Occupational Immunologic Lung Disease
Authors: Robert A. Goldstein, Roy Patterson
Journal: Annals of Internal Medicine (1982)

Symposium1 July 1982Occupational Immunologic Lung DiseaseROBERT A. GOLDSTEIN, M. D., PH.D., ROY PATTERSON, M.D.ROBERT A. GOLDSTEIN, M. D., PH.D.Search for more papers by this author, ROY PATTERSON, M.D.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-97-1-133_2 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptOccupational health hazards have been recognized for a long time. Lung disease caused by inhalation of dust was mentioned by Agricola in his treatise on mining in 1556 and described in stonecutters by Van Diemerbroeck in 1672 and by Ramazzini in 1713 (1). The subsequent elucidation of such disorders as silicosis, asbestosis, and anthracosis has been based on careful, arduous, and often retrospective, epidemiologic studies. In fact, because it has often been difficult to identify potentially causative agents ahead of time and because inexpensive diagnostic measures are not readily available for most of these disorders, few prospective studies have been...Symposium Papers1. SALVAGGIO J. Overview of occupational immunologic lung disease. Tulane University School of Medicine; New Orleans, Louisiana. Google Scholar2. BROOKS S. The evaluation of occupational airways disease in the laboratory and workplace. University of Cincinnati; Cincinnati, Ohio. Google Scholar3. KRAUSE R. A matter of opportunity. National Institute of Allergy and Infectious Diseases; Bethesda, Maryland. Google Scholar4. FINK J. Evaluation of the patient for occupational immunologic lung disease. Medical College of Wisconsin; Milwaukee, Wisconsin. Google Scholar5. WOLKONSKY P. The industrial physician and occupational lung disease. Standard Oil Company (Indiana); Chicago, Illinois. Google Scholar6. REED C. Detection and measurement of antigens. Mayo Clinic and Foundation; Rochester, Minnesota. Google Scholar7. SCHLEUTER D. Infiltrative lung disease: hypersensitivity pneumonitis. Medical College of Wisconsin; Milwaukee, Wisconsin. Google Scholar8. PATTERSONZEISSPRUZANSKY RCJ. Immunology and pathophysiology of trimellitic anhydride induced lung disease. Northwestern University Medical College; Chicago, Illinois. Google Scholar9. BERNSTEIN I. Toluene diisocyanate induced lung diseases. University of Cincinnati Medical Center; Cincinnati, Ohio. Google Scholar10. CHAN-YEUNG M. Immunologic and nonimmunologic mechanisms in western red cedar asthma. University of British Columbia; Vancouver, B.C., Canada. Google Scholar11. ZEISSWOLKONSKYPRUZANSKYPATTERSON CPJR. Clinical and immunologic evaluation of trimellitic anhydride workers in multiple industrial settings. Northwestern University Medical School; Chicago, Illinois. Google Scholar12. DE Shazo R. Current concepts about the pathogenesis of silicosis and asbestosis. Tulane University School of Medicine; New Orleans, Louisiana. Google Scholar13. McGiNNis J. Prevention of disease and promotion of health. Department of Health and Human Services; Washington, D.C. Google Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics 1 July 1982Volume 97, Issue 1Page: 133-135KeywordsAllergy and immunologyDustEpidemiologyInfectious diseasesInhalationLungsPrevention, policy, and public healthProspective studiesRetrospective studiesSilicosis Issue Published: 1 July 1982 PDF DownloadLoading ...