**Huat-iām** (ing-gú: Inflammation; lâi-tsū latin-gú: inflammatio; **huat-hông** (發癀); **iām-tsìng** (炎症); hàn-gú: 發炎) sī sin-thé ê tsoo-tsit tuì iú-hāi tshì-kik (pí-jû pīng-guân-thé (pathogen), siū-sún sè-pau hi̍k-tsiá tshì-kik bu̍t) ê ho̍k-tsa̍p sing-bu̍t huán-ìng ê tsi̍t-pōo-hūn, sī sia̍p-ki̍p bián-i̍k sè-pau, hiat-kuán kap hun-tsú kài-tsit (分子介質) tíng-tíng tsoo-tsit ê pó-hōo sìng huán-ìng. Huat-iām ê kong-lîng sī siau-tû sè-pau sún-siong ê siōng-thâu-á ê guân-in, tshing-tû guân-sí sún-siong kah iām-tsìng kuè-tîng jî-lâi siū-sún ê huāi-sí (壞死) sè-pau hām tsoo-tsit; pīng-tshiánn khé-tōng tsoo-tsit siu-ho̍k.

Ngóo ê tsú-iàu ê thé-ting sī jia̍t, thiànn, âng, tsíng kap kong-lîng sòng-sit (latin-gú: calor, dolor, rubor, tumor kap functio laesa). Huat-iām sī tsi̍t-tsióng t-it-puann ê huán-ìng, in-tshú huat-iām hông jīn-uî sī sian-thian bián-i̍k ê tsi̍t-tsióng ki-tsè; put-kò sik-ìng sìng bián-i̍k tio̍h-ē tsiam-tuì ta̍k-tsióng pīng-guân-thé (pathogen). Huat-iām siunn-tsió khó-lîng ē tì-sú ū-hāi ê tshì-kik (pí-jû ba̋i-khín (bacteria)) tuì tsoo-tsit ū tsìn-hîng sìng ê phò-hāi; pīng-tshiánn sún-hāi sing-bu̍t thé ê sing-tsûn. Tò-píng lâi kóng, kuè-to ê bān-sìng huat-iām hîng-sik ê huat-iām kah to-tsióng pēnn-tsìng ū kuan-hē, pí-jû hue-hún tsìng (花粉症), gê-tsiu pēnn(牙周病), tōng-me̍h tsiok-iōng ngē-huà (動脈粥樣硬化) kap kut kuan-tsat-iām (骨關節炎).

Huat-iām ē-tàng hun-tsò kip-sìng hām bān-sìng tsit nn̄g-tsióng. Kip-sìng huat-iām sī sin-thé tuì ū-hāi tshì-kik ê siōng-thâu-á ê huán-ìng, tō sī thong-kuè hiat-tsiong hām pe̍h sè-pau (ti̍k-pia̍t sī lia̍p sè-pau) uì hiat-i̍k tsìn-ji̍p siū-sún tsoo-tsit ê ūn-tōng tsing-ka lâi si̍t-hiān ê. It hē-lia̍t sing-huà sū-kiānn thuân-pò kap sîng-si̍k ê huat-iām huán-ìng, sia̍p-ki̍p kio̍k-pōo hiat-kuán hē-thóng, bián-i̍k hē-thóng kap siū-siong tsoo-tsit lāi ê kok-tsióng sè-pau. Tn̂g-kî huat-iām (kiò-tsò bān-sìng huat-iām) ē tì-sú huat-iām pōo-uī ê sè-pau luī-hîng (pí-jû tan-hi̍k (單核) sè-pau) huat-sing ta̍uh-ta̍uh-á piàn-huà, i-ê ti̍k-ting sī huat-iām kuè-tîng tang-tiong tsoo-tsit tông-sîphò-hāi hām siu-kian.

Tsù-kái
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1. ↑  Ferrero-Miliani L, Nielsen OH, Andersen PS, Girardin SE (February 2007). "Chronic inflammation: importance of NOD2 and NALP3 in interleukin-1beta generation". *Clinical and Experimental Immunology*. **147** (2): 227–235. doi:10.1111/j.1365-2249.2006.03261.x. PMC 1810472 Freely accessible. PMID 17223962.
2. ↑  Chen L, Deng H, Cui H, Fang J, Zuo Z, Deng J, et al. (January 2018). "Inflammatory responses and inflammation-associated diseases in organs". *Oncotarget*. Impact Journals, LLC. **9** (6): 7204–7218. doi:10.18632/oncotarget.23208. PMC 5805548 Freely accessible. PMID 29467962.
3. ↑  Abbas AB, Lichtman AH (2009). "Ch.2 Innate Immunity". Chū Saunders (Elsevier). *Basic Immunology. Functions and disorders of the immune system* (3rd pán.). Saunders/Elsevier. ISBN 978-1-4160-4688-2.

Tsham-ua̍t
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* Anaphylatoxin
* Anti-inflammatories
* Essential fatty acid interactions
* Healing
* Inflammaging
* Inflammatory cytokine
* Inflammatory reflex
* Interleukin
* Lipoxin
* Neurogenic inflammation
* Substance P
* Stress (biology)
* Endothelial Cell Tropism

Guā-pōo liân-kiat
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|  | Tī Wikimedia Commons téng ê siong-koan tóng-àn: ***Inflammations*** |

* Inflammation at the U.S. National Library of Medicine Medical Subject Headings (MeSH)

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| Sìn-kù khòng-chè | 
* LCCN: sh85066108
* GND: 4014975-4
* BNF: cb119720196 (data)
* NDL: 00562014
* NKC: ph135115

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