tbuckley/cabot-search huggingface dataset:
Tumor Control Achieved Despite Loss of Chimaerism in Renal Cell Carcinoma (RCC) Following Non-Myeloablative Allogeneic Peripheral Blood Stem Cell Transplantation (PBSCT): A Case Report.
Authors: Giovanna Andreola, Federica Gigli, Cristina Rabascio, Manuela Cazzanti, Fedro Peccatori, Saverio Cinieri, Rocco Pastano, Giovanni Martinelli
Journal: Blood (2005)
SAT0147 Is IT Possible to Quantify Morning Stiffness Objectively?: Table 1.
Authors: G. Mengi, F. Gogus, M. Beyazova
Journal: Annals of the Rheumatic Diseases (2014)

<h3>Background</h3> Morning stiffness exhibits a circadian rhythm. Its assessment is subjective and no method has been introduced yet to measure it quantitatively. Morning stiffness may cause a delay in response to a stimulus and this delay in response time can be measured electrophysiologically (1,2). <h3>Objectives</h3> To quantify morning stiffness and to determine the relationship between disease activity and morning stiffness in patients with rheumatoid arthritis. <h3>Methods</h3> 27 patients with rheumatoid arthritis (23 female, 4 male; with a median age 55.00 [24-74] years) were enrolled. Premotor reaction and response times of the participants were measured electromyographically. Premotor time is the interval between the stimulation and the beginning of muscle activation. Motor time is the interval between the beginning of muscle activation to perform a prespecified task and fulfillment of that task. Each subject was seated on a chair with the back supported and forearms on the lap. A button was located on a vertical platform placed in front of the patient at arm9s length. With a surface stimulation electrode, a painless electrical stimulus of 5 mA was applied on the shoulder of the non-dominant side. The patient was asked to press the button in response to the stimulus. The active recording surface electrode was placed over the anterior fibers of the deltoid muscle and the reference surface electrode was placed over the acromion on the dominant side. Recordings were performed twice at 8:00 AM and 17:00 PM on the same day. Grip strength was measured by a dynamometer. Quality of life and pain were evaluated by health assessment questionnaire (HAQ) and visual analogue scale (VAS), respectively. DAS28 was also evaluated. <h3>Results</h3> Motor time, grip strength, VAS and HAQ measurements improved significantly improved in the evening (Table 1). There was positive correlation between DAS28 and both duration (r=0.448, p=0.019) and severity (r=0.574, p=0.002) of morning stiffness, HAQ (r=0.553, p=0.003) and VAS (r=0.580, p=0.002). <h3>Conclusions</h3> This is a preliminary trial to quantify morning stiffness. We observed a decrease in the motor reaction time in the evening which may be due to amelioration of stiffness during the day. We believe that this electrophysiological method may permit assessment of morning stiffness more objectively. <h3>References</h3> Ozyemisci-Taskiran O, Gencay-Can A, Gunendi Z, Beyazova M. A Single Dose of Indomethacin Does Not Prolong Premotor Reaction Time in Young, Healthy Adults: A Randomized, Placebo-Controlled, Double-Blind, Cross-Over Study.Double-Blind, Cross-Over Study. Turk J Rheumatol 2012;27(1):24-30. Dickstein R, Hocherman S, Amdor G, Pillar T. Reaction and Movement Times in Patients with Hemiparesis for Unilateral and Bilateral Elbow Flexion. Phys Ther. 1993;73:374-380. <h3>Disclosure of Interest</h3> None declared <h3>DOI</h3> 10.1136/annrheumdis-2014-eular.4958
FRI0130 Efficacy of A Comprehensive Cardiovascular Risk Reduction Patient Education Programme in Patients with Early Inflammatory Arthritis Following A Treat To Target Therapeutic Regime: Table 1.
Authors: H. Inomata, L.-A. Bissell, K. Naraghi, J. Nam, A. Burska, E. Hensor, P. Emery, M. Buch, J. Andrews
Journal: Annals of the Rheumatic Diseases (2016)

<h3>Background</h3> Evidence suggests cardiovascular disease (CVD) risk in patients (pt, pts) with Inflammatory arthritis (IA), Rheumatoid arthritis (RA) is equivalent to that of type II diabetes mellitus<sup>1</sup>. The accepted guidance is lifestyle factor modifications should be addressed in IA<sup>2</sup>, but little is known of the impact of CVD risk reduction pt education programmes delivered to pts with IA within a Rheumatology service setting. <h3>Objectives</h3> To describe the utility of a comprehensive CVD risk reduction pt education programme in pts with newly diagnosed IA following a treat to target standard therapeutic regime. <h3>Methods</h3> 387 pts with IA (RA and Undifferentiated Inflammatory Arthritis: UIA) were assessed at baseline (BL) for CV risk factors (RFs) (see Table 1) Pts were; RA; pts (1987 or 2010 ACR criteria) (n=237) and UIA; pts not meeting ACR RA criteria (n=150). A standardized co-morbidity pt education programme, which included CVD risk reduction, was delivered throughout the first 12 months (M). The impact on modifiable CVD RFs was assessed at 12M. <h3>Results</h3> 108 RA, 34 UIA pts had complete datasets (see Table 1). Of those smoked at BL, 15% reported being ex-smokers at 12M and the number smoked had decreased in 44%. 10% who reported being ex-smokers at BL altered their status to never having smoked by 12M and their reported number previously smoked reduced by 43% at 12M. Alcohol use reduced in 39% and increased in 24% (overall decrease, p=0.033). At BL, 56% of pts reported doing no moderate exercise at all; at 12M around a third of the pts had either increased or decreased the amount of exercise (no overall change). Disease activity generally decreased (p&lt;0.001) and mobility increased (p=0.004). <h3>Conclusions</h3> A significant reduction in alcohol units and improvement in smoking habits were seen. However, despite significant improvements in IA disease activity and mobility, there was no improvement in exercise from the low level reported at BL. The need to address modifiable CVD RFs in IA is widely accepted by Rheumatologists but effecting lifestyle change is challenging. Behavioural change in exercise is complex, particularly in chronic disease. Further studies are required to optimise the approach in IA. <h3>References</h3> Ann Rheum Dis 2009; 68:1395–1400 ARD 2010; 69:325–331 <h3>Disclosure of Interest</h3> None declared
Follow-Up of WT1-Specific Cytotoxic T Lymphocytes (CTL) with Tetramer-Based Analysis after Allogeneic Stem Cell Transplantation (SCT) in Patients with Renal Cell Carcinoma (RCC).
Authors: Yuriko Morita, Mami Hosokawa, Yuji Heike, Tohru Sasaki, Michiko Ebisawa, Shin-ichiro Mori, Ryuji Tanosaki, Masahiro Kami, Kisato Nosaka, Mutsuko Ohnishi, Osamu Miura, Kensei Tobinai, Yoichi Takaue
Journal: Blood (2004)
SAT0286 Risk of Severe Herpes Simplex Virus Infection in Patients Sle: A Nationwide Population Cohort Study: Table 1.
Authors: T.-H. Li, C.-C. Lai, Y.-S. Chang
Journal: Annals of the Rheumatic Diseases (2016)

<h3>Background</h3> Severe herpes simplex virus (HSV) infections, especially encephalitis and keratitis, often affect immunocompromised patients and result in severe sequela. However, only few attempts have been made at the association between systemic lupus erythematosus (SLE) and HSV. <h3>Objectives</h3> To identify the risk of severe HSV infection in SLE patients by means of a nationwide population-based cohort study. <h3>Methods</h3> We identified SLE patients from the National Health Insurance research database between 1997 and 2012. We compared the incidence rate (IR) of severe HSV infection, including viral septicaemia, CNS infection, ocular infection, visceral infection and those with complications after infection, with that of non-SLE cohort. A Cox multivariable proportional hazards model was applied to evaluate the risk of severe HSV infection in the SLE cohort. <h3>Results</h3> A total of 122,520 subjects (24,504 SLE patients and 98,016 age- and gender-matched subjects as control group) were analyzed and revealed a significantly higher IR of severe HSV infection in SLE (Incidence rate ratio =3.52, <i>p</i> &lt;0.001). Previous skin infection (HR=2.17, <i>p</i>=0.047), intravenous steroid pulse therapy (HR=4.48, <i>p</i>&lt;0.001), oral daily steroid dose over 7.5mg prednisolone or equivalent (HR=1,60, <i>p</i>=0.010) were independent risk factors for severe HSV infection in SLE patients, while Age ≤18 (Hazard ratio [HR]=0.47, <i>p</i>=0.021) was a protective factor. <h3>Conclusions</h3> A higher risk of severe HSV infection was observed in SLE patients Risk factors for severe HSV infection were age over 18, previous skin infection, intravenous steroid pulse therapy and an oral daily steroid dose over 7.5mg. <h3>References</h3> Infection risk in systemic lupus erythematosus patients: susceptibility factors and preventive strategies. Lupus. 2013 Oct;22(12):1286–94 Bosch X, Guilabert A, Pallarés L, Cerveral R, Ramos-Casals M, Bové A, Ingelmo M, Font J. Infections in systemic lupus erythematosus: a prospective and controlled study of 110 patients. Lupus. 2006;15(9):584–9. The nature and outcome of infection in systemic lupus erythematosus. Lupus. 2002;11(4):234–9. <h3>Disclosure of Interest</h3> None declared
Zevalin®/BEAM/Rituximab vs BEAM/Rituximab and Autologous Stem Cell Transplantation (ASCT) for Relapsed Chemosensitive Diffuse Large B-Cell Lymphoma (DLBCL): Impact of the IPI and PET Status.
Authors: Amin M. Alousi, Chitra Hosing, Rima M. Saliba, Rosamar B. Valverde, Farzaneh Maadani, Martin Korbling, Grace J. Okoroji, Luis E. Fayad, Anne M. Stachowiak, William D. Erwin, Paolo Anderlini, Marcos J. de Lima, Sergio A. Giralt, Uday R. Popat, Partow Kebriaei, Naoto T. Ueno, Peter W. McLaughlin, Barbara Pro, Michael Wang, Alma Rodriguez, Fredrick B. Hagemeister, Homer A. Macapinlac, Donald A. Podoloff, Richard E. Champlin, Issa F. Khouri
Journal: Blood (2007)
Reduced Intensity Conditioning (RIC) with Fludarabine, Busulfan and Cyclophosphamide for High Risk Patients with Thalassemia Major Undergoing Allogeneic Bone Marrow Transplantation Results in High Rejection Rates.
Authors: Mammen Chandy, Vikram Mathews, Biju George, Auro Vishwabandya, Salamun Desire, M.L. Kavitha, Alok Srivastava
Journal: Blood (2007)
AB0976 Achilles Tendon Injury and Complex Patellar for Overuse in Soccer Players: Ultrasound Investigation before and after The Match
Authors: S. Tropea, C. Trovato, A. Tropea
Journal: Annals of the Rheumatic Diseases (2016)

<h3>Background</h3> Football is one of the most popular sports in the world. Many players are suffering from disease of the Achilles tendon and the complex of the patellar tendon during periods of intense training with characteristics painful conditions of the tendons. Football players use their tendons to the limit because of the 9intensity of your workouts and frequent games. They participate in tournaments with echographic signs of underlying disease, and most of them play with a certain degree of pain. <h3>Objectives</h3> To investigate the changes in the players ultrasound at baseline and after participating in a youth football tournament. <h3>Methods</h3> 72 players were interviewed. 64 of the respondents underwent ultrasound examination before the game. 45 of the 64 players (70%) were scanned again at follow-up after the match. The interviews were conducted by the same person and all scans were done by the same doctor. The players were chosen randomly among those controlled interview Scanning was performed with a 15 MHz linear transducer. E 9was performed a careful examination grayscale and Doppler of the Achilles tendon and patellar complex. Ultimate goal was to determine the amount and distribution of activities Doppler with a score of 0–4. <h3>Results</h3> Sixty-two players (86%) had severe damage to tendons 80 over the past three years. 45 (50%) covered the whole patellar and 34 (37%) the Achilles tendon. In 60% of the problems they had started slowly and the mean duration of symptoms was 4 months (0–36). The 986% accepted the pain, 21% took painkillers, especially NSAIDs, to be able to play. At baseline, the Doppler activity was evaluated in most players. After the match, these changes were not significantly changed in most investigated regions. However, a very significant increase was observed in the tendons on the non-dominant side: quadriceps (Willcoxon p&lt;0.001), patellar tendon (p&lt;0.05), and Achilles tendon central portion (p&lt;0.05). <h3>Conclusions</h3> Doppler activity was found in all the tendons of soccer players examined to indicate a high degree of tension of these tendons. An increase in the fraction of color Doppler was significant only at the non-dominant leg. This can be explained by the fact that players load more frequently on this side. Changes Doppler correspond to the highest number of accidents on this side and a possible prognostic value of Doppler information is to be tested in a prospective study. <h3>References</h3> Cook JL, Khan KM, Harcourt PR, Kiss ZS, Fehrmann MW, Griffiths L, Wark JD. Patellar Tendon Ultrasonography in Asymptomatic Active Athletes Reveals Hypoechoic Regions: A Study of 320 Tendons. Clin J Sport Med. 1998 Apr;8(2):73–7. Ohberg L, Alfredson H, and Khan K. Ultrasound guided sclerosis of neovessels in painful chronic Achilles tendinosis: pilot study of a new treatment. Br J Sports Med. 2002 Jun;36(3):173–177. <h3>Disclosure of Interest</h3> None declared
A Potential Therapeutic Strategy for Chronic Lymphocytic Leukemia by Combining GS-1101, a PI3 kinase Delta (PI3Kδ) Inhibitor and a Novel Highly Selective Spleen Tyrosine Kinase (Syk) Inhibitor, GS-9973
Authors: Russell T Burke, Astrid Clarke, Sarah A Meadows, Marc M Loriaux, Jennifer B Dunlap, Guang Fan, Patricia Maciejewski, Julie Di Paolo, Brian J. Lannutti, Brian J. Druker, Stephen E Spurgeon
Journal: Blood (2012)
Outcome in Unselected Patients with Relapsed/Refractory Diffuse Large B-Cell Lymphoma (DLBCL) Following R-CHOP When Stem Cell Transplantation Is Not Feasible
Authors: Roopesh R. Kansara, Kerry J. Savage, Diego Villa, Tamara Shenkier, Alina S. Gerrie, Richard Klasa, Felicitas Hitz, David W. Scott, Graham W. Slack, Randy D. Gascoyne, Joseph M Connors, Laurie H. Sehn
Journal: Blood (2014)
Human dendritic cells (DCs) are derived from distinct circulating precursors that are precommitted to become CD1c+ or CD141+ DCs
Authors: Gaëlle Breton, Shiwei Zheng, Renan Valieris, Israel Tojal da Silva, Rahul Satija, Michel C. Nussenzweig
Journal: The Journal of Experimental Medicine (2016)

In humans, conventional dendritic cells (cDCs) exist as two unique populations characterized by expression of CD1c and CD141. cDCs arise from increasingly restricted but well-defined bone marrow progenitors that include the common DC progenitor that differentiates into the pre-cDC, which is the direct precursor of cDCs. In this study, we show that pre-cDCs in humans are heterogeneous, consisting of two distinct populations of precursors that are precommitted to become either CD1c+ or CD141+ cDCs. The two groups of lineage-primed precursors can be distinguished based on differential expression of CD172a. Both subpopulations of pre-cDCs arise in the adult bone marrow and can be found in cord blood and adult peripheral blood. Gene expression analysis revealed that CD172a+ and CD172a− pre-cDCs represent developmentally discrete populations that differentially express lineage-restricted transcription factors. A clinical trial of Flt3L injection revealed that this cytokine increases the number of both CD172a− and CD172a+ pre-cDCs in human peripheral blood.
Proteasome Inhibitor/Imatinib Combinations Exert Potentiated Antileukemia Efficacy but Not Cardiotoxicity in Chronic Myeloid Leukemia.
Authors: Guangbiao Zhou, Zheng Hu, Xiaofen Pan, Jinglei Liu, Liyuan Ma, Xiaoli Liu, Fanyi Meng, Juan Li, Zhu Chen, Saijuan Chen
Journal: Blood (2007)
Prognostic Significance of Intracellular Survivin in Myeloid Blast Cells As an Inhibitor of Apoptosis in Egyptian Adult Acute Myeloid Leukemia (AML) Patients
Authors: Mohamed osman Azzazi, Mervat Mattar, Soha Ezz El-Arab, Hani Hegab, Rasha Ibrahim, Walaa Elsalakawy, Mohammad Shazly
Journal: Blood (2014)
AB0963 Inteference of ANAs in ANCa Analysis and Comparison To Clinical Outcome of Vasculitis
Authors: M.F. Benavides Solarte, S.M.C. Romero, A. Castro, I. Angarita, A.I. Ospina, V. Parra, I. Galindo, L. Chila, L. Rey, A. Villa, S. Pineda, M. Casas, J.M. Bello, R. Valle
Journal: Annals of the Rheumatic Diseases (2016)

<h3>Background</h3> Anti-neutrophil cytoplasmic antibodies (ANCAs) are directed against the primary granules of neutrophils and monocytes and are essential in the diagnosis of vasculitis pauciimmune. The positivity of anti nuclear antibodies (ANAs) interferes with the interpretation of ANCAs leading to false positives. <h3>Objectives</h3> Evaluate the interference of ANAs in the interpretation of ANCAs in a population with manifestations of autoimmune disease <h3>Methods</h3> 3330 patient data involving ANCA requests between 2013 and 2015 were analyzed. Indirect immunofluorescence (IIF) was used to determine whether the samples had interference with ANAs through the L-ANCA system (Inmmunoconcepts®). Results were evaluated by the Generic assay kit ELISA® and CytoBead® ANCA (Generic Assays) to confirm positivity to MPO and/or PR3.The technical performance was evaluated by correlation analysis with Kappa values for data in which ANCAs was assessed at least by two techniques. Subsequently positivity of ANCAs was determined in a small sample of patients with clinical manifestations of autoimmune disease <h3>Results</h3> Positive ANCAS were found in 10.21% (340/3330) of the data, 40% (136/340) were C-ANCA and 60% (204/340) P-ANCA. Only 67 requests included specific anti-MPO and anti-PR3 assays, and 62.7% of these (42/67) were positive for ANCAs. ANAs were requested in 538 data resulting positive in 68.21% (367), predominantly homogeneous pattern. 12,64% of ANCAs positive data were also positive for ANAs, with P-ANCA in 83.72% of cases. The technique comparison included 44 samples positive for ANCAs evaluated by L-ANCA, CytoBead ANCA and ELISA. The correlation analysis showed high concordance between <i>CytoBead</i> and ELISA for PR3 (K=1.00 p&lt;0.05) and MPO (K=0.94 p&lt;0.05). However, in the evaluation between <i>CytoBead</i> ANCA and ANCA-L, a very low concordance (K=0.17 p&lt;0.05) was observed <h3>Conclusions</h3> The results suggest there is interference of ANAs in the positive P-ANCA results. IIF is an excellent technique for screening the presence of ANCAs while other methods such as anti-PR3 and anti- MPO by ELISA or microparticles assays are required for confirmation <h3>References</h3> Jennette JC, Falk RJ. Pathogenesis of antineutrophil cytoplasmic autoantibody-mediated disease. Nat. Rev. Rheumatol. 10, 463–473 (2014). Csernok E, Moosig F. Current and emerging techniques for ANCA detection in vasculitis. Nat. Rev. Rheumatol. 2014;10(8):494–501. Radice A, Bianchi L, Sinico RA. Anti-neutrophil cytoplasmic autoantibodies: Methodological aspects and clinical significance in systemic vasculitis. Autoimmun Rev 2013;12:487–95. <h3>Disclosure of Interest</h3> None declared
Alpha-Catenin, ARHGAP21 and β-Catenin Are Abnormally Expressed in Bone Marrow Cells From Patients with Myelodysplatic Syndromes and Are a Possible Target for Decitabine Treatment.
Authors: Karin Spat Barcellos, Sheila Maria Winnischofer, Mariana Lazarini, Adriana Silva Santos Duarte, Carolina Louzao Bigarella, Fabiola Traina, Fernando Ferreira Costa, Sara T.O Saad
Journal: Blood (2009)
Natriuretic peptides and integrated risk assessment for cardiovascular disease: an individual-participant-data meta-analysis
Authors: Peter Willeit, Stephen Kaptoge, Paul Welsh, Adam S Butterworth, Rajiv Chowdhury, Sarah A Spackman, Lisa Pennells, Pei Gao, Stephen Burgess, Daniel F Freitag, Michael Sweeting, Angela M Wood, Nancy R Cook, Suzanne Judd, Stella Trompet, Vijay Nambi, Michael Hecht Olsen, Brendan M Everett, Frank Kee, Johan Ärnlöv, Veikko Salomaa, Daniel Levy, Jussi Kauhanen, Jari A Laukkanen, Maryam Kavousi, Toshiharu Ninomiya, Juan-Pablo Casas, Lori B Daniels, Lars Lind, Caroline N Kistorp, Jens Rosenberg, Thomas Mueller, Speranza Rubattu, Demosthenes B Panagiotakos, Oscar H Franco, James A de Lemos, Andreas Luchner, Jorge R Kizer, Stefan Kiechl, Jukka T Salonen, S Goya Wannamethee, Rudolf A de Boer, Børge G Nordestgaard, Jonas Andersson, Torben Jørgensen, Olle Melander, Christie M Ballantyne, Christopher DeFilippi, Paul M Ridker, Mary Cushman, Wayne D Rosamond, Simon G Thompson, Vilmundur Gudnason, Naveed Sattar, John Danesh, Emanuele Di Angelantonio
Journal: The Lancet Diabetes & Endocrinology (2016)

Guidelines for primary prevention of cardiovascular diseases focus on prediction of coronary heart disease and stroke. We assessed whether or not measurement of N-terminal-pro-B-type natriuretic peptide (NT-proBNP) concentration could enable a more integrated approach than at present by predicting heart failure and enhancing coronary heart disease and stroke risk assessment.
Alemtuzumab Use and Survival After Reduced Intensity Allogeneic Stem Cell Transplantation in High-Risk Chronic Lymphocytic Leukemia (CLL),
Authors: Wei Ding, Tait D. Shanafelt, Kari Rabe, Timothy G. Call, Clive S. Zent, Neil Kay, David James Inwards, Deborah Bowen, Mark R. Litzow, Susan L Slager, Susan Schwager, William J Hogan
Journal: Blood (2011)
Allograft After Pediatric-Inspired Therapy Does Not Improve Young patient's Outcome with High Risk Philadelphia- Chromosome-Negative Acute Lymphoblastic Leukemia (Ph-ALL). A Single Center Report
Authors: Thibaut Leguay, Arnaud Pigneux, Reza Tabrizi, Mathieu Sauvezie, Krimo Bouabdallah, Marie-Sarah Dilhuydy, Axelle Lascaux, Carine Foucaud, Anna Schmitt, Stephane Vigouroux, Gerald Marit, noel-Jean Milpied
Journal: Blood (2010)
Clinical Utility of Morphological Evaluation of Day 14 Bone Marrow Biopsies in Acute Myeloid Leukemia Patients Undergoing Standard Induction Chemotherapy: Time to Change Practice?
Authors: Yehuda E. Deutsch, German Campuzano-Zuluaga, Matthew P Salzberg, Alexandra Gomez Arteaga, Justin M. Watts, Jennifer R Chapman, Offiong F Ikpatt, Francisco Vega, Ronan T Swords
Journal: Blood (2014)
Hematopoietic Cell Transplantation-Specific Comorbidity Index To Predict Non-Relapse Mortality and Survival after Allografting.
Authors: Ruri Kato, Takahiro Fukuda, Eiji Usui, Satoshi Yamasaki, Dai Maruyama, Yuriko Morita, Sung-Won Kim, Shin-ichiro Mori, Ryuji Tanosaki, Kinuko Tajima, Yuji Heike, Atsushi Makimoto, Kensei Tobinai, Yoichi Takaue
Journal: Blood (2005)
Phase I Results of Combination Gemcitabine and Bortezomib (Velcade®) for Relapsed/Refractory Nodal T-Cell Non-Hodgkin Lymphoma (T-NHL) and Aggressive B-Cell NHL (B-NHL).
Authors: Andrew M. Evens, Leo I. Gordon, David Patton, Steven T. Rosen, Jane N. Winter, Justin Kline, Koen Van Besien, Sonali M. Smith
Journal: Blood (2008)
The Safety and Tolerability of Oral Fludarabine, ±oral Cyclophosphamide and Iv Rituximab Therapy In Previously Untreated Patients with Chronic Lymphocytic Leukaemia (CLL) Aged ≥65 Years – Interim Analysis From the Australasian Leukaemia and Lymphoma Group (ALLG) and CLL Australian Research Consortium (CLLARC) CLL5 Study.
Authors: Stephen P Mulligan, Devinder S Gill, William E. P. Renwick, Melanie L Sulda, O. Giles Best, Bryone J Kuss, John F Seymour
Journal: Blood (2010)
Combination Of 2-Deoxy-D-Glucose With ABT-199 Efficiently Kills All Molecular Subgroups Of Myeloma Cells
Authors: Maxime Halliez, Sophie Maïga, Cyrille Touzeau, Patricia Gomez-Bougie, Steven Le Gouill, Marie-Christine Bene, PharmSciD, Philippe Moreau, Catherine Pellat-Deceunynck, Martine Amiot
Journal: Blood (2013)
Left ventricular geometry and altered contractility in a large population sample: Insensitivity of standard methods compared to left ventricular non-invasive single-beat end-systolic elastance
Authors: Ernst Rietzschel, Tom Claessens, Marc De Buyzere, Dirk De Bacquer, Gui De Backer, Thierry Gillebert, Pascal Verdonck, Patrick Segers
Journal: Circulation (2005)
Impact Of Cytogenetics and Cytogenetic Response On Outcome In Myelodysplastic Syndromes (MDS) treated With Azacitidine (AZA). A Collaborative Study In 878 Patients
Authors: Marie Sebert, Rami S Komrokji, Mikkael A. Sekeres, Thomas Prebet, Thomas Cluzeau, Valeria Santini, Alessandro Sanna, Najla H Al Ali, Sean Hobson, Virginie Eclache, Alan List, Pierre Fenaux, Lionel Ades
Journal: Blood (2013)