tbuckley/cabot-search huggingface dataset:
Abstract 453: Young Adult Female Dahl Salt-Sensitive Rats with Heart Failure with Preserved Ejection Fraction have a Distinct Survival Advantage over Age-Matched Afflicted Males
Authors: David W Angert, Peter Kilfoil, Geoffrey De Couto, Stephan Aynaszyan, Jae H Cho, Antonio Echavez, Jackelyn Valle, Eva Jaghatspanyan, Eduardo Marban
Journal: Circulation Research (2018)

Introduction: Cardiovascular disease is the #1 killer of women. Heart failure with preserved ejection fraction (HFpEF) represents ≥50% of HF patients, and affects women ~2x more than men. With no treatment strategies showing decreased mortality, this indicates a critical unmet medical need. Objectives: To identify sex difference in HFpEF, we are establishing the female model of HFpEF in the Dahl salt-sensitive (DS) rat. We aim to compare male (M) and female (F) DS rats with HFpEF to identify sex-related differences in underlying mechanisms of HFpEF, for the first time. Methods: 7-week-old DS rats of both sexes are randomly assigned to low-salt (LS; control) or high-salt (HS; 8%) diets. At 7, 14 and 18-weeks of age, rats undergo echocardiography. The endpoint for LS and HS groups is 18-weeks, when HS diet groups have developed diastolic dysfunction and HFpEF. Invasive hemodynamics are performed prior to euthanasia. Results: HS (M) and (F) DS rats show a significant increase in systolic (M: 233±20; F: 243±15 bpm vs control; p&lt;0.001) and diastolic (M: 186±18; F: 183±25 bpm vs control; p&lt;0.001) blood pressure at 18-weeks, with no change in ejection fraction. Normalized LV wall thickness is differentially increased between sexes, with anterior wall thickness (systole) greater in (M) (M:F = 1.6-fold; p&lt;0.001), but posterior wall thickness (diastole) greater in (F) (F:M = 1.5-fold; p&lt;0.001), indicating structural differences. Normalized heart weight/tibia length in HS vs LS diets is increased in (F) (F: 0.39 vs. M: 0.23; p&lt;0.001), indicating increased cardiac hypertrophy in (F). Hemodynamics show increased normalized end-diastolic pressure volume relationship in (F) (F: 1.01 vs. M: 0.64; p&lt;0.01). Although (F) DS rats with HFpEF show increased cardiac hypertrophy and worse diastolic dysfunction compared with age-matched (M) DS rats with HFpEF, Kaplan-Meier survival analysis shows an 18-week mortality rate significantly greater in (M) (~50%) vs. (F) (~16%). Conclusion: This study characterizes the female DS model of HFpEF, and elucidates worsening (F) cardiac dysfunction vs. (M) at 18-weeks of age, with provocative results showing significantly decreased morbidity and mortality among females at the same time-point, indicating cardioprotective effects.
Anti-CD19 CAR-T as a feasible and safe treatment against central nervous system leukemia after intrathecal chemotherapy in adults with relapsed or refractory B-ALL
Authors: Xiaoyuan He, Xia Xiao, Qing Li, Yanyu Jiang, Yaqing Cao, Rui Sun, Xin Jin, Ting Yuan, Juanxia Meng, Li Ma, Wenyi Lu, Cuicui Lyu, Kaiqi Liu, Mingfeng Zhao
Journal: Leukemia (2019)

Chimeric antigen receptor (CAR)-T cell treatment as an emerging tumor immunotherapy has produced exciting results in relapsed or refractory B cell acute lymphoblastic leukemia (B-ALL) [1][2][3][4].However, few data are available on the therapeutic effect of
ASSOCIATION OF PRE-EXISTING MEDICAL COMORBIDITY WITH SHORT-TERM MORTALITY IN TAKOTSUBO CARDIOMYOPATHY
Authors: Arash Nayeri, Alexander Yuen, Cher Huang, Kathryn Cardoza, Andrea MacDonald, Kamran Shamsa, Quinn Wells, Gregg Fonarow
Journal: Journal of the American College of Cardiology (2019)
Abstract 15782: Effects of Ezetimibe+Statins and Statin Therapy on Changes in Remnant-lipoprotein Cholesterol
Authors: Peter P Toth, Harold E Bays, W Virgil Brown, Alberico L Catapano, Michael H Davidson, Michel Farnier, Joanne E Tomassini, Erin Jensen, Adam B Polis, Andrew M Tershakovec
Journal: Circulation (2017)

Introduction: Elevated remnant-lipoprotein cholesterol (RLP-C, cholesterol mass in VLDL and IDL) increases cardiovascular disease (CVD) risk, particularly for patients with residual risk on lipid-lowering therapy (LLT). RLP-C assessment methods are not standardized and include measurement by immunoseparation (IM) and vertical auto profile ultracentrifugation (VAP [IDL+VLDL3]) or calculation (total cholesterol – HDL-C – LDL-C). This study assessed the effects of Eze, Eze + statins and statin monotherapy on RLP-C measured by these 3 methods in 1st line (statin naive/wash-out) and 2nd line (ongoing statin/uptitration) studies. Methods: RLP-C change and percent change from baseline were evaluated using pooled data from randomized 1st line (Eze vs placebo, Eze+statinsξ vs statinsξ) and 2nd line (Eze+statin‖ vs double statin¶ dose) studies. RLP-C was measured by all 3 methods for 1st line studies; VAP and calculated methods for 2nd line studies. Relationships between baseline covariates (lipids, hsCRP, age, gen...
Abstract 18703: Novel MicroRNA 874 is a Unique Angiogenic Modulator of Stem Cell Paracrine Factor Epidermacan
Authors: Ying Zhang, Yangfan Liu, Pete Pow-anpongkul, Vinogran Naidoo, J.Alan Payne, Hui Mu, Nicholas Katsanis, Maria Mirotsou, Victor Dzau
Journal: Circulation (2012)

Introduction: We have previously shown that treatment with epidermacan a novel stem cell derived paracrine factor promoted angiogenesis in vitro accompanied with an increase in angiogenic miRNA expression. Here, we identified novel microRNAs regulated by epidermacan and validated their importance in modulating the epidermacan angiogenic effects in vitro and in vivo . Methods and Results: Increased levels of pro-angiogenic miRNAs such as miR-10b, miR-21 and miR-424 were discovered in MicroRNA profiling of HUVEC cells treated with epidermacan and then confirmed by qRT-PCR. We also identified miR-874, a relatively unknown miRNA but for its role as a tumor suppressor. Investigation of the downstream microRNA/gene networks revealed that epidermacan regulated known angiogenic genes such as VEGFC, TBX1, HIF-1a and NRP1 . Importantly, treatment with epidermacan decreased the levels of HoxD10, a known target of miR-10b in thrombin induced angiogenesis. Inhibition of miR-10b or miR-874 via anti-miR transfection repressed the effects of epidermacan in HUVEC tube formation on matrigel. Interestingly inhibition of miR-10b also reversed the angiogenic effects of VEGF; however inhibition of mir-874 only suppressed the effects of epidermacan, suggesting miR-874 as an epidermacan specific target. The fli:GFP transgenic zebrafish model was used to study the role of epidermacan in angiogenesis in vivo . Morpholino against zebrafish epidermacan gene was injected into the yolk of fli:GFP transgenic zebrafish embryos at 1-4 cell stage, and larvae were collected at 4 days post fertilization to monitor vessel development. The trunk vascular network was disorganized in larvae injected with the morpholino of epidermacan compared to uninjected control. The vascular defects were rescued by co-injection of 100 pg mRNA of human NDNF (epidermacan homolog), suggesting that vessel malformation is specifically due to knockdown of epidermacan. Conclusion: Our data confirm the role of epidermacan as an angiogenic factor and identify the novel miR-874 as specific modulator of epidermacan’s angiogenic effect. Further analysis to elucidate the microRNA/gene networks downstream of epidermacan will provide new insights on the regulatory networks of angiogenesis.
Early Melanoma Nodal Positivity and Biopsy Rates Before and After Implementation of the 7th Edition of the <i>AJCC Cancer Staging Manual</i>
Authors: Chelsea Isom, Lee Wheless, Mary A. Hooks, Rondi M. Kauffmann
Journal: JAMA Dermatology (2019)

There has been a continued increase in the incidence of newly diagnosed melanomas, most of which are T1 melanomas. The associations between changes in tumor staging, implemented with the 7th edition of the AJCC Cancer Staging Manual (AJCC 7), and sentinel lymph node biopsy rates and nodal positivity rates remain to be seen.To evaluate the change that the implementation of the AJCC 7 had on staging criteria and the distribution of thin melanomas requiring nodal surgery and nodal positivity rates.Retrospective cross-sectional study from 2004 through 2013 of all adults (≥18 years) diagnosed with a T1 (Breslow depth ≤1.0 mm) melanoma using The National Cancer Database that captures 70% of all newly diagnosed cancers from accredited Commission on Cancer organizations, including both academic and community settings. Data were analyzed in May 2017.Patients were grouped together based on year of diagnosis, before and after 2009.To determine the sentinel lymph node biopsy rate before and after the implementation of the AJCC 7.A total of 141 280 patients met inclusion criteria. Of 86 846 patients diagnosed from 2004 through 2009, 53.7% (49 644) were male and had a mean (SD) age of 57.7 (16.4) years. Of 54 434 patients diagnosed from 2010 through 2013, 54.3% (31 086) were male and had a mean (SD) age of 59.5 (15.9) years. After 2010, there was a 3.8% decrease in the number of nodal surgeries performed (32 485 of 86 846 patients [37.6%] vs 18 379 of 54 434 patients [33.8%]; P < .001). The nodal positivity rate decreased 1.0% from (9.8% [3166 of 86 846] to 8.8% [1618 of 54 434]) (P < .001). An increase in the proportion of T1b melanomas being evaluated, from 48.8% to 62.2%, was seen (P < .001). Of T1b melanomas that underwent nodal evaluation from 2004 through 2009, 74.0% had Clark level IV (invasion of the reticular dermis) or Clark level V (invasion of the deep, subcutaneous tissue) and 9.5% were ulcerated. From 2010 through 2013, of the T1b melanomas undergoing nodal evaluation, 82.6% had an elevated mitotic rate only, 3.7% were ulcerated, and 13.7% had both ulceration and an elevated mitotic rate.It appears that after the institution of AJCC 7, there was an overall decrease in the number of T1 melanomas undergoing nodal biopsy without a clinically relevant change in sentinel lymph node positivity, with an increase in the number of T1b melanomas undergoing nodal evaluation.
Risk Factors for Leukemic Transformation Among 1,306 Patients with Primary Myelofibrosis: Mutations Predict Early Events
Authors: Rangit Reddy Vallapureddy, Mythri Mudireddy, Natasha Szuber, Domenico Penna, Maura Nicolosi, Terra L. Lasho, Christy Finke, Curtis A. Hanson, Rhett P. Ketterling, Kebede H. Begna, Naseema Gangat, Animesh Pardanani, Ayalew Tefferi
Journal: Blood (2018)
Abstract 205: Cryopreserved Cardiac Tissue Graft (MyCardia) Improved Cardiac Function in Rats With Heart Failure
Authors: Jordan Lancaster, Jen Koevary, Steven Goldman
Journal: Circulation Research (2018)

Introduction: There are currently 2M patients with NYHA Class II-IV heart failure of ischemic etiology in the United States. Cell-based therapies hold tremendous potential to provide therapeutic benefit for treating degenerative diseases such as chronic heart failure (CHF). Therapeutic adoption of engineered grafts will require ease of use and demonstration of cost-effectiveness. Here we describe for the first time the ability to cryopreserve an engineered tissue (MyCardia™) and demonstrate functional recovery in a rat model of CHF. Methods: MyCardia grafts were cultured with human dermal fibroblasts (hNDF) and human iPSC derived cardiomyocytes (hiPSC-CMs) (FCDI) into a bioabsorbable mesh (Novus Scientific). Grafts were cryopreserved at -196C for 2-4weeks, thawed, reconstituted and implanted into rats with CHF. Myocardial infarction (MI) were generated in immune competent rats by ligating the left coronary artery. Three weeks after MI, rats received graft implants via median sternotomy. Echocardiography was performed at 3 and 7 weeks post graft implantation. In addition, terminal hemodynamics were performed. Results: MyCardia maintained spontaneous and synchronous contractions pre-freeze (65±10bpm) and post thaw (60±10bpm). Grafts were maintained to 40 days post thaw with no deterioration in composition or beat rate. MyCardia reversed maladaptive left ventricular (LV) remodeling by decreasing (p&lt;0.05) LV volume systole/diastole, LV diameter systole/diastole with trending improvement in EF and FS from 3 and 7 weeks post implantation as compared to CHF controls. Grafts decreased LV end diastolic pressure, 24±6 mmHg vs. 18±3mmHg and showed trending improvements in LV dP/dt(-) and dP/dt(+) as compared to controls. In addition, MyCardia results in 36% reduction in LV EDP operating volume as demonstrated by a leftward shift of the diastolic pressure-volume relationship. Conclusion: MyCardia can be cryopreserved, reconstituted and implanted in rats with CHF. Treated rats demonstrated reversal of maladaptive LV remodeling and continued functional improvement from 3 to 7 weeks post treatment. We anticipate that the ability to cryopreserve will allow long-term storage after large batch manufacture, providing cost and utility advantages.
A RANDOMIZED, PLACEBO-CONTROLLED, DOUBLE-BLIND, ASCENDING SINGLE-DOSE, PHASE 1 STUDY TO EVALUATE THE SAFETY, TOLERABILITY, PHARMACOKINETICS, AND PHARMACODYNAMICS OF AMG 529, A NOVEL ANTI-ASGR1 MONOCLONAL ANTIBODY, IN HEALTHY SUBJECTS
Authors: Mariano Janiszewski, Winnie Sohn, Cheng Su, Yi Hsiang Hsu, Elizabeth Finger, Allegra Kaufman
Journal: Journal of the American College of Cardiology (2019)
THE PROGNOSTIC ROLE OF VENTRICULAR-ARTERIAL COUPLING IN TRANSCATHETER AORTIC VALVE REPLACEMENT: THE IMPACT OF PULMONARY ARTERIAL PROPORTIONAL PULSE PRESSURE
Authors: Martin Miguel Inocencio Amor, Nicholas Chan, Brian Yuen, Gregory Maurer, Jacinda Berg, Bruce Rutkin, Alexander Lee
Journal: Journal of the American College of Cardiology (2019)
STENOSIS OF EXTERNAL ILIAC ARTERY DUE TO COMPRESSION FROM PREVIOUS STENT PLACEMENT IN ILIAC VEIN: AN INTRAVASUCLAR ULTRASOUND GUIDED APPROACH
Authors: Rahul Vasudev, Nirmal Guragai, Upamanyu Rampal, Hiten Patel, Hartaj Virk, Mahesh Bikkina, Shamoon Fayez
Journal: Journal of the American College of Cardiology (2019)
DIAGNOSTIC PERFORMANCE OF NOVAMEDS SENS-A-HEART POINT OF CARE QUALITATIVE ASSAY COMPARED WITH HIGH SENSITIVITY CARDIAC TROPONIN T FOR THE DIAGNOSIS OF ACUTE CORONARY SYNDROME
Authors: Ronny Alcalai, Boris Varshitzky, Ahmad Marhig, Gavriel Shalmiev, Emil Katz, David Stener, Larisa Boguslavsky, Chaim Lotan
Journal: Journal of the American College of Cardiology (2019)
Shared genetic factors and their causality in autoimmune diseases
Authors: Kazuhiko Yamamoto, Yukinori Okada
Journal: Annals of the Rheumatic Diseases (2019)

Autoimmune diseases including rheumatic diseases affect 3%–10% of the world population. The frequency of incidence is dependent on the geographical area, ethnicity, sex and environmental factors.1 Except for rare monogenic diseases, the majority of autoimmune diseases are defined as a complex trait disorder, where multiple genetic and environmental factors interact. For example, twin studies have estimated that the heritability of rheumatoid arthritis (RA) is ~60%.2 3 RA is classified into anti-citrullinated protein antibodies (ACPA) positive and negative subtypes. The above-mentioned heritability of RA applies primarily to patients with RA who are positive for ACPAs, whereas the heritability of seronegative RA appears to be low,4 suggesting the differences in the genetic background of these two conditions. Therefore, although the classification criteria are satisfied, each autoimmune disease appears to be heterogeneous. On the other hand, autoimmune diseases share several symptoms, such as Raynaud phenomenon and arthritis. In a clinical situation, the terms ‘poly-autoimmunity’ and ‘familial autoimmunity’ are used. The former suggests the presence of multiple autoimmune diseases in a single patient, and the latter implies the situation where diverse autoimmune diseases are persistent in a family.

Genes in the major histocompatibility complex (MHC) have been reported to be the strongest single genetic effect in the majority of autoimmune diseases. Although the precise mechanisms remain to be discovered, these MHC loci are the first and the most robust examples of shared genetic factors among several autoimmune diseases. Since 2007, rapid advances in genome-wide association study (GWAS) have enhanced the identification of hundreds of genetic risk factors for many complex diseases. Thereafter, one possible approach to expanding our understanding of the genetics of autoimmune disease using GWAS data is cross-trait analyses to identify genetic correlations with other complex traits. In fact, it has been reported that genetic variants associated with one …
Abstract 458: Role of Heterochromatin Protein 1 Binding Partner 3, HP1BP3, in Cardiomyocyte Stress Response
Authors: Cheryl X Chan, Wilson Tan, Roger Foo
Journal: Circulation Research (2018)

Cardiovascular disease (CVD) is a top cause of mortality and a serious economical burden. While many diseases fall under the umbrella of CVD, they all ultimately lead to heart failure. Little is known how and what epigenetic factors could be involved in cardiomyocytes stress response during such end stage event. The objective of this study is to uncover novel epigenetic proteins that might be nodule regulators of stress genes, to better understand and alter inducted stress pathways. Here, we found Heterochromatin Protein 1 Binding Partner 3, Hp1bp3 , in our single nuclear transcriptomics data set of sham and TAC murine cardiomyocytes. Hp1bp3 was up-regulated in a subset of TAC nuclei (53.8% in TAC versus 18.9% in sham) and appeared to be central in a module of disease-related cardiac genes, suggesting its possible role of stress response regulation. Indeed, a subset of DE genes seemed to be co-regulated with Hp1bp3 only in TAC nuclei amongst other subsets of genes that are not co-regulated. In contrast, stress gene regulation in sham nuclei seemed to not be controlled by Hp1bp3 . These data suggest that Hp1bp3 is unique in diseased nuclei and could be an important regulator of specific stress genes. When we performed the Ingenuity Pathway Analysis, we observed that Hp1bp3 -high TAC nuclei activated many cardioprotective pathways, like hypoxia-handling and ErBB-signaling pathways, suggesting Hp1bp3 was up-regulated to initiate a cardioprotective stress-handling response. Further experiments with a transgenic HL-1 cell line over-expressing Hp1bp3 showed a robust down-regulation of stress genes (Nppb, Myh7) after exposure to hypoxia.
TRENDS AND OUTCOMES OF GASTROINTESTINAL BLEEDING ADMISSIONS AMONG PATIENTS WITH LEFT VENTRICULAR ASSIST DEVICES
Authors: Dinesh Voruganti, Ghanshyam P.S. Shantha, Chakradhari Inampudi, Paulino Alvarez, Michael Giudici, Alexandros Briasoulis
Journal: Journal of the American College of Cardiology (2019)
Abstract #444: Molecular and clinical characterization of RICTOR (MTORC2) as a candidate oncogene in hepatocellular carcinoma
Authors: Augusto Villanueva, Helena Cornella, Victoria Tovar, Clara Alsinet, Derek Chiang, Beatriz Minguez, Sara Toffanin, Rad Savic, Yujin Hoshida, Anja Lechenmayer, Swan Thung, Sasan Roayaie, Myron Schwartz, Jordi Bruix, Vincenzo Mazzaferro, Scott Friedman, Josep Llovet
Journal: Cancer Research (2009)

Introduction: Recent data implicate mTOR signaling in human hepatocellular carcinoma (HCC). However, the specific contribution of mTOR complex components as either potential oncogenes or targets for therapies is unknown. Aims: (1) To evaluate the role of mTOR complex 2 (RICTOR) in human hepatocarcinogenesis and (2) to assess the impact of selective RICTOR blockade in cellular models of HCC. Methods: DNA copy number changes (Affymetrix 238K Sty-arrays) and mRNA levels of RICTOR (Affymetrix U133 Plus 2.0 arrays and qRTPCR using Taqman Probes) were analyzed in 102 human HCV-related HCC (fresh frozen samples). RICTOR over-expression was correlated with both signaling pathway activation (Wnt-sCatenin, IGF/AKT/MTOR and RAS/MAPK), and with the existing molecular classification of HCC (Chiang et al. Cancer Res 2008). Data were validated in a separate cohort of 164 HCC patients (paraffin-embedded samples), profiled using DASL (Illumina). Molecular data were correlated with clinico-pathological features and patient prognosis using Kaplan-Meier and Cox Proportional Hazard Test. For in vitro studies, Huh-7 cells were transfected with siRNA-RICTOR, and western blots done to evaluate downstream signals (p-AKT, p-RPS6 and p-ERK). Cell viability and proliferation were assessed by MTT and [H3]-Thymidine incorporation assay respectively, whereas FACS was used to evaluate cell cycle. Results: 25% (25/100) of HCV-related HCC samples had DNA gains in the RICTOR locus. Among them, 12% (11/87) had high mRNA levels of RICTOR (P=0.03). These patients had significantly worse survival (P=0.001) and higher rates of early recurrence (P  Citation Information: In: Proc Am Assoc Cancer Res; 2009 Apr 18-22; Denver, CO. Philadelphia (PA): AACR; 2009. Abstract nr 444.
LOW URINE SODIUM PREDICTS LENGTH OF STAY BUT NOT RESPONSE TO DOPAMINE OR NESIRITIDE IN HOSPITALIZED PATIENTS WITH ACUTE HEART FAILURE AND RENAL DYSFUNCTION
Authors: Jonathan Cunningham, Jie Sun, Kevin Anstrom, JoAnn Lindenfeld, Michael Givertz, Lynne Stevenson, Neal Lakdawala
Journal: Journal of the American College of Cardiology (2019)
IMPACT OF TREATMENT WITH ANGIOTENSIN-CONVERTING ENZYME INHIBITORS OR RECEPTOR BLOCKERS ON REVASCULARIZATION AFTER PERCUTANEOUS CORONARY INTERVENTION AND CORONARY ARTERY BYPASS GRAFTING FOR LEFT MAIN CORONARY ARTERY DISEASE: ANALYSIS FROM THE EXCEL TRIAL
Authors: Shmuel Chen, Bjorn Redfors, Patrick Serruys, A. Kappetein, Aaron Crowley, Ori Ben-Yehuda, Iva Srdanovic, Nicholas Lembo, W. Brown, Joseph Sabik, Gregg Stone
Journal: Journal of the American College of Cardiology (2019)
Abstract 15772: Cardiogenic Shock in Takotsubo Syndrome
Authors: Davide Di Vece, Rodolfo Citro, Victoria L Cammann, Ken Kato, Sebastiano Gili, Konrad A Szawan, Jozef Micek, Stjepan Jurisic, Katharina J Ding, Beatrice Bacchi, Moritz Schwyzer, Alessandro Candreva, Jelena R Ghadri, Christian Templin
Journal: Circulation (2018)

Background: The clinical course of takotsubo syndrome (TTS) can be complicated by several life-threatening conditions, including cardiogenic shock (CS). To date, the impact of CS on outcomes of pat...
18F-Fluoride Positron Emission Tomographic Imaging of Penile Arteries and Erectile Dysfunction
Authors: Takehiro Nakahara, Jagat Narula, Jan G.P. Tijssen, Sunil Agarwal, Mohammed M. Chowdhury, Patrick A. Coughlin, Marc R. Dweck, James H.F. Rudd, Masahiro Jinzaki, John Mulhall, H. William Strauss
Journal: Journal of the American College of Cardiology (2019)
Economic evaluation of a tailored therapist‐guided internet‐based cognitive behavioural treatment for patients with psoriasis: a randomized controlled trial
Authors: S. Beugen, M. Ferwerda, H. Middendorp, J.V. Smit, M.E.J. Zeeuwen‐Franssen, E.B.M. Kroft, E.M.G.J. Jong, P.C.M. Kerkhof, W. Kievit, A.W.M. Evers
Journal: British Journal of Dermatology (2019)
A SWITCH IN PLATELET SURFACE RECEPTOR SIGNALING AT THE TIME OF MYOCARDIAL INFRACTION OCCURS IN HUMANS AND MICE: IMPLICATIONS FOR ANTI-PLATELET THERAPY IN MEN AND WOMEN
Authors: Scott J. Cameron, Hamza Sadhra, Beom Soo Kim, David Auerbach, Frederick Ling, Amy Mohan, Sara Ture, David Rich, Craig Morrell
Journal: Journal of the American College of Cardiology (2019)
ASSOCIATION OF HIGH HDL IN PATIENTS WITH CORONARY ARTERY DISEASE: IMPACT OF RACE AND SEX
Authors: Devinder Dhindsa, Pratik Sandesara, Anurag Mehta, Matthew Topel, Marc Allard-Ratick, Wesley O'Neal, Jay Khambhati, Beka Bakthadze, Ayman Samman Tahhan, Salim Hayek, Laurence S. Sperling, Arshed Quyyumi
Journal: Journal of the American College of Cardiology (2019)
PREDICTION OF CARDIAC RESYNCHRONIZATION THERAPY RESPONSE IN DILATED CARDIOMYOPATHY USING VORTEX FLOW MAP OF CINE MAGNETIC RESONANCE IMAGING
Authors: Risako Nakao, Michinobu Nagao, Kenji Fukushima, Akiko Sakai, Eri Watanabe, Nobuhisa Hagiwara
Journal: Journal of the American College of Cardiology (2019)
LEFT VENTRICULAR END DIASTOLIC PRESSURES ARE ASSOCIATED WITH SIX MONTH MORTALITY RATES FOLLOWING TRANS-CATHETER AORTIC VALVE REPLACEMENT
Authors: Yaron Arbel, Yishay Szekely, Amir Halkin, Shmuel Banai, Samuel Bazan, Ariel Finkelstein
Journal: Journal of the American College of Cardiology (2019)