tbuckley/cabot-search huggingface dataset:
What does best practice care for musculoskeletal pain look like? Eleven consistent recommendations from high-quality clinical practice guidelines: systematic review
Authors: Ivan Lin, Louise Wiles, Rob Waller, Roger Goucke, Yusuf Nagree, Michael Gibberd, Leon Straker, Chris G Maher, Peter P B O’Sullivan
Journal: British Journal of Sports Medicine (2019)

Objectives To identify common recommendations for high-quality care for the most common musculoskeletal (MSK) pain sites encountered by clinicians in emergency and primary care (spinal (lumbar, thoracic and cervical), hip/knee (including osteoarthritis [OA] and shoulder) from contemporary, high-quality clinical practice guidelines (CPGs). Design Systematic review, critical appraisal and narrative synthesis of MSK pain CPG recommendations. Eligibility criteria Included MSK pain CPGs were written in English, rated as high quality, published from 2011, focused on adults and described development processes. Excluded CPGs were for: traumatic MSK pain, single modalities (eg, surgery), traditional healing/medicine, specific disease processes (eg, inflammatory arthropathies) or those that required payment. Data sources Four scientific databases (MEDLINE, Embase, CINAHL and Physiotherapy Evidence Database) and four guideline repositories. Results 6232 records were identified, 44 CPGs were appraised and 11 were rated as high quality (low back pain: 4, OA: 4, neck: 2 and shoulder: 1). We identified 11 recommendations for MSK pain care: ensure care is patient centred, screen for red flag conditions, assess psychosocial factors, use imaging selectively, undertake a physical examination, monitor patient progress, provide education/information, address physical activity/exercise, use manual therapy only as an adjunct to other treatments, offer high-quality non-surgical care prior to surgery and try to keep patients at work. Conclusion These 11 recommendations guide healthcare consumers, clinicians, researchers and policy makers to manage MSK pain. This should improve the quality of care of MSK pain.
TRANSCATHETER AORTIC VALVE REPLACEMENT VERSUS SURGICAL AORTIC VALVE REPLACEMENT OUTCOMES IN PATIENTS WITH PULMONARY HYPERTENSION AND SEVERE AORTIC VALVE STENOSIS
Authors: Yasser Al-Khadra, Fahed Darmoch, Homam Moussa Pacha, Mohamad Soud, Amir Kaki, M. Chadi Alraies
Journal: Journal of the American College of Cardiology (2019)
CARDIOVASCULAR OUTCOMES AND MORTALITY AFTER INITIATION OF CANAGLIFLOZIN VERSUS NON-SGLT2I ANTIHYPERGLYCEMIC AGENTS: ADDITIONAL ANALYSES FROM THE EASEL POPULATION-BASED COHORT STUDY
Authors: Jacob A. Udell, Zhong Yuan, Patrick Ryan, Toni Rush, Nicholas M. Sicignano, Michael Galitz, Norman Rosenthal
Journal: Journal of the American College of Cardiology (2019)
Prediction of Response to Fostamatinib Based on the Presence of Plasma Platelet Autoantibodies in Adult Patients with Immune Thrombocytopenia (ITP) — Exploratory Analyses from Phase 3 Studies
Authors: Vadim V. Markovtsov, Sothy Yi, Chi Young, Anne-Marie Duliege, Esteban Masuda
Journal: Blood (2018)
Abstract 17498: Cardiac-Specific Thioredoxin-1 Knockout Induces Cardiac Dysfunction
Authors: Shinichi Oka, Kevin Schesing, Narayani Nagarajan, Jaemin Byun, Shohei Ikeda, Wataru Mizushima, Junichi Sadoshima
Journal: Circulation (2017)

Thioredoxin-1 (Trx1) is a 12 KDa oxidoreductase, which reduces proteins with disulfide bonds through thiol-disulfide exchange reactions. Several lines of evidences show that Trx1 protects cardiomyocytes against stress, such as ischemia and pressure overload. However, due to the embryonic lethality of systemic Trx1 knockout mice, the role of endogenous Trx1 in cardiac function had not been tested with a loss-of-function model. In this study, we generated cardiac-specific Trx1 knockout (Trx1cKO) mice with Trx1floxflox and αMHC-Cre transgenic mice to elucidate the physiological role of Trx1. The protein levels of Trx1 were significantly reduced in Trx1cKO mice in a cardiac-specific manner. The Trx1cKO mice were viable but started dying at 2 weeks of age. The median survival age was 3.6 weeks. None of the mice survived for longer than 8 weeks. Histochemical analyses showed a trend of increased cardiomyocyte cell size in Trx1cKO mice (relative cell size: Wild-type (WT): 1; Trx1cKO: 1.8), whereas apoptotic cell...
NCAM1 (CD56) promotes leukemogenesis and confers drug resistance in AML
Authors: Daniel Sasca, Jakub Szybinski, Andrea Schüler, Viral Shah, Jan Heidelberger, Patricia S. Haehnel, Anna Dolnik, Oliver Kriege, Eva-Marie Fehr, Wolf H. Gebhardt, George Reid, Claudia Scholl, Matthias Theobald, Lars Bullinger, Petra Beli, Thomas Kindler
Journal: Blood (2019)
Abstract 106: Identifying Cardiac Conduction System-specific Enhancers That Link Cell-type Specification With Human Conduction Defects
Authors: Samadrita Bhattacharyya, Minoti Bhakta, Lin Wang, Nikhil V Munshi
Journal: Circulation Research (2018)

The Cardiac Conduction System (CCS) is composed of a small set of specialized cardiomyocytes (CMs) that generate and propagate the electrical impulse required for contraction of the cardiac chambers. Genome Wide Association Studies (GWAS) have identified nearly fifty cardiac rhythm associated Single Nucleotide Polymorphisms (SNPs), and recent reports have characterized how selected SNPs impact CCS gene regulatory networks to influence cardiac conduction. However, a global view of CCS regulatory networks remains to be elucidated, and the vast majority of cardiac conduction associated SNPs remain uncharacterized. Such in-depth analyses have been hampered by our inability to specifically interrogate genome accessibility and RNA expression in the CCS. In order to identify genome-wide cis regulatory elements that correlate with CCS cell-type specification, we generated and rigorously characterized novel knock-in (KI) Cre driver lines that label the Atrio-Ventricular Node (AVN) and ventricular conduction system (VCS) to add to the limited CCS-specific genetic toolkit. In our study, we also used a previously characterized Shox2 KI-Cre driver to label the sinoatrial node (SAN), the primary pacemaker of the heart. Given a variety of issues associated with FACS and Laser Capture Microdissection, we optimized the INTACT (Isolation of Nuclei TAgged in specific Cell Types) method for mammalian heart tissue. To isolate cell-type-specific nuclei, we used Rosa26-Sun1-myc-sfGFP-Tag mice in combination with CCS-specific Cre driver lines. Following isolation and enrichment of CCS-specific nuclei, we performed Omni-ATAC-seq to identify open regions of chromatin (e.g. enhancer elements) and differentially enriched transcription factor motifs for individual CCS cellular sub-types without prior knowledge of cell-type specific regulators. Most cardiac transcriptomes in the literature do not distinguish between individual subtypes. Therefore, using bulk nuclear RNA-Seq, we also generated CCS-subtype specific transcriptomes. We anticipate that our datasets will provide an important starting point for establishing mechanistic links between CCS-specific enhancer elements and disease-associated SNPs identified by GWAS.
COMMON PATHOPHYSIOLOGICAL PATHWAYS IN WOMEN WITH ISCHEMIA AND NO OBSTRUCTIVE CORONARY ARTERY DISEASE AND HEART FAILURE WITH PRESERVED EJECTION FRACTION: A REPORT FROM THE WOMEN'S ISCHEMIA SYNDROME EVALUATION-CORONARY VASCULAR DYSFUNCTION CONTINUATION STUDY
Authors: Haider Aldiwani, Janet Wei, Michael Nelson, Nissi Suppogu, Galen Cook-Wiens, Behzad Sharif, Lidia Szczepaniak, Louise Thomson, Balaji Tamarappoo, Chrisandra Shufelt, Daniel Berman, C. Noel Bairey Merz
Journal: Journal of the American College of Cardiology (2019)
Guidelines for Laboratory Evaluation in the Diagnosis of Lyme Disease
Journal: Annals of Internal Medicine (1997)

Lyme disease is the most common tick-borne disease in North America. From 1982 to 1994, more than 70 000 cases were reported in North America, most of them in the United States. It is important that clinicians diagnose Lyme disease correctly because efficacious therapy is available and delayed or inadequate treatment may lead to various morbid sequelae. On the other hand, inappropriate testing and therapy are costly and expose the patient to risk of the adverse effects of administered antibiotics.
VALUE OF SURFACE ELECTROCARDIOGRAPHIC CRITERIA IN LOCALIZING ORIGINS OF OUTFLOW TRACT VENTRICULAR ARRHYTHMIAS
Authors: Hung Manh Pham, Quang Nguyen, Van Dong Tran, Viet Phong Dang, Linh Dinh, Nhat Minh Pham
Journal: Journal of the American College of Cardiology (2019)
DOES RIGHT VENTRICULAR DILATION AFTER PULMONARY VALVE REPLACEMENT PREDICT ADVERSE CLINICAL OUTCOMES IN PATIENTS WITH REPAIRED TETRALOGY OF FALLOT?
Authors: Tony Pastor, Lynn Sleeper, Valeria E. Duarte, Sheila Drakeley, Minmin Lu, Tal Geva, Anne Marie Valente
Journal: Journal of the American College of Cardiology (2019)
Abstract 13535: Investigating Subtypes of the Metabolic Syndrome among US Hispanics/Latinos in Relation to Coronary Heart Disease Prevalence: Preliminary Results from the Hispanic Community Health Study/Study of Latinos (HCHS/SOL)
Authors: William Arguelles, Maria M Llabre, Frank J Penedo, Ralph L Sacco, David J Lee, Mercedes R Carnethon, Diane Catellier, Linda C Gallo, Hector M González, Christina K Holub, Laura R Loehr, Elsayed Z Soliman, Neil Schneiderman
Journal: Circulation (2012)

The validity and clinical utility of metabolic syndrome (MetS) diagnosis has been debated. MetS component cut-off scores may be viewed as arbitrary, and whether meaningful subtypes of MetS exist re...
Higher Expression of PALB2 Predict Poor Prognosis in AML Patients and Identifies Potential Targets of Synthetic Lethal Therapies
Authors: Antonella Padella, Giovanni Marconi, Giorgia Simonetti, Andrea Ghelli Luserna Di Rora, Maria Chiara Fontana, Cristina Papayannidis, Emanuela Ottaviani, Michele Cavo, Simona Soverini, Giovanni Martinelli
Journal: Blood (2018)
Abstract 287: Cardioprotection in Mice With a Knock-in Mutation (C202S) in Cyclophilin D
Authors: Georgios Amanakis, Junhui Sun, Jennifer Boylston, Chengyu Liu, Jeffery Molkentin, Elizabeth Murphy
Journal: Circulation Research (2018)

Our previous study in mouse embryonic fibroblasts showed that cysteine 202 of cyclophilin D (CypD) is necessary for redox stress-induced activation of the mitochondrial permeability transition pore (PTP). To further investigate the function of this cysteine residue in situ, we used CRISPR to develop a knock-in mouse model (C57BL/6N strain), where CypD cysteine 202 was mutated to a serine (C202S). The amount of total CypD expressed in CypD C202S mouse hearts did not differ compared to the wild-type (WT). However, CypD C202S hearts show significantly less ischemia-reperfusion (I/R) injury in the Langendorff perfused heart model; myocardial infarct size was decreased versus WT (24.5±4.7% CypD C202S vs 49.8±2.7% WT, p=0.003, n=4). Isolated cardiac mitochondria from CypD C202S mice also have a higher calcium retention capacity compared to WT (213.3±16.7 vs 140.0±20.8 umol Ca +2 /g protein, p=0.033, n=4). We previously found C202 to undergo S-nitrosylation with preconditioning and hypothesized that oxidation might compete with S-nitrosylation and might target CypD to the mitochondrial membrane to activate the PTP. Indeed, isolated cardiac mitochondria subjected to oxidative stress exhibit less binding of CypD C202S to the proposed PTP component ATP synthase compared to WT (78.7±6.9% vs WT, p=0.038, n=3). Cysteine 202 also matches an S-palmitoylation site motif found commonly in soluble proteins (-C--L-). To assess palmitoylation of C202 we employed a resin-assisted capture. Langendorff perfused mouse hearts were first treated with N-ethylmaleimide to block free cysteines, and then with hydroxylamine to free cysteines from palmitate. Free cysteines were subsequently captured by a resin. At baseline, CypD C202S exhibits less palmitoylation compared to WT (37.7±1.98% vs WT, p&lt;0.001, n=6) which might alter its membrane association and desensitize PTP. We are currently testing whether alterations in palmitoylation of C202 regulate PTP opening and are evaluating the physiological role of these post-translational modifications. In conclusion, these results show that mutated CypD C202S affords cardioprotection against I/R injury and that oxidation, S-nitrosylation, and S-palmitoylation of C202 are potential regulators of CypD and PTP.
COMPARISON OF MAJOR BLEEDING- AND STROKE-RELATED HOSPITAL READMISSIONS AMONG HOSPITALIZED NONVALVULAR ATRIAL FIBRILLATION PATIENTS TREATED WITH WARFARIN AND APIXABAN IN THE US
Authors: Steven Bruce Deitelzweig, Christine Baker, Amol Dhamane, Jack Mardekian, Oluwaseyi Dina, Lisa Rosenblatt, Cristina Russ, Tayla Poretta, Melissa Lingohr-Smith, Jay Lin
Journal: Journal of the American College of Cardiology (2019)
THE PREDICTIVE ROLE OF VISIT-TO-VISIT GLOMERULAR FILTRATION RATE VARIABILITY FOR CARDIOVASCULAR AND RENAL OUTCOMES IN ESSENTIAL HYPERTENSION: DATA FROM A GREEK 8-YEAR-FOLLOW-UP STUDY
Authors: Konstantinos P. Tsioufis, Kyriakos Dimitriadis, Alexandros Kasiakogias, Georgios Drosos, Nikos Karaminas, Panagiotis Iliakis, Katerina Damianaki, Dimitris Tousoulis
Journal: Journal of the American College of Cardiology (2019)
ULTRASOUND-FACILITATED THROMBOLYSIS AND RHEOLYTIC THROMBECTOMY IN PATIENTS WITH PULMONARY EMBOLISM AT HIGH RISK OR INTERMEDIATE-HIGH RISK: A SINGLE-CENTER EXPERIENCE
Authors: Ozgur Akbal, Cihangir Kaymaz, Aykun Hakgor, Seda Tanyeri, Ali Karagoz, Berhan Keskin, Hacer Ceren Tokgoz, Seyhmus Kulahcioglu, Emrah Erdogan, Sevim Turkday, Halil Tanboga, Cem Dogan, Zubeyde Bayram, Rezzan Deniz Acar, Nihal Ozdemir
Journal: Journal of the American College of Cardiology (2019)
OUTCOMES OF THORACIC ENDOVASCULAR AORTIC REPAIR FOR ACUTE UNCOMPLICATED STANFORD TYPE-B AORTIC DISSECTION: A SINGLE-CENTER EXPERIENCE WITH 522 CASES
Authors: Yi Zhu, Binquan Hu, Songyuan Luo, Huanyu Ding, Yuan Liu, Wenhui Huang, Nianjin Xie, Ling Xue, Jianfang Luo
Journal: Journal of the American College of Cardiology (2019)
EARLY DETECTION OF ANTHRACYCLINE CARDIOTOXICITY IN POPULATIONS WITH PREVALENT UNRELATED CARDIAC DISORDERS
Authors: Nathaniel Reichek, Derek Knight, Liana Haigis, Jonathan Weber, Alim Merchant, Marie Grgas, Yang Cheng, Sherly Jacob-Perez, Haoyi Zheng, Bhoomi Mehrotra
Journal: Journal of the American College of Cardiology (2019)
IMPACT OF RIGHT VENTRICULAR BRANCH SLOW FLOW PHENOMENON POST PERCUTANEOUS CORONARY INTERVENTION FOR ACUTE CORONARY SYNDROME TO PREDICT FUTURE PROLONGED RIGHT-VENTRICULAR DYSFUNCTION
Authors: Koshi Setoyama, Shinjo Sonoda, Konosuke Inoue, Toshiya Miura, Akiyoshi Shimizu, Reo Anai, Yoshinori Sanuki, Yuki Tsuda, Masaru Araki, Yutaka Otsuji
Journal: Journal of the American College of Cardiology (2019)
Association of Adding Aspirin to Warfarin Therapy Without an Apparent Indication With Bleeding and Other Adverse Events
Authors: Jordan K. Schaefer, Yun Li, Xiaokui Gu, Nicole M. Souphis, Brian Haymart, Eva Kline-Rogers, Steven L. Almany, Scott Kaatz, Jay H. Kozlowski, Gregory D. Krol, Suman L. Sood, James B. Froehlich, Geoffrey D. Barnes
Journal: JAMA Internal Medicine (2019)

It is not clear how often patients receive aspirin (acetylsalicylic acid) while receiving oral anticoagulation with warfarin sodium without a clear therapeutic indication for aspirin, such as a mechanical heart valve replacement, recent percutaneous coronary intervention, or acute coronary syndrome. The clinical outcomes of such patients treated with warfarin and aspirin therapy compared with warfarin monotherapy are not well defined to date.To evaluate the frequency and outcomes of adding aspirin to warfarin for patients without a clear therapeutic indication for combination therapy.A registry-based cohort study of adults enrolled at 6 anticoagulation clinics in Michigan (January 1, 2010, to December 31, 2017) who were receiving warfarin therapy for atrial fibrillation or venous thromboembolism without documentation of a recent myocardial infarction or history of valve replacement.Aspirin use without therapeutic indication.Rates of any bleeding, major bleeding events, emergency department visits, hospitalizations, and thrombotic events at 1, 2, and 3 years.Of the study cohort of 6539 patients (3326 men [50.9%]; mean [SD] age, 66.1 [15.5] years), 2453 patients (37.5%) without a clear therapeutic indication for aspirin were receiving combination warfarin and aspirin therapy. Data from 2 propensity score-matched cohorts of 1844 patients were analyzed (warfarin and aspirin vs warfarin only). At 1 year, patients receiving combination warfarin and aspirin compared with those receiving warfarin only had higher rates of overall bleeding (cumulative incidence, 26.0%; 95% CI, 23.8%-28.3% vs 20.3%; 95% CI, 18.3%-22.3%; P < .001), major bleeding (5.7%; 95% CI, 4.6%-7.1% vs 3.3%; 95% CI, 2.4%-4.3%; P < .001), emergency department visits for bleeding (13.3%; 95% CI, 11.6%-15.1% vs 9.8%; 95% CI, 8.4%-11.4%; P = .001), and hospitalizations for bleeding (8.1%; 6.8%-9.6% vs 5.2%; 4.1%-6.4%; P = .001). Rates of thrombosis were similar, with a 1-year cumulative incidence of 2.3% (95% CI, 1.6%-3.1%) for those receiving combination warfarin and aspirin therapy compared with 2.7% (95% CI, 2.0%-3.6%) for those receiving warfarin alone (P = .40). Similar findings persisted during 3 years of follow-up as well as in sensitivity analyses.Compared with warfarin monotherapy, receipt of combination warfarin and aspirin therapy was associated with increased bleeding and similar observed rates of thrombosis. Further research is needed to better stratify which patients may benefit from aspirin while anticoagulated with warfarin for atrial fibrillation or venous thromboembolism; clinicians should be judicious in selecting patients for combination therapy.
HIGHER ONE-YEAR MORTALITY IN PATIENTS WITH DEPRESSION REFERRED FOR CARDIAC CATHETERIZATION
Authors: Mohammed Al-Hijji, Nadeem Khan, S. Gharacholou, Rajiv Gulati, Robert Widmer, Abdallah El Sabbagh, Garrett Welle, Amrit Kanwar, Ryan Lennon, Amir Lerman, Mandeep Singh
Journal: Journal of the American College of Cardiology (2019)
THE DIAGNOSTIC VALUE OF C-REACTIVE PROTEIN TO TROPONIN RATIO FOR THE DIFFERENTIATION OF PERIMYOCARDITIS FROM ACUTE MYOCARDIAL INFARCTION
Authors: Simcha R. Meisel, Jamil Muhassen, Naama Amsalem, Rami Abu Fanne, Carmel Levin, Aaron Frimerman, Aya Asif, Yaniv Levi, David Blondheim, Avraham Shotan, Michael Kleiner Shochat
Journal: Journal of the American College of Cardiology (2019)
CORONARY ARTERY BYPASS GRAFTING USING RADIAL ARTERY OR SAPHENOUS VEIN VERSUS PERCUTANEOUS CORONARY INTERVENTION
Authors: Umberto Benedetto, Mario Gaudino, Rafael Cavalcante, Massimo Mancone, Jung-Min Ahn, Matthew Wingo, Irbaz Hameed, A. Kappetein, Friedrich W. Mohr, Seung-Jung Park, Patrick W. Serruys, David Taggart
Journal: Journal of the American College of Cardiology (2019)
THE RISK OF SUDDEN CARDIAC DEATH AMONG HEART FAILURE PATIENTS WITH HIV
Authors: Raza M. Alvi, Anne M. Neilan, Noor Tariq, Malek Z.O. Hassan, Magid Awadalla, Maryam Afshar, Adam Rokicki, Connor Mulligan, Virginia A. Triant, Markella V. Zanni, Tomas G. Neilan
Journal: Journal of the American College of Cardiology (2019)
Risk Factors for Kidney Disease in Type 1 Diabetes
Authors: Bruce A. Perkins, Ionut Bebu, Ian H. de Boer, Mark Molitch, William Tamborlane, Gayle Lorenzi, William Herman, Neil H. White, Rodica Pop-Busui, Andrew D. Paterson, Trevor Orchard, Catherine Cowie, John M. Lachin
Journal: Diabetes Care (2019)

OBJECTIVE In type 1 diabetes (T1D), the course of microalbuminuria is unpredictable and timing of glomerular filtration rate (GFR) loss is uncertain. Thus, there is a need to identify the risk factors associated with the development of more advanced stages of kidney disease through large, long-term systematic analysis. RESEARCH DESIGN AND METHODS Multivariable Cox proportional hazards models assessed the association of baseline and time-dependent glycemic and nonglycemic risk factors for incident macroalbuminuria and reduced estimated GFR (eGFR; defined as &amp;lt;60 mL/min/1.73 m2) over a mean of 27 years in the Diabetes Control and Complications Trial (DCCT) cohort. RESULTS Higher mean HbA1c (hazard ratio [HR] 1.969 per 1% higher level [95% CI 1.671–2.319]) and male sex (HR 2.767 [95% CI 1.951–3.923]) were the most significant factors independently associated with incident macroalbuminuria, whereas higher mean triglycerides, higher pulse, higher systolic blood pressure (BP), longer diabetes duration, higher current HbA1c, and lower mean weight had lower magnitude associations. For incident reduced eGFR, higher mean HbA1c (HR 1.952 per 1% higher level [95% CI 1.714–2.223]) followed by higher mean triglycerides, older age, and higher systolic BP were the most significant factors. CONCLUSIONS Although several risk factors associated with macroalbuminuria and reduced eGFR were identified, higher mean glycemic exposure was the strongest determinant of kidney disease among the modifiable risk factors. These findings may inform targeted clinical strategies for the frequency of screening, prevention, and treatment of kidney disease in T1D.
Upregulation of Tissue Factor May Contribute to Thrombosis in Polycythemia Vera and Essential Thrombocythemia
Authors: Brandi Reeves, Soo Jin Kim, Jihyun Song, Radhika Gangaraju, Rafal Pawlinski, Perumal Thiagarajan, Victor R. Gordeuk, Nigel S. Key, Josef T. Prchal
Journal: Blood (2018)
LACK OF OBSTRUCTIVE CORONARY ARTERY DISEASE ON INITIAL TESTING WITH CORONARY CT ANGIOGRAPHY (CCTA) IN SYMPTOMATIC PATIENTS LOWERS TOTAL AND DOWNSTREAM COSTS OVER A 5 YEAR PERIOD COMPARED TO NORMAL MYOCARDIAL PERFUSION IMAGING
Authors: Laith Dinkha, Jessica Rouse, Cory Madigan, Susan Williams, Brandon Walker, Andrew Wyatt, Samuel Farrell, Chu Chu, Mitchell Bowman, Dustin Thomas
Journal: Journal of the American College of Cardiology (2019)
LATE GADOLINIUM ENHANCEMENT CARDIAC MAGNETIC RESONANCE IMAGING (CMR) TISSUE CHARACTERIZATION FOR CANCER-ASSOCIATED CARDIAC MASSES: METABOLIC AND PROGNOSTIC MANIFESTATIONS IN RELATION TO WHOLE BODY POSITRON EMISSION TOMOGRAPHY (PET)
Authors: Angel Chan, Josef Fox, Rocio Perez Johnston, Jiwon Kim, John Grizzard, Raymond Kim, Chaya Moskowitz, Richard Steingart, Jonathan Weinsaft
Journal: Journal of the American College of Cardiology (2019)