tbuckley/cabot-search huggingface dataset:
Second Autologous Transplants for Multiple Myeloma (MM) Relapse After a Prior Autologous Transplant (AHCT) – a Report From the Center for International Blood and Marrow Transplant Research (CIBMTR)
Authors: Ayman A Saad, David H. Vesole, Jennifer Le-Rademacher, Xiaobo Zhong, Angela Dispenzieri, Sagar Lonial, Gustavo A. Milone, Parameswaran Hari
Journal: Blood (2011)
Inpatient Hospitalization Costs Associated with Birth Defects Among Persons of All Ages — United States, 2013
Authors: Annelise C. Arth, Sarah C. Tinker, Regina M. Simeone, Elizabeth C. Ailes, Janet D. Cragan, Scott D. Grosse
Journal: MMWR Morbidity and Mortality Weekly Report (2017)

In the United States, major structural or genetic birth defects affect approximately 3% of live births (1) and are responsible for 20% of infant deaths (2). Birth defects can affect persons across their lifespan and are the cause of significant lifelong disabilities. CDC used the Healthcare Cost and Utilization Project (HCUP) 2013 National Inpatient Sample (NIS), a 20% stratified sample of discharges from nonfederal community hospitals, to estimate the annual cost of birth defect-associated hospitalizations in the United States, both for persons of all ages and by age group. Birth defect-associated hospitalizations had disproportionately high costs, accounting for 3.0% of all hospitalizations and 5.2% of total hospital costs. The estimated annual cost of birth defect-associated hospitalizations in the United States in 2013 was $22.9 billion. Estimates of the cost of birth defect-associated hospitalizations offer important information about the impact of birth defects among persons of all ages on the overall health care system and can be used to prioritize prevention, early detection, and care.
Donor Lymphocyte Infusions and Second Transplantation as Salvage Treatment for Relapsed Myelofibrosis After Reduced-Intensity allografting.
Authors: Nicolaus Kröger, Evgeny Klyuchnikov, Daniel Wolff, Martin Bornhäuser, Guido Kobbe, Arnon Nagler, Avichai Shimoni, Christian Koenecke, Christine Wolschke, Ulrike Bacher, Axel R. Zander
Journal: Blood (2010)
Results From the Dose Escalation Phase of a Randomized Phase 1–2 First-in-Human (FIH) Study of SGI-110, a Novel Low Volume Stable Subcutaneous (SQ) Second Generation Hypomethylating Agent (HMA) in Patients with Relapsed/Refractory MDS and AML
Authors: Hagop M. Kantarjian, Gail J. Roboz, David A. Rizzieri, Wendy Stock, Casey L. O'Connell, Elizabeth A Griffiths, Karen Yee, Raoul Tibes, Guillermo Garcia-Manero, Farhad Ravandi, Katherine Walsh, Eric Feldman, Ellen Ritchie, Arati Rao, Carlos Decastro, Aaron D. Schimmer, Ruben A. Mesa, Ibrahim Syed, Gavin Choy, Aram Oganesian, Pietro Taverna, Mohammad Azab, Woonbok Chung, Jean-Pierre Issa
Journal: Blood (2012)
A MEDICO-LEGAL PROBLEM.
Journal: JAMA (1892)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA HomeNew OnlineCurrent IssueFor Authors Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) Podcasts Clinical Reviews Editors' Summary Medical News Author Interviews More JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA journal
A Photodynamic Pathway to Apoptosis Induced by Hypericin in Human Myeloma ARH-77 Cell Line: Possible Relevance to Photodynamic Therapy.
Authors: Ferit Avcu, Ali U. Ural, Yusuf Baran, Gungor Sobaci, Mustafa Deveci, Turker Cetin, Aysel Pekel, Meral Sarper, Pinar Elci, Ufuk Gunduz
Journal: Blood (2005)
New Evidence for the Complexity of the Population Structure of <i>Mycobacterium tuberculosis</i> Increases the Diagnostic and Biologic Challenges
Authors: Véronique Dartois, Kohta Saito, Thulasi Warrier, Carl Nathan
Journal: American Journal of Respiratory and Critical Care Medicine (2016)

developing serotype-independent vaccines on the basis of these targets are not promising.Major limitations of the study-which are probably unresolvable because of the human model-are that the results addressed only one pneumococcal serotype and that only easily accessible circulating B cells were measured, whereas typical B-cell reservoirs like spleen, lymph nodes, and bone marrow were omitted.Particularly, the longlived population of plasma cells residing in the bone marrow could not be assessed, and we still do not know their role in mucosal protection against pneumococci in men ( 14).Other limitations, such as using only simple methods like enzyme-linked immunospot assay to discriminate plasma cells from the memory B cells instead of flow cytometry, can and should be addressed in future studies.In conclusion, this study identified polysaccharide memory B cells as an immunological correlate for mucosal protection, which, if confirmed, could become an immunological outcome parameter in studies investigating pneumococcal vaccines.
AT (doxorubicin-Taxol) versus AC (doxorubicin-cyclophosphamide) as first line chemotherapy in metastatic breast cancer (MBC): A phase III study. (originally presented at ASCO 2000. Abstract 282)
Authors: Mj Piccart, T Cufer, P Bruning, Re Coleman, Ha Calvert, T Gamucci, C Twelves, Luc Duchateau, L Biganzoli, E Donato-Di Paola, C Yague, R Epelbaum
Journal: Annals of Oncology (2000)
The rice paradox: Multiple origins but single domestication in Asian rice
Authors: Jae Young Choi, Adrian E. Platts, Dorian Q. Fuller, Yue-Ie Hsing, Rod A. Wing, Michael D. Purugganan
Journal: Molecular Biology and Evolution (2017)

The origin of domesticated Asian rice (Oryza sativa) has been a contentious topic, with conflicting evidence for either single or multiple domestication of this key crop species. We examined the evolutionary history of domesticated rice by analyzing de novo assembled genomes from domesticated rice and its wild progenitors. Our results indicate multiple origins, where each domesticated rice subpopulation (japonica, indica, and aus) arose separately from progenitor O. rufipogon and/or O. nivara. Coalescence-based modeling of demographic parameters estimate that the first domesticated rice population to split off from O. rufipogon was O. sativa ssp. japonica, occurring at ∼13.1–24.1 ka, which is an order of magnitude older then the earliest archeological date of domestication. This date is consistent, however, with the expansion of O. rufipogon populations after the Last Glacial Maximum ∼18 ka and archeological evidence for early wild rice management in China. We also show that there is significant gene flow from japonica to both indica (∼17%) and aus (∼15%), which led to the transfer of domestication alleles from early-domesticated japonica to proto-indica and proto-aus populations. Our results provide support for a model in which different rice subspecies had separate origins, but that de novo domestication occurred only once, in O. sativa ssp. japonica, and introgressive hybridization from early japonica to proto-indica and proto-aus led to domesticated indica and aus rice.
The <i>goddard</i> and <i>saturn</i> genes are essential for <i>Drosophila</i> male fertility and may have arisen <i>de novo</i>
Authors: Anna M. Gubala, Jonathan F. Schmitz, Michael J. Kearns, Tery T. Vinh, Erich Bornberg-Bauer, Mariana F. Wolfner, Geoffrey D. Findlay
Journal: Molecular Biology and Evolution (2017)

New genes arise through a variety of mechanisms, including the duplication of existing genes and the de novo birth of genes from noncoding DNA sequences. While there are numerous examples of duplicated genes with important functional roles, the functions of de novo genes remain largely unexplored. Many newly evolved genes are expressed in the male reproductive tract, suggesting that these evolutionary innovations may provide advantages to males experiencing sexual selection. Using testis-specific RNA interference, we screened 11 putative de novo genes in Drosophila melanogaster for effects on male fertility and identified two, goddard and saturn, that are essential for spermatogenesis and sperm function. Goddard knockdown (KD) males fail to produce mature sperm, while saturn KD males produce few sperm, and these function inefficiently once transferred to females. Consistent with a de novo origin, both genes are identifiable only in Drosophila and are predicted to encode proteins with no sequence similarity to any annotated protein. However, since high levels of divergence prevented the unambiguous identification of the noncoding sequences from which each gene arose, we consider goddard and saturn to be putative de novo genes. Within Drosophila, both genes have been lost in certain lineages, but show conserved, male-specific patterns of expression in the species in which they are found. Goddard is consistently found in single-copy and evolves under purifying selection. In contrast, saturn has diversified through gene duplication and positive selection. These data suggest that de novo genes can acquire essential roles in male reproduction.
Prognosis and comorbidities in stage III and IV endometrial cancer
Authors: K. Wright, E. Munro, M. del Carmen, A. K. Goodman
Journal: Journal of Clinical Oncology (2009)

e16553 Background: While endometrial cancer may be associated with many comorbid conditions, none have been characterized as changing overall prognosis. The aim of this study was to identify medical conditions or laboratory values, that may serve as prognostic factors in stage III and IV endometrial cancer. Methods: A retrospective chart review identified 112 women with stage III or IV endometrial cancer between years 1993–1998. Information about medical comorbidities and presenting lab values were collected using electronic medical records. Progression free survival (PFS) and overall survival (OS) were analyzed using the Kaplan-Meier survival method and the log rank test. Results: The average age was 64.9 yrs. 79 women (70.5%) had stage III disease and 33 women (29.5%) had stage IV disease. For those with a baseline creatinine &lt;1.2 (n = 91), the PFS and OS were not significantly different from those with a baseline creatinine ≥1.2 (n = 17; p = 0.554 and p = 0.487, respectively). There was a non-significant trend toward worse PFS for the 41 patients with hypertension (HTN) compared to the 62 without (48.0 and 61.2 months, p = 0.191). The overall survival was significantly worse for those with HTN (38.7 months vs. 56.0 months p = 0.046). For those with known coronary artery disease, no significant difference in PFS or OS was found (p = 0.792 and p = 0.312 respectively). Those with diabetes (n = 15) did not have a significantly different PFS compared to those who did not (n = 88; p = 0.728). The OS was worse at 20.1 months for those with diabetes compared to 54.3 months for those without (p = 0.001). Baseline albumin had a significant effect on both PFS and OS. Those with an albumin &lt;3.5 (n = 54) had a PFS of 46.2 months compared to 94.0 months for those with an albumin ≥3.5 (n = 23; p = 0.007), and the OS for those with low albumin was 44.8 months compared to 83.4 months for those with the higher albumin (p = 0.005). Conclusions: These results suggest that past medical history and some baseline laboratory values may be important in considering prognosis. In particular, patients with a history of HTN or diabetes have a worse overall survival. Those with a baseline albumin of &lt;3.5 have a worse PFS and OS. No significant financial relationships to disclose.
Constitutively Activating Mutations At the FLT3 Activation Loop Residue D835 Are Associated with Clinical Resistance to AC220
Authors: Catherine C Smith, Jason Chin, Elisabeth Lasater, Amy L. Paguirigan, Kimberly Lin, Whitney Stewart, Sara Salerno, Lauren E Damon, Mark J. Levis, Alexander E Perl, Kevin Travers, Andrew Kasarskis, Jerald P. Radich, Neil Shah
Journal: Blood (2012)
Proteome Analysis of Apoptotic MR2 Cells, Acute Promyelocytic Leukemia Cell Line with All-Trans Retinoic Acid Resistance, Induced by Arsenic Trioxide
Authors: Pengcheng He, Mei Zhang, Xiaoning Wang, Huaiyu Wang, Jieying Xi, Jing Li, Di Wu, Kaihua Wei, Hongli Wang
Journal: Blood (2008)
High Resolution HLA Typing by Next Generation Exome Sequencing
Authors: Chang Liu, Yang Xiao, Brian Duffy, Michael Zody, Eric Tycksen, Savita Shrivastava, Rakesh Nagarajan, Brenda J. Grossman, Thalachallour Mohanakumar, Mitra D Robi, John D Pfeifer
Journal: Blood (2012)
Downregulation of the Cell-Cycle Regulating Ubiquitin-Ligase APC/CCdh1 May Contribute to the Differentiation Block of AML1/Eto Positive AML.
Authors: Manuel Hein, Dominik Schnerch, Andrea Schmidts, Julia Felthaus, Dagmar Wider, Gabriele Ihorst, Monika Engelhardt, Ralph M. Waesch
Journal: Blood (2009)
Leukemogenic Cascade Induced by a Constitutively Active Neurotrophin Receptor, ΔTrkA.
Authors: Zhixiong Li, Johann Meyer, Mathias Rhein, Olga Kustikova, Cornelia Rudolph, Kenji Kamino, Thomas Neumann, Min Yang, Anke Wahlers, Boris Fehse, Gary W. Reuther, Brigitte Schlegelberger, Arnold Ganser, Christopher Baum
Journal: Blood (2006)
A Novel, Highly Sensitive, Rapid, Non-PCR Based Method for the Detection of the JAK2 V617F Mutation in Chronic Myeloproliferative Neoplasms.
Authors: Giulia Minnucci, Giulia Amicarelli, Silvia Salmoiraghi, Orietta Spinelli, Daniel Adlerstein, Alessandro Rambaldi
Journal: Blood (2009)
Establishment of Effective B Lymphocyte Ex Vivo Expansion on Human Cord Blood Using TPO, SCF, FL, IL-4, IL-10, and CD40L.
Authors: Yeung-Chul Mun, Kyoung-Eun Lee, Jung Mi Kwon, Seung-Hyun Nam, Eun Sun Yoo, Yun-Kyung Bae, Seung-Eun Lee, Jee-Young Ahn, Myung Soo Hyun, Ki Young Kwon, Soon Nam Lee, Chu-Myong Seong
Journal: Blood (2004)
Integrating Newer with Traditional Prognostic Factors in Evaluating Patients with CLL Receiving Frontline Chemoimmunotherapy.
Authors: William G Wierda, Susan M O’Brien, Xuemei Wang, Stefan H Faderl, Alessandra Ferrajoli, Guillermo Garcia-Manero, Jorge Cortes, Constantine S Tam, Jan A Burger, Lynne V Abruzzo, Ellen Schlette, Susan Lerner, Michael J Keating
Journal: Blood (2008)
Pilot Study of Low Dose Melphalan and Bortezomib for Treatment of Acute Myelogenous Leukemia and High-Risk Myelodysplastic Syndromes.
Authors: Jeffrey A. Bubis, Pamela Ely, Kenneth R. Meehan, Diane Stearns, Anna D. Schaal, Elizabeth A. Kimtis, Christopher Lowrey, Elizabeth M. Bengtson, John Hill, Marc Gautier
Journal: Blood (2005)
CBM-12EXOSOMAL PTEN DETECTION IN GLIOMA PATIENTS
Authors: Sasidhar V. Manda, Yogesh Kataria, Babul Reddy TatiReddy, Alok Ranjan, Amitava Ray
Journal: Neuro-Oncology (2015)

BACKGROUND: Tyrosine kinases (TKs) are key regulators of intracellular signaling including cellular differentiation and proliferation. Phosphatase and tensin homologue deleted in chromosome 10 (PTEN) is a potent tumor suppressor protein, an inhibitor of PI3K signaling pathway. Gliomas are associated with a reduction or loss of PTEN expression. It has been recently shown that PTEN is transported through exosomes in various cancers. Herein, we report incorporation of PTEN in exosomal glioma cargo for the first time. METHODS: Serum and paired glioma tissue biopsies with a confirmed histopathology were collected as per approved ethics protocol. Both serum exosomal RNA and biopsy tissue RNA was isolated using commercial kits (Invitrogen). PTEN expression was quantified using a semi-nested PCR with GAPDH as an internal control. Clinical pathology was correlated with tissue and exosomal PTEN expression. RESULTS: Analysis of exosomal RNA isolated from exosomes showed the presence of small RNA. Exosomal PTEN was detected in 13 of the 72 glioma patients in the exosomal fraction, confirmed with sequencing. Exosomal PTEN was detected in 26% of Grade II, 22 % of Grade III, and 12% of Grade IV patients. Tissue PTEN could not be detected in 11 of the 72 glioma patients. CONCLUSION: These data suggest that PTEN expression can be detected in exosomes of glioma and that its expression correlates with the extent of tissue grade. Thus, PTEN detection through a simple blood test could be a promising clinical tool for early tumor diagnosis.
Angiographic comparison of the internal mammary artery graft and the saphenous vein graft within 2 months and 6 to 12 months after coronary artery bypass surgery.
Authors: Hisayoshi Suma, Shinjiro Sasaki, Atsuro Takeuchi, Yuzo Hirota, Takao Ishimura, Akira Wakabayashi
Journal: Circulation (1988)

Angiographic status of the saphenous vein graft (SVG) and the internal mammary artery graft (IMAG) anastomosed to the left anterior descending artery were compared at two different postoperative periods; - within 2 months and at 6 to 12 months after the operation. In 50 SVGs and 35 IMAGs which were studied at the early postoperative period, the rate of intact, stenosed and occluded grafts were almost the same in these two kinds of grafts. However, in 35 SVGs and 25 IMAGs which were studied at the later period, the stenosis of SVG increased significantly while IMAG remained intact. The rate of intact, stenosed and occluded grafts at postoperative 6 to 12 months were 71%, 23% and 6% in SVG, and 88%, 8& and 4% in IMAG, respectively. Considering the better angiographic quality of IMAG, use of IMAG to bypass the most important coronary artery should be considered especially when the patients are younger.
Bone Marrow Adipocytes Facilitate Fatty Acid Oxidation Activating AMPK and a Transcriptional Network Supporting Survival of Acute Monocytic Leukemia Cells
Authors: Yoko Tabe, Shinichi Yamamoto, Kaori Saitoh, Kazumasa Sekihara, Norikazu Monma, Kazuho Ikeo, Kaoru Mogushi, Masato Shikami, Vivian Ruvolo, Jo Ishizawa, Numsen Hail, Saiko Kazuno, Mamoru Igarashi, Hiromichi Matsushita, Yasunari Yamanaka, Hajime Arai, Isao Nagaoka, Takashi Miida, Yoshihide Hayashizaki, Marina Konopleva, Michael Andreeff
Journal: Cancer Research (2017)

Leukemia cells in the bone marrow must meet the biochemical demands of increased cell proliferation and also survive by continually adapting to fluctuations in nutrient and oxygen availability. Thus, targeting metabolic abnormalities in leukemia cells located in the bone marrow is a novel therapeutic approach. In this study, we investigated the metabolic role of bone marrow adipocytes in supporting the growth of leukemic blasts. Prevention of nutrient starvation-induced apoptosis of leukemic cells by bone marrow adipocytes, as well as the metabolic and molecular mechanisms involved in this process, was investigated using various analytic techniques. In acute monocytic leukemia (AMoL) cells, the prevention of spontaneous apoptosis by bone marrow adipocytes was associated with an increase in fatty acid β-oxidation (FAO) along with the upregulation of PPARγ, FABP4, CD36, and BCL2 genes. In AMoL cells, bone marrow adipocyte coculture increased adiponectin receptor gene expression and its downstream target stress response kinase AMPK, p38 MAPK with autophagy activation, and upregulated antiapoptotic chaperone HSPs. Inhibition of FAO disrupted metabolic homeostasis, increased reactive oxygen species production, and induced the integrated stress response mediator ATF4 and apoptosis in AMoL cells cocultured with bone marrow adipocytes. Our results suggest that bone marrow adipocytes support AMoL cell survival by regulating their metabolic energy balance and that the disruption of FAO in bone marrow adipocytes may be an alternative, novel therapeutic strategy for AMoL therapy. Cancer Res; 77(6); 1453-64. ©2017 AACR.
Climate change and lung health: the challenge for a new president
Authors: Nicholas S Hopkinson, Nicholas Hart, Gisli Jenkins, Naftali Kaminski, Margaret Rosenfeld, Alan Smyth, Alex Wilkinson
Journal: Thorax (2017)

This is a story from recent history that we believe incoming President Trump urgently needs to hear. In 1985, a huge and growing hole in the planet's ozone layer was identified. Ozone in the stratosphere blocks some of the sun's ultraviolet radiation from reaching the Earth's surface, thus protecting its biosphere (which includes humans) from DNA damage that would otherwise occur. The hole was caused by the action of chlorofluorocarbons (CFCs) used in refrigeration and as aerosol propellants.1 Margaret Thatcher is admired by many on both sides of the Atlantic, including President Trump2 as a strong politician, a person with clear beliefs on which she acted; the ‘Iron Lady’ as Donald Trump has described her on Twitter. A scientist by background, Thatcher appreciated the magnitude of the threat immediately, throwing her weight behind international efforts to address this. Working with Ronald Reagan, within 2 years the 1987 Montreal Protocol was signed to phase out the production and use of CFCs. Despite this, because CFCs persist in the atmosphere for more than a century, the ozone layer will not recover completely until 2060. A failure of leadership at that time would have been catastrophic. CFCs are also powerful greenhouse gases and without the Montreal agreement the world would already …
Functional Characterization of a Novel GFI1B Mutation Causing Congenital Macrothrombocytopenia
Authors: Katsumasa Kitamura, Yusuke Okuno, Kenichi Yoshida, Masashi Sanada, Yuichi Shiraishi, Hideki Muramatsu, Ryoji Kobayashi, Satoru Miyano, Seiji Kojima, Seishi Ogawa, Shinji Kunishima
Journal: Blood (2015)
Bayesian Multilevel Evaluation of Stem Cell Cryopreservation at the First National Umbilical Cord Blood Bank from the Public Health Sector in Mexico.
Authors: Eva D. Calderon-Garciduenas, Sergio A. Sanchez-Guerrero, Javier Fernandez-Torres, Miriam Millan-Rocha, Antonio Marin-Lopez, Leopoldo Valiente-Banuet, Monica Perez-Huizar
Journal: Blood (2004)
Does the Achievement of MR4.5 Improve the Outcome of Patients with Chronic Phase Chronic Myeloid Leukemia (CP-CML) Treated with Front Line Tyrosine Kinase Inhibitors (TKI)?
Authors: Lorenzo Falchi, Hagop M. Kantarjian, Xuemei Wang, Elias Jabbour, Farhad Ravandi, Guillermo Garcia-Manero, Zeev Estrov, Tapan Kadia, Naveen Pemmaraju, Alessandra Ferrajoli, Naval Daver, Sara Emogene Dellasala, Sherry Pierce, Srdan Verstovsek, Jorge E. Cortes
Journal: Blood (2015)
Steroid Refractoriness, but Not Rapidity of Response, to Induction Therapy Predicts for Outcome Post-Autologous Stem Cell Transplant (ASCT) in Patients (Pts) with Multiple Myeloma (MM)
Authors: Anca Prica, Young Trieu, Wei Xu, Donna E. Reece, Suzanne Trudel, Vishal Kukreti, Peter Anglin, Christine Chen
Journal: Blood (2008)
Long Term Results of Non-Myeloablative Stem Cell Transplantation in Non-Hodgkin’s Lymphomas: FLU150/MEL80 (Madrid Protocol).
Authors: Jorge Gayoso, Pascual Balsalobre, Jose F. Tomás, Javier de la Serna, David Serrano, Rafael Carrión, Ismael Buño, Alfonso Gómez-Pineda, Jose L. Díez-Martín
Journal: Blood (2007)
Peptides Inhibiting Tissue Factor Pathway Inhibitor Improve Hemostasis in Mice
Authors: Michael Dockal, Rudolf Hartmann, Thomas Polakowski, Osterkamp Frank, Hartmut J. Ehrlich, Alexandra Schiviz, Werner Hoellriegl, Eva-Maria Muchitsch, Ulrich Reinecke, Friedrich Scheiflinger
Journal: Blood (2011)
Darbepoetin-Alfa and I.V. Iron Administration after Autologous Hematopoietic Stem Cell Transplantation: A Prospective Randomized Multicenter Trial
Authors: Yves Beguin, Johan Maertens, Bernard De Prijck, Rik Schots, Pascale Frere, Christophe Bonnet, Kaoutar Hafraoui, Evelyne Willems, Gaetan Vanstraelen, Marie Lejeune, Koen Theunissen, Georges Fillet, Frederic Baron
Journal: Blood (2008)