The condition of electro-hypersensitivity mentioned in the article is real and research results into objective physiological markers were presented at a recent scientific conference in Belgium by Prof. Dominique Belpomme of the ARTAC institute. I expect this to be published soon but for now I copy here the abstract from Belpomme's presentation:

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"Since 2009, we prospectively investigated clinically and biologically 1200 consecutive EHS (Electro-HyperSensitivity) and/or MCS (Multiple Chemical-Sensitivity) self reported cases, in an attempt to establish objective diagnosis criteria and to elucidate the pathophysiological aspects of these two disorders. Inclusion criteria were: absence of diagnosed pathology; reproduced symptoms under the influence of electromagnetic fields (EMF) and/or chemicals, whatever their incriminated source; and regression or disappearance of symptoms in the case of EMF and/or chemical avoidance. We report here our preliminary results, based on the analysis of 839 originally enrolled cases of which 810 met the inclusion criteria and 727 were evaluable. 521 were diagnosed with EHS, 52 with MCS and 154 with both EHS and MCS. Sex ratio (F/M) was 495/232, meaning that 2 out of 3 patients with EHS and/or MCS were female; mean age (years) was 47.9 ± 12.4 and median age was 47 (range 16-83), with no significant difference between the 3 groups in those characteristics. Concomitant multiple food intolerance was found in 28.5%, 41.9% and 70.4% of the cases in the 3 groups respectively. Since inflammation appears to be a key process resulting from the EMF and/or chemical effects on tissues, and histamine release is potentially a major trait of inflammation, we systematically measured histamine in the blood of patients. Importantly, 37%, 36.7% and 41.5% of the patients respectively in the 3 above individualized groups showed a significant increase in histaminemia (>10 nmol/l), meaning that a chronic inflammatory response can be detected in these patients. As the release of oxidants is part of the basic process of inflammation and they are a key contributor to damage and response, we also measured nitrotyrosin (NTT), a marker of both peroxynitrite (ONOO .- ) production and opening of the blood brain barrier (BBB). NTT was increased in the blood (>0.90 μg/ml) in 29.7%, 26% and 28% of the cases in our 3 groups, respectively. Likewise protein S100B, another marker of BBB opening was found to be increased in the blood (>0.105 μg/L) in 14.7%, 19.7% and 10.7% of our cases, respectively. Circulating antibodies against O-myelin, Hsp 27 and/or Hsp 70 protein were also found to be increased in 43.1%, 25% and 52% of our cases respectively, indicating that the EHS and MCS disorders are associated with some autoimmune response. Since most patients reported chronic insomnia and fatigue, we also determined the 24h urine melatonin/creatinin ratio and found it was decreased (<0.8) in all investigated cases.

Finally, in order to gain further information about the underlying mechanisms of EHS and MCS, we serially measured the brain blood flow in the temporal lobes of each patient by using pulsed brain echodoppler. We found that both disorders were associated with an hypo-perfusion in the capsulothalamic area, suggesting that the inflammatory process may in fact involve the limbic system and the thalamus.

This ongoing study already strongly suggests that EHS and MCS can be objectively characterized and diagnosed with very simple tests. Both disorders appear to paint a common picture of inflammation-related hyper-histaminemia, oxidative stress, autoimmune response and BBB opening, and a deficit in melatonin excretion. EHS and MCS probably share a common pathological mechanism mainly involving the nervous central system."
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My note: The take home message here is that chronic, low-level, microwave exposure (like from cellphone & wi-fi) causes immune-system activation and inflammation, possibly leading to immune-system burnout if not controlled. Based on the results it is advised to reduce exposure now.