instruction:
Indicate the presence of numerical data in the abstract (whether numerical data plays a key role in medical records), solely using the information from the abstract.
question:
Abstract: The progeny of coisogenic C57BL/6J mice homozygous for either the tottering (tg/tg) or wildtype (+/+) allele were examined to determine the role of parental seizure disorders and paternal anticonvulsant drug therapy on the incidence of congenital malformations. Pregnancy outcomes in which one, both, or neither of the parents had an epileptic genotype were compared for various maternal and fetal reproductive parameters. In addition, the progeny from phenytoin-treated male tottering (tg/tg) mice with a spontaneous seizure disorder and from control untreated (+/+) male mice were similarly examined for evidence of congenital defects. The results of these experiments suggest that neither the parental genotype with respect to seizure disorders nor paternal anticonvulsant treatment with phenytoin is responsible for an increased incidence of congenital malformations.
Question: Does the abstract contain any numerical data?
answer:
Yes

question:
Abstract: We have determined the structures of high mannose (Man) oligosaccharide units of the alpha- and beta-subunits of mouse TSH heterodimers and of free alpha-subunits. Mouse thyrotropic tumor tissue, or pituitaries from euthyroid or hypothyroid mice, were incubated with D-[2-3H]Man, homogenized, and incubated in the presence or absence of pH 3 buffer to dissociate heterodimers. Highly enriched TSH beta-subunits, or TSH heterodimers, were obtained using anti-TSH beta serum and free alpha-subunits were subsequently obtained using anti-LH alpha-serum. High Man units released by endoglycosidase H were analyzed by paper chromatography. At 1 and 3 h oligosaccharides with 8 or 9 Man residues predominated in all subunits. In all three tissue types, units with 9 Man tended to accumulate in TSH beta-subunits, whereas Man-trimming from Man9GlcNAc to Man8GlcNAc proceeded more rapidly in TSH alpha-subunits, and in free alpha-subunits. The rate of TSH beta-subunit processing was also a function of tissue type; the rate in euthyroid pituitary tissue equaled that in tumor tissue and was greater than that in hypothyroid pituitary tissue, suggesting that processing may be physiologically regulated. Hypothyroid calf serum slowed TSH alpha-subunit processing in euthyroid pituitaries. Thus, the previously reported slower processing of Man9GlcNAc to Man8GlcNAc in heterodimers as compared to free alpha-subunits is in large part due to accumulation of Man9GlcNAc in TSH beta-subunits.
Question: Does the abstract contain any numerical data?
answer:
Yes

question:
Abstract: Head and neck cancer has rarely been reported to be a cause of meningeal carcinomatosis. These tumors are known more for their local invasiveness rather than distant metastasis. This would appear to preclude meningeal involvement, but close proximity to multiple cranial nerves may provide an access to the meninges. Six cases of head and neck cancer with meningeal invasion are presented. All six cases had malignant cells in their cerebrospinal fluid. Three cases had malignant cells recovered from a ventricular specimen after lumbar punctures were negative. The most common clinical finding on presentation was cranial nerve involvement. The optic nerve was involved most often with nerves VI and V the next most frequent. Headache was present in four patients and seizures occurred in two. No patient had meningismus. Our current treatment plan involves insertion of an Ommaya Reservoir and intraventricular methotrexate. Only patients whose primary head and neck tumor shows a response to systemic therapy undergo Ommaya placement. Meningeal carcinomatosis in head and neck cancer may be more prevalent than previously thought and the likely mechanism is via direct extension rather than hematogenous spread.
Question: Does the abstract contain any numerical data?
answer:
No