Cyanide poisoning

Cyanide poisoning be poisoning wey dey result from exposure to any of a number of forms of cyanide. Early symptoms dey include headache, dizziness, fast heart rate, shortness of breath, den vomiting. Dis phase then fi be followed by seizures, slow heart rate, low blood pressure, loss of consciousness, den cardiac arrest. Onset of symptoms usually dey occur within a few minutes. Sam survivors get long-term neurological problems.

Toxic cyanide-containing compounds dey include hydrogen cyanide gas den cyanide salts, such as potassium cyanide. Poisoning relatively be common dey follow breathing in smoke from a house fire. Oda potential routes of exposure dey include workplaces involved insyd metal polishing, certain insecticides, de medication sodium nitroprusside, den certain seeds such as those of apples den apricots. Liquid forms of cyanide fi be absorbed thru de skin. Cyanide ions interfere plus cellular respiration, wey dey result in de body ein tissues be unable to use oxygen.

Diagnosis often be difficult. E fi be suspected insyd a person following a house fire wey na e get a decreased level of consciousness, low blood pressure, anaa high lactic acid. Blood levels of cyanide fi be measured buh take time. Levels of 0.5–1 mg/L be mild, 1–2 mg/L be moderate, 2–3 mg/L be severe, den greater dan 3 mg/L generally result in death.

If exposure be suspected, dem for remove de person from de source of de exposure den decontaminated. Treatment dey involve supportive care den giving de person 100% oxygen. Hydroxocobalamin (vitamin B12a) dey appear to be useful as an antidote wey be generally first-line. Dem sanso fi give sodium thiosulfate. Historically, na dem dey use cyanide for mass suicide wey na dem use am for genocide by de Nazis.Longerich 2010, pp. 281–282.

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Category:Cyanides
Category:Neurotoxins
Category:Translated from MDWiki