補体制御因子
引用

↑  “Amino acid patterns within short consensus repeats define conserved duplicons shared by genes of the RCA complex”. Journal of Molecular Evolution 59 (2):  143–57. (August 2004). Bibcode: 2004JMolE..59..143M. doi:10.1007/s00239-004-2609-8. PMID 154866906
↑  “Discrimination between host and pathogens by the complement system”. Vaccine 26 Suppl 8 (Suppl 8):  I15-21. (December 2008). doi:10.1016/j.vaccine.2008.11.023. PMC 2673523. PMID 19388159. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2673523/.
↑  “Complementing the Sugar Code: Role of GAGs and Sialic Acid in Complement Regulation”. Frontiers in Immunology 6:  25. (2015-02-02). doi:10.3389/fimmu.2015.00025. PMC 4313701. PMID 25699044. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4313701/.
↑  “Complement regulators and inhibitory proteins”. Nature Reviews. Immunology 9 (10):  729–40. (October 2009). doi:10.1038/nri2620. PMID 19730437.
↑  “Factor H Family Proteins in Complement Evasion of Microorganisms”. Frontiers in Immunology 8:  571. (2017-05-18). doi:10.3389/fimmu.2017.00571. PMC 5435753. PMID 28572805. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5435753/.
↑  “Spatially conserved motifs in complement control protein domains determine functionality in regulators of complement activation-family proteins”. Communications Biology 2 (1):  290. (December 2019). doi:10.1038/s42003-019-0529-9. PMC 6683126. PMID 31396570. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6683126/.
↑  “Discrimination between host and pathogens by the complement system”. Vaccine 26 Suppl 8 (Suppl 8):  I15-21. (December 2008). doi:10.1016/j.vaccine.2008.11.023. PMC 2673523. PMID 19388159. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2673523/.
↑  “Complement factor H gene mutation associated with autosomal recessive atypical hemolytic uremic syndrome”. American Journal of Human Genetics 66 (5):  1721–2. (May 2000). doi:10.1086/302877. PMC 1378030. PMID 10762557. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1378030/.
↑  “A common haplotype in the complement regulatory gene factor H (HF1/CFH) predisposes individuals to age-related macular degeneration”. Proceedings of the National Academy of Sciences of the United States of America 102 (20):  7227–32. (May 2005). doi:10.1073/pnas.0501536102. PMC 1088171. PMID 15870199. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1088171/6
↑  “Progress in Xenotransplantation: Immunologic Barriers, Advances in Gene Editing, and Successful Tolerance Induction Strategies in Pig-To-Primate Transplantation”. Frontiers in Immunology 13:  899657. (2022). doi:10.3389/fimmu.2022.899657. PMC 9157571. PMID 35663933. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9157571/.
↑  “Xenotransplantation: Current Status in Preclinical Research”. Frontiers in Immunology 10:  3060. (2020). doi:10.3389/fimmu.2019.03060. PMC 6989439. PMID 32038617. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6989439/.
↑  “Complement: a key system for immune surveillance and homeostasis”. Nature Immunology 11 (9):  785–97. (September 2010). doi:10.1038/ni.1923. PMC 2924908. PMID 20720586. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2924908/.
↑  “Human complement control and complement evasion by pathogenic microbes--tipping the balance”. Molecular Immunology 56 (3):  152–60. (December 2013). doi:10.1016/j.molimm.2013.05.222. PMID 23810413. http://lup.lub.lu.se/record/4027108.
↑  Baum, Matthew L. (2018-09-16) (英語). The Schizophrenia-Associated Gene, CSMD1, Encodes a Brain-Specific Complement Inhibitor. https://dash.harvard.edu/handle/1/41121210.
↑  “Altered expression of the CSMD1 gene in the peripheral blood of schizophrenia patients”. BMC Psychiatry 19 (1):  113. (April 2019). doi:10.1186/s12888-019-2089-4. PMC 6466712. PMID 30987620. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6466712/6
↑  “The complement control-related genes CSMD1 and CSMD2 associate to schizophrenia” (English). Biological Psychiatry 70 (1):  35–42. (July 2011). doi:10.1016/j.biopsych.2011.01.030. PMID 21439553. https://www.biologicalpsychiatryjournal.com/article/S0006-3223(11)00122-3/abstract6