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This topic will review the treatment of Wilson disease. The epidemiology, pathogenesis, clinical manifestations, and diagnosis of Wilson disease are discussed separately. (See Wilson disease: Epidemiology and pathogenesis and Wilson disease: Clinical manifestations, diagnosis, and natural history and Wilson disease: Diagnostic tests.) Serum concentration of copper and of non-ceruloplasmin-bound copper. Most individuals with Wilson disease have a subnormal serum copper concentration that is proportional to the serum ceruloplasmin concentration. Note: Serum copper is low in healthy newborns. But in people with Wilson's disease, copper isn't eliminated properly and instead accumulates, possibly to a life-threatening level. When diagnosed early, Wilson's disease is treatable, and many people with the disorder live normal lives. Wilson's disease care at Mayo Clinic Molecular genetic studies that use DNA from blood cells to search for patterns of differences or similarities, a procedure called haplotype analysis may establish whether a full sibling of an affected patient has Wilson disease, is a carrier of the Wilson disease gene, or is not a carrier. With Wilson disease, the copper builds up in your liver, and it releases the copper directly into your bloodstream. This can cause damage to your brain, kidneys, and eyes. Wilson disease is present at birth, but symptoms usually start between ages 5 and 35. It first attacks the liver, the central nervous system or both. The most characteristic sign is a rusty brown ring around the cornea of the eye. A physical exam and laboratory tests can diagnose it. Treatment is with drugs to remove the extra copper from your body. Wilson disease is caused by mutations in the ATP7B gene. This gene provides instructions for making a protein called copper-transporting ATPase 2, which plays a role in the transport of copper from the liver to other parts of the body. A person's chances of having Wilson disease increase if a health care provider has diagnosed one or both parents with the condition. What are the signs and symptoms of Wilson disease? The signs and symptoms of Wilson disease vary, depending on what organs of the body are affected. Wilson disease is present at birth; however, the signs and symptoms of the disease do not appear until the copper builds up in the liver, the brain, or other organs. When people have signs and symptoms, they usually affect the liver, the central nervous system, or both. Points to Remember. 1 Wilson disease prevents the body from getting rid of extra copper. 2 Wilson disease first attacks the liver, the central nervous system, or both. 3 Anyone with unexplained liver disease or neurologic symptoms with evidence of liver disease should be screened for Wilson disease. WD, Wilson disease. Fromthe1DivisionofGastroenterology,HepatologyandNutrition,TheHospital for Sick Children, and Departments of Paediatrics, Medicine and Pharmacology, University of Toronto, Toronto, Ontario, Canada, and 2Department of Medicine. and Surgery, Division of Digestive Diseases, Section of Transplant and Immunol-. ogy, Yale University Medical Center, New Haven, CT. Copyright ©️ 2008 by the American Association for the Study of Liver Diseases. Either the liver or the brain can be harmed first, with signs as early as 4 years, or as late as 70 years of age. 1 Symptoms of liver disease include: 2 Jaundice, which is when the skin or the white part of the eye turns yellow. 3 Fatigue. 4 Loss of appetite. 5 Swelling in the abdomen. 6 Easy bruising. Query: how is wilson's disease treated
Output:
Wilson disease is treated with drugs to remove the extra copper from your body.