インターロイキン-22
関連文献

“Common polymorphisms in the interleukin-22 gene are not associated with chronic plaque psoriasis”. Experimental Dermatology 18 (9):  796–8. (September 2009). doi:10.1111/j.1600-0625.2009.00840.x. PMID 19469905.
“New genetic associations detected in a host response study to hepatitis B vaccine”. Genes and Immunity 11 (3):  232–8. (April 2010). doi:10.1038/gene.2010.1. PMID 20237496.
“Ulcerative colitis-risk loci on chromosomes 1p36 and 12q15 found by genome-wide association study”. Nature Genetics 41 (2):  216–20. (February 2009). doi:10.1038/ng.275. PMC 2652837. PMID 19122664. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2652837/.
“Crystal structure of a soluble decoy receptor IL-22BP bound to interleukin-22”. FEBS Letters 583 (7):  1072–7. (April 2009). doi:10.1016/j.febslet.2009.03.006. PMID 19285080.
“Activation of peripheral Th17 lymphocytes in patients with asthma”. Immunological Investigations 38 (7):  652–64. (2009). doi:10.1080/08820130903062756. PMID 19811428.
“Frequency and phenotype of peripheral blood Th17 cells in ankylosing spondylitis and rheumatoid arthritis”. Arthritis and Rheumatism 60 (6):  1647–56. (June 2009). doi:10.1002/art.24568. PMID 19479869.
“Interleukin-22 genetic polymorphisms and risk of colon cancer”. Cancer Causes & Control 21 (8):  1165–70. (August 2010). doi:10.1007/s10552-010-9542-5. PMID 20339910.
“Stage 3 immature human natural killer cells found in secondary lymphoid tissue constitutively and selectively express the TH 17 cytokine interleukin-22”. Blood 113 (17):  4008–10. (April 2009). doi:10.1182/blood-2008-12-192443. PMC 2673127. PMID 19244159. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2673127/.
“Genetic susceptibility to respiratory syncytial virus bronchiolitis in preterm children is associated with airway remodeling genes and innate immune genes”. The Pediatric Infectious Disease Journal 28 (4):  333–5. (April 2009). doi:10.1097/INF.0b013e31818e2aa9. PMID 19258923.
“IL-17 and IL-22 are associated with protection against human kala azar caused by Leishmania donovani”. The Journal of Clinical Investigation 119 (8):  2379–87. (August 2009). doi:10.1172/JCI38813. PMC 2719936. PMID 19620772. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2719936/.
“Decreased serum IL-22 levels in patients with systemic lupus erythematosus”. Clinica Chimica Acta; International Journal of Clinical Chemistry 401 (1–2):  179–80. (March 2009). doi:10.1016/j.cca.2008.11.009. PMID 19046958.
“Memory IL-22-producing CD4+ T cells specific for Candida albicans are present in humans”. European Journal of Immunology 39 (6):  1472–9. (June 2009). doi:10.1002/eji.200838811. PMID 19449309.
“Involvement of the IL-22/REG Ialpha axis in ulcerative colitis”. Laboratory Investigation; A Journal of Technical Methods and Pathology 90 (3):  496–505. (March 2010). doi:10.1038/labinvest.2009.147. PMID 20065946.
“IL-24 transgenic mice: in vivo evidence of overlapping functions for IL-20, IL-22, and IL-24 in the epidermis”. Journal of Immunology 184 (4):  1793–8. (February 2010). doi:10.4049/jimmunol.0901829. PMID 20061404.
“The Th17 cytokine IL-22 induces IL-20 production in keratinocytes: a novel immunological cascade with potential relevance in psoriasis”. European Journal of Immunology 39 (12):  3570–81. (December 2009). doi:10.1002/eji.200939687. PMID 19830738.
“Th22 cells represent a distinct human T cell subset involved in epidermal immunity and remodeling”. The Journal of Clinical Investigation 119 (12):  3573–85. (December 2009). doi:10.1172/JCI40202. PMC 2786807. PMID 19920355. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2786807/.
“IL-22 produced by human NK cells inhibits growth of Mycobacterium tuberculosis by enhancing phagolysosomal fusion”. Journal of Immunology 183 (10):  6639–45. (November 2009). doi:10.4049/jimmunol.0902587. PMID 19864591.
“A human natural killer cell subset provides an innate source of IL-22 for mucosal immunity”. Nature 457 (7230):  722–5. (February 2009). doi:10.1038/nature07537. PMC 3772687. PMID 18978771. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3772687/.
“Anti-inflammatory and pro-inflammatory roles of TGF-beta, IL-10, and IL-22 in immunity and autoimmunity”. Current Opinion in Pharmacology 9 (4):  447–53. (August 2009). doi:10.1016/j.coph.2009.04.008. PMC 2755239. PMID 19481975. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2755239/.
“Circulating Th17, Th22, and Th1 cells are increased in psoriasis”. The Journal of Investigative Dermatology 130 (5):  1373–83. (May 2010). doi:10.1038/jid.2009.399. PMC 2892169. PMID 20032993. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2892169/.