Atmospheric Contamination with Lead
Authors: BRYAN T. EMMERSON
Journal: Annals of Internal Medicine (1968)

Editorial Notes1 February 1968Atmospheric Contamination with LeadBRYAN T. EMMERSON, M.D., F.R.A.C.P.BRYAN T. EMMERSON, M.D., F.R.A.C.P.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-68-2-488 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptInterest in the deleterious effects of lead has shifted recently from the industrial field to that of potential hazards to the community at large. Lead is ubiquitous, and even primitive races have small quantities in their tissues. Persons living in modern communities in the United States ingest a large amount of lead, thought to amount to about 0.3 mg/day (1), due to the slight contamination of many foodstuffs. Only a small portion of ingested lead, probably about 10%, is absorbed from the alimentary tract, however, and the body can usually cope with this amount by excreting an equal amount in...References1. KEHOE RA: The metabolism of lead in man in health and disease. I. The normal metabolism of lead. J. Roy. Inst. Public Health 24: 81, 1961. MedlineGoogle Scholar2. HOFREUTERCATCOTTKEENANXINTARAS DHEJRGC: The public health significance of atmospheric lead. Arch. Environ. Health (Chicago) 3: 568, 1961. CrossrefMedlineGoogle Scholar3. U. S. PUBLIC HEALTH SERVICE: Survey of Lead in the Atmosphere of Three Urban Communitis. Publication No. 999-AP-12. U. S. Government Printing Office, Washington, D. C., 1965. Google Scholar4. GOLDSMITHHEXTER JRAC: Respiratory exposure to lead: epidemiological and experimental dose-response relationships. Science 158: 132, 1967. CrossrefMedlineGoogle Scholar5. PATTERSON CC: Contaminated and natural lead environment of man. Arch. Environ. Health (Chicago) 11: 344, 1965. CrossrefMedlineGoogle Scholar6. MACHLE W: Lead contamination. Arch. Environ. Health (Chicago) 12: 269, 1966. CrossrefGoogle Scholar7. SCHROEDERBALASSAVINTON HAJJWH: Chromium, cadmium, and lead in rats: effects on life span, tumors, and tissue levels. J. Nutr. 86: 51, 1965. CrossrefMedlineGoogle Scholar8. HASANHERNBERGMETSALAVIHKO JSPV: Enhanced potassium loss in blood cells from men exposed to lead. Arch. Environ. Health (Chicago) 14: 309, 1967. CrossrefMedlineGoogle Scholar9. HAEGER-ARONSEN B: Studies on urinary excretion of δ-amino-laevulinic acid and other haem precursors in lead workers and lead intoxicated rabbits. Scand. J. Clin. Lab. Invest. 12 (suppl. 47): 1960. MedlineGoogle Scholar10. SAITAMOREO GL: La determinazione dell'-acido delta aminolevulinico sierico ed urinario ai fini della diagnosi di pregressa intossicazione da piombo. Med. Lavoro 55: 357, 1964. Google Scholar11. HERNBERGVIHKOHASAN SVJ: Red cell membrane ATPases in workers exposed to inorganic lead. Arch. Environ. Health (Chicago) 14: 319, 1967. CrossrefMedlineGoogle Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: Duke University Medical Center, Durham, N. C. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics Cited ByChronic lead nephropathy 1 February 1968Volume 68, Issue 2Page: 488-490KeywordsRacial and ethnic issues Issue Published: 1 February 1968 PDF DownloadLoading ...