PANCREATIC ADENOMA (HYPERINSULINISM) ASSOCIATED WITH NEUROMUSCULAR DISORDERS
Authors: R W BARRIS
Journal: Annals of Internal Medicine (1953)

Case Reports1 January 1953PANCREATIC ADENOMA (HYPERINSULINISM) ASSOCIATED WITH NEUROMUSCULAR DISORDERSRALPH W. BARRIS, M.D., Ph.D.RALPH W. BARRIS, M.D., Ph.D.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-38-1-124 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptINTRODUCTIONThe neuromuscular disorders belong to a clinical group in which the cardinal signs and symptoms are related to a progressive wasting of muscles. As a group, these disorders are etiologically vague and pathogenically unspecific. In the broader classification of neuromuscular disorders, there are included those states characterized by some demonstrable alteration in the anterior horn cells, or in their peripheral processes, or by some alteration in structure in the muscle fibers, per se. The designation "progressive muscular atrophy," or the alternative designation "progressive spinal (or nuclear) muscular atrophy," is usually employed to designate that group of muscle wasting states...Bibliography1. HoussayBiasotti BAA: The hypophysis, carbohydrate metabolism, and diabetes, Endocrinology 15: 511, 1931. CrossrefGoogle Scholar2. LidzMillerPadgerStedem TJMPAF: Muscular atrophy and pseudologica fantastica associated with islet adenoma of the pancreas, Arch. Neurol. and Psychiat. 62: 304-313, 1949. CrossrefMedlineGoogle Scholar3. MoerschKernohan FPJW: Hypoglycemia, neurologic and neuropathologic studies, Arch. Neurol. and Psychiat. 39: 242-257, 1938. CrossrefGoogle Scholar4. ShyMcEachern GMD: Clinical features and response to cortisone of menopausal muscular dystrophy, J. Neurol., Neurosurg. and Psychiat. 14: 101, 1951. CrossrefGoogle Scholar5. Silfverskiöld BP: Polyneuritis hypoglycemia. Late peripheral paresis after hypoglycemic attacks in two insuloma patients, Acta med. Scandinav. 125: 502-504, 1946. CrossrefGoogle Scholar6. ThornEder GWHA: Studies on chronic thyrotoxic myopathy, Am. J. Med. 1: 583-601 (Dec.) 1946. CrossrefMedlineGoogle Scholar7. TomRichardson MIJC: Hypoglycemia from islet cell tumor of pancreas with amyotrophy and cerebrospinal nerve cell changes, J. Neuropath. and Exper. Neurol. 10: 57-66 (Jan.) 1951. CrossrefGoogle Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: LaJolla, California*Received for publication April 22, 1952. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics Cited byTheories of CausationEndocrine Function in Amyotrophic Lateral Sclerosis: A ReviewInsulin resistance in amyotrophic lateral sclerosisTumoren des PankreasPancreatic function in amyotrophic lateral sclerosisParalysis, Incoordination and Involuntary MovementsThe Zollinger-Ellison Syndrome. Its Place in the Pathophysiology of Gastric Acid Secretion and of its Hormonal RegulationDiscussion on Motor Neurone DiseaseDie Veränderungen des Zentralnervensystems beim Diabetes mellitusMultiple Sclerosis and Amyotrophic Lateral SclerosisVITAMIN E AND INTERMITTENT CLAUDICATIONCEREBRAL DISEASE DUE TO FUNCTIONING ISLET-CELL TUMOURS 1 January 1953Volume 38, Issue 1Page: 124-129KeywordsAdenomasAtrophyCellsHyperinsulinemiaMusclesPathogensSigns and symptoms ePublished: 1 December 2008 Issue Published: 1 January 1953 PDF downloadLoading ...