tbuckley/cabot-search huggingface dataset:
The influence of hypomagnesemia (hMg) on overall survival (OS) in a phase III randomized study of cetuximab (CET) plus best supportive care (BSC) versus BSC: NCIC CTG/AGITG CO.17.
Authors: M. M. Vickers, C. S. Karapetis, D. Tu, C. J. O'Callaghan, T. J. Price, N. C. Tebbutt, G. Van Hazel, J. D. Shapiro, N. Pavlakis, P. Gibbs, J. Blondal, U. J. Yu Min Lee, J. M. Meharchand, R. L. Burkes, S. H. Rubin, J. Simes, J. R. Zalcberg, M. J. Moore, L. Zhu, D. J. Jonker
Journal: Journal of Clinical Oncology (2011)

3601
Cryptic Truncating Rearrangements of the Erythropoietin Receptor in Ph-like Acute Lymphoblastic Leukemia
Authors: Ilaria Iacobucci, Kathryn G. Roberts, Yongjin Li, Debbie Payne-Turner, Marcus Valentine, Jinghui Zhang, Richard C. Harvey, Kelly McCastlain, John Easton, I-Ming L. Chen, Michael Rusch, Steven M. Kornblau, Marina Konopleva, Elisabeth M. Paietta, Jacob M. Rowe, Ching-Hon Pui, Julie Gastier-Foster, Mignon Loh, Cheryl L. Willman, James R Downing, Stephen Hunger, Charles G. Mullighan
Journal: Blood (2014)
Migraine and risk of perioperative ischemic stroke and hospital readmission: hospital based registry study
Authors: Fanny P Timm, Timothy T Houle, Stephanie D Grabitz, Anne-Louise Lihn, Janne B Stokholm, Katharina Eikermann-Haerter, Ala Nozari, Tobias Kurth, Matthias Eikermann
Journal: BMJ (2017)

<b>Objective</b>&nbsp;To evaluate whether patients with migraine are at increased risk of perioperative ischemic stroke and whether this may lead to an increased hospital readmission rate. <b>Design</b>&nbsp;Prospective hospital registry study. <b>Setting</b>&nbsp;Massachusetts General Hospital and two satellite campuses between January 2007 and August 2014. <b>Participants</b>&nbsp;124 558 surgical patients (mean age 52.6 years; 54.5% women). <b>Main outcome measures</b>&nbsp;The primary outcome was perioperative ischemic stroke occurring within 30 days after surgery in patients with and without migraine and migraine aura. The secondary outcome was hospital readmission within 30 days of surgery. Exploratory outcomes included post-discharge stroke and strata of neuroanatomical stroke location. <b>Results</b>&nbsp;10 179 (8.2%) patients had any migraine diagnosis, of whom 1278 (12.6%) had migraine with aura and 8901 (87.4%) had migraine without aura. 771 (0.6%) perioperative ischemic strokes occurred within 30 days of surgery. Patients with migraine were at increased risk of perioperative ischemic stroke (adjusted odds ratio 1.75, 95% confidence interval 1.39 to 2.21) compared with patients without migraine. The risk was higher in patients with migraine with aura (adjusted odds ratio 2.61, 1.59 to 4.29) than in those with migraine without aura (1.62, 1.26 to 2.09). The predicted absolute risk is 2.4 (2.1 to 2.8) perioperative ischemic strokes for every 1000 surgical patients. This increases to 4.3 (3.2 to 5.3) for every 1000 patients with any migraine diagnosis, 3.9 (2.9 to 5.0) for migraine without aura, and 6.3 (3.2 to 9.5) for migraine with aura.<b></b>Patients with migraine had a higher rate of readmission to hospital within 30 days of discharge (adjusted odds ratio 1.31, 1.22 to 1.41). <b>Conclusions</b>&nbsp;Surgical patients with a history of migraine are at increased risk of perioperative ischemic stroke and have an increased 30 day hospital readmission rate. Migraine should be considered in the risk assessment for perioperative ischemic stroke.
P1.02-049 EGFR, KRAS and ALK Gene Alterations in Lung Cancer Patients in Croatia
Authors: Marko Jakopovic, Luka Brcic, Marija Misic, Gordana Bubanovic, Fran Seiwerth, Gordana Drpa, Branka Cucevic, Mihovil Roglic, Sanja Plestina, Suazana Kukulj, Silvana Smojver-Jezek, Sven Seiwerth, Miroslav Samarzija
Journal: Journal of Thoracic Oncology (2017)
P3.02b-024 Dynamics of EGFR Mutational Load in Urine and Plasma Correlates with Treatment Response in Advanced NSCLC
Authors: Jhanelle Gray, Benjamin Creelan, Tawee Tanvetyanon, Scott Antonia, Charles Williams, Karen Johnson, Christine Sigua, Jongphil Kim, Richard Reich, Braydon Schaible, Cecile Rose Vibat, David Gustafson, Sandeep Pingle, Mark Erlander, Vlada Melnikova, Eric Haura
Journal: Journal of Thoracic Oncology (2017)
Targeting Transcriptional Dependency in Acute Myeloid Leukemia (AML) with a Covalent Inhibitor of Transcriptional Kinase CDK7
Authors: Yixuan Ren, Victoria Brown, Shanhu Hu, Jeremy Lopez, Sofija Miljovska, Darby Schmidt, Michael Bradley, Kevin Sprott, Eric Olson, Christian C. Fritz, Yoon Jong Choi
Journal: Blood (2015)
Implementation Science to Accelerate Clean Cooking for Public Health
Authors: Joshua Rosenthal, Kalpana Balakrishnan, Nigel Bruce, David Chambers, Jay Graham, Darby Jack, Lydia Kline, Omar Masera, Sumi Mehta, Ilse Ruiz Mercado, Gila Neta, Subhrendu Pattanayak, Elisa Puzzolo, Helen Petach, Antonello Punturieri, Adolfo Rubinstein, Michael Sage, Rachel Sturke, Anita Shankar, Kenny Sherr, Kirk Smith, Gautam Yadama
Journal: Environmental Health Perspectives (2017)

Summary:Clean cooking has emerged as a major concern for global health and development because of the enormous burden of disease caused by traditional cookstoves and fires. The World Health Organization has developed new indoor air quality guidelines that few homes will be able to achieve without replacing traditional methods with modern clean cooking technologies, including fuels and stoves. However, decades of experience with improved stove programs indicate that the challenge of modernizing cooking in impoverished communities includes a complex, multi-sectoral set of problems that require implementation research. The National Institutes of Health, in partnership with several government agencies and the Global Alliance for Clean Cookstoves, has launched the Clean Cooking Implementation Science Network that aims to address this issue. In this article, our focus is on building a knowledge base to accelerate scale-up and sustained use of the cleanest technologies in low- and middle-income countries. Implementation science provides a variety of analytical and planning tools to enhance effectiveness of clinical and public health interventions. These tools are being integrated with a growing body of knowledge and new research projects to yield new methods, consensus tools, and an evidence base to accelerate improvements in health promised by the renewed agenda of clean cooking.
Reversal of Dabigatran's Anticoagulant Activity in the Monkey by a Specific Antidote and Pharmacokinetic and Pharmacodynamic Modeling
Authors: Joshuaine Toth, Guanfa Gan, Joanne van Ryn, Holly Dursema, Jennifer Isler, Kelly Coble, John Burke, Bojan Lalovic, Stephen Olson
Journal: Blood (2012)
Comparison of risk scoring systems for patients presenting with upper gastrointestinal bleeding: international multicentre prospective study
Authors: Adrian J Stanley, Loren Laine, Harry R Dalton, Jing H Ngu, Michael Schultz, Roseta Abazi, Liam Zakko, Susan Thornton, Kelly Wilkinson, Cristopher J L Khor, Iain A Murray, Stig B Laursen
Journal: BMJ (2017)

<b>Objective</b>&nbsp;To compare the predictive accuracy and clinical utility of five risk scoring systems in the assessment of patients with upper gastrointestinal bleeding. <b>Design</b>&nbsp;International multicentre prospective study. <b>Setting</b>&nbsp;Six large hospitals in Europe, North America, Asia, and Oceania. <b>Participants</b>&nbsp;3012 consecutive patients presenting over 12 months with upper gastrointestinal bleeding. <b>Main outcome measures</b>&nbsp;Comparison of pre-endoscopy scores (admission Rockall, AIMS65, and Glasgow Blatchford) and post-endoscopy scores (full Rockall and PNED) for their ability to predict predefined clinical endpoints: a composite endpoint (transfusion, endoscopic treatment, interventional radiology, surgery, or 30 day mortality), endoscopic treatment, 30 day mortality, rebleeding, and length of hospital stay. Optimum score thresholds to identify low risk and high risk patients were determined. <b>Results</b>&nbsp;The Glasgow Blatchford score was best (area under the receiver operating characteristic curve (AUROC) 0.86) at predicting intervention or death compared with the full Rockall score (0.70), PNED score (0.69), admission Rockall score (0.66, and AIMS65 score (0.68) (all P&lt;0.001). A Glasgow Blatchford score of ≤1 was the optimum threshold to predict survival without intervention (sensitivity 98.6%, specificity 34.6%). The Glasgow Blatchford score was better at predicting endoscopic treatment (AUROC 0.75) than the AIMS65 (0.62) and admission Rockall scores (0.61) (both P&lt;0.001). A Glasgow Blatchford score of ≥7 was the optimum threshold to predict endoscopic treatment (sensitivity 80%, specificity 57%). The PNED (AUROC 0.77) and AIMS65 scores (0.77) were best at predicting mortality, with both superior to admission Rockall score (0.72) and Glasgow Blatchford score (0.64; P&lt;0.001). Score thresholds of ≥4 for PNED, ≥2 for AIMS65, ≥4 for admission Rockall, and ≥5 for full Rockall were optimal at predicting death, with sensitivities of 65.8-78.6% and specificities of 65.0-65.3%. No score was helpful at predicting rebleeding or length of stay. <b>Conclusions</b>&nbsp;The Glasgow Blatchford score has high accuracy at predicting need for hospital based intervention or death. Scores of ≤1 appear the optimum threshold for directing patients to outpatient management. AUROCs of scores for the other endpoints are less than 0.80, therefore their clinical utility for these outcomes seems to be limited. <b>Trial registration</b>&nbsp;Current Controlled Trials ISRCTN16235737.
Exosomal miR-142-3p is increased during cardiac allograft rejection and augments vascular permeability through down-regulation of endothelial RAB11FIP2 expression
Authors: Ihdina Sukma Dewi, Selvi Celik, Anna Karlsson, Zsuzsanna Hollander, Karen Lam, Janet-Wilson McManus, Scott Tebbutt, Raymond Ng, Paul Keown, Robert McMaster, Bruce McManus, Jenny Öhman, Olof Gidlöf
Journal: Cardiovascular Research (2016)

Exosome-mediated microRNA transfer is a recently discovered mode of cell-to-cell communication, in which microRNAs act as paracrine molecules, exerting their regulatory effects in recipient cells. T cells and endothelial cells are two main players in the mechanism of acute cellular cardiac rejection. The aim of this study was to investigate the role of exosomal microRNAs in the crosstalk between T cells and endothelial cells and its implications for the molecular mechanisms that drive acute cellular rejection in heart transplantation. Exosomes isolated from serum samples of heart transplant patients with and without acute cardiac allograft rejection were profiled and showed enrichment of miR-142-3p, miR-92a-3p, miR-339-3p and miR-21-5p. Treatment of endothelial cells with the respected serum exosomes resulted the increased of miR-142-3p level in endothelial cells. Using T cells isolated from healthy donors and activated with either anti-CD3/CD28 antibody or IL-2/PHA, we could show that miR-142-3p is released from activated cells, is contained in exosomes and can be transferred to human vascular endothelial cells in vitro. Transcriptome analysis of endothelial cells treated with activated T cell supernatant with or without exosomes was used to identify mRNA targets of transferred miR-142-3-p. Overexpression of miR-142-3p in endothelial cells resulted in a significant down-regulation of RAB11FIP2, and interaction of miR-142-3p with its predicted target site was confirmed with a reporter assay. Moreover, treatment of endothelial cells with serum exosomes from heart transplant patients with acute cellular rejection resulted in down-regulation of RAB11FIP2 expression and increase in vascular endothelial permeability. We have identified a novel mechanism whereby miR-142-3p, a microRNA enriched in exosomes during acute cellular rejection, is transferred to endothelial cells and compromises endothelial barrier function via down-regulation of RAB11FIP2. This study sheds new light on the interaction between host immune system and cardiac allograft endothelium during acute cellular rejection.
Synergistic Cytotoxicity of Ibrutinib and the BCL2 Antagonist, ABT-199(GDC-0199) in Mantle Cell Lymphoma (MCL) and Chronic Lymphocytic Leukemia (CLL): Molecular Analysis Reveals Mechanisms of Target Interactions
Authors: Craig A. Portell, Mark Axelrod, L. Kyle Brett, Vicki L Gordon, Brian Capaldo, Jeffrey C Xing, Stephan Bekiranov, Michael E. Williams, Michael J. Weber
Journal: Blood (2014)
The First-in-Class Alkylating Histone-Deacetylase Inhibitor (HDACi) Fusion Molecule Edo-S101 Exerts Potent Preclinical Activity Against Tumor Cells of Hodgkin Lymphoma (HL) Including Bendamustine-Resistant Clones
Authors: Rosaria De Filippi, Stefania Crisci, Michele Cillo, Sara Mele, Angela De Monaco, Domenico Galati, Serena Zanotta, Gaetana Capobianco, Francesco Volzone, Manuela Arcamone, Piera Maiolino, Mariangela Saggese, Gaetano Corazzelli, Ferdinando Frigeri, Thomas Mehrling, Antonello Pinto
Journal: Blood (2015)
P1.02-060 EGFR Mediates Activation of RET in Lung Adenocarcinoma with Neuroendocrine Differentiation Characterized by ASCL1 Expression
Authors: Farhad Kosari, Kaustubh Bhinge, Lin Yang, Simone Terra, Aqsa Nasir, Prasuna Muppa, Marie Christine Aubry, Joanne Yi, Nafiseh Janaki, Irina Kovtun, Stephen Murphy, Hamed Rahi, Aaron Mansfield, Mariza De Andrade, Ping Yang, George Vasmatzis, Tobias Peikert
Journal: Journal of Thoracic Oncology (2017)
Twice Daily Fludarabine and Cytarabine Combination Is Effective in Patients with Relapsed/Refractory Acute Myeloid Leukemia, High-Risk Myelodysplastic Syndromes, and Blast Phase Chronic Myeloid Leukemia,
Authors: Naval Daver, Hagop M. Kantarjian, Guillermo Garcia-Manero, Jorge E. Cortes, Farhad Ravandi, William Plunkett, Varsha Gandhi, Stefan Faderl, Susan o'Brien, Gautam Borthakur, Tapan Kadia, Jan A. Burger, Marina Konopleva, Mark Brandt, Xuelin Huang, Elias Jabbour
Journal: Blood (2011)
P2.03b-060 Baseline Skeletal Muscle Index (SMI) Values Are Associated with Biomarkers of Insulin Resistance in Stage IV Non-Small Cell Lung Cancer
Authors: Marta Batus, Samantha Kerns, Selina Savidine, Cristina Fhied, Sanjib Basu, Philip Bonomi, Jeffrey Borgia, Mary Fidler, Palmi Shah
Journal: Journal of Thoracic Oncology (2017)
Positively charged polypeptide nanogel enhances mucoadhesion and penetrability of 10-hydroxycamptothecin in orthotopic bladder carcinoma
Authors: Hui Guo, Weiguo Xu, Jinjin Chen, Lesan Yan, Jianxun Ding, Yuchuan Hou, Xuesi Chen
Journal: Journal of Controlled Release (2017)
Letter by Ryding et al Regarding Article, “Impact of Pretreatment With Clopidogrel on Initial Patency and Outcome in Patients Treated With Primary Percutaneous Coronary Intervention for ST-Segment Elevation Myocardial Infarction: A Systematic Review”
Authors: Alisdair Ryding, Saurabh Kumar, Ravinay Bhindi
Journal: Circulation (2009)
Increased canonical WNT/β-catenin signalling and myxomatous valve disease
Authors: Sunita Chopra, Nadia Al-Sammarraie, Yimu Lai, Mohamad Azhar
Journal: Cardiovascular Research (2016)

This editorial refers to ‘Loss of Axin2 results in impaired heart valve maturation and subsequent myxomatous valve disease’ by A. Hulin et al ., doi: 10.1093/cvr/cvw229. 

Myxomatous valve disease (MVD) is a common form of valvular heart disease which causes severe valvular regurgitation.1 Increased accumulation of glycosaminoglycans (GAG), disrupted collagen fiber organization, increased cell proliferation, increased presence of inflammatory cells, and induction of Wingless-type MMTV integration site family (WNT), Transforming Growth Factor-beta (TGFβ), and Bone Morphogenetic Protein (BMP) signalling is reported in late stage human MVD.2–4

Valve development involves cell–cell and cell–matrix interactions ( Figure 1 ). Recent studies suggest that defects in heart valve development can lead to heart valve disease in adults.5 Valve formation starts with an epithelial mesenchymal transition (EMT) (E9.5–E10.5) that gives rise to mesenchymal cushions in both the atrioventricular and outflow tract regions of the developing heart. Cardiac cushions become mature valves through mesenchymal expansion (E10.5–E12.5), differentiation (E12.5–E14.5), and maturation (E14.5-postnatal stages). Although heart valve remodelling can be conceptually or arbitrarily organized into multiple processes based on the molecular and cellular markers, it is very difficult to establish precisely when a developmental event (such as EMT) in valvulogenesis formally ends. In fact, it has been demonstrated that valvulogenesis continues after birth to adjust to the postnatal maturation of the heart. 



Figure 1 
Valve development and homeostasis and valve pathogenesis. Elucidation of the WNT/β-catenin mechanisms through which loss of Axin2 affects the developmental transition during valvulogenesis, from cushion formation – cushion remodeling – valve maturation – adult valve homeostasis, will improve basic understanding of the etiology of MVD.



WNT/β-catenin signalling is active during heart valve development6 and has been reported in human MVD.2,7 There are 19 WNT family members which signal through the canonical (via β-catenin) pathway but can also signal through non-canonical …
Diffuse B-Large Cell Lymphomas (DBLCL) with Hepatitis-C Virus (HCV) Infection: Clinical Outcome and Preliminary Results of a Pilot Study Combining R-CHOP with Antiviral Therapy.
Authors: Pellegrino Musto, Matteo Dell’Olio, Antonio La Sala, Saverio Mantuano, Nicola Cascavilla
Journal: Blood (2005)
Corrigendum to “Lipocalin-2 mediates non-alcoholic steatohepatitis by promoting neutrophil-macrophage crosstalk via the induction of CXCR2”
Authors: Dewei Ye, Kangmin Yang, Shufei Zang, Zhuofeng Lin, Hau-Tak Chau, Yudong Wang, Jialiang Zhang, Junping Shi, Aimin Xu, Shaoqiang Lin, Yu Wang
Journal: Journal of Hepatology (2017)
Autosomal Recessive Inherited Bleeding Disorders in Pakistan, a Cross Sectional Study from Selected Regions
Authors: Arshi Naz, Mohammad younus Jamal, Samina Tufail Amanat, Humayun Patel, Akbar Agha, fazal-E- Razik, Nisar Ahmed, Ayisha Imran, Tahir Shamsi
Journal: Blood (2015)
Macrophage Inflammatory Protein (MIP)-3α/CCL20 and Its Receptor CCR6 Are Overexpressed in the Bone Microenvironment and Involved in Osteoclast Formation in Multiple Myeloma Patients.
Authors: Nicola Giuliani, Gina Lisignoli, Sara Tagliaferri, Mirca Lazzaretti, Francesca Morandi, Simona Colla, Cristina Manferdini, Beatrice Tonnarelli, Vittorio Rizzoli
Journal: Blood (2007)
Donor Characteristics Affecting Graft Failure and Survival after Unrelated Donor Transplantation with Reduced Intensity Conditioning Regimens (RIC) for Hematologic Malignancies.
Authors: Jakob R. Passweg, Fangyu Kan, Mei-Jie Zhang, Vanderson Rocha, Luis M. Isola, Adrian P. Gee, John Gibson, Hillard M. Lazarus, Mary J. Laughlin, Alison Loren, David Marks, Alois Gratwohl, Mary Eapen
Journal: Blood (2008)
Abstract B13: Reprogramming glioblastoma multiforme cells to neurons by protein kinase inhibitors
Authors: Jie Yuan, Fan Zhang, Meng You, Qin Yang
Journal: Clinical Cancer Research (2017)

Abstract Reprogramming healthy somatic cells into pluripotent stem (iPS) cells with defined factors has been intensively investigated. However, reprogramming cancer cells has fallen much behind. Direct reversion of cancers into normal tissues is an ideal strategy for cancer treatment. Recent reports have showed that defined factors can induce reprogramming of cancer cells into iPS cells, supporting this notion. Glioblastoma multiforme (GBM) is the most common and most aggressive malignant primary brain tumor in humans. Despite multimodal therapy, the outcome for patients with GBM is still poor, with a median survival of just 14.2 months. Therefore, developing novel therapeutic strategy is an emergent task. The tumor suppressor p53 is considered today the most important tumor suppressor protein in humans and it suppresses both reprogramming and oncogenic transformation. p53 inactivation or mutations are the most common in GBM, and this p53 deficient GBM subgroup is currently the most difficult to treat, typically associated with rapid local recurrence after treatment. p53 mutations result in loss-of-function, gain-of-function, and dominant-negative mutational effects for p53. Thus, different p53 mutants may have various effects on reprogramming, promoting or inhibiting GBM reprogramming. Furthermore, p53 activity is often inactivated in GBM with wild-type p53 that may also affect reprogramming. We have demonstrated that the depletion of p53 reprograms human somatic cells into neurons by regulation of a set of transcription factors (Nature Communications, 2014; Cell Reports, 2014, International Journal of Stem Cell Research &amp; Therapy, 2016). Integration-free reprogramming cancer cells would be safer and preferable for clinical use. Along those lines, we screened a protein kinase inhibitor library and found that the kinase inhibitors can substitute for all transcription factors and convert human p53-deficient GBM cells into “normal” neurons (functional neurons). In vitro and in vivo tumorigenesis assays showed that induced neurons lose tumorigenicity. Moreover, the inhibitor reprogramming prevents GBM local recurrence in mice. Currently these inhibitors are used as anti-tumor drugs in patients, so this reprogramming strategy has significant potential to move rapidly toward clinical trials. Note: This abstract was not presented at the conference. Citation Format: Jie Yuan, Fan Zhang, Meng You, Qin Yang. Reprogramming glioblastoma multiforme cells to neurons by protein kinase inhibitors. [abstract]. In: Proceedings of the AACR Precision Medicine Series: Targeting the Vulnerabilities of Cancer; May 16-19, 2016; Miami, FL. Philadelphia (PA): AACR; Clin Cancer Res 2017;23(1_Suppl):Abstract nr B13.
Epidemiological Study of Mutiple Myeloma In Spain: Efectiveness and Survival Analysis
Authors: Consuelo González García, Ma Sol Durán, Jose Ramón Mayans, Javier De La Rubia, Alfons Soler, Inmaculada Castillo, Paz Ribas, Ma Carmen Menchaca Echevarría, Manuel Jurado, Miguel T. Hernandez, Ma Dolores López García-Carreño, Asunción Echeveste Gutierrez, Joaquín Ruiz Arredondo, Silvia López, Gemma Ramirez
Journal: Blood (2010)
P3.03-044 Is Toxicity Increased by Adding Intraoperative Chemotherapy to Preoperative Induction Chemotherapy for Mesothelioma Patients?
Authors: Olivia Lauk, Martina Friess, Mayura Meerang, Michaela Kirschner, Cordelia Bommeli, Rolf Stahel, Walter Weder, Isabelle Opitz
Journal: Journal of Thoracic Oncology (2017)
P2.05-053 Discussion and Analysis of Pneumonitis Related to Stereotactic Radiotherapy in Our Hospital
Authors: Keisuke Imasaka, Eriko Ikeya, Yuki Mitamura, Manabu Toyoda, Hiroki Takakura, Akio Komuro, Koichi Sunada, Nobuyuki Hamanaka, Miki Takahashi, Kunihiko Shimizu, Yoshimasa Inoue, Teruhiro Aoki, Shoji Sakai, Kazuhito Funakoshi
Journal: Journal of Thoracic Oncology (2017)
Serum Free Light Chain (SFLC) Concentration Kinetics in Patients Receiving Bortezomib: Temporary Inhibition of Protein Synthesis and Early Biomarker for Disease Response.
Authors: Mayukh Das, Graham P. Mead, Venkatasampath Sreekanth, Julia Anderson, Sue Blair, Tracy Howe, Jim Cavet, Effie Liakopoulou
Journal: Blood (2005)
Immunohistochemical Expression Patterns of Germinal Center and Activation B-Cell Markers in Diffuse Large B-Cell Lymphoma. Clinicopathologic Study of 62 Cases from Peru.
Authors: Shirley Quintana, Carlos Barrionuevo, Henry Gomez, Silvia Neciosup, Oswaldo Grados, Sandro Casavilca, Claudio Flores, Luis Casanova
Journal: Blood (2006)
P2.03b-065 Serum and Bronchoalveolar Lavage Levels of Adiponectin in Advanced Non-Small Cell Lung Cancer: Results of a Prospective Study
Authors: Paraskevi Boura, Dimitra Grapsa, Stylianos Loukides, Apostolos Achimastos, Kostas Syrigos
Journal: Journal of Thoracic Oncology (2017)
Dyadic Concordance of Sexuality Among Hematopoietic Stem Cell Transplantation Patients and Their Partners
Authors: Kwai Han Yoo, Jinhyun Cho, Haa-Na Song, Ki Sun Jung, Juhee Cho, Young Rok Do, Seok Jin Kim, Kihyun Kim, Chul Won Jung, Jun Ho Jang
Journal: Blood (2015)