Given a question and a context passage, generate the answer having the word or phrase from the context passage. Here, the answer should be a shortest continous span from the passage.

[Q]: Context: Mutations in the solute carrier family 9, subfamily A member 6 (SLC9A6) gene, encoding the endosomal Na+/H+ exchanger 6 (NHE6) are associated with Christianson syndrome, a syndromic form of X-linked intellectual disability characterized by microcephaly, severe global developmental delay, autistic behavior, early onset seizures and ataxia. In a 7-year-old boy with characteristic clinical and neuroimaging features of Christianson syndrome and epileptic encephalopathy with continuous spikes and waves during sleep, we identified a novel splice site mutation (IVS10-1G>A) in SLC9A6. These findings expand the clinical spectrum of the syndrome and indicate NHE6 dysfunction as a new cause of electrical status epilepticus during slow-wave sleep (ESES).
Question: Mutation of which gene is implicated in the Christianson syndrome?
[A]: slc9a6

[Q]: Context: We have dreams almost every night. Do you ever notice the colors of your dreams? Do you dream in black and white or do you dream in yellow, red and green? New research suggests that the type of television you watched as a child has a great effect on the color of your dreams. While almost all people under 25 dream in color, thousands of people over 55, all of whom were brought up with black and white TV sets, often dream in monochrome . "It suggests there could be a critical period in our childhood when watching films has a big impact on the way dreams are formed", said Eva Murzyn, a psychology student at Dundee University in Britain who carried out the study. Research from 1915 through the 1950s suggested that the vast majority of dreams are in black and white. But the tide turned in the sixties, and later results suggested that up to 83 percent of dreams contain some color. Since this period also marked the transition between black-and-white film and TV and Technicolor ,an obvious explanation was that the media had been painting people's dreams. However, there weren't any firm conclusions. But now Miss Murzyn believes she has proven the link. She made a survey of more than 60 people, half of whom were over 55 and half of whom were under 25. She asked the volunteers to answer a questionnaire on the color of their dreams and their childhood exposure to film and TV. She then analyzed her own data. Only 4.4 percent of the under-25s' dreams were black and white. The over-55s who had had access to color TV and film during their childhood also reported a very low proportion of just 7.3 percent. But the over-55s who only had access to black-and -white media reported dreaming in black and white about a quarter of the time. Even though they would have spent only a few hours a day watching TV or films, their attention and emotion would have been heightened during this time, leaving a deeper imprint on their mind, Miss Murzyn told the New Scientist. "The crucial time is between three and ten when we all begin to have the ability to dream", she said.
Question: In which magazine can you find the article?
[A]: new scientist

[Q]: Context: Diamond-Blackfan anemia (DBA) is a rare inherited bone marrow failure syndrome that is characterized by pure red-cell aplasia and associated physical deformities. It has been proven that defects of ribosomal proteins can lead to this disease and that RPS19 is the most frequently mutated gene in DBA patients. Previous studies suggest that p53-dependent genes and pathways play important roles in RPS19-deficient embryos. However, whether there are other vital factors linked to DBA has not been fully clarified. In this study, we compared the whole genome RNA-Seq data of zebrafish embryos injected with RPS19 morpholino (RPS19 MO), RPS19 and p53 morpholino simultaneously (RPS19+p53 MO) and control morpholino (control). We found that genes enriched in the functions of hematological systems, nervous system development and skeletal and muscular disorders had significant differential expression in RPS19 MO embryos compared with controls. Co-inhibition of p53 partially alleviates the abnormalities for RPS19-deficient embryos. However, the hematopoietic genes, which were down-regulated significantly in RPS19 MO embryos, were not completely recovered by the co-inhibition of p53. Furthermore, we identified the genome-wide p53-dependent and -independent genes and pathways. These results indicate that not only p53 family members but also other factors have important impacts on RPS19-deficient embryos. The detection of potential pathogenic genes and pathways provides us a new paradigm for future research on DBA, which is a systematic and complex hereditary disease.
Question: In which syndrome is the RPS19 gene most frequently mutated?
[A]:
dba