Question: What genetic condition did the actor who died of a heart defect on the same day his character did have?

Context articles: Eisenmenger's syndrome
Timmy Lenox
Cyanotic heart defect
Quadricuspid aortic valve
Acyanotic heart defect
Josh Ryan Evans
Atrial septal defect
Heart septal defect
Noonan syndrome
Cor triatriatum

Eisenmenger's syndrome (or ES, Eisenmenger's reaction, Eisenmenger physiology, or tardive cyanosis) is defined as the process in which a long-standing left-to-right cardiac shunt caused by a congenital heart defect (typically by a ventricular septal defect, atrial septal defect, or less commonly, patent ductus arteriosus) causes pulmonary hypertension and eventual reversal of the shunt into a cyanotic right-to-left shunt.
 Because of the advent of fetal screening with echocardiography early in life, the incidence of heart defects progressing to Eisenmenger's has decreased.
Timmy Lenox is a fictional character from the NBC/DirecTV daytime drama "Passions" portrayed by Josh Ryan Evans.
 In a tragically ironic twist of fate, the actor died of a congenital heart defect the very day his character Timmy died on the show and donated his heart to Charity Standish.
Cyanotic heart defect is a group-type of congenital heart defect (CHD) that occurs due to deoxygenated blood bypassing the lungs and entering the systemic circulation or a mixture of oxygenated and unoxygenated blood entering the systemic circulation.
 It is caused by structural defects of the heart (i.e.: right-to-left, bidirectional shunting, malposition of the great arteries), or any condition which increases pulmonary vascular resistance.
 The result being the development of collateral circulation.
A quadricuspid aortic valve (QAV) is a rare congenital heart defect characterized by the presence of four cusps, instead of the usual three found normally in the aortic valve.
 It is a defect that occurs during embryological development of the aortic trunk during gestation.
 There is an increased risk of developing post-natal aortic regurgitations and other heart-related diseases; therefore patients with the condition should be carefully monitored.
An acyanotic heart defect, also known as non-cyanotic heart defect, is a class of congenital heart defects.
 In these, blood is shunted (flows) from the left side of the heart to the right side of the heart due to a structural defect (hole) in the interventricular septum.
 People often retain normal levels of oxyhemoglobin saturation in systemic circulation.
Joshua Ryan "Josh" Evans (January 10, 1982 – August 5, 2002) was an American actor who became known for his role of Timmy Lenox in the soap opera "Passions".
 Though he was 17 years old when "Passions" debuted, Evans had the appearance and voice of a small child due to achondroplasia, a form of dwarfism.
 He was 3 ft tall.
Atrial septal defect (ASD) is a congenital heart defect in which blood flows between the atria (upper chambers) of the heart.
 Normally, the atria are separated by a dividing wall, the interatrial septum.
 If this septum is defective or absent, then oxygen-rich blood can flow directly from the left side of the heart to mix with the oxygen-poor blood in the right side of the heart, or vice versa.
 This can lead to lower-than-normal oxygen levels in the arterial blood that supplies the brain, organs, and tissues.
 However, an ASD may not produce noticeable signs or symptoms, especially if the defect is small.
Heart septal defect refers to a congenital heart defect of one of the septa of the heart.
Noonan syndrome (NS) is a relatively common autosomal dominant congenital disorder and is named after Jacqueline Noonan, a pediatric cardiologist.
 It is referred to as the male version of Turner's syndrome; however, the genetic causes of Noonan syndrome and Turner syndrome are distinct and both males and females are affected.
 The principal features include congenital heart defect (typically pulmonary valve stenosis with dysplastic pulmonary valve also atrial septal defect and hypertrophic cardiomyopathy), short stature, learning problems, pectus excavatum, impaired blood clotting, and a characteristic configuration of facial features including a webbed neck and a flat nose bridge.
 NS is a RASopathy, and is one of several disorders that are caused by a disruption of RAS-MAPK signaling pathway.
Cor triatriatum (or triatrial heart) is a congenital heart defect where the left atrium (cor triatriatum sinistrum) or right atrium (cor triatriatum dextrum) is subdivided by a thin membrane, resulting in three atrial chambers (hence the name).
 Cor triatriatum represents 0.1% of all congenital cardiac malformations and may be associated with other cardiac defects in as many as 50% of cases.
 The membrane may be complete or may contain one or more fenestrations of varying size.
 Cor triatrium sinistrum is more common.
 In this defect there is typically a proximal chamber that receives the pulmonic veins and a distal (true) chamber located more anteriorly where it empties into the mitral valve.
 The membrane that separates the atrium into two parts varies significantly in size and shape.
 It may appear similar to a diaphragm or be funnel-shaped, bandlike, entirely intact (imperforate) or contain one or more openings (fenestrations) ranging from small, restrictive-type to large and widely open.In the pediatric population, this anomaly may be associated with major congenital cardiac lesions such as tetralogy of Fallot, double outlet right ventricle, coarctation of the aorta, partial anomalous pulmonary venous connection, persistent left superior vena cava with unroofed coronary sinus, ventricular septal defect, atrioventricular septal (endocardial cushion) defect, and common atrioventricular canal.
 Rarely, asplenia or polysplenia has been reported in these patients.
 In the adult, cor triatriatum is frequently an isolated finding.