Generate an explanation for the given claim using the provided supporting material from the paragraph. Please consider the following points while generating an output. 1) The claim will always have supporting proof in the paragraph, and the paragraph will have a clear point of view supporting the claim. 2) There can be instances where direct correlation to the claim is not available. Make use of the sentences in the paragraph to provide a corresponding output supporting the claim. 3) The explanation should be at least one and at most twelve sentences long.

Paragraph: In 2004, a virulent form of canine influenza surfaced at greyhound racing parks in Florida. In that outbreak, it infected 24 greyhounds and killed 8 more. The contagion has since been confirmed in seven states, having killed greyhounds at tracks in Florida, Massachusetts, Arizona, West Virginia, Wisconsin, Texas and Iowa. It is highly contagious, spreading through shared items (like toys) or human contact (kennel workers have carried the virus home with them), in addition to dog-to-dog encounters. The virus that has been felling greyhounds is an H3N8 flu closely related to an equine flu strain. It is not related to typical human flus or to the H5N1 avian flu that killed about 100 people in Asia. Several states including Florida, Minnesota, and Louisiana,among others reported another rash of cases in late June 2017. According to the American Veterinary Medical Association (AVMA), the outbreaks in those states were caused by a strain known as canine H3N2 influenza, which was first reported in Chicago in 2015. An AVMA spokesperson, Sharon Granskog, told us in a phone interview that such outbreaks can be seasonal in nature: There's a time of year when more dogs are in contact with one another, they're at dog parks, it's highly contagious. We suggest that you make sure if your dog is going to a dog park that it's healthy and talk to a veterinarian about whether or not a vaccine would be right for your dog. The disease was first reported in the U.S. in 2004 after several racing greyhounds in Florida contracted it. The AVMA said that they believed that the dogs became infected by a strain of H3N8 influenza, which is particular to horses. Dr. Ruben Donis, chief of molecular genetics for the influenza branch of the Centers for Disease Control and Prevention, confirmed that the flu jumped from horses to dogs, "a very rare event of considerable scientific interest" and added "at this point, there is no reason to panic." How that jump occurred is either not known at this point or is not being commented on, so the email's assertion that the eating of raw meat was to blame should be regarded as speculation rather than as fact. While there is always the possibility the virus might again jump species, this time to humans, this strain of flu has been present in horses for more than 40 years without any documented cases of humans catching it or the related canine flu. But there are vaccines available for both the equine and canine flus. Many readers have been confused (and unnecessarily frightened) by the difference between the terms "morbidity rate" and "mortality rate." The morbidity rate describes the percentage of animals that will contract the disease after being exposed to the virus, but, despite its name, the term has nothing to do with the death rate associated with the flu. (Nearly 80 percent of dogs exposed to the canine flu virus will contract only a mild form of the disease which mimics kennel cough, a type of canine bronchitis that is rarely serious.) The mortality rate, which describes the percentage of animals that will die after contracting the disease, is in the much lower range of 5 to 8 percent, according to Dr. Cynda Crawford, the veterinary immunologist who first isolated the canine flu virus: The mortality rate is around 5 to 8 percent, says veterinarian Cynda Crawford at the University of Florida's College of Veterinary Medicine in Gainesville. "I want to stress that despite the rumors that are out on the Internet and other such sources, this disease is not as deadly as people want to make it," Crawford said. She says she receives more than 30 calls a day from concerned veterinarians. Dr. Crawford describes the contagion as producing in dogs "a moist, productive cough that ends in a gagging response, that will persist for one to four weeks, despite treatment with antibiotics or cough suppressants. Some dogs develop a thick, yellow discharge from the nose. A very few dogs will spike a high fever, between 105 to 107 degrees Fahrenheit. They become lethargic and weak, with rapid, shallow breathing. This is likely to progress to pneumonia." Other veterinary experts have estimated the potential death rate as between 1 and 10 percent, with the higher percentage applying to very young, very old, or infirm dogs. Presence of the virus in dogs can be confirmed only through blood tests performed at the Cornell University College of Veterinary Medicine. Results of such blood screens take as long as two weeks. Dr. Crawford recommends keeping dogs showing symptoms of respiratory disease at home and away from other dogs for up to two weeks. The CDC, which is tracking the disease, issued no official recommendations. Because the symptoms of this as yet unnamed virus somewhat mimic bordetella, a less virulent illness commonly known as kennel cough, it is hard to ascertain how widespread the flu has become. On the flipside of that confusion, vets in various parts of the country have been thrown into a panic when encountering run-of-the-mill kennel cough in any of their clients, fearing they are instead confronting cases of the new flu. The Animal Health Diagnostic Center at Cornell University inserted a caution against such hair-trigger diagnosis within a larger advisory about the potential for the flu to have spread to the state of New York: "One should not lose sight of the fact that all respiratory infections in dogs are not due to canine influenza virus. Adhering to the 'band wagon' approach could result in the failure to appropriately treat dogs with infections previously known to cause respiratory problems in dogs." We found this good advice for vets and dog owners in our inbox one day: PLEASE DO NOT PANIC, and do NOT assume that every cough is Canine Influenza. Kennel Cough from parainfluenza and Bordetella is more common. However, the Animal Health Diagnostic Center at Cornell is interested in receiving samples from dogs that appear to have kennel cough. Clinical Signs: Since this is a new pathogen in dogs, there is currently no natural immunity present in the unexposed canine population. Almost all exposed dogs will become infected, and nearly 80% have clinical signs. In the mild form the dogs will have a cough that persists for 10 to 21 days. The cough may be soft and moist or dry. Many dogs will have a nasal discharge from a secondary bacterial infection and low grade fever. The nasal discharge responds to broad spectrum antibiotics. In the severe form with pneumonia there is a high fever (104-106 F) and respiratory difficulties. X-rays may show consolidation. These dogs often have secondary bacterial infections and have responded to broad-spectrum antibiotics and supportive care including intravenous hydration. The incubation period is two to five days and dogs may shed virus for seven to 10 days. The disease can spread rapidly throughout a boarding kennel. Dogs that are coughing SHOULD NOT BE BROUGHT TO SHOWS or Performance EVENTS. In a 1 June 2017 advisory to its members, the AVMA suggested that vaccines against the H3N2 virus strain "might be another prevention option" for dogs who were at least 8 weeks old and also recommended isolating dogs who could be infected, as well as not sharing toys between dogs and disinfecting shelter, veterinary, and boarding facilities. Claim: A virus deadly to dogs has been spreading in the canine population of the U.S.
Has a virus deadly to dogs been spreading in the canine population of the U.S.?

Paragraph: "Even though the majority of strokes and heart attacks occur in people who are 65 and older, studies have found that cholesterol-lowering drugs are not prescribed as often for older people as they are for younger people," Dr. Seemant Chaturvedi of Wayne State University in Detroit, Michigan, said in a statement. "These results show that using these drugs is just as beneficial for people who are over 65 as they are for younger people." In April, British researchers reported in the New England Journal of Medicine that treating high blood pressure in people 80 and older cut their risk of fatal strokes and other heart problems. Statins cut the risk of heart attack or stroke by lowering levels of fatty substances such as low-density lipoprotein, or "bad" cholesterol, and triglycerides. They also raise levels of high-density lipoprotein or "good" cholesterol. They include atorvastatin, made by Pfizer Inc under the brand name Lipitor; pravastatin or Pravachol, made by Bristol-Myers Squibb; fluvastatin, made by Novartis AG under the brand name Lescol, and several others. Seemant and colleagues studied 4,731 people 18 and older who had a recent stroke or a mini-stroke as part of a study sponsored by Pfizer. About half were over 65, with an average age of 72. The other half were under 65 and had an average age of 54. In each age group, about half were taking Lipitor and the other half were on a placebo. The statin worked equally well in both groups, the researchers reported in the journal Neurology. It lowered LDL cholesterol by about 61 points in the group of older patients and by about 59 points in the younger age group. Stroke is the No. 3 killer in the United States behind heart disease and cancer. The U.S. Centres for Disease Control and Prevention estimate 780,000 Americans will have strokes this year. They will kill 150,000 people and permanently disable 15 percent to 30 percent of survivors. Claim: Statins fight stroke in older people too.
Older people who have had a stroke or mini-stroke benefit from cholesterol-lowering statin drugs just as much as younger people do, U.S. researchers said on Wednesday.

Paragraph: "The costs of foods containing vitamin E is not really in question, so we'll call this one not applicable. The story comes a whisker away from earning a satisfactory on this one. It leads by noting that participants with the highest vitamin E intakes had a 25% lower risk of dementia compared with those who had the lowest intakes, It more precisely quantifies this benefit later in the story, where itexplainsthat120 people developed dementia in the group with the highest vitamin E intake compared with 164people in the low- intakegroup. What would have been most informative, however,is if the storyhad provided somedenominators for this comparison that is,120 out of how many people developed dementia in the first group compared with 164 out of how many people in the second? This would given readers a better sense of theabsolute risk reduction associated with higher vitamin E intake. We know this sets the bar high, but for good reason, we think. There are no known harms to eating a diet rich in foods containing vitamin E, but this story goes the extra mile and notes that high vitamin E intake from supplements can cause excessive bleeding. The story uses appropriate language and caveatswhen reportingon the results of this observational study. It notes that the findings""do not prove that vitamin E itself protects the aging brain,"" and there is no talk about vitamin E ""lowering the risk"" of dementia. The story also provides the key details regarding how the researchers conducted the study, includingthe number of participants; how vitamin E intake was assessed; how long the participants were followed; what confounding factors were included in the analysis; and how much vitamin E participants were getting in the group that had the lowest risk. Had the story sought out a commentfrom anexpert in nutritional epidemiology, they likely would havepointed out someproblems with trying to assess dietary intakefrom a single baseline questionnaireas was used in this study. They also would have provided additional context about the MANY other studies (including other large cohorts) that have looked at associations between antioxidants and dementia/ cognitive decline. Nevertheless, readers can't miss the take-home message, which is that these findings are not conclusive. There was no disease-mongering of dementia in this story. Although the story is generally well balanced, it could have benefited from the inclusion of an independentcomment on the findings. Valuable context about prior studies and the serious limitations of the current study would have beenhelpful. There is currently no proven method for preventing age-related dementia. We might have wished that this story had mentioned some other diet andlifestyle choices that areassociated with reduced risk in observational studies. These include staying physically active, not smoking, eating a Mediterranean-style diet (richinfruits and vegetables and omega-3 fatty acids), and keepingyour mind engaged in intellectually challenging pursuits. But since the evidence is kind of squishy for all of this, we rule this satisfactory. The story provides a list of foods that are rich in this micronutrient. The idea that vitamin E and other antioxidants might protect against dementia is not new, and the story doesn't suggest that it is. While the story doesn't appear to have lifted anything directly from the press release issued for this study, it also doesn'tseem to have interviewed anyone about the research. Since we can't be sure to what extent the story relied ona release, we'll call it not applicable." Claim: Higher vitamin E intake tied to lower dementia risk
While the story has afew notable deficiencies no independent comment and insufficient statisticaldetail regarding the magnitudeof theeffect readers will undoubtedly come away with the rightbottom linemessage about this study: Interesting, but inconclusive. Dementia will place agrowingburdenon individuals and our society as the population ages, yet there are currently no effectiveways to prevent or treat this condition. Instead of offering false hope about strategies that have little chance of making a difference, stories such as this one help give readers a realistic view of where they should they spend their time and energy when trying to reducehealth risks.