Generate an explanation for the given claim using the provided supporting material from the paragraph. Please consider the following points while generating an output. 1) The claim will always have supporting proof in the paragraph, and the paragraph will have a clear point of view supporting the claim. 2) There can be instances where direct correlation to the claim is not available. Make use of the sentences in the paragraph to provide a corresponding output supporting the claim. 3) The explanation should be at least one and at most twelve sentences long.

[Q]: Paragraph: According to two studies published on Wednesday, DNA from these pre-modern humans may play a role in the appearance of hair and skin as well as the risk of certain diseases. Although Neanderthals became extinct 28,000 years ago in Europe, as much as one-fifth of their DNA has survived in human genomes due to interbreeding tens of thousands of years ago, one of the studies found, although any one individual has only about 2 percent of caveman DNA. "The 2 percent of your Neanderthal DNA might be different than my 2 percent of Neanderthal DNA, and it's found at different places in the genome," said geneticist Joshua Akey, who led one of the studies. Put it all together in a study of hundreds of people, and "you can recover a substantial proportion of the Neanderthal genome." Both studies confirmed earlier findings that the genomes of east Asians harbor more Neanderthal DNA than those of Europeans. This could be 21 percent more, according to an analysis by Akey and Benjamin Vernot, published online in the journal Science. Still, "more" is a relative term. According to the paper by geneticists at Harvard Medical School, published in Nature, about 1.4 percent of the genomes of Han Chinese in Beijing and south China, as well as Japanese in Tokyo come from Neanderthals, compared to 1.1 percent of the genomes of Europeans. Anthropologists expressed caution about the findings. Fewer than half a dozen Neanderthal fossils have yielded genetic material, said Erik Trinkaus of Washington University in St. Louis, one of the world's leading experts on early humans. Using this small sample to infer how much Neanderthal DNA persists in today's genome is therefore questionable, he said. As expected, since Neanderthals never existed in Africa, Africans and those who trace their ancestry to that continent have almost no Neanderthal DNA, the Harvard team found. Human ancestors began migrating out of humanity's natal continent as early as 1 million years ago, paleoanthropologists infer from fossil evidence, and between 500,000 and 200,000 years ago evolved into the robust, large-browed Homo neanderthalensis in Western Europe. Ever since scientists extracted DNA from the remains of Neanderthals, they have known that people today carry snippets of cavemen genes, in the amounts of 2 percent to 3 percent. That clinched the case that Neanderthals and modern humans interbred, probably 40,000 to 80,000 years ago, soon after the latter arrived in Europe from Africa. The new studies add details about how much DNA and of what kind we inherited. "The story of early human evolution is captivating in itself, yet it also has far-reaching implications for understanding the organization of the modern human genome," Irene Eckstrand of the National Institutes of Health's National Institute of General Medical Sciences, which partially funded the research, said in a statement. "Every piece of this story that we uncover tells us more about our ancestors' genetic contributions to modern human health and disease." The Harvard team used a novel genetic algorithm to estimate the probability that a particular genetic variant arose from Neanderthals. Basically, they started with the genome of a 50,000-year-old Neanderthal, and determined whether pieces of it also appeared in some non-Africans but not in sub-Saharan Africans. The algorithm identified remnants of Neanderthal DNA, called alleles, in genes associated with type 2 diabetes, Crohn's disease, lupus, biliary cirrhosis and smoking behavior. "But we can't tell if the Neanderthal alleles are contributing to disease," said Harvard's Sriram Sankararaman, lead author of the paper in Nature. Areas "with increased Neanderthal ancestry tend to be higher the farther away you go from genes," sitting, instead, in regions of the genome that do not actually produce the proteins, enzymes and other working molecules of the body. Neanderthal DNA does sit within some genes, however, such as those for keratin, a fibrous protein that makes skin, hair and nails tough and can be beneficial in colder environments by providing thicker insulation. The endurance of this Neanderthal DNA suggests that our caveman inheritance was adaptive, picked by natural selection to persist in our genome, generation after generation, because it conferred a survival advantage in individuals who carried it. The Harvard team analyzed the genomes of 1,004 people to estimate which populations got more or fewer Neanderthal DNA. Confirming a 2013 study, they found more in the genomes of east Asians than in Europeans. But they also dug deeper. Puerto Ricans and Spaniards have the least Neanderthal DNA, 1.05 percent and 1.07 percent respectively. That is curious because Neanderthals made their last stand on the Iberian peninsula, finally dying out there 28,000 years ago. Across all populations, Neanderthal DNA is conspicuously low in regions of the X chromosome and in testes-specific genes. The paucity of Neanderthal genes among those active in the testes suggests that interbreeding wasn't a resounding success: It produced viable children, but the males were infertile. Although as much as 20 percent of Neanderthal DNA persists in modern genomes, according to the Washington scientists, vast regions of the latter are Neanderthal deserts. Among them is a region of chromosome 7 that includes a gene called FOXP2. "FOXP2 has previously been implicated in speech and language and may be an example of a gene that influences a uniquely human trait, which is why we find no Neanderthal sequences in these regions," said Akey. Claim: Modern humans more Neanderthal than once thought, studies suggest.
[A]: It's getting harder and harder to take umbrage if someone calls you a Neanderthal.

[Q]: Paragraph: Scientists at Vienna University medical school's Institute of Hygiene and Applied Immunology came to the conclusion after analyzing the water quality at 21 "holy" springs and 18 fonts at churches and chapels at various times of year. Only 14 percent of the water samples from holy sources showed no fecal contamination, and none of the springs could be recommended as a source of drinking water, the study presented to a conference in Vienna this week found. The springs held not only fecal contamination - likely the result of poor hygiene - but many also had agricultural nitrates and bugs that can cause inflammatory diarrhoea. "We need to warn people against drinking from these sources," microbiologist Alexander Kirschner said in the study. Kirschner said the healing effects ascribed to holy sources arose from the hygienic conditions of the Middle Ages, when water quality in urban areas was generally so poor that people constantly contracted diarrhoea or other conditions. "If they then came across a protected spring in the forest that was not as polluted and drank from it for several days, their symptoms would disappear. So although in those days they were drinking healthier water, given the excellent quality of our drinking water today, the situation is now completely reversed." The study noted some ways to help address the problem, including regularly replacing holy water in church fonts. One Italian priest has invented a holy water dispenser that dispenses drops of holy water rather than having the faithful dip a hand in, it noted. Claim: Holy water in Austria unsafe to drink: researchers.
[A]: Holy water at religious shrines and churches in Austria is often contaminated with fecal matter and bacteria, researchers have found, advising the faithful not to drink it, especially in hospital chapels.

[Q]: Paragraph: Almost three years after he received bone marrow stem cells from a donor with a rare genetic mutation that resists HIV infection - and more than 18 months after he came off antiretroviral drugs - highly sensitive tests still show no trace of the man's previous HIV infection. "There is no virus there that we can measure. We can't detect anything," said Ravindra Gupta, a professor and HIV biologist who co-led a team of doctors treating the man. AIDS experts said the case is a proof of the concept that scientists will one day be able to end AIDS, and marks a "critical moment" in the search for an HIV cure, but does not mean that cure has already been found. Gupta described his patient as "functionally cured" and "in remission", but cautioned: "It's too early to say he's cured." The man is being called "the London patient", in part because his case is similar to the first known case of a functional cure of HIV - in an American man, Timothy Brown, who became known as the Berlin patient when he underwent similar treatment in Germany in 2007 which also cleared his HIV. Brown, who had been living in Berlin, has since moved to the United States and, according to HIV experts, is still HIV-free. Some 37 million people worldwide are currently infected with HIV and the AIDS pandemic has killed about 35 million people worldwide since it began in the 1980s. Scientific research into the complex virus has in recent years led to the development of drug combinations that can keep it at bay in most patients. Gupta, now at Cambridge University, treated the London patient when he was working at University College London. The man had contracted HIV in 2003, Gupta said, and in 2012 was also diagnosed with a type of blood cancer called Hodgkin's Lymphoma. In 2016, when he was very sick with cancer, doctors decided to seek a transplant match for him. "This was really his last chance of survival," Gupta told Reuters. The donor - who was unrelated - had a genetic mutation known as 'CCR5 delta 32', which confers resistance to HIV. The transplant went relatively smoothly, Gupta said, but there were some side effects, including the patient suffering a period of "graft-versus-host" disease - a condition in which donor immune cells attack the recipient's immune cells. Most experts say it is inconceivable such treatments could be a way of curing all patients. The procedure is expensive, complex and risky. To do this in others, exact match donors would have to be found in the tiny proportion of people most of them of northern European descent who have the CCR5 mutation that makes them resistant to the virus. "Although this is not a viable large-scale strategy for a cure, it does represent a critical moment," said Anton Pozniak, president of the International AIDS Society. "The hope is that this will eventually lead to a safe, cost-effective and easy strategy...using gene technology or antibody techniques." Specialists said it is also not yet clear whether the CCR5 resistance is the only key - or whether the graft-versus-host disease may have been just as important. Both the Berlin and London patients had this complication, which may have played a role in the loss of HIV-infected cells, Gupta said. His team plans to use these findings to explore potential new HIV treatment strategies. "We need to understand if we could knock out this (CCR5) receptor in people with HIV, which may be possible with gene therapy," he said. The London patient, whose case was set to be reported in the journal Nature and presented at a medical conference in Seattle on Tuesday, has asked his medical team not to reveal his name, age, nationality or other details. Claim: London HIV patient becomes world's second AIDS cure hope.
[A]:
An HIV-positive man in Britain has become the second known adult worldwide to be cleared of the AIDS virus after he received a bone marrow transplant from an HIV-resistant donor, his doctors said.