שגיב שיפמן
הערות שוליים

1 2  Sagiv Shifman, Michal Bronstein, Meira Sternfeld, Anne Pisanté-Shalom, Efrat Lev-Lehman, Avraham Weizman, Ilya Reznik, Baruch Spivak, Nimrod Grisaru, Leon Karp, Richard Schiffer, Moshe Kotler, Rael D. Strous, Marnina Swartz-Vanetik, Haim Y. Knobler, Eilat Shinar, Jacques S. Beckmann, Benjamin Yakir, Neil Risch, Naomi B. Zak, Ariel Darvasi, A highly significant association between a COMT haplotype and schizophrenia, American Journal of Human Genetics 71, 2002-12, עמ' 1296–1302 doi: 10.1086/344514
1 2  Sagiv Shifman, Jordana Tzenova Bell, Richard R. Copley, Martin S. Taylor, Robert W. Williams, Richard Mott, Jonathan Flint, A high-resolution single nucleotide polymorphism genetic map of the mouse genome, PLoS biology 4, 2006-11, עמ' e395 doi: 10.1371/journal.pbio.0040395
1 2  S. Shifman, A. Bhomra, S. Smiley, N. R. Wray, M. R. James, N. G. Martin, J. M. Hettema, S. S. An, M. C. Neale, E. J. C. G. van den Oord, K. S. Kendler, X. Chen, D. I. Boomsma, C. M. Middeldorp, J. J. Hottenga, P. E. Slagboom, J. Flint, A whole genome association study of neuroticism using DNA pooling, Molecular Psychiatry 13, 2008-03, עמ' 302–312 doi: 10.1038/sj.mp.4002048
↑  S. Shifman, A. Darvasi, The value of isolated populations, Nature Genetics 28, 2001-08, עמ' 309–310 doi: 10.1038/91060
↑  Sagiv Shifman, Jane Kuypers, Mark Kokoris, Benjamin Yakir, Ariel Darvasi, Linkage disequilibrium patterns of the human genome across populations, Human Molecular Genetics 12, 2003-04-01, עמ' 771–776 doi: 10.1093/hmg/ddg088
↑  Sagiv Shifman, Martina Johannesson, Michal Bronstein, Sam X. Chen, David A. Collier, Nicholas J. Craddock, Kenneth S. Kendler, Tao Li, Michael O'Donovan, F. Anthony O'Neill, Michael J. Owen, Dermot Walsh, Daniel R. Weinberger, Cuie Sun, Jonathan Flint, Ariel Darvasi, Genome-wide association identifies a common variant in the reelin gene that increases the risk of schizophrenia only in women, PLoS genetics 4, 2008-02, עמ' e28 doi: 10.1371/journal.pgen.0040028
↑  Eyal Ben-David, Sagiv Shifman, Networks of neuronal genes affected by common and rare variants in autism spectrum disorders, PLoS genetics 8, 2012, עמ' e1002556 doi: 10.1371/journal.pgen.1002556
↑  E Ben-David, S Shifman, Combined analysis of exome sequencing points toward a major role for transcription regulation during brain development in autism, Molecular Psychiatry 18, 2013-10, עמ' 1054–1056 doi: 10.1038/mp.2012.148
↑  Shahar Shohat, Eyal Ben-David, Sagiv Shifman, Varying Intolerance of Gene Pathways to Mutational Classes Explain Genetic Convergence across Neuropsychiatric Disorders, Cell Reports 18, 2017-02, עמ' 2217–2227 doi: 10.1016/j.celrep.2017.02.007
↑  Galya Monderer-Rothkoff, Nitzan Tal, Marina Risman, Odem Shani, Malka Nissim-Rafinia, Laura Malki-Feldman, Vera Medvedeva, Matthias Groszer, Eran Meshorer, Sagiv Shifman, AUTS2 isoforms control neuronal differentiation, Molecular Psychiatry 26, 2021-02, עמ' 666–681 doi: 10.1038/s41380-019-0409-1
↑  Reut Suliman-Lavie, Ben Title, Yahel Cohen, Nanako Hamada, Maayan Tal, Nitzan Tal, Galya Monderer-Rothkoff, Bjorg Gudmundsdottir, Kristbjorn O. Gudmundsson, Jonathan R. Keller, Guo-Jen Huang, Koh-Ichi Nagata, Yosef Yarom, Sagiv Shifman, Pogz deficiency leads to transcription dysregulation and impaired cerebellar activity underlying autism-like behavior in mice, Nature Communications 11, 2020-11-17, עמ' 5836 doi: 10.1038/s41467-020-19577-0
↑  Neta Herman, Sebastian Kadener, Sagiv Shifman, The chromatin factor ROW cooperates with BEAF ‐32 in regulating long‐range inducible genes, EMBO reports 23, 2022-12-06 doi: 10.15252/embr.202254720
↑  Shahar Shohat, Sagiv Shifman, Genes essential for embryonic stem cells are associated with neurodevelopmental disorders, Genome Research 29, 2019-11, עמ' 1910–1918 doi: 10.1101/gr.250019.119
↑  Elad Dvir, Shahar Shohat, Jonathan Flint, Sagiv Shifman, Identification of genetic mechanisms for tissue-specific genetic effects based on CRISPR screens, Genetics 222, 2022-11-01, עמ' iyac134 doi: 10.1093/genetics/iyac134
↑  Shahar Shohat, Ethel Vol, Sagiv Shifman, Gene essentiality in cancer cell lines is modified by the sex chromosomes, Genome Research, 2022-11-23 doi: 10.1101/gr.276488.121
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בקרת זהויות :  NLI: 987007268023105171  ORCID: 0000-0003-4071-5361  ResearcherID: D-8749-2017  VIAF: 98210544   WorldCat (עם VIAF): 98210544