tbuckley/cabot-search huggingface dataset:
Oxysterol Sensing through the Receptor GPR183 Promotes the Lymphoid-Tissue-Inducing Function of Innate Lymphoid Cells and Colonic Inflammation
Authors: Johanna Emgård, Hana Kammoun, Bethania García-Cassani, Julie Chesné, Sara M. Parigi, Jean-Marie Jacob, Hung-Wei Cheng, Elza Evren, Srustidhar Das, Paulo Czarnewski, Natalie Sleiers, Felipe Melo-Gonzalez, Egle Kvedaraite, Mattias Svensson, Elke Scandella, Matthew R. Hepworth, Samuel Huber, Burkhard Ludewig, Lucie Peduto, Eduardo J. Villablanca, Henrique Veiga-Fernandes, João P. Pereira, Richard A. Flavell, Tim Willinger
Journal: Immunity (2018)

Group 3 innate lymphoid cells (ILC3s) sense environmental signals and are critical for tissue integrity in the intestine. Yet, which signals are sensed and what receptors control ILC3 function remain poorly understood. Here, we show that ILC3s with a lymphoid-tissue-inducer (LTi) phenotype expressed G-protein-coupled receptor 183 (GPR183) and migrated to its oxysterol ligand 7α,25-hydroxycholesterol (7α,25-OHC). In mice lacking Gpr183 or 7α,25-OHC, ILC3s failed to localize to cryptopatches (CPs) and isolated lymphoid follicles (ILFs). Gpr183 deficiency in ILC3s caused a defect in CP and ILF formation in the colon, but not in the small intestine. Localized oxysterol production by fibroblastic stromal cells provided an essential signal for colonic lymphoid tissue development, and inflammation-induced increased oxysterol production caused colitis through GPR183-mediated cell recruitment. Our findings show that GPR183 promotes lymphoid organ development and indicate that oxysterol-GPR183-dependent positioning within tissues controls ILC3 activity and intestinal homeostasis.
Authorship trends in The Lancet Global Health
Authors: Adithi R Iyer
Journal: The Lancet Global Health (2018)

One of The Lancet Global Health's stated purposes is to represent “disadvantaged populations” in health-related scenarios around the world. To analyse to what extent the journal succeeds in its aim, I studied all research articles spanning the existence of the journal (June, 2013–July, 2017). I calculated what proportion of articles in the journal covered a research project in sub-Saharan Africa, the Middle East and north Africa, east Asia, south Asia, southeast Asia and Oceania, Europe, and the Americas, and what proportion of the contributing authors of an article was affiliated with or came from low-income and middle-income countries (LMICs).
Effect of Bariatric Surgery on CKD Risk
Authors: Allon N. Friedman, Abdus S. Wahed, Junyao Wang, Anita P. Courcoulas, Gregory Dakin, Marcelo W. Hinojosa, Paul L. Kimmel, James E. Mitchell, Alfons Pomp, Walter J. Pories, Jonathan Q. Purnell, Carel le Roux, Konstantinos Spaniolas, Kristine J. Steffen, Richard Thirlby, Bruce Wolfe
Journal: Journal of the American Society of Nephrology (2018)

Obesity is linked to the development and progression of CKD, but whether bariatric surgery protects against CKD is poorly understood. We, therefore, examined whether bariatric surgery influences CKD risk. The study included 2144 adults who underwent bariatric surgery from March of 2006 to April of 2009 and participated in the Longitudinal Assessment of Bariatric Surgery-2 Study cohort. The primary outcome was CKD risk categories as assessed by the Kidney Disease Improving Global Outcomes (KDIGO) consortium criteria using a combination of eGFR and albuminuria. Patients were 79% women and 87% white, with a median age of 46 years old. Improvements were observed in CKD risk at 1 and 7 years after surgery in patients with moderate baseline CKD risk (63% and 53%, respectively), high baseline risk (78% and 56%, respectively), and very high baseline risk (59% and 23%, respectively). The proportion of patients whose CKD risk worsened was ≤10%; five patients developed ESRD. Sensitivity analyses using year 1 as baseline to minimize the effect of weight loss on serum creatinine and differing eGFR equations offered qualitatively similar results. Treatment with bariatric surgery associated with an improvement in CKD risk categories in a large proportion of patients for up to 7 years, especially in those with moderate and high baseline risk. These findings support consideration of CKD risk in evaluation for bariatric surgery and further study of bariatric surgery as a treatment for high-risk obese patients with CKD.
Detection of MYD88 L265P Mutation By Next Generation Deep Sequencing in Peripheral Blood Mononuclear Cells of Waldenstrom Macroguloblinemia and IgM Monoclonal Gammopathy of Undetermined Significance
Authors: Ayako Nakamura, Chikako Ohwada, Masahiro Takeuchi, Yusuke Takeda, Shokichi Tsukamoto, Naoya Mimura, Nagisa Oshima-Hasegawa, Yuhei Nagao, Emi Togasaki, Hiroaki Tanaka, Chika Kawajiri-Manako, Yasumasa Sugita, Hisashi Wakita, Nobuyuki Aotsuka, Kosei Matsue, Kotaro Yokote, Osamu Ohara, Chiaki Nakaseko, Emiko Sakaida
Journal: Blood (2017)
Risk of Infection in Type 1 and Type 2 Diabetes Compared With the General Population: A Matched Cohort Study
Authors: Iain M. Carey, Julia A. Critchley, Stephen DeWilde, Tess Harris, Fay J. Hosking, Derek G. Cook
Journal: Diabetes Care (2018)

We describe in detail the burden of infections in adults with diabetes within a large national population cohort. We also compare infection rates between patients with type 1 and type 2 diabetes mellitus (T1DM and T2DM).A retrospective cohort study compared 102,493 English primary care patients aged 40-89 years with a diabetes diagnosis by 2008 (n = 5,863 T1DM and n = 96,630 T2DM) with 203,518 age-sex-practice-matched control subjects without diabetes. Infection rates during 2008-2015, compiled from primary care and linked hospital and mortality records, were compared across 19 individual infection categories. These were further summarized as any requiring a prescription or hospitalization or as cause of death. Poisson regression was used to estimate incidence rate ratios (IRRs) between 1) people with diabetes and control subjects and 2) T1DM and T2DM adjusted for age, sex, smoking, BMI, and deprivation.Compared with control subjects without diabetes, patients with diabetes had higher rates for all infections, with the highest IRRs seen for bone and joint infections, sepsis, and cellulitis. IRRs for infection-related hospitalizations were 3.71 (95% CI 3.27-4.21) for T1DM and 1.88 (95% CI 1.83-1.92) for T2DM. A direct comparison of types confirmed higher adjusted risks for T1DM versus T2DM (death from infection IRR 2.19 [95% CI 1.75-2.74]). We estimate that 6% of infection-related hospitalizations and 12% of infection-related deaths were attributable to diabetes.People with diabetes, particularly T1DM, are at increased risk of serious infection, representing an important population burden. Strategies that reduce the risk of developing severe infections and poor treatment outcomes are under-researched and should be explored.
Abstract 19919: Cardioprotection During Early Reperfusion via Complex I Inhibition by Metformin
Authors: Ahmed Mohsin, Qun Chen, Nanhu Quan, Michael Maceyka, Arun Samidurai, Jeremy Thompson, Ying Hu, Ji Li, Edward J Lesnefsky
Journal: Circulation (2016)

Introduction: Mitochondria (MITO) mediate cardiac injury during ischemia (ISC)-reperfusion (REP). Transient, reversible blockade of the proximal electron transport chain (ETC) during early REP limi...
Colorectal endoscopic submucosal dissection using the “dental floss with rubber band method”
Authors: Tatsuma Nomura, Akira Kamei, Shinya Sugimoto, Jun Oyamada
Journal: Endoscopy (2018)

Colorectal endoscopic submucosal dissection (ESD) is considered one of the most challenging procedures owing to the difficulty in maintaining good visualization of the submucosal layer. To improve the visibility of the submucosal layer, various traction methods have been invented [1−4]. We have developed a new and efficient traction method called the “dental-floss with rubber-band method” (DRM).
Nkx2.8 Inhibits Epithelial–Mesenchymal Transition in Bladder Urothelial Carcinoma via Transcriptional Repression of <i>Twist1</i>
Authors: Chunping Yu, Zhuowei Liu, Qiuhong Chen, Yonghong Li, Lijuan Jiang, Zhiling Zhang, Fangjian Zhou
Journal: Cancer Research (2018)

Abstract Epithelial-to-mesenchymal transition (EMT) promotes metastasis, which is the main cause of bladder urothelial carcinoma–related death. Loss of the candidate tumor-suppressor gene Nkx2.8 has been associated with urothelial carcinoma lymph node metastasis. Here, we show that enforced expression of Nkx2.8 is sufficient to inhibit EMT, reduce motility, and blunt invasiveness of urothelial carcinoma cells. Mechanistic investigations showed that Nkx2.8 negatively regulated expression of the EMT inducer Twist1 in urothelial carcinoma cells, at both the level of mRNA and protein accumulation. Nkx2.8 bound directly to the promoter region of this gene and transcriptionally repressed its expression. Twist1 upregulation reversed EMT inhibition by Nkx2.8, restoring the invasive phenotype of urothelial carcinoma cells. In clinical urothelial carcinoma specimens, expression of Nkx2.8 inversely correlated with Twist1 expression, and urothelial carcinoma patients with Nkx2.8 positivity and low Twist1 expression displayed the best prognosis. Our findings highlight the Nkx2.8–Twist1 axis as candidate target for therapeutic intervention in advanced urothelial carcinoma. Significance: These findings highlight a novel EMT signaling axis as a candidate target for therapeutic intervention in advanced urothelial carcinomas. Cancer Res; 78(5); 1241–52. ©2018 AACR.
Abstract 10007: Revascularization in Multivessel Coronary Artery Disease_Lower Early MACCE Rate With Multiple Arterial Bypass Grafting Than Percutaneous Coronary Intervention
Authors: Chaim Locker, Hartzell V Schaff, Richard C Daly, Bell R Malcolm, Robert L Frye, John M Stulak, Kevin L Greason, Joseph A Dearani, Lyle D Joyce, Alberto Pochettino, Zhuo Li, Ryan J Lennon, Amir Lerman
Journal: Circulation (2015)

Objectives: CABG and PCI are used for revascularization of multivessel coronary artery disease (MVCAD). However, limited information is available comparing the rates of in-hospital and 30-day major adverse cardiac and cerebrovascular event (early MACCE), including early death, stroke, myocardial infarction (MI), and repeat revascularization, among patients with multiple arterial CABG (MultArt) vs PCI. Methods: We reviewed 12,615 MVCAD patients with isolated primary CABG or PCI from 1993 through 2009. CABG patients (n=6,667) were grouped by number of arterial grafts into left internal mammary artery/saphenous vein (LIMA/SV) (n=5,712) or MultArt (n=955); PCI patients (n=5,948) into balloon angioplasty (BA) (n=1,020), bare metal stent (BMS) (n=3,242), and drug-eluting stent (DES) (n=1,686). Results: Early MACCE rate was significantly greater for PCI than CABG (8.5% vs 4.1% [P&lt;.001]) and lower for MultArt than LIMA/SV or PCI (1.5% vs 4.5% and 8.5%, respectively [P&lt;.001]). Stroke and early death rates were higher in CABG vs PCI (2.2% vs 0.4% [P&lt;.001], and 1.5% vs 1.0% [P=0.01], respectively), whereas MI and repeat revascularization were lower (0.5% vs 6.0% and 0.3% vs 2.2%, respectively [P&lt;.001]). Stroke and early death were similar in MultArt and PCI and lower vs LIMA/SV (0.6% and 0.4% vs 2.6%, and 0.5% and 1.0% vs 1.7%, respectively [P&lt;.001]); MI and repeat revascularization rates were similar in MultArt and LIMA/SV and lower vs PCI (0.4% and 0.6% vs 6.0%, and 0.3% and 0.3% vs 2.3%, respectively [P&lt;.001]). In propensity score-matched analysis, CABG had significantly improved early MACCE rate compared with BA (4.7% vs 13.2% [P&lt;.001]), BMS (4.3% vs 8.3% [P&lt;.001]), and DES (2.9% vs 5.5% [P=.008]), and MultArt compared with PCI (1.8% vs 7.8% [P&lt;.001]) fig. Conclusions: In MVCAD patients undergoing isolated primary revascularization, CABG conferred lower early MACCE rate vs PCI. Early MACCE rate was markedly decreased after MultArt compared with LIMA/SV, BA, BMS, and DES.
Leonine facies as a sign of leukaemia cutis
Authors: Yvonne Loh, Daryl Tan
Journal: BMJ (2018)

A 46 year old woman was referred for investigation of deranged blood counts. She had a white blood cell count of 20×109/L (normal range 4-10×109/L), haemoglobin 6.8 g/dL (11.5-16 g/dL), and …
Biocompatibility: Controlling Protein Adsorption through Nanostructured Polymeric Surfaces (Adv. Healthcare Mater. 1/2018)
Authors: Izabela Firkowska‐Boden, Xiaoyuan Zhang, Klaus D. Jandt
Journal: Advanced Healthcare Materials (2018)

Protein adsorption on biomaterials' surfaces is a very complex process impacting biocompatibility and functionality of healthcare materials. The introduction of specific surface nanostructures is recognized as a powerful tool to control protein-surface interactions. In article number 1700995, Klaus D. Jandt and co-workers review nanostructured polymer surfaces represented by synthetic homopolymers, semicrystalline polymers and block copolymers, and their ability to control the amount, conformation, and selectivity of protein adsorption.
Abstract 17978: Mlp Interacting Protein 1 (MIP1) Plays a Role for Cardiomyopathy
Authors: Byambajav Buyandelger, Catherine Mansfield, Sawa Kostin, Onjee Choi, Rivka L. Isaacson, Kar-Lai Poon, Gudrun Knöll, Sara McSweeney, Andreas Perrot, Mohammad R Toliat, Ju Chen, Ian Gould, Enrique Lara-Pezzi, Pradeep Luther, Enrico Petretto, Hendrik Milting, Thomas Brand, Ralph Knöll
Journal: Circulation (2015)

Mutations in genes involved in cardiac mechanosensation (mec), such as the muscle LIM protein (MLP) have been shown to cause heart failure. Identification of novel mec genes may provide new insights into underlying mechanisms. A yeast 2 hybrid screen identified a novel MIP, located at a chromosomal region recently implicated by GWASs in the pathogenesis of dilated cardiomyopathy (DCM), an interaction confirmed by various assays. We found that MIP1 is a stress inducible transcription factor, binds to the calcineurin promoter and activates NFAT, a known mediator of hypertrophy. Also overexpression of MIP1 in vitro promotes autophagy and activates expression of anti-apoptotic genes in cardiac myocytes. Mip1 conventional knockout animals die during gastrulation whereas Mip1 conditional knockout (cKO) animals failed to adapt to transverse aortic constriction. Four weeks after intervention, fractional shortening was significantly decreased, whereas LVEDD and LVESD were significantly increased in cKO mice compared to control littermates. TUNEL assays combined with activated caspase, whole gene expression arrays and qRT-PCR provide strong evidence that Mip1 cKO hearts are unable to undergo autophagy and as a result activate apoptotic processes which are responsible for the observed heart failure phenotype. However, overexpression of MIP1 in vivo resulted in marked cardiac hypertrophy at the organ and single cell levels, pointing to a link between autophagy and hypertrophy. As MIP1 contains the peak association signal (rs10927875) at the replicated 1p36 cardiomyopathy locus, we sequenced this gene in 575 unrelated individuals affected by DCM and identified 4 MIP1 missense mutations (T106M, G179S, A188V, Q531R) of which 2 co-segregate in two families with DCM and arrhythmia. All 4 variants show abnormalities with regards to their ability to interact with MLP, localization, structure, stability and potential to drive the calcineurin promoter. WT-MIP1 is able to efficiently protect cells from apoptosis, a property missing in all mutants. At least G179S and Q531R cause heart failure in the zebrafish. MIP1 is important for the initiation of survival pathways and autophagy upon biomechanical stress and represents a novel cardiomyopathy candidate gene.
iPhone–Based Device Helps Treat River Blindness
Authors: Jennifer Abbasi
Journal: JAMA (2018)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
Abstract P274: Pregnancy-induced-hypertension and the Risk of Postpartum Hypertension Among Gestational Diabetes Women
Authors: Leishen Wang, Junhong Leng, Huikun Liu, Shuang Zhang, Jing Wang, Weiqin Li, Wei Li, Nan Li, Tao Zhang, Gang Hu
Journal: Circulation (2017)

Background: Previous studies in the general people indicated that pregnancy-induced hypertension (PIH) increased the risk of subsequent hypertension after delivery. Some studies found that women with gestational diabetes mellitus (GDM) had an increased risk of PIH. However, very few studies have assessed the association between PIH and the risk of postpartum hypertension among GDM women. Aims: To evaluate the association between PIH and the risk of postpartum hypertension among GDM women. Methods: We performed a retrospective cohort study in 1261 GDM mother at 1-5 years after delivery using the baseline data from Tianjin Gestational Diabetes Mellitus Prevention Program. Cox regression models were applied to assess the single and joint associations of having a history of PIH, maternal pre-pregnancy BMI (normal weight, overweight and obesity), and weight change from pre-pregnancy to postpartum with the risk of subsequent hypertension among the GDM women. Results: GDM people with a history of PIH, high pre-pregnancy BMI, and weight gain more than 7 kg from pre-pregnancy to postpartum were associated with an increased risk of postpartum hypertension. Joint effects analysis revealed that the positive association between a history of PIH in the index pregnancy and the risk of postpartum hypertension was consistent in GDM women with different levels of pre-pregnancy BMI or weight gain from pre-pregnancy to postpartum. Conclusion: A history of PIH increases the risk of subsequent hypertension in postpartum 1-5 years among GDM women. We suggest highlight the pregnancy guide in different stages and pay more attention on this group of people.
Nasal IL-25 predicts the response to oral corticosteroids in chronic rhinosinusitis with nasal polyps
Authors: Haiyu Hong, Fenghong Chen, Yueqi Sun, Qintai Yang, Wenxiang Gao, Yujie Cao, Yunping Fan, Jianbo Shi, Huabin Li
Journal: Journal of Allergy and Clinical Immunology (2018)
Simultaneous inhibition of hedgehog signaling and tumor proliferation remodels stroma and enhances pancreatic cancer therapy
Authors: Jun Zhao, Huamin Wang, Cheng-Hui Hsiao, Diana S.-L. Chow, Eugene J. Koay, Yaan Kang, Xiaoxia Wen, Qian Huang, Ying Ma, James A. Bankson, Stephen E. Ullrich, Willem Overwijk, Anirban Maitra, David Piwnica-Worms, Jason B. Fleming, Chun Li
Journal: Biomaterials (2018)
Cost Effectiveness of Nivolumab for the Treatment of Relapsed/Refractory Classical Hodgkin Lymphoma (cHL) after Failure of Autologous Stem Cell Transplantation (ASCT) and Treatment with Brentuximab Vedotin (BV) Treatment in Australia
Authors: Marcus Tan, Greta Lozano-Ortega, Shelagh Szabo, Karissa Johnston, Joseph M Connors, Clara Chen, Hansoo Kim
Journal: Blood (2017)
Erratum: Corrigendum: Genome variation and evolution of the malaria parasite Plasmodium falciparum
Authors: Daniel C Jeffares, Arnab Pain, Andrew Berry, Anthony V Cox, James Stalker, Catherine E Ingle, Alan Thomas, Michael A Quail, Kyle Siebenthall, Anne-Catrin Uhlemann, Sue Kyes, Sanjeev Krishna, Chris Newbold, Emmanouil T Dermitzakis, Matthew Berriman
Journal: Nature Genetics (2007)
Mesenchymal Stromal Cells, Instigator or Suppressor for the Development of MLL-AF9 Induced Acute Myeloid Leukemia?
Authors: Pingnan Xiao, Yaser Heshmati, Thibault Bouderlique, Makoto Kondo, Monika Dolinska, Lakshmi Sandhow, Anne-Sofie Johansson, Julian Walfridsson, Hong Qian
Journal: Blood (2016)
Examining genetic susceptibility in acute exacerbations of COPD
Authors: Emily S Wan
Journal: Thorax (2018)

Acute exacerbations (AE) contribute significantly to morbidity and mortality, and account for a substantial proportion of the direct costs associated with chronic obstructive pulmonary disease (COPD).1 Although AE frequency generally increases with worsening spirometric airflow obstruction on the population level, considerable variation in the number of exacerbations experienced by individuals exists. Epidemiological studies support the existence of an ‘intrinsic susceptibility’ towards AE which is independent of the degree of impairment in forced expiratory volume in 1 s (FEV1) on spirometry.2 This has fuelled the search for genetic variants which may contribute to differential susceptibility towards AEs.

An overview of studies investigating associations between genetic sequence variants and AE risk in COPD is shown in table 1. The vast majority of studies published to date have used the ‘candidate gene’ approach, whereby a limited number of variants in genes felt to be plausible contributors to exacerbation susceptibility are examined. Unfortunately, the body of knowledge arising from these investigations is often inconsistent. Non-replication or conflicting reports of association between studies are common, even when identical variants are interrogated (as exemplified by studies on the adrenoreceptor beta 2 gene, ARDB2) . This is likely due, at least in part, to chance findings, limited power from small study sizes and inadequate statistical rigour. Recent advances in genotyping, computing power and statistical methodologies have facilitated the widespread application of ‘hypothesis-free’ genomics-based studies of complex diseases and traits, yet studies of AE risk in COPD using this approach have been conspicuously absent from the literature.

View this table:

Table 1 
Genetic variants investigated for association with AE of COPD



A major limitation of hypothesis-free approaches, which …
Depth of Organ Response in Newly Diagnosed AL Amyloidosis Is Associated with Improved Survival: Improved Outcome Discrimination with Graded Organ Response
Authors: Eli Muchtar, Angela Dispenzieri, Martha Q Lacy, Nelson Leung, Francis K Buadi, Martha Grogan, Suzanne R. Hayman, Prashant Kapoor, Yi L. Hwa, Amie Fonder, Miriam Hobbs, Rajshekhar Chakraborty, Wilson I Gonsalves, Taxiarchis Kourelis, Rahma Warsame, Stephen J. Russell, John A. Lust, Yi Lin, Ronald S. Go, Steven Zeldenrust, Robert A Kyle, S. Vincent Rajkumar, Shaji K Kumar, Morie A. Gertz
Journal: Blood (2017)
Associations of the Top 20 Alzheimer Disease Risk Variants With Brain Amyloidosis
Authors: Liana G. Apostolova, Shannon L. Risacher, Tugce Duran, Eddie C. Stage, Naira Goukasian, John D. West, Triet M. Do, Jonathan Grotts, Holly Wilhalme, Kwangsik Nho, Meredith Phillips, David Elashoff, Andrew J. Saykin
Journal: JAMA Neurology (2018)

Late-onset Alzheimer disease (AD) is highly heritable. Genome-wide association studies have identified more than 20 AD risk genes. The precise mechanism through which many of these genes are associated with AD remains unknown.
Comparative Analysis of Fecal Short Chain Fatty Acids Profiles in Atopic Dermatitis And Healthy Infants
Authors: Le Duc Huy Ta, James Chun Yip Chan, Chiung-Hui Huang, Gaik Chin Yap, Lynette Pei-Chi Shek, Anne Goh, Hugo P.S. Van Bever, Oon Hoe Teoh, Jian Yi Soh, Biju Thomas, Mahesh Babu Ramamurthy, Daniel Goh, Christophe Lay, Shu E. Soh, Fabian Yap, Kok Hian Tan, Yap-Seng Chong, Keith B.M. Godfrey, Peter D. Gluckman, Eric Chun Yong Chan, Bee Wah Lee
Journal: Journal of Allergy and Clinical Immunology (2018)