natgillin/translations-raw huggingface dataset:
Ved den mere følsomme SPME-GC/MS screening af samtlige produkter blev der registreret forekomst af butanon i produkter nr. 8 og 21, men uden målte koncentrationer. 
The more sensitive SPME-GC/MS screening of all products registered the occurrence of butanone in product no. 8 and 21, but without measured concentrations.
Ved de kvantitative analyser, blev der ikke fundet butanon i mængder over detektionsgrænsen i nogen af produkterne, heller ikke produkter nr. 8 og 21. 
In connection with the quantitative analyses, butanone was not found in amounts exceeding the detection limit in any of the products - and not in product no. 8 and 21.
8.2.6 Konklusion på butanon i tekstilimprægneringsspray 
8.2.6 Conclusion on butanone in textile proofing sprays
Der er identificeret butanon i produkterne nr. 8 og 21. 
Butanone has been identified in product no. 8 and 21.
Men i de kvantitative analyser af sprayprodukterne er butanon ikke fundet i mængder over detektionsgrænsen på 0,02 mg/g. 
However, in the quantitative analyses of spray products, butanone was not found in amounts exceeding the detection limit of 0.02 mg/g.
Butanon udgør derfor ikke i sig selv nogen sundhedsrisiko for forbrugerne i de undersøgte sprayprodukter til tekstilimprægnering. 
Therefore, butanone is not in itself hazardous to health for consumers in the investigated spray products for textile proofing.
Butan-1-ol bruges som opløsningsmiddel i farve- og lakindustrien, ved fremstilling af naturlige og syntetiske harpikser, vegetabilske olier, farvestoffer og alkaloider. 
1-Butanol is used as solvent in the dyestuff and the varnish industry when making natural and synthetic resins, vegetable oils, dyes and alkaloids.
Det bruges som mellemprodukt ved fremstilling af medicin og kemikalier og anvendes i industrier, der fremstiller kunstlæder, tekstiler, gummiklæbemidler, fotografiske film og parfumer (HSDB, 2007). 
It is used as an intermediate when making medicine and chemicals and is used in industries that make artificial leather, textiles, rubber adhesives, photographic film and perfume (HSDB, 2007).
Butan-1-ol er optaget på arbejdstilsynets liste over organiske opløsningsmidler. 
1-Butanol is included on the list of organic solvents of the Danish Working Environment Authority.
Butan-1-ol er en klar farveløs væske med en stærk karakteristisk (harsk og sød) svagt alkoholisk lugt. 
1-Butanol is a clear colourless liquid with a very characteristic (rancid and sweet) faint smell of alcohol.
Stoffet er noget opløseligt i vand, blandbart med ethanol og ether og meget letopløseligt i acetone. 
The substance is rather soluble is water, miscible with ethanol and ether and very easily soluble in acetone.
Opløseligheden i benzen er over 10 % (HSDB, 2007). 
The solubility in benzene exceeds 10 % (HSDB, 2007).
i vand 7,1 mg/L; i luft 0,83 ppm (HSDB, 2007) 
In water 7.1 mg/L; in air 0.83 ppm (HSDB, 2007)
Butan-1-ol optages i kroppen via lungerne, mavetarm-kanalen og huden. 
1-Butanol is absorbed in the body via the lungs, the gastrointestinal tract and the skin.
Optaget stof fordeles hurtigt ud i vævene, hvor der sker en ret omfattende omdannelse af stoffet. 
Absorbed substance is quickly distributed to the tissue where the substance is transformed considerably.
Størstedelen af optaget stof udskilles i form af CO2 via lungerne; men kun en mindre del udskilles via nyrerne (HSDB, 2007). 
The main part of absorbed substance is eliminated as CO2 via the lungs; but only a minor part is eliminated via the kidneys (HSDB, 2007).
Høje koncentrationer i luften forårsager hæmning af centralnervesystemet (søvninghed, hovedpine, muskelsvaghed, svimmelhed, stivhed, konfusion, delirium, koma) (HSDB, 2007; IPCS, 1992). 
High concentrations in the air cause inhibition of CNS (tiredness, headache, muscular weakness, dizziness, stiffness, confusion, delirium, coma) (HSDB, 2007; IPCS, 1992).
Der kan desuden være gastro-intestinale effekter som kvalme, opkastning og diarré. 
In addition, there might be gastrointestinal effects such as nausea, vomiting and diarrhea.
Eventuelt dødeligt forløbende forgiftning vil være på grund af åndedrætssvigt (HSDB, 2007). 
Possible lethal toxification would be due to respiratory failure (HSDB, 2007).
Butan-1-ol virker stærkt irriterende på slimhinder. 
1-Butanol is very irritating on the mucous membrane.
Der er fundet irritation af hud, øjne og hals ved udsættelse for væsken og dampe heraf. 
Irritation of skin, eyes and neck has been observed during exposure to the liquid and vapours.
De orale LD50 værdier for butan-1-ol i rotter varierer mellem 700 og 2100 mg/kg lgv. 
The oral LD50 values of 1-Butanol in rats vary between 700 mg and 2100 mg/kg bw.
De primære effekter af eksponering for dampene i kortere tid består i forskellige grader af irritation af slimhinder og hæmning af centralnervesystemet. 
The main effects from exposure to the vapour for a shorter time consist of different degrees of irritation of the mucous membrane and inhibition of CNS.
Det anføres fra flere kilder, at det anses for ca. 6 gange så toksisk som ethanol (IPCS, 1987). 
Several sources state that it is believed to be approx. 6 times as toxic as ethanol (IPCS, 1987).
Stoffet virker udpræget irriterende ved testning med væsken i øjne og moderat irriterende på hud (HSDB, 2007). 
The substance seems distinctly irritating during testing with liquid in the eyes and moderately irritating on the skin (HSDB, 2007).
Der er ikke foretaget testning af det hudsensibiliserende potentiale af butan-1-ol (IUCLID (ECB, 2007)). 
The skin sensitizing potential of 1-Butanol (IUCLID (ECB, 2007)) has not been tested.
8.3.4.4 Subkroniske effekter, dyr 
8.3.4.4 Subchronic effects, animals
Virkningerne af gentagne inhalationer omfatter patologiske forandringer i lungevæv og degenerative læsioner i lever og nyrer (IPCS, 1987). 
The effect of repeated inhalation comprises pathological changes in lung tissue and degenerative injuries in liver and kidneys (IPCS, 1987).
Dette er fundet i en række inhalationsstudier med gnavere med forskellige doseringer (fra 0,03 til ca. 40 ppm) og opsætninger varierende fra dosering i afmålte timer/dag i et vist antal dage til kontinuert eksponering i 30 dage, 4 måneder eller 92 dage (IUCLID). 
That was found in a number of inhalation studies carried out on rodents with different dosages (from 0.03 to approx. 40 ppm) and set ups varied from dosage in measured hours/day in a certain number of days to continuous exposure for 30 days, 4 months or 92 days (IUCLID).
Dyrestudierne der er til rådighed, er ikke egnede til at bestemme et no observed adverse effect level (NOAEL) til brug i risikobedømmelser. 
The available animal studies are not suited for determining a no observed adverse effect level (NOAEL) to be used in risk assessments.
Et inhalationsstudie med eksponering af drægtige hunrotter fra dag 1 til dag 19 i drægtighedsperioder 7 timer daglig med 3500, 6000 eller 8000 ppm fandt en NOAEC for mødrene på 3500, men en let stigning i antallet af rudimentære halshvirvler hos afkommet i det højst doserede hold, hvorfor NOAEC for udvikling/teratogenicitet blev 6000 ppm (svarende til 18000 mg/m³) (IUCLID fra (ECB, 2007)). 
An inhalation study with exposure of pregnant female rats from day 1 to day 19 during gestation periods 7 hours a day with 3500, 6000 or 8000 ppm, revealed a NOAEC in the dam of 3500, but there was a minor increase in the number of rudimentary cervical vertebra in the offspring in the highest dosed group, and therefore NOAEC for development/teratogenecity was 6000 ppm (corresponding to 18000 mg/m³) (IUCLID from (ECB, 2007)).
Der er ikke fundet tegn på mutagene eller genotoksiske egenskaber i butan-1-ol i en lang række korttidsstudier især in vitro. 
A wide range of short-term studies especially in vitro, showed no signs of mutagenic or genotoxic properties in 1-Butanol.
I såvel Environmental Health Criteria nr. 65: Butanols - four isomers, 1987, som i IUCLID fra (ECB, 2007) henvises til at der skulle findes 2 langtidsstudier af meget ringe kvalitet, men det er ikke muligt at finde yderligere referencer til disse studier. 
Environmental Health Criteria no. 65: Butanols - four isomers, 1987, as well as IUCLID from (ECB, 2007) refer to the fact that 2 long-term studies of very poor quality are supposed to exist, but it has not been possible to find further reference to these studies.
Der findes ingen studier med nogen administrationsvej, der gør det muligt at bedømme de kroniske effekter, for slet ikke at tale om det kræftfremkaldende potentiale af butan-1-ol i mennesker. 
No studies exist with a route of administration that makes it possible to evaluate the chronic effects - not to mention the carcinogenic potential of 1-Butanol in humans.
IARC har ikke vurderet butan-1-ol mht. carcinogenicitet i dyr eller mennesker. 
IARC has not assessed 1-Butanol with regard to carcinogenicity in animals or humans.
Butan-1-ol er en antændelig farveløs væske, der bruges som organisk opløsningsmiddel i mange industrielle sammenhænge. 
1-Butanol is an ignitable colourless liquid that is used as organic solvent in many industrial connections.
Det har lav akut giftighed uanset eksponeringsmåden. 
It has a low acute toxicity regardless of the exposure method.
Stoffet absorberes let med indåndingsluften, efter indtagelse eller via huden, og fordeles meget hurtigt ligeligt til alle væv. 
The substance is easily absorbed with the inhaled air, after intake or via the skin and it is distributed very quickly and evenly to all tissue.
Høje koncentrationer med indåndingsluften fremkalder tegn på hæmning af centralnervesystemet med døsighed, hovedpine (i mennesker) og svimmelhed i såvel dyr som mennesker. 
High concentrations with the inhaled air induce signs of inhibition of CNS such as drowsiness, headache (in humans) and dizziness in animals as well as in humans.
Ved gentagen dosering via inhalation ses i dyr patologiske forandringer i lungevævet og degenerative forandringer i lever og nyrer, og der fremkaldes til stadighed narkose. 
Pathological changes in the lung tissue and degenerative changes in liver and kidneys appear in animals after repeated dosage via inhalation and anaesthesia is constantly developed.
En anden fremherskende effekt af butan-1-ol er hud- og især slimhindeirritation, så irritation af øjne, næse og svælg er effekter, der registreres ved lave eksponeringer. 
Another predominating effect of 1-Butanol is skin and especially mucous membrane irritation, so irritation of eyes, nose and throat are effects that are registered at low exposures.
Der er ikke testet for sensibiliserende potentiale. 
Sensitizing potential tests have not been carried out.
Der er ikke fundet troværdige langtidsstudier i nogen dyreart, men stoffet har vist sig ikke mutagent ved omfattende testning in vitro. 
No trustworthy long-term studies have been found in any species of animal but the substance has proved to be non-mutagenic after substantial testing in vitro.
Der er fundet lav forekomst af udviklingsforstyrrelse ved doser, hvor der også er set toksisk påvirkning af moderdyrene i et udviklings/teratogenforsøg. 
A minor occurrence of developmental disturbance was found at doses where toxic effect on the dam also was observed in a development/teratogenic test.
En af de meget følsomme effekter er øjenirritationen ved udsættelse for dampe af butan-1-ol. 
One of the very sensitive effects is eye irritation on exposure to vapour from 1-Butanol.
Effektniveauet er her 153,9 mg/m³ svarende til 50 ppm i mennesker. 
In that connection, the effect level is 153.9 mg/m³ corresponding to 50 ppm in humans.
8.3.5 Sundhedsvurdering af butan-1-ol 
8.3.5 Health assessment of 1-Butanol
Der blev ikke fundet resultater for indhold af butan-1-ol ved den semikvantitative screening af samtlige produkter til tekstilimprægnering. 
No results were found for content of 1-Butanol during the semi-quantitative screening of all products for textile proofing.
Ved den mere følsomme SPME-GC/MS screening af samtlige produkter blev der registreret forekomst af butan-1-ol i produkter nr. 18, 20, 25 og 26. 
The more sensitive SPME-GC/MS screening of all products registered the occurrence of 1-Butanol in product no. 18, 20, 25 and 26.
Ved de kvantitative analyser, blev der ikke fundet butan-1-ol i mængder over detektionsgrænsen i nogen produkter, heller ikke produkter nr.18, 25 og 26, der blev analyseret kvantitativt. 
The quantitative analyses did not show 1-Butanol in amounts exceeding the detection limit in any product - and not in product no.18, 25 and 26 that were analysed quantitatively.
8.3.6 Konklusion på butan-1-ol i tekstilimprægneringsspray 
8.3.6 Conclusion on 1-Butanol in textile proofing spray
Butan-1-ol er ikke fundet i mængder over detektionsgrænsen (0,2 mg/g) i de kvantitative analyser i nogen spray. 
1-Butanol was not found in amounts exceeding the detection limit (0.2 mg/g) in the quantitative analyses in any spray.
Butan-1-ol i de undersøgte sprayprodukter til tekstilimprægnering udgør derfor ikke i sig selv nogen sundhedsrisiko for forbrugerne. 
1-Butanol in the investigated spray products for textile proofing is therefore in itself not a health hazard to consumers.
Hovedanvendelserne er som opløsningsmiddel for lakker og harpikser, som maling- og fernisfjerner, til ekstraktion af "essentielle olier", i den analytiske kemi til bestemmelse af molvægt, ved fremstilling af adipinsyre, benzen, cyclohexanon, cyclohexanol, cyclohexylchlorid, nitrocyclohexan, fast brændstof, til industriel omkrystallisering af steroider og i svampedræbende midler (HSDB, 2007). 
The main application is as solvent for varnishes and resins, as paint and varnish remover, for extraction of "essential oils" in the analytic chemistry for determination of molar weight, for making adipic acid, benzene, cyclohexanon, cyclohexanol, cyclohexyl chloride, nitrocyclohexane, solid fuel, for industrial re-crystallization of steroids and in fungicides (HDSB, 2007).
Cyclohexan er en farveløs, letflydende væske med en mild, sødlig petroleums eller chloroform lignende lugt. 
Cyclohexane is a colourless, easily flowing liquid with a mild, sweet petroleum or chloroform-like odour.
Den er meget letantændelig. 
It is very flammable.
Cyclohexan er praktisk taget uopløseligt i vand, men er opløseligt i ethanol, ether og acetone og er blandbar med olivenolie (HSDB, 2007). 
Cyclohexane is practically insoluble in water but is soluble in ethanol, ether and acetone and is miscible with olive oil (HDSB, 2007).
Lugtgrænsen er ca. 25 ppm i luft. 
The odour limit is approx. 25 ppm in air.
Cyclohexan er på Arbejdstilsynets liste over organiske opløsningsmidler. 
Cyclohexane is included on the list of organic solvents of the Danish Working Environment Authority.
Cyclohexan bliver næsten fuldstændig optaget via mave-tarmkanalen og efter indånding. 
Cyclohexane is almost completely absorbed via the gastrointestinal tract and after inhalation.
Cyclohexan fordeles i kroppen med højeste koncentrationer i fedtvæv. 
Cyclohexane is distributed in the body with highest concentrations in fatty tissue.
Udskillelsen sker fortrinsvis via lungerne, enten i uomdannet form eller som CO2 (ECB, 2004). 
Elimination mainly takes place via the lungs either unchanged or as CO2 (ECB, 2004).
8.4.4.2 Akutte toksiske effekter, mennesker 
8.4.4.2 Acute toxic effects, humans
I et nyligt udført studie blev humane frivillige eksponeret for 25 eller 250 ppm cyclohexan i en 4 timers periode. 
In a recent study, human volunteers were exposed to 25 or 250 ppm cyclohexane for a 4-hour period.
Der blev ikke fundet neuroadfærdseffekter ved nogen af doserne. 
No neuro behaviour effects were found in connection with any of the doses.
De 250 ppm (svarende til 860 mg/m³) anses derfor for at være en NOAEC for neuroadfærdstoksicitet (ECB, 2004). 
The 250 ppm (corresponding to 860 mg/m³) is therefore assessed to be a no observed adverse effect concentration (NOAEC) for neuro behaviour toxicity (ECB, 2004).
Man ser hudirritation efter gentagen dermal eksponering. 
Skin irritation appears after repeated dermal exposure.
Det skyldes, at cyclohexan har affedtende egenskaber. 
That is because cyclohexane has degreasing properties.
Der regnes ikke med hudsensibiliserende egenskaber (ECB, 2004). 
Skin sensitizing properties are not expected (ECB, 2004).
8.4.4.3 Akutte toksiske effekter, dyr 
8.4.4.3 Acute toxic effects, animals
Den laveste letale orale dosis i kanin er 6000 mg/kg; dette studie viste, at toksiciteten involverede centralnervesystemet (narkotisk effekt og kramper). 
The lowest lethal oral dose in rabbits is 6000 mg/kg; the study showed that toxicity involved the CNS (narcotic effect and cramps).
Den dermale LD50 i kaniner er større end 2000 mg/kg, som er den højeste dosis, der er testet (ECB, 2004). 
The dermal LD50 in rabbits is larger than 2000 mg/kg which is the highest dose that has been tested (ECB, 2004).
Eksponering af kaniner for cyclohexandampe i 1 time gav centralnervesystemseffekter (kramper, rysten, hurtig respiration, cyanose og diarré). 
Exposure of rabbits to cyclohexane vapour for 1 hour gave CNS effects (cramps, shaking, quick respiration, cyanosis and diarrhea).
Alle dyr udsat for 26.000 ppm (89.600 mg/m³) døde. 
All animals exposed to 26000 ppm (89600 mg/m³) died.
LC50 for eksponering af rotter i 4 timer oversteg 9500 ppm (32.800 mg/m³), idet ingen dødsfald indtraf (ECB, 2004). 
LC50 for exposure of rats for 4 hours exceeded 9500 ppm (32800 mg/m³) as no death occurred (ECB, 2004).
NOAEC var 2000 ppm (6880 mg/m³) for neurotoksicitet i rotter efter 6 timers helkrops-eksponering (ECB, 2004). 
NOAEC was 2000 ppm (6880 mg/m³) for neuro toxicity in rats after 6 hours of wholebody exposure (ECB, 2004).
En NOAEC på 400 ppm (1400 mg/m³) blev fundet for neurotoksiske effekter i et subakut rottestudie med 8 timers eksponering daglig i 6 dage (ECB, 2004). 
A NOAEC of 400 ppm (1400 mg/m³) was found for neuro toxic effects in a sub-acute rat study with 8 hours of exposure daily for 6 days (ECB, 2004).
Efter gentagen dosering ved inhalation var de systemiske effekter i både mus og rotter i løbet af de 28 og 90 dages studier begrænset til effekter på leveren: stigninger i absolutte og relative levervægte, stigning i mitotiske indeks tal og centrolobulær hypertrofi. 
After repeated dosage on inhalation, the systematic effects in both mice and rats in the course of the 28 and 90 day studies were limited to effects on the liver: increase in absolute and relative liver weight, increase in mitotic index figures and centrolobular hypertrophy.
Dette studie førte til en NOAEC på 2000 ppm (6880 mg/m³) (ECB, 2004). 
The study lead to a no observed adverse effect concentration (NOAEC) of 2000 ppm (6880 mg/m³) (ECB, 2004).
Et ældre studie gav ganske vist en NOAEC på 425 ppm, men studiet er for mangelfuldt til at denne værdi kan bruges ved risikovurderinger (ECB, 2004). 
It is true that an older study showed a NOAEC of 425 ppm, but the study is very insufficient and therefore this value cannot be used for health assessments (ECB, 2004).
Der findes ingen studier af subkroniske effekter af oral eksponering. 
No studies of subchronic effects from oral exposure exist.
Der findes et gammelt studie i kaniner af subkroniske effekter af dermal eksponering, men det var ikke muligt at udlede en NOAEL værdi ud fra dette (ECB, 2004). 
An old study exists for rabbits of subcronic effects from dermal exposure but it was not possible to derive a NOAEL value (ECB, 2004).
I et 2-generationers rottestudie (inhalation) blev der ikke fundet effekter på fertiliteten og kun små formindskelser i vægten på de nyfødte unger ved 7000 ppm, hvor man også så toksicitet i moderdyrene. 
In a 2-generation rat study (inhalation) no effects were found on fertility and only small weight reductions were found in the newly born offspring at 7000 ppm and toxicity in the dam also appeared.
I studiet var der NOAEC på 500 ppm (1720 mg/m³) for systemisk toksicitet (sedation) og på 2000 ppm (6880 mg/m³) for reproduktion. 
In the study, there was NOAEC of 500 ppm (1720 mg/m³) for systematic toxicity (sedation) and of 2000 ppm (6880 mg/m³) for reproduction.
Der er lavet 2 inhalationsstudier for toksicitet på udviklingen (teratogenicitetsstudier) - et i rotter og et i kaniner. 
2 inhalation studies were carried out for toxicity on the development (teratogenecity studies) - one in rats and one in rabbits.
Der blev benyttet koncentrationer op til 7000 ppm, 6 timer per dag på drægtighedsdagene 7-16 (i rotter) eller 7-19 (i kaniner). 
Concentrations of up to 7000 ppm, 6 hours a day on gestation day 7-16 (in rats) or on gestation day 7-19 (in rabbits) were used.
I rotter så man systemisk toksicitet i form af reduceret antal implantationer og nedsat legemsvægt og foderindtagelse ved 2000 og 7000 ppm hos moderdyrene, men ingen effekter på udviklingen af fostrene. 
In rats, there was systematic toxicity in the form of reduced number of implantations and the dam showed reduced body weight and feed consumption at 2000 and 7000 ppm, but no effects were seen in the development of the foetuses.
I kaniner så man hverken toksicitet i mødrene eller fostrene. 
In rabbits, no toxicity was seen in the dam or in the foetuses.
Der er derfor en NOAEC på 500 ppm (1,720 mg/m³) for systemiske effekter i rotter, mens der med hensyn til udvikling af fostrene er en NOAEC på 7000 ppm (24,080 mg/m³). 
Therefore, there is a NOAEC of 500 ppm (1.720 mg/m³) for systematic effects in rats, but with regard to the development of the foetuses there is a NOAEC of 7000 ppm (24.080 mg/m³).
I kaninstudiet er begge NOAEC-værdier 7000 ppm (24,080 mg/m³) (ECB, 2004). 
In the rabbit study, both NOAEC values are 7000 ppm (24.080 mg/m³) (ECB, 2004).
Cyclohexan er hverken fundet genotoksisk i korttids in vitro eller in vivo studier (ECB, 2004). 
Cyclohexane neither appeared genotoxic in short-term in vitro nor in vivo studies (ECB, 2004).
I et tvivlsomt studie blev det vist, at cyclohexan kunne have et svagt cancer promotor potentiale (ECB, 2004). 
In a doubtful study it appeared that cyclohexane might have a weak cancer promoter potential (ECB, 2004).
Der findes imidlertid intet konventionelt 2-års carcinogen-studie, men EU anser det for usandsynligt, at stoffet skulle være kræftfremkaldende. 
However, no conventional 2-year carcinogenic study exists, but the EU believes it is unlikely that the substance should be carcinogenic.
IARC har ikke vurderet cyclohexan med hensyn til kræftfremkaldende potentiale. 
IARC has not assessed cyclohexane with regard to carcinogenic potential.
Cyclohexan optages let via mavetarm-kanalen og ved indånding, og absorberes også i nogen grad via huden. 
Cyclohexane is absorbed easily via the gastrointestinal tract and on inhalation and it to some degree it is also absorbed via the skin.
Der er lav akut giftighed fra alle eksponeringsveje. 
There is low acute toxicity from all routes of exposure.
Effekterne såvel akut som efter gentagne doseringer er mest virkninger fra centralnervesystemet. 
The acute effects as well as the effects after repeated dosage are mainly effects from the CNS.
Desuden ser man virkninger på leveren i form af øget vægt og vækst af centrale celler i leveren i studier i gnavere af subkroniske effekter. 
In addition, liver effects appear as increased weight and growth of central cells in the liver in subchronic studies in rodents.
Cyclohexan har ingen toksiske virkninger på reproduktionen. 
Cyclohexane has no toxic effects on reproduction.
Det kritiske studie er et akut humant studie med 4 timers eksponering for 250 ppm svarende til 860 mg/m³ for neuroadfærdspåvirkning. 
The critical study is an acute human study with 4-hour exposure to 250 ppm corresponding to 860 mg/m³ for neuro behaviour effect.
Man så ingen effekter ved denne koncentration. 
No effects were seen with this concentration.
Den kritiske effekt er generel toksicitet på moderdyr i rotte teratogenforsøget. 
The critical effect is general toxicity in the dam in the rat teratogenic test.
Effekter ser man ved 500 ppm. 
Effects are seen at 500 ppm.
Cyclohexan er ikke mutagent, og selvom der ikke findes noget regulært carcinogenstudie anses det for usandsynligt at cyclohexan skulle have et kræftfremkaldende potentiale. 
Cyclohexane is not mutagenic and even though no regular carcinogenic study exists it is assessed to be unlikely that cyclohexane should have carcinogenic potential.