In this task, you're given a paragraph and title from the research paper. Your task is to classify whether the given title is suitable or not for the research paper based on the given paragraph. Return "True" if title is proper according to paragraph else "False".

Ex Input:
Paragraph: Community-acquired pneumonia is still a significant cause of morbidity and mortality and is often misdiagnosed and inappropriately treated. Although it can be caused by a wide variety of micro-organisms, the pneumococcus, atypicals, such as Mycoplasma pneumoniae and Chlamydophila pneumoniae, Staphylococcus aureus and certain Gram-negative rods are the usual pathogens encountered. The site-of-care decision is critical in determining the site and type of care as well as the extent of diagnostic workup. Antimicrobial therapy should be started as soon as possible particularly in those requiring admission to hospital, but typically the physician does not know with any degree of certainty the identity of the etiologic pathogen. A number of national guidelines have been published to help the physician with this choice. The initial drug(s) can be modified if necessary if the pathogen and its antimicrobial susceptibility pattern becomes known. Adjunctive therapy such as pressors and fluid replacement are of value and macrolides appear to help as well, likely secondary to their immunomodulatory effects. Recent data also suggest a role for steroids.
Title: Community-acquired pneumonia: An overview

Ex Output:
True

Ex Input:
Paragraph: Dendritic cells are potent antigen-capture and -presenting cells that play a key role in the initiation and regulation of the adaptive immune response. This process of immune homeostasis, as maintained by dendritic cells, is susceptible to dysregulation by certain pathogens during chronic infections. Such dysregulation may lead to disease perpetuation with potentially severe systemic consequences. Here we discuss in detail how intracellular pathogens exploit dendritic cells and evade degradation by altering or evading autophagy. This novel mechanism explains in part the chronic, persistent nature observed in several immuno-inflammatory diseases, including periodontal disease. Also, here we propose a hypothetical model on the plausible role of autophagy in the context of periodontal disease. Promotion of autophagy may open new therapeutic strategies in the search for a "cure" for periodontal disease in humans.
Title: Dendritic cells: microbial clearance via autophagy and potential immunobiological consequences for periodontal disease

Ex Output:
True

Ex Input:
Paragraph: Wild-type Middle East respiratory syndrome coronavirus (MERS-CoV) or I529T mutant MERS-CoV isolated from patients in South Korea (GenBank accession nos. KT029139.1 for wild type and KT868873.1 for I529T mutant) were cultured in a 24-well plate containing a monolayer of Vero E6 cells or 293T cells stably expressing CD26 (1). After 1 h incubation at 37C, viral supernatant was removed and cells were overlaid with 1 mL of 1% methylcellulose in Dulbecco modified Eagle medium, including 10% fetal bovine serum. Plates were incubated for 3 d at 37C, and then cells were fixed with 4% paraformaldehyde and 100% methanol. The MERS-CoV plaques were detected using rabbit anti-MERS-CoV N protein antibody (Sino Biologic Inc.) and goat anti-rabbit IgG secondary antibody conjugated with horseradish peroxidase (Invitrogen; https://www.thermofisher.com/us/en/home/brands/invitrogen.html). Viral plaques were visualized by incubation with 0.05% 33-diaminobenzidine tetrahydrochloride and 0.01% hydrogen peroxide in 50 mmol/L Tris-HCl (pH 8.0). Cellular layers were counterstained with trypan blue dye. Pseudotyped lentiviruses with wild type or mutant spikes of MERS-CoV were generated from 293T cells (Invitrogen) by cotransfection of human immunodeficiency virus backbone plasmids expressing firefly luciferase as previously described (1). We used the packaging plasmids, pLP1, pLP2, and pLP/VSV-G (Invitrogen) and pLVX-Luc-IRES-ZsGreen1 (Clontech; https://www.takarabio.com). For spike protein pseudotyping, condon-optimized cDNA of the
Title: Sequential Emergence and Wide Spread of Neutralization Escape Middle East Respiratory Syndrome Coronavirus Mutants, South Korea, 2015 Appendix Materials and Methods Middle East Respiratory Syndrome Coronavirus Culture and Plaque Assay

Ex Output:
True