ZEB2
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Wakamatsu N; Yamada Y; Yamada K;  외. (2001). “Mutations in SIP1, encoding Smad interacting protein-1, cause a form of Hirschsprung disease”. 《Nat. Genet.》 27 (4): 369–70. doi:10.1038/86860. PMID 11279515.
Comijn J; Berx G; Vermassen P;  외. (2001). “The two-handed E box binding zinc finger protein SIP1 downregulates E-cadherin and induces invasion”. 《Mol. Cell》 7 (6): 1267–78. doi:10.1016/S1097-2765(01)00260-X. PMID 11430829.
Cacheux V; Dastot-Le Moal F; Kääriäinen H;  외. (2001). “Loss-of-function mutations in SIP1 Smad interacting protein 1 result in a syndromic Hirschsprung disease”. 《Hum. Mol. Genet.》 10 (14): 1503–10. doi:10.1093/hmg/10.14.1503. PMID 11448942.
Tylzanowski P; Verschueren K; Huylebroeck D; Luyten FP (2001). “Smad-interacting protein 1 is a repressor of liver/bone/kidney alkaline phosphatase transcription in bone morphogenetic protein-induced osteogenic differentiation of C2C12 cells”. 《J. Biol. Chem.》 276 (43): 40001–7. doi:10.1074/jbc.M104112200. PMID 11477103.
Yamada K; Yamada Y; Nomura N;  외. (2002). “Nonsense and Frameshift Mutations in ZFHX1B, Encoding Smad-Interacting Protein 1, Cause a Complex Developmental Disorder with a Great Variety of Clinical Features”. 《Am. J. Hum. Genet.》 69 (6): 1178–85. doi:10.1086/324343. PMC 1235530. PMID 11592033.
Amiel J; Espinosa-Parrilla Y; Steffann J;  외. (2002). “Large-Scale Deletions and SMADIP1 Truncating Mutations in Syndromic Hirschsprung Disease with Involvement of Midline Structures”. 《Am. J. Hum. Genet.》 69 (6): 1370–7. doi:10.1086/324342. PMC 1235547. PMID 11595972.
Zweier C; Albrecht B; Mitulla B;  외. (2002). “"Mowat-Wilson" syndrome with and without Hirschsprung disease is a distinct, recognizable multiple congenital anomalies-mental retardation syndrome caused by mutations in the zinc finger homeo box 1B gene”. 《Am. J. Med. Genet.》 108 (3): 177–81. doi:10.1002/ajmg.10226. PMID 11891681.
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Guaita S; Puig I; Franci C;  외. (2002). “Snail induction of epithelial to mesenchymal transition in tumor cells is accompanied by MUC1 repression and ZEB1 expression”. 《J. Biol. Chem.》 277 (42): 39209–16. doi:10.1074/jbc.M206400200. PMID 12161443.
Espinosa-Parrilla Y; Amiel J; Augé J;  외. (2003). “Expression of the SMADIP1 gene during early human development”. 《Mech. Dev.》 114 (1–2): 187–91. doi:10.1016/S0925-4773(02)00062-X. PMID 12175509.
Yoneda M; Fujita T; Yamada Y;  외. (2002). “Late infantile Hirschsprung disease-mental retardation syndrome with a 3-bp deletion in ZFHX1B”. 《Neurology》 59 (10): 1637–40. PMID 12451214.
Strausberg RL; Feingold EA; Grouse LH;  외. (2003). “Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences”. 《Proc. Natl. Acad. Sci. U.S.A.》 99 (26): 16899–903. doi:10.1073/pnas.242603899. PMC 139241. PMID 12477932.
Postigo AA (2003). “Opposing functions of ZEB proteins in the regulation of the TGFβ/BMP signaling pathway”. 《EMBO J.》 22 (10): 2443–52. doi:10.1093/emboj/cdg225. PMC 155983. PMID 12743038.
Postigo AA; Depp JL; Taylor JJ; Kroll KL (2003). “Regulation of Smad signaling through a differential recruitment of coactivators and corepressors by ZEB proteins”. 《EMBO J.》 22 (10): 2453–62. doi:10.1093/emboj/cdg226. PMC 155984. PMID 12743039.
Zweier C; Temple IK; Beemer F;  외. (2003). “Characterisation of deletions of the ZFHX1B region and genotype-phenotype analysis in Mowat-Wilson syndrome”. 《J. Med. Genet.》 40 (8): 601–5. doi:10.1136/jmg.40.8.601. PMC 1735564. PMID 12920073.