tbuckley/cabot-search huggingface dataset:
Pals1 Haploinsufficiency Results in Proteinuria and Cyst Formation
Authors: Thomas Weide, Beate Vollenbröker, Ulf Schulze, Ivona Djuric, Maria Edeling, Jakob Bonse, Florian Hochapfel, Olga Panichkina, Dirk-Oliver Wennmann, Britta George, Seonhee Kim, Christoph Daniel, Jochen Seggewiß, Kerstin Amann, Wilhelm Kriz, Michael P. Krahn, Hermann Pavenstädt
Journal: Journal of the American Society of Nephrology (2017)

The nephron is the basic physiologic subunit of the mammalian kidney and is made up of several apicobasally polarized epithelial cell types. The process of apicobasal polarization in animal cells is controlled by the evolutionarily conserved Crumbs (CRB), Partitioning-defective, and Scribble protein complexes. Here, we investigated the role of protein associated with LIN-7 1 (Pals1, also known as Mpp5), a core component of the apical membrane–determining CRB complex in the nephron. Pals1 interacting proteins, including Crb3 and Wwtr1/Taz, have been linked to renal cyst formation in mice before. Immunohistologic analysis revealed Pals1 expression in renal tubular cells and podocytes of human kidneys. Mice lacking one Pals1 allele (functionally haploid for Pals1) in nephrons developed a fully penetrant phenotype, characterized by cyst formation and severe defects in renal barrier function, which led to death within 6–8 weeks. In Drosophila nephrocytes, deficiency of the Pals1 ortholog caused alterations in slit-diaphragm–like structures. Additional studies in epithelial cell culture models revealed that Pals1 functions as a dose-dependent upstream regulator of the crosstalk between Hippo- and TGF- β –mediated signaling. Furthermore, Pals1 haploinsufficiency in mouse kidneys associated with the upregulation of Hippo pathway target genes and marker genes of TGF- β signaling, including biomarkers of renal diseases. These findings support a link between apical polarity proteins and renal diseases, especially renal cyst diseases. Further investigation of the Pals1-linked networks is required to decipher the mechanisms underlying the pathogenesis of these diseases.
Imatinib in Children with Newly Diagnosed Chronic Phase Chronic Myelogenous Leukemia (CP CML): AAML0123 COG Study.
Authors: Martin A. Champagne, Cecilia Fu, Myron Chang, Linda Cooley, Nyla A. Heerema, Charlotte Wood, Mary Ellen French, Franklin O. Smith, Mark L. Bernstein
Journal: Blood (2006)
A Comprehensive Immunohistochemical Approach of AKT/mTOR Pathway and p-STAT3 in Mycosis Fungoides.
Authors: Marina Siakantaris, Georgia Levidou, Theodora Papadaki, Evangelia Papadavid, Theodoros P. Vassilakopoulos, Maria K. Angelopoulou, Leonidas Marinos, Vassiliki Nikolaou, Aphrodite Economidi, Christina Antoniou, Efstratios Patsouris, G.A. Pangalis, Penelope Korkolopoulou
Journal: Blood (2012)
Tropomodulin3-Null Mice Are Embryonic Lethal with Anemia Due to Defects in Erythroblast Enucleation and Erythroblast-Macrophage Islands in the Fetal Liver
Authors: Velia M. Fowler, Zhenhua Sui, Roberta B. Nowak, Nancy E. Kim, Andrea Bacconi
Journal: Blood (2012)
Breathlessness and neck swelling after a rugby game
Authors: Oliver M J Gittoes, Neil J L Gittoes
Journal: BMJ (2017)

A 16 year old boy presented to the emergency department with breathlessness and painful neck swelling after playing in a game of rugby. He had not sustained any trauma to the neck or chest. He had a history of asthma and had recently had an upper respiratory tract infection. His breathlessness was not helped by his salbutamol and he described the neck swelling and breathlessness progressively worsening since the match. What abnormalities are identified in the radiograph (fig 1⇓) and what do they signify …
ATPS-93CHEMO-NAÏVE GLIOBLASTOMA PATIENTS CONTAIN GENETICALLY HETEROGENEOUS SUB-CLONES CAPABLE OF TUMOUR PROPAGATION AND SPATIALLY HETEROGENEOUS DRUG-RESISTANT CELL POPULATIONS
Authors: Colin Watts, Sara Piccirillo, Sue Colman, Nicola Potter, Frederik van Delft, Suzanne Lillis, Maria-Jose Carnicer, Lyndal Kearney, Nicola-Jane Francis, Ashok Venkitaraman, Mel Greaves
Journal: Neuro-Oncology (2015)
Expression of HLA-G1 and G5 Enables Human Mesenchymal Stem Cells To Engraft at High Levels across Xenogeneic Barriers Following Transplantation into Immunocompetent Recipients.
Authors: Takashi Yamagami, Chad Sanada, Heinz Wiendl, Esmail D. Zanjani, Christopher D. Porada, Graça Almeida-Porada
Journal: Blood (2007)
Clinical Significance of Free Circulating CD3 and CD5 in Patients with Chronic Lymphocytic Leukemia.
Authors: Menna Hodge, Susan O’Brien, Adam Abdool, Michael Keating, Iman Jilani, Hagop Kantarjian, Alessandra Ferrajoli, Zeev Estrov, Francis Giles, Maher Albitar
Journal: Blood (2006)
Efficacy of the switch to oral antibiotics in the treatment of non- Staphylococcus aureus infective endocarditis in non-severely ill patients
Authors: S. Kernéis, A. Mzabi, C. Richaud, I. Podglajen, M.P. Fernandez-Gerlinger, J.L. Mainardi
Journal: Clinical Microbiology and Infection (2017)
Generation of Antigen Specific Cytotoxic T Lymphocytes (CTL) by Dendritic Cells (DCs) Transfected with In Vitro Transcribed (IVT) SART-1 and WT-1 mRNA.
Authors: Anri Saito, Miwako Narita, Toshio Yano, Naoko Sato, Asuka Sekiguchi, Norihiro Watanabe, Ayumi Yokoyama, Flavio Ayres, Louie Alldawi, Tatsuo Furukawa, Ken Toba, Yoko Watanabe, Yoshiaki Takahashi, Masuhiro Takahashi
Journal: Blood (2004)
Intracellular Nucleic Acid Detection in Autoimmunity
Authors: John T. Crowl, Elizabeth E. Gray, Kathleen Pestal, Hannah E. Volkman, Daniel B. Stetson
Journal: Annual Review of Immunology (2017)

Protective immune responses to viral infection are initiated by innate immune sensors that survey extracellular and intracellular space for foreign nucleic acids. The existence of these sensors raises fundamental questions about self/nonself discrimination because of the abundance of self-DNA and self-RNA that occupy these same compartments. Recent advances have revealed that enzymes that metabolize or modify endogenous nucleic acids are essential for preventing inappropriate activation of the innate antiviral response. In this review, we discuss rare human diseases caused by dysregulated nucleic acid sensing, focusing primarily on intracellular sensors of nucleic acids. We summarize lessons learned from these disorders, we rationalize the existence of these diseases in the context of evolution, and we propose that this framework may also apply to a number of more common autoimmune diseases for which the underlying genetics and mechanisms are not yet fully understood.
Bivariate Mixture Models of Serum Ferritin and Transferrin Saturation Predict Stable Components Measured 12 Years Apart in a Healthy Australian Population
Authors: Christine E. McLaren, Wen-Pin Chen, Nadine A. Bertalli, Lidija Turkovic, Martin B. Delatycki, Graham G. Giles, Dallas R. English, John L. Hopper, Katrina J. Allen, Lyle C. Gurrin
Journal: Blood (2011)
CD34+ Cell Dose Is the Major Cell Dose Determinant of Myeloid Engraftment After Single Umbilical Cord Blood Transplantation (sUCBT) for Adult Patients with High-Risk Hematologic Malignancies
Authors: Jaime Sanz, Pau Montesinos, David Martinez, Ignacio Lorenzo, Javier Palau, Luis Larrea, Dolores Planelles, Guillermo Martin, Isidro Jarque, Javier De La Rubia, Federico Moscardo, Jesus Martinez, Miguel Angel Sanz, Guillermo F Sanz
Journal: Blood (2010)
Abstract 364: Valproic Acid Induced Reprogramming of Adult Somatic Cells for Cardiac Differentiation
Authors: Zeeshan Pasha, Muhammad Ashraf
Journal: Circulation Research (2012)

Aims: Owing to the ethical concerns for use of embryonic stem cells (ESC), adult somatic cells are attractive stem cell sources for reprogramming to pluripotency. We report here non-viral approach for reprogramming of skeletal myoblasts (SM) using a small molecule. Methods and Results: SM purified from young male Oct3/4-GFP+ transgenic mouse were treated for 5 days with valproic acid (VPA), a histone deacetylase (HDAC) inhibitor. Three weeks later, GFP+ colonies of SM derived iPSC (Sk-iPS) resembling with mouse embryonic stem cells were observed and propagated in vitro. SiPS were positive for alkaline phosphatase, had normal karyotype, expressed SSEA1, and induced teratomas in nude mice containing tissue comprising all three germ layers. RT PCR analysis showed that Sk-iPS cells expressed Oct4, Sox2, KLF4, c-Myc, Nanog and ESC specific pluripotency genes. HDAC1 activity was significantly reduced in Sk-iPs generated with valproic acid treatment as compared to ES cells. Sk-iPS derived embryoid bodies (EBs) yielded spontaneously contracting cardiomyocytes with morphological, molecular, and ultrastructural features of developing cardiomyocytes. These cells were also positive for early and late cardiac markers such as myosin heavy chain, Gata4, Mef-2c and Nkx2.5, Connexin-43 (P&lt;0.01vs native SM). Micro RNA (miR) profiling showed abolition of let-7 family in Sk-iPS whereas ESC specific family of miR-290-295 was upregulated which indicated that Sk-iPS possessed miR profile similar to ESC. However muscle specific miRNAs (miR -133, -206) were identified in Sk-iPS cells as compared to ES cells indicating that the Sk-iPS retained the epigenetic memory of myogenic origin. Conclusions: We conclude that SM with endogenous expression of Sox2, KLF4, and cMyc are suitable candidates to generate iPS cells without viral vectors using a single small molecule.
Does enrollment setting influence patient attributes and outcomes in RTOG prostate cancer trials?
Authors: W. R. Lee, J. Dignam, D. Bruner, J. A. Efstathiou, Y. Yan, G. E. Hanks, M. Roach, M. V. Pilepich, H. M. Sandler
Journal: Journal of Clinical Oncology (2011)

4607 Background: The external validity of randomized trials (generalizability) has been called into question following the finding that patients treated on clinical trials vary substantially from patients treated in the usual care setting. The purpose of this secondary analysis is to examine whether results of prostate randomized trials within the RTOG differ according to enrollment setting. Methods: 4,154 patients accrued into one of 4 RTOG trials between 1987-1999 were included in this analysis. There were 224 North American institutions enrolling in one or more trials. Patient attributes were compared by enrollment settings, controlling for trial. Survival regression models stratified by trial and including patient attributes were used to evaluate survival differences by enrollment setting. Results: Academic institutions enrolled 36% of patients; community centers enrolled 49%; CCOPs enrolled 14%, and VHAs enrolled 2%. With respect to patient attributes, men from Academic and VHA centers tended to have better performance status, black men were less frequently enrolled from non-CCOP community institutions, and Gleason scores tended to be higher in men from CCOPs. Age at diagnosis did not differ materially by enrollment setting. Adjusted for patient attributes, survival by enrollment setting did not differ significantly (Table). Conclusions: Attributes of prostate cancer patients enrolled in these RTOG trials Phase III differed modestly according to enrollment site type. Accounting for these differences, overall survival did not depend on the type of institution from which patients were enrolled. These findings support the external validity of RTOG prostate trials.This project was supported by RTOG grant U10 CA21661, and CCOP grant U10 CA37422 from the National Cancer Institute (NCI). Contrast* Hazard ratio 95% confidence interval CCOP vs. academic 1.00 0.87–1.14 Other community vs. academic 0.91 0.83–1.00 VHA vs. academic 1.08 0.72–1.61 * Adjusted for age at diagnosis, race, performance status, Gleason score, node status.
Pre- and Post-National Coverage Determination (NCD) Hematologic Outcomes and Blood Utilization in Cancer Patients with Chemotherapy-Induced Anemia (CIA): Results From a Multicenter Chart Review.
Authors: David H. Henry, Corey J. Langer, R. Scott McKenzie, Catherine Tak Piech, Mekré Senbetta, Kathy L. Schulman, Edward J. Stepanski
Journal: Blood (2009)
The Immunohistochemical Pattern of Indoleamine 2,3-Dioxygenase in Tumor Tissue Indicates the Prognosis of Patients with Diffuse Large B-Cell Lymphoma.
Authors: Soranobu Ninomiya, Nobuhiro Kanemura, Hisashi Tsurumi, Takeshi Hara, Naoe Goto, Takeshi Yoshikawa, Senji Kasahara, Masahito Shimizu, Tsuyoshi Takami, Hisataka Moriwaki
Journal: Blood (2009)
Angioimmunoblastic T-Cell Lymphoma: A Clinicopathologic Study of 158 Patients Treated in GELA (Groupe d’ Etude des Lymphomes de l’ Adulte) Trials.
Authors: Nathalie Mourad, Nicolas Mounier, Josette Brière, Emmanue Raffoux, Thierry Molina, Bettina Fabiani, Tony Petrella, Jacques Diebold, Laurence de Leval, Felix Reyes, Christia Gisselbrecht, Philippe Gaulard
Journal: Blood (2006)
Downregulation of Receptor Activator of Nuclear Factor kB Ligand (RANKL) in Chronic Myeloid Leukemia Patients Receiving Imatinib Mesylate Is Dose-Dependent.
Authors: Francesco Albano, Antonella Zagaria, Luisa Anelli, Maria Guastadisegni, Alessandra Pannunzio, Giuseppina Spinosa, Mariano Rocchi, Vincenzo Liso, Giorgina Specchia
Journal: Blood (2006)
α-Tocopherol transfer protein mediates protective hypercapnia in murine ventilator-induced lung injury
Authors: Gail Otulakowski, Doreen Engelberts, Hajime Arima, Hiroyuki Hirate, Hülya Bayir, Martin Post, Brian P Kavanagh
Journal: Thorax (2017)

<h3>Rationale</h3> Hypercapnia is common in mechanically ventilated patients. Experimentally, 'therapeutic hypercapnia' can protect, but it can also cause harm, depending on the mechanism of injury. Hypercapnia suppresses multiple signalling pathways. Previous investigations have examined mechanisms that were known a priori, but only a limited number of pathways, each suppressed by CO<sub>2</sub>, have been reported. <h3>Objective</h3> Because of the complexity and interdependence of processes in acute lung injury, this study sought to fill in knowledge gaps using an unbiased screen, aiming to identify a specifically upregulated pathway. <h3>Methods and results</h3> Using genome-wide gene expression analysis in a mouse model of ventilator-induced lung injury, we discovered a previously unsuspected mechanism by which CO<sub>2</sub> can protect against injury: induction of the transporter protein for α-tocopherol, α-tocopherol transfer protein (αTTP). Pulmonary αTTP was induced by inspired CO<sub>2</sub> in two in vivo murine models of ventilator-induced lung injury; the level of αTTP expression correlated with degree of lung protection; and, absence of the αTTP gene significantly reduced the protective effects of CO<sub>2</sub>. α-Tocopherol is a potent antioxidant and hypercapnia increased lung α-tocopherol in wild-type mice, but this did not alter superoxide generation or expression of NRF2-dependent antioxidant response genes in wild-type or in αTTP<sup>−/−</sup> mice. In concordance with a regulatory role for α-tocopherol in lipid mediator synthesis, hypercapnia attenuated 5-lipoxygenase activity and this was dependent on the presence of αTTP. <h3>Conclusions</h3> Inspired CO<sub>2</sub> upregulates αTTP which increases lung α-tocopherol levels and inhibits synthesis of a pathogenic chemoattractant.
Whole Blood Donor Chimerism At Day 30 After Reduced Intensity Conditioned Allogeneic Stem-Cell Transplantation Predicts Disease Relapse, and Is Strongly Associated with Pretransplant Lymphodepletion
Authors: Ran Reshef, Rosemarie Mick, Pavel Vassilev, Jacqueline Smith, Elizabeth Hexner, Alison W. Loren, Noelle V. Frey, Steven C. Goldstein, Edward A. Stadtmauer, Selina M. Luger, David L. Porter
Journal: Blood (2011)
Identification of Non-Cardiotoxic hERG1 Blockers to Overcome Chemoresistance in Acute Lymphoblastic Leukemias
Authors: Marika Masselli, Serena Pillozzi, Massimo D'Amico, Luca Gasparoli, Olivia Crociani, Matteo Stefanini, Kennet Mugridge, Wolfang Tiedke, Andrea Becchetti, Annarosa Arcangeli
Journal: Blood (2012)
Predictive factors of failure of noninvasive mechanicalventilation in patients with acute respiratory failure in COPD
Authors: Pavel Matuška, Olga Pilařová, Jana Skřičková, Zdeněk Beránek
Journal: European Respiratory Journal (2003)

The authors analyse their experience with 41 patients with ARF
in COPD on the ICU. The APACHE score and concomitant disease,
among others, were regitred to pedict factors inluencihg the
sccess of noninvasive mechanical ventilation.
SURG-07A MULTI-INSTITUTIONAL EXPERIENCE ON LASER THERMAL ABLATION AS TREATMENT FOR CORPUS CALLOSAL GLIOBLASOTMAS
Authors: Clark Chen, Andrew Sloan, Alireza Mohammadi, Gene Barnett
Journal: Neuro-Oncology (2015)

INTRODUCTION: Surgical management of glioblastomas involving the corpus callosum is of particular challenge. Accordingly, patients afflicted with this disease entity exhibit poor survival relative to other forms of glioblastomas. The development of minimally invasive, stereotactic laser thermal ablation now offers a new surgical option against this disease. METHODS: We present a multi-institutional experience (Cleveland Clinic, University of California San Diego, University Hospital-Case Medical Center) in the treatment of glioblastoma that involve the corpus callosum. Radiographic control, peri-operative morbidities, time of hospitalization, and overall survival were examined. RESULTS: All 16 patients harbored histologically confirmed diagnosis of glioblastoma. Five patients were newly diagnosed and underwent ablation before TMZ/RT while the remaining 10 patients were treated after TMZ/RT failure. The median age of the cohort is 52 (range: 29-68), with a median KPS of 80 (range 60-100). Three patients suffered transient post-operative hemiparesis. The average length of hospital stay was 3.3 days (range: 1-11 days). 13 of the 16 patients underwent subsequent chemotherapy treatment. No tumor growth was observed within the region of thermal ablation in any of the treated patient. The median survival of the cohort has not been reached. Eleven of the treated patients (69%) survived > 3 months – the reported median survival for corpus callosal glioblastoma patients without surgical resection. The overall survival for these 11 patients ranged 5 to 35 months. CONCLUSIONS: Glioblastoma involving the corpus callosum can be safely treated through laser thermal ablation. Our multi-institutional experience provides evidence in support of safety and suggest potential efficacy in select patients.
Differential Effects of Wnt Signaling on Proliferation and Hematopoietic Support of Adult and Fetal Bone Marrow-Derived MSCs
Authors: Maja Maria Paciejewska, Marijke Maijenburg, Christian Gilissen, Kim Vermeul, Marion Kleijer, Kees Weijer, Marieke von Lindern, Ellen van der Schoot, Carlijn Voermans
Journal: Blood (2014)
Treatment of Post- HSCT Immunodeficiency By Infusion of Ex Vivo- Generated T Cell Precursors from Adult and Cord Blood Hematopoietic Stem and Progenitor Cells
Authors: Laura Simons, Corinne De La Chappedelaine, Christian Reimann, Elodie Elkaim, Sandrine Susini, Kuiying Ma, Fabien Touzot, Marina Cavazzana, Isabelle Andrè-Schmutz
Journal: Blood (2015)
Prevalence, Clinical and Instrumental Correlates of Myocardial Fibrosis and Necrosis by Delayed Contrast Enhancement Cardiovascular Magnetic Resonace in Thalassemia Major Patients
Authors: Alessia Pepe, Brunella Favilli, Vincenzo Positano, Marcello Capra, Aurelio Maggio, Carmen Lo Pinto, Anna Spasiano, Gianluca Forni, Aldo Filosa, Caterina Borgna-Pignatti, Paolo Cianciulli, Gaetano Giuffrida, Tommaso Casini, Giorgio Derchi, Massimo Lombardi
Journal: Blood (2008)
THE INCIDENCE OF SWIMMING-INDUCED PULMONARY OEDEMA DURING MASS-PARTICIPATION TRIATHLON RACES: OPTIMISING ATHLETE SAFETY
Authors: Ralph Smith, James Parkin, Nick Knight, Courtney Kipps
Journal: British Journal of Sports Medicine (2017)

Background Swimming Induced Pulmonary Edema (SIPE) is life-threatening condition that can affect healthy triathletes. There have been several reported cases amongst triathletes, however the current estimation of incidence is derived from a survey of triathletes with self-reported symptoms suggestive of SIPE. Objective To investigate the incidence of SIPE and associated risk factors in mass participation Triathlon Competitions (TC). Design A retrospective analysis of Competitors' Medical Records (CMR). Setting 11 consecutive UK-based TCs between 2011 and 2016. The competitions involved elite and non-elite competitors who raced Super Sprint, Sprint, Olympic and Olympic Plus distances. Participants CMR of patients presenting to Triathlon Medical Team (TMT) with medical complaints/injuries were analysed. Those diagnosed with SIPE were included. Diagnostic criteria included absence of water aspiration, acute onset of dyspnoea, cough and/or expectoration of frothy sputum, with evidence of pulmonary oedema on physical examination. Assessment of Risk Factors Patients' Age, gender, race distance, co-morbidities and medical management were recorded. Main Outcome Measurements The incidence of SIPE in TCs. Results 68557 competitors started the TCs and 429 competitors presented to the TMT. Five case of SIPE were recorded, giving rise to an incidence of 0.73/10,000 competitors and 1.2% of all presentations to the TMT. Mean age was 42 (21–58) and a third were female. All were non-elite athletes competing in a variety of race distances; one patient had pre-existing cardiac comorbidities; and in 3 cases participants were competing in their first triathlon. All required supplementary oxygen and transfer to hospital for definitive management. Conclusion This report is the first to describe the incidence of SIPE in mass participation triathlon competitions. Event organisers and TMTs should be prepared for competitors developing SIPE which appears not to be bound by age, gender, race distance or co-morbidities. Further research is required to identity those who are at risk of SIPE.
The Mitochondrial Metabolism Inhibitor Cpi-613 in Combination with High Dose Ara-C (HDAC) and Mitoxantrone Is Highly Active in Poor Risk Relapsed or Refractory Acute Myeloid Leukemia (AML)
Authors: Timothy Pardee, Kristin Pladna, Scott Isom, Leslie Renee Ellis, Dmitriy Berenzon, Dianna Howard, Megan Manuel, Sarah Dralle, David D Hurd, Susan Lyerly, Bayard L Powell
Journal: Blood (2015)
Usefulness of Differential Scanning Calorimetry for Monitoring Ex Vivo the Changes In Responses of CLL Cells to Anti-Cancer Drugs: Development of Personalized Therapy
Authors: Malgorzata Rogalinska, Jerzy Blonski, Pawel Goralski, Oxana Komina, Arleta Kazmierczuk, Ewa Wawrzyniak, Aleksandra Kotkowska, Pawel Robak, Henryk Piekarski, Tadeusz Robak, Zofia Kilianska, Jozefa Wesierska-Gadek
Journal: Blood (2010)
T(11;14)(q13;q32) in Mantle Cell Lymphoma Patients Is Present Only on Mature Malignant Cells. Fluorescence-Activated Cell Sorting and Fluorescence in Situ Hybridization Analysis
Authors: Vladimir I. Vorobyev, Evgenii’ M Gretsov, Tatyana N Obukhova, Elena V Domracheva, Yuri Yu Lorie, Sergey K Kravchenko, Ivan A Vorobjev
Journal: Blood (2011)
Melphalan Containing Cytoreduction Results in a Good Overall Survival (OS) in Pediatric Patients with Relapse or Refractory AML Undergoing Unrelated Umbilical Cord Blood Transplantation (UCBT).
Authors: Vinod K. Prasad, Juan Wu, Paul L. Martin, Timothy A. Driscoll, Suhag H. Parikh, Deborah Semmel, Jerelyn Moffet, Lauren Stafford, Ann Shonkwiler, Shelly Carter, Paul Szabolcs, Joanne Kurtzberg
Journal: Blood (2006)
Stromal Tumor Microenvironment in CLL: Regulation of Leukemic Progression
Authors: Nagendra K Chaturvedi, Adam K Ahrens, Ashima Shukla, Christine E Gilling, Amit K. Mittal, Philip Bierman, Dennis D Weisenburger, Runqing Lu, Shantaram S Joshi
Journal: Blood (2012)