Indicate the presence of numerical data in the abstract (whether numerical data plays a key role in medical records), solely using the information from the abstract.

[EX Q]: Abstract: We have induced a basophil hypersensitivity reaction in the upper tarsal conjunctiva of the guinea pig by methods that induce a comparable basophil hypersensitivity reaction in the flank. The inflammatory cell infiltrate in this reaction contained large numbers of basophils and eosinophils with accompanying neutrophils and monocytes. Ocular tissue can serve as a priming site for systemic immunization and also for elicitation of a secondary flare after challenge with antigen. Very few inflammatory cells were observed in the cutaneous epithelium of either primary or secondary flares. In contrast, the mucosal stroma and epithelium contained large numbers of inflammatory cells (basophils, eosinophils, and neutrophils), suggesting directed cellular movement onto the ocular surface. The lesion of ocular basophil hypersensitivity in the guinea pig has features in common with two human eye diseases, vernal conjunctivitis and contact lens-associated giant papillary conjunctivitis. We hypothesize that the acute basophil hypersensitivity reactions of the conjunctiva are transformed into chronic inflammatory and proliferative states in vernal conjunctivitis and giant papillary conjunctivitis.
Question: Does the abstract contain any numerical data?
[EX A]: No

[EX Q]: Abstract: We observed quinidine-induced prolongation of bleeding time without thrombocytopenia in three subjects. In addition, we noticed a cumulative prolongation of bleeding time by a combination of quinidine and aspirin. We postulated that because both quinidine and aspirin inhibit epinephrine-induced platelet aggregation, a cumulative effect of the two drugs might be responsible for the hemostatic defect. In studies using normal human platelets, we confirmed a marked reduction in epinephrine-induced platelet aggregation by the combination of these two agents. To further study the potential mechanism of this cumulative effect, platelet lysates were incubated with the alpha 2-adrenoceptor antagonist tritiated yohimbine in the presence of quinidine and aspirin. On the basis of the radioligand binding data, the dissociation constant (KD) of alpha 2-adrenoceptors was observed to increase in the presence of quinidine as well as aspirin. The combination of these two agents caused a marked increase in the KD of platelet alpha 2-adrenoceptors without alteration in the number of receptor sites. These data suggest that the cumulative effects of quinidine and aspirin on platelet alpha 2-adrenoceptor KD may relate to the significant reduction in epinephrine-induced platelet aggregation. This phenomenon, coupled with other well-known effects of aspirin on the platelet release reaction and arachidonate metabolism, may lead to bleeding problems in some patients receiving this combination.
Question: Does the abstract contain any numerical data?
[EX A]: Yes

[EX Q]: Abstract: A retrospective study of patients patch tested between 1968 and 1970 was undertaken and case records of 200 patients with unequivocal positive patch tests were studied for age and sex distribution. There was an overall female preponderance (61%), with 2 peaks of incidence in the 10-20 and 40-50 year age groups in females, but only 1 peak of incidence in the 40-50 year age group in males. There was a steady decline in all sensitivities after the 6th decade of life in both sexes. Nickel sensitivity was more common in females (92%), whereas dichromate sensitivity predominated in males (64%). Of the 200 patients, only 5(2.5%) were atopics, all of whom were females.
Question: Does the abstract contain any numerical data?
[EX A]:
Yes