Given an abstract, indicate which of the provided candidate titles (Option1 and Option2) best describes it (respond with the text of the title, not option1/2).

[EX Q]: Abstract: We did complete histologic study on 148 gravid tubes and found that nearly 90% showed chronic lesions. CS was the most frequent lesion encountered (95.5%), followed by SIN (32.6%). Most frequently the lesions were extensive and both the ampulla and isthmus were involved. We therefore question the conservative approach in the treatment of certain TPs.
Which is the apt title for the abstract? option1: Tubal causes of ectopic pregnancy. option2: AAMC longitudinal study of 1960 medical school graduates: a 20-year effort in 28 schools, 1956-1976 [editorial]
[EX A]: Tubal causes of ectopic pregnancy.

[EX Q]: Abstract: Thrombospondin with fibrinogen, fibronectin, and von Willebrand factor binds to platelets stimulated with agonists and support platelet adhesive functions. The receptors for the latter three proteins are associated with membrane glycoprotein GPIIb-IIIa. Thrombasthenic platelets deficient in GPIIb-IIIa have been utilized to examine the role of this membrane protein in the interactions of thrombospondin with platelets. Radioiodinated thrombospondin bound to thrombin-stimulated platelets from normal and thrombasthenic donors with a similar affinity and capacity. As monitored with a monoclonal antibody to thrombospondin, the divalent ion-dependent and -independent pathways for the expression of the endogenous pool of thrombospondin on the surface of thrombin-stimulated platelets from normal and thrombasthenic donors were also qualitatively and quantitatively similar. GPIIb-IIIa or ligands associated with GPIIb-IIIa thus are not essential for the binding of thrombospondin to platelets. Therefore, thrombospondin interacts with unique receptors on platelets.
Which is the apt title for the abstract? option1: Identification of a new class of inducible receptors on platelets. Thrombospondin interacts with platelets via a GPIIb-IIIa-independent mechanism. option2: Vacuolar encephalopathy of AIDS [letter]
[EX A]: Identification of a new class of inducible receptors on platelets. Thrombospondin interacts with platelets via a GPIIb-IIIa-independent mechanism.

[EX Q]: Abstract: Deformities are abnormalities of shape or form caused by abnormal or imbalanced mechanical forces. In prenatal deformation, most infants are intrinsically normal and the deformation results from extrinsic constraining forces. Most prenatal deformities will resolve spontaneously or with the application of counteracting forces. In contrast, postnatal deformities result from either extrinsic pathologic processes, affecting the bones, nervous system, or muscle and resulting in the application of abnormal forces to bone, or normal forces acting on intrinsic diseases of bone. While many postnatal deformities can be contained or corrected by the application of counteracting forces, aggressive physical therapy, casting, or surgery is often required for correction.
Which is the apt title for the abstract? option1: The origin of prenatal and postnatal deformities. option2: A concept of illness tested as an improved basis for surgical decisions in low-back disorders.
[EX A]:
The origin of prenatal and postnatal deformities.