Given an abstract, generate a keyword (a noun phrase) that best describes the focus or contribution of the paper. Such keywords can be directly from the given abstract or outside it.

Ex Input:
Abstract: The objective of this study was to find naturally occurring anti-idiotypic (anti-Id) antibodies to anti-human thyroglobulin (anti-hTg) idiotype in sera of patients with autoimmune thyroid disease. Sera from patients with Hashimoto's thyroiditis (HT), Graves' disease (GD), rheumatoid arthritis (RA), and systemic lupus erythematosus (SLE) and sera from normal subjects were tested for the presence of anti-Id antibodies against mouse anti-hTg monoclonal antibodies (McAb) in indirect ELISA and in indirect solid-phase RIA. Microtitration plates were coated with six McAb, five of them directed against different epitopes on hTg molecule, and then incubated with patients' sera. The bound antibody was detected with either peroxidase or 125I-labeled anti-human IgG. The specific positive reaction was observed in four of 40 patients with HT, in two of 26 patients with GD, in seven of 58 patients with RA, and in none of 20 normal subjects. The detected binding was due to the presence of anti-hTg anti-Id antibodies and not to Tg-anti-Tg circulating immune complexes, as the positive sera did not contain hTg when resolved on SDS-PAGE, nor did they bind to all anti-hTg McAb tested. The binding was dose dependent, and titers of anti-Id antibodies varied from 1:243 to 1:2187. The binding could be inhibited up to 50% by hTg, but not by the thyroid microsomal antigen, indicating that some of those anti-Id might represent the internal image of the antigen. Serum from the patient 3403, showing the strongest reactivity against McAb A-3, was chosen for IgG purification and F(ab')2 fragment isolation. The 3403 F(ab')2 fragment, but not the Fc fragment, was found to react specifically with four mouse anti-hTg McAb but not with the control mouse IgG. Thus, the obtained results permit the conclusion that anti-hTg anti-Id antibodies could occur naturally during the course of thyroid autoimmune disorders.

Ex Output:
Antibodies, Monoclonal

Ex Input:
Abstract: Seventy-six white and black low-income families were interviewed weekly during 1 month to determine how much they spent on food using Food Stamps, WIC coupons, and cash and how much additional food they obtained from different food and meals programs. After families had received their public assistance and Food Stamp allotments for the month, a 24-hour recall was obtained each week from each family member so that food intake could be monitored from the recalls as food-buying resources were depleted. Although families bought most of their food the first 2 weeks, they maintained a relatively constant food intake through the last week of the month. However, when nutrient intakes were compared with Recommended Dietary Allowances, the diets were found to be well supplied in protein, ascorbic acid, thiamin, niacin, riboflavin, vitamin B-12, vitamin A, and phosphorus, and inadequately supplied in vitamin B-6, vitamin D, vitamin E, iron, calcium, magnesium, zinc, and pantothenic acid. Since that finding was true not just at the end of the month but from the beginning, it indicated that the nutrition problems of low-income families are not intermittent, but continuous.

Ex Output:
Adolescence

Ex Input:
Abstract: Scintigraphy with radiolabeled metaiodobenzylguanidine was performed in a patient with MEN Type IIa having a pheochromocytoma of the right adrenal gland, adrenomedullary hyperplasia of the left adrenal gland and a primary medullary thyroid carcinoma. The scintigraphic findings demonstrate visualization of all the above mentioned pathologies.

Ex Output:
Adrenal Gland Hyperfunction