SCREENING: DATA FROM THE ATMOSPHERE-PROVAR STUDY
Authors: Bruno Ramos Nascimento, André L.S. Meirelles, Wagner Meira, Gisele L. Pappa, Maria do Carmo Nunes, Craig Sable, Andrea Beaton, Paulo R. Gomes, Fábio V. Modesto, George Luiz M. Teodoro, Antonio Ribeiro
Journal: Journal of the American College of Cardiology (2019)
REVERSIBLE LEFT VENTRICULAR SYSTOLIC DYSFUNCTION ASSOCIATED WITH SPONTANEOUS CORONARY ARTERY DISSECTION
Authors: Jessica Duran, Yousef Ghazzawi, Aferdita Spahillari, M. Ihsan Kaadan, Shelly Mishra, Francesca Ponzini, Nandita Scott, Sarah Tsiaras, Malissa Wood
Journal: Journal of the American College of Cardiology (2019)
CATHETER ABLATION COMPARED TO MEDICAL MANAGEMENT FOR ATRIAL FIBRILLATION IN PATIENTS WITH CAROTID ARTERIAL DISEASE IS ASSOCIATED WITH LOWER RATES OF DEMENTIA AND STROKE
Authors: Thomas Jared Bunch, Heidi May, Tami Bair, Brian G. Crandall, Michael J. Cutler, Kevin G. Graves, Victoria Jacobs, Charles Mallender, Jeffrey S. Osborn, J. Peter Weiss, John Day
Journal: Journal of the American College of Cardiology (2019)