التسمم بالزنك
مراجع

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التصنيفات الطبية:  رموز المراجعة التاسعة للتصنيف الدولي للأمراض (ICD-9): 985.8  رموز المراجعة العاشرة للتصنيف الدولي للأمراض (ICD-10): T56.5  
المعرفات الخارجية:  موقع مدلاين (MedlinePlus): 002570  
بوابة طب
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↑  Smith، SE؛ Larson, EJ (أبريل 1946). "Zinc toxicity in rats; antagonistic effects of copper and liver". The Journal of Biological Chemistry. ج. 163: 29–38. DOI:10.1016/S0021-9258(17)41344-5. PMID:21023625.
↑  Brita، T. A.؛ De Schamphelaere، Muyssen؛ Karel، A. C.؛ Janssen، Colin R. (2006). "Mechanisms of chronic waterborne Zn toxicity in Daphnia magna". Aquatic Toxicology. ج. 77 ع. 4: 393–401. DOI:10.1016/j.aquatox.2006.01.006. PMID:16472524.
1 2 3 4  Fosmire GJ (فبراير 1990). "Zinc toxicity". Am. J. Clin. Nutr. ج. 51 ع. 2: 225–227. DOI:10.1093/ajcn/51.2.225. PMID:2407097. مؤرشف من الأصل في 2023-10-28.
↑  Ciubotariu D، Ghiciuc CM، Lupușoru CE (2015). "Zinc involvement in opioid addiction and analgesia – should zinc supplementation be recommended for opioid-treated persons?". Subst Abuse Treat Prev Policy. ج. 10 ع. 1: 29. DOI:10.1186/s13011-015-0025-2. PMC:4523930. PMID:26238243.
↑  Pettilä، V.؛ Takkunen, O.؛ Tukiainen, P. (2000). "Zinc chloride smoke inhalation: a rare cause of severe acute respiratory distress syndrome". Intensive Care Medicine. ج. 26 ع. 2: 215–217. DOI:10.1007/s001340050049. PMID:10784312. S2CID:39004564.
↑  Pories، W.J. (1967). "Acceleration of healing with zinc sulfate". Ann Surg. ج. 165 ع. 3: 432–436. DOI:10.1097/00000658-196703000-00015. PMC:1617499. PMID:6019319.
↑  Simkin، P.A. (1976). "Oral zinc sulphate in rheumatoid arthritis". Lancet. ج. 2 ع. 7985: 539–542. DOI:10.1016/s0140-6736(76)91793-1. PMID:60622. S2CID:31836064.
1 2  Samman، S.؛ Roberts، D.C. (1987). "The effect of zinc supplements on plasma zinc and copper levels and the reported symptoms in healthy volunteers". Med J Aust. ج. 146 ع. 5: 246–249. DOI:10.5694/j.1326-5377.1987.tb120232.x. PMID:3547053. S2CID:10531701.
↑  Forman، W.B. (1990). "Zinc abuse: an unsuspected cause of sideroblastic anemia". West J Med. ج. 152 ع. 2: 190–192. PMC:1002314. PMID:2400417.
↑  Fiske، D.N. (1994). "Zinc-induced sideroblastic anemia: report of a case, review of the literature, and description of the hematologic syndrome". Am J Hematol. ج. 46 ع. 2: 147–150. DOI:10.1002/ajh.2830460217. PMID:8172183. S2CID:22495489.
↑  Hemilä، H. (23 يونيو 2011). "Zinc lozenges may shorten the duration of colds: a systematic review". Open Respir Med J. ج. 5 ع. 1: 51–58. DOI:10.2174/1874306401105010051. PMC:3136969. PMID:21769305. مؤرشف من الأصل في 2022-10-08.
↑  Lim KH، Riddell LJ، Nowson CA، Booth AO، Szymlek-Gay EA (2013). "Iron and zinc nutrition in the economically-developed world: a review". Nutrients. ج. 5 ع. 8: 3184–3211. DOI:10.3390/nu5083184. PMC:3775249. PMID:23945676. Homeostatic regulation of iron and zinc differ, with iron being regulated through absorption and zinc being regulated primarily through secretion. As the body does not have a means to eliminate excess iron, absorption from the small intestine is tightly regulated by hepcidin. Hepcidin is a peptide hormone that is present in higher concentrations when body iron is replete [52]. Higher concentrations of hepcidin prevent ingested iron from entering the bloodstream by trapping iron in enterocytes which are naturally shed every two days [112], thereby preventing body iron from escalating to dangerous levels. In comparison, endogenous (pancreatic, biliary and intestinal) secretions comprise the main route of zinc loss, with larger zinc intakes being balanced by larger zinc secretions [113, 114].