プロテアーゼ活性化受容体3
関連文献

“International Union of Pharmacology. XXVIII. Proteinase-activated receptors.”. Pharmacol. Rev. 54 (2):  203–17. (2002). doi:10.1124/pr.54.2.203. PMID 12037136.
“Protease-activated receptor 3 is a second thrombin receptor in humans.”. Nature 386 (6624):  502–6. (1997). Bibcode: 1997Natur.386..502I. doi:10.1038/386502a0. PMID 9087410.
“The human proteinase-activated receptor-3 (PAR-3) gene. Identification within a Par gene cluster and characterization in vascular endothelial cells and platelets.”. J. Biol. Chem. 273 (24):  15061–8. (1998). doi:10.1074/jbc.273.24.15061. PMID 9614115.
“Cloning and characterization of human protease-activated receptor 4”. Proc. Natl. Acad. Sci. U.S.A. 95 (12):  6642–6. (1998). Bibcode: 1998PNAS...95.6642X. doi:10.1073/pnas.95.12.6642. PMC 22580. PMID 9618465. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC22580/.
“Protease-activated receptor genes are clustered on 5q13”. Blood 92 (1):  25–31. (1998). doi:10.1182/blood.V92.1.25.413k41_25_31. PMID 9639495.
“A dual thrombin receptor system for platelet activation”. Nature 394 (6694):  690–4. (1998). Bibcode: 1998Natur.394..690K. doi:10.1038/29325. PMID 9716134.
“Gene and locus structure and chromosomal localization of the protease-activated receptor gene family”. J. Biol. Chem. 273 (36):  23290–6. (1998). doi:10.1074/jbc.273.36.23290. PMID 9722561.
“Protease-activated receptors 1 and 4 mediate activation of human platelets by thrombin”. J. Clin. Invest. 103 (6):  879–87. (1999). doi:10.1172/JCI6042. PMC 408153. PMID 10079109. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC408153/.
“alpha-Thrombin stimulates contraction of human bronchial rings by activation of protease-activated receptors”. Am. J. Physiol. 277 (1 Pt 1):  L22–9. (1999). doi:10.1152/ajplung.1999.277.1.L22. PMID 10409227.
“PAR3 is a cofactor for PAR4 activation by thrombin”. Nature 404 (6778):  609–13. (2000). Bibcode: 2000Natur.404..609N. doi:10.1038/35007085. PMID 10766244.
“Thrombin responses in human endothelial cells. Contributions from receptors other than PAR1 include the transactivation of PAR2 by thrombin-cleaved PAR1”. J. Biol. Chem. 275 (18):  13502–9. (2000). doi:10.1074/jbc.275.18.13502. PMID 10788464.
“Neutrophil proteases can inactivate human PAR3 and abolish the co-receptor function of PAR3 on murine platelets”. Thromb. Haemost. 85 (3):  533–8. (2001). doi:10.1055/s-0037-1615617. PMID 11307827.
“Role of thrombin signalling in platelets in haemostasis and thrombosis”. Nature 413 (6851):  74–8. (2001). Bibcode: 2001Natur.413...74S. doi:10.1038/35092573. PMID 11544528.
“Trypsin induces activation and inflammatory mediator release from human eosinophils through protease-activated receptor-2”. J. Immunol. 167 (11):  6615–22. (2002). doi:10.4049/jimmunol.167.11.6615. PMID 11714832.
“Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences”. Proc. Natl. Acad. Sci. U.S.A. 99 (26):  16899–903. (2003). doi:10.1073/pnas.242603899. PMC 139241. PMID 12477932. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC139241/.
“The pattern of expression of protease-activated receptors (PARs) during early trophoblast development”. J. Pathol. 200 (1):  47–52. (2003). doi:10.1002/path.1338. PMID 12692840.
Owen WG (2003). “PAR-3 is a low-affinity substrate, high affinity effector of thrombin”. Biochem. Biophys. Res. Commun. 305 (1):  166–8. doi:10.1016/S0006-291X(03)00732-0. PMID 12732212.
“Thrombin-induced Ca2+ mobilization in human gingival fibroblasts is mediated by protease-activated receptor-1 (PAR-1)”. Life Sci. 73 (3):  301–10. (2003). doi:10.1016/S0024-3205(03)00269-8. PMID 12757837.