tbuckley/cabot-search huggingface dataset:
EUS-guided sutured gastropexy for transgastric ERCP (ESTER) in patients with Roux-en-Y gastric bypass: a novel, single-session, minimally invasive approach
Authors: Rajeev Attam, Daniel Leslie, Mustafa Arain, Martin Freeman, Sayeed Ikramuddin
Journal: Endoscopy (2015)

<b>Background and study aims:</b> Endoscopic retrograde cholangiopancreatography (ERCP) in patients with Roux-en-Y gastric bypass (RYGB) is a challenging procedure. Available techniques are hampered by limited success, need for a separate delayed session for ERCP, potential complications, and/or invasiveness. This paper reports on a novel technique that involves endoscopic ultrasound (EUS)-guided access into the remnant stomach and sutured gastropexy for transgastric ERCP in a single combined session. <b>Patients and methods:</b> A total of 10 patients with RYGB underwent transgastric ERCP using the novel technique. EUS was used to puncture the excluded stomach through the gastric pouch or jejunum, the stomach was insufflated, and a direct percutaneous gastrostomy puncture followed by sutured gastropexy was performed. ERCP was performed by passing a duodenoscope through the gastrostomy. <b>Results: </b>Technical success of EUS-assisted sutured gastropexy was achieved in nine patients (90 %), with immediate, same-session ERCP in all nine. There were no adverse events. <b>Conclusions: </b>EUS-assisted, fluoroscopically guided sutured gastropexy is a safe and effective method to obtain access into the excluded gastric remnant for same-session transgastric ERCP in patients with Roux-en-Y gastric bypass anatomy.
Glycogen Synthase Kinase-3 (GSK-3)-Targeted Therapy and Imaging
Authors: Mukesh K. Pandey, Timothy R. DeGrado
Journal: Theranostics (2016)

Glycogen synthase kinase-3 (GSK-3) is associated with various key biological processes, including glucose regulation, apoptosis, protein synthesis, cell signaling, cellular transport, gene transcription, proliferation, and intracellular communication.Accordingly, GSK-3 has been implicated in a wide variety of diseases and specifically targeted for both therapeutic and imaging applications by a large number of academic laboratories and pharmaceutical companies.Here, we review the structure, function, expression levels, and ligand-binding properties of GSK-3 and its connection to various diseases.A selected list of highly potent GSK-3 inhibitors, with IC 50 <20 nM for adenosine triphosphate (ATP)-competitive inhibitors and IC 50 <5 μM for non-ATP-competitive inhibitors, were analyzed for structure activity relationships.Furthermore, ubiquitous expression of GSK-3 and its possible impact on therapy and imaging are also highlighted.Finally, a rational perspective and possible route to selective and effective GSK-3 inhibitors is discussed.
COST-EFFECTIVE AND SAFE MANAGEMENT OF LOW-RISK CHEST PAIN WITH AN OUTPATIENT PROTOCOL INVOLVING CONTRAST-ENHANCED STRESS ECHOCARDIOGRAPHY
Authors: David C. Yang, Rajiv S. Vasudevan, Golnar Rahbaran, Yvette Yeung, Riya Pulicharam, Steve Kawahara, Firas Dabbous, Swaminatha Gurudevan
Journal: Journal of the American College of Cardiology (2016)
SYMPTOMATIC CAROTID ARTERIES SHOW HIGHER THERMAL HETEROGENEITY IN PATIENTS WITH ISCHEMIC STROKE AND BILATERAL CAROTID ATHEROSCLEROSIS
Authors: Georgios Benetos, Konstantinos Toutouzas, Maria Drakopoulou, Christina Deligianni, Konstantinos Spengos, Andreas Synetos, Eleftherios Tsiamis, Elias Siores, Dimitris Tousoulis
Journal: Journal of the American College of Cardiology (2015)
Abstract 1867: DNA repair gene polymorphisms and tobacco smoking and the risk of colorectal adenoma
Authors: Ying Gao, Richard Hayes, Wen-Yi Huang, Neil Caporaso, Laurie Burdett, Meredith Yeager, Stephen Chanock, Sonja Berndt
Journal: Cancer Research (2010)

Abstract Genetic variants of DNA repair genes may contribute to their altered capacity to repair endogenous and exogenous DNA damage. Although rare mutations in mismatch repair genes are known to lead to hereditary non-polyposis colorectal cancer, to date studies investigating common single nucleotide polymorphisms (SNPs) in DNA repair genes and colorectal cancer or adenoma risk have only examined a small number of variants in a limited number of genes. We conducted a comprehensive analysis of DNA repair gene polymorphisms and colorectal adenoma risk using a nested case-control study in the Prostate, Lung, Colorectal, and Ovarian Cancer Screening Trial. Effect modification by smoking, which is a known risk factor for adenoma, was also explored. A total of 1,338 advanced, left-sided colorectal adenoma cases and 1,503 frequency-matched controls without evidence of a left-sided polyp on sigmoidoscopy were included in the current study. Using DNA extracted from blood specimens, 3,401 tag SNPs in 153 DNA repair genes were genotyped. Logistic regression was used to estimate the odds ratios (OR) and 95% confidence intervals (CI) for the associations between genetic variants and adenoma risk assuming a log-additive model for the genotype. Among Caucasians, 30 SNPs were associated with adenoma risk at p&amp;lt;0.01. The most significant finding was for a non-synonymous SNP in EXO1 (rs9350), which was predicted to be deleterious using the SIFT and PolyPhen databases. The C allele at EXO1 rs9350 was associated with a 1.30-fold increased risk (95% CI=1.11-1.51, P=0.001). The association appeared to be limited to smokers (OR=1.54, 95% CI=1.26-1.89) with the strongest risk for current smokers (OR=2.15, 95% CI=1.27-3.65), and an intermediate risk for former smokers (OR=1.45, 95% CI=1.14-1.82), compared to never smokers (OR=0.98, 95% CI=0.76-1.25) that exhibited no increased risk (Pinteraction=0.002). Among the top findings, a SNP (rs17503908) in ATM with an overall OR of 0.75 (95% CI=0.63-0.91) was also modified by smoking (Pinteraction=0.006); the T allele at ATM rs17503908 was protective among never smokers (OR=0.55, 95% CI=0.40-0.76) but not among smokers (OR=0.89, 95% CI=0.70-1.13). The study suggests that polymorphisms in EXO1 and ATM may be associated with risk of advanced colorectal adenoma with the effects modified by tobacco smoking status. Our findings warrant extended investigations of these associations for colorectal cancer risk. Citation Format: {Authors}. {Abstract title} [abstract]. In: Proceedings of the 101st Annual Meeting of the American Association for Cancer Research; 2010 Apr 17-21; Washington, DC. Philadelphia (PA): AACR; Cancer Res 2010;70(8 Suppl):Abstract nr 1867.
P984 EFFICACY OF SORAFENIB ACCORDING TO THE NUMBER OF PRIOR TACE PROCEDURES IN HEPATOCELLULAR CARCINOMA PATIENTS
Authors: R. Sacco, A. Romano, M. Bertini, S. Metrangolo, G. Parisi, M. Bertoni, G. Federici, A. Scaramuzzino, V. Mismas, B. Ginanni, I. Bargellini, L. Fornaro, G. Masi, G. Bresci
Journal: Journal of Hepatology (2014)
PMO-199 Comparative study of sample adequacy of 25G vs 22G needle in endoscopic ultrasound (EUS) guided fine needle aspirate (FNA) of solid lesions: Abstract PMO-199 Table 1
Authors: A U Murugananthan, T Hong, R Y Chen
Journal: Gut (2012)

<h3>Introduction</h3> Optimal needle size in achieving greatest diagnostic yield from EUS- guided FNA remains unclear. <h3>Aim</h3> We prospectively compared sample adequacy and safety of FNA of solid lesions between 25G and 22G (Cook<sup>TM</sup>) needle at two tertiary centres. <h3>Methods</h3> Prospective data from two sites was collected between November 2008 and November 2011. A single operator alternated on a case-by-case basis between a 25G and 22G needle. A cytopathologist was present to assess adequacy of sample. The operator could switch needle size if required. <h3>Results</h3> 152 patients undergoing 165 FNA were analysed (42M/30F, mean age 59). 76 patients had FNA with a 22 F needle and 76 with the 25F needle. Indications for EUS and FNA were pancreatic lesions 43%, lymph node enlargement 28%, biliary tract lesions 16%, submucosal lesion 8% and adrenal mass 1% and others 4%. Overall sample adequacy was 83.03% Adequacy per needle was 86.7% (22G) vs 79.2% (25G), p=0.22 Fischer9s Exact test. The number of passes used in successful FNA was higher with use of the 25G needle compared with the 22G needle. (2.42±0.11 SEM vs 1.962±0.15 SEM, p=0.015, t-test). In particular the use of a 25G needle had a higher number of passes in pancreatic lesions compared with the 22G needle (2.58±0.16 SEM vs 1.94±0.14, p=0.004, t-test). There was no difference in adequacy between the needle sizes for each type of lesion sampled (Abstract PMO-199 table 1). Two needle exchanges (25G to a 22G) occurred. One complication of local site bleeding occurred (22G) that settled during the test. <h3>Conclusion</h3> We show no difference in sample adequacy between the two needle sizes. Use of a 25G results is associated with a higher number of passes in pancreatic FNA. Both needle sizes appear safe. Operator choice and ease of passage of needle into anatomical location may also influence choice of needle. <h3>Competing interests</h3> None declared.
LONG-TERM MORTALITY IN A COHORT OF HYPERTENSIVE PATIENTS WITH CORONARY ARTERY DISEASE: RESULTS FROM AN EXTENDED FOLLOW UP OF THE INTERNATIONAL VERAPAMIL/ TRANDOLAPRIL STUDY (INVEST) PROJECT
Authors: Islam Elgendy, Anthony Bavry, Yan Gong, Eileen Handberg, Rhonda Cooper-DeHoff, Carl Pepine
Journal: Journal of the American College of Cardiology (2016)
Expression of adipocytokines at sites of bone remodelling in rheumatoid arthritis
Authors: G Krumbholz, A Lehr, M Rickert, S Rehart, G Schett, U Müller-Ladner, E Neumann
Journal: Annals of the Rheumatic Diseases (2012)

<h3>Background</h3> Rheumatoid arthritis (RA) is associated with increased production of adipocytokines, which are cytokine-like mediators. Increased levels of the adipokines adiponectin and resistin in synovial fluids of RA patients as well as their activating/inhibitory effect on cells of bone remodeling (eg, osteoblasts, osteoclasts) raise the question of immunomodulatory properties of these cytokines towards bone resorption. In this study, the expression of adiponectin and resistin in joint compartments and cells at the sites of bone remodelling in RA was analysed. <h3>Methods</h3> Bone tissue was obtained from RA patients during joint replacement surgery. Serial sections of decalcified and deparaffinised bone tissue were prepared. Immunohistochemistry was used to identify the localisation of adipokines at sites of bone remodelling in articular bone and for identification of cells expressing the adipokines adiponectin and resistin. Additionally, immunohistochemical staining was carried out by using antihuman ALP (alkaline phosphatase) antibodies for identifying osteoblasts and TRAP staining to identify osteoclasts. For histological overview, a Masson Trichrome staining was performed. <h3>Results</h3> Adiponectin and resistin were detectable in all joints of RA patients at sites of bone remodelling. An expression of adiponectin and resistin by osteoblasts and osteoclasts could be observed. Both adipokines were co-localised with osteoblasts at the border of newly formed, non-mineralised bone tissue. Interestingly, a correlation of the expression intensity of resistin with the expression of ALP in osteoblasts could be observed, indicating that active osteoblasts produce increased amounts of resistin. <h3>Conclusions</h3> The results indicate that resistin and adiponectin are expressed by osteoblasts and osteoclasts at sites of bone erosion. Therefore, these cell types are able to respond to resistin and adiponectin in bone tissue, which potentially further contribute to the differentiation and activation of these cell types. <h3>Acknowledgement</h3> Funded by the German Research Society (SPP1468, IMMUNOBONE, NE1174/6-1).
Alarmins S100A8 and S100A9 stimulate production of pro-inflammatory cytokines in M2 macrophages without changing their M2 membrane phenotype
Authors: R F Schelbergen, A B Blom, W de Munter, T Vogl, J Roth, W B van den Berg, P L van Lent
Journal: Annals of the Rheumatic Diseases (2012)

<h3>Background</h3> Synovial activation, which plays an important role in a subgroup of osteoarthritis (OA) patients, is regulated by M1/M2 macrophage subpopulations: pro-inflammatory M1-macrophages and regulatory M2-macrophages. Alarmins S100A8 and S100A9 are found in high concentrations in synovial fluid of rheumatoid arthritis (RA) and OA patients and are correlated to inflammation and cartilage destruction. In the current study, the authors investigated the production of S100A8 and/or S100A9 by M1- and M2-macrophages and whether S100A8 and/or S100A9 could stimulate M2-macrophages, giving them a more pro-inflammatory character. <h3>Methods</h3> Monocytes were isolated from blood of healthy volunteers by isolating peripheral blood mononuclear cells (PBMCs) using a Ficoll-gradient followed by isolating CD14+ monocytes using magnetic-activated cell sorting (MACS). CD14+ monocytes were differentiated into M1- or M2-macrophages by 6 day stimulation with either GM-CSF or M-CSF, respectively. mRNA levels of S100A8 and S100A9 in M1 and M2 macrophages were measured with RT-qPCR. Intracellular protein production was determined using FACS analysis and S100A8/A9 secretion was measured using an ELISA.M1 and M2 macrophages were stimulated with human recombinant S100A8 and S100A9. M1 and M2 markers (IL-1β, tumour necrosis factor α (TNFα), IL-6 and CD163, CD206 respectively) were measured using RT-qPCR, FACS and/or Luminex. <h3>Results</h3> S100A8 and S100A9 had a significantly higher mRNA expression (65- and 2.5-fold respectively, n=10) in human monocyte derived M1-macrophages compared to M2. Also, the levels of intracellular S100A8 and S100A9 were higher in M1 than in M2 macrophages (4.3-fold and 6.6-fold higher respectively, n=6). The secreted protein level of the S100A8/A9 heterodimer was elevated from 55.5 ng/ml in M2 to 102.8 ng/ml in M1 macrophages (n=10). Stimulation of human monocyte derived M2-macrophages for 24 h with S100A8 and S100A9 showed a marked upregulation of several pro-inflammatory markers: IL-1β expression was increased by S100A8 and S100A9 on mRNA level (33- and 16-fold, n=10). At the protein level IL-6 and TNFα were strongly upregulated by S100A8 71- and 146-fold and by S100A9 93- and 121-fold, respectively (n=6). Interestingly however, S100A8 or S100A9 stimulation did not alter the level of M2 membrane markers CD163 and CD206, both at mRNA as well as protein level. <h3>Conclusions</h3> Stimulation of M2 macrophages with S100A8 and S100A9, produced in high amounts by M1- macrophages, upregulates pro-inflammatory cytokines IL-1β, IL-6 and TNFα while M2 membrane markers CD163 and CD206 remain unchanged. S100A8 and S100A9 production during OA could therefore contribute to synovial activation by stimulating the M2 macrophage towards a more pro-inflammatory phenotype.
Leukemia Inhibitory Factor Has Multiple Personalities
Authors: C. Vanchieri
Journal: JNCI Journal of the National Cancer Institute (1994)

Journal Article Leukemia Inhibitory Factor Has Multiple Personalities Get access Cori Vanchieri Cori Vanchieri Search for other works by this author on: Oxford Academic PubMed Google Scholar JNCI: Journal of the National Cancer Institute, Volume 86, Issue 4, 16 February 1994, Page 262, https://doi.org/10.1093/jnci/86.4.262 Published: 16 February 1994
Dendritic Cells Coordinate the Development and Homeostasis of Organ-Specific Regulatory T Cells
Authors: Daniel S. Leventhal, Dana C. Gilmore, Julian M. Berger, Saki Nishi, Victoria Lee, Sven Malchow, Douglas E. Kline, Justin Kline, Donald J. Vander Griend, Haochu Huang, Nicholas D. Socci, Peter A. Savage
Journal: Immunity (2016)
TRATAMIENTO ENDOSCÓPICO DE LA ESTENOSIS DE LA ANASTOMOSIS BILIAR CON COLANGIO-PANCREATOGRAFÍA RETRÓGRADA ENDOSCÓPICA EN PACIENTES TRANSPLANTADOS HEPÁTICOS
Authors: D Royg, A Fernández-Simon, O Sendino, JM Bordas, G Fernández-Esparrach, M Pellisé, M López-Ceron, C Rodriguez D Miguel, A Ginés, J Llach, A Cárdenas
Journal: Endoscopy (2013)

Introducción: La incidencia de complicaciones biliares en trasplantados hepáticos oscila entre 10 y 30%. Las estenosis de la anastomosis biliar (EAB) son la complicación más frecuente y representan el 40% de todas las complicaciones biliares. La mayoría son tratadas con éxito por colangiopancreatografia endoscópica retrógrada CPRE con dilatación biliar y/o colocación de prótesis si existe anastomosis biliar de conducto biliar del donante a conducto biliar receptor. Las tasas de resolución reportadas con CPRE oscilan entre 65 – 100%. Objetivo: Analizar respuesta de curación al tratamiento endoscópico con prótesis y dilatación y dilatación sola en los pacientes trasplantados hepáticos con EAB. Material y Método: Estudio retrospectivo en pacientes trasplantados hepáticos con EAB desde Enero 2003 hasta Enero de 2013. Resultados: De 50 pacientes, 42 (84%) fueron sometidos a tratamiento con dilatación biliar más colocación de prótesis. De estos, 37 (88%) obtuvieron respuesta completa con recurrencia en 3 (8,2%) y todas resolvieron con re-tratamiento con tasa de curación del 100% en este grupo. De 8 pacientes tratados con solo dilatación biliar, 5 se curaron (62.5%) y no tuvieron recurrencia. En total, 8 (16%) pacientes no resolvieron con tratamiento endoscópico y fueron sometidos a cirugía. La tasa de curación global en los dos grupos fue del 84%. La tasa de complicaciones post CPRE fue del 5.2%. Conclusiones: El tratamiento de las EAB post-TH con dilatación biliar mas colocación de prótesis biliar plásticas es seguro y efectivo, con tasas de recurrencia baja y una alta tasa de curación.
ESCALA DE BLATCHFORD EN LA HEMORRAGIA DIGESTIVA ALTA EXPERENCIA EN EL DEPARTAMENTO DE URGENCIAS DEL COMPLEJO HOSPITALARIO TORRECARDENAS ALMERIA
Authors: G Patrón Román, M Rodríguez Manrique, A Gálvez Miras
Journal: Endoscopy (2011)

Introducción: La Hemorragia Digestiva Alta (HDA) es una causa frecuente de ingreso hospitalario, cuya incidencia se mantiene en el tiempo a pesar de los adelantos en la terapia farmacológica, presenta costos muy elevados y alta morbilidad-mortalidad. Las escalas de estratificación de riesgo, como herramientas de ayuda para la intervención clínica y endoscópica adecuado son recomendadas por múltiples estudios en la actualidad. En nuestra investigación seleccionados a todos los sujetos diagnosticados de HDA con alto riesgo en la escala de Blatchford en el servicio de urgencia y la relación con la intervención clínica y endoscópica.
A5.22 Membrane-Bound and Soluble Baff Expression by Human Rheumatoid Fibroblast-Like Synoviocytes in Response to TLR Stimulation
Authors: NW Kam, A Filer, C Buckley, M Bombardieri, C Pitzalis
Journal: Annals of the Rheumatic Diseases (2013)

<h3>Background and Objectives</h3> B cell activating factors of TNF family (BAFF) is associated with the survival and maturation of B cells. BAFF is widely expressed in the rheumatoid arthritis (RA) synovium which is characterised by the presence of synovial niches of autoreactive B cells and sustain in situ autoantibody production. Importantly, B cell niches remain functional in the RA-SCID model in the absence of recirculating cells, suggesting that autocrine mechanisms support ongoing B cell activation in the RA synovium. BAFF exerts its functional role both as a membrane bound protein and in soluble form. Here we investigated whether resident stromal cells in the RA synovium, synovial fibroblasts (RASF), are capable of producing either forms of BAFF and thus contribute to local B cell activation. <h3>Methods</h3> mRNA BAFF in RASF stimulated with TLR2, TLR3 and TLR4 ligands was assessed by quantitative Taqman PCR RA dermal fibroblasts (RADF) and osteoarthritis SF (OASF) were used as controls. The cytoplasmic, membrane bound and/or soluble forms of BAFF were investigated by 1) Western blot using total and membrane-enriched protein extracts, 2) flow cytometry, 3) ELISA and 4) immunocytochemistry. <h3>Results</h3> In vitro stimulation of TLR3, and to a significantly lesser extent TLR4, but not TLR2 on RASF led to strong induction of BAFF mRNA. In response to TLR3, soluble BAFF was time-dependently released in the supernatant of RASF (~600 pg/ml) and, to a lesser extent, OASF and RADF. RASF constitutively expressed both cytoplasmic and membrane bound BAFF as demonstrated by WB, FACS and immunocytochemistry which was upregulated upon TLR3 stimulation and was significantly increased as compared to RADF. <h3>Conclusions</h3> Here we provide conclusive evidence that SF in the RA synovium are a pivotal source of the B cell survival factor BAFF at both mRNA and protein level. In addition to their significant constitutive expression, RASF can further up-regulate cytoplasmic, membrane-bound and soluble BAFF in response to TLR3 stimulation. Overall, our data strongly support a fundamental role for RASF in sustaining functional B cell activation and antibody production in the inflamed RA synovium.
Investigation and Prevention of Fosaprepitant-Induced Vascular Pain
Authors: K. Imazu, K. Shinozaki, M. Kinoshita, Y. Mukaigawa, C. Nishikawa, Y. Nishi, M. Shirasuna, E. Kuwada, K. Nakata, M. Takaoka
Journal: Annals of Oncology (2013)
HIGH LEVEL OF BLOOD UREA NITROGEN IS A SIGNIFICANT PREDICTOR FOR IN-HOSPITAL MORTALITY IN PATIENT WITH ACUTE MYOCARDIAL INFRACTION
Authors: Yu Horiuchi, Masaharu Ishihara, Masashi Fujino, Hisao Ogawa, Koichi Nakao, Satoshi Yasuda, Teruo Noguchi, Yukio Ozaki, Kazuo Kimura, Satoru Suwa, Kazuteru Fujimoto, Yasuharu Nakama, Takashi Morita, Wataru Shimizu, Yoshihiko Saito, Atsushi Hirohata, Yasuhiro Morita, Teruo Inoue, Atsunori Okamura, Masaaki Uematsu, Kazuhito Hirata, Kunihiro Nishimura, Yoshihiro Miyamoto, Jiro Aoki, Kengo Tanabe
Journal: Journal of the American College of Cardiology (2016)
CS-28 * THE TRANSCRIPTION FACTOR POU3f2 UNIFORMLY EXPRESS IN HUMAN GLIOMAS AND PROMOTES TUMORIGENESIS AND OVERALL GROWTH RATE IN VIVO
Authors: M. A. Rahman, L. Leiss, M. S. Lellahi, C. H. Gjerde, H. S. Saed, E. Mutlu, P. O. Enger
Journal: Neuro-Oncology (2014)

The transcription factor POU3f2 is expressed during neurogenesis, and is essential for CNS development. Moreover, POU3f2 expression has been reported as a promoter of proliferation and invasion in malignant melanoma of the skin. Since both melanomas and CNS-malignancies arise in organs of neuroectodermal origin, we investigated whether human gliomas expressed POU3f2 and its potential role in glioma tumorigenesis. Immunohistochemistry results showed that POU3f2 was almost uniformly expressed in human gliomas, although at a varying degree. Microscopy revealed a predominantly nuclear staining pattern, but cytoplasmic immunopositivity for POU3f2 was also detected in some tumors. Moreover, western blot, qPCR and flow cytometry confirmed POU3f2 expression. Two independent observers estimated the nuclear staining index for all tumors, with 61% positive nuclei in GBM specimens, significantly higher than for grade II (37%, p = 0,0004) and grade III (36%, p = 0,0069) tumors. In acutely dissociated tumors, POU3f2 positive cells displayed an increased S- and G2/M-phase fraction. Moreover, overexpression of POU3f2 in the glioma cell lines increased its growth rate and colony formation, suggesting a role in regulating tumor cell proliferation, and hence possibly overall tumor aggressiveness. Cancer pathway reporter array showed that POU3f2 affects the NF-κB, MAPK/JNK and Myc/Max pathways. Overexpression of POU3f2 increase c-Myc expression and probably the POU3f2 drives cell proliferation elicited via activation of transcription of c-Myc target genes that are positive regulators of the cell cycle. On the other hand, POU3f2 regulates phosphorylation of histone H3 and might have an effect on epigenetic regulation through NF-kB signaling pathway. Ongoing studies aim at elucidating the mechanisms underlying the tumor-promoting effect of POU3f2, and will be presented at the meeting.
THE HASP VERSUS THE JASZCZAK PHANTOM FOR SPECT SYSTEM EVALUATION
Authors: D. M. Chiaramonte, P. F. Cutrera, M. W. Groch, S. M. Leonard, S. M. Spies, P. E. Mosebach
Journal: Clinical Nuclear Medicine (1996)

Chiaramonte, D. M.; Cutrera, P. F.; Groch, M. W.; Leonard, S. M.; Spies, S. M.; Mosebach, P. E. Author Information