ميثيل كولانثرين
مراجع

↑  Malins DC، Anderson KM، Gilman NK، Green VM، Barker EA، Hellström KE (يوليو 2004). "Development of a cancer DNA phenotype prior to tumor formation". Proceedings of the National Academy of Sciences of the United States of America. ج. 101 ع. 29: 10721–10725. DOI:10.1073/pnas.0403888101. JSTOR:3372726. PMC:490001. PMID:15249662.
↑  "3-methylcholanthrene".
↑  Lide DR (2007). "CRC Handbook of Chemistry and Physics 88th Edition". ص. 173.
↑  Duran-Reynals ML، Stanley B (ديسمبر 1961). "Vaccinia dermal infection and methylcholanthrene in cortisone-treated mice". Science. ج. 134 ع. 3494: 1984–1985. Bibcode:1961Sci...134.1984D. DOI:10.1126/science.134.3494.1984. PMID:13888610. S2CID:40961133.
↑  Cook JW، Haslewood GA (1934). "The synthesis of 5 : 6-dimethyl-1 : 2-benzanthraquinone, a degradation product of deoxycholic acid". Journal of the Chemical Society. ص. 428.
↑  Jacobs SA، Harvey RG (1981). "Synthesis of 3-methylcholanthrene". Tetrahedron Letters. ج. 22: 1093–1096.
↑  Harvey RG، Cortez C، Jacobs SA (1982). "Synthesis of polycyclic aromatic hydrocarbons via a novel annelation method". The Journal of Organic Chemistry. ج. 47 ع. 11: 2120–2125.
1 2  Venkatesan N، Argus MF، Arcos JC (أكتوبر 1970). "Mechanism of 3-methylcholanthrene-induced inhibition of dimethylnitrosamine demethylase in rat liver". Cancer Research. ج. 30 ع. 10: 2556–2562. PMID:5474178. مؤرشف من الأصل في 2020-09-17.
↑  Lasnitzki I، Bard DR، Franklin HR (أغسطس 1975). "3-Methylcholanthrene uptake and metabolism in organ culture". British Journal of Cancer. ج. 32 ع. 2: 219–229. DOI:10.1038/bjc.1975.152. PMC:2024855. PMID:1240005.
↑  Venkatesan N، Argus MF، Arcos JC (أكتوبر 1970). "Mechanism of 3-methylcholanthrene-induced inhibition of dimethylnitrosamine demethylase in rat liver". Cancer Research. ج. 30 ع. 10: 2556–2562. PMID:5474178. مؤرشف من الأصل في 2020-09-17.Venkatesan N, Argus MF, Arcos JC (October 1970). "Mechanism of 3-methylcholanthrene-induced inhibition of dimethylnitrosamine demethylase in rat liver". Cancer Research. 30 (10): 2556–2562. PMID 5474178.
↑  Eastman A، Sweetenham J، Bresnick E (ديسمبر 1978). "Comparison of in vivo and in vitro binding of polycyclic hydrocarbons to DNA". Chemico-Biological Interactions. ج. 23 ع. 3: 345–353. DOI:10.1016/0009-2797(78)90095-9. PMID:719814.
↑  Sheen YY، Kim SS، Yun HC (1993). "Effect of 3-methylcholanthrene on rat uterus: Uterine growth and mechanism of action of 3-methylcholanthrene". Archives of Pharmacal Research. ج. 16 ع. 4: 276. DOI:10.1007/bf02977516. S2CID:95829643.
1 2  Dauben WG، Mabee D (مارس 1951). "A study of the metabolism of 20-methylcholanthrene". Cancer Research. ج. 11 ع. 3: 216–220. PMID:14821965. مؤرشف من الأصل في 2020-09-17.
↑  Eastman A، Sweetenham J، Bresnick E (ديسمبر 1978). "Comparison of in vivo and in vitro binding of polycyclic hydrocarbons to DNA". Chemico-Biological Interactions. ج. 23 ع. 3: 345–353. DOI:10.1016/0009-2797(78)90095-9. PMID:719814.Eastman A, Sweetenham J, Bresnick E (December 1978). "Comparison of in vivo and in vitro binding of polycyclic hydrocarbons to DNA". Chemico-Biological Interactions. 23 (3): 345–353. doi:10.1016/0009-2797(78)90095-9. PMID 719814.
↑  Lasnitzki I، Bard DR، Franklin HR (أغسطس 1975). "3-Methylcholanthrene uptake and metabolism in organ culture". British Journal of Cancer. ج. 32 ع. 2: 219–229. DOI:10.1038/bjc.1975.152. PMC:2024855. PMID:1240005.Lasnitzki I, Bard DR, Franklin HR (August 1975). "3-Methylcholanthrene uptake and metabolism in organ culture". British Journal of Cancer. 32 (2): 219–229. doi:10.1038/bjc.1975.152. PMC 2024855. PMID 1240005.
↑  He R (أكتوبر 1991). "[Precancerous and cancerous lesions and their gamma-glutamyl transpeptidase expression in 3-methylcholanthrene-induced lung carcinogenesis in rats]". Zhongguo Yi Xue Ke Xue Yuan Xue Bao. Acta Academiae Medicinae Sinicae. ج. 13 ع. 5: 343–346. PMID:1686852.
↑  Jin Y، Miao W، Lin X، Pan X، Ye Y، Xu M، Fu Z (ديسمبر 2014). "Acute exposure to 3-methylcholanthrene induces hepatic oxidative stress via activation of the Nrf2/ARE signaling pathway in mice". Environmental Toxicology. ج. 29 ع. 12: 1399–1408. DOI:10.1002/tox.21870. PMID:23712962. S2CID:6071670.
↑  Shipley JM، Waxman DJ (يونيو 2006). "Aryl hydrocarbon receptor-independent activation of estrogen receptor-dependent transcription by 3-methylcholanthrene". Toxicology and Applied Pharmacology. ج. 213 ع. 2: 87–97. DOI:10.1016/j.taap.2005.09.011. PMID:16257430.
↑  "TOXNET". U.S. National Library of Medicine.
↑  Curren RD، Homer CJ، Price PJ، Freeman AE (مارس 1979). "Mutagenesis of human cells by 3-methylcholanthrene". Mutation Research. ج. 60 ع. 1: 109–113. DOI:10.1016/0027-5107(79)90214-8. PMID:431552.
↑  Hutton JJ، Meier J، Hackney C (يناير 1979). "Comparison of the in vitro mutagenicity and metabolism of dimethylnitrosamine and benzo[a]pyrene in tissues from inbred mice treated with phenobarbital, 3-methylcholanthrene or polychlorinated biphenyls". Mutation Research. ج. 66 ع. 1: 75–94. DOI:10.1016/0165-1218(79)90010-7. PMID:106274.
↑  Miller MS، Jones AB، Park SS، Anderson LM (يونيو 1990). "The formation of 3-methylcholanthrene-initiated lung tumors correlates with induction of cytochrome P450IA1 by the carcinogen in fetal but not adult mice". Toxicology and Applied Pharmacology. ج. 104 ع. 2: 235–245. DOI:10.1016/0041-008x(90)90298-9. PMID:2363175.