tbuckley/cabot-search huggingface dataset:
Associations of specific types of sports and exercise with all-cause and cardiovascular-disease mortality: a cohort study of 80 306 British adults
Authors: Pekka Oja, Paul Kelly, Zeljko Pedisic, Sylvia Titze, Adrian Bauman, Charlie Foster, Mark Hamer, Melvyn Hillsdon, Emmanuel Stamatakis
Journal: British Journal of Sports Medicine (2016)

Evidence for the long-term health effects of specific sport disciplines is scarce. Therefore, we examined the associations of six different types of sport/exercise with all-cause and cardiovascular disease (CVD) mortality risk in a large pooled Scottish and English population-based cohort.Cox proportional hazards regression was used to investigate the associations between each exposure and all-cause and CVD mortality with adjustment for potential confounders in 80 306 individuals (54% women; mean±SD age: 52±14 years).Significant reductions in all-cause mortality were observed for participation in cycling (HR=0.85, 95% CI 0.76 to 0.95), swimming (HR=0.72, 95% CI 0.65 to 0.80), racquet sports (HR=0.53, 95% CI 0.40 to 0.69) and aerobics (HR=0.73, 95% CI 0.63 to 0.85). No significant associations were found for participation in football and running. A significant reduction in CVD mortality was observed for participation in swimming (HR=0.59, 95% CI 0.46 to 0.75), racquet sports (HR=0.44, 95% CI 0.24 to 0.83) and aerobics (HR=0.64, 95% CI 0.45 to 0.92), but there were no significant associations for cycling, running and football. Variable dose-response patterns between the exposure and the outcomes were found across the sport disciplines.These findings demonstrate that participation in specific sports may have significant benefits for public health. Future research should aim to further strengthen the sport-specific epidemiological evidence base and understanding of how to promote greater sports participation.
Infer yourself: Interoception and internal “action” in conscious selfhood
Authors: Anil K Seth
Journal: Behavioral and Brain Sciences (2016)

Can consciousness be understood through an association with voluntary skeletomotor action selection? Although flexible and integrated action selection is a plausible function for consciousness, a narrow focus on skeletomotor control neglects the contributions to conscious selfhood and subjectivity that rest on interoception and autonomic regulation (internal "action"). I consider these issues from the perspective of predictive processing.
Myeloablative Versus Nonmyeloablative Hemopoietic Cell Transplantation (HCT) for Patients with Myelodysplasia (MDS) or AML with Multilineage Dysplasia Following MDS (tAML).
Authors: Bart L. Scott, Michael Maris, Brenda Sandmaier, Barry Storer, Thomas Chauncey, David G. Maloney, Mohamed Sorror, Rainer Storb, H. Joachim Deeg
Journal: Blood (2004)
Disorders of sex development: insights from targeted gene sequencing of a large international patient cohort
Authors: Stefanie Eggers, Simon Sadedin, Jocelyn A. van den Bergen, Gorjana Robevska, Thomas Ohnesorg, Jacqueline Hewitt, Luke Lambeth, Aurore Bouty, Ingrid M. Knarston, Tiong Yang Tan, Fergus Cameron, George Werther, John Hutson, Michele O’Connell, Sonia R. Grover, Yves Heloury, Margaret Zacharin, Philip Bergman, Chris Kimber, Justin Brown, Nathalie Webb, Matthew F. Hunter, Shubha Srinivasan, Angela Titmuss, Charles F. Verge, David Mowat, Grahame Smith, Janine Smith, Lisa Ewans, Carolyn Shalhoub, Patricia Crock, Chris Cowell, Gary M. Leong, Makato Ono, Antony R. Lafferty, Tony Huynh, Uma Visser, Catherine S. Choong, Fiona McKenzie, Nicholas Pachter, Elizabeth M. Thompson, Jennifer Couper, Anne Baxendale, Jozef Gecz, Benjamin J. Wheeler, Craig Jefferies, Karen MacKenzie, Paul Hofman, Philippa Carter, Richard I. King, Csilla Krausz, Conny M. A. van Ravenswaaij-Arts, Leendert Looijenga, Sten Drop, Stefan Riedl, Martine Cools, Angelika Dawson, Achmad Zulfa Juniarto, Vaman Khadilkar, Anuradha Khadilkar, Vijayalakshmi Bhatia, Vũ Chí Dũng, Irum Atta, Jamal Raza, Nguyen thi Diem Chi, Tran Kiem Hao, Vincent Harley, Peter Koopman, Garry Warne, Sultana Faradz, Alicia Oshlack, Katie L. Ayers, Andrew H. Sinclair
Journal: Genome biology (2016)

Disorders of sex development (DSD) are congenital conditions in which chromosomal, gonadal, or phenotypic sex is atypical. Clinical management of DSD is often difficult and currently only 13% of patients receive an accurate clinical genetic diagnosis. To address this we have developed a massively parallel sequencing targeted DSD gene panel which allows us to sequence all 64 known diagnostic DSD genes and candidate genes simultaneously. We analyzed DNA from the largest reported international cohort of patients with DSD (278 patients with 46,XY DSD and 48 with 46,XX DSD). Our targeted gene panel compares favorably with other sequencing platforms. We found a total of 28 diagnostic genes that are implicated in DSD, highlighting the genetic spectrum of this disorder. Sequencing revealed 93 previously unreported DSD gene variants. Overall, we identified a likely genetic diagnosis in 43% of patients with 46,XY DSD. In patients with 46,XY disorders of androgen synthesis and action the genetic diagnosis rate reached 60%. Surprisingly, little difference in diagnostic rate was observed between singletons and trios. In many cases our findings are informative as to the likely cause of the DSD, which will facilitate clinical management. Our massively parallel sequencing targeted DSD gene panel represents an economical means of improving the genetic diagnostic capability for patients affected by DSD. Implementation of this panel in a large cohort of patients has expanded our understanding of the underlying genetic etiology of DSD. The inclusion of research candidate genes also provides an invaluable resource for future identification of novel genes.
Secondary Central Nervous System Lymphoma: A Single Center Experience
Authors: Gregory M. Cote, Yang Feng, Aliyah R. Sohani, Zachary J. Roberts, Anna Colpo, Fred H Hochberg, Yi-Bin A. Chen, James S. Michaelson, Donna Neuberg, Jeremy S. Abramson, Ephraim P. Hochberg
Journal: Blood (2011)
Central Review of Cytogenetics Is Essential for Cooperative Group Clinical and Correlative Studies of Acute Leukemia: The Cancer and Leukemia Group B (CALGB) 8461 Experience.
Authors: Krzysztof Mrózek, Andrew J. Carroll, Kati Maharry, Kathleen W. Rao, Shivanand R. Patil, Mark J. Pettenati, Michael S. Watson, Diane C. Arthur, Ramana Tantravahi, Nyla A. Heerema, Prasad R.K. Koduru, Sandra H. Bigner, Judith Stamberg, AnneMarie W. Block, Colin G. Edwards, Lisa J. Sterling, Clara D. Bloomfield
Journal: Blood (2004)
Intra-arterial Treatment for Basilar Artery Occlusion
Authors: Alysha Bhatti, Prasanthi Sivakumaran
Journal: JAMA Neurology (2016)

To the Editor We thank van Houwelingen et al1 for their retrospective study exploring the efficacy and safety of intra-arterial (IA) treatment in basilar artery occlusion (BAO). They reported adequate recanalization and favorable outcomes in the cohort receiving IA therapy after BAO.
Arteriography for Lower Gastrointestinal Hemorrhage—Reply
Authors: Gregory Nadolski, Christina L. Jacovides, Jose L. Pascual
Journal: JAMA Surgery (2016)

Our website uses cookies to enhance your experience. By continuing to use our site, or clicking "Continue," you are agreeing to our Cookie Policy | Continue JAMA Surgery HomeNew OnlineCurrent IssueFor Authors Podcast Publications JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry (1919-1959) JN Learning / CMESubscribeJobsInstitutions / LibrariansReprints & Permissions Terms of Use | Privacy Policy | Accessibility Statement 2023 American Medical Association. All Rights Reserved Search All JAMA JAMA Network Open JAMA Cardiology JAMA Dermatology JAMA Forum Archive JAMA Health Forum JAMA Internal Medicine JAMA Neurology JAMA Oncology JAMA Ophthalmology JAMA Otolaryngology–Head & Neck Surgery JAMA Pediatrics JAMA Psychiatry JAMA Surgery Archives of Neurology & Psychiatry Input Search Term Sign In Individual Sign In Sign inCreate an Account Access through your institution Sign In Purchase Options: Buy this article Rent this article Subscribe to the JAMA Surgery journal
Successful Generation of Mature Professional Acute Myeloid Leukemia (AML) – Antigen Presenting Cells (APC) Does Not Predict Successful Induction of AML Reactive T Cells for Adoptive Immunotherapy.
Authors: Pauline Meij, Marian van de Meent, Inge Jedema, Roel Willemze, J.H. Frederik Falkenburg
Journal: Blood (2005)
Micafungin Versus Posaconazole Anti-Fungal Prophylaxis in Adult Patients with Acute Leukemia Undergoing Induction Chemotherapy
Authors: Elizabeth Halton, Dick Chung, Kun Xiao, Hilda Quintanilla, Christina Baldwin, Pamela Baird, Ellin Berman, Renier J Brentjens, Mark Heaney, Joseph G. Jurcic, Nicole Lamanna, Todd L. Rosenblat, Rana Kaplan, Genovefa Papanicolaou, Mark G. Frattini
Journal: Blood (2012)
Patterns of Acute and Chronic Graft-Versus-Host Disease Following ATG-Based Conditioning for Allogeneic Hematopoietic Cell Transplantation
Authors: Nandita Khera, Amylou C. Dueck, Veena Devi Salem Fauble, Lisa Sproat, Pierre Noel, Jose Leis, Roberta H. Adams, James L Slack
Journal: Blood (2012)
P0625 : A hyper-glycosylation of HBV surface major hydrophilic region characterizes HBV reactivation driven by immunosuppression and affects HBsAg recognition in vitro
Authors: R. Salpini, M. Surdo, L. Colagrossi, A. Battisti, M. Pollicita, A. Bertoli, F. Di Santo, C. Becker, C. Mastroianni, M. Marignani, S. Maylin, C. Delaugerre, F. Morisco, N. Coppola, A. Marrone, L. Sarmati, M. Andreoni, M. Angelico, J. Verheyen, C.F. Perno, V. Svicher
Journal: Journal of Hepatology (2015)
Acute Monocytic Leukemia Associated Antigen MLAA-34 up-Regulates JAK2/STAT3 Expression and JAK2/STAT3 Enhances MLAA-34 Activation in a Positive Feedback Loop
Authors: Bo Lei, Ju Bai, Wanggang Zhang, Aili He, Yinxia Chen, Pengyu Zhang, Lu Qian, Fuling Zhou
Journal: Blood (2015)
High CD69 Protein Expression Predicts a Poor Prognosis in B-Cell Chronic Lymphocytic Leukemia (B-CLL).
Authors: Giovanni Del Poeta, Maria Ilaria Del Principe, Fabrizio Luciano, Luca Maurillo, Francesco Buccisano, Gianfranco Catalano, Laura Giannì, Angela Coletta, Antonella Zucchetto, Pasquale Niscola, Adriano Venditti, Paolo de Fabritiis, Valter Gattei, Sergio Amadori
Journal: Blood (2007)
Crystal Structure of the Emerging Cancer Target MTHFD2 in Complex with a Substrate-Based Inhibitor
Authors: Robert Gustafsson, Ann-Sofie Jemth, Nina M.S. Gustafsson, Katarina Färnegårdh, Olga Loseva, Elisée Wiita, Nadilly Bonagas, Leif Dahllund, Sabin Llona-Minguez, Maria Häggblad, Martin Henriksson, Yasmin Andersson, Evert Homan, Thomas Helleday, Pål Stenmark
Journal: Cancer Research (2016)

To sustain their proliferation, cancer cells become dependent on one-carbon metabolism to support purine and thymidylate synthesis. Indeed, one of the most highly upregulated enzymes during neoplastic transformation is MTHFD2, a mitochondrial methylenetetrahydrofolate dehydrogenase and cyclohydrolase involved in one-carbon metabolism. Because MTHFD2 is expressed normally only during embryonic development, it offers a disease-selective therapeutic target for eradicating cancer cells while sparing healthy cells. Here we report the synthesis and preclinical characterization of the first inhibitor of human MTHFD2. We also disclose the first crystal structure of MTHFD2 in complex with a substrate-based inhibitor and the enzyme cofactors NAD+ and inorganic phosphate. Our work provides a rationale for continued development of a structural framework for the generation of potent and selective MTHFD2 inhibitors for cancer treatment. Cancer Res; 77(4); 937-48. ©2017 AACR.
JAK2V617F Mutation in Leukaemic Transformation of Philadelphia Negative Chronic Myeloproliferative Disorders.
Authors: Davide Rossi, Clara Deambrogi, Daniela Capello, Michaela Cerri, Monia Lunghi, Lorenzo De Paoli, Guido Parvis, Giuseppe Saglio, Gianluca Gaidano, Daniela Cilloni
Journal: Blood (2006)
Human Hematopoietic Stem/Progenitor Cells (HSPCs) and Mesenchymal Stromal Cells (MSCs) Express Several Functional Pituitary and Gonadal Sex Hormone Receptors - Identification of Follicle Stimulating Hormone (FSH) and Luteinizing Hormone (LH) As New Growth Factors for HSPCs and MSCs
Authors: Ahmed Abdelbaset-Ismail, Malwina Suszynska, Sylwia Julita Borkowska, Mateusz Adamiak, Janina Ratajczak, Mariusz Z Ratajczak
Journal: Blood (2015)
Association between screening and the thyroid cancer “epidemic” in South Korea: evidence from a nationwide study
Authors: Sohee Park, Chang-Mo Oh, Hyunsoon Cho, Joo Young Lee, Kyu-Won Jung, Jae Kwan Jun, Young-Joo Won, Hyun-Joo Kong, Kui Son Choi, You Jin Lee, Jin Soo Lee
Journal: BMJ (2016)

To investigate whether screening for thyroid cancer led to the current "epidemic" in South Korea. Review of the medical records of nationally representative samples of patients with a diagnosis of thyroid cancer in 1999, 2005, and 2008. Sample cases were randomly selected from South Korea's nationwide cancer registry, using a systematic sampling method after stratification by region. 5796 patients with thyroid cancer were included (891 in 1999, 2355 in 2005, and 2550 in 2008). The primary outcome was age standardised incidence of thyroid cancer and the changes in incidence between 1999 and 2008 according to the methods used to detect tumours (screen detection versus clinical detection versus unspecified). Between 1999 and 2008, the incidence of thyroid cancer increased 6.4-fold (95% confidence interval 4.9-fold to 8.4-fold), from 6.4 (95% confidence interval 6.2 to 6.6) per 100 000 population to 40.7 (40.2 to 41.2) per 100 000 population. Of the increase, 94.4% (34.4 per 100 000 population) were for tumours less than 20 mm, which were detected mainly by screening. 97.1% of the total increase was localised and regional tumours according to the Surveillance, Epidemiology, and End Results (SEER) summary stage. Where cases were clinically detected, 99.9% of the increased incidences (6.4 per 100 000 population) over the same period were tumours less than 20 mm. The current "epidemic" of thyroid cancer in South Korea is due to an increase in the detection of small tumours, most likely as a result of overdetection. Concerted efforts are needed at a national level to reduce unnecessary thyroid ultrasound examinations in the asymptomatic general population.
AUY922, a Heat Shock Protein 90 (Hsp90) Inhibitor, Demonstrates Activity in Patients with Myeloproliferative Neoplasms (MPNs)
Authors: Gabriella Hobbs, Rivka Litvin, Jihae Ahn, Anna Sophia Mckenney, Michael J. Mauro, Martin S Tallman, Ellin Berman, Mark Lawrence Heaney, Ross L. Levine, Raajit K. Rampal
Journal: Blood (2015)
Post-Transplant Cyclophosphomide Spares Granzyme B Expression in T Regulatory Cells (Treg), but Not in CD8+ T and NK Cells after Allogeneic HSCT
Authors: Mikhail Drokov, Elena N. Parovichnikova, Larisa A. Kuzmina, Vera Vasilieva, Ekaterina Mikhaltsova, Olga Koroleva, Daria Dubnyak, Irina Galtseva, Valeri Savchenko
Journal: Blood (2015)
Increased proportion of mature NK cells is associated with successful imatinib discontinuation in chronic myeloid leukemia
Authors: M Ilander, U Olsson-Strömberg, H Schlums, J Guilhot, O Brück, H Lähteenmäki, T Kasanen, P Koskenvesa, S Söderlund, M Höglund, B Markevärn, A Själander, K Lotfi, A Dreimane, A Lübking, E Holm, M Björeman, S Lehmann, L Stenke, L Ohm, T Gedde-Dahl, W Majeed, H Ehrencrona, S Koskela, S Saussele, F-X Mahon, K Porkka, H Hjorth-Hansen, Y T Bryceson, J Richter, S Mustjoki
Journal: Leukemia (2016)

Recent studies suggest that a proportion of chronic myeloid leukemia (CML) patients in deep molecular remission can discontinue the tyrosine kinase inhibitor (TKI) treatment without disease relapse. In this multi-center, prospective clinical trial (EURO-SKI, NCT01596114) we analyzed the function and phenotype of T and NK cells and their relation to successful TKI cessation. Lymphocyte subclasses were measured from 100 imatinib-treated patients at baseline and 1 month after the discontinuation, and functional characterization of NK and T cells was done from 45 patients. The proportion of NK cells was associated with the molecular relapse-free survival as patients with higher than median NK-cell percentage at the time of drug discontinuation had better probability to stay in remission. Similar association was not found with T or B cells or their subsets. In non-relapsing patients the NK-cell phenotype was mature, whereas patients with more naïve CD56bright NK cells had decreased relapse-free survival. In addition, the TNF-α/IFN-γ cytokine secretion by NK cells correlated with the successful drug discontinuation. Our results highlight the role of NK cells in sustaining remission and strengthen the status of CML as an immunogenic tumor warranting novel clinical trials with immunomodulating agents.
Ileocolonic anastomosis fecalith impaction in a patient with Crohn’s disease: needle-knife stricturotomy and stone retrieval
Authors: Vivian Chidi, Abigail Oberc, Bo Shen
Journal: Endoscopy (2016)

Stasis of intestinal contents is a proposed mechanism for intestinal fecalith formation [1] [2]. Obstruction and perforation requiring surgical intervention are reported in the literature as consequences of stercoral ulcers at the stone bed [2] [3] [4]. We present the case of an impacted anastomotic site fecalith removed after needle-knife stricturotomy treatment of the mucosa ([Video 1]).
Guiding hMSC Adhesion and Differentiation on Supported Lipid Bilayers
Authors: Gülistan Koçer, Pascal Jonkheijm
Journal: Advanced Healthcare Materials (2016)

Mesenchymal stem cells (MSCs) are intensively investigated for regenerative medicine applications due to their ease of isolation and multilineage differentiation capacity. Hence, designing instructive microenvironments to guide MSC behavior is important for the generation of smart interfaces to enhance biomaterial performance in guiding desired tissue formation. Supported lipid bilayers (SLBs) as cell membrane mimetics can be employed as biological interfaces with easily tunable characteristics such as biospecificity, mobility, and density of predesigned ligand molecules. Arg‐Gly‐Asp (RGD) ligand functionalized SLBs are explored for guiding human MSC (hMSC) adhesion and differentiation by studying the effect of changes in ligand density and mobility. Cellular and molecular analyses show that adhesion occurs through specific interactions with RGD ligands where the extent is positively correlated to changes in ligand density. Furthermore, cell area is significantly regulated by ligand density on ligand‐mobile SLBs when compared to ligand‐immobile SLBs. Finally, the osteogenic differentiation capacity of hMSCs is positively correlated to ligand density on ligand‐mobile SLBs indicating that regulation of cell spreading is linked to cell differentiation capacity. These results demonstrate that hMSC behavior can be directed on SLBs by molecular design and presents SLBs as versatile platforms for future engineering of smart biomaterial coatings.
Prognostic Factors of Response to Erythropoiesis Stimulating Agents (ESA) Treatment in Non RBC Transfusion Dependent Lower Risk MDS. Preliminary Results of a French and Italian Study (on behalf of the GFM and FISM).
Authors: Sophie Park, Gina Zini, Valérie Andrieu, Alessandro Levis, Daniela Gioia, Eliane Duchayne, Antonella Poloni, Enrico Balleari, Marco Calabrese, Isabelle Sudaka, Virginie Siguret, Martine Guesnu, Olivier Kosmider, Michaela Fontenay, Alessandro Sanna, Pierre Lemaire, Odile Beyne-Rauzy, Lionel Ades, Laurence Legros, Paolo Danise, Pellegrino Musto, Pierre Fenaux, Francois Dreyfus, Valeria Santini
Journal: Blood (2012)
Diagnostic Utility of Flow Cytometry in Myelodysplastic Syndromes: A Prospective Validation Study in Low-Risk Patients with Normal Karyotype
Authors: Kiyoyuki Ogata, Matteo G. Della Porta, Luca Malcovati, Cristina Picone, Norio Yokose, Akira Matsuda, Taishi Yamashita, Hideto Tamura, Junichi Tsukada, Kazuo Dan, Mario Cazzola
Journal: Blood (2008)
Novel Heritable Mutation of the Transcription Factor RUNX1 as a Cause of Autosomal Dominant Familial Platelet Disorder with Predisposition to Acute Myeloid Leukemia (FPD/AML).
Authors: William N. Patton, Graeme Suthers, Meryl Altree, Catherine Carmichael, Ella Wilkins, Jacqueline Carroll, Lesley Rawlings, Hamish Scott
Journal: Blood (2007)
TG101209, a Selective JAK2 Kinase Inhibitor, Suppresses Endogenous and Cytokine-Supported Colony Formation from Hematopoietic Progenitors Carrying JAK2V617F or MPLW515K/L Mutations.
Authors: Animesh Pardanani, John Hood, Terra Lasho, Glenn Noronha, Christy Finke, Chi Ching Mak, Ruben Mesa, Hong Zhu, Richard Soll, Ayalew Tefferi
Journal: Blood (2006)
Empirical Versus Pre-Emptive Antifungal Therapy in High-Risk Febrile Neutropenic Patients: A Prospective Randomized Study.
Authors: Catherine Cordonnier, Cecile Pautas, Sebastien Maury, Anne Vekhoff, Hassan Farhat, Felipe Suarez, Maria Basile, Francoise Isnard, Lionel Ades, Frederique Kuhnoski, Oumady Reman, Sami Chehata, Thierry De Revel, Stephane Lepretre, Emmanuel Raffoux, Stephane Bretagne, Michael Schwarzinger
Journal: Blood (2006)
Biomimetic Synthetic High Density Lipoprotein Nanostructures Target the SR-B1 Receptor and Differentially Manipulate Cellular Cholesterol Flux in Lymphoma Cells: A Novel Treatment Paradigm
Authors: Shuo Yang, Marina G Damiano, Heng Zhang, Sushant Tripathy, Andrey Ugolkov, Amareshwar T.K. Singh, Andrea Luthi, Sandeep S. Dave, Shad Thaxton, Leo I. Gordon
Journal: Blood (2012)
Maximising the potential of AKT inhibitors as anti-cancer treatments
Authors: Jessica S. Brown, Udai Banerji
Journal: Pharmacology & Therapeutics (2016)

PI3K/AKT signalling is commonly disrupted in human cancers, with AKT being a central component of the pathway, influencing multiple processes that are directly involved in tumourigenesis. Targeting AKT is therefore a highly attractive anti-cancer strategy with multiple AKT inhibitors now in various stages of clinical development. In this review, we summarise the role and regulation of AKT signalling in normal cellular physiology. We highlight the mechanisms by which AKT signalling can be hyperactivated in cancers and discuss the past, present and future clinical strategies for AKT inhibition in oncology.
Procoagulant Actions of Circulating Microparticles in Sickle Cell Anemia and Sepsis Associated Coagulopathy and Their Modulation By a Triblock Polymer MST 188
Authors: Jawed Fareed, Debra Hoppensteadt, Martin Emanuele, Daneyal Syed, Abro Schuharazad, Walter Jeske, Rakesh Wahi
Journal: Blood (2014)
GATA-1 Mutation R216Q: Increased Bone Marrow Angiogenesis and Reticulin Fibrosis in X-Linked Thrombocytopenia with Thalassemia Trait (XLTT).
Authors: Eva Zetterberg, Lovisa Olson, Victoria Hahn-Strömberg, Jan Palmblad, Ian Jones, Maria Åström
Journal: Blood (2009)
Active Immunotherapy with FavId® (Id/KLH) Following Rituximab Induction: Long-Term Follow-Up of Response Rate Improvement (RRI) and Disease Progression in Follicular Lymphoma Patients (pts).
Authors: Omer N. Koc, Charles Redfern, Peter H. Wiernik, Fred Rosenfelt, Jane N. Winter, Troy H. Guthrie, Lawrence Kaplan, Peter Holman, John Densmore, John Hainsworth, Thomas S. Lin, Rene A. Castillo, Nalini Janakiraman, Teresa Melink, Song Liu, John Bender
Journal: Blood (2006)
The CXCR4-Antagonist AMD3100 Augments the Number of Mobilized Peripheral Blood Progenitor Cells (PBPC) When Added to a G-CSF Standard Mobilization Regime and AMD3100-Mobilized PBPC Result in Rapid Hematopoietic Reconstitution after Autologous Transplantation.
Authors: Stefan Fruehauf, Timon Seeger, Julian Topaly, Doris Herrmann, Falk Dillmann, Per Humpert, Gary Calandra, Stephanie Laufs, Hartmut Goldschmidt, Anthony D. Ho
Journal: Blood (2005)