There were, however, mutations in four other genes in ALL patients that destabilized the MSH2 protein, and these were defective in 11% of children with ALL and 16% of adults with this cancer.
Porén, había mutacións noutros catro xenes en todos os pacientes de leucemia linfoblastoide aguda que desestabilizaban a proteína MSH2, e esta era defectiva no 11% dos nenos con esta leucemia e o 16% dos adultos.