tbuckley/cabot-search huggingface dataset:
External validation and comparison of three prediction tools for risk of osteoporotic fractures using data from population based electronic health records: retrospective cohort study
Authors: Noa Dagan, Chandra Cohen-Stavi, Maya Leventer-Roberts, Ran D Balicer
Journal: BMJ (2017)

<b>Objective</b>&nbsp;To directly compare the performance and externally validate the three most studied prediction tools for osteoporotic fractures—QFracture, FRAX, and Garvan—using data from electronic health records. <b>Design</b>&nbsp;Retrospective cohort study. <b>Setting</b>&nbsp;Payer provider healthcare organisation in Israel. <b>Participants</b>&nbsp;1 054 815 members aged 50 to 90 years for comparison between tools and cohorts of different age ranges, corresponding to those in each tools’ development study, for tool specific external validation. <b>Main outcome measure</b>&nbsp;First diagnosis of a major osteoporotic fracture (for QFracture and FRAX tools) and hip fractures (for all three tools) recorded in electronic health records from 2010 to 2014. Observed fracture rates were compared to probabilities predicted retrospectively as of 2010. <b>Results</b>&nbsp;The observed five year hip fracture rate was 2.7% and the rate for major osteoporotic fractures was 7.7%. The areas under the receiver operating curve (AUC) for hip fracture prediction were 82.7% for QFracture, 81.5% for FRAX, and 77.8% for Garvan. For major osteoporotic fractures, AUCs were 71.2% for QFracture and 71.4% for FRAX. All the tools underestimated the fracture risk, but the average observed to predicted ratios and the calibration slopes of FRAX were closest to 1. Tool specific validation analyses yielded hip fracture prediction AUCs of 88.0% for QFracture (among those aged 30-100 years), 81.5% for FRAX (50-90 years), and 71.2% for Garvan (60-95 years). <b>Conclusions</b>&nbsp;Both QFracture and FRAX had high discriminatory power for hip fracture prediction, with QFracture performing slightly better. This performance gap was more pronounced in previous studies, likely because of broader age inclusion criteria for QFracture validations. The simpler FRAX performed almost as well as QFracture for hip fracture prediction, and may have advantages if some of the input data required for QFracture are not available. However, both tools require calibration before implementation.
A Phase 1b Study of 30-Minute Infusion Carfilzomib 20/45 and 20/56 Mg/m2 Plus 40 Mg Weekly Dexamethasone in Patients with Relapsed and/or Refractory (R/R) Multiple Myeloma
Authors: Ashraf Z. Badros, Kyriakos P Papadopoulos, Naseem Zojwalla, Ju RueyJiuan Lee, David S. Siegel
Journal: Blood (2012)
High Pro-Apoptotic Effects of 17-Allylamino-17-Demethoxy Geldanamycin (17-AAG) In Philadelphia Acute Lymphoblastic Leukemia (Ph+ ALL)
Authors: Emmanuelle Tavernier-Tardy, Pascale Flandrin, Francoise Solly, Jérôme Cornillon, Karine Augeul-Meunier, Jean-Louis Stephan, Denis Guyotat, Lydia Campos
Journal: Blood (2010)
Profiling of Multiple Gene Mutations In Myelodysplastic Syndromes Using High-Throughput Resequenceing Combined with Barcode Labeling
Authors: Yasunobu Nagata, Masashi Sanada, Kenichi Yoshida, Takeo Nakaya, Aiko Matsubara, Naoshi Obara, Ken Ishiyama, Shuichi Miyawaki, Jyunko Takita, Shigeru Chiba, Lee-Yung Shih, H.Phillip Koeffler, Seishi Ogawa
Journal: Blood (2010)
Acquired central hypoventilation following<i>Listeria monocytogenes</i>rhombencephalitis
Authors: Sandrine H Launois, Natalia Siyanko, Marie Joyeux-Faure, Renaud Tamisier, Jean-Louis Pepin
Journal: Thorax (2017)

Acquired central hypoventilation syndrome (CHS) is a rare cause of respiratory failure. We report a case of acquired CHS, diagnosed several years after Listeria monocytogenes (LM) rhombencephalitis.

In 1993, a 46-year-old woman presented to our Sleep Clinic with poor sleep, nocturnal choking episodes and daytime fatigue. She denied habitual snoring and hypersomnolence. Her medical history was unremarkable except for severe LM rhombencephalitis in 1977. At that time, the patient had been hospitalised in the intensive care unit following the acute onset of fever, influenza-like symptoms, meningism, facial palsy and nystagmus. Blood and cerebral spinal fluid samples were positive for LM on direct microscopic exam as well cultures. Treatment was initiated but the patient acutely deteriorated, exhibiting tetra paresis, bilateral pontomedullary syndrome, impairment of vigilance, and experienced a cardiac arrest. She was successfully resuscitated and remained in the intensive care unit with gradual and slow recovery. After a lengthy hospital stay, she was eventually discharged from the hospital and resumed a normal life, with few neurological sequelae such as dysphagia and mild facial palsy. To investigate the patient's sleep complaints, an in-laboratory full polysomnography was obtained and revealed central sleep apnoeas and hypopnoeas (Apnoea–Hypopnoea index (AHI)=50 events/hour) with severe nocturnal hypoxaemia. Morning arterial blood gases revealed alveolar hypoventilation with partial pressure of arterial CO2 or PaCO2 of 6.7 kPa and PaO2 of 9.5 kPa. The patient refused further investigations and therapy but agreed to return to clinic for a follow-up in 1 year. In 1994, polysomnography and arterial blood gases showed persistent sleep apnoea and daytime hypoventilation but the patient continued to decline treatment. The patient was then lost to follow-up until 2011, when she presented for the third time to the Sleep Clinic complaining of daytime fatigue, unrefreshing sleep, choking episode during sleep and …
Donor Response to G-CSF Is the Best Predictive Factor of Extensive Chronic Graft-Versus-Host Disease after Non Myeloablative Allogeneic Peripheral Blood Stem Cell Transplantation.
Authors: N. Dhedin, T. Prebet, D. Rea, M.L. Tanguy, M. Kuentz, N. Piard, F. Norol, J.P. Jouet, Ja Rubeil, R. Tabrizi, B. Rio, B. Lioure, P. Tiberghien, J.H. Bourrhis, A. Sirvent, P. Bordigoni, D. Blaise, Mauricette Michallet, J.P. Vernant
Journal: Blood (2007)
Safety and efficacy of single bolus dose of recombinant factor VIIA in patients undergoing orthotopic liver transplantation : A randomized mulit-center study.
Authors: RM Planinsic, G Testa, S Emre, L Grande, A Candela, J van der Meer, RJ Porte, RM Ghobrial, H Isoniemi, P Schelde, E Erhardsen, G Klintmalm
Journal: Hepatology (2002)
Why have hospitalization rates for bullous pemphigoid soared?
Authors: D.F. Murrell
Journal: British Journal of Dermatology (2017)

Journal Article Why have hospitalization rates for bullous pemphigoid soared? Get access D.F. Murrell D.F. Murrell Department of Dermatology St George Hospital University of New South Wales, Faculty of Medicine Gray St, Kogarah Sydney NSW 2217 Australia Search for other works by this author on: Oxford Academic Google Scholar British Journal of Dermatology, Volume 176, Issue 1, 1 January 2017, Pages 5–6, https://doi.org/10.1111/bjd.15190 Published: 01 January 2017
The Bacterial T6SS Effector EvpP Prevents NLRP3 Inflammasome Activation by Inhibiting the Ca2+-Dependent MAPK-Jnk Pathway
Authors: Hao Chen, Dahai Yang, Fajun Han, Jinchao Tan, Lingzhi Zhang, Jingfan Xiao, Yuanxing Zhang, Qin Liu
Journal: Cell Host & Microbe (2017)
Establishment of An Angioimmunoblastic T-Cell Lymphoma (AITL) Model in the NOG Mouse
Authors: Asahi Ito, Takashi Ishida, Fumihiko Sato, Fumiko Mori, Ayako Masaki, Masaki Ri, Shigeru Kusumoto, Hirokazu Komatsu, Hiroshi Inagaki, Shinsuke Iida, Ryuzo Ueda
Journal: Blood (2011)
A quantitative reverse transcriptase polymerase chain reaction assay to identify metastatic carcinoma tissue of origin
Authors: D. Talantov, J. Baden, T. Jatkoe, J. Yu, D. Atkins, A. Mazumder, Y. Wang
Journal: Journal of Clinical Oncology (2006)

20024 Background: Carcinoma of unknown primary (CUP) wherein metastatic disease presents without an identifiable primary tumor site represents approximately 3–5% of all cancers. Identifying the origin of the primary tumor in patients with CUP can enable rational choice of therapeutic regimens. We developed an optimized set of ten gene markers for a quantitative reverse transcriptase polymerase chain reaction (qRTPCR) assay, and demonstrated high accuracy in predicting the tissue of origin when used on with formalin-fixed, paraffin-embedded (FFPE) metastatic carcinoma samples. Methods: Twenty-three putative tissue-specific markers for lung, colon, pancreas, breast, prostate and ovarian carcinomas were nominated by querying a gene expression profile database and by performing a literature search. Ten of these marker candidates were then selected based on validation by qRTPCR on 205 FFPE metastatic carcinomas of known tissue origin. Next, we optimized the RNA isolation and qRTPCR methods for these ten markers, and tested the qRTPCR assay on two sets of FFPE metastatic tumors. Results: We applied the 10-gene qRTPCR assay to a set of 260 metastatic tumors of known origin, generating an overall accuracy of 78%. Furthermore we tested an independent set of 48 metastatic samples, including thirty-seven samples where either the tissue of origin was known or which initially presented as CUP but were subsequently resolved. In these 48 samples, our assay demonstrated an accuracy of 76%. Conclusions: Our results suggested that optimized ten-gene markers qRTPCR assay reliably predicts tissue of origin of metastatic carcinomas in FFPE tissues. Such assay can significantly improve the rate of tissue of origin identification for carcinoma of unknown primary. [Table: see text]
Long-Term Survival and Late Relapse in 5-Year Survivors of Allogeneic Hematopoietic-Cell Transplantation (HCT) for Chronic Myeloid Leukemia (CML) in First Chronic Phase.
Authors: John M. Goldman, Navneet S. Majhail, John P. Klein, Zhiwei Wang, Kathleen A. Sobocinski, Mukta Arora, Mary M. Horowitz, J. Douglas Rizzo
Journal: Blood (2009)
Health-care-associated infections in neonates, children, and adolescents: an analysis of paediatric data from the European Centre for Disease Prevention and Control point-prevalence survey
Authors: Walter Zingg, Susan Hopkins, Angèle Gayet-Ageron, Alison Holmes, Mike Sharland, Carl Suetens, Maria Almeida, Jolanta Asembergiene, Michael A. Borg, Ana Budimir, Shona Cairns, Robert Cunney, Aleksander Deptula, Pilar Gallego Berciano, Olafur Gudlaugsson, Avgi Hadjiloucas, Naïma Hammami, Wendy Harrison, Elisabeth Heisbourg, Jana Kolman, Flora Kontopidou, Brian Kristensen, Outi Lyytikäinen, Pille Märtin, Gerry McIlvenny, Maria Luisa Moro, Brar Piening, Elisabeth Presterl, Roxana Serban, Emma Smid, Nina K. Sorknes, Maria Stefkovicova, Inese Sviestina, Rita Szabo, Hana Tkadlecova, Rossitza Vatcheva-Dobrevska, Delphine VerjatTrannoy
Journal: The Lancet Infectious Diseases (2017)
APC acts as a transcription factor and down-regulates the β-catenin gene, CTNNB1, and tumor suppressor gene SIAH1
Authors: Qingjie Li, Xiaoying Zhong, Melinda C. Myzak, Wan-Mohaiza Dashwood, Roderick H. Dashwood
Journal: Cancer Research (2004)

5076  Truncation of the tumor suppressor protein adenomatous polyposis coli (APC) constitutively activates the Wnt/β-catenin signalling pathway. Apc has a role in development: mouse embryos with truncated Apc do not complete gastrulation, and Apc mutation in zebrafish results in heart malformations. Recently, APC was found to shuttle in and out of the nucleus; however, the functional significance of this trafficking remains unclear. We examined the hypothesis that nuclear APC acts as a direct regulator of gene expression. Using a CTNNB1 promoter-luciferase reporter construct we showed that APC strongly inhibited the CTNNB1 promoter activity in HT29 cells. We also found that in HT29 cells transfected with APC there was a marked decrease in β-catenin mRNA levels. One possible explanation for this finding was that APC interacted directly with the promoter region of CTNNB1 and this was confirmed in mobility-shift and chromatin immunoprecipitation (ChIP) assays. To assess the role of other transcription factors, a 2.4 Kb promoter fragment of CTNNB1 was used to ‘pull down’ proteins from nuclear extracts of HT29 cells treated with APC, and the proteins were identified using TranSignal Protein/DNA arrays. Several transcription factors were identified through their interaction with the CTNNB1 promoter, and the profiles were qualitatively similar in the presence and absence of APC. However, APC increased in the expression of several bound transcription factors, most notably AP2, which was augmented more than four-fold. Reporter assays showed that over-expression of AP2 markedly decreased CTNNB1 promoter activity, but no increase in AP2 mRNA or protein was observed, suggesting that APC recruits AP2 to the CTNNB1 promoter. ChIP assays also showed that APC binds to the promoter of SIAH1, a p53-inducible tumor suppres sor involved in β-catenin degradation. Surprisingly, truncated APC was shown to bind the SIAH1 promoter and down-regulate SIAH1 mRNA and protein. Taken together, these results suggest that APC can act as a transcription factor and down-regulate CTNNB1 transcription by recruiting AP2 to CTNNB1 promoter. Truncated APC contributes to β-catenin accumulation by down-regulating SIAH1. These findings define an important new nuclear role for APC, namely as a transcriptional regulator of genes involved in differentiation, development, and cancer. Supported by NIH grants CA6552, CA80176, CA90890.
Low Dose-Radiation of Bone Marrow Stromal Cells Supports Pre-Leukemic Cell Survival Via Adhesion and Inflammatory Signaling Stimulation
Authors: Yoko Tabe, Linhua Jin, Yasuhito Hatanaka, Saiko Kazuno, Tsutomu Fujimura, Hiromichi Matsushita, Takashi Ueno, Keisuke Sasai, Takashi Miida
Journal: Blood (2012)
Prediction of Response to Imatinib by Prospective Quantitation of BCR-ABL Transcript in Late Chronic Phase Chronic Myeloid Leukemia PatientsBy GIMEMA Working Party on CML.
Authors: Giovanni Martinelli, Gianantonio Rosti, Fabrizio Pane, Marilina Amabile, Simona Bassi, Barbara Izzo, Angela Poerio, Barbara Giannini, Daniela Cilloni, Simona Soverini, Carolina Terragna, Emanuela Ottaviani, Tiziana Grafone, Antonio De Vivo, Nicoletta Testoni, Elena Trabacchi, Daniele Alberti, Francesco Salvatore, Giuseppe Saglio, Michele Baccarani
Journal: Blood (2004)
Crystal Structure of the Interaction of Human Urokinase Plasminogen Activitor with Its Receptor.
Authors: Qing Huai, Andrew P. Mazar, Graham Parry, Alice Kuo, Douglas B. Cines, Yongdong Li, Cai Yuan, Chuanbing Bian, Liqing Chen, Bruce Furie, Barbara C. Furie, Mingdong Huang
Journal: Blood (2005)
In the Absence of Preformed Antibody, Sensitized T Cells Mediate an Increased Barrier to Engraftment.
Authors: Hong Xu, Paula M. Chilton, Yiming Huang, Carrie L. Schanie, Michael K. Tanner, Mariano Dy-Liacco, Lala-Rukh Hussain, Suzanne T. Ildstad
Journal: Blood (2005)
Gastrointestinal Involvement in Mantle Cell Lymphoma (MCL). A Prospective Clinical, Endoscopical, Pathological and Molecular Study.
Authors: Antonio Salar, Nuria Juanpere, Eva Gonzalez-Barca, Beatriz Bellosillo, Blanca Espinet, Agustin Seoane, Agustin Panadés, Vicente Romagosa, Carmen Pedro, Miguel Nieto, Eugenia Abella, Eva Domingo-Domenech, Francesc Sole, Alberto Fernandez-Sevilla, Carlos Besses, Sergio Serrano
Journal: Blood (2005)
Intermediate Thrombin Activation Is Associated with Reduced Cardiovascular Death Independently of Markers of Lipid Metabolism and Inflammation
Authors: Berend H. Isermann, Jochen Schneider, Marcus E Kleber, Hongjie Wang, Bernhard O Boehm, Tanja B Grammer, Florian Prüller, Peter P Nawroth, Winfried März
Journal: Blood (2011)
PI3 Kinase, Phospholipase C (PLC)-γ, and RasGRPs Act Cooperatively to Activate the Ras-Extracellular-Related Kinase (ERK) Pathway in Response to Cytokines in Normal and Kras Mutant Myeloid Cells.
Authors: Ernesto Diaz-Flores, Hana Leah Goldschmidt, Philippe Depeille, Victor Ng, Jeroen Roose, Kevan Shokat, Benjamin S. Braun, Kevin Shannon
Journal: Blood (2009)
Sphingosine-1-phosphate signalling—a key player in the pathogenesis of Angiotensin II-induced hypertension
Authors: Anja Meissner, Francesc Miro, Francesc Jiménez-Altayó, Andrés Jurado, Elisabet Vila, Anna M. Planas
Journal: Cardiovascular Research (2016)

Hypertension is a complex condition involving functional and structural alterations of the microvasculature and an activation of the immune system. T-lymphocytes play a crucial role during the development of hypertension in experimental models, yet the underlying mechanisms remain elusive. Lymphocyte egress from lymph nodes is controlled by sphingosine-1-phosphate (S1P), a natural lipid mediator regulating immune cell and vascular function in health and disease. We therefore investigated the involvement of S1P signalling in the pathogenesis of hypertension. Angiotensin-II (AngII) treatment resulted in high blood pressure (BP) associated to increased plasma S1P and circulating T-cell counts. T-cell egress from lymph nodes was found to be a critical initial step for the onset of hypertension as fingolimod, a S1P-receptor agonist sequestering lymphocytes in the lymph nodes and inducing lymphopenia, blunted BP responses to AngII. Furthermore, activity of S1P-generating enzyme type 2 (SphK2) in haematopoietic cells critically contributed to AngII-induced lymphocyte mobilization from the lymph nodes as SphK2-/- mice and mice where SphK2 was ablated only in the haematopoietic system presented an accumulation of T-cells in mesenteric lymph nodes and a blunted BP response. In addition, deregulation of vascular SphK2 expression associated to a thrombo-inflammatory phenotype of the microvasculature, and to functional alterations of small resistance arteries. The presented results point to a critical involvement of S1P and its signalling axis in the pathogenesis of hypertension. Specifically, SphK2 evolves as key player in immune cell trafficking and vascular dysfunction contributing to the development of overt hypertension.
Burdern of Long-Term Morbidity after Hematopoietic Cell Transplantation: A Report from the Bone Marrow Transplant Survivor Study (BMTSS).
Authors: Can-Lan Sun, Liton Francisco, Toana Kawashima, Leslie L. Robison, K.S. Baker, Daniel Weisdorf, Stephen J. Forman, Smita Bhatia
Journal: Blood (2007)
Winter pressure leaves surgeons short of training opportunities
Authors: Abi Rimmer
Journal: BMJ (2017)

Surgeons are missing vital training opportunities because of the current winter crisis in the NHS, trainees have said.

Adam Williams, president of the Association of Surgeons in Training, told BMJ Careers that pressures on the NHS meant that opportunities to train were limited and trainees were often called out of operating theatres to assist on the wards.

“Ultimately, the emphasis has shifted from combining training with high quality service delivery, to maintaining basic service delivery at all costs. Patients are suffering, and …
Poor Outcomes in HIV Negative and HIV Positive Patients with Plasmablastic Lymphoma (PBL) in a Population Based Patient Study
Authors: Niamh Appleby, Ruth Cullen, Dan Painter, Alexandra Smith, Eve Roman, Sharon Barrans, Sheila J.M. O'Connor, Catherine H Burton, Andrew S Jack, Roger G Owen, Reuben M Tooze
Journal: Blood (2015)
Abstract #4295: Radiation induced migration of human head and neck squamous cell carcinoma (HNSCC) cells is inhibited by blocking the EGF-receptor
Authors: Anja Pickhard, Andreas Knopf, Claudius Fauser, Johanna Margraf, Anne-Laure Boulesteix, Henning Bier, Michael Molls, Jürgen Schlegel, Rudolf Reiter
Journal: Cancer Research (2009)

Purpose:  It is well documented that radiation induces the migration of tumor cells. This can facilitate further spread of tumor cells locally and systemically. The epidermal growth factor receptor (EGFR) is often overexpressed in HNSCC. The aim of this study was to evaluate whether blocking the EGFR and its downstream pathway reduces tumor cell migration in vitro.  Experimental design:  Migration was assessed via a wound healing assay. In addition, tumor cell proliferation was evaluated with a MTT colorimeritric assay using 3 HNSCC cell lines (BHY, CAL-27, HN). The cells were treated with increasing doses of irradiation (2Gy, 5Gy, 8Gy) in the presence or absence of EGFR, EGFR-antagonist (AG1478) or inhibitors of the downstream pathways (LY294002, rapamycin, PD98059). Biochemical activation of EGFR, Akt and MAPK was examined by Western blot analysis.  Results:  Increasing doses of irradiation induced a dose dependent enhancement of the count of migrating cells (p Conclusion:  Our results demonstrate that the EGFR is involved in radiation induced migration of HNSCC cells. Migration can be blocked by inhibition of the EGFR or its downstream pathways.   Citation Information:  In: Proc Am Assoc Cancer Res; 2009 Apr 18-22; Denver, CO. Philadelphia (PA): AACR; 2009. Abstract nr 4295.
Cancer Type and Risk of Newly Diagnosed Depression Among Elderly Medicare Beneficiaries With Incident Breast, Colorectal, and Prostate Cancers
Authors: Monira Alwhaibi, Usha Sambamoorthi, Suresh Madhavan, Thomas Bias, Kimberly Kelly, James Walkup
Journal: Journal of the National Comprehensive Cancer Network (2016)

Background: Elderly individuals (age >65 years) with cancer are at high risk for newly diagnosed depression after a cancer diagnosis. It is not known whether the risk of newly diagnosed depression varies by cancer type. Purpose: To examine the variations in the risk of newly diagnosed depression by cancer type among elderly individuals with cancer. Methods: This study used a retrospective cohort study design and data from the linked SEER-Medicare files. Elderly individuals (age >65 years) with incident breast, colorectal (CRC), and prostate cancers diagnosed between 2007 and 2011 (N=53,821) were followed for 12 months after cancer diagnosis. Depression diagnosis was identified during the 12-month follow-up period after cancer diagnosis using the ICD-9-Clinical Modification. Complementary log–log regression was used to examine the association between cancer type and risk of newly diagnosed depression after adjusting for other risk factors for depression. Results: We found a significantly higher percentage of newly diagnosed depression among women with CRC compared with those with breast cancer (5.8% vs 3.9%), and among men with CRC compared with those with prostate cancer (3.4% vs 1.6%). In the adjusted analysis, women with CRC had a 28.0% higher risk of newly diagnosed depression compared with women with breast cancer (adjusted risk ratio [ARR], 1.28; 95% CI, 1.12–1.46) and men with CRC had a 104.0% higher risk of newly diagnosed depression compared with those with prostate cancer (ARR, 2.04; 95% CI, 1.65–2.51). Conclusions: Our findings identified cancer types associated with a high risk of newly diagnosed depression after cancer diagnosis, who might benefit from routine depression screening to help in its early detection and treatment.
Direct in vivo evidence for increased proliferation of CLL cells in lymph nodes compared to bone marrow and peripheral blood
Authors: T M Herndon, S-S Chen, N S Saba, J Valdez, C Emson, M Gatmaitan, X Tian, T E Hughes, C Sun, D C Arthur, M Stetler-Stevenson, C M Yuan, C U Niemann, G E Marti, G Aue, S Soto, M Z H Farooqui, S E M Herman, N Chiorazzi, A Wiestner
Journal: Leukemia (2017)
Constitutive Expression of MEF2C, LYL1, or LMO2 in CD34+ HSCs Blocks the in Vitro Differentiation before the T-Cell Commitment Point and Could be Oncogenic in Early T-Cell Progenitor ALL
Authors: Kirsten Canté-Barrett, Rui D Mendes, Wilco K Smits, Rob Pieters, Jules PP Meijerink
Journal: Blood (2015)
Early Management of Heparin-Induced Thrombocytopenia Using a Simple Scoring System
Authors: Harry L. Messmore, Nancy Fabbrini, Mary L. Bird, Abdul M. Choudhury, Miguel Cerejo, M. Margaret Prechel, Walter Jeske, Arshea Siddiqui, Indermohan Thethi, William H. Wehrmacher, Jeanine M. Walenga
Journal: Blood (2011)